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You searched for subject:( pgc). Showing records 1 – 30 of 98 total matches.

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University of Cambridge

1. Ahmad, Shiraz. Mechanisms of ventricular arrhythmogenesis in the age dependent Pgc-1β -/- model of mitochondrial dysfunction.

Degree: 2019, University of Cambridge

 Sudden cardiac death is a leading cause of mortality worldwide. Ventricular arrhythmias, both ventricular tachycardia and ventricular fibrillation, are the primary arrhythmias that lead to… (more)

Subjects/Keywords: PGC; cardiac; arrhythmia

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APA (6th Edition):

Ahmad, S. (2019). Mechanisms of ventricular arrhythmogenesis in the age dependent Pgc-1β -/- model of mitochondrial dysfunction. (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/293627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahmad, Shiraz. “Mechanisms of ventricular arrhythmogenesis in the age dependent Pgc-1β -/- model of mitochondrial dysfunction. ” 2019. Thesis, University of Cambridge. Accessed July 21, 2019. https://www.repository.cam.ac.uk/handle/1810/293627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahmad, Shiraz. “Mechanisms of ventricular arrhythmogenesis in the age dependent Pgc-1β -/- model of mitochondrial dysfunction. ” 2019. Web. 21 Jul 2019.

Vancouver:

Ahmad S. Mechanisms of ventricular arrhythmogenesis in the age dependent Pgc-1β -/- model of mitochondrial dysfunction. [Internet] [Thesis]. University of Cambridge; 2019. [cited 2019 Jul 21]. Available from: https://www.repository.cam.ac.uk/handle/1810/293627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahmad S. Mechanisms of ventricular arrhythmogenesis in the age dependent Pgc-1β -/- model of mitochondrial dysfunction. [Thesis]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/293627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

2. Sutherland, Lindsey. Regulation of PGC-1 alpha in White Adipose Tissue by Exercise.

Degree: MS, Department of Agricultural, Food, and Nutritional Science, 2009, University of Alberta

 This project investigated the effects of exercise and epinephrine on the mRNA expression of peroxisome proliferator activated receptor gamma coactivator-1 alpha (PGC-1 alpha), a master… (more)

Subjects/Keywords: mitochondria; adipose; PGC-1

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APA (6th Edition):

Sutherland, L. (2009). Regulation of PGC-1 alpha in White Adipose Tissue by Exercise. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/vq27zp66j

Chicago Manual of Style (16th Edition):

Sutherland, Lindsey. “Regulation of PGC-1 alpha in White Adipose Tissue by Exercise.” 2009. Masters Thesis, University of Alberta. Accessed July 21, 2019. https://era.library.ualberta.ca/files/vq27zp66j.

MLA Handbook (7th Edition):

Sutherland, Lindsey. “Regulation of PGC-1 alpha in White Adipose Tissue by Exercise.” 2009. Web. 21 Jul 2019.

Vancouver:

Sutherland L. Regulation of PGC-1 alpha in White Adipose Tissue by Exercise. [Internet] [Masters thesis]. University of Alberta; 2009. [cited 2019 Jul 21]. Available from: https://era.library.ualberta.ca/files/vq27zp66j.

Council of Science Editors:

Sutherland L. Regulation of PGC-1 alpha in White Adipose Tissue by Exercise. [Masters Thesis]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/vq27zp66j


Virginia Commonwealth University

3. Hedrick, Shannon. IDENTIFICATION OF HUMAN PGC-1α-b ISOFORMS USING A NOVEL PGC-1α-b SPECIFIC ANTIBODY.

Degree: MS, Microbiology & Immunology, 2013, Virginia Commonwealth University

 Peroxisome proliferator-activated receptor gamma coactivator 1-alpha (PGC-1α) is known as the master regulator of mitochondrial biogenesis. PGC-1α holds this role by acting as a transcriptional… (more)

Subjects/Keywords: PGC-1a; PGC-1α; PGC-1α-b; PPARGC1A; PPARGC1α; PGC-1α-b antibody; PGC-1α isoforms; mitochondrial biogenesis; Medicine and Health Sciences

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APA (6th Edition):

Hedrick, S. (2013). IDENTIFICATION OF HUMAN PGC-1α-b ISOFORMS USING A NOVEL PGC-1α-b SPECIFIC ANTIBODY. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/3225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hedrick, Shannon. “IDENTIFICATION OF HUMAN PGC-1α-b ISOFORMS USING A NOVEL PGC-1α-b SPECIFIC ANTIBODY.” 2013. Thesis, Virginia Commonwealth University. Accessed July 21, 2019. https://scholarscompass.vcu.edu/etd/3225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hedrick, Shannon. “IDENTIFICATION OF HUMAN PGC-1α-b ISOFORMS USING A NOVEL PGC-1α-b SPECIFIC ANTIBODY.” 2013. Web. 21 Jul 2019.

Vancouver:

Hedrick S. IDENTIFICATION OF HUMAN PGC-1α-b ISOFORMS USING A NOVEL PGC-1α-b SPECIFIC ANTIBODY. [Internet] [Thesis]. Virginia Commonwealth University; 2013. [cited 2019 Jul 21]. Available from: https://scholarscompass.vcu.edu/etd/3225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hedrick S. IDENTIFICATION OF HUMAN PGC-1α-b ISOFORMS USING A NOVEL PGC-1α-b SPECIFIC ANTIBODY. [Thesis]. Virginia Commonwealth University; 2013. Available from: https://scholarscompass.vcu.edu/etd/3225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

4. Genge, Christine E. Scaling of Metabolic Enzymes: Transcriptional Basis of Interspecies Variation in Mitochondrial Content .

Degree: Biology, 2010, Queens University

 Mitochondrial content, an important determinant of muscle metabolic capacity, changes in individuals during development, and in response to physiological and environmental challenges. This phenotypic plasticity… (more)

Subjects/Keywords: Mitochondria; Allometric Scaling; Transcriptional Regulation; PGC-1alpha

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APA (6th Edition):

Genge, C. E. (2010). Scaling of Metabolic Enzymes: Transcriptional Basis of Interspecies Variation in Mitochondrial Content . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/5710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Genge, Christine E. “Scaling of Metabolic Enzymes: Transcriptional Basis of Interspecies Variation in Mitochondrial Content .” 2010. Thesis, Queens University. Accessed July 21, 2019. http://hdl.handle.net/1974/5710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Genge, Christine E. “Scaling of Metabolic Enzymes: Transcriptional Basis of Interspecies Variation in Mitochondrial Content .” 2010. Web. 21 Jul 2019.

