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You searched for subject:(AMPK). Showing records 1 – 30 of 291 total matches.

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Johannes Gutenberg Universität Mainz

1. Schuhmacher, Swenja. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation.

Degree: 2010, Johannes Gutenberg Universität Mainz

Die AMPK ist ein ubiquitär exprimiertes, heterotrimeres Enzym, das bei Energiemangel das Überleben der Zelle sichert. Um diese Funktion ausüben zu können fungiert die AMPK(more)

Subjects/Keywords: AMPK; AMPK; Natural sciences and mathematics

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APA (6th Edition):

Schuhmacher, S. (2010). Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/

Chicago Manual of Style (16th Edition):

Schuhmacher, Swenja. “Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation.” 2010. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 01, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/.

MLA Handbook (7th Edition):

Schuhmacher, Swenja. “Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation.” 2010. Web. 01 Mar 2021.

Vancouver:

Schuhmacher S. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2010. [cited 2021 Mar 01]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/.

Council of Science Editors:

Schuhmacher S. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2010. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/


Boston University

2. Allen, Katherine. The effects of troglitazone and PMA on AMPK in HepG2 cells.

Degree: MS, Medical Sciences, 2016, Boston University

 Type 2 diabetes, as well as other metabolic diseases, is an increasing global health concern and many of the mechanisms of both the disease and… (more)

Subjects/Keywords: Endocrinology; AMPK; Troglitazone

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APA (6th Edition):

Allen, K. (2016). The effects of troglitazone and PMA on AMPK in HepG2 cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16783

Chicago Manual of Style (16th Edition):

Allen, Katherine. “The effects of troglitazone and PMA on AMPK in HepG2 cells.” 2016. Masters Thesis, Boston University. Accessed March 01, 2021. http://hdl.handle.net/2144/16783.

MLA Handbook (7th Edition):

Allen, Katherine. “The effects of troglitazone and PMA on AMPK in HepG2 cells.” 2016. Web. 01 Mar 2021.

Vancouver:

Allen K. The effects of troglitazone and PMA on AMPK in HepG2 cells. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2144/16783.

Council of Science Editors:

Allen K. The effects of troglitazone and PMA on AMPK in HepG2 cells. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16783

3. 정, 재원. Roles of AMPK in Drosophila circadian clock.

Degree: 2015, Ajou University

국문요약 ⅰ 차례 ⅳ 그림 차례 ⅵ 표 차례 ⅶ Ⅰ. 서론 1 Ⅱ. 재료 및 방법 5 A. Schneider 2 (S2) 세포에서 발현시키기 위한… (more)

Subjects/Keywords: circadian clock; AMPK

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APA (6th Edition):

정, . (2015). Roles of AMPK in Drosophila circadian clock. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 재원. “Roles of AMPK in Drosophila circadian clock.” 2015. Thesis, Ajou University. Accessed March 01, 2021. http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 재원. “Roles of AMPK in Drosophila circadian clock.” 2015. Web. 01 Mar 2021.

Vancouver:

정 . Roles of AMPK in Drosophila circadian clock. [Internet] [Thesis]. Ajou University; 2015. [cited 2021 Mar 01]. Available from: http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . Roles of AMPK in Drosophila circadian clock. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

4. Day, Emily Anne. Mechanisms By Which Glucose Lowering Therapies Reduce Obesity And Atherosclerosis.

Degree: PhD, 2020, McMaster University

The incidence of obesity, type 2 diabetes and cardiovascular disease (CVD) is increasing at alarming rates worldwide. Obesity is associated with a chronic nutrient surplus… (more)

Subjects/Keywords: Diabetes; Obesity; Atherosclerosis; AMPK

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APA (6th Edition):

Day, E. A. (2020). Mechanisms By Which Glucose Lowering Therapies Reduce Obesity And Atherosclerosis. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/25312

Chicago Manual of Style (16th Edition):

Day, Emily Anne. “Mechanisms By Which Glucose Lowering Therapies Reduce Obesity And Atherosclerosis.” 2020. Doctoral Dissertation, McMaster University. Accessed March 01, 2021. http://hdl.handle.net/11375/25312.

MLA Handbook (7th Edition):

Day, Emily Anne. “Mechanisms By Which Glucose Lowering Therapies Reduce Obesity And Atherosclerosis.” 2020. Web. 01 Mar 2021.

Vancouver:

Day EA. Mechanisms By Which Glucose Lowering Therapies Reduce Obesity And Atherosclerosis. [Internet] [Doctoral dissertation]. McMaster University; 2020. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/11375/25312.

