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You searched for subject:(Adult T cell leukemia). Showing records 1 – 30 of 49674 total matches.

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1. Takachi, Takayuki. Human T-cell leukemia virus type 1 Tax oncoprotein represses the expression of the BCL11B tumor suppressor in T-cells : ヒトT細胞白血病ウイルス1型の癌蛋白Taxは癌抑制遺伝子BCL11Bの発現を抑制する.

Degree: 博士(医学), 2015, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第4067号. 学位記番号: 新大院博(医)甲第656号. 学位授与年月日: 平成27年9月24日

Cancer Science. 2015, 106(4), 461-465.

Human T-cell leukemia virus type 1 (HTLV-1) is the etiological agent of… (more)

Subjects/Keywords: Adult T cell leukemia; BCL11B; HBZ; HTLV-1; Tax

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Takachi, T. (2015). Human T-cell leukemia virus type 1 Tax oncoprotein represses the expression of the BCL11B tumor suppressor in T-cells : ヒトT細胞白血病ウイルス1型の癌蛋白Taxは癌抑制遺伝子BCL11Bの発現を抑制する. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/35559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takachi, Takayuki. “Human T-cell leukemia virus type 1 Tax oncoprotein represses the expression of the BCL11B tumor suppressor in T-cells : ヒトT細胞白血病ウイルス1型の癌蛋白Taxは癌抑制遺伝子BCL11Bの発現を抑制する.” 2015. Thesis, Niigata University / 新潟大学. Accessed March 05, 2021. http://hdl.handle.net/10191/35559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takachi, Takayuki. “Human T-cell leukemia virus type 1 Tax oncoprotein represses the expression of the BCL11B tumor suppressor in T-cells : ヒトT細胞白血病ウイルス1型の癌蛋白Taxは癌抑制遺伝子BCL11Bの発現を抑制する.” 2015. Web. 05 Mar 2021.

Vancouver:

Takachi T. Human T-cell leukemia virus type 1 Tax oncoprotein represses the expression of the BCL11B tumor suppressor in T-cells : ヒトT細胞白血病ウイルス1型の癌蛋白Taxは癌抑制遺伝子BCL11Bの発現を抑制する. [Internet] [Thesis]. Niigata University / 新潟大学; 2015. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/10191/35559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takachi T. Human T-cell leukemia virus type 1 Tax oncoprotein represses the expression of the BCL11B tumor suppressor in T-cells : ヒトT細胞白血病ウイルス1型の癌蛋白Taxは癌抑制遺伝子BCL11Bの発現を抑制する. [Thesis]. Niigata University / 新潟大学; 2015. Available from: http://hdl.handle.net/10191/35559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. 牧山, 純也. Treatment outcome of elderly patients with aggressive adult T cell leukemia-lymphoma: Nagasaki University Hospital experience : 長崎大学病院における高齢者成人T細胞白血病・リンパ腫 (ATL) の治療成績.

Degree: 博士(医学), 2014, Nagasaki University / 長崎大学

 VCAP (vincristine, cyclophosphamide, doxorubicin, and prednisone)-AMP (doxorubicin, ranimustine, and prednisone)-VECP (vindesine, etoposide, carboplatin, and prednisone) is a standard regimen for aggressive adult T cell leukemia-lymphoma… (more)

Subjects/Keywords: Adult T cell leukemia-lymphoma (ATL); Chemotherapy; Elderly patients

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APA (6th Edition):

牧山, . (2014). Treatment outcome of elderly patients with aggressive adult T cell leukemia-lymphoma: Nagasaki University Hospital experience : 長崎大学病院における高齢者成人T細胞白血病・リンパ腫 (ATL) の治療成績. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35926

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

牧山, 純也. “Treatment outcome of elderly patients with aggressive adult T cell leukemia-lymphoma: Nagasaki University Hospital experience : 長崎大学病院における高齢者成人T細胞白血病・リンパ腫 (ATL) の治療成績.” 2014. Thesis, Nagasaki University / 長崎大学. Accessed March 05, 2021. http://hdl.handle.net/10069/35926.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

牧山, 純也. “Treatment outcome of elderly patients with aggressive adult T cell leukemia-lymphoma: Nagasaki University Hospital experience : 長崎大学病院における高齢者成人T細胞白血病・リンパ腫 (ATL) の治療成績.” 2014. Web. 05 Mar 2021.

Vancouver:

牧山 . Treatment outcome of elderly patients with aggressive adult T cell leukemia-lymphoma: Nagasaki University Hospital experience : 長崎大学病院における高齢者成人T細胞白血病・リンパ腫 (ATL) の治療成績. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2014. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/10069/35926.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

牧山 . Treatment outcome of elderly patients with aggressive adult T cell leukemia-lymphoma: Nagasaki University Hospital experience : 長崎大学病院における高齢者成人T細胞白血病・リンパ腫 (ATL) の治療成績. [Thesis]. Nagasaki University / 長崎大学; 2014. Available from: http://hdl.handle.net/10069/35926

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Kohart, Nicole Ann, Kohart. Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis.

Degree: PhD, Comparative and Veterinary Medicine, 2017, The Ohio State University

Adult T-cell Leukemia/Lymphoma (ATL) is a rapidly fatal hematological malignancy that develops in a subset of Human T-cell Leukemia Virus Type 1 (HTLV-1) carriers after… (more)

Subjects/Keywords: Comparative; Pathology; Adult T cell Leukemia Lymphoma; bone metastasis

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APA (6th Edition):

Kohart, Nicole Ann, K. (2017). Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1503248777003095

Chicago Manual of Style (16th Edition):

Kohart, Nicole Ann, Kohart. “Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis.” 2017. Doctoral Dissertation, The Ohio State University. Accessed March 05, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1503248777003095.

MLA Handbook (7th Edition):

Kohart, Nicole Ann, Kohart. “Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis.” 2017. Web. 05 Mar 2021.

Vancouver:

Kohart, Nicole Ann K. Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis. [Internet] [Doctoral dissertation]. The Ohio State University; 2017. [cited 2021 Mar 05]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1503248777003095.

Council of Science Editors:

Kohart, Nicole Ann K. Models, Mechanisms, and Treatment of Adult T-cell Leukemia/Lymphoma Bone Metastasis. [Doctoral Dissertation]. The Ohio State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1503248777003095


Vanderbilt University

4. Elliott, Natalina Elizabeth. The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

Adult T-cell leukemia/lymphoma (ATLL) is an incurable peripheral T-cell malignancy where most patients succumb within the first year of diagnosis. Development of ATLL requires human… (more)

Subjects/Keywords: adult t-cell leukemia/lymphoma; Leukemia; FERM; JAK3; ATLL; HTLV-1; IL-2; mutation; t-cell

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APA (6th Edition):

Elliott, N. E. (2013). The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10404

Chicago Manual of Style (16th Edition):

Elliott, Natalina Elizabeth. “The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed March 05, 2021. http://hdl.handle.net/1803/10404.

