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You searched for subject:(AMPK). Showing records 1 – 30 of 237 total matches.

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Johannes Gutenberg Universität Mainz

1. Schuhmacher, Swenja. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation.

Degree: 2010, Johannes Gutenberg Universität Mainz

Die AMPK ist ein ubiquitär exprimiertes, heterotrimeres Enzym, das bei Energiemangel das Überleben der Zelle sichert. Um diese Funktion ausüben zu können fungiert die AMPK(more)

Subjects/Keywords: AMPK; AMPK; Natural sciences and mathematics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schuhmacher, S. (2010). Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/

Chicago Manual of Style (16th Edition):

Schuhmacher, Swenja. “Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation.” 2010. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed June 24, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/.

MLA Handbook (7th Edition):

Schuhmacher, Swenja. “Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation.” 2010. Web. 24 Jun 2019.

Vancouver:

Schuhmacher S. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2010. [cited 2019 Jun 24]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/.

Council of Science Editors:

Schuhmacher S. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2010. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2010/2540/

2. 정, 재원. Roles of AMPK in Drosophila circadian clock.

Degree: 2015, Ajou University

국문요약 ⅰ 차례 ⅳ 그림 차례 ⅵ 표 차례 ⅶ Ⅰ. 서론 1 Ⅱ. 재료 및 방법 5 A. Schneider 2 (S2) 세포에서 발현시키기 위한… (more)

Subjects/Keywords: circadian clock; AMPK

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APA (6th Edition):

정, . (2015). Roles of AMPK in Drosophila circadian clock. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 재원. “Roles of AMPK in Drosophila circadian clock.” 2015. Thesis, Ajou University. Accessed June 24, 2019. http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 재원. “Roles of AMPK in Drosophila circadian clock.” 2015. Web. 24 Jun 2019.

Vancouver:

정 . Roles of AMPK in Drosophila circadian clock. [Internet] [Thesis]. Ajou University; 2015. [cited 2019 Jun 24]. Available from: http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . Roles of AMPK in Drosophila circadian clock. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11829 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

3. Allen, Katherine. The effects of troglitazone and PMA on AMPK in HepG2 cells.

Degree: MS, Medical Sciences, 2016, Boston University

 Type 2 diabetes, as well as other metabolic diseases, is an increasing global health concern and many of the mechanisms of both the disease and… (more)

Subjects/Keywords: Endocrinology; AMPK; Troglitazone

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APA (6th Edition):

Allen, K. (2016). The effects of troglitazone and PMA on AMPK in HepG2 cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16783

Chicago Manual of Style (16th Edition):

Allen, Katherine. “The effects of troglitazone and PMA on AMPK in HepG2 cells.” 2016. Masters Thesis, Boston University. Accessed June 24, 2019. http://hdl.handle.net/2144/16783.

MLA Handbook (7th Edition):

Allen, Katherine. “The effects of troglitazone and PMA on AMPK in HepG2 cells.” 2016. Web. 24 Jun 2019.

Vancouver:

Allen K. The effects of troglitazone and PMA on AMPK in HepG2 cells. [Internet] [Masters thesis]. Boston University; 2016. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/2144/16783.

Council of Science Editors:

Allen K. The effects of troglitazone and PMA on AMPK in HepG2 cells. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16783


University of North Texas

4. LaRue, Bobby Lee, Jr. Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans.

Degree: 2010, University of North Texas

 Ischemic events of even a very short duration are not tolerated Ill in humans. The human cost of ischemia, when looked at as combined cardiovascular… (more)

Subjects/Keywords: Oxygen; ischemia; AMPK

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APA (6th Edition):

LaRue, Bobby Lee, J. (2010). Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc103350/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LaRue, Bobby Lee, Jr. “Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans.” 2010. Thesis, University of North Texas. Accessed June 24, 2019. https://digital.library.unt.edu/ark:/67531/metadc103350/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LaRue, Bobby Lee, Jr. “Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans.” 2010. Web. 24 Jun 2019.

Vancouver:

LaRue, Bobby Lee J. Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans. [Internet] [Thesis]. University of North Texas; 2010. [cited 2019 Jun 24]. Available from: https://digital.library.unt.edu/ark:/67531/metadc103350/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LaRue, Bobby Lee J. Genetic and Environmental Factors that Mediate Survival of Prolonged Oxygen Deprivation in the Nematode Caenorhabditis Elegans. [Thesis]. University of North Texas; 2010. Available from: https://digital.library.unt.edu/ark:/67531/metadc103350/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

5. Auciello, Francesca Romana. Canonical and non-canonical regulation of AMP-activated protein kinase.

Degree: PhD, 2015, University of Dundee

 The AMP-activated protein kinase (AMPK) is a sensor of cellular energy stress that, once activated, promotes ATP-producing process while it switches off ATP-consuming pathways, in… (more)

Subjects/Keywords: 616; AMPK; Oxidative stress

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Auciello, F. R. (2015). Canonical and non-canonical regulation of AMP-activated protein kinase. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/2720a2b7-3f1e-445c-b008-c5c235f35395

Chicago Manual of Style (16th Edition):

Auciello, Francesca Romana. “Canonical and non-canonical regulation of AMP-activated protein kinase.” 2015. Doctoral Dissertation, University of Dundee. Accessed June 24, 2019. http://hdl.handle.net/10588/2720a2b7-3f1e-445c-b008-c5c235f35395.

