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You searched for subject:(Amyloid beta). Showing records 1 – 30 of 523 total matches.

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University of Manchester

1. Moreth, Jens. Characterisation of different amyloid-ß aggregates in Alzheimer's Disease.

Degree: 2012, University of Manchester

 Alzheimer’s disease (AD) is the most common form of dementia, with more than 25 million people worldwide suffering this progressive intellectual failure. The disease was… (more)

Subjects/Keywords: Alzheimer's Disease; Amyloid beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moreth, J. (2012). Characterisation of different amyloid-ß aggregates in Alzheimer's Disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:164717

Chicago Manual of Style (16th Edition):

Moreth, Jens. “Characterisation of different amyloid-ß aggregates in Alzheimer's Disease.” 2012. Doctoral Dissertation, University of Manchester. Accessed September 26, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:164717.

MLA Handbook (7th Edition):

Moreth, Jens. “Characterisation of different amyloid-ß aggregates in Alzheimer's Disease.” 2012. Web. 26 Sep 2020.

Vancouver:

Moreth J. Characterisation of different amyloid-ß aggregates in Alzheimer's Disease. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Sep 26]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:164717.

Council of Science Editors:

Moreth J. Characterisation of different amyloid-ß aggregates in Alzheimer's Disease. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:164717


University of Manchester

2. Moreth, Jens. Characterisation of different amyloid-ß aggregates in Alzheimer's disease.

Degree: PhD, 2012, University of Manchester

 Alzheimer’s disease (AD) is the most common form of dementia, with more than 25 million people worldwide suffering this progressive intellectual failure. The disease was… (more)

Subjects/Keywords: 616.8; Alzheimer's Disease; Amyloid beta

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APA (6th Edition):

Moreth, J. (2012). Characterisation of different amyloid-ß aggregates in Alzheimer's disease. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/characterisation-of-different-amyloidss-aggregates-in-alzheimers-disease(88c7e7d8-dd08-4559-aa20-40f26950f0d5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632161

Chicago Manual of Style (16th Edition):

Moreth, Jens. “Characterisation of different amyloid-ß aggregates in Alzheimer's disease.” 2012. Doctoral Dissertation, University of Manchester. Accessed September 26, 2020. https://www.research.manchester.ac.uk/portal/en/theses/characterisation-of-different-amyloidss-aggregates-in-alzheimers-disease(88c7e7d8-dd08-4559-aa20-40f26950f0d5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632161.

MLA Handbook (7th Edition):

Moreth, Jens. “Characterisation of different amyloid-ß aggregates in Alzheimer's disease.” 2012. Web. 26 Sep 2020.

Vancouver:

Moreth J. Characterisation of different amyloid-ß aggregates in Alzheimer's disease. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Sep 26]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/characterisation-of-different-amyloidss-aggregates-in-alzheimers-disease(88c7e7d8-dd08-4559-aa20-40f26950f0d5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632161.

Council of Science Editors:

Moreth J. Characterisation of different amyloid-ß aggregates in Alzheimer's disease. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/characterisation-of-different-amyloidss-aggregates-in-alzheimers-disease(88c7e7d8-dd08-4559-aa20-40f26950f0d5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632161


University of Melbourne

3. JOHANSSEN, TIMOTHY. The Role of metals and Aβ in excitotoxicity and Alzheimer’s disease.

Degree: 2015, University of Melbourne

 Background: N-methyl-d-aspartate receptors (NMDARs) are ionotropic channels gated by the excitatory amino acid, glutamate. They play an essential role in synaptic plasticity, enhancing synaptic signal… (more)

Subjects/Keywords: Alzheimer's disease; beta amyloid; excitotoxicity

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APA (6th Edition):

JOHANSSEN, T. (2015). The Role of metals and Aβ in excitotoxicity and Alzheimer’s disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55550

Chicago Manual of Style (16th Edition):

JOHANSSEN, TIMOTHY. “The Role of metals and Aβ in excitotoxicity and Alzheimer’s disease.” 2015. Doctoral Dissertation, University of Melbourne. Accessed September 26, 2020. http://hdl.handle.net/11343/55550.

MLA Handbook (7th Edition):

JOHANSSEN, TIMOTHY. “The Role of metals and Aβ in excitotoxicity and Alzheimer’s disease.” 2015. Web. 26 Sep 2020.

Vancouver:

JOHANSSEN T. The Role of metals and Aβ in excitotoxicity and Alzheimer’s disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/11343/55550.

Council of Science Editors:

JOHANSSEN T. The Role of metals and Aβ in excitotoxicity and Alzheimer’s disease. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55550


University of Hawaii – Manoa

4. Lawrence, James Le Marchant. N-terminal beta amyloid fragments regulate nicotinic acetylcholine receptors.

Degree: 2015, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2014.

Soluble β-amyloid (Aβ) has been shown to regulate both presynaptic Ca2+ and synaptic plasticity. In particular picomolar concentrations… (more)

Subjects/Keywords: N-terminal beta amyloid fragments

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APA (6th Edition):

Lawrence, J. L. M. (2015). N-terminal beta amyloid fragments regulate nicotinic acetylcholine receptors. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/101105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lawrence, James Le Marchant. “N-terminal beta amyloid fragments regulate nicotinic acetylcholine receptors.” 2015. Thesis, University of Hawaii – Manoa. Accessed September 26, 2020. http://hdl.handle.net/10125/101105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lawrence, James Le Marchant. “N-terminal beta amyloid fragments regulate nicotinic acetylcholine receptors.” 2015. Web. 26 Sep 2020.

