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You searched for subject:(Allergy AND Immunology). Showing records 1 – 30 of 387 total matches.

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University of Wisconsin – Milwaukee

1. Matson, Vyara. Migration of Myeloid-derived Suppressor Cells to Tumor and Tumor-Draining Lymph Node in a Murine Model of Breast Cancer.

Degree: PhD, Biological Sciences, 2015, University of Wisconsin – Milwaukee

  Myeloid-derived suppressor cells (MDSC) consist of two major subsets, monocytic MDSC (M-MDSC) and polymorphonuclear MDSC (PMN-MDSC), both of which expand in cancer and suppress… (more)

Subjects/Keywords: Allergy and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Matson, V. (2015). Migration of Myeloid-derived Suppressor Cells to Tumor and Tumor-Draining Lymph Node in a Murine Model of Breast Cancer. (Doctoral Dissertation). University of Wisconsin – Milwaukee. Retrieved from https://dc.uwm.edu/etd/1012

Chicago Manual of Style (16th Edition):

Matson, Vyara. “Migration of Myeloid-derived Suppressor Cells to Tumor and Tumor-Draining Lymph Node in a Murine Model of Breast Cancer.” 2015. Doctoral Dissertation, University of Wisconsin – Milwaukee. Accessed April 05, 2020. https://dc.uwm.edu/etd/1012.

MLA Handbook (7th Edition):

Matson, Vyara. “Migration of Myeloid-derived Suppressor Cells to Tumor and Tumor-Draining Lymph Node in a Murine Model of Breast Cancer.” 2015. Web. 05 Apr 2020.

Vancouver:

Matson V. Migration of Myeloid-derived Suppressor Cells to Tumor and Tumor-Draining Lymph Node in a Murine Model of Breast Cancer. [Internet] [Doctoral dissertation]. University of Wisconsin – Milwaukee; 2015. [cited 2020 Apr 05]. Available from: https://dc.uwm.edu/etd/1012.

Council of Science Editors:

Matson V. Migration of Myeloid-derived Suppressor Cells to Tumor and Tumor-Draining Lymph Node in a Murine Model of Breast Cancer. [Doctoral Dissertation]. University of Wisconsin – Milwaukee; 2015. Available from: https://dc.uwm.edu/etd/1012


Drexel University

2. Lamontagne, Richard Jason. Global analysis of HBV-mediated changes to the primary hepatocyte transcriptome and metabolome.

Degree: 2015, Drexel University

Chronic infection with hepatitis B virus (HBV) remains a significant health concern, with between 350-500 million people chronically infected worldwide. Approximately 25% of chronically infected… (more)

Subjects/Keywords: Microbiology; Immunology; Allergy and Immunology

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APA (6th Edition):

Lamontagne, R. J. (2015). Global analysis of HBV-mediated changes to the primary hepatocyte transcriptome and metabolome. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lamontagne, Richard Jason. “Global analysis of HBV-mediated changes to the primary hepatocyte transcriptome and metabolome.” 2015. Thesis, Drexel University. Accessed April 05, 2020. http://hdl.handle.net/1860/idea:7152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lamontagne, Richard Jason. “Global analysis of HBV-mediated changes to the primary hepatocyte transcriptome and metabolome.” 2015. Web. 05 Apr 2020.

Vancouver:

Lamontagne RJ. Global analysis of HBV-mediated changes to the primary hepatocyte transcriptome and metabolome. [Internet] [Thesis]. Drexel University; 2015. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/1860/idea:7152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lamontagne RJ. Global analysis of HBV-mediated changes to the primary hepatocyte transcriptome and metabolome. [Thesis]. Drexel University; 2015. Available from: http://hdl.handle.net/1860/idea:7152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

3. Barsoumian, Hampartsoum. SA-4-1BBL as a modulator of innate, adaptive, and regulatory immunity : implications for cancer prevention and treatment.

Degree: PhD, 2016, University of Louisville

  SA-4-1BBL is a recombinant costimulatory molecule that is active in its soluble form and has pleiotropic effects on the functions of innate, adaptive, and… (more)

Subjects/Keywords: Allergy and Immunology; Microbiology

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APA (6th Edition):

Barsoumian, H. (2016). SA-4-1BBL as a modulator of innate, adaptive, and regulatory immunity : implications for cancer prevention and treatment. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2377 ; https://ir.library.louisville.edu/etd/2377

Chicago Manual of Style (16th Edition):

Barsoumian, Hampartsoum. “SA-4-1BBL as a modulator of innate, adaptive, and regulatory immunity : implications for cancer prevention and treatment.” 2016. Doctoral Dissertation, University of Louisville. Accessed April 05, 2020. 10.18297/etd/2377 ; https://ir.library.louisville.edu/etd/2377.

MLA Handbook (7th Edition):

Barsoumian, Hampartsoum. “SA-4-1BBL as a modulator of innate, adaptive, and regulatory immunity : implications for cancer prevention and treatment.” 2016. Web. 05 Apr 2020.

