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You searched for subject:( fetal liver). Showing records 1 – 26 of 26 total matches.

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Univerzitet u Beogradu

1. Stefanović, Tomislav Ž., 1963-. Korelacija ultrazvučnih pokazatelja fetalne makrozomijei parametara kontrole glikemije u trudnoći.

Degree: Medicinski fakultet, 2016, Univerzitet u Beogradu

Medicina - Humana reprodukcija / Medicine - Human reproduction

Cilj: Makrozomija i trudnički šećer (GDM) su poslednjih decenija rastući problem akušera, a njihova prevencija, dijagnostika… (more)

Subjects/Keywords: Gestational diabetes mellitus; macrosomy; ultrasound; fetal liver; fetal adipose tissue

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stefanović, Tomislav Ž., 1. (2016). Korelacija ultrazvučnih pokazatelja fetalne makrozomijei parametara kontrole glikemije u trudnoći. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:12566/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stefanović, Tomislav Ž., 1963-. “Korelacija ultrazvučnih pokazatelja fetalne makrozomijei parametara kontrole glikemije u trudnoći.” 2016. Thesis, Univerzitet u Beogradu. Accessed January 21, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:12566/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stefanović, Tomislav Ž., 1963-. “Korelacija ultrazvučnih pokazatelja fetalne makrozomijei parametara kontrole glikemije u trudnoći.” 2016. Web. 21 Jan 2020.

Vancouver:

Stefanović, Tomislav Ž. 1. Korelacija ultrazvučnih pokazatelja fetalne makrozomijei parametara kontrole glikemije u trudnoći. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2020 Jan 21]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12566/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stefanović, Tomislav Ž. 1. Korelacija ultrazvučnih pokazatelja fetalne makrozomijei parametara kontrole glikemije u trudnoći. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12566/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

2. Porayette, Prashanth. THE BMP4-SMAD5 SIGNALING PATHWAY IS REQUIRED FOR THE RAPID EXPANSION OF ERYTHROID PROGENITORS IN THE EMBRYO.

Degree: PhD, Integrative Biosciences, 2007, Penn State University

 During development, the embryo grows at a very rapid pace, unlike seen anytime during adult life. This rapid growth place a severe demand on the… (more)

Subjects/Keywords: fetal liver; BMP4; Smad5; erythropoiesis

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APA (6th Edition):

Porayette, P. (2007). THE BMP4-SMAD5 SIGNALING PATHWAY IS REQUIRED FOR THE RAPID EXPANSION OF ERYTHROID PROGENITORS IN THE EMBRYO. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8150

Chicago Manual of Style (16th Edition):

Porayette, Prashanth. “THE BMP4-SMAD5 SIGNALING PATHWAY IS REQUIRED FOR THE RAPID EXPANSION OF ERYTHROID PROGENITORS IN THE EMBRYO.” 2007. Doctoral Dissertation, Penn State University. Accessed January 21, 2020. https://etda.libraries.psu.edu/catalog/8150.

MLA Handbook (7th Edition):

Porayette, Prashanth. “THE BMP4-SMAD5 SIGNALING PATHWAY IS REQUIRED FOR THE RAPID EXPANSION OF ERYTHROID PROGENITORS IN THE EMBRYO.” 2007. Web. 21 Jan 2020.

Vancouver:

Porayette P. THE BMP4-SMAD5 SIGNALING PATHWAY IS REQUIRED FOR THE RAPID EXPANSION OF ERYTHROID PROGENITORS IN THE EMBRYO. [Internet] [Doctoral dissertation]. Penn State University; 2007. [cited 2020 Jan 21]. Available from: https://etda.libraries.psu.edu/catalog/8150.

Council of Science Editors:

Porayette P. THE BMP4-SMAD5 SIGNALING PATHWAY IS REQUIRED FOR THE RAPID EXPANSION OF ERYTHROID PROGENITORS IN THE EMBRYO. [Doctoral Dissertation]. Penn State University; 2007. Available from: https://etda.libraries.psu.edu/catalog/8150


Queens University

3. Platt, Alex. Alteration in MicroRNA Expression Pattern in CD-1 Mouse Fetal Liver Cells Following In Vitro Benzoquinone Exposure .

Degree: Biomedical and Molecular Sciences, Queens University

 The incidence of childhood leukemias in developed countries is increasing and in utero exposure to environmental toxicants such as benzene may be primarily responsible. The… (more)

Subjects/Keywords: Benzene; Fetal liver; microRNA

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APA (6th Edition):

Platt, A. (n.d.). Alteration in MicroRNA Expression Pattern in CD-1 Mouse Fetal Liver Cells Following In Vitro Benzoquinone Exposure . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/24440

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Platt, Alex. “Alteration in MicroRNA Expression Pattern in CD-1 Mouse Fetal Liver Cells Following In Vitro Benzoquinone Exposure .” Thesis, Queens University. Accessed January 21, 2020. http://hdl.handle.net/1974/24440.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Platt, Alex. “Alteration in MicroRNA Expression Pattern in CD-1 Mouse Fetal Liver Cells Following In Vitro Benzoquinone Exposure .” Web. 21 Jan 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Platt A. Alteration in MicroRNA Expression Pattern in CD-1 Mouse Fetal Liver Cells Following In Vitro Benzoquinone Exposure . [Internet] [Thesis]. Queens University; [cited 2020 Jan 21]. Available from: http://hdl.handle.net/1974/24440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Platt A. Alteration in MicroRNA Expression Pattern in CD-1 Mouse Fetal Liver Cells Following In Vitro Benzoquinone Exposure . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/24440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

4. Ortigosa, Cristiane. Doppler venoso fetal na insuficiência placentária: relação com o pH no nascimento.

Degree: PhD, Obstetrícia e Ginecologia, 2012, University of São Paulo

OBJETIVO: O presente estudo, realizado em gestantes de alto risco com diagnóstico de insuficiência placentária, tem como objetivo avaliar o fluxo sanguíneo fetal na veia… (more)

