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Dates: Last 2 Years

You searched for subject:(Angiogenesis). Showing records 1 – 30 of 97 total matches.

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NSYSU

1. Song , Yi-Chun. Coral-derived natural marine compound GB9 inhibits vascular development in zebrafish.

Degree: Master, Biological Sciences, 2017, NSYSU

 Vascular development is important for vertebrates, and genetic control vascular patterning has been intensively characterized. In addition, many studies have been show environmental hormones and… (more)

Subjects/Keywords: zebrafish; marine compound; angiogenesis; GB9

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APA (6th Edition):

Song , Y. (2017). Coral-derived natural marine compound GB9 inhibits vascular development in zebrafish. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115842

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Song , Yi-Chun. “Coral-derived natural marine compound GB9 inhibits vascular development in zebrafish.” 2017. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115842.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Song , Yi-Chun. “Coral-derived natural marine compound GB9 inhibits vascular development in zebrafish.” 2017. Web. 16 Jul 2019.

Vancouver:

Song Y. Coral-derived natural marine compound GB9 inhibits vascular development in zebrafish. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115842.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Song Y. Coral-derived natural marine compound GB9 inhibits vascular development in zebrafish. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115842

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

2. Herring, Cassandra Marie. Dietary Arginine Supplementation Enhances Placental Water Transport and Angiogenesis in Gestating Gilts.

Degree: MS, Physiology of Reproduction, 2018, Texas A&M University

 This study was conducted to test the hypothesis that dietary supplementation with 0.4% L-arginine between days 14 and 30 of gestation would enhance survival and… (more)

Subjects/Keywords: Reproduction; Nutrition; Placenta; Arginine; Angiogenesis

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APA (6th Edition):

Herring, C. M. (2018). Dietary Arginine Supplementation Enhances Placental Water Transport and Angiogenesis in Gestating Gilts. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174006

Chicago Manual of Style (16th Edition):

Herring, Cassandra Marie. “Dietary Arginine Supplementation Enhances Placental Water Transport and Angiogenesis in Gestating Gilts.” 2018. Masters Thesis, Texas A&M University. Accessed July 16, 2019. http://hdl.handle.net/1969.1/174006.

MLA Handbook (7th Edition):

Herring, Cassandra Marie. “Dietary Arginine Supplementation Enhances Placental Water Transport and Angiogenesis in Gestating Gilts.” 2018. Web. 16 Jul 2019.

Vancouver:

Herring CM. Dietary Arginine Supplementation Enhances Placental Water Transport and Angiogenesis in Gestating Gilts. [Internet] [Masters thesis]. Texas A&M University; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1969.1/174006.

Council of Science Editors:

Herring CM. Dietary Arginine Supplementation Enhances Placental Water Transport and Angiogenesis in Gestating Gilts. [Masters Thesis]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/174006


NSYSU

3. Liang, Shuo-Rong. NAD(P)H dependent steroid dehydrogenase-like (nsdhl) involved in cholesterol biosynthesis is critical for vascular development in zebrafish.

Degree: Master, Biological Sciences, 2018, NSYSU

 Genetic conrol of vascular development and patterning in vertebrates is not fully understood. We used zebrafish as a model organism to identify transcription factors Islet2… (more)

Subjects/Keywords: Nsdhl; VEGF; BMP; Angiogenesis; zebrafish

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APA (6th Edition):

Liang, S. (2018). NAD(P)H dependent steroid dehydrogenase-like (nsdhl) involved in cholesterol biosynthesis is critical for vascular development in zebrafish. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-131141

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liang, Shuo-Rong. “NAD(P)H dependent steroid dehydrogenase-like (nsdhl) involved in cholesterol biosynthesis is critical for vascular development in zebrafish.” 2018. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-131141.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liang, Shuo-Rong. “NAD(P)H dependent steroid dehydrogenase-like (nsdhl) involved in cholesterol biosynthesis is critical for vascular development in zebrafish.” 2018. Web. 16 Jul 2019.

Vancouver:

Liang S. NAD(P)H dependent steroid dehydrogenase-like (nsdhl) involved in cholesterol biosynthesis is critical for vascular development in zebrafish. [Internet] [Thesis]. NSYSU; 2018. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-131141.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liang S. NAD(P)H dependent steroid dehydrogenase-like (nsdhl) involved in cholesterol biosynthesis is critical for vascular development in zebrafish. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-131141

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

4. Shen, Ying-Ying. Generation and Characterization of Recombinant Adenovirus Encoding Irisin.

Degree: Master, Institute of Biomedical Sciences, 2017, NSYSU

 Exercise represents one of the most effective approaches for control of obesity and metabolic syndromes. Irisin is a 112-residue myokine secreted by skeletal muscle through… (more)

Subjects/Keywords: angiogenesis; gene therapy; obesity; myokines; Irisin; gluconeogenesis

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APA (6th Edition):

Shen, Y. (2017). Generation and Characterization of Recombinant Adenovirus Encoding Irisin. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0722117-235342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shen, Ying-Ying. “Generation and Characterization of Recombinant Adenovirus Encoding Irisin.” 2017. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0722117-235342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shen, Ying-Ying. “Generation and Characterization of Recombinant Adenovirus Encoding Irisin.” 2017. Web. 16 Jul 2019.

Vancouver:

Shen Y. Generation and Characterization of Recombinant Adenovirus Encoding Irisin. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0722117-235342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shen Y. Generation and Characterization of Recombinant Adenovirus Encoding Irisin. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0722117-235342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Ouarné, Marie. ALK1 : une nouvelle voie de signalisation dans le développement vasculaire physiologique et tumoral : ALK1 : a new pathway in physiological and tumoral vascular development.

