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You searched for subject:(Activated protein C). Showing records 1 – 30 of 53 total matches.

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Université Paris-Sud – Paris XI

1. Besbes, Samaher. Rôle de la Protéine C, un anticoagulant naturel, dans l’association thrombose et cancer : Role of Protein C, a Natural Anticoagulant, in Thrombosis and Cancer Association.

Degree: Docteur es, Sciences de la vie et de la santé, 2015, Université Paris-Sud – Paris XI

Il est désormais admis que le caractère invasif d'une tumeur est lié, non seulement, au génotype des cellules cancéreuses, mais aussi à leurs interactions avec… (more)

Subjects/Keywords: Cancer; Thrombose; Protéine C; Protéine C activée; Récepteur endothélial de la protéine C; Thrombopoïétine; Cancer; Thrombosis; Protein C; Activated protein C; Endothelial protein C receptor; Thrombopoietin

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APA (6th Edition):

Besbes, S. (2015). Rôle de la Protéine C, un anticoagulant naturel, dans l’association thrombose et cancer : Role of Protein C, a Natural Anticoagulant, in Thrombosis and Cancer Association. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2015PA11T048

Chicago Manual of Style (16th Edition):

Besbes, Samaher. “Rôle de la Protéine C, un anticoagulant naturel, dans l’association thrombose et cancer : Role of Protein C, a Natural Anticoagulant, in Thrombosis and Cancer Association.” 2015. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 25, 2020. http://www.theses.fr/2015PA11T048.

MLA Handbook (7th Edition):

Besbes, Samaher. “Rôle de la Protéine C, un anticoagulant naturel, dans l’association thrombose et cancer : Role of Protein C, a Natural Anticoagulant, in Thrombosis and Cancer Association.” 2015. Web. 25 Sep 2020.

Vancouver:

Besbes S. Rôle de la Protéine C, un anticoagulant naturel, dans l’association thrombose et cancer : Role of Protein C, a Natural Anticoagulant, in Thrombosis and Cancer Association. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2015. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2015PA11T048.

Council of Science Editors:

Besbes S. Rôle de la Protéine C, un anticoagulant naturel, dans l’association thrombose et cancer : Role of Protein C, a Natural Anticoagulant, in Thrombosis and Cancer Association. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2015. Available from: http://www.theses.fr/2015PA11T048


University of Manchester

2. Miranda, Charles Joseph. NOVEL APPROACHES TO THE DIAGNOSIS AND MANAGEMENT OF SEVERE ACUTE PANCREATITIS.

Degree: 2016, University of Manchester

 TITLE: Novel approaches to the diagnosis and management of severe acute pancreatitis.INTRODUCTION: Severe Acute Pancreatitis (SAP) is the rapid onset ofinflammation within the pancreatic organ.… (more)

Subjects/Keywords: Pancreatitis; Early warning score; Activated protein C; Xigris; Abdominal compartment syndrome

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APA (6th Edition):

Miranda, C. J. (2016). NOVEL APPROACHES TO THE DIAGNOSIS AND MANAGEMENT OF SEVERE ACUTE PANCREATITIS. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303467

Chicago Manual of Style (16th Edition):

Miranda, Charles Joseph. “NOVEL APPROACHES TO THE DIAGNOSIS AND MANAGEMENT OF SEVERE ACUTE PANCREATITIS.” 2016. Doctoral Dissertation, University of Manchester. Accessed September 25, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303467.

MLA Handbook (7th Edition):

Miranda, Charles Joseph. “NOVEL APPROACHES TO THE DIAGNOSIS AND MANAGEMENT OF SEVERE ACUTE PANCREATITIS.” 2016. Web. 25 Sep 2020.

Vancouver:

Miranda CJ. NOVEL APPROACHES TO THE DIAGNOSIS AND MANAGEMENT OF SEVERE ACUTE PANCREATITIS. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Sep 25]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303467.

Council of Science Editors:

Miranda CJ. NOVEL APPROACHES TO THE DIAGNOSIS AND MANAGEMENT OF SEVERE ACUTE PANCREATITIS. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303467


Cornell University

3. Alabanza, Leah. The Coagulation Factors, Activated Protein C And Thrombin, Modulate The Pathogenesis Of Experimental Autoimmune Encephalomyelitis.

Degree: PhD, Immunology, 2013, Cornell University

 The immune and coagulation systems are closely linked. Components of the coagulation system, notably activated protein C (APC) and thrombin, have potent effects on the… (more)

Subjects/Keywords: Activated Protein C; Thrombin; EAE; Multiple Sclerosis; Immunology; Coagulation

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APA (6th Edition):

Alabanza, L. (2013). The Coagulation Factors, Activated Protein C And Thrombin, Modulate The Pathogenesis Of Experimental Autoimmune Encephalomyelitis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33895

Chicago Manual of Style (16th Edition):

Alabanza, Leah. “The Coagulation Factors, Activated Protein C And Thrombin, Modulate The Pathogenesis Of Experimental Autoimmune Encephalomyelitis.” 2013. Doctoral Dissertation, Cornell University. Accessed September 25, 2020. http://hdl.handle.net/1813/33895.

MLA Handbook (7th Edition):

Alabanza, Leah. “The Coagulation Factors, Activated Protein C And Thrombin, Modulate The Pathogenesis Of Experimental Autoimmune Encephalomyelitis.” 2013. Web. 25 Sep 2020.

Vancouver:

Alabanza L. The Coagulation Factors, Activated Protein C And Thrombin, Modulate The Pathogenesis Of Experimental Autoimmune Encephalomyelitis. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1813/33895.

Council of Science Editors:

Alabanza L. The Coagulation Factors, Activated Protein C And Thrombin, Modulate The Pathogenesis Of Experimental Autoimmune Encephalomyelitis. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33895


University of Manchester

4. Idicula Babu, Benoy. Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis.

Degree: Thesis (M.D.), 2012, University of Manchester

 Effective management of acute pancreatitis has for centuries eluded mankind. The disease has a wide spectrum of presentation; the milder form is usually a self… (more)

Subjects/Keywords: 616.3; Green tea extracts; Recombinant human activated protein C; Acute pancreatitis

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APA (6th Edition):

Idicula Babu, B. (2012). Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/epigallocatechin3gallate-and-recombinant-human-activated-protein-c-and-the-modulation-of-acute-pancreatitis(5fc797e9-cf5b-4aab-841d-62c3a8cd82c6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566553

Chicago Manual of Style (16th Edition):

Idicula Babu, Benoy. “Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis.” 2012. Doctoral Dissertation, University of Manchester. Accessed September 25, 2020. https://www.research.manchester.ac.uk/portal/en/theses/epigallocatechin3gallate-and-recombinant-human-activated-protein-c-and-the-modulation-of-acute-pancreatitis(5fc797e9-cf5b-4aab-841d-62c3a8cd82c6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566553.

MLA Handbook (7th Edition):

Idicula Babu, Benoy. “Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis.” 2012. Web. 25 Sep 2020.