Subjects/Keywords: Circulação hepática; Fetal hypoxia; Hipoxia fetal; Insuficiência placentária; Liver circulation; Placental insufficiency; Ultrasonography Doppler; Ultrassonografia Doppler; Umbilical veins; Veias umbilicais

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APA (6th Edition):

Ortigosa, C. (2012). Doppler venoso fetal na insuficiência placentária: relação com o pH no nascimento. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5139/tde-10072012-161319/ ;

Chicago Manual of Style (16th Edition):

Ortigosa, Cristiane. “Doppler venoso fetal na insuficiência placentária: relação com o pH no nascimento.” 2012. Doctoral Dissertation, University of São Paulo. Accessed January 21, 2020. http://www.teses.usp.br/teses/disponiveis/5/5139/tde-10072012-161319/ ;.

MLA Handbook (7th Edition):

Ortigosa, Cristiane. “Doppler venoso fetal na insuficiência placentária: relação com o pH no nascimento.” 2012. Web. 21 Jan 2020.

Vancouver:

Ortigosa C. Doppler venoso fetal na insuficiência placentária: relação com o pH no nascimento. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2020 Jan 21]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5139/tde-10072012-161319/ ;.

Council of Science Editors:

Ortigosa C. Doppler venoso fetal na insuficiência placentária: relação com o pH no nascimento. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5139/tde-10072012-161319/ ;

5. Ferreira, Amanda Olivotti. Obtenção e caracterização de células do broto hepático de ratos para terapia celular.

Degree: Mestrado, Anatomia dos Animais Domésticos e Silvestres, 2011, University of São Paulo

 As células-tronco são capazes de se diferenciarem em praticamente todos os tipos celulares, podendo ser utilizadas em terapias de substituição em várias doenças. Células de… (more)

Subjects/Keywords: Broto hepático; Células-tronco embrionária e fetal; Citometria de fluxo; Embryonic stem cells and fetal; Flow cytometry; Lipid peroxidation; Liver bud; Liver transaminases; Peroxidação lipídica; Transaminases hepáticas

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APA (6th Edition):

Ferreira, A. O. (2011). Obtenção e caracterização de células do broto hepático de ratos para terapia celular. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10132/tde-05042012-085835/ ;

Chicago Manual of Style (16th Edition):

Ferreira, Amanda Olivotti. “Obtenção e caracterização de células do broto hepático de ratos para terapia celular.” 2011. Masters Thesis, University of São Paulo. Accessed January 21, 2020. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-05042012-085835/ ;.

MLA Handbook (7th Edition):

Ferreira, Amanda Olivotti. “Obtenção e caracterização de células do broto hepático de ratos para terapia celular.” 2011. Web. 21 Jan 2020.

Vancouver:

Ferreira AO. Obtenção e caracterização de células do broto hepático de ratos para terapia celular. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2020 Jan 21]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-05042012-085835/ ;.

Council of Science Editors:

Ferreira AO. Obtenção e caracterização de células do broto hepático de ratos para terapia celular. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-05042012-085835/ ;


IUPUI

6. Lee, Joonyong. The function of ASCL1 in pregnancy-induced maternal liver growth.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The maternal liver shows marked growth during pregnancy to accommodate the development and metabolic needs of the placenta and fetus.… (more)

Subjects/Keywords: Pregnancy  – Research; Hepatocyte growth factor  – Research; Liver  – Growth  – Research; Maternal-fetal exchange; Hyperplasia; Liver  – Hypertrophy; Transcription factors  – Research  – Evaluation  – Analysis; Gene expression  – Research; Developmental neurobiology; Fetal liver cells; Liver  – Regeneration; Stem cells  – Research; Nerves, Peripheral

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APA (6th Edition):

Lee, J. (2014). The function of ASCL1 in pregnancy-induced maternal liver growth. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5971

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Joonyong. “The function of ASCL1 in pregnancy-induced maternal liver growth.” 2014. Thesis, IUPUI. Accessed January 21, 2020. http://hdl.handle.net/1805/5971.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Joonyong. “The function of ASCL1 in pregnancy-induced maternal liver growth.” 2014. Web. 21 Jan 2020.

Vancouver:

Lee J. The function of ASCL1 in pregnancy-induced maternal liver growth. [Internet] [Thesis]. IUPUI; 2014. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/1805/5971.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee J. The function of ASCL1 in pregnancy-induced maternal liver growth. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5971

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Estado do Rio de Janeiro

7. Fernanda Ornellas Pinto da Cruz. Mães obesas, filhotes obesos: estudo experimental.

Degree: Master, 2012, Universidade do Estado do Rio de Janeiro

O aumento da obesidade materna pode refletir em efeitos deletérios na prole adulta, manifestos diferentemente de acordo com o gênero do indivíduo. Este trabalho teve… (more)

Subjects/Keywords: Dimorfismo sexual; Programação materna; Obesidade materna; Fígado; Adipocinas; Tecido adiposo; Maternal obesity; Fetal programming; Sexual dimorphism; Liver; Adipokines; Adipose tissue; MORFOLOGIA

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APA (6th Edition):

Cruz, F. O. P. d. (2012). Mães obesas, filhotes obesos: estudo experimental. (Masters Thesis). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9761 ;

Chicago Manual of Style (16th Edition):

Cruz, Fernanda Ornellas Pinto da. “Mães obesas, filhotes obesos: estudo experimental.” 2012. Masters Thesis, Universidade do Estado do Rio de Janeiro. Accessed January 21, 2020. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9761 ;.

MLA Handbook (7th Edition):

Cruz, Fernanda Ornellas Pinto da. “Mães obesas, filhotes obesos: estudo experimental.” 2012. Web. 21 Jan 2020.