Vancouver:

Barsoumian H. SA-4-1BBL as a modulator of innate, adaptive, and regulatory immunity : implications for cancer prevention and treatment. [Internet] [Doctoral dissertation]. University of Louisville; 2016. [cited 2020 Apr 05]. Available from: 10.18297/etd/2377 ; https://ir.library.louisville.edu/etd/2377.

Council of Science Editors:

Barsoumian H. SA-4-1BBL as a modulator of innate, adaptive, and regulatory immunity : implications for cancer prevention and treatment. [Doctoral Dissertation]. University of Louisville; 2016. Available from: 10.18297/etd/2377 ; https://ir.library.louisville.edu/etd/2377


University of Pennsylvania

4. Hill, David A. Microbial Regulation of Allergic Inflammation.

Degree: 2011, University of Pennsylvania

 Allergic diseases have reached pandemic levels and represent a significant source of morbidity, mortality and healthcare cost. These chronic inflammatory diseases are characterized by interleukin… (more)

Subjects/Keywords: immunology; antibiotic; allergy; microbiology; basophil; commensal bacteria; Allergy and Immunology

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APA (6th Edition):

Hill, D. A. (2011). Microbial Regulation of Allergic Inflammation. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/970

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hill, David A. “Microbial Regulation of Allergic Inflammation.” 2011. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/970.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hill, David A. “Microbial Regulation of Allergic Inflammation.” 2011. Web. 05 Apr 2020.

Vancouver:

Hill DA. Microbial Regulation of Allergic Inflammation. [Internet] [Thesis]. University of Pennsylvania; 2011. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/970.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hill DA. Microbial Regulation of Allergic Inflammation. [Thesis]. University of Pennsylvania; 2011. Available from: https://repository.upenn.edu/edissertations/970

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

5. Arias, Katherine. Immune-Effector Pathways Leading To Peanut-Induced Anaphylaxis.

Degree: PhD, 2011, McMaster University

Among food allergies, peanut has attracted the most research attention because the allergy is typically lifelong, often severe and potentially fatal. Furthermore, other than… (more)

Subjects/Keywords: Food allergy; Mast Cells; Platelet-activating Factor; IgE; IgG; Macrophages; Allergy and Immunology; Medical Immunology; Allergy and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arias, K. (2011). Immune-Effector Pathways Leading To Peanut-Induced Anaphylaxis. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/11274

Chicago Manual of Style (16th Edition):

Arias, Katherine. “Immune-Effector Pathways Leading To Peanut-Induced Anaphylaxis.” 2011. Doctoral Dissertation, McMaster University. Accessed April 05, 2020. http://hdl.handle.net/11375/11274.

MLA Handbook (7th Edition):

Arias, Katherine. “Immune-Effector Pathways Leading To Peanut-Induced Anaphylaxis.” 2011. Web. 05 Apr 2020.

Vancouver:

Arias K. Immune-Effector Pathways Leading To Peanut-Induced Anaphylaxis. [Internet] [Doctoral dissertation]. McMaster University; 2011. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/11375/11274.

Council of Science Editors:

Arias K. Immune-Effector Pathways Leading To Peanut-Induced Anaphylaxis. [Doctoral Dissertation]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11274


University of Pennsylvania

6. Obeng-Adjei, Nyamekye. Investigating and Manipulating Immune Responses to Hepatotropic Pathogens Using Synthetic DNA.

Degree: 2013, University of Pennsylvania

 Hepatotropic pathogens, such as Hepatitis B virus (HBV), Hepatitis C virus (HCV) and malaria Plasmodium often escape cellular immune clearance, resulting in chronic infections. With… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Pathology; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Obeng-Adjei, N. (2013). Investigating and Manipulating Immune Responses to Hepatotropic Pathogens Using Synthetic DNA. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/679

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Obeng-Adjei, Nyamekye. “Investigating and Manipulating Immune Responses to Hepatotropic Pathogens Using Synthetic DNA.” 2013. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/679.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Obeng-Adjei, Nyamekye. “Investigating and Manipulating Immune Responses to Hepatotropic Pathogens Using Synthetic DNA.” 2013. Web. 05 Apr 2020.

Vancouver:

Obeng-Adjei N. Investigating and Manipulating Immune Responses to Hepatotropic Pathogens Using Synthetic DNA. [Internet] [Thesis]. University of Pennsylvania; 2013. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/679.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Obeng-Adjei N. Investigating and Manipulating Immune Responses to Hepatotropic Pathogens Using Synthetic DNA. [Thesis]. University of Pennsylvania; 2013. Available from: https://repository.upenn.edu/edissertations/679

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

7. O'Hara, Aisling Catherine. Impact of IL-27 on regulatory T cell responses.

Degree: 2013, University of Pennsylvania

 Interleukin (IL)–27 is a heterodimeric cytokine with potent inhibitory properties. Thus, mice that lack IL–27–mediated signaling develop exaggerated inflammatory responses during toxoplasmosis as well as… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

O'Hara, A. C. (2013). Impact of IL-27 on regulatory T cell responses. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Hara, Aisling Catherine. “Impact of IL-27 on regulatory T cell responses.” 2013. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Hara, Aisling Catherine. “Impact of IL-27 on regulatory T cell responses.” 2013. Web. 05 Apr 2020.

