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You searched for subject:( hepatocytenuclear factor 4 alpha). Showing records 1 – 30 of 19272 total matches.

[1] [2] [3] [4] [5] … [643]

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Kyoto University

1. Nakajima, Naoki. GATA6-positive lung adenocarcinomas are associated with invasive mucinous adenocarcinoma morphology, hepatocyte nuclear factor 4α expression, and KRAS mutations .

Degree: 2020, Kyoto University

Subjects/Keywords: Lung adenocarcinoma; GATA6; immunohistochemistry; hepatocytenuclear factor 4 alpha; invasive mucinous carcinoma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nakajima, N. (2020). GATA6-positive lung adenocarcinomas are associated with invasive mucinous adenocarcinoma morphology, hepatocyte nuclear factor 4α expression, and KRAS mutations . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/253168

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakajima, Naoki. “GATA6-positive lung adenocarcinomas are associated with invasive mucinous adenocarcinoma morphology, hepatocyte nuclear factor 4α expression, and KRAS mutations .” 2020. Thesis, Kyoto University. Accessed October 31, 2020. http://hdl.handle.net/2433/253168.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakajima, Naoki. “GATA6-positive lung adenocarcinomas are associated with invasive mucinous adenocarcinoma morphology, hepatocyte nuclear factor 4α expression, and KRAS mutations .” 2020. Web. 31 Oct 2020.

Vancouver:

Nakajima N. GATA6-positive lung adenocarcinomas are associated with invasive mucinous adenocarcinoma morphology, hepatocyte nuclear factor 4α expression, and KRAS mutations . [Internet] [Thesis]. Kyoto University; 2020. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/2433/253168.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakajima N. GATA6-positive lung adenocarcinomas are associated with invasive mucinous adenocarcinoma morphology, hepatocyte nuclear factor 4α expression, and KRAS mutations . [Thesis]. Kyoto University; 2020. Available from: http://hdl.handle.net/2433/253168

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Riverside

2. Vuong, Linh. The Role of the P1 and P2 Promoter-Driven HNF4a Isoforms in Cellular Proliferation and Differentiation in Human Colon Cancer and Mouse Embryonic Stem Cells.

Degree: Cell, Molecular and Developmental Biology, 2014, University of California – Riverside

 Cellular proliferation and differentiation are critical events in normal development and cancer. Despite decades of research, much remains to be learned about how cells transition… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Developmental biology; Cancer; Colon; Differentiation; Embryonic Stem Cells; Hepatocyte Nuclear Factor 4 alpha; Proliferation

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APA (6th Edition):

Vuong, L. (2014). The Role of the P1 and P2 Promoter-Driven HNF4a Isoforms in Cellular Proliferation and Differentiation in Human Colon Cancer and Mouse Embryonic Stem Cells. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/0gb1j7p3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vuong, Linh. “The Role of the P1 and P2 Promoter-Driven HNF4a Isoforms in Cellular Proliferation and Differentiation in Human Colon Cancer and Mouse Embryonic Stem Cells.” 2014. Thesis, University of California – Riverside. Accessed October 31, 2020. http://www.escholarship.org/uc/item/0gb1j7p3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vuong, Linh. “The Role of the P1 and P2 Promoter-Driven HNF4a Isoforms in Cellular Proliferation and Differentiation in Human Colon Cancer and Mouse Embryonic Stem Cells.” 2014. Web. 31 Oct 2020.

Vancouver:

Vuong L. The Role of the P1 and P2 Promoter-Driven HNF4a Isoforms in Cellular Proliferation and Differentiation in Human Colon Cancer and Mouse Embryonic Stem Cells. [Internet] [Thesis]. University of California – Riverside; 2014. [cited 2020 Oct 31]. Available from: http://www.escholarship.org/uc/item/0gb1j7p3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vuong L. The Role of the P1 and P2 Promoter-Driven HNF4a Isoforms in Cellular Proliferation and Differentiation in Human Colon Cancer and Mouse Embryonic Stem Cells. [Thesis]. University of California – Riverside; 2014. Available from: http://www.escholarship.org/uc/item/0gb1j7p3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Al, Salihi Mohammed Omar. Tumor necrosis factor-α antagonist suppresses local inflammatory reaction and facilitates olfactory nerve recovery following injury.

Degree: 博士(医学), 2017, Mie University / 三重大学

Objective: Olfactory dysfunction is a common finding in head trauma due to injury to the olfactory nerve. We previously reported that anti-inflammatory treatment with steroids… (more)

Subjects/Keywords: Cycloheximide; Etanercept; Field potential recording; Olfactory nerve; Regeneration; Tumor necrosis factor-alpha (TNF-α)

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APA (6th Edition):

Al, S. M. O. (2017). Tumor necrosis factor-α antagonist suppresses local inflammatory reaction and facilitates olfactory nerve recovery following injury. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00017106

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al, Salihi Mohammed Omar. “Tumor necrosis factor-α antagonist suppresses local inflammatory reaction and facilitates olfactory nerve recovery following injury.” 2017. Thesis, Mie University / 三重大学. Accessed October 31, 2020. http://hdl.handle.net/10076/00017106.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al, Salihi Mohammed Omar. “Tumor necrosis factor-α antagonist suppresses local inflammatory reaction and facilitates olfactory nerve recovery following injury.” 2017. Web. 31 Oct 2020.

Vancouver:

Al SMO. Tumor necrosis factor-α antagonist suppresses local inflammatory reaction and facilitates olfactory nerve recovery following injury. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10076/00017106.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al SMO. Tumor necrosis factor-α antagonist suppresses local inflammatory reaction and facilitates olfactory nerve recovery following injury. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00017106

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. TAVARES, Mayara Costa Mansur. Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn .

Degree: 2016, Universidade Federal de Pernambuco

 Doença inflamatória intestinal descreve um grupo heterogêneo de doenças inflamatórias crônicas do trato gastrointestinal. Os dois principais tipos de DII são retocolite ulcerativa idiopática e… (more)

Subjects/Keywords: 1. Inflamassoma; 2. Interleucina; 1. 3. Fator de Necrose Tumoral alfa; 4. Polimorfismo de Única Base; 5. Doença inflamatória intestinal; 1. Inflammasome; 2. Interleukin; 1. 3. Tumour Necrosis Factor alpha; 4. Single Nucleotide Polymorphism; 5. Inflammatory bowel disease

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APA (6th Edition):

TAVARES, M. C. M. (2016). Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn . (Thesis). Universidade Federal de Pernambuco. Retrieved from https://repositorio.ufpe.br/handle/123456789/18518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

TAVARES, Mayara Costa Mansur. “Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn .” 2016. Thesis, Universidade Federal de Pernambuco. Accessed October 31, 2020. https://repositorio.ufpe.br/handle/123456789/18518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

TAVARES, Mayara Costa Mansur. “Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn .” 2016. Web. 31 Oct 2020.