MLA Handbook (7th Edition):

Elliott, Natalina Elizabeth. “The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma.” 2013. Web. 05 Mar 2021.

Vancouver:

Elliott NE. The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/1803/10404.

Council of Science Editors:

Elliott NE. The role of activating mutations in the FERM domain of janus kinase 3 in the development of adult t-cell leukemia/lymphoma. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/10404


Kyoto University / 京都大学

5. Nagai, Yuya. T memory stem cells are the hierarchical apex of adult T-cell leukemia. : 記憶型T幹細胞を頂点とした成人T細胞白血病の階層構造の解明.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第19267号

医博第4031号

Subjects/Keywords: adult T-cell leukemia; HTLV-1; T memory stem cell; T cell differentiation

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APA (6th Edition):

Nagai, Y. (2015). T memory stem cells are the hierarchical apex of adult T-cell leukemia. : 記憶型T幹細胞を頂点とした成人T細胞白血病の階層構造の解明. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/202670 ; http://dx.doi.org/10.14989/doctor.k19267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nagai, Yuya. “T memory stem cells are the hierarchical apex of adult T-cell leukemia. : 記憶型T幹細胞を頂点とした成人T細胞白血病の階層構造の解明.” 2015. Thesis, Kyoto University / 京都大学. Accessed March 05, 2021. http://hdl.handle.net/2433/202670 ; http://dx.doi.org/10.14989/doctor.k19267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nagai, Yuya. “T memory stem cells are the hierarchical apex of adult T-cell leukemia. : 記憶型T幹細胞を頂点とした成人T細胞白血病の階層構造の解明.” 2015. Web. 05 Mar 2021.

Vancouver:

Nagai Y. T memory stem cells are the hierarchical apex of adult T-cell leukemia. : 記憶型T幹細胞を頂点とした成人T細胞白血病の階層構造の解明. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/202670 ; http://dx.doi.org/10.14989/doctor.k19267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nagai Y. T memory stem cells are the hierarchical apex of adult T-cell leukemia. : 記憶型T幹細胞を頂点とした成人T細胞白血病の階層構造の解明. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/202670 ; http://dx.doi.org/10.14989/doctor.k19267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Nagai, Yuya. T memory stem cells are the hierarchical apex of adult T-cell leukemia.

Degree: 2015, Kyoto University

Subjects/Keywords: adult T-cell leukemia; HTLV-1; T memory stem cell; T cell differentiation

Page 1 Page 2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nagai, Y. (2015). T memory stem cells are the hierarchical apex of adult T-cell leukemia. (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/202670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nagai, Yuya. “T memory stem cells are the hierarchical apex of adult T-cell leukemia. ” 2015. Thesis, Kyoto University. Accessed March 05, 2021. http://hdl.handle.net/2433/202670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nagai, Yuya. “T memory stem cells are the hierarchical apex of adult T-cell leukemia. ” 2015. Web. 05 Mar 2021.

Vancouver:

Nagai Y. T memory stem cells are the hierarchical apex of adult T-cell leukemia. [Internet] [Thesis]. Kyoto University; 2015. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/202670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nagai Y. T memory stem cells are the hierarchical apex of adult T-cell leukemia. [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/202670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Tada, Kohei. Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia : 抗HIV薬アバカビルは、TDP1が欠損している成人T細胞白血病を標的とする.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第19363号

医博第4040号

Subjects/Keywords: Adult T cell leukemia; Abacavir; TDP1; NRTI; chemotherapy

Page 1 Page 2

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APA (6th Edition):

Tada, K. (2015). Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia : 抗HIV薬アバカビルは、TDP1が欠損している成人T細胞白血病を標的とする. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/202797 ; http://dx.doi.org/10.14989/doctor.k19363

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tada, Kohei. “Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia : 抗HIV薬アバカビルは、TDP1が欠損している成人T細胞白血病を標的とする.” 2015. Thesis, Kyoto University / 京都大学. Accessed March 05, 2021. http://hdl.handle.net/2433/202797 ; http://dx.doi.org/10.14989/doctor.k19363.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tada, Kohei. “Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia : 抗HIV薬アバカビルは、TDP1が欠損している成人T細胞白血病を標的とする.” 2015. Web. 05 Mar 2021.

Vancouver:

Tada K. Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia : 抗HIV薬アバカビルは、TDP1が欠損している成人T細胞白血病を標的とする. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/202797 ; http://dx.doi.org/10.14989/doctor.k19363.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tada K. Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia : 抗HIV薬アバカビルは、TDP1が欠損している成人T細胞白血病を標的とする. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/202797 ; http://dx.doi.org/10.14989/doctor.k19363

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Tada, Kohei. Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia .

Degree: 2015, Kyoto University

Subjects/Keywords: Adult T cell leukemia; Abacavir; TDP1; NRTI; chemotherapy

Page 1 Page 2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tada, K. (2015). Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/202797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tada, Kohei. “Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia .” 2015. Thesis, Kyoto University. Accessed March 05, 2021. http://hdl.handle.net/2433/202797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tada, Kohei. “Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia .” 2015. Web. 05 Mar 2021.

Vancouver:

Tada K. Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia . [Internet] [Thesis]. Kyoto University; 2015. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/202797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tada K. Abacavir, an anti-HIV-1 drug, targets TDP1-deficient adult T cell leukemia . [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/202797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Macaire, Héloïse. Régulation de l’expression des protéines anti-apoptotiques Bfl-1 et Bcl-xL par les protéines virales Tax et HBZ du virus HTLV-1 et identification de petites molécules anti-Bfl-1 à visée thérapeutique : Regulation of Bfl-1 and Bcl-xL anti-apoptotic protein expression by the HTLV-1 Tax and HBZ proteins and identification of small therapeutic molecules directed against Bfl-1 v.

Degree: Docteur es, Virologie-cancérologie, 2011, Université Claude Bernard – Lyon I

Le virus humain T lymphotrope de type 1 (HTLV-1) est l’agent étiologique de la leucémie/lymphome T de l’adulte (ATLL) qui se développe après plusieurs décennies… (more)

Subjects/Keywords: Virus humain T lymphotrope de type 1 (HTLV-1); Leucémie / lymphome T de l'adulte (ATLL); Tax; HBZ; Bfl-1; Bcl-xL; Apoptose; Cible thérapeutique; Human T-cell leukemia virus type 1; Adult T-cell leukemia / lymphoma (ATLL); Tax; HBZ; Bfl-1; Bcl-xL; Apoptosis; Therapeutic target; 579.2

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APA (6th Edition):

Macaire, H. (2011). Régulation de l’expression des protéines anti-apoptotiques Bfl-1 et Bcl-xL par les protéines virales Tax et HBZ du virus HTLV-1 et identification de petites molécules anti-Bfl-1 à visée thérapeutique : Regulation of Bfl-1 and Bcl-xL anti-apoptotic protein expression by the HTLV-1 Tax and HBZ proteins and identification of small therapeutic molecules directed against Bfl-1 v. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10357

Chicago Manual of Style (16th Edition):

Macaire, Héloïse. “Régulation de l’expression des protéines anti-apoptotiques Bfl-1 et Bcl-xL par les protéines virales Tax et HBZ du virus HTLV-1 et identification de petites molécules anti-Bfl-1 à visée thérapeutique : Regulation of Bfl-1 and Bcl-xL anti-apoptotic protein expression by the HTLV-1 Tax and HBZ proteins and identification of small therapeutic molecules directed against Bfl-1 v.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed March 05, 2021. http://www.theses.fr/2011LYO10357.