Vancouver:

Genge CE. Scaling of Metabolic Enzymes: Transcriptional Basis of Interspecies Variation in Mitochondrial Content . [Internet] [Thesis]. Queens University; 2010. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/1974/5710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Genge CE. Scaling of Metabolic Enzymes: Transcriptional Basis of Interspecies Variation in Mitochondrial Content . [Thesis]. Queens University; 2010. Available from: http://hdl.handle.net/1974/5710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

5. Thériault, Mathieu. Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal .

Degree: 2012, Université de Sherbrooke

 Le récepteur relié au récepteur à l'estrogène alpha (ERR?) est un récepteur orphelin de la superfamille des récepteurs nucléaires impliqué dans la régulation du métabolisme… (more)

Subjects/Keywords: IFRD1; PRC; PGC-1; ERR[alpha]

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APA (6th Edition):

Thériault, M. (2012). Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6364

Chicago Manual of Style (16th Edition):

Thériault, Mathieu. “Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal .” 2012. Masters Thesis, Université de Sherbrooke. Accessed July 21, 2019. http://hdl.handle.net/11143/6364.

MLA Handbook (7th Edition):

Thériault, Mathieu. “Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal .” 2012. Web. 21 Jul 2019.

Vancouver:

Thériault M. Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal . [Internet] [Masters thesis]. Université de Sherbrooke; 2012. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/11143/6364.

Council of Science Editors:

Thériault M. Étude de l'interaction de ERR[alpha] et de ses corégulateurs dans le carcinome colorectal . [Masters Thesis]. Université de Sherbrooke; 2012. Available from: http://hdl.handle.net/11143/6364


University of Toronto

6. Jiang, Qi. Signalling Through the PGC-1a Pathway Mediates an Inducible Stress Response in Retinal Astrocytes to Resist Oxidative and Metabolic Insults.

Degree: 2015, University of Toronto

Glaucoma is a neurodegenerative disease characterized by the loss of retinal ganglion cells (RGCs) in the eye. Astrocytes are an adjacent cell type that provides… (more)

Subjects/Keywords: Astrocytes; Glaucoma; Oxidative Stress; PGC-1a; 0317

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APA (6th Edition):

Jiang, Q. (2015). Signalling Through the PGC-1a Pathway Mediates an Inducible Stress Response in Retinal Astrocytes to Resist Oxidative and Metabolic Insults. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69646

Chicago Manual of Style (16th Edition):

Jiang, Qi. “Signalling Through the PGC-1a Pathway Mediates an Inducible Stress Response in Retinal Astrocytes to Resist Oxidative and Metabolic Insults.” 2015. Masters Thesis, University of Toronto. Accessed July 21, 2019. http://hdl.handle.net/1807/69646.

MLA Handbook (7th Edition):

Jiang, Qi. “Signalling Through the PGC-1a Pathway Mediates an Inducible Stress Response in Retinal Astrocytes to Resist Oxidative and Metabolic Insults.” 2015. Web. 21 Jul 2019.

Vancouver:

Jiang Q. Signalling Through the PGC-1a Pathway Mediates an Inducible Stress Response in Retinal Astrocytes to Resist Oxidative and Metabolic Insults. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/1807/69646.

Council of Science Editors:

Jiang Q. Signalling Through the PGC-1a Pathway Mediates an Inducible Stress Response in Retinal Astrocytes to Resist Oxidative and Metabolic Insults. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69646

7. Fogarty, Stuart A. SMURF1 as a Novel Regulator of PGC-1a.

Degree: Biological Sciences - Cell and Molecular: M.S., Biology, 2016, St. Cloud State University

  Parkinson’s disease is a neurodegenerative disorder caused by the impairment and/or death of the dopaminergic neurons in the area of the brain that controls… (more)

Subjects/Keywords: SMURF1 CRISPR siRNA PGC-1 Parkinson's Regulator

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APA (6th Edition):

Fogarty, S. A. (2016). SMURF1 as a Novel Regulator of PGC-1a. (Masters Thesis). St. Cloud State University. Retrieved from https://repository.stcloudstate.edu/biol_etds/9

Chicago Manual of Style (16th Edition):

Fogarty, Stuart A. “SMURF1 as a Novel Regulator of PGC-1a.” 2016. Masters Thesis, St. Cloud State University. Accessed July 21, 2019. https://repository.stcloudstate.edu/biol_etds/9.

MLA Handbook (7th Edition):

Fogarty, Stuart A. “SMURF1 as a Novel Regulator of PGC-1a.” 2016. Web. 21 Jul 2019.

Vancouver:

Fogarty SA. SMURF1 as a Novel Regulator of PGC-1a. [Internet] [Masters thesis]. St. Cloud State University; 2016. [cited 2019 Jul 21]. Available from: https://repository.stcloudstate.edu/biol_etds/9.

Council of Science Editors:

Fogarty SA. SMURF1 as a Novel Regulator of PGC-1a. [Masters Thesis]. St. Cloud State University; 2016. Available from: https://repository.stcloudstate.edu/biol_etds/9

8. Llimona, Flávia. Co-ativador de transcrição gênica PGC-1 na pancreatite aguda.

Degree: PhD, Processos Inflamatórios e Alérgicos, 2011, University of São Paulo

PGC-1 é uma família de coativadores de fatores de transcrição que controlam a expressão de diversos genes envolvidos na homeostase energética celular. As isoformas PGC-1… (more)

Subjects/Keywords: Bacterial translocation; Fagocitose; Pancreatite; Pancreatitis; PGC-1; PGC-1; Phagocytosis; Sepse; Sepsis; Translocação bacteriana

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APA (6th Edition):

Llimona, F. (2011). Co-ativador de transcrição gênica PGC-1 na pancreatite aguda. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;

Chicago Manual of Style (16th Edition):

Llimona, Flávia. “Co-ativador de transcrição gênica PGC-1 na pancreatite aguda.” 2011. Doctoral Dissertation, University of São Paulo. Accessed July 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;.

