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You searched for subject:(AMPAR). Showing records 1 – 29 of 29 total matches.

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University of Alberta

1. Chen, Yishen. Electrophysiological Signature of Neuropathic Pain.

Degree: PhD, Department of Pharmacology, 2012, University of Alberta

 Neuropathic pain afflicts 1.5-3% of the general population. It can be initiated by traumatic nerve injuries or diseases such as diabetic or post-herpetic neuropathy. Neuropathic… (more)

Subjects/Keywords: CCI; electrophysiology; AMPAR; pain; axotomy

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APA (6th Edition):

Chen, Y. (2012). Electrophysiological Signature of Neuropathic Pain. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/dv13zv08v

Chicago Manual of Style (16th Edition):

Chen, Yishen. “Electrophysiological Signature of Neuropathic Pain.” 2012. Doctoral Dissertation, University of Alberta. Accessed May 07, 2021. https://era.library.ualberta.ca/files/dv13zv08v.

MLA Handbook (7th Edition):

Chen, Yishen. “Electrophysiological Signature of Neuropathic Pain.” 2012. Web. 07 May 2021.

Vancouver:

Chen Y. Electrophysiological Signature of Neuropathic Pain. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2021 May 07]. Available from: https://era.library.ualberta.ca/files/dv13zv08v.

Council of Science Editors:

Chen Y. Electrophysiological Signature of Neuropathic Pain. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/dv13zv08v


Boston University

2. Lin, Amy Wei Pey. Regulation of glutamatergic AMPA receptor stability and trafficking by ubiquitination.

Degree: PhD, Neuroscience, 2013, Boston University

 AMPA-type glutamate receptors (AMPARs) play a critical role in mediating the majority of fast excitatory synaptic transmission in the brain, where alterations in receptor expression,… (more)

Subjects/Keywords: Neurosciences; AMPAR; Eps15; Trafficking; Ubiquitin

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APA (6th Edition):

Lin, A. W. P. (2013). Regulation of glutamatergic AMPA receptor stability and trafficking by ubiquitination. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13152

Chicago Manual of Style (16th Edition):

Lin, Amy Wei Pey. “Regulation of glutamatergic AMPA receptor stability and trafficking by ubiquitination.” 2013. Doctoral Dissertation, Boston University. Accessed May 07, 2021. http://hdl.handle.net/2144/13152.

MLA Handbook (7th Edition):

Lin, Amy Wei Pey. “Regulation of glutamatergic AMPA receptor stability and trafficking by ubiquitination.” 2013. Web. 07 May 2021.

Vancouver:

Lin AWP. Regulation of glutamatergic AMPA receptor stability and trafficking by ubiquitination. [Internet] [Doctoral dissertation]. Boston University; 2013. [cited 2021 May 07]. Available from: http://hdl.handle.net/2144/13152.

Council of Science Editors:

Lin AWP. Regulation of glutamatergic AMPA receptor stability and trafficking by ubiquitination. [Doctoral Dissertation]. Boston University; 2013. Available from: http://hdl.handle.net/2144/13152


University of Saskatchewan

3. Chen, Zhicheng 1981-. Interactions of AMPAR-Adenosine Receptor-Equilibrative Nucleoside Transporter in the Hippocampus: Implication for Stroke.

Degree: 2015, University of Saskatchewan

 The activation of presynaptic adenosine A1 receptors (A1Rs) is known to cause profound synaptic depression during hypoxia/cerebral ischemic insults, but postsynaptic function of A1Rs are… (more)

Subjects/Keywords: AMPAR; adenosine receptors; equilibrative nucleoside transporter; stroke

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APA (6th Edition):

Chen, Z. 1. (2015). Interactions of AMPAR-Adenosine Receptor-Equilibrative Nucleoside Transporter in the Hippocampus: Implication for Stroke. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/12653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Zhicheng 1981-. “Interactions of AMPAR-Adenosine Receptor-Equilibrative Nucleoside Transporter in the Hippocampus: Implication for Stroke.” 2015. Thesis, University of Saskatchewan. Accessed May 07, 2021. http://hdl.handle.net/10388/12653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Zhicheng 1981-. “Interactions of AMPAR-Adenosine Receptor-Equilibrative Nucleoside Transporter in the Hippocampus: Implication for Stroke.” 2015. Web. 07 May 2021.

Vancouver:

Chen Z1. Interactions of AMPAR-Adenosine Receptor-Equilibrative Nucleoside Transporter in the Hippocampus: Implication for Stroke. [Internet] [Thesis]. University of Saskatchewan; 2015. [cited 2021 May 07]. Available from: http://hdl.handle.net/10388/12653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Z1. Interactions of AMPAR-Adenosine Receptor-Equilibrative Nucleoside Transporter in the Hippocampus: Implication for Stroke. [Thesis]. University of Saskatchewan; 2015. Available from: http://hdl.handle.net/10388/12653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

4. Savtchouk, Iaroslav. Reciprocal interactions between synaptic AMPA receptors and action potential firing in cerebellar stellate cells.

Degree: 2011, Penn State University

 The cerebellum controls motor learning and is also implicated in higher cognitive functions including fear conditioning and emotional response. Inhibitory interneurons such as stellate cells… (more)

Subjects/Keywords: AMPAR; cerebellum; stellate cell; GluR2; synaptic plasticity

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APA (6th Edition):

Savtchouk, I. (2011). Reciprocal interactions between synaptic AMPA receptors and action potential firing in cerebellar stellate cells. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/12125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Savtchouk, Iaroslav. “Reciprocal interactions between synaptic AMPA receptors and action potential firing in cerebellar stellate cells.” 2011. Thesis, Penn State University. Accessed May 07, 2021. https://submit-etda.libraries.psu.edu/catalog/12125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Savtchouk, Iaroslav. “Reciprocal interactions between synaptic AMPA receptors and action potential firing in cerebellar stellate cells.” 2011. Web. 07 May 2021.

Vancouver:

Savtchouk I. Reciprocal interactions between synaptic AMPA receptors and action potential firing in cerebellar stellate cells. [Internet] [Thesis]. Penn State University; 2011. [cited 2021 May 07]. Available from: https://submit-etda.libraries.psu.edu/catalog/12125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Savtchouk I. Reciprocal interactions between synaptic AMPA receptors and action potential firing in cerebellar stellate cells. [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/12125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vrije Universiteit Amsterdam

5. Stroeder, J. Shisa proteins and AMPA-receptor function .

Degree: 2015, Vrije Universiteit Amsterdam

Subjects/Keywords: AMPAR; regulation; electrophysiology

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APA (6th Edition):

Stroeder, J. (2015). Shisa proteins and AMPA-receptor function . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/54967

Chicago Manual of Style (16th Edition):

Stroeder, J. “Shisa proteins and AMPA-receptor function .” 2015. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed May 07, 2021. http://hdl.handle.net/1871/54967.