Degree: Docteur es, Biologie cellulaire, 2017, Grenoble Alpes

ALK1 (Actlivin receptor-Like Kinase 1) est un récepteur exprimé spécifiquement sur les cellules endothéliales. ALK1 possède deux ligands de haute affinité BMP9 (Bone Morphogenic Protein)… (more)

Subjects/Keywords: Angiogenèse; Lymphangiogenèse; Cancer; Angiogenesis; Lymphangiogenesis; Cancer; 570

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APA (6th Edition):

Ouarné, M. (2017). ALK1 : une nouvelle voie de signalisation dans le développement vasculaire physiologique et tumoral : ALK1 : a new pathway in physiological and tumoral vascular development. (Doctoral Dissertation). Grenoble Alpes. Retrieved from http://www.theses.fr/2017GREAV052

Chicago Manual of Style (16th Edition):

Ouarné, Marie. “ALK1 : une nouvelle voie de signalisation dans le développement vasculaire physiologique et tumoral : ALK1 : a new pathway in physiological and tumoral vascular development.” 2017. Doctoral Dissertation, Grenoble Alpes. Accessed July 16, 2019. http://www.theses.fr/2017GREAV052.

MLA Handbook (7th Edition):

Ouarné, Marie. “ALK1 : une nouvelle voie de signalisation dans le développement vasculaire physiologique et tumoral : ALK1 : a new pathway in physiological and tumoral vascular development.” 2017. Web. 16 Jul 2019.

Vancouver:

Ouarné M. ALK1 : une nouvelle voie de signalisation dans le développement vasculaire physiologique et tumoral : ALK1 : a new pathway in physiological and tumoral vascular development. [Internet] [Doctoral dissertation]. Grenoble Alpes; 2017. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2017GREAV052.

Council of Science Editors:

Ouarné M. ALK1 : une nouvelle voie de signalisation dans le développement vasculaire physiologique et tumoral : ALK1 : a new pathway in physiological and tumoral vascular development. [Doctoral Dissertation]. Grenoble Alpes; 2017. Available from: http://www.theses.fr/2017GREAV052


University of Sydney

6. Danastas, Kevin. The role of vascular endothelial growth factor A in physiological and pathological angiogenesis .

Degree: 2017, University of Sydney

 Vascular endothelial growth factor A (VEGF-A) is a major contributor to physiological and pathological angiogenesis in the body. VEGF-A exists as several isoforms varying significantly… (more)

Subjects/Keywords: VEGF; angiogenesis; pregnancy; endo; etriosis; cancer

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APA (6th Edition):

Danastas, K. (2017). The role of vascular endothelial growth factor A in physiological and pathological angiogenesis . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Danastas, Kevin. “The role of vascular endothelial growth factor A in physiological and pathological angiogenesis .” 2017. Thesis, University of Sydney. Accessed July 16, 2019. http://hdl.handle.net/2123/17978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Danastas, Kevin. “The role of vascular endothelial growth factor A in physiological and pathological angiogenesis .” 2017. Web. 16 Jul 2019.

Vancouver:

Danastas K. The role of vascular endothelial growth factor A in physiological and pathological angiogenesis . [Internet] [Thesis]. University of Sydney; 2017. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2123/17978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Danastas K. The role of vascular endothelial growth factor A in physiological and pathological angiogenesis . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

7. Calvo Hoyas, Paula. Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio del potencial papel terapéutico del RNA de doble cadena (pIC-PEI) .

Degree: 2018, Universitat de Valencia

 La endometriosis es una enfermedad con elevada prevalencia en la población de mujeres en edad fértil (Goldstein y cols, 1980; Eskenazi y cols, 1997; Gazvani… (more)

Subjects/Keywords: endometriosis; angiogenesis; apoptosis; mouse model; pICPEI

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APA (6th Edition):

Calvo Hoyas, P. (2018). Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio del potencial papel terapéutico del RNA de doble cadena (pIC-PEI) . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/66945

Chicago Manual of Style (16th Edition):

Calvo Hoyas, Paula. “Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio del potencial papel terapéutico del RNA de doble cadena (pIC-PEI) .” 2018. Doctoral Dissertation, Universitat de Valencia. Accessed July 16, 2019. http://hdl.handle.net/10550/66945.

MLA Handbook (7th Edition):

Calvo Hoyas, Paula. “Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio del potencial papel terapéutico del RNA de doble cadena (pIC-PEI) .” 2018. Web. 16 Jul 2019.

Vancouver:

Calvo Hoyas P. Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio del potencial papel terapéutico del RNA de doble cadena (pIC-PEI) . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10550/66945.

Council of Science Editors:

Calvo Hoyas P. Establecimiento y optimización de modelo murino de la patología ginecológica endometriosis utilizando xenografts de endometrio humano para el estudio del potencial papel terapéutico del RNA de doble cadena (pIC-PEI) . [Doctoral Dissertation]. Universitat de Valencia; 2018. Available from: http://hdl.handle.net/10550/66945


University of Edinburgh

8. Ogley, Robert James. Role of WT1 in Ischaemic Angiogenesis.

Degree: PhD, 2018, University of Edinburgh

 Ischaemia causes irreversible tissue damage in cardiovascular disease. Since regenerative angiogenesis fails to consistently induce sufficient reperfusion to facilitate repair, targeted manipulation of angiogenesis is… (more)

Subjects/Keywords: angiogenesis; Wilms' tumour suppressor; WT1; transcription factors

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APA (6th Edition):

Ogley, R. J. (2018). Role of WT1 in Ischaemic Angiogenesis. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/31555

Chicago Manual of Style (16th Edition):

Ogley, Robert James. “Role of WT1 in Ischaemic Angiogenesis.” 2018. Doctoral Dissertation, University of Edinburgh. Accessed July 16, 2019. http://hdl.handle.net/1842/31555.

