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You searched for subject:(Airway Inflammation). Showing records 1 – 30 of 137 total matches.

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University of Manitoba

1. Ryu, Min Hyung. The effect of in-utero-through-postnatal exposure of mice to perfluorinated compounds on airway inflammation and function.

Degree: Physiology and Pathophysiology, 2014, University of Manitoba

 Perfluorinated compounds, non-degradable xenobiotics in many consumer products, can cause developmental toxicity in animals, and human exposure is associated with asthma symptoms. We tested the… (more)

Subjects/Keywords: airway hyperresponsiveness; perfluorinated compounds; environmental pollution; airway inflammation

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APA (6th Edition):

Ryu, M. H. (2014). The effect of in-utero-through-postnatal exposure of mice to perfluorinated compounds on airway inflammation and function. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/30195

Chicago Manual of Style (16th Edition):

Ryu, Min Hyung. “The effect of in-utero-through-postnatal exposure of mice to perfluorinated compounds on airway inflammation and function.” 2014. Masters Thesis, University of Manitoba. Accessed October 25, 2020. http://hdl.handle.net/1993/30195.

MLA Handbook (7th Edition):

Ryu, Min Hyung. “The effect of in-utero-through-postnatal exposure of mice to perfluorinated compounds on airway inflammation and function.” 2014. Web. 25 Oct 2020.

Vancouver:

Ryu MH. The effect of in-utero-through-postnatal exposure of mice to perfluorinated compounds on airway inflammation and function. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1993/30195.

Council of Science Editors:

Ryu MH. The effect of in-utero-through-postnatal exposure of mice to perfluorinated compounds on airway inflammation and function. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/30195


NSYSU

2. Fu, Yaw-syan. Inhibitory Effect of Warm Water Immersion-induced Hyperthermia on Neurogenic Inflammation in Rat Airways and the Possible Mechanisms.

Degree: PhD, Biological Sciences, 2010, NSYSU

 In mammals, the neurogenic inflammatory response can be induced by stimulation or activation on the peripheral sensory C-fibers to release neuropeptides from the peripheral terminals,… (more)

Subjects/Keywords: heat shock response; neurogenic inflammation; airway

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APA (6th Edition):

Fu, Y. (2010). Inhibitory Effect of Warm Water Immersion-induced Hyperthermia on Neurogenic Inflammation in Rat Airways and the Possible Mechanisms. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609110-134507

Chicago Manual of Style (16th Edition):

Fu, Yaw-syan. “Inhibitory Effect of Warm Water Immersion-induced Hyperthermia on Neurogenic Inflammation in Rat Airways and the Possible Mechanisms.” 2010. Doctoral Dissertation, NSYSU. Accessed October 25, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609110-134507.

MLA Handbook (7th Edition):

Fu, Yaw-syan. “Inhibitory Effect of Warm Water Immersion-induced Hyperthermia on Neurogenic Inflammation in Rat Airways and the Possible Mechanisms.” 2010. Web. 25 Oct 2020.

Vancouver:

Fu Y. Inhibitory Effect of Warm Water Immersion-induced Hyperthermia on Neurogenic Inflammation in Rat Airways and the Possible Mechanisms. [Internet] [Doctoral dissertation]. NSYSU; 2010. [cited 2020 Oct 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609110-134507.

Council of Science Editors:

Fu Y. Inhibitory Effect of Warm Water Immersion-induced Hyperthermia on Neurogenic Inflammation in Rat Airways and the Possible Mechanisms. [Doctoral Dissertation]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609110-134507

3. Gaurav, Rohit. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.

Degree: PhD, Biomedical Sciences (graduate program), 2014, Creighton University

 Allergic asthma is a complex and heterogeneous disease of the airways. Eosinophils migrate to the airways under the influence of chemokines to start an excessive… (more)

Subjects/Keywords: Chloride Channels – metabolism; Asthma – immunology; Airway Inflammation

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APA (6th Edition):

Gaurav, R. (2014). Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. (Doctoral Dissertation). Creighton University. Retrieved from http://hdl.handle.net/10504/62868

Chicago Manual of Style (16th Edition):

Gaurav, Rohit. “Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.” 2014. Doctoral Dissertation, Creighton University. Accessed October 25, 2020. http://hdl.handle.net/10504/62868.

MLA Handbook (7th Edition):

Gaurav, Rohit. “Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.” 2014. Web. 25 Oct 2020.

Vancouver:

Gaurav R. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. [Internet] [Doctoral dissertation]. Creighton University; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10504/62868.

Council of Science Editors:

Gaurav R. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. [Doctoral Dissertation]. Creighton University; 2014. Available from: http://hdl.handle.net/10504/62868


University of Adelaide

4. Jayaram, Lata. Airway inflammation, diagnosis, perception of asthma, and sputum zinc levels in a community cohort.

Degree: 2010, University of Adelaide

 Induced sputum examination (IS), an established research tool to measure airway inflammation (AI), is normally confined to specialised institutions and selected populations with airway disease,… (more)

Subjects/Keywords: asthma; airway inflammation; zinc levels in sputum

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APA (6th Edition):

Jayaram, L. (2010). Airway inflammation, diagnosis, perception of asthma, and sputum zinc levels in a community cohort. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/63531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jayaram, Lata. “Airway inflammation, diagnosis, perception of asthma, and sputum zinc levels in a community cohort.” 2010. Thesis, University of Adelaide. Accessed October 25, 2020. http://hdl.handle.net/2440/63531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jayaram, Lata. “Airway inflammation, diagnosis, perception of asthma, and sputum zinc levels in a community cohort.” 2010. Web. 25 Oct 2020.

Vancouver:

Jayaram L. Airway inflammation, diagnosis, perception of asthma, and sputum zinc levels in a community cohort. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2440/63531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jayaram L. Airway inflammation, diagnosis, perception of asthma, and sputum zinc levels in a community cohort. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/63531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

5. Prodanovic, Danica. Novel insights into mechanisms of glucocorticoid actions and sensitivity in the airway epithelium.