MLA Handbook (7th Edition):

Stroeder, J. “Shisa proteins and AMPA-receptor function .” 2015. Web. 07 May 2021.

Vancouver:

Stroeder J. Shisa proteins and AMPA-receptor function . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2015. [cited 2021 May 07]. Available from: http://hdl.handle.net/1871/54967.

Council of Science Editors:

Stroeder J. Shisa proteins and AMPA-receptor function . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2015. Available from: http://hdl.handle.net/1871/54967


Boston University

6. Guo, Ouyang. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.

Degree: MS, Anatomy & Neurobiology, 2016, Boston University

 Ubiquilin (UBQLN) is a member of type2 ubiquitin-like (UBL) protein family characterized by an UBL domain at the N-terminus and an ubiquitin associated (UBA) domain… (more)

Subjects/Keywords: Neurosciences; AMPAR; Neurodegenerative disease; Proteasome; Ubiquilin; Ubiquitination

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APA (6th Edition):

Guo, O. (2016). The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/17126

Chicago Manual of Style (16th Edition):

Guo, Ouyang. “The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.” 2016. Masters Thesis, Boston University. Accessed May 07, 2021. http://hdl.handle.net/2144/17126.

MLA Handbook (7th Edition):

Guo, Ouyang. “The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation.” 2016. Web. 07 May 2021.

Vancouver:

Guo O. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 May 07]. Available from: http://hdl.handle.net/2144/17126.

Council of Science Editors:

Guo O. The role of ubiquilin in AMPA receptor ubiquitination and proteasomal degradation. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/17126


University of Toronto

7. Lesperance, Lee Stephen. Activity Induced Plasticity in AMPAR Composition at the developing Calyx of Held / MNTB Synapse.

Degree: PhD, 2016, University of Toronto

Developing central synapses exhibit robust plasticity and undergo marked experience-dependent remodeling. This is particularly evident in sensory systems such as auditory brainstem circuits where synapses… (more)

Subjects/Keywords: activity; AMPAR; development; ion channel; MNTB; 0317

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APA (6th Edition):

Lesperance, L. S. (2016). Activity Induced Plasticity in AMPAR Composition at the developing Calyx of Held / MNTB Synapse. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80394

Chicago Manual of Style (16th Edition):

Lesperance, Lee Stephen. “Activity Induced Plasticity in AMPAR Composition at the developing Calyx of Held / MNTB Synapse.” 2016. Doctoral Dissertation, University of Toronto. Accessed May 07, 2021. http://hdl.handle.net/1807/80394.

MLA Handbook (7th Edition):

Lesperance, Lee Stephen. “Activity Induced Plasticity in AMPAR Composition at the developing Calyx of Held / MNTB Synapse.” 2016. Web. 07 May 2021.

Vancouver:

Lesperance LS. Activity Induced Plasticity in AMPAR Composition at the developing Calyx of Held / MNTB Synapse. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 May 07]. Available from: http://hdl.handle.net/1807/80394.

Council of Science Editors:

Lesperance LS. Activity Induced Plasticity in AMPAR Composition at the developing Calyx of Held / MNTB Synapse. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/80394


University of Toronto

8. Cai, Sammy. Investigating the Role of LIMK1 Signaling in PKA-dependent LTP in the Hippocampus.

Degree: 2019, University of Toronto

Long-term potentiation (LTP) in the hippocampus is the most extensive form of long-lasting synaptic plasticity and is widely regarded as the cellular basis of memory… (more)

Subjects/Keywords: CP-AMPAR; LIMK1; LTP; PKA; 0317

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APA (6th Edition):

Cai, S. (2019). Investigating the Role of LIMK1 Signaling in PKA-dependent LTP in the Hippocampus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/99896

Chicago Manual of Style (16th Edition):

Cai, Sammy. “Investigating the Role of LIMK1 Signaling in PKA-dependent LTP in the Hippocampus.” 2019. Masters Thesis, University of Toronto. Accessed May 07, 2021. http://hdl.handle.net/1807/99896.

MLA Handbook (7th Edition):

Cai, Sammy. “Investigating the Role of LIMK1 Signaling in PKA-dependent LTP in the Hippocampus.” 2019. Web. 07 May 2021.

Vancouver:

Cai S. Investigating the Role of LIMK1 Signaling in PKA-dependent LTP in the Hippocampus. [Internet] [Masters thesis]. University of Toronto; 2019. [cited 2021 May 07]. Available from: http://hdl.handle.net/1807/99896.

Council of Science Editors:

Cai S. Investigating the Role of LIMK1 Signaling in PKA-dependent LTP in the Hippocampus. [Masters Thesis]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/99896

9. Pougnet, Johan. Régulation du trafic des récepteurs AMPA et de la plasticité synaptique induite par les récepteurs P2X : ATP P2X receptors down-regulate ampa receptor trafficking and postsynaptic efficacy in hippocampal neurons.

Degree: Docteur es, Sciences, technologie, santé. Neurosciences, 2013, Université de Bordeaux Segalen

Les récepteurs ionotropiques AMPA (AMPAR) activés par le glutamate sont les principaux acteurs de la transmission synaptique excitatrice rapide du cerveau. Ils jouent également un… (more)

Subjects/Keywords: Récepteurs P2X; AMPAR; Endocytose; Neuromodulation; Synapse; P2X receptors; AMPAR; Endocytosis; Neuromodulation; Synapse

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APA (6th Edition):

Pougnet, J. (2013). Régulation du trafic des récepteurs AMPA et de la plasticité synaptique induite par les récepteurs P2X : ATP P2X receptors down-regulate ampa receptor trafficking and postsynaptic efficacy in hippocampal neurons. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2013BOR22062

Chicago Manual of Style (16th Edition):

Pougnet, Johan. “Régulation du trafic des récepteurs AMPA et de la plasticité synaptique induite par les récepteurs P2X : ATP P2X receptors down-regulate ampa receptor trafficking and postsynaptic efficacy in hippocampal neurons.” 2013. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed May 07, 2021. http://www.theses.fr/2013BOR22062.