Council of Science Editors:

Day EA. Mechanisms By Which Glucose Lowering Therapies Reduce Obesity And Atherosclerosis. [Doctoral Dissertation]. McMaster University; 2020. Available from: http://hdl.handle.net/11375/25312


Texas A&M University

5. Jing, Ji. ATM and AMPK Regulate Peroxisome Dynamics via PEX5 Phosphorylation in Response to Stress.

Degree: PhD, Biomedical Sciences, 2018, Texas A&M University

 Peroxisomes are single membrane-bound and autonomously replicating subcellular organelles. While their function as metabolic organelles is well known, their place in the world of cell… (more)

Subjects/Keywords: Peroxisome dynamics; ATM; AMPK

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APA (6th Edition):

Jing, J. (2018). ATM and AMPK Regulate Peroxisome Dynamics via PEX5 Phosphorylation in Response to Stress. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/173559

Chicago Manual of Style (16th Edition):

Jing, Ji. “ATM and AMPK Regulate Peroxisome Dynamics via PEX5 Phosphorylation in Response to Stress.” 2018. Doctoral Dissertation, Texas A&M University. Accessed March 01, 2021. http://hdl.handle.net/1969.1/173559.

MLA Handbook (7th Edition):

Jing, Ji. “ATM and AMPK Regulate Peroxisome Dynamics via PEX5 Phosphorylation in Response to Stress.” 2018. Web. 01 Mar 2021.

Vancouver:

Jing J. ATM and AMPK Regulate Peroxisome Dynamics via PEX5 Phosphorylation in Response to Stress. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1969.1/173559.

Council of Science Editors:

Jing J. ATM and AMPK Regulate Peroxisome Dynamics via PEX5 Phosphorylation in Response to Stress. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/173559


University of Dundee

6. Auciello, Francesca Romana. Canonical and non-canonical regulation of AMP-activated protein kinase.

Degree: PhD, 2015, University of Dundee

 The AMP-activated protein kinase (AMPK) is a sensor of cellular energy stress that, once activated, promotes ATP-producing process while it switches off ATP-consuming pathways, in… (more)

Subjects/Keywords: 616; AMPK; Oxidative stress

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APA (6th Edition):

Auciello, F. R. (2015). Canonical and non-canonical regulation of AMP-activated protein kinase. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087

Chicago Manual of Style (16th Edition):

Auciello, Francesca Romana. “Canonical and non-canonical regulation of AMP-activated protein kinase.” 2015. Doctoral Dissertation, University of Dundee. Accessed March 01, 2021. https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087.

MLA Handbook (7th Edition):

Auciello, Francesca Romana. “Canonical and non-canonical regulation of AMP-activated protein kinase.” 2015. Web. 01 Mar 2021.

Vancouver:

Auciello FR. Canonical and non-canonical regulation of AMP-activated protein kinase. [Internet] [Doctoral dissertation]. University of Dundee; 2015. [cited 2021 Mar 01]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087.

Council of Science Editors:

Auciello FR. Canonical and non-canonical regulation of AMP-activated protein kinase. [Doctoral Dissertation]. University of Dundee; 2015. Available from: https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087


IUPUI

7. Steele, Hannah E. Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration.

Degree: 2018, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The biophysical microenvironment of the tumor site has significant impact on breast cancer progression and metastasis. The importance of altered… (more)

Subjects/Keywords: Mechanotransduction; AMPK; Breast Cancer; Migration

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APA (6th Edition):

Steele, H. E. (2018). Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/15928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steele, Hannah E. “Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration.” 2018. Thesis, IUPUI. Accessed March 01, 2021. http://hdl.handle.net/1805/15928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steele, Hannah E. “Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration.” 2018. Web. 01 Mar 2021.

Vancouver:

Steele HE. Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration. [Internet] [Thesis]. IUPUI; 2018. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1805/15928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steele HE. Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration. [Thesis]. IUPUI; 2018. Available from: http://hdl.handle.net/1805/15928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

8. Ybanez, Maria Cecilia D. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Our previous studies have shown that acetaminophen (APAP)-induced hepatocyte necrosis is mediated by JNK. In the present study we show that protein kinase C (PKC)… (more)

Subjects/Keywords: acetaminophen (APAP); AMPK; p70S6K; necrosis

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APA (6th Edition):

Ybanez, M. C. D. (2013). Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301

Chicago Manual of Style (16th Edition):

Ybanez, Maria Cecilia D. “Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.” 2013. Masters Thesis, University of Southern California. Accessed March 01, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301.