MLA Handbook (7th Edition):

Auciello, Francesca Romana. “Canonical and non-canonical regulation of AMP-activated protein kinase.” 2015. Web. 24 Jun 2019.

Vancouver:

Auciello FR. Canonical and non-canonical regulation of AMP-activated protein kinase. [Internet] [Doctoral dissertation]. University of Dundee; 2015. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/10588/2720a2b7-3f1e-445c-b008-c5c235f35395.

Council of Science Editors:

Auciello FR. Canonical and non-canonical regulation of AMP-activated protein kinase. [Doctoral Dissertation]. University of Dundee; 2015. Available from: http://hdl.handle.net/10588/2720a2b7-3f1e-445c-b008-c5c235f35395


Queens University

6. Snider, Trevor. Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish .

Degree: Biology, 2013, Queens University

 The production of ATP is of the utmost importance to cell survival. To maintain energy homeostasis, cells regulate mitochondrial content through the control of degradative… (more)

Subjects/Keywords: PGC1; AMPK; Animal physiology

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APA (6th Edition):

Snider, T. (2013). Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Snider, Trevor. “Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish .” 2013. Thesis, Queens University. Accessed June 24, 2019. http://hdl.handle.net/1974/8254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Snider, Trevor. “Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish .” 2013. Web. 24 Jun 2019.

Vancouver:

Snider T. Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish . [Internet] [Thesis]. Queens University; 2013. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1974/8254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Snider T. Evaluating the Role of Peroxisome Proliferator-Activated Receptor Gama Coactivator-1 Alpha in Mitochondrial Biogenesis in Goldfish . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

7. Puthanveetil, Prasanth Nair. Glucocorticoid and its effect on cardiac glucose utilization .

Degree: 2008, University of British Columbia

 Glycogen is an immediate source of glucose for cardiac tissue to maintain its metabolic homeostasis. However, its excess brings about cardiac structural and physiological impairments.… (more)

Subjects/Keywords: AMPK; Glycogen

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APA (6th Edition):

Puthanveetil, P. N. (2008). Glucocorticoid and its effect on cardiac glucose utilization . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/5038

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Puthanveetil, Prasanth Nair. “Glucocorticoid and its effect on cardiac glucose utilization .” 2008. Thesis, University of British Columbia. Accessed June 24, 2019. http://hdl.handle.net/2429/5038.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Puthanveetil, Prasanth Nair. “Glucocorticoid and its effect on cardiac glucose utilization .” 2008. Web. 24 Jun 2019.

Vancouver:

Puthanveetil PN. Glucocorticoid and its effect on cardiac glucose utilization . [Internet] [Thesis]. University of British Columbia; 2008. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/2429/5038.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Puthanveetil PN. Glucocorticoid and its effect on cardiac glucose utilization . [Thesis]. University of British Columbia; 2008. Available from: http://hdl.handle.net/2429/5038

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

8. Auciello, Francesca Romana. Canonical and non-canonical regulation of AMP-activated protein kinase.

Degree: PhD, 2015, University of Dundee

 The AMP-activated protein kinase (AMPK) is a sensor of cellular energy stress that, once activated, promotes ATP-producing process while it switches off ATP-consuming pathways, in… (more)

Subjects/Keywords: 616; AMPK; Oxidative stress

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Auciello, F. R. (2015). Canonical and non-canonical regulation of AMP-activated protein kinase. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087

Chicago Manual of Style (16th Edition):

Auciello, Francesca Romana. “Canonical and non-canonical regulation of AMP-activated protein kinase.” 2015. Doctoral Dissertation, University of Dundee. Accessed June 24, 2019. https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087.

MLA Handbook (7th Edition):

Auciello, Francesca Romana. “Canonical and non-canonical regulation of AMP-activated protein kinase.” 2015. Web. 24 Jun 2019.

Vancouver:

Auciello FR. Canonical and non-canonical regulation of AMP-activated protein kinase. [Internet] [Doctoral dissertation]. University of Dundee; 2015. [cited 2019 Jun 24]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087.

Council of Science Editors:

Auciello FR. Canonical and non-canonical regulation of AMP-activated protein kinase. [Doctoral Dissertation]. University of Dundee; 2015. Available from: https://discovery.dundee.ac.uk/en/studentTheses/2720a2b7-3f1e-445c-b008-c5c235f35395 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679087

9. Ríos García, Marcos. Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis.

Degree: 2018, Universidad de Santiago de Compostela

Subjects/Keywords: Glioma; AMPK

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APA (6th Edition):

Ríos García, M. (2018). Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis. (Thesis). Universidad de Santiago de Compostela. Retrieved from http://hdl.handle.net/10347/3711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ríos García, Marcos. “Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis.” 2018. Thesis, Universidad de Santiago de Compostela. Accessed June 24, 2019. http://hdl.handle.net/10347/3711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ríos García, Marcos. “Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis.” 2018. Web. 24 Jun 2019.

