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You searched for subject:( inflammasome). Showing records 1 – 30 of 258 total matches.

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1. Riteau, Nicolas. Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals.

Degree: Docteur es, Science du vivant. Immunologie, 2011, Université d'Orléans

La théorie du danger développée par Polly Matzinger stipule que l’attrait principal du système immunitaire ne réside pas dans la distinction entre le soi «… (more)

Subjects/Keywords: Inflammasome; Inflammasome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Riteau, N. (2011). Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2011ORLE2038

Chicago Manual of Style (16th Edition):

Riteau, Nicolas. “Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals.” 2011. Doctoral Dissertation, Université d'Orléans. Accessed April 15, 2021. http://www.theses.fr/2011ORLE2038.

MLA Handbook (7th Edition):

Riteau, Nicolas. “Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals.” 2011. Web. 15 Apr 2021.

Vancouver:

Riteau N. Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals. [Internet] [Doctoral dissertation]. Université d'Orléans; 2011. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2011ORLE2038.

Council of Science Editors:

Riteau N. Immunité innée et inflammasome : rôle des signaux de dangers endogènes : Innate immunity and inflammasome : role of endogenous danger signals. [Doctoral Dissertation]. Université d'Orléans; 2011. Available from: http://www.theses.fr/2011ORLE2038


Vanderbilt University

2. Stouch, Ashley Nicole. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.

Degree: PhD, Cell and Developmental Biology, 2014, Vanderbilt University

 Bronchopulmonary dysplasia is a life-threatening lung disease affecting low birth weight preterm infants. While the occurrence of BPD is correlated with chorioamnionitis, the origination and… (more)

Subjects/Keywords: macrophage; inflammasome; inflammation

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APA (6th Edition):

Stouch, A. N. (2014). NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15353

Chicago Manual of Style (16th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/15353.

MLA Handbook (7th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Web. 15 Apr 2021.

Vancouver:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/15353.

Council of Science Editors:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/15353

3. Molyvdas, Adam. Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή.

Degree: 2017, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

Background: Chronic viral hepatitis is a prevalent disease with major health implications. The mechanisms that are involved in the pathophysiology of chronic viral hepatitis are… (more)

Subjects/Keywords: Φλεγμονόσωμα; Ηπατίτιτδα; inflammasome; Hepatitis

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APA (6th Edition):

Molyvdas, A. (2017). Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/41928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Molyvdas, Adam. “Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή.” 2017. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed April 15, 2021. http://hdl.handle.net/10442/hedi/41928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Molyvdas, Adam. “Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή.” 2017. Web. 15 Apr 2021.

Vancouver:

Molyvdas A. Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/10442/hedi/41928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Molyvdas A. Το NLRP3 φλεγμονόσωμα στην χρόνια ιογενή ηπατική φλεγμονή. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2017. Available from: http://hdl.handle.net/10442/hedi/41928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

4. Williams, Evan James. Strategies for reducing systemic inflammation in obesity.

Degree: PhD, 2019, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

The prevalence of overweight and obese individuals in Australia is currently 63% of adults according to current data… (more)

Subjects/Keywords: obesity; systemic inflammation; inflammasome; macrophages

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APA (6th Edition):

Williams, E. J. (2019). Strategies for reducing systemic inflammation in obesity. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1401110

Chicago Manual of Style (16th Edition):

Williams, Evan James. “Strategies for reducing systemic inflammation in obesity.” 2019. Doctoral Dissertation, University of Newcastle. Accessed April 15, 2021. http://hdl.handle.net/1959.13/1401110.

MLA Handbook (7th Edition):

Williams, Evan James. “Strategies for reducing systemic inflammation in obesity.” 2019. Web. 15 Apr 2021.

Vancouver:

Williams EJ. Strategies for reducing systemic inflammation in obesity. [Internet] [Doctoral dissertation]. University of Newcastle; 2019. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1959.13/1401110.

