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You searched for subject:( glutamate). Showing records 1 – 30 of 855 total matches.

[1] [2] [3] [4] [5] … [29]

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1. Davies, Don 1985-. The medial prefrontal cortex and the dorsomedial striatum are necessary for working memory in rats: role of NMDA receptors.

Degree: 2016, University of Saskatchewan

 Working memory is a form of short-term memory involved in the storage (maintenance) of information over time and reorganization (manipulation) of a memory set necessary… (more)

Subjects/Keywords: Glutamate

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APA (6th Edition):

Davies, D. 1. (2016). The medial prefrontal cortex and the dorsomedial striatum are necessary for working memory in rats: role of NMDA receptors. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davies, Don 1985-. “The medial prefrontal cortex and the dorsomedial striatum are necessary for working memory in rats: role of NMDA receptors.” 2016. Thesis, University of Saskatchewan. Accessed January 19, 2021. http://hdl.handle.net/10388/7626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davies, Don 1985-. “The medial prefrontal cortex and the dorsomedial striatum are necessary for working memory in rats: role of NMDA receptors.” 2016. Web. 19 Jan 2021.

Vancouver:

Davies D1. The medial prefrontal cortex and the dorsomedial striatum are necessary for working memory in rats: role of NMDA receptors. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10388/7626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davies D1. The medial prefrontal cortex and the dorsomedial striatum are necessary for working memory in rats: role of NMDA receptors. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/7626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Crawford, Jordan Thomas. THE GROUP II METABOTROPIC GLUTAMATE AGONIST, LY379268, DECREASES METHAMPHETAMINE SELF-ADMINISTRATION IN RATS.

Degree: 2011, Wake Forest University

 Rationale: Methamphetamine (METH) abuse is a growing problem worldwide, for which there is no effective pharmacotherapy. Group II metabotropic glutamate receptor (mGluR) agonists are proposed… (more)

Subjects/Keywords: Glutamate

…for which there is no effective pharmacotherapy. Group II metabotropic glutamate receptor… …extracellular glutamate within the anteromedial striatum. This increase in extracellular glutamate was… …MDMA administration, suggesting a link between METH induced glutamate rise and alterations… …investigating other psychostimulants revealed that stimulating ionotropic glutamate receptors (… …effects of cocaine, and implying a role for glutamate transmission in the relapse to drug… 

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APA (6th Edition):

Crawford, J. T. (2011). THE GROUP II METABOTROPIC GLUTAMATE AGONIST, LY379268, DECREASES METHAMPHETAMINE SELF-ADMINISTRATION IN RATS. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/36442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crawford, Jordan Thomas. “THE GROUP II METABOTROPIC GLUTAMATE AGONIST, LY379268, DECREASES METHAMPHETAMINE SELF-ADMINISTRATION IN RATS.” 2011. Thesis, Wake Forest University. Accessed January 19, 2021. http://hdl.handle.net/10339/36442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crawford, Jordan Thomas. “THE GROUP II METABOTROPIC GLUTAMATE AGONIST, LY379268, DECREASES METHAMPHETAMINE SELF-ADMINISTRATION IN RATS.” 2011. Web. 19 Jan 2021.

Vancouver:

Crawford JT. THE GROUP II METABOTROPIC GLUTAMATE AGONIST, LY379268, DECREASES METHAMPHETAMINE SELF-ADMINISTRATION IN RATS. [Internet] [Thesis]. Wake Forest University; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10339/36442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crawford JT. THE GROUP II METABOTROPIC GLUTAMATE AGONIST, LY379268, DECREASES METHAMPHETAMINE SELF-ADMINISTRATION IN RATS. [Thesis]. Wake Forest University; 2011. Available from: http://hdl.handle.net/10339/36442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

3. Retzlaff, Cassandra Lynn. Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone.

Degree: PhD, Neuroscience, 2017, Vanderbilt University

 Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone Cassandra Lynn Retzlaff Dissertation under the direction of Professor Randy Blakely,… (more)

Subjects/Keywords: Ceftriaxone; Glutamate; MBLAC1

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APA (6th Edition):

Retzlaff, C. L. (2017). Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14672

Chicago Manual of Style (16th Edition):

Retzlaff, Cassandra Lynn. “Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/14672.

MLA Handbook (7th Edition):

Retzlaff, Cassandra Lynn. “Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone.” 2017. Web. 19 Jan 2021.

Vancouver:

Retzlaff CL. Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/14672.

Council of Science Editors:

Retzlaff CL. Contributions of Metallo-Beta-Lactamase Domain Containing Protein 1 (MBLAC1) to the Neurobiological Actions of Ceftriaxone. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/14672


Université de Sherbrooke

4. Mahmoud, Shaimaa. Role of NLRX1 in glutamate uptake and release by astrocytes: Rôle de NLRX1 dans l'absorption et la libération de glutamate par les astrocytes.

Degree: 2019, Université de Sherbrooke

 Le glutamate est le principal neurotransmetteur excitateur du système nerveux central (SNC). Lorsque le glutamate est libéré dans la fente synaptique par les neurones présynaptiques,… (more)

Subjects/Keywords: NLRX1; Glutamate uptake; Glutamate release; Astrocytes; Excitotoxicity; Absorption de glutamate; Libération de glutamate; Excitotoxicité

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APA (6th Edition):

Mahmoud, S. (2019). Role of NLRX1 in glutamate uptake and release by astrocytes: Rôle de NLRX1 dans l'absorption et la libération de glutamate par les astrocytes. (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/15845

Chicago Manual of Style (16th Edition):

Mahmoud, Shaimaa. “Role of NLRX1 in glutamate uptake and release by astrocytes: Rôle de NLRX1 dans l'absorption et la libération de glutamate par les astrocytes.” 2019. Masters Thesis, Université de Sherbrooke. Accessed January 19, 2021. http://hdl.handle.net/11143/15845.

MLA Handbook (7th Edition):

Mahmoud, Shaimaa. “Role of NLRX1 in glutamate uptake and release by astrocytes: Rôle de NLRX1 dans l'absorption et la libération de glutamate par les astrocytes.” 2019. Web. 19 Jan 2021.

