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You searched for subject:( myofibroblast). Showing records 1 – 30 of 76 total matches.

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Cornell University

1. Brooks, Emily. Adipose-Derived Progenitor Cell Alterations To The Tumor Microenvironment Promote Tumorigenesis.

Degree: PhD, Biomedical Engineering, 2012, Cornell University

 Rather than simply focusing on the malignant cancer cells, the role of the tumor microenvironment as a whole is increasingly being studied. As this amalgamation… (more)

Subjects/Keywords: stiffness; myofibroblast; breast cancer

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APA (6th Edition):

Brooks, E. (2012). Adipose-Derived Progenitor Cell Alterations To The Tumor Microenvironment Promote Tumorigenesis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/31025

Chicago Manual of Style (16th Edition):

Brooks, Emily. “Adipose-Derived Progenitor Cell Alterations To The Tumor Microenvironment Promote Tumorigenesis.” 2012. Doctoral Dissertation, Cornell University. Accessed September 28, 2020. http://hdl.handle.net/1813/31025.

MLA Handbook (7th Edition):

Brooks, Emily. “Adipose-Derived Progenitor Cell Alterations To The Tumor Microenvironment Promote Tumorigenesis.” 2012. Web. 28 Sep 2020.

Vancouver:

Brooks E. Adipose-Derived Progenitor Cell Alterations To The Tumor Microenvironment Promote Tumorigenesis. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1813/31025.

Council of Science Editors:

Brooks E. Adipose-Derived Progenitor Cell Alterations To The Tumor Microenvironment Promote Tumorigenesis. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/31025


University of Toronto

2. Klingberg, Franco. Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin.

Degree: PhD, 2014, University of Toronto

Fibrosis is a detrimental disease that causes organ failure with no effective therapy available to date. Activated fibroblasts -myofibroblasts- are responsible for the excessive secretion… (more)

Subjects/Keywords: ECM; Myofibroblast; TGF-1; 0306

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APA (6th Edition):

Klingberg, F. (2014). Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/74490

Chicago Manual of Style (16th Edition):

Klingberg, Franco. “Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin.” 2014. Doctoral Dissertation, University of Toronto. Accessed September 28, 2020. http://hdl.handle.net/1807/74490.

MLA Handbook (7th Edition):

Klingberg, Franco. “Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin.” 2014. Web. 28 Sep 2020.

Vancouver:

Klingberg F. Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1807/74490.

Council of Science Editors:

Klingberg F. Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74490

3. Takatsuna, Masafumi. Myofibroblasts of the muscle layer stimulate the malignant potential of colorectal cancer : 固有筋層における筋線維芽細胞は大腸癌の悪性度に関与する.

Degree: 博士(医学), 2016, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第4210号. 学位記番号: 新大院博(医)甲第709号. 学位授与年月日: 平成28年9月20日

ONCOLOGY REPORTS

Myofibroblasts of colorectal cancer (CRC) have been associated with histopathological factors such as lymph node… (more)

Subjects/Keywords: colorectal cancer; myofibroblast; muscularis propria; prognosis

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APA (6th Edition):

Takatsuna, M. (2016). Myofibroblasts of the muscle layer stimulate the malignant potential of colorectal cancer : 固有筋層における筋線維芽細胞は大腸癌の悪性度に関与する. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/45096

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takatsuna, Masafumi. “Myofibroblasts of the muscle layer stimulate the malignant potential of colorectal cancer : 固有筋層における筋線維芽細胞は大腸癌の悪性度に関与する.” 2016. Thesis, Niigata University / 新潟大学. Accessed September 28, 2020. http://hdl.handle.net/10191/45096.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takatsuna, Masafumi. “Myofibroblasts of the muscle layer stimulate the malignant potential of colorectal cancer : 固有筋層における筋線維芽細胞は大腸癌の悪性度に関与する.” 2016. Web. 28 Sep 2020.

Vancouver:

Takatsuna M. Myofibroblasts of the muscle layer stimulate the malignant potential of colorectal cancer : 固有筋層における筋線維芽細胞は大腸癌の悪性度に関与する. [Internet] [Thesis]. Niigata University / 新潟大学; 2016. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/10191/45096.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takatsuna M. Myofibroblasts of the muscle layer stimulate the malignant potential of colorectal cancer : 固有筋層における筋線維芽細胞は大腸癌の悪性度に関与する. [Thesis]. Niigata University / 新潟大学; 2016. Available from: http://hdl.handle.net/10191/45096

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Khan, Zareen A. The role of integrins in the activation of fibroblasts from skin, lung and breast tissue.

Degree: PhD, 2017, Queen Mary, University of London

 Fibroblasts are abundant mesenchymal cells present in all tissues in a quiescent state, which contribute to wound healing when activated. Cytokine transforming growth factor-β1 (TGF-β1)… (more)

Subjects/Keywords: Cancer research; Fibroblasts; myofibroblast activity; organ fibrosis

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APA (6th Edition):

Khan, Z. A. (2017). The role of integrins in the activation of fibroblasts from skin, lung and breast tissue. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/25902 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765971

Chicago Manual of Style (16th Edition):

Khan, Zareen A. “The role of integrins in the activation of fibroblasts from skin, lung and breast tissue.” 2017. Doctoral Dissertation, Queen Mary, University of London. Accessed September 28, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/25902 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765971.

MLA Handbook (7th Edition):

Khan, Zareen A. “The role of integrins in the activation of fibroblasts from skin, lung and breast tissue.” 2017. Web. 28 Sep 2020.

Vancouver:

Khan ZA. The role of integrins in the activation of fibroblasts from skin, lung and breast tissue. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2017. [cited 2020 Sep 28]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/25902 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765971.

