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You searched for subject:(Antigens CD95 metabolism 60). Showing records 1 – 30 of 12882 total matches.

[1] [2] [3] [4] [5] … [430]

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1. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

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APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 15 Oct 2019.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

2. Swindall, Amanda F. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.

Degree: PhD, 2012, University of Alabama – Birmingham

The golgi glycosyltransferase, ST6Gal-I, adds a negatively-charged sialic acid in an alpha-2-6 linkage to N-linked glycans. ST6Gal-I is upregulated in many cancers, and is associated… (more)

Subjects/Keywords: Antigens, CD – metabolism.<; br>; Antigens, CD95 – metabolism.<; br>; Apoptosis<; br>; Tumor Cells, Cultured<; br>; Colonic Neoplasms<; br>; Neoplasm Proteins – metabolism.<; br>; Sialyltransferases – metabolism.

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APA (6th Edition):

Swindall, A. F. (2012). The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1392

Chicago Manual of Style (16th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1392.

MLA Handbook (7th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Web. 15 Oct 2019.

Vancouver:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392.

Council of Science Editors:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392

3. Brand, Jeffrey David. The role of CD11c+ cells in response to inhaled ozone.

Degree: PhD, 2012, University of Alabama – Birmingham

Environmental ozone exposure remains a major public health concern with over one-third of the United States population experiencing unhealthful levels. Epidemiological studies indicate exposure increases… (more)

Subjects/Keywords: Antigens, CD80.<; br>; Antigens, CD86.<; br>; Asthma – immunology<; br>; Dendritic Cells – metabolism.<; br>; Inhalation Exposure – adverse effects.<; br>; Ozone – toxicity.

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APA (6th Edition):

Brand, J. D. (2012). The role of CD11c+ cells in response to inhaled ozone. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1346

Chicago Manual of Style (16th Edition):

Brand, Jeffrey David. “The role of CD11c+ cells in response to inhaled ozone.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1346.

MLA Handbook (7th Edition):

Brand, Jeffrey David. “The role of CD11c+ cells in response to inhaled ozone.” 2012. Web. 15 Oct 2019.

Vancouver:

Brand JD. The role of CD11c+ cells in response to inhaled ozone. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1346.

Council of Science Editors:

Brand JD. The role of CD11c+ cells in response to inhaled ozone. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1346

4. Axtell, Robert C. (Robert Chaddon). The role of CD5 in experimental autoimmune encephalitomyelitis.

Degree: PhD, 2007, University of Alabama – Birmingham

The regulation of activation, differentiation and persistence of effector T-cells are critical for the development of Experimental Autoimmune Encephalomyelitis (EAE), the animal model of Multiple… (more)

Subjects/Keywords: Antigens, CD5  – metabolism <; br>; Encephalomyelitis, Autoimmune, Experimental  – immunology <; br>; Mice <; br>; Multiple Sclerosis

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APA (6th Edition):

Axtell, R. C. (. C. (2007). The role of CD5 in experimental autoimmune encephalitomyelitis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,84

Chicago Manual of Style (16th Edition):

Axtell, Robert C (Robert Chaddon). “The role of CD5 in experimental autoimmune encephalitomyelitis.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,84.

MLA Handbook (7th Edition):

Axtell, Robert C (Robert Chaddon). “The role of CD5 in experimental autoimmune encephalitomyelitis.” 2007. Web. 15 Oct 2019.

Vancouver:

Axtell RC(C. The role of CD5 in experimental autoimmune encephalitomyelitis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,84.

Council of Science Editors:

Axtell RC(C. The role of CD5 in experimental autoimmune encephalitomyelitis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,84

5. Fetterman, Jessica L. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.

Degree: PhD, 2011, University of Alabama – Birmingham

While progress has been made in understanding the development and progression of cardiovascular disease (CVD), the mechanisms of CVD risk and initiation are not completely… (more)

Subjects/Keywords: Antigens – metabolism.<; br>; Hematopoietic Stem Cell Transplantation – methods.<; br>; Hematopoietic Stem Cells – metabolism.<; br>; HIV-1<; br>; Immunologic Memory – genetics.<; br>; Lentivirus.<; br>; Neoplasms – immunology<; br>; T-Lymphocytes, Cytotoxic – immunology

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APA (6th Edition):

Fetterman, J. L. (2011). Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1395

Chicago Manual of Style (16th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1395.

MLA Handbook (7th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Web. 15 Oct 2019.

Vancouver:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395.