MLA Handbook (7th Edition):

Llimona, Flávia. “Co-ativador de transcrição gênica PGC-1 na pancreatite aguda.” 2011. Web. 21 Jul 2019.

Vancouver:

Llimona F. Co-ativador de transcrição gênica PGC-1 na pancreatite aguda. [Internet] [Doctoral dissertation]. University of São Paulo; 2011. [cited 2019 Jul 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;.

Council of Science Editors:

Llimona F. Co-ativador de transcrição gênica PGC-1 na pancreatite aguda. [Doctoral Dissertation]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-20052011-174649/ ;

9. Passos, Luis Augusto Abreu da Cunha. A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma.

Degree: PhD, Processos Inflamatórios e Alérgicos, 2015, University of São Paulo

PGC-1 beta é um co-ativador de transcrição gênica responsável pela regulação do metabolismo celular, principalmente na biogênese e função mitocondrial, disponibilidade de substrato e síntese… (more)

Subjects/Keywords: Cell proliferation; Cutaneous neoplasias; Melanoma; Melanoma; Neoplasias cutâneas; PGC-1; PGC-1beta; Proliferação de células

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APA (6th Edition):

Passos, L. A. A. d. C. (2015). A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;

Chicago Manual of Style (16th Edition):

Passos, Luis Augusto Abreu da Cunha. “A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma.” 2015. Doctoral Dissertation, University of São Paulo. Accessed July 21, 2019. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;.

MLA Handbook (7th Edition):

Passos, Luis Augusto Abreu da Cunha. “A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma.” 2015. Web. 21 Jul 2019.

Vancouver:

Passos LAAdC. A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma. [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2019 Jul 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;.

Council of Science Editors:

Passos LAAdC. A sinalização do co-ativador de transcrição PGC-1beta e sua relevância para a proliferação celular e desenvolvimento de melanoma. [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5165/tde-31032015-162021/ ;

10. Bamba Funck, Jessica. Etude des isoformes du gène PGC-1a dans le développement musculaire chez le bovin : Study of the PGC-1α gene isoforms in muscle development in cattle.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Limoges

Le coactivateur de facteurs de transcription PGC-1a (PPARC1A) est connu pour jouer un rôle clé dans la thermogénèse adaptative ainsi que dans l’homéostasie et la… (more)

Subjects/Keywords: PPARGC1A; PGC-1a; Muscle squelettique; Bovin; PPARGC1A; PGC-1a; Skeletal muscle; Bovin; 572.8

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APA (6th Edition):

Bamba Funck, J. (2018). Etude des isoformes du gène PGC-1a dans le développement musculaire chez le bovin : Study of the PGC-1α gene isoforms in muscle development in cattle. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2018LIMO0057

Chicago Manual of Style (16th Edition):

Bamba Funck, Jessica. “Etude des isoformes du gène PGC-1a dans le développement musculaire chez le bovin : Study of the PGC-1α gene isoforms in muscle development in cattle.” 2018. Doctoral Dissertation, Limoges. Accessed July 21, 2019. http://www.theses.fr/2018LIMO0057.

MLA Handbook (7th Edition):

Bamba Funck, Jessica. “Etude des isoformes du gène PGC-1a dans le développement musculaire chez le bovin : Study of the PGC-1α gene isoforms in muscle development in cattle.” 2018. Web. 21 Jul 2019.

Vancouver:

Bamba Funck J. Etude des isoformes du gène PGC-1a dans le développement musculaire chez le bovin : Study of the PGC-1α gene isoforms in muscle development in cattle. [Internet] [Doctoral dissertation]. Limoges; 2018. [cited 2019 Jul 21]. Available from: http://www.theses.fr/2018LIMO0057.

Council of Science Editors:

Bamba Funck J. Etude des isoformes du gène PGC-1a dans le développement musculaire chez le bovin : Study of the PGC-1α gene isoforms in muscle development in cattle. [Doctoral Dissertation]. Limoges; 2018. Available from: http://www.theses.fr/2018LIMO0057

11. Mazzucatto, Flávio. Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica.

Degree: Mestrado, Biodinâmica do Movimento Humano, 2013, University of São Paulo

O aumento do consumo de alimentos ricos em gorduras e carboidratos associado à reduzida prática de exercícios físicos pode ter como consequência o desenvolvimento da… (more)

Subjects/Keywords: Capilarização; Capillarity; Dieta hipercalórica; Glicemia; Glycemia; Hypercaloric diet; PGC-1?; PGC-1?; Physical training; Treinamento físico

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APA (6th Edition):

Mazzucatto, F. (2013). Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;

Chicago Manual of Style (16th Edition):

Mazzucatto, Flávio. “Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica.” 2013. Masters Thesis, University of São Paulo. Accessed July 21, 2019. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;.

MLA Handbook (7th Edition):

Mazzucatto, Flávio. “Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica.” 2013. Web. 21 Jul 2019.

Vancouver:

Mazzucatto F. Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2019 Jul 21]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;.

Council of Science Editors:

Mazzucatto F. Potencial do treinamento físico para a prevenção de distúrbios metabólicos induzidos por dieta hipercalórica. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-06082013-135619/ ;

12. Derbré, Frédéric. Etude des voies de signalisation impliquées dans la sarcopénie : rôle du stress oxydant et de l'inactivité physique : Cell signaling involved in sarcopenia : role of oxidative stress and physical inactivity.

Degree: Docteur es, Sciences et techniques des activités physiques et sportives (STAPS), 2011, Rennes 2

La sarcopénie est considérée comme un syndrome gériatrique se caractérisant par une diminution de la masse musculaire qui en s’aggravant sera à l’origine d’une détérioration… (more)

Subjects/Keywords: Sarcopénie; Stress oxydant; Vieillissement; Inactivité; Muscle, PGC-1α; NF-κB; Sarcopenia; Skeletal muscle; Oxidative stress; Inactivity; PGC-1α; NF-κB

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APA (6th Edition):

Derbré, F. (2011). Etude des voies de signalisation impliquées dans la sarcopénie : rôle du stress oxydant et de l'inactivité physique : Cell signaling involved in sarcopenia : role of oxidative stress and physical inactivity. (Doctoral Dissertation). Rennes 2. Retrieved from http://www.theses.fr/2011REN20036

Chicago Manual of Style (16th Edition):

Derbré, Frédéric. “Etude des voies de signalisation impliquées dans la sarcopénie : rôle du stress oxydant et de l'inactivité physique : Cell signaling involved in sarcopenia : role of oxidative stress and physical inactivity.” 2011. Doctoral Dissertation, Rennes 2. Accessed July 21, 2019. http://www.theses.fr/2011REN20036.