Vancouver:

Mahmoud S. Role of NLRX1 in glutamate uptake and release by astrocytes: Rôle de NLRX1 dans l'absorption et la libération de glutamate par les astrocytes. [Internet] [Masters thesis]. Université de Sherbrooke; 2019. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/11143/15845.

Council of Science Editors:

Mahmoud S. Role of NLRX1 in glutamate uptake and release by astrocytes: Rôle de NLRX1 dans l'absorption et la libération de glutamate par les astrocytes. [Masters Thesis]. Université de Sherbrooke; 2019. Available from: http://hdl.handle.net/11143/15845


California State University – Chico

5. Chin-Curtis, Alena. Neuroprotective effects of cyclosporine A and GluR2 AMPA receptors against excitotoxicity in FVB/N and C57BL/6 mice .

Degree: 2015, California State University – Chico

Glutamate (Glu) is an important excitatory neurotransmitter in the brain. It binds to ionotropic Glu receptors located in the postsynaptic membrane and allows calcium into… (more)

Subjects/Keywords: Cyclosporine; Glutamate; Neurotransmitters

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APA (6th Edition):

Chin-Curtis, A. (2015). Neuroprotective effects of cyclosporine A and GluR2 AMPA receptors against excitotoxicity in FVB/N and C57BL/6 mice . (Thesis). California State University – Chico. Retrieved from http://hdl.handle.net/10211.3/135745

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chin-Curtis, Alena. “Neuroprotective effects of cyclosporine A and GluR2 AMPA receptors against excitotoxicity in FVB/N and C57BL/6 mice .” 2015. Thesis, California State University – Chico. Accessed January 19, 2021. http://hdl.handle.net/10211.3/135745.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chin-Curtis, Alena. “Neuroprotective effects of cyclosporine A and GluR2 AMPA receptors against excitotoxicity in FVB/N and C57BL/6 mice .” 2015. Web. 19 Jan 2021.

Vancouver:

Chin-Curtis A. Neuroprotective effects of cyclosporine A and GluR2 AMPA receptors against excitotoxicity in FVB/N and C57BL/6 mice . [Internet] [Thesis]. California State University – Chico; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10211.3/135745.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chin-Curtis A. Neuroprotective effects of cyclosporine A and GluR2 AMPA receptors against excitotoxicity in FVB/N and C57BL/6 mice . [Thesis]. California State University – Chico; 2015. Available from: http://hdl.handle.net/10211.3/135745

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Montana

6. Hoffman, Katie Marie. THE EFFECT OF SPILLOVER ONTO IONOTROPIC GLUTAMATE RECEPTORS AT THE SCHAFFER-CA1 SYNAPSE IN HIPPOCAMPAL SLICE.

Degree: PhD, 2012, University of Montana

Glutamate is the major excitatory neurotransmitter in the brain and is crucial for processes such as learning and memory. Due its importance as a signaling… (more)

Subjects/Keywords: EAATs; glutamate; NMDARs

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APA (6th Edition):

Hoffman, K. M. (2012). THE EFFECT OF SPILLOVER ONTO IONOTROPIC GLUTAMATE RECEPTORS AT THE SCHAFFER-CA1 SYNAPSE IN HIPPOCAMPAL SLICE. (Doctoral Dissertation). University of Montana. Retrieved from https://scholarworks.umt.edu/etd/994

Chicago Manual of Style (16th Edition):

Hoffman, Katie Marie. “THE EFFECT OF SPILLOVER ONTO IONOTROPIC GLUTAMATE RECEPTORS AT THE SCHAFFER-CA1 SYNAPSE IN HIPPOCAMPAL SLICE.” 2012. Doctoral Dissertation, University of Montana. Accessed January 19, 2021. https://scholarworks.umt.edu/etd/994.

MLA Handbook (7th Edition):

Hoffman, Katie Marie. “THE EFFECT OF SPILLOVER ONTO IONOTROPIC GLUTAMATE RECEPTORS AT THE SCHAFFER-CA1 SYNAPSE IN HIPPOCAMPAL SLICE.” 2012. Web. 19 Jan 2021.

Vancouver:

Hoffman KM. THE EFFECT OF SPILLOVER ONTO IONOTROPIC GLUTAMATE RECEPTORS AT THE SCHAFFER-CA1 SYNAPSE IN HIPPOCAMPAL SLICE. [Internet] [Doctoral dissertation]. University of Montana; 2012. [cited 2021 Jan 19]. Available from: https://scholarworks.umt.edu/etd/994.

Council of Science Editors:

Hoffman KM. THE EFFECT OF SPILLOVER ONTO IONOTROPIC GLUTAMATE RECEPTORS AT THE SCHAFFER-CA1 SYNAPSE IN HIPPOCAMPAL SLICE. [Doctoral Dissertation]. University of Montana; 2012. Available from: https://scholarworks.umt.edu/etd/994


Universiteit Utrecht

7. Bink, D.I. Kainate receptors and cognitive enhancement.

Degree: 2011, Universiteit Utrecht

 The kainate (KA) receptor family is one of the three ionotropic glutamate receptors (iGluRs) families, consisting of five subunits. The receptors were first discovered by… (more)

Subjects/Keywords: Kainate receptors; Glutamate; LTP; Cognition

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APA (6th Edition):

Bink, D. I. (2011). Kainate receptors and cognitive enhancement. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/193556

Chicago Manual of Style (16th Edition):

Bink, D I. “Kainate receptors and cognitive enhancement.” 2011. Masters Thesis, Universiteit Utrecht. Accessed January 19, 2021. http://dspace.library.uu.nl:8080/handle/1874/193556.

MLA Handbook (7th Edition):

Bink, D I. “Kainate receptors and cognitive enhancement.” 2011. Web. 19 Jan 2021.

Vancouver:

Bink DI. Kainate receptors and cognitive enhancement. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2021 Jan 19]. Available from: http://dspace.library.uu.nl:8080/handle/1874/193556.

