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You searched for subject:(Arrestin). Showing records 1 – 30 of 73 total matches.

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1. Coffa, Sergio. Non-visual arrestins bind mitogen activated protein kinases and regulate their signaling.

Degree: PhD, Pharmacology, 2011, Vanderbilt University

 Arrestins are multifunctional signaling proteins, important for the regulation of signal transduction and the trafficking of G protein-coupled receptors (GPCRs). Recently, GPCR-arrestin interactions have been… (more)

Subjects/Keywords: arrestin

arrestin interactions have been proposed to be necessary for activation of G-protein-independent… …MAPKs). To investigate potential arrestin-MAPK interactions, we have used a variety of… …molecular tools including the co-expression of the individual domains of arrestin with single… …surface of arrestin that are conserved between arrestin-2 and arrestin-3. We found that the… …substitution of arginine 307 with an alanine significantly reduced arrestin-2 binding to c-Raf1… 

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APA (6th Edition):

Coffa, S. (2011). Non-visual arrestins bind mitogen activated protein kinases and regulate their signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-08042011-074210/ ;

Chicago Manual of Style (16th Edition):

Coffa, Sergio. “Non-visual arrestins bind mitogen activated protein kinases and regulate their signaling.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 22, 2019. http://etd.library.vanderbilt.edu/available/etd-08042011-074210/ ;.

MLA Handbook (7th Edition):

Coffa, Sergio. “Non-visual arrestins bind mitogen activated protein kinases and regulate their signaling.” 2011. Web. 22 Apr 2019.

Vancouver:

Coffa S. Non-visual arrestins bind mitogen activated protein kinases and regulate their signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Apr 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-08042011-074210/ ;.

Council of Science Editors:

Coffa S. Non-visual arrestins bind mitogen activated protein kinases and regulate their signaling. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-08042011-074210/ ;


Wake Forest University

2. Blume, Lawrence Christopher. CANNABINOID RECEPTOR INTERACTING PROTEIN 1A (CRIP1A): EFFECTS ON CB1 RECEPTOR FUNCTION AND CELLULAR REGULATION.

Degree: 2014, Wake Forest University

 Cannabis, or marijuana, is the most commonly used illicit drug in the United States. The CB1 cannabinoid receptor (CB1R) has been extensively studied since the… (more)

Subjects/Keywords: B-ARRESTIN

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APA (6th Edition):

Blume, L. C. (2014). CANNABINOID RECEPTOR INTERACTING PROTEIN 1A (CRIP1A): EFFECTS ON CB1 RECEPTOR FUNCTION AND CELLULAR REGULATION. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/39334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Blume, Lawrence Christopher. “CANNABINOID RECEPTOR INTERACTING PROTEIN 1A (CRIP1A): EFFECTS ON CB1 RECEPTOR FUNCTION AND CELLULAR REGULATION.” 2014. Thesis, Wake Forest University. Accessed April 22, 2019. http://hdl.handle.net/10339/39334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Blume, Lawrence Christopher. “CANNABINOID RECEPTOR INTERACTING PROTEIN 1A (CRIP1A): EFFECTS ON CB1 RECEPTOR FUNCTION AND CELLULAR REGULATION.” 2014. Web. 22 Apr 2019.

Vancouver:

Blume LC. CANNABINOID RECEPTOR INTERACTING PROTEIN 1A (CRIP1A): EFFECTS ON CB1 RECEPTOR FUNCTION AND CELLULAR REGULATION. [Internet] [Thesis]. Wake Forest University; 2014. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/10339/39334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Blume LC. CANNABINOID RECEPTOR INTERACTING PROTEIN 1A (CRIP1A): EFFECTS ON CB1 RECEPTOR FUNCTION AND CELLULAR REGULATION. [Thesis]. Wake Forest University; 2014. Available from: http://hdl.handle.net/10339/39334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

3. Huang, Shun-Ping. Exploring alternative roles of visual arrestin 1 in photoreceptor synaptic regulation and deciphering the molecular pathway of retinal degeneration using mouse knockout technology.

Degree: PhD, Genetic, Molecular & Cellular Biology, 2010, University of Southern California

 In the G-protein-coupled receptor (GPCR) phototransduction cascade, visual Arrestin1 (Arr1) binds to and deactivates phosphorylated light-activated opsins, a process that is critical for effective recovery… (more)

Subjects/Keywords: visual arrestin; S-antigen; cone arrestin; beta-arrestin; N-ethylmaleimide sensitive factor; SNARE; photoreceptor; synapse; FM1-43

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APA (6th Edition):

Huang, S. (2010). Exploring alternative roles of visual arrestin 1 in photoreceptor synaptic regulation and deciphering the molecular pathway of retinal degeneration using mouse knockout technology. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/370201/rec/2648

Chicago Manual of Style (16th Edition):

Huang, Shun-Ping. “Exploring alternative roles of visual arrestin 1 in photoreceptor synaptic regulation and deciphering the molecular pathway of retinal degeneration using mouse knockout technology.” 2010. Doctoral Dissertation, University of Southern California. Accessed April 22, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/370201/rec/2648.

MLA Handbook (7th Edition):

Huang, Shun-Ping. “Exploring alternative roles of visual arrestin 1 in photoreceptor synaptic regulation and deciphering the molecular pathway of retinal degeneration using mouse knockout technology.” 2010. Web. 22 Apr 2019.

Vancouver:

Huang S. Exploring alternative roles of visual arrestin 1 in photoreceptor synaptic regulation and deciphering the molecular pathway of retinal degeneration using mouse knockout technology. [Internet] [Doctoral dissertation]. University of Southern California; 2010. [cited 2019 Apr 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/370201/rec/2648.

