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You searched for subject:( Xenopus Proteins metabolism 60). Showing records 1 – 30 of 21335 total matches.

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1. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 14 Oct 2019.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

2. Harms, Paul William. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.

Degree: PhD, 2006, University of Alabama – Birmingham

Growth factor signals often regulate similar cellular processes both during embryogenesis and in adult homeostasis. Stringent control of these signals ensures proper embryonic development and… (more)

Subjects/Keywords: Homeodomain Proteins <; br>; Membrane Proteins  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Proteins <; br>; Signal Transduction  – physiology <; br>; Transcription Factors <; br>; Xenopus Proteins

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APA (6th Edition):

Harms, P. W. (2006). Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,369

Chicago Manual of Style (16th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,369.

MLA Handbook (7th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Web. 14 Oct 2019.

Vancouver:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369.

Council of Science Editors:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369

3. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

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APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 14 Oct 2019.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

4. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 14 Oct 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

5. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

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APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 14 Oct 2019.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

6. Marsh, Amie. Localization of proteins involved in trafficking in frog and mouse retina.

Degree: MS, 2012, University of Alabama – Birmingham

The connecting cilium serves as the major route for protein transport from the inner to outer segment of photoreceptor cells. The hypothesis that all Congenital… (more)

Subjects/Keywords: Cilia<; br>; Night Blindness<; br>; Protein Transport  – physiology<; br>; Rod Cell Outer Segment  – ultrastructure<; br>; Rhodopsin  – metabolism<; br>; Xenopus laevis

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APA (6th Edition):

Marsh, A. (2012). Localization of proteins involved in trafficking in frog and mouse retina. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1186

Chicago Manual of Style (16th Edition):

Marsh, Amie. “Localization of proteins involved in trafficking in frog and mouse retina.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1186.

MLA Handbook (7th Edition):

Marsh, Amie. “Localization of proteins involved in trafficking in frog and mouse retina.” 2012. Web. 14 Oct 2019.

Vancouver:

Marsh A. Localization of proteins involved in trafficking in frog and mouse retina. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1186.

Council of Science Editors:

Marsh A. Localization of proteins involved in trafficking in frog and mouse retina. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1186

7. Moore, Carlene Drucilla. The role of centaurin alpha-1 in the regulation of neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In the nervous system, PI 3-kinase has been implicated in neuronal differentiation. My studies have focused on a candidate neuronal PI 3-kinase target centaurin alpha-1,… (more)

Subjects/Keywords: 1-Phosphatidylinositol 3-Kinase <; br>; Adaptor Proteins, Signal Transducing  – metabolism <; br>; Dendrites  – metabolism <; br>; Dendrites  – ultrastructure <; br>; GTPase-Activating Proteins  – metabolism <; br>; Hippocampus  – cytology <; br>; Nerve Tissue Proteins  – metabolism <; br>; Neurons  – metabolism

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APA (6th Edition):

Moore, C. D. (2008). The role of centaurin alpha-1 in the regulation of neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,208

Chicago Manual of Style (16th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,208.

MLA Handbook (7th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Web. 14 Oct 2019.

Vancouver:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208.

Council of Science Editors:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208

8. Grabski, Robert. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.

Degree: PhD, 2008, University of Alabama – Birmingham

Metazoan cells are characterized with elaborate network of intracellular membranous compartments. These membranes allow the cell to spatially separate antagonistic processes and environments, and maintain… (more)

Subjects/Keywords: Carrier Proteins  – metabolism <; br>; Golgi Apparatus  – metabolism <; br>; Membrane Proteins  – metabolism <; br>; Protein Transport <; br>; RNA Interference

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APA (6th Edition):

Grabski, R. (2008). Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,328

Chicago Manual of Style (16th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,328.

MLA Handbook (7th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Web. 14 Oct 2019.

Vancouver:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328.

Council of Science Editors:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328

9. Cui, Wenjun. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.

Degree: PhD, 2010, University of Alabama – Birmingham

The unfolded protein response is one mechanism utilized by endoplasmic reticulum (ER) to maintain the homeostasis between ER protein folding machinery and ER proteins. UPR… (more)

Subjects/Keywords: Mitochondrial Membrane Transport Proteins  – chemistry<; br>; Mitochondrial Membrane Transport Proteins  – metabolism<; br>; Mitochondrial Membranes  – metabolism<; br>; Saccharomyces cerevisiae  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – chemistry<; br>; Saccharomyces cerevisiae Proteins  – metabolism<; br>; Unfolded Protein Response

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APA (6th Edition):

Cui, W. (2010). Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1088

Chicago Manual of Style (16th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1088.