MLA Handbook (7th Edition):

Ogley, Robert James. “Role of WT1 in Ischaemic Angiogenesis.” 2018. Web. 16 Jul 2019.

Vancouver:

Ogley RJ. Role of WT1 in Ischaemic Angiogenesis. [Internet] [Doctoral dissertation]. University of Edinburgh; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1842/31555.

Council of Science Editors:

Ogley RJ. Role of WT1 in Ischaemic Angiogenesis. [Doctoral Dissertation]. University of Edinburgh; 2018. Available from: http://hdl.handle.net/1842/31555


Rice University

9. Clements, Thomas P. A Rice CRISPy Treat: Improving CRISPR-Cas9 gene editing in the zebrafish to facilitate analysis of genes implicated in neural angiogenesis in an F0 screen.

Degree: PhD, Natural Sciences, 2018, Rice University

 Scientists are eager for novel mutants to study gene function, evolutionary relationships, and even perform drug screens. Zebrafish are a well-established model for scientific research… (more)

Subjects/Keywords: zebrafish; crispr; exocas9; f0; angiogenesis; hemorrhage

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APA (6th Edition):

Clements, T. P. (2018). A Rice CRISPy Treat: Improving CRISPR-Cas9 gene editing in the zebrafish to facilitate analysis of genes implicated in neural angiogenesis in an F0 screen. (Doctoral Dissertation). Rice University. Retrieved from http://hdl.handle.net/1911/105724

Chicago Manual of Style (16th Edition):

Clements, Thomas P. “A Rice CRISPy Treat: Improving CRISPR-Cas9 gene editing in the zebrafish to facilitate analysis of genes implicated in neural angiogenesis in an F0 screen.” 2018. Doctoral Dissertation, Rice University. Accessed July 16, 2019. http://hdl.handle.net/1911/105724.

MLA Handbook (7th Edition):

Clements, Thomas P. “A Rice CRISPy Treat: Improving CRISPR-Cas9 gene editing in the zebrafish to facilitate analysis of genes implicated in neural angiogenesis in an F0 screen.” 2018. Web. 16 Jul 2019.

Vancouver:

Clements TP. A Rice CRISPy Treat: Improving CRISPR-Cas9 gene editing in the zebrafish to facilitate analysis of genes implicated in neural angiogenesis in an F0 screen. [Internet] [Doctoral dissertation]. Rice University; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1911/105724.

Council of Science Editors:

Clements TP. A Rice CRISPy Treat: Improving CRISPR-Cas9 gene editing in the zebrafish to facilitate analysis of genes implicated in neural angiogenesis in an F0 screen. [Doctoral Dissertation]. Rice University; 2018. Available from: http://hdl.handle.net/1911/105724


University of Toronto

10. Kuzmic, Nikola. Modelling of Endothelial Cell Migration and Angiogenesis in Microfluidic Cell Culture Systems.

Degree: 2018, University of Toronto

Tumour-induced angiogenesis involves growth of new blood vessels from existing vasculature in response to signals induced by the undernourished part of tumour tissue. Due to… (more)

Subjects/Keywords: angiogenesis; computational modelling; endothelium; vasculature; 0541

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APA (6th Edition):

Kuzmic, N. (2018). Modelling of Endothelial Cell Migration and Angiogenesis in Microfluidic Cell Culture Systems. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91580

Chicago Manual of Style (16th Edition):

Kuzmic, Nikola. “Modelling of Endothelial Cell Migration and Angiogenesis in Microfluidic Cell Culture Systems.” 2018. Masters Thesis, University of Toronto. Accessed July 16, 2019. http://hdl.handle.net/1807/91580.

MLA Handbook (7th Edition):

Kuzmic, Nikola. “Modelling of Endothelial Cell Migration and Angiogenesis in Microfluidic Cell Culture Systems.” 2018. Web. 16 Jul 2019.

Vancouver:

Kuzmic N. Modelling of Endothelial Cell Migration and Angiogenesis in Microfluidic Cell Culture Systems. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1807/91580.

Council of Science Editors:

Kuzmic N. Modelling of Endothelial Cell Migration and Angiogenesis in Microfluidic Cell Culture Systems. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91580


Texas Tech University

11. -0519-8840. Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells.

Degree: MS, Nutritional Sciences, 2018, Texas Tech University

 Background: Breast cancer is considered as the most prevalent cancer in women world-wide. The average risk for a woman developing breast cancer in her life… (more)

Subjects/Keywords: Breast cancer; Parthenolide; Apoptosis; EMT; Angiogenesis

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APA (6th Edition):

-0519-8840. (2018). Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells. (Masters Thesis). Texas Tech University. Retrieved from http://hdl.handle.net/2346/74400

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-0519-8840. “Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells.” 2018. Masters Thesis, Texas Tech University. Accessed July 16, 2019. http://hdl.handle.net/2346/74400.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-0519-8840. “Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells.” 2018. Web. 16 Jul 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-0519-8840. Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells. [Internet] [Masters thesis]. Texas Tech University; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2346/74400.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-0519-8840. Studying the anticancer properties of Parthenolide (PTL) in MCF-7 breast cancer cells. [Masters Thesis]. Texas Tech University; 2018. Available from: http://hdl.handle.net/2346/74400

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Helsinki

12. Kallio, Pauliina; Jokinen, Elina; Das, Suvendu; Högström, Jenny; Heino, Sarika; Lähde, Marianne. Angiopoietin-2 blocking antibodies improve tumor growth inhibition and survival in mice treated with low doses of radiation.

