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You searched for subject:( induced contraction). Showing records 1 – 14 of 14 total matches.

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1. Hughes, William Edward. Dynamics of skeletal muscle blood flow and vasodilation with age.

Degree: PhD, Physical Rehabilitation Science, 2018, University of Iowa

  Aging is associated with attenuated blood flow and vasodilator responses during rhythmic exercise. Older adults also demonstrate attenuated blood flow and vasodilator responses following… (more)

Subjects/Keywords: Blood Flow; Contraction-Induced Rapid Vasodilation; Exercise; Skeletal Muscle; Vasodilation

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APA (6th Edition):

Hughes, W. E. (2018). Dynamics of skeletal muscle blood flow and vasodilation with age. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/6142

Chicago Manual of Style (16th Edition):

Hughes, William Edward. “Dynamics of skeletal muscle blood flow and vasodilation with age.” 2018. Doctoral Dissertation, University of Iowa. Accessed March 01, 2021. https://ir.uiowa.edu/etd/6142.

MLA Handbook (7th Edition):

Hughes, William Edward. “Dynamics of skeletal muscle blood flow and vasodilation with age.” 2018. Web. 01 Mar 2021.

Vancouver:

Hughes WE. Dynamics of skeletal muscle blood flow and vasodilation with age. [Internet] [Doctoral dissertation]. University of Iowa; 2018. [cited 2021 Mar 01]. Available from: https://ir.uiowa.edu/etd/6142.

Council of Science Editors:

Hughes WE. Dynamics of skeletal muscle blood flow and vasodilation with age. [Doctoral Dissertation]. University of Iowa; 2018. Available from: https://ir.uiowa.edu/etd/6142


Brigham Young University

2. Deyhle, Michael Roger. The Role of T Cells in Muscle Damage Protective Adaptation.

Degree: PhD, 2018, Brigham Young University

  Skeletal muscle is prone to damage from a range of stimuli. The muscle repair process that ensues is complex, involving several phases and requiring… (more)

Subjects/Keywords: inflammation; exercise-induced muscle damage; repeated bout effect; flow cytometry; T lymphocyte; lengthening contraction; eccentric contraction; Life Sciences

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APA (6th Edition):

Deyhle, M. R. (2018). The Role of T Cells in Muscle Damage Protective Adaptation. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=8455&context=etd

Chicago Manual of Style (16th Edition):

Deyhle, Michael Roger. “The Role of T Cells in Muscle Damage Protective Adaptation.” 2018. Doctoral Dissertation, Brigham Young University. Accessed March 01, 2021. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=8455&context=etd.

MLA Handbook (7th Edition):

Deyhle, Michael Roger. “The Role of T Cells in Muscle Damage Protective Adaptation.” 2018. Web. 01 Mar 2021.

Vancouver:

Deyhle MR. The Role of T Cells in Muscle Damage Protective Adaptation. [Internet] [Doctoral dissertation]. Brigham Young University; 2018. [cited 2021 Mar 01]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=8455&context=etd.

Council of Science Editors:

Deyhle MR. The Role of T Cells in Muscle Damage Protective Adaptation. [Doctoral Dissertation]. Brigham Young University; 2018. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=8455&context=etd


University of Michigan

3. Ng, Rainer N. The occurrence of contraction-induced lesions in the sarcolemma of skeletal muscles: Insights from a micro-sized whole muscle model.

Degree: PhD, Biomedical Engineering, 2008, University of Michigan

 Muscles exposed to unaccustomed exercise or injurious contractile activities are likely to sustain mechanical damage to muscle fibers, a characteristic of contraction-induced injuries. The susceptibility… (more)

Subjects/Keywords: Skeletal Muscles; Contraction-induced Injury; Biomedical Engineering; Engineering

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APA (6th Edition):

Ng, R. N. (2008). The occurrence of contraction-induced lesions in the sarcolemma of skeletal muscles: Insights from a micro-sized whole muscle model. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/60779

Chicago Manual of Style (16th Edition):

Ng, Rainer N. “The occurrence of contraction-induced lesions in the sarcolemma of skeletal muscles: Insights from a micro-sized whole muscle model.” 2008. Doctoral Dissertation, University of Michigan. Accessed March 01, 2021. http://hdl.handle.net/2027.42/60779.