MLA Handbook (7th Edition):

Ybanez, Maria Cecilia D. “Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.” 2013. Web. 01 Mar 2021.

Vancouver:

Ybanez MCD. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Mar 01]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301.

Council of Science Editors:

Ybanez MCD. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5301

9. Hurtado de Llera, Ana. Expresión y función de la proteína quinasa activada por AMP, AMPK, en la célula germinal masculina de cerdo .

Degree: 2014, Universidad de Extremadura

 El espermatozoide es una célula germinal altamente especializada que debe experimentar unos procesos funcionales característicos para lograr su función primordial: la fecundación del ovocito. Estos… (more)

Subjects/Keywords: Quinasa; AMPK; Espermatozoide; Kinase; Spermatozoa

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APA (6th Edition):

Hurtado de Llera, A. (2014). Expresión y función de la proteína quinasa activada por AMP, AMPK, en la célula germinal masculina de cerdo . (Thesis). Universidad de Extremadura. Retrieved from http://hdl.handle.net/10662/1357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hurtado de Llera, Ana. “Expresión y función de la proteína quinasa activada por AMP, AMPK, en la célula germinal masculina de cerdo .” 2014. Thesis, Universidad de Extremadura. Accessed March 01, 2021. http://hdl.handle.net/10662/1357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hurtado de Llera, Ana. “Expresión y función de la proteína quinasa activada por AMP, AMPK, en la célula germinal masculina de cerdo .” 2014. Web. 01 Mar 2021.

Vancouver:

Hurtado de Llera A. Expresión y función de la proteína quinasa activada por AMP, AMPK, en la célula germinal masculina de cerdo . [Internet] [Thesis]. Universidad de Extremadura; 2014. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10662/1357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hurtado de Llera A. Expresión y función de la proteína quinasa activada por AMP, AMPK, en la célula germinal masculina de cerdo . [Thesis]. Universidad de Extremadura; 2014. Available from: http://hdl.handle.net/10662/1357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

10. Snider, Trevor. Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish .

Degree: Biology, 2013, Queens University

 The production of ATP is of the utmost importance to cell survival. To maintain energy homeostasis, cells regulate mitochondrial content through the control of degradative… (more)

Subjects/Keywords: PGC1 ; AMPK ; Animal physiology

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APA (6th Edition):

Snider, T. (2013). Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Snider, Trevor. “Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish .” 2013. Thesis, Queens University. Accessed March 01, 2021. http://hdl.handle.net/1974/8254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Snider, Trevor. “Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish .” 2013. Web. 01 Mar 2021.

Vancouver:

Snider T. Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish . [Internet] [Thesis]. Queens University; 2013. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1974/8254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Snider T. Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

11. Puthanveetil, Prasanth Nair. Glucocorticoid and its effect on cardiac glucose utilization.

Degree: MS- MSc, Pharmaceutical Sciences, 2008, University of British Columbia

 Glycogen is an immediate source of glucose for cardiac tissue to maintain its metabolic homeostasis. However, its excess brings about cardiac structural and physiological impairments.… (more)

Subjects/Keywords: AMPK; Glycogen

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APA (6th Edition):

Puthanveetil, P. N. (2008). Glucocorticoid and its effect on cardiac glucose utilization. (Masters Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/5038

Chicago Manual of Style (16th Edition):

Puthanveetil, Prasanth Nair. “Glucocorticoid and its effect on cardiac glucose utilization.” 2008. Masters Thesis, University of British Columbia. Accessed March 01, 2021. http://hdl.handle.net/2429/5038.

MLA Handbook (7th Edition):

Puthanveetil, Prasanth Nair. “Glucocorticoid and its effect on cardiac glucose utilization.” 2008. Web. 01 Mar 2021.

Vancouver:

Puthanveetil PN. Glucocorticoid and its effect on cardiac glucose utilization. [Internet] [Masters thesis]. University of British Columbia; 2008. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2429/5038.

Council of Science Editors:

Puthanveetil PN. Glucocorticoid and its effect on cardiac glucose utilization. [Masters Thesis]. University of British Columbia; 2008. Available from: http://hdl.handle.net/2429/5038

12. Mohsen Mroueh, Fatima. Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université de Strasbourg

Le diabète est une dérégulation systémique chronique caractérisée par des perturbations métaboliques permanentes à l’origine de nombreuses complications, y compris le cancer. Le diabète augmente… (more)

Subjects/Keywords: Diabète; CRC; ROS; AMPK; Diabetes; CRC; ROS; AMPK; 572.8; 616.99; 616.4

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APA (6th Edition):

Mohsen Mroueh, F. (2017). Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ117

Chicago Manual of Style (16th Edition):

Mohsen Mroueh, Fatima. “Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed March 01, 2021. http://www.theses.fr/2017STRAJ117.