Vancouver:

TAVARES MCM. Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2016. [cited 2020 Oct 31]. Available from: https://repositorio.ufpe.br/handle/123456789/18518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

TAVARES MCM. Aspectos clínicos-epidemiológicos e análise de poliomorfirmos de genes relacionados à resposta imune em retocolite ulcerativa e doença de Crohn . [Thesis]. Universidade Federal de Pernambuco; 2016. Available from: https://repositorio.ufpe.br/handle/123456789/18518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Theologi, Vasiliki. Σύνδρομο αποφρακτικής άπνοιας στον ύπνο κατά την παιδική ηλικία: επιδράσεις στο κεντρικό νευρικό σύστημα.

Degree: 2016, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

BACKGROUND Obstructive sleep apnea (OSA) has been associated with increased frequency of excessive daytime sleepiness (EDS). Increased plasma TNF-α levels may mediate this association in… (more)

Subjects/Keywords: Άπνοια ύπνου; Ημερήσια υπνηλία; Παράγοντας νέκρωσης όγκων-α; Sleep apnea; Excessive daytime sleepiness; Tumor necrosis factor-alpha

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APA (6th Edition):

Theologi, V. (2016). Σύνδρομο αποφρακτικής άπνοιας στον ύπνο κατά την παιδική ηλικία: επιδράσεις στο κεντρικό νευρικό σύστημα. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/37690

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Theologi, Vasiliki. “Σύνδρομο αποφρακτικής άπνοιας στον ύπνο κατά την παιδική ηλικία: επιδράσεις στο κεντρικό νευρικό σύστημα.” 2016. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed October 31, 2020. http://hdl.handle.net/10442/hedi/37690.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Theologi, Vasiliki. “Σύνδρομο αποφρακτικής άπνοιας στον ύπνο κατά την παιδική ηλικία: επιδράσεις στο κεντρικό νευρικό σύστημα.” 2016. Web. 31 Oct 2020.

Vancouver:

Theologi V. Σύνδρομο αποφρακτικής άπνοιας στον ύπνο κατά την παιδική ηλικία: επιδράσεις στο κεντρικό νευρικό σύστημα. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2016. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10442/hedi/37690.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Theologi V. Σύνδρομο αποφρακτικής άπνοιας στον ύπνο κατά την παιδική ηλικία: επιδράσεις στο κεντρικό νευρικό σύστημα. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2016. Available from: http://hdl.handle.net/10442/hedi/37690

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Laurentian University

6. Edwardson, Derek William. Cancer cell behaviour in response to chemotherapeutics - a study of docetaxel induced inflammatory cytokine production and the effect of lipopolysaccharides .

Degree: 2018, Laurentian University

 The study of cancer is an ever‐evolving discipline and since the 1950's it has been driven by fundamental scientific research using cultured human tumour cell… (more)

Subjects/Keywords: cancer; chemotherapy; cytokines; taxanes; docetaxel; tumour necrosis factor (TNF‐α); interleukin‐8 (IL‐8; CXCL8); GRO‐α (MGSA; melanoma growth stimulatory activity; CXCL1); chemotherapy resistance; tumour progression; paclitaxel; doxorubicin; carboplatin; 5‐ fluorouracil (5‐FU); breast cancer; MCF‐7; MDA‐MB‐231; ovarian cancer; A2780; Lipopolysaccharides (LPS); Toll‐like receptor 4 (TLR4)

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APA (6th Edition):

Edwardson, D. W. (2018). Cancer cell behaviour in response to chemotherapeutics - a study of docetaxel induced inflammatory cytokine production and the effect of lipopolysaccharides . (Thesis). Laurentian University. Retrieved from https://zone.biblio.laurentian.ca/handle/10219/3166

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Edwardson, Derek William. “Cancer cell behaviour in response to chemotherapeutics - a study of docetaxel induced inflammatory cytokine production and the effect of lipopolysaccharides .” 2018. Thesis, Laurentian University. Accessed October 31, 2020. https://zone.biblio.laurentian.ca/handle/10219/3166.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Edwardson, Derek William. “Cancer cell behaviour in response to chemotherapeutics - a study of docetaxel induced inflammatory cytokine production and the effect of lipopolysaccharides .” 2018. Web. 31 Oct 2020.

Vancouver:

Edwardson DW. Cancer cell behaviour in response to chemotherapeutics - a study of docetaxel induced inflammatory cytokine production and the effect of lipopolysaccharides . [Internet] [Thesis]. Laurentian University; 2018. [cited 2020 Oct 31]. Available from: https://zone.biblio.laurentian.ca/handle/10219/3166.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Edwardson DW. Cancer cell behaviour in response to chemotherapeutics - a study of docetaxel induced inflammatory cytokine production and the effect of lipopolysaccharides . [Thesis]. Laurentian University; 2018. Available from: https://zone.biblio.laurentian.ca/handle/10219/3166

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

7. Leclerc, Simon. Étude du récepteur nucléaire FXR en contexte épithélial intestinal : activité et régulation de l'expression 3.

Degree: 2012, Université de Sherbrooke

 Le récepteur nucléaire FXR (NR1H4) possède quatre isoformes exprimés principalement dans le foie (FXR[alpha]1 et FXR[alpha]2) et dans l'intestin (FXR[alpha]3 et FXR[alpha]4). Le rôle de… (more)

Subjects/Keywords: Cdx2; FXR[alpha]3/[alpha]4; FXR[alpha]1/[alpha]2; Promoteur; Gènes cibles; Intestin; FXR

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APA (6th Edition):

Leclerc, S. (2012). Étude du récepteur nucléaire FXR en contexte épithélial intestinal : activité et régulation de l'expression 3. (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6333

Chicago Manual of Style (16th Edition):

Leclerc, Simon. “Étude du récepteur nucléaire FXR en contexte épithélial intestinal : activité et régulation de l'expression 3.” 2012. Masters Thesis, Université de Sherbrooke. Accessed October 31, 2020. http://hdl.handle.net/11143/6333.

MLA Handbook (7th Edition):

Leclerc, Simon. “Étude du récepteur nucléaire FXR en contexte épithélial intestinal : activité et régulation de l'expression 3.” 2012. Web. 31 Oct 2020.

Vancouver:

Leclerc S. Étude du récepteur nucléaire FXR en contexte épithélial intestinal : activité et régulation de l'expression 3. [Internet] [Masters thesis]. Université de Sherbrooke; 2012. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/11143/6333.

Council of Science Editors:

Leclerc S. Étude du récepteur nucléaire FXR en contexte épithélial intestinal : activité et régulation de l'expression 3. [Masters Thesis]. Université de Sherbrooke; 2012. Available from: http://hdl.handle.net/11143/6333


NSYSU

8. Lee, Pei-sang. A Multi-Factor Alpha Model Constructed Using Multi-lag-period Informationâ with Application in the Taiwan Market.