Vancouver:

O'Hara AC. Impact of IL-27 on regulatory T cell responses. [Internet] [Thesis]. University of Pennsylvania; 2013. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Hara AC. Impact of IL-27 on regulatory T cell responses. [Thesis]. University of Pennsylvania; 2013. Available from: https://repository.upenn.edu/edissertations/906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

8. Naradikian, Martin Souren. Interplay of Il-4, Il-21, and Ifnγ on Memory B Cell Fate Decisions.

Degree: 2016, University of Pennsylvania

 The ability to establish a durable pool of memory B (BMEM) cells is not only a key feature of adaptive immunity but also critical for… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Naradikian, M. S. (2016). Interplay of Il-4, Il-21, and Ifnγ on Memory B Cell Fate Decisions. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1909

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Naradikian, Martin Souren. “Interplay of Il-4, Il-21, and Ifnγ on Memory B Cell Fate Decisions.” 2016. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/1909.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Naradikian, Martin Souren. “Interplay of Il-4, Il-21, and Ifnγ on Memory B Cell Fate Decisions.” 2016. Web. 05 Apr 2020.

Vancouver:

Naradikian MS. Interplay of Il-4, Il-21, and Ifnγ on Memory B Cell Fate Decisions. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/1909.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Naradikian MS. Interplay of Il-4, Il-21, and Ifnγ on Memory B Cell Fate Decisions. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/1909

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

9. Rao, Sheila. Tyrosine Phosphorylation and Structural Requirements Mediate Toll-Like Receptor 9 Function.

Degree: 2013, University of Pennsylvania

 Upon invasion of microbial pathogens, cells of the innate immune system respond through the activation of pattern recognition receptors (PRRs) that recognize pattern associated molecular… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Rao, S. (2013). Tyrosine Phosphorylation and Structural Requirements Mediate Toll-Like Receptor 9 Function. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rao, Sheila. “Tyrosine Phosphorylation and Structural Requirements Mediate Toll-Like Receptor 9 Function.” 2013. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rao, Sheila. “Tyrosine Phosphorylation and Structural Requirements Mediate Toll-Like Receptor 9 Function.” 2013. Web. 05 Apr 2020.

Vancouver:

Rao S. Tyrosine Phosphorylation and Structural Requirements Mediate Toll-Like Receptor 9 Function. [Internet] [Thesis]. University of Pennsylvania; 2013. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rao S. Tyrosine Phosphorylation and Structural Requirements Mediate Toll-Like Receptor 9 Function. [Thesis]. University of Pennsylvania; 2013. Available from: https://repository.upenn.edu/edissertations/917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Swanstrom, Adrienne E. Dissociating Siv Env and Cd4: Consequenes for Virus and Host.

Degree: 2015, University of Pennsylvania

 CD4 tropism is conserved among all primate lentiviruses and likely contributes to viral pathogenesis by targeting cells that are critical for the adaptive anti-viral immune… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Microbiology; Virology

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APA (6th Edition):

Swanstrom, A. E. (2015). Dissociating Siv Env and Cd4: Consequenes for Virus and Host. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Swanstrom, Adrienne E. “Dissociating Siv Env and Cd4: Consequenes for Virus and Host.” 2015. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/2048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Swanstrom, Adrienne E. “Dissociating Siv Env and Cd4: Consequenes for Virus and Host.” 2015. Web. 05 Apr 2020.

Vancouver:

Swanstrom AE. Dissociating Siv Env and Cd4: Consequenes for Virus and Host. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/2048.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Swanstrom AE. Dissociating Siv Env and Cd4: Consequenes for Virus and Host. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/2048

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

11. Hergott, Christopher Bruce. Microbial Manipulation of Phagocyte Function During Infection and Health.

Degree: 2015, University of Pennsylvania

 Phagocytic cells comprise a central component of the inflammatory response to pathogens, particularly against extracellular bacteria that proliferate on mucosal surfaces. Mounting evidence suggests that… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Microbiology

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APA (6th Edition):

Hergott, C. B. (2015). Microbial Manipulation of Phagocyte Function During Infection and Health. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1760

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hergott, Christopher Bruce. “Microbial Manipulation of Phagocyte Function During Infection and Health.” 2015. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/1760.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hergott, Christopher Bruce. “Microbial Manipulation of Phagocyte Function During Infection and Health.” 2015. Web. 05 Apr 2020.

Vancouver:

Hergott CB. Microbial Manipulation of Phagocyte Function During Infection and Health. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/1760.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hergott CB. Microbial Manipulation of Phagocyte Function During Infection and Health. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/1760

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

12. Zou, Tao. Signals Controlling Regulatory T Cell Differentiation and Homeostasis.

Degree: 2012, University of Pennsylvania

 CD4+Foxp3+ regulatory T cells (Treg)s are essential for the prevention of autoimmunity. Treg lineage commitment requires T cell receptor (TCR) interactions that induce expression of… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Zou, T. (2012). Signals Controlling Regulatory T Cell Differentiation and Homeostasis. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zou, Tao. “Signals Controlling Regulatory T Cell Differentiation and Homeostasis.” 2012. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zou, Tao. “Signals Controlling Regulatory T Cell Differentiation and Homeostasis.” 2012. Web. 05 Apr 2020.

