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You searched for subject:( Ubiquitin Thiolesterase metabolism 60). Showing records 1 – 30 of 13039 total matches.

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1. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 04 Apr 2020.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

2. Crimmins, Stephen Lewis. Characterization and functional analysis of Usp14.

Degree: PhD, 2007, University of Alabama – Birmingham

The ubiquitin proteasome system (UPS) is essential for regulated protein degrada-tion, a requirement for numerous neuronal process, including vesicle cycling, neuro-transmitter release, spine morphology, and… (more)

Subjects/Keywords: Ataxia  – enzymology <; br>; Ataxia  – genetics <; br>; Gene Expression Regulation, Enzymologic  – physiology <; br>; Neurons  – enzymology <; br>; Ubiquitin  – metabolism <; br>; Ubiquitin Thiolesterase  – biosynthesis <; br>; Ubiquitin Thiolesterase  – genetics

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APA (6th Edition):

Crimmins, S. L. (2007). Characterization and functional analysis of Usp14. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,118

Chicago Manual of Style (16th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,118.

MLA Handbook (7th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Web. 04 Apr 2020.

Vancouver:

Crimmins SL. Characterization and functional analysis of Usp14. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118.

Council of Science Editors:

Crimmins SL. Characterization and functional analysis of Usp14. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118

3. Walters, John Brandon. USP14: a link between the proteasome and synaptic function.

Degree: PhD, 2010, University of Alabama – Birmingham

The ubiquitin proteasome system (UPS) is a coordinated process by which the cell can control protein distribution and abundance. Proteins are marked for turnover by… (more)

Subjects/Keywords: Neuronal Plasticity  – physiology<; br>; Proteasome Endopeptidase Complex  – metabolism<; br>; Synapses  – metabolism<; br>; Synaptic Transmission  – physiology<; br>; Ubiquitin  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism

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APA (6th Edition):

Walters, J. B. (2010). USP14: a link between the proteasome and synaptic function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1114

Chicago Manual of Style (16th Edition):

Walters, John Brandon. “USP14: a link between the proteasome and synaptic function.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1114.

MLA Handbook (7th Edition):

Walters, John Brandon. “USP14: a link between the proteasome and synaptic function.” 2010. Web. 04 Apr 2020.

Vancouver:

Walters JB. USP14: a link between the proteasome and synaptic function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1114.

Council of Science Editors:

Walters JB. USP14: a link between the proteasome and synaptic function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1114

4. Chen, Ping-Chung. The role of USP14 in regulating synaptic development and function of the neuromuscular junctions.

Degree: PhD, 2010, University of Alabama – Birmingham

In the nervous system, the ubiquitin proteasome system (UPS) plays critical roles in regulating a wide diversity of cellular process such as synaptic transmission, axon… (more)

Subjects/Keywords: Neuromuscular Junction  – enzymology<; br>; Neuromuscular Junction  – growth & development<; br>; Proteasome Endopeptidase Complex  – metabolism<; br>; Synapses  – enzymology<; br>; Ubiquitin  – metabolism<; br>; Ubiquitin Thiolesterase  – physiology

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APA (6th Edition):

Chen, P. (2010). The role of USP14 in regulating synaptic development and function of the neuromuscular junctions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,992

Chicago Manual of Style (16th Edition):

Chen, Ping-Chung. “The role of USP14 in regulating synaptic development and function of the neuromuscular junctions.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,992.

MLA Handbook (7th Edition):

Chen, Ping-Chung. “The role of USP14 in regulating synaptic development and function of the neuromuscular junctions.” 2010. Web. 04 Apr 2020.

Vancouver:

Chen P. The role of USP14 in regulating synaptic development and function of the neuromuscular junctions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,992.

Council of Science Editors:

Chen P. The role of USP14 in regulating synaptic development and function of the neuromuscular junctions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,992

5. Brooks, William Samuel. Localization and function of G2E3.

Degree: PhD, 2007, University of Alabama – Birmingham

A microarray study was performed which identified G2E3 as a novel, putative ubiquitin ligase that was both G2/M-specific in expression and down regulated at the… (more)

Subjects/Keywords: Cell Nucleus  – metabolism <; br>; DNA Damage  – physiology <; br>; Hela Cells <; br>; Nuclear Localization Signals  – analysis <; br>; Ubiquitin-Protein Ligases  – metabolism <; br>; Ubiquitin-Protein Ligases  – physiology

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APA (6th Edition):

Brooks, W. S. (2007). Localization and function of G2E3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,175

Chicago Manual of Style (16th Edition):

Brooks, William Samuel. “Localization and function of G2E3.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,175.

