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You searched for subject:(A precursor protein ). Showing records 1 – 30 of 65233 total matches.

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University of Kentucky

1. Zhang, Yu-Qian. INDUCTION OF THE HEAT SHOCK RESPONSE TO PROTECT AGAINST POLYGLUTAMINE DISEASES AND THE ROLE OF PROTEIN SUMOYLATION IN LAMINOPATHIES AND ALZHEIMER'S DISEASE.

Degree: 2008, University of Kentucky

 Heat shock proteins function as molecular chaperones which help protein folding and prevent protein aggregation. My study shows that celastrol, a pharmacological compound capable of… (more)

Subjects/Keywords: Celastrol; Polyglutamine Diseases; Protein Sumoylation; Lamin A; Amyloid Precursor Protein; Medical Biochemistry

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APA (6th Edition):

Zhang, Y. (2008). INDUCTION OF THE HEAT SHOCK RESPONSE TO PROTECT AGAINST POLYGLUTAMINE DISEASES AND THE ROLE OF PROTEIN SUMOYLATION IN LAMINOPATHIES AND ALZHEIMER'S DISEASE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/632

Chicago Manual of Style (16th Edition):

Zhang, Yu-Qian. “INDUCTION OF THE HEAT SHOCK RESPONSE TO PROTECT AGAINST POLYGLUTAMINE DISEASES AND THE ROLE OF PROTEIN SUMOYLATION IN LAMINOPATHIES AND ALZHEIMER'S DISEASE.” 2008. Doctoral Dissertation, University of Kentucky. Accessed April 11, 2021. https://uknowledge.uky.edu/gradschool_diss/632.

MLA Handbook (7th Edition):

Zhang, Yu-Qian. “INDUCTION OF THE HEAT SHOCK RESPONSE TO PROTECT AGAINST POLYGLUTAMINE DISEASES AND THE ROLE OF PROTEIN SUMOYLATION IN LAMINOPATHIES AND ALZHEIMER'S DISEASE.” 2008. Web. 11 Apr 2021.

Vancouver:

Zhang Y. INDUCTION OF THE HEAT SHOCK RESPONSE TO PROTECT AGAINST POLYGLUTAMINE DISEASES AND THE ROLE OF PROTEIN SUMOYLATION IN LAMINOPATHIES AND ALZHEIMER'S DISEASE. [Internet] [Doctoral dissertation]. University of Kentucky; 2008. [cited 2021 Apr 11]. Available from: https://uknowledge.uky.edu/gradschool_diss/632.

Council of Science Editors:

Zhang Y. INDUCTION OF THE HEAT SHOCK RESPONSE TO PROTECT AGAINST POLYGLUTAMINE DISEASES AND THE ROLE OF PROTEIN SUMOYLATION IN LAMINOPATHIES AND ALZHEIMER'S DISEASE. [Doctoral Dissertation]. University of Kentucky; 2008. Available from: https://uknowledge.uky.edu/gradschool_diss/632


University of Adelaide

2. Corrigan, Frances. The role of the amyloid precursor protein following traumatic brain injury.

Degree: 2011, University of Adelaide

 The amyloid precursor protein (APP) is known to increase following traumatic brain injury (TBI). It has been hypothesised that this increase in APP may be… (more)

Subjects/Keywords: amyloid precursor protein; traumatic brain injury; sAPPα

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APA (6th Edition):

Corrigan, F. (2011). The role of the amyloid precursor protein following traumatic brain injury. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/80625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Corrigan, Frances. “The role of the amyloid precursor protein following traumatic brain injury.” 2011. Thesis, University of Adelaide. Accessed April 11, 2021. http://hdl.handle.net/2440/80625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Corrigan, Frances. “The role of the amyloid precursor protein following traumatic brain injury.” 2011. Web. 11 Apr 2021.

Vancouver:

Corrigan F. The role of the amyloid precursor protein following traumatic brain injury. [Internet] [Thesis]. University of Adelaide; 2011. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2440/80625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Corrigan F. The role of the amyloid precursor protein following traumatic brain injury. [Thesis]. University of Adelaide; 2011. Available from: http://hdl.handle.net/2440/80625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Puig, Kendra L. Role Of The Alzheimer's Amyloid Precursor Protein In High Fat Diet Induced Obesity And Regulating Macrophage Phenotype.

Degree: PhD, Biomedical Sciences, 2013, University of North Dakota

  Amyloid precursor protein (APP) derived amyloid beta peptides have been extensively investigated in Alzheimer's disease pathology of the brain. However, the function of full… (more)

Subjects/Keywords: amyloid precursor protein; inflammation; intestine; macrophage; obesity

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APA (6th Edition):

Puig, K. L. (2013). Role Of The Alzheimer's Amyloid Precursor Protein In High Fat Diet Induced Obesity And Regulating Macrophage Phenotype. (Doctoral Dissertation). University of North Dakota. Retrieved from https://commons.und.edu/theses/1469

Chicago Manual of Style (16th Edition):

Puig, Kendra L. “Role Of The Alzheimer's Amyloid Precursor Protein In High Fat Diet Induced Obesity And Regulating Macrophage Phenotype.” 2013. Doctoral Dissertation, University of North Dakota. Accessed April 11, 2021. https://commons.und.edu/theses/1469.