Vancouver:

Lawrence JLM. N-terminal beta amyloid fragments regulate nicotinic acetylcholine receptors. [Internet] [Thesis]. University of Hawaii – Manoa; 2015. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/10125/101105.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lawrence JLM. N-terminal beta amyloid fragments regulate nicotinic acetylcholine receptors. [Thesis]. University of Hawaii – Manoa; 2015. Available from: http://hdl.handle.net/10125/101105

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Yıldız, Sema. İnsan embriyonik böbrek 293 (hek293) hücrelerinde amiloid beta toksisitesine karşı ısı şoku proteini 70 (hsp70)'in rolü: The role of heat shock protein 70 (hsp70) in toxicity of beta amyloid in human embryonic kidney 293 (hek293) cells.

Degree: Fen Fakültesi, 2018, University of Ankara

 Bu çalışmada insan embriyonik böbrek hücre hattında (HEK 293) amiloid beta ile indüklenen sitotoksisite üzerine Hsp70 şaperonunun koruyucu rolünün incelenmesi amaçlanmıştır. Toksisite oluşturmak için HEK… (more)

Subjects/Keywords: Hsp70; HEK 293; Amiloid beta; Amyloid beta

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APA (6th Edition):

Yıldız, S. (2018). İnsan embriyonik böbrek 293 (hek293) hücrelerinde amiloid beta toksisitesine karşı ısı şoku proteini 70 (hsp70)'in rolü: The role of heat shock protein 70 (hsp70) in toxicity of beta amyloid in human embryonic kidney 293 (hek293) cells. (Masters Thesis). University of Ankara. Retrieved from http://hdl.handle.net/20.500.12575/69293

Chicago Manual of Style (16th Edition):

Yıldız, Sema. “İnsan embriyonik böbrek 293 (hek293) hücrelerinde amiloid beta toksisitesine karşı ısı şoku proteini 70 (hsp70)'in rolü: The role of heat shock protein 70 (hsp70) in toxicity of beta amyloid in human embryonic kidney 293 (hek293) cells.” 2018. Masters Thesis, University of Ankara. Accessed September 26, 2020. http://hdl.handle.net/20.500.12575/69293.

MLA Handbook (7th Edition):

Yıldız, Sema. “İnsan embriyonik böbrek 293 (hek293) hücrelerinde amiloid beta toksisitesine karşı ısı şoku proteini 70 (hsp70)'in rolü: The role of heat shock protein 70 (hsp70) in toxicity of beta amyloid in human embryonic kidney 293 (hek293) cells.” 2018. Web. 26 Sep 2020.

Vancouver:

Yıldız S. İnsan embriyonik böbrek 293 (hek293) hücrelerinde amiloid beta toksisitesine karşı ısı şoku proteini 70 (hsp70)'in rolü: The role of heat shock protein 70 (hsp70) in toxicity of beta amyloid in human embryonic kidney 293 (hek293) cells. [Internet] [Masters thesis]. University of Ankara; 2018. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/20.500.12575/69293.

Council of Science Editors:

Yıldız S. İnsan embriyonik böbrek 293 (hek293) hücrelerinde amiloid beta toksisitesine karşı ısı şoku proteini 70 (hsp70)'in rolü: The role of heat shock protein 70 (hsp70) in toxicity of beta amyloid in human embryonic kidney 293 (hek293) cells. [Masters Thesis]. University of Ankara; 2018. Available from: http://hdl.handle.net/20.500.12575/69293


Drexel University

6. Nguyen, Phuong Thi Tam. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.

Degree: 2017, Drexel University

The average person spends several hours a day behind the wheel of their vehicles, which are usually equipped with on-board computers capable of collecting real-time… (more)

Subjects/Keywords: Biology; Alzheimer's disease; Drosophila; Amyloid beta-protein; Amyloid beta-protein precursor

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APA (6th Edition):

Nguyen, P. T. T. (2017). Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Phuong Thi Tam. “Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.” 2017. Thesis, Drexel University. Accessed September 26, 2020. http://hdl.handle.net/1860/idea:7473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Phuong Thi Tam. “Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.” 2017. Web. 26 Sep 2020.

Vancouver:

Nguyen PTT. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. [Internet] [Thesis]. Drexel University; 2017. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1860/idea:7473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen PTT. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. [Thesis]. Drexel University; 2017. Available from: http://hdl.handle.net/1860/idea:7473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of the Western Cape

7. Teponnou, Gerard A. Kenfack. Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy .

Degree: 2016, University of the Western Cape

 The cascade of neurotoxic events involved in the pathogenesis of Alzheimer's disease may explain the inefficacy of currently available treatment based on acetylcholinesterase inhibitors (AChEI… (more)

Subjects/Keywords: Amyloid Beta; Cholinesterases; Alzheimer's disease; Multifunctional drugs; Amyloid beta-protein

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APA (6th Edition):

Teponnou, G. A. K. (2016). Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/5344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Teponnou, Gerard A Kenfack. “Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy .” 2016. Thesis, University of the Western Cape. Accessed September 26, 2020. http://hdl.handle.net/11394/5344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Teponnou, Gerard A Kenfack. “Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy .” 2016. Web. 26 Sep 2020.

Vancouver:

Teponnou GAK. Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy . [Internet] [Thesis]. University of the Western Cape; 2016. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/11394/5344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Teponnou GAK. Tacrine, trolox and tryptoline as lead compounds for the design and synthesis of multi-target drugs for Alzheimer's disease therapy . [Thesis]. University of the Western Cape; 2016. Available from: http://hdl.handle.net/11394/5344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

8. Chia, Sean Keng Rui. Restoring the balance in the kinetic network of Aβ aggregation as a drug discovery strategy for Alzheimer’s Disease.

