Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Astrocytes physiology). Showing records 1 – 30 of 31 total matches.

[1] [2]

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


Michigan State University

1. Ramsell, Katrina Dee. Biochemical mechanisms mediating morphological plasticity of astrolgial cells in culture.

Degree: PhD, Department of Pharmacology and Toxicology, 1997, Michigan State University

Subjects/Keywords: Neurophysiology; Brain – Physiology; Astrocytes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ramsell, K. D. (1997). Biochemical mechanisms mediating morphological plasticity of astrolgial cells in culture. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:27084

Chicago Manual of Style (16th Edition):

Ramsell, Katrina Dee. “Biochemical mechanisms mediating morphological plasticity of astrolgial cells in culture.” 1997. Doctoral Dissertation, Michigan State University. Accessed March 06, 2021. http://etd.lib.msu.edu/islandora/object/etd:27084.

MLA Handbook (7th Edition):

Ramsell, Katrina Dee. “Biochemical mechanisms mediating morphological plasticity of astrolgial cells in culture.” 1997. Web. 06 Mar 2021.

Vancouver:

Ramsell KD. Biochemical mechanisms mediating morphological plasticity of astrolgial cells in culture. [Internet] [Doctoral dissertation]. Michigan State University; 1997. [cited 2021 Mar 06]. Available from: http://etd.lib.msu.edu/islandora/object/etd:27084.

Council of Science Editors:

Ramsell KD. Biochemical mechanisms mediating morphological plasticity of astrolgial cells in culture. [Doctoral Dissertation]. Michigan State University; 1997. Available from: http://etd.lib.msu.edu/islandora/object/etd:27084


University of Hong Kong

2. Ho, Chung-yin, Maggie. The functional role of endothelin-1 in astrocytes by making use of endothelin-1 knockout mice.

Degree: 2000, University of Hong Kong

Subjects/Keywords: Endothelins - Receptors.; Astrocytes.; Mice - Physiology.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ho, Chung-yin, M. (2000). The functional role of endothelin-1 in astrocytes by making use of endothelin-1 knockout mice. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/34005

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ho, Chung-yin, Maggie. “The functional role of endothelin-1 in astrocytes by making use of endothelin-1 knockout mice.” 2000. Thesis, University of Hong Kong. Accessed March 06, 2021. http://hdl.handle.net/10722/34005.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ho, Chung-yin, Maggie. “The functional role of endothelin-1 in astrocytes by making use of endothelin-1 knockout mice.” 2000. Web. 06 Mar 2021.

Vancouver:

Ho, Chung-yin M. The functional role of endothelin-1 in astrocytes by making use of endothelin-1 knockout mice. [Internet] [Thesis]. University of Hong Kong; 2000. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10722/34005.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ho, Chung-yin M. The functional role of endothelin-1 in astrocytes by making use of endothelin-1 knockout mice. [Thesis]. University of Hong Kong; 2000. Available from: http://hdl.handle.net/10722/34005

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

3. Huang, Yangyang, 1978-. Interleukin-1 mediated cell-type specific signaling in hippocampal neurons and astrocytes.

Degree: Biology, 2010, Rutgers University

Subjects/Keywords: Interleukin-1; Neurons – Physiology; Astrocytes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, Yangyang, 1. (2010). Interleukin-1 mediated cell-type specific signaling in hippocampal neurons and astrocytes. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10002600001.ETD.000052961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yangyang, 1978-. “Interleukin-1 mediated cell-type specific signaling in hippocampal neurons and astrocytes.” 2010. Thesis, Rutgers University. Accessed March 06, 2021. http://hdl.rutgers.edu/1782.2/rucore10002600001.ETD.000052961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yangyang, 1978-. “Interleukin-1 mediated cell-type specific signaling in hippocampal neurons and astrocytes.” 2010. Web. 06 Mar 2021.

Vancouver:

Huang, Yangyang 1. Interleukin-1 mediated cell-type specific signaling in hippocampal neurons and astrocytes. [Internet] [Thesis]. Rutgers University; 2010. [cited 2021 Mar 06]. Available from: http://hdl.rutgers.edu/1782.2/rucore10002600001.ETD.000052961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang, Yangyang 1. Interleukin-1 mediated cell-type specific signaling in hippocampal neurons and astrocytes. [Thesis]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.2/rucore10002600001.ETD.000052961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Bellot-Saez, Alba. Astrocytic modulation of neuronal network oscillations.

Degree: 2019, Western Sydney University

 The synchronization of the neuron’s membrane potential results in the emergence of neuronal oscillations at multiple frequencies that serve distinct physiological functions (e.g. facilitation of… (more)

Subjects/Keywords: astrocytes; central nervous system; diseases; neurons; physiology; neural transmission; Thesis (Ph.D.) – Western Sydney University, 2019

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bellot-Saez, A. (2019). Astrocytic modulation of neuronal network oscillations. (Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:54319

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bellot-Saez, Alba. “Astrocytic modulation of neuronal network oscillations.” 2019. Thesis, Western Sydney University. Accessed March 06, 2021. http://hdl.handle.net/1959.7/uws:54319.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bellot-Saez, Alba. “Astrocytic modulation of neuronal network oscillations.” 2019. Web. 06 Mar 2021.

Vancouver:

Bellot-Saez A. Astrocytic modulation of neuronal network oscillations. [Internet] [Thesis]. Western Sydney University; 2019. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1959.7/uws:54319.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bellot-Saez A. Astrocytic modulation of neuronal network oscillations. [Thesis]. Western Sydney University; 2019. Available from: http://hdl.handle.net/1959.7/uws:54319

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Dzubay, Jeffrey A. Determinants of the time course of excitatory synaptic events in the CNS.

Degree: PhD, 1999, Oregon Health Sciences University

Subjects/Keywords: Synaptic Transmission; Receptors, Glutamate; Glutamic Acid  – physiology; Neurons  – physiology; Astrocytes  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dzubay, J. A. (1999). Determinants of the time course of excitatory synaptic events in the CNS. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4H41PN1 ; http://digitalcommons.ohsu.edu/etd/2527

Chicago Manual of Style (16th Edition):

Dzubay, Jeffrey A. “Determinants of the time course of excitatory synaptic events in the CNS.” 1999. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 06, 2021. doi:10.6083/M4H41PN1 ; http://digitalcommons.ohsu.edu/etd/2527.