Council of Science Editors:

Williams EJ. Strategies for reducing systemic inflammation in obesity. [Doctoral Dissertation]. University of Newcastle; 2019. Available from: http://hdl.handle.net/1959.13/1401110


University of Oxford

5. Nordlander, Sofia. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.

Degree: PhD, 2013, University of Oxford

 Flagellin is a highly immunogenic, bacterial protein considered to be abundant in the intestinal lumen. It has been reported to be an immunodominant antigen in… (more)

Subjects/Keywords: 616.07; Immunology; Mucosal immunology; inflammasome

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APA (6th Edition):

Nordlander, S. (2013). Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535

Chicago Manual of Style (16th Edition):

Nordlander, Sofia. “Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.” 2013. Doctoral Dissertation, University of Oxford. Accessed April 15, 2021. http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535.

MLA Handbook (7th Edition):

Nordlander, Sofia. “Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation.” 2013. Web. 15 Apr 2021.

Vancouver:

Nordlander S. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2021 Apr 15]. Available from: http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535.

Council of Science Editors:

Nordlander S. Innate sensing of bacterial flagellin in acute and chronic intestinal inflammation. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:775c4150-9b22-4008-9288-3a6053cbf4cc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.647535


University of Toronto

6. Stanojcic, Mile. The Role of NLPR3 Inflammasome in Response to Thermal Injury and Sepsis.

Degree: PhD, 2017, University of Toronto

 Although the induction of acute-phase inflammation after burn injury is essential, if prolonged it can induce increased risk of infection and sepsis. The NLRP3 inflammasome(more)

Subjects/Keywords: Burn; Inflammasome; Inflammation; Sepsis; 0982

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APA (6th Edition):

Stanojcic, M. (2017). The Role of NLPR3 Inflammasome in Response to Thermal Injury and Sepsis. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80947

Chicago Manual of Style (16th Edition):

Stanojcic, Mile. “The Role of NLPR3 Inflammasome in Response to Thermal Injury and Sepsis.” 2017. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/80947.

MLA Handbook (7th Edition):

Stanojcic, Mile. “The Role of NLPR3 Inflammasome in Response to Thermal Injury and Sepsis.” 2017. Web. 15 Apr 2021.

Vancouver:

Stanojcic M. The Role of NLPR3 Inflammasome in Response to Thermal Injury and Sepsis. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/80947.

Council of Science Editors:

Stanojcic M. The Role of NLPR3 Inflammasome in Response to Thermal Injury and Sepsis. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/80947


University of Melbourne

7. Chow, Melvyn Teck Chye. Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3.

Degree: 2013, University of Melbourne

 Danger receptors are important sentinels in innate immunity. Once danger signals appear in the microenvironment due to tissue damage or injury, danger signal receptors recognize… (more)

Subjects/Keywords: NLRP3 inflammasome; Toll-like receptor3

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APA (6th Edition):

Chow, M. T. C. (2013). Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38120

Chicago Manual of Style (16th Edition):

Chow, Melvyn Teck Chye. “Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3.” 2013. Doctoral Dissertation, University of Melbourne. Accessed April 15, 2021. http://hdl.handle.net/11343/38120.

MLA Handbook (7th Edition):

Chow, Melvyn Teck Chye. “Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3.” 2013. Web. 15 Apr 2021.

Vancouver:

Chow MTC. Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/11343/38120.

Council of Science Editors:

Chow MTC. Danger signal receptors and cancer: NLRP3 inflammasome and Toll-like receptor 3. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38120


University of Texas Medical Branch – Galveston

8. [No author]. The Activation of the Inflammasome during Rickettsial Infection .

Degree: University of Texas Medical Branch – Galveston

 Rickettsiae are Gram-negative, obligately intracellular bacteria that infect endothelial cells and macrophages. The molecular mechanisms involved in interaction of rickettsiae with macrophages, however, remain poorly… (more)

Subjects/Keywords: Rickettsia; Inflammasome

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APA (6th Edition):

author], [. (n.d.). The Activation of the Inflammasome during Rickettsial Infection . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11179

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “The Activation of the Inflammasome during Rickettsial Infection .” Thesis, University of Texas Medical Branch – Galveston. Accessed April 15, 2021. http://hdl.handle.net/2152.3/11179.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “The Activation of the Inflammasome during Rickettsial Infection .” Web. 15 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. The Activation of the Inflammasome during Rickettsial Infection . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2152.3/11179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. The Activation of the Inflammasome during Rickettsial Infection . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Cambridge