MLA Handbook (7th Edition):

Pougnet, Johan. “Régulation du trafic des récepteurs AMPA et de la plasticité synaptique induite par les récepteurs P2X : ATP P2X receptors down-regulate ampa receptor trafficking and postsynaptic efficacy in hippocampal neurons.” 2013. Web. 07 May 2021.

Vancouver:

Pougnet J. Régulation du trafic des récepteurs AMPA et de la plasticité synaptique induite par les récepteurs P2X : ATP P2X receptors down-regulate ampa receptor trafficking and postsynaptic efficacy in hippocampal neurons. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2013. [cited 2021 May 07]. Available from: http://www.theses.fr/2013BOR22062.

Council of Science Editors:

Pougnet J. Régulation du trafic des récepteurs AMPA et de la plasticité synaptique induite par les récepteurs P2X : ATP P2X receptors down-regulate ampa receptor trafficking and postsynaptic efficacy in hippocampal neurons. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2013. Available from: http://www.theses.fr/2013BOR22062


University of Alberta

10. Cunningham, Marcus E. The Expression of Transmembrane AMPA Receptor Regulatory Proteins (TARPs) in Developing Zebrafish (Danio rerio).

Degree: MS, Department of Biological Sciences, 2013, University of Alberta

 In embryonic vertebrates, newly-formed synapses are quiet until they are unsilenced through a process that resembles synaptic plasticity but remains mechanistically unclear. I used gene… (more)

Subjects/Keywords: Zebrafish; AMPA Receptors; TARPs; AMPA; AMPAR; Stargazin; Developmental

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APA (6th Edition):

Cunningham, M. E. (2013). The Expression of Transmembrane AMPA Receptor Regulatory Proteins (TARPs) in Developing Zebrafish (Danio rerio). (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/h702q773v

Chicago Manual of Style (16th Edition):

Cunningham, Marcus E. “The Expression of Transmembrane AMPA Receptor Regulatory Proteins (TARPs) in Developing Zebrafish (Danio rerio).” 2013. Masters Thesis, University of Alberta. Accessed May 07, 2021. https://era.library.ualberta.ca/files/h702q773v.

MLA Handbook (7th Edition):

Cunningham, Marcus E. “The Expression of Transmembrane AMPA Receptor Regulatory Proteins (TARPs) in Developing Zebrafish (Danio rerio).” 2013. Web. 07 May 2021.

Vancouver:

Cunningham ME. The Expression of Transmembrane AMPA Receptor Regulatory Proteins (TARPs) in Developing Zebrafish (Danio rerio). [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2021 May 07]. Available from: https://era.library.ualberta.ca/files/h702q773v.

Council of Science Editors:

Cunningham ME. The Expression of Transmembrane AMPA Receptor Regulatory Proteins (TARPs) in Developing Zebrafish (Danio rerio). [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/h702q773v

11. Chevy, Quentin. Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses.

Degree: Docteur es, Neurosciences, 2015, Université Pierre et Marie Curie – Paris VI

L'efficacité de la transmission synaptique GABAergique est influencée par la concentration intracellulaire en ions chlorure. Dans les neurones matures, l'extrusion de ces ions par le… (more)

Subjects/Keywords: KCC2; Epine dendritique; Ltp; GluA1; AMPAR; Actine; Dendritic spine; Actin; 616.8

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APA (6th Edition):

Chevy, Q. (2015). Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066041

Chicago Manual of Style (16th Edition):

Chevy, Quentin. “Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed May 07, 2021. http://www.theses.fr/2015PA066041.

MLA Handbook (7th Edition):

Chevy, Quentin. “Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses.” 2015. Web. 07 May 2021.

Vancouver:

Chevy Q. Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2021 May 07]. Available from: http://www.theses.fr/2015PA066041.

Council of Science Editors:

Chevy Q. Rôle du transporteur neuronal Potassium/Chlore KCC2 dans la plasticité des synapses glutamatergiques : Involvement of the neuronal K/Cl cotransporter KCC2 in the plasticity of glutamatergic synapses. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066041


Boston University

12. Gilbert, James Patrick. Regulation of homeostatic synaptic plasticity by amyloid Beta in cultured rat hippocampal neurons.

Degree: 2014, Boston University

 Accumulation of amyloid beta (Aβ) in the brain is a pathological hallmark of Alzheimer's disease (AD) and has been shown to lead to synaptic dysfunction… (more)

Subjects/Keywords: Neurosciences; Alzheimer's disease; AMPAR; Amyloid beta; Homeostatic synaptic plasticity

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APA (6th Edition):

Gilbert, J. P. (2014). Regulation of homeostatic synaptic plasticity by amyloid Beta in cultured rat hippocampal neurons. (Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/14293

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gilbert, James Patrick. “Regulation of homeostatic synaptic plasticity by amyloid Beta in cultured rat hippocampal neurons.” 2014. Thesis, Boston University. Accessed May 07, 2021. http://hdl.handle.net/2144/14293.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gilbert, James Patrick. “Regulation of homeostatic synaptic plasticity by amyloid Beta in cultured rat hippocampal neurons.” 2014. Web. 07 May 2021.

Vancouver:

Gilbert JP. Regulation of homeostatic synaptic plasticity by amyloid Beta in cultured rat hippocampal neurons. [Internet] [Thesis]. Boston University; 2014. [cited 2021 May 07]. Available from: http://hdl.handle.net/2144/14293.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gilbert JP. Regulation of homeostatic synaptic plasticity by amyloid Beta in cultured rat hippocampal neurons. [Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14293

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

13. -5865-7115. Functional Characterization of an AMPA receptor auxiliary subunit GSG1L in vivo.

Degree: PhD, Neuroscience, 2020, Vanderbilt University

 AMPA receptors (AMPAR) are key mediators of fast excitatory neurotransmission within the brain. The functional identity of AMPAR at the synapses strongly contributes to molecular… (more)

Subjects/Keywords: AMPA; AMPAR; auxiliary subunits; GSG1L; stargazin; anterior thalamus; short-term plasticity

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APA (6th Edition):

-5865-7115. (2020). Functional Characterization of an AMPA receptor auxiliary subunit GSG1L in vivo. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/16364

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-5865-7115. “Functional Characterization of an AMPA receptor auxiliary subunit GSG1L in vivo.” 2020. Doctoral Dissertation, Vanderbilt University. Accessed May 07, 2021. http://hdl.handle.net/1803/16364.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-5865-7115. “Functional Characterization of an AMPA receptor auxiliary subunit GSG1L in vivo.” 2020. Web. 07 May 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-5865-7115. Functional Characterization of an AMPA receptor auxiliary subunit GSG1L in vivo. [Internet] [Doctoral dissertation]. Vanderbilt University; 2020. [cited 2021 May 07]. Available from: http://hdl.handle.net/1803/16364.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-5865-7115. Functional Characterization of an AMPA receptor auxiliary subunit GSG1L in vivo. [Doctoral Dissertation]. Vanderbilt University; 2020. Available from: http://hdl.handle.net/1803/16364