Council of Science Editors:

Huang S. Exploring alternative roles of visual arrestin 1 in photoreceptor synaptic regulation and deciphering the molecular pathway of retinal degeneration using mouse knockout technology. [Doctoral Dissertation]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/370201/rec/2648


University of Toronto

4. Robinson, Kelly Ann. Arrestin Interactions with Rhodopsin in the Squid Visual System.

Degree: 2015, University of Toronto

Light activation of squid rhodopsin results in stimulation of the Gq signalling cascade. Activated rhodopsin (metarhodopsin) is a target for squid arrestin and squid rhodopsin… (more)

Subjects/Keywords: arrestin; phosphorylation; rhodopsin; squid; vision; 0419

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APA (6th Edition):

Robinson, K. A. (2015). Arrestin Interactions with Rhodopsin in the Squid Visual System. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69696

Chicago Manual of Style (16th Edition):

Robinson, Kelly Ann. “Arrestin Interactions with Rhodopsin in the Squid Visual System.” 2015. Masters Thesis, University of Toronto. Accessed April 22, 2019. http://hdl.handle.net/1807/69696.

MLA Handbook (7th Edition):

Robinson, Kelly Ann. “Arrestin Interactions with Rhodopsin in the Squid Visual System.” 2015. Web. 22 Apr 2019.

Vancouver:

Robinson KA. Arrestin Interactions with Rhodopsin in the Squid Visual System. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/1807/69696.

Council of Science Editors:

Robinson KA. Arrestin Interactions with Rhodopsin in the Squid Visual System. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69696


University of Southern California

5. Deming, Janise D. Cone arrestin 4 contributes to vision, cone health, and desensitization of the dopamine receptor D4.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2015, University of Southern California

 In this dissertation, I investigated classical and alternative roles of Cone Arrestin 4 (ARR4). First, my collaborators and I described visual phenotypes of Arr4 null… (more)

Subjects/Keywords: arrestin; visual arrestin; cone arrestin; dopamine receptor; dopamine receptor D4; DRD4; ARR4; β ‐arrestin; retina; vision; G-protein coupled receptors; GPCR; phototransduction

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APA (6th Edition):

Deming, J. D. (2015). Cone arrestin 4 contributes to vision, cone health, and desensitization of the dopamine receptor D4. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/584780/rec/1575

Chicago Manual of Style (16th Edition):

Deming, Janise D. “Cone arrestin 4 contributes to vision, cone health, and desensitization of the dopamine receptor D4.” 2015. Doctoral Dissertation, University of Southern California. Accessed April 22, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/584780/rec/1575.

MLA Handbook (7th Edition):

Deming, Janise D. “Cone arrestin 4 contributes to vision, cone health, and desensitization of the dopamine receptor D4.” 2015. Web. 22 Apr 2019.

Vancouver:

Deming JD. Cone arrestin 4 contributes to vision, cone health, and desensitization of the dopamine receptor D4. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2019 Apr 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/584780/rec/1575.

Council of Science Editors:

Deming JD. Cone arrestin 4 contributes to vision, cone health, and desensitization of the dopamine receptor D4. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/584780/rec/1575


ETH Zürich

6. Mayer, Daniel. Molecular basis for the recognition of the phosphorylation pattern in the C-terminal tail of GPCRs by arrestins.

Degree: 2017, ETH Zürich

 Arrestins are responsible for desensitization and internalization of activated, phosphorylated G protein-coupled receptors (GPCRs). Over the years, it became clear that they have the potential… (more)

Subjects/Keywords: Arrestin; GPCR; phosphorylation barcode; phosphorylation sites

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APA (6th Edition):

Mayer, D. (2017). Molecular basis for the recognition of the phosphorylation pattern in the C-terminal tail of GPCRs by arrestins. (Doctoral Dissertation). ETH Zürich. Retrieved from http://hdl.handle.net/20.500.11850/215427

Chicago Manual of Style (16th Edition):

Mayer, Daniel. “Molecular basis for the recognition of the phosphorylation pattern in the C-terminal tail of GPCRs by arrestins.” 2017. Doctoral Dissertation, ETH Zürich. Accessed April 22, 2019. http://hdl.handle.net/20.500.11850/215427.

MLA Handbook (7th Edition):

Mayer, Daniel. “Molecular basis for the recognition of the phosphorylation pattern in the C-terminal tail of GPCRs by arrestins.” 2017. Web. 22 Apr 2019.

Vancouver:

Mayer D. Molecular basis for the recognition of the phosphorylation pattern in the C-terminal tail of GPCRs by arrestins. [Internet] [Doctoral dissertation]. ETH Zürich; 2017. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/20.500.11850/215427.

Council of Science Editors:

Mayer D. Molecular basis for the recognition of the phosphorylation pattern in the C-terminal tail of GPCRs by arrestins. [Doctoral Dissertation]. ETH Zürich; 2017. Available from: http://hdl.handle.net/20.500.11850/215427


University of Florida

7. Masannat, Jude Naser. Beta Arrestin 2 Mediates Renal Cell Carcinoma Tumor Growth and Metastasis.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2017, University of Florida

 Renal Cell Carcinoma (RCC) is one of the most lethal urological cancers worldwide, with incidence and mortality rates increasing in the past two decades. The… (more)

Subjects/Keywords: arrestin  – cancer  – metastasis  – proliferation  – rcc  – src

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APA (6th Edition):

Masannat, J. N. (2017). Beta Arrestin 2 Mediates Renal Cell Carcinoma Tumor Growth and Metastasis. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0051319

Chicago Manual of Style (16th Edition):

Masannat, Jude Naser. “Beta Arrestin 2 Mediates Renal Cell Carcinoma Tumor Growth and Metastasis.” 2017. Doctoral Dissertation, University of Florida. Accessed April 22, 2019. http://ufdc.ufl.edu/UFE0051319.

MLA Handbook (7th Edition):

Masannat, Jude Naser. “Beta Arrestin 2 Mediates Renal Cell Carcinoma Tumor Growth and Metastasis.” 2017. Web. 22 Apr 2019.