MLA Handbook (7th Edition):

Brooks, William Samuel. “Localization and function of G2E3.” 2007. Web. 04 Apr 2020.

Vancouver:

Brooks WS. Localization and function of G2E3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,175.

Council of Science Editors:

Brooks WS. Localization and function of G2E3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,175


University of Hong Kong

6. 王琳; Wang, Lin. The role of MDM2 in hepatic lipid metabolism.

Degree: Master of Medical Sciences, 2016, University of Hong Kong

 Background and objective: Liver is a core organ in regulation of lipid homeostasis, which is vital for life and health. Obesity is closely associated with… (more)

Subjects/Keywords: Ubiquitin; Lipids - Metabolism

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APA (6th Edition):

王琳; Wang, L. (2016). The role of MDM2 in hepatic lipid metabolism. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/236279

Chicago Manual of Style (16th Edition):

王琳; Wang, Lin. “The role of MDM2 in hepatic lipid metabolism.” 2016. Masters Thesis, University of Hong Kong. Accessed April 04, 2020. http://hdl.handle.net/10722/236279.

MLA Handbook (7th Edition):

王琳; Wang, Lin. “The role of MDM2 in hepatic lipid metabolism.” 2016. Web. 04 Apr 2020.

Vancouver:

王琳; Wang L. The role of MDM2 in hepatic lipid metabolism. [Internet] [Masters thesis]. University of Hong Kong; 2016. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10722/236279.

Council of Science Editors:

王琳; Wang L. The role of MDM2 in hepatic lipid metabolism. [Masters Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/236279


University of Texas Southwestern Medical Center

7. Myers, Kalisa Galina. UCHLI in the Mammalian Nervous System.

Degree: 2008, University of Texas Southwestern Medical Center

 UCHL1, or Ubiquitin Carboxy Terminal Hydrolase L1, is a de-ubiquitinating enzyme (DUB) thought to be involved in ubiquitin recycling. Beyond this, its natural function is… (more)

Subjects/Keywords: Ubiquitin Thiolesterase; Central Nervous System; Nerve Degeneration

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APA (6th Edition):

Myers, K. G. (2008). UCHLI in the Mammalian Nervous System. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Myers, Kalisa Galina. “UCHLI in the Mammalian Nervous System.” 2008. Thesis, University of Texas Southwestern Medical Center. Accessed April 04, 2020. http://hdl.handle.net/2152.5/403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Myers, Kalisa Galina. “UCHLI in the Mammalian Nervous System.” 2008. Web. 04 Apr 2020.

Vancouver:

Myers KG. UCHLI in the Mammalian Nervous System. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2008. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/2152.5/403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Myers KG. UCHLI in the Mammalian Nervous System. [Thesis]. University of Texas Southwestern Medical Center; 2008. Available from: http://hdl.handle.net/2152.5/403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

8. Hu, Wenbao. TRIP-Br1 mediates degradation of multiple adenylyl cyclase isoforms by XIAP E3 ligase.

Degree: 2015, Hong Kong University of Science and Technology

 Adenylyl cyclases (ACs) catalyze conversion of ATP to cAMP, a second messenger of utmost importance that regulates a vast array of biological processes in all… (more)

Subjects/Keywords: Adenylate cyclase ; Proteins ; Metabolism ; Ubiquitin ; Ligases

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APA (6th Edition):

Hu, W. (2015). TRIP-Br1 mediates degradation of multiple adenylyl cyclase isoforms by XIAP E3 ligase. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-80226 ; https://doi.org/10.14711/thesis-b1514880 ; http://repository.ust.hk/ir/bitstream/1783.1-80226/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Wenbao. “TRIP-Br1 mediates degradation of multiple adenylyl cyclase isoforms by XIAP E3 ligase.” 2015. Thesis, Hong Kong University of Science and Technology. Accessed April 04, 2020. http://repository.ust.hk/ir/Record/1783.1-80226 ; https://doi.org/10.14711/thesis-b1514880 ; http://repository.ust.hk/ir/bitstream/1783.1-80226/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Wenbao. “TRIP-Br1 mediates degradation of multiple adenylyl cyclase isoforms by XIAP E3 ligase.” 2015. Web. 04 Apr 2020.