Degree: Medicinska fakulteten, 2018, University of Helsinki

 Radiation induced tumor cell death is strongly dependent on oxygen. As abnormal tumor vasculature promotes tumor hypoxia, drugs that induce vascular normalization, such as the… (more)

Subjects/Keywords: Angiopoietin-2; radiotherapy; radiotherapy resistance; angiogenesis; radiation sensitizer; Angiopoietin-2; radiotherapy; radiotherapy resistance; angiogenesis; radiation sensitizer

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APA (6th Edition):

Kallio, Pauliina; Jokinen, Elina; Das, Suvendu; Högström, Jenny; Heino, Sarika; Lähde, M. (2018). Angiopoietin-2 blocking antibodies improve tumor growth inhibition and survival in mice treated with low doses of radiation. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/234044

Chicago Manual of Style (16th Edition):

Kallio, Pauliina; Jokinen, Elina; Das, Suvendu; Högström, Jenny; Heino, Sarika; Lähde, Marianne. “Angiopoietin-2 blocking antibodies improve tumor growth inhibition and survival in mice treated with low doses of radiation.” 2018. Masters Thesis, University of Helsinki. Accessed July 16, 2019. http://hdl.handle.net/10138/234044.

MLA Handbook (7th Edition):

Kallio, Pauliina; Jokinen, Elina; Das, Suvendu; Högström, Jenny; Heino, Sarika; Lähde, Marianne. “Angiopoietin-2 blocking antibodies improve tumor growth inhibition and survival in mice treated with low doses of radiation.” 2018. Web. 16 Jul 2019.

Vancouver:

Kallio, Pauliina; Jokinen, Elina; Das, Suvendu; Högström, Jenny; Heino, Sarika; Lähde M. Angiopoietin-2 blocking antibodies improve tumor growth inhibition and survival in mice treated with low doses of radiation. [Internet] [Masters thesis]. University of Helsinki; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10138/234044.

Council of Science Editors:

Kallio, Pauliina; Jokinen, Elina; Das, Suvendu; Högström, Jenny; Heino, Sarika; Lähde M. Angiopoietin-2 blocking antibodies improve tumor growth inhibition and survival in mice treated with low doses of radiation. [Masters Thesis]. University of Helsinki; 2018. Available from: http://hdl.handle.net/10138/234044

13. Zhao, Weilin. RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma.

Degree: 博士(医学), 2017, Mie University / 三重大学

Background: Nasopharyngeal carcinoma (NPC) is a malignancy of the head and neck that is prevalent in Southeast Asia and southern China. Recent studies in epigenetics… (more)

Subjects/Keywords: RERG; Nasopharyngeal carcinoma; Tumor suppressor gene; DNA methylation; Angiogenesis

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APA (6th Edition):

Zhao, W. (2017). RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00017324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhao, Weilin. “RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma.” 2017. Thesis, Mie University / 三重大学. Accessed July 16, 2019. http://hdl.handle.net/10076/00017324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhao, Weilin. “RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma.” 2017. Web. 16 Jul 2019.

Vancouver:

Zhao W. RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10076/00017324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhao W. RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00017324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

14. Chen, Zih-ying. Loss of lnx1 impairs zebrafish vascular development mediated by VEGF and BMP pathways.

Degree: Master, Biological Sciences, 2017, NSYSU

 The establishment of blood vessels is important for embryo growth and survival in vertebrate. Using zebrafish as a model organism to study vascular development has… (more)

Subjects/Keywords: lnx1; CVP, caudal vein plexus; ISV, intersegmental vessel; angiogenesis; zebrafish

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Z. (2017). Loss of lnx1 impairs zebrafish vascular development mediated by VEGF and BMP pathways. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115941

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Zih-ying. “Loss of lnx1 impairs zebrafish vascular development mediated by VEGF and BMP pathways.” 2017. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115941.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Zih-ying. “Loss of lnx1 impairs zebrafish vascular development mediated by VEGF and BMP pathways.” 2017. Web. 16 Jul 2019.

Vancouver:

Chen Z. Loss of lnx1 impairs zebrafish vascular development mediated by VEGF and BMP pathways. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115941.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Z. Loss of lnx1 impairs zebrafish vascular development mediated by VEGF and BMP pathways. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716117-115941

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

15. Hong, Jhu-ying. Modulation of atopic dermatitis by modifying marine-derived compound.

Degree: Master, Marine Biotechnology and Resources, 2017, NSYSU

 Atopic dermatitis (AD) is one of the most common chronic inflammatory skin diseases. Although the detailed mechanisms of AD remain unclear, some studies indicated that… (more)

Subjects/Keywords: anti-inflammation; anti-angiogenesis; anti-oxidation; Atopic dermatitis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hong, J. (2017). Modulation of atopic dermatitis by modifying marine-derived compound. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814117-100024

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hong, Jhu-ying. “Modulation of atopic dermatitis by modifying marine-derived compound.” 2017. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814117-100024.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hong, Jhu-ying. “Modulation of atopic dermatitis by modifying marine-derived compound.” 2017. Web. 16 Jul 2019.

Vancouver:

Hong J. Modulation of atopic dermatitis by modifying marine-derived compound. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814117-100024.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hong J. Modulation of atopic dermatitis by modifying marine-derived compound. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814117-100024

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

16. Arpino, Giovanni-Michele. Angiogenesis And Neo-Microcirculatory Function In Diseased Tissue Revealed By Intravital Microscopy.