Council of Science Editors:

Bink DI. Kainate receptors and cognitive enhancement. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/193556


University of California – Berkeley

8. Firl, Alana. Mechanisms underlying spontaneous glutamatergic activity in developing mouse retina.

Degree: Vision Science, 2014, University of California – Berkeley

 Throughout the developing nervous system, spontaneous oscillatory patterns of activity have been observed. The developing murine retina is no exception, where spontaneous activity manifests as… (more)

Subjects/Keywords: Neurosciences; development; glutamate; retina

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APA (6th Edition):

Firl, A. (2014). Mechanisms underlying spontaneous glutamatergic activity in developing mouse retina. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/3nx6s267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Firl, Alana. “Mechanisms underlying spontaneous glutamatergic activity in developing mouse retina.” 2014. Thesis, University of California – Berkeley. Accessed January 19, 2021. http://www.escholarship.org/uc/item/3nx6s267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Firl, Alana. “Mechanisms underlying spontaneous glutamatergic activity in developing mouse retina.” 2014. Web. 19 Jan 2021.

Vancouver:

Firl A. Mechanisms underlying spontaneous glutamatergic activity in developing mouse retina. [Internet] [Thesis]. University of California – Berkeley; 2014. [cited 2021 Jan 19]. Available from: http://www.escholarship.org/uc/item/3nx6s267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Firl A. Mechanisms underlying spontaneous glutamatergic activity in developing mouse retina. [Thesis]. University of California – Berkeley; 2014. Available from: http://www.escholarship.org/uc/item/3nx6s267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

9. Bhardwaj, Paramjit Paul. Cortisol decreases prefrontal glutamine concentrations.

Degree: MS, Department of Psychiatry, 2009, University of Alberta

 In rodents, stress and corticosteroids rapidly increase excitatory neurotransmission. During excitatory neurotransmission, glutamate concentrations are maintained by conversion of glutamine to glutamate. The hypothesis was… (more)

Subjects/Keywords: glutamine; MRS; cortisol; glutamate; spectroscopy

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APA (6th Edition):

Bhardwaj, P. P. (2009). Cortisol decreases prefrontal glutamine concentrations. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/3197xn19c

Chicago Manual of Style (16th Edition):

Bhardwaj, Paramjit Paul. “Cortisol decreases prefrontal glutamine concentrations.” 2009. Masters Thesis, University of Alberta. Accessed January 19, 2021. https://era.library.ualberta.ca/files/3197xn19c.

MLA Handbook (7th Edition):

Bhardwaj, Paramjit Paul. “Cortisol decreases prefrontal glutamine concentrations.” 2009. Web. 19 Jan 2021.

Vancouver:

Bhardwaj PP. Cortisol decreases prefrontal glutamine concentrations. [Internet] [Masters thesis]. University of Alberta; 2009. [cited 2021 Jan 19]. Available from: https://era.library.ualberta.ca/files/3197xn19c.

Council of Science Editors:

Bhardwaj PP. Cortisol decreases prefrontal glutamine concentrations. [Masters Thesis]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/3197xn19c


Vanderbilt University

10. Zike, Isaac Daniel. Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior.

Degree: PhD, Pharmacology, 2017, Vanderbilt University

 Obsessive-compulsive disorder (OCD) is a chronic, disabling condition with inadequate treatment options that leave most patients with substantial residual symptoms. Structural, neurochemical, and behavioral findings… (more)

Subjects/Keywords: obsessive; compulsive; glutamate; neuroscience

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APA (6th Edition):

Zike, I. D. (2017). Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12373

Chicago Manual of Style (16th Edition):

Zike, Isaac Daniel. “Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/12373.

MLA Handbook (7th Edition):

Zike, Isaac Daniel. “Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior.” 2017. Web. 19 Jan 2021.

Vancouver:

Zike ID. Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/12373.

Council of Science Editors:

Zike ID. Investigating the Role of Obsessive-Compulsive Disorder Candidate Gene SLC1A1 in Basal Ganglia and Repetitive Behavior. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/12373


Vanderbilt University

11. Wenthur, Cody James. Development and Characterization of Novel Allosteric Modulators Acting on Metabotropic Glutamate Receptors 2 and 3.

Degree: PhD, Pharmacology, 2015, Vanderbilt University

 The essential roles of glutamatergic signaling in both normal and impaired cognitive functioning have caused metabotropic glutamate (mGlu) receptors to become targets of interest for… (more)

Subjects/Keywords: allosteric; mglu2; mglu3; glutamate; metabotropic

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APA (6th Edition):

Wenthur, C. J. (2015). Development and Characterization of Novel Allosteric Modulators Acting on Metabotropic Glutamate Receptors 2 and 3. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12902

Chicago Manual of Style (16th Edition):

Wenthur, Cody James. “Development and Characterization of Novel Allosteric Modulators Acting on Metabotropic Glutamate Receptors 2 and 3.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/12902.

MLA Handbook (7th Edition):

Wenthur, Cody James. “Development and Characterization of Novel Allosteric Modulators Acting on Metabotropic Glutamate Receptors 2 and 3.” 2015. Web. 19 Jan 2021.

Vancouver:

Wenthur CJ. Development and Characterization of Novel Allosteric Modulators Acting on Metabotropic Glutamate Receptors 2 and 3. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/12902.

Council of Science Editors:

Wenthur CJ. Development and Characterization of Novel Allosteric Modulators Acting on Metabotropic Glutamate Receptors 2 and 3. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/12902


Université Montpellier II

12. Dalet, Antoine. Homéostasie glutamatergique des synapses en calice de l’appareil vestibulaire : implication de plusieurs transporteurs du glutamate de la famille des EAAT : Calyx synapses glutamatergic homeostasis in the vestibular system : implication of several EAAT family glutamate transporters.