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

14. Ferreira, Diana Aline Noga Morais. Papel de mTOR, BDNF, AMPAR E NMDAR na reconsolidação da memória de extinção.

Degree: 2019, Federal University of Rio Grande do Norte

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

A reativação de uma memória é um processo que pode induzir dois fenômenos aparentemente concorrentes, a extinção… (more)

Subjects/Keywords: CNPQ::CIENCIAS BIOLOGICAS; Reconsolidação; Extinção; mTOR; BDNF; GluN2A; GluN2B; AMPAR

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APA (6th Edition):

Ferreira, D. A. N. M. (2019). Papel de mTOR, BDNF, AMPAR E NMDAR na reconsolidação da memória de extinção. (Doctoral Dissertation). Federal University of Rio Grande do Norte. Retrieved from https://repositorio.ufrn.br/jspui/handle/123456789/27200

Chicago Manual of Style (16th Edition):

Ferreira, Diana Aline Noga Morais. “Papel de mTOR, BDNF, AMPAR E NMDAR na reconsolidação da memória de extinção.” 2019. Doctoral Dissertation, Federal University of Rio Grande do Norte. Accessed May 07, 2021. https://repositorio.ufrn.br/jspui/handle/123456789/27200.

MLA Handbook (7th Edition):

Ferreira, Diana Aline Noga Morais. “Papel de mTOR, BDNF, AMPAR E NMDAR na reconsolidação da memória de extinção.” 2019. Web. 07 May 2021.

Vancouver:

Ferreira DANM. Papel de mTOR, BDNF, AMPAR E NMDAR na reconsolidação da memória de extinção. [Internet] [Doctoral dissertation]. Federal University of Rio Grande do Norte; 2019. [cited 2021 May 07]. Available from: https://repositorio.ufrn.br/jspui/handle/123456789/27200.

Council of Science Editors:

Ferreira DANM. Papel de mTOR, BDNF, AMPAR E NMDAR na reconsolidação da memória de extinção. [Doctoral Dissertation]. Federal University of Rio Grande do Norte; 2019. Available from: https://repositorio.ufrn.br/jspui/handle/123456789/27200


Erasmus University Rotterdam

15. Sonderen, Agnes. Autoimmune encephalitis : The clinical value of antibodies directed to extracellular antigens.

Degree: Department of Neurology, 2018, Erasmus University Rotterdam

 textabstractHet doel van dit proefschrift is om meer inzicht te geven in de meest voorkomende ziekten die geassocieerd zijn met antistoffen tegen extracellulaire antigenen. Vroege… (more)

Subjects/Keywords: LGI1; Caspr2; VGKC; NMDAR; AMPAR; antibodies

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sonderen, A. (2018). Autoimmune encephalitis : The clinical value of antibodies directed to extracellular antigens. (Doctoral Dissertation). Erasmus University Rotterdam. Retrieved from http://hdl.handle.net/1765/113142

Chicago Manual of Style (16th Edition):

Sonderen, Agnes. “Autoimmune encephalitis : The clinical value of antibodies directed to extracellular antigens.” 2018. Doctoral Dissertation, Erasmus University Rotterdam. Accessed May 07, 2021. http://hdl.handle.net/1765/113142.

MLA Handbook (7th Edition):

Sonderen, Agnes. “Autoimmune encephalitis : The clinical value of antibodies directed to extracellular antigens.” 2018. Web. 07 May 2021.

Vancouver:

Sonderen A. Autoimmune encephalitis : The clinical value of antibodies directed to extracellular antigens. [Internet] [Doctoral dissertation]. Erasmus University Rotterdam; 2018. [cited 2021 May 07]. Available from: http://hdl.handle.net/1765/113142.

Council of Science Editors:

Sonderen A. Autoimmune encephalitis : The clinical value of antibodies directed to extracellular antigens. [Doctoral Dissertation]. Erasmus University Rotterdam; 2018. Available from: http://hdl.handle.net/1765/113142


University of Cambridge

16. Watson, Jake Frederick. Molecular mechanisms controlling excitatory synaptic transmission.

Degree: PhD, 2019, University of Cambridge

 At synapses, the sites of communication between neurons, neurotransmitter is released from the presynaptic cell and binds to postsynaptic receptors on another. In the case… (more)

Subjects/Keywords: AMPA receptor; AMPAR; LTP; long-term potentiation; synaptic plasticity; synaptic transmission; glutamate; glutamate receptors; iGluRs

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APA (6th Edition):

Watson, J. F. (2019). Molecular mechanisms controlling excitatory synaptic transmission. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.39689 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774798

Chicago Manual of Style (16th Edition):

Watson, Jake Frederick. “Molecular mechanisms controlling excitatory synaptic transmission.” 2019. Doctoral Dissertation, University of Cambridge. Accessed May 07, 2021. https://doi.org/10.17863/CAM.39689 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774798.

MLA Handbook (7th Edition):

Watson, Jake Frederick. “Molecular mechanisms controlling excitatory synaptic transmission.” 2019. Web. 07 May 2021.

Vancouver:

Watson JF. Molecular mechanisms controlling excitatory synaptic transmission. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 May 07]. Available from: https://doi.org/10.17863/CAM.39689 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774798.