MLA Handbook (7th Edition):

Dzubay, Jeffrey A. “Determinants of the time course of excitatory synaptic events in the CNS.” 1999. Web. 06 Mar 2021.

Vancouver:

Dzubay JA. Determinants of the time course of excitatory synaptic events in the CNS. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1999. [cited 2021 Mar 06]. Available from: doi:10.6083/M4H41PN1 ; http://digitalcommons.ohsu.edu/etd/2527.

Council of Science Editors:

Dzubay JA. Determinants of the time course of excitatory synaptic events in the CNS. [Doctoral Dissertation]. Oregon Health Sciences University; 1999. Available from: doi:10.6083/M4H41PN1 ; http://digitalcommons.ohsu.edu/etd/2527

6. Ma, Ying Jun. Transforming growth factor alpha (TGFα) of glial origin is a hypothalamic regulator of mammalian sexual development.

Degree: PhD, 1993, Oregon Health Sciences University

Subjects/Keywords: Transforming Growth Factor alpha; Sexual Maturation; Astrocytes  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ma, Y. J. (1993). Transforming growth factor alpha (TGFα) of glial origin is a hypothalamic regulator of mammalian sexual development. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M40G3H8K ; http://digitalcommons.ohsu.edu/etd/1695

Chicago Manual of Style (16th Edition):

Ma, Ying Jun. “Transforming growth factor alpha (TGFα) of glial origin is a hypothalamic regulator of mammalian sexual development.” 1993. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 06, 2021. doi:10.6083/M40G3H8K ; http://digitalcommons.ohsu.edu/etd/1695.

MLA Handbook (7th Edition):

Ma, Ying Jun. “Transforming growth factor alpha (TGFα) of glial origin is a hypothalamic regulator of mammalian sexual development.” 1993. Web. 06 Mar 2021.

Vancouver:

Ma YJ. Transforming growth factor alpha (TGFα) of glial origin is a hypothalamic regulator of mammalian sexual development. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1993. [cited 2021 Mar 06]. Available from: doi:10.6083/M40G3H8K ; http://digitalcommons.ohsu.edu/etd/1695.

Council of Science Editors:

Ma YJ. Transforming growth factor alpha (TGFα) of glial origin is a hypothalamic regulator of mammalian sexual development. [Doctoral Dissertation]. Oregon Health Sciences University; 1993. Available from: doi:10.6083/M40G3H8K ; http://digitalcommons.ohsu.edu/etd/1695


University of Oxford

7. Raimondo, Joseph Valentino. Intracellular chloride and hydrogen ion dynamics in the nervous system.

Degree: PhD, 2012, University of Oxford

 Synaptic transmission in the nervous system involves the activation of receptor proteins that permit rapid transmembrane fluxes of ions. Ionic gradients across the membrane determine… (more)

Subjects/Keywords: 612.8; Biosensors; Biology (medical sciences); Medical Sciences; Physiology and anatomy; epilepsy; neuroscience; chloride; pH; astrocytes; neurons

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raimondo, J. V. (2012). Intracellular chloride and hydrogen ion dynamics in the nervous system. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:0b69b409-46e5-41ac-88c4-f1cfaf08ad24 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588398

Chicago Manual of Style (16th Edition):

Raimondo, Joseph Valentino. “Intracellular chloride and hydrogen ion dynamics in the nervous system.” 2012. Doctoral Dissertation, University of Oxford. Accessed March 06, 2021. http://ora.ox.ac.uk/objects/uuid:0b69b409-46e5-41ac-88c4-f1cfaf08ad24 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588398.

MLA Handbook (7th Edition):

Raimondo, Joseph Valentino. “Intracellular chloride and hydrogen ion dynamics in the nervous system.” 2012. Web. 06 Mar 2021.

Vancouver:

Raimondo JV. Intracellular chloride and hydrogen ion dynamics in the nervous system. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2021 Mar 06]. Available from: http://ora.ox.ac.uk/objects/uuid:0b69b409-46e5-41ac-88c4-f1cfaf08ad24 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588398.

Council of Science Editors:

Raimondo JV. Intracellular chloride and hydrogen ion dynamics in the nervous system. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:0b69b409-46e5-41ac-88c4-f1cfaf08ad24 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588398


Michigan State University

8. Mohr, Margaret Ann. Neuro- and gliogenesis in the pubertal rat brain : implications for female reproduction.

Degree: 2015, Michigan State University

"The goal of the experiments conducted in this dissertation was to test the hypothesis that cells added to the AVPV [anteroventral periventricular nucleus] during puberty… (more)

Subjects/Keywords: Developmental neurobiology; Neuroendocrinology; Rats – Reproduction – Endocrine aspects; Veterinary neurology; Astrocytes; Luteinizing hormone; Rats – Physiology; Neurosciences; Endocrinology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mohr, M. A. (2015). Neuro- and gliogenesis in the pubertal rat brain : implications for female reproduction. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mohr, Margaret Ann. “Neuro- and gliogenesis in the pubertal rat brain : implications for female reproduction.” 2015. Thesis, Michigan State University. Accessed March 06, 2021. http://etd.lib.msu.edu/islandora/object/etd:3480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mohr, Margaret Ann. “Neuro- and gliogenesis in the pubertal rat brain : implications for female reproduction.” 2015. Web. 06 Mar 2021.

Vancouver:

Mohr MA. Neuro- and gliogenesis in the pubertal rat brain : implications for female reproduction. [Internet] [Thesis]. Michigan State University; 2015. [cited 2021 Mar 06]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mohr MA. Neuro- and gliogenesis in the pubertal rat brain : implications for female reproduction. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:3480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Reyes, Reno Cervo. The role of mitochondria and plasma membrane CA²⁺ transport systems in CA²⁺-dependent glutamate release from rat cortical astrocytes.

