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You searched for subject:(0383). Showing records 1 – 30 of 83 total matches.

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University of Toronto

1. MacAllister, Stephanie. Investigating the Cytotoxic Mechanisms of Hepatotoxicity Induced by Xenobiotics and their Metabolites.

Degree: 2013, University of Toronto

Xenobiotics or their metabolite(s) can be electrophilic or free radicals that elicit a variety of chemical reactions either directly or indirectly effecting the biochemistry of… (more)

Subjects/Keywords: Toxicology; 0383

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APA (6th Edition):

MacAllister, S. (2013). Investigating the Cytotoxic Mechanisms of Hepatotoxicity Induced by Xenobiotics and their Metabolites. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/70106

Chicago Manual of Style (16th Edition):

MacAllister, Stephanie. “Investigating the Cytotoxic Mechanisms of Hepatotoxicity Induced by Xenobiotics and their Metabolites.” 2013. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/70106.

MLA Handbook (7th Edition):

MacAllister, Stephanie. “Investigating the Cytotoxic Mechanisms of Hepatotoxicity Induced by Xenobiotics and their Metabolites.” 2013. Web. 15 Apr 2021.

Vancouver:

MacAllister S. Investigating the Cytotoxic Mechanisms of Hepatotoxicity Induced by Xenobiotics and their Metabolites. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/70106.

Council of Science Editors:

MacAllister S. Investigating the Cytotoxic Mechanisms of Hepatotoxicity Induced by Xenobiotics and their Metabolites. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/70106


University of Toronto

2. Sweeting, Jessica Nicole. Biochemical Determinants of Methanol Developmental Toxicity in Rabbits.

Degree: 2013, University of Toronto

The teratogenic potential of methanol (MeOH) in New Zealand white (NZW) rabbits was examined, with emphasis on the biochemical basis for species differences in susceptibility.… (more)

Subjects/Keywords: Methanol; Teratology; Rabbit; 0383; 0419

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APA (6th Edition):

Sweeting, J. N. (2013). Biochemical Determinants of Methanol Developmental Toxicity in Rabbits. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68918

Chicago Manual of Style (16th Edition):

Sweeting, Jessica Nicole. “Biochemical Determinants of Methanol Developmental Toxicity in Rabbits.” 2013. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/68918.

MLA Handbook (7th Edition):

Sweeting, Jessica Nicole. “Biochemical Determinants of Methanol Developmental Toxicity in Rabbits.” 2013. Web. 15 Apr 2021.

Vancouver:

Sweeting JN. Biochemical Determinants of Methanol Developmental Toxicity in Rabbits. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/68918.

Council of Science Editors:

Sweeting JN. Biochemical Determinants of Methanol Developmental Toxicity in Rabbits. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68918


University of Toronto

3. Feng, Yan. Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria.

Degree: 2010, University of Toronto

High chronic fructose consumption is linked to a nonalcoholic steatohepatitis (NASH) type of hepatotoxicity. Oxalate is the major endpoint of fructose metabolism, which accumulates in… (more)

Subjects/Keywords: Fructose; Hepatocyte toxicity; Nonalcoholic Steatohepatitis; Hyperoxaluria; 0383

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APA (6th Edition):

Feng, Y. (2010). Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24568

Chicago Manual of Style (16th Edition):

Feng, Yan. “Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria.” 2010. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/24568.

MLA Handbook (7th Edition):

Feng, Yan. “Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria.” 2010. Web. 15 Apr 2021.

Vancouver:

Feng Y. Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/24568.

Council of Science Editors:

Feng Y. Hepatocyte Molecular Cytotoxic Mechanism Study of Fructose and its Metabolites Involved in Nonalcoholic Steatohepatitis and Hyperoxaluria. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24568


University of Toronto

4. Sacco, Raffaele. Cockayne Syndrome B is Required for Neural Precursor Self-renewal and Neuritegenesis after DNA Damage.

Degree: 2010, University of Toronto

Neural precursor cells self-renew and differentiate throughout development and in response to neural injury in the adult brain. The DNA damage response in NPCs has… (more)

Subjects/Keywords: Neural Stem Cells; DNA damage; 0383

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APA (6th Edition):

Sacco, R. (2010). Cockayne Syndrome B is Required for Neural Precursor Self-renewal and Neuritegenesis after DNA Damage. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25785

Chicago Manual of Style (16th Edition):

Sacco, Raffaele. “Cockayne Syndrome B is Required for Neural Precursor Self-renewal and Neuritegenesis after DNA Damage.” 2010. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/25785.

MLA Handbook (7th Edition):

Sacco, Raffaele. “Cockayne Syndrome B is Required for Neural Precursor Self-renewal and Neuritegenesis after DNA Damage.” 2010. Web. 15 Apr 2021.

Vancouver:

Sacco R. Cockayne Syndrome B is Required for Neural Precursor Self-renewal and Neuritegenesis after DNA Damage. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/25785.

