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You searched for subject:( Tumor Suppressor Proteins). Showing records 1 – 30 of 13190 total matches.

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University of Hong Kong

1. Lo, Kwok-pui. Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies.

Degree: Master of Medical Sciences, 2012, University of Hong Kong

NK cell lymphoma is one of the cellular malignancies that arise from lymphocytes. Due to its rarity and aggressiveness the detailed molecular pathogenesis of NK… (more)

Subjects/Keywords: Lymphomas - Pathogenesis.; Tumor suppressor proteins.

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APA (6th Edition):

Lo, K. (2012). Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. (Masters Thesis). University of Hong Kong. Retrieved from Lo, K. [盧國培]. (2012). Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4842138 ; http://dx.doi.org/10.5353/th_b4842138 ; http://hdl.handle.net/10722/177294

Chicago Manual of Style (16th Edition):

Lo, Kwok-pui. “Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies.” 2012. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. Lo, K. [盧國培]. (2012). Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4842138 ; http://dx.doi.org/10.5353/th_b4842138 ; http://hdl.handle.net/10722/177294.

MLA Handbook (7th Edition):

Lo, Kwok-pui. “Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies.” 2012. Web. 13 Nov 2019.

Vancouver:

Lo K. Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. [Internet] [Masters thesis]. University of Hong Kong; 2012. [cited 2019 Nov 13]. Available from: Lo, K. [盧國培]. (2012). Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4842138 ; http://dx.doi.org/10.5353/th_b4842138 ; http://hdl.handle.net/10722/177294.

Council of Science Editors:

Lo K. Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. [Masters Thesis]. University of Hong Kong; 2012. Available from: Lo, K. [盧國培]. (2012). Tumor suppressive role of the α-isoform of transcriptional repressor PRDM1 in the pathogenesis of NK-cell malignancies. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4842138 ; http://dx.doi.org/10.5353/th_b4842138 ; http://hdl.handle.net/10722/177294


Hong Kong University of Science and Technology

2. Zuo, Hao LIFS. Regulation of the tumor suppressor Fhit by activated Gα subunits.

Degree: 2013, Hong Kong University of Science and Technology

 The FHIT tumor suppressor gene is arguably the most commonly altered gene in cancer since it is inactivated in about 60% of human tumors. Despite… (more)

Subjects/Keywords: Antioncogenes; Tumor suppressor proteins; G proteins; Receptors

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APA (6th Edition):

Zuo, H. L. (2013). Regulation of the tumor suppressor Fhit by activated Gα subunits. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zuo, Hao LIFS. “Regulation of the tumor suppressor Fhit by activated Gα subunits.” 2013. Thesis, Hong Kong University of Science and Technology. Accessed November 13, 2019. https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zuo, Hao LIFS. “Regulation of the tumor suppressor Fhit by activated Gα subunits.” 2013. Web. 13 Nov 2019.

Vancouver:

Zuo HL. Regulation of the tumor suppressor Fhit by activated Gα subunits. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2013. [cited 2019 Nov 13]. Available from: https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zuo HL. Regulation of the tumor suppressor Fhit by activated Gα subunits. [Thesis]. Hong Kong University of Science and Technology; 2013. Available from: https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

3. Kan, Pei-qi, Rebecca. Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma.

Degree: PhD, 2016, University of Hong Kong

 Nasopharyngeal carcinoma, poised as one of the top ten most common cancers in Hong Kong, displays a disproportionate clustering in specific ethnic groups and demonstrates… (more)

Subjects/Keywords: Nasopharynx - Cancer - Pathogenesis; Tumor suppressor proteins

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APA (6th Edition):

Kan, Pei-qi, R. (2016). Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Kan, P. R. [簡珮琪]. (2016). Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774068. ; http://dx.doi.org/10.5353/th_b5774068 ; http://hdl.handle.net/10722/238188

Chicago Manual of Style (16th Edition):

Kan, Pei-qi, Rebecca. “Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed November 13, 2019. Kan, P. R. [簡珮琪]. (2016). Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774068. ; http://dx.doi.org/10.5353/th_b5774068 ; http://hdl.handle.net/10722/238188.

MLA Handbook (7th Edition):

Kan, Pei-qi, Rebecca. “Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma.” 2016. Web. 13 Nov 2019.

Vancouver:

Kan, Pei-qi R. Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Nov 13]. Available from: Kan, P. R. [簡珮琪]. (2016). Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774068. ; http://dx.doi.org/10.5353/th_b5774068 ; http://hdl.handle.net/10722/238188.

Council of Science Editors:

Kan, Pei-qi R. Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: Kan, P. R. [簡珮琪]. (2016). Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5774068. ; http://dx.doi.org/10.5353/th_b5774068 ; http://hdl.handle.net/10722/238188


University of Hong Kong

4. Cheung, Yuk-lam. The role of P53 in nasal-type extranodal NK/T-cell lymphomas in Hong Kong patients.

Degree: Master of Medical Sciences, 2017, University of Hong Kong

Extranodal NK/T-cell lymphomas (ENKTCL) are the cancers originated from natural killer cells (NK cells) or T cells. ENKTCL are subdivided into two groups, the nasal-type… (more)

Subjects/Keywords: Lymphomas - Pathogenesis; p53 protein; Tumor suppressor proteins

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APA (6th Edition):

Cheung, Y. (2017). The role of P53 in nasal-type extranodal NK/T-cell lymphomas in Hong Kong patients. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/251317

Chicago Manual of Style (16th Edition):

Cheung, Yuk-lam. “The role of P53 in nasal-type extranodal NK/T-cell lymphomas in Hong Kong patients.” 2017. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. http://hdl.handle.net/10722/251317.

