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You searched for subject:(BACE1). Showing records 1 – 15 of 15 total matches.

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University of Dundee

1. Findlay, John Alexander. Investigation of BACE1 as a stress-induced regulator of neuronal metabolism.

Degree: PhD, 2014, University of Dundee

 Alzheimer’s disease (AD) is the most common cause of dementia, accounting for around 60-70% of cases. AD encompasses large-scale neuronal loss, resulting in progressive memory… (more)

Subjects/Keywords: 616.8; Alzheimer's disease; Glucose metabolism; BACE1; APP

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APA (6th Edition):

Findlay, J. A. (2014). Investigation of BACE1 as a stress-induced regulator of neuronal metabolism. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/2ba2c887-e38f-4563-b7e8-674b9e857d7d

Chicago Manual of Style (16th Edition):

Findlay, John Alexander. “Investigation of BACE1 as a stress-induced regulator of neuronal metabolism.” 2014. Doctoral Dissertation, University of Dundee. Accessed March 22, 2019. http://hdl.handle.net/10588/2ba2c887-e38f-4563-b7e8-674b9e857d7d.

MLA Handbook (7th Edition):

Findlay, John Alexander. “Investigation of BACE1 as a stress-induced regulator of neuronal metabolism.” 2014. Web. 22 Mar 2019.

Vancouver:

Findlay JA. Investigation of BACE1 as a stress-induced regulator of neuronal metabolism. [Internet] [Doctoral dissertation]. University of Dundee; 2014. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/10588/2ba2c887-e38f-4563-b7e8-674b9e857d7d.

Council of Science Editors:

Findlay JA. Investigation of BACE1 as a stress-induced regulator of neuronal metabolism. [Doctoral Dissertation]. University of Dundee; 2014. Available from: http://hdl.handle.net/10588/2ba2c887-e38f-4563-b7e8-674b9e857d7d


University of Dundee

2. Findlay, John Alexander. Investigation of BACE1 as a stress-induced regulator of neuronal metabolism.

Degree: PhD, 2014, University of Dundee

 Alzheimer’s disease (AD) is the most common cause of dementia, accounting for around 60-70% of cases. AD encompasses large-scale neuronal loss, resulting in progressive memory… (more)

Subjects/Keywords: 616.8; Alzheimer's disease; Glucose metabolism; BACE1; APP

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APA (6th Edition):

Findlay, J. A. (2014). Investigation of BACE1 as a stress-induced regulator of neuronal metabolism. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/2ba2c887-e38f-4563-b7e8-674b9e857d7d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.642893

Chicago Manual of Style (16th Edition):

Findlay, John Alexander. “Investigation of BACE1 as a stress-induced regulator of neuronal metabolism.” 2014. Doctoral Dissertation, University of Dundee. Accessed March 22, 2019. https://discovery.dundee.ac.uk/en/studentTheses/2ba2c887-e38f-4563-b7e8-674b9e857d7d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.642893.

MLA Handbook (7th Edition):

Findlay, John Alexander. “Investigation of BACE1 as a stress-induced regulator of neuronal metabolism.” 2014. Web. 22 Mar 2019.

Vancouver:

Findlay JA. Investigation of BACE1 as a stress-induced regulator of neuronal metabolism. [Internet] [Doctoral dissertation]. University of Dundee; 2014. [cited 2019 Mar 22]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/2ba2c887-e38f-4563-b7e8-674b9e857d7d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.642893.

Council of Science Editors:

Findlay JA. Investigation of BACE1 as a stress-induced regulator of neuronal metabolism. [Doctoral Dissertation]. University of Dundee; 2014. Available from: https://discovery.dundee.ac.uk/en/studentTheses/2ba2c887-e38f-4563-b7e8-674b9e857d7d ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.642893


NSYSU

3. Lee, Chiao-yen. 1,2-Diketone as Versatile Intermediate in Design and Synthesis of GSK3β and BACE1 Inhibitor.

Degree: Master, Chemistry, 2015, NSYSU

 1,2-Diketones is an important synthetic intermediate widely used for the synthesis of heterocycles, and have found wide applications in both medicinal and materials chemistry. In… (more)