MLA Handbook (7th Edition):

Derbré, Frédéric. “Etude des voies de signalisation impliquées dans la sarcopénie : rôle du stress oxydant et de l'inactivité physique : Cell signaling involved in sarcopenia : role of oxidative stress and physical inactivity.” 2011. Web. 21 Jul 2019.

Vancouver:

Derbré F. Etude des voies de signalisation impliquées dans la sarcopénie : rôle du stress oxydant et de l'inactivité physique : Cell signaling involved in sarcopenia : role of oxidative stress and physical inactivity. [Internet] [Doctoral dissertation]. Rennes 2; 2011. [cited 2019 Jul 21]. Available from: http://www.theses.fr/2011REN20036.

Council of Science Editors:

Derbré F. Etude des voies de signalisation impliquées dans la sarcopénie : rôle du stress oxydant et de l'inactivité physique : Cell signaling involved in sarcopenia : role of oxidative stress and physical inactivity. [Doctoral Dissertation]. Rennes 2; 2011. Available from: http://www.theses.fr/2011REN20036


Universidade do Estado do Rio de Janeiro

13. Annie Seixas Bello Moreira. Disfunção mitocondrial e expressão gênica alterada como mecanismos envolvidos na progressão da hipertrofia para insuficiência cardíaca em camundongos CIRSKO e PGC-1βKO.

Degree: PhD, 2009, Universidade do Estado do Rio de Janeiro

A insuficiência cardíaca (IC) é a evolução final das várias formas de doenças cardiovascular, sendo resultado de modificações estruturais, metabólicas e de contratilidade miocárdica. A… (more)

Subjects/Keywords: IRS; Heart failure; PGC-1b; Expressão gênica; Função mitocondrial; IRS; PGC-1b; Insuficiência cardíaca; FISIOLOGIA; Mitochondrial function; Gene expression

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APA (6th Edition):

Moreira, A. S. B. (2009). Disfunção mitocondrial e expressão gênica alterada como mecanismos envolvidos na progressão da hipertrofia para insuficiência cardíaca em camundongos CIRSKO e PGC-1βKO. (Doctoral Dissertation). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=1494 ;

Chicago Manual of Style (16th Edition):

Moreira, Annie Seixas Bello. “Disfunção mitocondrial e expressão gênica alterada como mecanismos envolvidos na progressão da hipertrofia para insuficiência cardíaca em camundongos CIRSKO e PGC-1βKO.” 2009. Doctoral Dissertation, Universidade do Estado do Rio de Janeiro. Accessed July 21, 2019. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=1494 ;.

MLA Handbook (7th Edition):

Moreira, Annie Seixas Bello. “Disfunção mitocondrial e expressão gênica alterada como mecanismos envolvidos na progressão da hipertrofia para insuficiência cardíaca em camundongos CIRSKO e PGC-1βKO.” 2009. Web. 21 Jul 2019.

Vancouver:

Moreira ASB. Disfunção mitocondrial e expressão gênica alterada como mecanismos envolvidos na progressão da hipertrofia para insuficiência cardíaca em camundongos CIRSKO e PGC-1βKO. [Internet] [Doctoral dissertation]. Universidade do Estado do Rio de Janeiro; 2009. [cited 2019 Jul 21]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=1494 ;.

Council of Science Editors:

Moreira ASB. Disfunção mitocondrial e expressão gênica alterada como mecanismos envolvidos na progressão da hipertrofia para insuficiência cardíaca em camundongos CIRSKO e PGC-1βKO. [Doctoral Dissertation]. Universidade do Estado do Rio de Janeiro; 2009. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=1494 ;

14. Pauly, Marion. La mitochondrie, une sentinelle dans le remodelage musculaire : réflexions autour du vieillissement et de la dystrophie de Duchenne : Mitochondria, a sentinel in muscle remodeling : new insights on aging and Duchenne muscular dystrophy.

Degree: Docteur es, Sciences et techniques des activités physiques et sportives, 2013, Université Montpellier I

Essentielle à l'équilibre énergétique de la cellule, la mitochondrie, véritable sentinelle, joue, un rôle majeur dans le destin de la cellule, en modulant les voies… (more)

Subjects/Keywords: Mdx; Myostatine; PGC-1a; Autophagie; Stress du réticulum endoplasmique; Aicar; Mdx; Myostatin; PGC-1a; Autophagy; Endoplasmic reticulum stress; Aicar

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pauly, M. (2013). La mitochondrie, une sentinelle dans le remodelage musculaire : réflexions autour du vieillissement et de la dystrophie de Duchenne : Mitochondria, a sentinel in muscle remodeling : new insights on aging and Duchenne muscular dystrophy. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2013MON1T016

Chicago Manual of Style (16th Edition):

Pauly, Marion. “La mitochondrie, une sentinelle dans le remodelage musculaire : réflexions autour du vieillissement et de la dystrophie de Duchenne : Mitochondria, a sentinel in muscle remodeling : new insights on aging and Duchenne muscular dystrophy.” 2013. Doctoral Dissertation, Université Montpellier I. Accessed July 21, 2019. http://www.theses.fr/2013MON1T016.

MLA Handbook (7th Edition):

Pauly, Marion. “La mitochondrie, une sentinelle dans le remodelage musculaire : réflexions autour du vieillissement et de la dystrophie de Duchenne : Mitochondria, a sentinel in muscle remodeling : new insights on aging and Duchenne muscular dystrophy.” 2013. Web. 21 Jul 2019.