9. Digby, Zsofia. Caspase isoforms in inflammasome activation.

Degree: PhD, 2020, University of Cambridge

 Inflammasomes are macromolecular signalling platforms composed of a receptor (for example NLRP3 or NLRC4), an adaptor (ASC) and effectors (caspases) that are thought to play… (more)

Subjects/Keywords: immunology; innate immunity; inflammasome

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APA (6th Edition):

Digby, Z. (2020). Caspase isoforms in inflammasome activation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/307833

Chicago Manual of Style (16th Edition):

Digby, Zsofia. “Caspase isoforms in inflammasome activation.” 2020. Doctoral Dissertation, University of Cambridge. Accessed April 15, 2021. https://www.repository.cam.ac.uk/handle/1810/307833.

MLA Handbook (7th Edition):

Digby, Zsofia. “Caspase isoforms in inflammasome activation.” 2020. Web. 15 Apr 2021.

Vancouver:

Digby Z. Caspase isoforms in inflammasome activation. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2021 Apr 15]. Available from: https://www.repository.cam.ac.uk/handle/1810/307833.

Council of Science Editors:

Digby Z. Caspase isoforms in inflammasome activation. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/307833

10. Paoletti, Audrey. Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2015, Université Paris-Saclay (ComUE)

Plus de 34 millions de personnes dans le monde vivent avec le virus de l’immunodéficience humaine de type 1 (VIH-1). Cette pandémie est partiellement contrôlée… (more)

Subjects/Keywords: Vih-1; Récepteur purinergique; Inflammasome; Hiv-1; Purinergic receptor; Inflammasome

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APA (6th Edition):

Paoletti, A. (2015). Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2015SACLS240

Chicago Manual of Style (16th Edition):

Paoletti, Audrey. “Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3.” 2015. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 15, 2021. http://www.theses.fr/2015SACLS240.

MLA Handbook (7th Edition):

Paoletti, Audrey. “Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3.” 2015. Web. 15 Apr 2021.

Vancouver:

Paoletti A. Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2015. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2015SACLS240.

Council of Science Editors:

Paoletti A. Étude des étapes précoces de l’infection par le VIH-1 : identification d’un nouveau point de contrôle immunitaire immunitaire impliquant le récepteur P2Y2 et la protéine NLRP3 : Study Of The Early Steps Of HIV-1 Infection : Identification Of A New Immune Checkpoint Involving P2Y2 And NLRP3. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2015. Available from: http://www.theses.fr/2015SACLS240

11. Groslambert, Marine. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.

Degree: Docteur es, Sciences de la Vie, 2019, Lyon

L'inflammation est un processus déclenché suite à la détection de pathogènes et de dommages tissulaires. Elle conduit à la sécrétion de cytokines pro-inflammatoires par les… (more)

Subjects/Keywords: NLRP3; Inflammasome; Ubiquitination; Phosphorylation; NLRP3; Inflammasome; Ubiquitination; Phosphorylation

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APA (6th Edition):

Groslambert, M. (2019). Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSEN022

Chicago Manual of Style (16th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Doctoral Dissertation, Lyon. Accessed April 15, 2021. http://www.theses.fr/2019LYSEN022.

MLA Handbook (7th Edition):

Groslambert, Marine. “Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation.” 2019. Web. 15 Apr 2021.

Vancouver:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2019LYSEN022.

Council of Science Editors:

Groslambert M. Etude de l'impact fonctionnel des modifications post-traductionnelles dans l'activation de l'inflammasome NLRP3 : Study of the functional impact of post-translational modifications on NLRP3 inflammasome activation. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSEN022


Texas A&M University

12. Tong, Yanzheng. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 The NOD-like receptor protein NLRC5 inhibits TLR-induced NF-ҡB and viral infection-mediated type I interferon signaling in vitro. In the first project, we generated NLRC5 knockout… (more)

Subjects/Keywords: innate immunity; NLRC5; inflammasome; NF-kB signaling

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APA (6th Edition):

Tong, Y. (2014). Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/186991

Chicago Manual of Style (16th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Doctoral Dissertation, Texas A&M University. Accessed April 15, 2021. http://hdl.handle.net/1969.1/186991.