Council of Science Editors:

Watson JF. Molecular mechanisms controlling excitatory synaptic transmission. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.39689 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774798

17. Watson, Jake Frederick. Molecular Mechanisms Controlling Excitatory Synaptic Transmission.

Degree: PhD, 2019, University of Cambridge

 At synapses, the sites of communication between neurons, neurotransmitter is released from the presynaptic cell and binds to postsynaptic receptors on another. In the case… (more)

Subjects/Keywords: AMPA receptor; AMPAR; LTP; long-term potentiation; synaptic plasticity; synaptic transmission; glutamate; glutamate receptors; iGluRs

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Watson, J. F. (2019). Molecular Mechanisms Controlling Excitatory Synaptic Transmission. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/292529https://www.repository.cam.ac.uk/bitstream/1810/292529/3/f1126f3d-42ec-40b5-9738-8b29bd65b3ea_confirmations.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/4/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/5/f1126f3d-42ec-40b5-9738-8b29bd65b3ea.zip ; https://www.repository.cam.ac.uk/bitstream/1810/292529/6/Thesis_JFW_Submission.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/7/Thesis_JFW_Submission.pdf.jpg

Chicago Manual of Style (16th Edition):

Watson, Jake Frederick. “Molecular Mechanisms Controlling Excitatory Synaptic Transmission.” 2019. Doctoral Dissertation, University of Cambridge. Accessed May 07, 2021. https://www.repository.cam.ac.uk/handle/1810/292529https://www.repository.cam.ac.uk/bitstream/1810/292529/3/f1126f3d-42ec-40b5-9738-8b29bd65b3ea_confirmations.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/4/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/5/f1126f3d-42ec-40b5-9738-8b29bd65b3ea.zip ; https://www.repository.cam.ac.uk/bitstream/1810/292529/6/Thesis_JFW_Submission.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/7/Thesis_JFW_Submission.pdf.jpg.

MLA Handbook (7th Edition):

Watson, Jake Frederick. “Molecular Mechanisms Controlling Excitatory Synaptic Transmission.” 2019. Web. 07 May 2021.

Vancouver:

Watson JF. Molecular Mechanisms Controlling Excitatory Synaptic Transmission. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 May 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/292529https://www.repository.cam.ac.uk/bitstream/1810/292529/3/f1126f3d-42ec-40b5-9738-8b29bd65b3ea_confirmations.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/4/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/5/f1126f3d-42ec-40b5-9738-8b29bd65b3ea.zip ; https://www.repository.cam.ac.uk/bitstream/1810/292529/6/Thesis_JFW_Submission.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/7/Thesis_JFW_Submission.pdf.jpg.

Council of Science Editors:

Watson JF. Molecular Mechanisms Controlling Excitatory Synaptic Transmission. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/292529https://www.repository.cam.ac.uk/bitstream/1810/292529/3/f1126f3d-42ec-40b5-9738-8b29bd65b3ea_confirmations.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/4/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/5/f1126f3d-42ec-40b5-9738-8b29bd65b3ea.zip ; https://www.repository.cam.ac.uk/bitstream/1810/292529/6/Thesis_JFW_Submission.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/292529/7/Thesis_JFW_Submission.pdf.jpg

18. Maxfield, Dane Arthur. Kinesin-1 regulates synaptic strength by mediating delivery, removal and redistribution of ampars.

Degree: PhD, Biology, 2014, University of Utah

 The cellular processes that govern neuronal function are highly complex and tightly regulated in order to perform the elaborate information processing achieved by the brain.… (more)

Subjects/Keywords: AMPAR; C. elegans; Kinesin; Synapse; Trafficking

…11 1.4 Overview of AMPAR transport… …localization and transport of the vertebrate GluA1 AMPAR subunit depends on UNC-116/KIF5… …gated current and AMPAR surface expression… …cellular trafficking of the AMPA-type glutamate receptor (AMPAR) due to its central… …the cell biological processes involved in synaptic function. AMPAR Localization and… 

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APA (6th Edition):

Maxfield, D. A. (2014). Kinesin-1 regulates synaptic strength by mediating delivery, removal and redistribution of ampars. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2963/rec/1453

Chicago Manual of Style (16th Edition):

Maxfield, Dane Arthur. “Kinesin-1 regulates synaptic strength by mediating delivery, removal and redistribution of ampars.” 2014. Doctoral Dissertation, University of Utah. Accessed May 07, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2963/rec/1453.

MLA Handbook (7th Edition):

Maxfield, Dane Arthur. “Kinesin-1 regulates synaptic strength by mediating delivery, removal and redistribution of ampars.” 2014. Web. 07 May 2021.

Vancouver:

Maxfield DA. Kinesin-1 regulates synaptic strength by mediating delivery, removal and redistribution of ampars. [Internet] [Doctoral dissertation]. University of Utah; 2014. [cited 2021 May 07]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2963/rec/1453.

Council of Science Editors:

Maxfield DA. Kinesin-1 regulates synaptic strength by mediating delivery, removal and redistribution of ampars. [Doctoral Dissertation]. University of Utah; 2014. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2963/rec/1453

19. Campelo, Tiago. De la diffusion latérale des récepteurs AMPA à la perception des whiskers : un nouveau modèle de cartographie corticale : From AMPAR lateral diffusion to whisker perception : a new model for cortical remapping.

Degree: Docteur es, Neurosciences, 2019, Bordeaux

 Les champs récepteurs corticaux se réorganisent en réponse aux changements de l'environnement. Par exemple, suite à une lésion périphérique, les modalités sensorielles préservées gagnent de… (more)

Subjects/Keywords: Récepteurs AMPA; Plasticité synaptique; Ltp; Réorganisation des cartes corticales; Imagerie et Electrophysiologie In vivo; Ampar; Synaptic Plasticity; Ltp; Cortical Remapping; In vivo Patch Clamp; In vivo 2-Photon Imaging

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APA (6th Edition):

Campelo, T. (2019). De la diffusion latérale des récepteurs AMPA à la perception des whiskers : un nouveau modèle de cartographie corticale : From AMPAR lateral diffusion to whisker perception : a new model for cortical remapping. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2019BORD0171

Chicago Manual of Style (16th Edition):

Campelo, Tiago. “De la diffusion latérale des récepteurs AMPA à la perception des whiskers : un nouveau modèle de cartographie corticale : From AMPAR lateral diffusion to whisker perception : a new model for cortical remapping.” 2019. Doctoral Dissertation, Bordeaux. Accessed May 07, 2021. http://www.theses.fr/2019BORD0171.

MLA Handbook (7th Edition):

Campelo, Tiago. “De la diffusion latérale des récepteurs AMPA à la perception des whiskers : un nouveau modèle de cartographie corticale : From AMPAR lateral diffusion to whisker perception : a new model for cortical remapping.” 2019. Web. 07 May 2021.

Vancouver:

Campelo T. De la diffusion latérale des récepteurs AMPA à la perception des whiskers : un nouveau modèle de cartographie corticale : From AMPAR lateral diffusion to whisker perception : a new model for cortical remapping. [Internet] [Doctoral dissertation]. Bordeaux; 2019. [cited 2021 May 07]. Available from: http://www.theses.fr/2019BORD0171.