Degree: PhD, 2020, University of Cambridge

 Alzheimer’s disease (AD) is the most prevalent cause of dementia that affects over 50 million people, and is associated strongly with the aggregation of the… (more)

Subjects/Keywords: Chemical Kinetics; Amyloid-β; Drug Discovery

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APA (6th Edition):

Chia, S. K. R. (2020). Restoring the balance in the kinetic network of Aβ aggregation as a drug discovery strategy for Alzheimer’s Disease. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301742

Chicago Manual of Style (16th Edition):

Chia, Sean Keng Rui. “Restoring the balance in the kinetic network of Aβ aggregation as a drug discovery strategy for Alzheimer’s Disease.” 2020. Doctoral Dissertation, University of Cambridge. Accessed September 26, 2020. https://www.repository.cam.ac.uk/handle/1810/301742.

MLA Handbook (7th Edition):

Chia, Sean Keng Rui. “Restoring the balance in the kinetic network of Aβ aggregation as a drug discovery strategy for Alzheimer’s Disease.” 2020. Web. 26 Sep 2020.

Vancouver:

Chia SKR. Restoring the balance in the kinetic network of Aβ aggregation as a drug discovery strategy for Alzheimer’s Disease. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Sep 26]. Available from: https://www.repository.cam.ac.uk/handle/1810/301742.

Council of Science Editors:

Chia SKR. Restoring the balance in the kinetic network of Aβ aggregation as a drug discovery strategy for Alzheimer’s Disease. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/301742


Massey University

9. Wang, Xiangli. Effects of environmental factors on heat-induced [beta]-lactoglobulin fibril formation : a thesis presented in partial fulfilment of the requirements for the degree of Master of Technology in Food Technology, Riddet Institute, Massey University, Palmerston North, New Zealand.

Degree: 2011, Massey University

 The heat-induced fibrillar aggregation of β-lactoglobulin was studied under various environmental conditions. The formation of β-lactoglobulin fibrils was monitored by Thioflavin T (ThT) fluorescence and… (more)

Subjects/Keywords: Milk proteins; Denaturation; Beta-lactoglobulin; Amyloid fibril; Thioflavin T fluorescence; TEM; β-Lactoglobulin

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APA (6th Edition):

Wang, X. (2011). Effects of environmental factors on heat-induced [beta]-lactoglobulin fibril formation : a thesis presented in partial fulfilment of the requirements for the degree of Master of Technology in Food Technology, Riddet Institute, Massey University, Palmerston North, New Zealand. (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/2227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Xiangli. “Effects of environmental factors on heat-induced [beta]-lactoglobulin fibril formation : a thesis presented in partial fulfilment of the requirements for the degree of Master of Technology in Food Technology, Riddet Institute, Massey University, Palmerston North, New Zealand. ” 2011. Thesis, Massey University. Accessed September 26, 2020. http://hdl.handle.net/10179/2227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Xiangli. “Effects of environmental factors on heat-induced [beta]-lactoglobulin fibril formation : a thesis presented in partial fulfilment of the requirements for the degree of Master of Technology in Food Technology, Riddet Institute, Massey University, Palmerston North, New Zealand. ” 2011. Web. 26 Sep 2020.

Vancouver:

Wang X. Effects of environmental factors on heat-induced [beta]-lactoglobulin fibril formation : a thesis presented in partial fulfilment of the requirements for the degree of Master of Technology in Food Technology, Riddet Institute, Massey University, Palmerston North, New Zealand. [Internet] [Thesis]. Massey University; 2011. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/10179/2227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang X. Effects of environmental factors on heat-induced [beta]-lactoglobulin fibril formation : a thesis presented in partial fulfilment of the requirements for the degree of Master of Technology in Food Technology, Riddet Institute, Massey University, Palmerston North, New Zealand. [Thesis]. Massey University; 2011. Available from: http://hdl.handle.net/10179/2227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

10. Ostrowski, Stephen M. Pleiotropic Mechanisms of Statin Action in Alzheimer's Disease.

Degree: PhD, Neurosciences, 2008, Case Western Reserve University

 Epidemiological evidence suggests that long term treatment with the cholesterol-lowering HMG-CoA reductase inhibitors, or statins, decreases the risk for developing Alzheimer’s Disease (AD). However, statin-mediated… (more)

Subjects/Keywords: Biology, Neuroscience; Alzheimers Disease; beta-amyloid; statins

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APA (6th Edition):

Ostrowski, S. M. (2008). Pleiotropic Mechanisms of Statin Action in Alzheimer's Disease. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1190669698

Chicago Manual of Style (16th Edition):

Ostrowski, Stephen M. “Pleiotropic Mechanisms of Statin Action in Alzheimer's Disease.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed September 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1190669698.

MLA Handbook (7th Edition):

Ostrowski, Stephen M. “Pleiotropic Mechanisms of Statin Action in Alzheimer's Disease.” 2008. Web. 26 Sep 2020.

Vancouver:

Ostrowski SM. Pleiotropic Mechanisms of Statin Action in Alzheimer's Disease. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2020 Sep 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1190669698.