Degree: 2017, University of Western Ontario

Angiogenesis is the process of generating new blood microvessels. In adults, angiogenesis is fundamental to tumor biology and also to tissues rendered ischemic from vascular… (more)

Subjects/Keywords: angiogenesis; microcirculation; ischemia; intravital microscopy; skeletal muscle; cancer; Medical Biophysics

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APA (6th Edition):

Arpino, G. (2017). Angiogenesis And Neo-Microcirculatory Function In Diseased Tissue Revealed By Intravital Microscopy. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arpino, Giovanni-Michele. “Angiogenesis And Neo-Microcirculatory Function In Diseased Tissue Revealed By Intravital Microscopy.” 2017. Thesis, University of Western Ontario. Accessed July 16, 2019. https://ir.lib.uwo.ca/etd/4731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arpino, Giovanni-Michele. “Angiogenesis And Neo-Microcirculatory Function In Diseased Tissue Revealed By Intravital Microscopy.” 2017. Web. 16 Jul 2019.

Vancouver:

Arpino G. Angiogenesis And Neo-Microcirculatory Function In Diseased Tissue Revealed By Intravital Microscopy. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2019 Jul 16]. Available from: https://ir.lib.uwo.ca/etd/4731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arpino G. Angiogenesis And Neo-Microcirculatory Function In Diseased Tissue Revealed By Intravital Microscopy. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

17. Kahramanoğlu, Zeynep Gülsüm. Mechanisms underlying chemotherapy-induced vascular proliferation in ovarian cancer.

Degree: 2017, University of Western Ontario

 Ovarian cancer is a leading cause of gynecological cancer-related death in Canadian women. Ovarian cancer is managed through surgical cytoreduction and carboplatin-based chemotherapy. Unfortunately, most… (more)

Subjects/Keywords: Serous ovarian cancer; angiogenesis; chemotherapy; carboplatin; vascular proliferation; Neoplasms

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APA (6th Edition):

Kahramanoğlu, Z. G. (2017). Mechanisms underlying chemotherapy-induced vascular proliferation in ovarian cancer. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4709

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kahramanoğlu, Zeynep Gülsüm. “Mechanisms underlying chemotherapy-induced vascular proliferation in ovarian cancer.” 2017. Thesis, University of Western Ontario. Accessed July 16, 2019. https://ir.lib.uwo.ca/etd/4709.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kahramanoğlu, Zeynep Gülsüm. “Mechanisms underlying chemotherapy-induced vascular proliferation in ovarian cancer.” 2017. Web. 16 Jul 2019.

Vancouver:

Kahramanoğlu ZG. Mechanisms underlying chemotherapy-induced vascular proliferation in ovarian cancer. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2019 Jul 16]. Available from: https://ir.lib.uwo.ca/etd/4709.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kahramanoğlu ZG. Mechanisms underlying chemotherapy-induced vascular proliferation in ovarian cancer. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4709

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

18. Marí Alexandre, Josep. Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for diagnosis and prognosis of endometriosis .

Degree: 2018, Universitat de Valencia

 Introduction: Endometriosis is an oestrogen-dependent inflammatory disorder defined by the presence of endometrial-like tissue in ectopic locations, which limits the quality of life of affected… (more)

Subjects/Keywords: endometriosis; epigenetics; miRNAs; DNA methylation; peritoneal fluid; angiogenesis; proteolysis

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APA (6th Edition):

Marí Alexandre, J. (2018). Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for diagnosis and prognosis of endometriosis . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/66183

Chicago Manual of Style (16th Edition):

Marí Alexandre, Josep. “Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for diagnosis and prognosis of endometriosis .” 2018. Doctoral Dissertation, Universitat de Valencia. Accessed July 16, 2019. http://hdl.handle.net/10550/66183.

MLA Handbook (7th Edition):

Marí Alexandre, Josep. “Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for diagnosis and prognosis of endometriosis .” 2018. Web. 16 Jul 2019.

Vancouver:

Marí Alexandre J. Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for diagnosis and prognosis of endometriosis . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10550/66183.

Council of Science Editors:

Marí Alexandre J. Characterization of a profile of epigenetic alterations involved in the aetiopathogenesis of endometriosis. Validation of molecular biomarkers for diagnosis and prognosis of endometriosis . [Doctoral Dissertation]. Universitat de Valencia; 2018. Available from: http://hdl.handle.net/10550/66183


Boston University

19. Chong, Brian Sung Ho. The role of glutaredoxin-1 on B16F0 melanoma growth and angiogenesis in diet-induced diabetic mice.

Degree: MS, Medical Sciences, 2018, Boston University

 OBJECTIVES: Recent studies have elucidated that diabetes mellitus (DM) patients exhibit an accelerated tumor progression, but the mechanism of its regulation is not yet fully… (more)

Subjects/Keywords: Medicine; Angiogenesis; Cancer; Diabetes; Glutaredoxin-1; Melanoma; Redox biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chong, B. S. H. (2018). The role of glutaredoxin-1 on B16F0 melanoma growth and angiogenesis in diet-induced diabetic mice. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/31165

Chicago Manual of Style (16th Edition):

Chong, Brian Sung Ho. “The role of glutaredoxin-1 on B16F0 melanoma growth and angiogenesis in diet-induced diabetic mice.” 2018. Masters Thesis, Boston University. Accessed July 16, 2019. http://hdl.handle.net/2144/31165.

MLA Handbook (7th Edition):

Chong, Brian Sung Ho. “The role of glutaredoxin-1 on B16F0 melanoma growth and angiogenesis in diet-induced diabetic mice.” 2018. Web. 16 Jul 2019.

Vancouver:

Chong BSH. The role of glutaredoxin-1 on B16F0 melanoma growth and angiogenesis in diet-induced diabetic mice. [Internet] [Masters thesis]. Boston University; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2144/31165.

