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You searched for subject:(Allograft Inflammatory Factor 1). Showing records 1 – 30 of 25015 total matches.

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Temple University

1. Sommerville, Laura Jean. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.

Degree: PhD, 2010, Temple University

Molecular and Cellular Physiology

The underlying cause of all vascular proliferative diseases is injury-induced activation of vascular endothelium and vascular smooth muscle cells (VSMC). Activated… (more)

Subjects/Keywords: Biology, Physiology; Allograft Inflammatory Factor-1; Atherosclerosis; Smooth Muscle Cell Activation; Vascular Proliferative Disease

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APA (6th Edition):

Sommerville, L. J. (2010). The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,76756

Chicago Manual of Style (16th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Doctoral Dissertation, Temple University. Accessed August 07, 2020. http://digital.library.temple.edu/u?/p245801coll10,76756.

MLA Handbook (7th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Web. 07 Aug 2020.

Vancouver:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 07]. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756.

Council of Science Editors:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756


University of Southern California

2. Nowitzki, Kristina M. The response of Allograft inflammatory factor-1 to neurotoxic injury, and its role as a secreted protein.

Degree: PhD, Biochemistry & Molecular Biology, 2008, University of Southern California

Allograft inflammatory factor-1 (AIF-1) is an evolutionary conserved protein important to inflammatory responses throughout the body including that of microglia in the central nervous system… (more)

Subjects/Keywords: migroglia; MPTP; AIF-1; Allograft inflammatory factor-1

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APA (6th Edition):

Nowitzki, K. M. (2008). The response of Allograft inflammatory factor-1 to neurotoxic injury, and its role as a secreted protein. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/147701/rec/7182

Chicago Manual of Style (16th Edition):

Nowitzki, Kristina M. “The response of Allograft inflammatory factor-1 to neurotoxic injury, and its role as a secreted protein.” 2008. Doctoral Dissertation, University of Southern California. Accessed August 07, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/147701/rec/7182.

MLA Handbook (7th Edition):

Nowitzki, Kristina M. “The response of Allograft inflammatory factor-1 to neurotoxic injury, and its role as a secreted protein.” 2008. Web. 07 Aug 2020.

Vancouver:

Nowitzki KM. The response of Allograft inflammatory factor-1 to neurotoxic injury, and its role as a secreted protein. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2020 Aug 07]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/147701/rec/7182.

Council of Science Editors:

Nowitzki KM. The response of Allograft inflammatory factor-1 to neurotoxic injury, and its role as a secreted protein. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/147701/rec/7182


Temple University

3. JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

Angiogenesis is an important process in maintaining normal physiology as well as in the pathology of many diseases. Angiogenesis based therapies have… (more)

Subjects/Keywords: Biology, Microbiology; Health Sciences, Immunology; Allograft Inflammatory Factor-1; Angiogenesis; Endothelial Cell; Interleukin-19; Migration; Proliferation

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APA (6th Edition):

JAIN,SURBHI. (2009). ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Doctoral Dissertation, Temple University. Accessed August 07, 2020. http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Web. 07 Aug 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Aug 07]. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Newcastle

4. Goggins, Bridie Jane. In vitro and in vivo investigations of the α-integrins regulated by Hypoxia Inducible Factor (HIF)-1 signalling during mucosal wound healing.

Degree: PhD, 2019, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Inflammatory Bowel Disease (IBD), which includes Crohn’s disease and ulcerative colitis, affects over 5 million people worldwide and… (more)

Subjects/Keywords: inflammatory bowel disease; hypoxia inducible factor; HIF-1; wound healing; cell migration

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APA (6th Edition):

Goggins, B. J. (2019). In vitro and in vivo investigations of the α-integrins regulated by Hypoxia Inducible Factor (HIF)-1 signalling during mucosal wound healing. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1397968

Chicago Manual of Style (16th Edition):

Goggins, Bridie Jane. “In vitro and in vivo investigations of the α-integrins regulated by Hypoxia Inducible Factor (HIF)-1 signalling during mucosal wound healing.” 2019. Doctoral Dissertation, University of Newcastle. Accessed August 07, 2020. http://hdl.handle.net/1959.13/1397968.

MLA Handbook (7th Edition):

Goggins, Bridie Jane. “In vitro and in vivo investigations of the α-integrins regulated by Hypoxia Inducible Factor (HIF)-1 signalling during mucosal wound healing.” 2019. Web. 07 Aug 2020.

Vancouver:

Goggins BJ. In vitro and in vivo investigations of the α-integrins regulated by Hypoxia Inducible Factor (HIF)-1 signalling during mucosal wound healing. [Internet] [Doctoral dissertation]. University of Newcastle; 2019. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1959.13/1397968.

Council of Science Editors:

Goggins BJ. In vitro and in vivo investigations of the α-integrins regulated by Hypoxia Inducible Factor (HIF)-1 signalling during mucosal wound healing. [Doctoral Dissertation]. University of Newcastle; 2019. Available from: http://hdl.handle.net/1959.13/1397968

5. TAVARES, Mayara Costa Mansur. Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn .

Degree: 2016, Universidade Federal de Pernambuco

 Doença inflamatória intestinal descreve um grupo heterogêneo de doenças inflamatórias crônicas do trato gastrointestinal. Os dois principais tipos de DII são retocolite ulcerativa idiopática e… (more)

Subjects/Keywords: 1. Inflamassoma; 2. Interleucina; 1. 3. Fator de Necrose Tumoral alfa; 4. Polimorfismo de Única Base; 5. Doença inflamatória intestinal; 1. Inflammasome; 2. Interleukin; 1. 3. Tumour Necrosis Factor alpha; 4. Single Nucleotide Polymorphism; 5. Inflammatory bowel disease

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APA (6th Edition):

TAVARES, M. C. M. (2016). Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn . (Thesis). Universidade Federal de Pernambuco. Retrieved from https://repositorio.ufpe.br/handle/123456789/18518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

TAVARES, Mayara Costa Mansur. “Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn .” 2016. Thesis, Universidade Federal de Pernambuco. Accessed August 07, 2020. https://repositorio.ufpe.br/handle/123456789/18518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

TAVARES, Mayara Costa Mansur. “Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn .” 2016. Web. 07 Aug 2020.

