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You searched for subject:(AhR). Showing records 1 – 30 of 101 total matches.

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University of Rochester

1. Kobielush, Brent R. S395, a Site Important for Regulating Transcriptional Activity of the Aryl Hydrocarbon Receptor (AhR), is Phosphorylated by Protein Kinase A (PKA).

Degree: PhD, 2009, University of Rochester

 The aryl hydrocarbon receptor (AhR) is a member of the basic helix-loop-helix transcription factor family. The AhR interacts with a wide variety of xenobiotic agonists,… (more)

Subjects/Keywords: AhR; Phosphorylation; PKA

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APA (6th Edition):

Kobielush, B. R. (2009). S395, a Site Important for Regulating Transcriptional Activity of the Aryl Hydrocarbon Receptor (AhR), is Phosphorylated by Protein Kinase A (PKA). (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8476

Chicago Manual of Style (16th Edition):

Kobielush, Brent R. “S395, a Site Important for Regulating Transcriptional Activity of the Aryl Hydrocarbon Receptor (AhR), is Phosphorylated by Protein Kinase A (PKA).” 2009. Doctoral Dissertation, University of Rochester. Accessed October 18, 2019. http://hdl.handle.net/1802/8476.

MLA Handbook (7th Edition):

Kobielush, Brent R. “S395, a Site Important for Regulating Transcriptional Activity of the Aryl Hydrocarbon Receptor (AhR), is Phosphorylated by Protein Kinase A (PKA).” 2009. Web. 18 Oct 2019.

Vancouver:

Kobielush BR. S395, a Site Important for Regulating Transcriptional Activity of the Aryl Hydrocarbon Receptor (AhR), is Phosphorylated by Protein Kinase A (PKA). [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1802/8476.

Council of Science Editors:

Kobielush BR. S395, a Site Important for Regulating Transcriptional Activity of the Aryl Hydrocarbon Receptor (AhR), is Phosphorylated by Protein Kinase A (PKA). [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8476


Universiteit Utrecht

2. Jong, T. de. Transcript detection of the AHR gene in the dog.

Degree: 2012, Universiteit Utrecht

 Summary Introduction: the Aryl Hydrocarbon Receptor is best known for its function in regulating xenobiotic metabolism and dioxin toxicity. [1] AHR activation by environmental contaminants… (more)

Subjects/Keywords: AHR; dog; transcript; sequencing

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APA (6th Edition):

Jong, T. d. (2012). Transcript detection of the AHR gene in the dog. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/289376

Chicago Manual of Style (16th Edition):

Jong, T de. “Transcript detection of the AHR gene in the dog.” 2012. Masters Thesis, Universiteit Utrecht. Accessed October 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/289376.

MLA Handbook (7th Edition):

Jong, T de. “Transcript detection of the AHR gene in the dog.” 2012. Web. 18 Oct 2019.

Vancouver:

Jong Td. Transcript detection of the AHR gene in the dog. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2019 Oct 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/289376.

Council of Science Editors:

Jong Td. Transcript detection of the AHR gene in the dog. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/289376


Penn State University

3. DiNatale, Brett C. The role of the aryl hydrocarbon receptor in tumor cell phenotype.

Degree: PhD, Molecular Medicine, 2010, Penn State University

 The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor widely associated with its function in xenobiotic metabolism and its ability to bind environmental pollutants… (more)

Subjects/Keywords: ahr; aryl hydrocarbon receptor; cancer

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APA (6th Edition):

DiNatale, B. C. (2010). The role of the aryl hydrocarbon receptor in tumor cell phenotype. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11597

Chicago Manual of Style (16th Edition):

DiNatale, Brett C. “The role of the aryl hydrocarbon receptor in tumor cell phenotype.” 2010. Doctoral Dissertation, Penn State University. Accessed October 18, 2019. https://etda.libraries.psu.edu/catalog/11597.

MLA Handbook (7th Edition):

DiNatale, Brett C. “The role of the aryl hydrocarbon receptor in tumor cell phenotype.” 2010. Web. 18 Oct 2019.

Vancouver:

DiNatale BC. The role of the aryl hydrocarbon receptor in tumor cell phenotype. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2019 Oct 18]. Available from: https://etda.libraries.psu.edu/catalog/11597.

Council of Science Editors:

DiNatale BC. The role of the aryl hydrocarbon receptor in tumor cell phenotype. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11597


Oregon State University

4. Koch, Daniel C. Ligand selective regulation of cell growth by the Ah receptor through activation of TGFβ signaling.

Degree: PhD, Toxicology, 2013, Oregon State University

 The Aryl hydrocarbon receptor (AhR) is a ligand-activated transcription factor and member of the basic helix-loop-helix Per/ARNT/Sim (bHLH/PAS) family of chemosensors and developmental regulators. As… (more)

Subjects/Keywords: AhR; Helix-loop-helix motifs

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APA (6th Edition):

Koch, D. C. (2013). Ligand selective regulation of cell growth by the Ah receptor through activation of TGFβ signaling. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/37899

Chicago Manual of Style (16th Edition):

Koch, Daniel C. “Ligand selective regulation of cell growth by the Ah receptor through activation of TGFβ signaling.” 2013. Doctoral Dissertation, Oregon State University. Accessed October 18, 2019. http://hdl.handle.net/1957/37899.

MLA Handbook (7th Edition):

Koch, Daniel C. “Ligand selective regulation of cell growth by the Ah receptor through activation of TGFβ signaling.” 2013. Web. 18 Oct 2019.

Vancouver:

Koch DC. Ligand selective regulation of cell growth by the Ah receptor through activation of TGFβ signaling. [Internet] [Doctoral dissertation]. Oregon State University; 2013. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1957/37899.

Council of Science Editors:

Koch DC. Ligand selective regulation of cell growth by the Ah receptor through activation of TGFβ signaling. [Doctoral Dissertation]. Oregon State University; 2013. Available from: http://hdl.handle.net/1957/37899


Université Catholique de Louvain

5. Cochez, Perrine. AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model.

Degree: 2016, Université Catholique de Louvain

IL-22 is a crucial cytokine in skin homeostasis by its action on keratinocytes. It induces the production of antimicrobial peptides, promotes epithelial regeneration and therefore… (more)

Subjects/Keywords: IL-22; AhR; Ccr6; Psoriasis

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APA (6th Edition):

Cochez, P. (2016). AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/176811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cochez, Perrine. “AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model.” 2016. Thesis, Université Catholique de Louvain. Accessed October 18, 2019. http://hdl.handle.net/2078.1/176811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cochez, Perrine. “AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model.” 2016. Web. 18 Oct 2019.

