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You searched for subject:(Akt). Showing records 1 – 30 of 540 total matches.

[1] [2] [3] [4] [5] … [18]

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1. Santos Ahmed, Jeena M. The Effect of Transient Neonatal Goitrogen Exposure on Akt1-Dependent Testicular Development and Gene Expression in the C57/Bl6 Mouse.

Degree: PhD, Pathobiology, 2011, Brown University

 The Sertoli cell is the "support cell" of the testis. The number of Sertoli cells which populate the testis during peri-pubertal development dictate the number… (more)

Subjects/Keywords: Akt

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APA (6th Edition):

Santos Ahmed, J. M. (2011). The Effect of Transient Neonatal Goitrogen Exposure on Akt1-Dependent Testicular Development and Gene Expression in the C57/Bl6 Mouse. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11350/

Chicago Manual of Style (16th Edition):

Santos Ahmed, Jeena M. “The Effect of Transient Neonatal Goitrogen Exposure on Akt1-Dependent Testicular Development and Gene Expression in the C57/Bl6 Mouse.” 2011. Doctoral Dissertation, Brown University. Accessed June 04, 2020. https://repository.library.brown.edu/studio/item/bdr:11350/.

MLA Handbook (7th Edition):

Santos Ahmed, Jeena M. “The Effect of Transient Neonatal Goitrogen Exposure on Akt1-Dependent Testicular Development and Gene Expression in the C57/Bl6 Mouse.” 2011. Web. 04 Jun 2020.

Vancouver:

Santos Ahmed JM. The Effect of Transient Neonatal Goitrogen Exposure on Akt1-Dependent Testicular Development and Gene Expression in the C57/Bl6 Mouse. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2020 Jun 04]. Available from: https://repository.library.brown.edu/studio/item/bdr:11350/.

Council of Science Editors:

Santos Ahmed JM. The Effect of Transient Neonatal Goitrogen Exposure on Akt1-Dependent Testicular Development and Gene Expression in the C57/Bl6 Mouse. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11350/

2. Axanova, Linara. 1á,25(OH)2-VITAMIN D3 IN PROSTATE: INTERSECTION WITH AKT/PTEN AXIS AND ROLE IN SENESCENCE.

Degree: 2010, Wake Forest University

 The PI3K-AKT pathway is frequently activated in prostate cancer and can lead to downregulation of p21 and p27 levels and function. 1-alpha, 25-dihydroxy vitamin D3(more)

Subjects/Keywords: AKT

…x28;OH)2D3 118 3.4.4. Inhibition of PI3K/AKT Partially Restores Sensitivity to 1α,25… …DISCUSSION 4.1. Synergistic Growth Inhibition by 1α,25(OH)2D3 and PI3K/AKT Inhibitors… …Interactions of PTEN/AKT Axis and p21/p27 53 Figure 2 Structure of tricyclic nucleoside API-2 56… …x29;2D3 synergistically inhibit growth of LNCaP and DU145 cells 64 AKT inhibitors API-2 and… …not synegize or cooperate to inhibit growth of DU145 cells 70 AKT inhibitor API-2 and 1α… 

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APA (6th Edition):

Axanova, L. (2010). 1á,25(OH)2-VITAMIN D3 IN PROSTATE: INTERSECTION WITH AKT/PTEN AXIS AND ROLE IN SENESCENCE. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/30431

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Axanova, Linara. “1á,25(OH)2-VITAMIN D3 IN PROSTATE: INTERSECTION WITH AKT/PTEN AXIS AND ROLE IN SENESCENCE.” 2010. Thesis, Wake Forest University. Accessed June 04, 2020. http://hdl.handle.net/10339/30431.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Axanova, Linara. “1á,25(OH)2-VITAMIN D3 IN PROSTATE: INTERSECTION WITH AKT/PTEN AXIS AND ROLE IN SENESCENCE.” 2010. Web. 04 Jun 2020.

Vancouver:

Axanova L. 1á,25(OH)2-VITAMIN D3 IN PROSTATE: INTERSECTION WITH AKT/PTEN AXIS AND ROLE IN SENESCENCE. [Internet] [Thesis]. Wake Forest University; 2010. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10339/30431.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Axanova L. 1á,25(OH)2-VITAMIN D3 IN PROSTATE: INTERSECTION WITH AKT/PTEN AXIS AND ROLE IN SENESCENCE. [Thesis]. Wake Forest University; 2010. Available from: http://hdl.handle.net/10339/30431

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

3. Sharpe, Laura Jane. Investigating the roles of Akt targets in cholesterol regulation and protein transport.

Degree: Biotechnology & Biomolecular Sciences, 2010, University of New South Wales

 It has previously been found that the Akt pathway influences cholesterolhomeostasis through effects on the endoplasmic reticulum (ER) to Golgi transport ofthe transcription factor, sterol-regulatory… (more)

Subjects/Keywords: Akt; Sec24

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APA (6th Edition):

Sharpe, L. J. (2010). Investigating the roles of Akt targets in cholesterol regulation and protein transport. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50209 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9087/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Sharpe, Laura Jane. “Investigating the roles of Akt targets in cholesterol regulation and protein transport.” 2010. Doctoral Dissertation, University of New South Wales. Accessed June 04, 2020. http://handle.unsw.edu.au/1959.4/50209 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9087/SOURCE02?view=true.

MLA Handbook (7th Edition):

Sharpe, Laura Jane. “Investigating the roles of Akt targets in cholesterol regulation and protein transport.” 2010. Web. 04 Jun 2020.