Degree: PhD, 2009, University of Alabama – Birmingham

Astrocytes, a type of glial cell in the central nervous system, are recognized for their support roles to neurons. They supply neurons with metabolites, maintain… (more)

Subjects/Keywords: Astrocytes  – metabolism<; br>; Astrocytes  – ultrastructure<; br>; Calcium  – metabolism<; br>; Exocytosis  – physiology<; br>; Glutamic Acid  – metabolism<; br>; Mitochondria  – physiology<; br>; Visual Cortex  – cytology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reyes, R. C. (2009). The role of mitochondria and plasma membrane CA²⁺ transport systems in CA²⁺-dependent glutamate release from rat cortical astrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,688

Chicago Manual of Style (16th Edition):

Reyes, Reno Cervo. “The role of mitochondria and plasma membrane CA²⁺ transport systems in CA²⁺-dependent glutamate release from rat cortical astrocytes.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 06, 2021. http://contentdm.mhsl.uab.edu/u?/etd,688.

MLA Handbook (7th Edition):

Reyes, Reno Cervo. “The role of mitochondria and plasma membrane CA²⁺ transport systems in CA²⁺-dependent glutamate release from rat cortical astrocytes.” 2009. Web. 06 Mar 2021.

Vancouver:

Reyes RC. The role of mitochondria and plasma membrane CA²⁺ transport systems in CA²⁺-dependent glutamate release from rat cortical astrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 06]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,688.

Council of Science Editors:

Reyes RC. The role of mitochondria and plasma membrane CA²⁺ transport systems in CA²⁺-dependent glutamate release from rat cortical astrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,688


Loma Linda University

10. Stout, Charles Emmet. Mechanisms of Calcium Wave Initiation and Propagation in Astrocytes.

Degree: PhD, Physiology, 2002, Loma Linda University

  Glial cells display propagated waves of cytoplasmic calcium (Ca2+) mobilization that spread outward from an initiating cell to involve hundreds to thousands of surrounding… (more)

Subjects/Keywords: Cellular and Molecular Physiology; Astrocytes  – physiology; Calcium Channels  – metabolism; Calcium Signaling.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stout, C. E. (2002). Mechanisms of Calcium Wave Initiation and Propagation in Astrocytes. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/925

Chicago Manual of Style (16th Edition):

Stout, Charles Emmet. “Mechanisms of Calcium Wave Initiation and Propagation in Astrocytes.” 2002. Doctoral Dissertation, Loma Linda University. Accessed March 06, 2021. https://scholarsrepository.llu.edu/etd/925.

MLA Handbook (7th Edition):

Stout, Charles Emmet. “Mechanisms of Calcium Wave Initiation and Propagation in Astrocytes.” 2002. Web. 06 Mar 2021.

Vancouver:

Stout CE. Mechanisms of Calcium Wave Initiation and Propagation in Astrocytes. [Internet] [Doctoral dissertation]. Loma Linda University; 2002. [cited 2021 Mar 06]. Available from: https://scholarsrepository.llu.edu/etd/925.

Council of Science Editors:

Stout CE. Mechanisms of Calcium Wave Initiation and Propagation in Astrocytes. [Doctoral Dissertation]. Loma Linda University; 2002. Available from: https://scholarsrepository.llu.edu/etd/925

11. Harrison, John M. (John Martin). Receptor occupancy and time course of excitatory synaptic events in the CNS.

Degree: PhD, 2005, Oregon Health Sciences University

Subjects/Keywords: Amino Acid Transport System X-AG  – physiology; Purkinje Cells  – physiology; Pyramidal Cells  – physiology; Glutamic Acid  – physiology; Astrocytes  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harrison, J. M. (. M. (2005). Receptor occupancy and time course of excitatory synaptic events in the CNS. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4B856DF ; http://digitalcommons.ohsu.edu/etd/2954

Chicago Manual of Style (16th Edition):

Harrison, John M (John Martin). “Receptor occupancy and time course of excitatory synaptic events in the CNS.” 2005. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 06, 2021. doi:10.6083/M4B856DF ; http://digitalcommons.ohsu.edu/etd/2954.

MLA Handbook (7th Edition):

Harrison, John M (John Martin). “Receptor occupancy and time course of excitatory synaptic events in the CNS.” 2005. Web. 06 Mar 2021.

Vancouver:

Harrison JM(M. Receptor occupancy and time course of excitatory synaptic events in the CNS. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2005. [cited 2021 Mar 06]. Available from: doi:10.6083/M4B856DF ; http://digitalcommons.ohsu.edu/etd/2954.

Council of Science Editors:

Harrison JM(M. Receptor occupancy and time course of excitatory synaptic events in the CNS. [Doctoral Dissertation]. Oregon Health Sciences University; 2005. Available from: doi:10.6083/M4B856DF ; http://digitalcommons.ohsu.edu/etd/2954


Texas Medical Center

12. Howe, Matthew D. The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy.

Degree: PhD, 2018, Texas Medical Center

  In healthy brain tissue, toxic amyloid-β (Aβ) proteins are transported by the pulsatile flow of cerebrospinal fluid (CSF) along perivascular drainage pathways. Ischemic stroke… (more)

Subjects/Keywords: Stroke; Fibronectin; Integrin; Amyloid; CAA; Glymphatic; CSF; TGF-beta; Astrocytes; Neurodegeneration; Biology; Cellular and Molecular Physiology; Medicine and Health Sciences; Molecular and Cellular Neuroscience; Pharmacology; Translational Medical Research

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Howe, M. D. (2018). The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875

Chicago Manual of Style (16th Edition):

Howe, Matthew D. “The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy.” 2018. Doctoral Dissertation, Texas Medical Center. Accessed March 06, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875.

MLA Handbook (7th Edition):

Howe, Matthew D. “The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy.” 2018. Web. 06 Mar 2021.

Vancouver:

Howe MD. The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2018. [cited 2021 Mar 06]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875.