Council of Science Editors:

Khan ZA. The role of integrins in the activation of fibroblasts from skin, lung and breast tissue. [Doctoral Dissertation]. Queen Mary, University of London; 2017. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/25902 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.765971


University of Gothenburg / Göteborgs Universitet

5. Hallberg, David. Myofibroblasts and polyploid cells in the conjunctival surface after allogeneic hematopoietic stem cell transplantation.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

 Allogeneic hematopoietic stem cell transplantation (allo-HSCT) is a potentially curative treatment modality for hematological malignancies, such as leukemia and lymphoma. However, a major complication of… (more)

Subjects/Keywords: Myofibroblast; Polyploid; Allogeneic; Graft-versus-Host Disease

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APA (6th Edition):

Hallberg, D. (2011). Myofibroblasts and polyploid cells in the conjunctival surface after allogeneic hematopoietic stem cell transplantation. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/24622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hallberg, David. “Myofibroblasts and polyploid cells in the conjunctival surface after allogeneic hematopoietic stem cell transplantation.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed September 28, 2020. http://hdl.handle.net/2077/24622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hallberg, David. “Myofibroblasts and polyploid cells in the conjunctival surface after allogeneic hematopoietic stem cell transplantation.” 2011. Web. 28 Sep 2020.

Vancouver:

Hallberg D. Myofibroblasts and polyploid cells in the conjunctival surface after allogeneic hematopoietic stem cell transplantation. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/2077/24622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hallberg D. Myofibroblasts and polyploid cells in the conjunctival surface after allogeneic hematopoietic stem cell transplantation. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/24622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

6. Rozycki, Matthew. Cytoskeletal Regulation of the Myofibroblast Phenotype: Central Effects on the Primary Cilium and Redox State.

Degree: PhD, 2015, University of Toronto

 The myofibroblast is a contractile cell which arises from progenitors to elicit wound healing. Dysregulation of these events are the basis of fibrosis, a feature… (more)

Subjects/Keywords: Cilium; Cytoskeleton; Fibrosis; Kidney; MRTF; Myofibroblast; 0307

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APA (6th Edition):

Rozycki, M. (2015). Cytoskeletal Regulation of the Myofibroblast Phenotype: Central Effects on the Primary Cilium and Redox State. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/71586

Chicago Manual of Style (16th Edition):

Rozycki, Matthew. “Cytoskeletal Regulation of the Myofibroblast Phenotype: Central Effects on the Primary Cilium and Redox State.” 2015. Doctoral Dissertation, University of Toronto. Accessed September 28, 2020. http://hdl.handle.net/1807/71586.

MLA Handbook (7th Edition):

Rozycki, Matthew. “Cytoskeletal Regulation of the Myofibroblast Phenotype: Central Effects on the Primary Cilium and Redox State.” 2015. Web. 28 Sep 2020.

Vancouver:

Rozycki M. Cytoskeletal Regulation of the Myofibroblast Phenotype: Central Effects on the Primary Cilium and Redox State. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1807/71586.

Council of Science Editors:

Rozycki M. Cytoskeletal Regulation of the Myofibroblast Phenotype: Central Effects on the Primary Cilium and Redox State. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71586


Kent State University

7. Shamhart, Patricia E. The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2010, Kent State University

 Cardiac fibroblasts (CFs) are the major non-contractile cells present in the myocardium and are the primary producers of the extracellular matrix (ECM) in the heart.… (more)

Subjects/Keywords: Biomedical Research; Cellular Biology; cardiac fibroblast; diabetes; angiotenin II; myofibroblast

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APA (6th Edition):

Shamhart, P. E. (2010). The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1283881511

Chicago Manual of Style (16th Edition):

Shamhart, Patricia E. “The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation.” 2010. Doctoral Dissertation, Kent State University. Accessed September 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent1283881511.

MLA Handbook (7th Edition):

Shamhart, Patricia E. “The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation.” 2010. Web. 28 Sep 2020.

Vancouver:

Shamhart PE. The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation. [Internet] [Doctoral dissertation]. Kent State University; 2010. [cited 2020 Sep 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1283881511.

Council of Science Editors:

Shamhart PE. The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation. [Doctoral Dissertation]. Kent State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1283881511


Vanderbilt University

8. Schroer, Alison Koelle. Mechanobiology of Cardiac Disease and Fibrosis: a Novel Role for Cadherin-11.

Degree: PhD, Biomedical Engineering, 2016, Vanderbilt University

 Heart disease is a leading cause of death worldwide, and many cardiac conditions that contribute to high morbidity and mortality are characterized by tissue fibrosis… (more)

Subjects/Keywords: mechanobiology; myofibroblast; cardiac fibrosis; cadherin-11; myocardial infarction

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APA (6th Edition):

Schroer, A. K. (2016). Mechanobiology of Cardiac Disease and Fibrosis: a Novel Role for Cadherin-11. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14583

Chicago Manual of Style (16th Edition):

Schroer, Alison Koelle. “Mechanobiology of Cardiac Disease and Fibrosis: a Novel Role for Cadherin-11.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 28, 2020. http://hdl.handle.net/1803/14583.

MLA Handbook (7th Edition):

Schroer, Alison Koelle. “Mechanobiology of Cardiac Disease and Fibrosis: a Novel Role for Cadherin-11.” 2016. Web. 28 Sep 2020.

Vancouver:

Schroer AK. Mechanobiology of Cardiac Disease and Fibrosis: a Novel Role for Cadherin-11. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1803/14583.

Council of Science Editors:

Schroer AK. Mechanobiology of Cardiac Disease and Fibrosis: a Novel Role for Cadherin-11. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/14583


Vanderbilt University

9. Bowler, Meghan Allardyce. Mechanobiology of cadherin-11 in calcific aortic valve disease.

Degree: PhD, Biomedical Engineering, 2018, Vanderbilt University

 Calcific aortic valve disease affects a quarter of those over 65 in the US alone. The only treatment is aortic valve replacement, but this intervention… (more)

Subjects/Keywords: calcification; myofibroblast; interstitial cell; heart valve; cadherin-11; mechanobiology

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APA (6th Edition):

Bowler, M. A. (2018). Mechanobiology of cadherin-11 in calcific aortic valve disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15400

Chicago Manual of Style (16th Edition):

Bowler, Meghan Allardyce. “Mechanobiology of cadherin-11 in calcific aortic valve disease.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed September 28, 2020. http://hdl.handle.net/1803/15400.

MLA Handbook (7th Edition):

Bowler, Meghan Allardyce. “Mechanobiology of cadherin-11 in calcific aortic valve disease.” 2018. Web. 28 Sep 2020.

Vancouver:

Bowler MA. Mechanobiology of cadherin-11 in calcific aortic valve disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1803/15400.

