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You searched for subject:(Autosomal dominant polycystic Kidney disease). Showing records 1 – 30 of 27772 total matches.

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Freie Universität Berlin

1. Oberländer, Natalie. Zystennierenentfernungen an der Charité Campus Mitte von 2005–2018.

Degree: 2020, Freie Universität Berlin

Autosomal dominant polycystic kidney disease (ADPKD) is one of the most common genetic disorders, affecting 12.5 million people worldwide. The disease causes the growth of… (more)

Subjects/Keywords: nephrektomy; kidney transplantation; autosomal dominant polycystic kidney disease; ddc:610

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oberländer, N. (2020). Zystennierenentfernungen an der Charité Campus Mitte von 2005–2018. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-26994

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oberländer, Natalie. “Zystennierenentfernungen an der Charité Campus Mitte von 2005–2018.” 2020. Thesis, Freie Universität Berlin. Accessed December 02, 2020. http://dx.doi.org/10.17169/refubium-26994.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oberländer, Natalie. “Zystennierenentfernungen an der Charité Campus Mitte von 2005–2018.” 2020. Web. 02 Dec 2020.

Vancouver:

Oberländer N. Zystennierenentfernungen an der Charité Campus Mitte von 2005–2018. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2020 Dec 02]. Available from: http://dx.doi.org/10.17169/refubium-26994.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oberländer N. Zystennierenentfernungen an der Charité Campus Mitte von 2005–2018. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-26994

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

2. Mallawaarachchi, Amali. Molecular diagnosis in inherited polycystic kidney disease.

Degree: Garvan Institute of Medical Research, 2019, University of New South Wales

Polycystic Kidney Disease (PKD) incorporates a number of genetically but not always phenotypically distinct inherited cystic kidney disorders. Autosomal Dominant Polycystic Kidney Disease (ADPKD) is… (more)

Subjects/Keywords: PKD2; Autosomal dominant polycystic kidney disease; PKD1; Whole genome sequencing

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APA (6th Edition):

Mallawaarachchi, A. (2019). Molecular diagnosis in inherited polycystic kidney disease. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/62980 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59489/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Mallawaarachchi, Amali. “Molecular diagnosis in inherited polycystic kidney disease.” 2019. Doctoral Dissertation, University of New South Wales. Accessed December 02, 2020. http://handle.unsw.edu.au/1959.4/62980 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59489/SOURCE02?view=true.

MLA Handbook (7th Edition):

Mallawaarachchi, Amali. “Molecular diagnosis in inherited polycystic kidney disease.” 2019. Web. 02 Dec 2020.

Vancouver:

Mallawaarachchi A. Molecular diagnosis in inherited polycystic kidney disease. [Internet] [Doctoral dissertation]. University of New South Wales; 2019. [cited 2020 Dec 02]. Available from: http://handle.unsw.edu.au/1959.4/62980 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59489/SOURCE02?view=true.

Council of Science Editors:

Mallawaarachchi A. Molecular diagnosis in inherited polycystic kidney disease. [Doctoral Dissertation]. University of New South Wales; 2019. Available from: http://handle.unsw.edu.au/1959.4/62980 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:59489/SOURCE02?view=true


University of Manitoba

3. Koohgoli, Roxanna Yasmin. Oxylipin profiles in an orthologous mouse model of autosomal dominant polycystic kidney disease.

Degree: Food and Human Nutritional Sciences, 2020, University of Manitoba

Autosomal dominant polycystic kidney disease (ADPKD) is characterized by the growth of renal cysts, which ultimately causes renal failure. Previous studies have suggested that whole… (more)

Subjects/Keywords: Autosomal dominant polycystic kidney disease; Oxylipins; Flaxseed; Sex effects; Healthy vs disease oxylipins

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APA (6th Edition):

Koohgoli, R. Y. (2020). Oxylipin profiles in an orthologous mouse model of autosomal dominant polycystic kidney disease. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34540

Chicago Manual of Style (16th Edition):

Koohgoli, Roxanna Yasmin. “Oxylipin profiles in an orthologous mouse model of autosomal dominant polycystic kidney disease.” 2020. Masters Thesis, University of Manitoba. Accessed December 02, 2020. http://hdl.handle.net/1993/34540.

MLA Handbook (7th Edition):

Koohgoli, Roxanna Yasmin. “Oxylipin profiles in an orthologous mouse model of autosomal dominant polycystic kidney disease.” 2020. Web. 02 Dec 2020.

Vancouver:

Koohgoli RY. Oxylipin profiles in an orthologous mouse model of autosomal dominant polycystic kidney disease. [Internet] [Masters thesis]. University of Manitoba; 2020. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1993/34540.

Council of Science Editors:

Koohgoli RY. Oxylipin profiles in an orthologous mouse model of autosomal dominant polycystic kidney disease. [Masters Thesis]. University of Manitoba; 2020. Available from: http://hdl.handle.net/1993/34540

4. Larissa Collis Vendramini. Avaliação do consumo de cafeína em pacientes com Doença Renal Policística Autossômica Dominante (DRPAD).

Degree: 2010, Universidade Federal de São Paulo

 A cafeína tem sido considerada um fator de risco para o crescimento dos cistos na Doença Renal Policística Autossômica Dominante (DRPAD) devido ao aumento de… (more)

Subjects/Keywords: Doença renal policística; Rins policísticos; Cafeína; Adenosina monofosfato cíclico; Volume Renal; NEFROLOGIA; Autosomal Dominant Polycystic Kidney Disease; Caffeine; Cyclic AMP; Nutrition

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APA (6th Edition):

Vendramini, L. C. (2010). Avaliação do consumo de cafeína em pacientes com Doença Renal Policística Autossômica Dominante (DRPAD). (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vendramini, Larissa Collis. “Avaliação do consumo de cafeína em pacientes com Doença Renal Policística Autossômica Dominante (DRPAD).” 2010. Thesis, Universidade Federal de São Paulo. Accessed December 02, 2020. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vendramini, Larissa Collis. “Avaliação do consumo de cafeína em pacientes com Doença Renal Policística Autossômica Dominante (DRPAD).” 2010. Web. 02 Dec 2020.