MLA Handbook (7th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Web. 15 Apr 2021.

Vancouver:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1969.1/186991.

Council of Science Editors:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/186991


McMaster University

13. Li, Yujin. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.

Degree: MSc, 2016, McMaster University

As a front-line treatment for cardiovascular disease, statins are among some of the most widely prescribed drugs worldwide. Statins are effective at lowering cholesterol, but… (more)

Subjects/Keywords: Inflammasome; Statins; Myopathy; Prenylation; Caspase-1; Atrogenes

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APA (6th Edition):

Li, Y. (2016). Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/20268

Chicago Manual of Style (16th Edition):

Li, Yujin. “Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.” 2016. Masters Thesis, McMaster University. Accessed April 15, 2021. http://hdl.handle.net/11375/20268.

MLA Handbook (7th Edition):

Li, Yujin. “Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy.” 2016. Web. 15 Apr 2021.

Vancouver:

Li Y. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. [Internet] [Masters thesis]. McMaster University; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/11375/20268.

Council of Science Editors:

Li Y. Investigating the Role of the NLRP3 Inflammasome in Statin-Induced Myopathy. [Masters Thesis]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/20268


Queens University

14. Wynick, Christopher. IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression .

Degree: Microbiology and Immunology, 2014, Queens University

 Inflammation plays an important role in responding to injury and combating infections. In this thesis, I examine how inflammation is regulated by cytokines responsible for… (more)

Subjects/Keywords: Inflammation ; IL-27 ; Monocytes ; Inflammasome ; IL-23

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APA (6th Edition):

Wynick, C. (2014). IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wynick, Christopher. “IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression .” 2014. Thesis, Queens University. Accessed April 15, 2021. http://hdl.handle.net/1974/12256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wynick, Christopher. “IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression .” 2014. Web. 15 Apr 2021.

Vancouver:

Wynick C. IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression . [Internet] [Thesis]. Queens University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1974/12256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wynick C. IL-27 Enhances LPS-Induced Proinflammatory Responses in Human Monocytes: Augmented Inflammasome Activity and IL-23 Expression . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/12256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

15. Maloy, Jeffrey Phillip. Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species.

Degree: Microbiology, Immunology, & Molecular Genetics, 2017, UCLA

 Burkholderia pseudomallei and Burkholderia thailandensis are related facultative intracellular pathogens that infect a variety of cell types, escape into the cytoplasm of host cells, and… (more)

Subjects/Keywords: Microbiology; Immunology; Burkholderia; Flagella; Inflammasome; Melioidosis; NLRC4

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APA (6th Edition):

Maloy, J. P. (2017). Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/11m9037f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maloy, Jeffrey Phillip. “Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species.” 2017. Thesis, UCLA. Accessed April 15, 2021. http://www.escholarship.org/uc/item/11m9037f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maloy, Jeffrey Phillip. “Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species.” 2017. Web. 15 Apr 2021.

Vancouver:

Maloy JP. Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species. [Internet] [Thesis]. UCLA; 2017. [cited 2021 Apr 15]. Available from: http://www.escholarship.org/uc/item/11m9037f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maloy JP. Characterization of an Intracellular Flagellar System in Pathogenic Burkholderia Species. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/11m9037f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

16. Hoyt, Laura Rose. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.

Degree: Medicine, 2015, University of Vermont

  Immunosuppression is a major complication of alcoholism and contributes to increased rates of opportunistic infections and sepsis associated with the addiction. The NLRP3 inflammasome(more)

Subjects/Keywords: Ethanol; Alcohol; NLRP3; Inflammasome; Inflammation; Immunosuppression

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hoyt, L. R. (2015). Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoyt, Laura Rose. “Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.” 2015. Thesis, University of Vermont. Accessed April 15, 2021. https://scholarworks.uvm.edu/hcoltheses/194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoyt, Laura Rose. “Inhibitory Effects of Ethanol on the NLRP3 Inflammasome.” 2015. Web. 15 Apr 2021.