Subjects/Keywords: alzheimer's disease; GSK3β inhibitors; anticancer activity; 1,2-diketone; BACE1 inhibitors

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APA (6th Edition):

Lee, C. (2015). 1,2-Diketone as Versatile Intermediate in Design and Synthesis of GSK3β and BACE1 Inhibitor. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0622115-155311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Chiao-yen. “1,2-Diketone as Versatile Intermediate in Design and Synthesis of GSK3β and BACE1 Inhibitor.” 2015. Thesis, NSYSU. Accessed March 22, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0622115-155311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Chiao-yen. “1,2-Diketone as Versatile Intermediate in Design and Synthesis of GSK3β and BACE1 Inhibitor.” 2015. Web. 22 Mar 2019.

Vancouver:

Lee C. 1,2-Diketone as Versatile Intermediate in Design and Synthesis of GSK3β and BACE1 Inhibitor. [Internet] [Thesis]. NSYSU; 2015. [cited 2019 Mar 22]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0622115-155311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee C. 1,2-Diketone as Versatile Intermediate in Design and Synthesis of GSK3β and BACE1 Inhibitor. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0622115-155311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

4. DOWN, RUSSELL. Development of a candidate for PET imaging of BACE1 in Alzheimer's disease.

Degree: 2015, University of Melbourne

 Alzheimer's disease (AD) is the most common form of dementia but there is currently no cure or disease modifying treatment. The pathological processes which lead… (more)

Subjects/Keywords: Alzheimer's disease; dementia; BACE1; PET imaging; diagnosis; radiochemistry; medicinal chemistry

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APA (6th Edition):

DOWN, R. (2015). Development of a candidate for PET imaging of BACE1 in Alzheimer's disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/56418

Chicago Manual of Style (16th Edition):

DOWN, RUSSELL. “Development of a candidate for PET imaging of BACE1 in Alzheimer's disease.” 2015. Doctoral Dissertation, University of Melbourne. Accessed March 22, 2019. http://hdl.handle.net/11343/56418.

MLA Handbook (7th Edition):

DOWN, RUSSELL. “Development of a candidate for PET imaging of BACE1 in Alzheimer's disease.” 2015. Web. 22 Mar 2019.

Vancouver:

DOWN R. Development of a candidate for PET imaging of BACE1 in Alzheimer's disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/11343/56418.

Council of Science Editors:

DOWN R. Development of a candidate for PET imaging of BACE1 in Alzheimer's disease. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/56418


University of Pennsylvania

5. Gannon, Patrick Joseph. Assessing the Contribution Antiretroviral Therapy to Neuronal Damage and Death as a Mediator of Cognitive Decline in HIV-Associated Neurocognitive Disorders.

Degree: 2014, University of Pennsylvania

 The advent of combination antiretroviral therapy (ART) in 1996 revolutionized the treatment of HIV/AIDS and significantly decreased the incidence of HIV-associated neurocognitive disorders (HAND), a… (more)

Subjects/Keywords: Antiretroviral; BACE1; HAND; HIV; Neurodegeneration; UPR; Neuroscience and Neurobiology; Pharmacology

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APA (6th Edition):

Gannon, P. J. (2014). Assessing the Contribution Antiretroviral Therapy to Neuronal Damage and Death as a Mediator of Cognitive Decline in HIV-Associated Neurocognitive Disorders. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1280

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gannon, Patrick Joseph. “Assessing the Contribution Antiretroviral Therapy to Neuronal Damage and Death as a Mediator of Cognitive Decline in HIV-Associated Neurocognitive Disorders.” 2014. Thesis, University of Pennsylvania. Accessed March 22, 2019. https://repository.upenn.edu/edissertations/1280.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gannon, Patrick Joseph. “Assessing the Contribution Antiretroviral Therapy to Neuronal Damage and Death as a Mediator of Cognitive Decline in HIV-Associated Neurocognitive Disorders.” 2014. Web. 22 Mar 2019.

