Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(3xTg). Showing records 1 – 18 of 18 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


University of Manitoba

1. Abyat, Zahra. Method optimization for Fourier transform infrared imaging of hippocampal sections from background and 3xTg (AD model) mice.

Degree: Biomedical Engineering, 2019, University of Manitoba

 The creatine/phosphocreatine system, regulated by Creatine kinase, plays a vital role in maintaining energy balance in the brain. Energy metabolism and the function of creatine… (more)

Subjects/Keywords: FTIR; 3XTg mouse model; creatine dietary enrichment

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abyat, Z. (2019). Method optimization for Fourier transform infrared imaging of hippocampal sections from background and 3xTg (AD model) mice. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34395

Chicago Manual of Style (16th Edition):

Abyat, Zahra. “Method optimization for Fourier transform infrared imaging of hippocampal sections from background and 3xTg (AD model) mice.” 2019. Masters Thesis, University of Manitoba. Accessed September 29, 2020. http://hdl.handle.net/1993/34395.

MLA Handbook (7th Edition):

Abyat, Zahra. “Method optimization for Fourier transform infrared imaging of hippocampal sections from background and 3xTg (AD model) mice.” 2019. Web. 29 Sep 2020.

Vancouver:

Abyat Z. Method optimization for Fourier transform infrared imaging of hippocampal sections from background and 3xTg (AD model) mice. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/1993/34395.

Council of Science Editors:

Abyat Z. Method optimization for Fourier transform infrared imaging of hippocampal sections from background and 3xTg (AD model) mice. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34395


Temple University

2. Joshi, Yash. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.

Degree: PhD, 2015, Temple University

Pharmacology

BACKGROUND: Alzheimer's disease (AD) is the most common aging-associated neurodegenerative dementia. Current epidemiological trends indicate that a rapidly aging population, in conjunction with the… (more)

Subjects/Keywords: Neurosciences; Biology; Biochemistry;

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Joshi, Y. (2015). The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,318405

Chicago Manual of Style (16th Edition):

Joshi, Yash. “The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.” 2015. Doctoral Dissertation, Temple University. Accessed September 29, 2020. http://digital.library.temple.edu/u?/p245801coll10,318405.

MLA Handbook (7th Edition):

Joshi, Yash. “The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.” 2015. Web. 29 Sep 2020.

Vancouver:

Joshi Y. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 29]. Available from: http://digital.library.temple.edu/u?/p245801coll10,318405.

Council of Science Editors:

Joshi Y. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,318405


University of Guelph

3. Creighton, Samantha. Disturbed Object Processing in 5xFAD and 3xTG Mouse Models of Alzheimer's Disease: Going Beyond "Object Recognition".

Degree: MS, Department of Psychology, 2016, University of Guelph

 Object recognition not a unitary process, and there are many uncharacterized facets of object processing that have relevance to Alzheimer’s disease (AD). To elucidate the… (more)

Subjects/Keywords: Object processing; Object recognition; Alzheimer's disease; 5xFAD; 3xTG

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Creighton, S. (2016). Disturbed Object Processing in 5xFAD and 3xTG Mouse Models of Alzheimer's Disease: Going Beyond "Object Recognition". (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9952

Chicago Manual of Style (16th Edition):

Creighton, Samantha. “Disturbed Object Processing in 5xFAD and 3xTG Mouse Models of Alzheimer's Disease: Going Beyond "Object Recognition".” 2016. Masters Thesis, University of Guelph. Accessed September 29, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9952.

MLA Handbook (7th Edition):

Creighton, Samantha. “Disturbed Object Processing in 5xFAD and 3xTG Mouse Models of Alzheimer's Disease: Going Beyond "Object Recognition".” 2016. Web. 29 Sep 2020.

Vancouver:

Creighton S. Disturbed Object Processing in 5xFAD and 3xTG Mouse Models of Alzheimer's Disease: Going Beyond "Object Recognition". [Internet] [Masters thesis]. University of Guelph; 2016. [cited 2020 Sep 29]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9952.