Vancouver:

Hoyt LR. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. [Internet] [Thesis]. University of Vermont; 2015. [cited 2021 Apr 15]. Available from: https://scholarworks.uvm.edu/hcoltheses/194.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoyt LR. Inhibitory Effects of Ethanol on the NLRP3 Inflammasome. [Thesis]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/hcoltheses/194

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. 김, 은형. Expression and function of the NLRP3 inflammasome in Behçet’s disease.

Degree: 2016, Ajou University

연구배경: 베체트병운 점막 및피부, 눈, 혈관, 신경 및 기타 여러 장기를 침범할 수 있는 임상적 특징을 가지는 만성, 재발성의 전신질환이다. 현재까지 베체트병에서 보이는 지속적인 염증… (more)

Subjects/Keywords: Behçet’s disease; inflammasome; NLRP3; interleukin-1β

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APA (6th Edition):

김, . (2016). Expression and function of the NLRP3 inflammasome in Behçet’s disease. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 은형. “Expression and function of the NLRP3 inflammasome in Behçet’s disease.” 2016. Thesis, Ajou University. Accessed April 15, 2021. http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 은형. “Expression and function of the NLRP3 inflammasome in Behçet’s disease.” 2016. Web. 15 Apr 2021.

Vancouver:

김 . Expression and function of the NLRP3 inflammasome in Behçet’s disease. [Internet] [Thesis]. Ajou University; 2016. [cited 2021 Apr 15]. Available from: http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Expression and function of the NLRP3 inflammasome in Behçet’s disease. [Thesis]. Ajou University; 2016. Available from: http://repository.ajou.ac.kr/handle/201003/13061 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000021737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

18. Srinivasan, N. The role of inflammasomes in intestinal inflammation.

Degree: PhD, 2014, University of Oxford

 Single Nucleotide Polymorphisms (SNPs) in the intracellular pattern recognition receptor gene NLRP3 are associated with susceptibility to Crohn’s disease, a form of inflammatory bowel disease… (more)

Subjects/Keywords: 616.3; Immunology; Inflammasome; intestinal inflammation; mucosal immunity

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APA (6th Edition):

Srinivasan, N. (2014). The role of inflammasomes in intestinal inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000

Chicago Manual of Style (16th Edition):

Srinivasan, N. “The role of inflammasomes in intestinal inflammation.” 2014. Doctoral Dissertation, University of Oxford. Accessed April 15, 2021. http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000.

MLA Handbook (7th Edition):

Srinivasan, N. “The role of inflammasomes in intestinal inflammation.” 2014. Web. 15 Apr 2021.

Vancouver:

Srinivasan N. The role of inflammasomes in intestinal inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2021 Apr 15]. Available from: http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000.

Council of Science Editors:

Srinivasan N. The role of inflammasomes in intestinal inflammation. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:04ad577c-a8dd-46eb-811a-79a3980ff806 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596000

19. Bergin, Ronan. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.

Degree: 2015, RIAN

 The innate immune system is the first line of defence against invading pathogens and is responsible for both initial pathogen recognition and mounting an effective… (more)

Subjects/Keywords: Analysis; role; Pellino2; Activation; NLRP3 Inflammasome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bergin, R. (2015). Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. (Thesis). RIAN. Retrieved from http://eprints.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Thesis, RIAN. Accessed April 15, 2021. http://eprints.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Web. 15 Apr 2021.

Vancouver:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Internet] [Thesis]. RIAN; 2015. [cited 2021 Apr 15]. Available from: http://eprints.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Thesis]. RIAN; 2015. Available from: http://eprints.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

20. Neiman-Zenevich, Jana. Characterization of NLRP1B Inflammasome Activation.

Degree: PhD, 2017, University of Toronto

An inflammasome is a multi-protein complex that consists of a sensor protein, caspase-1, and often an adaptor protein ASC. An inflammasome assembles in response to… (more)

Subjects/Keywords: ATP depletion; Inflammasome; Listeria; NLRP1B; 0306

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APA (6th Edition):

Neiman-Zenevich, J. (2017). Characterization of NLRP1B Inflammasome Activation. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/98773

Chicago Manual of Style (16th Edition):

Neiman-Zenevich, Jana. “Characterization of NLRP1B Inflammasome Activation.” 2017. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/98773.