Vancouver:

Gannon PJ. Assessing the Contribution Antiretroviral Therapy to Neuronal Damage and Death as a Mediator of Cognitive Decline in HIV-Associated Neurocognitive Disorders. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2019 Mar 22]. Available from: https://repository.upenn.edu/edissertations/1280.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gannon PJ. Assessing the Contribution Antiretroviral Therapy to Neuronal Damage and Death as a Mediator of Cognitive Decline in HIV-Associated Neurocognitive Disorders. [Thesis]. University of Pennsylvania; 2014. Available from: https://repository.upenn.edu/edissertations/1280

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Linköping University

6. Halvarsson, Camilla. Substitution of disulphide bonds to hydrophobic amino acids in BACE1.

Degree: Chemistry and Biology, 2009, Linköping University

  The study and understanding of Alzheimer’s disease on protein level is fundamentally important in the search for its treatment and there is a demand… (more)

Subjects/Keywords: BACE1; disulphide bonds substitution; hydrophobic amino acids; protein purification; refolding time; Biochemistry; Biokemi

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APA (6th Edition):

Halvarsson, C. (2009). Substitution of disulphide bonds to hydrophobic amino acids in BACE1. (Thesis). Linköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-52180

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Halvarsson, Camilla. “Substitution of disulphide bonds to hydrophobic amino acids in BACE1.” 2009. Thesis, Linköping University. Accessed March 22, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-52180.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Halvarsson, Camilla. “Substitution of disulphide bonds to hydrophobic amino acids in BACE1.” 2009. Web. 22 Mar 2019.

Vancouver:

Halvarsson C. Substitution of disulphide bonds to hydrophobic amino acids in BACE1. [Internet] [Thesis]. Linköping University; 2009. [cited 2019 Mar 22]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-52180.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Halvarsson C. Substitution of disulphide bonds to hydrophobic amino acids in BACE1. [Thesis]. Linköping University; 2009. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-52180

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

7. Stern, Anna Louise. Hiv And Antiretrovirals In The Central Nervous System: Molecular Mechanisms Of Cognitive Impairment.

Degree: 2017, University of Pennsylvania

 HIV-associated neurocognitive disorder (HAND) describes a wide range of cognitive impairments experienced by up to 55% of HIV+ individuals despite viral suppression by combined antiretroviral… (more)

Subjects/Keywords: antiretrovirals; APP; BACE1; excitotoxicity; HIV; HIV-associated neurocognitive disorders; Cell Biology; Neuroscience and Neurobiology; Virology

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APA (6th Edition):

Stern, A. L. (2017). Hiv And Antiretrovirals In The Central Nervous System: Molecular Mechanisms Of Cognitive Impairment. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2594

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stern, Anna Louise. “Hiv And Antiretrovirals In The Central Nervous System: Molecular Mechanisms Of Cognitive Impairment.” 2017. Thesis, University of Pennsylvania. Accessed March 22, 2019. https://repository.upenn.edu/edissertations/2594.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stern, Anna Louise. “Hiv And Antiretrovirals In The Central Nervous System: Molecular Mechanisms Of Cognitive Impairment.” 2017. Web. 22 Mar 2019.

Vancouver:

Stern AL. Hiv And Antiretrovirals In The Central Nervous System: Molecular Mechanisms Of Cognitive Impairment. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2019 Mar 22]. Available from: https://repository.upenn.edu/edissertations/2594.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stern AL. Hiv And Antiretrovirals In The Central Nervous System: Molecular Mechanisms Of Cognitive Impairment. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2594

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. [No author]. Regulation of BACE1 promoter activity by nuclear factor-kappaB\r\n .

Degree: 2005, University of Texas Medical Branch – Galveston

 The brains of Alzheimer’s disease (AD) patients display cerebrovascular and parenchymal deposits of beta-amyloid peptides, which are derived by proteolytic processing of the amyloid precursor… (more)

Subjects/Keywords: nuclear gactor-kappaB; BACE1; Alzheimer's disease

…40 BACE1 DETECTED BY ICC AND WESTERN BLOT ANALYSES… …40 BACE1 PROMOTER SPECIFICALLY BOUND SELECTIVE NF-κB SUBUNIT PROTEINS: EMSA… …42 BACE1 PROMOTER CONSTRUCT EXPRESSION… …45 BACE1 PROMOTER SPECIFICALLY BOUND SELECTIVE NF-κB SUBUNIT PROTEINS: LUCIFERASE ACTIVITY… …70 BACE1 PROMOTER CONSTRUCT EXPRESSION AS ALTERED AFTER ACUTE H2O2 EXPOSURE… 