Vancouver:

Ríos García M. Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis. [Internet] [Thesis]. Universidad de Santiago de Compostela; 2018. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/10347/3711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ríos García M. Papel de AMPK en la regulación del metabolismo y proliferación celular durante el proceso de tumorogénesis. [Thesis]. Universidad de Santiago de Compostela; 2018. Available from: http://hdl.handle.net/10347/3711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

10. Steele, Hannah E. Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration.

Degree: 2018, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The biophysical microenvironment of the tumor site has significant impact on breast cancer progression and metastasis. The importance of altered… (more)

Subjects/Keywords: Mechanotransduction; AMPK; Breast Cancer; Migration

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APA (6th Edition):

Steele, H. E. (2018). Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/15928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steele, Hannah E. “Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration.” 2018. Thesis, IUPUI. Accessed June 24, 2019. http://hdl.handle.net/1805/15928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steele, Hannah E. “Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration.” 2018. Web. 24 Jun 2019.

Vancouver:

Steele HE. Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration. [Internet] [Thesis]. IUPUI; 2018. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1805/15928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steele HE. Mechanotransduction of subcellular AMPK and its role in breast cancer cell migration. [Thesis]. IUPUI; 2018. Available from: http://hdl.handle.net/1805/15928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

11. Ybanez, Maria Cecilia D. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 Our previous studies have shown that acetaminophen (APAP)-induced hepatocyte necrosis is mediated by JNK. In the present study we show that protein kinase C (PKC)… (more)

Subjects/Keywords: acetaminophen (APAP); AMPK; p70S6K; necrosis

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APA (6th Edition):

Ybanez, M. C. D. (2013). Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5294

Chicago Manual of Style (16th Edition):

Ybanez, Maria Cecilia D. “Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.” 2013. Masters Thesis, University of Southern California. Accessed June 24, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5294.

MLA Handbook (7th Edition):

Ybanez, Maria Cecilia D. “Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways.” 2013. Web. 24 Jun 2019.

Vancouver:

Ybanez MCD. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Jun 24]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5294.

Council of Science Editors:

Ybanez MCD. Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through JNK dependent and independent pathways. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/247992/rec/5294

12. Mohsen Mroueh, Fatima. Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université de Strasbourg

Le diabète est une dérégulation systémique chronique caractérisée par des perturbations métaboliques permanentes à l’origine de nombreuses complications, y compris le cancer. Le diabète augmente… (more)

Subjects/Keywords: Diabète; CRC; ROS; AMPK; Diabetes; CRC; ROS; AMPK; 572.8; 616.99; 616.4

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APA (6th Edition):

Mohsen Mroueh, F. (2017). Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ117

Chicago Manual of Style (16th Edition):

Mohsen Mroueh, Fatima. “Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed June 24, 2019. http://www.theses.fr/2017STRAJ117.

MLA Handbook (7th Edition):

Mohsen Mroueh, Fatima. “Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies.” 2017. Web. 24 Jun 2019.

Vancouver:

Mohsen Mroueh F. Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2019 Jun 24]. Available from: http://www.theses.fr/2017STRAJ117.

Council of Science Editors:

Mohsen Mroueh F. Diabète et cancer colorectal : épidémiologie et physiopathologie : Diabetes and colorectal cancer : epidemiological and physiopathological studies. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ117

13. Gsaier, Linda. Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis.

Degree: Docteur es, Biologie cellulaire, 2018, Lyon

Durant la régénération du muscle suite à une lésion, les cellules souches musculaires, aussi appelées les cellules satellites, quittent leur état de quiescence et s’activent.… (more)

Subjects/Keywords: Muscle; Régénération; Métabolisme; Myogenèse; AMPK; Muscle; Regeneration; Metabolism; Myogenesis; AMPK; 570

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APA (6th Edition):

Gsaier, L. (2018). Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSE1208

Chicago Manual of Style (16th Edition):

Gsaier, Linda. “Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis.” 2018. Doctoral Dissertation, Lyon. Accessed June 24, 2019. http://www.theses.fr/2018LYSE1208.

MLA Handbook (7th Edition):

Gsaier, Linda. “Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis.” 2018. Web. 24 Jun 2019.

Vancouver:

Gsaier L. Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2019 Jun 24]. Available from: http://www.theses.fr/2018LYSE1208.