Vancouver:

TAVARES MCM. Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2016. [cited 2020 Aug 07]. Available from: https://repositorio.ufpe.br/handle/123456789/18518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

TAVARES MCM. Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn . [Thesis]. Universidade Federal de Pernambuco; 2016. Available from: https://repositorio.ufpe.br/handle/123456789/18518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

6. Bell, Iona Maree. The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease.

Degree: PhD, 2018, Queen Mary, University of London

 Tumour Necrosis Factor Alpha (TNFα) is central to the immunopathogenesis of inflammatory bowel disease. It is initially expressed on the cell surface as a trimer,… (more)

Subjects/Keywords: Inflammatory Bowel Disease; Tumour Necrosis Factor Alpha

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APA (6th Edition):

Bell, I. M. (2018). The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335

Chicago Manual of Style (16th Edition):

Bell, Iona Maree. “The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease.” 2018. Doctoral Dissertation, Queen Mary, University of London. Accessed August 07, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335.

MLA Handbook (7th Edition):

Bell, Iona Maree. “The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease.” 2018. Web. 07 Aug 2020.

Vancouver:

Bell IM. The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2018. [cited 2020 Aug 07]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335.

Council of Science Editors:

Bell IM. The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease. [Doctoral Dissertation]. Queen Mary, University of London; 2018. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335


University of Edinburgh

7. Al Samman, Khaldoon Mohammed A. Chemical genetic manipulation of interferon regulatory factor 1 (IRF-1) using synthetic biology.

Degree: PhD, 2012, University of Edinburgh

 Interferon regulatory factor 1 (IRF-1), the founding member of IRF family, is a nuclear transcription factor first described as a transcription factor that binds to… (more)

Subjects/Keywords: 612; Interferon regulatory factor 1; IRF-1

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APA (6th Edition):

Al Samman, K. M. A. (2012). Chemical genetic manipulation of interferon regulatory factor 1 (IRF-1) using synthetic biology. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8092

Chicago Manual of Style (16th Edition):

Al Samman, Khaldoon Mohammed A. “Chemical genetic manipulation of interferon regulatory factor 1 (IRF-1) using synthetic biology.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed August 07, 2020. http://hdl.handle.net/1842/8092.

MLA Handbook (7th Edition):

Al Samman, Khaldoon Mohammed A. “Chemical genetic manipulation of interferon regulatory factor 1 (IRF-1) using synthetic biology.” 2012. Web. 07 Aug 2020.

Vancouver:

Al Samman KMA. Chemical genetic manipulation of interferon regulatory factor 1 (IRF-1) using synthetic biology. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1842/8092.

Council of Science Editors:

Al Samman KMA. Chemical genetic manipulation of interferon regulatory factor 1 (IRF-1) using synthetic biology. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/8092


University of Georgia

8. Jardeleza, Sarah Elaine. Molecular evolution of EF1[alpha] and its utility for resolving phylogenies in the Euglenozoa and Euglenida.

Degree: PhD, Plant Biology, 2010, University of Georgia

 The gene elongation factor 1 alpha (EF1a) has had a complicated and somewhat unclear evolutionary history; however, it still remains a useful tool in the… (more)

Subjects/Keywords: elongation factor 1 alpha

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APA (6th Edition):

Jardeleza, S. E. (2010). Molecular evolution of EF1[alpha] and its utility for resolving phylogenies in the Euglenozoa and Euglenida. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/jardeleza_sarah_e_201005_phd

Chicago Manual of Style (16th Edition):

Jardeleza, Sarah Elaine. “Molecular evolution of EF1[alpha] and its utility for resolving phylogenies in the Euglenozoa and Euglenida.” 2010. Doctoral Dissertation, University of Georgia. Accessed August 07, 2020. http://purl.galileo.usg.edu/uga_etd/jardeleza_sarah_e_201005_phd.

MLA Handbook (7th Edition):

Jardeleza, Sarah Elaine. “Molecular evolution of EF1[alpha] and its utility for resolving phylogenies in the Euglenozoa and Euglenida.” 2010. Web. 07 Aug 2020.

Vancouver:

Jardeleza SE. Molecular evolution of EF1[alpha] and its utility for resolving phylogenies in the Euglenozoa and Euglenida. [Internet] [Doctoral dissertation]. University of Georgia; 2010. [cited 2020 Aug 07]. Available from: http://purl.galileo.usg.edu/uga_etd/jardeleza_sarah_e_201005_phd.