MLA Handbook (7th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Web. 14 Oct 2019.

Vancouver:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088.

Council of Science Editors:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088

10. Indran, Sabarish Vellatheri. Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions.

Degree: PhD, 2010, University of Alabama – Birmingham

Human cytomegalovirus, a ubiquitous human pathogen, establishes a persistent infection in the infected host. HCMV assembly takes place in the nucleus and cytoplasm of infected… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing – physiology.<; br>; Cytomegalovirus<; br>; Cytoskeletal Proteins – physiology<; br>; Host-Pathogen Interactions.<; br>; Phosphoproteins – metabolism<; br>; Viral Matrix Proteins – metabolism.<; br>; Virus Assembly.<; br>; rab GTP-Binding Proteins – metabolism.

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APA (6th Edition):

Indran, S. V. (2010). Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1351

Chicago Manual of Style (16th Edition):

Indran, Sabarish Vellatheri. “Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1351.

MLA Handbook (7th Edition):

Indran, Sabarish Vellatheri. “Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions.” 2010. Web. 14 Oct 2019.

Vancouver:

Indran SV. Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1351.

Council of Science Editors:

Indran SV. Role of the human cytomegalovirus tegument protein pp150 in the trafficking and assembly of infectious virions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1351

11. Balasubramani, Anand. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.

Degree: PhD, 2010, University of Alabama – Birmingham

The ability to differentially manipulate available genetic information in order to generate diverse cellular identities represents an innovation of complex multicellular eukaryotic organisms. Cis-acting modules… (more)

Subjects/Keywords: DNA Replication – physiology.<; br>; Drosophila – metabolism.<; br>; Drosophila Proteins – metabolism.<; br>; GTP Phosphohydrolases – metabolism.<; br>; Microfilament Proteins – metabolism.<; br>; Multiprotein Complexes – metabolism.<; br>; Origin Recognition Complex – metabolism.

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APA (6th Edition):

Balasubramani, A. (2010). Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1428

Chicago Manual of Style (16th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1428.

MLA Handbook (7th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Web. 14 Oct 2019.

Vancouver:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428.

Council of Science Editors:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428

12. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 14 Oct 2019.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

13. Masyukova, Svetlana V. Analysis of NPHP complex genetic interactions associated with human cilia disorders.

Degree: PhD, 2011, University of Alabama – Birmingham

Primary cilia are antenna-like organelles that extend from the surface of almost all mammalian cell types. They regulate many signaling pathways and sense physical and… (more)

Subjects/Keywords: Caenorhabditis elegans Proteins – metabolism.<; br>; Cilia – metabolism.<; br>; Membrane Proteins – metabolism<; br>; Mutation, Missense – physiology.<; br>; Proteins – genetics<; br>; Proteins – metabolism

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APA (6th Edition):

Masyukova, S. V. (2011). Analysis of NPHP complex genetic interactions associated with human cilia disorders. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1352

Chicago Manual of Style (16th Edition):

Masyukova, Svetlana V. “Analysis of NPHP complex genetic interactions associated with human cilia disorders.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1352.

MLA Handbook (7th Edition):

Masyukova, Svetlana V. “Analysis of NPHP complex genetic interactions associated with human cilia disorders.” 2011. Web. 14 Oct 2019.

Vancouver:

Masyukova SV. Analysis of NPHP complex genetic interactions associated with human cilia disorders. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1352.

Council of Science Editors:

Masyukova SV. Analysis of NPHP complex genetic interactions associated with human cilia disorders. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1352

14. Silveira, Alexandra C. Characterization of SUDS3 as a BRMS1 family member in breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

BRMS1 and SUDS3 belong to a protein family characterized by the Sds3-like domain. These proteins are core members of SIN3-HDAC chromatin remodeling complexes and thus,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism <; br>; Carrier Proteins  – metabolism <; br>; Chromatin Assembly and Disassembly <; br>; Histone Deacetylases  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Repressor Proteins/metabolism

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APA (6th Edition):

Silveira, A. C. (2008). Characterization of SUDS3 as a BRMS1 family member in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,339

Chicago Manual of Style (16th Edition):

Silveira, Alexandra C. “Characterization of SUDS3 as a BRMS1 family member in breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,339.

MLA Handbook (7th Edition):

Silveira, Alexandra C. “Characterization of SUDS3 as a BRMS1 family member in breast cancer.” 2008. Web. 14 Oct 2019.