MLA Handbook (7th Edition):

Cheung, Yuk-lam. “The role of P53 in nasal-type extranodal NK/T-cell lymphomas in Hong Kong patients.” 2017. Web. 13 Nov 2019.

Vancouver:

Cheung Y. The role of P53 in nasal-type extranodal NK/T-cell lymphomas in Hong Kong patients. [Internet] [Masters thesis]. University of Hong Kong; 2017. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10722/251317.

Council of Science Editors:

Cheung Y. The role of P53 in nasal-type extranodal NK/T-cell lymphomas in Hong Kong patients. [Masters Thesis]. University of Hong Kong; 2017. Available from: http://hdl.handle.net/10722/251317


University of Hong Kong

5. Hung, Wing-yan. The role of TAX1BP2 in hepatocellular carcinoma.

Degree: M. Phil., 2012, University of Hong Kong

 TAX1 Binding Protein 2 (TAX1BP2) has been found to be a centrosome duplication regulating protein. Previous findings have demonstrated that over-expression of TAX1BP2 suppresses centrosome… (more)

Subjects/Keywords: Tumor suppressor proteins; Liver - Cancer - Molecular aspects

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APA (6th Edition):

Hung, W. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070

Chicago Manual of Style (16th Edition):

Hung, Wing-yan. “The role of TAX1BP2 in hepatocellular carcinoma.” 2012. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070.

MLA Handbook (7th Edition):

Hung, Wing-yan. “The role of TAX1BP2 in hepatocellular carcinoma.” 2012. Web. 13 Nov 2019.

Vancouver:

Hung W. The role of TAX1BP2 in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2012. [cited 2019 Nov 13]. Available from: Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070.

Council of Science Editors:

Hung W. The role of TAX1BP2 in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2012. Available from: Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070


Hong Kong University of Science and Technology

6. Li, Yuanjun. Characterization of metastasis suppressor Nm23-H1 : the regulation mechanism and structure-function studies.

Degree: 2016, Hong Kong University of Science and Technology

 The Nm23 metastasis suppressor family is involved in diverse physiological and pathological processes including tumorigenesis and metastasis. Although the inverse correlation of Nm23 level with… (more)

Subjects/Keywords: Tumor suppressor proteins; Protein-protein interactions; G proteins

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APA (6th Edition):

Li, Y. (2016). Characterization of metastasis suppressor Nm23-H1 : the regulation mechanism and structure-function studies. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b1731862 ; http://repository.ust.hk/ir/bitstream/1783.1-87528/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Yuanjun. “Characterization of metastasis suppressor Nm23-H1 : the regulation mechanism and structure-function studies.” 2016. Thesis, Hong Kong University of Science and Technology. Accessed November 13, 2019. https://doi.org/10.14711/thesis-b1731862 ; http://repository.ust.hk/ir/bitstream/1783.1-87528/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Yuanjun. “Characterization of metastasis suppressor Nm23-H1 : the regulation mechanism and structure-function studies.” 2016. Web. 13 Nov 2019.

Vancouver:

Li Y. Characterization of metastasis suppressor Nm23-H1 : the regulation mechanism and structure-function studies. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2016. [cited 2019 Nov 13]. Available from: https://doi.org/10.14711/thesis-b1731862 ; http://repository.ust.hk/ir/bitstream/1783.1-87528/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. Characterization of metastasis suppressor Nm23-H1 : the regulation mechanism and structure-function studies. [Thesis]. Hong Kong University of Science and Technology; 2016. Available from: https://doi.org/10.14711/thesis-b1731862 ; http://repository.ust.hk/ir/bitstream/1783.1-87528/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Kovi, Ramesh C. Defining the Role of CtBP2 in p53-Independent Tumor Suppressor Function of ARF: A Dissertation.

Degree: Cancer Biology, Department of Medicine; Department of Cancer Biology, 2009, U of Massachusetts : Med

  ARF, a potent tumor suppressor, positively regulates p53 by antagonizing MDM2, a negative regulator of p53, which in turn, results in either apoptosis or… (more)

Subjects/Keywords: ADP-Ribosylation Factors; Apoptosis; Tumor Suppressor Protein p53; Phosphoproteins; DNA-Binding Proteins; Tumor Suppressor Protein p14ARF; Colorectal Neoplasms; Genes; Tumor Suppressor; Amino Acids, Peptides, and Proteins; Cells; Genetic Phenomena; Neoplasms

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APA (6th Edition):

Kovi, R. C. (2009). Defining the Role of CtBP2 in p53-Independent Tumor Suppressor Function of ARF: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/433

Chicago Manual of Style (16th Edition):

Kovi, Ramesh C. “Defining the Role of CtBP2 in p53-Independent Tumor Suppressor Function of ARF: A Dissertation.” 2009. Doctoral Dissertation, U of Massachusetts : Med. Accessed November 13, 2019. https://escholarship.umassmed.edu/gsbs_diss/433.

MLA Handbook (7th Edition):

Kovi, Ramesh C. “Defining the Role of CtBP2 in p53-Independent Tumor Suppressor Function of ARF: A Dissertation.” 2009. Web. 13 Nov 2019.