MLA Handbook (7th Edition):

Puig, Kendra L. “Role Of The Alzheimer's Amyloid Precursor Protein In High Fat Diet Induced Obesity And Regulating Macrophage Phenotype.” 2013. Web. 11 Apr 2021.

Vancouver:

Puig KL. Role Of The Alzheimer's Amyloid Precursor Protein In High Fat Diet Induced Obesity And Regulating Macrophage Phenotype. [Internet] [Doctoral dissertation]. University of North Dakota; 2013. [cited 2021 Apr 11]. Available from: https://commons.und.edu/theses/1469.

Council of Science Editors:

Puig KL. Role Of The Alzheimer's Amyloid Precursor Protein In High Fat Diet Induced Obesity And Regulating Macrophage Phenotype. [Doctoral Dissertation]. University of North Dakota; 2013. Available from: https://commons.und.edu/theses/1469


University of Melbourne

4. SPOERRI, LOREDANA. Investigating the functional role of the amyloid precursor protein’s copper binding domain.

Degree: 2011, University of Melbourne

 Alzheimer’s disease is a progressive and eventually fatal neurodegenerative disorder characterized by specific proteins deposition in the brain: amyloid plaques and tau tangles. While age… (more)

Subjects/Keywords: amyloid precursor protein; copper binding domain

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APA (6th Edition):

SPOERRI, L. (2011). Investigating the functional role of the amyloid precursor protein’s copper binding domain. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36752

Chicago Manual of Style (16th Edition):

SPOERRI, LOREDANA. “Investigating the functional role of the amyloid precursor protein’s copper binding domain.” 2011. Doctoral Dissertation, University of Melbourne. Accessed April 11, 2021. http://hdl.handle.net/11343/36752.

MLA Handbook (7th Edition):

SPOERRI, LOREDANA. “Investigating the functional role of the amyloid precursor protein’s copper binding domain.” 2011. Web. 11 Apr 2021.

Vancouver:

SPOERRI L. Investigating the functional role of the amyloid precursor protein’s copper binding domain. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/11343/36752.

Council of Science Editors:

SPOERRI L. Investigating the functional role of the amyloid precursor protein’s copper binding domain. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/36752

5. Luu, Luan. Defining the function of Amyloid Precursor Protein dimerisation in neuritogenesis.

Degree: 2017, University of Melbourne

 Amyloid Precursor Protein (APP) is known to be primarily involved with Alzheimer’s disease; however APP is also involved with neurogenesis, synaptic plasticity and neuroprotection. Many… (more)

Subjects/Keywords: Amyloid Precursor Protein; dimerisation; neurite outgrowth

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APA (6th Edition):

Luu, L. (2017). Defining the function of Amyloid Precursor Protein dimerisation in neuritogenesis. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/194187

Chicago Manual of Style (16th Edition):

Luu, Luan. “Defining the function of Amyloid Precursor Protein dimerisation in neuritogenesis.” 2017. Doctoral Dissertation, University of Melbourne. Accessed April 11, 2021. http://hdl.handle.net/11343/194187.

MLA Handbook (7th Edition):

Luu, Luan. “Defining the function of Amyloid Precursor Protein dimerisation in neuritogenesis.” 2017. Web. 11 Apr 2021.

Vancouver:

Luu L. Defining the function of Amyloid Precursor Protein dimerisation in neuritogenesis. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/11343/194187.

Council of Science Editors:

Luu L. Defining the function of Amyloid Precursor Protein dimerisation in neuritogenesis. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/194187


University of Georgia

6. Ha, Sangdeuk. Physiological roles of Hirano bodies.

Degree: 2014, University of Georgia

 Hirano bodies are actin rich, rod-shaped, cytoplasmic, paracrystalline inclusions found predominantly in the central nervous system in association with a variety of conditions including aging… (more)

Subjects/Keywords: Hirano bodies; F-actin; a C-terminal fragment of amyloid-Øâ precursor protein; Fe65; Alzheimer¡ s Disease; apoptosis; transcriptional activity; Cre/loxP recombination system; Mouse model.

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APA (6th Edition):

Ha, S. (2014). Physiological roles of Hirano bodies. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/24902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ha, Sangdeuk. “Physiological roles of Hirano bodies.” 2014. Thesis, University of Georgia. Accessed April 11, 2021. http://hdl.handle.net/10724/24902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ha, Sangdeuk. “Physiological roles of Hirano bodies.” 2014. Web. 11 Apr 2021.

Vancouver:

Ha S. Physiological roles of Hirano bodies. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10724/24902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ha S. Physiological roles of Hirano bodies. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/24902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

7. DelBove, Claire Elise. Visualizing APP trafficking and processing reveals its functional relationship with neuronal membrane cholesterol.

Degree: PhD, Pharmacology, 2018, Vanderbilt University

 Amyloid precursor protein (APP) is a type I transmembrane protein and has been studied extensively due to its relevance to Alzheimer’s disease. However, its subcellular… (more)

Subjects/Keywords: Alzheimers disease; choleserol; protein trafficking; amyloid precursor protein

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APA (6th Edition):

DelBove, C. E. (2018). Visualizing APP trafficking and processing reveals its functional relationship with neuronal membrane cholesterol. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13502

Chicago Manual of Style (16th Edition):

DelBove, Claire Elise. “Visualizing APP trafficking and processing reveals its functional relationship with neuronal membrane cholesterol.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 11, 2021. http://hdl.handle.net/1803/13502.

