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You searched for subject:(Acute lung injury). Showing records 1 – 30 of 97 total matches.

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1. 坪内, 拡伸. Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner : 六君子湯投与はブレオマイシン誘導性急性肺障害をグレリン非依存性に改善する.

Degree: 博士(医学), 2014, University of Miyazaki / 宮崎大学

以下に掲載:American Journal of Physiology - Lung Cellular and Molecular Physiology. 2014, 306, 3, p.L233-L245, doi:10.1152/ajplung.00096.2013.

Subjects/Keywords: rikkunshito; acute lung injury; ghrelin

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APA (6th Edition):

坪内, . (2014). Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner : 六君子湯投与はブレオマイシン誘導性急性肺障害をグレリン非依存性に改善する. (Thesis). University of Miyazaki / 宮崎大学. Retrieved from http://hdl.handle.net/10458/5039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

坪内, 拡伸. “Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner : 六君子湯投与はブレオマイシン誘導性急性肺障害をグレリン非依存性に改善する.” 2014. Thesis, University of Miyazaki / 宮崎大学. Accessed October 21, 2019. http://hdl.handle.net/10458/5039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

坪内, 拡伸. “Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner : 六君子湯投与はブレオマイシン誘導性急性肺障害をグレリン非依存性に改善する.” 2014. Web. 21 Oct 2019.

Vancouver:

坪内 . Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner : 六君子湯投与はブレオマイシン誘導性急性肺障害をグレリン非依存性に改善する. [Internet] [Thesis]. University of Miyazaki / 宮崎大学; 2014. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10458/5039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

坪内 . Rikkunshito ameliorates bleomycin-induced acute lung injury in a ghrelin-independent manner : 六君子湯投与はブレオマイシン誘導性急性肺障害をグレリン非依存性に改善する. [Thesis]. University of Miyazaki / 宮崎大学; 2014. Available from: http://hdl.handle.net/10458/5039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

2. Dhaliwal, Kanwaldeep. Developing novel therapeutic strategies for acute lung injury and infection-peripheral blood monocyte depletion and prophylactic antimicrobial therapy.

Degree: PhD, 2013, University of Edinburgh

 Background: Acute lung injury (ALI) and nosocomial pneumonia are major causes of morbidity and mortality. There are 200,000 cases per year of ALI in the… (more)

Subjects/Keywords: 616.2; Acute lung injury; neutrophils; monocytes; peptoids

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APA (6th Edition):

Dhaliwal, K. (2013). Developing novel therapeutic strategies for acute lung injury and infection-peripheral blood monocyte depletion and prophylactic antimicrobial therapy. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8100

Chicago Manual of Style (16th Edition):

Dhaliwal, Kanwaldeep. “Developing novel therapeutic strategies for acute lung injury and infection-peripheral blood monocyte depletion and prophylactic antimicrobial therapy.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed October 21, 2019. http://hdl.handle.net/1842/8100.

MLA Handbook (7th Edition):

Dhaliwal, Kanwaldeep. “Developing novel therapeutic strategies for acute lung injury and infection-peripheral blood monocyte depletion and prophylactic antimicrobial therapy.” 2013. Web. 21 Oct 2019.

Vancouver:

Dhaliwal K. Developing novel therapeutic strategies for acute lung injury and infection-peripheral blood monocyte depletion and prophylactic antimicrobial therapy. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1842/8100.

Council of Science Editors:

Dhaliwal K. Developing novel therapeutic strategies for acute lung injury and infection-peripheral blood monocyte depletion and prophylactic antimicrobial therapy. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/8100


University of Alberta

3. Ionescu, Lavinia Iuliana. Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases.

Degree: PhD, Department of Physiology, 2012, University of Alberta

 Recent discoveries in stem cell biology have generated enthusiasm about the possibility of harnessing stem cells for organ repair and regeneration. The ability of pluri-… (more)

Subjects/Keywords: asthma; mesenchymal stem cells; acute lung injury; lung; lipopolysaccharide; ovalbumin

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APA (6th Edition):

Ionescu, L. I. (2012). Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/kh04dq54w

Chicago Manual of Style (16th Edition):

Ionescu, Lavinia Iuliana. “Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases.” 2012. Doctoral Dissertation, University of Alberta. Accessed October 21, 2019. https://era.library.ualberta.ca/files/kh04dq54w.

MLA Handbook (7th Edition):

Ionescu, Lavinia Iuliana. “Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases.” 2012. Web. 21 Oct 2019.

Vancouver:

Ionescu LI. Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2019 Oct 21]. Available from: https://era.library.ualberta.ca/files/kh04dq54w.

Council of Science Editors:

Ionescu LI. Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/kh04dq54w


University of Manchester

4. Gieschen-Krische, Mary. The role of NKT cells following solid organ transplantation.

Degree: PhD, 2014, University of Manchester

 Introduction: NKT cells are categorised as borderline between NK and T cells, sharing phenotypic and functional characteristics of both cells, demonstrating their capacity to contritube… (more)

Subjects/Keywords: acute lung injury; immunosuppression; NKT cells; lung; transplantation

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APA (6th Edition):

Gieschen-Krische, M. (2014). The role of NKT cells following solid organ transplantation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287

Chicago Manual of Style (16th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Doctoral Dissertation, University of Manchester. Accessed October 21, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287.

MLA Handbook (7th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Web. 21 Oct 2019.

Vancouver:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Oct 21]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287.

Council of Science Editors:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287


University of Manchester

5. Gieschen-Krische, Mary. The role of NKT cells following solid organ transplantation.

Degree: 2014, University of Manchester

 Introduction: NKT cells are categorised as borderline between NK and T cells, sharing phenotypic and functional characteristics of both cells, demonstrating their capacity to contritube… (more)

Subjects/Keywords: NKT cells; lung; transplantation; immunosuppression; acute lung injury

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APA (6th Edition):

Gieschen-Krische, M. (2014). The role of NKT cells following solid organ transplantation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866

Chicago Manual of Style (16th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Doctoral Dissertation, University of Manchester. Accessed October 21, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866.

MLA Handbook (7th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Web. 21 Oct 2019.

Vancouver:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Oct 21]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866.