Vancouver:

Kovi RC. Defining the Role of CtBP2 in p53-Independent Tumor Suppressor Function of ARF: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2009. [cited 2019 Nov 13]. Available from: https://escholarship.umassmed.edu/gsbs_diss/433.

Council of Science Editors:

Kovi RC. Defining the Role of CtBP2 in p53-Independent Tumor Suppressor Function of ARF: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2009. Available from: https://escholarship.umassmed.edu/gsbs_diss/433

8. Mesrobian, Cristina M. The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators.

Degree: PhD, Division of Biology and Medicine. Pathobiology, 2009, Brown University

 Maintenance of an intricate balance between positive and negative regulators of the cell cycle is vital, as altered cell cycle regulation results in the uncontrolled… (more)

Subjects/Keywords: tumor suppressor

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APA (6th Edition):

Mesrobian, C. M. (2009). The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:173/

Chicago Manual of Style (16th Edition):

Mesrobian, Cristina M. “The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators.” 2009. Doctoral Dissertation, Brown University. Accessed November 13, 2019. https://repository.library.brown.edu/studio/item/bdr:173/.

MLA Handbook (7th Edition):

Mesrobian, Cristina M. “The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators.” 2009. Web. 13 Nov 2019.

Vancouver:

Mesrobian CM. The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators. [Internet] [Doctoral dissertation]. Brown University; 2009. [cited 2019 Nov 13]. Available from: https://repository.library.brown.edu/studio/item/bdr:173/.

Council of Science Editors:

Mesrobian CM. The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators. [Doctoral Dissertation]. Brown University; 2009. Available from: https://repository.library.brown.edu/studio/item/bdr:173/


East Carolina University

9. Loftin, Charles William. Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells.

Degree: 2010, East Carolina University

 DNA-dependent protein kinase (DNA-PK) is a holoenzyme of three subunits, Ku70, Ku80, and the DNA-Pk catalytic subunit (DNA-PKcs). DNA-PK serves a role in non-homologous end… (more)

Subjects/Keywords: Biology; DNA damage; Protein kinases; DNA repair; Tumor suppressor proteins

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APA (6th Edition):

Loftin, C. W. (2010). Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/2727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Loftin, Charles William. “Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells.” 2010. Thesis, East Carolina University. Accessed November 13, 2019. http://hdl.handle.net/10342/2727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Loftin, Charles William. “Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells.” 2010. Web. 13 Nov 2019.

Vancouver:

Loftin CW. Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells. [Internet] [Thesis]. East Carolina University; 2010. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10342/2727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loftin CW. Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells. [Thesis]. East Carolina University; 2010. Available from: http://hdl.handle.net/10342/2727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

10. 龔重儀; Kung, Chung-yee. Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma.

Degree: M. Phil., 2016, University of Hong Kong

Salt-Inducible Kinase 2 (SIK2) is a member of the AMP-activated kinase family. Initially characterized for its roles in cellular metabolism, SIK2 has recently been shown… (more)

Subjects/Keywords: Protein kinases; Pathogenesis - Cancer - Liver; Tumor suppressor proteins

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APA (6th Edition):

龔重儀; Kung, C. (2016). Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Kung, C. [龔重儀]. (2016). Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760911. ; http://dx.doi.org/10.5353/th_b5760911 ; http://hdl.handle.net/10722/239634

Chicago Manual of Style (16th Edition):

龔重儀; Kung, Chung-yee. “Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma.” 2016. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. Kung, C. [龔重儀]. (2016). Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760911. ; http://dx.doi.org/10.5353/th_b5760911 ; http://hdl.handle.net/10722/239634.

MLA Handbook (7th Edition):

龔重儀; Kung, Chung-yee. “Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma.” 2016. Web. 13 Nov 2019.

Vancouver:

龔重儀; Kung C. Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2016. [cited 2019 Nov 13]. Available from: Kung, C. [龔重儀]. (2016). Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760911. ; http://dx.doi.org/10.5353/th_b5760911 ; http://hdl.handle.net/10722/239634.

Council of Science Editors:

龔重儀; Kung C. Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2016. Available from: Kung, C. [龔重儀]. (2016). Functional characterization of salt-inducible kinase 2 and its interaction with Tax1 binding protein 2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760911. ; http://dx.doi.org/10.5353/th_b5760911 ; http://hdl.handle.net/10722/239634


University of Hong Kong

11. Lee, Ka-wai. Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma.

Degree: Master of Medical Sciences, 2015, University of Hong Kong

published_or_final_version

Pathology

Master

Master of Medical Sciences

Subjects/Keywords: Liver - Cancer; Tumor suppressor proteins

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APA (6th Edition):

Lee, K. (2015). Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Lee, K. [李家慧]. (2015). Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5633008 ; http://hdl.handle.net/10722/221470

Chicago Manual of Style (16th Edition):

Lee, Ka-wai. “Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma.” 2015. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. Lee, K. [李家慧]. (2015). Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5633008 ; http://hdl.handle.net/10722/221470.

MLA Handbook (7th Edition):

Lee, Ka-wai. “Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma.” 2015. Web. 13 Nov 2019.

Vancouver:

Lee K. Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Nov 13]. Available from: Lee, K. [李家慧]. (2015). Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5633008 ; http://hdl.handle.net/10722/221470.