MLA Handbook (7th Edition):

DelBove, Claire Elise. “Visualizing APP trafficking and processing reveals its functional relationship with neuronal membrane cholesterol.” 2018. Web. 11 Apr 2021.

Vancouver:

DelBove CE. Visualizing APP trafficking and processing reveals its functional relationship with neuronal membrane cholesterol. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1803/13502.

Council of Science Editors:

DelBove CE. Visualizing APP trafficking and processing reveals its functional relationship with neuronal membrane cholesterol. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/13502


Drexel University

8. Nguyen, Phuong Thi Tam. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.

Degree: 2017, Drexel University

The average person spends several hours a day behind the wheel of their vehicles, which are usually equipped with on-board computers capable of collecting real-time… (more)

Subjects/Keywords: Biology; Alzheimer's disease; Drosophila; Amyloid beta-protein; Amyloid beta-protein precursor

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APA (6th Edition):

Nguyen, P. T. T. (2017). Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Phuong Thi Tam. “Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.” 2017. Thesis, Drexel University. Accessed April 11, 2021. http://hdl.handle.net/1860/idea:7473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Phuong Thi Tam. “Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease.” 2017. Web. 11 Apr 2021.

Vancouver:

Nguyen PTT. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. [Internet] [Thesis]. Drexel University; 2017. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1860/idea:7473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen PTT. Phytosterol Cyclopamine Rescues Structural and Behavioral Deficits in a Drosophila model of Alzheimer's Disease. [Thesis]. Drexel University; 2017. Available from: http://hdl.handle.net/1860/idea:7473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

9. Truong, Phan Hong. Investigating the role of Amyloid Precursor-Like Protein 2 in Motor Neurone Disease.

Degree: 2019, University of Melbourne

 Motor neurone disease (MND) is a fatal human neurodegenerative disorder. The most common form of MND is amyotrophic lateral sclerosis (ALS). MND is characterised by… (more)

Subjects/Keywords: amyloid precursor – like protein 2; motor neurone disease; amyloid precursor protein; SOD1-G37R; superoxide dismutase; motor neurons; sex differences; knockout; transgenic

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APA (6th Edition):

Truong, P. H. (2019). Investigating the role of Amyloid Precursor-Like Protein 2 in Motor Neurone Disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/227648

Chicago Manual of Style (16th Edition):

Truong, Phan Hong. “Investigating the role of Amyloid Precursor-Like Protein 2 in Motor Neurone Disease.” 2019. Doctoral Dissertation, University of Melbourne. Accessed April 11, 2021. http://hdl.handle.net/11343/227648.

MLA Handbook (7th Edition):

Truong, Phan Hong. “Investigating the role of Amyloid Precursor-Like Protein 2 in Motor Neurone Disease.” 2019. Web. 11 Apr 2021.

Vancouver:

Truong PH. Investigating the role of Amyloid Precursor-Like Protein 2 in Motor Neurone Disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2019. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/11343/227648.

Council of Science Editors:

Truong PH. Investigating the role of Amyloid Precursor-Like Protein 2 in Motor Neurone Disease. [Doctoral Dissertation]. University of Melbourne; 2019. Available from: http://hdl.handle.net/11343/227648


NSYSU

10. Li, Yu-Ru. To Explore β-Amyloid Induced Oxidative DNA Damage and Repair in Rat Primary Cortical Neurons.

Degree: Master, Biological Sciences, 2016, NSYSU

 Alzheimer's disease (AD) is one of the common form of age-related neurodegenerative diseases and the leading cause of senile dementia. In the beta-amyloid (Aβ) cascade… (more)

Subjects/Keywords: β-amyloid; Amyloid precursor protein; Alzheimer's disease; DNA repair; Neurotoxicity

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APA (6th Edition):

Li, Y. (2016). To Explore β-Amyloid Induced Oxidative DNA Damage and Repair in Rat Primary Cortical Neurons. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1103115-165537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Yu-Ru. “To Explore β-Amyloid Induced Oxidative DNA Damage and Repair in Rat Primary Cortical Neurons.” 2016. Thesis, NSYSU. Accessed April 11, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1103115-165537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Yu-Ru. “To Explore β-Amyloid Induced Oxidative DNA Damage and Repair in Rat Primary Cortical Neurons.” 2016. Web. 11 Apr 2021.

Vancouver:

Li Y. To Explore β-Amyloid Induced Oxidative DNA Damage and Repair in Rat Primary Cortical Neurons. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Apr 11]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1103115-165537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. To Explore β-Amyloid Induced Oxidative DNA Damage and Repair in Rat Primary Cortical Neurons. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-1103115-165537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stony Brook University

11. Chiang, Hsueh-Chiang. Drosophila Model of Alzheimer's Disease .

Degree: 2009, Stony Brook University

Subjects/Keywords: Beta-amyloid precursor protein

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APA (6th Edition):

Chiang, H. (2009). Drosophila Model of Alzheimer's Disease . (Thesis). Stony Brook University. Retrieved from http://hdl.handle.net/1951/52212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chiang, Hsueh-Chiang. “Drosophila Model of Alzheimer's Disease .” 2009. Thesis, Stony Brook University. Accessed April 11, 2021. http://hdl.handle.net/1951/52212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chiang, Hsueh-Chiang. “Drosophila Model of Alzheimer's Disease .” 2009. Web. 11 Apr 2021.