Council of Science Editors:

Howe MD. The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy. [Doctoral Dissertation]. Texas Medical Center; 2018. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875


University of Oxford

13. Al Dalahmah, Osama Ahmad Odeh. The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3.

Degree: PhD, 2015, University of Oxford

 Neurogenesis continues postnatally in two major neural stem cell (NSC) niches: The subventricular zone (SVZ) and dentate gurus of the hippocampus. SVZ NSCs self-renew and… (more)

Subjects/Keywords: 612.8; Biology; Physiology and anatomy; Transgenics; Anatomy; Neuroscience; Neurogenetics; Stem cells; Subventricular zone; Neural stem cells; Astrocytes; Oligodendrocytes; Galectin-3; Wnt/B-catenin; cell signalling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Al Dalahmah, O. A. O. (2015). The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:cb9020e1-931f-44cd-8409-7d2c58817d6e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.682698

Chicago Manual of Style (16th Edition):

Al Dalahmah, Osama Ahmad Odeh. “The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3.” 2015. Doctoral Dissertation, University of Oxford. Accessed March 06, 2021. http://ora.ox.ac.uk/objects/uuid:cb9020e1-931f-44cd-8409-7d2c58817d6e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.682698.

MLA Handbook (7th Edition):

Al Dalahmah, Osama Ahmad Odeh. “The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3.” 2015. Web. 06 Mar 2021.

Vancouver:

Al Dalahmah OAO. The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3. [Internet] [Doctoral dissertation]. University of Oxford; 2015. [cited 2021 Mar 06]. Available from: http://ora.ox.ac.uk/objects/uuid:cb9020e1-931f-44cd-8409-7d2c58817d6e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.682698.

Council of Science Editors:

Al Dalahmah OAO. The molecular regulation of neural stem cell lineage progression in the postnatal subventricular zone by Galectin-3. [Doctoral Dissertation]. University of Oxford; 2015. Available from: http://ora.ox.ac.uk/objects/uuid:cb9020e1-931f-44cd-8409-7d2c58817d6e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.682698

14. Salameh, Ahlam Ibrahim. METABOLIC ACIDOSIS AND THE DIVERSE ROLES OF THE Cl/HCO3 EXCHANGER (AE3) IN INTRACELLULAR pH HOMEOSTASIS.

Degree: PhD, Physiology and Biophysics, 2016, Case Western Reserve University School of Graduate Studies

 In this dissertation, I examine the effects of extracellular metabolic acidosis (MAc)—a decrease in extracellular pH (pHo) caused by a decrease in [HCO3]o at fixed… (more)

Subjects/Keywords: Physiology; Nanoscience; Astrocytes; Cancer; Crosstalk; Dendritic Cells; Epilepsy; Hippocampus; Keratinocytes; Macrophages; Melanocytes; Mudullary raphe; Neurons; pH-regulation; Recovery Rate; Skeletal Muscles; Slc4

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salameh, A. I. (2016). METABOLIC ACIDOSIS AND THE DIVERSE ROLES OF THE Cl/HCO3 EXCHANGER (AE3) IN INTRACELLULAR pH HOMEOSTASIS. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1473852052811835

Chicago Manual of Style (16th Edition):

Salameh, Ahlam Ibrahim. “METABOLIC ACIDOSIS AND THE DIVERSE ROLES OF THE Cl/HCO3 EXCHANGER (AE3) IN INTRACELLULAR pH HOMEOSTASIS.” 2016. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed March 06, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1473852052811835.

MLA Handbook (7th Edition):

Salameh, Ahlam Ibrahim. “METABOLIC ACIDOSIS AND THE DIVERSE ROLES OF THE Cl/HCO3 EXCHANGER (AE3) IN INTRACELLULAR pH HOMEOSTASIS.” 2016. Web. 06 Mar 2021.

Vancouver:

Salameh AI. METABOLIC ACIDOSIS AND THE DIVERSE ROLES OF THE Cl/HCO3 EXCHANGER (AE3) IN INTRACELLULAR pH HOMEOSTASIS. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2016. [cited 2021 Mar 06]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1473852052811835.

Council of Science Editors:

Salameh AI. METABOLIC ACIDOSIS AND THE DIVERSE ROLES OF THE Cl/HCO3 EXCHANGER (AE3) IN INTRACELLULAR pH HOMEOSTASIS. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1473852052811835


University of St. Andrews

15. Acton, David. Regulation of mammalian spinal locomotor networks by glial cells .

Degree: 2017, University of St. Andrews

 Networks of interneurons within the spinal cord coordinate the rhythmic activation of muscles during locomotion. These networks are subject to extensive neuromodulation, ensuring appropriate behavioural… (more)

Subjects/Keywords: Spinal cord physiology; Locomotion; Central pattern generator; CPG; Motor control; NMDA receptors; Glia; Glial cells; Astrocytes; Neural networks; Gliotransmission; PAR1; Dopamine; Co-agonist; D-serine; Glycine; GlyT1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Acton, D. (2017). Regulation of mammalian spinal locomotor networks by glial cells . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/10133

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Acton, David. “Regulation of mammalian spinal locomotor networks by glial cells .” 2017. Thesis, University of St. Andrews. Accessed March 06, 2021. http://hdl.handle.net/10023/10133.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Acton, David. “Regulation of mammalian spinal locomotor networks by glial cells .” 2017. Web. 06 Mar 2021.

Vancouver:

Acton D. Regulation of mammalian spinal locomotor networks by glial cells . [Internet] [Thesis]. University of St. Andrews; 2017. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10023/10133.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Acton D. Regulation of mammalian spinal locomotor networks by glial cells . [Thesis]. University of St. Andrews; 2017. Available from: http://hdl.handle.net/10023/10133

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Baker, Brandi J. Oncostatin M-induced gene expression and regulation in astrocytes and microglia.