Council of Science Editors:

Ostrowski SM. Pleiotropic Mechanisms of Statin Action in Alzheimer's Disease. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1190669698


University of California – San Diego

11. Alfonso, Stephanie. The Role of PICK1 and PKC alpha in Alzheimer’s Disease.

Degree: Neurosciences, 2016, University of California – San Diego

Beta amyloid (Aβ), a key component in the pathophysiology of Alzheimer’s disease (AD) is thought to target excitatory synapses early in the disease. We show… (more)

Subjects/Keywords: Neurosciences; Alzheimer's Disease; amyloid beta; synapses

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APA (6th Edition):

Alfonso, S. (2016). The Role of PICK1 and PKC alpha in Alzheimer’s Disease. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/5t5592jz

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alfonso, Stephanie. “The Role of PICK1 and PKC alpha in Alzheimer’s Disease.” 2016. Thesis, University of California – San Diego. Accessed September 26, 2020. http://www.escholarship.org/uc/item/5t5592jz.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alfonso, Stephanie. “The Role of PICK1 and PKC alpha in Alzheimer’s Disease.” 2016. Web. 26 Sep 2020.

Vancouver:

Alfonso S. The Role of PICK1 and PKC alpha in Alzheimer’s Disease. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2020 Sep 26]. Available from: http://www.escholarship.org/uc/item/5t5592jz.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alfonso S. The Role of PICK1 and PKC alpha in Alzheimer’s Disease. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/5t5592jz

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

12. Le, Kim Virginia. Effect of Chirality and Chimeras on Amyloid Aggregation.

Degree: Chemistry, 2014, University of California – Irvine

 This thesis reports the activity of macrocyclic β-sheets and their activity against amyloid aggregation. Macrocyclic D-amino acid β-sheets delay or suppress amyloid aggregation at substoichiometric… (more)

Subjects/Keywords: Chemistry; Amyloid; beta-sheet; Chirality; macrocycle

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APA (6th Edition):

Le, K. V. (2014). Effect of Chirality and Chimeras on Amyloid Aggregation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/65c721q0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Le, Kim Virginia. “Effect of Chirality and Chimeras on Amyloid Aggregation.” 2014. Thesis, University of California – Irvine. Accessed September 26, 2020. http://www.escholarship.org/uc/item/65c721q0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Le, Kim Virginia. “Effect of Chirality and Chimeras on Amyloid Aggregation.” 2014. Web. 26 Sep 2020.

Vancouver:

Le KV. Effect of Chirality and Chimeras on Amyloid Aggregation. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2020 Sep 26]. Available from: http://www.escholarship.org/uc/item/65c721q0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Le KV. Effect of Chirality and Chimeras on Amyloid Aggregation. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/65c721q0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

13. Collins, JM. The effect of traumatic brain injury in experimental models of Alzheimer’s disease.

Degree: 2016, University of Tasmania

 Traumatic brain injury (TBI) can cause persistent cognitive changes and ongoing neurodegeneration in the brain. Accumulating epidemiological and pathological evidence implicates TBI in the development… (more)

Subjects/Keywords: Brain injury; Alzheimer's disease; beta amyloid; plaques

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APA (6th Edition):

Collins, J. (2016). The effect of traumatic brain injury in experimental models of Alzheimer’s disease. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/23007/1/Collins_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Collins, JM. “The effect of traumatic brain injury in experimental models of Alzheimer’s disease.” 2016. Thesis, University of Tasmania. Accessed September 26, 2020. https://eprints.utas.edu.au/23007/1/Collins_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Collins, JM. “The effect of traumatic brain injury in experimental models of Alzheimer’s disease.” 2016. Web. 26 Sep 2020.

Vancouver:

Collins J. The effect of traumatic brain injury in experimental models of Alzheimer’s disease. [Internet] [Thesis]. University of Tasmania; 2016. [cited 2020 Sep 26]. Available from: https://eprints.utas.edu.au/23007/1/Collins_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Collins J. The effect of traumatic brain injury in experimental models of Alzheimer’s disease. [Thesis]. University of Tasmania; 2016. Available from: https://eprints.utas.edu.au/23007/1/Collins_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

14. Vorobyeva, Anna Gennadiy. Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis.

Degree: 2014, Drexel University

Alzheimer's disease (AD) is the most common form of dementia for which there is no cure. AD is characterized by progressive neurotoxicity resulting in cell… (more)

Subjects/Keywords: Biological sciences; Alzheimer's disease; Amyloid beta-protein

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vorobyeva, A. G. (2014). Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:6998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vorobyeva, Anna Gennadiy. “Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis.” 2014. Thesis, Drexel University. Accessed September 26, 2020. http://hdl.handle.net/1860/idea:6998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vorobyeva, Anna Gennadiy. “Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis.” 2014. Web. 26 Sep 2020.

Vancouver:

Vorobyeva AG. Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis. [Internet] [Thesis]. Drexel University; 2014. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1860/idea:6998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vorobyeva AG. Sonic Hedgehog Signaling Pathway in Amyloid Precursor Protein Trafficking and Proteolysis. [Thesis]. Drexel University; 2014. Available from: http://hdl.handle.net/1860/idea:6998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

15. Utreja, Suruchi. Calcineurin regulates APP metabolism: In vitro & In vivo studies.

Degree: 2013, Drexel University

Alzheimer's disease (AD) is a complex neurodegenerative disease determined by the combination of environmental and genetic factors. AD is pathologically characterized by the accumulation of… (more)

Subjects/Keywords: Life sciences; Alzheimer's disease; Amyloid beta-protein

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APA (6th Edition):

Utreja, S. (2013). Calcineurin regulates APP metabolism: In vitro & In vivo studies. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:6997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Utreja, Suruchi. “Calcineurin regulates APP metabolism: In vitro & In vivo studies.” 2013. Thesis, Drexel University. Accessed September 26, 2020. http://hdl.handle.net/1860/idea:6997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Utreja, Suruchi. “Calcineurin regulates APP metabolism: In vitro & In vivo studies.” 2013. Web. 26 Sep 2020.