Vancouver:

Chiang H. Drosophila Model of Alzheimer's Disease . [Internet] [Thesis]. Stony Brook University; 2009. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1951/52212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chiang H. Drosophila Model of Alzheimer's Disease . [Thesis]. Stony Brook University; 2009. Available from: http://hdl.handle.net/1951/52212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

12. Dawkins, E. Role of Phosphoinositides in the biology of the amyloid precursor protein.

Degree: 2014, University of Tasmania

 Alzheimer’s disease (AD) is the leading cause of dementia in the elderly. In countries with aging populations, such as Australia, the prevalence of AD is… (more)

Subjects/Keywords: Alzheimer's disease; Phosphoinositides; beta-amyloid precursor protein; Phosphatidylinositol; phosphate; heparin.

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APA (6th Edition):

Dawkins, E. (2014). Role of Phosphoinositides in the biology of the amyloid precursor protein. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dawkins, E. “Role of Phosphoinositides in the biology of the amyloid precursor protein.” 2014. Thesis, University of Tasmania. Accessed April 11, 2021. https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dawkins, E. “Role of Phosphoinositides in the biology of the amyloid precursor protein.” 2014. Web. 11 Apr 2021.

Vancouver:

Dawkins E. Role of Phosphoinositides in the biology of the amyloid precursor protein. [Internet] [Thesis]. University of Tasmania; 2014. [cited 2021 Apr 11]. Available from: https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dawkins E. Role of Phosphoinositides in the biology of the amyloid precursor protein. [Thesis]. University of Tasmania; 2014. Available from: https://eprints.utas.edu.au/18274/1/front-Dawkins-thesis.pdf ; https://eprints.utas.edu.au/18274/2/Whole-Dawkins-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Warren, Rebekkah Lynn. Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan.

Degree: 2011, University of Texas Southwestern Medical Center

 In vitro alterations in cellular cholesterol content or synthesis affect the cleavage of amyloid precursor protein (APP) to amyloidogenic peptides characteristic of Alzheimer’s disease (AD).… (more)

Subjects/Keywords: Amyloid beta-Protein Precursor; Alzheimer Disease; Lipid Metabolism

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APA (6th Edition):

Warren, R. L. (2011). Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Warren, Rebekkah Lynn. “Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed April 11, 2021. http://hdl.handle.net/2152.5/891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Warren, Rebekkah Lynn. “Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan.” 2011. Web. 11 Apr 2021.

Vancouver:

Warren RL. Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2152.5/891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Warren RL. Genetic Reduction of Cholesterol Synthesis in the Mouse Brain Does Not Affect Amyloid Formation in an Alzheimer’s Disease Model, but Does Extend Lifespan. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

14. Plummer, Stephanie Lauren. Investigating amyloid precursor protein derivatives as novel therapeutic agents following traumatic brain injury.

Degree: 2017, University of Adelaide

 Despite the significant health and economic burden that traumatic brain injury (TBI) places on society, the development of successful therapeutic agents to reduce its burden… (more)

Subjects/Keywords: Amyloid precursor protein; traumatic brain injury; inflamamtion; axonal injury

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APA (6th Edition):

Plummer, S. L. (2017). Investigating amyloid precursor protein derivatives as novel therapeutic agents following traumatic brain injury. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/126463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Plummer, Stephanie Lauren. “Investigating amyloid precursor protein derivatives as novel therapeutic agents following traumatic brain injury.” 2017. Thesis, University of Adelaide. Accessed April 11, 2021. http://hdl.handle.net/2440/126463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Plummer, Stephanie Lauren. “Investigating amyloid precursor protein derivatives as novel therapeutic agents following traumatic brain injury.” 2017. Web. 11 Apr 2021.

Vancouver:

Plummer SL. Investigating amyloid precursor protein derivatives as novel therapeutic agents following traumatic brain injury. [Internet] [Thesis]. University of Adelaide; 2017. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2440/126463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Plummer SL. Investigating amyloid precursor protein derivatives as novel therapeutic agents following traumatic brain injury. [Thesis]. University of Adelaide; 2017. Available from: http://hdl.handle.net/2440/126463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

15. Morris, Gary Paul. Secreted amyloid precursor protein-α mediates neuroprotection and gene expression .

Degree: 2011, University of Otago

 Alzheimer’s disease (AD) is rapidly becoming one of the most significant challenges facing modern medicine. Currently there is no cure for this neurodegenerative disease. Memory… (more)

Subjects/Keywords: secreted amyloid precursor protein; Alzheimer's; gene expression; RER; sAPPα

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APA (6th Edition):

Morris, G. P. (2011). Secreted amyloid precursor protein-α mediates neuroprotection and gene expression . (Masters Thesis). University of Otago. Retrieved from http://hdl.handle.net/10523/2026

Chicago Manual of Style (16th Edition):

Morris, Gary Paul. “Secreted amyloid precursor protein-α mediates neuroprotection and gene expression .” 2011. Masters Thesis, University of Otago. Accessed April 11, 2021. http://hdl.handle.net/10523/2026.