Vancouver:

Sharpe LJ. Investigating the roles of Akt targets in cholesterol regulation and protein transport. [Internet] [Doctoral dissertation]. University of New South Wales; 2010. [cited 2020 Jun 04]. Available from: http://handle.unsw.edu.au/1959.4/50209 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9087/SOURCE02?view=true.

Council of Science Editors:

Sharpe LJ. Investigating the roles of Akt targets in cholesterol regulation and protein transport. [Doctoral Dissertation]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/50209 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9087/SOURCE02?view=true

4. Nelson, Ronald Alvin. Design, Synthesis, and Screening of Novel Phosphatidylinositol 3-Kinase (PI3K) Inhibitors Activated by Prostate Specific Antigen (PSA): Prodrug Treatment for Androgen-Independent Prostate Cancer.

Degree: 2017, Wake Forest University

 The PI3K/Akt pathway is one of the most frequently dysregulated signaling pathways in cancer and remains a crucial target in drug development. Current therapeutic drugs… (more)

Subjects/Keywords: Akt

…p-Akt T308 through a Western blot analysis ............... 91 Figure 27: Qualitative… …measure of Compound 71‘s PI3K pathway inhibition by measuring activated p-Akt T308 through a… …pathway inhibition by measuring activated p-Akt T308 through a Western blot analysis… …activated p-Akt S473 through a Western blot analysis ............... 98 viii Figure 31… …Qualitative measure of Compound 78‘s PI3K pathway inhibition by measuring activated p-Akt T308… 

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APA (6th Edition):

Nelson, R. A. (2017). Design, Synthesis, and Screening of Novel Phosphatidylinositol 3-Kinase (PI3K) Inhibitors Activated by Prostate Specific Antigen (PSA): Prodrug Treatment for Androgen-Independent Prostate Cancer. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/82252

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Ronald Alvin. “Design, Synthesis, and Screening of Novel Phosphatidylinositol 3-Kinase (PI3K) Inhibitors Activated by Prostate Specific Antigen (PSA): Prodrug Treatment for Androgen-Independent Prostate Cancer.” 2017. Thesis, Wake Forest University. Accessed June 04, 2020. http://hdl.handle.net/10339/82252.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Ronald Alvin. “Design, Synthesis, and Screening of Novel Phosphatidylinositol 3-Kinase (PI3K) Inhibitors Activated by Prostate Specific Antigen (PSA): Prodrug Treatment for Androgen-Independent Prostate Cancer.” 2017. Web. 04 Jun 2020.

Vancouver:

Nelson RA. Design, Synthesis, and Screening of Novel Phosphatidylinositol 3-Kinase (PI3K) Inhibitors Activated by Prostate Specific Antigen (PSA): Prodrug Treatment for Androgen-Independent Prostate Cancer. [Internet] [Thesis]. Wake Forest University; 2017. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10339/82252.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson RA. Design, Synthesis, and Screening of Novel Phosphatidylinositol 3-Kinase (PI3K) Inhibitors Activated by Prostate Specific Antigen (PSA): Prodrug Treatment for Androgen-Independent Prostate Cancer. [Thesis]. Wake Forest University; 2017. Available from: http://hdl.handle.net/10339/82252

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

5. Charette, Scott. Characterization of the Drosophila Tor Complex II Subunit Sin1 in Growth and Cellular Signaling.

Degree: PhD, 2010, University of Rochester

 Growth is an integral part of development in higher eukarya. It is a tightly controlled process regulated by both environmental and nutritional cues. Tor is… (more)

Subjects/Keywords: Sin1; TORC2; Akt

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APA (6th Edition):

Charette, S. (2010). Characterization of the Drosophila Tor Complex II Subunit Sin1 in Growth and Cellular Signaling. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/9864

Chicago Manual of Style (16th Edition):

Charette, Scott. “Characterization of the Drosophila Tor Complex II Subunit Sin1 in Growth and Cellular Signaling.” 2010. Doctoral Dissertation, University of Rochester. Accessed June 04, 2020. http://hdl.handle.net/1802/9864.

MLA Handbook (7th Edition):

Charette, Scott. “Characterization of the Drosophila Tor Complex II Subunit Sin1 in Growth and Cellular Signaling.” 2010. Web. 04 Jun 2020.

Vancouver:

Charette S. Characterization of the Drosophila Tor Complex II Subunit Sin1 in Growth and Cellular Signaling. [Internet] [Doctoral dissertation]. University of Rochester; 2010. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/1802/9864.

Council of Science Editors:

Charette S. Characterization of the Drosophila Tor Complex II Subunit Sin1 in Growth and Cellular Signaling. [Doctoral Dissertation]. University of Rochester; 2010. Available from: http://hdl.handle.net/1802/9864


Penn State University

6. Ye, Xin. GROWTH CONTROL BY HIPPO AND AKT SIGNALING PATHWAYS.

Degree: PhD, Genetics, 2010, Penn State University

 ABSTRACT Coordinated cell proliferation, cell growth and cell death are required to control normal organ size in multicellular organisms. The perturbations of the mechanisms that… (more)

Subjects/Keywords: growth control; Hippo; Akt; Drosophila

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APA (6th Edition):

Ye, X. (2010). GROWTH CONTROL BY HIPPO AND AKT SIGNALING PATHWAYS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11012

Chicago Manual of Style (16th Edition):

Ye, Xin. “GROWTH CONTROL BY HIPPO AND AKT SIGNALING PATHWAYS.” 2010. Doctoral Dissertation, Penn State University. Accessed June 04, 2020. https://etda.libraries.psu.edu/catalog/11012.