Vancouver:

Idicula Babu B. Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Sep 25]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/epigallocatechin3gallate-and-recombinant-human-activated-protein-c-and-the-modulation-of-acute-pancreatitis(5fc797e9-cf5b-4aab-841d-62c3a8cd82c6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566553.

Council of Science Editors:

Idicula Babu B. Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/epigallocatechin3gallate-and-recombinant-human-activated-protein-c-and-the-modulation-of-acute-pancreatitis(5fc797e9-cf5b-4aab-841d-62c3a8cd82c6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566553

5. Κρητικός, Όθων. Η επίδραση της ηπαρίνης, της ενεργοποιημένης πρωτεΐνης C και της αντιθρομβίνης ΙΙΙ στην ταχύτητα επούλωσης των επιπολής και εν τω βάθει θερμικών εγκαυμάτων μερικού πάχους: πειραματική μελέτη επί χοίρων.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

So far the use of rhAPC has been effectively applied in the treatment of patients with severe sepsis. We conducted an experimental study of the… (more)

Subjects/Keywords: Εγκαύματα; Ενεργοποιημένη πρωτεΐνη C; Επούλωση; Burns; Healing; Recombinant human activated protein C

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APA (6th Edition):

Κρητικός, . . (2012). Η επίδραση της ηπαρίνης, της ενεργοποιημένης πρωτεΐνης C και της αντιθρομβίνης ΙΙΙ στην ταχύτητα επούλωσης των επιπολής και εν τω βάθει θερμικών εγκαυμάτων μερικού πάχους: πειραματική μελέτη επί χοίρων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/29590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κρητικός, Όθων. “Η επίδραση της ηπαρίνης, της ενεργοποιημένης πρωτεΐνης C και της αντιθρομβίνης ΙΙΙ στην ταχύτητα επούλωσης των επιπολής και εν τω βάθει θερμικών εγκαυμάτων μερικού πάχους: πειραματική μελέτη επί χοίρων.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed September 25, 2020. http://hdl.handle.net/10442/hedi/29590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κρητικός, Όθων. “Η επίδραση της ηπαρίνης, της ενεργοποιημένης πρωτεΐνης C και της αντιθρομβίνης ΙΙΙ στην ταχύτητα επούλωσης των επιπολής και εν τω βάθει θερμικών εγκαυμάτων μερικού πάχους: πειραματική μελέτη επί χοίρων.” 2012. Web. 25 Sep 2020.

Vancouver:

Κρητικός . Η επίδραση της ηπαρίνης, της ενεργοποιημένης πρωτεΐνης C και της αντιθρομβίνης ΙΙΙ στην ταχύτητα επούλωσης των επιπολής και εν τω βάθει θερμικών εγκαυμάτων μερικού πάχους: πειραματική μελέτη επί χοίρων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10442/hedi/29590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κρητικός . Η επίδραση της ηπαρίνης, της ενεργοποιημένης πρωτεΐνης C και της αντιθρομβίνης ΙΙΙ στην ταχύτητα επούλωσης των επιπολής και εν τω βάθει θερμικών εγκαυμάτων μερικού πάχους: πειραματική μελέτη επί χοίρων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/29590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

6. Smith, Jenell Rene. Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma.

Degree: 2015, University of Pennsylvania

 Peripheral neural trauma is known to induce blood-spinal cord barrier (BSCB) breakdown, but if and how BSCB breakdown contributes to pain is unknown. The studies… (more)

Subjects/Keywords: activated protein C; blood-spinal cord barrier; nerve root injury; pain; protease-activated receptor; thrombin; Biomedical

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APA (6th Edition):

Smith, J. R. (2015). Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Jenell Rene. “Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma.” 2015. Thesis, University of Pennsylvania. Accessed September 25, 2020. https://repository.upenn.edu/edissertations/2025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Jenell Rene. “Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma.” 2015. Web. 25 Sep 2020.

Vancouver:

Smith JR. Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2020 Sep 25]. Available from: https://repository.upenn.edu/edissertations/2025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith JR. Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/2025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Pinkosky, Stephen. ROLE OF ATP-CITRATE LYASE AND AMP-ACTIVATED PROTEIN KINASE IN REGULATING LIVER LIPID SYNTHESIS.

Degree: PhD, 2017, McMaster University

Cholesterol and fatty acid homeostasis is maintained by a complex network of regulatory mechanisms that control the biosynthesis and deposition of lipids over diverse physiological… (more)

Subjects/Keywords: ATP-citrate lyase; AMP-activated protein kinase; NAFLD; ASCVD; Atherosclerosis; LDL-C

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APA (6th Edition):

Pinkosky, S. (2017). ROLE OF ATP-CITRATE LYASE AND AMP-ACTIVATED PROTEIN KINASE IN REGULATING LIVER LIPID SYNTHESIS. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/23456

Chicago Manual of Style (16th Edition):

Pinkosky, Stephen. “ROLE OF ATP-CITRATE LYASE AND AMP-ACTIVATED PROTEIN KINASE IN REGULATING LIVER LIPID SYNTHESIS.” 2017. Doctoral Dissertation, McMaster University. Accessed September 25, 2020. http://hdl.handle.net/11375/23456.

MLA Handbook (7th Edition):

Pinkosky, Stephen. “ROLE OF ATP-CITRATE LYASE AND AMP-ACTIVATED PROTEIN KINASE IN REGULATING LIVER LIPID SYNTHESIS.” 2017. Web. 25 Sep 2020.

Vancouver:

Pinkosky S. ROLE OF ATP-CITRATE LYASE AND AMP-ACTIVATED PROTEIN KINASE IN REGULATING LIVER LIPID SYNTHESIS. [Internet] [Doctoral dissertation]. McMaster University; 2017. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/11375/23456.

Council of Science Editors:

Pinkosky S. ROLE OF ATP-CITRATE LYASE AND AMP-ACTIVATED PROTEIN KINASE IN REGULATING LIVER LIPID SYNTHESIS. [Doctoral Dissertation]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/23456


University of Rochester

8. DeAngelis, Jennifer Patricia. The Mechanisms for Catalytic Inactivation of FVIIIa by APC and Factor Xa.

Degree: PhD, 2012, University of Rochester

 Factor VIII (FVIII) is activated by thrombin and factor Xa (FXa) through proteolysis at Arg372, Arg740 and Arg1689 and inactivated by activated protein C (APC)… (more)

Subjects/Keywords: Factor VIII; Activated Protein C; P4-P3' Residues; Mutagenesis; Western Blotting; Factor Xa; Exosites

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APA (6th Edition):

DeAngelis, J. P. (2012). The Mechanisms for Catalytic Inactivation of FVIIIa by APC and Factor Xa. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25503

Chicago Manual of Style (16th Edition):

DeAngelis, Jennifer Patricia. “The Mechanisms for Catalytic Inactivation of FVIIIa by APC and Factor Xa.” 2012. Doctoral Dissertation, University of Rochester. Accessed September 25, 2020. http://hdl.handle.net/1802/25503.