MLA Handbook (7th Edition):

Macaire, Héloïse. “Régulation de l’expression des protéines anti-apoptotiques Bfl-1 et Bcl-xL par les protéines virales Tax et HBZ du virus HTLV-1 et identification de petites molécules anti-Bfl-1 à visée thérapeutique : Regulation of Bfl-1 and Bcl-xL anti-apoptotic protein expression by the HTLV-1 Tax and HBZ proteins and identification of small therapeutic molecules directed against Bfl-1 v.” 2011. Web. 05 Mar 2021.

Vancouver:

Macaire H. Régulation de l’expression des protéines anti-apoptotiques Bfl-1 et Bcl-xL par les protéines virales Tax et HBZ du virus HTLV-1 et identification de petites molécules anti-Bfl-1 à visée thérapeutique : Regulation of Bfl-1 and Bcl-xL anti-apoptotic protein expression by the HTLV-1 Tax and HBZ proteins and identification of small therapeutic molecules directed against Bfl-1 v. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2021 Mar 05]. Available from: http://www.theses.fr/2011LYO10357.

Council of Science Editors:

Macaire H. Régulation de l’expression des protéines anti-apoptotiques Bfl-1 et Bcl-xL par les protéines virales Tax et HBZ du virus HTLV-1 et identification de petites molécules anti-Bfl-1 à visée thérapeutique : Regulation of Bfl-1 and Bcl-xL anti-apoptotic protein expression by the HTLV-1 Tax and HBZ proteins and identification of small therapeutic molecules directed against Bfl-1 v. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10357

10. 佐々木, 了政. YM155 suppresses cell proliferation and induces cell death in human adult T-cell leukemia/lymphoma cells : 成人T細胞白血病細胞株においてYM155は抗アポトーシスタンパクの発現を抑制しアポトーシスを誘導する.

Degree: 医学博士, 2016, Iwate Medical University / 岩手医科大学

2015

Subjects/Keywords: YM155; Surviving; STAT3; Mcl-1; Adult T-cell leukemia; YM155; Surviving; STAT3; Mcl-1; Adult T-cell leukemia

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APA (6th Edition):

佐々木, . (2016). YM155 suppresses cell proliferation and induces cell death in human adult T-cell leukemia/lymphoma cells : 成人T細胞白血病細胞株においてYM155は抗アポトーシスタンパクの発現を抑制しアポトーシスを誘導する. (Thesis). Iwate Medical University / 岩手医科大学. Retrieved from http://id.nii.ac.jp/1181/00005674/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

佐々木, 了政. “YM155 suppresses cell proliferation and induces cell death in human adult T-cell leukemia/lymphoma cells : 成人T細胞白血病細胞株においてYM155は抗アポトーシスタンパクの発現を抑制しアポトーシスを誘導する.” 2016. Thesis, Iwate Medical University / 岩手医科大学. Accessed March 05, 2021. http://id.nii.ac.jp/1181/00005674/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

佐々木, 了政. “YM155 suppresses cell proliferation and induces cell death in human adult T-cell leukemia/lymphoma cells : 成人T細胞白血病細胞株においてYM155は抗アポトーシスタンパクの発現を抑制しアポトーシスを誘導する.” 2016. Web. 05 Mar 2021.

Vancouver:

佐々木 . YM155 suppresses cell proliferation and induces cell death in human adult T-cell leukemia/lymphoma cells : 成人T細胞白血病細胞株においてYM155は抗アポトーシスタンパクの発現を抑制しアポトーシスを誘導する. [Internet] [Thesis]. Iwate Medical University / 岩手医科大学; 2016. [cited 2021 Mar 05]. Available from: http://id.nii.ac.jp/1181/00005674/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

佐々木 . YM155 suppresses cell proliferation and induces cell death in human adult T-cell leukemia/lymphoma cells : 成人T細胞白血病細胞株においてYM155は抗アポトーシスタンパクの発現を抑制しアポトーシスを誘導する. [Thesis]. Iwate Medical University / 岩手医科大学; 2016. Available from: http://id.nii.ac.jp/1181/00005674/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Pérès, Eléonore. La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1 : The humanized mouse : an in vivo model for the leukemogenic process induced by HTLV-1.

Degree: Docteur es, Sciences de la Vie, 2017, Lyon

La leucémie T de l’adulte (ATL), caractérisée par une prolifération dérégulée de lymphocytesT CD4+ activés, se développe chez des individus infectés par le virus T-lymphotropehumain… (more)

Subjects/Keywords: HTLV-1; Souris humanisée; Leucémie T de l’adulte; Leucémogenèse; Protéines PDZ; Chimiotactisme; HTLV-1; Humanized mouse; Adult t-cell Leukemia; Leukemogenesis; PDZ proteins; Chimiotaxis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pérès, E. (2017). La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1 : The humanized mouse : an in vivo model for the leukemogenic process induced by HTLV-1. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2017LYSEN043

Chicago Manual of Style (16th Edition):

Pérès, Eléonore. “La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1 : The humanized mouse : an in vivo model for the leukemogenic process induced by HTLV-1.” 2017. Doctoral Dissertation, Lyon. Accessed March 05, 2021. http://www.theses.fr/2017LYSEN043.

MLA Handbook (7th Edition):

Pérès, Eléonore. “La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1 : The humanized mouse : an in vivo model for the leukemogenic process induced by HTLV-1.” 2017. Web. 05 Mar 2021.

Vancouver:

Pérès E. La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1 : The humanized mouse : an in vivo model for the leukemogenic process induced by HTLV-1. [Internet] [Doctoral dissertation]. Lyon; 2017. [cited 2021 Mar 05]. Available from: http://www.theses.fr/2017LYSEN043.