Council of Science Editors:

Creighton S. Disturbed Object Processing in 5xFAD and 3xTG Mouse Models of Alzheimer's Disease: Going Beyond "Object Recognition". [Masters Thesis]. University of Guelph; 2016. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9952

4. Pais, Vera Cristina Martinho. Effect of class I histone deacetylase inhibitors in 3xTg-AD mice .

Degree: 2019, Universidade de Aveiro

 Alzheimer’s disease is the commonest form of dementia worldwide. Clinically, this neurodegenerative disorder is characterized by cognitive decline, changes in personality and mood, and disorientation.… (more)

Subjects/Keywords: Alzheimer’s disease; histone deacetylases; sodium butyrate; 3xTg-AD mice

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pais, V. C. M. (2019). Effect of class I histone deacetylase inhibitors in 3xTg-AD mice . (Thesis). Universidade de Aveiro. Retrieved from http://hdl.handle.net/10773/26899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pais, Vera Cristina Martinho. “Effect of class I histone deacetylase inhibitors in 3xTg-AD mice .” 2019. Thesis, Universidade de Aveiro. Accessed September 29, 2020. http://hdl.handle.net/10773/26899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pais, Vera Cristina Martinho. “Effect of class I histone deacetylase inhibitors in 3xTg-AD mice .” 2019. Web. 29 Sep 2020.

Vancouver:

Pais VCM. Effect of class I histone deacetylase inhibitors in 3xTg-AD mice . [Internet] [Thesis]. Universidade de Aveiro; 2019. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/10773/26899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pais VCM. Effect of class I histone deacetylase inhibitors in 3xTg-AD mice . [Thesis]. Universidade de Aveiro; 2019. Available from: http://hdl.handle.net/10773/26899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

5. Nguyen, Elizabeth T. Sex differences in stress responsivity, glucocorticoid signaling, and disease.

Degree: PhD, Medicine: Neuroscience/Medical Science Scholars Interdisciplinary, 2019, University of Cincinnati

 Excessive glucocorticoid (cortisol, corticosterone) secretion is associated with depression and Alzheimer’s disease (AD). Glucocorticoids act on low-affinity mineralocorticoid (MR) and high-affinity glucocorticoid (GR) receptors to… (more)

Subjects/Keywords: Neurology; corticosterone; hypothalamic-pituitary-adrenal axis; Alzheimer disease; depression; CORT118335; 3xTg

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nguyen, E. T. (2019). Sex differences in stress responsivity, glucocorticoid signaling, and disease. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin156327275069323

Chicago Manual of Style (16th Edition):

Nguyen, Elizabeth T. “Sex differences in stress responsivity, glucocorticoid signaling, and disease.” 2019. Doctoral Dissertation, University of Cincinnati. Accessed September 29, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin156327275069323.

MLA Handbook (7th Edition):

Nguyen, Elizabeth T. “Sex differences in stress responsivity, glucocorticoid signaling, and disease.” 2019. Web. 29 Sep 2020.

Vancouver:

Nguyen ET. Sex differences in stress responsivity, glucocorticoid signaling, and disease. [Internet] [Doctoral dissertation]. University of Cincinnati; 2019. [cited 2020 Sep 29]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin156327275069323.

Council of Science Editors:

Nguyen ET. Sex differences in stress responsivity, glucocorticoid signaling, and disease. [Doctoral Dissertation]. University of Cincinnati; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin156327275069323


University of Guelph

6. Collett, Heather. Effect of Early-life Enrichment on the Development of “Cognitive Reserve” in Mice.

Degree: MS, Department of Psychology, 2020, University of Guelph

 Resilience to Alzheimer’s disease (AD) pathology has been attributed to the development of cognitive reserve (CR) through life experiences. These experiences can be modeled with… (more)

Subjects/Keywords: Environmental Enrichment; Cognitive reserve; Enrichment; 3xTg; Alzheimer's Disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Collett, H. (2020). Effect of Early-life Enrichment on the Development of “Cognitive Reserve” in Mice. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/21176

Chicago Manual of Style (16th Edition):

Collett, Heather. “Effect of Early-life Enrichment on the Development of “Cognitive Reserve” in Mice.” 2020. Masters Thesis, University of Guelph. Accessed September 29, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/21176.