Council of Science Editors:

Chong BSH. The role of glutaredoxin-1 on B16F0 melanoma growth and angiogenesis in diet-induced diabetic mice. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/31165


University of Ottawa

20. Whitcomb, Elizabeth Jamieson. Identification of GATA4 Regulatory Mechanisms of Heart Development and Disease .

Degree: 2019, University of Ottawa

 The development and function of the heart is governed by a conserved set of transcription factors (TFs) that regulate gene expression in a cell-type, time… (more)

Subjects/Keywords: GATA4; Transcription Factors; Angiogenesis; Congenital Heart Disease; Heart Failure; Hypertrophy

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APA (6th Edition):

Whitcomb, E. J. (2019). Identification of GATA4 Regulatory Mechanisms of Heart Development and Disease . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/38830

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Whitcomb, Elizabeth Jamieson. “Identification of GATA4 Regulatory Mechanisms of Heart Development and Disease .” 2019. Thesis, University of Ottawa. Accessed July 16, 2019. http://hdl.handle.net/10393/38830.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Whitcomb, Elizabeth Jamieson. “Identification of GATA4 Regulatory Mechanisms of Heart Development and Disease .” 2019. Web. 16 Jul 2019.

Vancouver:

Whitcomb EJ. Identification of GATA4 Regulatory Mechanisms of Heart Development and Disease . [Internet] [Thesis]. University of Ottawa; 2019. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10393/38830.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Whitcomb EJ. Identification of GATA4 Regulatory Mechanisms of Heart Development and Disease . [Thesis]. University of Ottawa; 2019. Available from: http://hdl.handle.net/10393/38830

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

21. Bocci, Matteo. Growth factor signaling in the breast tumor microenvironment.

Degree: 2018, University of Lund

 Cancer represents a collection of malignancies characterized by an aberrant expansion of cells. This unrestrained growth is the result of the acquisition of several pro-survival… (more)

Subjects/Keywords: Breast cancer; Tumor microenvironment; Angiogenesis; Cancer-associated fibroblasts; PDGF-C; ALK1

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APA (6th Edition):

Bocci, M. (2018). Growth factor signaling in the breast tumor microenvironment. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/8006b63f-18c4-45cf-98c0-169c6fca798b ; http://portal.research.lu.se/ws/files/53404207/Matteo_Bocci_WEBB.pdf

Chicago Manual of Style (16th Edition):

Bocci, Matteo. “Growth factor signaling in the breast tumor microenvironment.” 2018. Doctoral Dissertation, University of Lund. Accessed July 16, 2019. http://lup.lub.lu.se/record/8006b63f-18c4-45cf-98c0-169c6fca798b ; http://portal.research.lu.se/ws/files/53404207/Matteo_Bocci_WEBB.pdf.

MLA Handbook (7th Edition):

Bocci, Matteo. “Growth factor signaling in the breast tumor microenvironment.” 2018. Web. 16 Jul 2019.

Vancouver:

Bocci M. Growth factor signaling in the breast tumor microenvironment. [Internet] [Doctoral dissertation]. University of Lund; 2018. [cited 2019 Jul 16]. Available from: http://lup.lub.lu.se/record/8006b63f-18c4-45cf-98c0-169c6fca798b ; http://portal.research.lu.se/ws/files/53404207/Matteo_Bocci_WEBB.pdf.

Council of Science Editors:

Bocci M. Growth factor signaling in the breast tumor microenvironment. [Doctoral Dissertation]. University of Lund; 2018. Available from: http://lup.lub.lu.se/record/8006b63f-18c4-45cf-98c0-169c6fca798b ; http://portal.research.lu.se/ws/files/53404207/Matteo_Bocci_WEBB.pdf


University of Minnesota

22. Verma, Mayank. Cross-talk between the skeletal muscle stem cells and endothelial cells.

Degree: PhD, Integrative Biology and Physiology, 2018, University of Minnesota

 Duchenne muscular dystrophy (DMD) is a progressive neurodegenerative muscle disease caused by the absence of the dystrophin protein. While the muscle develops normally, it is… (more)

Subjects/Keywords: Angiogenesis; capillary; Satellite cell; Skeletal muscle; Stem cell niche; VEGF

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APA (6th Edition):

Verma, M. (2018). Cross-talk between the skeletal muscle stem cells and endothelial cells. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/202120

Chicago Manual of Style (16th Edition):

Verma, Mayank. “Cross-talk between the skeletal muscle stem cells and endothelial cells.” 2018. Doctoral Dissertation, University of Minnesota. Accessed July 16, 2019. http://hdl.handle.net/11299/202120.

MLA Handbook (7th Edition):

Verma, Mayank. “Cross-talk between the skeletal muscle stem cells and endothelial cells.” 2018. Web. 16 Jul 2019.

Vancouver:

Verma M. Cross-talk between the skeletal muscle stem cells and endothelial cells. [Internet] [Doctoral dissertation]. University of Minnesota; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/11299/202120.

Council of Science Editors:

Verma M. Cross-talk between the skeletal muscle stem cells and endothelial cells. [Doctoral Dissertation]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/202120


University of Cambridge

23. Tulkki, Valtteri Heikki Juhani. Oncostatin M Receptor Overexpression Promotes Tumour Progression in Squamous Cell Carcinoma, via Hypoxia Signalling and Multiple Effects on the Tumour Microenvironment .