Degree: Master, Finance, 2013, NSYSU

 The main objective of this study is to generate values by combining the current and prior values of descriptors to improve the performance of a… (more)

Subjects/Keywords: Alpha model; Quantitative portfolio management; multi-factor

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APA (6th Edition):

Lee, P. (2013). A Multi-Factor Alpha Model Constructed Using Multi-lag-period Informationâ with Application in the Taiwan Market. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721113-014612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Pei-sang. “A Multi-Factor Alpha Model Constructed Using Multi-lag-period Informationâ with Application in the Taiwan Market.” 2013. Thesis, NSYSU. Accessed October 31, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721113-014612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Pei-sang. “A Multi-Factor Alpha Model Constructed Using Multi-lag-period Informationâ with Application in the Taiwan Market.” 2013. Web. 31 Oct 2020.

Vancouver:

Lee P. A Multi-Factor Alpha Model Constructed Using Multi-lag-period Informationâ with Application in the Taiwan Market. [Internet] [Thesis]. NSYSU; 2013. [cited 2020 Oct 31]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721113-014612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee P. A Multi-Factor Alpha Model Constructed Using Multi-lag-period Informationâ with Application in the Taiwan Market. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721113-014612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Wollongong

9. Liu, Zhenghao. Frequency-domain method for measuring alpha factor by self-mixing interferometry.

Degree: M. Phil., 2016, University of Wollongong

  Linewidth enhancement factor, also known as the alpha factor, is a fundamental characteristic parameter of a laser diode (LD). It characterises the broadening of… (more)

Subjects/Keywords: Semiconductor lasers; self-mixing interferometry; alpha factor

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APA (6th Edition):

Liu, Z. (2016). Frequency-domain method for measuring alpha factor by self-mixing interferometry. (Masters Thesis). University of Wollongong. Retrieved from 0205 OPTICAL PHYSICS, 0906 ELECTRICAL AND ELECTRONIC ENGINEERING ; https://ro.uow.edu.au/theses/4717

Chicago Manual of Style (16th Edition):

Liu, Zhenghao. “Frequency-domain method for measuring alpha factor by self-mixing interferometry.” 2016. Masters Thesis, University of Wollongong. Accessed October 31, 2020. 0205 OPTICAL PHYSICS, 0906 ELECTRICAL AND ELECTRONIC ENGINEERING ; https://ro.uow.edu.au/theses/4717.

MLA Handbook (7th Edition):

Liu, Zhenghao. “Frequency-domain method for measuring alpha factor by self-mixing interferometry.” 2016. Web. 31 Oct 2020.

Vancouver:

Liu Z. Frequency-domain method for measuring alpha factor by self-mixing interferometry. [Internet] [Masters thesis]. University of Wollongong; 2016. [cited 2020 Oct 31]. Available from: 0205 OPTICAL PHYSICS, 0906 ELECTRICAL AND ELECTRONIC ENGINEERING ; https://ro.uow.edu.au/theses/4717.

Council of Science Editors:

Liu Z. Frequency-domain method for measuring alpha factor by self-mixing interferometry. [Masters Thesis]. University of Wollongong; 2016. Available from: 0205 OPTICAL PHYSICS, 0906 ELECTRICAL AND ELECTRONIC ENGINEERING ; https://ro.uow.edu.au/theses/4717


Queen Mary, University of London

10. Bell, Iona Maree. The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease.

Degree: PhD, 2018, Queen Mary, University of London

 Tumour Necrosis Factor Alpha (TNFα) is central to the immunopathogenesis of inflammatory bowel disease. It is initially expressed on the cell surface as a trimer,… (more)

Subjects/Keywords: Inflammatory Bowel Disease; Tumour Necrosis Factor Alpha

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APA (6th Edition):

Bell, I. M. (2018). The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335

Chicago Manual of Style (16th Edition):

Bell, Iona Maree. “The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease.” 2018. Doctoral Dissertation, Queen Mary, University of London. Accessed October 31, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335.

MLA Handbook (7th Edition):

Bell, Iona Maree. “The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease.” 2018. Web. 31 Oct 2020.

Vancouver:

Bell IM. The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2018. [cited 2020 Oct 31]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335.

Council of Science Editors:

Bell IM. The role of membrane Tumour Necrosis Factor Alpha in the function and efficacy of anti-tumour necrosis factor antibodies in inflammatory bowel disease. [Doctoral Dissertation]. Queen Mary, University of London; 2018. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/43945 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786335

11. E. Lusito. A Network-based Approach to Breast Cancer Systems Medicine.

Degree: 2015, Università degli Studi di Milano

 Breast cancer is the most commonly diagnosed cancer and the second leading cause of cancer death in women. Although recent improvements in the prevention, early… (more)

Subjects/Keywords: Cancer Modules, Cancer Modules-genes, Gene Regulatory Networks, Transcriptional Master Regulator, Triple Negative Breast Cancer, Residual Disease, pathologic Complete Response, neoadjuvant chemotherapy, Transforming growth factor beta 1 induced transcript 1 (TGFB1I1), Transcription factor 4 (TCF4), Zinc finger protein, FOG family member 2 (ZFPM2), Paired related homeobox 1 (PRRX1), E74-like factor 4 (ELF4), Collagen, type I, alpha 1 (COL1A1); Settore MED/04 - Patologia Generale

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APA (6th Edition):

Lusito, E. (2015). A Network-based Approach to Breast Cancer Systems Medicine. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/265572

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lusito, E.. “A Network-based Approach to Breast Cancer Systems Medicine.” 2015. Thesis, Università degli Studi di Milano. Accessed October 31, 2020. http://hdl.handle.net/2434/265572.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lusito, E.. “A Network-based Approach to Breast Cancer Systems Medicine.” 2015. Web. 31 Oct 2020.

Vancouver:

Lusito E. A Network-based Approach to Breast Cancer Systems Medicine. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/2434/265572.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lusito E. A Network-based Approach to Breast Cancer Systems Medicine. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/265572

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. 大高, 行博; オオタカ, ヒロユキ. STXBP4 drives tumor growth and is associated with poor prognosis through PDGF Receptor signaling in lung squamous cell carcinoma : STXBP4は肺扁平上皮癌においてPDGF受容体シグナルを介して腫瘍増殖をもたらし予後不良と 関連する.