Vancouver:

Cochez P. AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model. [Internet] [Thesis]. Université Catholique de Louvain; 2016. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2078.1/176811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cochez P. AhR and Ccr6 are not required for IL-22 production in the imiquimod-induced psoriasis model. [Thesis]. Université Catholique de Louvain; 2016. Available from: http://hdl.handle.net/2078.1/176811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Medical Branch – Galveston

6. [No author]. A Rhapsody on the Aryl Hydrocarbon Receptor: Molecular and Environmental Health Insights Through Novel and Canonical Signaling Pathways .

Degree: University of Texas Medical Branch – Galveston

 The Aryl Hydrocarbon Receptor is a ubiquitously expressed, cytosolic transcription factor, which is activated by myriad structurally-diverse xenobiotic compounds, most notably 2,3,7,8-tetrechlorodibenzo-p-dioxin. In this role,… (more)

Subjects/Keywords: AhR; KLF6

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APA (6th Edition):

author], [. (n.d.). A Rhapsody on the Aryl Hydrocarbon Receptor: Molecular and Environmental Health Insights Through Novel and Canonical Signaling Pathways . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/656

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “A Rhapsody on the Aryl Hydrocarbon Receptor: Molecular and Environmental Health Insights Through Novel and Canonical Signaling Pathways .” Thesis, University of Texas Medical Branch – Galveston. Accessed October 18, 2019. http://hdl.handle.net/2152.3/656.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “A Rhapsody on the Aryl Hydrocarbon Receptor: Molecular and Environmental Health Insights Through Novel and Canonical Signaling Pathways .” Web. 18 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. A Rhapsody on the Aryl Hydrocarbon Receptor: Molecular and Environmental Health Insights Through Novel and Canonical Signaling Pathways . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2152.3/656.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. A Rhapsody on the Aryl Hydrocarbon Receptor: Molecular and Environmental Health Insights Through Novel and Canonical Signaling Pathways . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/656

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Rochester

7. Yin, Zhengyu. (-)-Epigallocatechin-3-gallate, Found in the Green Tea Extract, is a Novel Hsp90 Inhibitor.

Degree: PhD, 2009, University of Rochester

 Numerous animal studies have shown that epigallocatechin-3-gallate (EGCG), a major component of green tea, protects against certain types of cancers, although the mechanism has not… (more)

Subjects/Keywords: EGCG AhR Hsp90 Inhibitor

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APA (6th Edition):

Yin, Z. (2009). (-)-Epigallocatechin-3-gallate, Found in the Green Tea Extract, is a Novel Hsp90 Inhibitor. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8300

Chicago Manual of Style (16th Edition):

Yin, Zhengyu. “(-)-Epigallocatechin-3-gallate, Found in the Green Tea Extract, is a Novel Hsp90 Inhibitor.” 2009. Doctoral Dissertation, University of Rochester. Accessed October 18, 2019. http://hdl.handle.net/1802/8300.

MLA Handbook (7th Edition):

Yin, Zhengyu. “(-)-Epigallocatechin-3-gallate, Found in the Green Tea Extract, is a Novel Hsp90 Inhibitor.” 2009. Web. 18 Oct 2019.

Vancouver:

Yin Z. (-)-Epigallocatechin-3-gallate, Found in the Green Tea Extract, is a Novel Hsp90 Inhibitor. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1802/8300.

Council of Science Editors:

Yin Z. (-)-Epigallocatechin-3-gallate, Found in the Green Tea Extract, is a Novel Hsp90 Inhibitor. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8300


University of Rochester

8. Kung, Ming Hsien; Zuscik, Michael J. Investigating the Molecular Mechanisms of Cigarette Smoke Inhibition of Fracture Healing.

Degree: PhD, 2012, University of Rochester

 There is strong clinical evidence of a link between smoking and impaired fracture healing. Despite much effort, the molecular mechanisms underlying these effects remain poorly… (more)

Subjects/Keywords: Fracture; Smoking; Cigarette; Chondrogenesis; AhR

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APA (6th Edition):

Kung, Ming Hsien; Zuscik, M. J. (2012). Investigating the Molecular Mechanisms of Cigarette Smoke Inhibition of Fracture Healing. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/19823

Chicago Manual of Style (16th Edition):

Kung, Ming Hsien; Zuscik, Michael J. “Investigating the Molecular Mechanisms of Cigarette Smoke Inhibition of Fracture Healing.” 2012. Doctoral Dissertation, University of Rochester. Accessed October 18, 2019. http://hdl.handle.net/1802/19823.

MLA Handbook (7th Edition):

Kung, Ming Hsien; Zuscik, Michael J. “Investigating the Molecular Mechanisms of Cigarette Smoke Inhibition of Fracture Healing.” 2012. Web. 18 Oct 2019.

Vancouver:

Kung, Ming Hsien; Zuscik MJ. Investigating the Molecular Mechanisms of Cigarette Smoke Inhibition of Fracture Healing. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1802/19823.

Council of Science Editors:

Kung, Ming Hsien; Zuscik MJ. Investigating the Molecular Mechanisms of Cigarette Smoke Inhibition of Fracture Healing. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/19823


Texas A&M University

9. Lafage, Stephanie Isabelle. An alternative to the Winland R35 method for determining carbonate reservoir quality.

Degree: 2008, Texas A&M University

 The Winland R35 method [Log R35 = 0.732 + 0.588 (Log Kair) 0.864 (Log O)] is based on the relationship between porosity, permeability, and pore… (more)

Subjects/Keywords: Winland; Carbonate; R35; porosity; Ahr classification

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APA (6th Edition):

Lafage, S. I. (2008). An alternative to the Winland R35 method for determining carbonate reservoir quality. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/86031

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lafage, Stephanie Isabelle. “An alternative to the Winland R35 method for determining carbonate reservoir quality.” 2008. Thesis, Texas A&M University. Accessed October 18, 2019. http://hdl.handle.net/1969.1/86031.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lafage, Stephanie Isabelle. “An alternative to the Winland R35 method for determining carbonate reservoir quality.” 2008. Web. 18 Oct 2019.

Vancouver:

Lafage SI. An alternative to the Winland R35 method for determining carbonate reservoir quality. [Internet] [Thesis]. Texas A&M University; 2008. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1969.1/86031.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lafage SI. An alternative to the Winland R35 method for determining carbonate reservoir quality. [Thesis]. Texas A&M University; 2008. Available from: http://hdl.handle.net/1969.1/86031

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

10. Belton, Kerry R. ARYL HYDROCARBON RECEPTOR ACTIVATION DISRUPTS MOUSE MAMMARY GLAND AND LIVER FUNCTION DURING LACTATION.