Council of Science Editors:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866


University of Miami

6. Kerr, Nadine. Traumatic Brain Injury-Induced Acute Lung Injury: Evidence of Activation and Inhibition of a Neural-Respiratory Inflammasome Axis.

Degree: PhD, Neuroscience (Medicine), 2019, University of Miami

 Approximately 20-25 percent of Traumatic Brain Injury (TBI) subjects develop Acute Lung Injury (ALI), but the pathomechanisms of TBI-induced ALI remain poorly defined. In my… (more)

Subjects/Keywords: Traumatic Brain Injury; Inflammasome; Extracellular Vesicles; Neuroinflammation; Acute Lung Injury; Pyroptosis

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APA (6th Edition):

Kerr, N. (2019). Traumatic Brain Injury-Induced Acute Lung Injury: Evidence of Activation and Inhibition of a Neural-Respiratory Inflammasome Axis. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/2242

Chicago Manual of Style (16th Edition):

Kerr, Nadine. “Traumatic Brain Injury-Induced Acute Lung Injury: Evidence of Activation and Inhibition of a Neural-Respiratory Inflammasome Axis.” 2019. Doctoral Dissertation, University of Miami. Accessed October 21, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/2242.

MLA Handbook (7th Edition):

Kerr, Nadine. “Traumatic Brain Injury-Induced Acute Lung Injury: Evidence of Activation and Inhibition of a Neural-Respiratory Inflammasome Axis.” 2019. Web. 21 Oct 2019.

Vancouver:

Kerr N. Traumatic Brain Injury-Induced Acute Lung Injury: Evidence of Activation and Inhibition of a Neural-Respiratory Inflammasome Axis. [Internet] [Doctoral dissertation]. University of Miami; 2019. [cited 2019 Oct 21]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2242.

Council of Science Editors:

Kerr N. Traumatic Brain Injury-Induced Acute Lung Injury: Evidence of Activation and Inhibition of a Neural-Respiratory Inflammasome Axis. [Doctoral Dissertation]. University of Miami; 2019. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/2242


University of Toronto

7. Gao, Wenxi. The Effect of Alpha 1-Antitrypsin on Ischemia-Reperfusion Injury in Lung Transplantation.

Degree: 2012, University of Toronto

Ischemia-reperfusion (IR) injury is a severe complication in lung transplantation characterized by inflammation, alveolar damage, and hypoxemia. Alpha 1-antitrypsin (A1AT), a protease inhibitor, is currently… (more)

Subjects/Keywords: alpha 1-antitrypsin; lung transplantation; ischemia-reperfusion injury; acute lung injury; 0719

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APA (6th Edition):

Gao, W. (2012). The Effect of Alpha 1-Antitrypsin on Ischemia-Reperfusion Injury in Lung Transplantation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33210

Chicago Manual of Style (16th Edition):

Gao, Wenxi. “The Effect of Alpha 1-Antitrypsin on Ischemia-Reperfusion Injury in Lung Transplantation.” 2012. Masters Thesis, University of Toronto. Accessed October 21, 2019. http://hdl.handle.net/1807/33210.

MLA Handbook (7th Edition):

Gao, Wenxi. “The Effect of Alpha 1-Antitrypsin on Ischemia-Reperfusion Injury in Lung Transplantation.” 2012. Web. 21 Oct 2019.

Vancouver:

Gao W. The Effect of Alpha 1-Antitrypsin on Ischemia-Reperfusion Injury in Lung Transplantation. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1807/33210.

Council of Science Editors:

Gao W. The Effect of Alpha 1-Antitrypsin on Ischemia-Reperfusion Injury in Lung Transplantation. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33210


The Ohio State University

8. Chang, Christopher J. Using MicroRNAs 146a and 155 to Mitigate Barotrauma and Atelectrauma in Simulated Ventilator-Induced Lung Injury.

Degree: MS, Biomedical Engineering, 2018, The Ohio State University

Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are lung disorders characterized by increased permeability of the alveolar barrier, resulting in fluid buildup… (more)

Subjects/Keywords: Biomedical Engineering; lung injury; acute lung injury; acute respiratory distress syndrome; ventilator-induced lung injury; microRNA; miR-155; miR-146a; atelectrauma; barotrauma; extracellular vesicle; lung epithelial cell; alveolar macrophage

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APA (6th Edition):

Chang, C. J. (2018). Using MicroRNAs 146a and 155 to Mitigate Barotrauma and Atelectrauma in Simulated Ventilator-Induced Lung Injury. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1524188199388787

Chicago Manual of Style (16th Edition):

Chang, Christopher J. “Using MicroRNAs 146a and 155 to Mitigate Barotrauma and Atelectrauma in Simulated Ventilator-Induced Lung Injury.” 2018. Masters Thesis, The Ohio State University. Accessed October 21, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1524188199388787.

MLA Handbook (7th Edition):

Chang, Christopher J. “Using MicroRNAs 146a and 155 to Mitigate Barotrauma and Atelectrauma in Simulated Ventilator-Induced Lung Injury.” 2018. Web. 21 Oct 2019.

Vancouver:

Chang CJ. Using MicroRNAs 146a and 155 to Mitigate Barotrauma and Atelectrauma in Simulated Ventilator-Induced Lung Injury. [Internet] [Masters thesis]. The Ohio State University; 2018. [cited 2019 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1524188199388787.

Council of Science Editors:

Chang CJ. Using MicroRNAs 146a and 155 to Mitigate Barotrauma and Atelectrauma in Simulated Ventilator-Induced Lung Injury. [Masters Thesis]. The Ohio State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1524188199388787


University of Rochester

9. Graves, Brian T. Multilevel Modeling of ARDS Mortality Predictors.

Degree: PhD, 2012, University of Rochester

 Background The Acute Respiratory Distress Syndrome (ARDS) is associated with acute and persistent lung inflammation. Incidence and mortality is high, extensive complex critical care is… (more)

Subjects/Keywords: ARDS; ALI; Acute Respiratory Distress Syndrome; Acute Lung Injury; Hierarchical Generalized Linear Modeling; HGLM

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APA (6th Edition):

Graves, B. T. (2012). Multilevel Modeling of ARDS Mortality Predictors. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/21350

Chicago Manual of Style (16th Edition):

Graves, Brian T. “Multilevel Modeling of ARDS Mortality Predictors.” 2012. Doctoral Dissertation, University of Rochester. Accessed October 21, 2019. http://hdl.handle.net/1802/21350.