MLA Handbook (7th Edition):

Morris, Gary Paul. “Secreted amyloid precursor protein-α mediates neuroprotection and gene expression .” 2011. Web. 11 Apr 2021.

Vancouver:

Morris GP. Secreted amyloid precursor protein-α mediates neuroprotection and gene expression . [Internet] [Masters thesis]. University of Otago; 2011. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10523/2026.

Council of Science Editors:

Morris GP. Secreted amyloid precursor protein-α mediates neuroprotection and gene expression . [Masters Thesis]. University of Otago; 2011. Available from: http://hdl.handle.net/10523/2026


University of Manchester

16. Andrew, Robert. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.

Degree: 2015, University of Manchester

 Dementia, the most common cause of which is Alzheimer’s disease (AD), currently affects 850,000 people in the UK, a figure set to rise to over… (more)

Subjects/Keywords: Alzheimers disease; Amyloid precursor protein; Amyloid-beta; interactome; Proteolysis

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APA (6th Edition):

Andrew, R. (2015). Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080

Chicago Manual of Style (16th Edition):

Andrew, Robert. “Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.” 2015. Doctoral Dissertation, University of Manchester. Accessed April 11, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080.

MLA Handbook (7th Edition):

Andrew, Robert. “Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions.” 2015. Web. 11 Apr 2021.

Vancouver:

Andrew R. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 Apr 11]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080.

Council of Science Editors:

Andrew R. Differential Proteolysis of the Amyloid Precursor Protein Isoforms: The Role of Cellular Location and Protein-Protein Interactions. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:280080


University of Illinois – Chicago

17. Demars, Michael P. Soluble Amyloid Precursor Protein Regulates Neurogenesis: Implications for Brain Repair.

Degree: 2012, University of Illinois – Chicago

 Amyloid precursor protein (APP) has been studied extensively in the pathophysiology of Alzheimer’s disease due to the fact that mutations in APP are causative of… (more)

Subjects/Keywords: amyloid precursor protein; neurogenesis; neural progenitor cell; proliferation; Alzheimer's disease

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APA (6th Edition):

Demars, M. P. (2012). Soluble Amyloid Precursor Protein Regulates Neurogenesis: Implications for Brain Repair. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Demars, Michael P. “Soluble Amyloid Precursor Protein Regulates Neurogenesis: Implications for Brain Repair.” 2012. Thesis, University of Illinois – Chicago. Accessed April 11, 2021. http://hdl.handle.net/10027/9149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Demars, Michael P. “Soluble Amyloid Precursor Protein Regulates Neurogenesis: Implications for Brain Repair.” 2012. Web. 11 Apr 2021.

Vancouver:

Demars MP. Soluble Amyloid Precursor Protein Regulates Neurogenesis: Implications for Brain Repair. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10027/9149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Demars MP. Soluble Amyloid Precursor Protein Regulates Neurogenesis: Implications for Brain Repair. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bath

18. Hammond, Victoria. α7 nicotinic acetylcholine receptors at the glutamatergic synapse.

Degree: PhD, 2014, University of Bath

 Nicotinic acetylcholine receptor (nAChR) activation is neuroprotective and nicotine is a cognitive enhancer. Loss of nAChRs, deposition of tau neurofibrillary tangles, cleavage of amyloid precursor(more)

Subjects/Keywords: 612.8; nicotinic acetylcholine receptors; amyloid precursor protein; Neuron; Microglia

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APA (6th Edition):

Hammond, V. (2014). α7 nicotinic acetylcholine receptors at the glutamatergic synapse. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/7-nicotinic-acetylcholine-receptors-at-the-glutamatergic-synapse(8e4bcf14-fe9b-446a-a56e-fda3ba2c2eb4).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633163

Chicago Manual of Style (16th Edition):

Hammond, Victoria. “α7 nicotinic acetylcholine receptors at the glutamatergic synapse.” 2014. Doctoral Dissertation, University of Bath. Accessed April 11, 2021. https://researchportal.bath.ac.uk/en/studentthesis/7-nicotinic-acetylcholine-receptors-at-the-glutamatergic-synapse(8e4bcf14-fe9b-446a-a56e-fda3ba2c2eb4).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633163.

MLA Handbook (7th Edition):

Hammond, Victoria. “α7 nicotinic acetylcholine receptors at the glutamatergic synapse.” 2014. Web. 11 Apr 2021.

Vancouver:

Hammond V. α7 nicotinic acetylcholine receptors at the glutamatergic synapse. [Internet] [Doctoral dissertation]. University of Bath; 2014. [cited 2021 Apr 11]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/7-nicotinic-acetylcholine-receptors-at-the-glutamatergic-synapse(8e4bcf14-fe9b-446a-a56e-fda3ba2c2eb4).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633163.

Council of Science Editors:

Hammond V. α7 nicotinic acetylcholine receptors at the glutamatergic synapse. [Doctoral Dissertation]. University of Bath; 2014. Available from: https://researchportal.bath.ac.uk/en/studentthesis/7-nicotinic-acetylcholine-receptors-at-the-glutamatergic-synapse(8e4bcf14-fe9b-446a-a56e-fda3ba2c2eb4).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633163


Université Catholique de Louvain

19. Perrin, Florian. Molecular determinants regulating dimerization, processing and signaling of the amyloid precursor protein.