MLA Handbook (7th Edition):

Ye, Xin. “GROWTH CONTROL BY HIPPO AND AKT SIGNALING PATHWAYS.” 2010. Web. 04 Jun 2020.

Vancouver:

Ye X. GROWTH CONTROL BY HIPPO AND AKT SIGNALING PATHWAYS. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Jun 04]. Available from: https://etda.libraries.psu.edu/catalog/11012.

Council of Science Editors:

Ye X. GROWTH CONTROL BY HIPPO AND AKT SIGNALING PATHWAYS. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11012


NSYSU

7. Tsai, Ching-yi. Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death.

Degree: PhD, Biological Sciences, 2009, NSYSU

 As the origin of a âlife-and-deathâ signal that reflects central cardiovascular regulatory failure during brain stem death, the rostral ventrolateral medulla (RVLM) is a suitable… (more)

Subjects/Keywords: PKA; Akt; PI3K; mevinphos

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APA (6th Edition):

Tsai, C. (2009). Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732

Chicago Manual of Style (16th Edition):

Tsai, Ching-yi. “Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death.” 2009. Doctoral Dissertation, NSYSU. Accessed June 04, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732.

MLA Handbook (7th Edition):

Tsai, Ching-yi. “Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death.” 2009. Web. 04 Jun 2020.

Vancouver:

Tsai C. Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death. [Internet] [Doctoral dissertation]. NSYSU; 2009. [cited 2020 Jun 04]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732.

Council of Science Editors:

Tsai C. Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death. [Doctoral Dissertation]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732


NSYSU

8. Chuang, Chun-Wei. Study of the molecular mechanism by which COX-2 regulates CCR7 expression.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 The metastatic spread of tumor cells is the major lethal aspect of cancer, and lymphatic metastasis is one of the most important routes. Recent studies… (more)

Subjects/Keywords: Sp1; CCR7; AKT; COX-2

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APA (6th Edition):

Chuang, C. (2010). Study of the molecular mechanism by which COX-2 regulates CCR7 expression. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chuang, Chun-Wei. “Study of the molecular mechanism by which COX-2 regulates CCR7 expression.” 2010. Thesis, NSYSU. Accessed June 04, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chuang, Chun-Wei. “Study of the molecular mechanism by which COX-2 regulates CCR7 expression.” 2010. Web. 04 Jun 2020.

Vancouver:

Chuang C. Study of the molecular mechanism by which COX-2 regulates CCR7 expression. [Internet] [Thesis]. NSYSU; 2010. [cited 2020 Jun 04]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chuang C. Study of the molecular mechanism by which COX-2 regulates CCR7 expression. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

9. LEE, HSIAO-CHEN. Mitochondrial Akt Signaling and Embryonic Stem Cell Differentiation.

Degree: Biomedical Sciences, 2018, University of California – Irvine

 The ability of self-renewal and the unlimited potential to differentiate into three germ layers make pluripotent stem cells a key player in regenerative medicine and… (more)

Subjects/Keywords: Pathology; Akt; Mitochondria; Stem cell

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APA (6th Edition):

LEE, H. (2018). Mitochondrial Akt Signaling and Embryonic Stem Cell Differentiation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/9wt8v9t5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LEE, HSIAO-CHEN. “Mitochondrial Akt Signaling and Embryonic Stem Cell Differentiation.” 2018. Thesis, University of California – Irvine. Accessed June 04, 2020. http://www.escholarship.org/uc/item/9wt8v9t5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LEE, HSIAO-CHEN. “Mitochondrial Akt Signaling and Embryonic Stem Cell Differentiation.” 2018. Web. 04 Jun 2020.

Vancouver:

LEE H. Mitochondrial Akt Signaling and Embryonic Stem Cell Differentiation. [Internet] [Thesis]. University of California – Irvine; 2018. [cited 2020 Jun 04]. Available from: http://www.escholarship.org/uc/item/9wt8v9t5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LEE H. Mitochondrial Akt Signaling and Embryonic Stem Cell Differentiation. [Thesis]. University of California – Irvine; 2018. Available from: http://www.escholarship.org/uc/item/9wt8v9t5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

10. Phua, Yuwei. MicroRNA-184 (miR-184) represses tumour progression by negatively regulating the AKT/mTORC1 protein synthesis pathway.

Degree: Clinical School - St Vincent's Hospital, 2013, University of New South Wales

 Breast cancer patients who suffer from a relapse and develop metastatic disease are currently incurable due a lack of targeted therapies, in particular towards the… (more)

Subjects/Keywords: AKT/mTORC1; MicroRNA; Breast cancer

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APA (6th Edition):

Phua, Y. (2013). MicroRNA-184 (miR-184) represses tumour progression by negatively regulating the AKT/mTORC1 protein synthesis pathway. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53535 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12230/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Phua, Yuwei. “MicroRNA-184 (miR-184) represses tumour progression by negatively regulating the AKT/mTORC1 protein synthesis pathway.” 2013. Doctoral Dissertation, University of New South Wales. Accessed June 04, 2020. http://handle.unsw.edu.au/1959.4/53535 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12230/SOURCE02?view=true.

MLA Handbook (7th Edition):

Phua, Yuwei. “MicroRNA-184 (miR-184) represses tumour progression by negatively regulating the AKT/mTORC1 protein synthesis pathway.” 2013. Web. 04 Jun 2020.