MLA Handbook (7th Edition):

DeAngelis, Jennifer Patricia. “The Mechanisms for Catalytic Inactivation of FVIIIa by APC and Factor Xa.” 2012. Web. 25 Sep 2020.

Vancouver:

DeAngelis JP. The Mechanisms for Catalytic Inactivation of FVIIIa by APC and Factor Xa. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1802/25503.

Council of Science Editors:

DeAngelis JP. The Mechanisms for Catalytic Inactivation of FVIIIa by APC and Factor Xa. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25503


University of Manchester

9. Miranda, Charles Joseph. Novel approaches to the diagnosis and management of severe acute pancreatitis.

Degree: Thesis (M.D.), 2016, University of Manchester

 Severe Acute Pancreatitis (SAP) is the rapid onset of inflammation within the pancreatic organ. Unlike the milder form of this illness, SAP is associated with… (more)

Subjects/Keywords: 616.3; Pancreatitis; Early warning score; Activated protein C; Xigris; Abdominal compartment syndrome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Miranda, C. J. (2016). Novel approaches to the diagnosis and management of severe acute pancreatitis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/novel-approaches-to-the-diagnosis-and-management-of-severe-acute-pancreatitis(c24dab42-cd07-47bb-80ca-fcd3a5efcb44).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.694304

Chicago Manual of Style (16th Edition):

Miranda, Charles Joseph. “Novel approaches to the diagnosis and management of severe acute pancreatitis.” 2016. Doctoral Dissertation, University of Manchester. Accessed September 25, 2020. https://www.research.manchester.ac.uk/portal/en/theses/novel-approaches-to-the-diagnosis-and-management-of-severe-acute-pancreatitis(c24dab42-cd07-47bb-80ca-fcd3a5efcb44).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.694304.

MLA Handbook (7th Edition):

Miranda, Charles Joseph. “Novel approaches to the diagnosis and management of severe acute pancreatitis.” 2016. Web. 25 Sep 2020.

Vancouver:

Miranda CJ. Novel approaches to the diagnosis and management of severe acute pancreatitis. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Sep 25]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/novel-approaches-to-the-diagnosis-and-management-of-severe-acute-pancreatitis(c24dab42-cd07-47bb-80ca-fcd3a5efcb44).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.694304.

Council of Science Editors:

Miranda CJ. Novel approaches to the diagnosis and management of severe acute pancreatitis. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/novel-approaches-to-the-diagnosis-and-management-of-severe-acute-pancreatitis(c24dab42-cd07-47bb-80ca-fcd3a5efcb44).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.694304


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

10. Petras, Panagiotis. Ο ρόλος της ενεργοποιημένης πρωτεΐνης C (APC) στην βαριά οξεία παγκρεατίτιδα.

Degree: 2020, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Introduction: Acute pancreatitis is an inflammatory disease of the pancreas with significant morbidity and mortality. The aetiology of the disease is multiple, and its pathogenesis… (more)

Subjects/Keywords: Βαριά οξεία παγκρεατίτιδα; Ενεργοποιημένη πρωτεΐνη C; Φλεγμονώδης αντίδραση; Αντιπηκτική δράση; Severe acute pancreatitis; Activated protein C; Inflammatory response; Anti-thrombotic effect

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APA (6th Edition):

Petras, P. (2020). Ο ρόλος της ενεργοποιημένης πρωτεΐνης C (APC) στην βαριά οξεία παγκρεατίτιδα. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/47330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petras, Panagiotis. “Ο ρόλος της ενεργοποιημένης πρωτεΐνης C (APC) στην βαριά οξεία παγκρεατίτιδα.” 2020. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed September 25, 2020. http://hdl.handle.net/10442/hedi/47330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petras, Panagiotis. “Ο ρόλος της ενεργοποιημένης πρωτεΐνης C (APC) στην βαριά οξεία παγκρεατίτιδα.” 2020. Web. 25 Sep 2020.

Vancouver:

Petras P. Ο ρόλος της ενεργοποιημένης πρωτεΐνης C (APC) στην βαριά οξεία παγκρεατίτιδα. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2020. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10442/hedi/47330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petras P. Ο ρόλος της ενεργοποιημένης πρωτεΐνης C (APC) στην βαριά οξεία παγκρεατίτιδα. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2020. Available from: http://hdl.handle.net/10442/hedi/47330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Bouazza, Youcef. Effets de la protéine C activée et des glucocorticoïdes dans le choc septique expérimental : Activated protein C and glucocorticoid in experimental septic shock.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2011, Université Henri Poincaré – Nancy I

Le choc septique est la principale cause de mortalité dans les services de réanimation. Les glucocorticoïdes (GC) et la protéine C activée (APC) sont deux… (more)

Subjects/Keywords: Choc septique; Glucocorticoïdes; Protéine C activée; No; Septic shock; Glucocorticoids; Activated protein C; Nitric oxide; 616.944

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APA (6th Edition):

Bouazza, Y. (2011). Effets de la protéine C activée et des glucocorticoïdes dans le choc septique expérimental : Activated protein C and glucocorticoid in experimental septic shock. (Doctoral Dissertation). Université Henri Poincaré – Nancy I. Retrieved from http://www.theses.fr/2011NAN10098

Chicago Manual of Style (16th Edition):

Bouazza, Youcef. “Effets de la protéine C activée et des glucocorticoïdes dans le choc septique expérimental : Activated protein C and glucocorticoid in experimental septic shock.” 2011. Doctoral Dissertation, Université Henri Poincaré – Nancy I. Accessed September 25, 2020. http://www.theses.fr/2011NAN10098.

MLA Handbook (7th Edition):

Bouazza, Youcef. “Effets de la protéine C activée et des glucocorticoïdes dans le choc septique expérimental : Activated protein C and glucocorticoid in experimental septic shock.” 2011. Web. 25 Sep 2020.

Vancouver:

Bouazza Y. Effets de la protéine C activée et des glucocorticoïdes dans le choc septique expérimental : Activated protein C and glucocorticoid in experimental septic shock. [Internet] [Doctoral dissertation]. Université Henri Poincaré – Nancy I; 2011. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2011NAN10098.