Vancouver:

Vendramini LC. Avaliação do consumo de cafeína em pacientes com Doença Renal Policística Autossômica Dominante (DRPAD). [Internet] [Thesis]. Universidade Federal de São Paulo; 2010. [cited 2020 Dec 02]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vendramini LC. Avaliação do consumo de cafeína em pacientes com Doença Renal Policística Autossômica Dominante (DRPAD). [Thesis]. Universidade Federal de São Paulo; 2010. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

5. Raptis, Vasileios. Ο ρόλος της ασύμμετρης διμεθυλαργινίνης (ADMA) στην παθογένεια της νεφρικής ανεπάρκειας σε ασθενείς με αυτοσωματική επικρατούσα πολυκυστική νόσο νεφρών (ΑΕΠΚΝ).

Degree: 2015, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

 ADPKD is the most frequently occurring inherited disorder that causes CKD worldwide, leading to about 5% of patients in the final stage, both in Europe… (more)

Subjects/Keywords: Αυτοσωματική επικρατούσα πολυκυστική νόσος νεφρών; Χρόνια νεφρική νόσος; Ασύμμετρη Διμεθυλαργινίνη; Οξειδωτικό stress; Autosomal dominant polycystic kidney disease; Chronic kidney disease; Asymmetric dimethylarginine; Oxidative stress

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APA (6th Edition):

Raptis, V. (2015). Ο ρόλος της ασύμμετρης διμεθυλαργινίνης (ADMA) στην παθογένεια της νεφρικής ανεπάρκειας σε ασθενείς με αυτοσωματική επικρατούσα πολυκυστική νόσο νεφρών (ΑΕΠΚΝ). (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/35847

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raptis, Vasileios. “Ο ρόλος της ασύμμετρης διμεθυλαργινίνης (ADMA) στην παθογένεια της νεφρικής ανεπάρκειας σε ασθενείς με αυτοσωματική επικρατούσα πολυκυστική νόσο νεφρών (ΑΕΠΚΝ).” 2015. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed December 02, 2020. http://hdl.handle.net/10442/hedi/35847.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raptis, Vasileios. “Ο ρόλος της ασύμμετρης διμεθυλαργινίνης (ADMA) στην παθογένεια της νεφρικής ανεπάρκειας σε ασθενείς με αυτοσωματική επικρατούσα πολυκυστική νόσο νεφρών (ΑΕΠΚΝ).” 2015. Web. 02 Dec 2020.

Vancouver:

Raptis V. Ο ρόλος της ασύμμετρης διμεθυλαργινίνης (ADMA) στην παθογένεια της νεφρικής ανεπάρκειας σε ασθενείς με αυτοσωματική επικρατούσα πολυκυστική νόσο νεφρών (ΑΕΠΚΝ). [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2015. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/10442/hedi/35847.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raptis V. Ο ρόλος της ασύμμετρης διμεθυλαργινίνης (ADMA) στην παθογένεια της νεφρικής ανεπάρκειας σε ασθενείς με αυτοσωματική επικρατούσα πολυκυστική νόσο νεφρών (ΑΕΠΚΝ). [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2015. Available from: http://hdl.handle.net/10442/hedi/35847

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Balbo, Bruno Eduardo Pedroso. Camundongos com deficiência em Pkd1 apresentam  disfunção cardíaca, fenótipo atenuado por knockout de galectina-3.

Degree: PhD, Nefrologia, 2014, University of São Paulo

Anormalidades miocárdicas destacam-se entre as manifestações cardiovasculares da doença renal policística autossômica dominante (DRPAD). Para investigar a patogênese dessas manifestações, analisamos o fenótipo cardíaco em… (more)

Subjects/Keywords: Apoptose; Apoptosis; Autosomal dominant polycystic Kidney disease; Biologia molecular; Camundongos; Cardiomyopathies; Echocardiography; Ecocardiografia; Fibrose; Fibrosis; Galectin 3; Galectina 3; Mice; Miocardiopatias; Molecular biology; Rim policístico autossômico dominante; Ultrasonography; Ultrassonografia

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APA (6th Edition):

Balbo, B. E. P. (2014). Camundongos com deficiência em Pkd1 apresentam  disfunção cardíaca, fenótipo atenuado por knockout de galectina-3. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09122014-123524/ ;

Chicago Manual of Style (16th Edition):

Balbo, Bruno Eduardo Pedroso. “Camundongos com deficiência em Pkd1 apresentam  disfunção cardíaca, fenótipo atenuado por knockout de galectina-3.” 2014. Doctoral Dissertation, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09122014-123524/ ;.

MLA Handbook (7th Edition):

Balbo, Bruno Eduardo Pedroso. “Camundongos com deficiência em Pkd1 apresentam  disfunção cardíaca, fenótipo atenuado por knockout de galectina-3.” 2014. Web. 02 Dec 2020.

Vancouver:

Balbo BEP. Camundongos com deficiência em Pkd1 apresentam  disfunção cardíaca, fenótipo atenuado por knockout de galectina-3. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09122014-123524/ ;.

Council of Science Editors:

Balbo BEP. Camundongos com deficiência em Pkd1 apresentam  disfunção cardíaca, fenótipo atenuado por knockout de galectina-3. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09122014-123524/ ;


University of Otago

7. Bowden, Sarah. Genome-Wide DNA Methylation in Polycystic Kidney Disease .

Degree: University of Otago

Autosomal Dominant Polycystic Kidney Disease (ADPKD) is a heritable renal disease that causes the enlargement of kidneys due to the bilateral development of fluid-filled cysts.… (more)

Subjects/Keywords: Autosomal Dominant Polycystic Kidney Disease; ADPKD; DNA Methylation; miRNA; RRBS

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APA (6th Edition):

Bowden, S. (n.d.). Genome-Wide DNA Methylation in Polycystic Kidney Disease . (Masters Thesis). University of Otago. Retrieved from http://hdl.handle.net/10523/9536

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Bowden, Sarah. “Genome-Wide DNA Methylation in Polycystic Kidney Disease .” Masters Thesis, University of Otago. Accessed December 02, 2020. http://hdl.handle.net/10523/9536.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Bowden, Sarah. “Genome-Wide DNA Methylation in Polycystic Kidney Disease .” Web. 02 Dec 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Bowden S. Genome-Wide DNA Methylation in Polycystic Kidney Disease . [Internet] [Masters thesis]. University of Otago; [cited 2020 Dec 02]. Available from: http://hdl.handle.net/10523/9536.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Bowden S. Genome-Wide DNA Methylation in Polycystic Kidney Disease . [Masters Thesis]. University of Otago; Available from: http://hdl.handle.net/10523/9536

Note: this citation may be lacking information needed for this citation format:
No year of publication.