Vancouver:

Zou T. Signals Controlling Regulatory T Cell Differentiation and Homeostasis. [Internet] [Thesis]. University of Pennsylvania; 2012. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zou T. Signals Controlling Regulatory T Cell Differentiation and Homeostasis. [Thesis]. University of Pennsylvania; 2012. Available from: https://repository.upenn.edu/edissertations/602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

13. Zalinger, Zachary Bestor. Investigating the Role of Innate Inflammatory Pathways During Infection With Murine Coronavirus.

Degree: 2015, University of Pennsylvania

 Multicellular organisms are constantly exposed to microorganisms, such as viruses and bacteria, many of which are infectious pathogens. The immune system evolved to provide protection… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Virology

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APA (6th Edition):

Zalinger, Z. B. (2015). Investigating the Role of Innate Inflammatory Pathways During Infection With Murine Coronavirus. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2123

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zalinger, Zachary Bestor. “Investigating the Role of Innate Inflammatory Pathways During Infection With Murine Coronavirus.” 2015. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/2123.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zalinger, Zachary Bestor. “Investigating the Role of Innate Inflammatory Pathways During Infection With Murine Coronavirus.” 2015. Web. 05 Apr 2020.

Vancouver:

Zalinger ZB. Investigating the Role of Innate Inflammatory Pathways During Infection With Murine Coronavirus. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/2123.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zalinger ZB. Investigating the Role of Innate Inflammatory Pathways During Infection With Murine Coronavirus. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/2123

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

14. Ramos Hernández, Natalia M. The Role of NDFIP1 in T Cell Tolerance.

Degree: 2013, University of Pennsylvania

 Without the ability to suppress its responses, the immune system, instead of being advantageous to the individual, would elicit deleterious consequences. Thus, mechanisms to contain… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Ramos Hernández, N. M. (2013). The Role of NDFIP1 in T Cell Tolerance. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramos Hernández, Natalia M. “The Role of NDFIP1 in T Cell Tolerance.” 2013. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramos Hernández, Natalia M. “The Role of NDFIP1 in T Cell Tolerance.” 2013. Web. 05 Apr 2020.

Vancouver:

Ramos Hernández NM. The Role of NDFIP1 in T Cell Tolerance. [Internet] [Thesis]. University of Pennsylvania; 2013. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramos Hernández NM. The Role of NDFIP1 in T Cell Tolerance. [Thesis]. University of Pennsylvania; 2013. Available from: https://repository.upenn.edu/edissertations/686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

15. Freund, Jacquelyn Elizabeth. Regulation Of Natural Killer Cell Development And Function By Activating Receptor Signaling Pathways.

Degree: 2017, University of Pennsylvania

 Natural killer (NK) cells are lymphocytes of the innate immune system that recognize and eliminate virally infected and transformed cells through their release of cytotoxic… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Freund, J. E. (2017). Regulation Of Natural Killer Cell Development And Function By Activating Receptor Signaling Pathways. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Freund, Jacquelyn Elizabeth. “Regulation Of Natural Killer Cell Development And Function By Activating Receptor Signaling Pathways.” 2017. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/2290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Freund, Jacquelyn Elizabeth. “Regulation Of Natural Killer Cell Development And Function By Activating Receptor Signaling Pathways.” 2017. Web. 05 Apr 2020.

Vancouver:

Freund JE. Regulation Of Natural Killer Cell Development And Function By Activating Receptor Signaling Pathways. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/2290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Freund JE. Regulation Of Natural Killer Cell Development And Function By Activating Receptor Signaling Pathways. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

16. Glennie, Nelson D. The Role Of Skin Resident Cd4 T Cells In Cutaneous Leishmaniasis.

Degree: 2017, University of Pennsylvania

 Cutaneous leishmaniasis is a disease characterized by highly inflammatory, sometimes disfiguring lesions that nonetheless spontaneously resolve, resulting in robust protection against reinfection. It has been… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Glennie, N. D. (2017). The Role Of Skin Resident Cd4 T Cells In Cutaneous Leishmaniasis. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Glennie, Nelson D. “The Role Of Skin Resident Cd4 T Cells In Cutaneous Leishmaniasis.” 2017. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/2310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Glennie, Nelson D. “The Role Of Skin Resident Cd4 T Cells In Cutaneous Leishmaniasis.” 2017. Web. 05 Apr 2020.