MLA Handbook (7th Edition):

Neiman-Zenevich, Jana. “Characterization of NLRP1B Inflammasome Activation.” 2017. Web. 15 Apr 2021.

Vancouver:

Neiman-Zenevich J. Characterization of NLRP1B Inflammasome Activation. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/98773.

Council of Science Editors:

Neiman-Zenevich J. Characterization of NLRP1B Inflammasome Activation. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/98773


University of Iowa

21. Clay, Gwendolyn Mary. The role of NLR proteins in Leishmaniasis.

Degree: PhD, Molecular and Cell Biology, 2016, University of Iowa

  Leishmania species are vector-borne protozoan parasites that cause a spectrum of human diseases, with an estimated 12 million people infected in 88 countries. Inflammation… (more)

Subjects/Keywords: Inflammasome; Leishmania; Leishmaniasis; NLR; Cell Biology

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APA (6th Edition):

Clay, G. M. (2016). The role of NLR proteins in Leishmaniasis. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5442

Chicago Manual of Style (16th Edition):

Clay, Gwendolyn Mary. “The role of NLR proteins in Leishmaniasis.” 2016. Doctoral Dissertation, University of Iowa. Accessed April 15, 2021. https://ir.uiowa.edu/etd/5442.

MLA Handbook (7th Edition):

Clay, Gwendolyn Mary. “The role of NLR proteins in Leishmaniasis.” 2016. Web. 15 Apr 2021.

Vancouver:

Clay GM. The role of NLR proteins in Leishmaniasis. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2021 Apr 15]. Available from: https://ir.uiowa.edu/etd/5442.

Council of Science Editors:

Clay GM. The role of NLR proteins in Leishmaniasis. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/5442


University of Melbourne

22. Moecking, Jonas. Investigating the molecular basis of human NLRP1 inflammasome activation.

Degree: 2019, University of Melbourne

 Cytosolic sensor proteins like NLRP1 (NOD-like receptor containing a pyrin domain 1) play a fundamental role in mediating innate immunity. Upon activation they form signalling… (more)

Subjects/Keywords: NLRP1; DPP9; Inflammasome; Inflammation; Innate Immunity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moecking, J. (2019). Investigating the molecular basis of human NLRP1 inflammasome activation. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/233878

Chicago Manual of Style (16th Edition):

Moecking, Jonas. “Investigating the molecular basis of human NLRP1 inflammasome activation.” 2019. Doctoral Dissertation, University of Melbourne. Accessed April 15, 2021. http://hdl.handle.net/11343/233878.

MLA Handbook (7th Edition):

Moecking, Jonas. “Investigating the molecular basis of human NLRP1 inflammasome activation.” 2019. Web. 15 Apr 2021.

Vancouver:

Moecking J. Investigating the molecular basis of human NLRP1 inflammasome activation. [Internet] [Doctoral dissertation]. University of Melbourne; 2019. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/11343/233878.

Council of Science Editors:

Moecking J. Investigating the molecular basis of human NLRP1 inflammasome activation. [Doctoral Dissertation]. University of Melbourne; 2019. Available from: http://hdl.handle.net/11343/233878

23. Bergin, Ronan. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.

Degree: 2015, RIAN

 The innate immune system is the first line of defence against invading pathogens and is responsible for both initial pathogen recognition and mounting an effective… (more)

Subjects/Keywords: Analysis; role; Pellino2; Activation; NLRP3 Inflammasome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bergin, R. (2015). Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. (Thesis). RIAN. Retrieved from http://mural.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Thesis, RIAN. Accessed April 15, 2021. http://mural.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergin, Ronan. “Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome.” 2015. Web. 15 Apr 2021.