Vancouver:

Phua Y. MicroRNA-184 (miR-184) represses tumour progression by negatively regulating the AKT/mTORC1 protein synthesis pathway. [Internet] [Doctoral dissertation]. University of New South Wales; 2013. [cited 2020 Jun 04]. Available from: http://handle.unsw.edu.au/1959.4/53535 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12230/SOURCE02?view=true.

Council of Science Editors:

Phua Y. MicroRNA-184 (miR-184) represses tumour progression by negatively regulating the AKT/mTORC1 protein synthesis pathway. [Doctoral Dissertation]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/53535 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12230/SOURCE02?view=true


University of Illinois – Chicago

11. Chen, Xinyu. The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis.

Degree: 2018, University of Illinois – Chicago

 Our previous results suggest that pan-Akt inhibition could exert toxicity indicating that in order to move forward with Akt inhibition for cancer therapy selective Akt(more)

Subjects/Keywords: Akt; Breast Cancer; Metastasis

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APA (6th Edition):

Chen, X. (2018). The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Xinyu. “The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis.” 2018. Thesis, University of Illinois – Chicago. Accessed June 04, 2020. http://hdl.handle.net/10027/23330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Xinyu. “The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis.” 2018. Web. 04 Jun 2020.

Vancouver:

Chen X. The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10027/23330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen X. The Function of Akt Isoforms in Breast Cancer Tumorigenesis, Progression and Metastasis. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/23330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

12. Hu, Yusi. Development of Membrane-Protein Interaction Modulators as Allosteric Drug Candidates.

Degree: 2019, University of Illinois – Chicago

 The serine/threonine kinase Akt, also known as protein kinase B (PKB), plays a critical role in regulating diverse cellular functions, including cell survival, proliferation, migration… (more)

Subjects/Keywords: Akt Zap70 Allosteric Inhibitors

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APA (6th Edition):

Hu, Y. (2019). Development of Membrane-Protein Interaction Modulators as Allosteric Drug Candidates. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23733

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Yusi. “Development of Membrane-Protein Interaction Modulators as Allosteric Drug Candidates.” 2019. Thesis, University of Illinois – Chicago. Accessed June 04, 2020. http://hdl.handle.net/10027/23733.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Yusi. “Development of Membrane-Protein Interaction Modulators as Allosteric Drug Candidates.” 2019. Web. 04 Jun 2020.

Vancouver:

Hu Y. Development of Membrane-Protein Interaction Modulators as Allosteric Drug Candidates. [Internet] [Thesis]. University of Illinois – Chicago; 2019. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10027/23733.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu Y. Development of Membrane-Protein Interaction Modulators as Allosteric Drug Candidates. [Thesis]. University of Illinois – Chicago; 2019. Available from: http://hdl.handle.net/10027/23733

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

13. Mahesaniya, Afreeda. Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway .

Degree: 2017, University of Guelph

 Chronic kidney disease (CKD) is a debilitating illness which is increasing in frequency in the Canadian population as a consequence of the diabetes epidemic. Physicians… (more)

Subjects/Keywords: Soy; phytoestrogens; Akt; Podocyte; Nephrin

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APA (6th Edition):

Mahesaniya, A. (2017). Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mahesaniya, Afreeda. “Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway .” 2017. Thesis, University of Guelph. Accessed June 04, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mahesaniya, Afreeda. “Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway .” 2017. Web. 04 Jun 2020.

Vancouver:

Mahesaniya A. Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway . [Internet] [Thesis]. University of Guelph; 2017. [cited 2020 Jun 04]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mahesaniya A. Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway . [Thesis]. University of Guelph; 2017. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

14. Wey, Shiuan. The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2013, University of Southern California

 The endoplasmic reticulum (ER) is an intracellular organelle for protein folding, lipid synthesis and Ca²⁺ storage. It is also responsible for the transportation for most… (more)

Subjects/Keywords: GRP78; AKT; PTEN; leukemia; hematopoiesis

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APA (6th Edition):

Wey, S. (2013). The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218

Chicago Manual of Style (16th Edition):

Wey, Shiuan. “The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis.” 2013. Doctoral Dissertation, University of Southern California. Accessed June 04, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218.

MLA Handbook (7th Edition):

Wey, Shiuan. “The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis.” 2013. Web. 04 Jun 2020.

Vancouver:

Wey S. The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2020 Jun 04]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218.

Council of Science Editors:

Wey S. The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218


University of Rochester

15. Chugh, Pauline E. Elucidation of the mechanism involved in long-term survival of HIV-1 infected macrophages: A new strategy targeting HIV reservoirs.

Degree: PhD, 2009, University of Rochester

 The interaction of Human Immunodeficiency Virus Type 1 (HIV-1) with target CD4+ T lymphocytes is well-studied and typically leads to virally-induced cytolysis, resulting in the… (more)

Subjects/Keywords: HIV-1; Reservoirs; AKT; Macrophages; Tat; AKT Inhibitors; Survival

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APA (6th Edition):

Chugh, P. E. (2009). Elucidation of the mechanism involved in long-term survival of HIV-1 infected macrophages: A new strategy targeting HIV reservoirs. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6628

Chicago Manual of Style (16th Edition):

Chugh, Pauline E. “Elucidation of the mechanism involved in long-term survival of HIV-1 infected macrophages: A new strategy targeting HIV reservoirs.” 2009. Doctoral Dissertation, University of Rochester. Accessed June 04, 2020. http://hdl.handle.net/1802/6628.

MLA Handbook (7th Edition):

Chugh, Pauline E. “Elucidation of the mechanism involved in long-term survival of HIV-1 infected macrophages: A new strategy targeting HIV reservoirs.” 2009. Web. 04 Jun 2020.