Vancouver:

Cruz FOPd. Mães obesas, filhotes obesos: estudo experimental. [Internet] [Masters thesis]. Universidade do Estado do Rio de Janeiro; 2012. [cited 2020 Jan 21]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9761 ;.

Council of Science Editors:

Cruz FOPd. Mães obesas, filhotes obesos: estudo experimental. [Masters Thesis]. Universidade do Estado do Rio de Janeiro; 2012. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9761 ;


University of Cincinnati

8. Ghiaur, Gabriel. The role of Rho GTPases in hematopoietic stem cell biology: RhoA GTPase regulates adult HSC engraftment and Rac1 GTPases is important for embryonic HSC migration.

Degree: PhD, Medicine : Molecular and Developmental Biology, 2008, University of Cincinnati

 Two closely related Rho GTPases (Rac1 and RhoA) that influence basic biological functions of hematopoietic stem cells were studied during the work included in this… (more)

Subjects/Keywords: Molecular Biology; Hematopoietic stem cell; Hematopoietic ontogeny; yolk sac; AGM; Fetal Liver; Bone marrow transplantation; gene therapy; Rho GTPase; Rac; RhoA

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APA (6th Edition):

Ghiaur, G. (2008). The role of Rho GTPases in hematopoietic stem cell biology: RhoA GTPase regulates adult HSC engraftment and Rac1 GTPases is important for embryonic HSC migration. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1204374567

Chicago Manual of Style (16th Edition):

Ghiaur, Gabriel. “The role of Rho GTPases in hematopoietic stem cell biology: RhoA GTPase regulates adult HSC engraftment and Rac1 GTPases is important for embryonic HSC migration.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed January 21, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1204374567.

MLA Handbook (7th Edition):

Ghiaur, Gabriel. “The role of Rho GTPases in hematopoietic stem cell biology: RhoA GTPase regulates adult HSC engraftment and Rac1 GTPases is important for embryonic HSC migration.” 2008. Web. 21 Jan 2020.

Vancouver:

Ghiaur G. The role of Rho GTPases in hematopoietic stem cell biology: RhoA GTPase regulates adult HSC engraftment and Rac1 GTPases is important for embryonic HSC migration. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2020 Jan 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1204374567.

Council of Science Editors:

Ghiaur G. The role of Rho GTPases in hematopoietic stem cell biology: RhoA GTPase regulates adult HSC engraftment and Rac1 GTPases is important for embryonic HSC migration. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1204374567

9. Vlassaks, E. Elucidating the underlying mechanisms of inflammation caused by perinatal asphyxia and chorioamnionitis.

Degree: 2012, Uitgeverij BOXPress

 Perinatal asphyxia and chorioamnionitis are both important neonatal complications that can lead to serious organ disorders and even neonatal death. This dissertation studied the underlying… (more)

Subjects/Keywords: perinatal asphyxia; fetal asphyxia; chorioamnionitis; inflammation; liver; brain

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APA (6th Edition):

Vlassaks, E. (2012). Elucidating the underlying mechanisms of inflammation caused by perinatal asphyxia and chorioamnionitis. (Doctoral Dissertation). Uitgeverij BOXPress. Retrieved from https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; a1004488-4288-449d-a78d-c2e7b79ab873 ; urn:isbn:9789088915154 ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html

Chicago Manual of Style (16th Edition):

Vlassaks, E. “Elucidating the underlying mechanisms of inflammation caused by perinatal asphyxia and chorioamnionitis.” 2012. Doctoral Dissertation, Uitgeverij BOXPress. Accessed January 21, 2020. https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; a1004488-4288-449d-a78d-c2e7b79ab873 ; urn:isbn:9789088915154 ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html.

MLA Handbook (7th Edition):

Vlassaks, E. “Elucidating the underlying mechanisms of inflammation caused by perinatal asphyxia and chorioamnionitis.” 2012. Web. 21 Jan 2020.

Vancouver:

Vlassaks E. Elucidating the underlying mechanisms of inflammation caused by perinatal asphyxia and chorioamnionitis. [Internet] [Doctoral dissertation]. Uitgeverij BOXPress; 2012. [cited 2020 Jan 21]. Available from: https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; a1004488-4288-449d-a78d-c2e7b79ab873 ; urn:isbn:9789088915154 ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html.

Council of Science Editors:

Vlassaks E. Elucidating the underlying mechanisms of inflammation caused by perinatal asphyxia and chorioamnionitis. [Doctoral Dissertation]. Uitgeverij BOXPress; 2012. Available from: https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; a1004488-4288-449d-a78d-c2e7b79ab873 ; urn:isbn:9789088915154 ; urn:nbn:nl:ui:27-a1004488-4288-449d-a78d-c2e7b79ab873 ; https://cris.maastrichtuniversity.nl/portal/en/publications/elucidating-the-underlying-mechanisms-of-inflammation-caused-by-perinatal-asphyxia-and-chorioamnionitis(a1004488-4288-449d-a78d-c2e7b79ab873).html


Freie Universität Berlin

10. Haumann, Hannah Marie. Hepato-metabolic phenotype in wild-type offspring to heterozygous eNOS deficient mice in regard to the concept of Fetal Programming.

Degree: 2012, Freie Universität Berlin

 Background: The Advanced Fetal Programming Hypothesis proposes that maternal genes are regulative factors of fetal growth and disease risk in later life. NO is an… (more)

Subjects/Keywords: Advanced Fetal Programming; eNOS; liver; metabolic syndrome; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Haumann, H. M. (2012). Hepato-metabolic phenotype in wild-type offspring to heterozygous eNOS deficient mice in regard to the concept of Fetal Programming. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haumann, Hannah Marie. “Hepato-metabolic phenotype in wild-type offspring to heterozygous eNOS deficient mice in regard to the concept of Fetal Programming.” 2012. Thesis, Freie Universität Berlin. Accessed January 21, 2020. http://dx.doi.org/10.17169/refubium-8750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haumann, Hannah Marie. “Hepato-metabolic phenotype in wild-type offspring to heterozygous eNOS deficient mice in regard to the concept of Fetal Programming.” 2012. Web. 21 Jan 2020.