MLA Handbook (7th Edition):

Collett, Heather. “Effect of Early-life Enrichment on the Development of “Cognitive Reserve” in Mice.” 2020. Web. 29 Sep 2020.

Vancouver:

Collett H. Effect of Early-life Enrichment on the Development of “Cognitive Reserve” in Mice. [Internet] [Masters thesis]. University of Guelph; 2020. [cited 2020 Sep 29]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/21176.

Council of Science Editors:

Collett H. Effect of Early-life Enrichment on the Development of “Cognitive Reserve” in Mice. [Masters Thesis]. University of Guelph; 2020. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/21176

7. Kinoshita, Denise. Análise temporal de mediadores inflamatórios no tecido neuronal e na periferia em camundongos 3xTg-AD, um modelo animal para a Doença de Alzheimer.

Degree: PhD, Patologia Experimental e Comparada, 2012, University of São Paulo

A Doença de Alzheimer é a causa mais freqüente de demência senil e os gastos com pacientes com demência já supera os de pacientes com… (more)

Subjects/Keywords: 3xTg-AD mice; Alzheimer's Disease; Amyloid-β peptide; Behavior; Camundongo 3xTg-AD; Citocinas; Comportamento; Cytokines; Doença de Alzheimer; Neuroimmunology; Neuroimunologia; Peptídeo β-amilóide

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kinoshita, D. (2012). Análise temporal de mediadores inflamatórios no tecido neuronal e na periferia em camundongos 3xTg-AD, um modelo animal para a Doença de Alzheimer. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10133/tde-26092012-140403/ ;

Chicago Manual of Style (16th Edition):

Kinoshita, Denise. “Análise temporal de mediadores inflamatórios no tecido neuronal e na periferia em camundongos 3xTg-AD, um modelo animal para a Doença de Alzheimer.” 2012. Doctoral Dissertation, University of São Paulo. Accessed September 29, 2020. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-26092012-140403/ ;.

MLA Handbook (7th Edition):

Kinoshita, Denise. “Análise temporal de mediadores inflamatórios no tecido neuronal e na periferia em camundongos 3xTg-AD, um modelo animal para a Doença de Alzheimer.” 2012. Web. 29 Sep 2020.

Vancouver:

Kinoshita D. Análise temporal de mediadores inflamatórios no tecido neuronal e na periferia em camundongos 3xTg-AD, um modelo animal para a Doença de Alzheimer. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2020 Sep 29]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10133/tde-26092012-140403/ ;.

Council of Science Editors:

Kinoshita D. Análise temporal de mediadores inflamatórios no tecido neuronal e na periferia em camundongos 3xTg-AD, um modelo animal para a Doença de Alzheimer. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/10/10133/tde-26092012-140403/ ;

8. 손, 경선. Ischemic Vulnerability in the Brain of an Alzheimer’s Mouse Model.

Degree: 2019, Ajou University

Alzheimer's disease (AD) is a condition that mostly affects elderly individuals who are also at an increased risk of cerebrovascular disease. It is not well… (more)

Subjects/Keywords: Alzheimer’s disease; cerebral ischemia; 3xTG-AD mice; amyloid beta; tau protein; neuronal change; 알츠하이머병; 뇌 허혈; 3xTG-AD 마우스; 아밀로이드 베타; 타우 단백질; 신경 병리 변화

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

손, . (2019). Ischemic Vulnerability in the Brain of an Alzheimer’s Mouse Model. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/17904 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000028838

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

손, 경선. “Ischemic Vulnerability in the Brain of an Alzheimer’s Mouse Model.” 2019. Thesis, Ajou University. Accessed September 29, 2020. http://repository.ajou.ac.kr/handle/201003/17904 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000028838.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

손, 경선. “Ischemic Vulnerability in the Brain of an Alzheimer’s Mouse Model.” 2019. Web. 29 Sep 2020.