MLA Handbook (7th Edition):

Ng, Rainer N. “The occurrence of contraction-induced lesions in the sarcolemma of skeletal muscles: Insights from a micro-sized whole muscle model.” 2008. Web. 01 Mar 2021.

Vancouver:

Ng RN. The occurrence of contraction-induced lesions in the sarcolemma of skeletal muscles: Insights from a micro-sized whole muscle model. [Internet] [Doctoral dissertation]. University of Michigan; 2008. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2027.42/60779.

Council of Science Editors:

Ng RN. The occurrence of contraction-induced lesions in the sarcolemma of skeletal muscles: Insights from a micro-sized whole muscle model. [Doctoral Dissertation]. University of Michigan; 2008. Available from: http://hdl.handle.net/2027.42/60779


University of Michigan

4. Hunter, Kam D. Contraction-induced injury: Interaction of initial length and work input.

Degree: PhD, Kinesiology, 1997, University of Michigan

 For single pliometric (lengthening) contractions of whole skeletal muscles initiated solely from optimal fiber length (Lf), the most important factor determining the subsequent deficit in… (more)

Subjects/Keywords: Contraction; Induced; Initial; Injury; Input; Interaction; Length; Work

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APA (6th Edition):

Hunter, K. D. (1997). Contraction-induced injury: Interaction of initial length and work input. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/130263

Chicago Manual of Style (16th Edition):

Hunter, Kam D. “Contraction-induced injury: Interaction of initial length and work input.” 1997. Doctoral Dissertation, University of Michigan. Accessed March 01, 2021. http://hdl.handle.net/2027.42/130263.

MLA Handbook (7th Edition):

Hunter, Kam D. “Contraction-induced injury: Interaction of initial length and work input.” 1997. Web. 01 Mar 2021.

Vancouver:

Hunter KD. Contraction-induced injury: Interaction of initial length and work input. [Internet] [Doctoral dissertation]. University of Michigan; 1997. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2027.42/130263.

Council of Science Editors:

Hunter KD. Contraction-induced injury: Interaction of initial length and work input. [Doctoral Dissertation]. University of Michigan; 1997. Available from: http://hdl.handle.net/2027.42/130263


University of Michigan

5. Rader, Erik Paul. Contraction -induced injury of weight -bearing muscles and the role of reactive oxygen species.

Degree: PhD, Biomedical engineering, 2004, University of Michigan

 For small, non-weight bearing muscles of old mice, severe contraction-induced injury causes a permanent 20% reduction in mass and maximum force. With aging, large, weight-bearing… (more)

Subjects/Keywords: Contraction-induced Injury; Reactive Oxygen Species; Role; Sarcomeres; Weight-bearing Muscles

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APA (6th Edition):

Rader, E. P. (2004). Contraction -induced injury of weight -bearing muscles and the role of reactive oxygen species. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/124538

Chicago Manual of Style (16th Edition):

Rader, Erik Paul. “Contraction -induced injury of weight -bearing muscles and the role of reactive oxygen species.” 2004. Doctoral Dissertation, University of Michigan. Accessed March 01, 2021. http://hdl.handle.net/2027.42/124538.

MLA Handbook (7th Edition):

Rader, Erik Paul. “Contraction -induced injury of weight -bearing muscles and the role of reactive oxygen species.” 2004. Web. 01 Mar 2021.

Vancouver:

Rader EP. Contraction -induced injury of weight -bearing muscles and the role of reactive oxygen species. [Internet] [Doctoral dissertation]. University of Michigan; 2004. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2027.42/124538.

Council of Science Editors:

Rader EP. Contraction -induced injury of weight -bearing muscles and the role of reactive oxygen species. [Doctoral Dissertation]. University of Michigan; 2004. Available from: http://hdl.handle.net/2027.42/124538


University of Michigan

6. Lockhart, Nicole C. Mechanisms underlying the protection from contraction -induced skeletal muscle injury provided by exercising with passive stretches.