Vancouver:

Hu W. TRIP-Br1 mediates degradation of multiple adenylyl cyclase isoforms by XIAP E3 ligase. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2015. [cited 2020 Apr 04]. Available from: http://repository.ust.hk/ir/Record/1783.1-80226 ; https://doi.org/10.14711/thesis-b1514880 ; http://repository.ust.hk/ir/bitstream/1783.1-80226/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu W. TRIP-Br1 mediates degradation of multiple adenylyl cyclase isoforms by XIAP E3 ligase. [Thesis]. Hong Kong University of Science and Technology; 2015. Available from: http://repository.ust.hk/ir/Record/1783.1-80226 ; https://doi.org/10.14711/thesis-b1514880 ; http://repository.ust.hk/ir/bitstream/1783.1-80226/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 04 Apr 2020.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956

10. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 04 Apr 2020.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

11. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

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APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 04 Apr 2020.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

12. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 04 Apr 2020.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

13. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

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APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 04 Apr 2020.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

14. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 04 Apr 2020.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

15. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 04 Apr 2020.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

16. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

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APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 04 Apr 2020.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

17. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

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APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 04 Apr 2020.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

18. Durham, Carolyn G. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.

Degree: PhD, 2010, University of Alabama – Birmingham

The inverse correlation between the industrialization and disease prevalence is termed the "hygiene hypothesis." Supporting this, immunological studies show Th1 cytokines modulate Th2 immune responses.… (more)

Subjects/Keywords: Asthma<; br>; Gastric Mucosa  – metabolism<; br>; Gastritis<; br>; Helicobacter Infections  – metabolism<; br>; Helicobacter felis<; br>; Mucus  – metabolism

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APA (6th Edition):

Durham, C. G. (2010). Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,707

Chicago Manual of Style (16th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,707.

MLA Handbook (7th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Web. 04 Apr 2020.

Vancouver:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707.

Council of Science Editors:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707

19. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 04 Apr 2020.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

20. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 04 Apr 2020.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

21. 加藤, 智弘. ラット顎下腺主導管における刷子細胞に関する免疫組織化学的研究.

Degree: 博士(歯学), 2016, Kanagawa Dental University / 神奈川歯科大学

光学顕微鏡レベルにおいてラット顎下腺主導管にみられる刷子細胞を多列上皮細胞より区別して標識可能とするため、マーカー候補のProtein Gene Product(PGP)9.5、血管運動腸管ペプチド(VIP)、サイトケラチン(CK)18、ニューロフィラメント(NF)に対する抗体を用い、免疫組織化学的に二重標識法を行った。その結果、CK18に対する免疫陽性反応は全ての上皮細胞に観察されたが、PGP9.5、VIP、NFは粘膜上皮において少数の細胞にのみ陽性反応を認めた。この陽性細胞を免疫電顕法により観察したところ、刷子細胞であることが確認され、これらが刷子細胞の特異的な標識マーカーとして有用と考えられた。また刷子細胞が示す形態学的な特徴は舌の味蕾の細胞にみられるIII型細胞に類似しており、この細胞はPGP9.5およびVIPに対して陽性を示すことより、顎下腺の刷子細胞は味蕾細胞と同様に化学受容細胞であることが推察された。

2014

Subjects/Keywords: *顎下腺; 顕微鏡検査法; 電子顕微鏡検査法; *上皮細胞; *免疫組織化学; Wistarラット; Ubiquitin Thiolesterase; Keratin-18; *刷子細胞; ラット; 動物; オス; 歯学

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APA (6th Edition):

加藤, . (2016). ラット顎下腺主導管における刷子細胞に関する免疫組織化学的研究. (Thesis). Kanagawa Dental University / 神奈川歯科大学. Retrieved from http://id.nii.ac.jp/1421/00000012/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

加藤, 智弘. “ラット顎下腺主導管における刷子細胞に関する免疫組織化学的研究.” 2016. Thesis, Kanagawa Dental University / 神奈川歯科大学. Accessed April 04, 2020. http://id.nii.ac.jp/1421/00000012/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

加藤, 智弘. “ラット顎下腺主導管における刷子細胞に関する免疫組織化学的研究.” 2016. Web. 04 Apr 2020.

Vancouver:

加藤 . ラット顎下腺主導管における刷子細胞に関する免疫組織化学的研究. [Internet] [Thesis]. Kanagawa Dental University / 神奈川歯科大学; 2016. [cited 2020 Apr 04]. Available from: http://id.nii.ac.jp/1421/00000012/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

加藤 . ラット顎下腺主導管における刷子細胞に関する免疫組織化学的研究. [Thesis]. Kanagawa Dental University / 神奈川歯科大学; 2016. Available from: http://id.nii.ac.jp/1421/00000012/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 04 Apr 2020.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

23. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

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APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 04 Apr 2020.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

24. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

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APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 04 Apr 2020.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194

25. Moore, Carlene Drucilla. The role of centaurin alpha-1 in the regulation of neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In the nervous system, PI 3-kinase has been implicated in neuronal differentiation. My studies have focused on a candidate neuronal PI 3-kinase target centaurin alpha-1,… (more)

Subjects/Keywords: 1-Phosphatidylinositol 3-Kinase <; br>; Adaptor Proteins, Signal Transducing  – metabolism <; br>; Dendrites  – metabolism <; br>; Dendrites  – ultrastructure <; br>; GTPase-Activating Proteins  – metabolism <; br>; Hippocampus  – cytology <; br>; Nerve Tissue Proteins  – metabolism <; br>; Neurons  – metabolism

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APA (6th Edition):

Moore, C. D. (2008). The role of centaurin alpha-1 in the regulation of neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,208

Chicago Manual of Style (16th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,208.