Degree: Docteur es, Biologie Santé, 2011, Université Montpellier II

L'homéostasie glutamatergique dans les fentes synaptiques régule la neurotransmission et préserve de l'excitotoxicité. Cela est particulièrement important dans l'oreille interne où il y a une… (more)

Subjects/Keywords: Vestibule; Synapse en calice; Ruban synaptique; Glutamate; Transporteurs du glutamate; Excitotoxicité; Vestibule; Calyx synapse; Synaptic ribbon; Glutamate; Glutamate transporters; Excitotoxicity

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APA (6th Edition):

Dalet, A. (2011). Homéostasie glutamatergique des synapses en calice de l’appareil vestibulaire : implication de plusieurs transporteurs du glutamate de la famille des EAAT : Calyx synapses glutamatergic homeostasis in the vestibular system : implication of several EAAT family glutamate transporters. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2011MON20115

Chicago Manual of Style (16th Edition):

Dalet, Antoine. “Homéostasie glutamatergique des synapses en calice de l’appareil vestibulaire : implication de plusieurs transporteurs du glutamate de la famille des EAAT : Calyx synapses glutamatergic homeostasis in the vestibular system : implication of several EAAT family glutamate transporters.” 2011. Doctoral Dissertation, Université Montpellier II. Accessed January 19, 2021. http://www.theses.fr/2011MON20115.

MLA Handbook (7th Edition):

Dalet, Antoine. “Homéostasie glutamatergique des synapses en calice de l’appareil vestibulaire : implication de plusieurs transporteurs du glutamate de la famille des EAAT : Calyx synapses glutamatergic homeostasis in the vestibular system : implication of several EAAT family glutamate transporters.” 2011. Web. 19 Jan 2021.

Vancouver:

Dalet A. Homéostasie glutamatergique des synapses en calice de l’appareil vestibulaire : implication de plusieurs transporteurs du glutamate de la famille des EAAT : Calyx synapses glutamatergic homeostasis in the vestibular system : implication of several EAAT family glutamate transporters. [Internet] [Doctoral dissertation]. Université Montpellier II; 2011. [cited 2021 Jan 19]. Available from: http://www.theses.fr/2011MON20115.

Council of Science Editors:

Dalet A. Homéostasie glutamatergique des synapses en calice de l’appareil vestibulaire : implication de plusieurs transporteurs du glutamate de la famille des EAAT : Calyx synapses glutamatergic homeostasis in the vestibular system : implication of several EAAT family glutamate transporters. [Doctoral Dissertation]. Université Montpellier II; 2011. Available from: http://www.theses.fr/2011MON20115


University of Illinois – Chicago

13. Stevenson, James M. Antipsychotic Pharmacogenetics in First Episode Psychosis: Is There a Role for Glutamate Genes?.

Degree: 2014, University of Illinois – Chicago

 Symptomatic response to antipsychotic treatment in patients treated for psychotic disorders is highly heterogeneous. The reasons for variable response and tolerability are unclear, but likely… (more)

Subjects/Keywords: antipsychotic; pharmacogenetics; glutamate; pharmacogenomics; psychosis

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APA (6th Edition):

Stevenson, J. M. (2014). Antipsychotic Pharmacogenetics in First Episode Psychosis: Is There a Role for Glutamate Genes?. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/19020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stevenson, James M. “Antipsychotic Pharmacogenetics in First Episode Psychosis: Is There a Role for Glutamate Genes?.” 2014. Thesis, University of Illinois – Chicago. Accessed January 19, 2021. http://hdl.handle.net/10027/19020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stevenson, James M. “Antipsychotic Pharmacogenetics in First Episode Psychosis: Is There a Role for Glutamate Genes?.” 2014. Web. 19 Jan 2021.

Vancouver:

Stevenson JM. Antipsychotic Pharmacogenetics in First Episode Psychosis: Is There a Role for Glutamate Genes?. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10027/19020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stevenson JM. Antipsychotic Pharmacogenetics in First Episode Psychosis: Is There a Role for Glutamate Genes?. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/19020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

14. Beeler-Muscat, Dina M. Characterization of Optimus Prime, A Novel Regulator Of Synaptic Transmission in Drosophila.

Degree: 2018, University of Illinois – Chicago

Glutamate is the most abundant neurotransmitter in the human brain and is used for the fast transfer of information between neurons. Glutamate gated ion channels… (more)

Subjects/Keywords: Synaptic transmission; Drosophila; glutamate

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APA (6th Edition):

Beeler-Muscat, D. M. (2018). Characterization of Optimus Prime, A Novel Regulator Of Synaptic Transmission in Drosophila. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23258

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Beeler-Muscat, Dina M. “Characterization of Optimus Prime, A Novel Regulator Of Synaptic Transmission in Drosophila.” 2018. Thesis, University of Illinois – Chicago. Accessed January 19, 2021. http://hdl.handle.net/10027/23258.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Beeler-Muscat, Dina M. “Characterization of Optimus Prime, A Novel Regulator Of Synaptic Transmission in Drosophila.” 2018. Web. 19 Jan 2021.

Vancouver:

Beeler-Muscat DM. Characterization of Optimus Prime, A Novel Regulator Of Synaptic Transmission in Drosophila. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10027/23258.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Beeler-Muscat DM. Characterization of Optimus Prime, A Novel Regulator Of Synaptic Transmission in Drosophila. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/23258

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Hull, Jonathan. The investigation of the hBCAT proteins in control and diseased human brains : implications for glutamate toxicity in Alzheimer's disease.

Degree: PhD, 2014, University of the West of England, Bristol

 Introduction & Aims: The distribution of the BCAT proteins has been extensively mapped in rodent models, and metabolic studies have established that BCAT transamination in… (more)

Subjects/Keywords: 612.8; BCAT glutamate; Alzheimer's

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APA (6th Edition):

Hull, J. (2014). The investigation of the hBCAT proteins in control and diseased human brains : implications for glutamate toxicity in Alzheimer's disease. (Doctoral Dissertation). University of the West of England, Bristol. Retrieved from https://uwe-repository.worktribe.com/output/824745 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619058

Chicago Manual of Style (16th Edition):

Hull, Jonathan. “The investigation of the hBCAT proteins in control and diseased human brains : implications for glutamate toxicity in Alzheimer's disease.” 2014. Doctoral Dissertation, University of the West of England, Bristol. Accessed January 19, 2021. https://uwe-repository.worktribe.com/output/824745 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619058.

MLA Handbook (7th Edition):

Hull, Jonathan. “The investigation of the hBCAT proteins in control and diseased human brains : implications for glutamate toxicity in Alzheimer's disease.” 2014. Web. 19 Jan 2021.