Vancouver:

Silveira AC. Characterization of SUDS3 as a BRMS1 family member in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,339.

Council of Science Editors:

Silveira AC. Characterization of SUDS3 as a BRMS1 family member in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,339

15. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 14 Oct 2019.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

16. Yu, Jei-Hwa. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.

Degree: PhD, 2008, University of Alabama – Birmingham

Papillomaviruses (PV) are prevalent pathogens that infect human or animal squamous epithelia. Its genome is a double strand circular DNA of approximately 7.9 kb. It… (more)

Subjects/Keywords: DNA Helicases  – metabolism <; br>; DNA-Binding Proteins  – metabolism <; br>; Human papillomavirus 11  – physiology <; br>; Mitogen-Activated Protein Kinases  – metabolism <; br>; Nuclear Localization Signals  – metabolism <; br>; Replication Origin <; br>; Viral Proteins  – metabolism

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APA (6th Edition):

Yu, J. (2008). MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,213

Chicago Manual of Style (16th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,213.

MLA Handbook (7th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Web. 14 Oct 2019.

Vancouver:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213.

Council of Science Editors:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213

17. Parish, Lindsay A. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.

Degree: PhD, 2009, University of Alabama – Birmingham

Malaria is a mosquito-borne infectious disease that is caused by parasites in the genus Plasmodium. There are four species of malaria that routinely infect humans,… (more)

Subjects/Keywords: Erythrocytes  – metabolism<; br>; Plasmodium falciparum  – metabolism<; br>; Protozoan Proteins  – genetics<; br>; Qa-SNARE Proteins  – genetics<; br>; Secretory Pathway  – genetics

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APA (6th Edition):

Parish, L. A. (2009). Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,503

Chicago Manual of Style (16th Edition):

Parish, Lindsay A. “Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,503.

MLA Handbook (7th Edition):

Parish, Lindsay A. “Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes.” 2009. Web. 14 Oct 2019.

Vancouver:

Parish LA. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,503.

Council of Science Editors:

Parish LA. Protein trafficking and 4.1R relocalization in Plasmodium falciparum-infected erythrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,503

18. Qadri, Yawar J. Small molecule inhibitors of acid sensing ion channel-1.

Degree: PhD, 2009, University of Alabama – Birmingham

Acid Sensing Ion Channel 1 is one of the many proteins in the Epithelial Sodium Channel/Degenerin family. The proteins in this family interact to form… (more)

Subjects/Keywords: Amiloride  – pharmacology<; br>; Nerve Tissue Proteins  – chemistry<; br>; Nerve Tissue Proteins  – metabolism<; br>; Sodium Channels  – chemistry<; br>; Spider Venoms  – metabolism

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APA (6th Edition):

Qadri, Y. J. (2009). Small molecule inhibitors of acid sensing ion channel-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1174

Chicago Manual of Style (16th Edition):

Qadri, Yawar J. “Small molecule inhibitors of acid sensing ion channel-1.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1174.

MLA Handbook (7th Edition):

Qadri, Yawar J. “Small molecule inhibitors of acid sensing ion channel-1.” 2009. Web. 14 Oct 2019.

Vancouver:

Qadri YJ. Small molecule inhibitors of acid sensing ion channel-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1174.

Council of Science Editors:

Qadri YJ. Small molecule inhibitors of acid sensing ion channel-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1174

19. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 14 Oct 2019.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

20. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 14 Oct 2019.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

21. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 14 Oct 2019.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

22. Williams, Corey L. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.

Degree: PhD, 2009, University of Alabama – Birmingham

Cilia are evolutionarily conserved, membrane-bound, microtubule-based organelles found on a diverse array of cell types in eukaryotic organisms. Inherited diseases of cilia protein dysfunction include… (more)

Subjects/Keywords: Caenorhabditis elegans  – cytology<; br>; Caenorhabditis elegans  – metabolism<; br>; Caenorhabditis elegans Proteins  – metabolism<; br>; Cilia  – metabolism<; br>; Protein Transport

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APA (6th Edition):

Williams, C. L. (2009). Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,407

Chicago Manual of Style (16th Edition):

Williams, Corey L. “Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,407.

MLA Handbook (7th Edition):

Williams, Corey L. “Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.” 2009. Web. 14 Oct 2019.

Vancouver:

Williams CL. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,407.