Degree: 博士(医学), 2017, Gunma University / 群馬大学

学位記番号:医博甲1634

Subjects/Keywords: syntaxin-binding protein 4; platelet-derived growth factor receptor alpha; lung squamous cell carcinoma; p63

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APA (6th Edition):

大高, 行博; オオタカ, . (2017). STXBP4 drives tumor growth and is associated with poor prognosis through PDGF Receptor signaling in lung squamous cell carcinoma : STXBP4は肺扁平上皮癌においてPDGF受容体シグナルを介して腫瘍増殖をもたらし予後不良と 関連する. (Thesis). Gunma University / 群馬大学. Retrieved from http://hdl.handle.net/10087/11441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

大高, 行博; オオタカ, ヒロユキ. “STXBP4 drives tumor growth and is associated with poor prognosis through PDGF Receptor signaling in lung squamous cell carcinoma : STXBP4は肺扁平上皮癌においてPDGF受容体シグナルを介して腫瘍増殖をもたらし予後不良と 関連する.” 2017. Thesis, Gunma University / 群馬大学. Accessed October 31, 2020. http://hdl.handle.net/10087/11441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

大高, 行博; オオタカ, ヒロユキ. “STXBP4 drives tumor growth and is associated with poor prognosis through PDGF Receptor signaling in lung squamous cell carcinoma : STXBP4は肺扁平上皮癌においてPDGF受容体シグナルを介して腫瘍増殖をもたらし予後不良と 関連する.” 2017. Web. 31 Oct 2020.

Vancouver:

大高, 行博; オオタカ . STXBP4 drives tumor growth and is associated with poor prognosis through PDGF Receptor signaling in lung squamous cell carcinoma : STXBP4は肺扁平上皮癌においてPDGF受容体シグナルを介して腫瘍増殖をもたらし予後不良と 関連する. [Internet] [Thesis]. Gunma University / 群馬大学; 2017. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10087/11441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

大高, 行博; オオタカ . STXBP4 drives tumor growth and is associated with poor prognosis through PDGF Receptor signaling in lung squamous cell carcinoma : STXBP4は肺扁平上皮癌においてPDGF受容体シグナルを介して腫瘍増殖をもたらし予後不良と 関連する. [Thesis]. Gunma University / 群馬大学; 2017. Available from: http://hdl.handle.net/10087/11441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vrije Universiteit Amsterdam

13. Silva Aresta Belo van Wijk, A.I. da. The Sweet and Sorrow of Pancreatic cancer: Galectin-4 and glycosylation in pancreatic cancer .

Degree: 2015, Vrije Universiteit Amsterdam

Subjects/Keywords: Pancreatic ductal adenocarcinoma; Galectin-4; glycosylation; glycosyltransferases; colorectal cancer; lectin binding proteins; Hypoxia Inducible Factor 1 alpha; fucosyltransferase

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APA (6th Edition):

Silva Aresta Belo van Wijk, A. I. d. (2015). The Sweet and Sorrow of Pancreatic cancer: Galectin-4 and glycosylation in pancreatic cancer . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/54686

Chicago Manual of Style (16th Edition):

Silva Aresta Belo van Wijk, A I da. “The Sweet and Sorrow of Pancreatic cancer: Galectin-4 and glycosylation in pancreatic cancer .” 2015. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed October 31, 2020. http://hdl.handle.net/1871/54686.

MLA Handbook (7th Edition):

Silva Aresta Belo van Wijk, A I da. “The Sweet and Sorrow of Pancreatic cancer: Galectin-4 and glycosylation in pancreatic cancer .” 2015. Web. 31 Oct 2020.

Vancouver:

Silva Aresta Belo van Wijk AId. The Sweet and Sorrow of Pancreatic cancer: Galectin-4 and glycosylation in pancreatic cancer . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2015. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/1871/54686.

Council of Science Editors:

Silva Aresta Belo van Wijk AId. The Sweet and Sorrow of Pancreatic cancer: Galectin-4 and glycosylation in pancreatic cancer . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2015. Available from: http://hdl.handle.net/1871/54686

14. Jansson, Camilla. Mekanismer för inaktivering av lipoproteinlipas i 3T3-L1 celler : En studie av hur aktiviteten regleras med Angiopoietin-likt protein 4, Actinomycin D och Tumor Necrosis Factor alpha.

Degree: Biomedical Laboratory Science, 2012, Umeå University

Subjects/Keywords: Angiopoietin-likt protein 4; Actinomycin D; Tumor Necrosis Factor alpha; Lipoproteinlipas-aktivitet; 3T3-L1 adipocyter

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APA (6th Edition):

Jansson, C. (2012). Mekanismer för inaktivering av lipoproteinlipas i 3T3-L1 celler : En studie av hur aktiviteten regleras med Angiopoietin-likt protein 4, Actinomycin D och Tumor Necrosis Factor alpha. (Thesis). Umeå University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-57972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jansson, Camilla. “Mekanismer för inaktivering av lipoproteinlipas i 3T3-L1 celler : En studie av hur aktiviteten regleras med Angiopoietin-likt protein 4, Actinomycin D och Tumor Necrosis Factor alpha.” 2012. Thesis, Umeå University. Accessed October 31, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-57972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jansson, Camilla. “Mekanismer för inaktivering av lipoproteinlipas i 3T3-L1 celler : En studie av hur aktiviteten regleras med Angiopoietin-likt protein 4, Actinomycin D och Tumor Necrosis Factor alpha.” 2012. Web. 31 Oct 2020.

Vancouver:

Jansson C. Mekanismer för inaktivering av lipoproteinlipas i 3T3-L1 celler : En studie av hur aktiviteten regleras med Angiopoietin-likt protein 4, Actinomycin D och Tumor Necrosis Factor alpha. [Internet] [Thesis]. Umeå University; 2012. [cited 2020 Oct 31]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-57972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jansson C. Mekanismer för inaktivering av lipoproteinlipas i 3T3-L1 celler : En studie av hur aktiviteten regleras med Angiopoietin-likt protein 4, Actinomycin D och Tumor Necrosis Factor alpha. [Thesis]. Umeå University; 2012. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-57972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Dalhousie University

15. McDonald, William. Characterization of the Interaction of Alpha4 Phosphoprotein with Novel Binding Partners: EDD E3 Ubiquitin Ligase and Poly(A)-Binding Protein.

Degree: MS, Department of Biochemistry & Molecular Biology, 2011, Dalhousie University

 ?4 phosphoprotein (also known as IGBP1) is a component of the mammalian target-of-rapamycin (mTOR) pathway that controls the initiation of translation and cell-cycle progression in… (more)

Subjects/Keywords: Alpha 4 phosphoprotein; EDD E3 ubiquitin ligase; PABP

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APA (6th Edition):

McDonald, W. (2011). Characterization of the Interaction of Alpha4 Phosphoprotein with Novel Binding Partners: EDD E3 Ubiquitin Ligase and Poly(A)-Binding Protein. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/13336

Chicago Manual of Style (16th Edition):

McDonald, William. “Characterization of the Interaction of Alpha4 Phosphoprotein with Novel Binding Partners: EDD E3 Ubiquitin Ligase and Poly(A)-Binding Protein.” 2011. Masters Thesis, Dalhousie University. Accessed October 31, 2020. http://hdl.handle.net/10222/13336.

