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You searched for subject:(Acetaminophen Toxicology ). Showing records 1 – 27 of 27 total matches.

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University of South Florida

1. Haire, Kambria. Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity.

Degree: 2015, University of South Florida

 Drug toxicity may cause liver injury, resulting in damage to cells and tissues. This damage can lead to cytotoxic events that may result in an… (more)

Subjects/Keywords: PARP; Heptotoxicity; Cocaine; Acetaminophen; Toxicology

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APA (6th Edition):

Haire, K. (2015). Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/5959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haire, Kambria. “Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity.” 2015. Thesis, University of South Florida. Accessed June 19, 2019. https://scholarcommons.usf.edu/etd/5959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haire, Kambria. “Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity.” 2015. Web. 19 Jun 2019.

Vancouver:

Haire K. Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity. [Internet] [Thesis]. University of South Florida; 2015. [cited 2019 Jun 19]. Available from: https://scholarcommons.usf.edu/etd/5959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haire K. Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity. [Thesis]. University of South Florida; 2015. Available from: https://scholarcommons.usf.edu/etd/5959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

2. McGreal, Steven. The Role of O-GlcNAc in Liver Injury and Regeneration.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 O-GlcNAcylation is a covalent attachment of a single N-acetyl glucosamine to a serine or threonine residue of a protein. Unlike other forms of protein glycosylation,… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Liver; O-GlcNAc; regeneration

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APA (6th Edition):

McGreal, S. (2017). The Role of O-GlcNAc in Liver Injury and Regeneration. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27029

Chicago Manual of Style (16th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/27029.

MLA Handbook (7th Edition):

McGreal, Steven. “The Role of O-GlcNAc in Liver Injury and Regeneration.” 2017. Web. 19 Jun 2019.

Vancouver:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/27029.

Council of Science Editors:

McGreal S. The Role of O-GlcNAc in Liver Injury and Regeneration. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27029


University of Arizona

3. MACDONALD, JOHN ROBERT. CYSTAMINE TREATMENT OF CHEMICALLY-INDUCED HEPATOTOXICITY.

Degree: 1984, University of Arizona

 There are few examples of therapeutic treatments in chemically-induced toxicity compared to pretreatments that protect against chemical injury. Cystamine treatment 12 hours after carbon tetrachloride… (more)

Subjects/Keywords: Toxicology, Experimental.; Acetaminophen  – Toxicology.

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APA (6th Edition):

MACDONALD, J. R. (1984). CYSTAMINE TREATMENT OF CHEMICALLY-INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/187813

Chicago Manual of Style (16th Edition):

MACDONALD, JOHN ROBERT. “CYSTAMINE TREATMENT OF CHEMICALLY-INDUCED HEPATOTOXICITY. ” 1984. Doctoral Dissertation, University of Arizona. Accessed June 19, 2019. http://hdl.handle.net/10150/187813.

MLA Handbook (7th Edition):

MACDONALD, JOHN ROBERT. “CYSTAMINE TREATMENT OF CHEMICALLY-INDUCED HEPATOTOXICITY. ” 1984. Web. 19 Jun 2019.

Vancouver:

MACDONALD JR. CYSTAMINE TREATMENT OF CHEMICALLY-INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Arizona; 1984. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/10150/187813.

Council of Science Editors:

MACDONALD JR. CYSTAMINE TREATMENT OF CHEMICALLY-INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Arizona; 1984. Available from: http://hdl.handle.net/10150/187813


University of Adelaide

4. Pramyothin, Pornpen. Metabolism and toxicity of paracetamol in isolated rat and mouse hepatocytes / by Pornpen Pramyothin.

Degree: 1980, University of Adelaide

Subjects/Keywords: Acetaminophen Toxicology.; Acetaminophen Metabolism; Liver cells.

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APA (6th Edition):

Pramyothin, P. (1980). Metabolism and toxicity of paracetamol in isolated rat and mouse hepatocytes / by Pornpen Pramyothin. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/20272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pramyothin, Pornpen. “Metabolism and toxicity of paracetamol in isolated rat and mouse hepatocytes / by Pornpen Pramyothin.” 1980. Thesis, University of Adelaide. Accessed June 19, 2019. http://hdl.handle.net/2440/20272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pramyothin, Pornpen. “Metabolism and toxicity of paracetamol in isolated rat and mouse hepatocytes / by Pornpen Pramyothin.” 1980. Web. 19 Jun 2019.