Council of Science Editors:

Bowler MA. Mechanobiology of cadherin-11 in calcific aortic valve disease. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/15400


University of Rochester

10. Epa, Amali Prasadini. The Role of Cellular Cross-Talk in Regulating Human Lung Fibroblast to Myofibroblast Differentiation.

Degree: PhD, 2016, University of Rochester

 Fibrosis refers to the process of excessive accumulation of scar tissue, and occurs in a variety of chronic diseases affecting organs as diverse as the… (more)

Subjects/Keywords: Fibrosis; Pulmonary fibrosis; Cellular cross-talk; Fibroblast; Myofibroblast differentiation

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APA (6th Edition):

Epa, A. P. (2016). The Role of Cellular Cross-Talk in Regulating Human Lung Fibroblast to Myofibroblast Differentiation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30497

Chicago Manual of Style (16th Edition):

Epa, Amali Prasadini. “The Role of Cellular Cross-Talk in Regulating Human Lung Fibroblast to Myofibroblast Differentiation.” 2016. Doctoral Dissertation, University of Rochester. Accessed September 28, 2020. http://hdl.handle.net/1802/30497.

MLA Handbook (7th Edition):

Epa, Amali Prasadini. “The Role of Cellular Cross-Talk in Regulating Human Lung Fibroblast to Myofibroblast Differentiation.” 2016. Web. 28 Sep 2020.

Vancouver:

Epa AP. The Role of Cellular Cross-Talk in Regulating Human Lung Fibroblast to Myofibroblast Differentiation. [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1802/30497.

Council of Science Editors:

Epa AP. The Role of Cellular Cross-Talk in Regulating Human Lung Fibroblast to Myofibroblast Differentiation. [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/30497


University of Texas Southwestern Medical Center

11. Russell, C. Hunter, III. The Use of Ultrasound-Targeted Microbubble Destruction in Mediated Delivery of a Gadolinium Magnetic Resonance Imaging Contrast Agent Into Fibroblast Cells In Vitro.

Degree: 2004, University of Texas Southwestern Medical Center

 A method for delivering Gd-based MRI contrast agents by ultrasound-targeted microbubble destruction into fibroblast cells in vitro is presented. First, MRI contrast agents Gd-F-DOTP-ME and… (more)

Subjects/Keywords: Microbubbles; Magnetic Resonance Imaging; Myofibroblast

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APA (6th Edition):

Russell, C. Hunter, I. (2004). The Use of Ultrasound-Targeted Microbubble Destruction in Mediated Delivery of a Gadolinium Magnetic Resonance Imaging Contrast Agent Into Fibroblast Cells In Vitro. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/716

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Russell, C. Hunter, III. “The Use of Ultrasound-Targeted Microbubble Destruction in Mediated Delivery of a Gadolinium Magnetic Resonance Imaging Contrast Agent Into Fibroblast Cells In Vitro.” 2004. Thesis, University of Texas Southwestern Medical Center. Accessed September 28, 2020. http://hdl.handle.net/2152.5/716.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Russell, C. Hunter, III. “The Use of Ultrasound-Targeted Microbubble Destruction in Mediated Delivery of a Gadolinium Magnetic Resonance Imaging Contrast Agent Into Fibroblast Cells In Vitro.” 2004. Web. 28 Sep 2020.

Vancouver:

Russell, C. Hunter I. The Use of Ultrasound-Targeted Microbubble Destruction in Mediated Delivery of a Gadolinium Magnetic Resonance Imaging Contrast Agent Into Fibroblast Cells In Vitro. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2004. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/2152.5/716.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Russell, C. Hunter I. The Use of Ultrasound-Targeted Microbubble Destruction in Mediated Delivery of a Gadolinium Magnetic Resonance Imaging Contrast Agent Into Fibroblast Cells In Vitro. [Thesis]. University of Texas Southwestern Medical Center; 2004. Available from: http://hdl.handle.net/2152.5/716

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

12. Rumora, Amy. Molecular Characterization Of Purβ: A Purine-Rich Single-Stranded Dna-Binding Repressor Of Myofibroblast Differentiation.

Degree: PhD, Biochemistry, 2014, University of Vermont

  The trans-differentiation of injury-activated fibroblasts to myofibroblasts is a process that provides contractile strength for wound closure. Persistent myofibroblast differentiation, however, is associated with… (more)

Subjects/Keywords: ACTA2; fibrosis; myofibroblast; Purβ,; smooth muscle alpha-actin; Biochemistry

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APA (6th Edition):

Rumora, A. (2014). Molecular Characterization Of Purβ: A Purine-Rich Single-Stranded Dna-Binding Repressor Of Myofibroblast Differentiation. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/277

Chicago Manual of Style (16th Edition):

Rumora, Amy. “Molecular Characterization Of Purβ: A Purine-Rich Single-Stranded Dna-Binding Repressor Of Myofibroblast Differentiation.” 2014. Doctoral Dissertation, University of Vermont. Accessed September 28, 2020. https://scholarworks.uvm.edu/graddis/277.

MLA Handbook (7th Edition):

Rumora, Amy. “Molecular Characterization Of Purβ: A Purine-Rich Single-Stranded Dna-Binding Repressor Of Myofibroblast Differentiation.” 2014. Web. 28 Sep 2020.

Vancouver:

Rumora A. Molecular Characterization Of Purβ: A Purine-Rich Single-Stranded Dna-Binding Repressor Of Myofibroblast Differentiation. [Internet] [Doctoral dissertation]. University of Vermont; 2014. [cited 2020 Sep 28]. Available from: https://scholarworks.uvm.edu/graddis/277.

Council of Science Editors:

Rumora A. Molecular Characterization Of Purβ: A Purine-Rich Single-Stranded Dna-Binding Repressor Of Myofibroblast Differentiation. [Doctoral Dissertation]. University of Vermont; 2014. Available from: https://scholarworks.uvm.edu/graddis/277


University of Toronto

13. Pakshir, Pardis. Dynamic Cross-Talk between Macrophages and Contractile Fibroblasts in Fibrosis.

Degree: PhD, 2019, University of Toronto

Accumulation and contraction of collagen extracellular matrix (ECM) by myofibroblasts (MFs) is a hallmark of fibrosis that can culminate in loss of organ function. In… (more)

Subjects/Keywords: Biomechanics; collagen; intracellular communication; macrophages; myofibroblast; wound repair; 0202

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APA (6th Edition):

Pakshir, P. (2019). Dynamic Cross-Talk between Macrophages and Contractile Fibroblasts in Fibrosis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/101751

Chicago Manual of Style (16th Edition):

Pakshir, Pardis. “Dynamic Cross-Talk between Macrophages and Contractile Fibroblasts in Fibrosis.” 2019. Doctoral Dissertation, University of Toronto. Accessed September 28, 2020. http://hdl.handle.net/1807/101751.