MLA Handbook (7th Edition):

Graves, Brian T. “Multilevel Modeling of ARDS Mortality Predictors.” 2012. Web. 21 Oct 2019.

Vancouver:

Graves BT. Multilevel Modeling of ARDS Mortality Predictors. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1802/21350.

Council of Science Editors:

Graves BT. Multilevel Modeling of ARDS Mortality Predictors. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/21350


The Ohio State University

10. Lai, Ju-Ping. Phosphatase and tensin homolog deleted on chromosome Ten (PTEN) as a molecular target in lung epithelial wound repair and protection.

Degree: PhD, Pharmacy, 2008, The Ohio State University

 The long-term goal of this study is to identify a potential innovative therapeutic target to prevent or treat Acute Respiratory Distress Syndrome (ARDS), a condition… (more)

Subjects/Keywords: Pharmaceuticals; Pharmacology; PTEN inhibition; acute lung injury; wound repair; protection; lung epithelium

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APA (6th Edition):

Lai, J. (2008). Phosphatase and tensin homolog deleted on chromosome Ten (PTEN) as a molecular target in lung epithelial wound repair and protection. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1206120012

Chicago Manual of Style (16th Edition):

Lai, Ju-Ping. “Phosphatase and tensin homolog deleted on chromosome Ten (PTEN) as a molecular target in lung epithelial wound repair and protection.” 2008. Doctoral Dissertation, The Ohio State University. Accessed October 21, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1206120012.

MLA Handbook (7th Edition):

Lai, Ju-Ping. “Phosphatase and tensin homolog deleted on chromosome Ten (PTEN) as a molecular target in lung epithelial wound repair and protection.” 2008. Web. 21 Oct 2019.

Vancouver:

Lai J. Phosphatase and tensin homolog deleted on chromosome Ten (PTEN) as a molecular target in lung epithelial wound repair and protection. [Internet] [Doctoral dissertation]. The Ohio State University; 2008. [cited 2019 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1206120012.

Council of Science Editors:

Lai J. Phosphatase and tensin homolog deleted on chromosome Ten (PTEN) as a molecular target in lung epithelial wound repair and protection. [Doctoral Dissertation]. The Ohio State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1206120012


Dalhousie University

11. Ismaiel, Nada. Protective Ventilation vs. Hypercapnia for the Attenuation of Ventilator-Associated Lung Injury.

Degree: MS, Department of Physiology & Biophysics, 2011, Dalhousie University

 Mechanically ventilated patients are at risk of developing Ventilator-Associated Lung Injury (VALI). Improved ventilation strategies by lung-protective settings may cause hypercapnia. This study investigated whether… (more)

Subjects/Keywords: Acute Lung Injury; Mechanical Ventilation; Hypercapnia; Inflammation; Carbon Dioxide

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APA (6th Edition):

Ismaiel, N. (2011). Protective Ventilation vs. Hypercapnia for the Attenuation of Ventilator-Associated Lung Injury. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14227

Chicago Manual of Style (16th Edition):

Ismaiel, Nada. “Protective Ventilation vs. Hypercapnia for the Attenuation of Ventilator-Associated Lung Injury.” 2011. Masters Thesis, Dalhousie University. Accessed October 21, 2019. http://hdl.handle.net/10222/14227.

MLA Handbook (7th Edition):

Ismaiel, Nada. “Protective Ventilation vs. Hypercapnia for the Attenuation of Ventilator-Associated Lung Injury.” 2011. Web. 21 Oct 2019.

Vancouver:

Ismaiel N. Protective Ventilation vs. Hypercapnia for the Attenuation of Ventilator-Associated Lung Injury. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10222/14227.

Council of Science Editors:

Ismaiel N. Protective Ventilation vs. Hypercapnia for the Attenuation of Ventilator-Associated Lung Injury. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14227


Carnegie Mellon University

12. Nelson, Diane L. Pulmonary Drug Delivery via Reverse Perfluorocarbon Emulsions: A Novel Method for Bacterial Respiratory Infections and Acute Respiratory Failure.

Degree: 2018, Carnegie Mellon University

 Inhaled drug delivery is currently the gold standard for treating many respiratory diseases. However, improved treatments are needed for lung diseases like Cystic Fibrosis (CF)… (more)

Subjects/Keywords: acute lung injury; cystic fibrosis; drug delivery; fluorosurfactant; liquid ventilation; perfluorocarbon

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APA (6th Edition):

Nelson, D. L. (2018). Pulmonary Drug Delivery via Reverse Perfluorocarbon Emulsions: A Novel Method for Bacterial Respiratory Infections and Acute Respiratory Failure. (Thesis). Carnegie Mellon University. Retrieved from http://repository.cmu.edu/dissertations/1147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Diane L. “Pulmonary Drug Delivery via Reverse Perfluorocarbon Emulsions: A Novel Method for Bacterial Respiratory Infections and Acute Respiratory Failure.” 2018. Thesis, Carnegie Mellon University. Accessed October 21, 2019. http://repository.cmu.edu/dissertations/1147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Diane L. “Pulmonary Drug Delivery via Reverse Perfluorocarbon Emulsions: A Novel Method for Bacterial Respiratory Infections and Acute Respiratory Failure.” 2018. Web. 21 Oct 2019.