Degree: PhD, 2009, University of Alabama – Birmingham

Astrocytes and microglia are specialized glial cells of the Central Nervous System (CNS) that modulate neural activity and regulate immunological and inflammatory events. These cells… (more)

Subjects/Keywords: Astrocytes  – physiology<; br>; Central Nervous System  – immunology<; br>; Central Nervous System Diseases  – immunology<; br>; Gene Expression Regulation  – physiology<; br>; Microglia  – physiology<; br>; Oncostatin M  – physiology<; br>; Suppressor of Cytokine Signaling Proteins  – biosynthesis<; br>; Suppressor of Cytokine Signaling Proteins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baker, B. J. (2009). Oncostatin M-induced gene expression and regulation in astrocytes and microglia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,560

Chicago Manual of Style (16th Edition):

Baker, Brandi J. “Oncostatin M-induced gene expression and regulation in astrocytes and microglia.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 06, 2021. http://contentdm.mhsl.uab.edu/u?/etd,560.

MLA Handbook (7th Edition):

Baker, Brandi J. “Oncostatin M-induced gene expression and regulation in astrocytes and microglia.” 2009. Web. 06 Mar 2021.

Vancouver:

Baker BJ. Oncostatin M-induced gene expression and regulation in astrocytes and microglia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 06]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,560.

Council of Science Editors:

Baker BJ. Oncostatin M-induced gene expression and regulation in astrocytes and microglia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,560

17. Yeo, Sujeong. Transcriptional Regulation Of Gfap.

Degree: 2012, University of Alabama – Birmingham

Glial fibrillary acidic protein (GFAP) is an astrocytic intermediate filament protein whose levels are increased in response to CNS injuries. Aim one of my thesis… (more)

Subjects/Keywords: Astrocytes – metabolism<; /br>; Brain Injuries – metabolism.<; /br>; Epigenesis, Genetic.<; /br>; Gene Expression Regulation – physiology<; /br>; Glial Fibrillary Acidic Protein – genetics.<; /br>; Mice, Transgenic<; /br>; Promoter Regions, Genetic – physiology<; /br>; Transcription Factors – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yeo, S. (2012). Transcriptional Regulation Of Gfap. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yeo, Sujeong. “Transcriptional Regulation Of Gfap.” 2012. Thesis, University of Alabama – Birmingham. Accessed March 06, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yeo, Sujeong. “Transcriptional Regulation Of Gfap.” 2012. Web. 06 Mar 2021.

Vancouver:

Yeo S. Transcriptional Regulation Of Gfap. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 06]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeo S. Transcriptional Regulation Of Gfap. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loma Linda University

18. Fukuda, Andrew Minoru. Role of Astrocyte Network in Edema after Juvenile Traumatic Brain Injury.

Degree: PhD, Basic Sciences, 2014, Loma Linda University

  Juvenile traumatic brain injury (jTBI) is the leading cause of death and disability in young children and adolescents. Despite its lasting detrimental effects on… (more)

Subjects/Keywords: Anatomy; Medical Physiology; Medicine and Health Sciences; Musculoskeletal, Neural, and Ocular Physiology; Neurology; Neurosciences; Pediatrics; Brain Injuries - Physiopathology; Brain Edema - Physiopathology; Astrocytes - Metabolism; Aquaporins - Metabolism; Glial Fibrillary Acidic Protein; Juvenile Traumatic Brain Injury; jTBI; Edema; Connexin 43; Reactive Astrogliosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fukuda, A. M. (2014). Role of Astrocyte Network in Edema after Juvenile Traumatic Brain Injury. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/212

Chicago Manual of Style (16th Edition):

Fukuda, Andrew Minoru. “Role of Astrocyte Network in Edema after Juvenile Traumatic Brain Injury.” 2014. Doctoral Dissertation, Loma Linda University. Accessed March 06, 2021. https://scholarsrepository.llu.edu/etd/212.

MLA Handbook (7th Edition):

Fukuda, Andrew Minoru. “Role of Astrocyte Network in Edema after Juvenile Traumatic Brain Injury.” 2014. Web. 06 Mar 2021.

Vancouver:

Fukuda AM. Role of Astrocyte Network in Edema after Juvenile Traumatic Brain Injury. [Internet] [Doctoral dissertation]. Loma Linda University; 2014. [cited 2021 Mar 06]. Available from: https://scholarsrepository.llu.edu/etd/212.

Council of Science Editors:

Fukuda AM. Role of Astrocyte Network in Edema after Juvenile Traumatic Brain Injury. [Doctoral Dissertation]. Loma Linda University; 2014. Available from: https://scholarsrepository.llu.edu/etd/212


University of Missouri – Columbia

19. Hu, Chunhua. Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2.

Degree: 2007, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Reactive oxygen species (ROS) are produced in cells by enzymic and non-enzymic mechanisms and play… (more)

Subjects/Keywords: Astrocytes; NAD (Coenzyme); Apoptosis; Cell membranes; Phospholipase A2  – Inhibitors; Cytosol; Oxidative stress; Active oxygen; Astrocytes  – physiology; NADPH Oxidase  – physiology; Apoptosis  – physiology; Cell Membrane; Central Nervous System  – physiology; NADP  – antagonists & inhibitors; Oxidative Stress  – physiology; Phospholipases A  – metabolism; Reactive Oxygen Species  – metabolism; Vitamin K3

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hu, C. (2007). Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/6270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Chunhua. “Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2.” 2007. Thesis, University of Missouri – Columbia. Accessed March 06, 2021. https://doi.org/10.32469/10355/6270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Chunhua. “Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2.” 2007. Web. 06 Mar 2021.

Vancouver:

Hu C. Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2. [Internet] [Thesis]. University of Missouri – Columbia; 2007. [cited 2021 Mar 06]. Available from: https://doi.org/10.32469/10355/6270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu C. Activation of astrocytes: involvement of NADPH oxidase and cytosolic phospholipase A2. [Thesis]. University of Missouri – Columbia; 2007. Available from: https://doi.org/10.32469/10355/6270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Gottipati, Manoj K. Modulation Of Morpho-Functional Characteristics Of Astrocytes Using Chemically-Functionalized Water-Soluble Single-Walled Carbon Nanotubes.