Degree: PhD, Physical therapy, 2006, University of Michigan

Contraction-induced injury can occur during everyday activities when muscles are stretched while activated (lengthening contractions). Administration of passive stretches during which the muscle is lengthened… (more)

Subjects/Keywords: Contraction-induced; Exercising; Mechanisms; Muscle Injury; Passive Stretches; Protection; Provided; Skeletal Muscle; Underlying

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APA (6th Edition):

Lockhart, N. C. (2006). Mechanisms underlying the protection from contraction -induced skeletal muscle injury provided by exercising with passive stretches. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/125698

Chicago Manual of Style (16th Edition):

Lockhart, Nicole C. “Mechanisms underlying the protection from contraction -induced skeletal muscle injury provided by exercising with passive stretches.” 2006. Doctoral Dissertation, University of Michigan. Accessed March 01, 2021. http://hdl.handle.net/2027.42/125698.

MLA Handbook (7th Edition):

Lockhart, Nicole C. “Mechanisms underlying the protection from contraction -induced skeletal muscle injury provided by exercising with passive stretches.” 2006. Web. 01 Mar 2021.

Vancouver:

Lockhart NC. Mechanisms underlying the protection from contraction -induced skeletal muscle injury provided by exercising with passive stretches. [Internet] [Doctoral dissertation]. University of Michigan; 2006. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2027.42/125698.

Council of Science Editors:

Lockhart NC. Mechanisms underlying the protection from contraction -induced skeletal muscle injury provided by exercising with passive stretches. [Doctoral Dissertation]. University of Michigan; 2006. Available from: http://hdl.handle.net/2027.42/125698

7. 王, 志丹. Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する.

Degree: 修士(健康科学), 2017, Tokyo Metropolitan University / 首都大学東京

Caffeine is known to have inhibitory effects on Ca^<2+>-induced contraction in smooth muscle, and possible mechanisms may contribute the inhibitory effects. Recently, it was found… (more)

Subjects/Keywords: caffeine; Ca^<; 2+>; -induced contraction; myosin-actin interaction; myosin light chain phosphorylation; カフェイン; Ca^<; 2+>; 活性化収縮; ミオシン-アクチン相互作用; ミオシン軽鎖リン酸化

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APA (6th Edition):

王, . (2017). Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する. (Thesis). Tokyo Metropolitan University / 首都大学東京. Retrieved from http://hdl.handle.net/10748/00009635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

王, 志丹. “Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する.” 2017. Thesis, Tokyo Metropolitan University / 首都大学東京. Accessed March 01, 2021. http://hdl.handle.net/10748/00009635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

王, 志丹. “Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する.” 2017. Web. 01 Mar 2021.

Vancouver:

王 . Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する. [Internet] [Thesis]. Tokyo Metropolitan University / 首都大学東京; 2017. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10748/00009635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

王 . Caffeine inhibits smooth muscle actin-myosin interaction : カフェインは平滑筋ミオシンアクチン相互作用を抑制する. [Thesis]. Tokyo Metropolitan University / 首都大学東京; 2017. Available from: http://hdl.handle.net/10748/00009635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

8. Panchangam, Appaji. Sarcomere Dynamics During Contraction-Induced Injury to Permeabilized Single Muscle Fibers.

Degree: PhD, Biomedical Engineering, 2008, University of Michigan

 A contraction-induced injury results when an activated skeletal muscle or a single fiber is stretched in a ‘lengthening contraction’. This type of injury to muscle… (more)

Subjects/Keywords: Sarcomere Dynamics; Sarcomere Non-uniformity; Sarcomere Heterogeneity; Contraction-induced Injury; Laser Scanning; Sarcomere Length; Engineering; Health Sciences

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APA (6th Edition):

Panchangam, A. (2008). Sarcomere Dynamics During Contraction-Induced Injury to Permeabilized Single Muscle Fibers. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/58375

Chicago Manual of Style (16th Edition):

Panchangam, Appaji. “Sarcomere Dynamics During Contraction-Induced Injury to Permeabilized Single Muscle Fibers.” 2008. Doctoral Dissertation, University of Michigan. Accessed March 01, 2021. http://hdl.handle.net/2027.42/58375.

MLA Handbook (7th Edition):

Panchangam, Appaji. “Sarcomere Dynamics During Contraction-Induced Injury to Permeabilized Single Muscle Fibers.” 2008. Web. 01 Mar 2021.

Vancouver:

Panchangam A. Sarcomere Dynamics During Contraction-Induced Injury to Permeabilized Single Muscle Fibers. [Internet] [Doctoral dissertation]. University of Michigan; 2008. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2027.42/58375.