Council of Science Editors:

Lee K. Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2015. Available from: Lee, K. [李家慧]. (2015). Functional characterization of novel tumor suppressor galectin-2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5633008 ; http://hdl.handle.net/10722/221470


University of Hong Kong

12. Liu, Heong-fai, Michael. Characterization of the functional role of AMP-activated protein kinase in tumor suppression.

Degree: M. Phil., 2007, University of Hong Kong

abstract

published_or_final_version

Pathology

Master

Master of Philosophy

Advisors/Committee Members: Ng, IOL, Ching, YP.

Subjects/Keywords: Tumor suppressor proteins.; Protein kinases.

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APA (6th Edition):

Liu, Heong-fai, M. (2007). Characterization of the functional role of AMP-activated protein kinase in tumor suppression. (Masters Thesis). University of Hong Kong. Retrieved from Liu, H. M. [呂向暉]. (2007). Characterization of the functional role of AMP-activated protein kinase in tumor suppression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955715 ; http://dx.doi.org/10.5353/th_b3955715 ; http://hdl.handle.net/10722/52285

Chicago Manual of Style (16th Edition):

Liu, Heong-fai, Michael. “Characterization of the functional role of AMP-activated protein kinase in tumor suppression.” 2007. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. Liu, H. M. [呂向暉]. (2007). Characterization of the functional role of AMP-activated protein kinase in tumor suppression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955715 ; http://dx.doi.org/10.5353/th_b3955715 ; http://hdl.handle.net/10722/52285.

MLA Handbook (7th Edition):

Liu, Heong-fai, Michael. “Characterization of the functional role of AMP-activated protein kinase in tumor suppression.” 2007. Web. 13 Nov 2019.

Vancouver:

Liu, Heong-fai M. Characterization of the functional role of AMP-activated protein kinase in tumor suppression. [Internet] [Masters thesis]. University of Hong Kong; 2007. [cited 2019 Nov 13]. Available from: Liu, H. M. [呂向暉]. (2007). Characterization of the functional role of AMP-activated protein kinase in tumor suppression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955715 ; http://dx.doi.org/10.5353/th_b3955715 ; http://hdl.handle.net/10722/52285.

Council of Science Editors:

Liu, Heong-fai M. Characterization of the functional role of AMP-activated protein kinase in tumor suppression. [Masters Thesis]. University of Hong Kong; 2007. Available from: Liu, H. M. [呂向暉]. (2007). Characterization of the functional role of AMP-activated protein kinase in tumor suppression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955715 ; http://dx.doi.org/10.5353/th_b3955715 ; http://hdl.handle.net/10722/52285


IUPUI

13. Walker, Joanna Antigone. Expression of Human Papillomavirus Type 16 E7 Is Sufficient To Significantly Increase Expression of Angiogenic Factors But Is Not Sufficient To Induce Endothelial Cell Migration.

Degree: 2010, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Human papillomavirus 16 (HPV 16) causes cancer. Two viral oncoproteins of HPV 16, E6 and E7, are consistently expressed in… (more)

Subjects/Keywords: Papillomaviruses  – Research; Neovascularization inhibitors; Tumor suppressor proteins; Cancer

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APA (6th Edition):

Walker, J. A. (2010). Expression of Human Papillomavirus Type 16 E7 Is Sufficient To Significantly Increase Expression of Angiogenic Factors But Is Not Sufficient To Induce Endothelial Cell Migration. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2291

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walker, Joanna Antigone. “Expression of Human Papillomavirus Type 16 E7 Is Sufficient To Significantly Increase Expression of Angiogenic Factors But Is Not Sufficient To Induce Endothelial Cell Migration.” 2010. Thesis, IUPUI. Accessed November 13, 2019. http://hdl.handle.net/1805/2291.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walker, Joanna Antigone. “Expression of Human Papillomavirus Type 16 E7 Is Sufficient To Significantly Increase Expression of Angiogenic Factors But Is Not Sufficient To Induce Endothelial Cell Migration.” 2010. Web. 13 Nov 2019.

Vancouver:

Walker JA. Expression of Human Papillomavirus Type 16 E7 Is Sufficient To Significantly Increase Expression of Angiogenic Factors But Is Not Sufficient To Induce Endothelial Cell Migration. [Internet] [Thesis]. IUPUI; 2010. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/1805/2291.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walker JA. Expression of Human Papillomavirus Type 16 E7 Is Sufficient To Significantly Increase Expression of Angiogenic Factors But Is Not Sufficient To Induce Endothelial Cell Migration. [Thesis]. IUPUI; 2010. Available from: http://hdl.handle.net/1805/2291

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

14. Billing, David. The BARD1 BRCT Domain in Tumor Suppression and Genome Stability.

Degree: 2018, Columbia University

 BRCA1 preserves genome integrity through both homology-directed repair (HDR) and stalled fork protection (SFP). In vivo, BRCA1 exists as a heterodimer with the BARD1 tumor(more)

Subjects/Keywords: Molecular biology; Cytology; Biochemistry; Tumor suppressor proteins; Genomes

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APA (6th Edition):

Billing, D. (2018). The BARD1 BRCT Domain in Tumor Suppression and Genome Stability. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8Z339N4

Chicago Manual of Style (16th Edition):

Billing, David. “The BARD1 BRCT Domain in Tumor Suppression and Genome Stability.” 2018. Doctoral Dissertation, Columbia University. Accessed November 13, 2019. https://doi.org/10.7916/D8Z339N4.