Vancouver:

Utreja S. Calcineurin regulates APP metabolism: In vitro & In vivo studies. [Internet] [Thesis]. Drexel University; 2013. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1860/idea:6997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Utreja S. Calcineurin regulates APP metabolism: In vitro & In vivo studies. [Thesis]. Drexel University; 2013. Available from: http://hdl.handle.net/1860/idea:6997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

16. Smith, Micholas Dean. Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment.

Degree: 2014, Drexel University

The behavior of the Alzheimer's related peptide Aβ is the subject of much study. In typical computational studies the environment local to the peptide is… (more)

Subjects/Keywords: Physics; Alzheimer's disease; Amyloid beta-protein

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APA (6th Edition):

Smith, M. D. (2014). Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/4569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Micholas Dean. “Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment.” 2014. Thesis, Drexel University. Accessed September 26, 2020. http://hdl.handle.net/1860/4569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Micholas Dean. “Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment.” 2014. Web. 26 Sep 2020.

Vancouver:

Smith MD. Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment. [Internet] [Thesis]. Drexel University; 2014. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1860/4569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith MD. Influence of Aqueous-Salt Conditions on the Structure and Dynamics of the Monomeric and Novel Dimeric forms of the Alzheimer's A[beta]21-30 protein fragment. [Thesis]. Drexel University; 2014. Available from: http://hdl.handle.net/1860/4569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Mathew, Justin George. The growth factor effect on endothelial cell dysfunction in the presence of glycated collagen and A[beta] peptide : implications for decreased angiogenesis in diabetes and Alzheimer’s disease.

Degree: 2014, Drexel University

 Reduced angiogenesis, the growth of new blood vessels from existing vessels, is important in disease states including diabetes and Alzheimer’s disease. People with diabetes experience… (more)

Subjects/Keywords: Mechanical engineering; Amyloid beta-protein; Neovascularization

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APA (6th Edition):

Mathew, J. G. (2014). The growth factor effect on endothelial cell dysfunction in the presence of glycated collagen and A[beta] peptide : implications for decreased angiogenesis in diabetes and Alzheimer’s disease. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/4555

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mathew, Justin George. “The growth factor effect on endothelial cell dysfunction in the presence of glycated collagen and A[beta] peptide : implications for decreased angiogenesis in diabetes and Alzheimer’s disease.” 2014. Thesis, Drexel University. Accessed September 26, 2020. http://hdl.handle.net/1860/4555.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mathew, Justin George. “The growth factor effect on endothelial cell dysfunction in the presence of glycated collagen and A[beta] peptide : implications for decreased angiogenesis in diabetes and Alzheimer’s disease.” 2014. Web. 26 Sep 2020.

Vancouver:

Mathew JG. The growth factor effect on endothelial cell dysfunction in the presence of glycated collagen and A[beta] peptide : implications for decreased angiogenesis in diabetes and Alzheimer’s disease. [Internet] [Thesis]. Drexel University; 2014. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1860/4555.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mathew JG. The growth factor effect on endothelial cell dysfunction in the presence of glycated collagen and A[beta] peptide : implications for decreased angiogenesis in diabetes and Alzheimer’s disease. [Thesis]. Drexel University; 2014. Available from: http://hdl.handle.net/1860/4555

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

18. Dhar, Devjani. Rational Design of sym-Triazines For Multitarget-directed Modulation of Cholinesterases and Amyloid-beta in Alzheimer’s Disease.

Degree: 2013, University of Toronto

Alzheimer’s disease (AD), a progressive age-related neurodegenerative disorder is characterized by impairments in memory and cognitive functions. The two main pathogenic hallmarks associated with the… (more)

Subjects/Keywords: Alzheimer's disease; amyloid-beta; acetylcholinesterase; 0490

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APA (6th Edition):

Dhar, D. (2013). Rational Design of sym-Triazines For Multitarget-directed Modulation of Cholinesterases and Amyloid-beta in Alzheimer’s Disease. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35601

Chicago Manual of Style (16th Edition):

Dhar, Devjani. “Rational Design of sym-Triazines For Multitarget-directed Modulation of Cholinesterases and Amyloid-beta in Alzheimer’s Disease.” 2013. Masters Thesis, University of Toronto. Accessed September 26, 2020. http://hdl.handle.net/1807/35601.

MLA Handbook (7th Edition):

Dhar, Devjani. “Rational Design of sym-Triazines For Multitarget-directed Modulation of Cholinesterases and Amyloid-beta in Alzheimer’s Disease.” 2013. Web. 26 Sep 2020.

Vancouver:

Dhar D. Rational Design of sym-Triazines For Multitarget-directed Modulation of Cholinesterases and Amyloid-beta in Alzheimer’s Disease. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1807/35601.

Council of Science Editors:

Dhar D. Rational Design of sym-Triazines For Multitarget-directed Modulation of Cholinesterases and Amyloid-beta in Alzheimer’s Disease. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35601


East Carolina University

19. Sacco, Cristina. Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide.

Degree: 2010, East Carolina University

 Alzheimer's disease is a fatal and neurodegenerative disease with as many as 5.3 million Americans living with the disease. One of the characteristic hallmarks of… (more)

Subjects/Keywords: Alzheimer's disease; Copper; Amyloid beta-protein; Peptides

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sacco, C. (2010). Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide. (Masters Thesis). East Carolina University. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14792

Chicago Manual of Style (16th Edition):

Sacco, Cristina. “Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide.” 2010. Masters Thesis, East Carolina University. Accessed September 26, 2020. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14792.