Vancouver:

Haumann HM. Hepato-metabolic phenotype in wild-type offspring to heterozygous eNOS deficient mice in regard to the concept of Fetal Programming. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2020 Jan 21]. Available from: http://dx.doi.org/10.17169/refubium-8750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haumann HM. Hepato-metabolic phenotype in wild-type offspring to heterozygous eNOS deficient mice in regard to the concept of Fetal Programming. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-8750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

11. Topletz, Ariel Rebecca. The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites.

Degree: PhD, 2013, University of Washington

 All-trans-retinoic acid (atRA), the active metabolite of Vitamin A (retinol), is an essential nutrient during both fetal development and adult life. The concentration of atRA… (more)

Subjects/Keywords: 18-OH-RA; 4-oxo-RA; all-trans-retinoic acid; CYP26A1; CYP26B1; fetal liver; Pharmaceutical sciences; pharmaceutics

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APA (6th Edition):

Topletz, A. R. (2013). The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/23714

Chicago Manual of Style (16th Edition):

Topletz, Ariel Rebecca. “The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites.” 2013. Doctoral Dissertation, University of Washington. Accessed January 21, 2020. http://hdl.handle.net/1773/23714.

MLA Handbook (7th Edition):

Topletz, Ariel Rebecca. “The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites.” 2013. Web. 21 Jan 2020.

Vancouver:

Topletz AR. The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/1773/23714.

Council of Science Editors:

Topletz AR. The Relative Importance of CYP26A1 and CYP26B1 in Mediating Retinoid Homeostasis: Studies on the Formation, Elimination and Biological Activity of All-trans-Retinoic Acid Metabolites. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/23714

12. Crabtree, Roy Herbert. A Qualitative Histological Study in Prenatal and Postnatal Liver of Laboratory Mice .

Degree: 1968, Drake U

Subjects/Keywords: Liver cells – Development; Fetal liver cells – Development; Rats as laboratory animals

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APA (6th Edition):

Crabtree, R. H. (1968). A Qualitative Histological Study in Prenatal and Postnatal Liver of Laboratory Mice . (Thesis). Drake U. Retrieved from http://hdl.handle.net/2092/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crabtree, Roy Herbert. “A Qualitative Histological Study in Prenatal and Postnatal Liver of Laboratory Mice .” 1968. Thesis, Drake U. Accessed January 21, 2020. http://hdl.handle.net/2092/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crabtree, Roy Herbert. “A Qualitative Histological Study in Prenatal and Postnatal Liver of Laboratory Mice .” 1968. Web. 21 Jan 2020.

Vancouver:

Crabtree RH. A Qualitative Histological Study in Prenatal and Postnatal Liver of Laboratory Mice . [Internet] [Thesis]. Drake U; 1968. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/2092/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crabtree RH. A Qualitative Histological Study in Prenatal and Postnatal Liver of Laboratory Mice . [Thesis]. Drake U; 1968. Available from: http://hdl.handle.net/2092/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Ogiso, Satoshi. Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes : 胆管経路を利用した胎仔肝前駆細胞による脱細胞化肝臓グラフトの効率的な再細胞化.

Degree: 博士(医学), 2017, Kyoto University / 京都大学

新制・課程博士

甲第20280号

医博第4239号

Subjects/Keywords: liver regeneration; organ regeneration; fetal hepatocyte; decellularization; recellularization; biliary tree

Page 1 Page 2

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APA (6th Edition):

Ogiso, S. (2017). Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes : 胆管経路を利用した胎仔肝前駆細胞による脱細胞化肝臓グラフトの効率的な再細胞化. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/225505 ; http://dx.doi.org/10.14989/doctor.k20280

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ogiso, Satoshi. “Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes : 胆管経路を利用した胎仔肝前駆細胞による脱細胞化肝臓グラフトの効率的な再細胞化.” 2017. Thesis, Kyoto University / 京都大学. Accessed January 21, 2020. http://hdl.handle.net/2433/225505 ; http://dx.doi.org/10.14989/doctor.k20280.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ogiso, Satoshi. “Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes : 胆管経路を利用した胎仔肝前駆細胞による脱細胞化肝臓グラフトの効率的な再細胞化.” 2017. Web. 21 Jan 2020.

Vancouver:

Ogiso S. Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes : 胆管経路を利用した胎仔肝前駆細胞による脱細胞化肝臓グラフトの効率的な再細胞化. [Internet] [Thesis]. Kyoto University / 京都大学; 2017. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/2433/225505 ; http://dx.doi.org/10.14989/doctor.k20280.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ogiso S. Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes : 胆管経路を利用した胎仔肝前駆細胞による脱細胞化肝臓グラフトの効率的な再細胞化. [Thesis]. Kyoto University / 京都大学; 2017. Available from: http://hdl.handle.net/2433/225505 ; http://dx.doi.org/10.14989/doctor.k20280

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

14. Ogiso, Satoshi. Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes .

Degree: 2017, Kyoto University

Subjects/Keywords: liver regeneration; organ regeneration; fetal hepatocyte; decellularization; recellularization; biliary tree

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APA (6th Edition):

Ogiso, S. (2017). Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/225505

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ogiso, Satoshi. “Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes .” 2017. Thesis, Kyoto University. Accessed January 21, 2020. http://hdl.handle.net/2433/225505.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ogiso, Satoshi. “Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes .” 2017. Web. 21 Jan 2020.