Vancouver:

Nelson DL. Pulmonary Drug Delivery via Reverse Perfluorocarbon Emulsions: A Novel Method for Bacterial Respiratory Infections and Acute Respiratory Failure. [Internet] [Thesis]. Carnegie Mellon University; 2018. [cited 2019 Oct 21]. Available from: http://repository.cmu.edu/dissertations/1147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson DL. Pulmonary Drug Delivery via Reverse Perfluorocarbon Emulsions: A Novel Method for Bacterial Respiratory Infections and Acute Respiratory Failure. [Thesis]. Carnegie Mellon University; 2018. Available from: http://repository.cmu.edu/dissertations/1147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

13. Musah, Sadiatu. Repair of the airway epithelium after chlorine-induced injury.

Degree: PhD, 2013, University of Louisville

  Chlorine is a widely used toxic chemical that is considered a chemical threat agent. Chlorine inhalation injures airway epithelium, and efficient epithelial repair is… (more)

Subjects/Keywords: Chlorine; Nerve growth factor; Repair; Beta-catenin; Acute lung injury

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APA (6th Edition):

Musah, S. (2013). Repair of the airway epithelium after chlorine-induced injury. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031

Chicago Manual of Style (16th Edition):

Musah, Sadiatu. “Repair of the airway epithelium after chlorine-induced injury.” 2013. Doctoral Dissertation, University of Louisville. Accessed October 21, 2019. 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031.

MLA Handbook (7th Edition):

Musah, Sadiatu. “Repair of the airway epithelium after chlorine-induced injury.” 2013. Web. 21 Oct 2019.

Vancouver:

Musah S. Repair of the airway epithelium after chlorine-induced injury. [Internet] [Doctoral dissertation]. University of Louisville; 2013. [cited 2019 Oct 21]. Available from: 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031.

Council of Science Editors:

Musah S. Repair of the airway epithelium after chlorine-induced injury. [Doctoral Dissertation]. University of Louisville; 2013. Available from: 10.18297/etd/1031 ; https://ir.library.louisville.edu/etd/1031

14. Ζέρβα, Αγάθη. Η επίδραση της χορήγησης του σεβοφλουρανίου στην οξεία πνευμονική βλάβη.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Objective: Sevoflurane exerts effects on pulmonary cells that could protectagainst lung injury. We evaluated the potential of pretreatment withsevoflurane to attenuate lipopolysaccharide (LPS)-induced lung injury.Design:… (more)

Subjects/Keywords: Οξεία πνευμονική βλάβη; Προγύμναση; Σεβοφλουράνιο; Acute lung injury; Pre-treatment; Sevoflurane

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APA (6th Edition):

Ζέρβα, . . (2013). Η επίδραση της χορήγησης του σεβοφλουρανίου στην οξεία πνευμονική βλάβη. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41121

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ζέρβα, Αγάθη. “Η επίδραση της χορήγησης του σεβοφλουρανίου στην οξεία πνευμονική βλάβη.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed October 21, 2019. http://hdl.handle.net/10442/hedi/41121.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ζέρβα, Αγάθη. “Η επίδραση της χορήγησης του σεβοφλουρανίου στην οξεία πνευμονική βλάβη.” 2013. Web. 21 Oct 2019.

Vancouver:

Ζέρβα . Η επίδραση της χορήγησης του σεβοφλουρανίου στην οξεία πνευμονική βλάβη. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10442/hedi/41121.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ζέρβα . Η επίδραση της χορήγησης του σεβοφλουρανίου στην οξεία πνευμονική βλάβη. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/41121

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Oklahoma State University

15. Guo, Yujie. Developmental Signaling and Acute Lung Injury.

Degree: Veterinary Biomedical Sciences, 2014, Oklahoma State University

 The present study is initiated to explore the potential implication of developmental signaling in AEC I injury, acute inflammatory response, and host defense against pathogen… (more)

Subjects/Keywords: acute lung injury; cell death; host defense; inflammation; wnt/beta-catenin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guo, Y. (2014). Developmental Signaling and Acute Lung Injury. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/14862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Yujie. “Developmental Signaling and Acute Lung Injury.” 2014. Thesis, Oklahoma State University. Accessed October 21, 2019. http://hdl.handle.net/11244/14862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Yujie. “Developmental Signaling and Acute Lung Injury.” 2014. Web. 21 Oct 2019.

Vancouver:

Guo Y. Developmental Signaling and Acute Lung Injury. [Internet] [Thesis]. Oklahoma State University; 2014. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/11244/14862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo Y. Developmental Signaling and Acute Lung Injury. [Thesis]. Oklahoma State University; 2014. Available from: http://hdl.handle.net/11244/14862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

16. Silberberg, Amanda. Evaluating the Impact of Novel Bone Marrow Cell-secreted Factors, C19orf10 and C1orf54, on Immunomodulation, Lung Cell Proliferation, and Resistance to Injury.

Degree: 2017, University of Toronto

Bone marrow-derived mesenchymal stem cells possess significant immunomodulatory and tissue-reparative capacity, and have been shown to promote recovery from lung injury, predominantly through their secretion… (more)

Subjects/Keywords: Acute lung injury; Bone marrow; Paracrine factors; 0379

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APA (6th Edition):

Silberberg, A. (2017). Evaluating the Impact of Novel Bone Marrow Cell-secreted Factors, C19orf10 and C1orf54, on Immunomodulation, Lung Cell Proliferation, and Resistance to Injury. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/76668

Chicago Manual of Style (16th Edition):

Silberberg, Amanda. “Evaluating the Impact of Novel Bone Marrow Cell-secreted Factors, C19orf10 and C1orf54, on Immunomodulation, Lung Cell Proliferation, and Resistance to Injury.” 2017. Masters Thesis, University of Toronto. Accessed October 21, 2019. http://hdl.handle.net/1807/76668.

MLA Handbook (7th Edition):

Silberberg, Amanda. “Evaluating the Impact of Novel Bone Marrow Cell-secreted Factors, C19orf10 and C1orf54, on Immunomodulation, Lung Cell Proliferation, and Resistance to Injury.” 2017. Web. 21 Oct 2019.

Vancouver:

Silberberg A. Evaluating the Impact of Novel Bone Marrow Cell-secreted Factors, C19orf10 and C1orf54, on Immunomodulation, Lung Cell Proliferation, and Resistance to Injury. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1807/76668.