Council of Science Editors:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395

6. You, Yuying. Cross-talk between marginal zone B cells and marginal zone macrophages.

Degree: PhD, 2010, University of Alabama – Birmingham

The marginal zone (MZ) in the spleen provides a critical defense against bloodborne pathogens. Specialized MZ macrophages (MZM) identified previously based on scavenger receptor (MARCO)… (more)

Subjects/Keywords: Antigens, CD19  – immunology<; br>; Antigens, CD19  – metabolism<; br>; B-Lymphocytes  – cytology<; br>; B-Lymphocytes  – immunology<; br>; Spleen  – cytology<; br>; Spleen  – immunology

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APA (6th Edition):

You, Y. (2010). Cross-talk between marginal zone B cells and marginal zone macrophages. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,659

Chicago Manual of Style (16th Edition):

You, Yuying. “Cross-talk between marginal zone B cells and marginal zone macrophages.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,659.

MLA Handbook (7th Edition):

You, Yuying. “Cross-talk between marginal zone B cells and marginal zone macrophages.” 2010. Web. 15 Oct 2019.

Vancouver:

You Y. Cross-talk between marginal zone B cells and marginal zone macrophages. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,659.

Council of Science Editors:

You Y. Cross-talk between marginal zone B cells and marginal zone macrophages. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,659

7. Luo, Wenyi. Identification and characterization of virulence factors of mycoplasmas.

Degree: PhD, 2009, University of Alabama – Birmingham

Mycoplasmas cause a group of diseases that are characterized by their chronicity and resistance to treatment. Diseases caused by the murine pathogens Mycoplasma pulmonis and… (more)

Subjects/Keywords: Antigens, Bacterial  – toxicity<; br>; Arthritis, Infectious  – microbiology<; br>; DNA Modification Methylases  – metabolism<; br>; Deoxyribonucleases, Type II Site-Specific  – metabolism<; br>; Mycoplasma arthritidis<; br>; Mycoplasma Infections  – microbiology<; br>; Superantigens  – toxicity

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APA (6th Edition):

Luo, W. (2009). Identification and characterization of virulence factors of mycoplasmas. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,649

Chicago Manual of Style (16th Edition):

Luo, Wenyi. “Identification and characterization of virulence factors of mycoplasmas.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,649.

MLA Handbook (7th Edition):

Luo, Wenyi. “Identification and characterization of virulence factors of mycoplasmas.” 2009. Web. 15 Oct 2019.

Vancouver:

Luo W. Identification and characterization of virulence factors of mycoplasmas. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,649.

Council of Science Editors:

Luo W. Identification and characterization of virulence factors of mycoplasmas. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,649

8. Larson, Matthew Rodney. Elucidation of the elongated fibrillar structure of Streptococcus mutans antigen I/II.

Degree: PhD, 2011, University of Alabama – Birmingham

Streptococcus mutans (S. mutans) is the causative agent behind dental caries, an infectious disease also known as tooth decay or dental cavities. S. mutans has… (more)

Subjects/Keywords: Adhesins, Bacterial  – chemistry<; br>; Agglutinins  – chemistry<; br>; Antigens, Bacterial  – chemistry<; br>; Saliva  – metabolism<; br>; Streptococcus mutans  – chemistry<; br>; Streptococcus mutans  – metabolism<; br>

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APA (6th Edition):

Larson, M. R. (2011). Elucidation of the elongated fibrillar structure of Streptococcus mutans antigen I/II. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1093

Chicago Manual of Style (16th Edition):

Larson, Matthew Rodney. “Elucidation of the elongated fibrillar structure of Streptococcus mutans antigen I/II.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1093.

MLA Handbook (7th Edition):

Larson, Matthew Rodney. “Elucidation of the elongated fibrillar structure of Streptococcus mutans antigen I/II.” 2011. Web. 15 Oct 2019.

Vancouver:

Larson MR. Elucidation of the elongated fibrillar structure of Streptococcus mutans antigen I/II. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1093.