Vancouver:

Ogiso S. Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes . [Internet] [Thesis]. Kyoto University; 2017. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/2433/225505.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ogiso S. Efficient recellularisation of decellularised whole-liver grafts using biliary tree and foetal hepatocytes . [Thesis]. Kyoto University; 2017. Available from: http://hdl.handle.net/2433/225505

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Löwenberg, Bob. Fetal liver cell transplantation : role and nature of the fetal haemopoietic stem cell.

Degree: 1975, Erasmus University Medical Center

 textabstractFetal liver cell transplantation deserves intensified interest because, according to previous experimental evidence, it may represent a useful approach to reduce or avoid severe Graft-versus-Host… (more)

Subjects/Keywords: Fetal blood; Graft versus host reaction; Hematopoietic stem cells; Liver cells

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APA (6th Edition):

Löwenberg, B. (1975). Fetal liver cell transplantation : role and nature of the fetal haemopoietic stem cell. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/31320

Chicago Manual of Style (16th Edition):

Löwenberg, Bob. “Fetal liver cell transplantation : role and nature of the fetal haemopoietic stem cell.” 1975. Doctoral Dissertation, Erasmus University Medical Center. Accessed January 21, 2020. http://hdl.handle.net/1765/31320.

MLA Handbook (7th Edition):

Löwenberg, Bob. “Fetal liver cell transplantation : role and nature of the fetal haemopoietic stem cell.” 1975. Web. 21 Jan 2020.

Vancouver:

Löwenberg B. Fetal liver cell transplantation : role and nature of the fetal haemopoietic stem cell. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 1975. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/1765/31320.

Council of Science Editors:

Löwenberg B. Fetal liver cell transplantation : role and nature of the fetal haemopoietic stem cell. [Doctoral Dissertation]. Erasmus University Medical Center; 1975. Available from: http://hdl.handle.net/1765/31320


Universidade do Estado do Rio de Janeiro

16. Bianca Martins Gregório. Dieta hiperlipídica materna e pós-natal promove remodelamento adverso do fígado, pâncreas e tecido adiposo na prole.

Degree: PhD, 2010, Universidade do Estado do Rio de Janeiro

A dieta hiperlipídica (high-fat, HF) materna durante a gestação e/ou lactação aumenta a susceptibilidade da prole para o desenvolvimento de doenças crônicas na fase adulta.… (more)

Subjects/Keywords: programação fetal; camundongos C57BL/6; dieta hiperlipídica materna; gestação/lactação; esteatose hepática; alterações pancreáticas; remodelamento do tecido adiposo; maternal high-fat diet; C57BL/6 mice; fetal programming; gestation/lactation; liver steatosis; pancreatic alteration; adipose tissue remodeling; MORFOLOGIA

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APA (6th Edition):

Gregório, B. M. (2010). Dieta hiperlipídica materna e pós-natal promove remodelamento adverso do fígado, pâncreas e tecido adiposo na prole. (Doctoral Dissertation). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3850 ;

Chicago Manual of Style (16th Edition):

Gregório, Bianca Martins. “Dieta hiperlipídica materna e pós-natal promove remodelamento adverso do fígado, pâncreas e tecido adiposo na prole.” 2010. Doctoral Dissertation, Universidade do Estado do Rio de Janeiro. Accessed January 21, 2020. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3850 ;.

MLA Handbook (7th Edition):

Gregório, Bianca Martins. “Dieta hiperlipídica materna e pós-natal promove remodelamento adverso do fígado, pâncreas e tecido adiposo na prole.” 2010. Web. 21 Jan 2020.

Vancouver:

Gregório BM. Dieta hiperlipídica materna e pós-natal promove remodelamento adverso do fígado, pâncreas e tecido adiposo na prole. [Internet] [Doctoral dissertation]. Universidade do Estado do Rio de Janeiro; 2010. [cited 2020 Jan 21]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3850 ;.

Council of Science Editors:

Gregório BM. Dieta hiperlipídica materna e pós-natal promove remodelamento adverso do fígado, pâncreas e tecido adiposo na prole. [Doctoral Dissertation]. Universidade do Estado do Rio de Janeiro; 2010. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3850 ;


University of Oulu

17. Syrjänen, R. (Riikka). TIM family molecules in hematopoiesis.

Degree: 2014, University of Oulu

Abstract Hematopoietic cells, i.e., erythrocytes, platelets and white blood cells, differentiate from hematopoietic stem cells in a process that is similar in vertebrates. Hematopoiesis is… (more)

Subjects/Keywords: B cell development; fetal liver; gene expression; hematopoiesis; myeloid progenitor cells; para-aortic region; transmembrane immunoglobulin and mucin domain containing molecule; B-solujen kehitys; alkion maksa; geeniekspressio; hematopoieesi; myeloidiset esisolut; para-aortaalinen alue; transmembrane immunoglobulin and mucin domain containing -molekyyli

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APA (6th Edition):

Syrjänen, R. (. (2014). TIM family molecules in hematopoiesis. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526204246

Chicago Manual of Style (16th Edition):

Syrjänen, R (Riikka). “TIM family molecules in hematopoiesis.” 2014. Doctoral Dissertation, University of Oulu. Accessed January 21, 2020. http://urn.fi/urn:isbn:9789526204246.

MLA Handbook (7th Edition):

Syrjänen, R (Riikka). “TIM family molecules in hematopoiesis.” 2014. Web. 21 Jan 2020.

Vancouver:

Syrjänen R(. TIM family molecules in hematopoiesis. [Internet] [Doctoral dissertation]. University of Oulu; 2014. [cited 2020 Jan 21]. Available from: http://urn.fi/urn:isbn:9789526204246.

Council of Science Editors:

Syrjänen R(. TIM family molecules in hematopoiesis. [Doctoral Dissertation]. University of Oulu; 2014. Available from: http://urn.fi/urn:isbn:9789526204246


The Ohio State University

18. Irshaid, Fawzi Irshaid. Characterization of CI1L gene expression on human tissues: identificaiton of CR1L-2, a two SCR transcript from human fetal liver and bone marrow.