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APA (6th Edition):

author], [. (2005). Regulation of BACE1 promoter activity by nuclear factor-kappaB\r\n . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Regulation of BACE1 promoter activity by nuclear factor-kappaB\r\n .” 2005. Thesis, University of Texas Medical Branch – Galveston. Accessed March 22, 2019. http://hdl.handle.net/2152.3/164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Regulation of BACE1 promoter activity by nuclear factor-kappaB\r\n .” 2005. Web. 22 Mar 2019.

Vancouver:

author] [. Regulation of BACE1 promoter activity by nuclear factor-kappaB\r\n . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; 2005. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/2152.3/164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Regulation of BACE1 promoter activity by nuclear factor-kappaB\r\n . [Thesis]. University of Texas Medical Branch – Galveston; 2005. Available from: http://hdl.handle.net/2152.3/164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

9. Monceaux, Christopher Jon. Triazole-linked reduced amide isosteres: An approach for the fragment-based drug discovery of anti-Alzheimer's BACE1 inhibitors and NH-assisted Fürst-Plattner opening of cyclohexene oxides.

Degree: PhD, Chemistry, 2010, Virginia Tech

 In the scope of our BACE1 inhibitor project we used an originally designed microtiter plate-based screening to discover 4 triazole-linked reduced amide isosteres that showed… (more)

Subjects/Keywords: Fürst-Plattner (trans diaxial effect); epoxidation; epoxide opening; click-chemistry; diazotransfer; microtiter plate-based screening; reduced amide BACE1 inhibitors

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APA (6th Edition):

Monceaux, C. J. (2010). Triazole-linked reduced amide isosteres: An approach for the fragment-based drug discovery of anti-Alzheimer's BACE1 inhibitors and NH-assisted Fürst-Plattner opening of cyclohexene oxides. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/30221

Chicago Manual of Style (16th Edition):

Monceaux, Christopher Jon. “Triazole-linked reduced amide isosteres: An approach for the fragment-based drug discovery of anti-Alzheimer's BACE1 inhibitors and NH-assisted Fürst-Plattner opening of cyclohexene oxides.” 2010. Doctoral Dissertation, Virginia Tech. Accessed March 22, 2019. http://hdl.handle.net/10919/30221.

MLA Handbook (7th Edition):

Monceaux, Christopher Jon. “Triazole-linked reduced amide isosteres: An approach for the fragment-based drug discovery of anti-Alzheimer's BACE1 inhibitors and NH-assisted Fürst-Plattner opening of cyclohexene oxides.” 2010. Web. 22 Mar 2019.

Vancouver:

Monceaux CJ. Triazole-linked reduced amide isosteres: An approach for the fragment-based drug discovery of anti-Alzheimer's BACE1 inhibitors and NH-assisted Fürst-Plattner opening of cyclohexene oxides. [Internet] [Doctoral dissertation]. Virginia Tech; 2010. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/10919/30221.

Council of Science Editors:

Monceaux CJ. Triazole-linked reduced amide isosteres: An approach for the fragment-based drug discovery of anti-Alzheimer's BACE1 inhibitors and NH-assisted Fürst-Plattner opening of cyclohexene oxides. [Doctoral Dissertation]. Virginia Tech; 2010. Available from: http://hdl.handle.net/10919/30221

10. Li, Qian. GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer's disease-related protein β-secretase (BACE1).

Degree: PhD, 2014, University of Edinburgh

 GPR50, an X-linked orphan G protein-coupled receptor (GPCR), is a risk factor for bipolar disorder (BD) in female subjects. It has been shown that GPR50… (more)

Subjects/Keywords: 616.8; brain diseases; Alzheimer’s disease; GPR50; BACE1;