Council of Science Editors:

Pérès E. La souris humanisée : modèle d'étude in vivo du processus leucémogène induit par HTLV-1 : The humanized mouse : an in vivo model for the leukemogenic process induced by HTLV-1. [Doctoral Dissertation]. Lyon; 2017. Available from: http://www.theses.fr/2017LYSEN043


Kyoto University / 京都大学

12. Mitagami, Yu. Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice : インターフェロンγはHBZトランスジェニックマウスの炎症とTリンパ腫の発症を促進する.

Degree: 博士(医科学), 2016, Kyoto University / 京都大学

新制・課程博士

甲第19628号

医科博第66号

Subjects/Keywords: human T-cell leukemia virus type 1; adult T-cell leukemia; HTLV-1 bZIP factor; IFN-gamma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mitagami, Y. (2016). Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice : インターフェロンγはHBZトランスジェニックマウスの炎症とTリンパ腫の発症を促進する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215454 ; http://dx.doi.org/10.14989/doctor.k19628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mitagami, Yu. “Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice : インターフェロンγはHBZトランスジェニックマウスの炎症とTリンパ腫の発症を促進する.” 2016. Thesis, Kyoto University / 京都大学. Accessed March 05, 2021. http://hdl.handle.net/2433/215454 ; http://dx.doi.org/10.14989/doctor.k19628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mitagami, Yu. “Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice : インターフェロンγはHBZトランスジェニックマウスの炎症とTリンパ腫の発症を促進する.” 2016. Web. 05 Mar 2021.

Vancouver:

Mitagami Y. Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice : インターフェロンγはHBZトランスジェニックマウスの炎症とTリンパ腫の発症を促進する. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/215454 ; http://dx.doi.org/10.14989/doctor.k19628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mitagami Y. Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice : インターフェロンγはHBZトランスジェニックマウスの炎症とTリンパ腫の発症を促進する. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215454 ; http://dx.doi.org/10.14989/doctor.k19628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Mitagami, Yu. Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice .

Degree: 2016, Kyoto University

Subjects/Keywords: human T-cell leukemia virus type 1; adult T-cell leukemia; HTLV-1 bZIP factor; IFN-gamma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mitagami, Y. (2016). Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/215454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mitagami, Yu. “Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice .” 2016. Thesis, Kyoto University. Accessed March 05, 2021. http://hdl.handle.net/2433/215454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mitagami, Yu. “Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice .” 2016. Web. 05 Mar 2021.

Vancouver:

Mitagami Y. Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice . [Internet] [Thesis]. Kyoto University; 2016. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/215454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mitagami Y. Interferon-γ promotes inflammation and development of T-cell lymphoma in HTLV-1 bZIP factor transgenic mice . [Thesis]. Kyoto University; 2016. Available from: http://hdl.handle.net/2433/215454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Cheminant, Morgane. Rôle des récepteurs NK dans les lymphoproliférations T matures. Exemple des leucémies/lymphomes T de l'adulte associés à l'HTLV-1 et des lymphoproliférations T intestinales primitives : Role of NK Receptors in Peripheral T-cell Lymphoma. Example of Adult T-cell Leukemia/Lymphoma and Primary Gastro-intestinal T-cell Lymphoproliferative Disease.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Université Paris-Saclay (ComUE)

Les lymphomes T périphériques (PTCLs) sont des entités hétérogènes dont la classification a récemment été révisée. Plus de 25 entités de lymphomes T ou NK… (more)

Subjects/Keywords: Récepteurs NK; Leucémies/lymphomes T de l'adulte associés à l'HLTV-1; Kir3dl2; NKp46; Lymphoproliferations T intestinales primitives; Maladie coeliaque réfractaire; NK receptors; Adult T-Cell Leukemia/Lymphoma; Kir3dl2; NKp46; Primary Gastro-Intestinal T-Cell Lymphoproliferative Disease; Refractory Celiac Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cheminant, M. (2018). Rôle des récepteurs NK dans les lymphoproliférations T matures. Exemple des leucémies/lymphomes T de l'adulte associés à l'HTLV-1 et des lymphoproliférations T intestinales primitives : Role of NK Receptors in Peripheral T-cell Lymphoma. Example of Adult T-cell Leukemia/Lymphoma and Primary Gastro-intestinal T-cell Lymphoproliferative Disease. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS301

Chicago Manual of Style (16th Edition):

Cheminant, Morgane. “Rôle des récepteurs NK dans les lymphoproliférations T matures. Exemple des leucémies/lymphomes T de l'adulte associés à l'HTLV-1 et des lymphoproliférations T intestinales primitives : Role of NK Receptors in Peripheral T-cell Lymphoma. Example of Adult T-cell Leukemia/Lymphoma and Primary Gastro-intestinal T-cell Lymphoproliferative Disease.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed March 05, 2021. http://www.theses.fr/2018SACLS301.

MLA Handbook (7th Edition):

Cheminant, Morgane. “Rôle des récepteurs NK dans les lymphoproliférations T matures. Exemple des leucémies/lymphomes T de l'adulte associés à l'HTLV-1 et des lymphoproliférations T intestinales primitives : Role of NK Receptors in Peripheral T-cell Lymphoma. Example of Adult T-cell Leukemia/Lymphoma and Primary Gastro-intestinal T-cell Lymphoproliferative Disease.” 2018. Web. 05 Mar 2021.

Vancouver:

Cheminant M. Rôle des récepteurs NK dans les lymphoproliférations T matures. Exemple des leucémies/lymphomes T de l'adulte associés à l'HTLV-1 et des lymphoproliférations T intestinales primitives : Role of NK Receptors in Peripheral T-cell Lymphoma. Example of Adult T-cell Leukemia/Lymphoma and Primary Gastro-intestinal T-cell Lymphoproliferative Disease. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2021 Mar 05]. Available from: http://www.theses.fr/2018SACLS301.

Council of Science Editors:

Cheminant M. Rôle des récepteurs NK dans les lymphoproliférations T matures. Exemple des leucémies/lymphomes T de l'adulte associés à l'HTLV-1 et des lymphoproliférations T intestinales primitives : Role of NK Receptors in Peripheral T-cell Lymphoma. Example of Adult T-cell Leukemia/Lymphoma and Primary Gastro-intestinal T-cell Lymphoproliferative Disease. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS301


Kyoto University / 京都大学

15. Tanaka, Azusa. HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization : HTLV-1 bZIP Factorは転写因子FoxO3aの機能および局在に影響を及ぼしアポトーシスを阻害する.

Degree: 博士(医科学), 2014, Kyoto University / 京都大学

新制・課程博士

甲第17979号

医科博第48号

Subjects/Keywords: HTLV-1 bZIP Factor; Apoptosis; FoxO3a; Adult T-cell leukemia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tanaka, A. (2014). HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization : HTLV-1 bZIP Factorは転写因子FoxO3aの機能および局在に影響を及ぼしアポトーシスを阻害する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/185199 ; http://dx.doi.org/10.14989/doctor.k17979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tanaka, Azusa. “HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization : HTLV-1 bZIP Factorは転写因子FoxO3aの機能および局在に影響を及ぼしアポトーシスを阻害する.” 2014. Thesis, Kyoto University / 京都大学. Accessed March 05, 2021. http://hdl.handle.net/2433/185199 ; http://dx.doi.org/10.14989/doctor.k17979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tanaka, Azusa. “HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization : HTLV-1 bZIP Factorは転写因子FoxO3aの機能および局在に影響を及ぼしアポトーシスを阻害する.” 2014. Web. 05 Mar 2021.