MLA Handbook (7th Edition):

Mohsen Mroueh, Fatima. “Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies.” 2017. Web. 01 Mar 2021.

Vancouver:

Mohsen Mroueh F. Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2021 Mar 01]. Available from: http://www.theses.fr/2017STRAJ117.

Council of Science Editors:

Mohsen Mroueh F. Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ117

13. Desgeorges, Thibaut. Crosstalk of Glucocorticoid Receptor and AMP-activated protein kinase in macrophages during skeletal muscle regeneration : Interactions entre le Récepteur aux glucocorticoïdes et l'AMP-activated protein kinase dans les macrophages au cours de la régénération du muscle strié squelettique.

Degree: Docteur es, Biologie cellulaire, 2019, Lyon

Le muscle strié squelettique régénère ad integrum après une lésion aigüe stérile grâce aux cellules satellites qui sont les cellules souches du muscle strié squelettique.… (more)

Subjects/Keywords: Muscle; Régénération; Macrophages; AMPK; Glucocorticoïdes; Muscle; Regeneration; Macrophages; AMPK; Glucocorticoids; 570

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APA (6th Edition):

Desgeorges, T. (2019). Crosstalk of Glucocorticoid Receptor and AMP-activated protein kinase in macrophages during skeletal muscle regeneration : Interactions entre le Récepteur aux glucocorticoïdes et l'AMP-activated protein kinase dans les macrophages au cours de la régénération du muscle strié squelettique. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSE1058

Chicago Manual of Style (16th Edition):

Desgeorges, Thibaut. “Crosstalk of Glucocorticoid Receptor and AMP-activated protein kinase in macrophages during skeletal muscle regeneration : Interactions entre le Récepteur aux glucocorticoïdes et l'AMP-activated protein kinase dans les macrophages au cours de la régénération du muscle strié squelettique.” 2019. Doctoral Dissertation, Lyon. Accessed March 01, 2021. http://www.theses.fr/2019LYSE1058.

MLA Handbook (7th Edition):

Desgeorges, Thibaut. “Crosstalk of Glucocorticoid Receptor and AMP-activated protein kinase in macrophages during skeletal muscle regeneration : Interactions entre le Récepteur aux glucocorticoïdes et l'AMP-activated protein kinase dans les macrophages au cours de la régénération du muscle strié squelettique.” 2019. Web. 01 Mar 2021.

Vancouver:

Desgeorges T. Crosstalk of Glucocorticoid Receptor and AMP-activated protein kinase in macrophages during skeletal muscle regeneration : Interactions entre le Récepteur aux glucocorticoïdes et l'AMP-activated protein kinase dans les macrophages au cours de la régénération du muscle strié squelettique. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2021 Mar 01]. Available from: http://www.theses.fr/2019LYSE1058.

Council of Science Editors:

Desgeorges T. Crosstalk of Glucocorticoid Receptor and AMP-activated protein kinase in macrophages during skeletal muscle regeneration : Interactions entre le Récepteur aux glucocorticoïdes et l'AMP-activated protein kinase dans les macrophages au cours de la régénération du muscle strié squelettique. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSE1058

14. Gsaier, Linda. Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis.

Degree: Docteur es, Biologie cellulaire, 2018, Lyon

Durant la régénération du muscle suite à une lésion, les cellules souches musculaires, aussi appelées les cellules satellites, quittent leur état de quiescence et s’activent.… (more)

Subjects/Keywords: Muscle; Régénération; Métabolisme; Myogenèse; AMPK; Muscle; Regeneration; Metabolism; Myogenesis; AMPK; 570

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APA (6th Edition):

Gsaier, L. (2018). Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSE1208

Chicago Manual of Style (16th Edition):

Gsaier, Linda. “Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis.” 2018. Doctoral Dissertation, Lyon. Accessed March 01, 2021. http://www.theses.fr/2018LYSE1208.

MLA Handbook (7th Edition):

Gsaier, Linda. “Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis.” 2018. Web. 01 Mar 2021.

Vancouver:

Gsaier L. Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2021 Mar 01]. Available from: http://www.theses.fr/2018LYSE1208.