Vancouver:

Masannat JN. Beta Arrestin 2 Mediates Renal Cell Carcinoma Tumor Growth and Metastasis. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2019 Apr 22]. Available from: http://ufdc.ufl.edu/UFE0051319.

Council of Science Editors:

Masannat JN. Beta Arrestin 2 Mediates Renal Cell Carcinoma Tumor Growth and Metastasis. [Doctoral Dissertation]. University of Florida; 2017. Available from: http://ufdc.ufl.edu/UFE0051319


Duke University

8. Nobles, Kelly Nicole. Phosphorylation Bar Codes Induce Distinct Conformations and Functionalities of beta-Arrestin .

Degree: 2010, Duke University

  Seven transmembrane spanning receptors (7TMRs), or G-protein coupled receptors (GPCRs), represent the largest and most ubiquitous of the several families of plasma membrane receptors… (more)

Subjects/Keywords: Biochemistry; 7TMRs; beta-arrestin; GRKs; Phosphorylation

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APA (6th Edition):

Nobles, K. N. (2010). Phosphorylation Bar Codes Induce Distinct Conformations and Functionalities of beta-Arrestin . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/3117

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nobles, Kelly Nicole. “Phosphorylation Bar Codes Induce Distinct Conformations and Functionalities of beta-Arrestin .” 2010. Thesis, Duke University. Accessed April 22, 2019. http://hdl.handle.net/10161/3117.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nobles, Kelly Nicole. “Phosphorylation Bar Codes Induce Distinct Conformations and Functionalities of beta-Arrestin .” 2010. Web. 22 Apr 2019.

Vancouver:

Nobles KN. Phosphorylation Bar Codes Induce Distinct Conformations and Functionalities of beta-Arrestin . [Internet] [Thesis]. Duke University; 2010. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/10161/3117.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nobles KN. Phosphorylation Bar Codes Induce Distinct Conformations and Functionalities of beta-Arrestin . [Thesis]. Duke University; 2010. Available from: http://hdl.handle.net/10161/3117

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

9. Wagener, Brant. Regulation of N-formyl peptide receptor trafficking and signaling by arrestins.

Degree: Biomedical Sciences Graduate Program, 2009, University of New Mexico

 This work focuses on how arrestin regulates trafficking and signaling of the N-formyl peptide receptor (FPR), a G protein-coupled receptor (GPCR). GPCRs are involved in… (more)

Subjects/Keywords: arrestin; FPR; apoptosis; signaling; trafficking; recycling

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APA (6th Edition):

Wagener, B. (2009). Regulation of N-formyl peptide receptor trafficking and signaling by arrestins. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/105

Chicago Manual of Style (16th Edition):

Wagener, Brant. “Regulation of N-formyl peptide receptor trafficking and signaling by arrestins.” 2009. Doctoral Dissertation, University of New Mexico. Accessed April 22, 2019. https://digitalrepository.unm.edu/biom_etds/105.

MLA Handbook (7th Edition):

Wagener, Brant. “Regulation of N-formyl peptide receptor trafficking and signaling by arrestins.” 2009. Web. 22 Apr 2019.

Vancouver:

Wagener B. Regulation of N-formyl peptide receptor trafficking and signaling by arrestins. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2019 Apr 22]. Available from: https://digitalrepository.unm.edu/biom_etds/105.

Council of Science Editors:

Wagener B. Regulation of N-formyl peptide receptor trafficking and signaling by arrestins. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: https://digitalrepository.unm.edu/biom_etds/105


University of Rochester

10. Jones, Brian William. Phosphorylation of the Thyrotropin-releasing Hormone Receptor.

Degree: PhD, 2009, University of Rochester

 G protein-coupled receptors (GPCRs) regulate numerous signaling pathways. Their activity is tightly regulated through phosphorylation at multiple sites. These studies were performed to elucidate the… (more)

Subjects/Keywords: Arrestin; Rab GTPase; Trafficking; GRK; Desensitization

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APA (6th Edition):

Jones, B. W. (2009). Phosphorylation of the Thyrotropin-releasing Hormone Receptor. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6618

Chicago Manual of Style (16th Edition):

Jones, Brian William. “Phosphorylation of the Thyrotropin-releasing Hormone Receptor.” 2009. Doctoral Dissertation, University of Rochester. Accessed April 22, 2019. http://hdl.handle.net/1802/6618.

MLA Handbook (7th Edition):

Jones, Brian William. “Phosphorylation of the Thyrotropin-releasing Hormone Receptor.” 2009. Web. 22 Apr 2019.

Vancouver:

Jones BW. Phosphorylation of the Thyrotropin-releasing Hormone Receptor. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/1802/6618.

Council of Science Editors:

Jones BW. Phosphorylation of the Thyrotropin-releasing Hormone Receptor. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6618


University of Western Australia

11. Jaeger, Werner Christian. Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling.

Degree: PhD, 2011, University of Western Australia

[Truncated abstract] The orexin endocrine system has been associated with a range of physiological functions including regulation of sleep and wake states, energy metabolism, addictive… (more)

Subjects/Keywords: Energy metabolism; Sleep/wake; Orexin; GPCR; β-arrestin; BRET; Ubiquitin; MAPK

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APA (6th Edition):

Jaeger, W. C. (2011). Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Jaeger, Werner Christian. “Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling.” 2011. Doctoral Dissertation, University of Western Australia. Accessed April 22, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Jaeger, Werner Christian. “Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling.” 2011. Web. 22 Apr 2019.

Vancouver:

Jaeger WC. Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling. [Internet] [Doctoral dissertation]. University of Western Australia; 2011. [cited 2019 Apr 22]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01.

Council of Science Editors:

Jaeger WC. Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling. [Doctoral Dissertation]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01


University of Illinois – Chicago

12. Hoeppner, Crystal Z. Identification of a Nuclear Localization Sequence in Beta-Arrestin1:Implications in NF- kB Activation.