Vancouver:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Internet] [Thesis]. RIAN; 2015. [cited 2021 Apr 15]. Available from: http://mural.maynoothuniversity.ie/10393/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergin R. Analysis of the role of Pellino2 in the Activation of the NLRP3 Inflammasome. [Thesis]. RIAN; 2015. Available from: http://mural.maynoothuniversity.ie/10393/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

24. Alegre Guerra, Fernando. Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz .

Degree: 2017, Universitat de Valencia

 RESUMEN INTRODUCCIÓN Diversos agentes hepatotóxicos y enfermedades hepáticas pueden conducir al desarrollo de fibrosis hepática debido a la presencia de respuestas inflamatorias desreguladas (Kubes and… (more)

Subjects/Keywords: liver; hiv; efavirenz; inflammation; inflammasome; fibrogenesis

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APA (6th Edition):

Alegre Guerra, F. (2017). Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/58371

Chicago Manual of Style (16th Edition):

Alegre Guerra, Fernando. “Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz .” 2017. Doctoral Dissertation, Universitat de Valencia. Accessed April 15, 2021. http://hdl.handle.net/10550/58371.

MLA Handbook (7th Edition):

Alegre Guerra, Fernando. “Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz .” 2017. Web. 15 Apr 2021.

Vancouver:

Alegre Guerra F. Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/10550/58371.

Council of Science Editors:

Alegre Guerra F. Acute inflammatory and fibrogenic responses induced in liver cells by efavirenz . [Doctoral Dissertation]. Universitat de Valencia; 2017. Available from: http://hdl.handle.net/10550/58371


University of Cambridge

25. Bittante, Alessandra. Molecular basis of NAIP/NLRC4 inflammasome activation by flagellin.

Degree: PhD, 2018, University of Cambridge

 The overall aim of this project was to determine the molecular mechanisms by which the flagellin gene from Salmonella enterica serovar Typhimurium (S. Typhimurium) activates… (more)

Subjects/Keywords: 636.089; Salmonella typhimurium; flagellin; inflammasome; innate immunity

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APA (6th Edition):

Bittante, A. (2018). Molecular basis of NAIP/NLRC4 inflammasome activation by flagellin. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.23007 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744836

Chicago Manual of Style (16th Edition):

Bittante, Alessandra. “Molecular basis of NAIP/NLRC4 inflammasome activation by flagellin.” 2018. Doctoral Dissertation, University of Cambridge. Accessed April 15, 2021. https://doi.org/10.17863/CAM.23007 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744836.

MLA Handbook (7th Edition):

Bittante, Alessandra. “Molecular basis of NAIP/NLRC4 inflammasome activation by flagellin.” 2018. Web. 15 Apr 2021.

Vancouver:

Bittante A. Molecular basis of NAIP/NLRC4 inflammasome activation by flagellin. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Apr 15]. Available from: https://doi.org/10.17863/CAM.23007 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744836.

Council of Science Editors:

Bittante A. Molecular basis of NAIP/NLRC4 inflammasome activation by flagellin. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.23007 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744836


University of Tasmania

26. Ranson, NM. Inflammasome activity in the inflammatory bowel diseases.

Degree: 2018, University of Tasmania

 Ulcerative colitis (UC) and Crohn’s disease (CD) are characterised by chronic and recurrent inflammation of the gastrointestinal tract. The development of Inflammatory Bowel Diseases (IBD)… (more)

Subjects/Keywords: inflammation; inflammasome complexes; ulcerative colitis; Crohn's disease

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APA (6th Edition):

Ranson, N. (2018). Inflammasome activity in the inflammatory bowel diseases. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ranson, NM. “Inflammasome activity in the inflammatory bowel diseases.” 2018. Thesis, University of Tasmania. Accessed April 15, 2021. https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ranson, NM. “Inflammasome activity in the inflammatory bowel diseases.” 2018. Web. 15 Apr 2021.