Vancouver:

Glennie ND. The Role Of Skin Resident Cd4 T Cells In Cutaneous Leishmaniasis. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/2310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Glennie ND. The Role Of Skin Resident Cd4 T Cells In Cutaneous Leishmaniasis. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

17. Liu, Mingen. Metabolic Rewiring Of Macrophages Promotes Anti-Tumor Activity In Pancreatic Cancer.

Degree: 2018, University of Pennsylvania

 Macrophages abound in the tumor microenvironment of pancreatic cancer and other solid malignancies. Although macrophages typically promote tumorigenesis, they also represent key targets for immunotherapy… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Liu, M. (2018). Metabolic Rewiring Of Macrophages Promotes Anti-Tumor Activity In Pancreatic Cancer. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/3145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Mingen. “Metabolic Rewiring Of Macrophages Promotes Anti-Tumor Activity In Pancreatic Cancer.” 2018. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/3145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Mingen. “Metabolic Rewiring Of Macrophages Promotes Anti-Tumor Activity In Pancreatic Cancer.” 2018. Web. 05 Apr 2020.

Vancouver:

Liu M. Metabolic Rewiring Of Macrophages Promotes Anti-Tumor Activity In Pancreatic Cancer. [Internet] [Thesis]. University of Pennsylvania; 2018. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/3145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu M. Metabolic Rewiring Of Macrophages Promotes Anti-Tumor Activity In Pancreatic Cancer. [Thesis]. University of Pennsylvania; 2018. Available from: https://repository.upenn.edu/edissertations/3145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

18. Mack, Ethan. Tribbles Homologue 1 Controls Granulocyte Progenitor Commitment And Terminal Cell Identity And Function.

Degree: 2018, University of Pennsylvania

 Eosinophils and neutrophils are critical for host defense, yet gaps in understanding how granulocytes differentiate from HSCs into mature effectors remain. The pseudokinase Trib1 is… (more)

Subjects/Keywords: Allergy and Immunology; Biology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Mack, E. (2018). Tribbles Homologue 1 Controls Granulocyte Progenitor Commitment And Terminal Cell Identity And Function. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/3155

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mack, Ethan. “Tribbles Homologue 1 Controls Granulocyte Progenitor Commitment And Terminal Cell Identity And Function.” 2018. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/3155.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mack, Ethan. “Tribbles Homologue 1 Controls Granulocyte Progenitor Commitment And Terminal Cell Identity And Function.” 2018. Web. 05 Apr 2020.

Vancouver:

Mack E. Tribbles Homologue 1 Controls Granulocyte Progenitor Commitment And Terminal Cell Identity And Function. [Internet] [Thesis]. University of Pennsylvania; 2018. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/3155.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mack E. Tribbles Homologue 1 Controls Granulocyte Progenitor Commitment And Terminal Cell Identity And Function. [Thesis]. University of Pennsylvania; 2018. Available from: https://repository.upenn.edu/edissertations/3155

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

19. Khan, Omar. The Hmg Transcription Factor Tox Induces A Transcriptional And Epigenetic Program Of Cd8+ T Cell Exhaustion In Chronic Infection And Cancer.

Degree: 2019, University of Pennsylvania

 Exhausted CD8+ T cells (TEX) in chronic infections and cancer are characterized by loss of optimal function, high co-expression of inhibitory receptors and extensive transcriptional… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Khan, O. (2019). The Hmg Transcription Factor Tox Induces A Transcriptional And Epigenetic Program Of Cd8+ T Cell Exhaustion In Chronic Infection And Cancer. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/3673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Khan, Omar. “The Hmg Transcription Factor Tox Induces A Transcriptional And Epigenetic Program Of Cd8+ T Cell Exhaustion In Chronic Infection And Cancer.” 2019. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/3673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Khan, Omar. “The Hmg Transcription Factor Tox Induces A Transcriptional And Epigenetic Program Of Cd8+ T Cell Exhaustion In Chronic Infection And Cancer.” 2019. Web. 05 Apr 2020.

Vancouver:

Khan O. The Hmg Transcription Factor Tox Induces A Transcriptional And Epigenetic Program Of Cd8+ T Cell Exhaustion In Chronic Infection And Cancer. [Internet] [Thesis]. University of Pennsylvania; 2019. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/3673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Khan O. The Hmg Transcription Factor Tox Induces A Transcriptional And Epigenetic Program Of Cd8+ T Cell Exhaustion In Chronic Infection And Cancer. [Thesis]. University of Pennsylvania; 2019. Available from: https://repository.upenn.edu/edissertations/3673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Wymore Brand, Meghan Joyce. Age-related impact of proteobacteria colonization on mucosal homeostasis and the microbial community in gastrointestinal health and disease.

Degree: 2017, Iowa State University

 Inflammatory bowel disease is a group of chronic intestinal inflammatory disorders with a complex etiology, and has been associated with a microbial dysbiosis and presence… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Wymore Brand, M. J. (2017). Age-related impact of proteobacteria colonization on mucosal homeostasis and the microbial community in gastrointestinal health and disease. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/16112

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wymore Brand, Meghan Joyce. “Age-related impact of proteobacteria colonization on mucosal homeostasis and the microbial community in gastrointestinal health and disease.” 2017. Thesis, Iowa State University. Accessed April 05, 2020. https://lib.dr.iastate.edu/etd/16112.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wymore Brand, Meghan Joyce. “Age-related impact of proteobacteria colonization on mucosal homeostasis and the microbial community in gastrointestinal health and disease.” 2017. Web. 05 Apr 2020.