Degree: 2012, University of Illinois – Chicago

 A mounting body of evidence suggests that beta-arrestin1 plays important roles in the nucleus, but how beta-arrestin1 enters the nucleus remains unclear since no nuclear… (more)

Subjects/Keywords: G Protein-Coupled Receptors; Transcription; Beta-Arrestin; Nuclear Localization

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APA (6th Edition):

Hoeppner, C. Z. (2012). Identification of a Nuclear Localization Sequence in Beta-Arrestin1:Implications in NF- kB Activation. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoeppner, Crystal Z. “Identification of a Nuclear Localization Sequence in Beta-Arrestin1:Implications in NF- kB Activation.” 2012. Thesis, University of Illinois – Chicago. Accessed April 22, 2019. http://hdl.handle.net/10027/9243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoeppner, Crystal Z. “Identification of a Nuclear Localization Sequence in Beta-Arrestin1:Implications in NF- kB Activation.” 2012. Web. 22 Apr 2019.

Vancouver:

Hoeppner CZ. Identification of a Nuclear Localization Sequence in Beta-Arrestin1:Implications in NF- kB Activation. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/10027/9243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoeppner CZ. Identification of a Nuclear Localization Sequence in Beta-Arrestin1:Implications in NF- kB Activation. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

13. Chen, Qiuyan. The structure basis of arrestin mediated GPCR signaling.

Degree: PhD, Pharmacology, 2015, Vanderbilt University

Arrestin selectively binds the phosphorylated active receptor to either terminate the G protein dependent signaling or initiate G protein independent signaling. Receptor binding induces global… (more)

Subjects/Keywords: signaling; structural basis; arrestin; G protein coupled receptor

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APA (6th Edition):

Chen, Q. (2015). The structure basis of arrestin mediated GPCR signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;

Chicago Manual of Style (16th Edition):

Chen, Qiuyan. “The structure basis of arrestin mediated GPCR signaling.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 22, 2019. http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;.

MLA Handbook (7th Edition):

Chen, Qiuyan. “The structure basis of arrestin mediated GPCR signaling.” 2015. Web. 22 Apr 2019.

Vancouver:

Chen Q. The structure basis of arrestin mediated GPCR signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Apr 22]. Available from: http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;.

Council of Science Editors:

Chen Q. The structure basis of arrestin mediated GPCR signaling. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-11162015-201134/ ;


Vanderbilt University

14. Cleghorn, Whitney Marie. Arrestins regulate cell spreading and motility via focal adhesion dynamics.

Degree: PhD, Pharmacology, 2012, Vanderbilt University

 Arrestins bind G protein-coupled receptors and more than 100 non-receptor partners, regulating various signaling pathways and cellular functions. The interactions of many proteins (e.g., Src,… (more)

Subjects/Keywords: structural biology; pharmacology; morphology; cell biology; GPCR; arrestin

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APA (6th Edition):

Cleghorn, W. M. (2012). Arrestins regulate cell spreading and motility via focal adhesion dynamics. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06222012-142651/ ;

Chicago Manual of Style (16th Edition):

Cleghorn, Whitney Marie. “Arrestins regulate cell spreading and motility via focal adhesion dynamics.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 22, 2019. http://etd.library.vanderbilt.edu/available/etd-06222012-142651/ ;.

MLA Handbook (7th Edition):

Cleghorn, Whitney Marie. “Arrestins regulate cell spreading and motility via focal adhesion dynamics.” 2012. Web. 22 Apr 2019.

Vancouver:

Cleghorn WM. Arrestins regulate cell spreading and motility via focal adhesion dynamics. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Apr 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-06222012-142651/ ;.

Council of Science Editors:

Cleghorn WM. Arrestins regulate cell spreading and motility via focal adhesion dynamics. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-06222012-142651/ ;


Duquesne University

15. Sethi, Shalini. Effect of Melatonin on Differentiation of Human Mesenchymal Stem Cells and a Study on C-Terminal Domains of MT1 and MT2 Melatonin Receptors.

Degree: PhD, Pharmacology, 2010, Duquesne University

 Melatonin has been reported to enhance the differentiation of osteoblasts. The purpose of this study was to determine the melatonin treatment that would differentiate human… (more)

Subjects/Keywords: Beta-arrestin; Desensitization; Melatonin receptors; Mesenchymal stem cells; Mutations; Osteoblasts

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APA (6th Edition):

Sethi, S. (2010). Effect of Melatonin on Differentiation of Human Mesenchymal Stem Cells and a Study on C-Terminal Domains of MT1 and MT2 Melatonin Receptors. (Doctoral Dissertation). Duquesne University. Retrieved from https://dsc.duq.edu/etd/1173

Chicago Manual of Style (16th Edition):

Sethi, Shalini. “Effect of Melatonin on Differentiation of Human Mesenchymal Stem Cells and a Study on C-Terminal Domains of MT1 and MT2 Melatonin Receptors.” 2010. Doctoral Dissertation, Duquesne University. Accessed April 22, 2019. https://dsc.duq.edu/etd/1173.

MLA Handbook (7th Edition):

Sethi, Shalini. “Effect of Melatonin on Differentiation of Human Mesenchymal Stem Cells and a Study on C-Terminal Domains of MT1 and MT2 Melatonin Receptors.” 2010. Web. 22 Apr 2019.

Vancouver:

Sethi S. Effect of Melatonin on Differentiation of Human Mesenchymal Stem Cells and a Study on C-Terminal Domains of MT1 and MT2 Melatonin Receptors. [Internet] [Doctoral dissertation]. Duquesne University; 2010. [cited 2019 Apr 22]. Available from: https://dsc.duq.edu/etd/1173.