Council of Science Editors:

Campelo T. De la diffusion latérale des récepteurs AMPA à la perception des whiskers : un nouveau modèle de cartographie corticale : From AMPAR lateral diffusion to whisker perception : a new model for cortical remapping. [Doctoral Dissertation]. Bordeaux; 2019. Available from: http://www.theses.fr/2019BORD0171

20. Geddes, Sean D. Dynamic Regulation of Synaptic Transmission onto Serotonin Neurons by Antidepressants .

Degree: 2012, University of Ottawa

 Antidepressants are generally believed to exert their clinical efficacy by enhancing 5-HT transmission. Interestingly, sustained administration of selective serotonin (5-HT) reuptake inhibitors (SSRIs) strongly suppresses… (more)

Subjects/Keywords: Electrophysiology; Glutamate; NMDAR; AMPAR; Serotonin; SSRI; Whole-Cell Recording

…after-depolarizing potential AMPAR α-amino-3-hydroxy-5-methylisoxazole-4-propionic acid… …5-methyl-4isoxazolepropionic acid (AMPAR), N-methyl-D-aspartate (NMDAR)… …The GriA1- GriA4 genes encode the GluA1- GluA4 AMPAR subunits respectively. AMPAR subunits… …can exist as both homo- and heteromers. It is their composition that in turn dictates AMPAR… …2011). Moreover, when comparing AMPAR- and KAR13 mediated currents, KAR currents have… 

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APA (6th Edition):

Geddes, S. D. (2012). Dynamic Regulation of Synaptic Transmission onto Serotonin Neurons by Antidepressants . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/23532

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Geddes, Sean D. “Dynamic Regulation of Synaptic Transmission onto Serotonin Neurons by Antidepressants .” 2012. Thesis, University of Ottawa. Accessed May 07, 2021. http://hdl.handle.net/10393/23532.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Geddes, Sean D. “Dynamic Regulation of Synaptic Transmission onto Serotonin Neurons by Antidepressants .” 2012. Web. 07 May 2021.

Vancouver:

Geddes SD. Dynamic Regulation of Synaptic Transmission onto Serotonin Neurons by Antidepressants . [Internet] [Thesis]. University of Ottawa; 2012. [cited 2021 May 07]. Available from: http://hdl.handle.net/10393/23532.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Geddes SD. Dynamic Regulation of Synaptic Transmission onto Serotonin Neurons by Antidepressants . [Thesis]. University of Ottawa; 2012. Available from: http://hdl.handle.net/10393/23532

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Lincoln University

21. Liang, Jingyi. Modelling and investigation of NMDAR-mediated calcium signalling at Hippocampal Dendritic Spine in Alzheimer’s disease.

Degree: 2016, Lincoln University

 Alzheimer’s disease (AD) is a devastating, incurable neurodegenerative disease affecting millions of people worldwide. Dysregulation of intracellular Ca2+ signalling has been observed as an early… (more)

Subjects/Keywords: NMDAR; CaMKII-NMDAR formation; parameter esitimation; ordinary differential equations; AMPAR; Alzheimer's disease; Amyloid beta; calcium ions; glutamate transmission; endoplasmic reticulum; mathematical modelling; 010202 Biological Mathematics; 060114 Systems Biology; 080110 Simulation and Modelling

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APA (6th Edition):

Liang, J. (2016). Modelling and investigation of NMDAR-mediated calcium signalling at Hippocampal Dendritic Spine in Alzheimer’s disease. (Thesis). Lincoln University. Retrieved from http://hdl.handle.net/10182/7661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liang, Jingyi. “Modelling and investigation of NMDAR-mediated calcium signalling at Hippocampal Dendritic Spine in Alzheimer’s disease.” 2016. Thesis, Lincoln University. Accessed May 07, 2021. http://hdl.handle.net/10182/7661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liang, Jingyi. “Modelling and investigation of NMDAR-mediated calcium signalling at Hippocampal Dendritic Spine in Alzheimer’s disease.” 2016. Web. 07 May 2021.

Vancouver:

Liang J. Modelling and investigation of NMDAR-mediated calcium signalling at Hippocampal Dendritic Spine in Alzheimer’s disease. [Internet] [Thesis]. Lincoln University; 2016. [cited 2021 May 07]. Available from: http://hdl.handle.net/10182/7661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liang J. Modelling and investigation of NMDAR-mediated calcium signalling at Hippocampal Dendritic Spine in Alzheimer’s disease. [Thesis]. Lincoln University; 2016. Available from: http://hdl.handle.net/10182/7661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

22. Dukoff, David. The Impact of ROS Scavenging on NMDA and AMPA Receptor Whole Cell Currents in Pyramidal Neurons of the Anoxia Tolerant Western Painted Turtle.

Degree: 2013, University of Toronto

Extended periods of oxygen deprivation cause brain death in mammals but the western painted turtle overwinters in anoxic mud for months without damage. Neural protection… (more)

Subjects/Keywords: anoxia; reactive oxidative speecies; NMDAR; AMPAR; western painted turtle; mitochondria; ATP-sensitive potassium channels; calcium; hydrogen peroxide; N-acetylcysteine; N-2-mercaptopropionylglycine; NMDAR redox site; electrophysiological patch clamping; fluorescent microscopy; 0379

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APA (6th Edition):

Dukoff, D. (2013). The Impact of ROS Scavenging on NMDA and AMPA Receptor Whole Cell Currents in Pyramidal Neurons of the Anoxia Tolerant Western Painted Turtle. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42826

Chicago Manual of Style (16th Edition):

Dukoff, David. “The Impact of ROS Scavenging on NMDA and AMPA Receptor Whole Cell Currents in Pyramidal Neurons of the Anoxia Tolerant Western Painted Turtle.” 2013. Masters Thesis, University of Toronto. Accessed May 07, 2021. http://hdl.handle.net/1807/42826.

MLA Handbook (7th Edition):

Dukoff, David. “The Impact of ROS Scavenging on NMDA and AMPA Receptor Whole Cell Currents in Pyramidal Neurons of the Anoxia Tolerant Western Painted Turtle.” 2013. Web. 07 May 2021.

Vancouver:

Dukoff D. The Impact of ROS Scavenging on NMDA and AMPA Receptor Whole Cell Currents in Pyramidal Neurons of the Anoxia Tolerant Western Painted Turtle. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 May 07]. Available from: http://hdl.handle.net/1807/42826.