Council of Science Editors:

Jardeleza SE. Molecular evolution of EF1[alpha] and its utility for resolving phylogenies in the Euglenozoa and Euglenida. [Doctoral Dissertation]. University of Georgia; 2010. Available from: http://purl.galileo.usg.edu/uga_etd/jardeleza_sarah_e_201005_phd


Harvard University

9. Li, Ping. Plasma Endothelin in Patients With End-Stage Renal Disease on Hemodialysis.

Degree: Master of Medical Sciences, 2019, Harvard University

End-stage renal disease (ESRD) is a worldwide public health problem. The main treatment for ESRD is still hemodialysis (HD). Despite the substantial improvements in dialysis… (more)

Subjects/Keywords: Endothelin-1; Hemodialysis; risk factor

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APA (6th Edition):

Li, P. (2019). Plasma Endothelin in Patients With End-Stage Renal Disease on Hemodialysis. (Masters Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42061458

Chicago Manual of Style (16th Edition):

Li, Ping. “Plasma Endothelin in Patients With End-Stage Renal Disease on Hemodialysis.” 2019. Masters Thesis, Harvard University. Accessed August 07, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42061458.

MLA Handbook (7th Edition):

Li, Ping. “Plasma Endothelin in Patients With End-Stage Renal Disease on Hemodialysis.” 2019. Web. 07 Aug 2020.

Vancouver:

Li P. Plasma Endothelin in Patients With End-Stage Renal Disease on Hemodialysis. [Internet] [Masters thesis]. Harvard University; 2019. [cited 2020 Aug 07]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42061458.

Council of Science Editors:

Li P. Plasma Endothelin in Patients With End-Stage Renal Disease on Hemodialysis. [Masters Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42061458


Universidad de Cantabria

10. Muñiz Diego, María del Carmen. The role of Smad Ubiquitin Regulatory Factor-1: SMURF1 in the differentiation 3T3-L1.

Degree: Máster en Biología Molecular y Biomedicina, 2013, Universidad de Cantabria

 The Smad Ubiquitin Regulatory Factor-1: SMURF1 is an E3 ligase. This E3 ligase has been linked to several important biological pathways, including the bone morphogenetic… (more)

Subjects/Keywords: Smad Ubiquitin Regulatory Factor-1

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APA (6th Edition):

Muñiz Diego, M. d. C. (2013). The role of Smad Ubiquitin Regulatory Factor-1: SMURF1 in the differentiation 3T3-L1. (Masters Thesis). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10902/4198

Chicago Manual of Style (16th Edition):

Muñiz Diego, María del Carmen. “The role of Smad Ubiquitin Regulatory Factor-1: SMURF1 in the differentiation 3T3-L1.” 2013. Masters Thesis, Universidad de Cantabria. Accessed August 07, 2020. http://hdl.handle.net/10902/4198.

MLA Handbook (7th Edition):

Muñiz Diego, María del Carmen. “The role of Smad Ubiquitin Regulatory Factor-1: SMURF1 in the differentiation 3T3-L1.” 2013. Web. 07 Aug 2020.

Vancouver:

Muñiz Diego MdC. The role of Smad Ubiquitin Regulatory Factor-1: SMURF1 in the differentiation 3T3-L1. [Internet] [Masters thesis]. Universidad de Cantabria; 2013. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/10902/4198.

Council of Science Editors:

Muñiz Diego MdC. The role of Smad Ubiquitin Regulatory Factor-1: SMURF1 in the differentiation 3T3-L1. [Masters Thesis]. Universidad de Cantabria; 2013. Available from: http://hdl.handle.net/10902/4198

11. Suppa, Alessandra Paes. Estudo da expressão dos genes regulatórios de hipóxia durante a inflamação pulmonar produzida pela isquemia e reperfusão intestinal em camundongos AIRmax e AIRmin.

Degree: Mestrado, Biotecnologia, 2009, University of São Paulo

Homeostase do O2 é essencial para a sobrevivência e desenvolvimento fisiológico dos organismos. A falta de O2 nos tecidos é um fator subjacente comum na… (more)

Subjects/Keywords: Acute inflammatory response; Acute respiratory distress syndrome; Camundongos; Expressão Gênica; Fator indutor de Hipóxia -1α; Gene Expresion; Hipoxia inducible factor -1α; Ischemia; Isquemia; Mice; Resposta inflamatória aguda; Síndrome do desconforto respiratório agudo

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APA (6th Edition):

Suppa, A. P. (2009). Estudo da expressão dos genes regulatórios de hipóxia durante a inflamação pulmonar produzida pela isquemia e reperfusão intestinal em camundongos AIRmax e AIRmin. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/87/87131/tde-29042010-103419/ ;

Chicago Manual of Style (16th Edition):

Suppa, Alessandra Paes. “Estudo da expressão dos genes regulatórios de hipóxia durante a inflamação pulmonar produzida pela isquemia e reperfusão intestinal em camundongos AIRmax e AIRmin.” 2009. Masters Thesis, University of São Paulo. Accessed August 07, 2020. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-29042010-103419/ ;.

MLA Handbook (7th Edition):

Suppa, Alessandra Paes. “Estudo da expressão dos genes regulatórios de hipóxia durante a inflamação pulmonar produzida pela isquemia e reperfusão intestinal em camundongos AIRmax e AIRmin.” 2009. Web. 07 Aug 2020.

Vancouver:

Suppa AP. Estudo da expressão dos genes regulatórios de hipóxia durante a inflamação pulmonar produzida pela isquemia e reperfusão intestinal em camundongos AIRmax e AIRmin. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2020 Aug 07]. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-29042010-103419/ ;.