Council of Science Editors:

Gsaier L. Rôle du métabolisme sur le devenir des cellules souches musculaires et l'homéostasie du muscle squelettique : Role of cell-autonomous regulation of metabolism on muscle stem cell fate and skeletal muscle homeostasis. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSE1208


NSYSU

14. Chen, Shin-yi. The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer.

Degree: Master, Institute of Biomedical Sciences, 2011, NSYSU

 STK11/LKB1, a serine/threonine protein kinase, is a key upstream kinase of adenine monophosphate-activated protein kinase (AMPK), a necessary kinase in the control of metabolism for… (more)

Subjects/Keywords: Wnt pathway; LKB1; AMPK; PJS; hypoxia

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APA (6th Edition):

Chen, S. (2011). The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Shin-yi. “The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer.” 2011. Thesis, NSYSU. Accessed June 24, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Shin-yi. “The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer.” 2011. Web. 24 Jun 2019.

Vancouver:

Chen S. The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer. [Internet] [Thesis]. NSYSU; 2011. [cited 2019 Jun 24]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen S. The role of LKB1 in the regulation of energetic checkpoints and DNA damage in the lung cancer. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-135626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

15. Cansby, Emmelie. Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25.

Degree: 2014, University of Gothenburg / Göteborgs Universitet

 Type 2 diabetes mellitus (T2DM) is a widespread metabolic disorder that has reached epidemic proportions globally, and is now considered to be one of the… (more)

Subjects/Keywords: AMPK; STK25; Metabolism; Type 2 diabetes; Inflammation

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APA (6th Edition):

Cansby, E. (2014). Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/35938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cansby, Emmelie. “Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25.” 2014. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed June 24, 2019. http://hdl.handle.net/2077/35938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cansby, Emmelie. “Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25.” 2014. Web. 24 Jun 2019.

Vancouver:

Cansby E. Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2014. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/2077/35938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cansby E. Regulation of metabolism and inflammation by two protein kinases - AMPK and STK25. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2014. Available from: http://hdl.handle.net/2077/35938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

16. Ehsan, Mehroz. Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction.

Degree: 2015, University of Toronto

Monocyte-derived microparticles (MPs) have been suggested to link cardiovascular risk factors to vascular injury. We hypothesized that adiponectin, an anti-inflammatory and vasculoprotective adipokine, limits the… (more)

Subjects/Keywords: Adiponectin; AMPK; inflammation; Microparticles; microvesicles; 0564

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APA (6th Edition):

Ehsan, M. (2015). Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71230

Chicago Manual of Style (16th Edition):

Ehsan, Mehroz. “Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction.” 2015. Masters Thesis, University of Toronto. Accessed June 24, 2019. http://hdl.handle.net/1807/71230.

MLA Handbook (7th Edition):

Ehsan, Mehroz. “Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction.” 2015. Web. 24 Jun 2019.

Vancouver:

Ehsan M. Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1807/71230.

Council of Science Editors:

Ehsan M. Role of Adiponectin in Monocytic Microparticle-Induced Endothelial Dysfunction. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71230


University of California – San Diego

17. Lombardo, Portia Sachi. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.

Degree: Biology, 2017, University of California – San Diego

 Our laboratory recently identified the autophagy initiating kinase ULK1 as a novel AMPK substrate, which directly connects the highly conserved AMPK-mediated energy sensing pathway to… (more)

Subjects/Keywords: Molecular biology; AMPK; mitophagy; Parkin; ULK1

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APA (6th Edition):

Lombardo, P. S. (2017). Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/515006px

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lombardo, Portia Sachi. “Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.” 2017. Thesis, University of California – San Diego. Accessed June 24, 2019. http://www.escholarship.org/uc/item/515006px.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lombardo, Portia Sachi. “Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.” 2017. Web. 24 Jun 2019.

Vancouver:

Lombardo PS. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2019 Jun 24]. Available from: http://www.escholarship.org/uc/item/515006px.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lombardo PS. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/515006px

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Riverside

18. Marin, Traci. AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes.

Degree: Biochemistry and Molecular Biology, 2013, University of California – Riverside

 Mitochondria serve to regulate energy and redox homeostasis, ultimately determining cell fate. While mitochondrial dysfunction is characteristic of many metabolic, cardiovascular and neurodegenerative diseases, AMP-activated… (more)

Subjects/Keywords: Biochemistry; AMPK; chromatin; DNMT1; epigenetic; Mitochondria; transcription

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APA (6th Edition):

Marin, T. (2013). AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/5z46r2s4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marin, Traci. “AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes.” 2013. Thesis, University of California – Riverside. Accessed June 24, 2019. http://www.escholarship.org/uc/item/5z46r2s4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marin, Traci. “AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes.” 2013. Web. 24 Jun 2019.