…3.3.5 lines Exogenous BACE1 is co-immunoprecipitated with GPR50 in mammalian cell… …101 3.3.6 Endogenous BACE1 is co-immunoprecipitated with GPR50 in HEK293 cells… …104 3.4.1 3.4.2 4 The interaction between GPR50 and BACE1… …109 4.1.3 The role of GPR50 in regulating BACE1 activity… …115 4.3.2 Detection of Bace1/BACE1 activity levels in mouse primary cortical neurons and… 

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APA (6th Edition):

Li, Q. (2014). GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer's disease-related protein β-secretase (BACE1). (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/10042

Chicago Manual of Style (16th Edition):

Li, Qian. “GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer's disease-related protein β-secretase (BACE1).” 2014. Doctoral Dissertation, University of Edinburgh. Accessed March 22, 2019. http://hdl.handle.net/1842/10042.

MLA Handbook (7th Edition):

Li, Qian. “GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer's disease-related protein β-secretase (BACE1).” 2014. Web. 22 Mar 2019.

Vancouver:

Li Q. GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer's disease-related protein β-secretase (BACE1). [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/1842/10042.

Council of Science Editors:

Li Q. GPR50, a potential factor involved in psychiatric disorders interacts with Alzheimer's disease-related protein β-secretase (BACE1). [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/10042

11. Holler, Christopher J. THE CELLULAR NUCLEIC ACID BINDING PROTEIN REGULATES THE ALZHEIMER’S DISEASE β-SECRETASE PROTEIN BACE1.

Degree: 2012, University of Kentucky

 Alzheimer’s disease (AD) is the most common neurodegenerative disease affecting the elderly population and is believed to be caused by the overproduction and accumulation of… (more)

Subjects/Keywords: Alzheimer’s Disease; BACE1; CNBP; RNA Binding Protein; Translation; Molecular and Cellular Neuroscience; Nervous System Diseases

…2 Discovery of the Brain β-secretase: BACE1… …4 Synthesis, Maturation, and Trafficking of BACE1… …6 BACE1 Substrates… …8 BACE1 Expression and Regulation in Normal and AD Brain… …47 BACE1 and BACE2 Protein and Activity Increase in Neurodegenerative Disease . 47 BACE1… 

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APA (6th Edition):

Holler, C. J. (2012). THE CELLULAR NUCLEIC ACID BINDING PROTEIN REGULATES THE ALZHEIMER’S DISEASE β-SECRETASE PROTEIN BACE1. (Doctoral Dissertation). University of Kentucky. Retrieved from http://uknowledge.uky.edu/biochem_etds/12

Chicago Manual of Style (16th Edition):

Holler, Christopher J. “THE CELLULAR NUCLEIC ACID BINDING PROTEIN REGULATES THE ALZHEIMER’S DISEASE β-SECRETASE PROTEIN BACE1.” 2012. Doctoral Dissertation, University of Kentucky. Accessed March 22, 2019. http://uknowledge.uky.edu/biochem_etds/12.

MLA Handbook (7th Edition):

Holler, Christopher J. “THE CELLULAR NUCLEIC ACID BINDING PROTEIN REGULATES THE ALZHEIMER’S DISEASE β-SECRETASE PROTEIN BACE1.” 2012. Web. 22 Mar 2019.

Vancouver:

Holler CJ. THE CELLULAR NUCLEIC ACID BINDING PROTEIN REGULATES THE ALZHEIMER’S DISEASE β-SECRETASE PROTEIN BACE1. [Internet] [Doctoral dissertation]. University of Kentucky; 2012. [cited 2019 Mar 22]. Available from: http://uknowledge.uky.edu/biochem_etds/12.

Council of Science Editors:

Holler CJ. THE CELLULAR NUCLEIC ACID BINDING PROTEIN REGULATES THE ALZHEIMER’S DISEASE β-SECRETASE PROTEIN BACE1. [Doctoral Dissertation]. University of Kentucky; 2012. Available from: http://uknowledge.uky.edu/biochem_etds/12


Universitat Pompeu Fabra

12. Guix Ràfols, Francesc Xavier. Study of the pathophysiological role of nitric oxide on the amyloid-induced toxicity attending to the biochemical modifications and cellular damages.