Vancouver:

Hull J. The investigation of the hBCAT proteins in control and diseased human brains : implications for glutamate toxicity in Alzheimer's disease. [Internet] [Doctoral dissertation]. University of the West of England, Bristol; 2014. [cited 2021 Jan 19]. Available from: https://uwe-repository.worktribe.com/output/824745 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619058.

Council of Science Editors:

Hull J. The investigation of the hBCAT proteins in control and diseased human brains : implications for glutamate toxicity in Alzheimer's disease. [Doctoral Dissertation]. University of the West of England, Bristol; 2014. Available from: https://uwe-repository.worktribe.com/output/824745 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.619058


Colorado State University

16. Giovanni, Maria Elizabeth. Sensory and functional properties of monosodium glutamate.

Degree: PhD, Food Science and Human Nutrition, 2002, Colorado State University

 Flavor potentiators have been used for centuries to improve food flavor. However, neither the taste transduction mechanisms nor the behavior of flavor potentiators in food… (more)

Subjects/Keywords: Monosodium glutamate

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APA (6th Edition):

Giovanni, M. E. (2002). Sensory and functional properties of monosodium glutamate. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/86392

Chicago Manual of Style (16th Edition):

Giovanni, Maria Elizabeth. “Sensory and functional properties of monosodium glutamate.” 2002. Doctoral Dissertation, Colorado State University. Accessed January 19, 2021. http://hdl.handle.net/10217/86392.

MLA Handbook (7th Edition):

Giovanni, Maria Elizabeth. “Sensory and functional properties of monosodium glutamate.” 2002. Web. 19 Jan 2021.

Vancouver:

Giovanni ME. Sensory and functional properties of monosodium glutamate. [Internet] [Doctoral dissertation]. Colorado State University; 2002. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10217/86392.

Council of Science Editors:

Giovanni ME. Sensory and functional properties of monosodium glutamate. [Doctoral Dissertation]. Colorado State University; 2002. Available from: http://hdl.handle.net/10217/86392


University of Toronto

17. Zhang, Jane. Transition to Seizure in the CA3 Hippocampal Network: Predominant Preictal GABAergic Potentials, followed by Predominant Ictal Glutamatergic Potentials.

Degree: 2010, University of Toronto

The mechanisms underlying the transition to seizure are still unresolved. Proposed mechanisms include excitatory GABAergic drive, loss of interneuron-mediated inhibition, and glutamatergic input potentiation. The… (more)

Subjects/Keywords: epilepsy; hippocampus; GABA; glutamate; 0719

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APA (6th Edition):

Zhang, J. (2010). Transition to Seizure in the CA3 Hippocampal Network: Predominant Preictal GABAergic Potentials, followed by Predominant Ictal Glutamatergic Potentials. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30134

Chicago Manual of Style (16th Edition):

Zhang, Jane. “Transition to Seizure in the CA3 Hippocampal Network: Predominant Preictal GABAergic Potentials, followed by Predominant Ictal Glutamatergic Potentials.” 2010. Masters Thesis, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/30134.

MLA Handbook (7th Edition):

Zhang, Jane. “Transition to Seizure in the CA3 Hippocampal Network: Predominant Preictal GABAergic Potentials, followed by Predominant Ictal Glutamatergic Potentials.” 2010. Web. 19 Jan 2021.

Vancouver:

Zhang J. Transition to Seizure in the CA3 Hippocampal Network: Predominant Preictal GABAergic Potentials, followed by Predominant Ictal Glutamatergic Potentials. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/30134.

Council of Science Editors:

Zhang J. Transition to Seizure in the CA3 Hippocampal Network: Predominant Preictal GABAergic Potentials, followed by Predominant Ictal Glutamatergic Potentials. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/30134


University of Houston

18. -9202-6472. Regulating Neuronal Hyperexcitability: The Role of Glutamate Transport, Cannabinoids, and Platinum-Based Anti-Cancer Drugs.

Degree: PhD, Cell and Molecular Biology, 2017, University of Houston

 Our studies consist of three distinct projects addressing the modulation of neuronal hyperexcitability states. Hyperexcitability is linked to many neurological disorders and can occur as… (more)

Subjects/Keywords: Neuronal hyperexcitability; Glutamate transporter; Cannabis

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APA (6th Edition):

-9202-6472. (2017). Regulating Neuronal Hyperexcitability: The Role of Glutamate Transport, Cannabinoids, and Platinum-Based Anti-Cancer Drugs. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/4821

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-9202-6472. “Regulating Neuronal Hyperexcitability: The Role of Glutamate Transport, Cannabinoids, and Platinum-Based Anti-Cancer Drugs.” 2017. Doctoral Dissertation, University of Houston. Accessed January 19, 2021. http://hdl.handle.net/10657/4821.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-9202-6472. “Regulating Neuronal Hyperexcitability: The Role of Glutamate Transport, Cannabinoids, and Platinum-Based Anti-Cancer Drugs.” 2017. Web. 19 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-9202-6472. Regulating Neuronal Hyperexcitability: The Role of Glutamate Transport, Cannabinoids, and Platinum-Based Anti-Cancer Drugs. [Internet] [Doctoral dissertation]. University of Houston; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10657/4821.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-9202-6472. Regulating Neuronal Hyperexcitability: The Role of Glutamate Transport, Cannabinoids, and Platinum-Based Anti-Cancer Drugs. [Doctoral Dissertation]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/4821

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Rice University

19. Chatterjee, Sudeshna. Understanding the activation-deactivation mechanism of glutamate receptor proteins at the single molecule level.

Degree: PhD, Natural Sciences, 2019, Rice University

 Ionotropic glutamate receptor proteins form ion channels at the neuronal membrane and mediate excitatory synaptic neurotransmission in the mammalian central nervous system. These receptor proteins,… (more)

Subjects/Keywords: Glutamate receptors; NMDA; AMPA; smFRET

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APA (6th Edition):

Chatterjee, S. (2019). Understanding the activation-deactivation mechanism of glutamate receptor proteins at the single molecule level. (Doctoral Dissertation). Rice University. Retrieved from http://hdl.handle.net/1911/105998

Chicago Manual of Style (16th Edition):

Chatterjee, Sudeshna. “Understanding the activation-deactivation mechanism of glutamate receptor proteins at the single molecule level.” 2019. Doctoral Dissertation, Rice University. Accessed January 19, 2021. http://hdl.handle.net/1911/105998.