Vancouver:

Pauly M. La mitochondrie, une sentinelle dans le remodelage musculaire : réflexions autour du vieillissement et de la dystrophie de Duchenne : Mitochondria, a sentinel in muscle remodeling : new insights on aging and Duchenne muscular dystrophy. [Internet] [Doctoral dissertation]. Université Montpellier I; 2013. [cited 2019 Jul 21]. Available from: http://www.theses.fr/2013MON1T016.

Council of Science Editors:

Pauly M. La mitochondrie, une sentinelle dans le remodelage musculaire : réflexions autour du vieillissement et de la dystrophie de Duchenne : Mitochondria, a sentinel in muscle remodeling : new insights on aging and Duchenne muscular dystrophy. [Doctoral Dissertation]. Université Montpellier I; 2013. Available from: http://www.theses.fr/2013MON1T016


Princeton University

15. Eagle, Whitby Vernon Ishmael. CIS AND TRANS REGULATION OF GERM PLASM mRNA LOCALIZATION IN DROSOPHILA .

Degree: PhD, 2017, Princeton University

 ABSTRACT Subcellular mRNA localization is an efficacious and pervasive strategy for generating the asymmetric protein distributions necessary for cellular and developmental polarity. During early Drosophila… (more)

Subjects/Keywords: Drosophila; gcl; germ plasm; mRNA localization; pgc; polar granule

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Eagle, W. V. I. (2017). CIS AND TRANS REGULATION OF GERM PLASM mRNA LOCALIZATION IN DROSOPHILA . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01p2676z200

Chicago Manual of Style (16th Edition):

Eagle, Whitby Vernon Ishmael. “CIS AND TRANS REGULATION OF GERM PLASM mRNA LOCALIZATION IN DROSOPHILA .” 2017. Doctoral Dissertation, Princeton University. Accessed July 21, 2019. http://arks.princeton.edu/ark:/88435/dsp01p2676z200.

MLA Handbook (7th Edition):

Eagle, Whitby Vernon Ishmael. “CIS AND TRANS REGULATION OF GERM PLASM mRNA LOCALIZATION IN DROSOPHILA .” 2017. Web. 21 Jul 2019.

Vancouver:

Eagle WVI. CIS AND TRANS REGULATION OF GERM PLASM mRNA LOCALIZATION IN DROSOPHILA . [Internet] [Doctoral dissertation]. Princeton University; 2017. [cited 2019 Jul 21]. Available from: http://arks.princeton.edu/ark:/88435/dsp01p2676z200.

Council of Science Editors:

Eagle WVI. CIS AND TRANS REGULATION OF GERM PLASM mRNA LOCALIZATION IN DROSOPHILA . [Doctoral Dissertation]. Princeton University; 2017. Available from: http://arks.princeton.edu/ark:/88435/dsp01p2676z200


Kyoto University / 京都大学

16. Matsuoka, Tatsuhiko. Critical roles of nardilysin in the maintenance of body temperature homoeostasis : ナルディライジンは体温恒常性維持に重要な役割を果たす.

Degree: 博士(医学), 2014, Kyoto University / 京都大学

Yoshinori Hiraoka, Tatsuhiko Matsuoka, Mikiko Ohno, Kazuhiro Nakamura, Sayaka Saijo, Shigenobu Matsumura, Kiyoto Nishi, Jiro Sakamoto, Po-Min Chen, Kazuo Inoue, Tohru Fushiki, Toru Kita, Takeshi Kimura & Eiichiro Nishi "Critical roles of nardilysin in the maintenance of body temperature homoeostasis" Nature Communications 5, Article number: 3224 doi:10.1038/ncomms4224

新制・課程博士

甲第18454号

医博第3909号

Subjects/Keywords: body temperature homoeostasis; brown adipose tissue; UCP1; PGC-1α; adaptive thermogenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Matsuoka, T. (2014). Critical roles of nardilysin in the maintenance of body temperature homoeostasis : ナルディライジンは体温恒常性維持に重要な役割を果たす. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/189347 ; http://dx.doi.org/10.14989/doctor.k18454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matsuoka, Tatsuhiko. “Critical roles of nardilysin in the maintenance of body temperature homoeostasis : ナルディライジンは体温恒常性維持に重要な役割を果たす.” 2014. Thesis, Kyoto University / 京都大学. Accessed July 21, 2019. http://hdl.handle.net/2433/189347 ; http://dx.doi.org/10.14989/doctor.k18454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matsuoka, Tatsuhiko. “Critical roles of nardilysin in the maintenance of body temperature homoeostasis : ナルディライジンは体温恒常性維持に重要な役割を果たす.” 2014. Web. 21 Jul 2019.

Vancouver:

Matsuoka T. Critical roles of nardilysin in the maintenance of body temperature homoeostasis : ナルディライジンは体温恒常性維持に重要な役割を果たす. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/2433/189347 ; http://dx.doi.org/10.14989/doctor.k18454.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matsuoka T. Critical roles of nardilysin in the maintenance of body temperature homoeostasis : ナルディライジンは体温恒常性維持に重要な役割を果たす. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/189347 ; http://dx.doi.org/10.14989/doctor.k18454

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. 小山, 淑正. Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Reversed feeding uncouples peripheral and master clock gene rhythms and leads to an increased risk of disease development. The aim of this study was to… (more)

Subjects/Keywords: sepsis; sirtuin-1; PGC-1α; pro-inflammatory cytokines; survival rate

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APA (6th Edition):

小山, . (2016). Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Thesis, Oita University / 大分大学. Accessed July 21, 2019. http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Web. 21 Jul 2019.

Vancouver:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

18. Zarebidaki, Eleen. Browning of white adipose tissue by melatonin.

Degree: MS, Biology, 2015, Georgia State University

  There are two distinct types of adipose tissue which have different functions within the body, white (WAT) and brown (BAT). Browning of WAT occurs… (more)

Subjects/Keywords: Brown adipose tissue; Lipolysis; Obesity; UCP1; PGC-1α

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zarebidaki, E. (2015). Browning of white adipose tissue by melatonin. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zarebidaki, Eleen. “Browning of white adipose tissue by melatonin.” 2015. Thesis, Georgia State University. Accessed July 21, 2019. https://scholarworks.gsu.edu/biology_theses/65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zarebidaki, Eleen. “Browning of white adipose tissue by melatonin.” 2015. Web. 21 Jul 2019.