Vancouver:

Chugh PE. Elucidation of the mechanism involved in long-term survival of HIV-1 infected macrophages: A new strategy targeting HIV reservoirs. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/1802/6628.

Council of Science Editors:

Chugh PE. Elucidation of the mechanism involved in long-term survival of HIV-1 infected macrophages: A new strategy targeting HIV reservoirs. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6628

16. Houssaini, Amal. La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension.

Degree: Docteur es, Physiopathologie, 2012, Université Paris-Est

 Les travaux de la thèse portent sur l'implication de la voie de signalisation Akt (sérine/thréonine kinase Akt) et mTOR (mammalian target of rapamycin) dans la… (more)

Subjects/Keywords: Akt; Mtor; Htap; Proliferation; Akt; Mtor; Pah; Proliferation

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APA (6th Edition):

Houssaini, A. (2012). La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0069

Chicago Manual of Style (16th Edition):

Houssaini, Amal. “La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed June 04, 2020. http://www.theses.fr/2012PEST0069.

MLA Handbook (7th Edition):

Houssaini, Amal. “La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension.” 2012. Web. 04 Jun 2020.

Vancouver:

Houssaini A. La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2020 Jun 04]. Available from: http://www.theses.fr/2012PEST0069.

Council of Science Editors:

Houssaini A. La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0069

17. Baliou, Evangelia. Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού.

Degree: 2018, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

PI3K/Akt pathway is one of the most important signal transduction pathways activated by many stimuli, such as hormones and growth factors. It’s role in modulating… (more)

Subjects/Keywords: Μονοπάτι PI3K/Akt; Μαστός; Pathway PI3K/Akt; Breast

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APA (6th Edition):

Baliou, E. (2018). Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/44625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baliou, Evangelia. “Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού.” 2018. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed June 04, 2020. http://hdl.handle.net/10442/hedi/44625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baliou, Evangelia. “Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού.” 2018. Web. 04 Jun 2020.

Vancouver:

Baliou E. Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10442/hedi/44625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baliou E. Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. Available from: http://hdl.handle.net/10442/hedi/44625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

18. WOODMAN, KERYN. Increasing Akt signalling protects dystrophic skeletal muscle.

Degree: 2015, University of Melbourne

 The muscular dystrophies are a group of genetic neuromuscular disorders that result in the progressive deterioration of skeletal muscle. Duchenne muscular dystrophy (DMD) is the… (more)

Subjects/Keywords: muscle; dystrophy; mdx; Akt signalling; Akt, benfotiamine, mTOR, resveratrol

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APA (6th Edition):

WOODMAN, K. (2015). Increasing Akt signalling protects dystrophic skeletal muscle. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55494

Chicago Manual of Style (16th Edition):

WOODMAN, KERYN. “Increasing Akt signalling protects dystrophic skeletal muscle.” 2015. Doctoral Dissertation, University of Melbourne. Accessed June 04, 2020. http://hdl.handle.net/11343/55494.

MLA Handbook (7th Edition):

WOODMAN, KERYN. “Increasing Akt signalling protects dystrophic skeletal muscle.” 2015. Web. 04 Jun 2020.

Vancouver:

WOODMAN K. Increasing Akt signalling protects dystrophic skeletal muscle. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/11343/55494.

Council of Science Editors:

WOODMAN K. Increasing Akt signalling protects dystrophic skeletal muscle. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55494


Virginia Tech

19. Qin, Qizhi. Evaluation of the therapeutic potential of Akt inhibition in a translational model of histiocytic sarcoma.

Degree: PhD, Biomedical and Veterinary Sciences, 2018, Virginia Tech

 Histiocytic sarcoma (HS) is an exceptionally rare malignant neoplasm derived from dendritic cells and histiocytes, with no available effective treatment options. Akt signaling and proteasome… (more)

Subjects/Keywords: Histiocytic sarcoma; PI3K/Akt signaling; Akt-targeted therapy

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APA (6th Edition):

Qin, Q. (2018). Evaluation of the therapeutic potential of Akt inhibition in a translational model of histiocytic sarcoma. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/97520

Chicago Manual of Style (16th Edition):

Qin, Qizhi. “Evaluation of the therapeutic potential of Akt inhibition in a translational model of histiocytic sarcoma.” 2018. Doctoral Dissertation, Virginia Tech. Accessed June 04, 2020. http://hdl.handle.net/10919/97520.

MLA Handbook (7th Edition):

Qin, Qizhi. “Evaluation of the therapeutic potential of Akt inhibition in a translational model of histiocytic sarcoma.” 2018. Web. 04 Jun 2020.

Vancouver:

Qin Q. Evaluation of the therapeutic potential of Akt inhibition in a translational model of histiocytic sarcoma. [Internet] [Doctoral dissertation]. Virginia Tech; 2018. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10919/97520.

Council of Science Editors:

Qin Q. Evaluation of the therapeutic potential of Akt inhibition in a translational model of histiocytic sarcoma. [Doctoral Dissertation]. Virginia Tech; 2018. Available from: http://hdl.handle.net/10919/97520

20. Pizon, Mathieu. L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis.

Degree: Docteur es, Sciences, technologie, santé. Microbiologie, 2010, Université de Bordeaux Segalen

Le CD95 appartient à la famille du TNF-R. Il est capable de déclencher un signal apoptotique et joue un rôle prépondérant dans le maintien de… (more)

Subjects/Keywords: Apoptose; Cd95; Pi3k; Akt; Microdomaines; Apoptosis; Microdomains

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APA (6th Edition):

Pizon, M. (2010). L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2010BOR21710

Chicago Manual of Style (16th Edition):

Pizon, Mathieu. “L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis.” 2010. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed June 04, 2020. http://www.theses.fr/2010BOR21710.