Degree: 2019, Université Catholique de Louvain

Alzheimer’s disease (AD) is characterized by cognitive impairments such as apraxia, aphasia and memory decline. The two majors hallmarks of AD are neurofibrillary tangles and… (more)

Subjects/Keywords: Alzheimer's disease; Amyloid Precursor Protein; Dimerization; Oligomerization; Orientation; Processing

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APA (6th Edition):

Perrin, F. (2019). Molecular determinants regulating dimerization, processing and signaling of the amyloid precursor protein. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/219632

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perrin, Florian. “Molecular determinants regulating dimerization, processing and signaling of the amyloid precursor protein.” 2019. Thesis, Université Catholique de Louvain. Accessed April 11, 2021. http://hdl.handle.net/2078.1/219632.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perrin, Florian. “Molecular determinants regulating dimerization, processing and signaling of the amyloid precursor protein.” 2019. Web. 11 Apr 2021.

Vancouver:

Perrin F. Molecular determinants regulating dimerization, processing and signaling of the amyloid precursor protein. [Internet] [Thesis]. Université Catholique de Louvain; 2019. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2078.1/219632.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perrin F. Molecular determinants regulating dimerization, processing and signaling of the amyloid precursor protein. [Thesis]. Université Catholique de Louvain; 2019. Available from: http://hdl.handle.net/2078.1/219632

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

20. BARAKAT, ADEL. Investigation of neuregulin1 processing by BACE1 and gamma-secretase in schizophrenia.

Degree: 2014, University of Melbourne

 Schizophrenia is a multifactorial complex psychiatric illness, which implicates many genes, among which is neuregulin1 (Nrg1). BACE1 and γ-secretase are known to be involved in… (more)

Subjects/Keywords: neuroscience; schizophrenia; secretase; BACE1; gamma secretase; neuregulin1; amyloid precursor protein

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APA (6th Edition):

BARAKAT, A. (2014). Investigation of neuregulin1 processing by BACE1 and gamma-secretase in schizophrenia. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/39642

Chicago Manual of Style (16th Edition):

BARAKAT, ADEL. “Investigation of neuregulin1 processing by BACE1 and gamma-secretase in schizophrenia.” 2014. Doctoral Dissertation, University of Melbourne. Accessed April 11, 2021. http://hdl.handle.net/11343/39642.

MLA Handbook (7th Edition):

BARAKAT, ADEL. “Investigation of neuregulin1 processing by BACE1 and gamma-secretase in schizophrenia.” 2014. Web. 11 Apr 2021.

Vancouver:

BARAKAT A. Investigation of neuregulin1 processing by BACE1 and gamma-secretase in schizophrenia. [Internet] [Doctoral dissertation]. University of Melbourne; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/11343/39642.

Council of Science Editors:

BARAKAT A. Investigation of neuregulin1 processing by BACE1 and gamma-secretase in schizophrenia. [Doctoral Dissertation]. University of Melbourne; 2014. Available from: http://hdl.handle.net/11343/39642


University of Melbourne

21. GAO, CHEN. Structural and biophysical characterisation of Alzheimer’s disease amyloid precursor protein.

Degree: 2015, University of Melbourne

 Alzheimer’s disease (AD) is an irreversible, neurodegenerative disorder most commonly affecting the aged population. Despite the growing incidence world wide, the disease remains untreatable due… (more)

Subjects/Keywords: amyloid precursor protein; Alzheimer's; dimerisation; X-ray crystallography; structural biology

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APA (6th Edition):

GAO, C. (2015). Structural and biophysical characterisation of Alzheimer’s disease amyloid precursor protein. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55553

Chicago Manual of Style (16th Edition):

GAO, CHEN. “Structural and biophysical characterisation of Alzheimer’s disease amyloid precursor protein.” 2015. Doctoral Dissertation, University of Melbourne. Accessed April 11, 2021. http://hdl.handle.net/11343/55553.

MLA Handbook (7th Edition):

GAO, CHEN. “Structural and biophysical characterisation of Alzheimer’s disease amyloid precursor protein.” 2015. Web. 11 Apr 2021.

Vancouver:

GAO C. Structural and biophysical characterisation of Alzheimer’s disease amyloid precursor protein. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/11343/55553.

Council of Science Editors:

GAO C. Structural and biophysical characterisation of Alzheimer’s disease amyloid precursor protein. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55553


University of Melbourne

22. TOH, WEI. The intracellular trafficking pathways of β-secretase and the amyloid precursor protein in Alzheimer’s disease.

Degree: 2015, University of Melbourne

 Alzheimer’s disease (AD) is characterized by the accumulation of amyloid plaques in the brain consisting of an aggregated form of β-amyloid peptide (Aβ) derived from… (more)

Subjects/Keywords: Amyloid precursor protein; Beta-secretase, BACE1; Membrane trafficking; Alzheimer's disease

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APA (6th Edition):

TOH, W. (2015). The intracellular trafficking pathways of β-secretase and the amyloid precursor protein in Alzheimer’s disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55767

Chicago Manual of Style (16th Edition):

TOH, WEI. “The intracellular trafficking pathways of β-secretase and the amyloid precursor protein in Alzheimer’s disease.” 2015. Doctoral Dissertation, University of Melbourne. Accessed April 11, 2021. http://hdl.handle.net/11343/55767.