Degree: 2018, University of Cambridge

 Cervical cancer still represents the fourth most common cause of cancer deaths in women worldwide. Human papilloma virus (HPV) infection plays a role in cervical… (more)

Subjects/Keywords: Cervical Cancer; Tumour microenvironment; OSM; OSMR; STAT3; M2 macrophages; Angiogenesis; EMT

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APA (6th Edition):

Tulkki, V. H. J. (2018). Oncostatin M Receptor Overexpression Promotes Tumour Progression in Squamous Cell Carcinoma, via Hypoxia Signalling and Multiple Effects on the Tumour Microenvironment . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/275416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tulkki, Valtteri Heikki Juhani. “Oncostatin M Receptor Overexpression Promotes Tumour Progression in Squamous Cell Carcinoma, via Hypoxia Signalling and Multiple Effects on the Tumour Microenvironment .” 2018. Thesis, University of Cambridge. Accessed July 16, 2019. https://www.repository.cam.ac.uk/handle/1810/275416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tulkki, Valtteri Heikki Juhani. “Oncostatin M Receptor Overexpression Promotes Tumour Progression in Squamous Cell Carcinoma, via Hypoxia Signalling and Multiple Effects on the Tumour Microenvironment .” 2018. Web. 16 Jul 2019.

Vancouver:

Tulkki VHJ. Oncostatin M Receptor Overexpression Promotes Tumour Progression in Squamous Cell Carcinoma, via Hypoxia Signalling and Multiple Effects on the Tumour Microenvironment . [Internet] [Thesis]. University of Cambridge; 2018. [cited 2019 Jul 16]. Available from: https://www.repository.cam.ac.uk/handle/1810/275416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tulkki VHJ. Oncostatin M Receptor Overexpression Promotes Tumour Progression in Squamous Cell Carcinoma, via Hypoxia Signalling and Multiple Effects on the Tumour Microenvironment . [Thesis]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/275416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

24. Hedhli, Jamila. A multimodal lens into vascular recovery in a preclinical model of peripheral arterial disease.

Degree: PhD, Bioengineering, 2018, University of Illinois – Urbana-Champaign

 In the week of February 2nd (21 days into gestation) my parents Ali, and Aicha heard my first heart beat. This shouldn't be a surprise… (more)

Subjects/Keywords: angiogenesis; Stem-Cell; peripheral vascular disease; Imaging; Diabetes

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APA (6th Edition):

Hedhli, J. (2018). A multimodal lens into vascular recovery in a preclinical model of peripheral arterial disease. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/102382

Chicago Manual of Style (16th Edition):

Hedhli, Jamila. “A multimodal lens into vascular recovery in a preclinical model of peripheral arterial disease.” 2018. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed July 16, 2019. http://hdl.handle.net/2142/102382.

MLA Handbook (7th Edition):

Hedhli, Jamila. “A multimodal lens into vascular recovery in a preclinical model of peripheral arterial disease.” 2018. Web. 16 Jul 2019.

Vancouver:

Hedhli J. A multimodal lens into vascular recovery in a preclinical model of peripheral arterial disease. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2142/102382.

Council of Science Editors:

Hedhli J. A multimodal lens into vascular recovery in a preclinical model of peripheral arterial disease. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2018. Available from: http://hdl.handle.net/2142/102382


University of Texas – Austin

25. -9024-5383. Glypican-1 proteoliposomes enhance growth factor activity for therapeutic angiogenesis.

Degree: Biomedical Engineering, 2018, University of Texas – Austin

 Peripheral arterial disease affects more than 27 million patients in the United States. PAD can lead to peripheral limb ischemia and result in non-healing foot… (more)

Subjects/Keywords: Glypican-1; FGF-2; VEGF; Ischemia; Angiogenesis; Growth factor

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APA (6th Edition):

-9024-5383. (2018). Glypican-1 proteoliposomes enhance growth factor activity for therapeutic angiogenesis. (Thesis). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/68372

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

-9024-5383. “Glypican-1 proteoliposomes enhance growth factor activity for therapeutic angiogenesis.” 2018. Thesis, University of Texas – Austin. Accessed July 16, 2019. http://hdl.handle.net/2152/68372.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

-9024-5383. “Glypican-1 proteoliposomes enhance growth factor activity for therapeutic angiogenesis.” 2018. Web. 16 Jul 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-9024-5383. Glypican-1 proteoliposomes enhance growth factor activity for therapeutic angiogenesis. [Internet] [Thesis]. University of Texas – Austin; 2018. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2152/68372.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

-9024-5383. Glypican-1 proteoliposomes enhance growth factor activity for therapeutic angiogenesis. [Thesis]. University of Texas – Austin; 2018. Available from: http://hdl.handle.net/2152/68372

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

26. Zhou, Jun-Qing. Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development.

Degree: Master, Biological Sciences, 2018, NSYSU

 Genetic programs and signaling pathways are required for proper growth and patterning of blood vessels. Zebrafish is a powerful vertebrate model organism to study genetic… (more)

Subjects/Keywords: angiogenesis; zebrafish; ect2; wasf1; VEGF; BMP; ISV; CVP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, J. (2018). Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Jun-Qing. “Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development.” 2018. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Jun-Qing. “Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development.” 2018. Web. 16 Jul 2019.

Vancouver:

Zhou J. Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development. [Internet] [Thesis]. NSYSU; 2018. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou J. Study the function of GTPase related proteins wasf1 and ect2 in zebrafish vascular development. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727118-130736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wright State University

27. Ye, Qinmao. Exosomes Released from Multiple Myeloma Cells Influence the Angiogenic Function of Endothelial Cells by Regulating MicroRNA-29b.