Vancouver:

Marin T. AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes. [Internet] [Thesis]. University of California – Riverside; 2013. [cited 2019 Jun 24]. Available from: http://www.escholarship.org/uc/item/5z46r2s4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marin T. AMPK Targets Involved in Energy Homeostasis and Epigenetic Regulation of Mitochondrial Function-related Genes. [Thesis]. University of California – Riverside; 2013. Available from: http://www.escholarship.org/uc/item/5z46r2s4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

19. Shim, Hong Seok. Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis.

Degree: PhD, Integrative and Evolutionary Biology, 2016, University of Southern California

 Dietary or calorie restriction has been a well‐studied strategy for increasing survival and preventing carcinogenesis in mammals. Short‐term starvation, an extreme form of dietary restriction,… (more)

Subjects/Keywords: starvation; REV1; sumoylation; p53; EGR1; AMPK; PKA

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APA (6th Edition):

Shim, H. S. (2016). Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1984

Chicago Manual of Style (16th Edition):

Shim, Hong Seok. “Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis.” 2016. Doctoral Dissertation, University of Southern California. Accessed June 24, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1984.

MLA Handbook (7th Edition):

Shim, Hong Seok. “Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis.” 2016. Web. 24 Jun 2019.

Vancouver:

Shim HS. Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis. [Internet] [Doctoral dissertation]. University of Southern California; 2016. [cited 2019 Jun 24]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1984.

Council of Science Editors:

Shim HS. Differential effects of starvation in normal and cancer cells: from EGR1-dependent protection to p53-mediated apoptosis. [Doctoral Dissertation]. University of Southern California; 2016. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/566322/rec/1984

20. 정, 종갑. Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice.

Degree: 2016, Ajou University

PART 1. TM-25659-induced activation of FGF21 level decreases insulin resistance and inflammation in skeletal muscle via GCN2 pathways 1 Ⅰ. INTRODUCTION 2 Ⅱ. MATERIALS AND… (more)

Subjects/Keywords: TAZ; TM-25659; insulin resistance; FGF21; AMPK

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APA (6th Edition):

정, . (2016). Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

정, 종갑. “Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice.” 2016. Thesis, Ajou University. Accessed June 24, 2019. http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

정, 종갑. “Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice.” 2016. Web. 24 Jun 2019.

Vancouver:

정 . Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice. [Internet] [Thesis]. Ajou University; 2016. [cited 2019 Jun 24]. Available from: http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

정 . Protective effect of TM-25659, known as TAZ activator, on high-fat diet-induced insulin resistance in C57BL/6J mice. [Thesis]. Ajou University; 2016. Available from: http://repository.ajou.ac.kr/handle/201003/13021 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

21. Xu, Pingwen. Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight.

Degree: PhD, Animal and Poultry Sciences, 2011, Virginia Tech

 This study was to determine (1) if genetic selection for high (HWS) or low (LWS) body weight in chickens has altered the hypothalamic AMP-activated protein… (more)

Subjects/Keywords: ghrelin; food intake; chicken; AMPK; obestatin

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APA (6th Edition):

Xu, P. (2011). Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/37680

Chicago Manual of Style (16th Edition):

Xu, Pingwen. “Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight.” 2011. Doctoral Dissertation, Virginia Tech. Accessed June 24, 2019. http://hdl.handle.net/10919/37680.

MLA Handbook (7th Edition):

Xu, Pingwen. “Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight.” 2011. Web. 24 Jun 2019.

Vancouver:

Xu P. Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight. [Internet] [Doctoral dissertation]. Virginia Tech; 2011. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/10919/37680.

Council of Science Editors:

Xu P. Involvement of AMP-activated protein kinase in differential regulation of appetite between lines of chickens selected for low or high juvenile body weight. [Doctoral Dissertation]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/37680


University of Ottawa

22. Al-Rewashdy, Hasanen. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .

Degree: 2014, University of Ottawa

 Duchenne Muscular Dystrophy (DMD) results from the absence of a functional dystrophin protein. Among its possible therapeutic options is the upregulation of dystrophin’s autosomal analogue,… (more)

Subjects/Keywords: Utrophin; Duchenne Muscular Dystrophy; Dystrophin; AMPK; AICAR

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APA (6th Edition):

Al-Rewashdy, H. (2014). Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Rewashdy, Hasanen. “Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .” 2014. Thesis, University of Ottawa. Accessed June 24, 2019. http://hdl.handle.net/10393/31470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Rewashdy, Hasanen. “Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .” 2014. Web. 24 Jun 2019.

Vancouver:

Al-Rewashdy H. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/10393/31470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Rewashdy H. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

23. Mahmoud, Amira Dia. The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body.

Degree: PhD, 2015, University of Edinburgh

 Ventilatory drive is mediated by respiratory central pattern generators that are located in the brainstem, which are continuously modulated by specialised peripheral and central chemoreceptors… (more)

Subjects/Keywords: 616.2; hypoxia; Lkb1; AMPK; apnoeas; ventilatory response

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APA (6th Edition):

Mahmoud, A. D. (2015). The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17886

Chicago Manual of Style (16th Edition):

Mahmoud, Amira Dia. “The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed June 24, 2019. http://hdl.handle.net/1842/17886.

MLA Handbook (7th Edition):

Mahmoud, Amira Dia. “The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body.” 2015. Web. 24 Jun 2019.

Vancouver:

Mahmoud AD. The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/1842/17886.