MLA Handbook (7th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Web. 04 Apr 2020.

Vancouver:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208.

Council of Science Editors:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208

26. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 04 Apr 2020.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

27. Mavalli, Mahendra D. Mechanisms of growth hormone action in skeletal muscle.

Degree: PhD, 2009, University of Alabama – Birmingham

Growth hormone (GH) and insulin like growth factor-1 (IGF-1) exert profound growth promoting actions during pre and postnatal skeletal muscle development. GH and IGF-1 seem… (more)

Subjects/Keywords: Growth Hormone  – metabolism<; br>; Insulin  – metabolism<; br>; Insulin Resistance<; br>; Muscle, Skeletal  – growth & development<; br>; Muscle, Skeletal  – metabolism<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptors, Somatotropin  – metabolism<; br>; Sarcopenia

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APA (6th Edition):

Mavalli, M. D. (2009). Mechanisms of growth hormone action in skeletal muscle. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1103

Chicago Manual of Style (16th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1103.

MLA Handbook (7th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Web. 04 Apr 2020.

Vancouver:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103.

Council of Science Editors:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103

28. Tang, Yi, Ph.D. The role of transforming growth factor beta-1 in bone remodeling.

Degree: PhD, 2009, University of Alabama – Birmingham

Bone remodeling depends on the precise coordination of bone resorption by the osteoclasts and bone formation by the osteoblasts. It is proposed that osteoclastic bone… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Bone Regeneration  – drug effects<; br>; Cadherins  – metabolism<; br>; Cell Membrane  – metabolism<; br>; Multiprotein Complexes  – metabolism<; br>; Smad7 Protein  – metabolism<; br>; Transforming Growth Factor beta  – metabolism

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APA (6th Edition):

Tang, Yi, P. D. (2009). The role of transforming growth factor beta-1 in bone remodeling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,405

Chicago Manual of Style (16th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,405.

MLA Handbook (7th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Web. 04 Apr 2020.

Vancouver:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405.

Council of Science Editors:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405

29. Balasubramani, Anand. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.

Degree: PhD, 2010, University of Alabama – Birmingham

The ability to differentially manipulate available genetic information in order to generate diverse cellular identities represents an innovation of complex multicellular eukaryotic organisms. Cis-acting modules… (more)

Subjects/Keywords: DNA Replication – physiology.<; br>; Drosophila – metabolism.<; br>; Drosophila Proteins – metabolism.<; br>; GTP Phosphohydrolases – metabolism.<; br>; Microfilament Proteins – metabolism.<; br>; Multiprotein Complexes – metabolism.<; br>; Origin Recognition Complex – metabolism.

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APA (6th Edition):

Balasubramani, A. (2010). Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1428

Chicago Manual of Style (16th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 04, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1428.

MLA Handbook (7th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Web. 04 Apr 2020.

Vancouver:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 04]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428.

Council of Science Editors:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428


University of Western Australia

30. Jaeger, Werner Christian. Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling.

Degree: PhD, 2011, University of Western Australia

[Truncated abstract] The orexin endocrine system has been associated with a range of physiological functions including regulation of sleep and wake states, energy metabolism, addictive… (more)

Subjects/Keywords: Energy metabolism; Sleep/wake; Orexin; GPCR; β-arrestin; BRET; Ubiquitin; MAPK

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APA (6th Edition):

Jaeger, W. C. (2011). Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Jaeger, Werner Christian. “Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling.” 2011. Doctoral Dissertation, University of Western Australia. Accessed April 04, 2020. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Jaeger, Werner Christian. “Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling.” 2011. Web. 04 Apr 2020.

Vancouver:

Jaeger WC. Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling. [Internet] [Doctoral dissertation]. University of Western Australia; 2011. [cited 2020 Apr 04]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01.

Council of Science Editors:

Jaeger WC. Biophysical insights into orexin receptor-β-arrestin complexes and their role in cellular signalling. [Doctoral Dissertation]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=32908&local_base=GEN01-INS01

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