Council of Science Editors:

Williams CL. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,407

23. Mans, Keri April. Translocation and function of Akt in the mitochondria.

Degree: PhD, 2010, University of Alabama – Birmingham

The ubiquitously expressed kinase Akt is a known survival protein, and is involved in multiple cell signaling cascades, notably the phosphatidylinositol 3-kinase (PI3K) pathway. Active… (more)

Subjects/Keywords: HSP90 Heat-Shock Proteins  – metabolism<; br>; Mitochondria  – metabolism<; br>; Oncogene Protein v-akt  – metabolism<; br>; Phosphatidylinositol 3-Kinase<; br>; Postmortem Changes

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APA (6th Edition):

Mans, K. A. (2010). Translocation and function of Akt in the mitochondria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,831

Chicago Manual of Style (16th Edition):

Mans, Keri April. “Translocation and function of Akt in the mitochondria.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,831.

MLA Handbook (7th Edition):

Mans, Keri April. “Translocation and function of Akt in the mitochondria.” 2010. Web. 14 Oct 2019.

Vancouver:

Mans KA. Translocation and function of Akt in the mitochondria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,831.

Council of Science Editors:

Mans KA. Translocation and function of Akt in the mitochondria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,831

24. Krzyzaniak, Magdalena Anna. Role of the gM/gN glycoprotein complex in the final assembly and egress of the human cytomegalovirus (HCMV).

Degree: PhD, 2008, University of Alabama – Birmingham

HCMV consists of a dsDNA genome enclosed by, an icosahedral capsid surrounded by a layer of tegument proteins; the virion structure is enclosed in a… (more)

Subjects/Keywords: Cytomegalovirus  – physiology <; br>; Glycoproteins  – metabolism <; br>; Protein Sorting Signals <; br>; Viral Envelope Proteins  – metabolism <; br>; Virus Assembly <; br>; Virus Replication

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APA (6th Edition):

Krzyzaniak, M. A. (2008). Role of the gM/gN glycoprotein complex in the final assembly and egress of the human cytomegalovirus (HCMV). (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,231

Chicago Manual of Style (16th Edition):

Krzyzaniak, Magdalena Anna. “Role of the gM/gN glycoprotein complex in the final assembly and egress of the human cytomegalovirus (HCMV).” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,231.

MLA Handbook (7th Edition):

Krzyzaniak, Magdalena Anna. “Role of the gM/gN glycoprotein complex in the final assembly and egress of the human cytomegalovirus (HCMV).” 2008. Web. 14 Oct 2019.

Vancouver:

Krzyzaniak MA. Role of the gM/gN glycoprotein complex in the final assembly and egress of the human cytomegalovirus (HCMV). [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,231.

Council of Science Editors:

Krzyzaniak MA. Role of the gM/gN glycoprotein complex in the final assembly and egress of the human cytomegalovirus (HCMV). [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,231

25. Dolan, Philip J. The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau.

Degree: PhD, 2010, University of Alabama – Birmingham

Alzheimer Disease (AD) is pathologically characterized by the appearance of senile plaques composed of β-amyloid (Aβ) and neurofibrillary tangles composed of the microtubule-associated protein tau.… (more)

Subjects/Keywords: Alzheimer Disease  – enzymology<; br>; Autophagy<; br>; Caspases  – metabolism<; br>; Protein Kinases  – metabolism<; br>; Signal Transduction<; br>; tau Proteins  – physiology

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APA (6th Edition):

Dolan, P. J. (2010). The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1090

Chicago Manual of Style (16th Edition):

Dolan, Philip J. “The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1090.

MLA Handbook (7th Edition):

Dolan, Philip J. “The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau.” 2010. Web. 14 Oct 2019.

Vancouver:

Dolan PJ. The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1090.

Council of Science Editors:

Dolan PJ. The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1090

26. Carter, Robert J. (Robert Joseph). HPV DNA partitioning during mitosis as followed by fluorescence microscopy.

Degree: PhD, 2010, University of Alabama – Birmingham

Human papillomaviruses (HPVs) are small, double-stranded deoxyribonucleic acid (DNA) tumor viruses capable of establishing persistent infections in the epithelia. After infecting actively-dividing basal cells, the… (more)

Subjects/Keywords: DNA-Binding Proteins<; br>; DNA, Viral  – metabolism<; br>; Microscopy, Fluorescence<; br>; Mitosis<; br>; Papillomaviridae  – metabolism<; br>; Plasmids

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APA (6th Edition):

Carter, R. J. (. J. (2010). HPV DNA partitioning during mitosis as followed by fluorescence microscopy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1253

Chicago Manual of Style (16th Edition):

Carter, Robert J (Robert Joseph). “HPV DNA partitioning during mitosis as followed by fluorescence microscopy.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1253.