MLA Handbook (7th Edition):

Pizon, Mathieu. “L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis.” 2010. Web. 04 Jun 2020.

Vancouver:

Pizon M. L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2010. [cited 2020 Jun 04]. Available from: http://www.theses.fr/2010BOR21710.

Council of Science Editors:

Pizon M. L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2010. Available from: http://www.theses.fr/2010BOR21710


Penn State University

21. Facompre, Nicole Danielle. Organoselenium-mediated alteration of prostate cancer cell signaling pathways.

Degree: PhD, Biochemistry and Molecular Biology, 2010, Penn State University

 Prostate cancer is the most commonly diagnosed malignancy and second leading cause of cancer related death in men in the United States. The lack of… (more)

Subjects/Keywords: androgen receptor; selenium; prostate cancer; Akt

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APA (6th Edition):

Facompre, N. D. (2010). Organoselenium-mediated alteration of prostate cancer cell signaling pathways. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11123

Chicago Manual of Style (16th Edition):

Facompre, Nicole Danielle. “Organoselenium-mediated alteration of prostate cancer cell signaling pathways.” 2010. Doctoral Dissertation, Penn State University. Accessed June 04, 2020. https://etda.libraries.psu.edu/catalog/11123.

MLA Handbook (7th Edition):

Facompre, Nicole Danielle. “Organoselenium-mediated alteration of prostate cancer cell signaling pathways.” 2010. Web. 04 Jun 2020.

Vancouver:

Facompre ND. Organoselenium-mediated alteration of prostate cancer cell signaling pathways. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Jun 04]. Available from: https://etda.libraries.psu.edu/catalog/11123.

Council of Science Editors:

Facompre ND. Organoselenium-mediated alteration of prostate cancer cell signaling pathways. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11123


University of California – Berkeley

22. Weilhammer, Dina. Host factor regulation of Toxoplasma gondii growth and differentiation.

Degree: Molecular & Cell Biology, 2010, University of California – Berkeley

 Toxoplasma gondii is an obligate intracellular protozoan parasite that infects a wide range of mammalian and avian hosts, including up to one third of the… (more)

Subjects/Keywords: Biology, Molecular; Akt; cell-extrinsic; glycolysis; Toxoplasma

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APA (6th Edition):

Weilhammer, D. (2010). Host factor regulation of Toxoplasma gondii growth and differentiation. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/0kv4540j

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weilhammer, Dina. “Host factor regulation of Toxoplasma gondii growth and differentiation.” 2010. Thesis, University of California – Berkeley. Accessed June 04, 2020. http://www.escholarship.org/uc/item/0kv4540j.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weilhammer, Dina. “Host factor regulation of Toxoplasma gondii growth and differentiation.” 2010. Web. 04 Jun 2020.

Vancouver:

Weilhammer D. Host factor regulation of Toxoplasma gondii growth and differentiation. [Internet] [Thesis]. University of California – Berkeley; 2010. [cited 2020 Jun 04]. Available from: http://www.escholarship.org/uc/item/0kv4540j.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weilhammer D. Host factor regulation of Toxoplasma gondii growth and differentiation. [Thesis]. University of California – Berkeley; 2010. Available from: http://www.escholarship.org/uc/item/0kv4540j

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

23. Norris, Dougall. Mechanistic Insights into Insulin-Stimulated Akt Activation and GLUT4 Response Heterogeneity .

Degree: 2017, University of Sydney

 Glucose Transporter 4 (GLUT4) undergoes membrane translocation in response to insulin, facilitating uptake of glucose from the blood into muscle and fat, a process that… (more)

Subjects/Keywords: Akt; GLUT4; insulin; signalling; imaging; microscopy

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APA (6th Edition):

Norris, D. (2017). Mechanistic Insights into Insulin-Stimulated Akt Activation and GLUT4 Response Heterogeneity . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17943

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Norris, Dougall. “Mechanistic Insights into Insulin-Stimulated Akt Activation and GLUT4 Response Heterogeneity .” 2017. Thesis, University of Sydney. Accessed June 04, 2020. http://hdl.handle.net/2123/17943.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Norris, Dougall. “Mechanistic Insights into Insulin-Stimulated Akt Activation and GLUT4 Response Heterogeneity .” 2017. Web. 04 Jun 2020.

Vancouver:

Norris D. Mechanistic Insights into Insulin-Stimulated Akt Activation and GLUT4 Response Heterogeneity . [Internet] [Thesis]. University of Sydney; 2017. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/2123/17943.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Norris D. Mechanistic Insights into Insulin-Stimulated Akt Activation and GLUT4 Response Heterogeneity . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17943

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

24. Wagner, Melany. Herpes Simplex Virus Requires VP11/12 to Activate Src Family Kinase-PI3 Kinase-Akt Signalling.

Degree: PhD, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 This thesis defines a novel role for the Herpes Simplex Virus (HSV) tegument protein, virion protein (VP) 11/12 as a modulator of host cell signalling.… (more)

Subjects/Keywords: Akt; PI3 Kinase; VP11/12; Lck; Herpes

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APA (6th Edition):

Wagner, M. (2010). Herpes Simplex Virus Requires VP11/12 to Activate Src Family Kinase-PI3 Kinase-Akt Signalling. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/pz50gx756

Chicago Manual of Style (16th Edition):

Wagner, Melany. “Herpes Simplex Virus Requires VP11/12 to Activate Src Family Kinase-PI3 Kinase-Akt Signalling.” 2010. Doctoral Dissertation, University of Alberta. Accessed June 04, 2020. https://era.library.ualberta.ca/files/pz50gx756.