Vancouver:

Zarebidaki E. Browning of white adipose tissue by melatonin. [Internet] [Thesis]. Georgia State University; 2015. [cited 2019 Jul 21]. Available from: https://scholarworks.gsu.edu/biology_theses/65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zarebidaki E. Browning of white adipose tissue by melatonin. [Thesis]. Georgia State University; 2015. Available from: https://scholarworks.gsu.edu/biology_theses/65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

19. Miller, Spencer. AMP Deaminase 3 overexpression regulates cellular energetics and signaling for PGC-1α activation.

Degree: 2017, East Carolina University

 Skeletal muscles undergoing atrophy have decreased [ATP], PGC-1α expression, and mitochondrial content. This combination of findings is unexpected because decreased [ATP] is often associated with… (more)

Subjects/Keywords: Skeletal muscle; mitochondria; atrophy; AMPK; PGC-1α; ATP

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APA (6th Edition):

Miller, S. (2017). AMP Deaminase 3 overexpression regulates cellular energetics and signaling for PGC-1α activation. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/6376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miller, Spencer. “AMP Deaminase 3 overexpression regulates cellular energetics and signaling for PGC-1α activation.” 2017. Thesis, East Carolina University. Accessed July 21, 2019. http://hdl.handle.net/10342/6376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miller, Spencer. “AMP Deaminase 3 overexpression regulates cellular energetics and signaling for PGC-1α activation.” 2017. Web. 21 Jul 2019.

Vancouver:

Miller S. AMP Deaminase 3 overexpression regulates cellular energetics and signaling for PGC-1α activation. [Internet] [Thesis]. East Carolina University; 2017. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/10342/6376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miller S. AMP Deaminase 3 overexpression regulates cellular energetics and signaling for PGC-1α activation. [Thesis]. East Carolina University; 2017. Available from: http://hdl.handle.net/10342/6376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oslo

20. Staurseth, Julie. PGC-1β's role in the regulation of adult mice muscle plasticity.

Degree: 2009, University of Oslo

 Adult skeletal muscle fibers show an ability to undergo phenotypic alterations without cell death or regeneration in response to environmental changes. Important factors affecting the… (more)

Subjects/Keywords: PGC-1B skjelettmuskel fiber typing mus; VDP::473

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APA (6th Edition):

Staurseth, J. (2009). PGC-1β's role in the regulation of adult mice muscle plasticity. (Thesis). University of Oslo. Retrieved from http://urn.nb.no/URN:NBN:no-23864 ; https://www.duo.uio.no/handle/10852/11422 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/11422/1/JuliexStaursethxOppgave.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Staurseth, Julie. “PGC-1β's role in the regulation of adult mice muscle plasticity.” 2009. Thesis, University of Oslo. Accessed July 21, 2019. http://urn.nb.no/URN:NBN:no-23864 ; https://www.duo.uio.no/handle/10852/11422 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/11422/1/JuliexStaursethxOppgave.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Staurseth, Julie. “PGC-1β's role in the regulation of adult mice muscle plasticity.” 2009. Web. 21 Jul 2019.

Vancouver:

Staurseth J. PGC-1β's role in the regulation of adult mice muscle plasticity. [Internet] [Thesis]. University of Oslo; 2009. [cited 2019 Jul 21]. Available from: http://urn.nb.no/URN:NBN:no-23864 ; https://www.duo.uio.no/handle/10852/11422 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/11422/1/JuliexStaursethxOppgave.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Staurseth J. PGC-1β's role in the regulation of adult mice muscle plasticity. [Thesis]. University of Oslo; 2009. Available from: http://urn.nb.no/URN:NBN:no-23864 ; https://www.duo.uio.no/handle/10852/11422 ; Fulltext https://www.duo.uio.no/bitstream/handle/10852/11422/1/JuliexStaursethxOppgave.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Baylor University

21. Schwarz, Neil Andrew. Effect of resistance exercise intensity on the expression of PGC-1alpha isoforms and the anabolic and catabolic signaling mediators, IGF-1 and myostatin, in human skeletal muscle.

Degree: Health, Human Performance and Recreation., 2014, Baylor University

 Proliferator-activated receptor gamma coactivator-1 alpha (PGC-1α) is a protein mechanistically involved in skeletal muscle adaptations to exercise. Recently, novel isoforms of PGC-1α have been identified,… (more)

Subjects/Keywords: Resistance exercise.; PGC-1alpha.; Skeletal muscle.; Gene expression.

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APA (6th Edition):

Schwarz, N. A. (2014). Effect of resistance exercise intensity on the expression of PGC-1alpha isoforms and the anabolic and catabolic signaling mediators, IGF-1 and myostatin, in human skeletal muscle. (Thesis). Baylor University. Retrieved from http://hdl.handle.net/2104/9104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schwarz, Neil Andrew. “Effect of resistance exercise intensity on the expression of PGC-1alpha isoforms and the anabolic and catabolic signaling mediators, IGF-1 and myostatin, in human skeletal muscle. ” 2014. Thesis, Baylor University. Accessed July 21, 2019. http://hdl.handle.net/2104/9104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schwarz, Neil Andrew. “Effect of resistance exercise intensity on the expression of PGC-1alpha isoforms and the anabolic and catabolic signaling mediators, IGF-1 and myostatin, in human skeletal muscle. ” 2014. Web. 21 Jul 2019.

Vancouver:

Schwarz NA. Effect of resistance exercise intensity on the expression of PGC-1alpha isoforms and the anabolic and catabolic signaling mediators, IGF-1 and myostatin, in human skeletal muscle. [Internet] [Thesis]. Baylor University; 2014. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/2104/9104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schwarz NA. Effect of resistance exercise intensity on the expression of PGC-1alpha isoforms and the anabolic and catabolic signaling mediators, IGF-1 and myostatin, in human skeletal muscle. [Thesis]. Baylor University; 2014. Available from: http://hdl.handle.net/2104/9104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

22. O'Donnell, Kelley. Mitochondrial Transport and Function in Axon Degeneration.

Degree: Neuroscience, 2013, UCLA

 Axon degeneration plays a critical but ill-defined role in Parkinson disease (PD), and is actively regulated by pathways that are not well understood. Mitochondria orchestrate… (more)

Subjects/Keywords: Neurosciences; alpha-synuclein; axon degeneration; mitochondria; Parkinson disease; PGC-1; zebrafish

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APA (6th Edition):

O'Donnell, K. (2013). Mitochondrial Transport and Function in Axon Degeneration. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/0q20j9bg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Donnell, Kelley. “Mitochondrial Transport and Function in Axon Degeneration.” 2013. Thesis, UCLA. Accessed July 21, 2019. http://www.escholarship.org/uc/item/0q20j9bg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Donnell, Kelley. “Mitochondrial Transport and Function in Axon Degeneration.” 2013. Web. 21 Jul 2019.