Vancouver:

Ranson N. Inflammasome activity in the inflammatory bowel diseases. [Internet] [Thesis]. University of Tasmania; 2018. [cited 2021 Apr 15]. Available from: https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ranson N. Inflammasome activity in the inflammatory bowel diseases. [Thesis]. University of Tasmania; 2018. Available from: https://eprints.utas.edu.au/29986/1/Ranson_whole_thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

27. Edye, Michelle. Interleukin-1 signalling in disease.

Degree: 2015, University of Manchester

 The pro-inflammatory cytokine interleukin 1 (IL-1) is involved in numerous physiological and pathological processes. It contributes to thermoregulation, sleep, feeding behaviour and notably to the… (more)

Subjects/Keywords: Interleukin-1; Inflammation; Acidosis; Inflammasome; Seizure

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APA (6th Edition):

Edye, M. (2015). Interleukin-1 signalling in disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336

Chicago Manual of Style (16th Edition):

Edye, Michelle. “Interleukin-1 signalling in disease.” 2015. Doctoral Dissertation, University of Manchester. Accessed April 15, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336.

MLA Handbook (7th Edition):

Edye, Michelle. “Interleukin-1 signalling in disease.” 2015. Web. 15 Apr 2021.

Vancouver:

Edye M. Interleukin-1 signalling in disease. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 Apr 15]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336.

Council of Science Editors:

Edye M. Interleukin-1 signalling in disease. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:293336


Georgia State University

28. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed April 15, 2021. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 15 Apr 2021.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2021 Apr 15]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

29. Lin, Jie. Einfluss von Colchicin auf experimentelle Coxsackievirus B3-induzierte Myokarditis.

Degree: 2019, Freie Universität Berlin

 Hintergrund Das nucleotide-binding oligomerization domain-like receptor pyrin domain-containing-3 (NLRP3) Inflammasom, aktiviert die proteolytische Spaltung von pro-Interleukin (IL)-1β in seine aktive Form IL-1β und ist ein… (more)

Subjects/Keywords: colchicine; coxsackievirus; myocarditis; inflammasome; ddc:610

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, J. (2019). Einfluss von Colchicin auf experimentelle Coxsackievirus B3-induzierte Myokarditis. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-27078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Jie. “Einfluss von Colchicin auf experimentelle Coxsackievirus B3-induzierte Myokarditis.” 2019. Thesis, Freie Universität Berlin. Accessed April 15, 2021. http://dx.doi.org/10.17169/refubium-27078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Jie. “Einfluss von Colchicin auf experimentelle Coxsackievirus B3-induzierte Myokarditis.” 2019. Web. 15 Apr 2021.

Vancouver:

Lin J. Einfluss von Colchicin auf experimentelle Coxsackievirus B3-induzierte Myokarditis. [Internet] [Thesis]. Freie Universität Berlin; 2019. [cited 2021 Apr 15]. Available from: http://dx.doi.org/10.17169/refubium-27078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin J. Einfluss von Colchicin auf experimentelle Coxsackievirus B3-induzierte Myokarditis. [Thesis]. Freie Universität Berlin; 2019. Available from: http://dx.doi.org/10.17169/refubium-27078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Benaoudia, Sacha. Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4.

Degree: Docteur es, Science de la vie, 2018, Lyon

Le lipopolysaccharide (LPS), un composant de la membrane des bactéries à Gram-négatif, active l’inflammasome non-canonique constitué chez l’homme des caspase-4/5 et chez la souris de… (more)

Subjects/Keywords: Caspase; Inflammasome; Mort cellulaire; LPS; Immunité innée; Caspase; Inflammasome; Cell death; LPS; Innate immunity; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Benaoudia, S. (2018). Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSE1219

Chicago Manual of Style (16th Edition):

Benaoudia, Sacha. “Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4.” 2018. Doctoral Dissertation, Lyon. Accessed April 15, 2021. http://www.theses.fr/2018LYSE1219.

MLA Handbook (7th Edition):

Benaoudia, Sacha. “Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4.” 2018. Web. 15 Apr 2021.

Vancouver:

Benaoudia S. Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2018LYSE1219.

Council of Science Editors:

Benaoudia S. Détection du LPS cytosolique : un crible CRISPR/Cas9 pangénomique identifie IRF2 comme régulateur de l’inflammasome non-canonique caspase-4 : Detection of cytosolic LPS : a genome-wide CRISPR/Cas9 screen identifies IRF2 as a regulator of the non-canonical inflammasome caspase-4. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSE1219

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