MLA Handbook (7th Edition):

Pakshir, Pardis. “Dynamic Cross-Talk between Macrophages and Contractile Fibroblasts in Fibrosis.” 2019. Web. 28 Sep 2020.

Vancouver:

Pakshir P. Dynamic Cross-Talk between Macrophages and Contractile Fibroblasts in Fibrosis. [Internet] [Doctoral dissertation]. University of Toronto; 2019. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1807/101751.

Council of Science Editors:

Pakshir P. Dynamic Cross-Talk between Macrophages and Contractile Fibroblasts in Fibrosis. [Doctoral Dissertation]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/101751


University of Manitoba

14. Jahan, Fahmida. Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition.

Degree: Biochemistry and Medical Genetics, 2014, University of Manitoba

 Cardiac fibroblast to myofibroblast phenoconversion is a critical step during the development of cardiac fibrosis. Myofibroblasts chronically remodel extracellular matrix that results in myocardial stiffening,… (more)

Subjects/Keywords: Zeb2; Meox2; Cardiac fibroblast; Myofibroblast; Phenoconversion; Cardiac fibrosis

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APA (6th Edition):

Jahan, F. (2014). Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31090

Chicago Manual of Style (16th Edition):

Jahan, Fahmida. “Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition.” 2014. Masters Thesis, University of Manitoba. Accessed September 28, 2020. http://hdl.handle.net/1993/31090.

MLA Handbook (7th Edition):

Jahan, Fahmida. “Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition.” 2014. Web. 28 Sep 2020.

Vancouver:

Jahan F. Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition. [Internet] [Masters thesis]. University of Manitoba; 2014. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1993/31090.

Council of Science Editors:

Jahan F. Zeb2: A novel regulator of cardiac fibroblast to myofibroblast transition. [Masters Thesis]. University of Manitoba; 2014. Available from: http://hdl.handle.net/1993/31090

15. Cunnington, Ryan H. Modulation of myofibroblast phenotype and function by c-Ski.

Degree: Physiology, 2011, University of Manitoba

 Cardiovascular disease is a leading cause of death and a major economic burden in the developed and developing world. Many heart diseases, including post-myocardial infarction,… (more)

Subjects/Keywords: myofibroblast; c-Ski

…secretion 58 6. Effects of c-Ski on myofibroblast phenotype 62 7. Effects of c-Ski on… …myofibroblast cell viability 62 8. c-Ski effects on Meox1 and Meox2 mRNA expression 69 9. Effects… …of Meox2 protein overexpression on myofibroblast phenotype 71 10. Mechanism for c-Ski… …overexpression reduces rat ventricular myofibroblast viability Figure 15. c-Ski overexpression induces… …Meox2 mRNA expression Figure 17. Meox2 overexpression causes diminution of myofibroblast… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cunnington, R. H. (2011). Modulation of myofibroblast phenotype and function by c-Ski. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4854

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cunnington, Ryan H. “Modulation of myofibroblast phenotype and function by c-Ski.” 2011. Thesis, University of Manitoba. Accessed September 28, 2020. http://hdl.handle.net/1993/4854.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cunnington, Ryan H. “Modulation of myofibroblast phenotype and function by c-Ski.” 2011. Web. 28 Sep 2020.

Vancouver:

Cunnington RH. Modulation of myofibroblast phenotype and function by c-Ski. [Internet] [Thesis]. University of Manitoba; 2011. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1993/4854.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cunnington RH. Modulation of myofibroblast phenotype and function by c-Ski. [Thesis]. University of Manitoba; 2011. Available from: http://hdl.handle.net/1993/4854

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

16. Walker, Matthew. Dynamic Mechanical Regulation of Cells in 3D Microtissues .

Degree: 2020, University of Ottawa

 It has been well established that the fundamental behaviors of mammalian cells are influenced by the physical cues that they experience from their surrounding environment.… (more)

Subjects/Keywords: Mechanobiology; Microtissue; Cytoskeleton; Myofibroblast; Cell mechanics; 3D cell culture

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APA (6th Edition):

Walker, M. (2020). Dynamic Mechanical Regulation of Cells in 3D Microtissues . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40555

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walker, Matthew. “Dynamic Mechanical Regulation of Cells in 3D Microtissues .” 2020. Thesis, University of Ottawa. Accessed September 28, 2020. http://hdl.handle.net/10393/40555.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walker, Matthew. “Dynamic Mechanical Regulation of Cells in 3D Microtissues .” 2020. Web. 28 Sep 2020.

Vancouver:

Walker M. Dynamic Mechanical Regulation of Cells in 3D Microtissues . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/10393/40555.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walker M. Dynamic Mechanical Regulation of Cells in 3D Microtissues . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40555

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

17. Cheung, Jane Wing Chi. A Highly Perfused Tissue-engineered Construct for Gingival Tissue Regeneration using Degradable/Polar/Hydrophobic/Ionic Polyurethanes and A Dynamic Co-culture System.

Degree: PhD, 2015, University of Toronto

 Gingival tissue engineering represents a promising method for regenerating the gingival lamina propria. In this thesis, a degradable polyurethane (degradable/polar/hydrophobic/ionic polyurethane (D-PHI)) scaffold was shown… (more)

Subjects/Keywords: Endothelial Cells; Fibroblasts; Gingival Tissues; Myofibroblast Differentiation; Polyurethane; Tissue Engineering; 0541

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APA (6th Edition):

Cheung, J. W. C. (2015). A Highly Perfused Tissue-engineered Construct for Gingival Tissue Regeneration using Degradable/Polar/Hydrophobic/Ionic Polyurethanes and A Dynamic Co-culture System. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69300

Chicago Manual of Style (16th Edition):

Cheung, Jane Wing Chi. “A Highly Perfused Tissue-engineered Construct for Gingival Tissue Regeneration using Degradable/Polar/Hydrophobic/Ionic Polyurethanes and A Dynamic Co-culture System.” 2015. Doctoral Dissertation, University of Toronto. Accessed September 28, 2020. http://hdl.handle.net/1807/69300.