8. Cornec-Le Gall, Emilie. Polykystose rénale autosomique dominante : de la génétique moléculaire au développement d'outils pronostiques : Autosomal dominant holycystic kidney disease (ADPKD) : from molecular genetics to the development of prognostic tools.

Degree: Docteur es, Biologie-santé, 2015, Brest

La Polykystose Rénale Autosomique Dominante (PKRAD) est une des pathologies héréditaires les plus fréquentes et affecte environ un individu sur 1000. Elle se caractérise par… (more)

Subjects/Keywords: Polykystose rénale autosomique dominante; Génétique moléculaire; PKD1; PKD2; Insuffisance rénale terminale; Dialyse; Transplantation rénale; Facteurs prédictifs; Médecine personnalisée; Autosomal dominant polycystic kidney disease; Molecular genetics; PKD1; PKD2; End-stage renal disease; Dialysis; Kidney transplantation; Predictive factors; Personalized medicine; 572.8; 616.61

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APA (6th Edition):

Cornec-Le Gall, E. (2015). Polykystose rénale autosomique dominante : de la génétique moléculaire au développement d'outils pronostiques : Autosomal dominant holycystic kidney disease (ADPKD) : from molecular genetics to the development of prognostic tools. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2015BRES0030

Chicago Manual of Style (16th Edition):

Cornec-Le Gall, Emilie. “Polykystose rénale autosomique dominante : de la génétique moléculaire au développement d'outils pronostiques : Autosomal dominant holycystic kidney disease (ADPKD) : from molecular genetics to the development of prognostic tools.” 2015. Doctoral Dissertation, Brest. Accessed December 02, 2020. http://www.theses.fr/2015BRES0030.

MLA Handbook (7th Edition):

Cornec-Le Gall, Emilie. “Polykystose rénale autosomique dominante : de la génétique moléculaire au développement d'outils pronostiques : Autosomal dominant holycystic kidney disease (ADPKD) : from molecular genetics to the development of prognostic tools.” 2015. Web. 02 Dec 2020.

Vancouver:

Cornec-Le Gall E. Polykystose rénale autosomique dominante : de la génétique moléculaire au développement d'outils pronostiques : Autosomal dominant holycystic kidney disease (ADPKD) : from molecular genetics to the development of prognostic tools. [Internet] [Doctoral dissertation]. Brest; 2015. [cited 2020 Dec 02]. Available from: http://www.theses.fr/2015BRES0030.

Council of Science Editors:

Cornec-Le Gall E. Polykystose rénale autosomique dominante : de la génétique moléculaire au développement d'outils pronostiques : Autosomal dominant holycystic kidney disease (ADPKD) : from molecular genetics to the development of prognostic tools. [Doctoral Dissertation]. Brest; 2015. Available from: http://www.theses.fr/2015BRES0030

9. Bastos, Ana Paula Almeida. A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos.

Degree: PhD, Nefrologia, 2010, University of São Paulo

A maior parte dos casos de doença renal policística autossômica dominante (DRPAD) é causada por mutações no gene PKD1 (Polycystic Kidney Disease 1). O insulto… (more)

Subjects/Keywords: Autosomal dominant polycystic kidney disease; Cell proliferation; Cyclin-dependent kinase Inhibitor p21; Cystic kidney diseases; Doenças renais císticas; Inibidor de quinase dependente de ciclina p21; Ischemia; Isquemia; Mutação; Mutation; Proliferação de células; Reperfusion injury; Rim policístico autossômico dominante; Traumatismo por reperfusão

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APA (6th Edition):

Bastos, A. P. A. (2010). A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;

Chicago Manual of Style (16th Edition):

Bastos, Ana Paula Almeida. “A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos.” 2010. Doctoral Dissertation, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;.

MLA Handbook (7th Edition):

Bastos, Ana Paula Almeida. “A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos.” 2010. Web. 02 Dec 2020.

Vancouver:

Bastos APA. A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;.

Council of Science Editors:

Bastos APA. A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;

10. Raman, Archana. THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE.

Degree: PhD, Molecular & Integrative Physiology, 2017, University of Kansas

Polycystic kidney disease (PKD) is characterized by excessive enlargement of the kidney, due to the hyperplastic growth of renal epithelial cells, giving rise to fluid-filled… (more)

Subjects/Keywords: Physiology; Autosomal dominant Polycystic kidney disease; ECM; Integrin-linked kinase; Integrins; Kidney; Periostin

…xvi Chapter 1. Pathophysiology of autosomal dominant polycystic kidney disease… …1 1.2. Autosomal Dominant Polycystic Kidney Disease (ADPKD)… …vasopressin ADPKD: autosomal dominant polycystic kidney disease ARPKD: autosomal Recessive… …AUTOSOMAL DOMINANT POLYCYSTIC KIDNEY DISEASE 1.1. Polycystic Kidney Disease “Though of simple… …1 1.1. Polycystic Kidney Disease… 

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APA (6th Edition):

Raman, A. (2017). THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26892

Chicago Manual of Style (16th Edition):

Raman, Archana. “THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE.” 2017. Doctoral Dissertation, University of Kansas. Accessed December 02, 2020. http://hdl.handle.net/1808/26892.

MLA Handbook (7th Edition):

Raman, Archana. “THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE.” 2017. Web. 02 Dec 2020.

Vancouver:

Raman A. THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1808/26892.