MLA Handbook (7th Edition):

Chatterjee, Sudeshna. “Understanding the activation-deactivation mechanism of glutamate receptor proteins at the single molecule level.” 2019. Web. 19 Jan 2021.

Vancouver:

Chatterjee S. Understanding the activation-deactivation mechanism of glutamate receptor proteins at the single molecule level. [Internet] [Doctoral dissertation]. Rice University; 2019. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1911/105998.

Council of Science Editors:

Chatterjee S. Understanding the activation-deactivation mechanism of glutamate receptor proteins at the single molecule level. [Doctoral Dissertation]. Rice University; 2019. Available from: http://hdl.handle.net/1911/105998


University of New Mexico

20. Puglia, Michael. Ethanol exposure impairs glutamatergic synaptic transmission and plasticity in the ca1 hippocampal region during the third trimester-equivalent of human pregnancy : implications for fetal alcohol spectrum disorder.

Degree: Biomedical Sciences Graduate Program, 2012, University of New Mexico

 Fetal alcohol spectrum disorder results from developmental exposure to ethanol. Although many organ systems are targeted by this teratogen, the central nervous system is exquisitely… (more)

Subjects/Keywords: ethanol; development; plasticity; glutamate; syanptic

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APA (6th Edition):

Puglia, M. (2012). Ethanol exposure impairs glutamatergic synaptic transmission and plasticity in the ca1 hippocampal region during the third trimester-equivalent of human pregnancy : implications for fetal alcohol spectrum disorder. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/56

Chicago Manual of Style (16th Edition):

Puglia, Michael. “Ethanol exposure impairs glutamatergic synaptic transmission and plasticity in the ca1 hippocampal region during the third trimester-equivalent of human pregnancy : implications for fetal alcohol spectrum disorder.” 2012. Doctoral Dissertation, University of New Mexico. Accessed January 19, 2021. https://digitalrepository.unm.edu/biom_etds/56.

MLA Handbook (7th Edition):

Puglia, Michael. “Ethanol exposure impairs glutamatergic synaptic transmission and plasticity in the ca1 hippocampal region during the third trimester-equivalent of human pregnancy : implications for fetal alcohol spectrum disorder.” 2012. Web. 19 Jan 2021.

Vancouver:

Puglia M. Ethanol exposure impairs glutamatergic synaptic transmission and plasticity in the ca1 hippocampal region during the third trimester-equivalent of human pregnancy : implications for fetal alcohol spectrum disorder. [Internet] [Doctoral dissertation]. University of New Mexico; 2012. [cited 2021 Jan 19]. Available from: https://digitalrepository.unm.edu/biom_etds/56.

Council of Science Editors:

Puglia M. Ethanol exposure impairs glutamatergic synaptic transmission and plasticity in the ca1 hippocampal region during the third trimester-equivalent of human pregnancy : implications for fetal alcohol spectrum disorder. [Doctoral Dissertation]. University of New Mexico; 2012. Available from: https://digitalrepository.unm.edu/biom_etds/56


University of Georgia

21. Schaeffer, Lauren Kay Hayrynen. The systemic effects of glutamatergic dysfunction in schizophrenia: from receptors to networks.

Degree: 2018, University of Georgia

 Schizophrenia is a devastating psychiatric illness characterized by a diverse array of structural, functional, and molecular brain abnormalities and a concomitant range of psychotic, affective,… (more)

Subjects/Keywords: Glutamate; Gamma Oscillations; Schizophrenia; Psychosis

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APA (6th Edition):

Schaeffer, L. K. H. (2018). The systemic effects of glutamatergic dysfunction in schizophrenia: from receptors to networks. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/37490

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schaeffer, Lauren Kay Hayrynen. “The systemic effects of glutamatergic dysfunction in schizophrenia: from receptors to networks.” 2018. Thesis, University of Georgia. Accessed January 19, 2021. http://hdl.handle.net/10724/37490.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schaeffer, Lauren Kay Hayrynen. “The systemic effects of glutamatergic dysfunction in schizophrenia: from receptors to networks.” 2018. Web. 19 Jan 2021.

Vancouver:

Schaeffer LKH. The systemic effects of glutamatergic dysfunction in schizophrenia: from receptors to networks. [Internet] [Thesis]. University of Georgia; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10724/37490.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schaeffer LKH. The systemic effects of glutamatergic dysfunction in schizophrenia: from receptors to networks. [Thesis]. University of Georgia; 2018. Available from: http://hdl.handle.net/10724/37490

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

22. Gregg, Laura. Group I metabotropic glutamate receptors modulate endocannabinoid-mediated stress-induced analgesia.

Degree: 2014, University of Georgia

 Stress-induced analgesia (SIA) is mediated by mobilization of endocannabinoid lipids such as 2-arachidonoylglycerol (2-AG) in the midbrain periaqueductal gray (PAG). 2-AG may be synthesized on… (more)

Subjects/Keywords: endocannabinoid; antinociception; metabotropic glutamate receptors

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APA (6th Edition):

Gregg, L. (2014). Group I metabotropic glutamate receptors modulate endocannabinoid-mediated stress-induced analgesia. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/24617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gregg, Laura. “Group I metabotropic glutamate receptors modulate endocannabinoid-mediated stress-induced analgesia.” 2014. Thesis, University of Georgia. Accessed January 19, 2021. http://hdl.handle.net/10724/24617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gregg, Laura. “Group I metabotropic glutamate receptors modulate endocannabinoid-mediated stress-induced analgesia.” 2014. Web. 19 Jan 2021.