MLA Handbook (7th Edition):

Cheung, Jane Wing Chi. “A Highly Perfused Tissue-engineered Construct for Gingival Tissue Regeneration using Degradable/Polar/Hydrophobic/Ionic Polyurethanes and A Dynamic Co-culture System.” 2015. Web. 28 Sep 2020.

Vancouver:

Cheung JWC. A Highly Perfused Tissue-engineered Construct for Gingival Tissue Regeneration using Degradable/Polar/Hydrophobic/Ionic Polyurethanes and A Dynamic Co-culture System. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1807/69300.

Council of Science Editors:

Cheung JWC. A Highly Perfused Tissue-engineered Construct for Gingival Tissue Regeneration using Degradable/Polar/Hydrophobic/Ionic Polyurethanes and A Dynamic Co-culture System. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69300

18. Quinlan, Angela. Mechanical Activation Of Valvular Interstitial Cell Phenotype.

Degree: PhD, 2012, Worcester Polytechnic Institute

 "During heart valve remodeling, and in many disease states, valvular interstitial cells (VICs) shift to an activated myofibroblast phenotype which is characterized by enhanced synthetic… (more)

Subjects/Keywords: stiffness; myofibroblast; VICs; stretch; transforming growth factor beta

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APA (6th Edition):

Quinlan, A. (2012). Mechanical Activation Of Valvular Interstitial Cell Phenotype. (Doctoral Dissertation). Worcester Polytechnic Institute. Retrieved from etd-082012-190459 ; https://digitalcommons.wpi.edu/etd-dissertations/355

Chicago Manual of Style (16th Edition):

Quinlan, Angela. “Mechanical Activation Of Valvular Interstitial Cell Phenotype.” 2012. Doctoral Dissertation, Worcester Polytechnic Institute. Accessed September 28, 2020. etd-082012-190459 ; https://digitalcommons.wpi.edu/etd-dissertations/355.

MLA Handbook (7th Edition):

Quinlan, Angela. “Mechanical Activation Of Valvular Interstitial Cell Phenotype.” 2012. Web. 28 Sep 2020.

Vancouver:

Quinlan A. Mechanical Activation Of Valvular Interstitial Cell Phenotype. [Internet] [Doctoral dissertation]. Worcester Polytechnic Institute; 2012. [cited 2020 Sep 28]. Available from: etd-082012-190459 ; https://digitalcommons.wpi.edu/etd-dissertations/355.

Council of Science Editors:

Quinlan A. Mechanical Activation Of Valvular Interstitial Cell Phenotype. [Doctoral Dissertation]. Worcester Polytechnic Institute; 2012. Available from: etd-082012-190459 ; https://digitalcommons.wpi.edu/etd-dissertations/355


Georgia State University

19. Zhang, Yinwei. Functions of Extracellular Pyruvate Kinase M2 in Tissue Repair and Regeneration.

Degree: PhD, Biology, 2016, Georgia State University

  Pyruvate kinase M2 (PKM2) is a glycolytic enzyme expressed in highly proliferating cells. Studies of PKM2 have been focused on its function of promoting… (more)

Subjects/Keywords: Pyruvate kinase M2; Wound healing; Angiogenesis; Myofibroblast differentiation; Liver fibrosis; Apoptosis

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APA (6th Edition):

Zhang, Y. (2016). Functions of Extracellular Pyruvate Kinase M2 in Tissue Repair and Regeneration. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/166

Chicago Manual of Style (16th Edition):

Zhang, Yinwei. “Functions of Extracellular Pyruvate Kinase M2 in Tissue Repair and Regeneration.” 2016. Doctoral Dissertation, Georgia State University. Accessed September 28, 2020. https://scholarworks.gsu.edu/biology_diss/166.

MLA Handbook (7th Edition):

Zhang, Yinwei. “Functions of Extracellular Pyruvate Kinase M2 in Tissue Repair and Regeneration.” 2016. Web. 28 Sep 2020.

Vancouver:

Zhang Y. Functions of Extracellular Pyruvate Kinase M2 in Tissue Repair and Regeneration. [Internet] [Doctoral dissertation]. Georgia State University; 2016. [cited 2020 Sep 28]. Available from: https://scholarworks.gsu.edu/biology_diss/166.

Council of Science Editors:

Zhang Y. Functions of Extracellular Pyruvate Kinase M2 in Tissue Repair and Regeneration. [Doctoral Dissertation]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/biology_diss/166

20. Δρυγιαννάκης, Ιωάννης. Ο ρόλος των μυοϊνοβλαστών στην ινωτική διεργασία στη νόσο του Crohn.

Degree: 2012, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Background and Aims. Colonic epithelial cells and adjacent subepithelial myofibroblasts are important counterparts in the pathophysiology of intestinal inflammation and fibrosis, respectively. We investigated the… (more)

Subjects/Keywords: Ενδοθηλίνη; Μεταλλοπρωτεάσες; Κολλαγόνο; Μυοϊνοβλάστες; Εντερικό επιθηλιακό κύτταρο; Εντερική ίνωση; Μετανάστευση μυοϊνοβλαστών; Αυξητικός παράγοντας μεταμόρφωσης TGF-B1; Endothelin; Matrix metalloproteinases; Collagen; Myofibroblast; Intestinal fibrosis; Myofibroblast migration; Transforming growth factor

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APA (6th Edition):

Δρυγιαννάκης, . . (2012). Ο ρόλος των μυοϊνοβλαστών στην ινωτική διεργασία στη νόσο του Crohn. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/28436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Δρυγιαννάκης, Ιωάννης. “Ο ρόλος των μυοϊνοβλαστών στην ινωτική διεργασία στη νόσο του Crohn.” 2012. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed September 28, 2020. http://hdl.handle.net/10442/hedi/28436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Δρυγιαννάκης, Ιωάννης. “Ο ρόλος των μυοϊνοβλαστών στην ινωτική διεργασία στη νόσο του Crohn.” 2012. Web. 28 Sep 2020.