Degree: PhD, Molecular Toxicology, 2016, Penn State University

 Environmental exposure to pesticides and xenobiotics poses a significant risk to human reproductive and lactation health. The liver and the mammary gland have complementary metabolic… (more)

Subjects/Keywords: AHR; Mammary gland; Metabolism; Lactation; NMR

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APA (6th Edition):

Belton, K. R. (2016). ARYL HYDROCARBON RECEPTOR ACTIVATION DISRUPTS MOUSE MAMMARY GLAND AND LIVER FUNCTION DURING LACTATION. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/13516krb22

Chicago Manual of Style (16th Edition):

Belton, Kerry R. “ARYL HYDROCARBON RECEPTOR ACTIVATION DISRUPTS MOUSE MAMMARY GLAND AND LIVER FUNCTION DURING LACTATION.” 2016. Doctoral Dissertation, Penn State University. Accessed October 18, 2019. https://etda.libraries.psu.edu/catalog/13516krb22.

MLA Handbook (7th Edition):

Belton, Kerry R. “ARYL HYDROCARBON RECEPTOR ACTIVATION DISRUPTS MOUSE MAMMARY GLAND AND LIVER FUNCTION DURING LACTATION.” 2016. Web. 18 Oct 2019.

Vancouver:

Belton KR. ARYL HYDROCARBON RECEPTOR ACTIVATION DISRUPTS MOUSE MAMMARY GLAND AND LIVER FUNCTION DURING LACTATION. [Internet] [Doctoral dissertation]. Penn State University; 2016. [cited 2019 Oct 18]. Available from: https://etda.libraries.psu.edu/catalog/13516krb22.

Council of Science Editors:

Belton KR. ARYL HYDROCARBON RECEPTOR ACTIVATION DISRUPTS MOUSE MAMMARY GLAND AND LIVER FUNCTION DURING LACTATION. [Doctoral Dissertation]. Penn State University; 2016. Available from: https://etda.libraries.psu.edu/catalog/13516krb22


Penn State University

11. Borland, Michael Gregory. PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS.

Degree: PhD, Biochemistry, Microbiology, and Molecular Biology, 2010, Penn State University

 Since its identification in the early 1990fs, the physiological roles of the nuclear hormone receptor peroxisome proliferator-activated receptor-ƒÀ/ƒÂ (PPARƒÀ/ƒÂ) have become better understood. This ligand-activated… (more)

Subjects/Keywords: PPARBeta/Delta; AHR; PAH; Bioactivation; Skin carcinogenesis

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APA (6th Edition):

Borland, M. G. (2010). PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11381

Chicago Manual of Style (16th Edition):

Borland, Michael Gregory. “PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS.” 2010. Doctoral Dissertation, Penn State University. Accessed October 18, 2019. https://etda.libraries.psu.edu/catalog/11381.

MLA Handbook (7th Edition):

Borland, Michael Gregory. “PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS.” 2010. Web. 18 Oct 2019.

Vancouver:

Borland MG. PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2019 Oct 18]. Available from: https://etda.libraries.psu.edu/catalog/11381.

Council of Science Editors:

Borland MG. PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11381


Texas A&M University

12. Klemashevich, Cory Lee. The Microbially Derived Metabolite Indole Attenuates Obesity Associated Inflammatory Processes in Adipocytes and Macrophages.

Degree: PhD, Chemical Engineering, 2016, Texas A&M University

 The human gastrointestinal (GI) tract is colonized by ~1014 bacteria belonging to ~1,000 species that are collectively termed the intestinal microbiota. Recent studies show that… (more)

Subjects/Keywords: indole; inflammation; obesity; microbiota; mTOR; AhR

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APA (6th Edition):

Klemashevich, C. L. (2016). The Microbially Derived Metabolite Indole Attenuates Obesity Associated Inflammatory Processes in Adipocytes and Macrophages. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/159093

Chicago Manual of Style (16th Edition):

Klemashevich, Cory Lee. “The Microbially Derived Metabolite Indole Attenuates Obesity Associated Inflammatory Processes in Adipocytes and Macrophages.” 2016. Doctoral Dissertation, Texas A&M University. Accessed October 18, 2019. http://hdl.handle.net/1969.1/159093.

MLA Handbook (7th Edition):

Klemashevich, Cory Lee. “The Microbially Derived Metabolite Indole Attenuates Obesity Associated Inflammatory Processes in Adipocytes and Macrophages.” 2016. Web. 18 Oct 2019.

Vancouver:

Klemashevich CL. The Microbially Derived Metabolite Indole Attenuates Obesity Associated Inflammatory Processes in Adipocytes and Macrophages. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1969.1/159093.

Council of Science Editors:

Klemashevich CL. The Microbially Derived Metabolite Indole Attenuates Obesity Associated Inflammatory Processes in Adipocytes and Macrophages. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/159093

13. Inácio, Ana Filipa Ferreira da Costa Palma. A terapêutica com o omeprazol: avaliação da potencial relação com a suscetibilidade ao cancro.

Degree: 2012, RCAAP

Dissertação de mest., Ciências Farmacêuticas, Faculdade de Ciências e Tecnologia, Univ. do Algarve, 2012

O omeprazol, principal benzimidazol substituído utilizado na inibição da secreção gástrica,… (more)

Subjects/Keywords: Omeprazol; AhR; Indução enzimática; CYP1A1; CYP1A2; Cancro

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APA (6th Edition):

Inácio, A. F. F. d. C. P. (2012). A terapêutica com o omeprazol: avaliação da potencial relação com a suscetibilidade ao cancro. (Thesis). RCAAP. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/3016

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Inácio, Ana Filipa Ferreira da Costa Palma. “A terapêutica com o omeprazol: avaliação da potencial relação com a suscetibilidade ao cancro.” 2012. Thesis, RCAAP. Accessed October 18, 2019. http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/3016.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Inácio, Ana Filipa Ferreira da Costa Palma. “A terapêutica com o omeprazol: avaliação da potencial relação com a suscetibilidade ao cancro.” 2012. Web. 18 Oct 2019.

