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You searched for subject:(BEZ235). Showing records 1 – 7 of 7 total matches.

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1. Fourneaux, Benjamin. Ciblage de la voie PI3K/mTOR dans les léiomyosarcomes : sensibilité et mécanismes de résistance : Targeting PI3K/mTOR pathway in leiomyosarcomas : sensitivity and mechanisms of resistance.

Degree: Docteur es, Biologie Cellulaire et Physiopathologie, 2017, Bordeaux

Les léiomyosarcomes (LMS) sont des tumeurs d’origine mésenchymateuse caractérisées par une différenciation musculaire lisse. La voie de signalisation PI3K/mTOR (qui contrôle la prolifération et la… (more)

Subjects/Keywords: Léiomyosarcome; Voie PI3K/mTOR; BEZ235; Voie RAS/MAPK; Résistance secondaire; Leiomyosarcoma; PI3K/mTOR pathway; BEZ235; RAS/MAPK pathway; Secondary resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fourneaux, B. (2017). Ciblage de la voie PI3K/mTOR dans les léiomyosarcomes : sensibilité et mécanismes de résistance : Targeting PI3K/mTOR pathway in leiomyosarcomas : sensitivity and mechanisms of resistance. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2017BORD0742

Chicago Manual of Style (16th Edition):

Fourneaux, Benjamin. “Ciblage de la voie PI3K/mTOR dans les léiomyosarcomes : sensibilité et mécanismes de résistance : Targeting PI3K/mTOR pathway in leiomyosarcomas : sensitivity and mechanisms of resistance.” 2017. Doctoral Dissertation, Bordeaux. Accessed April 10, 2021. http://www.theses.fr/2017BORD0742.

MLA Handbook (7th Edition):

Fourneaux, Benjamin. “Ciblage de la voie PI3K/mTOR dans les léiomyosarcomes : sensibilité et mécanismes de résistance : Targeting PI3K/mTOR pathway in leiomyosarcomas : sensitivity and mechanisms of resistance.” 2017. Web. 10 Apr 2021.

Vancouver:

Fourneaux B. Ciblage de la voie PI3K/mTOR dans les léiomyosarcomes : sensibilité et mécanismes de résistance : Targeting PI3K/mTOR pathway in leiomyosarcomas : sensitivity and mechanisms of resistance. [Internet] [Doctoral dissertation]. Bordeaux; 2017. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2017BORD0742.

Council of Science Editors:

Fourneaux B. Ciblage de la voie PI3K/mTOR dans les léiomyosarcomes : sensibilité et mécanismes de résistance : Targeting PI3K/mTOR pathway in leiomyosarcomas : sensitivity and mechanisms of resistance. [Doctoral Dissertation]. Bordeaux; 2017. Available from: http://www.theses.fr/2017BORD0742


University of Melbourne

2. SHORTT, JAKE. Targeting the phosphatidylinositol-3-kinase and mammalian target of rapamycin signalling pathway in MYC-driven models of B-cell neoplasia.

Degree: 2011, University of Melbourne

 The phosphatidylinositol-3-kinase (PI3K) / mammalian target of rapamycin (mTOR) pathway links afferent signals from the microenvironment to the cellular machinery of growth, proliferation and survival.… (more)

Subjects/Keywords: MYC; lymphoma; myeloma; mTOR; PI3K; BEZ235; rapamycin; methyl-pyrrolidone; IMiDs

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SHORTT, J. (2011). Targeting the phosphatidylinositol-3-kinase and mammalian target of rapamycin signalling pathway in MYC-driven models of B-cell neoplasia. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36787

Chicago Manual of Style (16th Edition):

SHORTT, JAKE. “Targeting the phosphatidylinositol-3-kinase and mammalian target of rapamycin signalling pathway in MYC-driven models of B-cell neoplasia.” 2011. Doctoral Dissertation, University of Melbourne. Accessed April 10, 2021. http://hdl.handle.net/11343/36787.

MLA Handbook (7th Edition):

SHORTT, JAKE. “Targeting the phosphatidylinositol-3-kinase and mammalian target of rapamycin signalling pathway in MYC-driven models of B-cell neoplasia.” 2011. Web. 10 Apr 2021.