Vancouver:

O'Donnell K. Mitochondrial Transport and Function in Axon Degeneration. [Internet] [Thesis]. UCLA; 2013. [cited 2019 Jul 21]. Available from: http://www.escholarship.org/uc/item/0q20j9bg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Donnell K. Mitochondrial Transport and Function in Axon Degeneration. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/0q20j9bg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

23. Liu, Chien-Ting. Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes.

Degree: Integrative Biology, 2011, University of California – Berkeley

 During endurance exercise, most (about 75%) of the energy derived from the oxidation of metabolic fuels and ATP hydrolysis of muscle contraction is liberated as… (more)

Subjects/Keywords: Physiology; Cellular biology; AMPK; heat shock; mitochondria; PGC-1alpha; SIRT1; Sirtuin

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APA (6th Edition):

Liu, C. (2011). Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/1s48q979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Chien-Ting. “Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes.” 2011. Thesis, University of California – Berkeley. Accessed July 21, 2019. http://www.escholarship.org/uc/item/1s48q979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Chien-Ting. “Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes.” 2011. Web. 21 Jul 2019.

Vancouver:

Liu C. Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes. [Internet] [Thesis]. University of California – Berkeley; 2011. [cited 2019 Jul 21]. Available from: http://www.escholarship.org/uc/item/1s48q979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu C. Mild Heat Stress Induces Mitochondrial Biogenesis Associated with Activation of the AMPK-SIRT1-PGC-1alpha Pathway in C2C12 Myotubes. [Thesis]. University of California – Berkeley; 2011. Available from: http://www.escholarship.org/uc/item/1s48q979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

24. Ha, Ainhi Duy. Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease .

Degree: 2014, University of Sydney

 There is increasing evidence to suggest that mitochondrial dysfunction plays a key role in the patho-physiology of Parkinson’s Disease (PD). The transcriptional coactivator PGC-1alpha is… (more)

Subjects/Keywords: Parkinson’s Disease; PGC-1alpha; mitochondria; PINK1; parkin; ATP13A2; LRRK2

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APA (6th Edition):

Ha, A. D. (2014). Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/12264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ha, Ainhi Duy. “Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease .” 2014. Thesis, University of Sydney. Accessed July 21, 2019. http://hdl.handle.net/2123/12264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ha, Ainhi Duy. “Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease .” 2014. Web. 21 Jul 2019.

Vancouver:

Ha AD. Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease . [Internet] [Thesis]. University of Sydney; 2014. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/2123/12264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ha AD. Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/12264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

25. Shen, Nicole. CHARACTERIZING PROTEIN ARGININE METHYLTRANSFERASE EXPRESSION AND ACTIVITY DURING MYOGENESIS.

Degree: MSc, 2017, McMaster University

Despite the emerging importance of protein arginine methyltransferases (PRMTs) in regulating skeletal muscle plasticity, the biology of these enzymes during muscle development remains poorly understood.… (more)

Subjects/Keywords: Protein arginine methyltransferase; skeletal muscle; myogenesis; PGC-1α; mitochondria

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shen, N. (2017). CHARACTERIZING PROTEIN ARGININE METHYLTRANSFERASE EXPRESSION AND ACTIVITY DURING MYOGENESIS. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/22267

Chicago Manual of Style (16th Edition):

Shen, Nicole. “CHARACTERIZING PROTEIN ARGININE METHYLTRANSFERASE EXPRESSION AND ACTIVITY DURING MYOGENESIS.” 2017. Masters Thesis, McMaster University. Accessed July 21, 2019. http://hdl.handle.net/11375/22267.

MLA Handbook (7th Edition):

Shen, Nicole. “CHARACTERIZING PROTEIN ARGININE METHYLTRANSFERASE EXPRESSION AND ACTIVITY DURING MYOGENESIS.” 2017. Web. 21 Jul 2019.

Vancouver:

Shen N. CHARACTERIZING PROTEIN ARGININE METHYLTRANSFERASE EXPRESSION AND ACTIVITY DURING MYOGENESIS. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/11375/22267.

Council of Science Editors:

Shen N. CHARACTERIZING PROTEIN ARGININE METHYLTRANSFERASE EXPRESSION AND ACTIVITY DURING MYOGENESIS. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/22267


McMaster University

26. vanLieshout, Tiffany. PRMT Biology During Acute Exercise.

Degree: MSc, 2017, McMaster University

Protein arginine methyltransferase 1 (PRMT1), -4 (also known as coactivator-associated arginine methyltransferase 1; CARM1), and -5 catalyze the methylation of arginine residues on target proteins.… (more)

Subjects/Keywords: Protein arginine methyltransferase; Exercise; Skeletal muscle; Fiber types; PGC-1alpha

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

vanLieshout, T. (2017). PRMT Biology During Acute Exercise. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/22264

Chicago Manual of Style (16th Edition):

vanLieshout, Tiffany. “PRMT Biology During Acute Exercise.” 2017. Masters Thesis, McMaster University. Accessed July 21, 2019. http://hdl.handle.net/11375/22264.

MLA Handbook (7th Edition):

vanLieshout, Tiffany. “PRMT Biology During Acute Exercise.” 2017. Web. 21 Jul 2019.

Vancouver:

vanLieshout T. PRMT Biology During Acute Exercise. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/11375/22264.

Council of Science Editors:

vanLieshout T. PRMT Biology During Acute Exercise. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/22264


University of Gothenburg / Göteborgs Universitet

27. Karlsson, Lars. Effects of PGC1a-induced exercise adaptations in muscle on plasticity and recovery mechanisms in the CNS.