Vancouver:

Inácio AFFdCP. A terapêutica com o omeprazol: avaliação da potencial relação com a suscetibilidade ao cancro. [Internet] [Thesis]. RCAAP; 2012. [cited 2019 Oct 18]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/3016.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Inácio AFFdCP. A terapêutica com o omeprazol: avaliação da potencial relação com a suscetibilidade ao cancro. [Thesis]. RCAAP; 2012. Available from: http://www.rcaap.pt/detail.jsp?id=oai:sapientia.ualg.pt:10400.1/3016

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Medical Branch – Galveston

14. [No author]. Novel Insights Into Normal Aryl Hydrocarbon Receptor Biology Through the Regulation of Stanniocalcin 2 .

Degree: 2013, University of Texas Medical Branch – Galveston

 Proper hepatocyte function is vital for survival; hence unrepaired destruction of the parenchymal tissue leading to liver decompensation is devastating. Therefore, understanding the homeostatic process… (more)

Subjects/Keywords: AhR; Stc2; Hepatocytes; Apoptosis; ER Stress

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APA (6th Edition):

author], [. (2013). Novel Insights Into Normal Aryl Hydrocarbon Receptor Biology Through the Regulation of Stanniocalcin 2 . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/542

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Novel Insights Into Normal Aryl Hydrocarbon Receptor Biology Through the Regulation of Stanniocalcin 2 .” 2013. Thesis, University of Texas Medical Branch – Galveston. Accessed October 18, 2019. http://hdl.handle.net/2152.3/542.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Novel Insights Into Normal Aryl Hydrocarbon Receptor Biology Through the Regulation of Stanniocalcin 2 .” 2013. Web. 18 Oct 2019.

Vancouver:

author] [. Novel Insights Into Normal Aryl Hydrocarbon Receptor Biology Through the Regulation of Stanniocalcin 2 . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; 2013. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2152.3/542.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Novel Insights Into Normal Aryl Hydrocarbon Receptor Biology Through the Regulation of Stanniocalcin 2 . [Thesis]. University of Texas Medical Branch – Galveston; 2013. Available from: http://hdl.handle.net/2152.3/542

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

15. Dever, Daniel Patrick. The Aryl Hydrocarbon Receptor and Cerebellar Granule Cell Neurogenesis: Implications for Development and Medulloblastoma Pathogenesis.

Degree: PhD, 2014, University of Rochester

 The aryl hydrocarbon receptor (AhR) is a ligand-activated member of the basic-helixloop- helix (bHLH)/PER-ARNT-SIM transcription factor superfamily that mediates 2,3,7,8- tetrachlorodibenzo-p-dioxin (TCDD) toxicity. Increasing evidence… (more)

Subjects/Keywords: AhR; Cerebellum; Cerebellar granule cells; Development; Medulloblastoma

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APA (6th Edition):

Dever, D. P. (2014). The Aryl Hydrocarbon Receptor and Cerebellar Granule Cell Neurogenesis: Implications for Development and Medulloblastoma Pathogenesis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/28437

Chicago Manual of Style (16th Edition):

Dever, Daniel Patrick. “The Aryl Hydrocarbon Receptor and Cerebellar Granule Cell Neurogenesis: Implications for Development and Medulloblastoma Pathogenesis.” 2014. Doctoral Dissertation, University of Rochester. Accessed October 18, 2019. http://hdl.handle.net/1802/28437.

MLA Handbook (7th Edition):

Dever, Daniel Patrick. “The Aryl Hydrocarbon Receptor and Cerebellar Granule Cell Neurogenesis: Implications for Development and Medulloblastoma Pathogenesis.” 2014. Web. 18 Oct 2019.

Vancouver:

Dever DP. The Aryl Hydrocarbon Receptor and Cerebellar Granule Cell Neurogenesis: Implications for Development and Medulloblastoma Pathogenesis. [Internet] [Doctoral dissertation]. University of Rochester; 2014. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1802/28437.

Council of Science Editors:

Dever DP. The Aryl Hydrocarbon Receptor and Cerebellar Granule Cell Neurogenesis: Implications for Development and Medulloblastoma Pathogenesis. [Doctoral Dissertation]. University of Rochester; 2014. Available from: http://hdl.handle.net/1802/28437


University of Texas – Austin

16. Gardella, Kacie Alicia Thomas. Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator.

Degree: PhD, Cell and Molecular Biology, 2015, University of Texas – Austin

 Nuclear factor-[kappa]B (NF-[kappa]B) signaling is critical for the proper function of the immune system, and when deregulated, promotes the development of immune disorders and cancer.… (more)

Subjects/Keywords: ARNT; NF-kappaB; AHR; Lymphoid malignancies

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APA (6th Edition):

Gardella, K. A. T. (2015). Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/63854

Chicago Manual of Style (16th Edition):

Gardella, Kacie Alicia Thomas. “Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator.” 2015. Doctoral Dissertation, University of Texas – Austin. Accessed October 18, 2019. http://hdl.handle.net/2152/63854.

MLA Handbook (7th Edition):

Gardella, Kacie Alicia Thomas. “Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator.” 2015. Web. 18 Oct 2019.

Vancouver:

Gardella KAT. Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2015. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2152/63854.

Council of Science Editors:

Gardella KAT. Regulation of the NF-kappaB and p53 pathways by the aryl hydrocarbon receptor nuclear translocator. [Doctoral Dissertation]. University of Texas – Austin; 2015. Available from: http://hdl.handle.net/2152/63854

17. Lecoq, Anne-Lise. Implication d'AIP (Aryl hydrocarbon receptor Interacting Protein) dans la tumorigenèse des adénomes hypophysaires. : Role of AIP (Aryl Hydrocarbon Receptor Interacting Protein) in Pituitary Adenoma Tumorigenesis.

Degree: Docteur es, Sciences de la vie et de la santé, 2015, Paris Saclay

Nous avons souhaité, dans ce travail de Thèse, préciser l'impact de l'invalidation d'AIP sur la fonction sécrétoire et la prolifération des cellules somatotropes in vivo… (more)

Subjects/Keywords: Acromégalie; Adénome hypophysaire; Tumorigenèse; Aip; AhR; AMPc; Acromegaly; Pituitary adenoma; Tumorigenesis; Aip; AhR; Camp

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APA (6th Edition):

Lecoq, A. (2015). Implication d'AIP (Aryl hydrocarbon receptor Interacting Protein) dans la tumorigenèse des adénomes hypophysaires. : Role of AIP (Aryl Hydrocarbon Receptor Interacting Protein) in Pituitary Adenoma Tumorigenesis. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2015SACLS028

Chicago Manual of Style (16th Edition):

Lecoq, Anne-Lise. “Implication d'AIP (Aryl hydrocarbon receptor Interacting Protein) dans la tumorigenèse des adénomes hypophysaires. : Role of AIP (Aryl Hydrocarbon Receptor Interacting Protein) in Pituitary Adenoma Tumorigenesis.” 2015. Doctoral Dissertation, Paris Saclay. Accessed October 18, 2019. http://www.theses.fr/2015SACLS028.