MLA Handbook (7th Edition):

Billing, David. “The BARD1 BRCT Domain in Tumor Suppression and Genome Stability.” 2018. Web. 13 Nov 2019.

Vancouver:

Billing D. The BARD1 BRCT Domain in Tumor Suppression and Genome Stability. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2019 Nov 13]. Available from: https://doi.org/10.7916/D8Z339N4.

Council of Science Editors:

Billing D. The BARD1 BRCT Domain in Tumor Suppression and Genome Stability. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8Z339N4


University of Texas Southwestern Medical Center

15. Yue, Tao. Studies of the Hippo Signaling Pathway.

Degree: 2012, University of Texas Southwestern Medical Center

 How multicellular organisms control their growth to reach proper organ size during development is a fascinating question. Recent studies, initially from Drosophila, have identified the… (more)

Subjects/Keywords: Tumor Suppressor Proteins; Protein-Serine-Threonine Kinases; Drosophila melanogaster

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APA (6th Edition):

Yue, T. (2012). Studies of the Hippo Signaling Pathway. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yue, Tao. “Studies of the Hippo Signaling Pathway.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed November 13, 2019. http://hdl.handle.net/2152.5/1104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yue, Tao. “Studies of the Hippo Signaling Pathway.” 2012. Web. 13 Nov 2019.

Vancouver:

Yue T. Studies of the Hippo Signaling Pathway. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2152.5/1104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yue T. Studies of the Hippo Signaling Pathway. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Ram, Rosalyn Ruanga. The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1 Checkpoint Mechanisms.

Degree: 2012, University of Texas Southwestern Medical Center

 The RASSF1A tumor suppressor is one of the most commonly inactivated genes in cancer. To understand why epigenetic silencing of RASSF1A promotes tumorigenesis, I employed… (more)

Subjects/Keywords: Tumor Suppressor Proteins; Ubiquitin-Protein Ligase Complexes; Cell Cycle

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APA (6th Edition):

Ram, R. R. (2012). The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1 Checkpoint Mechanisms. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ram, Rosalyn Ruanga. “The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1 Checkpoint Mechanisms.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed November 13, 2019. http://hdl.handle.net/2152.5/1004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ram, Rosalyn Ruanga. “The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1 Checkpoint Mechanisms.” 2012. Web. 13 Nov 2019.

Vancouver:

Ram RR. The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1 Checkpoint Mechanisms. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2152.5/1004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ram RR. The RASSF1A Tumor Suppressor Regulates a Cascade of Oncogenic Signals That Are Restrained by G1 Checkpoint Mechanisms. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Lu, Wan-Jin. Illuminating the P53 Regulatory Network in Genetic Models.

Degree: 2011, University of Texas Southwestern Medical Center

The file named "thesis.pdf" is the primary dissertation file. Supplemental videos ("Movie1.avi", "Movie2.avi") are also provided (in Audio Video Interleaved format).

The tumor suppressor gene… (more)

Subjects/Keywords: Genes, P53; Drosophila melanogaster; Gene Regulatory Networks; Tumor Suppressor Proteins

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APA (6th Edition):

Lu, W. (2011). Illuminating the P53 Regulatory Network in Genetic Models. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lu, Wan-Jin. “Illuminating the P53 Regulatory Network in Genetic Models.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed November 13, 2019. http://hdl.handle.net/2152.5/857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lu, Wan-Jin. “Illuminating the P53 Regulatory Network in Genetic Models.” 2011. Web. 13 Nov 2019.

Vancouver:

Lu W. Illuminating the P53 Regulatory Network in Genetic Models. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2152.5/857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lu W. Illuminating the P53 Regulatory Network in Genetic Models. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Pereira, Larissa Miranda. Clonagem, expressão, purificação e caracterização estrutural da proteína ribossomal L10 humana recombinante.

Degree: Mestrado, Tecnologia Nuclear - Aplicações, 2009, University of São Paulo

A proteína ribossomal L10 (RP L10) é uma forte candidata a ser incluída na classe de proteínas supressoras de tumor. Também denominada QM, a proteína… (more)

Subjects/Keywords: proteína ribossomal L10; proteína supressora de tumor; QM; QM; ribosomal protein L10; tumor suppressor proteins

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APA (6th Edition):

Pereira, L. M. (2009). Clonagem, expressão, purificação e caracterização estrutural da proteína ribossomal L10 humana recombinante. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/85/85131/tde-22092011-101810/ ;

Chicago Manual of Style (16th Edition):

Pereira, Larissa Miranda. “Clonagem, expressão, purificação e caracterização estrutural da proteína ribossomal L10 humana recombinante.” 2009. Masters Thesis, University of São Paulo. Accessed November 13, 2019. http://www.teses.usp.br/teses/disponiveis/85/85131/tde-22092011-101810/ ;.

MLA Handbook (7th Edition):

Pereira, Larissa Miranda. “Clonagem, expressão, purificação e caracterização estrutural da proteína ribossomal L10 humana recombinante.” 2009. Web. 13 Nov 2019.