Degree: 2018, University of Melbourne

 Glucocorticoids (GCs) remain the frontline treatment in the management of chronic inflammatory diseases, as they are the most potent and effective anti-inflammatory agents available so… (more)

Subjects/Keywords: glucocorticoids; corticosteroids; epithelium; airway epithelium; asthma; inflammation

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APA (6th Edition):

Prodanovic, D. (2018). Novel insights into mechanisms of glucocorticoid actions and sensitivity in the airway epithelium. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/212276

Chicago Manual of Style (16th Edition):

Prodanovic, Danica. “Novel insights into mechanisms of glucocorticoid actions and sensitivity in the airway epithelium.” 2018. Doctoral Dissertation, University of Melbourne. Accessed October 25, 2020. http://hdl.handle.net/11343/212276.

MLA Handbook (7th Edition):

Prodanovic, Danica. “Novel insights into mechanisms of glucocorticoid actions and sensitivity in the airway epithelium.” 2018. Web. 25 Oct 2020.

Vancouver:

Prodanovic D. Novel insights into mechanisms of glucocorticoid actions and sensitivity in the airway epithelium. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/11343/212276.

Council of Science Editors:

Prodanovic D. Novel insights into mechanisms of glucocorticoid actions and sensitivity in the airway epithelium. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/212276


University of Newcastle

6. Essilfie, Ama-Tawiah. Modulation of responses in allergic airways disease by Haemophilus influenzae infection.

Degree: PhD, 2012, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Asthma is a common chronic inflammatory disease of the airways that affects over 2.2 million people in Australia.… (more)

Subjects/Keywords: asthma; airway diseases; eosinophilic inflammation; neutrophilic inflammation; Haemophilus influenzae

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APA (6th Edition):

Essilfie, A. (2012). Modulation of responses in allergic airways disease by Haemophilus influenzae infection. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/927983

Chicago Manual of Style (16th Edition):

Essilfie, Ama-Tawiah. “Modulation of responses in allergic airways disease by Haemophilus influenzae infection.” 2012. Doctoral Dissertation, University of Newcastle. Accessed October 25, 2020. http://hdl.handle.net/1959.13/927983.

MLA Handbook (7th Edition):

Essilfie, Ama-Tawiah. “Modulation of responses in allergic airways disease by Haemophilus influenzae infection.” 2012. Web. 25 Oct 2020.

Vancouver:

Essilfie A. Modulation of responses in allergic airways disease by Haemophilus influenzae infection. [Internet] [Doctoral dissertation]. University of Newcastle; 2012. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1959.13/927983.

Council of Science Editors:

Essilfie A. Modulation of responses in allergic airways disease by Haemophilus influenzae infection. [Doctoral Dissertation]. University of Newcastle; 2012. Available from: http://hdl.handle.net/1959.13/927983


University of Toronto

7. Salehi, Sepehr. The Role of Syk in Airway Hyperresponsiveness and Remodeling in House Dust Mite Induced Murine Models of Allergic Airways Inflammation.

Degree: 2013, University of Toronto

Spleen tyrosine kinase (Syk) plays a critical role in regulation of immune and inflammatory responses. This thesis investigated the role of Syk in the development… (more)

Subjects/Keywords: Allergic Airway Inflammation; Airway Hyperresponsiveness; House Dust Mite; Murine Model; 0680; 0714

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APA (6th Edition):

Salehi, S. (2013). The Role of Syk in Airway Hyperresponsiveness and Remodeling in House Dust Mite Induced Murine Models of Allergic Airways Inflammation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42908

Chicago Manual of Style (16th Edition):

Salehi, Sepehr. “The Role of Syk in Airway Hyperresponsiveness and Remodeling in House Dust Mite Induced Murine Models of Allergic Airways Inflammation.” 2013. Masters Thesis, University of Toronto. Accessed October 25, 2020. http://hdl.handle.net/1807/42908.

MLA Handbook (7th Edition):

Salehi, Sepehr. “The Role of Syk in Airway Hyperresponsiveness and Remodeling in House Dust Mite Induced Murine Models of Allergic Airways Inflammation.” 2013. Web. 25 Oct 2020.

Vancouver:

Salehi S. The Role of Syk in Airway Hyperresponsiveness and Remodeling in House Dust Mite Induced Murine Models of Allergic Airways Inflammation. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1807/42908.

Council of Science Editors:

Salehi S. The Role of Syk in Airway Hyperresponsiveness and Remodeling in House Dust Mite Induced Murine Models of Allergic Airways Inflammation. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42908


University of Manitoba

8. Kashani, Hessam Hassanzadeh. Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells.

Degree: Physiology, 2014, University of Manitoba

 Asthma is a chronic disorder highlighted by intermittent airway inflammation and characterized by paroxysmal dyspnea and airway hyperresponsiveness (AHR). A key feature of severe asthma… (more)

Subjects/Keywords: high mobility group box-1 (HMGB1); airway smooth muscle; fibrosis; inflammation; airway remodeling; asthma

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APA (6th Edition):

Kashani, H. H. (2014). Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/23677

Chicago Manual of Style (16th Edition):

Kashani, Hessam Hassanzadeh. “Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells.” 2014. Masters Thesis, University of Manitoba. Accessed October 25, 2020. http://hdl.handle.net/1993/23677.

MLA Handbook (7th Edition):

Kashani, Hessam Hassanzadeh. “Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells.” 2014. Web. 25 Oct 2020.

Vancouver:

Kashani HH. Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1993/23677.