Vancouver:

SHORTT J. Targeting the phosphatidylinositol-3-kinase and mammalian target of rapamycin signalling pathway in MYC-driven models of B-cell neoplasia. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11343/36787.

Council of Science Editors:

SHORTT J. Targeting the phosphatidylinositol-3-kinase and mammalian target of rapamycin signalling pathway in MYC-driven models of B-cell neoplasia. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/36787


University of Melbourne

3. AZAD, ARUN. Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells.

Degree: 2012, University of Melbourne

 Ionizing radiation is a widely used anti-cancer modality. Unfortunately however, relapse rates are high following radiation treatment indicating an urgent need for novel radiosensitizing strategies.… (more)

Subjects/Keywords: DNA-PK; DNA-PKCs; senescence; radiation; BEZ235; DNA damage

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

AZAD, A. (2012). Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37954

Chicago Manual of Style (16th Edition):

AZAD, ARUN. “Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells.” 2012. Doctoral Dissertation, University of Melbourne. Accessed April 10, 2021. http://hdl.handle.net/11343/37954.

MLA Handbook (7th Edition):

AZAD, ARUN. “Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells.” 2012. Web. 10 Apr 2021.

Vancouver:

AZAD A. Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11343/37954.

Council of Science Editors:

AZAD A. Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37954


Duke University

4. Herbert, James Taylor. The Role of Autophagy and Translation Initiation Factors in Overcoming Resistance to mTOR Inhibitors in Prostate Cancer.

Degree: 2013, Duke University

  Castration resistant prostate cancer (CRPC) causes significant morbidity and mortality around the world and improving treatment options for patients with CRPC is a major… (more)

Subjects/Keywords: Oncology; Pathology; Cellular biology; 4EBP; autophagy; BEZ235; mTOR; Prostate Cancer; RAD001

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APA (6th Edition):

Herbert, J. T. (2013). The Role of Autophagy and Translation Initiation Factors in Overcoming Resistance to mTOR Inhibitors in Prostate Cancer. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/7097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Herbert, James Taylor. “The Role of Autophagy and Translation Initiation Factors in Overcoming Resistance to mTOR Inhibitors in Prostate Cancer. ” 2013. Thesis, Duke University. Accessed April 10, 2021. http://hdl.handle.net/10161/7097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Herbert, James Taylor. “The Role of Autophagy and Translation Initiation Factors in Overcoming Resistance to mTOR Inhibitors in Prostate Cancer. ” 2013. Web. 10 Apr 2021.

Vancouver:

Herbert JT. The Role of Autophagy and Translation Initiation Factors in Overcoming Resistance to mTOR Inhibitors in Prostate Cancer. [Internet] [Thesis]. Duke University; 2013. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10161/7097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Herbert JT. The Role of Autophagy and Translation Initiation Factors in Overcoming Resistance to mTOR Inhibitors in Prostate Cancer. [Thesis]. Duke University; 2013. Available from: http://hdl.handle.net/10161/7097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

5. Moorthy, Ganesh. Clinical Pharmacokinetics of the Novel Combination of BEZ235, PI3K/mTOR Inhibitor, and Everolimus, mTOR Inhibitor: Phase I Clinical Studies and Non-clinical Mechanistic Assessment.

Degree: PhD, Pharmacy: Pharmaceutical Sciences/Biopharmaceutics, 2015, University of Cincinnati

 Dysregulation of PI3K/Akt/mTOR pathway has been implicated in tumorigenesis and malignancy in numerous solid tumors. Everolimus is a potent allosteric inhibitor of mTORC1, and has… (more)

Subjects/Keywords: Pharmaceuticals; pharmacokinetics; Phase I; PBPK; DDI; BEZ235; Everolimus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moorthy, G. (2015). Clinical Pharmacokinetics of the Novel Combination of BEZ235, PI3K/mTOR Inhibitor, and Everolimus, mTOR Inhibitor: Phase I Clinical Studies and Non-clinical Mechanistic Assessment. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439296033

Chicago Manual of Style (16th Edition):

Moorthy, Ganesh. “Clinical Pharmacokinetics of the Novel Combination of BEZ235, PI3K/mTOR Inhibitor, and Everolimus, mTOR Inhibitor: Phase I Clinical Studies and Non-clinical Mechanistic Assessment.” 2015. Doctoral Dissertation, University of Cincinnati. Accessed April 10, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439296033.