MLA Handbook (7th Edition):

Sacco, Cristina. “Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide.” 2010. Web. 26 Sep 2020.

Vancouver:

Sacco C. Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide. [Internet] [Masters thesis]. East Carolina University; 2010. [cited 2020 Sep 26]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14792.

Council of Science Editors:

Sacco C. Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide. [Masters Thesis]. East Carolina University; 2010. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14792


University of Manitoba

20. Wang, Yiran. The effect of β-amyloid on thioredoxin antioxidant system.

Degree: Pharmacology and Therapeutics, 2018, University of Manitoba

Beta-amyloid peptide (Aβ) contributes significantly to neurodegeneration during the development of Alzheimer’s disease (AD). Many studies showed that Aβ can cause oxidative damage. Thioredoxin (Trx)… (more)

Subjects/Keywords: Beta-amyloid; Thioredoxin-interacting protein; oxidative damage

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APA (6th Edition):

Wang, Y. (2018). The effect of β-amyloid on thioredoxin antioxidant system. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/33239

Chicago Manual of Style (16th Edition):

Wang, Yiran. “The effect of β-amyloid on thioredoxin antioxidant system.” 2018. Masters Thesis, University of Manitoba. Accessed September 26, 2020. http://hdl.handle.net/1993/33239.

MLA Handbook (7th Edition):

Wang, Yiran. “The effect of β-amyloid on thioredoxin antioxidant system.” 2018. Web. 26 Sep 2020.

Vancouver:

Wang Y. The effect of β-amyloid on thioredoxin antioxidant system. [Internet] [Masters thesis]. University of Manitoba; 2018. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/1993/33239.

Council of Science Editors:

Wang Y. The effect of β-amyloid on thioredoxin antioxidant system. [Masters Thesis]. University of Manitoba; 2018. Available from: http://hdl.handle.net/1993/33239


Hong Kong University of Science and Technology

21. Tang, Siqi LIFS. Characterization of the function of EphA4 signaling in Alzheimer's disease.

Degree: 2015, Hong Kong University of Science and Technology

 Alzheimer’s disease (AD) is one most common dementia. Although there is currently no effective treatment, the reversal of amyloid beta (Aβ)-induced synaptic deficits is believed… (more)

Subjects/Keywords: Alzheimer's disease ; Amyloid beta-protein ; Erythropoietin ; Receptors

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APA (6th Edition):

Tang, S. L. (2015). Characterization of the function of EphA4 signaling in Alzheimer's disease. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tang, Siqi LIFS. “Characterization of the function of EphA4 signaling in Alzheimer's disease.” 2015. Thesis, Hong Kong University of Science and Technology. Accessed September 26, 2020. http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tang, Siqi LIFS. “Characterization of the function of EphA4 signaling in Alzheimer's disease.” 2015. Web. 26 Sep 2020.

Vancouver:

Tang SL. Characterization of the function of EphA4 signaling in Alzheimer's disease. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2015. [cited 2020 Sep 26]. Available from: http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tang SL. Characterization of the function of EphA4 signaling in Alzheimer's disease. [Thesis]. Hong Kong University of Science and Technology; 2015. Available from: http://repository.ust.hk/ir/Record/1783.1-92215 ; https://doi.org/10.14711/thesis-b1514949 ; http://repository.ust.hk/ir/bitstream/1783.1-92215/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

22. Jana, Metta Karunia. Identifying the key Aβ oligomer species by correlating cell neurotoxicity and binding properties.

Degree: 2016, University of Melbourne

 Alzheimer’s disease (AD) represents 60-80% of dementia cases and predominantly affects elderly people above 65 years of age. While it is the second most leading… (more)

Subjects/Keywords: alzheimer's disease; amyloid beta; neurotoxicity; oligomer

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APA (6th Edition):

Jana, M. K. (2016). Identifying the key Aβ oligomer species by correlating cell neurotoxicity and binding properties. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/116121

Chicago Manual of Style (16th Edition):

Jana, Metta Karunia. “Identifying the key Aβ oligomer species by correlating cell neurotoxicity and binding properties.” 2016. Doctoral Dissertation, University of Melbourne. Accessed September 26, 2020. http://hdl.handle.net/11343/116121.

MLA Handbook (7th Edition):

Jana, Metta Karunia. “Identifying the key Aβ oligomer species by correlating cell neurotoxicity and binding properties.” 2016. Web. 26 Sep 2020.

Vancouver:

Jana MK. Identifying the key Aβ oligomer species by correlating cell neurotoxicity and binding properties. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/11343/116121.

Council of Science Editors:

Jana MK. Identifying the key Aβ oligomer species by correlating cell neurotoxicity and binding properties. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/116121


University of Melbourne

23. Wagen, Aaron Zvi. Applying matrix assisted laser desorption-ionization mass spectrometry to identify and quantify isoforms of amyloid beta in human brain.

Degree: 2017, University of Melbourne

Amyloid beta (Aβ) plays a central pathogenic role in Alzheimer's Disease (AD), however its great heterogeneity remains largely unexplored as most research focuses on only… (more)

Subjects/Keywords: Alzheimer's Disease; amyloid beta; mass spectrometry

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APA (6th Edition):

Wagen, A. Z. (2017). Applying matrix assisted laser desorption-ionization mass spectrometry to identify and quantify isoforms of amyloid beta in human brain. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/213485

Chicago Manual of Style (16th Edition):

Wagen, Aaron Zvi. “Applying matrix assisted laser desorption-ionization mass spectrometry to identify and quantify isoforms of amyloid beta in human brain.” 2017. Masters Thesis, University of Melbourne. Accessed September 26, 2020. http://hdl.handle.net/11343/213485.