Council of Science Editors:

Raman A. THE ROLE OF MATRICELLULAR SIGNALING IN POLYCYSTIC KIDNEY DISEASE. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26892


Universitat Pompeu Fabra

11. Trujillano Lidón, Daniel, 1987-. Mendelian disease gene identification and diagnosis using targeted next generation sequencing.

Degree: Departament de Ciències Experimentals i de la Salut, 2013, Universitat Pompeu Fabra

 Next Generation Sequencing (NGS) technologies have emerged as a powerful tool for the discovery of causative mutations and novel Mendelian disease genes, and are rapidly… (more)

Subjects/Keywords: Mendelian disease; Diagnostics; Next generation sequencing; Cystic fibrosis; Hyperphenylalaninemias; Autosomal domynant polycystic kidney disease; Malaltia mendeliana; Diagnòstic; Seqüenciació de nova generació; Hipertensió; Fibrosi quística; Hiperfenilalaninèmies; Malaltia poliquística renal autosòmica dominant; 575

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APA (6th Edition):

Trujillano Lidón, Daniel, 1. (2013). Mendelian disease gene identification and diagnosis using targeted next generation sequencing. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/295582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Trujillano Lidón, Daniel, 1987-. “Mendelian disease gene identification and diagnosis using targeted next generation sequencing.” 2013. Thesis, Universitat Pompeu Fabra. Accessed December 02, 2020. http://hdl.handle.net/10803/295582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Trujillano Lidón, Daniel, 1987-. “Mendelian disease gene identification and diagnosis using targeted next generation sequencing.” 2013. Web. 02 Dec 2020.

Vancouver:

Trujillano Lidón, Daniel 1. Mendelian disease gene identification and diagnosis using targeted next generation sequencing. [Internet] [Thesis]. Universitat Pompeu Fabra; 2013. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/10803/295582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Trujillano Lidón, Daniel 1. Mendelian disease gene identification and diagnosis using targeted next generation sequencing. [Thesis]. Universitat Pompeu Fabra; 2013. Available from: http://hdl.handle.net/10803/295582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Δημητρίου, Κυπρούλα. Μελέτη αυτοσωματικής επικρατούσας μορφής πολυκυστικής νόσου των νεφρών (ΑΕΠΝΝ) τύπου 1 και 2 στην Κύπρο: κλινικά χαρακτηριστικά και επιδημιολογία της νόσου.

Degree: 2004, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Subjects/Keywords: Αυτοσωματική επικρατούσα πολυκυστική νόσος νεφρών; Τύπος 1; Τύπος 2; Autosomal dominant polycystic kidney disease; Type 1; Type 2

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APA (6th Edition):

Δημητρίου, . . (2004). Μελέτη αυτοσωματικής επικρατούσας μορφής πολυκυστικής νόσου των νεφρών (ΑΕΠΝΝ) τύπου 1 και 2 στην Κύπρο: κλινικά χαρακτηριστικά και επιδημιολογία της νόσου. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/15929

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Δημητρίου, Κυπρούλα. “Μελέτη αυτοσωματικής επικρατούσας μορφής πολυκυστικής νόσου των νεφρών (ΑΕΠΝΝ) τύπου 1 και 2 στην Κύπρο: κλινικά χαρακτηριστικά και επιδημιολογία της νόσου.” 2004. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed December 02, 2020. http://hdl.handle.net/10442/hedi/15929.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Δημητρίου, Κυπρούλα. “Μελέτη αυτοσωματικής επικρατούσας μορφής πολυκυστικής νόσου των νεφρών (ΑΕΠΝΝ) τύπου 1 και 2 στην Κύπρο: κλινικά χαρακτηριστικά και επιδημιολογία της νόσου.” 2004. Web. 02 Dec 2020.

Vancouver:

Δημητρίου . Μελέτη αυτοσωματικής επικρατούσας μορφής πολυκυστικής νόσου των νεφρών (ΑΕΠΝΝ) τύπου 1 και 2 στην Κύπρο: κλινικά χαρακτηριστικά και επιδημιολογία της νόσου. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2004. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/10442/hedi/15929.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Δημητρίου . Μελέτη αυτοσωματικής επικρατούσας μορφής πολυκυστικής νόσου των νεφρών (ΑΕΠΝΝ) τύπου 1 και 2 στην Κύπρο: κλινικά χαρακτηριστικά και επιδημιολογία της νόσου. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2004. Available from: http://hdl.handle.net/10442/hedi/15929

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

13. Wallin, Ashley Kay. Renal Arterial Blood Flow Quantification by Breath-held Phase-velocity Encoded MRI.

Degree: MS, Bioengineering, 2004, Georgia Tech

Autosomal dominant polycystic disease (ADPKD) is the most common hereditary renal disease and is characterized by renal cyst growth and enlargement. Hypertension occurs early when… (more)

Subjects/Keywords: Bland-Altman plot; Magnetic resonance imaging; Blood flow; Polyvinyl alcohol; Phase-velocity encoded MRI; Autosomal dominant polycystic kidney disease; PC-MRI accuracy and precision; Renal circulation; Polycystic kidney disease Magnetic resonance imaging; Magnetic resonance imaging

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APA (6th Edition):

Wallin, A. K. (2004). Renal Arterial Blood Flow Quantification by Breath-held Phase-velocity Encoded MRI. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/4982

Chicago Manual of Style (16th Edition):

Wallin, Ashley Kay. “Renal Arterial Blood Flow Quantification by Breath-held Phase-velocity Encoded MRI.” 2004. Masters Thesis, Georgia Tech. Accessed December 02, 2020. http://hdl.handle.net/1853/4982.

MLA Handbook (7th Edition):

Wallin, Ashley Kay. “Renal Arterial Blood Flow Quantification by Breath-held Phase-velocity Encoded MRI.” 2004. Web. 02 Dec 2020.

Vancouver:

Wallin AK. Renal Arterial Blood Flow Quantification by Breath-held Phase-velocity Encoded MRI. [Internet] [Masters thesis]. Georgia Tech; 2004. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1853/4982.