Council of Science Editors:

Sacco R. Cockayne Syndrome B is Required for Neural Precursor Self-renewal and Neuritegenesis after DNA Damage. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25785


University of Toronto

5. Moller, Monique. Detection of Prenatal Opiate Exposures in Alternative Matrices.

Degree: 2010, University of Toronto

Identification of maternal opioid abuse in pregnancy is often difficult to ascertain in the absence of reliable self report. For this reason, physicians and child… (more)

Subjects/Keywords: Drug; Opiate; Hair; Prenatal; 0419; 0383

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APA (6th Edition):

Moller, M. (2010). Detection of Prenatal Opiate Exposures in Alternative Matrices. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25865

Chicago Manual of Style (16th Edition):

Moller, Monique. “Detection of Prenatal Opiate Exposures in Alternative Matrices.” 2010. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/25865.

MLA Handbook (7th Edition):

Moller, Monique. “Detection of Prenatal Opiate Exposures in Alternative Matrices.” 2010. Web. 15 Apr 2021.

Vancouver:

Moller M. Detection of Prenatal Opiate Exposures in Alternative Matrices. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/25865.

Council of Science Editors:

Moller M. Detection of Prenatal Opiate Exposures in Alternative Matrices. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25865

6. Grey, Anne Mullen. Adrenalectomy and Glucocorticoid Effects on the Rat Hepatic Aryl Hydrocarbon Receptor Pathway and the Response to Aromatic Hydrocarbons.

Degree: 2011, University of Toronto

The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor that mediates the effects of aromatic hydrocarbons, such as 2,3,7,8-tetrachlorodibenzo-p-dioxin and 3-methylcholanthrene (MC); the prototypical… (more)

Subjects/Keywords: 0419; 0383

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APA (6th Edition):

Grey, A. M. (2011). Adrenalectomy and Glucocorticoid Effects on the Rat Hepatic Aryl Hydrocarbon Receptor Pathway and the Response to Aromatic Hydrocarbons. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/31874

Chicago Manual of Style (16th Edition):

Grey, Anne Mullen. “Adrenalectomy and Glucocorticoid Effects on the Rat Hepatic Aryl Hydrocarbon Receptor Pathway and the Response to Aromatic Hydrocarbons.” 2011. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/31874.

MLA Handbook (7th Edition):

Grey, Anne Mullen. “Adrenalectomy and Glucocorticoid Effects on the Rat Hepatic Aryl Hydrocarbon Receptor Pathway and the Response to Aromatic Hydrocarbons.” 2011. Web. 15 Apr 2021.

Vancouver:

Grey AM. Adrenalectomy and Glucocorticoid Effects on the Rat Hepatic Aryl Hydrocarbon Receptor Pathway and the Response to Aromatic Hydrocarbons. [Internet] [Doctoral dissertation]. University of Toronto; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/31874.

Council of Science Editors:

Grey AM. Adrenalectomy and Glucocorticoid Effects on the Rat Hepatic Aryl Hydrocarbon Receptor Pathway and the Response to Aromatic Hydrocarbons. [Doctoral Dissertation]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31874


University of Toronto

7. Mak, Alastair. Investigation of the Mechanism of Idiosyncratic Drug-Induced Liver Injury by Immune System Modulation.

Degree: PhD, 2018, University of Toronto

 Idiosyncratic drug-induced liver injury (IDILI) continues to be an important issue. There is increasing clinical evidence that most IDILI is immune mediated. Extensive mechanistic studies… (more)

Subjects/Keywords: Animal Models; Idiosyncratic Drug Reactions; Immunomodulation; 0383

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APA (6th Edition):

Mak, A. (2018). Investigation of the Mechanism of Idiosyncratic Drug-Induced Liver Injury by Immune System Modulation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89812

Chicago Manual of Style (16th Edition):

Mak, Alastair. “Investigation of the Mechanism of Idiosyncratic Drug-Induced Liver Injury by Immune System Modulation.” 2018. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/89812.

MLA Handbook (7th Edition):

Mak, Alastair. “Investigation of the Mechanism of Idiosyncratic Drug-Induced Liver Injury by Immune System Modulation.” 2018. Web. 15 Apr 2021.

Vancouver:

Mak A. Investigation of the Mechanism of Idiosyncratic Drug-Induced Liver Injury by Immune System Modulation. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/89812.

Council of Science Editors:

Mak A. Investigation of the Mechanism of Idiosyncratic Drug-Induced Liver Injury by Immune System Modulation. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89812


University of Toronto

8. Ahmadi, Bilal. Cadmium-induced Autophagy and Autophagic Contribution to Cell Death in Cadmium-treated Mesangial Cells.

Degree: 2016, University of Toronto

Cadmium (Cd) is a nephrotoxic metal that has a number of consequences for renal function. Cells exposed to Cd may have methods to counteract toxic… (more)

Subjects/Keywords: Apoptosis; Autophagy; Cadmium; Cell Death; Cell Survival; Mesangial Cells; 0383

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APA (6th Edition):

Ahmadi, B. (2016). Cadmium-induced Autophagy and Autophagic Contribution to Cell Death in Cadmium-treated Mesangial Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71663

Chicago Manual of Style (16th Edition):

Ahmadi, Bilal. “Cadmium-induced Autophagy and Autophagic Contribution to Cell Death in Cadmium-treated Mesangial Cells.” 2016. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/71663.