Council of Science Editors:

Dukoff D. The Impact of ROS Scavenging on NMDA and AMPA Receptor Whole Cell Currents in Pyramidal Neurons of the Anoxia Tolerant Western Painted Turtle. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42826


University of Florida

23. Lee, Wei-Hua. The Influence of Viral Vector Delivery of Superoxide Dismutase and Catalase to the Hippocampus on Spatial Learning, Memory and NMDA Receptor Function during Aging.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2012, University of Florida

 Studies employing transgenic mice indicate overexpression of superoxide dismutase 1 (SOD1) improves memory during aging. It is unclear whether the improvement is due to a… (more)

Subjects/Keywords: Antioxidants; Enzymes; Hippocampus; Learning; Lipids; Mazes; Memory; Oxidative stress; Rats; Superoxides; aav  – ampar  – behavior  – cat  – catalase  – damage  – glutathione  – hippocampus  – learning  – lentivirus  – memory  – nmdar  – oxidative  – rat  – redox  – ros  – sod1  – sod2  – spatial  – virus

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APA (6th Edition):

Lee, W. (2012). The Influence of Viral Vector Delivery of Superoxide Dismutase and Catalase to the Hippocampus on Spatial Learning, Memory and NMDA Receptor Function during Aging. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0044560

Chicago Manual of Style (16th Edition):

Lee, Wei-Hua. “The Influence of Viral Vector Delivery of Superoxide Dismutase and Catalase to the Hippocampus on Spatial Learning, Memory and NMDA Receptor Function during Aging.” 2012. Doctoral Dissertation, University of Florida. Accessed May 07, 2021. https://ufdc.ufl.edu/UFE0044560.

MLA Handbook (7th Edition):

Lee, Wei-Hua. “The Influence of Viral Vector Delivery of Superoxide Dismutase and Catalase to the Hippocampus on Spatial Learning, Memory and NMDA Receptor Function during Aging.” 2012. Web. 07 May 2021.

Vancouver:

Lee W. The Influence of Viral Vector Delivery of Superoxide Dismutase and Catalase to the Hippocampus on Spatial Learning, Memory and NMDA Receptor Function during Aging. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2021 May 07]. Available from: https://ufdc.ufl.edu/UFE0044560.

Council of Science Editors:

Lee W. The Influence of Viral Vector Delivery of Superoxide Dismutase and Catalase to the Hippocampus on Spatial Learning, Memory and NMDA Receptor Function during Aging. [Doctoral Dissertation]. University of Florida; 2012. Available from: https://ufdc.ufl.edu/UFE0044560

24. Evelyn I. Nwabuisi (7973555). Mechanisms of ApoEIsoform Effects in Alzheimer’s Disease.

Degree: 2013, University of Illinois – Chicago

 Alzheimer’s disease (AD) is the primary cause of dementia in the elderly. Mutations that increase the 42 amino acid form of the peptide amyloid-beta (Aβ42)… (more)

Subjects/Keywords: Uncategorized; Alzheimer's disease; dendritic spines; AMPAR; NMDAR; synapses; spine morphology; oligomers; apolipoprotein E; amyloid beta

…9 4. Aβ42 reduces AMPAR surface expression and function… …transporter A1 AD Alzheimer’s disease AICD APP intracellular domain AMPAR alpha-amino-3… …localized on dendritic spines. Both NMDAR and AMPAR are cation channels gated by the… …outcomes. The Mg2+ block is released by enhanced AMPAR-mediated depolarization, which results… …recruitment and expression of AMPAR at the synapse, and increases the stability, size and number of… 

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APA (6th Edition):

(7973555), E. I. N. (2013). Mechanisms of ApoEIsoform Effects in Alzheimer’s Disease. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9755

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

(7973555), Evelyn I. Nwabuisi. “Mechanisms of ApoEIsoform Effects in Alzheimer’s Disease.” 2013. Thesis, University of Illinois – Chicago. Accessed May 07, 2021. http://hdl.handle.net/10027/9755.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

(7973555), Evelyn I. Nwabuisi. “Mechanisms of ApoEIsoform Effects in Alzheimer’s Disease.” 2013. Web. 07 May 2021.

Vancouver:

(7973555) EIN. Mechanisms of ApoEIsoform Effects in Alzheimer’s Disease. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2021 May 07]. Available from: http://hdl.handle.net/10027/9755.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

(7973555) EIN. Mechanisms of ApoEIsoform Effects in Alzheimer’s Disease. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/9755

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

25. Cueva Vargas, Jorge Luis. Role of Reactive Gliosis and Neuroinflammation in Experimental Glaucoma.

Degree: 2016, Université de Montréal

Subjects/Keywords: Cellules ganglionnaires de la rétine; Ibudilast; Glaucome; Phosphodiestérase-4; Adénosine monophosphate cyclique; Neuroinflammation; Facteur de nécrose tumorale alpha; CP-AMPAR; Cyclic adenosine monophosphate; Tumor necrosis factor alpha; Retinal ganglion cell; Biology - Neuroscience / Biologie - Neurologie (UMI : 0317)

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APA (6th Edition):

Cueva Vargas, J. L. (2016). Role of Reactive Gliosis and Neuroinflammation in Experimental Glaucoma. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cueva Vargas, Jorge Luis. “Role of Reactive Gliosis and Neuroinflammation in Experimental Glaucoma.” 2016. Thesis, Université de Montréal. Accessed May 07, 2021. http://hdl.handle.net/1866/13531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cueva Vargas, Jorge Luis. “Role of Reactive Gliosis and Neuroinflammation in Experimental Glaucoma.” 2016. Web. 07 May 2021.

Vancouver:

Cueva Vargas JL. Role of Reactive Gliosis and Neuroinflammation in Experimental Glaucoma. [Internet] [Thesis]. Université de Montréal; 2016. [cited 2021 May 07]. Available from: http://hdl.handle.net/1866/13531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cueva Vargas JL. Role of Reactive Gliosis and Neuroinflammation in Experimental Glaucoma. [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

26. Xing, Paul. Implication de Syngap1 dans la transmission GABAergique et la plasticité synaptique.

Degree: 2016, Université de Montréal

Subjects/Keywords: autisme; AMPAR; déficience intellectuelle; équilibre excitation/inhibition; hippocampe; interneurones GABAergiques; LTP; LTD; néocortex; mEPSCs; mIPSCs; NMDAR; Syngap1; autism; excitation/inhibition balance; GABAergic interneurons; hippocampus; intellectual disability; Biology - Neuroscience / Biologie - Neurologie (UMI : 0317)

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APA (6th Edition):

Xing, P. (2016). Implication de Syngap1 dans la transmission GABAergique et la plasticité synaptique. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13793

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xing, Paul. “Implication de Syngap1 dans la transmission GABAergique et la plasticité synaptique.” 2016. Thesis, Université de Montréal. Accessed May 07, 2021. http://hdl.handle.net/1866/13793.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xing, Paul. “Implication de Syngap1 dans la transmission GABAergique et la plasticité synaptique.” 2016. Web. 07 May 2021.