Vancouver:

Pereira LM. Clonagem, expressão, purificação e caracterização estrutural da proteína ribossomal L10 humana recombinante. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2019 Nov 13]. Available from: http://www.teses.usp.br/teses/disponiveis/85/85131/tde-22092011-101810/ ;.

Council of Science Editors:

Pereira LM. Clonagem, expressão, purificação e caracterização estrutural da proteína ribossomal L10 humana recombinante. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/85/85131/tde-22092011-101810/ ;


University of Hong Kong

19. Li, Hung-sing. Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer.

Degree: M. Phil., 2014, University of Hong Kong

 Polycomb group (PcG) proteins governs the regulation of diverse cellular functions, such as cell fate decision, cell cycle progression, maintenance of embryonic stem cell pluripotency,… (more)

Subjects/Keywords: Rectum - Cancer - Genetic aspects; Chromosomal proteins; Colon (Anatomy) - Cancer - Genetic aspects; Tumor suppressor proteins

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APA (6th Edition):

Li, H. (2014). Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. (Masters Thesis). University of Hong Kong. Retrieved from Li, H. [李鴻陞]. (2014). Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194758 ; http://dx.doi.org/10.5353/th_b5194758 ; http://hdl.handle.net/10722/197534

Chicago Manual of Style (16th Edition):

Li, Hung-sing. “Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer.” 2014. Masters Thesis, University of Hong Kong. Accessed November 13, 2019. Li, H. [李鴻陞]. (2014). Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194758 ; http://dx.doi.org/10.5353/th_b5194758 ; http://hdl.handle.net/10722/197534.

MLA Handbook (7th Edition):

Li, Hung-sing. “Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer.” 2014. Web. 13 Nov 2019.

Vancouver:

Li H. Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2019 Nov 13]. Available from: Li, H. [李鴻陞]. (2014). Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194758 ; http://dx.doi.org/10.5353/th_b5194758 ; http://hdl.handle.net/10722/197534.

Council of Science Editors:

Li H. Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. [Masters Thesis]. University of Hong Kong; 2014. Available from: Li, H. [李鴻陞]. (2014). Identification of polycomb group protein CBX8 as a novel tumor suppressor in human colorectal cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194758 ; http://dx.doi.org/10.5353/th_b5194758 ; http://hdl.handle.net/10722/197534

20. Rohatgi, Rasika. Autophagy-Independent Role for Beclin 1 in the Regulation of Growth Factor Receptor Signaling: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology Department, 2015, U of Massachusetts : Med

  Beclin 1 is a haplo-insufficient tumor suppressor that is decreased in many human tumors. The function of Beclin 1 in cancer has been attributed… (more)

Subjects/Keywords: Signal Transducing Adaptor Proteins; Apoptosis Regulatory Proteins; Tumor Suppressor Proteins; Autophagy; Breast Neoplasms; Neoplastic Cell Transformation; Cancer Biology; Cell Biology; Neoplasms

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APA (6th Edition):

Rohatgi, R. (2015). Autophagy-Independent Role for Beclin 1 in the Regulation of Growth Factor Receptor Signaling: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/873

Chicago Manual of Style (16th Edition):

Rohatgi, Rasika. “Autophagy-Independent Role for Beclin 1 in the Regulation of Growth Factor Receptor Signaling: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed November 13, 2019. http://escholarship.umassmed.edu/gsbs_diss/873.

MLA Handbook (7th Edition):

Rohatgi, Rasika. “Autophagy-Independent Role for Beclin 1 in the Regulation of Growth Factor Receptor Signaling: A Dissertation.” 2015. Web. 13 Nov 2019.

Vancouver:

Rohatgi R. Autophagy-Independent Role for Beclin 1 in the Regulation of Growth Factor Receptor Signaling: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Nov 13]. Available from: http://escholarship.umassmed.edu/gsbs_diss/873.

Council of Science Editors:

Rohatgi R. Autophagy-Independent Role for Beclin 1 in the Regulation of Growth Factor Receptor Signaling: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/873


University of Hong Kong

21. Kan, Pei-qi, Rebecca. Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma.

Degree: PhD, 2016, University of Hong Kong

abstract

Clinical Oncology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Nasopharynx - Cancer - Pathogenesis; Tumor suppressor proteins

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APA (6th Edition):

Kan, Pei-qi, R. (2016). Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/227933

Chicago Manual of Style (16th Edition):

Kan, Pei-qi, Rebecca. “Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed November 13, 2019. http://hdl.handle.net/10722/227933.

MLA Handbook (7th Edition):

Kan, Pei-qi, Rebecca. “Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma.” 2016. Web. 13 Nov 2019.

Vancouver:

Kan, Pei-qi R. Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/10722/227933.

Council of Science Editors:

Kan, Pei-qi R. Characterization of the extracellular matrix tumor suppressor protein, latent transforming growth factor-beta binding protein 2 (LTBP2), and in-depth mechanistic studies of its signaling pathway in nasopharyngeal carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/227933


University of Hong Kong

22. 黃澤蕾.; Wong, Chak-lui, Carmen. Regulations and functions of rho-kinases in hepatocellular carcinoma.

Degree: PhD, 2009, University of Hong Kong

The Best PhD Thesis in the Faculties of Dentistry, Engineering, Medicine and Science (University of Hong Kong), Li Ka Shing Prize,2008-2009

published_or_final_version

Pathology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Ng, IOL.