Vancouver:

Tanaka A. HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization : HTLV-1 bZIP Factorは転写因子FoxO3aの機能および局在に影響を及ぼしアポトーシスを阻害する. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/185199 ; http://dx.doi.org/10.14989/doctor.k17979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tanaka A. HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization : HTLV-1 bZIP Factorは転写因子FoxO3aの機能および局在に影響を及ぼしアポトーシスを阻害する. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/185199 ; http://dx.doi.org/10.14989/doctor.k17979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. 国廣, 真里枝. Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells : ヒートショックは、ヒトT細胞白血病ウイルスI型(HTLV-1)感染者由来の初代培養と継代培養されたT細胞のTax抗原発現を促進する.

Degree: 博士(医学), 2017, University of the Ryukyus / 琉球大学

Subjects/Keywords: HTLV-I; Tax; heat shock; adult T cell leukemia (ATL)

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APA (6th Edition):

国廣, . (2017). Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells : ヒートショックは、ヒトT細胞白血病ウイルスI型(HTLV-1)感染者由来の初代培養と継代培養されたT細胞のTax抗原発現を促進する. (Thesis). University of the Ryukyus / 琉球大学. Retrieved from http://hdl.handle.net/20.500.12000/36646

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

国廣, 真里枝. “Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells : ヒートショックは、ヒトT細胞白血病ウイルスI型(HTLV-1)感染者由来の初代培養と継代培養されたT細胞のTax抗原発現を促進する.” 2017. Thesis, University of the Ryukyus / 琉球大学. Accessed March 05, 2021. http://hdl.handle.net/20.500.12000/36646.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

国廣, 真里枝. “Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells : ヒートショックは、ヒトT細胞白血病ウイルスI型(HTLV-1)感染者由来の初代培養と継代培養されたT細胞のTax抗原発現を促進する.” 2017. Web. 05 Mar 2021.

Vancouver:

国廣 . Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells : ヒートショックは、ヒトT細胞白血病ウイルスI型(HTLV-1)感染者由来の初代培養と継代培養されたT細胞のTax抗原発現を促進する. [Internet] [Thesis]. University of the Ryukyus / 琉球大学; 2017. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/20.500.12000/36646.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

国廣 . Heat Shock Enhances the Expression of the Human T Cell Leukemia Virus Type-I (HTLV-I) Trans-Activator (Tax) Antigen in Human HTLV-I Infected Primary and Cultured T Cells : ヒートショックは、ヒトT細胞白血病ウイルスI型(HTLV-1)感染者由来の初代培養と継代培養されたT細胞のTax抗原発現を促進する. [Thesis]. University of the Ryukyus / 琉球大学; 2017. Available from: http://hdl.handle.net/20.500.12000/36646

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

17. Tanaka, Azusa. HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization .

Degree: 2014, Kyoto University

Subjects/Keywords: HTLV-1 bZIP Factor; Apoptosis; FoxO3a; Adult T-cell leukemia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tanaka, A. (2014). HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/185199

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tanaka, Azusa. “HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization .” 2014. Thesis, Kyoto University. Accessed March 05, 2021. http://hdl.handle.net/2433/185199.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tanaka, Azusa. “HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization .” 2014. Web. 05 Mar 2021.

Vancouver:

Tanaka A. HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization . [Internet] [Thesis]. Kyoto University; 2014. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/185199.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tanaka A. HTLV-1 bZIP Factor Suppresses Apoptosis by Attenuating the Function of FoxO3a and Altering its Localization . [Thesis]. Kyoto University; 2014. Available from: http://hdl.handle.net/2433/185199

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Kawata, Takahito. Dual inhibition of the mTORC1 and mTORC2 signaling pathways is a promising therapeutic target for Adult T-cell Leukemia .

Degree: 2018, Kyoto University

Subjects/Keywords: adult t cell leukemia; Akt; mTORC; TOR Serinr-Threonine Kinases; HTLV-1

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APA (6th Edition):

Kawata, T. (2018). Dual inhibition of the mTORC1 and mTORC2 signaling pathways is a promising therapeutic target for Adult T-cell Leukemia . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232109

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kawata, Takahito. “Dual inhibition of the mTORC1 and mTORC2 signaling pathways is a promising therapeutic target for Adult T-cell Leukemia .” 2018. Thesis, Kyoto University. Accessed March 05, 2021. http://hdl.handle.net/2433/232109.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kawata, Takahito. “Dual inhibition of the mTORC1 and mTORC2 signaling pathways is a promising therapeutic target for Adult T-cell Leukemia .” 2018. Web. 05 Mar 2021.

Vancouver:

Kawata T. Dual inhibition of the mTORC1 and mTORC2 signaling pathways is a promising therapeutic target for Adult T-cell Leukemia . [Internet] [Thesis]. Kyoto University; 2018. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/232109.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kawata T. Dual inhibition of the mTORC1 and mTORC2 signaling pathways is a promising therapeutic target for Adult T-cell Leukemia . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232109

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Mohamed, Mahgoub Mohamed Ahmed Mohamed. Sporadic on/off switching of HTLV-1 Tax expression is crucial to maintain the whole population of virus-induced leukemic cells .

Degree: 2018, Kyoto University

Subjects/Keywords: HTLV-1; Tax; HBZ; adult T-cell leukemia-lymphoma; computational simulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mohamed, M. M. A. M. (2018). Sporadic on/off switching of HTLV-1 Tax expression is crucial to maintain the whole population of virus-induced leukemic cells . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232139

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mohamed, Mahgoub Mohamed Ahmed Mohamed. “Sporadic on/off switching of HTLV-1 Tax expression is crucial to maintain the whole population of virus-induced leukemic cells .” 2018. Thesis, Kyoto University. Accessed March 05, 2021. http://hdl.handle.net/2433/232139.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mohamed, Mahgoub Mohamed Ahmed Mohamed. “Sporadic on/off switching of HTLV-1 Tax expression is crucial to maintain the whole population of virus-induced leukemic cells .” 2018. Web. 05 Mar 2021.

Vancouver:

Mohamed MMAM. Sporadic on/off switching of HTLV-1 Tax expression is crucial to maintain the whole population of virus-induced leukemic cells . [Internet] [Thesis]. Kyoto University; 2018. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/2433/232139.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mohamed MMAM. Sporadic on/off switching of HTLV-1 Tax expression is crucial to maintain the whole population of virus-induced leukemic cells . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232139

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Meireles, Ana Luísa Langanke Pedroso. Quantificação de células endoteliais circulantes em portadores assintomáticos do vírus linfotrópico humano de células T do tipo 1 (HTLV1) por citometria de fluxo.