Vancouver:

Pramyothin P. Metabolism and toxicity of paracetamol in isolated rat and mouse hepatocytes / by Pornpen Pramyothin. [Internet] [Thesis]. University of Adelaide; 1980. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/2440/20272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pramyothin P. Metabolism and toxicity of paracetamol in isolated rat and mouse hepatocytes / by Pornpen Pramyothin. [Thesis]. University of Adelaide; 1980. Available from: http://hdl.handle.net/2440/20272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

5. Boudreau, Jordache. Electrochemical Generation of Reactive Species and their Application as Chemotherapeutics .

Degree: 2012, University of Guelph

 A major limitation in developing a successful cancer treatment is the need for a distinction between normal and cancerous tissue. For solid tumors, this distinction… (more)

Subjects/Keywords: electrochemistry; toxicology; cancer biology; cyclophosphamide; acetaminophen; focal therapy; graphite; oxidation; bioactivation

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APA (6th Edition):

Boudreau, J. (2012). Electrochemical Generation of Reactive Species and their Application as Chemotherapeutics . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boudreau, Jordache. “Electrochemical Generation of Reactive Species and their Application as Chemotherapeutics .” 2012. Thesis, University of Guelph. Accessed June 19, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boudreau, Jordache. “Electrochemical Generation of Reactive Species and their Application as Chemotherapeutics .” 2012. Web. 19 Jun 2019.

Vancouver:

Boudreau J. Electrochemical Generation of Reactive Species and their Application as Chemotherapeutics . [Internet] [Thesis]. University of Guelph; 2012. [cited 2019 Jun 19]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boudreau J. Electrochemical Generation of Reactive Species and their Application as Chemotherapeutics . [Thesis]. University of Guelph; 2012. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

6. Weemhoff, James Lawrence. Mechanistic Biomarkers in Acute Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

 Acute liver failure continues to be a major medical problem. There are many underlying causes of acute liver failure, but drug induced liver injury is… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Biomarkers; HepaRG; Hypoxic Hepatitis; Liver Injury; Liver Transplantation

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APA (6th Edition):

Weemhoff, J. L. (2017). Mechanistic Biomarkers in Acute Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/27044

Chicago Manual of Style (16th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/27044.

MLA Handbook (7th Edition):

Weemhoff, James Lawrence. “Mechanistic Biomarkers in Acute Liver Injury.” 2017. Web. 19 Jun 2019.

Vancouver:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/27044.

Council of Science Editors:

Weemhoff JL. Mechanistic Biomarkers in Acute Liver Injury. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/27044

7. Dragomir, Ana-Cristina, 1981-. Mechanisms mediating macrophage activation during acetaminophen-induced hepatotoxicity: role of high mobility group box-1 and galectin-3.

Degree: Toxicology, 2012, Rutgers University

Subjects/Keywords: Acetaminophen – Toxicology; Macrophages – Activation

acetaminophen ASC apoptosis-associated speck-like protein ATCC American Type Culture Collection… 

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APA (6th Edition):

Dragomir, Ana-Cristina, 1. (2012). Mechanisms mediating macrophage activation during acetaminophen-induced hepatotoxicity: role of high mobility group box-1 and galectin-3. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dragomir, Ana-Cristina, 1981-. “Mechanisms mediating macrophage activation during acetaminophen-induced hepatotoxicity: role of high mobility group box-1 and galectin-3.” 2012. Thesis, Rutgers University. Accessed June 19, 2019. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dragomir, Ana-Cristina, 1981-. “Mechanisms mediating macrophage activation during acetaminophen-induced hepatotoxicity: role of high mobility group box-1 and galectin-3.” 2012. Web. 19 Jun 2019.

Vancouver:

Dragomir, Ana-Cristina 1. Mechanisms mediating macrophage activation during acetaminophen-induced hepatotoxicity: role of high mobility group box-1 and galectin-3. [Internet] [Thesis]. Rutgers University; 2012. [cited 2019 Jun 19]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066724.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dragomir, Ana-Cristina 1. Mechanisms mediating macrophage activation during acetaminophen-induced hepatotoxicity: role of high mobility group box-1 and galectin-3. [Thesis]. Rutgers University; 2012. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066724

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Williams, Jessica A. R. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

Acetaminophen (APAP) is the leading cause of acute liver failure in the United States, and alcoholic liver disease (ALD) is a worldwide health problem that… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alcohol; Autophagy; Liver Injury; Mitophagy; Parkin

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APA (6th Edition):

Williams, J. A. R. (2015). The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19470

Chicago Manual of Style (16th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/19470.

MLA Handbook (7th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Web. 19 Jun 2019.

Vancouver:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/19470.

Council of Science Editors:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19470


Michigan State University

9. Miyakawa, Kazuhisa. Contribution of thrombin and platelets to acetaminophen hepatotoxicity in mice.

Degree: 2014, Michigan State University

Thesis Ph. D. Michigan State University. Pathobiology-Environmental Toxicology 2014.