Council of Science Editors:

Larson MR. Elucidation of the elongated fibrillar structure of Streptococcus mutans antigen I/II. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1093

9. Woodard-Grice, Alencia V. (Alencia Vanay). Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.

Degree: PhD, 2008, University of Alabama – Birmingham

During monocyte activation and differentiation along the macrophage lineage, the activity of [alpha]4[beta]1 integrins is upregulated, which promotes extravasation from the vasculature and migration through… (more)

Subjects/Keywords: Amyloid Precursor Protein Secretases  – metabolism<; br>; Antigens, CD  – biosynthesis<; br>; Aspartic Endopeptidases  – metabolism<; br>; Cell Differentiation  – physiology<; br>; Integrin alpha4beta1  – metabolism<; br>; Macrophages  – enzymology<; br>; Monocytes  – enzymology<; br>; Protein Modification, Translational  – physiology<; br>; Sialyltransferases  – biosynthesis

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APA (6th Edition):

Woodard-Grice, A. V. (. V. (2008). Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,473

Chicago Manual of Style (16th Edition):

Woodard-Grice, Alencia V (Alencia Vanay). “Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,473.

MLA Handbook (7th Edition):

Woodard-Grice, Alencia V (Alencia Vanay). “Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.” 2008. Web. 15 Oct 2019.

Vancouver:

Woodard-Grice AV(V. Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,473.

Council of Science Editors:

Woodard-Grice AV(V. Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,473

10. Lai, Jen-Fang. The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae.

Degree: PhD, 2009, University of Alabama – Birmingham

Several reports have suggested that Mycoplasma pneumoniae (Mp) can contribute importantly to the expression of symptoms in asthmatic human subjects. As a foundation for understanding… (more)

Subjects/Keywords: Antigens, CD11b  – metabolism<; br>; Macrophages  – immunology<; br>; Mice<; br>; Myeloid Differentiation Factor 88<; br>; Pneumonia, Mycoplasma  – metabolism<; br>; Toll-Like Receptors

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APA (6th Edition):

Lai, J. (2009). The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,680

Chicago Manual of Style (16th Edition):

Lai, Jen-Fang. “The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,680.

MLA Handbook (7th Edition):

Lai, Jen-Fang. “The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae.” 2009. Web. 15 Oct 2019.

Vancouver:

Lai J. The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,680.

Council of Science Editors:

Lai J. The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,680

11. Schreeder, Daniel M. Biological characterization of Fc receptor-like 6 (FCRL6).

Degree: PhD, 2009, University of Alabama – Birmingham

Members of the Fc receptor-like (FCRL) family are cell-surface proteins with ancient conservation, distinct lymphocyte expression patterns, and tyrosine-based signaling capabilities that imply a fundamental… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes  – immunology<; br>; HLA-DR Antigens  – metabolism<; br>; Killer Cells, Natural  – immunology<; br>; Leukemia, Lymphocytic, Chronic, B-Cell  – immunology<; br>; Receptors, Cell Surface  – physiology<; br>; Receptors, Fc  – metabolism

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APA (6th Edition):

Schreeder, D. M. (2009). Biological characterization of Fc receptor-like 6 (FCRL6). (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1112

Chicago Manual of Style (16th Edition):

Schreeder, Daniel M. “Biological characterization of Fc receptor-like 6 (FCRL6).” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1112.

MLA Handbook (7th Edition):

Schreeder, Daniel M. “Biological characterization of Fc receptor-like 6 (FCRL6).” 2009. Web. 15 Oct 2019.

Vancouver:

Schreeder DM. Biological characterization of Fc receptor-like 6 (FCRL6). [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1112.

Council of Science Editors:

Schreeder DM. Biological characterization of Fc receptor-like 6 (FCRL6). [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1112

12. gupte. Cellular mechanisms involved in host responses to Porphyromonas gingivalis and its virulence factor Hemagglutinin B.

Degree: PhD, 2010, University of Alabama – Birmingham

Porphyromonas gingivalis is a main causative agent for adult chronic periodontitis and immunization with its virulence factor Hemagglutinin B (HagB) provides protection against infection. Toll-like… (more)

Subjects/Keywords: Adaptor Proteins, Vesicular Transport – metabolism.<; br>; Adhesins, Bacterial – immunology.<; br>; Antigens, CD14 – metabolism.<; br>; Bacterial Proteins – pharmacology.<; br>; CD4-Positive T-Lymphocytes – immunology<; br>; Dendritic Cells – drug effects.<; br>; Myeloid Differentiation Factor 88 – metabolism.<; br>; Porphyromonas gingivalis – immunology<; br>; Signal Transduction<; br>; Toll-Like Receptor 4 – metabolism.