MLA Handbook (7th Edition):

Wagen, Aaron Zvi. “Applying matrix assisted laser desorption-ionization mass spectrometry to identify and quantify isoforms of amyloid beta in human brain.” 2017. Web. 26 Sep 2020.

Vancouver:

Wagen AZ. Applying matrix assisted laser desorption-ionization mass spectrometry to identify and quantify isoforms of amyloid beta in human brain. [Internet] [Masters thesis]. University of Melbourne; 2017. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/11343/213485.

Council of Science Editors:

Wagen AZ. Applying matrix assisted laser desorption-ionization mass spectrometry to identify and quantify isoforms of amyloid beta in human brain. [Masters Thesis]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/213485


Louisiana State University

24. Picou, Ryan Andrew. Analysis of amyloid beta (A [beta]) peptides by capillary electrophoresis and laser-induced fluorescence anisotropy detection.

Degree: PhD, Chemistry, 2011, Louisiana State University

 The goal of the research presented in this dissertation is to develop laser-induced fluorescence anisotropy (LIFA) detection for detecting and characterizing amyloid beta (Aβ) peptide… (more)

Subjects/Keywords: Capillary Electrophoresis; Fluorescence Anisotropy; Amyloid Beta Peptide

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APA (6th Edition):

Picou, R. A. (2011). Analysis of amyloid beta (A [beta]) peptides by capillary electrophoresis and laser-induced fluorescence anisotropy detection. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-11072011-135711 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1503

Chicago Manual of Style (16th Edition):

Picou, Ryan Andrew. “Analysis of amyloid beta (A [beta]) peptides by capillary electrophoresis and laser-induced fluorescence anisotropy detection.” 2011. Doctoral Dissertation, Louisiana State University. Accessed September 26, 2020. etd-11072011-135711 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1503.

MLA Handbook (7th Edition):

Picou, Ryan Andrew. “Analysis of amyloid beta (A [beta]) peptides by capillary electrophoresis and laser-induced fluorescence anisotropy detection.” 2011. Web. 26 Sep 2020.

Vancouver:

Picou RA. Analysis of amyloid beta (A [beta]) peptides by capillary electrophoresis and laser-induced fluorescence anisotropy detection. [Internet] [Doctoral dissertation]. Louisiana State University; 2011. [cited 2020 Sep 26]. Available from: etd-11072011-135711 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1503.

Council of Science Editors:

Picou RA. Analysis of amyloid beta (A [beta]) peptides by capillary electrophoresis and laser-induced fluorescence anisotropy detection. [Doctoral Dissertation]. Louisiana State University; 2011. Available from: etd-11072011-135711 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1503


University of South Carolina

25. Moore, Kelly Ann. Gold Nanoparticles and Peptoids as Novel Inhibitors of Amyloid Beta Aggregation in Alzheimer's Disease.

Degree: PhD, Biomedical Engineering, 2013, University of South Carolina

  In 2012 it was estimated that one in every nine Americans over the age of 65 has Alzheimer's disease (AD) (1). These patients experience… (more)

Subjects/Keywords: Alzheimer's Disease; Amyloid Beta; Gold Nanoparticles; Peptoids

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APA (6th Edition):

Moore, K. A. (2013). Gold Nanoparticles and Peptoids as Novel Inhibitors of Amyloid Beta Aggregation in Alzheimer's Disease. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/531

Chicago Manual of Style (16th Edition):

Moore, Kelly Ann. “Gold Nanoparticles and Peptoids as Novel Inhibitors of Amyloid Beta Aggregation in Alzheimer's Disease.” 2013. Doctoral Dissertation, University of South Carolina. Accessed September 26, 2020. https://scholarcommons.sc.edu/etd/531.

MLA Handbook (7th Edition):

Moore, Kelly Ann. “Gold Nanoparticles and Peptoids as Novel Inhibitors of Amyloid Beta Aggregation in Alzheimer's Disease.” 2013. Web. 26 Sep 2020.

Vancouver:

Moore KA. Gold Nanoparticles and Peptoids as Novel Inhibitors of Amyloid Beta Aggregation in Alzheimer's Disease. [Internet] [Doctoral dissertation]. University of South Carolina; 2013. [cited 2020 Sep 26]. Available from: https://scholarcommons.sc.edu/etd/531.

Council of Science Editors:

Moore KA. Gold Nanoparticles and Peptoids as Novel Inhibitors of Amyloid Beta Aggregation in Alzheimer's Disease. [Doctoral Dissertation]. University of South Carolina; 2013. Available from: https://scholarcommons.sc.edu/etd/531


University of Manchester

26. Phillips, Ashley. Probing Early Stage Aggregates of Amyloidogenic Proteins using Mass Spectrometry Based Methods.

Degree: 2016, University of Manchester

 Mass Spectrometry (MS) and Ion Mobility – Mass Spectrometry (IM-MS) can be used to investigate protein structure and dynamics and are ideally positioned to study… (more)

Subjects/Keywords: alpha-Synuclein; Amyloid-beta; MS; IM-MS

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APA (6th Edition):

Phillips, A. (2016). Probing Early Stage Aggregates of Amyloidogenic Proteins using Mass Spectrometry Based Methods. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306171

Chicago Manual of Style (16th Edition):

Phillips, Ashley. “Probing Early Stage Aggregates of Amyloidogenic Proteins using Mass Spectrometry Based Methods.” 2016. Doctoral Dissertation, University of Manchester. Accessed September 26, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306171.