Council of Science Editors:

Suppa AP. Estudo da expressão dos genes regulatórios de hipóxia durante a inflamação pulmonar produzida pela isquemia e reperfusão intestinal em camundongos AIRmax e AIRmin. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-29042010-103419/ ;


University of Toronto

12. Tajdaran, Kasra. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).

Degree: 2015, University of Toronto

Nerve injuries cause severe disability. The present investigational drug delivery strategies for enhancing peripheral nerve regeneration after nerve transection are not yet clinically translatable due… (more)

Subjects/Keywords: acellular nerve allograft; biomaterials; drug delivery; glial cell line-derived neurotrophic factor; nerve injury; regenerative medicine; 0541

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APA (6th Edition):

Tajdaran, K. (2015). Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74747

Chicago Manual of Style (16th Edition):

Tajdaran, Kasra. “Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).” 2015. Masters Thesis, University of Toronto. Accessed August 07, 2020. http://hdl.handle.net/1807/74747.

MLA Handbook (7th Edition):

Tajdaran, Kasra. “Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).” 2015. Web. 07 Aug 2020.

Vancouver:

Tajdaran K. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1807/74747.

Council of Science Editors:

Tajdaran K. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74747


University of Toronto

13. Tajdaran, Kasra. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).

Degree: 2015, University of Toronto

Nerve injuries cause severe disability. The present investigational drug delivery strategies for enhancing peripheral nerve regeneration after nerve transection are not yet clinically translatable due… (more)

Subjects/Keywords: acellular nerve allograft; biomaterials; drug delivery; glial cell line-derived neurotrophic factor; nerve injury; regenerative medicine; 0541

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APA (6th Edition):

Tajdaran, K. (2015). Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74749

Chicago Manual of Style (16th Edition):

Tajdaran, Kasra. “Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).” 2015. Masters Thesis, University of Toronto. Accessed August 07, 2020. http://hdl.handle.net/1807/74749.

MLA Handbook (7th Edition):

Tajdaran, Kasra. “Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).” 2015. Web. 07 Aug 2020.

Vancouver:

Tajdaran K. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1807/74749.

Council of Science Editors:

Tajdaran K. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74749


University of Toronto

14. Tajdaran, Kasra. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).

Degree: 2015, University of Toronto

Nerve injuries cause severe disability. The present investigational drug delivery strategies for enhancing peripheral nerve regeneration after nerve transection are not yet clinically translatable due… (more)

Subjects/Keywords: acellular nerve allograft; biomaterials; drug delivery; glial cell line-derived neurotrophic factor; nerve injury; regenerative medicine; 0541

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APA (6th Edition):

Tajdaran, K. (2015). Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74480

Chicago Manual of Style (16th Edition):

Tajdaran, Kasra. “Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).” 2015. Masters Thesis, University of Toronto. Accessed August 07, 2020. http://hdl.handle.net/1807/74480.

MLA Handbook (7th Edition):

Tajdaran, Kasra. “Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF).” 2015. Web. 07 Aug 2020.

Vancouver:

Tajdaran K. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1807/74480.

Council of Science Editors:

Tajdaran K. Enhancement of Peripheral Nerve Regeneration with Controlled Release of Glial Cell Line-derived Neurotrophic Factor (GDNF). [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74480


Penn State University

15. Pastor, Danielle Marie. Inflammatory mediator-induced dysregulation of colorectal carcinogenic pathways.

Degree: PhD, Cell and Molecular Biology, 2010, Penn State University

 Colorectal cancer (CRC) is a leading cause of cancer-related mortality in the U.S. Although CRC is associated with a lifetime risk of 5-6% in the… (more)

Subjects/Keywords: colorectal cancer; inflammatory bowel disease; interferon gamma; tumor necrosis factor; PUMA

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APA (6th Edition):

Pastor, D. M. (2010). Inflammatory mediator-induced dysregulation of colorectal carcinogenic pathways. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11281

Chicago Manual of Style (16th Edition):

Pastor, Danielle Marie. “Inflammatory mediator-induced dysregulation of colorectal carcinogenic pathways.” 2010. Doctoral Dissertation, Penn State University. Accessed August 07, 2020. https://etda.libraries.psu.edu/catalog/11281.

MLA Handbook (7th Edition):

Pastor, Danielle Marie. “Inflammatory mediator-induced dysregulation of colorectal carcinogenic pathways.” 2010. Web. 07 Aug 2020.

Vancouver:

Pastor DM. Inflammatory mediator-induced dysregulation of colorectal carcinogenic pathways. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Aug 07]. Available from: https://etda.libraries.psu.edu/catalog/11281.

Council of Science Editors:

Pastor DM. Inflammatory mediator-induced dysregulation of colorectal carcinogenic pathways. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11281

16. 小山, 淑正. Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Reversed feeding uncouples peripheral and master clock gene rhythms and leads to an increased risk of disease development. The aim of this study was to… (more)

Subjects/Keywords: sepsis; sirtuin-1; PGC-1α; pro-inflammatory cytokines; survival rate

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APA (6th Edition):

小山, . (2016). Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Thesis, Oita University / 大分大学. Accessed August 07, 2020. http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Web. 07 Aug 2020.