Degree: Mestrado, Distúrbios do Crescimento Celular, Hemodinâmicos e da Hemostasia, 2009, University of São Paulo

Células endoteliais provenientes da medula óssea (MO) participam da fisiopatologia de várias doenças que possuem dano vascular como fator em comum. Apesar de consideradas evento… (more)

Subjects/Keywords: Células endoteliais; Citometria de fluxo; Endothelial cells; Flow cytometry; Human T-lymphotropic virus 1; Leucemia-linfoma de células T do adulto; Leukemia-lymphoma adult T-cell; Neovascularização patológica; Neovascularization pathologic; Virus linfotrópico de células T humanas tipo 1

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APA (6th Edition):

Meireles, A. L. L. P. (2009). Quantificação de células endoteliais circulantes em portadores assintomáticos do vírus linfotrópico humano de células T do tipo 1 (HTLV1) por citometria de fluxo. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5167/tde-09062009-170516/ ;

Chicago Manual of Style (16th Edition):

Meireles, Ana Luísa Langanke Pedroso. “Quantificação de células endoteliais circulantes em portadores assintomáticos do vírus linfotrópico humano de células T do tipo 1 (HTLV1) por citometria de fluxo.” 2009. Masters Thesis, University of São Paulo. Accessed March 05, 2021. http://www.teses.usp.br/teses/disponiveis/5/5167/tde-09062009-170516/ ;.

MLA Handbook (7th Edition):

Meireles, Ana Luísa Langanke Pedroso. “Quantificação de células endoteliais circulantes em portadores assintomáticos do vírus linfotrópico humano de células T do tipo 1 (HTLV1) por citometria de fluxo.” 2009. Web. 05 Mar 2021.

Vancouver:

Meireles ALLP. Quantificação de células endoteliais circulantes em portadores assintomáticos do vírus linfotrópico humano de células T do tipo 1 (HTLV1) por citometria de fluxo. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2021 Mar 05]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5167/tde-09062009-170516/ ;.

Council of Science Editors:

Meireles ALLP. Quantificação de células endoteliais circulantes em portadores assintomáticos do vírus linfotrópico humano de células T do tipo 1 (HTLV1) por citometria de fluxo. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5167/tde-09062009-170516/ ;


University of Iowa

21. Vrieze, Katherine Elna. Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia.

Degree: PhD, Molecular and Cellular Biology, 2011, University of Iowa

T-cell acute lymphocytic leukemia (T-ALL) affects approximately 1,500 people per year in the United States, many of them children. The overall survival rate of… (more)

Subjects/Keywords: ETP-ALL; NOTCH1; T-ALL; T-cell Acute Lymphocytic Leukemia; Cell Biology

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APA (6th Edition):

Vrieze, K. E. (2011). Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2785

Chicago Manual of Style (16th Edition):

Vrieze, Katherine Elna. “Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia.” 2011. Doctoral Dissertation, University of Iowa. Accessed March 05, 2021. https://ir.uiowa.edu/etd/2785.

MLA Handbook (7th Edition):

Vrieze, Katherine Elna. “Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia.” 2011. Web. 05 Mar 2021.

Vancouver:

Vrieze KE. Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia. [Internet] [Doctoral dissertation]. University of Iowa; 2011. [cited 2021 Mar 05]. Available from: https://ir.uiowa.edu/etd/2785.

Council of Science Editors:

Vrieze KE. Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia. [Doctoral Dissertation]. University of Iowa; 2011. Available from: https://ir.uiowa.edu/etd/2785


University of Toronto

22. Suliman, Sara. Functions of Notch1, Notch3, and Fringe Glycosyltransferases during Thymocyte β-Selection and T-cell Leukemogenesis.

Degree: 2013, University of Toronto

T-cells play critical roles in the immune system, from fighting infection and tissue remodeling to anti-tumor immunity. Notch1 (N1) signaling is a conserved signaling pathway… (more)

Subjects/Keywords: Notch Signaling; T-Cell Development; Leukemia; Thymus; 0982

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APA (6th Edition):

Suliman, S. (2013). Functions of Notch1, Notch3, and Fringe Glycosyltransferases during Thymocyte β-Selection and T-cell Leukemogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68967

Chicago Manual of Style (16th Edition):

Suliman, Sara. “Functions of Notch1, Notch3, and Fringe Glycosyltransferases during Thymocyte β-Selection and T-cell Leukemogenesis.” 2013. Doctoral Dissertation, University of Toronto. Accessed March 05, 2021. http://hdl.handle.net/1807/68967.

MLA Handbook (7th Edition):

Suliman, Sara. “Functions of Notch1, Notch3, and Fringe Glycosyltransferases during Thymocyte β-Selection and T-cell Leukemogenesis.” 2013. Web. 05 Mar 2021.

Vancouver:

Suliman S. Functions of Notch1, Notch3, and Fringe Glycosyltransferases during Thymocyte β-Selection and T-cell Leukemogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/1807/68967.

Council of Science Editors:

Suliman S. Functions of Notch1, Notch3, and Fringe Glycosyltransferases during Thymocyte β-Selection and T-cell Leukemogenesis. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68967

23. 정, 현주. Effect of Tissue Transglutaminase on Steroid Resistance in T-Cell Acute Lymphoblastic Leukemia.

Degree: 2020, Ajou University

AIM: To improve survival in patients with glucocorticoid-resistant T-cell acute lymphoblastic leukemia (T-ALL), it is critical to develop new therapeutic strategies to overcome steroid resistance.… (more)

Subjects/Keywords: Tissue transglutaminase; steroid resistance; T-cell acute lymphoblastic leukemia

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APA (6th Edition):

정, . (2020). Effect of Tissue Transglutaminase on Steroid Resistance in T-Cell Acute Lymphoblastic Leukemia. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/19265 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000030197

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 현주. “Effect of Tissue Transglutaminase on Steroid Resistance in T-Cell Acute Lymphoblastic Leukemia.” 2020. Thesis, Ajou University. Accessed March 05, 2021. http://repository.ajou.ac.kr/handle/201003/19265 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000030197.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 현주. “Effect of Tissue Transglutaminase on Steroid Resistance in T-Cell Acute Lymphoblastic Leukemia.” 2020. Web. 05 Mar 2021.

Vancouver:

정 . Effect of Tissue Transglutaminase on Steroid Resistance in T-Cell Acute Lymphoblastic Leukemia. [Internet] [Thesis]. Ajou University; 2020. [cited 2021 Mar 05]. Available from: http://repository.ajou.ac.kr/handle/201003/19265 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000030197.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . Effect of Tissue Transglutaminase on Steroid Resistance in T-Cell Acute Lymphoblastic Leukemia. [Thesis]. Ajou University; 2020. Available from: http://repository.ajou.ac.kr/handle/201003/19265 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000030197

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

24. Phelan, James D., B.S. Transcriptional Control of Normal Lymphopoiesis and T-cell Neoplasia by Growth Factor Independent 1.