Acetaminophen (APAP) is one of the most widely used antipyretic and analgesic drugs in both children… (more)

Subjects/Keywords: Acetaminophen – Toxicology; Hepatotoxicology; Thrombin; Blood platelets; Mice – Effect of drugs on; Toxicology; Pharmacology; Pathology

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APA (6th Edition):

Miyakawa, K. (2014). Contribution of thrombin and platelets to acetaminophen hepatotoxicity in mice. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3071

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miyakawa, Kazuhisa. “Contribution of thrombin and platelets to acetaminophen hepatotoxicity in mice.” 2014. Thesis, Michigan State University. Accessed June 19, 2019. http://etd.lib.msu.edu/islandora/object/etd:3071.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miyakawa, Kazuhisa. “Contribution of thrombin and platelets to acetaminophen hepatotoxicity in mice.” 2014. Web. 19 Jun 2019.

Vancouver:

Miyakawa K. Contribution of thrombin and platelets to acetaminophen hepatotoxicity in mice. [Internet] [Thesis]. Michigan State University; 2014. [cited 2019 Jun 19]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3071.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miyakawa K. Contribution of thrombin and platelets to acetaminophen hepatotoxicity in mice. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:3071

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

10. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 19 Jun 2019.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369

11. Williams, Clarence David. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

Acetaminophen (APAP) overdose is the leading cause of acute liver failure in the US. APAP is metabolized to a reactive metabolite that causes hepatotoxicity in… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Il-1beta; Neutrophil

Acetaminophen hepatotoxicity and repair: the role of sterile inflammation and innate immunity, 2012… …with permission from Wiley. 1 1.1 History of APAP The history of acetaminophen (APAP… …APAP It is estimated that each week approximately 50 million Americans consume acetaminophen… …APAP-induced injury  If IL-1β is responsible for enhanced injury during acetaminophen… 

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APA (6th Edition):

Williams, C. D. (2012). Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10290

Chicago Manual of Style (16th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/10290.

MLA Handbook (7th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Web. 19 Jun 2019.

Vancouver:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/10290.

Council of Science Editors:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10290


University of Kansas

12. Du, Kuo. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2017, University of Kansas

Acetaminophen (APAP) overdose causes severe hepatotoxicity in animals and humans. Although numerous studies have established the existence of an extensive oxidative stress during APAP hepatotoxicity,… (more)

Subjects/Keywords: Toxicology; Pharmacology; Molecular biology; Acetaminophen; liver toxicity; metformin; mitochondria; mito-tempo; oxidative stress

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APA (6th Edition):

Du, K. (2017). MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/26928

Chicago Manual of Style (16th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/26928.

MLA Handbook (7th Edition):

Du, Kuo. “MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY.” 2017. Web. 19 Jun 2019.

Vancouver:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2017. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/26928.

Council of Science Editors:

Du K. MITOCHONDRIAL OXIDATIVE STRESS AND BIOGENESIS DURING ACETAMINOPHEN HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2017. Available from: http://hdl.handle.net/1808/26928


University of Kansas

13. Bhushan, Bharat. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Overdose of acetaminophen (APAP) is the major cause of acute liver failure (ALF) in the western world with very limited treatment options. Recent studies suggest… (more)

Subjects/Keywords: Toxicology; Pharmacology; Acetaminophen; EGF receptor; Liver; mice model; Regeneration; Wnt-Beta Catenin-GSK3 Beta pathway

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APA (6th Edition):

Bhushan, B. (2016). MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/25366

Chicago Manual of Style (16th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/25366.

MLA Handbook (7th Edition):

Bhushan, Bharat. “MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE.” 2016. Web. 19 Jun 2019.

Vancouver:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/25366.

Council of Science Editors:

Bhushan B. MECHANISMS OF LIVER REGENERATION AFTER ACETAMINOPHEN-INDUCED HEPATOTOXICITY IN MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/25366


Central Connecticut State University

14. Benitex, Yulianingsih. The effects of phenetyl isothiocyanate and benzyl isothiocyanate on acetaminophen metabolism and toxicity in freshly isolated rat hepatocytes in cell suspension.

Degree: Department of Chemistry and Biochemistry, 1999, Central Connecticut State University

Acetaminophen is extensively used as an analgesic and antipyretic agent, but at high doses it causes liver damage. The major metabolic pathways of acetaminophen are… (more)

Subjects/Keywords: Acetaminophen – Toxicity testing.; Liver cells.; Toxicology, Experimental.