Vancouver:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

17. Saeed, Muhammad Azeem. The role of Protease-activated receptor 1 in inflammatory bowel disease.

Degree: 2015, University of Melbourne

Inflammatory bowel disease (IBD), a group of chronic, debilitating inflammatory disorders of the gastrointestinal tract, is a common cause of morbidity and mortality worldwide. Crohn’s… (more)

Subjects/Keywords: Protease-activated receptor 1; inflammatory bowel disease; colitis; citrobacter rodentium

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APA (6th Edition):

Saeed, M. A. (2015). The role of Protease-activated receptor 1 in inflammatory bowel disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/59618

Chicago Manual of Style (16th Edition):

Saeed, Muhammad Azeem. “The role of Protease-activated receptor 1 in inflammatory bowel disease.” 2015. Doctoral Dissertation, University of Melbourne. Accessed August 07, 2020. http://hdl.handle.net/11343/59618.

MLA Handbook (7th Edition):

Saeed, Muhammad Azeem. “The role of Protease-activated receptor 1 in inflammatory bowel disease.” 2015. Web. 07 Aug 2020.

Vancouver:

Saeed MA. The role of Protease-activated receptor 1 in inflammatory bowel disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/11343/59618.

Council of Science Editors:

Saeed MA. The role of Protease-activated receptor 1 in inflammatory bowel disease. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/59618


Tulane University

18. Pollard, Kevin. Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens.

Degree: 2017, Tulane University

The goal of the following experiments was to determine the cellular mechanisms through which estrogen receptor activity is maintained in hippocampal cells following termination of… (more)

Subjects/Keywords: estrogen; hippocampus; insulin-like growth factor-1

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APA (6th Edition):

Pollard, K. (2017). Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:76927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pollard, Kevin. “Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens.” 2017. Thesis, Tulane University. Accessed August 07, 2020. https://digitallibrary.tulane.edu/islandora/object/tulane:76927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pollard, Kevin. “Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens.” 2017. Web. 07 Aug 2020.

Vancouver:

Pollard K. Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens. [Internet] [Thesis]. Tulane University; 2017. [cited 2020 Aug 07]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:76927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pollard K. Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens. [Thesis]. Tulane University; 2017. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:76927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Exeter

19. Clissold, Rhian. Identification of hepatocyte nuclear factor 1β-associated disease.

Degree: PhD, 2017, University of Exeter

 Heterozygous mutations and deletions of the gene that encodes the transcription factor hepatocyte nuclear factor 1β (HNF1B) are the commonest known monogenic cause of developmental… (more)

Subjects/Keywords: 616.4; hepatocyte nuclear factor 1 beta (HNF1B)

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APA (6th Edition):

Clissold, R. (2017). Identification of hepatocyte nuclear factor 1β-associated disease. (Doctoral Dissertation). University of Exeter. Retrieved from http://hdl.handle.net/10871/31132

Chicago Manual of Style (16th Edition):

Clissold, Rhian. “Identification of hepatocyte nuclear factor 1β-associated disease.” 2017. Doctoral Dissertation, University of Exeter. Accessed August 07, 2020. http://hdl.handle.net/10871/31132.

MLA Handbook (7th Edition):

Clissold, Rhian. “Identification of hepatocyte nuclear factor 1β-associated disease.” 2017. Web. 07 Aug 2020.

Vancouver:

Clissold R. Identification of hepatocyte nuclear factor 1β-associated disease. [Internet] [Doctoral dissertation]. University of Exeter; 2017. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/10871/31132.

Council of Science Editors:

Clissold R. Identification of hepatocyte nuclear factor 1β-associated disease. [Doctoral Dissertation]. University of Exeter; 2017. Available from: http://hdl.handle.net/10871/31132

20. DRAKEFORD, CLIVE. Investigating the Role of Von Willebrand Factor in Regulating Macrophage Biology and Innate Immunity.

Degree: School of Medicine. Discipline of Haematology, 2019, Trinity College Dublin

 The plasma multimeric glycoprotein von Willebrand factor (VWF) plays a critical role in primary haemostasis by tethering platelets to exposed collagen at sites of vascular… (more)

Subjects/Keywords: Von Willebrand factor; Haemostasis; HIF-1?; Immunity

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APA (6th Edition):

DRAKEFORD, C. (2019). Investigating the Role of Von Willebrand Factor in Regulating Macrophage Biology and Innate Immunity. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/90724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DRAKEFORD, CLIVE. “Investigating the Role of Von Willebrand Factor in Regulating Macrophage Biology and Innate Immunity.” 2019. Thesis, Trinity College Dublin. Accessed August 07, 2020. http://hdl.handle.net/2262/90724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DRAKEFORD, CLIVE. “Investigating the Role of Von Willebrand Factor in Regulating Macrophage Biology and Innate Immunity.” 2019. Web. 07 Aug 2020.

Vancouver:

DRAKEFORD C. Investigating the Role of Von Willebrand Factor in Regulating Macrophage Biology and Innate Immunity. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/2262/90724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DRAKEFORD C. Investigating the Role of Von Willebrand Factor in Regulating Macrophage Biology and Innate Immunity. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/90724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Chile

21. Sanhueza Muñoz, Carlos Joaquín. Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales .

Degree: 2013, Universidad de Chile

 El cáncer es la 2° causa de muerte en Chile. El desarrollo del cáncer se ha propuesto que es consecuencia de la pérdida de función… (more)

Subjects/Keywords: Caveolinas; Factor 1 Inducible por Hipoxia

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APA (6th Edition):

Sanhueza Muñoz, C. J. (2013). Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales . (Thesis). Universidad de Chile. Retrieved from http://repositorio.uchile.cl/handle/2250/114871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sanhueza Muñoz, Carlos Joaquín. “Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales .” 2013. Thesis, Universidad de Chile. Accessed August 07, 2020. http://repositorio.uchile.cl/handle/2250/114871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sanhueza Muñoz, Carlos Joaquín. “Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales .” 2013. Web. 07 Aug 2020.