Council of Science Editors:

Silberberg A. Evaluating the Impact of Novel Bone Marrow Cell-secreted Factors, C19orf10 and C1orf54, on Immunomodulation, Lung Cell Proliferation, and Resistance to Injury. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/76668


University of Vermont

17. Christopher, Massa. Modeling Recruitment/Derecruitment.

Degree: MS, Biomedical Engineering, 2008, University of Vermont

 Recruitment and derecruitment (R/D) of airways is known to significantly influence mechanical properties of the respiratory system during artificial ventilation, particularly in states of lung(more)

Subjects/Keywords: acute lung injury; mathematical modeling

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APA (6th Edition):

Christopher, M. (2008). Modeling Recruitment/Derecruitment. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/47

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Christopher, Massa. “Modeling Recruitment/Derecruitment.” 2008. Thesis, University of Vermont. Accessed October 21, 2019. https://scholarworks.uvm.edu/graddis/47.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Christopher, Massa. “Modeling Recruitment/Derecruitment.” 2008. Web. 21 Oct 2019.

Vancouver:

Christopher M. Modeling Recruitment/Derecruitment. [Internet] [Thesis]. University of Vermont; 2008. [cited 2019 Oct 21]. Available from: https://scholarworks.uvm.edu/graddis/47.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Christopher M. Modeling Recruitment/Derecruitment. [Thesis]. University of Vermont; 2008. Available from: https://scholarworks.uvm.edu/graddis/47

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

18. Puntorieri, Valeria. Surfactant and Matrix Metalloproteinase 3 in the Pathogenesis of Acute Lung Injury.

Degree: 2015, University of Western Ontario

Acute lung injury (ALI) is a pulmonary inflammatory disorder resulting in respiratory failure that is initiated by a number of different insults to the lung.… (more)

Subjects/Keywords: Acute Lung Injury; lung inflammation; cytokines; chemokines; mechanical ventilation; lung surfactant; exogenous surfactant; matrix metalloproteinase-3; lipopolysaccharide; acid-induced lung injury.; Circulatory and Respiratory Physiology

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APA (6th Edition):

Puntorieri, V. (2015). Surfactant and Matrix Metalloproteinase 3 in the Pathogenesis of Acute Lung Injury. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Puntorieri, Valeria. “Surfactant and Matrix Metalloproteinase 3 in the Pathogenesis of Acute Lung Injury.” 2015. Thesis, University of Western Ontario. Accessed October 21, 2019. https://ir.lib.uwo.ca/etd/2661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Puntorieri, Valeria. “Surfactant and Matrix Metalloproteinase 3 in the Pathogenesis of Acute Lung Injury.” 2015. Web. 21 Oct 2019.

Vancouver:

Puntorieri V. Surfactant and Matrix Metalloproteinase 3 in the Pathogenesis of Acute Lung Injury. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2019 Oct 21]. Available from: https://ir.lib.uwo.ca/etd/2661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Puntorieri V. Surfactant and Matrix Metalloproteinase 3 in the Pathogenesis of Acute Lung Injury. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/2661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Gonçalves, Cintia Tokio Reis. Efeitos do exercício físico aeróbico na lesão pulmonar aguda induzida por lipopolissacarídeo em camundongos.

Degree: PhD, Patologia, 2012, University of São Paulo

Introdução: A prática regular de exercício tem sido grandemente associada a efeitos benéficos em doenças pulmonares crônicas como asma e doença pulmonar obstrutiva crônica. Poucos… (more)

Subjects/Keywords: Acute lung injury; Aerobic exercise; Exercício; Exercício aeróbico; Exercise; Lesão pulmonar aguda; Lipopolissacarídeos; Lipopolysaccharides

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APA (6th Edition):

Gonçalves, C. T. R. (2012). Efeitos do exercício físico aeróbico na lesão pulmonar aguda induzida por lipopolissacarídeo em camundongos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-18022013-111359/ ;

Chicago Manual of Style (16th Edition):

Gonçalves, Cintia Tokio Reis. “Efeitos do exercício físico aeróbico na lesão pulmonar aguda induzida por lipopolissacarídeo em camundongos.” 2012. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-18022013-111359/ ;.

MLA Handbook (7th Edition):

Gonçalves, Cintia Tokio Reis. “Efeitos do exercício físico aeróbico na lesão pulmonar aguda induzida por lipopolissacarídeo em camundongos.” 2012. Web. 21 Oct 2019.

Vancouver:

Gonçalves CTR. Efeitos do exercício físico aeróbico na lesão pulmonar aguda induzida por lipopolissacarídeo em camundongos. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-18022013-111359/ ;.

Council of Science Editors:

Gonçalves CTR. Efeitos do exercício físico aeróbico na lesão pulmonar aguda induzida por lipopolissacarídeo em camundongos. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-18022013-111359/ ;


Universidade do Estado do Rio de Janeiro

20. Eduardo Tavares Lima Trajano. Participação do estresse oxidativo na lesão pulmonar induzida por lipopolissacarídeo: repercussões inflamatórias estruturais e funcionais.

Degree: Master, 2011, Universidade do Estado do Rio de Janeiro

O objetivo do presente estudo foi investigar o envolvimento do estresse oxidativo na lesão pulmonar aguda (LPA) induzida por lipopolissacarídeo (LPS) e as repercussões inflamatórias,… (more)

Subjects/Keywords: Lipopolissacarídeo; Lesão pulmonar aguda; Estresse oxidativo; Lipopolysaccharide. Acute lung injury. Oxidative stress; MORFOLOGIA

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APA (6th Edition):

Trajano, E. T. L. (2011). Participação do estresse oxidativo na lesão pulmonar induzida por lipopolissacarídeo: repercussões inflamatórias estruturais e funcionais. (Masters Thesis). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3466 ;

Chicago Manual of Style (16th Edition):

Trajano, Eduardo Tavares Lima. “Participação do estresse oxidativo na lesão pulmonar induzida por lipopolissacarídeo: repercussões inflamatórias estruturais e funcionais.” 2011. Masters Thesis, Universidade do Estado do Rio de Janeiro. Accessed October 21, 2019. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3466 ;.

MLA Handbook (7th Edition):

Trajano, Eduardo Tavares Lima. “Participação do estresse oxidativo na lesão pulmonar induzida por lipopolissacarídeo: repercussões inflamatórias estruturais e funcionais.” 2011. Web. 21 Oct 2019.