Subjects/Keywords: Cancer cells.; Metastasis.; Liver - Cancer - Molecular aspects.; Serine proteinases.; Protein kinases.; Tumor suppressor proteins.

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APA (6th Edition):

黃澤蕾.; Wong, Chak-lui, C. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132

Chicago Manual of Style (16th Edition):

黃澤蕾.; Wong, Chak-lui, Carmen. “Regulations and functions of rho-kinases in hepatocellular carcinoma.” 2009. Doctoral Dissertation, University of Hong Kong. Accessed November 13, 2019. Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132.

MLA Handbook (7th Edition):

黃澤蕾.; Wong, Chak-lui, Carmen. “Regulations and functions of rho-kinases in hepatocellular carcinoma.” 2009. Web. 13 Nov 2019.

Vancouver:

黃澤蕾.; Wong, Chak-lui C. Regulations and functions of rho-kinases in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2009. [cited 2019 Nov 13]. Available from: Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132.

Council of Science Editors:

黃澤蕾.; Wong, Chak-lui C. Regulations and functions of rho-kinases in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2009. Available from: Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132


IUPUI

23. Hardy, Tabitha M. XPC DNA REPAIR PROTEIN REGULATION IN THE CONTEXT OF THE G1/S CELL CYCLE CHECKPOINT.

Degree: 2010, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

DNA is subject to various types of damage that can impair cellular function or cause cell death. DNA damage blocks… (more)

Subjects/Keywords: XPC, DNA repair, retinoblastoma protein, cell cycle; DNA repair; Tumor suppressor proteins; Cell cycle

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APA (6th Edition):

Hardy, T. M. (2010). XPC DNA REPAIR PROTEIN REGULATION IN THE CONTEXT OF THE G1/S CELL CYCLE CHECKPOINT. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hardy, Tabitha M. “XPC DNA REPAIR PROTEIN REGULATION IN THE CONTEXT OF THE G1/S CELL CYCLE CHECKPOINT.” 2010. Thesis, IUPUI. Accessed November 13, 2019. http://hdl.handle.net/1805/2279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hardy, Tabitha M. “XPC DNA REPAIR PROTEIN REGULATION IN THE CONTEXT OF THE G1/S CELL CYCLE CHECKPOINT.” 2010. Web. 13 Nov 2019.

Vancouver:

Hardy TM. XPC DNA REPAIR PROTEIN REGULATION IN THE CONTEXT OF THE G1/S CELL CYCLE CHECKPOINT. [Internet] [Thesis]. IUPUI; 2010. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/1805/2279.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hardy TM. XPC DNA REPAIR PROTEIN REGULATION IN THE CONTEXT OF THE G1/S CELL CYCLE CHECKPOINT. [Thesis]. IUPUI; 2010. Available from: http://hdl.handle.net/1805/2279

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

24. Yoh, Kathryn Elizabeth. Ras, p63 and breast cancer.

Degree: 2016, Columbia University

 As a master regulator of the epithelial state, p63 is a family member of the well-known tumor suppressor p53. It has previously been connected to… (more)

Subjects/Keywords: Breast – Cancer; Carcinogenesis; Breast – Cancer – Etiology; Mesenchyme; Tumor suppressor proteins; Oncology; Molecular biology

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APA (6th Edition):

Yoh, K. E. (2016). Ras, p63 and breast cancer. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8ZS2WQK

Chicago Manual of Style (16th Edition):

Yoh, Kathryn Elizabeth. “Ras, p63 and breast cancer.” 2016. Doctoral Dissertation, Columbia University. Accessed November 13, 2019. https://doi.org/10.7916/D8ZS2WQK.

MLA Handbook (7th Edition):

Yoh, Kathryn Elizabeth. “Ras, p63 and breast cancer.” 2016. Web. 13 Nov 2019.

Vancouver:

Yoh KE. Ras, p63 and breast cancer. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2019 Nov 13]. Available from: https://doi.org/10.7916/D8ZS2WQK.

Council of Science Editors:

Yoh KE. Ras, p63 and breast cancer. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8ZS2WQK


Columbia University

25. Ou, Yang. Dissecting the role of p53-mediated metabolic regulation in tumor suppression.

Degree: 2016, Columbia University

 The p53 tumor suppressor protein has been well-characterized for its role in inducing growth arrest, senescence, and apoptosis upon various types of stresses. Recently, however,… (more)

Subjects/Keywords: p53 antioncogene; Tumor suppressor proteins; Antioncogenes; Metabolism – Regulation; p53 protein; Biology; Oncology; Cytology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ou, Y. (2016). Dissecting the role of p53-mediated metabolic regulation in tumor suppression. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8BZ66K5

Chicago Manual of Style (16th Edition):

Ou, Yang. “Dissecting the role of p53-mediated metabolic regulation in tumor suppression.” 2016. Doctoral Dissertation, Columbia University. Accessed November 13, 2019. https://doi.org/10.7916/D8BZ66K5.

MLA Handbook (7th Edition):

Ou, Yang. “Dissecting the role of p53-mediated metabolic regulation in tumor suppression.” 2016. Web. 13 Nov 2019.

Vancouver:

Ou Y. Dissecting the role of p53-mediated metabolic regulation in tumor suppression. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2019 Nov 13]. Available from: https://doi.org/10.7916/D8BZ66K5.