Council of Science Editors:

Kashani HH. Role of high mobility group box-1 in the pro-fibrotic response of human airway smooth muscle cells. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/23677


University of Manitoba

9. Piyadasa, Hadeesha. Immunomodulatory innate defense regulator (IDR)-1002 peptide in airway inflammation.

Degree: Immunology, 2019, University of Manitoba

 Allergic asthma is a chronic respiratory disease characterized by airway inflammation and hyper-responsiveness (AHR). Despite available treatments such as inhaled corticosteroids (ICS), ~20% patients do… (more)

Subjects/Keywords: Immunomodulatory peptides; Asthma; Airway inflammation; IL-33; Allergy; Airway hyper-responsiveness; IDR peptides

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APA (6th Edition):

Piyadasa, H. (2019). Immunomodulatory innate defense regulator (IDR)-1002 peptide in airway inflammation. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Piyadasa, Hadeesha. “Immunomodulatory innate defense regulator (IDR)-1002 peptide in airway inflammation.” 2019. Thesis, University of Manitoba. Accessed October 25, 2020. http://hdl.handle.net/1993/34464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Piyadasa, Hadeesha. “Immunomodulatory innate defense regulator (IDR)-1002 peptide in airway inflammation.” 2019. Web. 25 Oct 2020.

Vancouver:

Piyadasa H. Immunomodulatory innate defense regulator (IDR)-1002 peptide in airway inflammation. [Internet] [Thesis]. University of Manitoba; 2019. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1993/34464.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Piyadasa H. Immunomodulatory innate defense regulator (IDR)-1002 peptide in airway inflammation. [Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34464

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Scheerens, J. Eotaxin, airway inflammation and hyperresponsiveness.

Degree: 2001, University Utrecht

 Eotaxin is a chemokine involved in migration and activation of inflammatory cells. CC-chemokine receptor-3 (CCR3), the receptor for eotaxin and other chemokines, is expressed by… (more)

Subjects/Keywords: eotaxin; asthma; airway inflammation; airway hyperresponsiveness

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APA (6th Edition):

Scheerens, J. (2001). Eotaxin, airway inflammation and hyperresponsiveness. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/332 ; URN:NBN:NL:UI:10-1874-332 ; URN:NBN:NL:UI:10-1874-332 ; https://dspace.library.uu.nl/handle/1874/332

Chicago Manual of Style (16th Edition):

Scheerens, J. “Eotaxin, airway inflammation and hyperresponsiveness.” 2001. Doctoral Dissertation, University Utrecht. Accessed October 25, 2020. https://dspace.library.uu.nl/handle/1874/332 ; URN:NBN:NL:UI:10-1874-332 ; URN:NBN:NL:UI:10-1874-332 ; https://dspace.library.uu.nl/handle/1874/332.

MLA Handbook (7th Edition):

Scheerens, J. “Eotaxin, airway inflammation and hyperresponsiveness.” 2001. Web. 25 Oct 2020.

Vancouver:

Scheerens J. Eotaxin, airway inflammation and hyperresponsiveness. [Internet] [Doctoral dissertation]. University Utrecht; 2001. [cited 2020 Oct 25]. Available from: https://dspace.library.uu.nl/handle/1874/332 ; URN:NBN:NL:UI:10-1874-332 ; URN:NBN:NL:UI:10-1874-332 ; https://dspace.library.uu.nl/handle/1874/332.

Council of Science Editors:

Scheerens J. Eotaxin, airway inflammation and hyperresponsiveness. [Doctoral Dissertation]. University Utrecht; 2001. Available from: https://dspace.library.uu.nl/handle/1874/332 ; URN:NBN:NL:UI:10-1874-332 ; URN:NBN:NL:UI:10-1874-332 ; https://dspace.library.uu.nl/handle/1874/332


University of Sydney

11. Tonga, Katrina ‘Oto’ota. Complex lung physiology and airway inflammation in adults with asthma and fixed airflow obstruction .

Degree: 2018, University of Sydney

 Background: Fixed airflow obstruction (FAO) occurs in asthma despite adequate treatment and no or minimal smoking history. FAO in asthma is more common in older… (more)

Subjects/Keywords: asthma; fixed airflow obstruction; lung elastic recoil; ventilation heterogeneity; airway inflammation; small airway dysfunction

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APA (6th Edition):

Tonga, K. . (2018). Complex lung physiology and airway inflammation in adults with asthma and fixed airflow obstruction . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tonga, Katrina ‘Oto’ota. “Complex lung physiology and airway inflammation in adults with asthma and fixed airflow obstruction .” 2018. Thesis, University of Sydney. Accessed October 25, 2020. http://hdl.handle.net/2123/20295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tonga, Katrina ‘Oto’ota. “Complex lung physiology and airway inflammation in adults with asthma and fixed airflow obstruction .” 2018. Web. 25 Oct 2020.

Vancouver:

Tonga K. Complex lung physiology and airway inflammation in adults with asthma and fixed airflow obstruction . [Internet] [Thesis]. University of Sydney; 2018. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/2123/20295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tonga K. Complex lung physiology and airway inflammation in adults with asthma and fixed airflow obstruction . [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/20295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

12. Larson, Jennifer Lynn. The Comparison of Airway Responses of Normal Horses Fed Round Bale versus Square Bale Hay.

Degree: MS, Biomedical and Veterinary Sciences, 2012, Virginia Tech

 Background – Feeding horses round bale hay (RBH) has been associated with airway inflammation. The purpose of this study was to determine if horses fed… (more)

Subjects/Keywords: botulism; farmer's lung disease; inflammatoroy airway disease; recurrent airway obstruction; airway neutrophilia; airway inflammation; tracheal aspirate fluid; bronchoalveolar lavage fluid; fungal growth; Round bale hay; square bale hay

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APA (6th Edition):

Larson, J. L. (2012). The Comparison of Airway Responses of Normal Horses Fed Round Bale versus Square Bale Hay. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/43464

Chicago Manual of Style (16th Edition):

Larson, Jennifer Lynn. “The Comparison of Airway Responses of Normal Horses Fed Round Bale versus Square Bale Hay.” 2012. Masters Thesis, Virginia Tech. Accessed October 25, 2020. http://hdl.handle.net/10919/43464.

MLA Handbook (7th Edition):

Larson, Jennifer Lynn. “The Comparison of Airway Responses of Normal Horses Fed Round Bale versus Square Bale Hay.” 2012. Web. 25 Oct 2020.