Vancouver:

Sanhueza Muñoz CJ. Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales . [Internet] [Thesis]. Universidad de Chile; 2013. [cited 2020 Aug 07]. Available from: http://repositorio.uchile.cl/handle/2250/114871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sanhueza Muñoz CJ. Caveolina-1 reduce la transcripción dependiente de hif1α en un mecanismo dependiente del óxido nítrico en células tumorales . [Thesis]. Universidad de Chile; 2013. Available from: http://repositorio.uchile.cl/handle/2250/114871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

22. Russell, Fiona Margaret M. Role of C-terminal phosphorylation in the regulation of the tumour suppressor IRF-1.

Degree: PhD, 2013, University of Edinburgh

 The transcription factor Interferon Regulatory Factor-1 (IRF-1) has been demonstrated to suppress tumour growth through the regulation of many anti-oncogenic genes. Pro- and anti-apoptotic factors,… (more)

Subjects/Keywords: interferon regulatory factor-1; IRF-1; autoimmune; tumour suppressor activity

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APA (6th Edition):

Russell, F. M. M. (2013). Role of C-terminal phosphorylation in the regulation of the tumour suppressor IRF-1. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8078

Chicago Manual of Style (16th Edition):

Russell, Fiona Margaret M. “Role of C-terminal phosphorylation in the regulation of the tumour suppressor IRF-1.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed August 07, 2020. http://hdl.handle.net/1842/8078.

MLA Handbook (7th Edition):

Russell, Fiona Margaret M. “Role of C-terminal phosphorylation in the regulation of the tumour suppressor IRF-1.” 2013. Web. 07 Aug 2020.

Vancouver:

Russell FMM. Role of C-terminal phosphorylation in the regulation of the tumour suppressor IRF-1. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1842/8078.

Council of Science Editors:

Russell FMM. Role of C-terminal phosphorylation in the regulation of the tumour suppressor IRF-1. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/8078


University of Edinburgh

23. Mallin, Lucy Janet. Understanding the relationship between IRF-1 and the transcriptional repressor ZNF350.

Degree: PhD, 2015, University of Edinburgh

 Interferon regulatory factor-1 (IRF-1) is a transcription factor and tumour suppressor, involved in many diverse cellular processes including immune responses and growth regulation. An interesting… (more)

Subjects/Keywords: 616.07; transcription repression; Interferon regulatory factor-1; IRF-1; ZNF350

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APA (6th Edition):

Mallin, L. J. (2015). Understanding the relationship between IRF-1 and the transcriptional repressor ZNF350. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/15893

Chicago Manual of Style (16th Edition):

Mallin, Lucy Janet. “Understanding the relationship between IRF-1 and the transcriptional repressor ZNF350.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed August 07, 2020. http://hdl.handle.net/1842/15893.

MLA Handbook (7th Edition):

Mallin, Lucy Janet. “Understanding the relationship between IRF-1 and the transcriptional repressor ZNF350.” 2015. Web. 07 Aug 2020.

Vancouver:

Mallin LJ. Understanding the relationship between IRF-1 and the transcriptional repressor ZNF350. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1842/15893.

Council of Science Editors:

Mallin LJ. Understanding the relationship between IRF-1 and the transcriptional repressor ZNF350. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/15893


Oklahoma State University

24. Hart, James Charles Albert. In Vivo Evaluation of a Naturally Derived Cytocompatibile and Architecturally Optimized Tendon Allograft in the Horse.

Degree: Veterinary Pathobiology, 2011, Oklahoma State University

 In horses, debilitating tendon injuries can result from external trauma. Currently, the repair of tendon defects arising from trauma relies heavily upon de novo tissue… (more)

Subjects/Keywords: allograft; horse; tendon

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APA (6th Edition):

Hart, J. C. A. (2011). In Vivo Evaluation of a Naturally Derived Cytocompatibile and Architecturally Optimized Tendon Allograft in the Horse. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/9817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hart, James Charles Albert. “In Vivo Evaluation of a Naturally Derived Cytocompatibile and Architecturally Optimized Tendon Allograft in the Horse.” 2011. Thesis, Oklahoma State University. Accessed August 07, 2020. http://hdl.handle.net/11244/9817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hart, James Charles Albert. “In Vivo Evaluation of a Naturally Derived Cytocompatibile and Architecturally Optimized Tendon Allograft in the Horse.” 2011. Web. 07 Aug 2020.

Vancouver:

Hart JCA. In Vivo Evaluation of a Naturally Derived Cytocompatibile and Architecturally Optimized Tendon Allograft in the Horse. [Internet] [Thesis]. Oklahoma State University; 2011. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/11244/9817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hart JCA. In Vivo Evaluation of a Naturally Derived Cytocompatibile and Architecturally Optimized Tendon Allograft in the Horse. [Thesis]. Oklahoma State University; 2011. Available from: http://hdl.handle.net/11244/9817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

25. Walters, Stacey Nicole. The role of BAFF in T cell mediated allograft rejection.

Degree: Garvan Institute of Medical Research, 2010, University of New South Wales

 Understanding the factors that control T cell responses is a major focus of immunology. Despite this the factors that control T cell development, homeostasis and… (more)

Subjects/Keywords: REJECTION; BAFF; ALLOGRAFT

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APA (6th Edition):

Walters, S. N. (2010). The role of BAFF in T cell mediated allograft rejection. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/45047 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8342/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Walters, Stacey Nicole. “The role of BAFF in T cell mediated allograft rejection.” 2010. Masters Thesis, University of New South Wales. Accessed August 07, 2020. http://handle.unsw.edu.au/1959.4/45047 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8342/SOURCE02?view=true.