Vancouver:

Trajano ETL. Participação do estresse oxidativo na lesão pulmonar induzida por lipopolissacarídeo: repercussões inflamatórias estruturais e funcionais. [Internet] [Masters thesis]. Universidade do Estado do Rio de Janeiro; 2011. [cited 2019 Oct 21]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3466 ;.

Council of Science Editors:

Trajano ETL. Participação do estresse oxidativo na lesão pulmonar induzida por lipopolissacarídeo: repercussões inflamatórias estruturais e funcionais. [Masters Thesis]. Universidade do Estado do Rio de Janeiro; 2011. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3466 ;

21. Barry, Katherine C. Pharmacokinetic Modeling of 99mTc-HMPAO Images of Isolated Perfused Rat Lungs.

Degree: 2018, Marquette University

 The single-photon emission tomography (SPECT) imaging biomarker technetium-labeled hexamethylpropyleneamine oxime (99mTc-HMPAO) exists in two forms, the oxidized, cell-permeable form and the reduced, cell-impermeable form. Recent… (more)

Subjects/Keywords: Acute Lung Injury; Pharmacokinetic modeling; SPECT; Biological Engineering; Biomedical Engineering and Bioengineering

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APA (6th Edition):

Barry, K. C. (2018). Pharmacokinetic Modeling of 99mTc-HMPAO Images of Isolated Perfused Rat Lungs. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/theses_open/456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barry, Katherine C. “Pharmacokinetic Modeling of 99mTc-HMPAO Images of Isolated Perfused Rat Lungs.” 2018. Thesis, Marquette University. Accessed October 21, 2019. https://epublications.marquette.edu/theses_open/456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barry, Katherine C. “Pharmacokinetic Modeling of 99mTc-HMPAO Images of Isolated Perfused Rat Lungs.” 2018. Web. 21 Oct 2019.

Vancouver:

Barry KC. Pharmacokinetic Modeling of 99mTc-HMPAO Images of Isolated Perfused Rat Lungs. [Internet] [Thesis]. Marquette University; 2018. [cited 2019 Oct 21]. Available from: https://epublications.marquette.edu/theses_open/456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barry KC. Pharmacokinetic Modeling of 99mTc-HMPAO Images of Isolated Perfused Rat Lungs. [Thesis]. Marquette University; 2018. Available from: https://epublications.marquette.edu/theses_open/456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

22. Jackson, Isabel Lauren. Mechanisms Regulating Pulmonary Sensitivity to Radiation .

Degree: 2012, Duke University

  At the present time, here is no approved medical countermeasure (MCM) for mitigating or treating pneumonitis/fibrosis following acute radiation exposure. Since it is neither… (more)

Subjects/Keywords: Pathology; Medicine; acute radiation sickness; hypoxia; lung injury; oxidative stress; radiation biology

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APA (6th Edition):

Jackson, I. L. (2012). Mechanisms Regulating Pulmonary Sensitivity to Radiation . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5471

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jackson, Isabel Lauren. “Mechanisms Regulating Pulmonary Sensitivity to Radiation .” 2012. Thesis, Duke University. Accessed October 21, 2019. http://hdl.handle.net/10161/5471.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jackson, Isabel Lauren. “Mechanisms Regulating Pulmonary Sensitivity to Radiation .” 2012. Web. 21 Oct 2019.

Vancouver:

Jackson IL. Mechanisms Regulating Pulmonary Sensitivity to Radiation . [Internet] [Thesis]. Duke University; 2012. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10161/5471.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jackson IL. Mechanisms Regulating Pulmonary Sensitivity to Radiation . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5471

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

23. Pacheco, Shaun. Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury.

Degree: 2014, University of Toronto

Acute lung injury is a morbid complication with numerous causes and currently no effective clinical treatment. Studies have shown that Src protein tyrosine kinases (PTK's)… (more)

Subjects/Keywords: Acute lung injury; Drug delivery; High-throughput screen; Hydrophobic drug; Nanotechnology; Self-assembling peptide; 0719

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APA (6th Edition):

Pacheco, S. (2014). Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69966

Chicago Manual of Style (16th Edition):

Pacheco, Shaun. “Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury.” 2014. Masters Thesis, University of Toronto. Accessed October 21, 2019. http://hdl.handle.net/1807/69966.

MLA Handbook (7th Edition):

Pacheco, Shaun. “Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury.” 2014. Web. 21 Oct 2019.

Vancouver:

Pacheco S. Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1807/69966.

Council of Science Editors:

Pacheco S. Nanoscale formulation strategy for therapeutic agents in the prevention of acute lung injury. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/69966

24. Fernandes, Lucas Matos. Comparação entre duas soluções de recondicionamento pulmonar em pulmões humanos não-aceitos para transplante em modelo de avaliação e recondicionamento pulmonar ex vivo.

Degree: PhD, Cirurgia Torácica e Cardiovascular, 2015, University of São Paulo

 INTRODUÇÃO: O transplante pulmonar é terapia reconhecida de tratamento de doenças terminais pulmonares. Os pulmões, entretanto, são muito susceptíveis às transformações hormonais e hidroeletrolíticas ocorridas… (more)

Subjects/Keywords: Acute lung injury; Comparative study; Estudo comparativo; Lesão pulmonar aguda; Lung transplantation; Organ preservation; Preservação de órgãos; Transplante de pulmão

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APA (6th Edition):

Fernandes, L. M. (2015). Comparação entre duas soluções de recondicionamento pulmonar em pulmões humanos não-aceitos para transplante em modelo de avaliação e recondicionamento pulmonar ex vivo. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5156/tde-09112015-161415/ ;

Chicago Manual of Style (16th Edition):

Fernandes, Lucas Matos. “Comparação entre duas soluções de recondicionamento pulmonar em pulmões humanos não-aceitos para transplante em modelo de avaliação e recondicionamento pulmonar ex vivo.” 2015. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5156/tde-09112015-161415/ ;.

MLA Handbook (7th Edition):

Fernandes, Lucas Matos. “Comparação entre duas soluções de recondicionamento pulmonar em pulmões humanos não-aceitos para transplante em modelo de avaliação e recondicionamento pulmonar ex vivo.” 2015. Web. 21 Oct 2019.