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APA (6th Edition):

gupte. (2010). Cellular mechanisms involved in host responses to Porphyromonas gingivalis and its virulence factor Hemagglutinin B. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1347

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

gupte. “Cellular mechanisms involved in host responses to Porphyromonas gingivalis and its virulence factor Hemagglutinin B.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1347.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

gupte. “Cellular mechanisms involved in host responses to Porphyromonas gingivalis and its virulence factor Hemagglutinin B.” 2010. Web. 15 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

gupte. Cellular mechanisms involved in host responses to Porphyromonas gingivalis and its virulence factor Hemagglutinin B. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1347.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

gupte. Cellular mechanisms involved in host responses to Porphyromonas gingivalis and its virulence factor Hemagglutinin B. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1347

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

13. Ashley, Jason Waid. Significance and regulation of CD68 expression in the osteoclast.

Degree: PhD, 2011, University of Alabama – Birmingham

The mucin-like Lysosome Associated Membrane Protein (LAMP) family member CD68 is a primarily myeloid lineage restricted transmembrane protein that is expressed in macrophages and osteoclasts.… (more)

Subjects/Keywords: Antigens, CD  – genetics<; br>; Antigens, Differentiation, Myelomonocytic  – genetics<; br>; Biological Assay  – methods<; br>; Bone and Bones<; br>; Drug Evaluation, Preclinical  – methods<; br>; Gene Deletion<; br>; High-Throughput Screening Assays  – methods<; br>; Macrophages  – drug effects<; br>; Osteoclasts<; br>; Receptor Activator of Nuclear Factor-kappa B  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Ashley, J. W. (2011). Significance and regulation of CD68 expression in the osteoclast. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1033

Chicago Manual of Style (16th Edition):

Ashley, Jason Waid. “Significance and regulation of CD68 expression in the osteoclast.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1033.

MLA Handbook (7th Edition):

Ashley, Jason Waid. “Significance and regulation of CD68 expression in the osteoclast.” 2011. Web. 15 Oct 2019.

Vancouver:

Ashley JW. Significance and regulation of CD68 expression in the osteoclast. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1033.

Council of Science Editors:

Ashley JW. Significance and regulation of CD68 expression in the osteoclast. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1033

14. Myers, Riley Croom. The role of CD19 in follicular dendritic cell activation.

Degree: PhD, 2011, University of Alabama – Birmingham

Germinal Centers (GC) are organized foci in secondary lymphoid organs of birds and mammals and are the principle sites of memory B cell generation and… (more)

Subjects/Keywords: Antigens, CD19  – metabolism<; br>; Lymphocytes  – cytology<; br>; B-Lymphocytes  – immunology<; br>; Dendritic Cells  – immunology<; br>; Germinal Center<; br>; Vascular Cell Adhesion Molecule-1

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APA (6th Edition):

Myers, R. C. (2011). The role of CD19 in follicular dendritic cell activation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1053

Chicago Manual of Style (16th Edition):

Myers, Riley Croom. “The role of CD19 in follicular dendritic cell activation.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1053.

MLA Handbook (7th Edition):

Myers, Riley Croom. “The role of CD19 in follicular dendritic cell activation.” 2011. Web. 15 Oct 2019.

Vancouver:

Myers RC. The role of CD19 in follicular dendritic cell activation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1053.

Council of Science Editors:

Myers RC. The role of CD19 in follicular dendritic cell activation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1053

15. Hong, Sang Yong. Pax5 controls B lineage specific gene expression program through association with the nuclear matrix.

Degree: PhD, 2011, University of Alabama – Birmingham

Pax5 is an essential regulator for B lineage cell development and controls hundreds of positive and negative target genes. The nuclear matrix (NM) has long… (more)

Subjects/Keywords: Antigens, CD19 – biosynthesis.<; br>; B-Cell-Specific Activator Protein – metabolism.<; br>; Lysine – chemistry.<; br>; Molecular Sequence Data<; br>; Nuclear Matrix<; br>; Protein Binding

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APA (6th Edition):

Hong, S. Y. (2011). Pax5 controls B lineage specific gene expression program through association with the nuclear matrix. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1354

Chicago Manual of Style (16th Edition):

Hong, Sang Yong. “Pax5 controls B lineage specific gene expression program through association with the nuclear matrix.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1354.

MLA Handbook (7th Edition):

Hong, Sang Yong. “Pax5 controls B lineage specific gene expression program through association with the nuclear matrix.” 2011. Web. 15 Oct 2019.

Vancouver:

Hong SY. Pax5 controls B lineage specific gene expression program through association with the nuclear matrix. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1354.