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APA (6th Edition):

Benitex, Y. (1999). The effects of phenetyl isothiocyanate and benzyl isothiocyanate on acetaminophen metabolism and toxicity in freshly isolated rat hepatocytes in cell suspension. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,2130

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Benitex, Yulianingsih. “The effects of phenetyl isothiocyanate and benzyl isothiocyanate on acetaminophen metabolism and toxicity in freshly isolated rat hepatocytes in cell suspension.” 1999. Thesis, Central Connecticut State University. Accessed June 19, 2019. http://content.library.ccsu.edu/u?/ccsutheses,2130.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Benitex, Yulianingsih. “The effects of phenetyl isothiocyanate and benzyl isothiocyanate on acetaminophen metabolism and toxicity in freshly isolated rat hepatocytes in cell suspension.” 1999. Web. 19 Jun 2019.

Vancouver:

Benitex Y. The effects of phenetyl isothiocyanate and benzyl isothiocyanate on acetaminophen metabolism and toxicity in freshly isolated rat hepatocytes in cell suspension. [Internet] [Thesis]. Central Connecticut State University; 1999. [cited 2019 Jun 19]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2130.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Benitex Y. The effects of phenetyl isothiocyanate and benzyl isothiocyanate on acetaminophen metabolism and toxicity in freshly isolated rat hepatocytes in cell suspension. [Thesis]. Central Connecticut State University; 1999. Available from: http://content.library.ccsu.edu/u?/ccsutheses,2130

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kansas State University

15. Nash, Sherrie LeRew. The effect of acetaminophen toxicity on selected blood biochemical parameters in the cat.

Degree: 1983, Kansas State University

Subjects/Keywords: Acetaminophen – Toxicology.; Cats – Physiology.; Veterinary hematology; Blood – Analysis.

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APA (6th Edition):

Nash, S. L. (1983). The effect of acetaminophen toxicity on selected blood biochemical parameters in the cat. (Thesis). Kansas State University. Retrieved from http://hdl.handle.net/2097/12987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nash, Sherrie LeRew. “The effect of acetaminophen toxicity on selected blood biochemical parameters in the cat.” 1983. Thesis, Kansas State University. Accessed June 19, 2019. http://hdl.handle.net/2097/12987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nash, Sherrie LeRew. “The effect of acetaminophen toxicity on selected blood biochemical parameters in the cat.” 1983. Web. 19 Jun 2019.

Vancouver:

Nash SL. The effect of acetaminophen toxicity on selected blood biochemical parameters in the cat. [Internet] [Thesis]. Kansas State University; 1983. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/2097/12987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nash SL. The effect of acetaminophen toxicity on selected blood biochemical parameters in the cat. [Thesis]. Kansas State University; 1983. Available from: http://hdl.handle.net/2097/12987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Sullivan, Bradley P. Role of Coagulation in Xenobiotic-Induced Liver Injury.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 The liver is a common target for xenobiotic-induced toxicity. Of importance, synthesis of soluble coagulation factors by the liver plays an essential role in hemostasis.… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alpha-naphthylisothiocyanate; Blood coagulation; Fibrosis; Liver injury; Platelet

…115 Fibrin(ogen)-Independent Role of Plasminogen Activators in Acetaminophen… …Alpha-naphthylisothiocyanate APAP Acetaminophen asTF alternatively spliced tissue factor αVβ6… 

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APA (6th Edition):

Sullivan, B. P. (2012). Role of Coagulation in Xenobiotic-Induced Liver Injury. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10291

Chicago Manual of Style (16th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/10291.

MLA Handbook (7th Edition):

Sullivan, Bradley P. “Role of Coagulation in Xenobiotic-Induced Liver Injury.” 2012. Web. 19 Jun 2019.

Vancouver:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/10291.

Council of Science Editors:

Sullivan BP. Role of Coagulation in Xenobiotic-Induced Liver Injury. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10291

17. McGill, Mitchell Ryan. Acetaminophen Hepatotoxicity in Humans and Mice.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2013, University of Kansas

Acetaminophen (APAP) is a popular analgesic and antipyretic. Most of a therapeutic dose is glucuronidated or sulfated and excreted. A small amount is converted by… (more)

Subjects/Keywords: Toxicology; Physiology; Pathology; Acetaminophen; Human; Liver; Mitochondria; Translational research

…Serum acylcarnitines as biomarkers of mitochondrial dysfunction after acetaminophen overdose… …liver acetaminophenprotein adduct levels in mice after acetaminophen treatment: dose-response… …Williams CD*, Xie Y, Ramachandran A, Jaeschke H. (2012) Acetaminophen-induced liver… …acetaminophen hepatotoxicity in humans and mice involve mitochondrial damage and nuclear DNA… …during clinical acetaminophen hepatotoxicity. J Hepatol. 56, 1070-9. XII 6) McGill MR… 

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APA (6th Edition):

McGill, M. R. (2013). Acetaminophen Hepatotoxicity in Humans and Mice. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/12178

Chicago Manual of Style (16th Edition):

McGill, Mitchell Ryan. “Acetaminophen Hepatotoxicity in Humans and Mice.” 2013. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/12178.