Degree: PhD, Molecular, Cellular, and Developmental Biology, 2005, The Ohio State University

 CR1-like (CR1L) is a genetic element, identified from human genomic clones, with high homology to complement receptor type one (CR1). Though CR1L is expressed in… (more)

Subjects/Keywords: complement receptor type one (CR1); CR1-like; short consensus repeats; northern blot; human fetal liver; bone marrow; C4d

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APA (6th Edition):

Irshaid, F. I. (2005). Characterization of CI1L gene expression on human tissues: identificaiton of CR1L-2, a two SCR transcript from human fetal liver and bone marrow. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1111530577

Chicago Manual of Style (16th Edition):

Irshaid, Fawzi Irshaid. “Characterization of CI1L gene expression on human tissues: identificaiton of CR1L-2, a two SCR transcript from human fetal liver and bone marrow.” 2005. Doctoral Dissertation, The Ohio State University. Accessed January 21, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1111530577.

MLA Handbook (7th Edition):

Irshaid, Fawzi Irshaid. “Characterization of CI1L gene expression on human tissues: identificaiton of CR1L-2, a two SCR transcript from human fetal liver and bone marrow.” 2005. Web. 21 Jan 2020.

Vancouver:

Irshaid FI. Characterization of CI1L gene expression on human tissues: identificaiton of CR1L-2, a two SCR transcript from human fetal liver and bone marrow. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2020 Jan 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1111530577.

Council of Science Editors:

Irshaid FI. Characterization of CI1L gene expression on human tissues: identificaiton of CR1L-2, a two SCR transcript from human fetal liver and bone marrow. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1111530577


Università Cattolica del Sacro Cuore

19. TONDELLI, BARBARA. TECNICHE AVANZATE NELLA MESSA A PUNTO DI TECNOLOGIE TRANSGENICHE E NON NELLA SPECIE MURINA.

Degree: 2009, Università Cattolica del Sacro Cuore

L’osteopetrosi autosomale recessiva (ARO) è un gruppo di malattie dovute a un difettoso funzionamento degli osteoclasti che preclude un rimodellamento osseo corretto. Nel 50% dei… (more)

Subjects/Keywords: AGR/16: MICROBIOLOGIA AGRARIA; osteoclasta, trapianto in utero, epatociti fetali, osteopetrosi, cellule stminali ematopoietiche, GOF, Oct-4, cellule staminali embrionali, osteoclasts, in utero transplantation, fetal liver, osteopetrosis, hematopoietic stem cells, GOF, Oct-4, embryonic stem cells

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APA (6th Edition):

TONDELLI, B. (2009). TECNICHE AVANZATE NELLA MESSA A PUNTO DI TECNOLOGIE TRANSGENICHE E NON NELLA SPECIE MURINA. (Doctoral Dissertation). Università Cattolica del Sacro Cuore. Retrieved from http://hdl.handle.net/10280/406

Chicago Manual of Style (16th Edition):

TONDELLI, BARBARA. “TECNICHE AVANZATE NELLA MESSA A PUNTO DI TECNOLOGIE TRANSGENICHE E NON NELLA SPECIE MURINA.” 2009. Doctoral Dissertation, Università Cattolica del Sacro Cuore. Accessed January 21, 2020. http://hdl.handle.net/10280/406.

MLA Handbook (7th Edition):

TONDELLI, BARBARA. “TECNICHE AVANZATE NELLA MESSA A PUNTO DI TECNOLOGIE TRANSGENICHE E NON NELLA SPECIE MURINA.” 2009. Web. 21 Jan 2020.

Vancouver:

TONDELLI B. TECNICHE AVANZATE NELLA MESSA A PUNTO DI TECNOLOGIE TRANSGENICHE E NON NELLA SPECIE MURINA. [Internet] [Doctoral dissertation]. Università Cattolica del Sacro Cuore; 2009. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/10280/406.

Council of Science Editors:

TONDELLI B. TECNICHE AVANZATE NELLA MESSA A PUNTO DI TECNOLOGIE TRANSGENICHE E NON NELLA SPECIE MURINA. [Doctoral Dissertation]. Università Cattolica del Sacro Cuore; 2009. Available from: http://hdl.handle.net/10280/406

20. ANTONY SAGAYARAJ. I. GENETIC REGULATION OF HUMAN FETAL LIVER AND ITS CLINICAL APPLICATION.

Degree: 2015, National University of Singapore

Subjects/Keywords: Human fetal Liver; developmental biology; Transcript signatures; 5 phases; growth factors and extracellular matrix

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APA (6th Edition):

I, A. S. (2015). GENETIC REGULATION OF HUMAN FETAL LIVER AND ITS CLINICAL APPLICATION. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/119899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

I, ANTONY SAGAYARAJ.. “GENETIC REGULATION OF HUMAN FETAL LIVER AND ITS CLINICAL APPLICATION.” 2015. Thesis, National University of Singapore. Accessed January 21, 2020. http://scholarbank.nus.edu.sg/handle/10635/119899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

I, ANTONY SAGAYARAJ.. “GENETIC REGULATION OF HUMAN FETAL LIVER AND ITS CLINICAL APPLICATION.” 2015. Web. 21 Jan 2020.

Vancouver:

I AS. GENETIC REGULATION OF HUMAN FETAL LIVER AND ITS CLINICAL APPLICATION. [Internet] [Thesis]. National University of Singapore; 2015. [cited 2020 Jan 21]. Available from: http://scholarbank.nus.edu.sg/handle/10635/119899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

I AS. GENETIC REGULATION OF HUMAN FETAL LIVER AND ITS CLINICAL APPLICATION. [Thesis]. National University of Singapore; 2015. Available from: http://scholarbank.nus.edu.sg/handle/10635/119899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Ma, Noelle L. Fetal and Neonatal Exposure to Nicotine Augments Hepatic Fatty Acid Synthesis in Rat Offspring Long-Term.