Degree: MS, Pharmacology and Toxicology, 2018, Wright State University

 Multiple myeloma is a hematological malignancy characterized by clonal proliferation of plasma cells generally caused by chromosomal abnormalities. It occurs in the bone marrow, which… (more)

Subjects/Keywords: Biology; Biomedical Research; Pharmacology; Exosomes; MicroRNA; Multiple Myeloma; Endothelial cells; Angiogenesis

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APA (6th Edition):

Ye, Q. (2018). Exosomes Released from Multiple Myeloma Cells Influence the Angiogenic Function of Endothelial Cells by Regulating MicroRNA-29b. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1532474227694747

Chicago Manual of Style (16th Edition):

Ye, Qinmao. “Exosomes Released from Multiple Myeloma Cells Influence the Angiogenic Function of Endothelial Cells by Regulating MicroRNA-29b.” 2018. Masters Thesis, Wright State University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1532474227694747.

MLA Handbook (7th Edition):

Ye, Qinmao. “Exosomes Released from Multiple Myeloma Cells Influence the Angiogenic Function of Endothelial Cells by Regulating MicroRNA-29b.” 2018. Web. 16 Jul 2019.

Vancouver:

Ye Q. Exosomes Released from Multiple Myeloma Cells Influence the Angiogenic Function of Endothelial Cells by Regulating MicroRNA-29b. [Internet] [Masters thesis]. Wright State University; 2018. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1532474227694747.

Council of Science Editors:

Ye Q. Exosomes Released from Multiple Myeloma Cells Influence the Angiogenic Function of Endothelial Cells by Regulating MicroRNA-29b. [Masters Thesis]. Wright State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1532474227694747


NSYSU

28. Chen, Yi-Ting. Characterization of the roles of Connexin 32 in zebrafish vascular development.

Degree: Master, Biological Sciences, 2018, NSYSU

 The process of vascular development in vertebrates are mainly divided into vasculogenesis and angiogenesis. We previously identified the transcription factors Islet2 (isl2) and Nr2f1b were… (more)

Subjects/Keywords: VEGF signal pathway; CVP; ISV; Connexin; zebrafish; angiogenesis; BMP signal pathway

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Y. (2018). Characterization of the roles of Connexin 32 in zebrafish vascular development. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0707118-155606

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Yi-Ting. “Characterization of the roles of Connexin 32 in zebrafish vascular development.” 2018. Thesis, NSYSU. Accessed July 16, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0707118-155606.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Yi-Ting. “Characterization of the roles of Connexin 32 in zebrafish vascular development.” 2018. Web. 16 Jul 2019.

Vancouver:

Chen Y. Characterization of the roles of Connexin 32 in zebrafish vascular development. [Internet] [Thesis]. NSYSU; 2018. [cited 2019 Jul 16]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0707118-155606.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Y. Characterization of the roles of Connexin 32 in zebrafish vascular development. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0707118-155606

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

29. Zakhari, Joseph Samir. Tissue engineered micro and macrovasculature utilizing stromal vascular fraction.

Degree: PhD, 2018, University of Louisville

  This dissertation describes the use of stromal vascular fraction to tissue engineer 3D microvasculature and macrovasculature. Stromal vascular fraction is an easily isolatable cell… (more)

Subjects/Keywords: stromal vascular fraction; angiogenesis; tissue engineering; microvessels; electrospinning; bioprinting; Medical Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zakhari, J. S. (2018). Tissue engineered micro and macrovasculature utilizing stromal vascular fraction. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2974 ; https://ir.library.louisville.edu/etd/2974

Chicago Manual of Style (16th Edition):

Zakhari, Joseph Samir. “Tissue engineered micro and macrovasculature utilizing stromal vascular fraction.” 2018. Doctoral Dissertation, University of Louisville. Accessed July 16, 2019. 10.18297/etd/2974 ; https://ir.library.louisville.edu/etd/2974.

MLA Handbook (7th Edition):

Zakhari, Joseph Samir. “Tissue engineered micro and macrovasculature utilizing stromal vascular fraction.” 2018. Web. 16 Jul 2019.

Vancouver:

Zakhari JS. Tissue engineered micro and macrovasculature utilizing stromal vascular fraction. [Internet] [Doctoral dissertation]. University of Louisville; 2018. [cited 2019 Jul 16]. Available from: 10.18297/etd/2974 ; https://ir.library.louisville.edu/etd/2974.

Council of Science Editors:

Zakhari JS. Tissue engineered micro and macrovasculature utilizing stromal vascular fraction. [Doctoral Dissertation]. University of Louisville; 2018. Available from: 10.18297/etd/2974 ; https://ir.library.louisville.edu/etd/2974

30. TAN EVAN. ROLE OF JAG1-EXTRACELLULAR VESICLES ON ANGIOGENESIS.

Degree: 2017, National University of Singapore

Subjects/Keywords: Angiogenesis; Notch signalling; Extracellular vesicles

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

EVAN, T. (2017). ROLE OF JAG1-EXTRACELLULAR VESICLES ON ANGIOGENESIS. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/139717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

EVAN, TAN. “ROLE OF JAG1-EXTRACELLULAR VESICLES ON ANGIOGENESIS.” 2017. Thesis, National University of Singapore. Accessed July 16, 2019. http://scholarbank.nus.edu.sg/handle/10635/139717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

EVAN, TAN. “ROLE OF JAG1-EXTRACELLULAR VESICLES ON ANGIOGENESIS.” 2017. Web. 16 Jul 2019.

Vancouver:

EVAN T. ROLE OF JAG1-EXTRACELLULAR VESICLES ON ANGIOGENESIS. [Internet] [Thesis]. National University of Singapore; 2017. [cited 2019 Jul 16]. Available from: http://scholarbank.nus.edu.sg/handle/10635/139717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

EVAN T. ROLE OF JAG1-EXTRACELLULAR VESICLES ON ANGIOGENESIS. [Thesis]. National University of Singapore; 2017. Available from: http://scholarbank.nus.edu.sg/handle/10635/139717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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