MLA Handbook (7th Edition):

McGill, Mitchell Ryan. “Acetaminophen Hepatotoxicity in Humans and Mice.” 2013. Web. 19 Jun 2019.

Vancouver:

McGill MR. Acetaminophen Hepatotoxicity in Humans and Mice. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/12178.

Council of Science Editors:

McGill MR. Acetaminophen Hepatotoxicity in Humans and Mice. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/12178


University of Otago

18. Owens, Katie Heather. Clinical pharmacology and toxicology of paracetamol in patient populations .

Degree: 2014, University of Otago

 Introduction: Paracetamol has been used as an analgesic and antipyretic drug in patient populations for decades. Recently, it has become available as an intravenous (IV)… (more)

Subjects/Keywords: clinical pharmacology; toxicology; pharmacy; paracetamol; acetaminophen; patient populations; clinical research; surgery; pharmacokinetics; pharmacodynamics; PKPD; modelling; Phoenix NLME; prothrombin time; INR; drug metabolism; Phoenix WinNonlin

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APA (6th Edition):

Owens, K. H. (2014). Clinical pharmacology and toxicology of paracetamol in patient populations . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/4675

Chicago Manual of Style (16th Edition):

Owens, Katie Heather. “Clinical pharmacology and toxicology of paracetamol in patient populations .” 2014. Doctoral Dissertation, University of Otago. Accessed June 19, 2019. http://hdl.handle.net/10523/4675.

MLA Handbook (7th Edition):

Owens, Katie Heather. “Clinical pharmacology and toxicology of paracetamol in patient populations .” 2014. Web. 19 Jun 2019.

Vancouver:

Owens KH. Clinical pharmacology and toxicology of paracetamol in patient populations . [Internet] [Doctoral dissertation]. University of Otago; 2014. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/10523/4675.

Council of Science Editors:

Owens KH. Clinical pharmacology and toxicology of paracetamol in patient populations . [Doctoral Dissertation]. University of Otago; 2014. Available from: http://hdl.handle.net/10523/4675

19. Ward, Jeanine. MicroRNA Markers of Acetaminophen Toxicity: A Master's Thesis.

Degree: MSin Clinical Investigation, Emergency Medicine, 2012, U of Massachusetts : Med

  Background To investigate plasma microRNA (miRNA) profiles indicative of hepatotoxicity in the setting of lethal acetaminophen (APAP) toxicity in mice. Methods Using plasma from… (more)

Subjects/Keywords: MicroRNAs; Acetaminophen; Animal Experimentation and Research; Digestive System; Nucleic Acids, Nucleotides, and Nucleosides; Organic Chemicals; Pharmaceutical Preparations; Pharmacology, Toxicology and Environmental Health; Therapeutics

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APA (6th Edition):

Ward, J. (2012). MicroRNA Markers of Acetaminophen Toxicity: A Master's Thesis. (Masters Thesis). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/625

Chicago Manual of Style (16th Edition):

Ward, Jeanine. “MicroRNA Markers of Acetaminophen Toxicity: A Master's Thesis.” 2012. Masters Thesis, U of Massachusetts : Med. Accessed June 19, 2019. https://escholarship.umassmed.edu/gsbs_diss/625.

MLA Handbook (7th Edition):

Ward, Jeanine. “MicroRNA Markers of Acetaminophen Toxicity: A Master's Thesis.” 2012. Web. 19 Jun 2019.

Vancouver:

Ward J. MicroRNA Markers of Acetaminophen Toxicity: A Master's Thesis. [Internet] [Masters thesis]. U of Massachusetts : Med; 2012. [cited 2019 Jun 19]. Available from: https://escholarship.umassmed.edu/gsbs_diss/625.

Council of Science Editors:

Ward J. MicroRNA Markers of Acetaminophen Toxicity: A Master's Thesis. [Masters Thesis]. U of Massachusetts : Med; 2012. Available from: https://escholarship.umassmed.edu/gsbs_diss/625

20. Woods, Sally. Comparison of metformin, rosiglitazone, and acetaminophen in the prevention of olanzapine toxicity in mice.