MLA Handbook (7th Edition):

Ahmadi, Bilal. “Cadmium-induced Autophagy and Autophagic Contribution to Cell Death in Cadmium-treated Mesangial Cells.” 2016. Web. 15 Apr 2021.

Vancouver:

Ahmadi B. Cadmium-induced Autophagy and Autophagic Contribution to Cell Death in Cadmium-treated Mesangial Cells. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/71663.

Council of Science Editors:

Ahmadi B. Cadmium-induced Autophagy and Autophagic Contribution to Cell Death in Cadmium-treated Mesangial Cells. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/71663


University of Toronto

9. Cho, Tiffany Elizabeth. 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice.

Degree: 2015, University of Toronto

The aryl hydrocarbon receptor (AHR) is a ligand-regulated transcription factor that is activated upon binding to various ligands. The activated AHR modulates the expression of… (more)

Subjects/Keywords: 3-Methylcholanthrene; Aryl Hydrocarbon Receptor; Chylous Ascites; Lethality; TIPARP; Toxicity; 0383

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APA (6th Edition):

Cho, T. E. (2015). 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74562

Chicago Manual of Style (16th Edition):

Cho, Tiffany Elizabeth. “3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice.” 2015. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/74562.

MLA Handbook (7th Edition):

Cho, Tiffany Elizabeth. “3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice.” 2015. Web. 15 Apr 2021.

Vancouver:

Cho TE. 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/74562.

Council of Science Editors:

Cho TE. 3-Methylcholanthrene Induces Chylous Ascites and Lethality in Tiparp Knockout Mice. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74562


University of Toronto

10. Gulersen, Moti. Characterization of NP22 and its Potential Role in NMDA Receptor-mediated Transmission.

Degree: 2011, University of Toronto

N-methyl D-aspartate (NMDA) receptors represent integral signal transducers for excitatory glutamate neurotransmission. While NMDA receptors are critical for synaptic plasticity, the molecular events underlying this… (more)

Subjects/Keywords: NMDA; neuroscience; protein interactions; NP22; synaptic plasticity; affinity purification; 0419; 0383

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APA (6th Edition):

Gulersen, M. (2011). Characterization of NP22 and its Potential Role in NMDA Receptor-mediated Transmission. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30617

Chicago Manual of Style (16th Edition):

Gulersen, Moti. “Characterization of NP22 and its Potential Role in NMDA Receptor-mediated Transmission.” 2011. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/30617.

MLA Handbook (7th Edition):

Gulersen, Moti. “Characterization of NP22 and its Potential Role in NMDA Receptor-mediated Transmission.” 2011. Web. 15 Apr 2021.

Vancouver:

Gulersen M. Characterization of NP22 and its Potential Role in NMDA Receptor-mediated Transmission. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/30617.

Council of Science Editors:

Gulersen M. Characterization of NP22 and its Potential Role in NMDA Receptor-mediated Transmission. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30617


University of Toronto

11. Yang, Kai. Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes.

Degree: 2011, University of Toronto

High chronic fructose consumption has been linked to many diseases. Sugar metabolites, especially glyoxal and methylglyoxal can form advanced glycation products, which contribute to the… (more)

Subjects/Keywords: Dicarbonyls; Oxidative Stress; Fructose; Protein carbonylation; Diabetes; AGEs; 0383; 0491

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APA (6th Edition):

Yang, K. (2011). Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/31650

Chicago Manual of Style (16th Edition):

Yang, Kai. “Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes.” 2011. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/31650.

MLA Handbook (7th Edition):

Yang, Kai. “Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes.” 2011. Web. 15 Apr 2021.

Vancouver:

Yang K. Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/31650.

Council of Science Editors:

Yang K. Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31650


University of Toronto

12. Novalen, Maria. Investigation of a Metabolic Pathway Leading to an Idiosyncratic Drug Reaction: Is the Sulfate of 12-Hydroxynevirapine Responsible for the Skin Rash in Brown Norway rats?.

Degree: 2010, University of Toronto

An animal model of nevirapine (NVP)-induced skin rash was used to test the hypothesis that sulfonation of 12-OH NVP, a metabolite of NVP proven essential… (more)

Subjects/Keywords: nevirapine; drug metabolism; adverse drug reactions; idiosyncratic drug reactions; 0383

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APA (6th Edition):

Novalen, M. (2010). Investigation of a Metabolic Pathway Leading to an Idiosyncratic Drug Reaction: Is the Sulfate of 12-Hydroxynevirapine Responsible for the Skin Rash in Brown Norway rats?. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25882

Chicago Manual of Style (16th Edition):

Novalen, Maria. “Investigation of a Metabolic Pathway Leading to an Idiosyncratic Drug Reaction: Is the Sulfate of 12-Hydroxynevirapine Responsible for the Skin Rash in Brown Norway rats?.” 2010. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/25882.

MLA Handbook (7th Edition):

Novalen, Maria. “Investigation of a Metabolic Pathway Leading to an Idiosyncratic Drug Reaction: Is the Sulfate of 12-Hydroxynevirapine Responsible for the Skin Rash in Brown Norway rats?.” 2010. Web. 15 Apr 2021.