Council of Science Editors:

Bouazza Y. Effets de la protéine C activée et des glucocorticoïdes dans le choc septique expérimental : Activated protein C and glucocorticoid in experimental septic shock. [Doctoral Dissertation]. Université Henri Poincaré – Nancy I; 2011. Available from: http://www.theses.fr/2011NAN10098


Université de Lorraine

12. Popovic, Batric. Caractérisation du phénotype hypercoagulable et de ses déterminants dans l’insuffisance cardiaque aiguë : Characterization of the hypercoagulable phenotype in patients with acute heart failure.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Lorraine

Malgré le bénéfice observé par de nouvelles thérapeutiques, le devenir des patients hospitalisés pour une insuffisance cardiaque aigue (ICA) reste sombre. Ce pronostic péjoratif est… (more)

Subjects/Keywords: Insuffisance cardiaque aiguë; Protéine C activée; Microparticules; Génération de thrombine; Acute heart failure; Activated C protein; Thrombin generation; Microparticles; 616.129

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APA (6th Edition):

Popovic, B. (2016). Caractérisation du phénotype hypercoagulable et de ses déterminants dans l’insuffisance cardiaque aiguë : Characterization of the hypercoagulable phenotype in patients with acute heart failure. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2016LORR0336

Chicago Manual of Style (16th Edition):

Popovic, Batric. “Caractérisation du phénotype hypercoagulable et de ses déterminants dans l’insuffisance cardiaque aiguë : Characterization of the hypercoagulable phenotype in patients with acute heart failure.” 2016. Doctoral Dissertation, Université de Lorraine. Accessed September 25, 2020. http://www.theses.fr/2016LORR0336.

MLA Handbook (7th Edition):

Popovic, Batric. “Caractérisation du phénotype hypercoagulable et de ses déterminants dans l’insuffisance cardiaque aiguë : Characterization of the hypercoagulable phenotype in patients with acute heart failure.” 2016. Web. 25 Sep 2020.

Vancouver:

Popovic B. Caractérisation du phénotype hypercoagulable et de ses déterminants dans l’insuffisance cardiaque aiguë : Characterization of the hypercoagulable phenotype in patients with acute heart failure. [Internet] [Doctoral dissertation]. Université de Lorraine; 2016. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2016LORR0336.

Council of Science Editors:

Popovic B. Caractérisation du phénotype hypercoagulable et de ses déterminants dans l’insuffisance cardiaque aiguë : Characterization of the hypercoagulable phenotype in patients with acute heart failure. [Doctoral Dissertation]. Université de Lorraine; 2016. Available from: http://www.theses.fr/2016LORR0336

13. Delabranche, Xavier. Microparticules membranaires au cours des états septiques graves : aspects cellulaires, physiopathologiques et cliniques : Menbrane microparticles during severe septic challenge : cellular, pathophysiological and clinical aspects.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université de Strasbourg

Ce travail porte sur le rôle des microparticules procoagulantes (MPs) générées par les cellules vasculaires en réponse à un état septique. Après une introduction rappelant… (more)

Subjects/Keywords: Microparticules; Choc septique; Coagulation intravasculaire disséminée; Protéine C activée; Endothélium; Hémostase; Inflammation; Procoagulant microparticles shed; Lymphocyte apoptosis; Septic shock; Activated protein C; Disseminated intravascular coagulopathy; 572.8; 612.1; 616.1

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APA (6th Edition):

Delabranche, X. (2013). Microparticules membranaires au cours des états septiques graves : aspects cellulaires, physiopathologiques et cliniques : Menbrane microparticles during severe septic challenge : cellular, pathophysiological and clinical aspects. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ087

Chicago Manual of Style (16th Edition):

Delabranche, Xavier. “Microparticules membranaires au cours des états septiques graves : aspects cellulaires, physiopathologiques et cliniques : Menbrane microparticles during severe septic challenge : cellular, pathophysiological and clinical aspects.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed September 25, 2020. http://www.theses.fr/2013STRAJ087.

MLA Handbook (7th Edition):

Delabranche, Xavier. “Microparticules membranaires au cours des états septiques graves : aspects cellulaires, physiopathologiques et cliniques : Menbrane microparticles during severe septic challenge : cellular, pathophysiological and clinical aspects.” 2013. Web. 25 Sep 2020.

Vancouver:

Delabranche X. Microparticules membranaires au cours des états septiques graves : aspects cellulaires, physiopathologiques et cliniques : Menbrane microparticles during severe septic challenge : cellular, pathophysiological and clinical aspects. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2013STRAJ087.

Council of Science Editors:

Delabranche X. Microparticules membranaires au cours des états septiques graves : aspects cellulaires, physiopathologiques et cliniques : Menbrane microparticles during severe septic challenge : cellular, pathophysiological and clinical aspects. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ087

14. Helms, Julie. Les microparticules dans le choc septique, marqueurs pathogènes et cibles thérapeutiques potentielles : Microparticles in septic shock, pathogenic biomarkers and potential therapeutic targets.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2014, Université de Strasbourg

Le choc septique est caractérisé par une intense activation cellulaire marquée par une génération excessive de microparticules (MPs), libérées dans l’espace extracellulaire suite à un… (more)

Subjects/Keywords: Microparticules; Choc septique; Coagulation intravasculaire disséminée; Protéine C activée; Endothélium; Inflammation; Hémostase; Microparticles; Septic shock; Disseminated intravascular coagulation; Activated protein C; Endothelium; Inflammation; Hemostasis; 572.8; 612.1; 616.1

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APA (6th Edition):

Helms, J. (2014). Les microparticules dans le choc septique, marqueurs pathogènes et cibles thérapeutiques potentielles : Microparticles in septic shock, pathogenic biomarkers and potential therapeutic targets. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2014STRAJ033

Chicago Manual of Style (16th Edition):

Helms, Julie. “Les microparticules dans le choc septique, marqueurs pathogènes et cibles thérapeutiques potentielles : Microparticles in septic shock, pathogenic biomarkers and potential therapeutic targets.” 2014. Doctoral Dissertation, Université de Strasbourg. Accessed September 25, 2020. http://www.theses.fr/2014STRAJ033.

MLA Handbook (7th Edition):

Helms, Julie. “Les microparticules dans le choc septique, marqueurs pathogènes et cibles thérapeutiques potentielles : Microparticles in septic shock, pathogenic biomarkers and potential therapeutic targets.” 2014. Web. 25 Sep 2020.

Vancouver:

Helms J. Les microparticules dans le choc septique, marqueurs pathogènes et cibles thérapeutiques potentielles : Microparticles in septic shock, pathogenic biomarkers and potential therapeutic targets. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2014. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2014STRAJ033.

Council of Science Editors:

Helms J. Les microparticules dans le choc septique, marqueurs pathogènes et cibles thérapeutiques potentielles : Microparticles in septic shock, pathogenic biomarkers and potential therapeutic targets. [Doctoral Dissertation]. Université de Strasbourg; 2014. Available from: http://www.theses.fr/2014STRAJ033

15. Καζιάνη, Αικατερίνη. Η επίδραση της χορήγησης αντιπηκτικής αγωγής στην πνευμονική κυκλοφορία ασθενών με σήψη και ασθενών με οξεία πνευμονική βλάβη (ALI).