Council of Science Editors:

Ou Y. Dissecting the role of p53-mediated metabolic regulation in tumor suppression. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8BZ66K5


Columbia University

26. Pappas, Kyrie Jean. Transcriptional control of tumor suppressor genes in cancer.

Degree: 2017, Columbia University

 An important hallmark of cancer is the inactivation of tumor suppressor genes. The most common genetic alteration in cancer is the mutation of the TP53… (more)

Subjects/Keywords: Antioncogenes; Cancer – Genetic aspects; Tumor suppressor proteins; Oncology; Genetic transcription – Regulation; Cancer; Biology; Genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pappas, K. J. (2017). Transcriptional control of tumor suppressor genes in cancer. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8MW2NQM

Chicago Manual of Style (16th Edition):

Pappas, Kyrie Jean. “Transcriptional control of tumor suppressor genes in cancer.” 2017. Doctoral Dissertation, Columbia University. Accessed November 13, 2019. https://doi.org/10.7916/D8MW2NQM.

MLA Handbook (7th Edition):

Pappas, Kyrie Jean. “Transcriptional control of tumor suppressor genes in cancer.” 2017. Web. 13 Nov 2019.

Vancouver:

Pappas KJ. Transcriptional control of tumor suppressor genes in cancer. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2019 Nov 13]. Available from: https://doi.org/10.7916/D8MW2NQM.

Council of Science Editors:

Pappas KJ. Transcriptional control of tumor suppressor genes in cancer. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D8MW2NQM


University of Texas Southwestern Medical Center

27. Kurtz, Paula S. In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network.

Degree: 2017, University of Texas Southwestern Medical Center

 p53 is the most commonly mutated gene in human cancers. Despite decades of p53 studies we do not fully understand how p53 suppresses tumors. Similar… (more)

Subjects/Keywords: Drosophila Proteins; Gene Expression Regulation; Genome-Wide Association Study; Tumor Suppressor Protein p53

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kurtz, P. S. (2017). In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7197

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kurtz, Paula S. “In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed November 13, 2019. http://hdl.handle.net/2152.5/7197.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kurtz, Paula S. “In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network.” 2017. Web. 13 Nov 2019.

Vancouver:

Kurtz PS. In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/2152.5/7197.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kurtz PS. In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7197

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

28. Cho, Chi-kong, Lawrence. Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP.

Degree: PhD, 2011, University of Hong Kong

published_or_final_version

Chemistry

Doctoral

Doctor of Philosophy

Subjects/Keywords: Tumor suppressor proteins.; Apoptosis - Molecular aspects.; Zinc-finger proteins.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cho, Chi-kong, L. (2011). Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cho, C. L. [曹智剛]. (2011). Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4704412 ; http://dx.doi.org/10.5353/th_b4704412 ; http://hdl.handle.net/10722/177199

Chicago Manual of Style (16th Edition):

Cho, Chi-kong, Lawrence. “Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed November 13, 2019. Cho, C. L. [曹智剛]. (2011). Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4704412 ; http://dx.doi.org/10.5353/th_b4704412 ; http://hdl.handle.net/10722/177199.

MLA Handbook (7th Edition):

Cho, Chi-kong, Lawrence. “Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP.” 2011. Web. 13 Nov 2019.

Vancouver:

Cho, Chi-kong L. Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2019 Nov 13]. Available from: Cho, C. L. [曹智剛]. (2011). Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4704412 ; http://dx.doi.org/10.5353/th_b4704412 ; http://hdl.handle.net/10722/177199.

Council of Science Editors:

Cho, Chi-kong L. Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: Cho, C. L. [曹智剛]. (2011). Structural characterization of C-terminal zinc finger domain of XIAP associated factor 1 (XAF1) and its interaction studies with XIAP. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4704412 ; http://dx.doi.org/10.5353/th_b4704412 ; http://hdl.handle.net/10722/177199


Queens University

29. Chen, Jiamin. MICRORNA-193B FUNCTIONS AS A TUMOR SUPPRESSOR IN MALIGNANT MELANOMA .

Degree: Pathology and Molecular Medicine, 2012, Queens University

 Cutaneous melanoma is an increasingly common skin cancer characterized by aggressive metastatic growth and poor prognosis. The mechanisms behind melanoma progression are not fully understood,… (more)

Subjects/Keywords: melanoma; miR-193b; tumor suppressor

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APA (6th Edition):

Chen, J. (2012). MICRORNA-193B FUNCTIONS AS A TUMOR SUPPRESSOR IN MALIGNANT MELANOMA . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jiamin. “MICRORNA-193B FUNCTIONS AS A TUMOR SUPPRESSOR IN MALIGNANT MELANOMA .” 2012. Thesis, Queens University. Accessed November 13, 2019. http://hdl.handle.net/1974/7224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jiamin. “MICRORNA-193B FUNCTIONS AS A TUMOR SUPPRESSOR IN MALIGNANT MELANOMA .” 2012. Web. 13 Nov 2019.

Vancouver:

Chen J. MICRORNA-193B FUNCTIONS AS A TUMOR SUPPRESSOR IN MALIGNANT MELANOMA . [Internet] [Thesis]. Queens University; 2012. [cited 2019 Nov 13]. Available from: http://hdl.handle.net/1974/7224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen J. MICRORNA-193B FUNCTIONS AS A TUMOR SUPPRESSOR IN MALIGNANT MELANOMA . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed November 13, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 13 Nov 2019.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Nov 13]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045

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