Council of Science Editors:

Panchangam A. Sarcomere Dynamics During Contraction-Induced Injury to Permeabilized Single Muscle Fibers. [Doctoral Dissertation]. University of Michigan; 2008. Available from: http://hdl.handle.net/2027.42/58375


University of Michigan

9. Consolino, Christina Marie. The role of the dystrophin -glycoprotein complex in the structure, function, and susceptibility to contraction -induced injury of limb muscles in dystrophic mice.

Degree: PhD, Kinesiology, 2003, University of Michigan

 The dystrophin glycoprotein complex (DGC) physically links cytoskeletal actin to the extracellular matrix (ECM) of skeletal muscle fibers, and mutations in the genes encoding many… (more)

Subjects/Keywords: Complex; Contraction-induced Injury; Dystrophic; Dystrophin-glycoprotein; Function; Limb Muscles; Mice; Muscular Dystrophy; Role; Structure; Susceptibility

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APA (6th Edition):

Consolino, C. M. (2003). The role of the dystrophin -glycoprotein complex in the structure, function, and susceptibility to contraction -induced injury of limb muscles in dystrophic mice. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/123367

Chicago Manual of Style (16th Edition):

Consolino, Christina Marie. “The role of the dystrophin -glycoprotein complex in the structure, function, and susceptibility to contraction -induced injury of limb muscles in dystrophic mice.” 2003. Doctoral Dissertation, University of Michigan. Accessed March 01, 2021. http://hdl.handle.net/2027.42/123367.

MLA Handbook (7th Edition):

Consolino, Christina Marie. “The role of the dystrophin -glycoprotein complex in the structure, function, and susceptibility to contraction -induced injury of limb muscles in dystrophic mice.” 2003. Web. 01 Mar 2021.

Vancouver:

Consolino CM. The role of the dystrophin -glycoprotein complex in the structure, function, and susceptibility to contraction -induced injury of limb muscles in dystrophic mice. [Internet] [Doctoral dissertation]. University of Michigan; 2003. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/2027.42/123367.

Council of Science Editors:

Consolino CM. The role of the dystrophin -glycoprotein complex in the structure, function, and susceptibility to contraction -induced injury of limb muscles in dystrophic mice. [Doctoral Dissertation]. University of Michigan; 2003. Available from: http://hdl.handle.net/2027.42/123367

10. PORKODI PANNEERSELVAM. MITOCHONDRIA LOCALIZED TLR ADAPTOR SARM IS PROAPOPTOTIC DURING T-CELL IMMUNE RESPONSE.

Degree: 2012, National University of Singapore

Subjects/Keywords: Influenza infection mouse model; proapoptotic SARM; T-cell contraction; neglect- and activation-induced T-cell death; NK/T cell lymphoma

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APA (6th Edition):

PANNEERSELVAM, P. (2012). MITOCHONDRIA LOCALIZED TLR ADAPTOR SARM IS PROAPOPTOTIC DURING T-CELL IMMUNE RESPONSE. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/35245

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

PANNEERSELVAM, PORKODI. “MITOCHONDRIA LOCALIZED TLR ADAPTOR SARM IS PROAPOPTOTIC DURING T-CELL IMMUNE RESPONSE.” 2012. Thesis, National University of Singapore. Accessed March 01, 2021. http://scholarbank.nus.edu.sg/handle/10635/35245.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

PANNEERSELVAM, PORKODI. “MITOCHONDRIA LOCALIZED TLR ADAPTOR SARM IS PROAPOPTOTIC DURING T-CELL IMMUNE RESPONSE.” 2012. Web. 01 Mar 2021.

Vancouver:

PANNEERSELVAM P. MITOCHONDRIA LOCALIZED TLR ADAPTOR SARM IS PROAPOPTOTIC DURING T-CELL IMMUNE RESPONSE. [Internet] [Thesis]. National University of Singapore; 2012. [cited 2021 Mar 01]. Available from: http://scholarbank.nus.edu.sg/handle/10635/35245.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

PANNEERSELVAM P. MITOCHONDRIA LOCALIZED TLR ADAPTOR SARM IS PROAPOPTOTIC DURING T-CELL IMMUNE RESPONSE. [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/35245

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Krishna, Abhilash. Multiphysics model of a cardiac myocyte: A voltage-clamp study.