MLA Handbook (7th Edition):

Wagner, Melany. “Herpes Simplex Virus Requires VP11/12 to Activate Src Family Kinase-PI3 Kinase-Akt Signalling.” 2010. Web. 04 Jun 2020.

Vancouver:

Wagner M. Herpes Simplex Virus Requires VP11/12 to Activate Src Family Kinase-PI3 Kinase-Akt Signalling. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2020 Jun 04]. Available from: https://era.library.ualberta.ca/files/pz50gx756.

Council of Science Editors:

Wagner M. Herpes Simplex Virus Requires VP11/12 to Activate Src Family Kinase-PI3 Kinase-Akt Signalling. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/pz50gx756


University of Alberta

25. Urbanowski, Matthew D. Characterization of the anti-apoptotic properties of flavivirus capsid proteins.

Degree: PhD, Department of Cell Biology, 2014, University of Alberta

 The introduction of WNV into North America in 1999 was followed by rapid spread throughout the continent. Today, WNV is an endemic pathogen in the… (more)

Subjects/Keywords: apoptosis; virus; capsid; PI3K; flavivirus; akt

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APA (6th Edition):

Urbanowski, M. D. (2014). Characterization of the anti-apoptotic properties of flavivirus capsid proteins. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/sb3978635

Chicago Manual of Style (16th Edition):

Urbanowski, Matthew D. “Characterization of the anti-apoptotic properties of flavivirus capsid proteins.” 2014. Doctoral Dissertation, University of Alberta. Accessed June 04, 2020. https://era.library.ualberta.ca/files/sb3978635.

MLA Handbook (7th Edition):

Urbanowski, Matthew D. “Characterization of the anti-apoptotic properties of flavivirus capsid proteins.” 2014. Web. 04 Jun 2020.

Vancouver:

Urbanowski MD. Characterization of the anti-apoptotic properties of flavivirus capsid proteins. [Internet] [Doctoral dissertation]. University of Alberta; 2014. [cited 2020 Jun 04]. Available from: https://era.library.ualberta.ca/files/sb3978635.

Council of Science Editors:

Urbanowski MD. Characterization of the anti-apoptotic properties of flavivirus capsid proteins. [Doctoral Dissertation]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/sb3978635

26. Zeybek, Ayça. Essai du traitement pré-clinique du carcinome hépatocellulaire sur la cirrhose dans le modèle de rat : Pre-trial of hepatocellular carcinoma on cirrhosis in a rat model.

Degree: Docteur es, Chimie Biologie, 2016, Grenoble Alpes; İzmir Yüksek Teknoloji Enstitüsü (Izmir, Turquie)

Hepatocellular carcinoma (HCC) is the second most common cause of cancerrelated mortality worldwide. AKT pathway has been found activated in 50% of HCC cases, making… (more)

Subjects/Keywords: Hepatocellular carcinoma; Sorafenib; AKT inhibitor; PI3K/AKT/mTOR pathway; Rat model; Den; Hepatocellular carcinoma; Sorafenib; AKT inhibitor; PI3K/AKT/mTOR pathway; Rat model; Den; 540

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zeybek, A. (2016). Essai du traitement pré-clinique du carcinome hépatocellulaire sur la cirrhose dans le modèle de rat : Pre-trial of hepatocellular carcinoma on cirrhosis in a rat model. (Doctoral Dissertation). Grenoble Alpes; İzmir Yüksek Teknoloji Enstitüsü (Izmir, Turquie). Retrieved from http://www.theses.fr/2016GREAV054

Chicago Manual of Style (16th Edition):

Zeybek, Ayça. “Essai du traitement pré-clinique du carcinome hépatocellulaire sur la cirrhose dans le modèle de rat : Pre-trial of hepatocellular carcinoma on cirrhosis in a rat model.” 2016. Doctoral Dissertation, Grenoble Alpes; İzmir Yüksek Teknoloji Enstitüsü (Izmir, Turquie). Accessed June 04, 2020. http://www.theses.fr/2016GREAV054.

MLA Handbook (7th Edition):

Zeybek, Ayça. “Essai du traitement pré-clinique du carcinome hépatocellulaire sur la cirrhose dans le modèle de rat : Pre-trial of hepatocellular carcinoma on cirrhosis in a rat model.” 2016. Web. 04 Jun 2020.

Vancouver:

Zeybek A. Essai du traitement pré-clinique du carcinome hépatocellulaire sur la cirrhose dans le modèle de rat : Pre-trial of hepatocellular carcinoma on cirrhosis in a rat model. [Internet] [Doctoral dissertation]. Grenoble Alpes; İzmir Yüksek Teknoloji Enstitüsü (Izmir, Turquie); 2016. [cited 2020 Jun 04]. Available from: http://www.theses.fr/2016GREAV054.