Degree: 2012, University of Alabama – Birmingham

In this thesis, I report the use of chemically functionalized water-soluble single-walled carbon nanotubes (ws-SWCNTs) for the modulation of morpho-functional characteristics of astrocytes. When added… (more)

Subjects/Keywords: Astrocytes.<; br>; Cells – Growth.<; br>; Cells – Morphology.<; br>; Cell physiology.<; br>; Nanotubes – Therapeutic use.<; br>; Nerve tissue – Wounds and injuries – Treatment.<; br>; Cytoplasmic filaments – Reactivity.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gottipati, M. K. (2012). Modulation Of Morpho-Functional Characteristics Of Astrocytes Using Chemically-Functionalized Water-Soluble Single-Walled Carbon Nanotubes. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1469

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gottipati, Manoj K. “Modulation Of Morpho-Functional Characteristics Of Astrocytes Using Chemically-Functionalized Water-Soluble Single-Walled Carbon Nanotubes.” 2012. Thesis, University of Alabama – Birmingham. Accessed March 06, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1469.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gottipati, Manoj K. “Modulation Of Morpho-Functional Characteristics Of Astrocytes Using Chemically-Functionalized Water-Soluble Single-Walled Carbon Nanotubes.” 2012. Web. 06 Mar 2021.

Vancouver:

Gottipati MK. Modulation Of Morpho-Functional Characteristics Of Astrocytes Using Chemically-Functionalized Water-Soluble Single-Walled Carbon Nanotubes. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 06]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1469.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gottipati MK. Modulation Of Morpho-Functional Characteristics Of Astrocytes Using Chemically-Functionalized Water-Soluble Single-Walled Carbon Nanotubes. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1469

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. McCoy, Eric. Expression and function of aquaporins in malignant and non-malignant astrocytes.

Degree: PhD, 2008, University of Alabama – Birmingham

Aquaporins (AQP) constitute the primary pathway for water movement across cellular membrances. As a result, their expression and function are important for regulating cell volume.… (more)

Subjects/Keywords: Aquaporin 1 Aquaporin 4<; br>; Aquaporins  – metabolism<; br>; Astrocytes  – physiology<; br>; Brain Injuries  – physiopathology<; br>; Brain Neoplasms  – metabolism<; br>; Cell Movement<; br>; Neoplasm Invasiveness<; br>; Wounds, Stab  – physiopathology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McCoy, E. (2008). Expression and function of aquaporins in malignant and non-malignant astrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,774

Chicago Manual of Style (16th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 06, 2021. http://contentdm.mhsl.uab.edu/u?/etd,774.

MLA Handbook (7th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Web. 06 Mar 2021.

Vancouver:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 06]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774.

Council of Science Editors:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774


Virginia Commonwealth University

22. Park, Sung H. High Affinity Block of ICl,swell by Thiol-Reactive Small Molecules.

Degree: PhD, Physiology and Biophysics, 2016, Virginia Commonwealth University

  Ebselen (Ebs) is considered as a glutathione peroxidase (GPx) mimetic and primarily thought to function by scavenging intracellular reactive oxygen species (ROS). Previous to… (more)

Subjects/Keywords: Ebs; high affinity block of; DI TNC1 astrocytes; thiol-reactive small molecules; Ebs-p-yne; VRAC; Biophysics; Cellular and Molecular Physiology; Medicinal Chemistry and Pharmaceutics; Molecular and Cellular Neuroscience; Neuroscience and Neurobiology; Pharmacology, Toxicology and Environmental Health

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Park, S. H. (2016). High Affinity Block of ICl,swell by Thiol-Reactive Small Molecules. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/ACX8-TR69 ; https://scholarscompass.vcu.edu/etd/4433

Chicago Manual of Style (16th Edition):

Park, Sung H. “High Affinity Block of ICl,swell by Thiol-Reactive Small Molecules.” 2016. Doctoral Dissertation, Virginia Commonwealth University. Accessed March 06, 2021. https://doi.org/10.25772/ACX8-TR69 ; https://scholarscompass.vcu.edu/etd/4433.

MLA Handbook (7th Edition):

Park, Sung H. “High Affinity Block of ICl,swell by Thiol-Reactive Small Molecules.” 2016. Web. 06 Mar 2021.

Vancouver:

Park SH. High Affinity Block of ICl,swell by Thiol-Reactive Small Molecules. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2016. [cited 2021 Mar 06]. Available from: https://doi.org/10.25772/ACX8-TR69 ; https://scholarscompass.vcu.edu/etd/4433.

Council of Science Editors:

Park SH. High Affinity Block of ICl,swell by Thiol-Reactive Small Molecules. [Doctoral Dissertation]. Virginia Commonwealth University; 2016. Available from: https://doi.org/10.25772/ACX8-TR69 ; https://scholarscompass.vcu.edu/etd/4433


Texas Medical Center

23. Vincent, Bryce H. The Role of the Androgen Receptor Cofactor p44/WDR77 in Astrocyte Activation.

Degree: PhD, 2011, Texas Medical Center

  Astrogliosis is induced by neuronal damage and is also a pathological feature of the major aging-related neurodegenerative disorders. The mechanisms that control the cascade… (more)

Subjects/Keywords: p44; NF-kappa B; p21; astrocytes; androgen receptor; astrogliosis; Biochemistry; Cell Biology; Cellular and Molecular Physiology; Laboratory and Basic Science Research; Medical Biochemistry; Medical Neurobiology; Medical Sciences; Molecular and Cellular Neuroscience; Molecular Biology; Neurosciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vincent, B. H. (2011). The Role of the Androgen Receptor Cofactor p44/WDR77 in Astrocyte Activation. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/134

Chicago Manual of Style (16th Edition):

Vincent, Bryce H. “The Role of the Androgen Receptor Cofactor p44/WDR77 in Astrocyte Activation.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed March 06, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/134.

MLA Handbook (7th Edition):

Vincent, Bryce H. “The Role of the Androgen Receptor Cofactor p44/WDR77 in Astrocyte Activation.” 2011. Web. 06 Mar 2021.

Vancouver:

Vincent BH. The Role of the Androgen Receptor Cofactor p44/WDR77 in Astrocyte Activation. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2021 Mar 06]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/134.