Council of Science Editors:

Gsaier L. Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSE1208


NSYSU

15. Chen, Shin-yi. The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer.

Degree: Master, Institute of Biomedical Sciences, 2011, NSYSU

 STK11/LKB1, a serine/threonine protein kinase, is a key upstream kinase of adenine monophosphate-activated protein kinase (AMPK), a necessary kinase in the control of metabolism for… (more)

Subjects/Keywords: Wnt pathway; LKB1; AMPK; PJS; hypoxia

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APA (6th Edition):

Chen, S. (2011). The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Shin-yi. “The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer.” 2011. Thesis, NSYSU. Accessed March 01, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Shin-yi. “The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer.” 2011. Web. 01 Mar 2021.

Vancouver:

Chen S. The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer. [Internet] [Thesis]. NSYSU; 2011. [cited 2021 Mar 01]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen S. The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

16. Lombardo, Portia Sachi. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.

Degree: Biology, 2017, University of California – San Diego

 Our laboratory recently identified the autophagy initiating kinase ULK1 as a novel AMPK substrate, which directly connects the highly conserved AMPK-mediated energy sensing pathway to… (more)

Subjects/Keywords: Molecular biology; AMPK; mitophagy; Parkin; ULK1

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APA (6th Edition):

Lombardo, P. S. (2017). Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/515006px

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lombardo, Portia Sachi. “Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.” 2017. Thesis, University of California – San Diego. Accessed March 01, 2021. http://www.escholarship.org/uc/item/515006px.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lombardo, Portia Sachi. “Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.” 2017. Web. 01 Mar 2021.

Vancouver:

Lombardo PS. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2021 Mar 01]. Available from: http://www.escholarship.org/uc/item/515006px.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lombardo PS. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/515006px

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Riverside

17. Marin, Traci. AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes.

Degree: Biochemistry and Molecular Biology, 2013, University of California – Riverside

 Mitochondria serve to regulate energy and redox homeostasis, ultimately determining cell fate. While mitochondrial dysfunction is characteristic of many metabolic, cardiovascular and neurodegenerative diseases, AMP-activated… (more)

Subjects/Keywords: Biochemistry; AMPK; chromatin; DNMT1; epigenetic; Mitochondria; transcription

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APA (6th Edition):

Marin, T. (2013). AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/5z46r2s4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marin, Traci. “AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes.” 2013. Thesis, University of California – Riverside. Accessed March 01, 2021. http://www.escholarship.org/uc/item/5z46r2s4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marin, Traci. “AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes.” 2013. Web. 01 Mar 2021.

Vancouver:

Marin T. AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes. [Internet] [Thesis]. University of California – Riverside; 2013. [cited 2021 Mar 01]. Available from: http://www.escholarship.org/uc/item/5z46r2s4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marin T. AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes. [Thesis]. University of California – Riverside; 2013. Available from: http://www.escholarship.org/uc/item/5z46r2s4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

18. Ehsan, Mehroz. Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction.

Degree: 2015, University of Toronto

Monocyte-derived microparticles (MPs) have been suggested to link cardiovascular risk factors to vascular injury. We hypothesized that adiponectin, an anti-inflammatory and vasculoprotective adipokine, limits the… (more)

Subjects/Keywords: Adiponectin; AMPK; inflammation; Microparticles; microvesicles; 0564

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APA (6th Edition):

Ehsan, M. (2015). Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71230

Chicago Manual of Style (16th Edition):

Ehsan, Mehroz. “Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction.” 2015. Masters Thesis, University of Toronto. Accessed March 01, 2021. http://hdl.handle.net/1807/71230.

MLA Handbook (7th Edition):

Ehsan, Mehroz. “Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction.” 2015. Web. 01 Mar 2021.

Vancouver:

Ehsan M. Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1807/71230.

Council of Science Editors:

Ehsan M. Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71230

19. 정, 종갑. Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice.

Degree: 2016, Ajou University

PART 1. TM-25659-induced activation of FGF21 level decreases insulin resistance and inflammation in skeletal muscle via GCN2 pathways 1 Ⅰ. INTRODUCTION 2 Ⅱ. MATERIALS AND… (more)

Subjects/Keywords: TAZ; TM-25659; insulin resistance; FGF21; AMPK

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APA (6th Edition):

정, . (2016). Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 종갑. “Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice.” 2016. Thesis, Ajou University. Accessed March 01, 2021. http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 종갑. “Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice.” 2016. Web. 01 Mar 2021.