MLA Handbook (7th Edition):

TOH, WEI. “The intracellular trafficking pathways of β-secretase and the amyloid precursor protein in Alzheimer’s disease.” 2015. Web. 11 Apr 2021.

Vancouver:

TOH W. The intracellular trafficking pathways of β-secretase and the amyloid precursor protein in Alzheimer’s disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/11343/55767.

Council of Science Editors:

TOH W. The intracellular trafficking pathways of β-secretase and the amyloid precursor protein in Alzheimer’s disease. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55767

23. Kirouac, Lisa. The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide.

Degree: 2016, University of South Florida

 It is widely accepted that A-beta (Aβ) generated from amyloid precursor protein (APP) oligomerizes and fibrillizes to form neuritic plaques in the Alzheimer’s disease (AD)… (more)

Subjects/Keywords: Alzheimer’s disease; amyloid-beta; amyloid precursor protein; mitosis; proliferation; Neurosciences

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APA (6th Edition):

Kirouac, L. (2016). The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kirouac, Lisa. “The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide.” 2016. Thesis, University of South Florida. Accessed April 11, 2021. https://scholarcommons.usf.edu/etd/6278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kirouac, Lisa. “The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide.” 2016. Web. 11 Apr 2021.

Vancouver:

Kirouac L. The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide. [Internet] [Thesis]. University of South Florida; 2016. [cited 2021 Apr 11]. Available from: https://scholarcommons.usf.edu/etd/6278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kirouac L. The Concerted Regulation of Intracellular Signaling by Amyloid Precursor Protein and Aβ Peptide. [Thesis]. University of South Florida; 2016. Available from: https://scholarcommons.usf.edu/etd/6278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

24. Christianson, Melissa Gottron. Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy .

Degree: 2012, Duke University

  Hypoxia is a profound stressor of the central nervous system implicated in numerous neurodegenerative diseases. While it is increasingly evident that the early effects… (more)

Subjects/Keywords: Neurosciences; Alzheimer's disease; Amyloid Beta; Amyloid Precursor Protein; Axon; Hypoxia; Neurodegeneration

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APA (6th Edition):

Christianson, M. G. (2012). Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Christianson, Melissa Gottron. “Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy .” 2012. Thesis, Duke University. Accessed April 11, 2021. http://hdl.handle.net/10161/5427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Christianson, Melissa Gottron. “Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy .” 2012. Web. 11 Apr 2021.

Vancouver:

Christianson MG. Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy . [Internet] [Thesis]. Duke University; 2012. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10161/5427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Christianson MG. Amyloid Precursor Protein-Dependent and -Independent Mechanisms in Hypoxia-Induced Axonopathy . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

25. Gupta, Nisha. Association of Hirano bodies and C31 fragment of the amyloid precursor protein.

Degree: 2014, University of Georgia

 Hirano bodies are cellular inclusions found in patients suffering from neurodegenerative diseases. However, the physiological function of Hirano bodies is not understood. A previous study… (more)

Subjects/Keywords: Hirano bodies; amyloid precursor protein; C31; Alzheimer’s disease

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APA (6th Edition):

Gupta, N. (2014). Association of Hirano bodies and C31 fragment of the amyloid precursor protein. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/25811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gupta, Nisha. “Association of Hirano bodies and C31 fragment of the amyloid precursor protein.” 2014. Thesis, University of Georgia. Accessed April 11, 2021. http://hdl.handle.net/10724/25811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gupta, Nisha. “Association of Hirano bodies and C31 fragment of the amyloid precursor protein.” 2014. Web. 11 Apr 2021.

Vancouver:

Gupta N. Association of Hirano bodies and C31 fragment of the amyloid precursor protein. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10724/25811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gupta N. Association of Hirano bodies and C31 fragment of the amyloid precursor protein. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/25811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

26. Adeniran, Charles. Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex.

Degree: MS, Chemistry, 2017, Georgia State University

  The NF-κB family of transcription factors controls a number of essential cellular functions. Pirin is a non-heme iron (Fe) redox specific co-regulator of NF-κB… (more)

Subjects/Keywords: NF-κB; Pirin; DNA; Transcription Factor; Precursor Protein; Iron

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APA (6th Edition):

Adeniran, C. (2017). Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/chemistry_theses/108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adeniran, Charles. “Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex.” 2017. Thesis, Georgia State University. Accessed April 11, 2021. https://scholarworks.gsu.edu/chemistry_theses/108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adeniran, Charles. “Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex.” 2017. Web. 11 Apr 2021.

Vancouver:

Adeniran C. Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex. [Internet] [Thesis]. Georgia State University; 2017. [cited 2021 Apr 11]. Available from: https://scholarworks.gsu.edu/chemistry_theses/108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adeniran C. Pirin Allosterically Modulates The Dynamics And Interactions Of The κB DNA In The NF-κB Supramolecular Complex. [Thesis]. Georgia State University; 2017. Available from: https://scholarworks.gsu.edu/chemistry_theses/108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

27. Andrew, Robert. Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions.

Degree: PhD, 2015, University of Manchester

 Dementia, the most common cause of which is Alzheimer's disease (AD), currently affects 850,000 people in the UK, a figure set to rise to over… (more)

Subjects/Keywords: 616.8; Alzheimers disease; Amyloid precursor protein; Amyloid-beta; interactome; Proteolysis

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APA (6th Edition):

Andrew, R. (2015). Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346

Chicago Manual of Style (16th Edition):

Andrew, Robert. “Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions.” 2015. Doctoral Dissertation, University of Manchester. Accessed April 11, 2021. https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346.