Degree: PhD, Medicine: Immunology, 2012, University of Cincinnati

 Hematopoietic development is a tightly controlled process in which environmental cues promote or restrict the proliferation and differentiation of bone marrow derived cells. These signals… (more)

Subjects/Keywords: Biology; Gfi1; Notch; Leukemia; Hematopoiesis; T cell; p53

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APA (6th Edition):

Phelan, James D., B. S. (2012). Transcriptional Control of Normal Lymphopoiesis and T-cell Neoplasia by Growth Factor Independent 1. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337351444

Chicago Manual of Style (16th Edition):

Phelan, James D., B S. “Transcriptional Control of Normal Lymphopoiesis and T-cell Neoplasia by Growth Factor Independent 1.” 2012. Doctoral Dissertation, University of Cincinnati. Accessed March 05, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337351444.

MLA Handbook (7th Edition):

Phelan, James D., B S. “Transcriptional Control of Normal Lymphopoiesis and T-cell Neoplasia by Growth Factor Independent 1.” 2012. Web. 05 Mar 2021.

Vancouver:

Phelan, James D. BS. Transcriptional Control of Normal Lymphopoiesis and T-cell Neoplasia by Growth Factor Independent 1. [Internet] [Doctoral dissertation]. University of Cincinnati; 2012. [cited 2021 Mar 05]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337351444.

Council of Science Editors:

Phelan, James D. BS. Transcriptional Control of Normal Lymphopoiesis and T-cell Neoplasia by Growth Factor Independent 1. [Doctoral Dissertation]. University of Cincinnati; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337351444


Johannes Gutenberg Universität Mainz

25. Albrecht, Jana. In vitro characterization of human AML-reactive CTL clones generated from the naive subset of healthy donors and adoptive transfer into leukemia-engrafted NSG mice.

Degree: 2012, Johannes Gutenberg Universität Mainz

Acute myeloid leukemia (AML) is a very aggressive cancer of the hematopoietic system. Chemotherapy and immunotherapeutical approaches including hematopoietic stem cell transplantation (HSCT) and donor… (more)

Subjects/Keywords: T-Zell-Therapie, Leukämie-reaktive CTL, Präklinisches Leukämie-Mausmodell; T-cell therapy, leukemia-reactive CTL, preclinical leukemia-mousemodel; Life sciences

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APA (6th Edition):

Albrecht, J. (2012). In vitro characterization of human AML-reactive CTL clones generated from the naive subset of healthy donors and adoptive transfer into leukemia-engrafted NSG mice. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2013/3368/

Chicago Manual of Style (16th Edition):

Albrecht, Jana. “In vitro characterization of human AML-reactive CTL clones generated from the naive subset of healthy donors and adoptive transfer into leukemia-engrafted NSG mice.” 2012. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 05, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2013/3368/.

MLA Handbook (7th Edition):

Albrecht, Jana. “In vitro characterization of human AML-reactive CTL clones generated from the naive subset of healthy donors and adoptive transfer into leukemia-engrafted NSG mice.” 2012. Web. 05 Mar 2021.

Vancouver:

Albrecht J. In vitro characterization of human AML-reactive CTL clones generated from the naive subset of healthy donors and adoptive transfer into leukemia-engrafted NSG mice. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2012. [cited 2021 Mar 05]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2013/3368/.

Council of Science Editors:

Albrecht J. In vitro characterization of human AML-reactive CTL clones generated from the naive subset of healthy donors and adoptive transfer into leukemia-engrafted NSG mice. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2012. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2013/3368/


Universitat Pompeu Fabra

26. Colomer-Lahiguera, Sara, 1981-. The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia.

Degree: Departament de Ciències Experimentals i de la Salut, 2015, Universitat Pompeu Fabra

 La leucemia linfoblástica aguda de células T (LLA-T) representa el 10 15% de las leucemias pediátricas. La transformación oncogénica de precursores de células T es… (more)

Subjects/Keywords: CDKN1B-p27kip1; T-ALL; Lymphoblastic; MEF2C; SKP2; T-cell; Leukemia; Pediatric; Leucemia; Pediátrica; 616

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APA (6th Edition):

Colomer-Lahiguera, Sara, 1. (2015). The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/523487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Colomer-Lahiguera, Sara, 1981-. “The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia.” 2015. Thesis, Universitat Pompeu Fabra. Accessed March 05, 2021. http://hdl.handle.net/10803/523487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Colomer-Lahiguera, Sara, 1981-. “The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia.” 2015. Web. 05 Mar 2021.

Vancouver:

Colomer-Lahiguera, Sara 1. The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia. [Internet] [Thesis]. Universitat Pompeu Fabra; 2015. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/10803/523487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Colomer-Lahiguera, Sara 1. The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia. [Thesis]. Universitat Pompeu Fabra; 2015. Available from: http://hdl.handle.net/10803/523487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Calvo, Julien. Etude de l'impact des adipocytes de la moelle osseuse sur le développement et la chimiorésistance des leucémies aigues lymphoblastiques T (LAL-T) : Study of the impact of bone marrow adipocytes on the development and chemoresistance of T cell actue lymphoblastic leukemia (T-ALL).

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université de Paris (2019-....)

 Les leucémies aiguës lymphoblastiques T (LAL-T) sont des maladies des progéniteurs T qui surviennent principalement chez l'enfant et le jeune adulte. Des facteurs intrinsèques et… (more)

Subjects/Keywords: Leucémies aiguës lymphoblastiques T; Microenvironnement; Adipocytes; Chimiorésistance; T cell acute leukemia; Microenvironment; Adipocytes; Chemoresistance

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APA (6th Edition):

Calvo, J. (2019). Etude de l'impact des adipocytes de la moelle osseuse sur le développement et la chimiorésistance des leucémies aigues lymphoblastiques T (LAL-T) : Study of the impact of bone marrow adipocytes on the development and chemoresistance of T cell actue lymphoblastic leukemia (T-ALL). (Doctoral Dissertation). Université de Paris (2019-....). Retrieved from http://www.theses.fr/2019UNIP7114

Chicago Manual of Style (16th Edition):

Calvo, Julien. “Etude de l'impact des adipocytes de la moelle osseuse sur le développement et la chimiorésistance des leucémies aigues lymphoblastiques T (LAL-T) : Study of the impact of bone marrow adipocytes on the development and chemoresistance of T cell actue lymphoblastic leukemia (T-ALL).” 2019. Doctoral Dissertation, Université de Paris (2019-....). Accessed March 05, 2021. http://www.theses.fr/2019UNIP7114.

MLA Handbook (7th Edition):

Calvo, Julien. “Etude de l'impact des adipocytes de la moelle osseuse sur le développement et la chimiorésistance des leucémies aigues lymphoblastiques T (LAL-T) : Study of the impact of bone marrow adipocytes on the development and chemoresistance of T cell actue lymphoblastic leukemia (T-ALL).” 2019. Web. 05 Mar 2021.