Council of Science Editors:

Sethi S. Effect of Melatonin on Differentiation of Human Mesenchymal Stem Cells and a Study on C-Terminal Domains of MT1 and MT2 Melatonin Receptors. [Doctoral Dissertation]. Duquesne University; 2010. Available from: https://dsc.duq.edu/etd/1173


Purdue University

16. Brust Fernandes, Tarsis. FUNCTIONAL SELECTIVITY DOWNSTREAM OF Gαi/o-COUPLED RECEPTORS.

Degree: PhD, Medicinal Chemistry and Molecular Pharmacology, 2015, Purdue University

 G protein-coupled receptors (GPCRs) are drug targets that often activate multiple signaling pathways. The multiple GPCR responses provide opportunities for biased or functionally selective ligands… (more)

Subjects/Keywords: adenylyl cyclase; beta-arrestin; biased signaling; functional selectivity; GPCR; G protein

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APA (6th Edition):

Brust Fernandes, T. (2015). FUNCTIONAL SELECTIVITY DOWNSTREAM OF Gαi/o-COUPLED RECEPTORS. (Doctoral Dissertation). Purdue University. Retrieved from https://docs.lib.purdue.edu/open_access_dissertations/1448

Chicago Manual of Style (16th Edition):

Brust Fernandes, Tarsis. “FUNCTIONAL SELECTIVITY DOWNSTREAM OF Gαi/o-COUPLED RECEPTORS.” 2015. Doctoral Dissertation, Purdue University. Accessed April 22, 2019. https://docs.lib.purdue.edu/open_access_dissertations/1448.

MLA Handbook (7th Edition):

Brust Fernandes, Tarsis. “FUNCTIONAL SELECTIVITY DOWNSTREAM OF Gαi/o-COUPLED RECEPTORS.” 2015. Web. 22 Apr 2019.

Vancouver:

Brust Fernandes T. FUNCTIONAL SELECTIVITY DOWNSTREAM OF Gαi/o-COUPLED RECEPTORS. [Internet] [Doctoral dissertation]. Purdue University; 2015. [cited 2019 Apr 22]. Available from: https://docs.lib.purdue.edu/open_access_dissertations/1448.

Council of Science Editors:

Brust Fernandes T. FUNCTIONAL SELECTIVITY DOWNSTREAM OF Gαi/o-COUPLED RECEPTORS. [Doctoral Dissertation]. Purdue University; 2015. Available from: https://docs.lib.purdue.edu/open_access_dissertations/1448


University of Illinois – Urbana-Champaign

17. Cervantes, David. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.

Degree: PhD, 0325, 2012, University of Illinois – Urbana-Champaign

 Heart disease and diabetes mellitus are growing epidemics, consistently ranking within the top ten causes of death in the United States. Both diseases are associated… (more)

Subjects/Keywords: adrenergic receptor; insulin receptor; extracellular signal-regulated kinase (ERK); heart; arrestin

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APA (6th Edition):

Cervantes, D. (2012). Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29837

Chicago Manual of Style (16th Edition):

Cervantes, David. “Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed April 22, 2019. http://hdl.handle.net/2142/29837.

MLA Handbook (7th Edition):

Cervantes, David. “Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.” 2012. Web. 22 Apr 2019.

Vancouver:

Cervantes D. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/2142/29837.

Council of Science Editors:

Cervantes D. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29837


Duke University

18. Cahill, Thomas J. New Insights into GPCR–Transducer Coupling .

Degree: 2018, Duke University

  β-arrestins (βarrs) interact with G protein-coupled receptors (GPCRs) to desensitize G protein signaling, initiate signaling on their own, and mediate receptor endocytosis.  Using a… (more)

Subjects/Keywords: Biochemistry; arrestin; desensitization; electron microscopy; endocytosis; GPCR; signaling

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APA (6th Edition):

Cahill, T. J. (2018). New Insights into GPCR–Transducer Coupling . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/16774

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cahill, Thomas J. “New Insights into GPCR–Transducer Coupling .” 2018. Thesis, Duke University. Accessed April 22, 2019. http://hdl.handle.net/10161/16774.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cahill, Thomas J. “New Insights into GPCR–Transducer Coupling .” 2018. Web. 22 Apr 2019.

Vancouver:

Cahill TJ. New Insights into GPCR–Transducer Coupling . [Internet] [Thesis]. Duke University; 2018. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/10161/16774.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cahill TJ. New Insights into GPCR–Transducer Coupling . [Thesis]. Duke University; 2018. Available from: http://hdl.handle.net/10161/16774

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Groer, Chad E. Agonist-selective regulation of the mu opioid receptor by ßarrestins.

Degree: PhD, Integrated Biomedical Science Graduate Program, 2010, The Ohio State University

  Morphine and other opiates mediate their effects through activation of the mu opioid receptor (MOR). Activation of the MOR results in recruitment of regulatory… (more)

Subjects/Keywords: Pharmacology; opioid arrestin herkinorin morphine

…with Differential β-Arrestin-2 Interactions. J. Med. Chem. 2008 Apr 24;51(8):2421… …An opioid agonist that does not induce mu-opioid receptor-arrestin interactions or receptor… …classical regulatory mechanisms including phosphorylation, arrestin binding, desensitization, and… …respectively [47]. The phosphorylated GPCR can serve as a substrate for arrestin binding… …x5B;48-50]. Arrestin protein binding is most robust when GPCRs are both… 

Page 1 Page 2 Page 3 Page 4 Page 5

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APA (6th Edition):

Groer, C. E. (2010). Agonist-selective regulation of the mu opioid receptor by ßarrestins. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1284396146

Chicago Manual of Style (16th Edition):

Groer, Chad E. “Agonist-selective regulation of the mu opioid receptor by ßarrestins.” 2010. Doctoral Dissertation, The Ohio State University. Accessed April 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1284396146.

MLA Handbook (7th Edition):

Groer, Chad E. “Agonist-selective regulation of the mu opioid receptor by ßarrestins.” 2010. Web. 22 Apr 2019.