MLA Handbook (7th Edition):

Phillips, Ashley. “Probing Early Stage Aggregates of Amyloidogenic Proteins using Mass Spectrometry Based Methods.” 2016. Web. 26 Sep 2020.

Vancouver:

Phillips A. Probing Early Stage Aggregates of Amyloidogenic Proteins using Mass Spectrometry Based Methods. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Sep 26]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306171.

Council of Science Editors:

Phillips A. Probing Early Stage Aggregates of Amyloidogenic Proteins using Mass Spectrometry Based Methods. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:306171

27. NC DOCKS at East Carolina University; Sacco, Cristina. Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide.

Degree: 2012, NC Docks

 Alzheimer's disease is a fatal and neurodegenerative disease with as many as 5.3 million Americans living with the disease. One of the characteristic hallmarks of… (more)

Subjects/Keywords: Alzheimer's disease; Copper; Amyloid beta-protein; Peptides

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APA (6th Edition):

NC DOCKS at East Carolina University; Sacco, C. (2012). Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

NC DOCKS at East Carolina University; Sacco, Cristina. “Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide.” 2012. Thesis, NC Docks. Accessed September 26, 2020. http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

NC DOCKS at East Carolina University; Sacco, Cristina. “Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide.” 2012. Web. 26 Sep 2020.

Vancouver:

NC DOCKS at East Carolina University; Sacco C. Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide. [Internet] [Thesis]. NC Docks; 2012. [cited 2020 Sep 26]. Available from: http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

NC DOCKS at East Carolina University; Sacco C. Thermodynamic Investigation of Copper Binding to the Amyloid-Beta (A[beta]) Peptide. [Thesis]. NC Docks; 2012. Available from: http://libres.uncg.edu/ir/ecu/f/0000-embargo-holder.txt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

28. Shabrokh, Elika. Mitochondrial Biology in Sporadic Inclusion Body Myositis.

Degree: PhD, Human Nutrition, Foods, and Exercise, 2014, Virginia Tech

 Sporadic Inclusion Body Myositis (sIBM) is an inflammatory muscle disease that strikes individuals at random and accounts for approximately 1/3 of all idiopathic inflammatory myopathies.… (more)

Subjects/Keywords: Amyloid beta; Inclusion Body Myositis; Mitochondria

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shabrokh, E. (2014). Mitochondrial Biology in Sporadic Inclusion Body Myositis. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/47782

Chicago Manual of Style (16th Edition):

Shabrokh, Elika. “Mitochondrial Biology in Sporadic Inclusion Body Myositis.” 2014. Doctoral Dissertation, Virginia Tech. Accessed September 26, 2020. http://hdl.handle.net/10919/47782.

MLA Handbook (7th Edition):

Shabrokh, Elika. “Mitochondrial Biology in Sporadic Inclusion Body Myositis.” 2014. Web. 26 Sep 2020.

Vancouver:

Shabrokh E. Mitochondrial Biology in Sporadic Inclusion Body Myositis. [Internet] [Doctoral dissertation]. Virginia Tech; 2014. [cited 2020 Sep 26]. Available from: http://hdl.handle.net/10919/47782.

Council of Science Editors:

Shabrokh E. Mitochondrial Biology in Sporadic Inclusion Body Myositis. [Doctoral Dissertation]. Virginia Tech; 2014. Available from: http://hdl.handle.net/10919/47782


Texas State University – San Marcos

29. Campbell, James. Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein.

Degree: MS, Biochemistry, 2014, Texas State University – San Marcos

 Many common diseases are caused by amyloid proteins. Amyloid structures are β sheet rich, protease resistant, and can form polydisperse insoluble fibers. Due to these… (more)

Subjects/Keywords: SDD-AGE, Prion, Amyloid; Prion; Amyloid; Amyloid beta-protein; Amyloidosis; Prions; Recombinant proteins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Campbell, J. (2014). Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein. (Masters Thesis). Texas State University – San Marcos. Retrieved from https://digital.library.txstate.edu/handle/10877/5272

Chicago Manual of Style (16th Edition):

Campbell, James. “Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein.” 2014. Masters Thesis, Texas State University – San Marcos. Accessed September 26, 2020. https://digital.library.txstate.edu/handle/10877/5272.

MLA Handbook (7th Edition):

Campbell, James. “Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein.” 2014. Web. 26 Sep 2020.

Vancouver:

Campbell J. Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein. [Internet] [Masters thesis]. Texas State University – San Marcos; 2014. [cited 2020 Sep 26]. Available from: https://digital.library.txstate.edu/handle/10877/5272.

Council of Science Editors:

Campbell J. Optimization of SDD-AGE as a Method to Study Amyloid Conversion of Human Recombinant Prion Protein. [Masters Thesis]. Texas State University – San Marcos; 2014. Available from: https://digital.library.txstate.edu/handle/10877/5272


University of Manchester

30. Andrew, Robert. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.

Degree: 2015, University of Manchester

 Dementia, the most common cause of which is Alzheimer’s disease (AD), currently affects 850,000 people in the UK, a figure set to rise to over… (more)

Subjects/Keywords: Alzheimers disease; Amyloid precursor protein; Amyloid-beta; interactome; Proteolysis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andrew, R. (2015). Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080

Chicago Manual of Style (16th Edition):

Andrew, Robert. “Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.” 2015. Doctoral Dissertation, University of Manchester. Accessed September 26, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080.

MLA Handbook (7th Edition):

Andrew, Robert. “Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.” 2015. Web. 26 Sep 2020.

Vancouver:

Andrew R. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2020 Sep 26]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080.

Council of Science Editors:

Andrew R. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080

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