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Severe sepsis and sepsis-related acute respiratory distress syndrome (ARDS), are associated with a major insult on endothelial anatomical and functional integrity that compromises microcirculation and… (more)

Subjects/Keywords: Βαρεία σήψη; Οξύ σύνδρομο αναπνευστικής δυσχέρειας; Ενεργοποιημένη πρωτεΐνη C; Πολυοργανική ανεπάρκεια; Αγγειομετατρεπτικό ένζυμο; Ενδοθηλιακή δυσλειτουργία; Severe sepsis; ARDS; Activated protein C; Multiorgan dysfunction; Endothelial dysfunction; ACE

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APA (6th Edition):

Καζιάνη, . . (2013). Η επίδραση της χορήγησης αντιπηκτικής αγωγής στην πνευμονική κυκλοφορία ασθενών με σήψη και ασθενών με οξεία πνευμονική βλάβη (ALI). (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/39052

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Καζιάνη, Αικατερίνη. “Η επίδραση της χορήγησης αντιπηκτικής αγωγής στην πνευμονική κυκλοφορία ασθενών με σήψη και ασθενών με οξεία πνευμονική βλάβη (ALI).” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed September 25, 2020. http://hdl.handle.net/10442/hedi/39052.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Καζιάνη, Αικατερίνη. “Η επίδραση της χορήγησης αντιπηκτικής αγωγής στην πνευμονική κυκλοφορία ασθενών με σήψη και ασθενών με οξεία πνευμονική βλάβη (ALI).” 2013. Web. 25 Sep 2020.

Vancouver:

Καζιάνη . Η επίδραση της χορήγησης αντιπηκτικής αγωγής στην πνευμονική κυκλοφορία ασθενών με σήψη και ασθενών με οξεία πνευμονική βλάβη (ALI). [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10442/hedi/39052.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Καζιάνη . Η επίδραση της χορήγησης αντιπηκτικής αγωγής στην πνευμονική κυκλοφορία ασθενών με σήψη και ασθενών με οξεία πνευμονική βλάβη (ALI). [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/39052

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

16. Cheng, Kwan-wai. Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase.

Degree: 2005, University of Hong Kong

Subjects/Keywords: Nucleosides.; Mitogen-activated protein kinases.; Genetic regulation.; Protein kinase C.

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APA (6th Edition):

Cheng, K. (2005). Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/131551

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheng, Kwan-wai. “Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase.” 2005. Thesis, University of Hong Kong. Accessed September 25, 2020. http://hdl.handle.net/10722/131551.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheng, Kwan-wai. “Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase.” 2005. Web. 25 Sep 2020.

Vancouver:

Cheng K. Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase. [Internet] [Thesis]. University of Hong Kong; 2005. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10722/131551.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheng K. Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase. [Thesis]. University of Hong Kong; 2005. Available from: http://hdl.handle.net/10722/131551

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

17. Kerkelä, R. (Risto). Signaling pathways in myocyte hypertrophy:role of GATA4, mitogen-activated protein kinases and protein kinase C.

Degree: 2003, University of Oulu

 Abstract Cardiac myocytes react to increased workload and hypertrophic neurohumoral stimuli by increasing protein synthesis, reinitiating expression of fetal forms of structural genes, α-skeletal actin… (more)

Subjects/Keywords: endothelin-1; hypertrophy; mitogen-activated protein kinase; protein kinase C; GATA4

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APA (6th Edition):

Kerkelä, R. (. (2003). Signaling pathways in myocyte hypertrophy:role of GATA4, mitogen-activated protein kinases and protein kinase C. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9514269950

Chicago Manual of Style (16th Edition):

Kerkelä, R (Risto). “Signaling pathways in myocyte hypertrophy:role of GATA4, mitogen-activated protein kinases and protein kinase C.” 2003. Doctoral Dissertation, University of Oulu. Accessed September 25, 2020. http://urn.fi/urn:isbn:9514269950.

MLA Handbook (7th Edition):

Kerkelä, R (Risto). “Signaling pathways in myocyte hypertrophy:role of GATA4, mitogen-activated protein kinases and protein kinase C.” 2003. Web. 25 Sep 2020.

Vancouver:

Kerkelä R(. Signaling pathways in myocyte hypertrophy:role of GATA4, mitogen-activated protein kinases and protein kinase C. [Internet] [Doctoral dissertation]. University of Oulu; 2003. [cited 2020 Sep 25]. Available from: http://urn.fi/urn:isbn:9514269950.

Council of Science Editors:

Kerkelä R(. Signaling pathways in myocyte hypertrophy:role of GATA4, mitogen-activated protein kinases and protein kinase C. [Doctoral Dissertation]. University of Oulu; 2003. Available from: http://urn.fi/urn:isbn:9514269950


Vrije Universiteit Amsterdam

18. Cramer, T.J. Inactivation of coagulation factors Va and VIIIa by activated protein C .

Degree: 2011, Vrije Universiteit Amsterdam

Subjects/Keywords: Blood coagulation; Activated factor VIII; Activated factor V; Anticoagulant factor V; Activated protein C; Autolysis loop; Anticoagulant

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APA (6th Edition):

Cramer, T. J. (2011). Inactivation of coagulation factors Va and VIIIa by activated protein C . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/19959

Chicago Manual of Style (16th Edition):

Cramer, T J. “Inactivation of coagulation factors Va and VIIIa by activated protein C .” 2011. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed September 25, 2020. http://hdl.handle.net/1871/19959.

MLA Handbook (7th Edition):

Cramer, T J. “Inactivation of coagulation factors Va and VIIIa by activated protein C .” 2011. Web. 25 Sep 2020.

Vancouver:

Cramer TJ. Inactivation of coagulation factors Va and VIIIa by activated protein C . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2011. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1871/19959.

Council of Science Editors:

Cramer TJ. Inactivation of coagulation factors Va and VIIIa by activated protein C . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2011. Available from: http://hdl.handle.net/1871/19959


University of New South Wales

19. Parsi, Kurosh. Interaction of detergent sclerosants with coagulation, antithrombotic and fibrinolytic mecanisms.

Degree: Clinical School - St Vincent's Hospital, 2011, University of New South Wales

 The effects of detergent sclerosants, sodium tetradecyl sulphate (STS) and polidocanol (POL), on coagulation, antithrombotic and fibrinolytic mechanisms were investigated in vitro.All samples were spiked… (more)

Subjects/Keywords: Detergents; Sclerosants; Sclerotherapy; Surfactants; Clotting factors; GPIIb/IIIa; Activated protein C; Annexin V; Phosphatidyl serine; Polidocanol; Coagulation; Clot formation; Thrombosis; Antithrombotic proteins; Plasminogen; Protein C; Protein S; Antithrombin; Fibrinolysis; Platelets; Microparticles; t-PA; PAI-1; Antiplasmin; Fibrinogen; Sodium tetradecyl sulphate; Fibrin; Thromboelastometry; Multiplate; Factor XIII; Albumin

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APA (6th Edition):

Parsi, K. (2011). Interaction of detergent sclerosants with coagulation, antithrombotic and fibrinolytic mecanisms. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50868 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9762/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Parsi, Kurosh. “Interaction of detergent sclerosants with coagulation, antithrombotic and fibrinolytic mecanisms.” 2011. Doctoral Dissertation, University of New South Wales. Accessed September 25, 2020. http://handle.unsw.edu.au/1959.4/50868 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9762/SOURCE02?view=true.