Vancouver:

Fernandes LM. Comparação entre duas soluções de recondicionamento pulmonar em pulmões humanos não-aceitos para transplante em modelo de avaliação e recondicionamento pulmonar ex vivo. [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5156/tde-09112015-161415/ ;.

Council of Science Editors:

Fernandes LM. Comparação entre duas soluções de recondicionamento pulmonar em pulmões humanos não-aceitos para transplante em modelo de avaliação e recondicionamento pulmonar ex vivo. [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5156/tde-09112015-161415/ ;


University of Toronto

25. Lee, Dai Yoon. Therapeutic Peptide-functionalized Gold Nanoparticles for the Treatment of Acute Lung Injury.

Degree: 2013, University of Toronto

Acute lung injury (ALI) is a major cause of mortality after lung transplantation. Recent studies indicate protein kinase C delta (PKCδ) could be an effective… (more)

Subjects/Keywords: acute lung injury; gold nanoparticle; peptide; drug delivery; protein kinase C delta; lung transplant; 0379; 0719; 0794; 0491

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APA (6th Edition):

Lee, D. Y. (2013). Therapeutic Peptide-functionalized Gold Nanoparticles for the Treatment of Acute Lung Injury. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43003

Chicago Manual of Style (16th Edition):

Lee, Dai Yoon. “Therapeutic Peptide-functionalized Gold Nanoparticles for the Treatment of Acute Lung Injury.” 2013. Masters Thesis, University of Toronto. Accessed October 21, 2019. http://hdl.handle.net/1807/43003.

MLA Handbook (7th Edition):

Lee, Dai Yoon. “Therapeutic Peptide-functionalized Gold Nanoparticles for the Treatment of Acute Lung Injury.” 2013. Web. 21 Oct 2019.

Vancouver:

Lee DY. Therapeutic Peptide-functionalized Gold Nanoparticles for the Treatment of Acute Lung Injury. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1807/43003.

Council of Science Editors:

Lee DY. Therapeutic Peptide-functionalized Gold Nanoparticles for the Treatment of Acute Lung Injury. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43003

26. Freitas, Cláudia Regina da Costa. Avaliação do efeito da manutenção da perfusão e ventilação dos pulmões durante a circulação extracorpórea sobre a resposta inflamatória: estudo experimental.

Degree: PhD, Anestesiologia, 2013, University of São Paulo

 INTRODUÇÃO: A isquemia-reperfusão pulmonar e o uso do oxigenador de membranas são considerados fatores importantes na resposta inflamatória após a cirurgia cardíaca (CC) com utilização… (more)

Subjects/Keywords: Acute lung injury; Circulação extracorpórea; Cirurgia torácica; Extracorporeal circulation; Lesão pulmonar aguda; Reperfusion injury; Thoracic surgery; Traumatismo por reperfusão

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APA (6th Edition):

Freitas, C. R. d. C. (2013). Avaliação do efeito da manutenção da perfusão e ventilação dos pulmões durante a circulação extracorpórea sobre a resposta inflamatória: estudo experimental. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5152/tde-01082013-143111/ ;

Chicago Manual of Style (16th Edition):

Freitas, Cláudia Regina da Costa. “Avaliação do efeito da manutenção da perfusão e ventilação dos pulmões durante a circulação extracorpórea sobre a resposta inflamatória: estudo experimental.” 2013. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5152/tde-01082013-143111/ ;.

MLA Handbook (7th Edition):

Freitas, Cláudia Regina da Costa. “Avaliação do efeito da manutenção da perfusão e ventilação dos pulmões durante a circulação extracorpórea sobre a resposta inflamatória: estudo experimental.” 2013. Web. 21 Oct 2019.

Vancouver:

Freitas CRdC. Avaliação do efeito da manutenção da perfusão e ventilação dos pulmões durante a circulação extracorpórea sobre a resposta inflamatória: estudo experimental. [Internet] [Doctoral dissertation]. University of São Paulo; 2013. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5152/tde-01082013-143111/ ;.

Council of Science Editors:

Freitas CRdC. Avaliação do efeito da manutenção da perfusão e ventilação dos pulmões durante a circulação extracorpórea sobre a resposta inflamatória: estudo experimental. [Doctoral Dissertation]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5152/tde-01082013-143111/ ;

27. Campos, Renata. Efeitos da N-acetilcisteína na função pulmonar e renal em ratos com sepse sob ventilação mecânica invasiva.

Degree: PhD, Nefrologia, 2011, University of São Paulo

Introdução: A sepse pode causar alterações renais e pulmonares. Nos casos mais graves, a ventilação mecânica invasiva (VMI) torna-se necessária para melhorar as trocas gasosas… (more)

Subjects/Keywords: Acetilcisteína; Acetylcysteine; Acute lung injury; Acute renal injury; Estresse oxidativo; Insuficiência renal aguda; Lesão pulmonar aguda; Mecânica respiratória; Oxidative stress; Ratos Wistar; Respiratory mechanics; Sepse; Sepsis; Wistar rats

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Campos, R. (2011). Efeitos da N-acetilcisteína na função pulmonar e renal em ratos com sepse sob ventilação mecânica invasiva. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-12072011-163117/ ;

Chicago Manual of Style (16th Edition):

Campos, Renata. “Efeitos da N-acetilcisteína na função pulmonar e renal em ratos com sepse sob ventilação mecânica invasiva.” 2011. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-12072011-163117/ ;.

MLA Handbook (7th Edition):

Campos, Renata. “Efeitos da N-acetilcisteína na função pulmonar e renal em ratos com sepse sob ventilação mecânica invasiva.” 2011. Web. 21 Oct 2019.

Vancouver:

Campos R. Efeitos da N-acetilcisteína na função pulmonar e renal em ratos com sepse sob ventilação mecânica invasiva. [Internet] [Doctoral dissertation]. University of São Paulo; 2011. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-12072011-163117/ ;.