Vancouver:

Larson JL. The Comparison of Airway Responses of Normal Horses Fed Round Bale versus Square Bale Hay. [Internet] [Masters thesis]. Virginia Tech; 2012. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10919/43464.

Council of Science Editors:

Larson JL. The Comparison of Airway Responses of Normal Horses Fed Round Bale versus Square Bale Hay. [Masters Thesis]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/43464


University of Hong Kong

13. 葉秀文. A pathophysiologic study of airway inflammation in bronchiectasis.

Degree: 1991, University of Hong Kong

Subjects/Keywords: Bronchiectasis.; Airway (Medicine) - Inflammation.

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APA (6th Edition):

葉秀文. (1991). A pathophysiologic study of airway inflammation in bronchiectasis. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/27412

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

葉秀文. “A pathophysiologic study of airway inflammation in bronchiectasis.” 1991. Thesis, University of Hong Kong. Accessed October 25, 2020. http://hdl.handle.net/10722/27412.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

葉秀文. “A pathophysiologic study of airway inflammation in bronchiectasis.” 1991. Web. 25 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

葉秀文. A pathophysiologic study of airway inflammation in bronchiectasis. [Internet] [Thesis]. University of Hong Kong; 1991. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10722/27412.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

葉秀文. A pathophysiologic study of airway inflammation in bronchiectasis. [Thesis]. University of Hong Kong; 1991. Available from: http://hdl.handle.net/10722/27412

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

14. Young Hwang, Ji. Functional Role of Inducible Bronchus-Associated Lymphoid Tissue (iBALT) in Allergic Airway Inflammation.

Degree: PhD, 2016, University of Rochester

 Inducible bronchus-associated lymphoid tissue (iBALT) is an ectopic lymphoid tissue formed in the lung after pulmonary infection or inflammation. This local lymphoid tissue is structurally… (more)

Subjects/Keywords: iBALT; Allergic airway inflammation; OVA; HDM; Transgene; and CXCL13

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APA (6th Edition):

Young Hwang, J. (2016). Functional Role of Inducible Bronchus-Associated Lymphoid Tissue (iBALT) in Allergic Airway Inflammation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30506

Chicago Manual of Style (16th Edition):

Young Hwang, Ji. “Functional Role of Inducible Bronchus-Associated Lymphoid Tissue (iBALT) in Allergic Airway Inflammation.” 2016. Doctoral Dissertation, University of Rochester. Accessed October 25, 2020. http://hdl.handle.net/1802/30506.

MLA Handbook (7th Edition):

Young Hwang, Ji. “Functional Role of Inducible Bronchus-Associated Lymphoid Tissue (iBALT) in Allergic Airway Inflammation.” 2016. Web. 25 Oct 2020.

Vancouver:

Young Hwang J. Functional Role of Inducible Bronchus-Associated Lymphoid Tissue (iBALT) in Allergic Airway Inflammation. [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1802/30506.

Council of Science Editors:

Young Hwang J. Functional Role of Inducible Bronchus-Associated Lymphoid Tissue (iBALT) in Allergic Airway Inflammation. [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/30506


University of Newcastle

15. Fu, Juan-juan. Systemic inflammation in obstructive airway disease.

Degree: PhD, 2014, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Obstructive airway disease (OAD) such as asthma and chronic obstructive pulmonary disease (COPD) are common respiratory conditions affecting… (more)

Subjects/Keywords: systemic inflammation; obstructive airway disease; chronic obstructive pulmonary disease; immune dysfunction

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APA (6th Edition):

Fu, J. (2014). Systemic inflammation in obstructive airway disease. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1050186

Chicago Manual of Style (16th Edition):

Fu, Juan-juan. “Systemic inflammation in obstructive airway disease.” 2014. Doctoral Dissertation, University of Newcastle. Accessed October 25, 2020. http://hdl.handle.net/1959.13/1050186.

MLA Handbook (7th Edition):

Fu, Juan-juan. “Systemic inflammation in obstructive airway disease.” 2014. Web. 25 Oct 2020.

Vancouver:

Fu J. Systemic inflammation in obstructive airway disease. [Internet] [Doctoral dissertation]. University of Newcastle; 2014. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1959.13/1050186.

Council of Science Editors:

Fu J. Systemic inflammation in obstructive airway disease. [Doctoral Dissertation]. University of Newcastle; 2014. Available from: http://hdl.handle.net/1959.13/1050186


University of Vermont

16. Rattu, Gurkiranjit Kaur. Use of a Chemical Chaperone to Attenuate Allergic Airway Inflammation.

Degree: Department of Biology; Department of Pathology and Laboratory Medicine, 2016, University of Vermont

  Asthma is a chronic inflammatory disease characterized by increased airway inflammation and fibrosis. So far, endoplasmic reticulum (ER) stress has been shown to play… (more)

Subjects/Keywords: allergic airway inflammation; asthma; ER stress; immunohistochemistry; TUDCA; periodic-acid schiff

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APA (6th Edition):

Rattu, G. K. (2016). Use of a Chemical Chaperone to Attenuate Allergic Airway Inflammation. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/116

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rattu, Gurkiranjit Kaur. “Use of a Chemical Chaperone to Attenuate Allergic Airway Inflammation.” 2016. Thesis, University of Vermont. Accessed October 25, 2020. https://scholarworks.uvm.edu/hcoltheses/116.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rattu, Gurkiranjit Kaur. “Use of a Chemical Chaperone to Attenuate Allergic Airway Inflammation.” 2016. Web. 25 Oct 2020.

Vancouver:

Rattu GK. Use of a Chemical Chaperone to Attenuate Allergic Airway Inflammation. [Internet] [Thesis]. University of Vermont; 2016. [cited 2020 Oct 25]. Available from: https://scholarworks.uvm.edu/hcoltheses/116.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rattu GK. Use of a Chemical Chaperone to Attenuate Allergic Airway Inflammation. [Thesis]. University of Vermont; 2016. Available from: https://scholarworks.uvm.edu/hcoltheses/116

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queensland University of Technology

17. Hadaway, Matthew. Effect of induced airway inflammation in asthma : the expression of trefoil factors, secretoglobins and genes involved in histone acetylation.