Vancouver:

손 . Ischemic Vulnerability in the Brain of an Alzheimer’s Mouse Model. [Internet] [Thesis]. Ajou University; 2019. [cited 2020 Sep 29]. Available from: http://repository.ajou.ac.kr/handle/201003/17904 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000028838.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

손 . Ischemic Vulnerability in the Brain of an Alzheimer’s Mouse Model. [Thesis]. Ajou University; 2019. Available from: http://repository.ajou.ac.kr/handle/201003/17904 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000028838

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

9. Park, Keigan Merrick; Bowers, William J. Regulation of the Inositol (1,4,5) Trisphosphate Receptor by Tumor Necrosis Factor-alpha.

Degree: PhD, 2010, University of Rochester

 Inflammatory events have long been implicated in initiating and/or propagating the pathophysiology associated with a number of neurological diseases. During neuroinflammation, the activation of brain-resident… (more)

Subjects/Keywords: TNF-Q; Alzheimer's; Calcium; Neuroinflammation; JNK; SP-1; 3xTg-AD; IP3R; Muscarinic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Park, Keigan Merrick; Bowers, W. J. (2010). Regulation of the Inositol (1,4,5) Trisphosphate Receptor by Tumor Necrosis Factor-alpha. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/10504

Chicago Manual of Style (16th Edition):

Park, Keigan Merrick; Bowers, William J. “Regulation of the Inositol (1,4,5) Trisphosphate Receptor by Tumor Necrosis Factor-alpha.” 2010. Doctoral Dissertation, University of Rochester. Accessed September 29, 2020. http://hdl.handle.net/1802/10504.

MLA Handbook (7th Edition):

Park, Keigan Merrick; Bowers, William J. “Regulation of the Inositol (1,4,5) Trisphosphate Receptor by Tumor Necrosis Factor-alpha.” 2010. Web. 29 Sep 2020.

Vancouver:

Park, Keigan Merrick; Bowers WJ. Regulation of the Inositol (1,4,5) Trisphosphate Receptor by Tumor Necrosis Factor-alpha. [Internet] [Doctoral dissertation]. University of Rochester; 2010. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/1802/10504.

Council of Science Editors:

Park, Keigan Merrick; Bowers WJ. Regulation of the Inositol (1,4,5) Trisphosphate Receptor by Tumor Necrosis Factor-alpha. [Doctoral Dissertation]. University of Rochester; 2010. Available from: http://hdl.handle.net/1802/10504


East Carolina University

10. Do, Khoa. Early Metabolic Differences in Alzheimer’s Disease Mouse Model and Exercise Intervention Treatment.

Degree: MS, MS-Kinesiology, 2017, East Carolina University

 Alzheimer's disease (AD) is a neurodegenerative disorder that affects the central nervous system (CNS). In this study, we characterized and examined the early metabolic changes… (more)

Subjects/Keywords: neuropeptides; AgRP; NPY; POMC; 3xtg; Alzheimer's disease; Exercise therapy; Hypothalamus; Metabolism; Mice as laboratory animals

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Do, K. (2017). Early Metabolic Differences in Alzheimer’s Disease Mouse Model and Exercise Intervention Treatment. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/6345

Chicago Manual of Style (16th Edition):

Do, Khoa. “Early Metabolic Differences in Alzheimer’s Disease Mouse Model and Exercise Intervention Treatment.” 2017. Masters Thesis, East Carolina University. Accessed September 29, 2020. http://hdl.handle.net/10342/6345.

MLA Handbook (7th Edition):

Do, Khoa. “Early Metabolic Differences in Alzheimer’s Disease Mouse Model and Exercise Intervention Treatment.” 2017. Web. 29 Sep 2020.