Degree: MS, Medicine: Toxicology (Environmental Health), 2011, University of Cincinnati

 BACKGROUND: Olanzapine (OLZ) is an atypical antipsychotic drug that induces weight gain and diabetogenic effects in a subset of patients. Conventional anti-diabetic drugs such as… (more)

Subjects/Keywords: Toxicology; metformin; acetaminophen; rosiglitazone; olanzapine; metabolic disease; mice

…and cardiovascular disease (32);(39). Acetaminophen Acetaminophen as an… …antioxidant Acetaminophen (APAP) is a commonly used analgesic and antipyretic. Its… …x28;33). 12 Potenital role of acetaminophen in type 2 diabetes treatment One… …diabetes in vivo (36). Potenital role of acetaminophen in olanzapine treatment In mice… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Woods, S. (2011). Comparison of metformin, rosiglitazone, and acetaminophen in the prevention of olanzapine toxicity in mice. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305892985

Chicago Manual of Style (16th Edition):

Woods, Sally. “Comparison of metformin, rosiglitazone, and acetaminophen in the prevention of olanzapine toxicity in mice.” 2011. Masters Thesis, University of Cincinnati. Accessed June 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305892985.

MLA Handbook (7th Edition):

Woods, Sally. “Comparison of metformin, rosiglitazone, and acetaminophen in the prevention of olanzapine toxicity in mice.” 2011. Web. 19 Jun 2019.

Vancouver:

Woods S. Comparison of metformin, rosiglitazone, and acetaminophen in the prevention of olanzapine toxicity in mice. [Internet] [Masters thesis]. University of Cincinnati; 2011. [cited 2019 Jun 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305892985.

Council of Science Editors:

Woods S. Comparison of metformin, rosiglitazone, and acetaminophen in the prevention of olanzapine toxicity in mice. [Masters Thesis]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305892985


University of North Carolina – Greensboro

21. Tan, Xiao. Analysis of ginger root essential oil and hydrosol: CYP450 inhibition.

Degree: 2015, University of North Carolina – Greensboro

 Former researches showed ginger extracts could contribute to hepatic protection. Two species of cytochrome P450 enzymes, CYP2E1 and CYP2A6, are both important heme-containing liver enzymes… (more)

Subjects/Keywords: Cytochrome P-450 – Inhibitors; Cytochrome P-450 CYP2E1 – Inhibitors; Enzyme inhibitors; Ginger – Physiological effect; Essences and essential oils – Physiological effect; Acetaminophen – Toxicology; Hepatotoxicology

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APA (6th Edition):

Tan, X. (2015). Analysis of ginger root essential oil and hydrosol: CYP450 inhibition. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=19129

Chicago Manual of Style (16th Edition):

Tan, Xiao. “Analysis of ginger root essential oil and hydrosol: CYP450 inhibition.” 2015. Masters Thesis, University of North Carolina – Greensboro. Accessed June 19, 2019. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=19129.

MLA Handbook (7th Edition):

Tan, Xiao. “Analysis of ginger root essential oil and hydrosol: CYP450 inhibition.” 2015. Web. 19 Jun 2019.

Vancouver:

Tan X. Analysis of ginger root essential oil and hydrosol: CYP450 inhibition. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2015. [cited 2019 Jun 19]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=19129.

Council of Science Editors:

Tan X. Analysis of ginger root essential oil and hydrosol: CYP450 inhibition. [Masters Thesis]. University of North Carolina – Greensboro; 2015. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=19129


University of Adelaide

22. Nicholls-Grzemski, Felicity April. The effect of short-term pretreatment with peroxisome proliferators on the acute toxicity of various toxicants, including paracetamol / Felicity April Nicholls-Grzemski.

Degree: 1998, University of Adelaide

Shows that pretreatment with peroxisome proliferators protects mice against the acute hepatotoxicity of paracetamol, in addition to a number of other toxicants. Advisors/Committee Members: Dept. of Clinical and Experimental Pharmacology (school).

Subjects/Keywords: Acetaminophen Toxicology.; Liver cells Effect of drugs on.; Toxicity testing In vitro.

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APA (6th Edition):

Nicholls-Grzemski, F. A. (1998). The effect of short-term pretreatment with peroxisome proliferators on the acute toxicity of various toxicants, including paracetamol / Felicity April Nicholls-Grzemski. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/19434

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nicholls-Grzemski, Felicity April. “The effect of short-term pretreatment with peroxisome proliferators on the acute toxicity of various toxicants, including paracetamol / Felicity April Nicholls-Grzemski.” 1998. Thesis, University of Adelaide. Accessed June 19, 2019. http://hdl.handle.net/2440/19434.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nicholls-Grzemski, Felicity April. “The effect of short-term pretreatment with peroxisome proliferators on the acute toxicity of various toxicants, including paracetamol / Felicity April Nicholls-Grzemski.” 1998. Web. 19 Jun 2019.