Degree: 2012, Queensland University of Technology

 Asthma is an incapacitating disease of the respiratory system, which causes extensive morbidity and mortality worldwide. Asthma affects more than 300 million people globally(Masoli et… (more)

Subjects/Keywords: airway inflammation; asthma; bronchodilator; corticosteroid; trefoil factor; secretoglobin; histone acetylation

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APA (6th Edition):

Hadaway, M. (2012). Effect of induced airway inflammation in asthma : the expression of trefoil factors, secretoglobins and genes involved in histone acetylation. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/62168/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hadaway, Matthew. “Effect of induced airway inflammation in asthma : the expression of trefoil factors, secretoglobins and genes involved in histone acetylation.” 2012. Thesis, Queensland University of Technology. Accessed October 25, 2020. https://eprints.qut.edu.au/62168/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hadaway, Matthew. “Effect of induced airway inflammation in asthma : the expression of trefoil factors, secretoglobins and genes involved in histone acetylation.” 2012. Web. 25 Oct 2020.

Vancouver:

Hadaway M. Effect of induced airway inflammation in asthma : the expression of trefoil factors, secretoglobins and genes involved in histone acetylation. [Internet] [Thesis]. Queensland University of Technology; 2012. [cited 2020 Oct 25]. Available from: https://eprints.qut.edu.au/62168/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hadaway M. Effect of induced airway inflammation in asthma : the expression of trefoil factors, secretoglobins and genes involved in histone acetylation. [Thesis]. Queensland University of Technology; 2012. Available from: https://eprints.qut.edu.au/62168/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

18. Redhu, Naresh Singh. Molecular regulation and effector functions of the high affinity IgE receptor (FcεRI) in human airway smooth muscle cells.

Degree: Immunology, 2012, University of Manitoba

 The prevalence and economic burden of chronic airway disorders such as asthma is on the rise annually. Allergic asthma is characterized by chronic airway inflammation,… (more)

Subjects/Keywords: asthma; airway smooth muscle; IgE; Fc receptor; TSLP; inflammation

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APA (6th Edition):

Redhu, N. S. (2012). Molecular regulation and effector functions of the high affinity IgE receptor (FcεRI) in human airway smooth muscle cells. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/5305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Redhu, Naresh Singh. “Molecular regulation and effector functions of the high affinity IgE receptor (FcεRI) in human airway smooth muscle cells.” 2012. Thesis, University of Manitoba. Accessed October 25, 2020. http://hdl.handle.net/1993/5305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Redhu, Naresh Singh. “Molecular regulation and effector functions of the high affinity IgE receptor (FcεRI) in human airway smooth muscle cells.” 2012. Web. 25 Oct 2020.

Vancouver:

Redhu NS. Molecular regulation and effector functions of the high affinity IgE receptor (FcεRI) in human airway smooth muscle cells. [Internet] [Thesis]. University of Manitoba; 2012. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1993/5305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Redhu NS. Molecular regulation and effector functions of the high affinity IgE receptor (FcεRI) in human airway smooth muscle cells. [Thesis]. University of Manitoba; 2012. Available from: http://hdl.handle.net/1993/5305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Tham, Rachel. The contribution of outdoor fungal spores to child and adolescent asthma hospitalisations: lung function and airway inflammation.

Degree: 2017, University of Melbourne

 Outdoor fungal spores are among the most common aerobiological particles in the air we breathe. Although a limited number of outdoor fungal species are recognised… (more)

Subjects/Keywords: outdoor fungal spores; asthma hospitalisations; lung function; airway inflammation; epidemiology

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APA (6th Edition):

Tham, R. (2017). The contribution of outdoor fungal spores to child and adolescent asthma hospitalisations: lung function and airway inflammation. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/191669

Chicago Manual of Style (16th Edition):

Tham, Rachel. “The contribution of outdoor fungal spores to child and adolescent asthma hospitalisations: lung function and airway inflammation.” 2017. Doctoral Dissertation, University of Melbourne. Accessed October 25, 2020. http://hdl.handle.net/11343/191669.

MLA Handbook (7th Edition):

Tham, Rachel. “The contribution of outdoor fungal spores to child and adolescent asthma hospitalisations: lung function and airway inflammation.” 2017. Web. 25 Oct 2020.

Vancouver:

Tham R. The contribution of outdoor fungal spores to child and adolescent asthma hospitalisations: lung function and airway inflammation. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/11343/191669.

Council of Science Editors:

Tham R. The contribution of outdoor fungal spores to child and adolescent asthma hospitalisations: lung function and airway inflammation. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/191669


University of Tennessee – Knoxville

20. Brown, Patricia Louise. Calcium and Leucine Modulation of Airway Inflammation.

Degree: 2014, University of Tennessee – Knoxville

 Over the past several decades the prevalence of obesity and asthma have increased in a parallel fashion. Recent studies reported a positive relationship between the… (more)

Subjects/Keywords: leucine; asthma; calcium; obesity; airway inflammation; Molecular, Genetic, and Biochemical Nutrition

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APA (6th Edition):

Brown, P. L. (2014). Calcium and Leucine Modulation of Airway Inflammation. (Doctoral Dissertation). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_graddiss/3188

Chicago Manual of Style (16th Edition):

Brown, Patricia Louise. “Calcium and Leucine Modulation of Airway Inflammation.” 2014. Doctoral Dissertation, University of Tennessee – Knoxville. Accessed October 25, 2020. https://trace.tennessee.edu/utk_graddiss/3188.

MLA Handbook (7th Edition):

Brown, Patricia Louise. “Calcium and Leucine Modulation of Airway Inflammation.” 2014. Web. 25 Oct 2020.