MLA Handbook (7th Edition):

Walters, Stacey Nicole. “The role of BAFF in T cell mediated allograft rejection.” 2010. Web. 07 Aug 2020.

Vancouver:

Walters SN. The role of BAFF in T cell mediated allograft rejection. [Internet] [Masters thesis]. University of New South Wales; 2010. [cited 2020 Aug 07]. Available from: http://handle.unsw.edu.au/1959.4/45047 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8342/SOURCE02?view=true.

Council of Science Editors:

Walters SN. The role of BAFF in T cell mediated allograft rejection. [Masters Thesis]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/45047 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:8342/SOURCE02?view=true


Dalhousie University

26. Stillie, RoseMarie. INSIGHTS INTO THE ROLE OF INFLAMMATION IN COLITIS-ASSOCIATED CANCER: TARGETING TUMOR NECROSIS FACTOR RECEPTORS.

Degree: PhD, Department of Microbiology & Immunology, 2011, Dalhousie University

Inflammatory bowel diseases (IBD) are associated with an elevated risk of colorectal cancer that increases with disease duration and severity. Tumor necrosis factor (TNF) is… (more)

Subjects/Keywords: Colitis; cancer; inflammatory bowel disease; animal model of colitis; tumor necrosis factor; NADPH oxidase

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APA (6th Edition):

Stillie, R. (2011). INSIGHTS INTO THE ROLE OF INFLAMMATION IN COLITIS-ASSOCIATED CANCER: TARGETING TUMOR NECROSIS FACTOR RECEPTORS. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14393

Chicago Manual of Style (16th Edition):

Stillie, RoseMarie. “INSIGHTS INTO THE ROLE OF INFLAMMATION IN COLITIS-ASSOCIATED CANCER: TARGETING TUMOR NECROSIS FACTOR RECEPTORS.” 2011. Doctoral Dissertation, Dalhousie University. Accessed August 07, 2020. http://hdl.handle.net/10222/14393.

MLA Handbook (7th Edition):

Stillie, RoseMarie. “INSIGHTS INTO THE ROLE OF INFLAMMATION IN COLITIS-ASSOCIATED CANCER: TARGETING TUMOR NECROSIS FACTOR RECEPTORS.” 2011. Web. 07 Aug 2020.

Vancouver:

Stillie R. INSIGHTS INTO THE ROLE OF INFLAMMATION IN COLITIS-ASSOCIATED CANCER: TARGETING TUMOR NECROSIS FACTOR RECEPTORS. [Internet] [Doctoral dissertation]. Dalhousie University; 2011. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/10222/14393.

Council of Science Editors:

Stillie R. INSIGHTS INTO THE ROLE OF INFLAMMATION IN COLITIS-ASSOCIATED CANCER: TARGETING TUMOR NECROSIS FACTOR RECEPTORS. [Doctoral Dissertation]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14393


Texas A&M University

27. Siska, Karla P. Anti-inflammatory Effect of Vigna Unguiculata Polyphenols in Raw 264.7 Macrophages.

Degree: 2013, Texas A&M University

 This study investigated the association between flavonoid profiles of different cowpea (Vigna Unguiculata) varieties with anti-inflammatory properties as a possible benefit against inflammatory bowel disease.… (more)

Subjects/Keywords: Vigna Unguiculata; Cowpea; Phenolics; Inflammatory bowel disease; NF-?B; tumor necrosis factor alfa

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APA (6th Edition):

Siska, K. P. (2013). Anti-inflammatory Effect of Vigna Unguiculata Polyphenols in Raw 264.7 Macrophages. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Siska, Karla P. “Anti-inflammatory Effect of Vigna Unguiculata Polyphenols in Raw 264.7 Macrophages.” 2013. Thesis, Texas A&M University. Accessed August 07, 2020. http://hdl.handle.net/1969.1/149934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Siska, Karla P. “Anti-inflammatory Effect of Vigna Unguiculata Polyphenols in Raw 264.7 Macrophages.” 2013. Web. 07 Aug 2020.

Vancouver:

Siska KP. Anti-inflammatory Effect of Vigna Unguiculata Polyphenols in Raw 264.7 Macrophages. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/1969.1/149934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Siska KP. Anti-inflammatory Effect of Vigna Unguiculata Polyphenols in Raw 264.7 Macrophages. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

28. Trauernicht, Anna. Genetic Variation in Janus Associated Kinase 2 and Signal Transducers and Activators of Transcription 3 is Associated with Granulocyte-Macrophage Colony Stimulating Factor Auto-antibodies in Pediatric Crohn’s Disease.

Degree: MS, Medicine: Clinical and Translational Research, 2011, University of Cincinnati

 Background: High levels of neutralizing Granulocyte-Macrophage Colony Stimulating Factor auto-antibodies (GM-CSF Ab) in Crohn’s Disease (CD) are associated with ileal involvement, stricturing behavior, and surgery.… (more)

Subjects/Keywords: Surgery; Crohn's; Genetics; Granulocyte-Macrophage Stimulating Factor auto-ant; Inflammatory Bowel Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Trauernicht, A. (2011). Genetic Variation in Janus Associated Kinase 2 and Signal Transducers and Activators of Transcription 3 is Associated with Granulocyte-Macrophage Colony Stimulating Factor Auto-antibodies in Pediatric Crohn’s Disease. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321370394

Chicago Manual of Style (16th Edition):

Trauernicht, Anna. “Genetic Variation in Janus Associated Kinase 2 and Signal Transducers and Activators of Transcription 3 is Associated with Granulocyte-Macrophage Colony Stimulating Factor Auto-antibodies in Pediatric Crohn’s Disease.” 2011. Masters Thesis, University of Cincinnati. Accessed August 07, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321370394.