Vancouver:

Wymore Brand MJ. Age-related impact of proteobacteria colonization on mucosal homeostasis and the microbial community in gastrointestinal health and disease. [Internet] [Thesis]. Iowa State University; 2017. [cited 2020 Apr 05]. Available from: https://lib.dr.iastate.edu/etd/16112.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wymore Brand MJ. Age-related impact of proteobacteria colonization on mucosal homeostasis and the microbial community in gastrointestinal health and disease. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/16112

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

21. O'Neill, Kevin Charles. Efficacy and impact of current commercial porcine circovirus type 2 (PCV2) vaccines in dams and growing pigs.

Degree: 2012, Iowa State University

 Porcine circovirus (PCV) was initially described as a contaminant of the continuous porcine kidney cell line PK-15 in 1974 (Tischer et al., 1974). It is… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Veterinary Medicine

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APA (6th Edition):

O'Neill, K. C. (2012). Efficacy and impact of current commercial porcine circovirus type 2 (PCV2) vaccines in dams and growing pigs. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/12837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Neill, Kevin Charles. “Efficacy and impact of current commercial porcine circovirus type 2 (PCV2) vaccines in dams and growing pigs.” 2012. Thesis, Iowa State University. Accessed April 05, 2020. https://lib.dr.iastate.edu/etd/12837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Neill, Kevin Charles. “Efficacy and impact of current commercial porcine circovirus type 2 (PCV2) vaccines in dams and growing pigs.” 2012. Web. 05 Apr 2020.

Vancouver:

O'Neill KC. Efficacy and impact of current commercial porcine circovirus type 2 (PCV2) vaccines in dams and growing pigs. [Internet] [Thesis]. Iowa State University; 2012. [cited 2020 Apr 05]. Available from: https://lib.dr.iastate.edu/etd/12837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Neill KC. Efficacy and impact of current commercial porcine circovirus type 2 (PCV2) vaccines in dams and growing pigs. [Thesis]. Iowa State University; 2012. Available from: https://lib.dr.iastate.edu/etd/12837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

22. Rome, Kelly. Tribbles Homologue 1 Controls Antiviral Immunity By Restraining T Cell Effector Programming And Function.

Degree: 2019, University of Pennsylvania

 In chronic disease, persistent antigen stimulation results in a gradual loss in function of the exhausted T cell (TEX) pool, and a subsequent loss of… (more)

Subjects/Keywords: Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Molecular Biology

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APA (6th Edition):

Rome, K. (2019). Tribbles Homologue 1 Controls Antiviral Immunity By Restraining T Cell Effector Programming And Function. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/3620

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rome, Kelly. “Tribbles Homologue 1 Controls Antiviral Immunity By Restraining T Cell Effector Programming And Function.” 2019. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/3620.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rome, Kelly. “Tribbles Homologue 1 Controls Antiviral Immunity By Restraining T Cell Effector Programming And Function.” 2019. Web. 05 Apr 2020.

Vancouver:

Rome K. Tribbles Homologue 1 Controls Antiviral Immunity By Restraining T Cell Effector Programming And Function. [Internet] [Thesis]. University of Pennsylvania; 2019. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/3620.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rome K. Tribbles Homologue 1 Controls Antiviral Immunity By Restraining T Cell Effector Programming And Function. [Thesis]. University of Pennsylvania; 2019. Available from: https://repository.upenn.edu/edissertations/3620

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Zhao, Jiangyang. Epigenetic Activation of the Mouse T Cell Receptor Beta Recombination Center.

Degree: PhD, Biology & Biomedical Sciences (Molecular Genetics & Genomics), 2017, Washington University in St. Louis

 Lymphocytes are the work horses of adaptive immunity. Compared to the B lymphocyte lineage, early stage progenitors of T lymphocytes maintain considerable potential for differentiation… (more)

Subjects/Keywords: Allergy and Immunology; Genetics; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Zhao, J. (2017). Epigenetic Activation of the Mouse T Cell Receptor Beta Recombination Center. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/art_sci_etds/1157

Chicago Manual of Style (16th Edition):

Zhao, Jiangyang. “Epigenetic Activation of the Mouse T Cell Receptor Beta Recombination Center.” 2017. Doctoral Dissertation, Washington University in St. Louis. Accessed April 05, 2020. https://openscholarship.wustl.edu/art_sci_etds/1157.

MLA Handbook (7th Edition):

Zhao, Jiangyang. “Epigenetic Activation of the Mouse T Cell Receptor Beta Recombination Center.” 2017. Web. 05 Apr 2020.

Vancouver:

Zhao J. Epigenetic Activation of the Mouse T Cell Receptor Beta Recombination Center. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2017. [cited 2020 Apr 05]. Available from: https://openscholarship.wustl.edu/art_sci_etds/1157.