MLA Handbook (7th Edition):

Lecoq, Anne-Lise. “Implication d'AIP (Aryl hydrocarbon receptor Interacting Protein) dans la tumorigenèse des adénomes hypophysaires. : Role of AIP (Aryl Hydrocarbon Receptor Interacting Protein) in Pituitary Adenoma Tumorigenesis.” 2015. Web. 18 Oct 2019.

Vancouver:

Lecoq A. Implication d'AIP (Aryl hydrocarbon receptor Interacting Protein) dans la tumorigenèse des adénomes hypophysaires. : Role of AIP (Aryl Hydrocarbon Receptor Interacting Protein) in Pituitary Adenoma Tumorigenesis. [Internet] [Doctoral dissertation]. Paris Saclay; 2015. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2015SACLS028.

Council of Science Editors:

Lecoq A. Implication d'AIP (Aryl hydrocarbon receptor Interacting Protein) dans la tumorigenèse des adénomes hypophysaires. : Role of AIP (Aryl Hydrocarbon Receptor Interacting Protein) in Pituitary Adenoma Tumorigenesis. [Doctoral Dissertation]. Paris Saclay; 2015. Available from: http://www.theses.fr/2015SACLS028

18. Chevallier, Aline. Etude du rôle du récepteur aux hydrocarbures aromatiques ou AhR dans le développement et l’homéostasie du système nerveux de la souris C57BL/6J : Investigation of the role of the Aryl hydrocarbon Receptor (AhR) in the nervous system of C57BL/6J mice.

Degree: Docteur es, Neurotoxicologie, 2012, Université Paris Descartes – Paris V

Le récepteur aux hydrocarbures aromatiques (AhR) est un facteur de transcription de la famille bHLH/PAS, activé par différents ligands exogènes dont les hydrocarbures aromatiques polycycliques… (more)

Subjects/Keywords: AhR; Système nerveux; Réflexe vestibulaire; Vidéo-oculographie; Dioxine; AhR; Nervous system; Vestibulo-ocular reflex; Video-oculography; Dioxin; 616.81071

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APA (6th Edition):

Chevallier, A. (2012). Etude du rôle du récepteur aux hydrocarbures aromatiques ou AhR dans le développement et l’homéostasie du système nerveux de la souris C57BL/6J : Investigation of the role of the Aryl hydrocarbon Receptor (AhR) in the nervous system of C57BL/6J mice. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2012PA05P638

Chicago Manual of Style (16th Edition):

Chevallier, Aline. “Etude du rôle du récepteur aux hydrocarbures aromatiques ou AhR dans le développement et l’homéostasie du système nerveux de la souris C57BL/6J : Investigation of the role of the Aryl hydrocarbon Receptor (AhR) in the nervous system of C57BL/6J mice.” 2012. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed October 18, 2019. http://www.theses.fr/2012PA05P638.

MLA Handbook (7th Edition):

Chevallier, Aline. “Etude du rôle du récepteur aux hydrocarbures aromatiques ou AhR dans le développement et l’homéostasie du système nerveux de la souris C57BL/6J : Investigation of the role of the Aryl hydrocarbon Receptor (AhR) in the nervous system of C57BL/6J mice.” 2012. Web. 18 Oct 2019.

Vancouver:

Chevallier A. Etude du rôle du récepteur aux hydrocarbures aromatiques ou AhR dans le développement et l’homéostasie du système nerveux de la souris C57BL/6J : Investigation of the role of the Aryl hydrocarbon Receptor (AhR) in the nervous system of C57BL/6J mice. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2012. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2012PA05P638.

Council of Science Editors:

Chevallier A. Etude du rôle du récepteur aux hydrocarbures aromatiques ou AhR dans le développement et l’homéostasie du système nerveux de la souris C57BL/6J : Investigation of the role of the Aryl hydrocarbon Receptor (AhR) in the nervous system of C57BL/6J mice. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2012. Available from: http://www.theses.fr/2012PA05P638


University of Cincinnati

19. CLAY, COREY DAVIS. AN EVALUATION OF TCDD AND POLYHALOGENATED BIPHENYL MEDIATED REACTIVE OXYGEN GENERATION BY CYTOCHROMES P4501A1, P4501A2 AND P4502E1.

Degree: MS, Medicine : Environmental Health Sciences, 2003, University of Cincinnati

 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is an environmentally persistent compound that causes cancer and toxicity in multiple organ systems. Because of its potency, other structurally similar hazardous toxicants… (more)

Subjects/Keywords: AHR; AHR gene battery; PCB; PBB; microsome

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APA (6th Edition):

CLAY, C. D. (2003). AN EVALUATION OF TCDD AND POLYHALOGENATED BIPHENYL MEDIATED REACTIVE OXYGEN GENERATION BY CYTOCHROMES P4501A1, P4501A2 AND P4502E1. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061220136

Chicago Manual of Style (16th Edition):

CLAY, COREY DAVIS. “AN EVALUATION OF TCDD AND POLYHALOGENATED BIPHENYL MEDIATED REACTIVE OXYGEN GENERATION BY CYTOCHROMES P4501A1, P4501A2 AND P4502E1.” 2003. Masters Thesis, University of Cincinnati. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061220136.

MLA Handbook (7th Edition):

CLAY, COREY DAVIS. “AN EVALUATION OF TCDD AND POLYHALOGENATED BIPHENYL MEDIATED REACTIVE OXYGEN GENERATION BY CYTOCHROMES P4501A1, P4501A2 AND P4502E1.” 2003. Web. 18 Oct 2019.

Vancouver:

CLAY CD. AN EVALUATION OF TCDD AND POLYHALOGENATED BIPHENYL MEDIATED REACTIVE OXYGEN GENERATION BY CYTOCHROMES P4501A1, P4501A2 AND P4502E1. [Internet] [Masters thesis]. University of Cincinnati; 2003. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061220136.

Council of Science Editors:

CLAY CD. AN EVALUATION OF TCDD AND POLYHALOGENATED BIPHENYL MEDIATED REACTIVE OXYGEN GENERATION BY CYTOCHROMES P4501A1, P4501A2 AND P4502E1. [Masters Thesis]. University of Cincinnati; 2003. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061220136

20. Gondouin, Bertrand. Les toxines urémiques provoquent un phénotype procoagulant de l'endothelium par la voie du facteur de transcription AHR : Indolic uremic solutes induce an endothelial procoagulant phenotype via the AHR pathway.