Vancouver:

Novalen M. Investigation of a Metabolic Pathway Leading to an Idiosyncratic Drug Reaction: Is the Sulfate of 12-Hydroxynevirapine Responsible for the Skin Rash in Brown Norway rats?. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/25882.

Council of Science Editors:

Novalen M. Investigation of a Metabolic Pathway Leading to an Idiosyncratic Drug Reaction: Is the Sulfate of 12-Hydroxynevirapine Responsible for the Skin Rash in Brown Norway rats?. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25882


University of Toronto

13. English, Simon G. An Integrative Analysis of the Effects of Neonicotinoid Pesticides on North American Hummingbirds.

Degree: 2020, University of Toronto

Neonicotinoids are neurotoxic systemic insecticides applied extensively worldwide. The impacts of neonicotinoid exposure on invertebrates are widely studied, however effects on essential vertebrate pollinators like… (more)

Subjects/Keywords: dose-response; hummingbirds; imidacloprid; integrative biology; neonicotinoid pesticides; 0383

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APA (6th Edition):

English, S. G. (2020). An Integrative Analysis of the Effects of Neonicotinoid Pesticides on North American Hummingbirds. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/103588

Chicago Manual of Style (16th Edition):

English, Simon G. “An Integrative Analysis of the Effects of Neonicotinoid Pesticides on North American Hummingbirds.” 2020. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/103588.

MLA Handbook (7th Edition):

English, Simon G. “An Integrative Analysis of the Effects of Neonicotinoid Pesticides on North American Hummingbirds.” 2020. Web. 15 Apr 2021.

Vancouver:

English SG. An Integrative Analysis of the Effects of Neonicotinoid Pesticides on North American Hummingbirds. [Internet] [Masters thesis]. University of Toronto; 2020. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/103588.

Council of Science Editors:

English SG. An Integrative Analysis of the Effects of Neonicotinoid Pesticides on North American Hummingbirds. [Masters Thesis]. University of Toronto; 2020. Available from: http://hdl.handle.net/1807/103588

14. Abramov, Julia. Embryoprotective Role of Endogenous Catalase.

Degree: 2011, University of Toronto

Oxidative stress and reactive oxygen species (ROS) such as hydrogen peroxide (H2O2), which is detoxified by catalase, are implicated in fetal death and birth defects,… (more)

Subjects/Keywords: Catalase; embryonic development; 0383

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APA (6th Edition):

Abramov, J. (2011). Embryoprotective Role of Endogenous Catalase. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/31672

Chicago Manual of Style (16th Edition):

Abramov, Julia. “Embryoprotective Role of Endogenous Catalase.” 2011. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/31672.

MLA Handbook (7th Edition):

Abramov, Julia. “Embryoprotective Role of Endogenous Catalase.” 2011. Web. 15 Apr 2021.

Vancouver:

Abramov J. Embryoprotective Role of Endogenous Catalase. [Internet] [Doctoral dissertation]. University of Toronto; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/31672.

Council of Science Editors:

Abramov J. Embryoprotective Role of Endogenous Catalase. [Doctoral Dissertation]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31672

15. Gunness, Patrina. New insight into Acyclovir Renal Handling and Nephrotoxicity.

Degree: 2011, University of Toronto

Drug – induced nephrotoxicity is a serious adverse reaction that can have deleterious effects on a patient’s health and well-being. Acyclovir is an example of… (more)

Subjects/Keywords: Acyclovir; Nephrotoxicity; 0383

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APA (6th Edition):

Gunness, P. (2011). New insight into Acyclovir Renal Handling and Nephrotoxicity. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/31771

Chicago Manual of Style (16th Edition):

Gunness, Patrina. “New insight into Acyclovir Renal Handling and Nephrotoxicity.” 2011. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/31771.

MLA Handbook (7th Edition):

Gunness, Patrina. “New insight into Acyclovir Renal Handling and Nephrotoxicity.” 2011. Web. 15 Apr 2021.

Vancouver:

Gunness P. New insight into Acyclovir Renal Handling and Nephrotoxicity. [Internet] [Doctoral dissertation]. University of Toronto; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/31771.

Council of Science Editors:

Gunness P. New insight into Acyclovir Renal Handling and Nephrotoxicity. [Doctoral Dissertation]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31771


University of Toronto

16. Chan, Katherine. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.

Degree: 2008, University of Toronto

Hepatotoxicity is a serious adverse health effect caused by drugs and other chemical toxins generally detected in the later stages of drug development or in… (more)

Subjects/Keywords: xenobiotic metabolism; hepatotoxicity; QSAR; 0383

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APA (6th Edition):

Chan, K. (2008). Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/17290

Chicago Manual of Style (16th Edition):

Chan, Katherine. “Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.” 2008. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/17290.

MLA Handbook (7th Edition):

Chan, Katherine. “Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems.” 2008. Web. 15 Apr 2021.

Vancouver:

Chan K. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/17290.