Degree: 2013, University of Western Ontario

 While nicotine replacement therapy (NRT) is presumed to be a safer alternative to smoking in pregnancy, the long-term consequences in offspring are still largely unknown.… (more)

Subjects/Keywords: Nicotine Replacement Therapy (NRT); Triglycerides; Fatty Acid Synthase (FAS); Liver; Liver X Receptor (LXR); Obesity; Fetal Programming; Medical Pharmacology; Medical Physiology; Obstetrics and Gynecology

…60 4.6 The role of epigenetics in the fetal programming of hypertriglyceridemia in adult… …liver of PND180 male offspring x 47 3.3.1 MNE-PL increases hepatic the acetylation of… …Kilogram LiCl Lithium chloride LXR Liver X Receptor α and Liver X Receptor β LXRE LXR… …x29;, impaired fetal growth (2, 3) and perinatal mortality (2, 3)… …x28;36, 39). Nicotine is absorbed with high affinity in the brain, muscle, liver, lung… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

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APA (6th Edition):

Ma, N. L. (2013). Fetal and Neonatal Exposure to Nicotine Augments Hepatic Fatty Acid Synthesis in Rat Offspring Long-Term. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1374

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ma, Noelle L. “Fetal and Neonatal Exposure to Nicotine Augments Hepatic Fatty Acid Synthesis in Rat Offspring Long-Term.” 2013. Thesis, University of Western Ontario. Accessed January 21, 2020. https://ir.lib.uwo.ca/etd/1374.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ma, Noelle L. “Fetal and Neonatal Exposure to Nicotine Augments Hepatic Fatty Acid Synthesis in Rat Offspring Long-Term.” 2013. Web. 21 Jan 2020.

Vancouver:

Ma NL. Fetal and Neonatal Exposure to Nicotine Augments Hepatic Fatty Acid Synthesis in Rat Offspring Long-Term. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2020 Jan 21]. Available from: https://ir.lib.uwo.ca/etd/1374.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ma NL. Fetal and Neonatal Exposure to Nicotine Augments Hepatic Fatty Acid Synthesis in Rat Offspring Long-Term. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1374

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Iqbal, Waseem. Prenatal Programming of Hepatic Glucose and Cholesterol Regulation in Male Rat Offspring by Chronic Intermittent Hypoxia.

Degree: 2013, University of Western Ontario

 Obstructive sleep apnea (OSA) is a chronic disorder involving repetitive interruptions in breathing during sleep. Sufferers of OSA are exposed to chronic intermittent hypoxia (CIH),… (more)

Subjects/Keywords: Obstructive sleep apnea; pregnancy; fetal programming; intrauterine growth restriction; liver metabolism; liver x receptor; Developmental Biology; Endocrinology; Nutritional and Metabolic Diseases; Obstetrics and Gynecology

…INTERMITTENT HYPOXIA IMPAIRS HEPATIC GLUCOSE HOMEOSTASIS VIA REDUCED LIVER X RECEPTOR REGULATION OF… …lipoprotein LDLR Low-density lipoprotein receptor LXR Liver X receptor MAP Mitogen activated… …protein MLK Mixed-lineage kinase NAFLD Non-alcoholic fatty liver disease NASH Non… …al. 2008). 1.3.5.3 – Liver disease Non-alcoholic fatty liver disease (NAFLD)… …is a persistent liver disease that is estimated to affect as many as two thirds of… 

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APA (6th Edition):

Iqbal, W. (2013). Prenatal Programming of Hepatic Glucose and Cholesterol Regulation in Male Rat Offspring by Chronic Intermittent Hypoxia. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iqbal, Waseem. “Prenatal Programming of Hepatic Glucose and Cholesterol Regulation in Male Rat Offspring by Chronic Intermittent Hypoxia.” 2013. Thesis, University of Western Ontario. Accessed January 21, 2020. https://ir.lib.uwo.ca/etd/1817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iqbal, Waseem. “Prenatal Programming of Hepatic Glucose and Cholesterol Regulation in Male Rat Offspring by Chronic Intermittent Hypoxia.” 2013. Web. 21 Jan 2020.

Vancouver:

Iqbal W. Prenatal Programming of Hepatic Glucose and Cholesterol Regulation in Male Rat Offspring by Chronic Intermittent Hypoxia. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2020 Jan 21]. Available from: https://ir.lib.uwo.ca/etd/1817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iqbal W. Prenatal Programming of Hepatic Glucose and Cholesterol Regulation in Male Rat Offspring by Chronic Intermittent Hypoxia. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

23. Ducsay, Charles Andrew, 1953- ( Dissertant ). Uteroferrin : a mechanism of maternal to fetal iron transport in swine.

Degree: 1980, University of Florida

 Uteroferrin (UF) is an iron-containing, progesteroneinduced glycoprotein with an approximate molecular weight of 35,000. This protein is present in uterine secretions of non-pregnant swine between… (more)

Subjects/Keywords: Allantoic fluid; Fetus; Gilts; Iron; Liver; Phosphatases; Placenta; Pregnancy; Secretion; Swine; Animal Science thesis Ph. D; Iron  – Metabolism; Maternal-fetal exchange; Swine  – Embryos; Swine  – Physiology

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APA (6th Edition):

Ducsay, Charles Andrew, 1. (. D. ). (1980). Uteroferrin : a mechanism of maternal to fetal iron transport in swine. (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UF00099376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ducsay, Charles Andrew, 1953- ( Dissertant ). “Uteroferrin : a mechanism of maternal to fetal iron transport in swine.” 1980. Thesis, University of Florida. Accessed January 21, 2020. http://ufdc.ufl.edu/UF00099376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ducsay, Charles Andrew, 1953- ( Dissertant ). “Uteroferrin : a mechanism of maternal to fetal iron transport in swine.” 1980. Web. 21 Jan 2020.