Council of Science Editors:

Wallin AK. Renal Arterial Blood Flow Quantification by Breath-held Phase-velocity Encoded MRI. [Masters Thesis]. Georgia Tech; 2004. Available from: http://hdl.handle.net/1853/4982


University of Otago

14. Lett, Bron. Genetics of an Ovine PKD Model .

Degree: 2011, University of Otago

Autosomal recessive polycystic kidney disease (ARPKD) is an important cause of renal and liver related perinatal and neonatal death. ARPKD is presented as a gross… (more)

Subjects/Keywords: Polycystic Kidney Disease; Ovine Model

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APA (6th Edition):

Lett, B. (2011). Genetics of an Ovine PKD Model . (Masters Thesis). University of Otago. Retrieved from http://hdl.handle.net/10523/2070

Chicago Manual of Style (16th Edition):

Lett, Bron. “Genetics of an Ovine PKD Model .” 2011. Masters Thesis, University of Otago. Accessed December 02, 2020. http://hdl.handle.net/10523/2070.

MLA Handbook (7th Edition):

Lett, Bron. “Genetics of an Ovine PKD Model .” 2011. Web. 02 Dec 2020.

Vancouver:

Lett B. Genetics of an Ovine PKD Model . [Internet] [Masters thesis]. University of Otago; 2011. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/10523/2070.

Council of Science Editors:

Lett B. Genetics of an Ovine PKD Model . [Masters Thesis]. University of Otago; 2011. Available from: http://hdl.handle.net/10523/2070


University of Manitoba

15. Monirujjaman, Md. Effect of dietary oils and protein and cyclooxygenase-2 inhibition on disease progression and oxylipin alterations in cystic kidney disease.

Degree: Food and Human Nutritional Sciences, 2019, University of Manitoba

Autosomal dominant polycystic kidney disease (ADPKD) is the most common inherited kidney disease in humans. In animal models that are non-orthologous to ADPKD, dietary soy… (more)

Subjects/Keywords: Polycystic kidney disease; Oxylipin

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APA (6th Edition):

Monirujjaman, M. (2019). Effect of dietary oils and protein and cyclooxygenase-2 inhibition on disease progression and oxylipin alterations in cystic kidney disease. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Monirujjaman, Md. “Effect of dietary oils and protein and cyclooxygenase-2 inhibition on disease progression and oxylipin alterations in cystic kidney disease.” 2019. Thesis, University of Manitoba. Accessed December 02, 2020. http://hdl.handle.net/1993/34233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Monirujjaman, Md. “Effect of dietary oils and protein and cyclooxygenase-2 inhibition on disease progression and oxylipin alterations in cystic kidney disease.” 2019. Web. 02 Dec 2020.

Vancouver:

Monirujjaman M. Effect of dietary oils and protein and cyclooxygenase-2 inhibition on disease progression and oxylipin alterations in cystic kidney disease. [Internet] [Thesis]. University of Manitoba; 2019. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1993/34233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Monirujjaman M. Effect of dietary oils and protein and cyclooxygenase-2 inhibition on disease progression and oxylipin alterations in cystic kidney disease. [Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cape Town

16. de Villiers, Jacquez Charl. Polycystic Disease of the Kidneys with special reference to its Clinical features, Radiological diagnosis and Genetic Nature.

Degree: Doctoral, Human Biology, 1958, University of Cape Town

 This work on polycystic disease of the kidney commenced while the author was engaged in general practice in Swellendam, in the South Western Cape from… (more)

Subjects/Keywords: kidney; polycystic disease

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APA (6th Edition):

de Villiers, J. C. (1958). Polycystic Disease of the Kidneys with special reference to its Clinical features, Radiological diagnosis and Genetic Nature. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/31923

Chicago Manual of Style (16th Edition):

de Villiers, Jacquez Charl. “Polycystic Disease of the Kidneys with special reference to its Clinical features, Radiological diagnosis and Genetic Nature.” 1958. Doctoral Dissertation, University of Cape Town. Accessed December 02, 2020. http://hdl.handle.net/11427/31923.

MLA Handbook (7th Edition):

de Villiers, Jacquez Charl. “Polycystic Disease of the Kidneys with special reference to its Clinical features, Radiological diagnosis and Genetic Nature.” 1958. Web. 02 Dec 2020.

Vancouver:

de Villiers JC. Polycystic Disease of the Kidneys with special reference to its Clinical features, Radiological diagnosis and Genetic Nature. [Internet] [Doctoral dissertation]. University of Cape Town; 1958. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/11427/31923.

Council of Science Editors:

de Villiers JC. Polycystic Disease of the Kidneys with special reference to its Clinical features, Radiological diagnosis and Genetic Nature. [Doctoral Dissertation]. University of Cape Town; 1958. Available from: http://hdl.handle.net/11427/31923

17. Boddu, Ravindra. Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1.

Degree: 2012, University of Alabama – Birmingham

Mutations in <italic>PKHD1</italic> gene cause autosomal recessive polycystic kidney disease. Our previous studies have shown that human <italic>PKHD1</italic> and its mouse orthologue, <italic>Pkhd1</italic>, undergo an… (more)

Subjects/Keywords: Alternative Splicing<; br>; Disease Models, Animal<; br>; Kidney Tubules, Collecting – metabolism.<; br>; Polycystic Kidney, Autosomal Recessive – genetics.<; br>; Receptors, Cell Surface – genetics.<; br>; Translocation, Genetic

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APA (6th Edition):

Boddu, R. (2012). Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boddu, Ravindra. “Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1.” 2012. Thesis, University of Alabama – Birmingham. Accessed December 02, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boddu, Ravindra. “Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1.” 2012. Web. 02 Dec 2020.