Council of Science Editors:

Hong SY. Pax5 controls B lineage specific gene expression program through association with the nuclear matrix. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1354

16. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 15 Oct 2019.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956

17. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 15 Oct 2019.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

18. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 15 Oct 2019.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

19. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

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APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 15 Oct 2019.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

20. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 15 Oct 2019.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

21. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

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APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 15 Oct 2019.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

22. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 15 Oct 2019.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

23. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 15 Oct 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

24. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

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APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 15 Oct 2019.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

25. Lazaryan, Aleksandr. Human leukocyte antigen supertypes in relation to human imunodeficiency virus infection among populations of African ancestry.

Degree: PhD, 2008, University of Alabama – Birmingham

The role of human leukocyte antigen (HLA)-A and HLA-B supertypes in control-ling human immunodeficiency virus type 1 (HIV-1) infection in African-Americans re-mains poorly understood. We… (more)

Subjects/Keywords: African Continental Ancestry Group<; br>; Alleles<; br>; Antigens, Differentiation  – genetics<; br>; HIV Infections  – epidemiology<; br>; HIV Infections  – immunology<; br>; HIV-1  – genetics<; br>; HIV-1  – pathogenicity<; br>; HLA-B Antigens/immunology

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APA (6th Edition):

Lazaryan, A. (2008). Human leukocyte antigen supertypes in relation to human imunodeficiency virus infection among populations of African ancestry. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,525

Chicago Manual of Style (16th Edition):

Lazaryan, Aleksandr. “Human leukocyte antigen supertypes in relation to human imunodeficiency virus infection among populations of African ancestry.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,525.

MLA Handbook (7th Edition):

Lazaryan, Aleksandr. “Human leukocyte antigen supertypes in relation to human imunodeficiency virus infection among populations of African ancestry.” 2008. Web. 15 Oct 2019.

Vancouver:

Lazaryan A. Human leukocyte antigen supertypes in relation to human imunodeficiency virus infection among populations of African ancestry. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,525.

Council of Science Editors:

Lazaryan A. Human leukocyte antigen supertypes in relation to human imunodeficiency virus infection among populations of African ancestry. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,525

26. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

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APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 15 Oct 2019.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

27. Durham, Carolyn G. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.

Degree: PhD, 2010, University of Alabama – Birmingham

The inverse correlation between the industrialization and disease prevalence is termed the "hygiene hypothesis." Supporting this, immunological studies show Th1 cytokines modulate Th2 immune responses.… (more)

Subjects/Keywords: Asthma<; br>; Gastric Mucosa  – metabolism<; br>; Gastritis<; br>; Helicobacter Infections  – metabolism<; br>; Helicobacter felis<; br>; Mucus  – metabolism

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APA (6th Edition):

Durham, C. G. (2010). Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,707

Chicago Manual of Style (16th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,707.

MLA Handbook (7th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Web. 15 Oct 2019.

Vancouver:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707.

Council of Science Editors:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707

28. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 15 Oct 2019.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

29. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 15 Oct 2019.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

30. Ingram, Jennifer T. Exhaustion renders CD8 T cells unresponsive to antigen-independent activation and is characterized by IL-18 receptor downregulation.

Degree: MS, 2011, University of Alabama – Birmingham

During many chronic infections virus-specific CD8 T cells succumb to exhaustion as they lose their ability to respond to antigenic activation. Combinations of interleukin (IL)-12,… (more)

Subjects/Keywords: T cells  – Immunology <; br>; T cells  – Receptors <; br>; Interleukins <; br>; CD antigens <; br>; Interferon inducers <; br>; Activation (Chemistry)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ingram, J. T. (2011). Exhaustion renders CD8 T cells unresponsive to antigen-independent activation and is characterized by IL-18 receptor downregulation. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,887

Chicago Manual of Style (16th Edition):

Ingram, Jennifer T. “Exhaustion renders CD8 T cells unresponsive to antigen-independent activation and is characterized by IL-18 receptor downregulation.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed October 15, 2019. http://contentdm.mhsl.uab.edu/u?/etd,887.

MLA Handbook (7th Edition):

Ingram, Jennifer T. “Exhaustion renders CD8 T cells unresponsive to antigen-independent activation and is characterized by IL-18 receptor downregulation.” 2011. Web. 15 Oct 2019.

Vancouver:

Ingram JT. Exhaustion renders CD8 T cells unresponsive to antigen-independent activation and is characterized by IL-18 receptor downregulation. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 15]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,887.

Council of Science Editors:

Ingram JT. Exhaustion renders CD8 T cells unresponsive to antigen-independent activation and is characterized by IL-18 receptor downregulation. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,887

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