Vancouver:

Gregg L. Group I metabotropic glutamate receptors modulate endocannabinoid-mediated stress-induced analgesia. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10724/24617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gregg L. Group I metabotropic glutamate receptors modulate endocannabinoid-mediated stress-induced analgesia. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/24617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Flanagan, Bronac. A biophysical model of the role of astrocytes in hyper-excitability.

Degree: PhD, 2019, Ulster University

 The human brain consists of numerous networks of cells, working in harmony to operate one of the most intricate structures in existence. A fine balance… (more)

Subjects/Keywords: Glutamate; GABA; Computational model

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APA (6th Edition):

Flanagan, B. (2019). A biophysical model of the role of astrocytes in hyper-excitability. (Doctoral Dissertation). Ulster University. Retrieved from https://ulster.pure.elsevier.com/en/studentTheses/e24b9d38-3ec8-4e4c-8653-e87e5278fad6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.793777

Chicago Manual of Style (16th Edition):

Flanagan, Bronac. “A biophysical model of the role of astrocytes in hyper-excitability.” 2019. Doctoral Dissertation, Ulster University. Accessed January 19, 2021. https://ulster.pure.elsevier.com/en/studentTheses/e24b9d38-3ec8-4e4c-8653-e87e5278fad6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.793777.

MLA Handbook (7th Edition):

Flanagan, Bronac. “A biophysical model of the role of astrocytes in hyper-excitability.” 2019. Web. 19 Jan 2021.

Vancouver:

Flanagan B. A biophysical model of the role of astrocytes in hyper-excitability. [Internet] [Doctoral dissertation]. Ulster University; 2019. [cited 2021 Jan 19]. Available from: https://ulster.pure.elsevier.com/en/studentTheses/e24b9d38-3ec8-4e4c-8653-e87e5278fad6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.793777.

Council of Science Editors:

Flanagan B. A biophysical model of the role of astrocytes in hyper-excitability. [Doctoral Dissertation]. Ulster University; 2019. Available from: https://ulster.pure.elsevier.com/en/studentTheses/e24b9d38-3ec8-4e4c-8653-e87e5278fad6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.793777


University of Georgia

24. Gregg, Laura Corrine. Activation of group III metabotropic glutamate receptors regulates stress-induced analgesia through an endocannabinoid mechanism.

Degree: 2014, University of Georgia

 The endocannabinoid 2-arachidonoylglycerol (2-AG) produces adaptive changes in pain responses following exposure to environmental stressors. This phenomenon, termed stress-induced analgesia (SIA), is dependent upon mobilization… (more)

Subjects/Keywords: endocannabinoid; antinociception; metabotropic glutamate receptor

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APA (6th Edition):

Gregg, L. C. (2014). Activation of group III metabotropic glutamate receptors regulates stress-induced analgesia through an endocannabinoid mechanism. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/26333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gregg, Laura Corrine. “Activation of group III metabotropic glutamate receptors regulates stress-induced analgesia through an endocannabinoid mechanism.” 2014. Thesis, University of Georgia. Accessed January 19, 2021. http://hdl.handle.net/10724/26333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gregg, Laura Corrine. “Activation of group III metabotropic glutamate receptors regulates stress-induced analgesia through an endocannabinoid mechanism.” 2014. Web. 19 Jan 2021.

Vancouver:

Gregg LC. Activation of group III metabotropic glutamate receptors regulates stress-induced analgesia through an endocannabinoid mechanism. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10724/26333.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gregg LC. Activation of group III metabotropic glutamate receptors regulates stress-induced analgesia through an endocannabinoid mechanism. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/26333

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Montana Tech

25. Matti, Afnan Lilly. Microwave-Accelerated Synthesis of Isoxazole Inhibitors of The System Xc- Transnporter.

Degree: MIS, 2011, Montana Tech

 The purpose of this study was the synthesis of selective ligands that are important to understand the classes of glutamate binding proteins (i.e. Glu receptors… (more)

Subjects/Keywords: Glutamate; Isoxazole; MARS; Transporter; Xc-

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APA (6th Edition):

Matti, A. L. (2011). Microwave-Accelerated Synthesis of Isoxazole Inhibitors of The System Xc- Transnporter. (Thesis). Montana Tech. Retrieved from https://scholarworks.umt.edu/etd/662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matti, Afnan Lilly. “Microwave-Accelerated Synthesis of Isoxazole Inhibitors of The System Xc- Transnporter.” 2011. Thesis, Montana Tech. Accessed January 19, 2021. https://scholarworks.umt.edu/etd/662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matti, Afnan Lilly. “Microwave-Accelerated Synthesis of Isoxazole Inhibitors of The System Xc- Transnporter.” 2011. Web. 19 Jan 2021.

Vancouver:

Matti AL. Microwave-Accelerated Synthesis of Isoxazole Inhibitors of The System Xc- Transnporter. [Internet] [Thesis]. Montana Tech; 2011. [cited 2021 Jan 19]. Available from: https://scholarworks.umt.edu/etd/662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matti AL. Microwave-Accelerated Synthesis of Isoxazole Inhibitors of The System Xc- Transnporter. [Thesis]. Montana Tech; 2011. Available from: https://scholarworks.umt.edu/etd/662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University College London (University of London)

26. Groom, Anthony J. The involvement of ionotropic AMPA receptors in the pathogenesis of neurological diseases.

Degree: PhD, 2002, University College London (University of London)

 The interest in glutamate in the pathogenesis of neurodegenerative disorders has rapidly expanded since the early descriptions of the excitotoxic properties of amino acids in… (more)

Subjects/Keywords: 616; Glutamate

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APA (6th Edition):

Groom, A. J. (2002). The involvement of ionotropic AMPA receptors in the pathogenesis of neurological diseases. (Doctoral Dissertation). University College London (University of London). Retrieved from https://discovery.ucl.ac.uk/id/eprint/10104751/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268875

Chicago Manual of Style (16th Edition):

Groom, Anthony J. “The involvement of ionotropic AMPA receptors in the pathogenesis of neurological diseases.” 2002. Doctoral Dissertation, University College London (University of London). Accessed January 19, 2021. https://discovery.ucl.ac.uk/id/eprint/10104751/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268875.