Council of Science Editors:

Zhao J. Epigenetic Activation of the Mouse T Cell Receptor Beta Recombination Center. [Doctoral Dissertation]. Washington University in St. Louis; 2017. Available from: https://openscholarship.wustl.edu/art_sci_etds/1157

24. Fernandez, Estefania. Characterizing the Humoral Response to Flavivirus Infection.

Degree: PhD, Biology & Biomedical Sciences (Immunology), 2019, Washington University in St. Louis

  Flaviviruses are positive (+) sense, single-stranded RNA viruses of the Flaviviridae family that are transmitted by mosquitoes. For our studies, we focused on Zika… (more)

Subjects/Keywords: JEV, ZIKV; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Fernandez, E. (2019). Characterizing the Humoral Response to Flavivirus Infection. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/art_sci_etds/1775

Chicago Manual of Style (16th Edition):

Fernandez, Estefania. “Characterizing the Humoral Response to Flavivirus Infection.” 2019. Doctoral Dissertation, Washington University in St. Louis. Accessed April 05, 2020. https://openscholarship.wustl.edu/art_sci_etds/1775.

MLA Handbook (7th Edition):

Fernandez, Estefania. “Characterizing the Humoral Response to Flavivirus Infection.” 2019. Web. 05 Apr 2020.

Vancouver:

Fernandez E. Characterizing the Humoral Response to Flavivirus Infection. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2019. [cited 2020 Apr 05]. Available from: https://openscholarship.wustl.edu/art_sci_etds/1775.

Council of Science Editors:

Fernandez E. Characterizing the Humoral Response to Flavivirus Infection. [Doctoral Dissertation]. Washington University in St. Louis; 2019. Available from: https://openscholarship.wustl.edu/art_sci_etds/1775


Michigan State University

25. Ortiz, Tina Consetta. Effect of extrusion processing on in vivo allergenicity of hazelnut protein extract in an adjuvant-free mouse model.

Degree: 2014, Michigan State University

Thesis M.S. Michigan State University. Food Science - Master of Science 2014.

Life-threatening nut allergy is a growing public health problem in many countries including… (more)

Subjects/Keywords: Food allergy; Food allergy – Animal models; Hazelnuts; Food science; Immunology

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APA (6th Edition):

Ortiz, T. C. (2014). Effect of extrusion processing on in vivo allergenicity of hazelnut protein extract in an adjuvant-free mouse model. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3159

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ortiz, Tina Consetta. “Effect of extrusion processing on in vivo allergenicity of hazelnut protein extract in an adjuvant-free mouse model.” 2014. Thesis, Michigan State University. Accessed April 05, 2020. http://etd.lib.msu.edu/islandora/object/etd:3159.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ortiz, Tina Consetta. “Effect of extrusion processing on in vivo allergenicity of hazelnut protein extract in an adjuvant-free mouse model.” 2014. Web. 05 Apr 2020.

Vancouver:

Ortiz TC. Effect of extrusion processing on in vivo allergenicity of hazelnut protein extract in an adjuvant-free mouse model. [Internet] [Thesis]. Michigan State University; 2014. [cited 2020 Apr 05]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3159.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ortiz TC. Effect of extrusion processing on in vivo allergenicity of hazelnut protein extract in an adjuvant-free mouse model. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:3159

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Qaseem, Asif Shehzad. Modulation of immune cell functions by human lung surfactant protein SP-D in allergic asthma.

Degree: PhD, 2016, Brunel University

 Lung surfactant protein D (SP-D) is a soluble pattern recognition and innate immune molecule, which has been shown to be protective against lung infection, allergy,… (more)

Subjects/Keywords: 616.2; Immunology; Asthma; Allergy; Molecular biology

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APA (6th Edition):

Qaseem, A. S. (2016). Modulation of immune cell functions by human lung surfactant protein SP-D in allergic asthma. (Doctoral Dissertation). Brunel University. Retrieved from http://bura.brunel.ac.uk/handle/2438/13893 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701946

Chicago Manual of Style (16th Edition):

Qaseem, Asif Shehzad. “Modulation of immune cell functions by human lung surfactant protein SP-D in allergic asthma.” 2016. Doctoral Dissertation, Brunel University. Accessed April 05, 2020. http://bura.brunel.ac.uk/handle/2438/13893 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701946.

MLA Handbook (7th Edition):

Qaseem, Asif Shehzad. “Modulation of immune cell functions by human lung surfactant protein SP-D in allergic asthma.” 2016. Web. 05 Apr 2020.

Vancouver:

Qaseem AS. Modulation of immune cell functions by human lung surfactant protein SP-D in allergic asthma. [Internet] [Doctoral dissertation]. Brunel University; 2016. [cited 2020 Apr 05]. Available from: http://bura.brunel.ac.uk/handle/2438/13893 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701946.

Council of Science Editors:

Qaseem AS. Modulation of immune cell functions by human lung surfactant protein SP-D in allergic asthma. [Doctoral Dissertation]. Brunel University; 2016. Available from: http://bura.brunel.ac.uk/handle/2438/13893 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701946


University of Louisville

27. Waterstone, M. Lacefield. Studies in clinical allergy.

Degree: MS, 1941, University of Louisville

Subjects/Keywords: Allergy and Immunology

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APA (6th Edition):

Waterstone, M. L. (1941). Studies in clinical allergy. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/1939 ; https://ir.library.louisville.edu/etd/1939

Chicago Manual of Style (16th Edition):

Waterstone, M Lacefield. “Studies in clinical allergy.” 1941. Masters Thesis, University of Louisville. Accessed April 05, 2020. 10.18297/etd/1939 ; https://ir.library.louisville.edu/etd/1939.