Vancouver:

Do K. Early Metabolic Differences in Alzheimer’s Disease Mouse Model and Exercise Intervention Treatment. [Internet] [Masters thesis]. East Carolina University; 2017. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/10342/6345.

Council of Science Editors:

Do K. Early Metabolic Differences in Alzheimer’s Disease Mouse Model and Exercise Intervention Treatment. [Masters Thesis]. East Carolina University; 2017. Available from: http://hdl.handle.net/10342/6345

11. Palmer, Daniel. Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease.

Degree: PhD, Department of Psychology, 2017, University of Guelph

 In the course of Alzheimer’s Disease (AD) pathology, there is a progressive worsening of cognitive symptoms. One of the important steps in AD research has… (more)

Subjects/Keywords: Alzheimer's; Transgenic Mouse Models; Visual Spatial Learning; Attention; 3xTG; 5xFAD; APP/PS1; Touchscreen Operant Chamber

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Palmer, D. (2017). Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148

Chicago Manual of Style (16th Edition):

Palmer, Daniel. “Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease.” 2017. Doctoral Dissertation, University of Guelph. Accessed September 29, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148.

MLA Handbook (7th Edition):

Palmer, Daniel. “Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease.” 2017. Web. 29 Sep 2020.

Vancouver:

Palmer D. Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease. [Internet] [Doctoral dissertation]. University of Guelph; 2017. [cited 2020 Sep 29]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148.

Council of Science Editors:

Palmer D. Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease. [Doctoral Dissertation]. University of Guelph; 2017. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148


University of Georgia

12. McManus, Meagan Joy. The role of mitochondrial dysfunction and oxidative stress in age-dependent neurodegeneration.

Degree: 2014, University of Georgia

 The work presented herein illustrates the multifaceted role of mitochondria in neurodegeneration and provides evidence supporting the use of mitochondria-targeted therapeutics in diseases involving oxidative… (more)

Subjects/Keywords: mitochondria; oxidative stress; neuronal apoptosis; mitochondrial therapeutics; MitoQ; neurodegenerative disease; Alzheimer's disease; 3xTg-AD; learning and memory

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McManus, M. J. (2014). The role of mitochondrial dysfunction and oxidative stress in age-dependent neurodegeneration. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/26967

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McManus, Meagan Joy. “The role of mitochondrial dysfunction and oxidative stress in age-dependent neurodegeneration.” 2014. Thesis, University of Georgia. Accessed September 29, 2020. http://hdl.handle.net/10724/26967.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McManus, Meagan Joy. “The role of mitochondrial dysfunction and oxidative stress in age-dependent neurodegeneration.” 2014. Web. 29 Sep 2020.

Vancouver:

McManus MJ. The role of mitochondrial dysfunction and oxidative stress in age-dependent neurodegeneration. [Internet] [Thesis]. University of Georgia; 2014. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/10724/26967.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McManus MJ. The role of mitochondrial dysfunction and oxidative stress in age-dependent neurodegeneration. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/26967

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

13. Baker, Kelly E. Targeting Kremen1 Downregulation with RVG-9R/siRNA Complexes in the Triple-Transgenic Mouse Model of Alzheimer’s Disease.

Degree: MS, MS-Biomedical Science, 2017, East Carolina University

 Alzheimer's disease (AD) is a progressive disease characterized by cognitive decline and memory loss. Memory loss observed in AD results from the loss of neurons… (more)

Subjects/Keywords: Kremen1; Wnt Signaling; 3xTg-AD Mouse Model; Alzheimer Disease; Down-Regulation; RNA, Small Interfering; Mice, Ttransgenic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baker, K. E. (2017). Targeting Kremen1 Downregulation with RVG-9R/siRNA Complexes in the Triple-Transgenic Mouse Model of Alzheimer’s Disease. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/6400

Chicago Manual of Style (16th Edition):

Baker, Kelly E. “Targeting Kremen1 Downregulation with RVG-9R/siRNA Complexes in the Triple-Transgenic Mouse Model of Alzheimer’s Disease.” 2017. Masters Thesis, East Carolina University. Accessed September 29, 2020. http://hdl.handle.net/10342/6400.