Vancouver:

Boddu R. Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Dec 02]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boddu R. Characterizing The Transcriptional Complexity Of Pkhd1/pkhd1. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

18. Hadimeri, Henrik 1962-. Renal and extrarenal signs of autosomal dominant polycystic kidney disease.

Degree: 2000, University of Gothenburg / Göteborgs Universitet

 The aim of the study was to evaluate the clinical impact of renal and extrarenal manifestations of ADPKD (Autosomal Dominant Polycystic Kidney Disease). For this… (more)

Subjects/Keywords: autosomal dominant polycystic disease; extrarenal manifestations; arterial aneurysms; arterio-venous fistulas; continuous ambulatory peritoneal dialysis; echocardiography

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APA (6th Edition):

Hadimeri, H. 1. (2000). Renal and extrarenal signs of autosomal dominant polycystic kidney disease. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/11338

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hadimeri, Henrik 1962-. “Renal and extrarenal signs of autosomal dominant polycystic kidney disease.” 2000. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed December 02, 2020. http://hdl.handle.net/2077/11338.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hadimeri, Henrik 1962-. “Renal and extrarenal signs of autosomal dominant polycystic kidney disease.” 2000. Web. 02 Dec 2020.

Vancouver:

Hadimeri H1. Renal and extrarenal signs of autosomal dominant polycystic kidney disease. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2000. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/2077/11338.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hadimeri H1. Renal and extrarenal signs of autosomal dominant polycystic kidney disease. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2000. Available from: http://hdl.handle.net/2077/11338

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Sousa, Mauri Félix de. "Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos".

Degree: PhD, Nefrologia, 2005, University of São Paulo

Vários estudos mostram que na doença renal policística autossômica dominante os cistos surgem a partir de um mecanismo de "dois-golpes". A patogênese das manifestações não-císticas,… (more)

Subjects/Keywords: CAMUNDONGOS KNOCKOUT; CAPACIDADE DE CONCENTRAÇÃO RENAL; GLOMERULAR FILTRATION RATE; GLOMÉRULOS RENAIS/anatomia & histologia; KIDNEY CONCENTRATING ABILITY; KIDNEY GLOMERULUS/anatomy & histology; MICE KNOCKOUT; POLYCYSTIC KIDNEY AUTOSOMAL DOMINANT/physiopathology; RIM POLICÍSTICO AUTOSSÔMICO DOMINANTE/fisiopatologia; TAXA DE FILTRAÇÃO GLOMERULAR

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APA (6th Edition):

Sousa, M. F. d. (2005). "Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos". (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-21122005-163447/ ;

Chicago Manual of Style (16th Edition):

Sousa, Mauri Félix de. “"Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos".” 2005. Doctoral Dissertation, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-21122005-163447/ ;.

MLA Handbook (7th Edition):

Sousa, Mauri Félix de. “"Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos".” 2005. Web. 02 Dec 2020.

Vancouver:

Sousa MFd. "Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos". [Internet] [Doctoral dissertation]. University of São Paulo; 2005. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-21122005-163447/ ;.

Council of Science Editors:

Sousa MFd. "Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos". [Doctoral Dissertation]. University of São Paulo; 2005. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-21122005-163447/ ;


Macquarie University

20. Underwood, Conor Francis. Hypothalamic control of blood pressure in polycystic kidney disease.

Degree: 2019, Macquarie University

Thesis by publication.

Bibliography: pages 205-294.

1. Introduction  – 2. Increased excitatory regulation of the hypothalamic paraventricular nucleus circulating vasopressin results in the hypertension observed… (more)

Subjects/Keywords: Renal hypertension; Polycystic kidney disease; blood pressure; polycystic kidney disease; sympathetic nervous system; hypothalamus; vasopressin; baroreflex

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APA (6th Edition):

Underwood, C. F. (2019). Hypothalamic control of blood pressure in polycystic kidney disease. (Doctoral Dissertation). Macquarie University. Retrieved from http://hdl.handle.net/1959.14/1274839

Chicago Manual of Style (16th Edition):

Underwood, Conor Francis. “Hypothalamic control of blood pressure in polycystic kidney disease.” 2019. Doctoral Dissertation, Macquarie University. Accessed December 02, 2020. http://hdl.handle.net/1959.14/1274839.

MLA Handbook (7th Edition):

Underwood, Conor Francis. “Hypothalamic control of blood pressure in polycystic kidney disease.” 2019. Web. 02 Dec 2020.

Vancouver:

Underwood CF. Hypothalamic control of blood pressure in polycystic kidney disease. [Internet] [Doctoral dissertation]. Macquarie University; 2019. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1959.14/1274839.

Council of Science Editors:

Underwood CF. Hypothalamic control of blood pressure in polycystic kidney disease. [Doctoral Dissertation]. Macquarie University; 2019. Available from: http://hdl.handle.net/1959.14/1274839

21. Fonseca, Jonathan Mackowiak da. O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1.

Degree: Mestrado, Fisiopatologia Experimental, 2012, University of São Paulo

O desenvolvimento de hipertensão arterial (HAS) ocorre dez anos mais cedo em pacientes com doença renal policística autossômica dominante (DRPAD) comparados à população geral, estando… (more)

Subjects/Keywords: Autossomal dominant polycystic kidney disease; Capacidade de concentração renal; Cystic kidney diseases; Doenças renais císticas; Hipertensão; Hypertension; Mutação; Mutation; Nitric oxide; Óxido nítrico; Renal concentration deficit; Renin-angiotensin system; Rim policístico autossômico dominante; Sistema renina-angiotensina

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APA (6th Edition):

Fonseca, J. M. d. (2012). O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;

Chicago Manual of Style (16th Edition):

Fonseca, Jonathan Mackowiak da. “O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1.” 2012. Masters Thesis, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;.

MLA Handbook (7th Edition):

Fonseca, Jonathan Mackowiak da. “O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1.” 2012. Web. 02 Dec 2020.

Vancouver:

Fonseca JMd. O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;.

Council of Science Editors:

Fonseca JMd. O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;

22. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 02, 2020. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 02 Dec 2020.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Dec 02]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610


University of Alberta

23. Lara, Carlos J. The Herp and HRD1-dependent degradation of TRPP2.

Degree: MS, Department of Physiology, 2012, University of Alberta

Autosomal Dominant Polycystic Kidney Disease (ADPKD) is a prevalent genetic disorder where multiple fluid-filled cysts destroy kidney architecture, eventually requiring hemodialysis or kidney transplant. Approximately… (more)

Subjects/Keywords: calcium signaling; endoplasmic reticulum stress; polycystic kidney disease; Herp; HRD1; TRPP2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lara, C. J. (2012). The Herp and HRD1-dependent degradation of TRPP2. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/p8418n98g

Chicago Manual of Style (16th Edition):

Lara, Carlos J. “The Herp and HRD1-dependent degradation of TRPP2.” 2012. Masters Thesis, University of Alberta. Accessed December 02, 2020. https://era.library.ualberta.ca/files/p8418n98g.