Council of Science Editors:

Vincent BH. The Role of the Androgen Receptor Cofactor p44/WDR77 in Astrocyte Activation. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/134


University of Florida

24. Boyd, Frederick Tilghman, 1959-. Characterization and physiological significance of brain insulin receptors.

Degree: 1985, University of Florida

Subjects/Keywords: Astrocytes; Brain; Cells; Cultured cells; Insulin; Neuroglia; Neurons; Norepinephrine; Rats; Receptors; Binding Sites ( mesh ); Brain Chemistry ( mesh ); Physiology thesis Ph.D ( mesh ); Receptor, Insulin  – physiology ( mesh )

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Boyd, Frederick Tilghman, 1. (1985). Characterization and physiological significance of brain insulin receptors. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00053670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boyd, Frederick Tilghman, 1959-. “Characterization and physiological significance of brain insulin receptors.” 1985. Thesis, University of Florida. Accessed March 06, 2021. https://ufdc.ufl.edu/AA00053670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boyd, Frederick Tilghman, 1959-. “Characterization and physiological significance of brain insulin receptors.” 1985. Web. 06 Mar 2021.

Vancouver:

Boyd, Frederick Tilghman 1. Characterization and physiological significance of brain insulin receptors. [Internet] [Thesis]. University of Florida; 1985. [cited 2021 Mar 06]. Available from: https://ufdc.ufl.edu/AA00053670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boyd, Frederick Tilghman 1. Characterization and physiological significance of brain insulin receptors. [Thesis]. University of Florida; 1985. Available from: https://ufdc.ufl.edu/AA00053670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

25. Olson, John Ackerman, 1964-. Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain.

Degree: 1990, University of Florida

Subjects/Keywords: Astrocytes; Brain; Cell growth; Cells; Cultural studies; Cultured cells; Neurons; Rats; Receptors; Secretion; Angiotensin II  – physiology ( mesh ); Astrocytes  – growth &; development ( mesh ); Astrocytes  – secretion ( mesh ); Brain Chemistry ( mesh ); Department of Pharmacology and Therapeutics thesis Ph.D ( mesh ); Insulin-Like Growth Factor-Binding Protein 2 ( mesh ); Proteins  – secretion ( mesh ); Rats ( mesh ); Research ( mesh )

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Olson, John Ackerman, 1. (1990). Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00022809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Olson, John Ackerman, 1964-. “Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain.” 1990. Thesis, University of Florida. Accessed March 06, 2021. https://ufdc.ufl.edu/AA00022809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Olson, John Ackerman, 1964-. “Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain.” 1990. Web. 06 Mar 2021.

Vancouver:

Olson, John Ackerman 1. Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain. [Internet] [Thesis]. University of Florida; 1990. [cited 2021 Mar 06]. Available from: https://ufdc.ufl.edu/AA00022809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Olson, John Ackerman 1. Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain. [Thesis]. University of Florida; 1990. Available from: https://ufdc.ufl.edu/AA00022809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Augustine, Claudia. Traumatic Injury in vitro Elicits JNK-mediated Human Astrocyte Retraction, but Spares Cerebrovascular Endothelial Cells.

Degree: 2013, University of Western Ontario

 The pathophysiology of a traumatic brain injury (TBI) involves the dysfunction of the blood-brain barrier (BBB). The lumen of the BBB is lined with cerebrovascular… (more)

Subjects/Keywords: traumatic brain injury; human astrocytes; human cerebrovascular endothelial cells; blood-brain barrier; MAP kinases; cellular retraction; Physiology

…Dehydrogenase GDNF Glial-derived Neurotrophic Factor HA Human Astrocytes HRP Horseradish… …astrocytes, with smaller numbers of pericytes, and pericytic macrophages [3]. These cells… …Perivascular astrocytes project their specialized endfeet to surround the cerebral microvasculature… …perivascular astrocytes. Together, they function to infer a low permeability of the blood vessel and… …Perivascular astrocytes project their endfeet to almost entirely cover the basolateral side of the… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Augustine, C. (2013). Traumatic Injury in vitro Elicits JNK-mediated Human Astrocyte Retraction, but Spares Cerebrovascular Endothelial Cells. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Augustine, Claudia. “Traumatic Injury in vitro Elicits JNK-mediated Human Astrocyte Retraction, but Spares Cerebrovascular Endothelial Cells.” 2013. Thesis, University of Western Ontario. Accessed March 06, 2021. https://ir.lib.uwo.ca/etd/1411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Augustine, Claudia. “Traumatic Injury in vitro Elicits JNK-mediated Human Astrocyte Retraction, but Spares Cerebrovascular Endothelial Cells.” 2013. Web. 06 Mar 2021.

Vancouver:

Augustine C. Traumatic Injury in vitro Elicits JNK-mediated Human Astrocyte Retraction, but Spares Cerebrovascular Endothelial Cells. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2021 Mar 06]. Available from: https://ir.lib.uwo.ca/etd/1411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Augustine C. Traumatic Injury in vitro Elicits JNK-mediated Human Astrocyte Retraction, but Spares Cerebrovascular Endothelial Cells. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

27. Paulding, Waltke Reche, 1964-. Regulation of atrial natriuretic peptide receptors in astroglia.

Degree: 1996, University of Florida

Subjects/Keywords: Research ( mesh ); Receptors, Atrial Natriuretic Factor  – physiology ( mesh ); Astrocytes ( mesh ); Cells, Cultured ( mesh ); Protein Kinase C  – physiology ( mesh ); Angiotensin II  – physiology ( mesh ); Rats, Sprague-Dawley ( mesh ); Rats, Inbred SHR ( mesh ); Rats, Inbred WKY ( mesh ); Department of Physiology thesis Ph.D ( mesh )

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paulding, Waltke Reche, 1. (1996). Regulation of atrial natriuretic peptide receptors in astroglia. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00067432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paulding, Waltke Reche, 1964-. “Regulation of atrial natriuretic peptide receptors in astroglia.” 1996. Thesis, University of Florida. Accessed March 06, 2021. https://ufdc.ufl.edu/AA00067432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paulding, Waltke Reche, 1964-. “Regulation of atrial natriuretic peptide receptors in astroglia.” 1996. Web. 06 Mar 2021.