Degree: Docteur es, Pathologie humaine. Pathologie vasculaire et nutrition, 2013, Aix Marseille Université

L'insuffisance rénale chronique (IRC) est associée à une importante morbidité et mortalité cardio-vasculaire, à laquelle participent la dysfonction endothéliale. Les patients IRC présentent de plus… (more)

Subjects/Keywords: Endothelium; Insuffisance renale chronique; Facteur tissulaire; AHR; Dioxine; Mortalité cardiovasculaire; Endothelium; Chronic kidney disease; Tissue factor; AHR; Dioxin; Cardiovascular mortality

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APA (6th Edition):

Gondouin, B. (2013). Les toxines urémiques provoquent un phénotype procoagulant de l'endothelium par la voie du facteur de transcription AHR : Indolic uremic solutes induce an endothelial procoagulant phenotype via the AHR pathway. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2013AIXM5054

Chicago Manual of Style (16th Edition):

Gondouin, Bertrand. “Les toxines urémiques provoquent un phénotype procoagulant de l'endothelium par la voie du facteur de transcription AHR : Indolic uremic solutes induce an endothelial procoagulant phenotype via the AHR pathway.” 2013. Doctoral Dissertation, Aix Marseille Université. Accessed October 18, 2019. http://www.theses.fr/2013AIXM5054.

MLA Handbook (7th Edition):

Gondouin, Bertrand. “Les toxines urémiques provoquent un phénotype procoagulant de l'endothelium par la voie du facteur de transcription AHR : Indolic uremic solutes induce an endothelial procoagulant phenotype via the AHR pathway.” 2013. Web. 18 Oct 2019.

Vancouver:

Gondouin B. Les toxines urémiques provoquent un phénotype procoagulant de l'endothelium par la voie du facteur de transcription AHR : Indolic uremic solutes induce an endothelial procoagulant phenotype via the AHR pathway. [Internet] [Doctoral dissertation]. Aix Marseille Université 2013. [cited 2019 Oct 18]. Available from: http://www.theses.fr/2013AIXM5054.

Council of Science Editors:

Gondouin B. Les toxines urémiques provoquent un phénotype procoagulant de l'endothelium par la voie du facteur de transcription AHR : Indolic uremic solutes induce an endothelial procoagulant phenotype via the AHR pathway. [Doctoral Dissertation]. Aix Marseille Université 2013. Available from: http://www.theses.fr/2013AIXM5054

21. 厳, 向紅. The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-.

Degree: 博士(理工学), 2016, Konan University / 甲南大学

皮膚は人体最大の臓器であり、外部刺激や環境因子から生体内部を防御する上で重要な役割を担う。その皮膚老化は、外見に影響を与えるだけでなく、生体防御機構の乱れとして一連の病患を引き起こし、個人の生活の質(QOL; Quality of Life)の低下をもたらすことから、皮膚老化の進行を制御することがますます重要となっている。そこで、皮膚老化に大きく影響する要素を解明し、それぞれの要素の皮膚老化メカニズムを分子生物学的に解析することで、皮膚に対するアンチエイジング効果を持つ生体機能性成分の作用機序について理解することを目的として研究を行った。はじめに、皮膚老化過程を経年的に調査することが被験者の個人差などを排除できる有効な調査方法であることを示し、その経年的な疫学調査から、肌理、シワ、シミの三点が皮膚の見た目の老化にとって最も重要な要素だと認めるとともに、皮膚老化の有効な指標となることを示した。そこでまず、肌理に関わる角化細胞に対してバリア機能を高める効果があるとして知られるOpuntia ficus-indica抽出物(OFIE)の作用について検討を行った。その結果、OFIEはAhR-Nrf2-NQO1(アリール炭化水素受容体—NF-E2関連因子—NAD(P)H酸化還元酵素、キノン1)経路を活性化することで抗酸化作用を持つことが示された。さらに、OFIEがフィラグリンやロリクリンの発現をAhR依存的に増強することで皮膚バリアを保護することを示した。さらに、ヒトの老化した真皮および若い真皮におけるマイクロRNAの発現を網羅的に比較するとともに、生体機能性成分によるマイクロRNA発現様式への影響を調べた。その結果、miRNA 34および29が、線維芽細胞の老化および細胞外マトリックスの再構築を制御する上で中心的な役割を果たし、シワの形成に関与することが明らかとなった。最後に、角化細胞におけるメラニンの生物学的作用を明らかにするため、単純化細胞モデルを構築し、生体機能性成分を評価したところ、ナイアシンアミドおよびトリプシン阻害剤が角化細胞におけるメラニン取り込みを抑制することが示された。さらに、取り込まれたメラニンは角化細胞の細胞周期を停止させ、色素細胞の機能を抑制することが知られるDickkopf 1遺伝子の発現低下を引き起こすことが明らかとなった。これらの結果は、皮膚老化要素に影響を与える角化細胞および線維芽細胞の働きについて新たな知見を提供するものであり、ヒトにおける皮膚老化の抑制に向けた新たな戦略を立てるうえで大きな示唆を与えている。さらに、生体機能性成分が持つ皮膚老化要素に対する有効性をより強固にサポートしている。これらの成果は、今後の新たな皮膚アンチエイジング技術の開発を通して、肌理の乱れ、シワやシミといった多くの人の皮膚老化への懸念を解消し、高齢化社会における人々のQOLの向上に資するものと期待される。

平成27年(2015年度)

Subjects/Keywords: Skin; aging; anti-aging; AhR-Nrf2 signaling; microRNA; melanin uptake

Page 1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

厳, . (2016). The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-. (Thesis). Konan University / 甲南大学. Retrieved from http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

厳, 向紅. “The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-.” 2016. Thesis, Konan University / 甲南大学. Accessed October 18, 2019. http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

厳, 向紅. “The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-.” 2016. Web. 18 Oct 2019.

Vancouver:

厳 . The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-. [Internet] [Thesis]. Konan University / 甲南大学; 2016. [cited 2019 Oct 18]. Available from: http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

厳 . The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-. [Thesis]. Konan University / 甲南大学; 2016. Available from: http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

22. Wahlen, S. Innate lymphoid cells in the intestines and the role of dietary compounds.

Degree: 2014, Universiteit Utrecht

 The incidence of intestinal related diseases such as inflammatory bowel disease annually increases and progress in research is impeded by the complexity of the intestinal… (more)

Subjects/Keywords: Innate lymphoid cells; AhR; Vitamin A; Vitamin D

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APA (6th Edition):

Wahlen, S. (2014). Innate lymphoid cells in the intestines and the role of dietary compounds. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/301212

Chicago Manual of Style (16th Edition):

Wahlen, S. “Innate lymphoid cells in the intestines and the role of dietary compounds.” 2014. Masters Thesis, Universiteit Utrecht. Accessed October 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/301212.