Vancouver:

Groer CE. Agonist-selective regulation of the mu opioid receptor by ßarrestins. [Internet] [Doctoral dissertation]. The Ohio State University; 2010. [cited 2019 Apr 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1284396146.

Council of Science Editors:

Groer CE. Agonist-selective regulation of the mu opioid receptor by ßarrestins. [Doctoral Dissertation]. The Ohio State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1284396146


The Ohio State University

20. Raehal, Kirsten Michele. Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice.

Degree: PhD, Integrated Biomedical Sciences, 2009, The Ohio State University

 Opioid drugs are potent analgesics; however, they also produce several adverse side effects including constipation, antinociceptive tolerance, and physical dependence by activating the mu opioid… (more)

Subjects/Keywords: Pharmacology; opioid; arrestin; G protein-coupled receptor kinase; gastrointestinal; physical dependence; tolerance

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APA (6th Edition):

Raehal, K. M. (2009). Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585

Chicago Manual of Style (16th Edition):

Raehal, Kirsten Michele. “Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice.” 2009. Doctoral Dissertation, The Ohio State University. Accessed April 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585.

MLA Handbook (7th Edition):

Raehal, Kirsten Michele. “Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice.” 2009. Web. 22 Apr 2019.

Vancouver:

Raehal KM. Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2019 Apr 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585.

Council of Science Editors:

Raehal KM. Opioid-Induced Side Effects in Beta-arrestin2 adn G Protein-Coupled Receptor Kinase Knockout Mice. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1236884585


University of Southern California

21. Chan, Sanny Kai-Wai. Signaling cascades: a functional characterization of cone arrestin and a differential gene expression analysis of developing retinal ganglion cells.

Degree: PhD, Cell & Neurobiology, 2007, University of Southern California

 The mature retina is a specialized tissue critical for vision. Individual cell-types that comprise the retina have specialized functional roles mediated by differential gene expression.… (more)

Subjects/Keywords: phototransduction; cone arrestin; retinal ganglion cells

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APA (6th Edition):

Chan, S. K. (2007). Signaling cascades: a functional characterization of cone arrestin and a differential gene expression analysis of developing retinal ganglion cells. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/583837/rec/5846

Chicago Manual of Style (16th Edition):

Chan, Sanny Kai-Wai. “Signaling cascades: a functional characterization of cone arrestin and a differential gene expression analysis of developing retinal ganglion cells.” 2007. Doctoral Dissertation, University of Southern California. Accessed April 22, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/583837/rec/5846.

MLA Handbook (7th Edition):

Chan, Sanny Kai-Wai. “Signaling cascades: a functional characterization of cone arrestin and a differential gene expression analysis of developing retinal ganglion cells.” 2007. Web. 22 Apr 2019.

Vancouver:

Chan SK. Signaling cascades: a functional characterization of cone arrestin and a differential gene expression analysis of developing retinal ganglion cells. [Internet] [Doctoral dissertation]. University of Southern California; 2007. [cited 2019 Apr 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/583837/rec/5846.

Council of Science Editors:

Chan SK. Signaling cascades: a functional characterization of cone arrestin and a differential gene expression analysis of developing retinal ganglion cells. [Doctoral Dissertation]. University of Southern California; 2007. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/583837/rec/5846


University of Western Ontario

22. Murphy-Marshman, Hannah E. Identification Of A Novel Interaction Between Integrin-Linked Kinase And Beta-Arrestin 1.

Degree: 2016, University of Western Ontario

 Integrin-linked kinase (ILK) is a ubiquitous scaffold protein essential for the development of front-rear polarity and directional migration of epidermal keratinocytes. β-arrestin 1 is another… (more)

Subjects/Keywords: Integrin-linked kinase; Beta-arrestin; protein-protein interactions; epidermis; keratinocytes; Cellular and Molecular Physiology

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APA (6th Edition):

Murphy-Marshman, H. E. (2016). Identification Of A Novel Interaction Between Integrin-Linked Kinase And Beta-Arrestin 1. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Murphy-Marshman, Hannah E. “Identification Of A Novel Interaction Between Integrin-Linked Kinase And Beta-Arrestin 1.” 2016. Thesis, University of Western Ontario. Accessed April 22, 2019. https://ir.lib.uwo.ca/etd/4182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Murphy-Marshman, Hannah E. “Identification Of A Novel Interaction Between Integrin-Linked Kinase And Beta-Arrestin 1.” 2016. Web. 22 Apr 2019.

Vancouver:

Murphy-Marshman HE. Identification Of A Novel Interaction Between Integrin-Linked Kinase And Beta-Arrestin 1. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2019 Apr 22]. Available from: https://ir.lib.uwo.ca/etd/4182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Murphy-Marshman HE. Identification Of A Novel Interaction Between Integrin-Linked Kinase And Beta-Arrestin 1. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

23. McLean, Sarah E.A. Transforming Growth Factor Beta Receptor Partitioning: Molecular Mechanisms and Functional Consequences.

Degree: 2012, University of Western Ontario

 The transforming growth factor beta (TGFbeta) pathway has been conserved throughout evolution and plays important roles in tissue homeostasis. Dysregulation of the TGFbeta pathway has… (more)

Subjects/Keywords: Transforming growth factor beta; Membrane raft; Endocytosis; Beta arrestin; Intracellular Trafficking; Medical Sciences

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APA (6th Edition):

McLean, S. E. A. (2012). Transforming Growth Factor Beta Receptor Partitioning: Molecular Mechanisms and Functional Consequences. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McLean, Sarah E A. “Transforming Growth Factor Beta Receptor Partitioning: Molecular Mechanisms and Functional Consequences.” 2012. Thesis, University of Western Ontario. Accessed April 22, 2019. https://ir.lib.uwo.ca/etd/752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McLean, Sarah E A. “Transforming Growth Factor Beta Receptor Partitioning: Molecular Mechanisms and Functional Consequences.” 2012. Web. 22 Apr 2019.