Vancouver:

Brown PL. Calcium and Leucine Modulation of Airway Inflammation. [Internet] [Doctoral dissertation]. University of Tennessee – Knoxville; 2014. [cited 2020 Oct 25]. Available from: https://trace.tennessee.edu/utk_graddiss/3188.

Council of Science Editors:

Brown PL. Calcium and Leucine Modulation of Airway Inflammation. [Doctoral Dissertation]. University of Tennessee – Knoxville; 2014. Available from: https://trace.tennessee.edu/utk_graddiss/3188


Massey University

21. Brooks, Collin Richard. Non-invasive assessment of airway inflammation in asthma : a thesis by publications presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Public Health, Massey University, Wellington, New Zealand .

Degree: 2013, Massey University

Inflammation is a central feature in current definitions of asthma. Despite this, airway inflammation remains infrequently assessed in either population-based studies or clinical practice. In… (more)

Subjects/Keywords: Asthma; Airway inflammation; Sputum; Flow cytometry; Sputum leukocyte populations; Asthma pathophysiology

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APA (6th Edition):

Brooks, C. R. (2013). Non-invasive assessment of airway inflammation in asthma : a thesis by publications presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Public Health, Massey University, Wellington, New Zealand . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/5154

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brooks, Collin Richard. “Non-invasive assessment of airway inflammation in asthma : a thesis by publications presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Public Health, Massey University, Wellington, New Zealand .” 2013. Thesis, Massey University. Accessed October 25, 2020. http://hdl.handle.net/10179/5154.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brooks, Collin Richard. “Non-invasive assessment of airway inflammation in asthma : a thesis by publications presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Public Health, Massey University, Wellington, New Zealand .” 2013. Web. 25 Oct 2020.

Vancouver:

Brooks CR. Non-invasive assessment of airway inflammation in asthma : a thesis by publications presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Public Health, Massey University, Wellington, New Zealand . [Internet] [Thesis]. Massey University; 2013. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10179/5154.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brooks CR. Non-invasive assessment of airway inflammation in asthma : a thesis by publications presented in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Public Health, Massey University, Wellington, New Zealand . [Thesis]. Massey University; 2013. Available from: http://hdl.handle.net/10179/5154

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Maryland

22. Palaniyandi, Senthilkumar. CD23 MEDIATED IGE TRANSCYTOSIS IN AIRWAY INFLAMMATION.

Degree: Veterinary Medical Science, 2012, University of Maryland

 CD23 (FceRII), a C-type lectin type II membrane glycoprotein, plays an important role in IgE homeostasis and development of allergic inflammation. I showed that CD23… (more)

Subjects/Keywords: Immunology; Cellular biology; airway; asthma; Fc receptors; immune complexes; inflammation; transcytosis

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APA (6th Edition):

Palaniyandi, S. (2012). CD23 MEDIATED IGE TRANSCYTOSIS IN AIRWAY INFLAMMATION. (Thesis). University of Maryland. Retrieved from http://hdl.handle.net/1903/13084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Palaniyandi, Senthilkumar. “CD23 MEDIATED IGE TRANSCYTOSIS IN AIRWAY INFLAMMATION.” 2012. Thesis, University of Maryland. Accessed October 25, 2020. http://hdl.handle.net/1903/13084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Palaniyandi, Senthilkumar. “CD23 MEDIATED IGE TRANSCYTOSIS IN AIRWAY INFLAMMATION.” 2012. Web. 25 Oct 2020.

Vancouver:

Palaniyandi S. CD23 MEDIATED IGE TRANSCYTOSIS IN AIRWAY INFLAMMATION. [Internet] [Thesis]. University of Maryland; 2012. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1903/13084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Palaniyandi S. CD23 MEDIATED IGE TRANSCYTOSIS IN AIRWAY INFLAMMATION. [Thesis]. University of Maryland; 2012. Available from: http://hdl.handle.net/1903/13084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

23. Andrews, Carla S. The Regulation of Nontypeable Haemophilus influezae-Induced Inflammation and an Alternative Therapeutic Strategy.

Degree: PhD, Biology, 2016, Georgia State University

Airway diseases such as asthma and chronic obstructive pulmonary disease (COPD) affect more than one-half billion people globally resulting in billions of dollars in… (more)

Subjects/Keywords: Airway disease; Inflammation; NTHi; Resveratrol; Negative regulation; PDE inhibitor

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APA (6th Edition):

Andrews, C. S. (2016). The Regulation of Nontypeable Haemophilus influezae-Induced Inflammation and an Alternative Therapeutic Strategy. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/168

Chicago Manual of Style (16th Edition):

Andrews, Carla S. “The Regulation of Nontypeable Haemophilus influezae-Induced Inflammation and an Alternative Therapeutic Strategy.” 2016. Doctoral Dissertation, Georgia State University. Accessed October 25, 2020. https://scholarworks.gsu.edu/biology_diss/168.

MLA Handbook (7th Edition):

Andrews, Carla S. “The Regulation of Nontypeable Haemophilus influezae-Induced Inflammation and an Alternative Therapeutic Strategy.” 2016. Web. 25 Oct 2020.

Vancouver:

Andrews CS. The Regulation of Nontypeable Haemophilus influezae-Induced Inflammation and an Alternative Therapeutic Strategy. [Internet] [Doctoral dissertation]. Georgia State University; 2016. [cited 2020 Oct 25]. Available from: https://scholarworks.gsu.edu/biology_diss/168.

Council of Science Editors:

Andrews CS. The Regulation of Nontypeable Haemophilus influezae-Induced Inflammation and an Alternative Therapeutic Strategy. [Doctoral Dissertation]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/biology_diss/168


University of Montana

24. Hong, Soram. THE EFFECT OF A CANNABINOID RECEPTOR 2 (CB2) AGONIST ON ALLERGIC AIRWAY INFLAMMATION IN A MOUSE MODEL OF ASTHMA.