Vancouver:

Δρυγιαννάκης . Ο ρόλος των μυοϊνοβλαστών στην ινωτική διεργασία στη νόσο του Crohn. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/10442/hedi/28436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Δρυγιαννάκης . Ο ρόλος των μυοϊνοβλαστών στην ινωτική διεργασία στη νόσο του Crohn. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. Available from: http://hdl.handle.net/10442/hedi/28436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

21. Olsen, Abby L. The Fibronectin EIIIA Splice Variant Promotes Hepatic Stellate Cell Motility and Liver Fibrosis via α9β1 Integrin.

Degree: 2011, University of Pennsylvania

 Cellular fibronectin that contains the alternatively spliced extra domain A (EIIIA+ cFN) is upregulated after liver injury and has been reported to promote myofibroblast differentiation… (more)

Subjects/Keywords: EIIIA fibronectin; hepatic stellate cells; liver fibrosis; alpha 9 integrin; myofibroblast; cell motility; Cell Biology

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APA (6th Edition):

Olsen, A. L. (2011). The Fibronectin EIIIA Splice Variant Promotes Hepatic Stellate Cell Motility and Liver Fibrosis via α9β1 Integrin. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Olsen, Abby L. “The Fibronectin EIIIA Splice Variant Promotes Hepatic Stellate Cell Motility and Liver Fibrosis via α9β1 Integrin.” 2011. Thesis, University of Pennsylvania. Accessed September 28, 2020. https://repository.upenn.edu/edissertations/997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Olsen, Abby L. “The Fibronectin EIIIA Splice Variant Promotes Hepatic Stellate Cell Motility and Liver Fibrosis via α9β1 Integrin.” 2011. Web. 28 Sep 2020.

Vancouver:

Olsen AL. The Fibronectin EIIIA Splice Variant Promotes Hepatic Stellate Cell Motility and Liver Fibrosis via α9β1 Integrin. [Internet] [Thesis]. University of Pennsylvania; 2011. [cited 2020 Sep 28]. Available from: https://repository.upenn.edu/edissertations/997.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Olsen AL. The Fibronectin EIIIA Splice Variant Promotes Hepatic Stellate Cell Motility and Liver Fibrosis via α9β1 Integrin. [Thesis]. University of Pennsylvania; 2011. Available from: https://repository.upenn.edu/edissertations/997

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. ZHU HONGYUAN. Cytoskeleton Regulators in the Control of Corneal Fibrosis.

Degree: 2011, National University of Singapore

Subjects/Keywords: fibrosis; moesin; cytoskeleton; cornea; myofibroblast; keratocytes

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APA (6th Edition):

HONGYUAN, Z. (2011). Cytoskeleton Regulators in the Control of Corneal Fibrosis. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/36108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

HONGYUAN, ZHU. “Cytoskeleton Regulators in the Control of Corneal Fibrosis.” 2011. Thesis, National University of Singapore. Accessed September 28, 2020. http://scholarbank.nus.edu.sg/handle/10635/36108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

HONGYUAN, ZHU. “Cytoskeleton Regulators in the Control of Corneal Fibrosis.” 2011. Web. 28 Sep 2020.

Vancouver:

HONGYUAN Z. Cytoskeleton Regulators in the Control of Corneal Fibrosis. [Internet] [Thesis]. National University of Singapore; 2011. [cited 2020 Sep 28]. Available from: http://scholarbank.nus.edu.sg/handle/10635/36108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

HONGYUAN Z. Cytoskeleton Regulators in the Control of Corneal Fibrosis. [Thesis]. National University of Singapore; 2011. Available from: http://scholarbank.nus.edu.sg/handle/10635/36108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. G.L. Perrucci. INVOLVEMENT OF THE 'TRACTION MODEL' AS TGF-BETA1 ACTIVATION MECHANISM IN THE DEVELOPMENT OF REACTIVE FIBROSIS IN SPONTANEOUSLY HYPERTENSIVE RATS CARDIAC FIBROBLASTS.

Degree: 2014, Università degli Studi di Milano

 Cardiac reactive fibrosis is a well-known pathological effect derived from arterial hypertension. TGF-β plays a key role in the progression from the inflammation state to… (more)

Subjects/Keywords: hypertension; cardiac fibrosis; myofibroblast; SHR; TGF-beta activation; integrin; Settore BIO/13 - Biologia Applicata

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APA (6th Edition):

Perrucci, G. (2014). INVOLVEMENT OF THE 'TRACTION MODEL' AS TGF-BETA1 ACTIVATION MECHANISM IN THE DEVELOPMENT OF REACTIVE FIBROSIS IN SPONTANEOUSLY HYPERTENSIVE RATS CARDIAC FIBROBLASTS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/233163

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perrucci, G.L.. “INVOLVEMENT OF THE 'TRACTION MODEL' AS TGF-BETA1 ACTIVATION MECHANISM IN THE DEVELOPMENT OF REACTIVE FIBROSIS IN SPONTANEOUSLY HYPERTENSIVE RATS CARDIAC FIBROBLASTS.” 2014. Thesis, Università degli Studi di Milano. Accessed September 28, 2020. http://hdl.handle.net/2434/233163.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perrucci, G.L.. “INVOLVEMENT OF THE 'TRACTION MODEL' AS TGF-BETA1 ACTIVATION MECHANISM IN THE DEVELOPMENT OF REACTIVE FIBROSIS IN SPONTANEOUSLY HYPERTENSIVE RATS CARDIAC FIBROBLASTS.” 2014. Web. 28 Sep 2020.

Vancouver:

Perrucci G. INVOLVEMENT OF THE 'TRACTION MODEL' AS TGF-BETA1 ACTIVATION MECHANISM IN THE DEVELOPMENT OF REACTIVE FIBROSIS IN SPONTANEOUSLY HYPERTENSIVE RATS CARDIAC FIBROBLASTS. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/2434/233163.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perrucci G. INVOLVEMENT OF THE 'TRACTION MODEL' AS TGF-BETA1 ACTIVATION MECHANISM IN THE DEVELOPMENT OF REACTIVE FIBROSIS IN SPONTANEOUSLY HYPERTENSIVE RATS CARDIAC FIBROBLASTS. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/233163

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

24. Ratti, James A. INVESTIGATING SMOKE EXPOSURE AND CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) WITH A CALIBRATED AGENT BASED MODEL (ABM) OF IN VITRO FIBROBLAST WOUND HEALING.