MLA Handbook (7th Edition):

McDonald, William. “Characterization of the Interaction of Alpha4 Phosphoprotein with Novel Binding Partners: EDD E3 Ubiquitin Ligase and Poly(A)-Binding Protein.” 2011. Web. 31 Oct 2020.

Vancouver:

McDonald W. Characterization of the Interaction of Alpha4 Phosphoprotein with Novel Binding Partners: EDD E3 Ubiquitin Ligase and Poly(A)-Binding Protein. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10222/13336.

Council of Science Editors:

McDonald W. Characterization of the Interaction of Alpha4 Phosphoprotein with Novel Binding Partners: EDD E3 Ubiquitin Ligase and Poly(A)-Binding Protein. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/13336


Colorado State University

16. Warro, Daniel. Effects of a single bout of exercise, with energy replacement, on plasma concentrations of TNF-α following consumption of a high-fat, high-carbohydrate mixed meal, The.

Degree: MS(M.S.), Food Science and Human Nutrition, 2011, Colorado State University

 Background: Cardiovascular disease (CVD) is a major contributor to all-cause mortality in the United States and is a major cause of long-term disability. The risk… (more)

Subjects/Keywords: exercise; acute exercise; meal replacement; nutrition; TNF alpha; tumor necrosis factor alpha

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APA (6th Edition):

Warro, D. (2011). Effects of a single bout of exercise, with energy replacement, on plasma concentrations of TNF-α following consumption of a high-fat, high-carbohydrate mixed meal, The. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/47420

Chicago Manual of Style (16th Edition):

Warro, Daniel. “Effects of a single bout of exercise, with energy replacement, on plasma concentrations of TNF-α following consumption of a high-fat, high-carbohydrate mixed meal, The.” 2011. Masters Thesis, Colorado State University. Accessed October 31, 2020. http://hdl.handle.net/10217/47420.

MLA Handbook (7th Edition):

Warro, Daniel. “Effects of a single bout of exercise, with energy replacement, on plasma concentrations of TNF-α following consumption of a high-fat, high-carbohydrate mixed meal, The.” 2011. Web. 31 Oct 2020.

Vancouver:

Warro D. Effects of a single bout of exercise, with energy replacement, on plasma concentrations of TNF-α following consumption of a high-fat, high-carbohydrate mixed meal, The. [Internet] [Masters thesis]. Colorado State University; 2011. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10217/47420.

Council of Science Editors:

Warro D. Effects of a single bout of exercise, with energy replacement, on plasma concentrations of TNF-α following consumption of a high-fat, high-carbohydrate mixed meal, The. [Masters Thesis]. Colorado State University; 2011. Available from: http://hdl.handle.net/10217/47420


University of Alberta

17. Xia, Li. Studies of a mycobacterial α-(1→4)-mannosyltransferase involved in 3-O-methyl-mannose polysaccharide biosynthesis.

Degree: PhD, Department of Chemistry, 2013, University of Alberta

 Mycobacteria produce a number of unique carbohydrates, the majority of which are found in the cell wall of the organism. In addition to these extracellular… (more)

Subjects/Keywords: Regioselective polymethylation; MMP biosynthesis; α-(1→4)-mannosyltransferase; 3-O-methyl-mannose polysaccharide

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APA (6th Edition):

Xia, L. (2013). Studies of a mycobacterial α-(1→4)-mannosyltransferase involved in 3-O-methyl-mannose polysaccharide biosynthesis. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/1v53jz121

Chicago Manual of Style (16th Edition):

Xia, Li. “Studies of a mycobacterial α-(1→4)-mannosyltransferase involved in 3-O-methyl-mannose polysaccharide biosynthesis.” 2013. Doctoral Dissertation, University of Alberta. Accessed October 31, 2020. https://era.library.ualberta.ca/files/1v53jz121.

MLA Handbook (7th Edition):

Xia, Li. “Studies of a mycobacterial α-(1→4)-mannosyltransferase involved in 3-O-methyl-mannose polysaccharide biosynthesis.” 2013. Web. 31 Oct 2020.

Vancouver:

Xia L. Studies of a mycobacterial α-(1→4)-mannosyltransferase involved in 3-O-methyl-mannose polysaccharide biosynthesis. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2020 Oct 31]. Available from: https://era.library.ualberta.ca/files/1v53jz121.

Council of Science Editors:

Xia L. Studies of a mycobacterial α-(1→4)-mannosyltransferase involved in 3-O-methyl-mannose polysaccharide biosynthesis. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/1v53jz121


University of New South Wales

18. Zhang, Ning. Transcriptional regulation of PDGF receptor alpha in fibroblasts.

Degree: Centre for Vascular Research, 2011, University of New South Wales

 Wound healing is a process of stimulating cell growth and proliferation to achieve the result of repair. This process is controlled by cytokines and growth… (more)

Subjects/Keywords: PDGF receptor alpha; fibroblasts; interleukin 1 beta (IL-1beta); tumour necrosis factor alpha (TNF-alpha); receptor alpha (PDGF-R-alpha); wound healing

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APA (6th Edition):

Zhang, N. (2011). Transcriptional regulation of PDGF receptor alpha in fibroblasts. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Doctoral Dissertation, University of New South Wales. Accessed October 31, 2020. http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

MLA Handbook (7th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Web. 31 Oct 2020.

Vancouver:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2020 Oct 31]. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

Council of Science Editors:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

19. Rocha, Tatiana Marques Ferreira da. Associação entre polimorfismos nos genes SLC2A1, SLC2A2, HNF1A, TGFB1 e DCP1A e nefropatia em portadores de diabetes mellitus tipo 1.

Degree: Mestrado, Endocrinologia, 2013, University of São Paulo

 A nefropatia diabética (ND) decorre da hiperglicemia crônica, de fatores de risco como a hipertensão arterial e a dislipidemia e de uma susceptibilidade genética já… (more)

Subjects/Keywords: DCP1A; DCP1A; Diabetes mellitus tipo 1; Diabetic nephropaty; Fator 1-alfa nuclear de hepatócito; Fator de crescimento transformador beta; Glucose transporter type 1; Glucose transporter type 2; Nefropatias diabéticas; Nuclear factor 1-alpha hepatocyte; Polimorfismo de nucleotídeo único; Protein smad 4; Proteína smad 4; Single nucleotide polymorphism; Transforming growth factor beta; Transportador de glucose tipo 1; Transportador de glucose tipo 2; Type 1 diabetes mellitus

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APA (6th Edition):

Rocha, T. M. F. d. (2013). Associação entre polimorfismos nos genes SLC2A1, SLC2A2, HNF1A, TGFB1 e DCP1A e nefropatia em portadores de diabetes mellitus tipo 1. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26032013-141528/ ;

Chicago Manual of Style (16th Edition):

Rocha, Tatiana Marques Ferreira da. “Associação entre polimorfismos nos genes SLC2A1, SLC2A2, HNF1A, TGFB1 e DCP1A e nefropatia em portadores de diabetes mellitus tipo 1.” 2013. Masters Thesis, University of São Paulo. Accessed October 31, 2020. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26032013-141528/ ;.