Vancouver:

Ducsay, Charles Andrew 1(D). Uteroferrin : a mechanism of maternal to fetal iron transport in swine. [Internet] [Thesis]. University of Florida; 1980. [cited 2020 Jan 21]. Available from: http://ufdc.ufl.edu/UF00099376.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ducsay, Charles Andrew 1(D). Uteroferrin : a mechanism of maternal to fetal iron transport in swine. [Thesis]. University of Florida; 1980. Available from: http://ufdc.ufl.edu/UF00099376

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Karkoulia, Elena. Characterization of the molecular functions of the transcription factor GATA-1 in hematopoiesis.

Degree: 2013, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Subjects/Keywords: Μοριακή λειτουργία; Μεταγραφικός παράγοντας; Βιοτινυλίωση; Αιμοποίηση; Εμβρυϊκό ήπαρ; Ερυθρά κύτταρα; Molecular function; Transcription factor; GATA-1; biotin tag; Knock-in; Hematopoiesis; Fetal liver; Erythroid cells

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APA (6th Edition):

Karkoulia, E. (2013). Characterization of the molecular functions of the transcription factor GATA-1 in hematopoiesis. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/38926

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Karkoulia, Elena. “Characterization of the molecular functions of the transcription factor GATA-1 in hematopoiesis.” 2013. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed January 21, 2020. http://hdl.handle.net/10442/hedi/38926.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Karkoulia, Elena. “Characterization of the molecular functions of the transcription factor GATA-1 in hematopoiesis.” 2013. Web. 21 Jan 2020.

Vancouver:

Karkoulia E. Characterization of the molecular functions of the transcription factor GATA-1 in hematopoiesis. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2013. [cited 2020 Jan 21]. Available from: http://hdl.handle.net/10442/hedi/38926.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Karkoulia E. Characterization of the molecular functions of the transcription factor GATA-1 in hematopoiesis. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2013. Available from: http://hdl.handle.net/10442/hedi/38926

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Montwill, Natalie. Characterization of Hemangioma-initiating Stem Cells.

Degree: 2017, University of Western Ontario

 Infantile hemangioma (IH) is the most common vascular tumour of infancy. IH undergoes a unique life cycle consisting of robust endothelial cell proliferation and vessel… (more)

Subjects/Keywords: infantile hemangioma; hemangioma stem cells; fetal liver stem cells; bone marrow progenitor cells; hematopoiesis; endothelial-to-hematopoietic transition; Congenital, Hereditary, and Neonatal Diseases and Abnormalities

liver FLI1 Fli-1 proto-oncogene FLK1 Fetal liver kinase-1 (Vascular endothelial… …growth factor receptor 2) FLSC Fetal liver stem cell FLT1 Fms related tyrosine kinase… …most to the HSC pool.114 Following definitive hematopoiesis, HSCs migrate to the fetal liver… …only give rise to endothelial or hematopoietic colonies, but not both.120 Fetal liver kinase… …The fetal liver The fetal liver (FL) is a well-known site of definitive… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Montwill, N. (2017). Characterization of Hemangioma-initiating Stem Cells. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Montwill, Natalie. “Characterization of Hemangioma-initiating Stem Cells.” 2017. Thesis, University of Western Ontario. Accessed January 21, 2020. https://ir.lib.uwo.ca/etd/4873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Montwill, Natalie. “Characterization of Hemangioma-initiating Stem Cells.” 2017. Web. 21 Jan 2020.

Vancouver:

Montwill N. Characterization of Hemangioma-initiating Stem Cells. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Jan 21]. Available from: https://ir.lib.uwo.ca/etd/4873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Montwill N. Characterization of Hemangioma-initiating Stem Cells. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Sohi, Gurjeev. Molecular Mechanisms Underlying the Early Life Programming of the Liver.

Degree: 2013, University of Western Ontario

 Clinical studies have demonstrated that intrauterine growth restriction (IUGR) offspring, faced with a nutritional mismatch postpartum, have an increased risk of developing the metabolic syndrome.… (more)

Subjects/Keywords: Fetal Programming; Intrauterine Growth Restriction; Thrifty Phenotype; Low Protein Diet; Metabolic Syndrome; Cholesterol; Insulin Resistance; Liver; Epigenetics; Posttranslational Histone Modifications; Endoplasmic Reticulum Stress; Protein Translation; Growth; Drug Metabolism; Developmental Biology; Endocrinology; Life Sciences; Molecular Biology; Nutrition; Pharmacology; Physiology

…suggests that iron restriction in utero has short-term effects on fetal liver function which may… …liver growth 68 3.3.2 A low protein diet during pregnancy and lactation leads to augmented… …initiation site throughout fetal and postnatal development 76 3.3.5 Decreased recruitment of RNA… …protein levels were unaltered by maternal protein restriction in the fetal livers at embryonic… …Primers 62 Table 3.2: The effect of LP dietary regimes LP1, LP2, LP3 on liver growth and body… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sohi, G. (2013). Molecular Mechanisms Underlying the Early Life Programming of the Liver. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sohi, Gurjeev. “Molecular Mechanisms Underlying the Early Life Programming of the Liver.” 2013. Thesis, University of Western Ontario. Accessed January 21, 2020. https://ir.lib.uwo.ca/etd/1455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sohi, Gurjeev. “Molecular Mechanisms Underlying the Early Life Programming of the Liver.” 2013. Web. 21 Jan 2020.

Vancouver:

Sohi G. Molecular Mechanisms Underlying the Early Life Programming of the Liver. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2020 Jan 21]. Available from: https://ir.lib.uwo.ca/etd/1455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sohi G. Molecular Mechanisms Underlying the Early Life Programming of the Liver. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.