Council of Science Editors:

Campos R. Efeitos da N-acetilcisteína na função pulmonar e renal em ratos com sepse sob ventilação mecânica invasiva. [Doctoral Dissertation]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-12072011-163117/ ;

28. Medeiros, Israel Lopes de. Comparação entre as soluções de preservação pulmonar Perfadex® e LPD-G nacional em pulmões com um modelo de perfusão pulmonar ex vivo.

Degree: PhD, Cirurgia Torácica e Cardiovascular, 2012, University of São Paulo

INTRODUÇÃO: As técnicas de preservação pulmonar visam a melhorar a qualidade do enxerto e aumentar sua tolerância ao período de isquemia fria. A técnica mais… (more)

Subjects/Keywords: Acute lung injury; Lesão pulmonar aguda; Lung transplantation; Organ preservation solutions; Reperfusion injury; Soluções para preservação de órgãos; Transplante de pulmão; Traumatismo por reperfusão

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Medeiros, I. L. d. (2012). Comparação entre as soluções de preservação pulmonar Perfadex® e LPD-G nacional em pulmões com um modelo de perfusão pulmonar ex vivo. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5156/tde-25042012-104103/ ;

Chicago Manual of Style (16th Edition):

Medeiros, Israel Lopes de. “Comparação entre as soluções de preservação pulmonar Perfadex® e LPD-G nacional em pulmões com um modelo de perfusão pulmonar ex vivo.” 2012. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5156/tde-25042012-104103/ ;.

MLA Handbook (7th Edition):

Medeiros, Israel Lopes de. “Comparação entre as soluções de preservação pulmonar Perfadex® e LPD-G nacional em pulmões com um modelo de perfusão pulmonar ex vivo.” 2012. Web. 21 Oct 2019.

Vancouver:

Medeiros ILd. Comparação entre as soluções de preservação pulmonar Perfadex® e LPD-G nacional em pulmões com um modelo de perfusão pulmonar ex vivo. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5156/tde-25042012-104103/ ;.

Council of Science Editors:

Medeiros ILd. Comparação entre as soluções de preservação pulmonar Perfadex® e LPD-G nacional em pulmões com um modelo de perfusão pulmonar ex vivo. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5156/tde-25042012-104103/ ;

29. Calciolari, Christiane Costa. Modulação da expressão gênica de componentes envolvidos no processo de remodelamento pulmonar em diferentes modelos de lesão induzida pela ventilação mecânica.

Degree: PhD, Patologia, 2010, University of São Paulo

INTRODUÇÂO: Apesar de ser essencial, em algumas situações a ventilação mecânica (VM) pode acarretar danos ao pulmão sadio, evento conhecido como lesão pulmonar induzida pela… (more)

Subjects/Keywords: Acute lung injury; Expressão gênica; Extracellular matrix; Gene expression; Lesão pulmonar aguda; Matriz extracelular; Mecanotransdução celular; Mechanical ventilation; Mechanotransduction; Ventilação mecânica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Calciolari, C. C. (2010). Modulação da expressão gênica de componentes envolvidos no processo de remodelamento pulmonar em diferentes modelos de lesão induzida pela ventilação mecânica. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-03112010-155159/ ;

Chicago Manual of Style (16th Edition):

Calciolari, Christiane Costa. “Modulação da expressão gênica de componentes envolvidos no processo de remodelamento pulmonar em diferentes modelos de lesão induzida pela ventilação mecânica.” 2010. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-03112010-155159/ ;.

MLA Handbook (7th Edition):

Calciolari, Christiane Costa. “Modulação da expressão gênica de componentes envolvidos no processo de remodelamento pulmonar em diferentes modelos de lesão induzida pela ventilação mecânica.” 2010. Web. 21 Oct 2019.

Vancouver:

Calciolari CC. Modulação da expressão gênica de componentes envolvidos no processo de remodelamento pulmonar em diferentes modelos de lesão induzida pela ventilação mecânica. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-03112010-155159/ ;.

Council of Science Editors:

Calciolari CC. Modulação da expressão gênica de componentes envolvidos no processo de remodelamento pulmonar em diferentes modelos de lesão induzida pela ventilação mecânica. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-03112010-155159/ ;

30. Holms, Carla Augusto Thomaz de Aquino. Avaliação da resposta inflamatória pulmonar de suínos submetidos a lesão pulmonar aguda induzida por ácido clorídrico e tratados com solução salina hipertônica.

Degree: PhD, Anestesiologia, 2012, University of São Paulo

INTRODUÇÃO: A lesão pulmonar aguda (LPA) decorrente de aspiração do conteúdo gástrico é freqüente em pacientes com nível de consciência comprometido e depressão dos reflexos… (more)

Subjects/Keywords: Ácido clorídrico; Acute lung injury; Citocinas; Cytokine; Hydrochloric acid; Hypertonic saline; Inflamação; Inflammation; Lesão pulmonar aguda; Solução salina hipertônica; Suínos; Swine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Holms, C. A. T. d. A. (2012). Avaliação da resposta inflamatória pulmonar de suínos submetidos a lesão pulmonar aguda induzida por ácido clorídrico e tratados com solução salina hipertônica. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5152/tde-22012013-174923/ ;

Chicago Manual of Style (16th Edition):

Holms, Carla Augusto Thomaz de Aquino. “Avaliação da resposta inflamatória pulmonar de suínos submetidos a lesão pulmonar aguda induzida por ácido clorídrico e tratados com solução salina hipertônica.” 2012. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5152/tde-22012013-174923/ ;.

MLA Handbook (7th Edition):

Holms, Carla Augusto Thomaz de Aquino. “Avaliação da resposta inflamatória pulmonar de suínos submetidos a lesão pulmonar aguda induzida por ácido clorídrico e tratados com solução salina hipertônica.” 2012. Web. 21 Oct 2019.

Vancouver:

Holms CATdA. Avaliação da resposta inflamatória pulmonar de suínos submetidos a lesão pulmonar aguda induzida por ácido clorídrico e tratados com solução salina hipertônica. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5152/tde-22012013-174923/ ;.

Council of Science Editors:

Holms CATdA. Avaliação da resposta inflamatória pulmonar de suínos submetidos a lesão pulmonar aguda induzida por ácido clorídrico e tratados com solução salina hipertônica. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5152/tde-22012013-174923/ ;

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