MLA Handbook (7th Edition):

Rocha, Tatiana Marques Ferreira da. “Associação entre polimorfismos nos genes SLC2A1, SLC2A2, HNF1A, TGFB1 e DCP1A e nefropatia em portadores de diabetes mellitus tipo 1.” 2013. Web. 31 Oct 2020.

Vancouver:

Rocha TMFd. Associação entre polimorfismos nos genes SLC2A1, SLC2A2, HNF1A, TGFB1 e DCP1A e nefropatia em portadores de diabetes mellitus tipo 1. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Oct 31]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26032013-141528/ ;.

Council of Science Editors:

Rocha TMFd. Associação entre polimorfismos nos genes SLC2A1, SLC2A2, HNF1A, TGFB1 e DCP1A e nefropatia em portadores de diabetes mellitus tipo 1. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26032013-141528/ ;

20. Biel, Stephanie E. A Biologically Active Tnf-Alpha Inhibitor Fails To Suppress Colitis In Balb/C Mice.

Degree: MSin Cell and Molecular Biology, Biomedical Sciences, 2016, Missouri State University

 Tumor necrosis factor a (TNFα), a potent inflammatory cytokine, has long been established as a major driving force for pathologic inflammation. Currently, anti-TNFα therapies are… (more)

Subjects/Keywords: inflammatory bowel disease; tumor necrosis factor alpha; tumor necrosis factor alpha converting enzyme; ulcerative colitis; Crohn’s disease; Medical Molecular Biology

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APA (6th Edition):

Biel, S. E. (2016). A Biologically Active Tnf-Alpha Inhibitor Fails To Suppress Colitis In Balb/C Mice. (Masters Thesis). Missouri State University. Retrieved from https://bearworks.missouristate.edu/theses/3026

Chicago Manual of Style (16th Edition):

Biel, Stephanie E. “A Biologically Active Tnf-Alpha Inhibitor Fails To Suppress Colitis In Balb/C Mice.” 2016. Masters Thesis, Missouri State University. Accessed October 31, 2020. https://bearworks.missouristate.edu/theses/3026.

MLA Handbook (7th Edition):

Biel, Stephanie E. “A Biologically Active Tnf-Alpha Inhibitor Fails To Suppress Colitis In Balb/C Mice.” 2016. Web. 31 Oct 2020.

Vancouver:

Biel SE. A Biologically Active Tnf-Alpha Inhibitor Fails To Suppress Colitis In Balb/C Mice. [Internet] [Masters thesis]. Missouri State University; 2016. [cited 2020 Oct 31]. Available from: https://bearworks.missouristate.edu/theses/3026.

Council of Science Editors:

Biel SE. A Biologically Active Tnf-Alpha Inhibitor Fails To Suppress Colitis In Balb/C Mice. [Masters Thesis]. Missouri State University; 2016. Available from: https://bearworks.missouristate.edu/theses/3026


Oklahoma State University

21. Semola, Oluwayemisi. Effect of advanced glycation end products on regulation of placental growth factor.

Degree: Veterinary Biomedical Sciences, 2019, Oklahoma State University

 Cardiovascular disease affects nearly half (121.5 million) of the adult US population. In many of these patients, atherosclerotic lesions lead to coronary and peripheral artery… (more)

Subjects/Keywords: advanced glycation end products (age); arteriogenesis; collateral artery growth; diabetes; placental growth factor (plgf); tumor necrosis factor alpha (tnf alpha)

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APA (6th Edition):

Semola, O. (2019). Effect of advanced glycation end products on regulation of placental growth factor. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/324911

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Semola, Oluwayemisi. “Effect of advanced glycation end products on regulation of placental growth factor.” 2019. Thesis, Oklahoma State University. Accessed October 31, 2020. http://hdl.handle.net/11244/324911.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Semola, Oluwayemisi. “Effect of advanced glycation end products on regulation of placental growth factor.” 2019. Web. 31 Oct 2020.

Vancouver:

Semola O. Effect of advanced glycation end products on regulation of placental growth factor. [Internet] [Thesis]. Oklahoma State University; 2019. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/11244/324911.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Semola O. Effect of advanced glycation end products on regulation of placental growth factor. [Thesis]. Oklahoma State University; 2019. Available from: http://hdl.handle.net/11244/324911

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

22. McLachlan, Michael J. Transcription Factor Engineering: Tools and Applications.

Degree: PhD, 0319, 2011, University of Illinois – Urbana-Champaign

 Transcription factors play a vital role in the biology of every organism. By controlling gene expression they regulate growth, development, metabolism, reproduction, signaling, and response… (more)

Subjects/Keywords: Transcription factor; estrogen receptor alpha; protein engineering; biosensor; vascular endothelial growth factor-A (VEGF-A)

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APA (6th Edition):

McLachlan, M. J. (2011). Transcription Factor Engineering: Tools and Applications. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/18624

Chicago Manual of Style (16th Edition):

McLachlan, Michael J. “Transcription Factor Engineering: Tools and Applications.” 2011. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 31, 2020. http://hdl.handle.net/2142/18624.

MLA Handbook (7th Edition):

McLachlan, Michael J. “Transcription Factor Engineering: Tools and Applications.” 2011. Web. 31 Oct 2020.

Vancouver:

McLachlan MJ. Transcription Factor Engineering: Tools and Applications. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2011. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/2142/18624.

Council of Science Editors:

McLachlan MJ. Transcription Factor Engineering: Tools and Applications. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2011. Available from: http://hdl.handle.net/2142/18624

23. Kojima, Anri. Tumor necrosis factor-alpha gene promoter methylation in Japanese adults with chronic periodontitis and rheumatoid arthritis : 日本人慢性歯周炎および関節リウマチ罹患成人におけるTNF-α遺伝子プロモーター領域メチル化の解析.

Degree: 博士(歯学), 2016, Niigata University / 新潟大学

学位の種類: 博士(歯学). 報告番号: 甲第4148号. 学位記番号: 新大院博(歯)甲第346号. 学位授与年月日: 平成28年3月23日

Journal of Periodontal Research. 2016, 51(3), 350-358.