Degree: MS, Biomedical Engineering, 2018, Virginia Commonwealth University

  COPD is characterized by tissue inflammation and impaired remodeling that suggests fibroblast maintenance of structural homeostasis is dysregulated. Thus, we performed in vitro wound… (more)

Subjects/Keywords: cellular automata; contact inhibition; senescence; myofibroblast; oxidative stress; hypoxia; Computational Engineering; Disease Modeling; Systems Biology

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APA (6th Edition):

Ratti, J. A. (2018). INVESTIGATING SMOKE EXPOSURE AND CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) WITH A CALIBRATED AGENT BASED MODEL (ABM) OF IN VITRO FIBROBLAST WOUND HEALING. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/Z38X-QX71 ; https://scholarscompass.vcu.edu/etd/5441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ratti, James A. “INVESTIGATING SMOKE EXPOSURE AND CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) WITH A CALIBRATED AGENT BASED MODEL (ABM) OF IN VITRO FIBROBLAST WOUND HEALING.” 2018. Thesis, Virginia Commonwealth University. Accessed September 28, 2020. https://doi.org/10.25772/Z38X-QX71 ; https://scholarscompass.vcu.edu/etd/5441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ratti, James A. “INVESTIGATING SMOKE EXPOSURE AND CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) WITH A CALIBRATED AGENT BASED MODEL (ABM) OF IN VITRO FIBROBLAST WOUND HEALING.” 2018. Web. 28 Sep 2020.

Vancouver:

Ratti JA. INVESTIGATING SMOKE EXPOSURE AND CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) WITH A CALIBRATED AGENT BASED MODEL (ABM) OF IN VITRO FIBROBLAST WOUND HEALING. [Internet] [Thesis]. Virginia Commonwealth University; 2018. [cited 2020 Sep 28]. Available from: https://doi.org/10.25772/Z38X-QX71 ; https://scholarscompass.vcu.edu/etd/5441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ratti JA. INVESTIGATING SMOKE EXPOSURE AND CHRONIC OBSTRUCTIVE PULMONARY DISEASE (COPD) WITH A CALIBRATED AGENT BASED MODEL (ABM) OF IN VITRO FIBROBLAST WOUND HEALING. [Thesis]. Virginia Commonwealth University; 2018. Available from: https://doi.org/10.25772/Z38X-QX71 ; https://scholarscompass.vcu.edu/etd/5441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

25. Adhikari Bagchi, Rushita. Transcriptional regulation of cardiac extracellular matrix gene expression and fibroblast phenotype by scleraxis.

Degree: Physiology and Pathophysiology, 2015, University of Manitoba

 Cardiac fibrosis contributes to heart failure by dramatically impairing cardiac function, increasing patient morbidity and mortality. The primary fibrillar collagen expressed in the heart is… (more)

Subjects/Keywords: fibroblast; myofibroblast; transcription; gene regulation; transgenic mice; extracellular matrix; conditional knockout; fibrosis

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APA (6th Edition):

Adhikari Bagchi, R. (2015). Transcriptional regulation of cardiac extracellular matrix gene expression and fibroblast phenotype by scleraxis. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adhikari Bagchi, Rushita. “Transcriptional regulation of cardiac extracellular matrix gene expression and fibroblast phenotype by scleraxis.” 2015. Thesis, University of Manitoba. Accessed September 28, 2020. http://hdl.handle.net/1993/31243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adhikari Bagchi, Rushita. “Transcriptional regulation of cardiac extracellular matrix gene expression and fibroblast phenotype by scleraxis.” 2015. Web. 28 Sep 2020.

Vancouver:

Adhikari Bagchi R. Transcriptional regulation of cardiac extracellular matrix gene expression and fibroblast phenotype by scleraxis. [Internet] [Thesis]. University of Manitoba; 2015. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1993/31243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adhikari Bagchi R. Transcriptional regulation of cardiac extracellular matrix gene expression and fibroblast phenotype by scleraxis. [Thesis]. University of Manitoba; 2015. Available from: http://hdl.handle.net/1993/31243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Roche, Patricia. Scleraxis is a mechanoresponsive regulator of the cardiac myofibroblast phenotype.

Degree: Physiology and Pathophysiology, 2015, University of Manitoba

 Cardiac fibrosis is the excess deposition of myocardial extracellular matrix components, which increases tissue stiffness and heterogeneity, causing impaired diastolic/systolic function and arrhythmias, and eventually… (more)

Subjects/Keywords: Fibroblast; Myofibroblast; Cardiac fibrosis; Scleraxis

…72 Figure 6: Effect of cyclic stretch on expression of myofibroblast markers in cardiac… …80 Figure 10: Effect of Scleraxis over-expression on collagen and myofibroblast marker… …myofibroblast phenotype… …Although myofibroblast function is critical to the wound healing process, their persistence in… …myofibroblast phenotype, after proper wound healing has concluded, represents a novel approach to the… 

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APA (6th Edition):

Roche, P. (2015). Scleraxis is a mechanoresponsive regulator of the cardiac myofibroblast phenotype. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/30354

Chicago Manual of Style (16th Edition):

Roche, Patricia. “Scleraxis is a mechanoresponsive regulator of the cardiac myofibroblast phenotype.” 2015. Masters Thesis, University of Manitoba. Accessed September 28, 2020. http://hdl.handle.net/1993/30354.

MLA Handbook (7th Edition):

Roche, Patricia. “Scleraxis is a mechanoresponsive regulator of the cardiac myofibroblast phenotype.” 2015. Web. 28 Sep 2020.

Vancouver:

Roche P. Scleraxis is a mechanoresponsive regulator of the cardiac myofibroblast phenotype. [Internet] [Masters thesis]. University of Manitoba; 2015. [cited 2020 Sep 28]. Available from: http://hdl.handle.net/1993/30354.

Council of Science Editors:

Roche P. Scleraxis is a mechanoresponsive regulator of the cardiac myofibroblast phenotype. [Masters Thesis]. University of Manitoba; 2015. Available from: http://hdl.handle.net/1993/30354


Freie Universität Berlin

27. Weiß, Markus. Crimp frequency is strongly correlated to myofibroblast density in the human anterior cruciate ligament and its autologous tendon grafts.