Vancouver:

Nicholls-Grzemski FA. The effect of short-term pretreatment with peroxisome proliferators on the acute toxicity of various toxicants, including paracetamol / Felicity April Nicholls-Grzemski. [Internet] [Thesis]. University of Adelaide; 1998. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/2440/19434.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nicholls-Grzemski FA. The effect of short-term pretreatment with peroxisome proliferators on the acute toxicity of various toxicants, including paracetamol / Felicity April Nicholls-Grzemski. [Thesis]. University of Adelaide; 1998. Available from: http://hdl.handle.net/2440/19434

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

23. Reisman, Scott Aaron. PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2008, University of Kansas

 Nuclear factor erythroid 2-related factor 2 (Nrf2) is a transcription factor, which, upon translocation into the nucleus, is capable of inducing a variety of cytoprotective… (more)

Subjects/Keywords: Health sciences; Toxicology; Pharmacology; Acetaminophen; Electrophilic stress; Glutathione-s-transferases; Nad(p)h:quinone oxidoreductase; Nrf2; Oxidative stress

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APA (6th Edition):

Reisman, S. A. (2008). PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/4325

Chicago Manual of Style (16th Edition):

Reisman, Scott Aaron. “PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS.” 2008. Doctoral Dissertation, University of Kansas. Accessed June 19, 2019. http://hdl.handle.net/1808/4325.

MLA Handbook (7th Edition):

Reisman, Scott Aaron. “PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS.” 2008. Web. 19 Jun 2019.

Vancouver:

Reisman SA. PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS. [Internet] [Doctoral dissertation]. University of Kansas; 2008. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/1808/4325.

Council of Science Editors:

Reisman SA. PHARMACOLOGIC AND TRANSGENIC ACTIVATION OF NUCLEAR FACTOR-ERYTHROID 2-RELATED FACTOR 2 (NRF2) ALTERS KINETICS AND TOXICODYNAMICS OF XENOBIOTICS. [Doctoral Dissertation]. University of Kansas; 2008. Available from: http://hdl.handle.net/1808/4325


University of Adelaide

24. Bruschi, Sam A. (Sam Anthony). Investigations into mechanisms of paracetamol-induced toxicity using in vitro' systems / by Sam A. Bruschi.

Degree: 1987, University of Adelaide

Subjects/Keywords: 615.907 20; Acetaminophen Toxicology.; Liver cells Effect of drugs on.; Toxicity testing In vitro.

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APA (6th Edition):

Bruschi, S. A. (. A. (1987). Investigations into mechanisms of paracetamol-induced toxicity using in vitro' systems / by Sam A. Bruschi. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/18564

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bruschi, Sam A (Sam Anthony). “Investigations into mechanisms of paracetamol-induced toxicity using in vitro' systems / by Sam A. Bruschi.” 1987. Thesis, University of Adelaide. Accessed June 19, 2019. http://hdl.handle.net/2440/18564.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bruschi, Sam A (Sam Anthony). “Investigations into mechanisms of paracetamol-induced toxicity using in vitro' systems / by Sam A. Bruschi.” 1987. Web. 19 Jun 2019.

Vancouver:

Bruschi SA(A. Investigations into mechanisms of paracetamol-induced toxicity using in vitro' systems / by Sam A. Bruschi. [Internet] [Thesis]. University of Adelaide; 1987. [cited 2019 Jun 19]. Available from: http://hdl.handle.net/2440/18564.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bruschi SA(A. Investigations into mechanisms of paracetamol-induced toxicity using in vitro' systems / by Sam A. Bruschi. [Thesis]. University of Adelaide; 1987. Available from: http://hdl.handle.net/2440/18564

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Marshall University

25. Harmon, R. Christopher. A Mechanistic Study in the Nephrotoxicity of p-Aminophenol.

Degree: 2003, Marshall University

 The acetaminophen metabolite, p-aminophenol (PAP), is a known nephrotoxicant. The current study is directed at understanding the mechanism of this nephrotoxicity. Renal cortical slices isolated… (more)

Subjects/Keywords: <; p>; Acetaminophen - Toxicology.<; /p>; <; p>; Nephrotoxicology.<; /p>;

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APA (6th Edition):

Harmon, R. C. (2003). A Mechanistic Study in the Nephrotoxicity of p-Aminophenol. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harmon, R Christopher. “A Mechanistic Study in the Nephrotoxicity of p-Aminophenol.” 2003. Thesis, Marshall University. Accessed June 19, 2019. http://mds.marshall.edu/etd/625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harmon, R Christopher. “A Mechanistic Study in the Nephrotoxicity of p-Aminophenol.” 2003. Web. 19 Jun 2019.