Vancouver:

정 . Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice. [Internet] [Thesis]. Ajou University; 2016. [cited 2021 Mar 01]. Available from: http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice. [Thesis]. Ajou University; 2016. Available from: http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Texas

20. LaRue, Bobby Lee, Jr. Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans.

Degree: 2010, University of North Texas

 Ischemic events of even a very short duration are not tolerated Ill in humans. The human cost of ischemia, when looked at as combined cardiovascular… (more)

Subjects/Keywords: Oxygen; ischemia; AMPK

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APA (6th Edition):

LaRue, Bobby Lee, J. (2010). Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc103350/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LaRue, Bobby Lee, Jr. “Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans.” 2010. Thesis, University of North Texas. Accessed March 01, 2021. https://digital.library.unt.edu/ark:/67531/metadc103350/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LaRue, Bobby Lee, Jr. “Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans.” 2010. Web. 01 Mar 2021.

Vancouver:

LaRue, Bobby Lee J. Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans. [Internet] [Thesis]. University of North Texas; 2010. [cited 2021 Mar 01]. Available from: https://digital.library.unt.edu/ark:/67531/metadc103350/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LaRue, Bobby Lee J. Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans. [Thesis]. University of North Texas; 2010. Available from: https://digital.library.unt.edu/ark:/67531/metadc103350/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Colorado State University

21. Mudron, Megan Reese. Molecular regulation of growth and molting in decapod crustaceans.

Degree: MS(M.S.), Biology, 2014, Colorado State University

 The green shore crab, Carcinus maenas, is a highly invasive species that inhabits coastal temperate zones worldwide. The reaction of C. maenas to acute temperature… (more)

Subjects/Keywords: AMPK; crustacean; HSP70; molting; mTOR; myostatin

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APA (6th Edition):

Mudron, M. R. (2014). Molecular regulation of growth and molting in decapod crustaceans. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/84008

Chicago Manual of Style (16th Edition):

Mudron, Megan Reese. “Molecular regulation of growth and molting in decapod crustaceans.” 2014. Masters Thesis, Colorado State University. Accessed March 01, 2021. http://hdl.handle.net/10217/84008.

MLA Handbook (7th Edition):

Mudron, Megan Reese. “Molecular regulation of growth and molting in decapod crustaceans.” 2014. Web. 01 Mar 2021.

Vancouver:

Mudron MR. Molecular regulation of growth and molting in decapod crustaceans. [Internet] [Masters thesis]. Colorado State University; 2014. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10217/84008.

Council of Science Editors:

Mudron MR. Molecular regulation of growth and molting in decapod crustaceans. [Masters Thesis]. Colorado State University; 2014. Available from: http://hdl.handle.net/10217/84008


University of Ottawa

22. Al-Rewashdy, Hasanen. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .

Degree: 2014, University of Ottawa

 Duchenne Muscular Dystrophy (DMD) results from the absence of a functional dystrophin protein. Among its possible therapeutic options is the upregulation of dystrophin’s autosomal analogue,… (more)

Subjects/Keywords: Utrophin; Duchenne Muscular Dystrophy; Dystrophin; AMPK; AICAR

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APA (6th Edition):

Al-Rewashdy, H. (2014). Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Rewashdy, Hasanen. “Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .” 2014. Thesis, University of Ottawa. Accessed March 01, 2021. http://hdl.handle.net/10393/31470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Rewashdy, Hasanen. “Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .” 2014. Web. 01 Mar 2021.

Vancouver:

Al-Rewashdy H. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10393/31470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Rewashdy H. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

23. Dufeys, Cécile. Exploring the role of fibroblastic AMPKalpha1 in ventricular remodelling after myocardial infarction : implication of connexin 43 and miR-125b.

Degree: 2019, Université Catholique de Louvain

Myofibroblasts (MFs) are crucial components of the fibrotic remodelling after cardiac injury. In the present work, we investigate whether AMP-activated protein kinase α1 (AMPKα1) from… (more)

Subjects/Keywords: AMPK; Connexin 43; MiR-125b; Cardiac fibrosis

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APA (6th Edition):

Dufeys, C. (2019). Exploring the role of fibroblastic AMPKalpha1 in ventricular remodelling after myocardial infarction : implication of connexin 43 and miR-125b. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/216751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dufeys, Cécile. “Exploring the role of fibroblastic AMPKalpha1 in ventricular remodelling after myocardial infarction : implication of connexin 43 and miR-125b.” 2019. Thesis, Université Catholique de Louvain. Accessed March 01, 2021. http://hdl.handle.net/2078.1/216751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dufeys, Cécile. “Exploring the role of fibroblastic AMPKalpha1 in ventricular remodelling after myocardial infarction : implication of connexin 43 and miR-125b.” 2019. Web. 01 Mar 2021.