MLA Handbook (7th Edition):

Moorthy, Ganesh. “Clinical Pharmacokinetics of the Novel Combination of BEZ235, PI3K/mTOR Inhibitor, and Everolimus, mTOR Inhibitor: Phase I Clinical Studies and Non-clinical Mechanistic Assessment.” 2015. Web. 10 Apr 2021.

Vancouver:

Moorthy G. Clinical Pharmacokinetics of the Novel Combination of BEZ235, PI3K/mTOR Inhibitor, and Everolimus, mTOR Inhibitor: Phase I Clinical Studies and Non-clinical Mechanistic Assessment. [Internet] [Doctoral dissertation]. University of Cincinnati; 2015. [cited 2021 Apr 10]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439296033.

Council of Science Editors:

Moorthy G. Clinical Pharmacokinetics of the Novel Combination of BEZ235, PI3K/mTOR Inhibitor, and Everolimus, mTOR Inhibitor: Phase I Clinical Studies and Non-clinical Mechanistic Assessment. [Doctoral Dissertation]. University of Cincinnati; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439296033

6. Colaço, Laura Guerreiro. Investigating the role and function of Tribbles 2 (TRIB2) in drug resistance within cancer.

Degree: 2014, RCAAP

Dissertação de Mestrado, Ciências Biomédicas, Departamento de Ciências Biomédicas e Medicina, Universidade do Algarve, 2014

O cancro é uma das maiores causas de morte em… (more)

Subjects/Keywords: Cancro; Melanoma; TRIB2; FOXO3a; p53; BEZ235; Sinalização; Domínio/Área Científica::Engenharia e Tecnologia::Outras Engenharias e Tecnologias

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Colaço, L. G. (2014). Investigating the role and function of Tribbles 2 (TRIB2) in drug resistance within cancer. (Thesis). RCAAP. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8366

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Colaço, Laura Guerreiro. “Investigating the role and function of Tribbles 2 (TRIB2) in drug resistance within cancer.” 2014. Thesis, RCAAP. Accessed April 10, 2021. http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8366.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Colaço, Laura Guerreiro. “Investigating the role and function of Tribbles 2 (TRIB2) in drug resistance within cancer.” 2014. Web. 10 Apr 2021.

Vancouver:

Colaço LG. Investigating the role and function of Tribbles 2 (TRIB2) in drug resistance within cancer. [Internet] [Thesis]. RCAAP; 2014. [cited 2021 Apr 10]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8366.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Colaço LG. Investigating the role and function of Tribbles 2 (TRIB2) in drug resistance within cancer. [Thesis]. RCAAP; 2014. Available from: http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8366

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Santos, Marta Machado Pereira dos. Characterization of TRIB2-mediated resistance to anti-cancer drugs.

Degree: 2016, RCAAP

Dissertação de Mestrado, Oncobiologia: Mecanismos Moleculares do Cancro, Departamento de Ciências Biomédicas e Medicina, Universidade do Algarve, 2016

Cancer results from the accumulation of multiple… (more)

Subjects/Keywords: Cancer; Melanoma; TRIB2; Cancer cell signaling; Drug resistance; Cancro; FOXO3a; PI3K; AKT; BEZ235; Domínio/Área Científica::Ciências Médicas::Outras Ciências Médicas

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Santos, M. M. P. d. (2016). Characterization of TRIB2-mediated resistance to anti-cancer drugs. (Thesis). RCAAP. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, Marta Machado Pereira dos. “Characterization of TRIB2-mediated resistance to anti-cancer drugs.” 2016. Thesis, RCAAP. Accessed April 10, 2021. https://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, Marta Machado Pereira dos. “Characterization of TRIB2-mediated resistance to anti-cancer drugs.” 2016. Web. 10 Apr 2021.

Vancouver:

Santos MMPd. Characterization of TRIB2-mediated resistance to anti-cancer drugs. [Internet] [Thesis]. RCAAP; 2016. [cited 2021 Apr 10]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos MMPd. Characterization of TRIB2-mediated resistance to anti-cancer drugs. [Thesis]. RCAAP; 2016. Available from: https://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/8442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.