Degree: PhD, Engineering, 2013, Rice University

 We develop a composite multiphysics model of excitation-contraction coupling for a rat ventricular myocyte under voltage clamp (VC) conditions to: (1) probe mechanisms underlying the… (more)

Subjects/Keywords: Multiphysics model; Excitation-contraction coupling; Rats; Ventricular myocyte; Voltage clamp; Electromechanics; FFR; Force-frequency response; CICR; Calcium-induced calcium-release; Myofilament contraction

…Secondary [Ca2+ ]myo Transients Induced by “Tail Currents” . 96 3.3.16 Cytosolic… …Contraction on the [Ca2+ ]myo Transient . . . . . . . 122 4.3.3 Regulation of Isometric… …Secondary [Ca2+ ]myo transients induced by “tail currents” . . . . . . 99 3.16… …contraction . . . . . . . . . . . . . . . . . . . . . . . . 115 4.2 Unloaded contraction… …117 4.3 Isometric contraction . . . . . . . . . . . . . . . . . . . . . . . . . . . 119… 

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APA (6th Edition):

Krishna, A. (2013). Multiphysics model of a cardiac myocyte: A voltage-clamp study. (Doctoral Dissertation). Rice University. Retrieved from http://hdl.handle.net/1911/71664

Chicago Manual of Style (16th Edition):

Krishna, Abhilash. “Multiphysics model of a cardiac myocyte: A voltage-clamp study.” 2013. Doctoral Dissertation, Rice University. Accessed March 01, 2021. http://hdl.handle.net/1911/71664.

MLA Handbook (7th Edition):

Krishna, Abhilash. “Multiphysics model of a cardiac myocyte: A voltage-clamp study.” 2013. Web. 01 Mar 2021.

Vancouver:

Krishna A. Multiphysics model of a cardiac myocyte: A voltage-clamp study. [Internet] [Doctoral dissertation]. Rice University; 2013. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1911/71664.

Council of Science Editors:

Krishna A. Multiphysics model of a cardiac myocyte: A voltage-clamp study. [Doctoral Dissertation]. Rice University; 2013. Available from: http://hdl.handle.net/1911/71664


Leiden University

12. Meer, van, B.J. Quantifying functional phenotypes in human pluripotent stem cell derived cardiomyocytes for disease modelling and drug discovery.

Degree: 2020, Leiden University

 Studying diseases or effects of new drugs on the human body is challenging, not least because of the lack of proper testing models that recapitulate… (more)

Subjects/Keywords: Human induced pluripotent stem cell (hiPSC); Human induced pluripotent stem cell-derived cardiomyocyte (hiPSC-CM); Cardiomyocytes; Cardiac contractility; Organ-on-Chip (OoC); Microphysiological systems (MPS); Drug testing; Disease modelling; Contraction-excitation coupling; Heart; Human induced pluripotent stem cell (hiPSC); Human induced pluripotent stem cell-derived cardiomyocyte (hiPSC-CM); Cardiomyocytes; Cardiac contractility; Organ-on-Chip (OoC); Microphysiological systems (MPS); Drug testing; Disease modelling; Contraction-excitation coupling; Heart

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APA (6th Edition):

Meer, van, B. J. (2020). Quantifying functional phenotypes in human pluripotent stem cell derived cardiomyocytes for disease modelling and drug discovery. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/138008

Chicago Manual of Style (16th Edition):

Meer, van, B J. “Quantifying functional phenotypes in human pluripotent stem cell derived cardiomyocytes for disease modelling and drug discovery.” 2020. Doctoral Dissertation, Leiden University. Accessed March 01, 2021. http://hdl.handle.net/1887/138008.

MLA Handbook (7th Edition):

Meer, van, B J. “Quantifying functional phenotypes in human pluripotent stem cell derived cardiomyocytes for disease modelling and drug discovery.” 2020. Web. 01 Mar 2021.

Vancouver:

Meer, van BJ. Quantifying functional phenotypes in human pluripotent stem cell derived cardiomyocytes for disease modelling and drug discovery. [Internet] [Doctoral dissertation]. Leiden University; 2020. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1887/138008.