MLA Handbook (7th Edition):

Lara, Carlos J. “The Herp and HRD1-dependent degradation of TRPP2.” 2012. Web. 02 Dec 2020.

Vancouver:

Lara CJ. The Herp and HRD1-dependent degradation of TRPP2. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2020 Dec 02]. Available from: https://era.library.ualberta.ca/files/p8418n98g.

Council of Science Editors:

Lara CJ. The Herp and HRD1-dependent degradation of TRPP2. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/p8418n98g


Hong Kong University of Science and Technology

24. Li, Ching Ki. Characterization of a TRPP complex and its interacting components acting in sex pheromone perception of male C. elegans.

Degree: 2013, Hong Kong University of Science and Technology

 In nature, chemical communication between animals is important for species survival. Finding a mating partner in dioecious species is one of these survival strategies. Caenorhabditis… (more)

Subjects/Keywords: TRP channels ; Caenorhabditis elegans  – Genetics ; Polycystic kidney disease

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APA (6th Edition):

Li, C. K. (2013). Characterization of a TRPP complex and its interacting components acting in sex pheromone perception of male C. elegans. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-7857 ; https://doi.org/10.14711/thesis-b1213356 ; http://repository.ust.hk/ir/bitstream/1783.1-7857/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Ching Ki. “Characterization of a TRPP complex and its interacting components acting in sex pheromone perception of male C. elegans.” 2013. Thesis, Hong Kong University of Science and Technology. Accessed December 02, 2020. http://repository.ust.hk/ir/Record/1783.1-7857 ; https://doi.org/10.14711/thesis-b1213356 ; http://repository.ust.hk/ir/bitstream/1783.1-7857/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Ching Ki. “Characterization of a TRPP complex and its interacting components acting in sex pheromone perception of male C. elegans.” 2013. Web. 02 Dec 2020.

Vancouver:

Li CK. Characterization of a TRPP complex and its interacting components acting in sex pheromone perception of male C. elegans. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2013. [cited 2020 Dec 02]. Available from: http://repository.ust.hk/ir/Record/1783.1-7857 ; https://doi.org/10.14711/thesis-b1213356 ; http://repository.ust.hk/ir/bitstream/1783.1-7857/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li CK. Characterization of a TRPP complex and its interacting components acting in sex pheromone perception of male C. elegans. [Thesis]. Hong Kong University of Science and Technology; 2013. Available from: http://repository.ust.hk/ir/Record/1783.1-7857 ; https://doi.org/10.14711/thesis-b1213356 ; http://repository.ust.hk/ir/bitstream/1783.1-7857/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

25. McGreal, Kerri McGreal. TRANSMEMBRANE PROTEIN 2: A NOVEL PROTEIN IN POLYCYSTIC KIDNEY DISEASE.

Degree: MS, Clinical Research, 2016, University of Kansas

 ABSTRACT Background: Autosomal Dominant Polycystic Kidney Disease (ADPKD) is a common hereditary cause of end stage renal disease. Affected individuals have mutations in PKD1 or… (more)

Subjects/Keywords: Medicine; Biochemistry; Polycystic Kidney Disease; Polycystin-1; Transmembrane Protein 2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McGreal, K. M. (2016). TRANSMEMBRANE PROTEIN 2: A NOVEL PROTEIN IN POLYCYSTIC KIDNEY DISEASE. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/22495

Chicago Manual of Style (16th Edition):

McGreal, Kerri McGreal. “TRANSMEMBRANE PROTEIN 2: A NOVEL PROTEIN IN POLYCYSTIC KIDNEY DISEASE.” 2016. Masters Thesis, University of Kansas. Accessed December 02, 2020. http://hdl.handle.net/1808/22495.

MLA Handbook (7th Edition):

McGreal, Kerri McGreal. “TRANSMEMBRANE PROTEIN 2: A NOVEL PROTEIN IN POLYCYSTIC KIDNEY DISEASE.” 2016. Web. 02 Dec 2020.

Vancouver:

McGreal KM. TRANSMEMBRANE PROTEIN 2: A NOVEL PROTEIN IN POLYCYSTIC KIDNEY DISEASE. [Internet] [Masters thesis]. University of Kansas; 2016. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1808/22495.

Council of Science Editors:

McGreal KM. TRANSMEMBRANE PROTEIN 2: A NOVEL PROTEIN IN POLYCYSTIC KIDNEY DISEASE. [Masters Thesis]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/22495


University of Toledo Health Science Campus

26. Hossain Saad, Md Zubayer. Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease.

Degree: MSP, Pharmaceutical Sciences (Pharmacology/Toxicology), 2016, University of Toledo Health Science Campus

Autosomal Dominant Polycystic Kidney Disease (ADPKD) is the most common life-threatening hereditary genetic disease. Early cardiovascular complications, including hypertension, have been witnessed in the majority… (more)

Subjects/Keywords: Pharmacology; Cellular Biology; Molecular Biology; cilia, Polycystic kidney disease, hypertension, endothelium

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hossain Saad, M. Z. (2016). Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903

Chicago Manual of Style (16th Edition):

Hossain Saad, Md Zubayer. “Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease.” 2016. Masters Thesis, University of Toledo Health Science Campus. Accessed December 02, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903.

MLA Handbook (7th Edition):

Hossain Saad, Md Zubayer. “Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease.” 2016. Web. 02 Dec 2020.

Vancouver:

Hossain Saad MZ. Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2016. [cited 2020 Dec 02]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903.