MLA Handbook (7th Edition):

Parsi, Kurosh. “Interaction of detergent sclerosants with coagulation, antithrombotic and fibrinolytic mecanisms.” 2011. Web. 25 Sep 2020.

Vancouver:

Parsi K. Interaction of detergent sclerosants with coagulation, antithrombotic and fibrinolytic mecanisms. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2020 Sep 25]. Available from: http://handle.unsw.edu.au/1959.4/50868 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9762/SOURCE02?view=true.

Council of Science Editors:

Parsi K. Interaction of detergent sclerosants with coagulation, antithrombotic and fibrinolytic mecanisms. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50868 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9762/SOURCE02?view=true


University of Georgia

20. Nash, Rodney James. Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling.

Degree: 2014, University of Georgia

 CD9, a member of the tetraspanin superfamily, is expressed in several cell lines and is associated with cellular activities such as migration, proliferation and metastasis… (more)

Subjects/Keywords: Cluster of differentiation 9; Forssman antigen; Dolichos biflorus agglutinin; Mitogen activated protein kinase; Stress activated protein kinase/ C-Jun N terminal kinase; Extracellular signal regulated kinase; Apoptosis; Rescue and Juxtacrine Organization

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APA (6th Edition):

Nash, R. J. (2014). Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/23477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nash, Rodney James. “Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling.” 2014. Thesis, University of Georgia. Accessed September 25, 2020. http://hdl.handle.net/10724/23477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nash, Rodney James. “Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling.” 2014. Web. 25 Sep 2020.

Vancouver:

Nash RJ. Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling. [Internet] [Thesis]. University of Georgia; 2014. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10724/23477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nash RJ. Tetraspanin CD9 regulates apoptosis in mouse embryonic stem cells through the suppression of epidermal growth factor receptor signaling. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/23477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

21. Yang, Tony L. Cellular origins and function of murine coagulation factor V.

Degree: PhD, Genetics, 1999, University of Michigan

 Coagulation factor V (FV) is a central regulator of the coagulation cascade, serving as a critical cofactor for procoagulant factor Xa, and as a major… (more)

Subjects/Keywords: Activated Protein C; Cellular; Coagulation Factor V; Function; Murine; Origins; Platelets; Procoagulants

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APA (6th Edition):

Yang, T. L. (1999). Cellular origins and function of murine coagulation factor V. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/131823

Chicago Manual of Style (16th Edition):

Yang, Tony L. “Cellular origins and function of murine coagulation factor V.” 1999. Doctoral Dissertation, University of Michigan. Accessed September 25, 2020. http://hdl.handle.net/2027.42/131823.

MLA Handbook (7th Edition):

Yang, Tony L. “Cellular origins and function of murine coagulation factor V.” 1999. Web. 25 Sep 2020.

Vancouver:

Yang TL. Cellular origins and function of murine coagulation factor V. [Internet] [Doctoral dissertation]. University of Michigan; 1999. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/2027.42/131823.

Council of Science Editors:

Yang TL. Cellular origins and function of murine coagulation factor V. [Doctoral Dissertation]. University of Michigan; 1999. Available from: http://hdl.handle.net/2027.42/131823


Brigham Young University

22. Ong, Kai Li. SNFing Glucose to PASs Mitochondrial Dysfunction: The Role of Two Sensory Protein Kinases in Metabolic Diseases.

Degree: PhD, 2019, Brigham Young University

 Mitochondria is no longer viewed as merely a powerhouse of the cell. It is now apparentthat mitochondria play a central role in signaling, maintaining cellular… (more)

Subjects/Keywords: AMP-activated Kinase; SNF1; Oxysterol binding protein; OSBP; Osh6; Osh7;

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APA (6th Edition):

Ong, K. L. (2019). SNFing Glucose to PASs Mitochondrial Dysfunction: The Role of Two Sensory Protein Kinases in Metabolic Diseases. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=9587&context=etd

Chicago Manual of Style (16th Edition):

Ong, Kai Li. “SNFing Glucose to PASs Mitochondrial Dysfunction: The Role of Two Sensory Protein Kinases in Metabolic Diseases.” 2019. Doctoral Dissertation, Brigham Young University. Accessed September 25, 2020. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=9587&context=etd.

MLA Handbook (7th Edition):

Ong, Kai Li. “SNFing Glucose to PASs Mitochondrial Dysfunction: The Role of Two Sensory Protein Kinases in Metabolic Diseases.” 2019. Web. 25 Sep 2020.

Vancouver:

Ong KL. SNFing Glucose to PASs Mitochondrial Dysfunction: The Role of Two Sensory Protein Kinases in Metabolic Diseases. [Internet] [Doctoral dissertation]. Brigham Young University; 2019. [cited 2020 Sep 25]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=9587&context=etd.

Council of Science Editors:

Ong KL. SNFing Glucose to PASs Mitochondrial Dysfunction: The Role of Two Sensory Protein Kinases in Metabolic Diseases. [Doctoral Dissertation]. Brigham Young University; 2019. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=9587&context=etd

23. Idicula Babu, Benoy. Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis.

Degree: 2012, University of Manchester

 Effective management of acute pancreatitis has for centuries eluded mankind. The disease has a wide spectrum of presentation; the milder form is usually a self… (more)

Subjects/Keywords: Green tea extracts; Recombinant human activated protein C; Acute pancreatitis

…146 8.3 Recombinant human activated protein C modulates the sub-cellular expression of… …Human recombinant activated protein C (Xigris) early in severe acute pancreatitis… …ratio Recombinant human activated protein C Soluble thrombomodulin Systemic inflammatory… …human Activated Protein C (rhAPC) has identified a potential treatment for acute… …Recombinant human activated protein C (Xigris) attenuates murine cerulein-induced acute… 

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APA (6th Edition):

Idicula Babu, B. (2012). Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:160046

Chicago Manual of Style (16th Edition):

Idicula Babu, Benoy. “Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis.” 2012. Doctoral Dissertation, University of Manchester. Accessed September 25, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:160046.

MLA Handbook (7th Edition):

Idicula Babu, Benoy. “Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis.” 2012. Web. 25 Sep 2020.

Vancouver:

Idicula Babu B. Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Sep 25]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:160046.

Council of Science Editors:

Idicula Babu B. Epigallocatechin-3-gallate and recombinant human activated protein C and the modulation of acute pancreatitis. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:160046


University of Adelaide

24. Melino, Michelle. The role of c-jun N-terminal kinase (JNK) in human T cell function.

Degree: 2009, University of Adelaide

 T cells are involved in cellular pathways which enable the immune system to protect us against infection and cancer. However, the same mechanisms also allow… (more)

Subjects/Keywords: T cells; cytokines; c-jun N-terminal kinase; mitogen-activated protein kinases

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APA (6th Edition):

Melino, M. (2009). The role of c-jun N-terminal kinase (JNK) in human T cell function. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/56209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melino, Michelle. “The role of c-jun N-terminal kinase (JNK) in human T cell function.” 2009. Thesis, University of Adelaide. Accessed September 25, 2020. http://hdl.handle.net/2440/56209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melino, Michelle. “The role of c-jun N-terminal kinase (JNK) in human T cell function.” 2009. Web. 25 Sep 2020.