Vancouver:

Dufeys C. Exploring the role of fibroblastic AMPKalpha1 in ventricular remodelling after myocardial infarction : implication of connexin 43 and miR-125b. [Internet] [Thesis]. Université Catholique de Louvain; 2019. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2078.1/216751.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dufeys C. Exploring the role of fibroblastic AMPKalpha1 in ventricular remodelling after myocardial infarction : implication of connexin 43 and miR-125b. [Thesis]. Université Catholique de Louvain; 2019. Available from: http://hdl.handle.net/2078.1/216751

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

24. Mahmoud, Amira Dia. The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body.

Degree: PhD, 2015, University of Edinburgh

 Ventilatory drive is mediated by respiratory central pattern generators that are located in the brainstem, which are continuously modulated by specialised peripheral and central chemoreceptors… (more)

Subjects/Keywords: 616.2; hypoxia; Lkb1; AMPK; apnoeas; ventilatory response

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APA (6th Edition):

Mahmoud, A. D. (2015). The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17886

Chicago Manual of Style (16th Edition):

Mahmoud, Amira Dia. “The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed March 01, 2021. http://hdl.handle.net/1842/17886.

MLA Handbook (7th Edition):

Mahmoud, Amira Dia. “The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body.” 2015. Web. 01 Mar 2021.

Vancouver:

Mahmoud AD. The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1842/17886.

Council of Science Editors:

Mahmoud AD. The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/17886


University of Gothenburg / Göteborgs Universitet

25. Cansby, Emmelie. Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25.

Degree: 2014, University of Gothenburg / Göteborgs Universitet

 Type 2 diabetes mellitus (T2DM) is a widespread metabolic disorder that has reached epidemic proportions globally, and is now considered to be one of the… (more)

Subjects/Keywords: AMPK; STK25; Metabolism; Type 2 diabetes; Inflammation

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APA (6th Edition):

Cansby, E. (2014). Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/35938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cansby, Emmelie. “Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25.” 2014. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed March 01, 2021. http://hdl.handle.net/2077/35938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cansby, Emmelie. “Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25.” 2014. Web. 01 Mar 2021.

Vancouver:

Cansby E. Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2014. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2077/35938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cansby E. Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2014. Available from: http://hdl.handle.net/2077/35938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

26. Xu, Pingwen. Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight.

Degree: PhD, Animal and Poultry Sciences, 2011, Virginia Tech

 This study was to determine (1) if genetic selection for high (HWS) or low (LWS) body weight in chickens has altered the hypothalamic AMP-activated protein… (more)

Subjects/Keywords: ghrelin; food intake; chicken; AMPK; obestatin

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APA (6th Edition):

Xu, P. (2011). Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/37680

Chicago Manual of Style (16th Edition):

Xu, Pingwen. “Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight.” 2011. Doctoral Dissertation, Virginia Tech. Accessed March 01, 2021. http://hdl.handle.net/10919/37680.

MLA Handbook (7th Edition):

Xu, Pingwen. “Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight.” 2011. Web. 01 Mar 2021.

Vancouver:

Xu P. Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight. [Internet] [Doctoral dissertation]. Virginia Tech; 2011. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10919/37680.

Council of Science Editors:

Xu P. Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight. [Doctoral Dissertation]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/37680


University of Minnesota

27. Yan, Yan. Regulation and Function of the Phosphatase PHLPP2 in Leukemia.

Degree: PhD, Pharmacology, 2017, University of Minnesota

 PHLPP2, a member of the PHLPP phosphatase family, which targets oncogenic kinases, has been actively investigated as a tumor suppressor in solid tumors. Little was… (more)

Subjects/Keywords: AMPK; ATRA; leukemia; microRNA; PHLPP2; phosphatase

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APA (6th Edition):

Yan, Y. (2017). Regulation and Function of the Phosphatase PHLPP2 in Leukemia. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191472

Chicago Manual of Style (16th Edition):

Yan, Yan. “Regulation and Function of the Phosphatase PHLPP2 in Leukemia.” 2017. Doctoral Dissertation, University of Minnesota. Accessed March 01, 2021. http://hdl.handle.net/11299/191472.

MLA Handbook (7th Edition):

Yan, Yan. “Regulation and Function of the Phosphatase PHLPP2 in Leukemia.” 2017. Web. 01 Mar 2021.

Vancouver:

Yan Y. Regulation and Function of the Phosphatase PHLPP2 in Leukemia. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/11299/191472.