Council of Science Editors:

Mahmoud AD. The LKB1-AMPK signalling pathway drives the hypoxic ventilatory response by regulating brainstem nuclei but not the carotid body. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/17886


University of Minnesota

24. Yan, Yan. Regulation and Function of the Phosphatase PHLPP2 in Leukemia.

Degree: PhD, Pharmacology, 2017, University of Minnesota

 PHLPP2, a member of the PHLPP phosphatase family, which targets oncogenic kinases, has been actively investigated as a tumor suppressor in solid tumors. Little was… (more)

Subjects/Keywords: AMPK; ATRA; leukemia; microRNA; PHLPP2; phosphatase

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APA (6th Edition):

Yan, Y. (2017). Regulation and Function of the Phosphatase PHLPP2 in Leukemia. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191472

Chicago Manual of Style (16th Edition):

Yan, Yan. “Regulation and Function of the Phosphatase PHLPP2 in Leukemia.” 2017. Doctoral Dissertation, University of Minnesota. Accessed June 24, 2019. http://hdl.handle.net/11299/191472.

MLA Handbook (7th Edition):

Yan, Yan. “Regulation and Function of the Phosphatase PHLPP2 in Leukemia.” 2017. Web. 24 Jun 2019.

Vancouver:

Yan Y. Regulation and Function of the Phosphatase PHLPP2 in Leukemia. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2019 Jun 24]. Available from: http://hdl.handle.net/11299/191472.

Council of Science Editors:

Yan Y. Regulation and Function of the Phosphatase PHLPP2 in Leukemia. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/191472

25. Obba, Sandrine. Caractérisation et implication de l'autophagie au cours de la différenciation macrophagique des monocytes. Application à la Leucémie Myélomonocytaire Chronique : Characterization and implication of autophagy during the macrophagic differentiation. Application to Chronic MyeloMonocytic Leukemia.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2015, Nice

La LMMC est une hémopathie est caractérisée par une monocytose persistante (>1.109/L) ainsi que par la présence anormale de granulocytes immatures dans le sang des… (more)

Subjects/Keywords: Autophagie; Monocytes; Macrophages; AMPK; Différenciation cellulaire; Autophagy; Monocytes; Macrophages; AMPK; Cell differentiation

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APA (6th Edition):

Obba, S. (2015). Caractérisation et implication de l'autophagie au cours de la différenciation macrophagique des monocytes. Application à la Leucémie Myélomonocytaire Chronique : Characterization and implication of autophagy during the macrophagic differentiation. Application to Chronic MyeloMonocytic Leukemia. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2015NICE4035

Chicago Manual of Style (16th Edition):

Obba, Sandrine. “Caractérisation et implication de l'autophagie au cours de la différenciation macrophagique des monocytes. Application à la Leucémie Myélomonocytaire Chronique : Characterization and implication of autophagy during the macrophagic differentiation. Application to Chronic MyeloMonocytic Leukemia.” 2015. Doctoral Dissertation, Nice. Accessed June 24, 2019. http://www.theses.fr/2015NICE4035.

MLA Handbook (7th Edition):

Obba, Sandrine. “Caractérisation et implication de l'autophagie au cours de la différenciation macrophagique des monocytes. Application à la Leucémie Myélomonocytaire Chronique : Characterization and implication of autophagy during the macrophagic differentiation. Application to Chronic MyeloMonocytic Leukemia.” 2015. Web. 24 Jun 2019.

Vancouver:

Obba S. Caractérisation et implication de l'autophagie au cours de la différenciation macrophagique des monocytes. Application à la Leucémie Myélomonocytaire Chronique : Characterization and implication of autophagy during the macrophagic differentiation. Application to Chronic MyeloMonocytic Leukemia. [Internet] [Doctoral dissertation]. Nice; 2015. [cited 2019 Jun 24]. Available from: http://www.theses.fr/2015NICE4035.

Council of Science Editors:

Obba S. Caractérisation et implication de l'autophagie au cours de la différenciation macrophagique des monocytes. Application à la Leucémie Myélomonocytaire Chronique : Characterization and implication of autophagy during the macrophagic differentiation. Application to Chronic MyeloMonocytic Leukemia. [Doctoral Dissertation]. Nice; 2015. Available from: http://www.theses.fr/2015NICE4035

26. Philippe, Chloe. Modulation de la voie HIPPO par un métabolite aux propriétés anti-tumorales : l'AICAR : Modulation of the HIPPO pathway by a metabolite with anti-tumor properties : AICAR.