Council of Science Editors:

Chan K. Application of Quantitative Structure-activity Relationships to Investigate Xenobiotic Cytotoxicity Mechanisms in Hepatocyte Systems. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17290


University of Toronto

17. Tafazoli, Shahrzad. Mechanisms of Drug-induced Oxidative Stress in the Hepatocyte Inflammation Model.

Degree: 2008, University of Toronto

Drug induced idiosyncratic agranulocytosis has been attributed to oxidation by hypochlorite formed by bone marrow myeloperoxidase (MPO). Idiosyncratic liver toxicity could also involve drug oxidative… (more)

Subjects/Keywords: Inflammation, Oxidative Stress, Hepatocytes; 0383

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APA (6th Edition):

Tafazoli, S. (2008). Mechanisms of Drug-induced Oxidative Stress in the Hepatocyte Inflammation Model. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/17294

Chicago Manual of Style (16th Edition):

Tafazoli, Shahrzad. “Mechanisms of Drug-induced Oxidative Stress in the Hepatocyte Inflammation Model.” 2008. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/17294.

MLA Handbook (7th Edition):

Tafazoli, Shahrzad. “Mechanisms of Drug-induced Oxidative Stress in the Hepatocyte Inflammation Model.” 2008. Web. 15 Apr 2021.

Vancouver:

Tafazoli S. Mechanisms of Drug-induced Oxidative Stress in the Hepatocyte Inflammation Model. [Internet] [Doctoral dissertation]. University of Toronto; 2008. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/17294.

Council of Science Editors:

Tafazoli S. Mechanisms of Drug-induced Oxidative Stress in the Hepatocyte Inflammation Model. [Doctoral Dissertation]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/17294


University of Toronto

18. Shapiro, Aaron Michael. Free Radical Determinants of Teratogenesis: Oxidative Damage and Biochemical Signalling by Reactive Oxygen Species.

Degree: PhD, 2014, University of Toronto

Prenatal exposure to xenobiotics can lead to the production of reactive oxygen species (ROS) that alter signal transduction or oxidatively damage cellular macromolecules such as… (more)

Subjects/Keywords: Bisphenol A; DNA damage; Ethanol; Methamphetamine; Oxidative Stress; ROS signalling; 0383

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APA (6th Edition):

Shapiro, A. M. (2014). Free Radical Determinants of Teratogenesis: Oxidative Damage and Biochemical Signalling by Reactive Oxygen Species. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/94553

Chicago Manual of Style (16th Edition):

Shapiro, Aaron Michael. “Free Radical Determinants of Teratogenesis: Oxidative Damage and Biochemical Signalling by Reactive Oxygen Species.” 2014. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/94553.

MLA Handbook (7th Edition):

Shapiro, Aaron Michael. “Free Radical Determinants of Teratogenesis: Oxidative Damage and Biochemical Signalling by Reactive Oxygen Species.” 2014. Web. 15 Apr 2021.

Vancouver:

Shapiro AM. Free Radical Determinants of Teratogenesis: Oxidative Damage and Biochemical Signalling by Reactive Oxygen Species. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/94553.

Council of Science Editors:

Shapiro AM. Free Radical Determinants of Teratogenesis: Oxidative Damage and Biochemical Signalling by Reactive Oxygen Species. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/94553


University of Toronto

19. Ng, Winnie Wing Yee. Investigation of the Mechanisms of Aromatic Amine-induced Idiosyncratic Drug Reactions.

Degree: 2013, University of Toronto

Drugs with the primary aromatic amine structure are notorious structural alerts in drug development because of their association with a high incidence of idiosyncratic drug… (more)

Subjects/Keywords: idiosyncratic drug reactions; aromatic amine drugs; immunotoxicity; biomarkers; 0383

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APA (6th Edition):

Ng, W. W. Y. (2013). Investigation of the Mechanisms of Aromatic Amine-induced Idiosyncratic Drug Reactions. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68900

Chicago Manual of Style (16th Edition):

Ng, Winnie Wing Yee. “Investigation of the Mechanisms of Aromatic Amine-induced Idiosyncratic Drug Reactions.” 2013. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/68900.

MLA Handbook (7th Edition):

Ng, Winnie Wing Yee. “Investigation of the Mechanisms of Aromatic Amine-induced Idiosyncratic Drug Reactions.” 2013. Web. 15 Apr 2021.

Vancouver:

Ng WWY. Investigation of the Mechanisms of Aromatic Amine-induced Idiosyncratic Drug Reactions. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/68900.

Council of Science Editors:

Ng WWY. Investigation of the Mechanisms of Aromatic Amine-induced Idiosyncratic Drug Reactions. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68900


University of Toronto

20. Johnston, Alexander Scott. Insights into the Mechanism of Idiosyncratic Drug-induced Agranulocytosis.

Degree: 2017, University of Toronto

Many drugs are known to cause idiosyncratic drug-induced agranulocytosis, although the mechanism is not well understood. This adverse event is suspected to be the result… (more)

Subjects/Keywords: Adverse Drug Reactions; Agranulocytosis; Animal Model; Clozapine; Idiosyncratic Drug Reactions; Myeloperoxidase; 0383

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APA (6th Edition):

Johnston, A. S. (2017). Insights into the Mechanism of Idiosyncratic Drug-induced Agranulocytosis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/77802

Chicago Manual of Style (16th Edition):

Johnston, Alexander Scott. “Insights into the Mechanism of Idiosyncratic Drug-induced Agranulocytosis.” 2017. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/77802.