"Background and Objective: Over-expression of tumor necrosis factor-alpha (TNF-a) plays… (more)

Subjects/Keywords: DNA methylation; periodontitis; rheumatoid arthritis; tumor necrosis factor-alpha

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APA (6th Edition):

Kojima, A. (2016). Tumor necrosis factor-alpha gene promoter methylation in Japanese adults with chronic periodontitis and rheumatoid arthritis : 日本人慢性歯周炎および関節リウマチ罹患成人におけるTNF-α遺伝子プロモーター領域メチル化の解析. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/42134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kojima, Anri. “Tumor necrosis factor-alpha gene promoter methylation in Japanese adults with chronic periodontitis and rheumatoid arthritis : 日本人慢性歯周炎および関節リウマチ罹患成人におけるTNF-α遺伝子プロモーター領域メチル化の解析.” 2016. Thesis, Niigata University / 新潟大学. Accessed October 31, 2020. http://hdl.handle.net/10191/42134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kojima, Anri. “Tumor necrosis factor-alpha gene promoter methylation in Japanese adults with chronic periodontitis and rheumatoid arthritis : 日本人慢性歯周炎および関節リウマチ罹患成人におけるTNF-α遺伝子プロモーター領域メチル化の解析.” 2016. Web. 31 Oct 2020.

Vancouver:

Kojima A. Tumor necrosis factor-alpha gene promoter methylation in Japanese adults with chronic periodontitis and rheumatoid arthritis : 日本人慢性歯周炎および関節リウマチ罹患成人におけるTNF-α遺伝子プロモーター領域メチル化の解析. [Internet] [Thesis]. Niigata University / 新潟大学; 2016. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10191/42134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kojima A. Tumor necrosis factor-alpha gene promoter methylation in Japanese adults with chronic periodontitis and rheumatoid arthritis : 日本人慢性歯周炎および関節リウマチ罹患成人におけるTNF-α遺伝子プロモーター領域メチル化の解析. [Thesis]. Niigata University / 新潟大学; 2016. Available from: http://hdl.handle.net/10191/42134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Dalhousie University

24. Parlee, Sebastian Demian. Tumor necrosis factor-{alpha} amplifies adipose-derived chemerin production and bioactivation.

Degree: PhD, Department of Pharmacology, 2013, Dalhousie University

 Due to its escalating prevalence, obesity is becoming a leading cause of morbidity and mortality worldwide. Obesity is a complex health problem accompanied by metabolic… (more)

Subjects/Keywords: Chemerin; Adipose; Tumor Necrosis Factor-{alpha}; Obesity; Inflammation

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APA (6th Edition):

Parlee, S. D. (2013). Tumor necrosis factor-{alpha} amplifies adipose-derived chemerin production and bioactivation. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/21406

Chicago Manual of Style (16th Edition):

Parlee, Sebastian Demian. “Tumor necrosis factor-{alpha} amplifies adipose-derived chemerin production and bioactivation.” 2013. Doctoral Dissertation, Dalhousie University. Accessed October 31, 2020. http://hdl.handle.net/10222/21406.

MLA Handbook (7th Edition):

Parlee, Sebastian Demian. “Tumor necrosis factor-{alpha} amplifies adipose-derived chemerin production and bioactivation.” 2013. Web. 31 Oct 2020.

Vancouver:

Parlee SD. Tumor necrosis factor-{alpha} amplifies adipose-derived chemerin production and bioactivation. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10222/21406.

Council of Science Editors:

Parlee SD. Tumor necrosis factor-{alpha} amplifies adipose-derived chemerin production and bioactivation. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/21406

25. 小林, 信博. Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.

Degree: 博士(歯学), 2017, Osaka Dental University / 大阪歯科大学

The effect of porous alpha-tricalcium phosphate (-TCP) with immobilized basic fibroblast growth factor (bFGF) on bone regeneration was evaluated in a canine mandibular bone defect… (more)

Subjects/Keywords: alpha-tricalcium phosphate |basic fibroblast growth factor | bone regeneration

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APA (6th Edition):

小林, . (2017). Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000138/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小林, 信博. “Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.” 2017. Thesis, Osaka Dental University / 大阪歯科大学. Accessed October 31, 2020. http://id.nii.ac.jp/1392/00000138/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小林, 信博. “Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる.” 2017. Web. 31 Oct 2020.

Vancouver:

小林 . Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. [cited 2020 Oct 31]. Available from: http://id.nii.ac.jp/1392/00000138/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小林 . Porous Alpha-Tricalcium Phosphate with Immobilized Basic Fibroblast Growth Factor Enhances Bone Regeneration in a Canine Mandibular Bone Defect Model : イヌ下顎骨骨欠損モデルにおいて塩基性線維芽細胞増殖因子を固定させたα-リン酸三カルシウム多孔質体は骨再生を促進させる. [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. Available from: http://id.nii.ac.jp/1392/00000138/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

26. Montgomery, Sara L. Cell- and Stage-specific Impact of TNF-alpha Receptor Signaling in Alzheimer's Disease.

Degree: PhD, 2013, University of Rochester

 Alzheimer’s disease (AD) is a progressive degenerative disorder characterized by severe memory loss and cognitive impairment that elaborates in a temporal and spatial manner. Neuropathological… (more)

Subjects/Keywords: Neuroinflammation; Tumor Necrosis Factor-Alpha; Amyloid; Alzheimer's Disease

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APA (6th Edition):

Montgomery, S. L. (2013). Cell- and Stage-specific Impact of TNF-alpha Receptor Signaling in Alzheimer's Disease. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/27246

Chicago Manual of Style (16th Edition):

Montgomery, Sara L. “Cell- and Stage-specific Impact of TNF-alpha Receptor Signaling in Alzheimer's Disease.” 2013. Doctoral Dissertation, University of Rochester. Accessed October 31, 2020. http://hdl.handle.net/1802/27246.

MLA Handbook (7th Edition):

Montgomery, Sara L. “Cell- and Stage-specific Impact of TNF-alpha Receptor Signaling in Alzheimer's Disease.” 2013. Web. 31 Oct 2020.

Vancouver:

Montgomery SL. Cell- and Stage-specific Impact of TNF-alpha Receptor Signaling in Alzheimer's Disease. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/1802/27246.

Council of Science Editors:

Montgomery SL. Cell- and Stage-specific Impact of TNF-alpha Receptor Signaling in Alzheimer's Disease. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/27246

27. Brazee, Nicholas Justin. Armillaria in Massachusetts Forests: Ecology, Species Distribution, and Population Structure, with an Emphasis on Mixed Oak Forests.