Vancouver:

Xing P. Implication de Syngap1 dans la transmission GABAergique et la plasticité synaptique. [Internet] [Thesis]. Université de Montréal; 2016. [cited 2021 May 07]. Available from: http://hdl.handle.net/1866/13793.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xing P. Implication de Syngap1 dans la transmission GABAergique et la plasticité synaptique. [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13793

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

27. Livingstone, Rhys Warren. The role of calcium-permeable AMPA receptors and arc in secreted amyloid precursor protein alpha-mediated plasticity .

Degree: University of Otago

 The orchestrated regulation of the α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-subtype of glutamate receptors by neuronal activity and neuromodulators is critical to the expression of both long-term potentiation… (more)

Subjects/Keywords: New Zealand; Alzheimer's Disease; Arc; AMPA receptor; Calcium-Permeable AMPA receptor; CP-AMPAR; Hippocampus; Long-term Potentiation; LTP; Immunohistochemistry; Immunocytochemistry; FUNCAT-PLA; sAPPα; Neurotrophin; Cell Culture; Protein Synthesis; Electrophysiology; Gene Expression; Neuron; Proximity Ligase Assay

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Livingstone, R. W. (n.d.). The role of calcium-permeable AMPA receptors and arc in secreted amyloid precursor protein alpha-mediated plasticity . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/10485

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Livingstone, Rhys Warren. “The role of calcium-permeable AMPA receptors and arc in secreted amyloid precursor protein alpha-mediated plasticity .” Doctoral Dissertation, University of Otago. Accessed May 07, 2021. http://hdl.handle.net/10523/10485.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Livingstone, Rhys Warren. “The role of calcium-permeable AMPA receptors and arc in secreted amyloid precursor protein alpha-mediated plasticity .” Web. 07 May 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Livingstone RW. The role of calcium-permeable AMPA receptors and arc in secreted amyloid precursor protein alpha-mediated plasticity . [Internet] [Doctoral dissertation]. University of Otago; [cited 2021 May 07]. Available from: http://hdl.handle.net/10523/10485.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Livingstone RW. The role of calcium-permeable AMPA receptors and arc in secreted amyloid precursor protein alpha-mediated plasticity . [Doctoral Dissertation]. University of Otago; Available from: http://hdl.handle.net/10523/10485

Note: this citation may be lacking information needed for this citation format:
No year of publication.

28. James Gocel (7922525). Ionotropic Glutamate Receptors in Anterior Piriform Cortex.

Degree: 2012, University of Illinois – Chicago

 The influence of anatomical, developmental and degenerative factors on the function of synaptically expressed ionotropic glutamate receptors was assessed in murine models. Recordings were obtained… (more)

Subjects/Keywords: Uncategorized; NMDAR; AMPAR; glutamate; piriform cortex; cognition; electrophysiology; α-amino-3-hydroxyl-5-methyl-4-isoxazole propionic acid receptor; N-methyl-D-aspartate receptor

…current TARP transmembrane AMPAR regulatory protein TTX tetrodotoxin UTR untranslated… …transient changes in voltage at the synapse. Temporal and spatial summation of AMPAR-mediated… …types; however, AMPAR and NMDAR function is essential for subsequent signaling processes that… …residues on AMPAR subunits prohibit calcium influx constitutively whereas NMDAR subunits form… …consequence of postsynaptic organization of NMDARs, unlike that of AMPAR organization, is that… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

(7922525), J. G. (2012). Ionotropic Glutamate Receptors in Anterior Piriform Cortex. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

(7922525), James Gocel. “Ionotropic Glutamate Receptors in Anterior Piriform Cortex.” 2012. Thesis, University of Illinois – Chicago. Accessed May 07, 2021. http://hdl.handle.net/10027/9598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

(7922525), James Gocel. “Ionotropic Glutamate Receptors in Anterior Piriform Cortex.” 2012. Web. 07 May 2021.

Vancouver:

(7922525) JG. Ionotropic Glutamate Receptors in Anterior Piriform Cortex. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2021 May 07]. Available from: http://hdl.handle.net/10027/9598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

(7922525) JG. Ionotropic Glutamate Receptors in Anterior Piriform Cortex. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Lado, Wudu E. The Neural Substrate of Sex Pheromone Signalling in Male Goldfish (Carassius auratus) .

Degree: 2012, University of Ottawa

 The transmission of sex pheromone-mediated signals is essential for goldfish reproduction. However, the neural pathways underlying this reproductive signalling pathway in the goldfish brain is… (more)

Subjects/Keywords: Goldfish, reproduction, male, preoptic area, ventral telencephalon, sex pheromones; NMDAR, AMPAR, NMDA, AMAPA, glutamate,; ventral telencephali pars ventralis, septal region; Prostaglandin F 2alpha, 17 alpha,20 beta-dihydroxy-4-pregnen-3-one; spawning, microarray analysis, patch-clamp electrophysiology; goldfish

…CVL: coefficient of variation of the methylisoxazole-4-propionate latency AMPAR: AMPA… …AMPAR MWW: Mann-Whitney-Wilcoxon subunit) NaCl: sodium chloride GnRH: gonadotropin… …Meldrum, 2000). α-Amino-3-hydroxy-5-methylisoxazole-4-propionate receptors (AMPAR)… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lado, W. E. (2012). The Neural Substrate of Sex Pheromone Signalling in Male Goldfish (Carassius auratus) . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/23460

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lado, Wudu E. “The Neural Substrate of Sex Pheromone Signalling in Male Goldfish (Carassius auratus) .” 2012. Thesis, University of Ottawa. Accessed May 07, 2021. http://hdl.handle.net/10393/23460.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lado, Wudu E. “The Neural Substrate of Sex Pheromone Signalling in Male Goldfish (Carassius auratus) .” 2012. Web. 07 May 2021.

Vancouver:

Lado WE. The Neural Substrate of Sex Pheromone Signalling in Male Goldfish (Carassius auratus) . [Internet] [Thesis]. University of Ottawa; 2012. [cited 2021 May 07]. Available from: http://hdl.handle.net/10393/23460.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lado WE. The Neural Substrate of Sex Pheromone Signalling in Male Goldfish (Carassius auratus) . [Thesis]. University of Ottawa; 2012. Available from: http://hdl.handle.net/10393/23460

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.