Degree: 2012, Freie Universität Berlin

 Purpose: Collagen crimp is essential for maintaining viscoelastic properties of normal ligament and tendon tissue. The actin isoform α-smooth muscle actin (ASMA) has been identified… (more)

Subjects/Keywords: Tendon; ACL; crimp; myofibroblast; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Weiß, M. (2012). Crimp frequency is strongly correlated to myofibroblast density in the human anterior cruciate ligament and its autologous tendon grafts. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/11178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weiß, Markus. “Crimp frequency is strongly correlated to myofibroblast density in the human anterior cruciate ligament and its autologous tendon grafts.” 2012. Thesis, Freie Universität Berlin. Accessed September 28, 2020. https://refubium.fu-berlin.de/handle/fub188/11178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weiß, Markus. “Crimp frequency is strongly correlated to myofibroblast density in the human anterior cruciate ligament and its autologous tendon grafts.” 2012. Web. 28 Sep 2020.

Vancouver:

Weiß M. Crimp frequency is strongly correlated to myofibroblast density in the human anterior cruciate ligament and its autologous tendon grafts. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2020 Sep 28]. Available from: https://refubium.fu-berlin.de/handle/fub188/11178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weiß M. Crimp frequency is strongly correlated to myofibroblast density in the human anterior cruciate ligament and its autologous tendon grafts. [Thesis]. Freie Universität Berlin; 2012. Available from: https://refubium.fu-berlin.de/handle/fub188/11178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

28. Elliott, Christopher G. Evaluation of matricellular proteins as potential therapeutics for the treatment of human chronic skin wounds.

Degree: 2013, University of Western Ontario

 There is currently an unmet need for treatments to enhance healing of human chronic skin wounds. Previously, therapy development has focused on growth factors and… (more)

Subjects/Keywords: CCN2; Chronic Skin Wounds; Fibrosis; Matricellular Protein; Myofibroblast; Periostin; Skin and Connective Tissue Diseases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Elliott, C. G. (2013). Evaluation of matricellular proteins as potential therapeutics for the treatment of human chronic skin wounds. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elliott, Christopher G. “Evaluation of matricellular proteins as potential therapeutics for the treatment of human chronic skin wounds.” 2013. Thesis, University of Western Ontario. Accessed September 28, 2020. https://ir.lib.uwo.ca/etd/1501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elliott, Christopher G. “Evaluation of matricellular proteins as potential therapeutics for the treatment of human chronic skin wounds.” 2013. Web. 28 Sep 2020.

Vancouver:

Elliott CG. Evaluation of matricellular proteins as potential therapeutics for the treatment of human chronic skin wounds. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2020 Sep 28]. Available from: https://ir.lib.uwo.ca/etd/1501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elliott CG. Evaluation of matricellular proteins as potential therapeutics for the treatment of human chronic skin wounds. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

29. Crawford, Justin D. The Roles of Matrix-Associated Periostin in an In Vitro Model of Hypertrophic Scarring.

Degree: 2014, University of Western Ontario

 Abnormal scarring is a type of benign fibrosis of the skin that can restrict mobility, dexterity and quality of life. There are few, if any,… (more)

Subjects/Keywords: Hypertrophic scarring; periostin; fibroblasts; in vitro human model; proliferation; myofibroblast differentiation; Biochemistry

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APA (6th Edition):

Crawford, J. D. (2014). The Roles of Matrix-Associated Periostin in an In Vitro Model of Hypertrophic Scarring. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crawford, Justin D. “The Roles of Matrix-Associated Periostin in an In Vitro Model of Hypertrophic Scarring.” 2014. Thesis, University of Western Ontario. Accessed September 28, 2020. https://ir.lib.uwo.ca/etd/2228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crawford, Justin D. “The Roles of Matrix-Associated Periostin in an In Vitro Model of Hypertrophic Scarring.” 2014. Web. 28 Sep 2020.

Vancouver:

Crawford JD. The Roles of Matrix-Associated Periostin in an In Vitro Model of Hypertrophic Scarring. [Internet] [Thesis]. University of Western Ontario; 2014. [cited 2020 Sep 28]. Available from: https://ir.lib.uwo.ca/etd/2228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crawford JD. The Roles of Matrix-Associated Periostin in an In Vitro Model of Hypertrophic Scarring. [Thesis]. University of Western Ontario; 2014. Available from: https://ir.lib.uwo.ca/etd/2228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington University in St. Louis

30. Lee, Sheng-Lin. Cellular and Cytoskeletal Responses of Myofibroblasts in Three Dimensional Culture to Mechanical Stretch.

Degree: PhD, Mechanical Engineering and Materials Science, 2011, Washington University in St. Louis

 Myofibroblasts play important roles in wound healing and pathological organ remodeling, such as hypertensive cardiac fibrosis and promotion of metastasis. Differentiated myofibroblasts are characterized by… (more)

Subjects/Keywords: Biomedical Engineering; Mechanical Engineering; Biophysics; Cytoskeleton, Extracellular matrix, Focal adhesion, Myofibroblast, Stress fiber, Tissue engineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, S. (2011). Cellular and Cytoskeletal Responses of Myofibroblasts in Three Dimensional Culture to Mechanical Stretch. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/etd/201

Chicago Manual of Style (16th Edition):

Lee, Sheng-Lin. “Cellular and Cytoskeletal Responses of Myofibroblasts in Three Dimensional Culture to Mechanical Stretch.” 2011. Doctoral Dissertation, Washington University in St. Louis. Accessed September 28, 2020. https://openscholarship.wustl.edu/etd/201.

MLA Handbook (7th Edition):

Lee, Sheng-Lin. “Cellular and Cytoskeletal Responses of Myofibroblasts in Three Dimensional Culture to Mechanical Stretch.” 2011. Web. 28 Sep 2020.

Vancouver:

Lee S. Cellular and Cytoskeletal Responses of Myofibroblasts in Three Dimensional Culture to Mechanical Stretch. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2011. [cited 2020 Sep 28]. Available from: https://openscholarship.wustl.edu/etd/201.

Council of Science Editors:

Lee S. Cellular and Cytoskeletal Responses of Myofibroblasts in Three Dimensional Culture to Mechanical Stretch. [Doctoral Dissertation]. Washington University in St. Louis; 2011. Available from: https://openscholarship.wustl.edu/etd/201

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