MLA Handbook (7th Edition):

Baker, Kelly E. “Targeting Kremen1 Downregulation with RVG-9R/siRNA Complexes in the Triple-Transgenic Mouse Model of Alzheimer’s Disease.” 2017. Web. 29 Sep 2020.

Vancouver:

Baker KE. Targeting Kremen1 Downregulation with RVG-9R/siRNA Complexes in the Triple-Transgenic Mouse Model of Alzheimer’s Disease. [Internet] [Masters thesis]. East Carolina University; 2017. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/10342/6400.

Council of Science Editors:

Baker KE. Targeting Kremen1 Downregulation with RVG-9R/siRNA Complexes in the Triple-Transgenic Mouse Model of Alzheimer’s Disease. [Masters Thesis]. East Carolina University; 2017. Available from: http://hdl.handle.net/10342/6400

14. Torres-Lista, Virginia. Conducta social, anidación y respuesta a antipsicóticos Atípicos en el modelo triple transgénico 3xtg-AD para la Enfermedad de alzheimer.

Degree: Departament de Psiquiatria i de Medicina Legal, 2012, Universitat Autònoma de Barcelona

 Although dementia is the characteristic symptom of the Alzheimer’s disease, Behavioral and Psychological Symptoms of Dementia (BPSD) and alterations in Daily Life Activities (DLA) are… (more)

Subjects/Keywords: Risperidona; SCPD; 3xtg-AD; Ciències de la Salut; 616.8

…ABREVIATURAS Abreviaturas más utilizadas en este trabajo. NTg 3xTg-AD (s) (r)… …conducta sociales en ratones 3xTg-AD ….. 59 Materiales y métodos… …conducta de anidación en ratones 3xTg-AD …... 77 Materiales y métodos… …95 Estudio 3: Caracterización del perfil cognitivo y tipo SCPD de los ratones 3xTg-AD y… …inducida por el tratamiento crónico con el antipsicótico atípico risperidona en ratones 3xTg-AD y… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Torres-Lista, V. (2012). Conducta social, anidación y respuesta a antipsicóticos Atípicos en el modelo triple transgénico 3xtg-AD para la Enfermedad de alzheimer. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/117537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Torres-Lista, Virginia. “Conducta social, anidación y respuesta a antipsicóticos Atípicos en el modelo triple transgénico 3xtg-AD para la Enfermedad de alzheimer.” 2012. Thesis, Universitat Autònoma de Barcelona. Accessed September 29, 2020. http://hdl.handle.net/10803/117537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Torres-Lista, Virginia. “Conducta social, anidación y respuesta a antipsicóticos Atípicos en el modelo triple transgénico 3xtg-AD para la Enfermedad de alzheimer.” 2012. Web. 29 Sep 2020.

Vancouver:

Torres-Lista V. Conducta social, anidación y respuesta a antipsicóticos Atípicos en el modelo triple transgénico 3xtg-AD para la Enfermedad de alzheimer. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2012. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/10803/117537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Torres-Lista V. Conducta social, anidación y respuesta a antipsicóticos Atípicos en el modelo triple transgénico 3xtg-AD para la Enfermedad de alzheimer. [Thesis]. Universitat Autònoma de Barcelona; 2012. Available from: http://hdl.handle.net/10803/117537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

15. Sancheti, Harshkumar. Age dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a 3xTg-AD mouse model of Alzheimer's disease: implications as a therapeutic/nutraceutical agent.