Vancouver:

McLean SEA. Transforming Growth Factor Beta Receptor Partitioning: Molecular Mechanisms and Functional Consequences. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2019 Apr 22]. Available from: https://ir.lib.uwo.ca/etd/752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McLean SEA. Transforming Growth Factor Beta Receptor Partitioning: Molecular Mechanisms and Functional Consequences. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. C. Cancellieri. BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2.

Degree: 2014, Università degli Studi di Milano

 Chemokines promote leukocyte migration through the activation of dedicated G-protein coupled receptors. Beyond conventional chemokine receptors, which directly induce cell migration through heterotrimeric Gαi-mediated signalling… (more)

Subjects/Keywords: chemokine; atypical chemokine receptor; sytoskeleton; actin; microtubules; myosin; beta-arrestin; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Cancellieri, C. (2014). BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cancellieri, C.. “BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2.” 2014. Thesis, Università degli Studi di Milano. Accessed April 22, 2019. http://hdl.handle.net/2434/229565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cancellieri, C.. “BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2.” 2014. Web. 22 Apr 2019.

Vancouver:

Cancellieri C. BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/2434/229565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cancellieri C. BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

25. Kuhar, Jamie Rose. Mechanisms of Opioid Receptor Desensitization.

Degree: PhD, 2015, University of Washington

 Agonists targeting MOR are effective analgesics, but their clinical use is hindered by side effects, including tolerance and addiction. KOR agonists also produce analgesia, but… (more)

Subjects/Keywords: Arrestin; c-Jun N-terminal kinase; Fentanyl; Morphine; Opioid; p38; Pharmacology; pharmacology

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APA (6th Edition):

Kuhar, J. R. (2015). Mechanisms of Opioid Receptor Desensitization. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34122

Chicago Manual of Style (16th Edition):

Kuhar, Jamie Rose. “Mechanisms of Opioid Receptor Desensitization.” 2015. Doctoral Dissertation, University of Washington. Accessed April 22, 2019. http://hdl.handle.net/1773/34122.

MLA Handbook (7th Edition):

Kuhar, Jamie Rose. “Mechanisms of Opioid Receptor Desensitization.” 2015. Web. 22 Apr 2019.

Vancouver:

Kuhar JR. Mechanisms of Opioid Receptor Desensitization. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/1773/34122.

Council of Science Editors:

Kuhar JR. Mechanisms of Opioid Receptor Desensitization. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34122


Université Montpellier II

26. Armando, Sylvain. Structure quaternaire des récepteurs de chimiokines CXCR4 et CCR2 et interaction avec leur effecteurs. : Quaternary arrangements of the CXCR4-CCR2 homo- and hetero-oligomers and of their complexes with their signaling effectors.

Degree: Docteur es, Neurosciences, 2010, Université Montpellier II

Les récepteurs couplés aux protéines G (RCPG) sont la famille de récepteurs membranaires la plus représentée chez les vertébrés, et la plus grande cible thérapeutique… (more)

Subjects/Keywords: Cxcr4; Tétramère; Bret; Asymétrie; Protéines G; Arrestines; Cxcr4; Tetramer; Bret; Asymetry; G protein; Arrestin

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APA (6th Edition):

Armando, S. (2010). Structure quaternaire des récepteurs de chimiokines CXCR4 et CCR2 et interaction avec leur effecteurs. : Quaternary arrangements of the CXCR4-CCR2 homo- and hetero-oligomers and of their complexes with their signaling effectors. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2010MON20208

Chicago Manual of Style (16th Edition):

Armando, Sylvain. “Structure quaternaire des récepteurs de chimiokines CXCR4 et CCR2 et interaction avec leur effecteurs. : Quaternary arrangements of the CXCR4-CCR2 homo- and hetero-oligomers and of their complexes with their signaling effectors.” 2010. Doctoral Dissertation, Université Montpellier II. Accessed April 22, 2019. http://www.theses.fr/2010MON20208.

MLA Handbook (7th Edition):

Armando, Sylvain. “Structure quaternaire des récepteurs de chimiokines CXCR4 et CCR2 et interaction avec leur effecteurs. : Quaternary arrangements of the CXCR4-CCR2 homo- and hetero-oligomers and of their complexes with their signaling effectors.” 2010. Web. 22 Apr 2019.

Vancouver:

Armando S. Structure quaternaire des récepteurs de chimiokines CXCR4 et CCR2 et interaction avec leur effecteurs. : Quaternary arrangements of the CXCR4-CCR2 homo- and hetero-oligomers and of their complexes with their signaling effectors. [Internet] [Doctoral dissertation]. Université Montpellier II; 2010. [cited 2019 Apr 22]. Available from: http://www.theses.fr/2010MON20208.

Council of Science Editors:

Armando S. Structure quaternaire des récepteurs de chimiokines CXCR4 et CCR2 et interaction avec leur effecteurs. : Quaternary arrangements of the CXCR4-CCR2 homo- and hetero-oligomers and of their complexes with their signaling effectors. [Doctoral Dissertation]. Université Montpellier II; 2010. Available from: http://www.theses.fr/2010MON20208


Vanderbilt University

27. Walker III, William Benjamin. On the nature of the sensory arrestins of the dipteran insects Anopheles gambiae and Drosophila melanogaster.

Degree: PhD, Neuroscience, 2009, Vanderbilt University

 This project is concerned with the functional roles of the sensory arrestin genes, arr1 and arr2, in the regulation of dipteran olfactory signal transduction. Specifically,… (more)

Subjects/Keywords: Anopheles gambiae; Arrestin; Drosophila melanogaster; Olfaction; Desensitization; Smell  – Molecular aspects; Insects  – Physiology

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APA (6th Edition):

Walker III, W. B. (2009). On the nature of the sensory arrestins of the dipteran insects Anopheles gambiae and Drosophila melanogaster. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03312008-162553/ ;

Chicago Manual of Style (16th Edition):

Walker III, William Benjamin. “On the nature of the sensory arrestins of the dipteran insects Anopheles gambiae and Drosophila melanogaster.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed April 22, 2019. http://etd.library.vanderbilt.edu/available/etd-03312008-162553/ ;.