Degree: 2019, University of Gothenburg / Göteborgs Universitet

 In this thesis, we sought to determine if muscle-derived exercise-induced signaling via PGC-1α muscle activation influences neuroplasticity under physiological or pathophysiological conditions. For this purpose,… (more)

Subjects/Keywords: muscle; brain; exercise; transgenic; PGC-1α; FNDC5; irisin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Karlsson, L. (2019). Effects of PGC1a-induced exercise adaptations in muscle on plasticity and recovery mechanisms in the CNS. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/59545

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Karlsson, Lars. “Effects of PGC1a-induced exercise adaptations in muscle on plasticity and recovery mechanisms in the CNS.” 2019. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed July 21, 2019. http://hdl.handle.net/2077/59545.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Karlsson, Lars. “Effects of PGC1a-induced exercise adaptations in muscle on plasticity and recovery mechanisms in the CNS.” 2019. Web. 21 Jul 2019.

Vancouver:

Karlsson L. Effects of PGC1a-induced exercise adaptations in muscle on plasticity and recovery mechanisms in the CNS. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/2077/59545.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Karlsson L. Effects of PGC1a-induced exercise adaptations in muscle on plasticity and recovery mechanisms in the CNS. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. Available from: http://hdl.handle.net/2077/59545

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

28. Jin, Youngnam. Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation.

Degree: PhD, 2011, University of Rochester

 Huntington’s disease (HD) is an inherited neurodegenerative disease resulting from an abnormal expansion of polyglutamine in huntingtin (Htt). Many studies have shown that energetic deficits… (more)

Subjects/Keywords: Huntington Disease; Transcription; Metabolism; PGC-lalpha; PPARgamma; Superoxide; Mitochondria

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jin, Y. (2011). Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14596

Chicago Manual of Style (16th Edition):

Jin, Youngnam. “Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation.” 2011. Doctoral Dissertation, University of Rochester. Accessed July 21, 2019. http://hdl.handle.net/1802/14596.

MLA Handbook (7th Edition):

Jin, Youngnam. “Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation.” 2011. Web. 21 Jul 2019.

Vancouver:

Jin Y. Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/1802/14596.

Council of Science Editors:

Jin Y. Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14596


Université de Montréal

29. Keil, Sarah. Rôle de la ghréline dans la régulation du coactivateur transcriptionnel PGC-1alpha .

Degree: 2014, Université de Montréal

 L’adaptation de l’organisme à son environnement est essentielle à sa survie. L’homéostasie énergétique permet l’équilibre entre les apports, les dépenses et le stockage d’énergie. Un… (more)

Subjects/Keywords: PGC-1alpha; GHS-R1a; Ghréline; Coactivateur transcriptionnel; Récepteurs nucléaires; PPARbeta; PGC-1alpha; GHS-R1a; Ghrelin; Nuclear receptors; Transcriptional coactivator; Metabolic syndrome; Syndrome métabolique; PPARbeta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Keil, S. (2014). Rôle de la ghréline dans la régulation du coactivateur transcriptionnel PGC-1alpha . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/10863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keil, Sarah. “Rôle de la ghréline dans la régulation du coactivateur transcriptionnel PGC-1alpha .” 2014. Thesis, Université de Montréal. Accessed July 21, 2019. http://hdl.handle.net/1866/10863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keil, Sarah. “Rôle de la ghréline dans la régulation du coactivateur transcriptionnel PGC-1alpha .” 2014. Web. 21 Jul 2019.

Vancouver:

Keil S. Rôle de la ghréline dans la régulation du coactivateur transcriptionnel PGC-1alpha . [Internet] [Thesis]. Université de Montréal; 2014. [cited 2019 Jul 21]. Available from: http://hdl.handle.net/1866/10863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keil S. Rôle de la ghréline dans la régulation du coactivateur transcriptionnel PGC-1alpha . [Thesis]. Université de Montréal; 2014. Available from: http://hdl.handle.net/1866/10863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Gali Ramamoorthy, Thanuja. Role of PGC-1β and TIF2 co-regulators in mouse skeletal muscle function : Rôle des co-régulateurs PGC-1ß et TIF2 dans la fonction du muscle squelettique chez la souris.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université de Strasbourg

Le muscle squelettique (MS) est un tissu métabolique important. L'objectif de ma thèse était de caractériser le rôle des corégulateurs de la transcription, PGC-1β (transcriptional… (more)

Subjects/Keywords: Corégulateurs de la transcription; PGC-1β; TIF2; Mitochondries; Stress oxydatif; Muscle squelettique; Métabolisme; Longévité; Transcriptional coregulators; PGC-1β; TIF2; Mitochondria; Oxidative stress; Skeletal muscle; Metabolism; Longevity; 572.4

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gali Ramamoorthy, T. (2013). Role of PGC-1β and TIF2 co-regulators in mouse skeletal muscle function : Rôle des co-régulateurs PGC-1ß et TIF2 dans la fonction du muscle squelettique chez la souris. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ095

Chicago Manual of Style (16th Edition):

Gali Ramamoorthy, Thanuja. “Role of PGC-1β and TIF2 co-regulators in mouse skeletal muscle function : Rôle des co-régulateurs PGC-1ß et TIF2 dans la fonction du muscle squelettique chez la souris.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed July 21, 2019. http://www.theses.fr/2013STRAJ095.

MLA Handbook (7th Edition):

Gali Ramamoorthy, Thanuja. “Role of PGC-1β and TIF2 co-regulators in mouse skeletal muscle function : Rôle des co-régulateurs PGC-1ß et TIF2 dans la fonction du muscle squelettique chez la souris.” 2013. Web. 21 Jul 2019.

Vancouver:

Gali Ramamoorthy T. Role of PGC-1β and TIF2 co-regulators in mouse skeletal muscle function : Rôle des co-régulateurs PGC-1ß et TIF2 dans la fonction du muscle squelettique chez la souris. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2019 Jul 21]. Available from: http://www.theses.fr/2013STRAJ095.

Council of Science Editors:

Gali Ramamoorthy T. Role of PGC-1β and TIF2 co-regulators in mouse skeletal muscle function : Rôle des co-régulateurs PGC-1ß et TIF2 dans la fonction du muscle squelettique chez la souris. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ095

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