Vancouver:

Calvo J. Etude de l'impact des adipocytes de la moelle osseuse sur le développement et la chimiorésistance des leucémies aigues lymphoblastiques T (LAL-T) : Study of the impact of bone marrow adipocytes on the development and chemoresistance of T cell actue lymphoblastic leukemia (T-ALL). [Internet] [Doctoral dissertation]. Université de Paris (2019-....); 2019. [cited 2021 Mar 05]. Available from: http://www.theses.fr/2019UNIP7114.

Council of Science Editors:

Calvo J. Etude de l'impact des adipocytes de la moelle osseuse sur le développement et la chimiorésistance des leucémies aigues lymphoblastiques T (LAL-T) : Study of the impact of bone marrow adipocytes on the development and chemoresistance of T cell actue lymphoblastic leukemia (T-ALL). [Doctoral Dissertation]. Université de Paris (2019-....); 2019. Available from: http://www.theses.fr/2019UNIP7114


University of South Florida

28. Dubovsky, Jason A. Epigenetic Modifiers to Augment the Immunogenicity of Chronic Lymphocytic Leukemia.

Degree: 2013, University of South Florida

 Cancer cells employ a litany of immunosuppressive and immunevasive strategies to avoid detection and elimination by the various arms of the innate and adaptive immune… (more)

Subjects/Keywords: Epigenetic; Histone; Leukemia; Lymphocyte; Methylation; T cell; Cell Biology; Immunology and Infectious Disease; Molecular Biology

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APA (6th Edition):

Dubovsky, J. A. (2013). Epigenetic Modifiers to Augment the Immunogenicity of Chronic Lymphocytic Leukemia. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/4623

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dubovsky, Jason A. “Epigenetic Modifiers to Augment the Immunogenicity of Chronic Lymphocytic Leukemia.” 2013. Thesis, University of South Florida. Accessed March 05, 2021. https://scholarcommons.usf.edu/etd/4623.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dubovsky, Jason A. “Epigenetic Modifiers to Augment the Immunogenicity of Chronic Lymphocytic Leukemia.” 2013. Web. 05 Mar 2021.

Vancouver:

Dubovsky JA. Epigenetic Modifiers to Augment the Immunogenicity of Chronic Lymphocytic Leukemia. [Internet] [Thesis]. University of South Florida; 2013. [cited 2021 Mar 05]. Available from: https://scholarcommons.usf.edu/etd/4623.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dubovsky JA. Epigenetic Modifiers to Augment the Immunogenicity of Chronic Lymphocytic Leukemia. [Thesis]. University of South Florida; 2013. Available from: https://scholarcommons.usf.edu/etd/4623

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Helsinki

29. Ilander, Mette. NK-and T-cell function in chronic myeloid leukemia patients treated with interferon-α monotherapy: T-ja NK-solujen toiminta interferoni-α hoidetuilla kroonista myeloista leukemiaa sairastavilla potilailla.

Degree: Agrikultur- och forstvetenskapliga fakulteten, 2011, University of Helsinki

 Krooninen myeloinen leukemia (KML) aiheutuu hematopoieettisessa kantasolussa tapahtuvasta kromosomien 9 ja 22 välisestä translokaatiosta. Siinä syntyy Philadelphia kromosomi (Ph) ja itsenäisesti toimiva tyrosiinikinaasi BCR-ABL. BCR-ABL… (more)

Subjects/Keywords: KML; BCR-ABL; interferon-α; NK-cell; T-cell; NK-solu; T-solu; interferoni-α; chronic myeloid leukemia; Bioteknik; Biotechnology; Biotekniikka

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APA (6th Edition):

Ilander, M. (2011). NK-and T-cell function in chronic myeloid leukemia patients treated with interferon-α monotherapy: T-ja NK-solujen toiminta interferoni-α hoidetuilla kroonista myeloista leukemiaa sairastavilla potilailla. (Masters Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/28476

Chicago Manual of Style (16th Edition):

Ilander, Mette. “NK-and T-cell function in chronic myeloid leukemia patients treated with interferon-α monotherapy: T-ja NK-solujen toiminta interferoni-α hoidetuilla kroonista myeloista leukemiaa sairastavilla potilailla.” 2011. Masters Thesis, University of Helsinki. Accessed March 05, 2021. http://hdl.handle.net/10138/28476.

MLA Handbook (7th Edition):

Ilander, Mette. “NK-and T-cell function in chronic myeloid leukemia patients treated with interferon-α monotherapy: T-ja NK-solujen toiminta interferoni-α hoidetuilla kroonista myeloista leukemiaa sairastavilla potilailla.” 2011. Web. 05 Mar 2021.

Vancouver:

Ilander M. NK-and T-cell function in chronic myeloid leukemia patients treated with interferon-α monotherapy: T-ja NK-solujen toiminta interferoni-α hoidetuilla kroonista myeloista leukemiaa sairastavilla potilailla. [Internet] [Masters thesis]. University of Helsinki; 2011. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/10138/28476.

Council of Science Editors:

Ilander M. NK-and T-cell function in chronic myeloid leukemia patients treated with interferon-α monotherapy: T-ja NK-solujen toiminta interferoni-α hoidetuilla kroonista myeloista leukemiaa sairastavilla potilailla. [Masters Thesis]. University of Helsinki; 2011. Available from: http://hdl.handle.net/10138/28476


University of Toronto

30. Matei, Irina. The Role of ATM in Promoting Normal T cell Development and Preventing T Cell Leukemogenesis.

Degree: 2009, University of Toronto

The immune system recognizes and eliminates an enormous array of pathogens due to the diverse antigen receptor repertoire of T and B lymphocytes. However, the… (more)

Subjects/Keywords: ATM; T cell development; T cell leukemia; 0379

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APA (6th Edition):

Matei, I. (2009). The Role of ATM in Promoting Normal T cell Development and Preventing T Cell Leukemogenesis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/17799

Chicago Manual of Style (16th Edition):

Matei, Irina. “The Role of ATM in Promoting Normal T cell Development and Preventing T Cell Leukemogenesis.” 2009. Doctoral Dissertation, University of Toronto. Accessed March 05, 2021. http://hdl.handle.net/1807/17799.

MLA Handbook (7th Edition):

Matei, Irina. “The Role of ATM in Promoting Normal T cell Development and Preventing T Cell Leukemogenesis.” 2009. Web. 05 Mar 2021.

Vancouver:

Matei I. The Role of ATM in Promoting Normal T cell Development and Preventing T Cell Leukemogenesis. [Internet] [Doctoral dissertation]. University of Toronto; 2009. [cited 2021 Mar 05]. Available from: http://hdl.handle.net/1807/17799.

Council of Science Editors:

Matei I. The Role of ATM in Promoting Normal T cell Development and Preventing T Cell Leukemogenesis. [Doctoral Dissertation]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/17799

[1] [2] [3] [4] [5] … [1656]

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