Degree: Departament de Ciències Experimentals i de la Salut, 2009, Universitat Pompeu Fabra

 This thesis demonstrates that amyloid ß-peptide (Aß)-induced peroxynitrite contributes to the switch of the Aβ42/Aβ40 ratio that occurs in Alzheimer's disease (AD). Since Aβ42 is… (more)

Subjects/Keywords: GAP; free radicals; fibrils; familiar AD; DHAP; cerebral cortex; BACE1; APH-1; antioxidants; amyloid-beta peptide; protein; amyloid precursor; alzheimer's disease; advanced glycation end products; acetylcholine; plaques; peroxinitrite; peròxid d'hidrogen; pèptid beta-amiloide; Pen2; òxid nítric sintasa; òxid nítric; oligomers; nitrotirosinasa; nicastrina; neurona; metilglioxal; malaltia d'alzheimer; hipocamp; glioxalase; glicolisis; GAP; helicoidals aparellats; filaments; fibriles; èstres oxidatiu; DHAP; cortex cerebral; cabdells neurofibrilars; BACE1; APH-1; antioxidants; anió superòxid; AD familiar; AD esporàdic; acetilcolina; glycolysis; glyoxalase; hippocampus; hydrogen peroxide; methylglyoxal; neurofibrillary tangles; neuron; nicastrin; nitric oxide; nitric oxide synthase; nitrotyrosination; oligomers; oxidative stress; pair helical filaments; 616.8

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APA (6th Edition):

Guix Ràfols, F. X. (2009). Study of the pathophysiological role of nitric oxide on the amyloid-induced toxicity attending to the biochemical modifications and cellular damages. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/7162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guix Ràfols, Francesc Xavier. “Study of the pathophysiological role of nitric oxide on the amyloid-induced toxicity attending to the biochemical modifications and cellular damages.” 2009. Thesis, Universitat Pompeu Fabra. Accessed March 22, 2019. http://hdl.handle.net/10803/7162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guix Ràfols, Francesc Xavier. “Study of the pathophysiological role of nitric oxide on the amyloid-induced toxicity attending to the biochemical modifications and cellular damages.” 2009. Web. 22 Mar 2019.

Vancouver:

Guix Ràfols FX. Study of the pathophysiological role of nitric oxide on the amyloid-induced toxicity attending to the biochemical modifications and cellular damages. [Internet] [Thesis]. Universitat Pompeu Fabra; 2009. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/10803/7162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guix Ràfols FX. Study of the pathophysiological role of nitric oxide on the amyloid-induced toxicity attending to the biochemical modifications and cellular damages. [Thesis]. Universitat Pompeu Fabra; 2009. Available from: http://hdl.handle.net/10803/7162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Axelsson, Markus. Investigation of the pathophysiology of progression in multiple sclerosis: studies on cerebrospinal fluid biomarkers.

Degree: 2013, University of Gothenburg / Göteborgs Universitet

 Multiple Sclerosis (MS) is considered an autoimmune disease of the central nervous system (CNS). It usually starts with a relapsing remitting (RR) course that eventually… (more)

Subjects/Keywords: multiple scerosos; cerebrospinal fluud; biomarker; disease progression; NFL; GFAP; CXCL13; BACE1; sAPP/Aß; IgG; oligoligoclonal igG bands; IgG index

Page 1 Page 2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Axelsson, M. (2013). Investigation of the pathophysiology of progression in multiple sclerosis: studies on cerebrospinal fluid biomarkers. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/32007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Axelsson, Markus. “Investigation of the pathophysiology of progression in multiple sclerosis: studies on cerebrospinal fluid biomarkers.” 2013. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed March 22, 2019. http://hdl.handle.net/2077/32007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Axelsson, Markus. “Investigation of the pathophysiology of progression in multiple sclerosis: studies on cerebrospinal fluid biomarkers.” 2013. Web. 22 Mar 2019.