Vancouver:

Paulding, Waltke Reche 1. Regulation of atrial natriuretic peptide receptors in astroglia. [Internet] [Thesis]. University of Florida; 1996. [cited 2021 Mar 06]. Available from: https://ufdc.ufl.edu/AA00067432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paulding, Waltke Reche 1. Regulation of atrial natriuretic peptide receptors in astroglia. [Thesis]. University of Florida; 1996. Available from: https://ufdc.ufl.edu/AA00067432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

28. Rydzewski, Bartosz, 1967-. Angiotensin II regulates the expression of extracellular protease inhibitors in the central nervous system.

Degree: 1992, University of Florida

Subjects/Keywords: Research ( mesh ); Angiotensin II  – physiology ( mesh ); Angiotensin II  – pharmacology ( mesh ); Protease Inhibitors  – metabolism ( mesh ); Plasminogen Activator Inhibitor 1  – metabolism ( mesh ); RNA, Messenger  – metabolism ( mesh ); Central Nervous System  – physiology ( mesh ); Gene Expression Regulation ( mesh ); Rats, Inbred WKY ( mesh ); Astrocytes ( mesh ); Brain  – physiology ( mesh ); Department of Physiology thesis Ph.D ( mesh )

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rydzewski, Bartosz, 1. (1992). Angiotensin II regulates the expression of extracellular protease inhibitors in the central nervous system. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00066134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rydzewski, Bartosz, 1967-. “Angiotensin II regulates the expression of extracellular protease inhibitors in the central nervous system.” 1992. Thesis, University of Florida. Accessed March 06, 2021. https://ufdc.ufl.edu/AA00066134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rydzewski, Bartosz, 1967-. “Angiotensin II regulates the expression of extracellular protease inhibitors in the central nervous system.” 1992. Web. 06 Mar 2021.

Vancouver:

Rydzewski, Bartosz 1. Angiotensin II regulates the expression of extracellular protease inhibitors in the central nervous system. [Internet] [Thesis]. University of Florida; 1992. [cited 2021 Mar 06]. Available from: https://ufdc.ufl.edu/AA00066134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rydzewski, Bartosz 1. Angiotensin II regulates the expression of extracellular protease inhibitors in the central nervous system. [Thesis]. University of Florida; 1992. Available from: https://ufdc.ufl.edu/AA00066134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

29. Masters, Brian Andrew, 1962-. Insulin-like growth factors in cell differentiation and development in the nervous system.

Degree: 1990, University of Florida

Subjects/Keywords: Astrocytes; Brain; Cultured cells; Insulin; Neurons; Oligodendroglia; Progenitor cells; Rats; Receptors; Somatomedins; Department of Physiology thesis Ph.D ( mesh ); Insulin-Like Growth Factor I ( mesh ); Nervous System  – growth &; development ( mesh ); Oligodendroglia ( mesh ); Receptors, Somatomedin ( mesh ); Research ( mesh )

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Masters, Brian Andrew, 1. (1990). Insulin-like growth factors in cell differentiation and development in the nervous system. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00056906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Masters, Brian Andrew, 1962-. “Insulin-like growth factors in cell differentiation and development in the nervous system.” 1990. Thesis, University of Florida. Accessed March 06, 2021. https://ufdc.ufl.edu/AA00056906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Masters, Brian Andrew, 1962-. “Insulin-like growth factors in cell differentiation and development in the nervous system.” 1990. Web. 06 Mar 2021.

Vancouver:

Masters, Brian Andrew 1. Insulin-like growth factors in cell differentiation and development in the nervous system. [Internet] [Thesis]. University of Florida; 1990. [cited 2021 Mar 06]. Available from: https://ufdc.ufl.edu/AA00056906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Masters, Brian Andrew 1. Insulin-like growth factors in cell differentiation and development in the nervous system. [Thesis]. University of Florida; 1990. Available from: https://ufdc.ufl.edu/AA00056906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

30. Reutter, Michael A., 1959-. Regulation of Alpha2A-Adrenergic receptor expression in cultured rat astroglia by Michael A. Reutter.

Degree: 1997, University of Florida

Subjects/Keywords: Adrenergic receptors; Agonists; Astrocytes; Cell lines; Cultured cells; Inurement; Messenger RNA; Norepinephrine; Rats; Receptors; Angiotensin II ( mesh ); Astrocytes ( mesh ); Cells, Cultured ( mesh ); Cyclic AMP ( mesh ); Department of Physiology thesis Ph.D ( mesh ); Epinephrine ( mesh ); Gene Expression Regulation ( mesh ); Protein Kinase C ( mesh ); RNA, Messenger ( mesh ); Rats, Sprague-Dawley ( mesh ); Receptors, Adrenergic, alpha-2  – biosynthesis ( mesh ); Receptors, Adrenergic, alpha-2  – metabolism ( mesh ); Research ( mesh ); Signal Transduction ( mesh ); Transcription, Genetic ( mesh )

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reutter, Michael A., 1. (1997). Regulation of Alpha2A-Adrenergic receptor expression in cultured rat astroglia by Michael A. Reutter. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00011813

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reutter, Michael A., 1959-. “Regulation of Alpha2A-Adrenergic receptor expression in cultured rat astroglia by Michael A. Reutter.” 1997. Thesis, University of Florida. Accessed March 06, 2021. https://ufdc.ufl.edu/AA00011813.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reutter, Michael A., 1959-. “Regulation of Alpha2A-Adrenergic receptor expression in cultured rat astroglia by Michael A. Reutter.” 1997. Web. 06 Mar 2021.

Vancouver:

Reutter, Michael A. 1. Regulation of Alpha2A-Adrenergic receptor expression in cultured rat astroglia by Michael A. Reutter. [Internet] [Thesis]. University of Florida; 1997. [cited 2021 Mar 06]. Available from: https://ufdc.ufl.edu/AA00011813.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reutter, Michael A. 1. Regulation of Alpha2A-Adrenergic receptor expression in cultured rat astroglia by Michael A. Reutter. [Thesis]. University of Florida; 1997. Available from: https://ufdc.ufl.edu/AA00011813

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.