Vancouver:

Harmon RC. A Mechanistic Study in the Nephrotoxicity of p-Aminophenol. [Internet] [Thesis]. Marshall University; 2003. [cited 2019 Jun 19]. Available from: http://mds.marshall.edu/etd/625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Harmon RC. A Mechanistic Study in the Nephrotoxicity of p-Aminophenol. [Thesis]. Marshall University; 2003. Available from: http://mds.marshall.edu/etd/625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Marshall University

26. Meduru, Sarath. Chronic Acetaminophen Treatment Influences Indices of Reactive Oxygen Species Accumulation in the Aging Fisher 344 X Brown Norway rat Aorta.

Degree: 2007, Marshall University

 We have previously reported that aging in the Fisher 344 X Brown Norway (FBN) rat aorta is characterized by increased levels of ROS and alterations… (more)

Subjects/Keywords: aging; aorta; acetaminophen; ROS; cell signaling; <; p>; Aorta  – Aging.<; /p>; <; p>; Acetaminophen  – Toxicology.<; /p>;

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APA (6th Edition):

Meduru, S. (2007). Chronic Acetaminophen Treatment Influences Indices of Reactive Oxygen Species Accumulation in the Aging Fisher 344 X Brown Norway rat Aorta. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/764

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Meduru, Sarath. “Chronic Acetaminophen Treatment Influences Indices of Reactive Oxygen Species Accumulation in the Aging Fisher 344 X Brown Norway rat Aorta.” 2007. Thesis, Marshall University. Accessed June 19, 2019. http://mds.marshall.edu/etd/764.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Meduru, Sarath. “Chronic Acetaminophen Treatment Influences Indices of Reactive Oxygen Species Accumulation in the Aging Fisher 344 X Brown Norway rat Aorta.” 2007. Web. 19 Jun 2019.

Vancouver:

Meduru S. Chronic Acetaminophen Treatment Influences Indices of Reactive Oxygen Species Accumulation in the Aging Fisher 344 X Brown Norway rat Aorta. [Internet] [Thesis]. Marshall University; 2007. [cited 2019 Jun 19]. Available from: http://mds.marshall.edu/etd/764.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Meduru S. Chronic Acetaminophen Treatment Influences Indices of Reactive Oxygen Species Accumulation in the Aging Fisher 344 X Brown Norway rat Aorta. [Thesis]. Marshall University; 2007. Available from: http://mds.marshall.edu/etd/764

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Brown, James Michael. A Mechanistic Study of S-Adenosyl-L-methionine Protection Against Acetaminophen Hepatotoxicity.

Degree: 2012, Marshall University

Acetaminophen (APAP) toxicity remains the leading cause of drug induced liver failure in the United States. The current therapy for APAP toxicity is N-acetylcysteine (NAC).… (more)

Subjects/Keywords: 4-Hydroxynonenal; Acetaminophen; Hepatotoxicity; Oxidative Stress; S-Adenosylhomocysteine; S-Adenosyl-L-methionine; <; p>; <; strong>; <; /strong>; Acetaminophen - Overdose.<; /p>; <; p>; Acetaminophen - Toxicology<; /p>;

…108 Chapter V: S-Adenosyl-L-methionine protection of acetaminophen mediated oxidative stress… …Analysis of Variance APAP….Acetaminophen APAP-SG…GSH:APAP conjugate ASK1…Apoptosis Signal… …A…N-acetylcysteine given 1 hour after Acetaminophen NAC…N-acetylcysteine NADPH…Nicotine… …after Acetaminophen SAH…S-Adenosyl-L-homocysteine SAMC…S-Adenosyl-L-methionine mitochondrial… …Necrosis Factor UDPGA…Uridine Diphosphate-Glucuronic Acid xv Abstract Acetaminophen (APAP… 

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APA (6th Edition):

Brown, J. M. (2012). A Mechanistic Study of S-Adenosyl-L-methionine Protection Against Acetaminophen Hepatotoxicity. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brown, James Michael. “A Mechanistic Study of S-Adenosyl-L-methionine Protection Against Acetaminophen Hepatotoxicity.” 2012. Thesis, Marshall University. Accessed June 19, 2019. http://mds.marshall.edu/etd/347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brown, James Michael. “A Mechanistic Study of S-Adenosyl-L-methionine Protection Against Acetaminophen Hepatotoxicity.” 2012. Web. 19 Jun 2019.

Vancouver:

Brown JM. A Mechanistic Study of S-Adenosyl-L-methionine Protection Against Acetaminophen Hepatotoxicity. [Internet] [Thesis]. Marshall University; 2012. [cited 2019 Jun 19]. Available from: http://mds.marshall.edu/etd/347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brown JM. A Mechanistic Study of S-Adenosyl-L-methionine Protection Against Acetaminophen Hepatotoxicity. [Thesis]. Marshall University; 2012. Available from: http://mds.marshall.edu/etd/347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.