Degree: MS, 2013, University of Montana

 Allergic asthma is a chronic inflammatory disease that affects approximately 300 million people worldwide. The health problem is compounded by the fact that the prevalence… (more)

Subjects/Keywords: ALLERGIC AIRWAY INFLAMMATION; ASTHMA; CANNABINOID RECEPTOR 2 (CB2) AGONIST; MOUSE

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APA (6th Edition):

Hong, S. (2013). THE EFFECT OF A CANNABINOID RECEPTOR 2 (CB2) AGONIST ON ALLERGIC AIRWAY INFLAMMATION IN A MOUSE MODEL OF ASTHMA. (Masters Thesis). University of Montana. Retrieved from https://scholarworks.umt.edu/etd/1398

Chicago Manual of Style (16th Edition):

Hong, Soram. “THE EFFECT OF A CANNABINOID RECEPTOR 2 (CB2) AGONIST ON ALLERGIC AIRWAY INFLAMMATION IN A MOUSE MODEL OF ASTHMA.” 2013. Masters Thesis, University of Montana. Accessed October 25, 2020. https://scholarworks.umt.edu/etd/1398.

MLA Handbook (7th Edition):

Hong, Soram. “THE EFFECT OF A CANNABINOID RECEPTOR 2 (CB2) AGONIST ON ALLERGIC AIRWAY INFLAMMATION IN A MOUSE MODEL OF ASTHMA.” 2013. Web. 25 Oct 2020.

Vancouver:

Hong S. THE EFFECT OF A CANNABINOID RECEPTOR 2 (CB2) AGONIST ON ALLERGIC AIRWAY INFLAMMATION IN A MOUSE MODEL OF ASTHMA. [Internet] [Masters thesis]. University of Montana; 2013. [cited 2020 Oct 25]. Available from: https://scholarworks.umt.edu/etd/1398.

Council of Science Editors:

Hong S. THE EFFECT OF A CANNABINOID RECEPTOR 2 (CB2) AGONIST ON ALLERGIC AIRWAY INFLAMMATION IN A MOUSE MODEL OF ASTHMA. [Masters Thesis]. University of Montana; 2013. Available from: https://scholarworks.umt.edu/etd/1398


Universiteit Utrecht

25. Scheerens, J. Eotaxin, airway inflammation and hyperresponsiveness.

Degree: 2001, Universiteit Utrecht

 Eotaxin is a chemokine involved in migration and activation of inflammatory cells. CC-chemokine receptor-3 (CCR3), the receptor for eotaxin and other chemokines, is expressed by… (more)

Subjects/Keywords: Farmacie; eotaxin; asthma; airway inflammation; airway hyperresponsiveness

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APA (6th Edition):

Scheerens, J. (2001). Eotaxin, airway inflammation and hyperresponsiveness. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/332

Chicago Manual of Style (16th Edition):

Scheerens, J. “Eotaxin, airway inflammation and hyperresponsiveness.” 2001. Doctoral Dissertation, Universiteit Utrecht. Accessed October 25, 2020. http://dspace.library.uu.nl:8080/handle/1874/332.

MLA Handbook (7th Edition):

Scheerens, J. “Eotaxin, airway inflammation and hyperresponsiveness.” 2001. Web. 25 Oct 2020.

Vancouver:

Scheerens J. Eotaxin, airway inflammation and hyperresponsiveness. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2001. [cited 2020 Oct 25]. Available from: http://dspace.library.uu.nl:8080/handle/1874/332.

Council of Science Editors:

Scheerens J. Eotaxin, airway inflammation and hyperresponsiveness. [Doctoral Dissertation]. Universiteit Utrecht; 2001. Available from: http://dspace.library.uu.nl:8080/handle/1874/332


University of Saskatchewan

26. Davis, Beth E. Protective effect of H1 and CysLT1 antagonists on allergen induced airway responses in atopic asthma.

Degree: 2009, University of Saskatchewan

 Background The mechanism by which allergies trigger asthma occurs through the interaction of antigen, IgE and the FcεR1 receptor on mast cells resulting in the… (more)

Subjects/Keywords: asthma; airway inflammation; montelukast; desloratadine; airway responses

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APA (6th Edition):

Davis, B. E. (2009). Protective effect of H1 and CysLT1 antagonists on allergen induced airway responses in atopic asthma. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-01202010-215614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davis, Beth E. “Protective effect of H1 and CysLT1 antagonists on allergen induced airway responses in atopic asthma.” 2009. Thesis, University of Saskatchewan. Accessed October 25, 2020. http://hdl.handle.net/10388/etd-01202010-215614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davis, Beth E. “Protective effect of H1 and CysLT1 antagonists on allergen induced airway responses in atopic asthma.” 2009. Web. 25 Oct 2020.

Vancouver:

Davis BE. Protective effect of H1 and CysLT1 antagonists on allergen induced airway responses in atopic asthma. [Internet] [Thesis]. University of Saskatchewan; 2009. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/10388/etd-01202010-215614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davis BE. Protective effect of H1 and CysLT1 antagonists on allergen induced airway responses in atopic asthma. [Thesis]. University of Saskatchewan; 2009. Available from: http://hdl.handle.net/10388/etd-01202010-215614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

27. Eapen, MS. Understanding cellular changes and mediators of airway remodelling in COPD.

Degree: 2018, University of Tasmania

 (Background) (and) (Aims:) Chronic obstructive pulmonary disease (COPD) is emerging as a substantial global health problem, with an estimated annual mortality of over 3 million… (more)

Subjects/Keywords: Chronic Obstructive Pulmonary Disease (COPD); airway wall; cellularity; inflammation; immune dysfunction; myofibroblast; extracellular matrix; airway remodeling

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APA (6th Edition):

Eapen, M. (2018). Understanding cellular changes and mediators of airway remodelling in COPD. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eapen, MS. “Understanding cellular changes and mediators of airway remodelling in COPD.” 2018. Thesis, University of Tasmania. Accessed October 25, 2020. https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eapen, MS. “Understanding cellular changes and mediators of airway remodelling in COPD.” 2018. Web. 25 Oct 2020.