MLA Handbook (7th Edition):

Wahlen, S. “Innate lymphoid cells in the intestines and the role of dietary compounds.” 2014. Web. 18 Oct 2019.

Vancouver:

Wahlen S. Innate lymphoid cells in the intestines and the role of dietary compounds. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2019 Oct 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/301212.

Council of Science Editors:

Wahlen S. Innate lymphoid cells in the intestines and the role of dietary compounds. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/301212


University of Alberta

23. Amara, Issa. Modulation of Aryl Hydrocarbon Receptor Regulated-Genes by Mercury.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2013, University of Alberta

 Aryl hydrocarbon receptor (AhR) ligands such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and metals, such as mercury (Hg), are environmental co-contaminants with multiple biological consequences. Therefore, the objectives… (more)

Subjects/Keywords: AhR, Mercury, TCDD CYP1A1, NQO1, in vitro and in vivo

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APA (6th Edition):

Amara, I. (2013). Modulation of Aryl Hydrocarbon Receptor Regulated-Genes by Mercury. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/4m90dv732

Chicago Manual of Style (16th Edition):

Amara, Issa. “Modulation of Aryl Hydrocarbon Receptor Regulated-Genes by Mercury.” 2013. Doctoral Dissertation, University of Alberta. Accessed October 18, 2019. https://era.library.ualberta.ca/files/4m90dv732.

MLA Handbook (7th Edition):

Amara, Issa. “Modulation of Aryl Hydrocarbon Receptor Regulated-Genes by Mercury.” 2013. Web. 18 Oct 2019.

Vancouver:

Amara I. Modulation of Aryl Hydrocarbon Receptor Regulated-Genes by Mercury. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2019 Oct 18]. Available from: https://era.library.ualberta.ca/files/4m90dv732.

Council of Science Editors:

Amara I. Modulation of Aryl Hydrocarbon Receptor Regulated-Genes by Mercury. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/4m90dv732


Université de Sherbrooke

24. Bergeron, Sandra. Effets d'un mélange d'ingrédients actifs de pesticides sur l'activation de la voie du récepteur aux hydrocarbures d'aryle .

Degree: 2017, Université de Sherbrooke

 Depuis bon nombre d’années déjà, la question ne se pose plus ; l’utilisation des pesticides en agriculture est nécessaire puisque sans ces derniers les chances… (more)

Subjects/Keywords: AhR; CYP1A1; CYP1B1; ERα; MCF-7; Interaction croisée; Pesticides; Transcription génique

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APA (6th Edition):

Bergeron, S. (2017). Effets d'un mélange d'ingrédients actifs de pesticides sur l'activation de la voie du récepteur aux hydrocarbures d'aryle . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/11066

Chicago Manual of Style (16th Edition):

Bergeron, Sandra. “Effets d'un mélange d'ingrédients actifs de pesticides sur l'activation de la voie du récepteur aux hydrocarbures d'aryle .” 2017. Masters Thesis, Université de Sherbrooke. Accessed October 18, 2019. http://hdl.handle.net/11143/11066.

MLA Handbook (7th Edition):

Bergeron, Sandra. “Effets d'un mélange d'ingrédients actifs de pesticides sur l'activation de la voie du récepteur aux hydrocarbures d'aryle .” 2017. Web. 18 Oct 2019.

Vancouver:

Bergeron S. Effets d'un mélange d'ingrédients actifs de pesticides sur l'activation de la voie du récepteur aux hydrocarbures d'aryle . [Internet] [Masters thesis]. Université de Sherbrooke; 2017. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/11143/11066.

Council of Science Editors:

Bergeron S. Effets d'un mélange d'ingrédients actifs de pesticides sur l'activation de la voie du récepteur aux hydrocarbures d'aryle . [Masters Thesis]. Université de Sherbrooke; 2017. Available from: http://hdl.handle.net/11143/11066


Wright State University

25. Henseler, Rebecca Anne. Modulation of the 3'IgH Regulatory Region (3'IgH RR), a prospective in vitro screening tool for identifying potential immunotoxicants.

Degree: MS, Biological Sciences, 2007, Wright State University

 The immune system is critical to human survival. However, assessing alterations of immune function by potential immunotoxicants is complicated by the diffuse nature of the… (more)

Subjects/Keywords: IgH; AhR; IgH RR; TCDD; RR; Primaquine; ¿¿¿¿2b

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APA (6th Edition):

Henseler, R. A. (2007). Modulation of the 3'IgH Regulatory Region (3'IgH RR), a prospective in vitro screening tool for identifying potential immunotoxicants. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1196883435

Chicago Manual of Style (16th Edition):

Henseler, Rebecca Anne. “Modulation of the 3'IgH Regulatory Region (3'IgH RR), a prospective in vitro screening tool for identifying potential immunotoxicants.” 2007. Masters Thesis, Wright State University. Accessed October 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1196883435.

MLA Handbook (7th Edition):

Henseler, Rebecca Anne. “Modulation of the 3'IgH Regulatory Region (3'IgH RR), a prospective in vitro screening tool for identifying potential immunotoxicants.” 2007. Web. 18 Oct 2019.

Vancouver:

Henseler RA. Modulation of the 3'IgH Regulatory Region (3'IgH RR), a prospective in vitro screening tool for identifying potential immunotoxicants. [Internet] [Masters thesis]. Wright State University; 2007. [cited 2019 Oct 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1196883435.

Council of Science Editors:

Henseler RA. Modulation of the 3'IgH Regulatory Region (3'IgH RR), a prospective in vitro screening tool for identifying potential immunotoxicants. [Masters Thesis]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1196883435


Universiteit Utrecht

26. Mooij, F.A. de. The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food.

Degree: 2015, Universiteit Utrecht

 Layman’s summary Everybody knows that eating vegetables and overall healthy food is beneficial for your health. In recent years advertisers also started to tell us… (more)

Subjects/Keywords: ahr; aryl hydrocarbon receptor; probiotics; microbiotics; indole-3-carbinol; tryptophan

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APA (6th Edition):

Mooij, F. A. d. (2015). The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/309341

Chicago Manual of Style (16th Edition):

Mooij, F A de. “The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food.” 2015. Masters Thesis, Universiteit Utrecht. Accessed October 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/309341.