MLA Handbook (7th Edition):

Johnston, Alexander Scott. “Insights into the Mechanism of Idiosyncratic Drug-induced Agranulocytosis.” 2017. Web. 15 Apr 2021.

Vancouver:

Johnston AS. Insights into the Mechanism of Idiosyncratic Drug-induced Agranulocytosis. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/77802.

Council of Science Editors:

Johnston AS. Insights into the Mechanism of Idiosyncratic Drug-induced Agranulocytosis. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/77802


University of Toronto

21. Hanna, Daniel. Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse.

Degree: 2013, University of Toronto

We previously observed that postnatal exposure of mice to the procarcinogen 4-aminobiphenyl (ABP) produced liver tumors only in wild-type males, while arylamine N-acetyltransferase deficient males… (more)

Subjects/Keywords: 4-aminobiphenyl; Aromatic amines; Diethylnitrosamine; Carcinogenesis; Hepatocellular carcinoma; Hepatotoxicity; Inflammation; 0419; 0383; 0433; 0307

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APA (6th Edition):

Hanna, D. (2013). Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35616

Chicago Manual of Style (16th Edition):

Hanna, Daniel. “Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse.” 2013. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/35616.

MLA Handbook (7th Edition):

Hanna, Daniel. “Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse.” 2013. Web. 15 Apr 2021.

Vancouver:

Hanna D. Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/35616.

Council of Science Editors:

Hanna D. Sex and Strain Differences in Acute Hepatotoxic and Inflammatory Responses to Liver Procarcinogens in the Developing Mouse. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35616


University of Toronto

22. Choong, Grace Mei Yee. Effects of Cadmium on Actin Glutathionylation and Focal Adhesions.

Degree: 2013, University of Toronto

The toxic metal ion cadmium (Cd2+) is pro-oxidant and specifically disrupts the actin cytoskeleton in renal mesangial cells. This study investigated the role of Cd2+-mediated… (more)

Subjects/Keywords: cadmium toxicology; glutathionylation; ROS; actin cytoskeleton; focal adhesions; CaMK-II; 0383; 0379

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APA (6th Edition):

Choong, G. M. Y. (2013). Effects of Cadmium on Actin Glutathionylation and Focal Adhesions. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42739

Chicago Manual of Style (16th Edition):

Choong, Grace Mei Yee. “Effects of Cadmium on Actin Glutathionylation and Focal Adhesions.” 2013. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/42739.

MLA Handbook (7th Edition):

Choong, Grace Mei Yee. “Effects of Cadmium on Actin Glutathionylation and Focal Adhesions.” 2013. Web. 15 Apr 2021.

Vancouver:

Choong GMY. Effects of Cadmium on Actin Glutathionylation and Focal Adhesions. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/42739.

Council of Science Editors:

Choong GMY. Effects of Cadmium on Actin Glutathionylation and Focal Adhesions. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42739


University of Toronto

23. Lankadurai, Brian. 1H NMR-based Metabolomics for Elucidating the Mode of Action of Ccontaminants in the Earthworm Eisenia Fetida after Sub-lethal Exposure.

Degree: 2013, University of Toronto

There is a growing need to develop rapid and cost-effective ecotoxicological tools for risk assessment because traditional methods examine endpoints such as mortality, which do… (more)

Subjects/Keywords: Ecotoxicology; Environmental Chemistry; Metabolic Profiling; PAHs; PFOA; PFOS; Soil contamination; Earthworms; 0486; 0383; 0487

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APA (6th Edition):

Lankadurai, B. (2013). 1H NMR-based Metabolomics for Elucidating the Mode of Action of Ccontaminants in the Earthworm Eisenia Fetida after Sub-lethal Exposure. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/35873

Chicago Manual of Style (16th Edition):

Lankadurai, Brian. “1H NMR-based Metabolomics for Elucidating the Mode of Action of Ccontaminants in the Earthworm Eisenia Fetida after Sub-lethal Exposure.” 2013. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/35873.

MLA Handbook (7th Edition):

Lankadurai, Brian. “1H NMR-based Metabolomics for Elucidating the Mode of Action of Ccontaminants in the Earthworm Eisenia Fetida after Sub-lethal Exposure.” 2013. Web. 15 Apr 2021.

Vancouver:

Lankadurai B. 1H NMR-based Metabolomics for Elucidating the Mode of Action of Ccontaminants in the Earthworm Eisenia Fetida after Sub-lethal Exposure. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/35873.

Council of Science Editors:

Lankadurai B. 1H NMR-based Metabolomics for Elucidating the Mode of Action of Ccontaminants in the Earthworm Eisenia Fetida after Sub-lethal Exposure. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35873


University of Toronto

24. Siu, Michelle Tiffani. Oxidative Stress and an Alternative Glutathione-dependent Mechanism in the Developmental Toxicity of Methanol.