Council of Science Editors:

Hossain Saad MZ. Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease. [Masters Thesis]. University of Toledo Health Science Campus; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903


Wright State University

27. Obidan, Amnah Mahmoud. Urinary Metabolomics to Detect Polycystic Kidney Disease at Early Stage.

Degree: MS, Biochemistry and Molecular Biology, 2017, Wright State University

 Urinary metabolomics abilities in detection of biological effects (i.e., toxicity, disease) is compounded by high background variability. Previously, we showed mild kidney dysfunction was detectable… (more)

Subjects/Keywords: Biochemistry; 1H NMR; Metabolomics; Polycystic Kidney Disease; Furosemide

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Obidan, A. M. (2017). Urinary Metabolomics to Detect Polycystic Kidney Disease at Early Stage. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1514093416800549

Chicago Manual of Style (16th Edition):

Obidan, Amnah Mahmoud. “Urinary Metabolomics to Detect Polycystic Kidney Disease at Early Stage.” 2017. Masters Thesis, Wright State University. Accessed December 02, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1514093416800549.

MLA Handbook (7th Edition):

Obidan, Amnah Mahmoud. “Urinary Metabolomics to Detect Polycystic Kidney Disease at Early Stage.” 2017. Web. 02 Dec 2020.

Vancouver:

Obidan AM. Urinary Metabolomics to Detect Polycystic Kidney Disease at Early Stage. [Internet] [Masters thesis]. Wright State University; 2017. [cited 2020 Dec 02]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1514093416800549.

Council of Science Editors:

Obidan AM. Urinary Metabolomics to Detect Polycystic Kidney Disease at Early Stage. [Masters Thesis]. Wright State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1514093416800549


University of Kansas

28. Paul, Binu M. Regulation of cell proliferation in autosomal dominant polycystic kidney disease.

Degree: PhD, Anatomy & Cell Biology, 2011, University of Kansas

Autosomal dominant polycystic kidney disease (ADPKD) is a life-threatening genetic disorder characterized by the presence of fluid-filled cysts primarily in the kidneys. Mutations in either… (more)

Subjects/Keywords: Genetics; Molecular biology; Developmental biology; Cell proliferation; Cux1; Kidney development; P27; Polycystic kidney disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paul, B. M. (2011). Regulation of cell proliferation in autosomal dominant polycystic kidney disease. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/8384

Chicago Manual of Style (16th Edition):

Paul, Binu M. “Regulation of cell proliferation in autosomal dominant polycystic kidney disease.” 2011. Doctoral Dissertation, University of Kansas. Accessed December 02, 2020. http://hdl.handle.net/1808/8384.

MLA Handbook (7th Edition):

Paul, Binu M. “Regulation of cell proliferation in autosomal dominant polycystic kidney disease.” 2011. Web. 02 Dec 2020.

Vancouver:

Paul BM. Regulation of cell proliferation in autosomal dominant polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1808/8384.

Council of Science Editors:

Paul BM. Regulation of cell proliferation in autosomal dominant polycystic kidney disease. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/8384


Edith Cowan University

29. Schiavello, Tina. Genetic and non-genetic factors involved in modifying the clinical severity of autosomal dominant polycystic kidney disease.

Degree: 2002, Edith Cowan University

Autosomal dominant polycystic kidney disease (ADPKD) is one of the most common Mendelian disorders, affecting approximately 1 in 1000 individuals. The disease is recognised as… (more)

Subjects/Keywords: Polycystic kidney disease; Genetic aspects; Polycystic kidney disease.; Medicine and Health Sciences

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APA (6th Edition):

Schiavello, T. (2002). Genetic and non-genetic factors involved in modifying the clinical severity of autosomal dominant polycystic kidney disease. (Thesis). Edith Cowan University. Retrieved from https://ro.ecu.edu.au/theses/762

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schiavello, Tina. “Genetic and non-genetic factors involved in modifying the clinical severity of autosomal dominant polycystic kidney disease.” 2002. Thesis, Edith Cowan University. Accessed December 02, 2020. https://ro.ecu.edu.au/theses/762.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schiavello, Tina. “Genetic and non-genetic factors involved in modifying the clinical severity of autosomal dominant polycystic kidney disease.” 2002. Web. 02 Dec 2020.

Vancouver:

Schiavello T. Genetic and non-genetic factors involved in modifying the clinical severity of autosomal dominant polycystic kidney disease. [Internet] [Thesis]. Edith Cowan University; 2002. [cited 2020 Dec 02]. Available from: https://ro.ecu.edu.au/theses/762.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schiavello T. Genetic and non-genetic factors involved in modifying the clinical severity of autosomal dominant polycystic kidney disease. [Thesis]. Edith Cowan University; 2002. Available from: https://ro.ecu.edu.au/theses/762

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

30. Yau, Chung-fai, Forrest. DNA microsatellites co-segregation of polycystic kidney diseasegenes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease(ADPKD) families & cell culture models for ADPKD.

Degree: 1999, University of Hong Kong

Subjects/Keywords: Polycystic kidney disease - Molecular diagnosis.; Polycystic kidney disease - Genetic aspects.

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APA (6th Edition):

Yau, Chung-fai, F. (1999). DNA microsatellites co-segregation of polycystic kidney diseasegenes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease(ADPKD) families & cell culture models for ADPKD. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/33365

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yau, Chung-fai, Forrest. “DNA microsatellites co-segregation of polycystic kidney diseasegenes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease(ADPKD) families & cell culture models for ADPKD.” 1999. Thesis, University of Hong Kong. Accessed December 02, 2020. http://hdl.handle.net/10722/33365.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yau, Chung-fai, Forrest. “DNA microsatellites co-segregation of polycystic kidney diseasegenes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease(ADPKD) families & cell culture models for ADPKD.” 1999. Web. 02 Dec 2020.

Vancouver:

Yau, Chung-fai F. DNA microsatellites co-segregation of polycystic kidney diseasegenes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease(ADPKD) families & cell culture models for ADPKD. [Internet] [Thesis]. University of Hong Kong; 1999. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/10722/33365.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yau, Chung-fai F. DNA microsatellites co-segregation of polycystic kidney diseasegenes (PKD1 & PKD2) in autosomal dominant polycystic kidney disease(ADPKD) families & cell culture models for ADPKD. [Thesis]. University of Hong Kong; 1999. Available from: http://hdl.handle.net/10722/33365

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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