Degree: Docteur es, Biologie cellulaire et physiopathologie, 2016, Bordeaux

L’AICAR (5-Aminoimidazole-4-carboxamide ribonucleotide) est un intermédiaire de la voie de biosynthèse des purines. A des concentrations importantes, ce métabolite a un effet cytotoxique sur les… (more)

Subjects/Keywords: AICAR; Voie HIPPO; Effets cytotoxiques; AMPK; AICAR; HIPPO pathway; Cytotoxic effect; AMPK

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APA (6th Edition):

Philippe, C. (2016). Modulation de la voie HIPPO par un métabolite aux propriétés anti-tumorales : l'AICAR : Modulation of the HIPPO pathway by a metabolite with anti-tumor properties : AICAR. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2016BORD0454

Chicago Manual of Style (16th Edition):

Philippe, Chloe. “Modulation de la voie HIPPO par un métabolite aux propriétés anti-tumorales : l'AICAR : Modulation of the HIPPO pathway by a metabolite with anti-tumor properties : AICAR.” 2016. Doctoral Dissertation, Bordeaux. Accessed June 24, 2019. http://www.theses.fr/2016BORD0454.

MLA Handbook (7th Edition):

Philippe, Chloe. “Modulation de la voie HIPPO par un métabolite aux propriétés anti-tumorales : l'AICAR : Modulation of the HIPPO pathway by a metabolite with anti-tumor properties : AICAR.” 2016. Web. 24 Jun 2019.

Vancouver:

Philippe C. Modulation de la voie HIPPO par un métabolite aux propriétés anti-tumorales : l'AICAR : Modulation of the HIPPO pathway by a metabolite with anti-tumor properties : AICAR. [Internet] [Doctoral dissertation]. Bordeaux; 2016. [cited 2019 Jun 24]. Available from: http://www.theses.fr/2016BORD0454.

Council of Science Editors:

Philippe C. Modulation de la voie HIPPO par un métabolite aux propriétés anti-tumorales : l'AICAR : Modulation of the HIPPO pathway by a metabolite with anti-tumor properties : AICAR. [Doctoral Dissertation]. Bordeaux; 2016. Available from: http://www.theses.fr/2016BORD0454


Brigham Young University

27. Merrill, John. Iron Deficiency Causes a Shift in AMP-Activated Protein Kinase (AMPK) Catalytic Subunit Composition in Rat Skeletal Muscle.

Degree: MS, 2012, Brigham Young University

 To determine effects of iron deficiency on AMPK activation and signaling, as well as the AMPKα subunit composition in skeletal muscle, rats were fed a… (more)

Subjects/Keywords: AMPK; AMPK alpha; iron deficiency; anemia; energy metabolism; skeletal muscle; Cell and Developmental Biology; Physiology

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APA (6th Edition):

Merrill, J. (2012). Iron Deficiency Causes a Shift in AMP-Activated Protein Kinase (AMPK) Catalytic Subunit Composition in Rat Skeletal Muscle. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4482&context=etd

Chicago Manual of Style (16th Edition):

Merrill, John. “Iron Deficiency Causes a Shift in AMP-Activated Protein Kinase (AMPK) Catalytic Subunit Composition in Rat Skeletal Muscle.” 2012. Masters Thesis, Brigham Young University. Accessed June 24, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4482&context=etd.

MLA Handbook (7th Edition):

Merrill, John. “Iron Deficiency Causes a Shift in AMP-Activated Protein Kinase (AMPK) Catalytic Subunit Composition in Rat Skeletal Muscle.” 2012. Web. 24 Jun 2019.

Vancouver:

Merrill J. Iron Deficiency Causes a Shift in AMP-Activated Protein Kinase (AMPK) Catalytic Subunit Composition in Rat Skeletal Muscle. [Internet] [Masters thesis]. Brigham Young University; 2012. [cited 2019 Jun 24]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4482&context=etd.

Council of Science Editors:

Merrill J. Iron Deficiency Causes a Shift in AMP-Activated Protein Kinase (AMPK) Catalytic Subunit Composition in Rat Skeletal Muscle. [Masters Thesis]. Brigham Young University; 2012. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4482&context=etd


Université Paris-Sud – Paris XI

28. Lissa, Delphine. Le resvératrol et l’aspirine éliminent les cellules tétraploïdes pour la chimioprévention du cancer : Resveratrol and aspirin eliminate tetraploid cells for anticancer chemoprevention.

Degree: Docteur es, Oncologie, 2014, Université Paris-Sud – Paris XI

La tétraploïdie – cellule contenant le double du génome d’une cellule diploïde – est considérée comme un état métastable, à l’origine de l’aneuploïdie des cancers.… (more)

Subjects/Keywords: Resveratrol; Aspirine; Tétraploïdie; AMPK; ApcMin/+; Chimioprévention; Cancer; Resveratrol; Aspirin; Tetraploidy; AMPK; ApcMin/+; Chemoprevention; Cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lissa, D. (2014). Le resvératrol et l’aspirine éliminent les cellules tétraploïdes pour la chimioprévention du cancer : Resveratrol and aspirin eliminate tetraploid cells for anticancer chemoprevention. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA11T019

Chicago Manual of Style (16th Edition):

Lissa, Delphine. “Le resvératrol et l’aspirine éliminent les cellules tétraploïdes pour la chimioprévention du cancer : Resveratrol and aspirin eliminate tetraploid cells for anticancer chemoprevention.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed June 24, 2019. http://www.theses.fr/2014PA11T019.