MLA Handbook (7th Edition):

Groom, Anthony J. “The involvement of ionotropic AMPA receptors in the pathogenesis of neurological diseases.” 2002. Web. 19 Jan 2021.

Vancouver:

Groom AJ. The involvement of ionotropic AMPA receptors in the pathogenesis of neurological diseases. [Internet] [Doctoral dissertation]. University College London (University of London); 2002. [cited 2021 Jan 19]. Available from: https://discovery.ucl.ac.uk/id/eprint/10104751/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268875.

Council of Science Editors:

Groom AJ. The involvement of ionotropic AMPA receptors in the pathogenesis of neurological diseases. [Doctoral Dissertation]. University College London (University of London); 2002. Available from: https://discovery.ucl.ac.uk/id/eprint/10104751/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268875


Rutgers University

27. Bignell Jr, Henry. Maternal ingestion of glutamine and glutamate during sow pregnancy and lactation: lipid profile analysis of milk and neonatal adipose tissues.

Degree: MS, Animal Sciences, 2014, Rutgers University

 Glutamine and glutamate are the most abundant amino acids found in milk and are thought to be important for rapid growth of neonatal tissues. Although… (more)

Subjects/Keywords: Glutamine; Glutamate; Swine – Reproduction

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APA (6th Edition):

Bignell Jr, H. (2014). Maternal ingestion of glutamine and glutamate during sow pregnancy and lactation: lipid profile analysis of milk and neonatal adipose tissues. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/45208/

Chicago Manual of Style (16th Edition):

Bignell Jr, Henry. “Maternal ingestion of glutamine and glutamate during sow pregnancy and lactation: lipid profile analysis of milk and neonatal adipose tissues.” 2014. Masters Thesis, Rutgers University. Accessed January 19, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/45208/.

MLA Handbook (7th Edition):

Bignell Jr, Henry. “Maternal ingestion of glutamine and glutamate during sow pregnancy and lactation: lipid profile analysis of milk and neonatal adipose tissues.” 2014. Web. 19 Jan 2021.

Vancouver:

Bignell Jr H. Maternal ingestion of glutamine and glutamate during sow pregnancy and lactation: lipid profile analysis of milk and neonatal adipose tissues. [Internet] [Masters thesis]. Rutgers University; 2014. [cited 2021 Jan 19]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/45208/.

Council of Science Editors:

Bignell Jr H. Maternal ingestion of glutamine and glutamate during sow pregnancy and lactation: lipid profile analysis of milk and neonatal adipose tissues. [Masters Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/45208/


Oklahoma State University

28. Ibitokun, Bernadette Olayi. Role of Glutamate in Corneal Nociception.

Degree: Department of Biochemistry and Molecular Biology, 2012, Oklahoma State University

 At sensory peripheral nerve terminals, glutamate is released into tissues, e.g., skin, where it sensitizes surrounding afferent nerves and resident cells. Sensitization of peripheral sensory… (more)

Subjects/Keywords: cornea; glutamate; ionotropic receptors; nociception

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ibitokun, B. O. (2012). Role of Glutamate in Corneal Nociception. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/6650

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ibitokun, Bernadette Olayi. “Role of Glutamate in Corneal Nociception.” 2012. Thesis, Oklahoma State University. Accessed January 19, 2021. http://hdl.handle.net/11244/6650.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ibitokun, Bernadette Olayi. “Role of Glutamate in Corneal Nociception.” 2012. Web. 19 Jan 2021.

Vancouver:

Ibitokun BO. Role of Glutamate in Corneal Nociception. [Internet] [Thesis]. Oklahoma State University; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/11244/6650.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ibitokun BO. Role of Glutamate in Corneal Nociception. [Thesis]. Oklahoma State University; 2012. Available from: http://hdl.handle.net/11244/6650

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Charles Cano, Jonathan Rene. Metabotropic Glutamate Receptors Effects on Feeding.

Degree: Neuroscience, 2015, University of California – Riverside

 Every living organism has two biological driving forces: the need to reproduce and the need to feed. The need to feed is not merely a… (more)

Subjects/Keywords: Neurosciences; feeding; glutamate; lateral hypothalamus; metabotropic glutamate receptors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Charles Cano, J. R. (2015). Metabotropic Glutamate Receptors Effects on Feeding. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/4525s93t

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Charles Cano, Jonathan Rene. “Metabotropic Glutamate Receptors Effects on Feeding.” 2015. Thesis, University of California – Riverside. Accessed January 19, 2021. http://www.escholarship.org/uc/item/4525s93t.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Charles Cano, Jonathan Rene. “Metabotropic Glutamate Receptors Effects on Feeding.” 2015. Web. 19 Jan 2021.

Vancouver:

Charles Cano JR. Metabotropic Glutamate Receptors Effects on Feeding. [Internet] [Thesis]. University of California – Riverside; 2015. [cited 2021 Jan 19]. Available from: http://www.escholarship.org/uc/item/4525s93t.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Charles Cano JR. Metabotropic Glutamate Receptors Effects on Feeding. [Thesis]. University of California – Riverside; 2015. Available from: http://www.escholarship.org/uc/item/4525s93t

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

30. Langer, Elizabeth A. Behavioral Characterization of System xc- Mutant Mice.

Degree: 2014, University of Illinois – Chicago

 System xc- is a membrane-bound transporter that exchanges one glutamate for intake of a cystine molecule. It is thought to be important in regulating extracellular… (more)

Subjects/Keywords: system xc-; cystine glutamate transporter (xCT); extracellular glutamate; mouse behavior

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Langer, E. A. (2014). Behavioral Characterization of System xc- Mutant Mice. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/18879

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Langer, Elizabeth A. “Behavioral Characterization of System xc- Mutant Mice.” 2014. Thesis, University of Illinois – Chicago. Accessed January 19, 2021. http://hdl.handle.net/10027/18879.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Langer, Elizabeth A. “Behavioral Characterization of System xc- Mutant Mice.” 2014. Web. 19 Jan 2021.

Vancouver:

Langer EA. Behavioral Characterization of System xc- Mutant Mice. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10027/18879.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Langer EA. Behavioral Characterization of System xc- Mutant Mice. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/18879

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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