Council of Science Editors:

Yan Y. Regulation and Function of the Phosphatase PHLPP2 in Leukemia. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/191472


University of Melbourne

28. EMANUELLE, SHANE. The regulation of SnRK1 from Arabidopsis thaliana.

Degree: 2013, University of Melbourne

 An intrinsic characteristic of all life is the ability to regulate the organism’s internal energy status despite sometimes wildly fluctuating conditions in the external environment.… (more)

Subjects/Keywords: SnRK1; SNF1; AMPK; enzyme; biochemistry; botany

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APA (6th Edition):

EMANUELLE, S. (2013). The regulation of SnRK1 from Arabidopsis thaliana. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38299

Chicago Manual of Style (16th Edition):

EMANUELLE, SHANE. “The regulation of SnRK1 from Arabidopsis thaliana.” 2013. Doctoral Dissertation, University of Melbourne. Accessed March 01, 2021. http://hdl.handle.net/11343/38299.

MLA Handbook (7th Edition):

EMANUELLE, SHANE. “The regulation of SnRK1 from Arabidopsis thaliana.” 2013. Web. 01 Mar 2021.

Vancouver:

EMANUELLE S. The regulation of SnRK1 from Arabidopsis thaliana. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/11343/38299.

Council of Science Editors:

EMANUELLE S. The regulation of SnRK1 from Arabidopsis thaliana. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38299


University of Southern California

29. Shim, Hong Seok. Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis.

Degree: PhD, Integrative and Evolutionary Biology, 2016, University of Southern California

 Dietary or calorie restriction has been a well‐studied strategy for increasing survival and preventing carcinogenesis in mammals. Short‐term starvation, an extreme form of dietary restriction,… (more)

Subjects/Keywords: starvation; REV1; sumoylation; p53; EGR1; AMPK; PKA

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APA (6th Edition):

Shim, H. S. (2016). Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1987

Chicago Manual of Style (16th Edition):

Shim, Hong Seok. “Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis.” 2016. Doctoral Dissertation, University of Southern California. Accessed March 01, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1987.

MLA Handbook (7th Edition):

Shim, Hong Seok. “Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis.” 2016. Web. 01 Mar 2021.

Vancouver:

Shim HS. Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis. [Internet] [Doctoral dissertation]. University of Southern California; 2016. [cited 2021 Mar 01]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1987.

Council of Science Editors:

Shim HS. Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis. [Doctoral Dissertation]. University of Southern California; 2016. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1987


Univerzitet u Beogradu

30. Pantović, Aleksandar, 1982-, 59207177. Uloga adenozin-monofosfatom aktivirane protein-kinaze i mTOR kompleksa 1 u in vitro citotoksičnom dejstvu nesteroidnih anti-inflamatornih lekova na ćelije glioma.

Degree: Medicinski fakultet, 2020, Univerzitet u Beogradu

Medicina - Molekularna medicina / Medicine - Molecular medicine

Gliomi su najčešći primarni tumori centralnog nervnog sistema. Glioblastom je gliom gradusa IV, terapijski rezistentan maligni… (more)

Subjects/Keywords: glioma; NSAID; indomethacin; AMPK; mTOR; apoptosis; autophagy

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APA (6th Edition):

Pantović, Aleksandar, 1982-, 5. (2020). Uloga adenozin-monofosfatom aktivirane protein-kinaze i mTOR kompleksa 1 u in vitro citotoksičnom dejstvu nesteroidnih anti-inflamatornih lekova na ćelije glioma. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:23025/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pantović, Aleksandar, 1982-, 59207177. “Uloga adenozin-monofosfatom aktivirane protein-kinaze i mTOR kompleksa 1 u in vitro citotoksičnom dejstvu nesteroidnih anti-inflamatornih lekova na ćelije glioma.” 2020. Thesis, Univerzitet u Beogradu. Accessed March 01, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:23025/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pantović, Aleksandar, 1982-, 59207177. “Uloga adenozin-monofosfatom aktivirane protein-kinaze i mTOR kompleksa 1 u in vitro citotoksičnom dejstvu nesteroidnih anti-inflamatornih lekova na ćelije glioma.” 2020. Web. 01 Mar 2021.

Vancouver:

Pantović, Aleksandar, 1982- 5. Uloga adenozin-monofosfatom aktivirane protein-kinaze i mTOR kompleksa 1 u in vitro citotoksičnom dejstvu nesteroidnih anti-inflamatornih lekova na ćelije glioma. [Internet] [Thesis]. Univerzitet u Beogradu; 2020. [cited 2021 Mar 01]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:23025/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pantović, Aleksandar, 1982- 5. Uloga adenozin-monofosfatom aktivirane protein-kinaze i mTOR kompleksa 1 u in vitro citotoksičnom dejstvu nesteroidnih anti-inflamatornih lekova na ćelije glioma. [Thesis]. Univerzitet u Beogradu; 2020. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:23025/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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