MLA Handbook (7th Edition):

Lissa, Delphine. “Le resvératrol et l’aspirine éliminent les cellules tétraploïdes pour la chimioprévention du cancer : Resveratrol and aspirin eliminate tetraploid cells for anticancer chemoprevention.” 2014. Web. 24 Jun 2019.

Vancouver:

Lissa D. Le resvératrol et l’aspirine éliminent les cellules tétraploïdes pour la chimioprévention du cancer : Resveratrol and aspirin eliminate tetraploid cells for anticancer chemoprevention. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2019 Jun 24]. Available from: http://www.theses.fr/2014PA11T019.

Council of Science Editors:

Lissa D. Le resvératrol et l’aspirine éliminent les cellules tétraploïdes pour la chimioprévention du cancer : Resveratrol and aspirin eliminate tetraploid cells for anticancer chemoprevention. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA11T019


Universidade Federal de Santa Maria

29. Juliana Trevisan da Rocha. EFEITOS FARMACOLÓGICOS DO DISSELENETO DE DIFENILA EM UM MODELO DE MENOPAUSA INDUZIDA POR OVARIECTOMIA EM ROEDORES.

Degree: 2012, Universidade Federal de Santa Maria

 A menopausa caracteriza-se pela suspensão irreversível da função ovariana, com declínio da produção de hormônios estrogênicos. A falta desses hormônios culmina no aparecimento de alterações… (more)

Subjects/Keywords: disseleneto de difenila; episódios depressivos; prejuízo cognitivo; AMPK; dislipidemia; ovariectomia; CIENCIAS BIOLOGICAS; dyslipidemia; ovariectomy; AMPK; cognitive impairment; depressive state; diphenyl diselenide

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rocha, J. T. d. (2012). EFEITOS FARMACOLÓGICOS DO DISSELENETO DE DIFENILA EM UM MODELO DE MENOPAUSA INDUZIDA POR OVARIECTOMIA EM ROEDORES. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4965

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rocha, Juliana Trevisan da. “EFEITOS FARMACOLÓGICOS DO DISSELENETO DE DIFENILA EM UM MODELO DE MENOPAUSA INDUZIDA POR OVARIECTOMIA EM ROEDORES.” 2012. Thesis, Universidade Federal de Santa Maria. Accessed June 24, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4965.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rocha, Juliana Trevisan da. “EFEITOS FARMACOLÓGICOS DO DISSELENETO DE DIFENILA EM UM MODELO DE MENOPAUSA INDUZIDA POR OVARIECTOMIA EM ROEDORES.” 2012. Web. 24 Jun 2019.

Vancouver:

Rocha JTd. EFEITOS FARMACOLÓGICOS DO DISSELENETO DE DIFENILA EM UM MODELO DE MENOPAUSA INDUZIDA POR OVARIECTOMIA EM ROEDORES. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2012. [cited 2019 Jun 24]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4965.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rocha JTd. EFEITOS FARMACOLÓGICOS DO DISSELENETO DE DIFENILA EM UM MODELO DE MENOPAUSA INDUZIDA POR OVARIECTOMIA EM ROEDORES. [Thesis]. Universidade Federal de Santa Maria; 2012. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4965

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. 김, 주미. AMP-activated protein kinase-mTOR signaling pathway and hepatitis B virus replication.

Degree: 2015, Ajou University

B형간염 바이러스(hepatitis B virus, HBV)는 Hepadnaviridae family에 속하며, genome 크기가 약 3.2kb의 작은 DNA 바이러스이다. 전 세계적으로 약 3억 5천만 명 이상이 만성으로 HBV에 감염되어… (more)

Subjects/Keywords: B형간염 바이러스; AMPK-mTOR 신호 전달과정; Akt PP2A; S6K1; 4E-BP1; HBV; AMPK-mTOR signal; Akt

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

김, . (2015). AMP-activated protein kinase-mTOR signaling pathway and hepatitis B virus replication. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/11795 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 주미. “AMP-activated protein kinase-mTOR signaling pathway and hepatitis B virus replication.” 2015. Thesis, Ajou University. Accessed June 24, 2019. http://repository.ajou.ac.kr/handle/201003/11795 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 주미. “AMP-activated protein kinase-mTOR signaling pathway and hepatitis B virus replication.” 2015. Web. 24 Jun 2019.

Vancouver:

김 . AMP-activated protein kinase-mTOR signaling pathway and hepatitis B virus replication. [Internet] [Thesis]. Ajou University; 2015. [cited 2019 Jun 24]. Available from: http://repository.ajou.ac.kr/handle/201003/11795 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . AMP-activated protein kinase-mTOR signaling pathway and hepatitis B virus replication. [Thesis]. Ajou University; 2015. Available from: http://repository.ajou.ac.kr/handle/201003/11795 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000019519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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