MLA Handbook (7th Edition):

Trauernicht, Anna. “Genetic Variation in Janus Associated Kinase 2 and Signal Transducers and Activators of Transcription 3 is Associated with Granulocyte-Macrophage Colony Stimulating Factor Auto-antibodies in Pediatric Crohn’s Disease.” 2011. Web. 07 Aug 2020.

Vancouver:

Trauernicht A. Genetic Variation in Janus Associated Kinase 2 and Signal Transducers and Activators of Transcription 3 is Associated with Granulocyte-Macrophage Colony Stimulating Factor Auto-antibodies in Pediatric Crohn’s Disease. [Internet] [Masters thesis]. University of Cincinnati; 2011. [cited 2020 Aug 07]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321370394.

Council of Science Editors:

Trauernicht A. Genetic Variation in Janus Associated Kinase 2 and Signal Transducers and Activators of Transcription 3 is Associated with Granulocyte-Macrophage Colony Stimulating Factor Auto-antibodies in Pediatric Crohn’s Disease. [Masters Thesis]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321370394


NSYSU

29. Hung, Han-Chun. Proteomics Analysis of an Anti-inflammatory Marine-derived Compound.

Degree: Master, Marine Biotechnology and Resources, 2011, NSYSU

 Many inflammatory diseases are growing increasing common in the aging society of Taiwan. Inflammation cascades can cause diseases such as rheumatoid arthritis, osteoarthritis, chronic asthma,… (more)

Subjects/Keywords: pro-inflammatory; nucleophosmin; inducible nitric oxide synthase; RAW 264.7; nuclear factor-kappaB; proteomics; lipopolysaccharide

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hung, H. (2011). Proteomics Analysis of an Anti-inflammatory Marine-derived Compound. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0829111-140131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hung, Han-Chun. “Proteomics Analysis of an Anti-inflammatory Marine-derived Compound.” 2011. Thesis, NSYSU. Accessed August 07, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0829111-140131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hung, Han-Chun. “Proteomics Analysis of an Anti-inflammatory Marine-derived Compound.” 2011. Web. 07 Aug 2020.

Vancouver:

Hung H. Proteomics Analysis of an Anti-inflammatory Marine-derived Compound. [Internet] [Thesis]. NSYSU; 2011. [cited 2020 Aug 07]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0829111-140131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hung H. Proteomics Analysis of an Anti-inflammatory Marine-derived Compound. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0829111-140131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington State University

30. [No author]. CONTRIBUTION OF TRPV1 VAGAL AFFERENT NEURONS IN THE DETECTION OF PATHOGENIC BACTERIAL COLONIZATION IN THE GUT: PUTATIVE ROLE(S) OF PRO-INFLAMMATORY CYTOKINES .

Degree: 2012, Washington State University

 Vagal efferent activation can reduce inflammation and disease activity in various animal models of intestinal inflammation, likely via a mechanism involving activation of a a7nAChR… (more)

Subjects/Keywords: Neurosciences; Microbiology; Immunology; anti-inflammatory reflex; campylobacter; lipopolysaccharide; salmonella; tumor necrosis factor; vagus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (2012). CONTRIBUTION OF TRPV1 VAGAL AFFERENT NEURONS IN THE DETECTION OF PATHOGENIC BACTERIAL COLONIZATION IN THE GUT: PUTATIVE ROLE(S) OF PRO-INFLAMMATORY CYTOKINES . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/4174

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “CONTRIBUTION OF TRPV1 VAGAL AFFERENT NEURONS IN THE DETECTION OF PATHOGENIC BACTERIAL COLONIZATION IN THE GUT: PUTATIVE ROLE(S) OF PRO-INFLAMMATORY CYTOKINES .” 2012. Thesis, Washington State University. Accessed August 07, 2020. http://hdl.handle.net/2376/4174.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “CONTRIBUTION OF TRPV1 VAGAL AFFERENT NEURONS IN THE DETECTION OF PATHOGENIC BACTERIAL COLONIZATION IN THE GUT: PUTATIVE ROLE(S) OF PRO-INFLAMMATORY CYTOKINES .” 2012. Web. 07 Aug 2020.

Vancouver:

author] [. CONTRIBUTION OF TRPV1 VAGAL AFFERENT NEURONS IN THE DETECTION OF PATHOGENIC BACTERIAL COLONIZATION IN THE GUT: PUTATIVE ROLE(S) OF PRO-INFLAMMATORY CYTOKINES . [Internet] [Thesis]. Washington State University; 2012. [cited 2020 Aug 07]. Available from: http://hdl.handle.net/2376/4174.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. CONTRIBUTION OF TRPV1 VAGAL AFFERENT NEURONS IN THE DETECTION OF PATHOGENIC BACTERIAL COLONIZATION IN THE GUT: PUTATIVE ROLE(S) OF PRO-INFLAMMATORY CYTOKINES . [Thesis]. Washington State University; 2012. Available from: http://hdl.handle.net/2376/4174

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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