MLA Handbook (7th Edition):

Walker III, William Benjamin. “On the nature of the sensory arrestins of the dipteran insects Anopheles gambiae and Drosophila melanogaster.” 2009. Web. 22 Apr 2019.

Vancouver:

Walker III WB. On the nature of the sensory arrestins of the dipteran insects Anopheles gambiae and Drosophila melanogaster. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2019 Apr 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-03312008-162553/ ;.

Council of Science Editors:

Walker III WB. On the nature of the sensory arrestins of the dipteran insects Anopheles gambiae and Drosophila melanogaster. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://etd.library.vanderbilt.edu/available/etd-03312008-162553/ ;


The Ohio State University

28. Davidson, Reshma. Regulation of Septum Formation by Two Novel Proteins Art1 and Bga1 in Fission Yeast Cytokinesis.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2016, The Ohio State University

 Cytokinesis is the final step in the cell cycle that partitions a mother cell into two daughter cells. The final step in cytokinesis involves extracellular… (more)

Subjects/Keywords: Biology; Genetics; Molecular Biology; Cytokinesis; Schizosaccharomyces pombe; Septum; Arrestin; SKN1; Kre6; Rgf3; Rho GTPase

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APA (6th Edition):

Davidson, R. (2016). Regulation of Septum Formation by Two Novel Proteins Art1 and Bga1 in Fission Yeast Cytokinesis. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1469185292

Chicago Manual of Style (16th Edition):

Davidson, Reshma. “Regulation of Septum Formation by Two Novel Proteins Art1 and Bga1 in Fission Yeast Cytokinesis.” 2016. Doctoral Dissertation, The Ohio State University. Accessed April 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1469185292.

MLA Handbook (7th Edition):

Davidson, Reshma. “Regulation of Septum Formation by Two Novel Proteins Art1 and Bga1 in Fission Yeast Cytokinesis.” 2016. Web. 22 Apr 2019.

Vancouver:

Davidson R. Regulation of Septum Formation by Two Novel Proteins Art1 and Bga1 in Fission Yeast Cytokinesis. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2019 Apr 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1469185292.

Council of Science Editors:

Davidson R. Regulation of Septum Formation by Two Novel Proteins Art1 and Bga1 in Fission Yeast Cytokinesis. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1469185292


Université de Montréal

29. Bonneterre, Julien. Impact de différentes modalités de recrutement de la β-arrestine au récepteur de chimiokine CXCR4 .

Degree: 2018, Université de Montréal

Subjects/Keywords: CXCR4; β-arrestin; GRK; BRET; WHIM

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bonneterre, J. (2018). Impact de différentes modalités de recrutement de la β-arrestine au récepteur de chimiokine CXCR4 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/20403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonneterre, Julien. “Impact de différentes modalités de recrutement de la β-arrestine au récepteur de chimiokine CXCR4 .” 2018. Thesis, Université de Montréal. Accessed April 22, 2019. http://hdl.handle.net/1866/20403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonneterre, Julien. “Impact de différentes modalités de recrutement de la β-arrestine au récepteur de chimiokine CXCR4 .” 2018. Web. 22 Apr 2019.

Vancouver:

Bonneterre J. Impact de différentes modalités de recrutement de la β-arrestine au récepteur de chimiokine CXCR4 . [Internet] [Thesis]. Université de Montréal; 2018. [cited 2019 Apr 22]. Available from: http://hdl.handle.net/1866/20403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonneterre J. Impact de différentes modalités de recrutement de la β-arrestine au récepteur de chimiokine CXCR4 . [Thesis]. Université de Montréal; 2018. Available from: http://hdl.handle.net/1866/20403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

30. Nguyen, Peter. CANNABINOID RECEPTORS IN THE 3D RECONSTRUCTED MOUSE BRAIN: FUNCTION AND REGULATION.

Degree: PhD, Pharmacology & Toxicology, 2010, Virginia Commonwealth University

 CB1 receptors (CB1R) mediate the psychoactive and therapeutic effects of cannabinoids including ∆9-tetrahydrocannabinol (THC), the main psychoactive constituent in marijuana. However, therapeutic use is limited… (more)

Subjects/Keywords: cannabinoid receptors; imaging; statistical parametric mapping; beta-arrestin; Medical Pharmacology; Medical Sciences; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nguyen, P. (2010). CANNABINOID RECEPTORS IN THE 3D RECONSTRUCTED MOUSE BRAIN: FUNCTION AND REGULATION. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/2274

Chicago Manual of Style (16th Edition):

Nguyen, Peter. “CANNABINOID RECEPTORS IN THE 3D RECONSTRUCTED MOUSE BRAIN: FUNCTION AND REGULATION.” 2010. Doctoral Dissertation, Virginia Commonwealth University. Accessed April 22, 2019. https://scholarscompass.vcu.edu/etd/2274.

MLA Handbook (7th Edition):

Nguyen, Peter. “CANNABINOID RECEPTORS IN THE 3D RECONSTRUCTED MOUSE BRAIN: FUNCTION AND REGULATION.” 2010. Web. 22 Apr 2019.

Vancouver:

Nguyen P. CANNABINOID RECEPTORS IN THE 3D RECONSTRUCTED MOUSE BRAIN: FUNCTION AND REGULATION. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2010. [cited 2019 Apr 22]. Available from: https://scholarscompass.vcu.edu/etd/2274.

Council of Science Editors:

Nguyen P. CANNABINOID RECEPTORS IN THE 3D RECONSTRUCTED MOUSE BRAIN: FUNCTION AND REGULATION. [Doctoral Dissertation]. Virginia Commonwealth University; 2010. Available from: https://scholarscompass.vcu.edu/etd/2274

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