Degree: 2013, University of Toronto

The in vivo teratogenicity of methanol (MeOH) and its underlying molecular mechanisms were investigated, particularly the pathogenic contribution of reactive oxygen species (ROS) and oxidative… (more)

Subjects/Keywords: Methanol; Oxidative stress; Teratology; Mouse model; Reactive oxygen species; Alcohol; 0307; 0383; 0758

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APA (6th Edition):

Siu, M. T. (2013). Oxidative Stress and an Alternative Glutathione-dependent Mechanism in the Developmental Toxicity of Methanol. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68965

Chicago Manual of Style (16th Edition):

Siu, Michelle Tiffani. “Oxidative Stress and an Alternative Glutathione-dependent Mechanism in the Developmental Toxicity of Methanol.” 2013. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/68965.

MLA Handbook (7th Edition):

Siu, Michelle Tiffani. “Oxidative Stress and an Alternative Glutathione-dependent Mechanism in the Developmental Toxicity of Methanol.” 2013. Web. 15 Apr 2021.

Vancouver:

Siu MT. Oxidative Stress and an Alternative Glutathione-dependent Mechanism in the Developmental Toxicity of Methanol. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/68965.

Council of Science Editors:

Siu MT. Oxidative Stress and an Alternative Glutathione-dependent Mechanism in the Developmental Toxicity of Methanol. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68965


University of Toronto

25. Miller, Lutfiya. Oxidative Stress Mechanisms in Alcohol Developmental Toxicity.

Degree: 2014, University of Toronto

Consumption of alcohol (ethanol, EtOH) during pregnancy can result in a spectrum of anomalies termed the Fetal Alcohol Spectrum Disorders (FASD), characterized by structural and… (more)

Subjects/Keywords: oxidative stress; fetal alcohol spectrum disorder; teratogenesis; embryology; methanol; ethanol; 0419; 0383; 0758

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APA (6th Edition):

Miller, L. (2014). Oxidative Stress Mechanisms in Alcohol Developmental Toxicity. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72528

Chicago Manual of Style (16th Edition):

Miller, Lutfiya. “Oxidative Stress Mechanisms in Alcohol Developmental Toxicity.” 2014. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/72528.

MLA Handbook (7th Edition):

Miller, Lutfiya. “Oxidative Stress Mechanisms in Alcohol Developmental Toxicity.” 2014. Web. 15 Apr 2021.

Vancouver:

Miller L. Oxidative Stress Mechanisms in Alcohol Developmental Toxicity. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/72528.

Council of Science Editors:

Miller L. Oxidative Stress Mechanisms in Alcohol Developmental Toxicity. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/72528

26. Issa, Moustapha Soumaila. Molecular characterization and functional analysis of cytochrome P450 genes in the yellow fever mosquito Aedes aegypti (Diptera: Culicidae).

Degree: MS, Department of Entomology, 2014, Kansas State University

 Cytochrome P450 monooxygenases (P450s) are important enzymes involved in the metabolism of a variety of xenobiotics, including insecticides and plant allelochemicals, and endogenous compounds, including… (more)

Subjects/Keywords: Cytochrome P450; Aedes aegypti; RNA interference; Detoxification; Pyrethroids; Entomology (0353); Molecular Biology (0307); Toxicology (0383)

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APA (6th Edition):

Issa, M. S. (2014). Molecular characterization and functional analysis of cytochrome P450 genes in the yellow fever mosquito Aedes aegypti (Diptera: Culicidae). (Masters Thesis). Kansas State University. Retrieved from http://hdl.handle.net/2097/18253

Chicago Manual of Style (16th Edition):

Issa, Moustapha Soumaila. “Molecular characterization and functional analysis of cytochrome P450 genes in the yellow fever mosquito Aedes aegypti (Diptera: Culicidae).” 2014. Masters Thesis, Kansas State University. Accessed April 15, 2021. http://hdl.handle.net/2097/18253.

MLA Handbook (7th Edition):

Issa, Moustapha Soumaila. “Molecular characterization and functional analysis of cytochrome P450 genes in the yellow fever mosquito Aedes aegypti (Diptera: Culicidae).” 2014. Web. 15 Apr 2021.

Vancouver:

Issa MS. Molecular characterization and functional analysis of cytochrome P450 genes in the yellow fever mosquito Aedes aegypti (Diptera: Culicidae). [Internet] [Masters thesis]. Kansas State University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2097/18253.

Council of Science Editors:

Issa MS. Molecular characterization and functional analysis of cytochrome P450 genes in the yellow fever mosquito Aedes aegypti (Diptera: Culicidae). [Masters Thesis]. Kansas State University; 2014. Available from: http://hdl.handle.net/2097/18253


University of Toronto

27. Dedina, Liana. Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland.

Degree: 2012, University of Toronto

Mother's milk provides multiple benefits to the offspring. However, xenobiotics transferred into breast milk may pose a risk to the nursing infant. The breast cancer… (more)

Subjects/Keywords: Pharmacology; Toxicology; Riboflavin; Cimetidine; Breast cancer resistance protein (BCRP); Riboflavin transporters; Breast milk; Mammary Gland; Lactation; 0419; 0383

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APA (6th Edition):

Dedina, L. (2012). Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33644

Chicago Manual of Style (16th Edition):

Dedina, Liana. “Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland.” 2012. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/33644.