Vancouver:

Axelsson M. Investigation of the pathophysiology of progression in multiple sclerosis: studies on cerebrospinal fluid biomarkers. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2013. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/2077/32007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Axelsson M. Investigation of the pathophysiology of progression in multiple sclerosis: studies on cerebrospinal fluid biomarkers. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2013. Available from: http://hdl.handle.net/2077/32007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

14. Pachaiyappan, Boobalan. Structure- and Ligand-Based Modeling of Beta-Secretase 1 (BACE1) Inhibitors.

Degree: 2012, University of Illinois – Chicago

 There exists a broad consensus among the Alzheimer's disease research community that the key to successful treatment lies in the specific inhibition of beta-amyloid converting… (more)

Subjects/Keywords: BACE1; Alzheimer's; SBB; LBDD; Ab40; CADD; RACHEL; Docking; GOLD; Molecular Modeling; CoMFA; QSAR; Tripos; Openeye; insilico log BB; Clark Equation; β-site Amyloid Precursor Protein Cleaving Enzyme 1; Ligand-Based Drug Design; Computer-Assisted Drug Design; Real-Time Automated Combinatorial Heuristic Enhancement of Lead Compounds; Genetic Optimization for Ligand Docking; Comparative Molecular Field Analysis; Quantitative Structure-Activity Relationship

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pachaiyappan, B. (2012). Structure- and Ligand-Based Modeling of Beta-Secretase 1 (BACE1) Inhibitors. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pachaiyappan, Boobalan. “Structure- and Ligand-Based Modeling of Beta-Secretase 1 (BACE1) Inhibitors.” 2012. Thesis, University of Illinois – Chicago. Accessed March 22, 2019. http://hdl.handle.net/10027/9616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pachaiyappan, Boobalan. “Structure- and Ligand-Based Modeling of Beta-Secretase 1 (BACE1) Inhibitors.” 2012. Web. 22 Mar 2019.

Vancouver:

Pachaiyappan B. Structure- and Ligand-Based Modeling of Beta-Secretase 1 (BACE1) Inhibitors. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/10027/9616.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pachaiyappan B. Structure- and Ligand-Based Modeling of Beta-Secretase 1 (BACE1) Inhibitors. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9616

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Long, Justin M. Novel regulation of neuronal genes implicated in Alzheimer disease by microRNA.

Degree: 2013, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Alzheimer disease (AD) results, in part, from the excess accumulation of the amyloid-β peptide (Aβ) as neuritic plaques in the… (more)

Subjects/Keywords: Alzheimer; APP; BACE1; microRNA; post-transcriptional; gene regulation; amyloid; fetal neurons; Alzheimer's disease  – Molecular aspects; Alzheimer's disease  – Research; Nervous system  – Degeneration  – Pathophysiology; Amyloid beta-protein  – Research; Alzheimer's disease  – Pathophysiology; Small interfering RNA  – Therapeutic use; Proteins  – Physiological transport; Cellular signal transduction; Nervous system  – Degeneration; Protein precursors  – Research; Cognitive neuroscience  – Research; Genetic translation  – Regulation; Neuroplasticity  – Research

…104 II. Aim 2: Identify miRNA that endogenously regulate BACE1 expression ............ 117 A… …Global knockdown of miRNA function elevates BACE1 expression .............117 B. Bioinformatic… …analysis of putative miRNA targets in BACE1 3’-UTR ............119 C. Reporter validation of… …putative miRNA targets in BACE1 3’-UTR.................121 D. miR-339-5p negatively regulates… …BACE1 expression ..................................126 III. Aim 3: Investigate the relevance… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Long, J. M. (2013). Novel regulation of neuronal genes implicated in Alzheimer disease by microRNA. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3758

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Long, Justin M. “Novel regulation of neuronal genes implicated in Alzheimer disease by microRNA.” 2013. Thesis, IUPUI. Accessed March 22, 2019. http://hdl.handle.net/1805/3758.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Long, Justin M. “Novel regulation of neuronal genes implicated in Alzheimer disease by microRNA.” 2013. Web. 22 Mar 2019.

Vancouver:

Long JM. Novel regulation of neuronal genes implicated in Alzheimer disease by microRNA. [Internet] [Thesis]. IUPUI; 2013. [cited 2019 Mar 22]. Available from: http://hdl.handle.net/1805/3758.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Long JM. Novel regulation of neuronal genes implicated in Alzheimer disease by microRNA. [Thesis]. IUPUI; 2013. Available from: http://hdl.handle.net/1805/3758

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.