MLA Handbook (7th Edition):

Mooij, F A de. “The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food.” 2015. Web. 18 Oct 2019.

Vancouver:

Mooij FAd. The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food. [Internet] [Masters thesis]. Universiteit Utrecht; 2015. [cited 2019 Oct 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/309341.

Council of Science Editors:

Mooij FAd. The Aryl Hydrocarbon receptor and intestinal immunity: an overview of ligands derived from microbial metabolism and food. [Masters Thesis]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/309341


University of Gothenburg / Göteborgs Universitet

27. Albertsson, Eva. From Proteomic Analysis to Biomarker Application - Studies of Carbonyl Reductase in Fish.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

 Many anthropogenic substances are present in the aquatic environment, but there is limited information on how this combination of chemicals affects exposed wildlife. To assess… (more)

Subjects/Keywords: Fish; Proteomics; Biomarker; Carbonyl Reductase; AhR agonists; Oxidative stress; Biomonitoring

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APA (6th Edition):

Albertsson, E. (2011). From Proteomic Analysis to Biomarker Application - Studies of Carbonyl Reductase in Fish. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/26664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Albertsson, Eva. “From Proteomic Analysis to Biomarker Application - Studies of Carbonyl Reductase in Fish.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed October 18, 2019. http://hdl.handle.net/2077/26664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Albertsson, Eva. “From Proteomic Analysis to Biomarker Application - Studies of Carbonyl Reductase in Fish.” 2011. Web. 18 Oct 2019.

Vancouver:

Albertsson E. From Proteomic Analysis to Biomarker Application - Studies of Carbonyl Reductase in Fish. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/2077/26664.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Albertsson E. From Proteomic Analysis to Biomarker Application - Studies of Carbonyl Reductase in Fish. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/26664

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

28. Tanos , Rachel. Novel regulation of lipid homeostasis by the Ah receptor.

Degree: PhD, Biochemistry and Molecular Biology, 2012, Penn State University

 The aryl hydrocarbon receptor (AHR) is a ligand activated transcription factor. Activation of AHR has been associated with toxicity through its binding to dioxin response… (more)

Subjects/Keywords: Ah receptor; AHR; cholesterol; Fatty acid; lipid; SREBP

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APA (6th Edition):

Tanos , R. (2012). Novel regulation of lipid homeostasis by the Ah receptor. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/15460

Chicago Manual of Style (16th Edition):

Tanos , Rachel. “Novel regulation of lipid homeostasis by the Ah receptor.” 2012. Doctoral Dissertation, Penn State University. Accessed October 18, 2019. https://etda.libraries.psu.edu/catalog/15460.

MLA Handbook (7th Edition):

Tanos , Rachel. “Novel regulation of lipid homeostasis by the Ah receptor.” 2012. Web. 18 Oct 2019.

Vancouver:

Tanos R. Novel regulation of lipid homeostasis by the Ah receptor. [Internet] [Doctoral dissertation]. Penn State University; 2012. [cited 2019 Oct 18]. Available from: https://etda.libraries.psu.edu/catalog/15460.

Council of Science Editors:

Tanos R. Novel regulation of lipid homeostasis by the Ah receptor. [Doctoral Dissertation]. Penn State University; 2012. Available from: https://etda.libraries.psu.edu/catalog/15460

29. Cheng, Yating. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.

Degree: PhD, Toxicology, 2016, Texas A&M University

 LncRNAs are a group of non-coding RNAs containing >200 nucleotides and these RNAs have no significant protein coding potential. In the past 10-15 years the… (more)

Subjects/Keywords: Long noncoding RNAs; Pancreatic cancer; Gut microbiota, AhR

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APA (6th Edition):

Cheng, Y. (2016). The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/159030

Chicago Manual of Style (16th Edition):

Cheng, Yating. “The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.” 2016. Doctoral Dissertation, Texas A&M University. Accessed October 18, 2019. http://hdl.handle.net/1969.1/159030.

MLA Handbook (7th Edition):

Cheng, Yating. “The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.” 2016. Web. 18 Oct 2019.

Vancouver:

Cheng Y. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/1969.1/159030.

Council of Science Editors:

Cheng Y. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/159030


Université de Sherbrooke

30. Doyon, Kathy. Clonage de gènes de petits vertébrés susceptibles de voir leur expression induite par des pesticides environnementaux et séquençage et assemblage du génome de l'hirondelle bicolore (Tachycineta bicolor).

Degree: 2015, Université de Sherbrooke

 Environ 3500 tonnes de pesticides sont étendues chaque année sur les terres agricoles du Québec. L’utilisation de plusieurs de ces substances a été interdite, car… (more)

Subjects/Keywords: AhR; CYP1A1; CYP1B1; Régulation génique; Pesticides; Blarina brevicauda; Tachycineta bicolor

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APA (6th Edition):

Doyon, K. (2015). Clonage de gènes de petits vertébrés susceptibles de voir leur expression induite par des pesticides environnementaux et séquençage et assemblage du génome de l'hirondelle bicolore (Tachycineta bicolor). (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6869

Chicago Manual of Style (16th Edition):

Doyon, Kathy. “Clonage de gènes de petits vertébrés susceptibles de voir leur expression induite par des pesticides environnementaux et séquençage et assemblage du génome de l'hirondelle bicolore (Tachycineta bicolor). ” 2015. Masters Thesis, Université de Sherbrooke. Accessed October 18, 2019. http://hdl.handle.net/11143/6869.

MLA Handbook (7th Edition):

Doyon, Kathy. “Clonage de gènes de petits vertébrés susceptibles de voir leur expression induite par des pesticides environnementaux et séquençage et assemblage du génome de l'hirondelle bicolore (Tachycineta bicolor). ” 2015. Web. 18 Oct 2019.

Vancouver:

Doyon K. Clonage de gènes de petits vertébrés susceptibles de voir leur expression induite par des pesticides environnementaux et séquençage et assemblage du génome de l'hirondelle bicolore (Tachycineta bicolor). [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2019 Oct 18]. Available from: http://hdl.handle.net/11143/6869.

Council of Science Editors:

Doyon K. Clonage de gènes de petits vertébrés susceptibles de voir leur expression induite par des pesticides environnementaux et séquençage et assemblage du génome de l'hirondelle bicolore (Tachycineta bicolor). [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://hdl.handle.net/11143/6869

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