Degree: PhD, Molecular Pharmacology and Toxicology, 2016, University of Southern California

 Alzheimer’s disease is a type of dementia that causes problems with memory, thinking and behavior. Currently, there are no approved drugs to treat Alzheimer’s disease;… (more)

Subjects/Keywords: Alzheimer'; s disease; triple transgenic mice (3xTg-AD); lipoic acid; brain; glucose uptake; glucose metabolism; PI3K/Akt; IRS; glutamate; hypometabolism; hypermetabolism; synaptic plasticity; long term potentiation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sancheti, H. (2016). Age dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a 3xTg-AD mouse model of Alzheimer's disease: implications as a therapeutic/nutraceutical agent. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/376878/rec/577

Chicago Manual of Style (16th Edition):

Sancheti, Harshkumar. “Age dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a 3xTg-AD mouse model of Alzheimer's disease: implications as a therapeutic/nutraceutical agent.” 2016. Doctoral Dissertation, University of Southern California. Accessed September 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/376878/rec/577.

MLA Handbook (7th Edition):

Sancheti, Harshkumar. “Age dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a 3xTg-AD mouse model of Alzheimer's disease: implications as a therapeutic/nutraceutical agent.” 2016. Web. 29 Sep 2020.

Vancouver:

Sancheti H. Age dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a 3xTg-AD mouse model of Alzheimer's disease: implications as a therapeutic/nutraceutical agent. [Internet] [Doctoral dissertation]. University of Southern California; 2016. [cited 2020 Sep 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/376878/rec/577.

Council of Science Editors:

Sancheti H. Age dependent modulation of synaptic plasticity and insulin mimetic effect of lipoic acid on a 3xTg-AD mouse model of Alzheimer's disease: implications as a therapeutic/nutraceutical agent. [Doctoral Dissertation]. University of Southern California; 2016. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/376878/rec/577


University College Cork

16. Kaar, Aidan. Investigating neuronal calcium homeostasis in murine models of Alzheimer’s disease.

Degree: 2019, University College Cork

 This thesis is primarily concerned with the pathological remodelling of calcium homeostasis and signalling in murine Alzheimer’s disease (AD) models, namely the 3xTg-AD (APPSWE/PS1M146V/TauP301L) mouse… (more)

Subjects/Keywords: Primary neuronal culture; Hippocampal neurons; Trituration; 3xTg-AD mouse; TgF344-AD rat; Presenilin; Endoplasmic reticulum; Metabotropic glutamate receptor; mGluR; I-mGluR; cADPR; ADP ribosyl cyclase; glutamate; Bcl-2; Bcl-XL; IP3R; RyR; Alzheimer's disease; Calcium signalling; Calcium homeostasis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kaar, A. (2019). Investigating neuronal calcium homeostasis in murine models of Alzheimer’s disease. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/7453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kaar, Aidan. “Investigating neuronal calcium homeostasis in murine models of Alzheimer’s disease.” 2019. Thesis, University College Cork. Accessed September 29, 2020. http://hdl.handle.net/10468/7453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kaar, Aidan. “Investigating neuronal calcium homeostasis in murine models of Alzheimer’s disease.” 2019. Web. 29 Sep 2020.

Vancouver:

Kaar A. Investigating neuronal calcium homeostasis in murine models of Alzheimer’s disease. [Internet] [Thesis]. University College Cork; 2019. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/10468/7453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kaar A. Investigating neuronal calcium homeostasis in murine models of Alzheimer’s disease. [Thesis]. University College Cork; 2019. Available from: http://hdl.handle.net/10468/7453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

17. Moreau, Mireille. Expression profile of plasticity-related mRNAs in the cortex and hippocampus of young and aged rats and of 3xTg and wild type mice.