MLA Handbook (7th Edition):

Dedina, Liana. “Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland.” 2012. Web. 15 Apr 2021.

Vancouver:

Dedina L. Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/33644.

Council of Science Editors:

Dedina L. Riboflavin Transporters and Breast Cancer Resistance Protein: Cimetidine-Riboflavin Interactions in the Mammary Gland. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33644


University of Toronto

28. Carnevale, Amanda. Investigating the Use of Hair to Assess Polybrominated Diphenyl Ether (PBDE) Exposure Retrospectively, and in Male Infants with Genitourinary Tract Malformations.

Degree: 2013, University of Toronto

Polybrominated diphenyl ethers (PBDEs) are synthetic chemicals that are added to a variety of consumer products as flame-retardants. The ubiquitous nature and endocrine disrupting properties… (more)

Subjects/Keywords: environmental exposure; hair; polybrominated diphenyl ethers (PBDE); genitourinary tract malformations; male reproduction; brominated flame retardants; 0419; 0383

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APA (6th Edition):

Carnevale, A. (2013). Investigating the Use of Hair to Assess Polybrominated Diphenyl Ether (PBDE) Exposure Retrospectively, and in Male Infants with Genitourinary Tract Malformations. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35595

Chicago Manual of Style (16th Edition):

Carnevale, Amanda. “Investigating the Use of Hair to Assess Polybrominated Diphenyl Ether (PBDE) Exposure Retrospectively, and in Male Infants with Genitourinary Tract Malformations.” 2013. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/35595.

MLA Handbook (7th Edition):

Carnevale, Amanda. “Investigating the Use of Hair to Assess Polybrominated Diphenyl Ether (PBDE) Exposure Retrospectively, and in Male Infants with Genitourinary Tract Malformations.” 2013. Web. 15 Apr 2021.

Vancouver:

Carnevale A. Investigating the Use of Hair to Assess Polybrominated Diphenyl Ether (PBDE) Exposure Retrospectively, and in Male Infants with Genitourinary Tract Malformations. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/35595.

Council of Science Editors:

Carnevale A. Investigating the Use of Hair to Assess Polybrominated Diphenyl Ether (PBDE) Exposure Retrospectively, and in Male Infants with Genitourinary Tract Malformations. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35595


Universidade de Lisboa

29. Teixeira, Rafael Leite. A espada do Basileus: a política imperial e a cristianização do exército romano, século IV d.C.

Degree: 2012, Universidade de Lisboa

Tese de mestrado, História (História Antiga), Universidade de Lisboa, Faculdade de Letras, 2012

Nesta dissertação, discutiu-se como o Estado romano lidou com os cristãos em… (more)

Subjects/Keywords: Vegécio,0383?-0450? - Crítica e interpretação; Cristianismo - Roma (Itália) - séc.04; Roma(Itália) - Exército - séc.04; Teses de mestrado - 2012

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APA (6th Edition):

Teixeira, R. L. (2012). A espada do Basileus: a política imperial e a cristianização do exército romano, século IV d.C. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7936

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Teixeira, Rafael Leite. “A espada do Basileus: a política imperial e a cristianização do exército romano, século IV d.C.” 2012. Thesis, Universidade de Lisboa. Accessed April 15, 2021. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7936.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Teixeira, Rafael Leite. “A espada do Basileus: a política imperial e a cristianização do exército romano, século IV d.C.” 2012. Web. 15 Apr 2021.

Vancouver:

Teixeira RL. A espada do Basileus: a política imperial e a cristianização do exército romano, século IV d.C. [Internet] [Thesis]. Universidade de Lisboa; 2012. [cited 2021 Apr 15]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7936.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Teixeira RL. A espada do Basileus: a política imperial e a cristianização do exército romano, século IV d.C. [Thesis]. Universidade de Lisboa; 2012. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7936

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

30. Malczyk, Evan. Assessing Mercury Exposure Risk in the Lake Zapotlán Watershed, Mexico.

Degree: 2009, University of Toronto

Mercury is an environmental contaminant of global concern. The distribution of mercury in freshwater systems is poorly characterized in Mexico, despite widespread contamination from industrial… (more)

Subjects/Keywords: mercury; effluents; Mexico; fisheries; wetland; 0425; 0768; 0383

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APA (6th Edition):

Malczyk, E. (2009). Assessing Mercury Exposure Risk in the Lake Zapotlán Watershed, Mexico. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18904

Chicago Manual of Style (16th Edition):

Malczyk, Evan. “Assessing Mercury Exposure Risk in the Lake Zapotlán Watershed, Mexico.” 2009. Masters Thesis, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/18904.

MLA Handbook (7th Edition):

Malczyk, Evan. “Assessing Mercury Exposure Risk in the Lake Zapotlán Watershed, Mexico.” 2009. Web. 15 Apr 2021.

Vancouver:

Malczyk E. Assessing Mercury Exposure Risk in the Lake Zapotlán Watershed, Mexico. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/18904.

Council of Science Editors:

Malczyk E. Assessing Mercury Exposure Risk in the Lake Zapotlán Watershed, Mexico. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18904

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