MLA Handbook (7th Edition):

Waterstone, M Lacefield. “Studies in clinical allergy.” 1941. Web. 05 Apr 2020.

Vancouver:

Waterstone ML. Studies in clinical allergy. [Internet] [Masters thesis]. University of Louisville; 1941. [cited 2020 Apr 05]. Available from: 10.18297/etd/1939 ; https://ir.library.louisville.edu/etd/1939.

Council of Science Editors:

Waterstone ML. Studies in clinical allergy. [Masters Thesis]. University of Louisville; 1941. Available from: 10.18297/etd/1939 ; https://ir.library.louisville.edu/etd/1939

28. Azzaoui, Imane. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.

Degree: Docteur es, Immunologie (Médecine), 2011, Université Lille II – Droit et Santé

 Les chimiokines sont un élément essentiel du trafic cellulaire aussi bien homéostatique que dans des situations pathologiques. Outre cette fonction chimiotactique spécifique à ce type… (more)

Subjects/Keywords: Chimiokine CCL18; Allergie; Chemokines; Allergy; Immunology

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APA (6th Edition):

Azzaoui, I. (2011). CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2011LIL2S019

Chicago Manual of Style (16th Edition):

Azzaoui, Imane. “CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.” 2011. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed April 05, 2020. http://www.theses.fr/2011LIL2S019.

MLA Handbook (7th Edition):

Azzaoui, Imane. “CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.” 2011. Web. 05 Apr 2020.

Vancouver:

Azzaoui I. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2011. [cited 2020 Apr 05]. Available from: http://www.theses.fr/2011LIL2S019.

Council of Science Editors:

Azzaoui I. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2011. Available from: http://www.theses.fr/2011LIL2S019


University of Wisconsin – Milwaukee

29. Bunnag, Sopitsuda. Impaired T Lymphocyte Responses in Older Macaques: Possible Implications for Lentiviral Disease Progression.

Degree: MS, Biomedical Sciences, 2015, University of Wisconsin – Milwaukee

  Lentiviral infections of humans and rhesus macaques result in acquired immunodeficiency almost invariably. Yet the duration between the initial infection and the onset of… (more)

Subjects/Keywords: Lentiviral; Macaques; Rhesus; Allergy and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bunnag, S. (2015). Impaired T Lymphocyte Responses in Older Macaques: Possible Implications for Lentiviral Disease Progression. (Thesis). University of Wisconsin – Milwaukee. Retrieved from https://dc.uwm.edu/etd/796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bunnag, Sopitsuda. “Impaired T Lymphocyte Responses in Older Macaques: Possible Implications for Lentiviral Disease Progression.” 2015. Thesis, University of Wisconsin – Milwaukee. Accessed April 05, 2020. https://dc.uwm.edu/etd/796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bunnag, Sopitsuda. “Impaired T Lymphocyte Responses in Older Macaques: Possible Implications for Lentiviral Disease Progression.” 2015. Web. 05 Apr 2020.

Vancouver:

Bunnag S. Impaired T Lymphocyte Responses in Older Macaques: Possible Implications for Lentiviral Disease Progression. [Internet] [Thesis]. University of Wisconsin – Milwaukee; 2015. [cited 2020 Apr 05]. Available from: https://dc.uwm.edu/etd/796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bunnag S. Impaired T Lymphocyte Responses in Older Macaques: Possible Implications for Lentiviral Disease Progression. [Thesis]. University of Wisconsin – Milwaukee; 2015. Available from: https://dc.uwm.edu/etd/796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

30. Johnson-Weaver, Brandi Tranae. Modulation of Allergic Disease through the use of Th1-associated Vaccine Adjuvants .

Degree: 2015, Duke University

  The prevalence of allergic disease such as peanut (PN) allergy has increased within the last century. Environmental factors have been associated with an increased… (more)

Subjects/Keywords: Immunology; adjuvant; immunotherapy; peanut allergy; vaccine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johnson-Weaver, B. T. (2015). Modulation of Allergic Disease through the use of Th1-associated Vaccine Adjuvants . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/10495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johnson-Weaver, Brandi Tranae. “Modulation of Allergic Disease through the use of Th1-associated Vaccine Adjuvants .” 2015. Thesis, Duke University. Accessed April 05, 2020. http://hdl.handle.net/10161/10495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johnson-Weaver, Brandi Tranae. “Modulation of Allergic Disease through the use of Th1-associated Vaccine Adjuvants .” 2015. Web. 05 Apr 2020.

Vancouver:

Johnson-Weaver BT. Modulation of Allergic Disease through the use of Th1-associated Vaccine Adjuvants . [Internet] [Thesis]. Duke University; 2015. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/10161/10495.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johnson-Weaver BT. Modulation of Allergic Disease through the use of Th1-associated Vaccine Adjuvants . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/10495

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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