Council of Science Editors:

Meer, van BJ. Quantifying functional phenotypes in human pluripotent stem cell derived cardiomyocytes for disease modelling and drug discovery. [Doctoral Dissertation]. Leiden University; 2020. Available from: http://hdl.handle.net/1887/138008


Colorado State University

13. Simpson, Carrie Beth. Acute ascorbic acid administration improves exercise hyperemia during rhythmic but not single contractions in aging humans.

Degree: MS(M.S.), Health and Exercise Science, 2009, Colorado State University

 Age-related increases in oxidative stress are known to impair endothelium dependent vasodilation in older healthy humans. As a result, many researchers have speculated that endothelial… (more)

Subjects/Keywords: acute AA administration; acute ascorbic acid administration; oxidative stress; endothelial dysfunction; contraction-induced rapid vasodilation; older adults; aging; exercise hyperaemia; Vitamin C  – Physiological effect; Older people  – Exercise  – Physiological aspects; Oxidative stress; Vascular endothelium; Blood-vessels  – Dilatation

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APA (6th Edition):

Simpson, C. B. (2009). Acute ascorbic acid administration improves exercise hyperemia during rhythmic but not single contractions in aging humans. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/23287

Chicago Manual of Style (16th Edition):

Simpson, Carrie Beth. “Acute ascorbic acid administration improves exercise hyperemia during rhythmic but not single contractions in aging humans.” 2009. Masters Thesis, Colorado State University. Accessed March 01, 2021. http://hdl.handle.net/10217/23287.

MLA Handbook (7th Edition):

Simpson, Carrie Beth. “Acute ascorbic acid administration improves exercise hyperemia during rhythmic but not single contractions in aging humans.” 2009. Web. 01 Mar 2021.

Vancouver:

Simpson CB. Acute ascorbic acid administration improves exercise hyperemia during rhythmic but not single contractions in aging humans. [Internet] [Masters thesis]. Colorado State University; 2009. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/10217/23287.

Council of Science Editors:

Simpson CB. Acute ascorbic acid administration improves exercise hyperemia during rhythmic but not single contractions in aging humans. [Masters Thesis]. Colorado State University; 2009. Available from: http://hdl.handle.net/10217/23287


Leiden University

14. Halaidych, O.V. Towards functional analysis of cerebrovascular cell types derived from human induced pluripotent stem cells.

Degree: 2019, Leiden University

 The work presented in this thesis provides novel contributions to advanced quantitative functional assessment of vascular endothelial and smooth muscle cells differentiated from hiPSCs. The automated assays… (more)

Subjects/Keywords: Human Induced Pluripotent Stem Cells; hiPSC-derived endothelial cells; hiPSC-derived smooth muscle cells; endothelial cell barrier function; impedance spectroscopy; junctional integrity; inflammatory responses; Ca2+ release; cell contraction; automated image analysis; Human Induced Pluripotent Stem Cells; hiPSC-derived endothelial cells; hiPSC-derived smooth muscle cells; endothelial cell barrier function; impedance spectroscopy; junctional integrity; inflammatory responses; Ca2+ release; cell contraction; automated image analysis

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APA (6th Edition):

Halaidych, O. V. (2019). Towards functional analysis of cerebrovascular cell types derived from human induced pluripotent stem cells. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/80758

Chicago Manual of Style (16th Edition):

Halaidych, O V. “Towards functional analysis of cerebrovascular cell types derived from human induced pluripotent stem cells.” 2019. Doctoral Dissertation, Leiden University. Accessed March 01, 2021. http://hdl.handle.net/1887/80758.

MLA Handbook (7th Edition):

Halaidych, O V. “Towards functional analysis of cerebrovascular cell types derived from human induced pluripotent stem cells.” 2019. Web. 01 Mar 2021.

Vancouver:

Halaidych OV. Towards functional analysis of cerebrovascular cell types derived from human induced pluripotent stem cells. [Internet] [Doctoral dissertation]. Leiden University; 2019. [cited 2021 Mar 01]. Available from: http://hdl.handle.net/1887/80758.

Council of Science Editors:

Halaidych OV. Towards functional analysis of cerebrovascular cell types derived from human induced pluripotent stem cells. [Doctoral Dissertation]. Leiden University; 2019. Available from: http://hdl.handle.net/1887/80758

.