MLA Handbook (7th Edition):

Carter, Robert J (Robert Joseph). “HPV DNA partitioning during mitosis as followed by fluorescence microscopy.” 2010. Web. 14 Oct 2019.

Vancouver:

Carter RJ(J. HPV DNA partitioning during mitosis as followed by fluorescence microscopy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1253.

Council of Science Editors:

Carter RJ(J. HPV DNA partitioning during mitosis as followed by fluorescence microscopy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1253

27. Genovese, Nicholas J. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.

Degree: PhD, 2010, University of Alabama – Birmingham

Though human papillomavirus infection of the human epidermis is epidemiologically widespread and typically benign, manipulation of the cell cycle within host tissues during infections can… (more)

Subjects/Keywords: Cell Cycle<; br>; Cell Transformation, Viral<; br>; Human papillomavirus 16  – metabolism<; br>; Keratinocytes<; br>; Oncogene Proteins, Viral  – metabolism<; br>; Papillomaviridae  – physiology<; br>; Papillomavirus E7 Proteins  – metabolism<; br>; Receptors, Estrogen  – metabolism<; br>; Retinoblastoma-Like Protein p130  – metabolism<; br>; S Phase

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APA (6th Edition):

Genovese, N. J. (2010). The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1158

Chicago Manual of Style (16th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1158.

MLA Handbook (7th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Web. 14 Oct 2019.

Vancouver:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158.

Council of Science Editors:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158

28. Hertz, Marla (Marla Ilene). In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.

Degree: PhD, 2011, University of Alabama – Birmingham

Translation of the majority of eukaryotic mRNAs is initiated upon recognition of its 5′ cap structure by translation initiation factors in so-called cap-dependent translation. Capdependent… (more)

Subjects/Keywords: Dicistroviridae  – metabolism<; br>; Gene Expression Regulation<; br>; Hepacivirus  – metabolism<; br>; Prostatic Neoplasms<; br>; Protein Biosynthesis<; br>; Ribosomal Proteins  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – metabolism

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APA (6th Edition):

Hertz, M. (. I. (2011). In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,953

Chicago Manual of Style (16th Edition):

Hertz, Marla (Marla Ilene). “In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,953.

MLA Handbook (7th Edition):

Hertz, Marla (Marla Ilene). “In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.” 2011. Web. 14 Oct 2019.

Vancouver:

Hertz M(I. In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,953.

Council of Science Editors:

Hertz M(I. In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,953

29. Maitra, Sushmit. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulated mRNA decay is a highly important process for the tight control of gene expression. Inherently unstable mRNAs contain AU-rich elements (AREs) in the 3’… (more)

Subjects/Keywords: Intracellular Signaling Peptides and Proteins  – metabolism <; br>; Protein-Serine-Threonine Kinases  – metabolism <; br>; RNA, Messenger  – metabolism <; br>; RNA Stability  – physiology <; br>; RNA-Binding Proteins  – metabolism <; br>; TATA-Binding Protein Associated Factors  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Maitra, S. (2008). The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,379

Chicago Manual of Style (16th Edition):

Maitra, Sushmit. “The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,379.

MLA Handbook (7th Edition):

Maitra, Sushmit. “The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.” 2008. Web. 14 Oct 2019.

Vancouver:

Maitra S. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,379.

Council of Science Editors:

Maitra S. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,379

30. Byon, Chang Hyun. Oxidative stress-stimulated vascular calcification.

Degree: PhD, 2009, University of Alabama – Birmingham

Oxidative stress plays a critical role in pathogenesis of atherosclerosis. However, the effect of oxidative stress-induced molecular signaling in development of vascular calcification, a feature… (more)

Subjects/Keywords: Calcinosis  – metabolism<; br>; Core Binding Factor Alpha 1 Subunit  – metabolism<; br>; Hydrogen Peroxide  – metabolism<; br>; Muscle, smooth, Vascular  – metabolism<; br>; Oxidative Stress<; br>; Proto-Oncogene Proteins c-akt  – metabolism<; br>; Signal Transduction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Byon, C. H. (2009). Oxidative stress-stimulated vascular calcification. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,662

Chicago Manual of Style (16th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 14, 2019. http://contentdm.mhsl.uab.edu/u?/etd,662.

MLA Handbook (7th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Web. 14 Oct 2019.

Vancouver:

Byon CH. Oxidative stress-stimulated vascular calcification. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 14]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662.

Council of Science Editors:

Byon CH. Oxidative stress-stimulated vascular calcification. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662

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