Vancouver:

Melino M. The role of c-jun N-terminal kinase (JNK) in human T cell function. [Internet] [Thesis]. University of Adelaide; 2009. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/2440/56209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melino M. The role of c-jun N-terminal kinase (JNK) in human T cell function. [Thesis]. University of Adelaide; 2009. Available from: http://hdl.handle.net/2440/56209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

25. Sampram, Ellis. Arterial Thrombosis in Factor V Leiden or Activated Protein C Resistance. Clinical and Experimental Studies.

Degree: 2012, University of Lund

 Abstract The last two decades has seen an avalanche of studies establishing Activated protein C (APC) resistance due to Factor V Leiden mutation as the… (more)

Subjects/Keywords: Cardiac and Cardiovascular Systems; Activated protein c resistance; Factor V Leiden mutation; thrombophilia; hypercoagulable state; Factor V Leiden mice; arterial thrombosis; venous thromboembolism; peripheral vascular reconstructions; peripheral vascular reconstruction occlusion

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APA (6th Edition):

Sampram, E. (2012). Arterial Thrombosis in Factor V Leiden or Activated Protein C Resistance. Clinical and Experimental Studies. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/3164139 ; https://portal.research.lu.se/ws/files/3494208/3165175.pdf

Chicago Manual of Style (16th Edition):

Sampram, Ellis. “Arterial Thrombosis in Factor V Leiden or Activated Protein C Resistance. Clinical and Experimental Studies.” 2012. Doctoral Dissertation, University of Lund. Accessed September 25, 2020. https://lup.lub.lu.se/record/3164139 ; https://portal.research.lu.se/ws/files/3494208/3165175.pdf.

MLA Handbook (7th Edition):

Sampram, Ellis. “Arterial Thrombosis in Factor V Leiden or Activated Protein C Resistance. Clinical and Experimental Studies.” 2012. Web. 25 Sep 2020.

Vancouver:

Sampram E. Arterial Thrombosis in Factor V Leiden or Activated Protein C Resistance. Clinical and Experimental Studies. [Internet] [Doctoral dissertation]. University of Lund; 2012. [cited 2020 Sep 25]. Available from: https://lup.lub.lu.se/record/3164139 ; https://portal.research.lu.se/ws/files/3494208/3165175.pdf.

Council of Science Editors:

Sampram E. Arterial Thrombosis in Factor V Leiden or Activated Protein C Resistance. Clinical and Experimental Studies. [Doctoral Dissertation]. University of Lund; 2012. Available from: https://lup.lub.lu.se/record/3164139 ; https://portal.research.lu.se/ws/files/3494208/3165175.pdf


Université de Lorraine

26. Issa, Khodr. Effet protecteur du sulfure d'hydrogène, de la protéine C activée et de la dexamétasone dans la modulation hémodynamique et inflammatoire de l'ischémie/reperfusion : Protector effect of hydrogen sulfure, protein C activated and dexamethason in the hemodynamic and inflammatory modulation in ischemia-reperfusion.

Degree: Docteur es, Sciences de la vie et de la santé, 2013, Université de Lorraine

L'ischémie/reperfusion (I/R) est un phénomène très fréquent en clinique humaine. Ce phénomène est observé lors de la désobstruction d'une artère digestive, du traitement d'un état… (more)

Subjects/Keywords: Choc hémorragique; Ischémie/Reperfusion; Monoxyde d'azote (NO); Sulfure d'hydrogène (H2S); Protéine C activée (PCa); Dexamethasone (Dexa); Hemorrhagic shock; Ischemia/Reperfusion; Nitric oxide (NO); Hydrogen sulfide (H2S); Activated Protein C (APC); Dexamethason (Dexa); 617.919

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APA (6th Edition):

Issa, K. (2013). Effet protecteur du sulfure d'hydrogène, de la protéine C activée et de la dexamétasone dans la modulation hémodynamique et inflammatoire de l'ischémie/reperfusion : Protector effect of hydrogen sulfure, protein C activated and dexamethason in the hemodynamic and inflammatory modulation in ischemia-reperfusion. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2013LORR0059

Chicago Manual of Style (16th Edition):

Issa, Khodr. “Effet protecteur du sulfure d'hydrogène, de la protéine C activée et de la dexamétasone dans la modulation hémodynamique et inflammatoire de l'ischémie/reperfusion : Protector effect of hydrogen sulfure, protein C activated and dexamethason in the hemodynamic and inflammatory modulation in ischemia-reperfusion.” 2013. Doctoral Dissertation, Université de Lorraine. Accessed September 25, 2020. http://www.theses.fr/2013LORR0059.

MLA Handbook (7th Edition):

Issa, Khodr. “Effet protecteur du sulfure d'hydrogène, de la protéine C activée et de la dexamétasone dans la modulation hémodynamique et inflammatoire de l'ischémie/reperfusion : Protector effect of hydrogen sulfure, protein C activated and dexamethason in the hemodynamic and inflammatory modulation in ischemia-reperfusion.” 2013. Web. 25 Sep 2020.

Vancouver:

Issa K. Effet protecteur du sulfure d'hydrogène, de la protéine C activée et de la dexamétasone dans la modulation hémodynamique et inflammatoire de l'ischémie/reperfusion : Protector effect of hydrogen sulfure, protein C activated and dexamethason in the hemodynamic and inflammatory modulation in ischemia-reperfusion. [Internet] [Doctoral dissertation]. Université de Lorraine; 2013. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2013LORR0059.

Council of Science Editors:

Issa K. Effet protecteur du sulfure d'hydrogène, de la protéine C activée et de la dexamétasone dans la modulation hémodynamique et inflammatoire de l'ischémie/reperfusion : Protector effect of hydrogen sulfure, protein C activated and dexamethason in the hemodynamic and inflammatory modulation in ischemia-reperfusion. [Doctoral Dissertation]. Université de Lorraine; 2013. Available from: http://www.theses.fr/2013LORR0059


Université de Lorraine

27. Said, Rose. Caractérisation des propriétés pro- et anti-coagulantes associées aux cellules musculaires lisses vasculaires : Characterization of pro-and anti-coagulant properties of vascular smooth muscle cells.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2012, Université de Lorraine

L'objectif principal de ce travail était de comparer l'implication (i) de cellules vasculaires, cellules musculaires lisses vasculaires (CML) et cellules endothéliales (CE), ou des cellules… (more)

Subjects/Keywords: Cellules musculaires lisses vasculaires, Microparticules, Génération de thrombine, Sensibilité à la protéine C activée, Intégrine [alpha]v[gamma]3; Vascular smooth muscle cells, Microparticles, Thrombin generation, Sensitivity to activated protein C, [alpha]v[gamma]3 integrin; 612.13; 611.018 6

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APA (6th Edition):

Said, R. (2012). Caractérisation des propriétés pro- et anti-coagulantes associées aux cellules musculaires lisses vasculaires : Characterization of pro-and anti-coagulant properties of vascular smooth muscle cells. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2012LORR0004

Chicago Manual of Style (16th Edition):

Said, Rose. “Caractérisation des propriétés pro- et anti-coagulantes associées aux cellules musculaires lisses vasculaires : Characterization of pro-and anti-coagulant properties of vascular smooth muscle cells.” 2012. Doctoral Dissertation, Université de Lorraine. Accessed September 25, 2020. http://www.theses.fr/2012LORR0004.