Council of Science Editors:

Zeybek A. Essai du traitement pré-clinique du carcinome hépatocellulaire sur la cirrhose dans le modèle de rat : Pre-trial of hepatocellular carcinoma on cirrhosis in a rat model. [Doctoral Dissertation]. Grenoble Alpes; İzmir Yüksek Teknoloji Enstitüsü (Izmir, Turquie); 2016. Available from: http://www.theses.fr/2016GREAV054


NSYSU

27. Tseng, Yu-chen. Functional and Receptor-interacting Studies of Hepatoma-derived growth factor (HDGF).

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

 Hepatoma-derived growth factor (HDGF) is a nuclear protein with both mitogenic and angiogenic activity that trigger tumor progression in various types of cancer including hepatocellular… (more)

Subjects/Keywords: Hepatoma; HDGF; Akt pathway; Epithelialâmesenchymal transition

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tseng, Y. (2013). Functional and Receptor-interacting Studies of Hepatoma-derived growth factor (HDGF). (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623113-200356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tseng, Yu-chen. “Functional and Receptor-interacting Studies of Hepatoma-derived growth factor (HDGF).” 2013. Thesis, NSYSU. Accessed June 04, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623113-200356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tseng, Yu-chen. “Functional and Receptor-interacting Studies of Hepatoma-derived growth factor (HDGF).” 2013. Web. 04 Jun 2020.

Vancouver:

Tseng Y. Functional and Receptor-interacting Studies of Hepatoma-derived growth factor (HDGF). [Internet] [Thesis]. NSYSU; 2013. [cited 2020 Jun 04]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623113-200356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tseng Y. Functional and Receptor-interacting Studies of Hepatoma-derived growth factor (HDGF). [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623113-200356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

28. Arsenault, Marie-Pier. Étude de facteurs génétiques prédictifs dans le neuroblastome, en particulier les anomalies du chromosmoe 14q .

Degree: 2011, Université de Montréal

 Le neuroblastome (NB) représente 8% de tous les cancers pédiatriques et est caractérisé par sa grande hétérogénéité clinique. Afin d’évaluer son pronostic, plusieurs facteurs génétiques… (more)

Subjects/Keywords: Neuroblastome; Neuroblastoma; FISH; SNP; 14q; AKT

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arsenault, M. (2011). Étude de facteurs génétiques prédictifs dans le neuroblastome, en particulier les anomalies du chromosmoe 14q . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/4867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arsenault, Marie-Pier. “Étude de facteurs génétiques prédictifs dans le neuroblastome, en particulier les anomalies du chromosmoe 14q .” 2011. Thesis, Université de Montréal. Accessed June 04, 2020. http://hdl.handle.net/1866/4867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arsenault, Marie-Pier. “Étude de facteurs génétiques prédictifs dans le neuroblastome, en particulier les anomalies du chromosmoe 14q .” 2011. Web. 04 Jun 2020.

Vancouver:

Arsenault M. Étude de facteurs génétiques prédictifs dans le neuroblastome, en particulier les anomalies du chromosmoe 14q . [Internet] [Thesis]. Université de Montréal; 2011. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/1866/4867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arsenault M. Étude de facteurs génétiques prédictifs dans le neuroblastome, en particulier les anomalies du chromosmoe 14q . [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/4867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. 馬, 良. Dual targeting of heat shock proteins 90 and 70 promotes cell death and enhances the anticancer effect of chemotherapeutic agents in bladder cancer.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Heat shock proteins (HSPs), which are molecular chaperones that stabilize numerous vital proteins, may be attractive targets for cancer therapy. The aim of the present… (more)

Subjects/Keywords: HSP90; HSP70; Akt; bladder cancer; chemotherapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

馬, . (2016). Dual targeting of heat shock proteins 90 and 70 promotes cell death and enhances the anticancer effect of chemotherapeutic agents in bladder cancer. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

馬, 良. “Dual targeting of heat shock proteins 90 and 70 promotes cell death and enhances the anticancer effect of chemotherapeutic agents in bladder cancer.” 2016. Thesis, Oita University / 大分大学. Accessed June 04, 2020. http://hdl.handle.net/10559/15614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

馬, 良. “Dual targeting of heat shock proteins 90 and 70 promotes cell death and enhances the anticancer effect of chemotherapeutic agents in bladder cancer.” 2016. Web. 04 Jun 2020.

Vancouver:

馬 . Dual targeting of heat shock proteins 90 and 70 promotes cell death and enhances the anticancer effect of chemotherapeutic agents in bladder cancer. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2020 Jun 04]. Available from: http://hdl.handle.net/10559/15614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

馬 . Dual targeting of heat shock proteins 90 and 70 promotes cell death and enhances the anticancer effect of chemotherapeutic agents in bladder cancer. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

30. Paramo Sanchez, Blanca Estela. The role of Sin1 in cell survival.

Degree: 2015, University of Manchester

 Cancer and neurodegeneration are detrimental conditions associated with an inappropriate regulation of cell survival and cell death, causing compromised cells to evade death or excessive… (more)

Subjects/Keywords: survival; apoptosis; necroptosis; mTORC2 Akt; neuronal survival

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paramo Sanchez, B. E. (2015). The role of Sin1 in cell survival. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:263792

Chicago Manual of Style (16th Edition):

Paramo Sanchez, Blanca Estela. “The role of Sin1 in cell survival.” 2015. Doctoral Dissertation, University of Manchester. Accessed June 04, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:263792.

MLA Handbook (7th Edition):

Paramo Sanchez, Blanca Estela. “The role of Sin1 in cell survival.” 2015. Web. 04 Jun 2020.

Vancouver:

Paramo Sanchez BE. The role of Sin1 in cell survival. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2020 Jun 04]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:263792.

Council of Science Editors:

Paramo Sanchez BE. The role of Sin1 in cell survival. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:263792

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