MLA Handbook (7th Edition):

Andrew, Robert. “Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions.” 2015. Web. 11 Apr 2021.

Vancouver:

Andrew R. Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 Apr 11]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346.

Council of Science Editors:

Andrew R. Differential proteolysis of the amyloid precursor protein isoforms : the role of cellular location and protein-protein interactions. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/differential-proteolysis-of-the-amyloid-precursor-protein-isoforms-the-role-of-cellular-location-and-proteinprotein-interactions(5390e8fa-fc5e-4357-8109-cb2bb1c49212).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764346


NSYSU

28. Chen, Chueh-Tan. The Possible Role of Neuron Autophagy on Amyloidogenesis Disorderswith Lead Exposure.

Degree: Master, Biological Sciences, 2012, NSYSU

 Lead (Pb) is one of the most well known toxic heavy metals in human beings and animals, which leads to toxic neurological disorders, cognitive problems,… (more)

Subjects/Keywords: Alzheimerâs Disease; amyloid precursor protein; autophagy; amyloidogensis disorders; apoptosis; β-amyloid protein; lead exposure

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, C. (2012). The Possible Role of Neuron Autophagy on Amyloidogenesis Disorderswith Lead Exposure. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0216112-112117

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Chueh-Tan. “The Possible Role of Neuron Autophagy on Amyloidogenesis Disorderswith Lead Exposure.” 2012. Thesis, NSYSU. Accessed April 11, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0216112-112117.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Chueh-Tan. “The Possible Role of Neuron Autophagy on Amyloidogenesis Disorderswith Lead Exposure.” 2012. Web. 11 Apr 2021.

Vancouver:

Chen C. The Possible Role of Neuron Autophagy on Amyloidogenesis Disorderswith Lead Exposure. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Apr 11]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0216112-112117.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen C. The Possible Role of Neuron Autophagy on Amyloidogenesis Disorderswith Lead Exposure. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0216112-112117

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

29. Pituch, Katarzyna C. Regulation of Oligodendrogenesis by Sulfatides and PDGFrα.

Degree: 2014, University of Illinois – Chicago

 Sulfatides, a significant constituent of myelin, regulate oligodendrogenesis by unknown mechanism. Therefore, we hypothesized that sulfatides affect very early events during gliogenesis regulating the differentiation… (more)

Subjects/Keywords: Sulfatides; oligodendrogenesis; PDGFrα; oligodendrocyte precursor cells; multipotential neural progenitors; arylsulfatase A

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pituch, K. C. (2014). Regulation of Oligodendrogenesis by Sulfatides and PDGFrα. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/19127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pituch, Katarzyna C. “Regulation of Oligodendrogenesis by Sulfatides and PDGFrα.” 2014. Thesis, University of Illinois – Chicago. Accessed April 11, 2021. http://hdl.handle.net/10027/19127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pituch, Katarzyna C. “Regulation of Oligodendrogenesis by Sulfatides and PDGFrα.” 2014. Web. 11 Apr 2021.

Vancouver:

Pituch KC. Regulation of Oligodendrogenesis by Sulfatides and PDGFrα. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10027/19127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pituch KC. Regulation of Oligodendrogenesis by Sulfatides and PDGFrα. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/19127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

30. Chang, Qing, 1989-. Effect of staphylococcal enterotoxin A (SEA) on cell proliferation in the dentate gyrus and hippocampal-dependent learning and memory.

Degree: MS, Psychology, 2015, Rutgers University

 Staphylococcal enterotoxin A (SEA) is known as a T cell superantigen, with well-characterized neurobiological and endocrine effects. We have quantified the survival of new cells,… (more)

Subjects/Keywords: Staphylococcal protein A

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chang, Qing, 1. (2015). Effect of staphylococcal enterotoxin A (SEA) on cell proliferation in the dentate gyrus and hippocampal-dependent learning and memory. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/48412/

Chicago Manual of Style (16th Edition):

Chang, Qing, 1989-. “Effect of staphylococcal enterotoxin A (SEA) on cell proliferation in the dentate gyrus and hippocampal-dependent learning and memory.” 2015. Masters Thesis, Rutgers University. Accessed April 11, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/48412/.

MLA Handbook (7th Edition):

Chang, Qing, 1989-. “Effect of staphylococcal enterotoxin A (SEA) on cell proliferation in the dentate gyrus and hippocampal-dependent learning and memory.” 2015. Web. 11 Apr 2021.

Vancouver:

Chang, Qing 1. Effect of staphylococcal enterotoxin A (SEA) on cell proliferation in the dentate gyrus and hippocampal-dependent learning and memory. [Internet] [Masters thesis]. Rutgers University; 2015. [cited 2021 Apr 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48412/.

Council of Science Editors:

Chang, Qing 1. Effect of staphylococcal enterotoxin A (SEA) on cell proliferation in the dentate gyrus and hippocampal-dependent learning and memory. [Masters Thesis]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48412/

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