Vancouver:

Eapen M. Understanding cellular changes and mediators of airway remodelling in COPD. [Internet] [Thesis]. University of Tasmania; 2018. [cited 2020 Oct 25]. Available from: https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eapen M. Understanding cellular changes and mediators of airway remodelling in COPD. [Thesis]. University of Tasmania; 2018. Available from: https://eprints.utas.edu.au/28492/1/Eapen_whole_thesis.pdf ; Eapen, MS ORCID: 0000-0003-0570-7059 <https://orcid.org/0000-0003-0570-7059> 2018 , 'Understanding cellular changes and mediators of airway remodelling in COPD', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Movassagh, Hesamaldin. Expression of Semaphorin 3E in Asthma and its role in Allergic Airway Disease.

Degree: Immunology, 2015, University of Manitoba

 Asthma is a chronic condition characterized by variable airflow obstruction, bronchial hyper-responsiveness, airway inflammation and remodeling. In spite of tremendous advances, the regulatory mechanisms controlling… (more)

Subjects/Keywords: Asthma; Semaphorin; Airway Inflammation; Airway Remodeling

…allergen-induced neutrophilic airway inflammation 162 4.4 Supplemental figures… …178 4.6.9 Allergen-induced airway inflammation model… …186 5.5 Versatile role of Sema3E in allergen-induced airway inflammation… …145 X LIST OF FIGURES FIGURE 1-1 CHRONIC STAGE OF ALLERGEN-INDUCED AIRWAY INFLAMMATION… …100 FIGURE 3-2 SEMA3E DEFICIENT MICE HAVE ELEVATED AHR, AIRWAY INFLAMMATION AND REMODELING… 

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APA (6th Edition):

Movassagh, H. (2015). Expression of Semaphorin 3E in Asthma and its role in Allergic Airway Disease. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31121

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Movassagh, Hesamaldin. “Expression of Semaphorin 3E in Asthma and its role in Allergic Airway Disease.” 2015. Thesis, University of Manitoba. Accessed October 25, 2020. http://hdl.handle.net/1993/31121.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Movassagh, Hesamaldin. “Expression of Semaphorin 3E in Asthma and its role in Allergic Airway Disease.” 2015. Web. 25 Oct 2020.

Vancouver:

Movassagh H. Expression of Semaphorin 3E in Asthma and its role in Allergic Airway Disease. [Internet] [Thesis]. University of Manitoba; 2015. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1993/31121.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Movassagh H. Expression of Semaphorin 3E in Asthma and its role in Allergic Airway Disease. [Thesis]. University of Manitoba; 2015. Available from: http://hdl.handle.net/1993/31121

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Leiden University

29. Slats, A.M. Towards restoring the physiological protection against airway narrowing in asthma: take a deep breath!.

Degree: 2011, Leiden University

 Asthma is characterized by chronic airway inflammation and airway hyperresponsiveness. Deep inspirations affect airway narrowing and may therefore play a role in airway hyperresponsiveness in… (more)

Subjects/Keywords: Deep inspiration; Bronchoprotection; Bronchodilation; Asthma; Airway smooth muscle; Airway inflammation; Deep inspiration; Bronchoprotection; Bronchodilation; Asthma; Airway smooth muscle; Airway inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Slats, A. M. (2011). Towards restoring the physiological protection against airway narrowing in asthma: take a deep breath!. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/18221

Chicago Manual of Style (16th Edition):

Slats, A M. “Towards restoring the physiological protection against airway narrowing in asthma: take a deep breath!.” 2011. Doctoral Dissertation, Leiden University. Accessed October 25, 2020. http://hdl.handle.net/1887/18221.

MLA Handbook (7th Edition):

Slats, A M. “Towards restoring the physiological protection against airway narrowing in asthma: take a deep breath!.” 2011. Web. 25 Oct 2020.

Vancouver:

Slats AM. Towards restoring the physiological protection against airway narrowing in asthma: take a deep breath!. [Internet] [Doctoral dissertation]. Leiden University; 2011. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1887/18221.

Council of Science Editors:

Slats AM. Towards restoring the physiological protection against airway narrowing in asthma: take a deep breath!. [Doctoral Dissertation]. Leiden University; 2011. Available from: http://hdl.handle.net/1887/18221

30. Zuyderduyn, Suzanne. Airway smooth muscle cells: regulators of airway inflammation.

Degree: 2007, Department of Pulmonology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University

 Airways from asthmatic subjects are more responsive to bronchoconstrictive stimuli than airways from healthy subjects. Airway smooth muscle (ASM) cells mediate contraction of the airways… (more)

Subjects/Keywords: Airway inflammation; Airway smooth muscle cells; Asthma; Chemokines; Airway inflammation; Airway smooth muscle cells; Asthma; Chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zuyderduyn, S. (2007). Airway smooth muscle cells: regulators of airway inflammation. (Doctoral Dissertation). Department of Pulmonology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University. Retrieved from http://hdl.handle.net/1887/12464

Chicago Manual of Style (16th Edition):

Zuyderduyn, Suzanne. “Airway smooth muscle cells: regulators of airway inflammation.” 2007. Doctoral Dissertation, Department of Pulmonology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University. Accessed October 25, 2020. http://hdl.handle.net/1887/12464.

MLA Handbook (7th Edition):

Zuyderduyn, Suzanne. “Airway smooth muscle cells: regulators of airway inflammation.” 2007. Web. 25 Oct 2020.

Vancouver:

Zuyderduyn S. Airway smooth muscle cells: regulators of airway inflammation. [Internet] [Doctoral dissertation]. Department of Pulmonology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University; 2007. [cited 2020 Oct 25]. Available from: http://hdl.handle.net/1887/12464.

Council of Science Editors:

Zuyderduyn S. Airway smooth muscle cells: regulators of airway inflammation. [Doctoral Dissertation]. Department of Pulmonology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University; 2007. Available from: http://hdl.handle.net/1887/12464

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