Degree: PhD, Plant, Soil & Insect Sciences, 2011, U of Massachusetts : PhD

  The ecology, species distribution, and population structure of Armillaria was investigated in the forests of Massachusetts. From 64 plots at 16 sites, 640 isolates… (more)

Subjects/Keywords: AFLPs; Armillaria; elongation factor 1-alpha; polyphenols; Quercus; Plant Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brazee, N. J. (2011). Armillaria in Massachusetts Forests: Ecology, Species Distribution, and Population Structure, with an Emphasis on Mixed Oak Forests. (Doctoral Dissertation). U of Massachusetts : PhD. Retrieved from https://scholarworks.umass.edu/open_access_dissertations/402

Chicago Manual of Style (16th Edition):

Brazee, Nicholas Justin. “Armillaria in Massachusetts Forests: Ecology, Species Distribution, and Population Structure, with an Emphasis on Mixed Oak Forests.” 2011. Doctoral Dissertation, U of Massachusetts : PhD. Accessed October 31, 2020. https://scholarworks.umass.edu/open_access_dissertations/402.

MLA Handbook (7th Edition):

Brazee, Nicholas Justin. “Armillaria in Massachusetts Forests: Ecology, Species Distribution, and Population Structure, with an Emphasis on Mixed Oak Forests.” 2011. Web. 31 Oct 2020.

Vancouver:

Brazee NJ. Armillaria in Massachusetts Forests: Ecology, Species Distribution, and Population Structure, with an Emphasis on Mixed Oak Forests. [Internet] [Doctoral dissertation]. U of Massachusetts : PhD; 2011. [cited 2020 Oct 31]. Available from: https://scholarworks.umass.edu/open_access_dissertations/402.

Council of Science Editors:

Brazee NJ. Armillaria in Massachusetts Forests: Ecology, Species Distribution, and Population Structure, with an Emphasis on Mixed Oak Forests. [Doctoral Dissertation]. U of Massachusetts : PhD; 2011. Available from: https://scholarworks.umass.edu/open_access_dissertations/402


Boston University

28. Tahhan, Georges. Control and induction of tumor necrosis factor and its receptors on human lymphocytes: a critical structure for immune regulation.

Degree: MS, Medical Sciences, 2015, Boston University

 Type I diabetes (T1D) is an autoimmune disease characterized by the destruction of insulin-producing β cells in the pancreas. Destruction of the body's own proteins,… (more)

Subjects/Keywords: Medicine; TNF; TNFR1; TNFR2; Tumor necrosis factor alpha; Type I diabetes

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APA (6th Edition):

Tahhan, G. (2015). Control and induction of tumor necrosis factor and its receptors on human lymphocytes: a critical structure for immune regulation. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16138

Chicago Manual of Style (16th Edition):

Tahhan, Georges. “Control and induction of tumor necrosis factor and its receptors on human lymphocytes: a critical structure for immune regulation.” 2015. Masters Thesis, Boston University. Accessed October 31, 2020. http://hdl.handle.net/2144/16138.

MLA Handbook (7th Edition):

Tahhan, Georges. “Control and induction of tumor necrosis factor and its receptors on human lymphocytes: a critical structure for immune regulation.” 2015. Web. 31 Oct 2020.

Vancouver:

Tahhan G. Control and induction of tumor necrosis factor and its receptors on human lymphocytes: a critical structure for immune regulation. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/2144/16138.

Council of Science Editors:

Tahhan G. Control and induction of tumor necrosis factor and its receptors on human lymphocytes: a critical structure for immune regulation. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16138


University of Wollongong

29. Sun, Yuan. Design a real-time system for measuring the alpha factor of semiconductor lasers.

Degree: Master of Engineering - Research, 2012, University of Wollongong

  Linewidth enhancement factor, also called α , is a key parameter of a semiconductor laser (SL). This factor determines the characteristic of SL, such… (more)

Subjects/Keywords: alpha factor; semiconductor; real-time; field programmable gate arrays (FPGAs)

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APA (6th Edition):

Sun, Y. (2012). Design a real-time system for measuring the alpha factor of semiconductor lasers. (Masters Thesis). University of Wollongong. Retrieved from ; https://ro.uow.edu.au/theses/3771

Chicago Manual of Style (16th Edition):

Sun, Yuan. “Design a real-time system for measuring the alpha factor of semiconductor lasers.” 2012. Masters Thesis, University of Wollongong. Accessed October 31, 2020. ; https://ro.uow.edu.au/theses/3771.

MLA Handbook (7th Edition):

Sun, Yuan. “Design a real-time system for measuring the alpha factor of semiconductor lasers.” 2012. Web. 31 Oct 2020.

Vancouver:

Sun Y. Design a real-time system for measuring the alpha factor of semiconductor lasers. [Internet] [Masters thesis]. University of Wollongong; 2012. [cited 2020 Oct 31]. Available from: ; https://ro.uow.edu.au/theses/3771.

Council of Science Editors:

Sun Y. Design a real-time system for measuring the alpha factor of semiconductor lasers. [Masters Thesis]. University of Wollongong; 2012. Available from: ; https://ro.uow.edu.au/theses/3771


University of Wisconsin – Milwaukee

30. Medic, Velinka. Modulation of Interleukin-17 By Tumor Necrosis Factor Alpha During the Immune Response to Borrelia Burgdorferi.

Degree: MS, Biomedical Sciences, 2013, University of Wisconsin – Milwaukee

  Arthritis is one of the main complications of late-stage Lyme borreliosis. Cytokines play an important role in the persistent inflammation that is elicited by… (more)

Subjects/Keywords: Interleukin-17; Lyme Arthritis; Tumor Necrosis Factor-Alpha; Allergy and Immunology

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APA (6th Edition):

Medic, V. (2013). Modulation of Interleukin-17 By Tumor Necrosis Factor Alpha During the Immune Response to Borrelia Burgdorferi. (Thesis). University of Wisconsin – Milwaukee. Retrieved from https://dc.uwm.edu/etd/729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Medic, Velinka. “Modulation of Interleukin-17 By Tumor Necrosis Factor Alpha During the Immune Response to Borrelia Burgdorferi.” 2013. Thesis, University of Wisconsin – Milwaukee. Accessed October 31, 2020. https://dc.uwm.edu/etd/729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Medic, Velinka. “Modulation of Interleukin-17 By Tumor Necrosis Factor Alpha During the Immune Response to Borrelia Burgdorferi.” 2013. Web. 31 Oct 2020.

Vancouver:

Medic V. Modulation of Interleukin-17 By Tumor Necrosis Factor Alpha During the Immune Response to Borrelia Burgdorferi. [Internet] [Thesis]. University of Wisconsin – Milwaukee; 2013. [cited 2020 Oct 31]. Available from: https://dc.uwm.edu/etd/729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Medic V. Modulation of Interleukin-17 By Tumor Necrosis Factor Alpha During the Immune Response to Borrelia Burgdorferi. [Thesis]. University of Wisconsin – Milwaukee; 2013. Available from: https://dc.uwm.edu/etd/729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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