MLA Handbook (7th Edition):

Said, Rose. “Caractérisation des propriétés pro- et anti-coagulantes associées aux cellules musculaires lisses vasculaires : Characterization of pro-and anti-coagulant properties of vascular smooth muscle cells.” 2012. Web. 25 Sep 2020.

Vancouver:

Said R. Caractérisation des propriétés pro- et anti-coagulantes associées aux cellules musculaires lisses vasculaires : Characterization of pro-and anti-coagulant properties of vascular smooth muscle cells. [Internet] [Doctoral dissertation]. Université de Lorraine; 2012. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2012LORR0004.

Council of Science Editors:

Said R. Caractérisation des propriétés pro- et anti-coagulantes associées aux cellules musculaires lisses vasculaires : Characterization of pro-and anti-coagulant properties of vascular smooth muscle cells. [Doctoral Dissertation]. Université de Lorraine; 2012. Available from: http://www.theses.fr/2012LORR0004

28. Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, Hisashi. p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells.

Degree: 博士(歯学), 2015, Fukuoka Dental College / 福岡歯科大学

To investigate the involvement of stress-activated protein kinases, JNK and p38 MAPK, in the assembly of tight junctions in keratinocytes, we treated HaCaT cells with… (more)

Subjects/Keywords: MAPK; JNK; p38; タイト結合; ZO-1; HaCaT細胞; mitogen-activated protein kinase (MAPK); c-Jun NH2-terminal protein kinase (JNK); p38 MAPK; tight junction; ZO-1; HaCaT cell

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APA (6th Edition):

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, H. (2015). p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000024/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, Hisashi. “p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells.” 2015. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed September 25, 2020. http://id.nii.ac.jp/1167/00000024/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, Hisashi. “p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells.” 2015. Web. 25 Sep 2020.

Vancouver:

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan H. p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. [cited 2020 Sep 25]. Available from: http://id.nii.ac.jp/1167/00000024/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan H. p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. Available from: http://id.nii.ac.jp/1167/00000024/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

29. Ginneken, Mireille Maria Elisabeth van. Adaptation of signal transduction and muscle proteome in trained horses.

Degree: 2006, Universiteit Utrecht

 In the present thesis the localization and activation of signaling proteins, known from human studies, in equine muscle were investigated under conditions of rest, after… (more)

Subjects/Keywords: Geneeskunde; exercise physiology; horses; signal transduction; overtraining; proteomics; mitogen-activated protein kinase; protein kinase c

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APA (6th Edition):

Ginneken, M. M. E. v. (2006). Adaptation of signal transduction and muscle proteome in trained horses. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/8484

Chicago Manual of Style (16th Edition):

Ginneken, Mireille Maria Elisabeth van. “Adaptation of signal transduction and muscle proteome in trained horses.” 2006. Doctoral Dissertation, Universiteit Utrecht. Accessed September 25, 2020. http://dspace.library.uu.nl:8080/handle/1874/8484.

MLA Handbook (7th Edition):

Ginneken, Mireille Maria Elisabeth van. “Adaptation of signal transduction and muscle proteome in trained horses.” 2006. Web. 25 Sep 2020.

Vancouver:

Ginneken MMEv. Adaptation of signal transduction and muscle proteome in trained horses. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2006. [cited 2020 Sep 25]. Available from: http://dspace.library.uu.nl:8080/handle/1874/8484.

Council of Science Editors:

Ginneken MMEv. Adaptation of signal transduction and muscle proteome in trained horses. [Doctoral Dissertation]. Universiteit Utrecht; 2006. Available from: http://dspace.library.uu.nl:8080/handle/1874/8484

30. Ginneken, Mireille Maria Elisabeth van. Adaptation of signal transduction and muscle proteome in trained horses.

Degree: 2006, University Utrecht

 In the present thesis the localization and activation of signaling proteins, known from human studies, in equine muscle were investigated under conditions of rest, after… (more)

Subjects/Keywords: exercise physiology; horses; signal transduction; overtraining; proteomics; mitogen-activated protein kinase; protein kinase c

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ginneken, M. M. E. v. (2006). Adaptation of signal transduction and muscle proteome in trained horses. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/8484 ; URN:NBN:NL:UI:10-1874-8484 ; urn:isbn:90-9020511-X ; URN:NBN:NL:UI:10-1874-8484 ; https://dspace.library.uu.nl/handle/1874/8484

Chicago Manual of Style (16th Edition):

Ginneken, Mireille Maria Elisabeth van. “Adaptation of signal transduction and muscle proteome in trained horses.” 2006. Doctoral Dissertation, University Utrecht. Accessed September 25, 2020. https://dspace.library.uu.nl/handle/1874/8484 ; URN:NBN:NL:UI:10-1874-8484 ; urn:isbn:90-9020511-X ; URN:NBN:NL:UI:10-1874-8484 ; https://dspace.library.uu.nl/handle/1874/8484.

MLA Handbook (7th Edition):

Ginneken, Mireille Maria Elisabeth van. “Adaptation of signal transduction and muscle proteome in trained horses.” 2006. Web. 25 Sep 2020.

Vancouver:

Ginneken MMEv. Adaptation of signal transduction and muscle proteome in trained horses. [Internet] [Doctoral dissertation]. University Utrecht; 2006. [cited 2020 Sep 25]. Available from: https://dspace.library.uu.nl/handle/1874/8484 ; URN:NBN:NL:UI:10-1874-8484 ; urn:isbn:90-9020511-X ; URN:NBN:NL:UI:10-1874-8484 ; https://dspace.library.uu.nl/handle/1874/8484.

Council of Science Editors:

Ginneken MMEv. Adaptation of signal transduction and muscle proteome in trained horses. [Doctoral Dissertation]. University Utrecht; 2006. Available from: https://dspace.library.uu.nl/handle/1874/8484 ; URN:NBN:NL:UI:10-1874-8484 ; urn:isbn:90-9020511-X ; URN:NBN:NL:UI:10-1874-8484 ; https://dspace.library.uu.nl/handle/1874/8484

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