Degree: 2012, Université de Montréal

Subjects/Keywords: Plasticité synaptique; ARNm; Déclins cognitifs; Maladie d’Alzheimer; Rats jeunes; Rats âgés; Souris 3xTg; Souris WT; Cortex; Hippocampe; Synaptic plasticity; mRNAs; Cognitive decline; Alzheimer disease; Young rats; Aged rats; 3xTg mice; WT mice; Cortex; Hippocampus; Biology - Molecular / Biologie - Biologie moléculaire (UMI : 0307)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moreau, M. (2012). Expression profile of plasticity-related mRNAs in the cortex and hippocampus of young and aged rats and of 3xTg and wild type mice. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/7047

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moreau, Mireille. “Expression profile of plasticity-related mRNAs in the cortex and hippocampus of young and aged rats and of 3xTg and wild type mice.” 2012. Thesis, Université de Montréal. Accessed September 29, 2020. http://hdl.handle.net/1866/7047.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moreau, Mireille. “Expression profile of plasticity-related mRNAs in the cortex and hippocampus of young and aged rats and of 3xTg and wild type mice.” 2012. Web. 29 Sep 2020.

Vancouver:

Moreau M. Expression profile of plasticity-related mRNAs in the cortex and hippocampus of young and aged rats and of 3xTg and wild type mice. [Internet] [Thesis]. Université de Montréal; 2012. [cited 2020 Sep 29]. Available from: http://hdl.handle.net/1866/7047.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moreau M. Expression profile of plasticity-related mRNAs in the cortex and hippocampus of young and aged rats and of 3xTg and wild type mice. [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/7047

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Cavendish, John Zachary. Mitochondrial Aspects of Neuronal Pathology in Triple-Transgenic Alzheimer’s Disease Mice.

Degree: PhD, Not Listed, 2021, West Virginia University

  Alzheimer’s disease (AD) is a fatal, progressive neurodegenerative disease afflicting millions of people in the United States alone and is the only one of… (more)

Subjects/Keywords: Alzheimer’s Disease; Neurodegeneration; 3xTg-AD Mice; Mitochondrial Structure; Mitochondrial Movement; Sigma-1 Receptors; Animal Experimentation and Research; Biochemical Phenomena, Metabolism, and Nutrition; Biological Phenomena, Cell Phenomena, and Immunity; Cell Biology; Laboratory and Basic Science Research; Molecular and Cellular Neuroscience; Nervous System Diseases; Neurosciences

…model to date has been able to fully recapitulate every single aspect of human disease. 3xTg… …controls. Nevertheless, mouse models such as 3xTg-AD mice are valuable tools for understanding… …x5D;. These alterations correlate strongly with the clinical stage of disease. The 3xTg-AD… …x5D; and more recently that mitochondrial movement was greatly impaired in 3xTg-AD primary… …3xTg-AD mice (Quintana et. al; publication in revision) and AD patients often have… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cavendish, J. Z. (2021). Mitochondrial Aspects of Neuronal Pathology in Triple-Transgenic Alzheimer’s Disease Mice. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.4024 ; https://researchrepository.wvu.edu/etd/4024

Chicago Manual of Style (16th Edition):

Cavendish, John Zachary. “Mitochondrial Aspects of Neuronal Pathology in Triple-Transgenic Alzheimer’s Disease Mice.” 2021. Doctoral Dissertation, West Virginia University. Accessed September 29, 2020. https://doi.org/10.33915/etd.4024 ; https://researchrepository.wvu.edu/etd/4024.

MLA Handbook (7th Edition):

Cavendish, John Zachary. “Mitochondrial Aspects of Neuronal Pathology in Triple-Transgenic Alzheimer’s Disease Mice.” 2021. Web. 29 Sep 2020.

Vancouver:

Cavendish JZ. Mitochondrial Aspects of Neuronal Pathology in Triple-Transgenic Alzheimer’s Disease Mice. [Internet] [Doctoral dissertation]. West Virginia University; 2021. [cited 2020 Sep 29]. Available from: https://doi.org/10.33915/etd.4024 ; https://researchrepository.wvu.edu/etd/4024.

Council of Science Editors:

Cavendish JZ. Mitochondrial Aspects of Neuronal Pathology in Triple-Transgenic Alzheimer’s Disease Mice. [Doctoral Dissertation]. West Virginia University; 2021. Available from: https://doi.org/10.33915/etd.4024 ; https://researchrepository.wvu.edu/etd/4024

.