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You searched for subject:( Transcription Factors metabolism 60). Showing records 1 – 30 of 28946 total matches.

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1. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 18 Oct 2019.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

2. Ho, Shiuh-Rong. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.

Degree: PhD, 2010, University of Alabama – Birmingham

O-GlcNAcylation is an abundant and dynamic post-translational modification on serine and threonine residues of nuclear and cytoplasmic proteins. O-GlcNAc Transferase (OGT) and Nuclear Cytoplasmic O-GlcNAcase… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Gene Expression Regulation<; br>; Oxidative Stress  – genetics<; br>; Transcription Factors  – metabolism<; br>; Transcription, Genetic

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APA (6th Edition):

Ho, S. (2010). O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1162

Chicago Manual of Style (16th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1162.

MLA Handbook (7th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Web. 18 Oct 2019.

Vancouver:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162.

Council of Science Editors:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162

3. Sarof, Arjun. Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells.

Degree: MS, 2009, University of Alabama – Birmingham

Runx2 is a runt domain transcription factor essential for bone development and tooth morphogenesis and is required for epithelial-mesenchymal interactions that regulate skeletogenesis. Fibroblast growth… (more)

Subjects/Keywords: Epithelium  – physiology<; br>; Fibroblast Growth Factor 3<; br>; Tooth  – embryology<; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Sarof, A. (2009). Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,802

Chicago Manual of Style (16th Edition):

Sarof, Arjun. “Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,802.

MLA Handbook (7th Edition):

Sarof, Arjun. “Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells.” 2009. Web. 18 Oct 2019.

Vancouver:

Sarof A. Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,802.

Council of Science Editors:

Sarof A. Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,802

4. Hock, Thomas D. Regulation of the human heme oxygenase-1 gene.

Degree: PhD, 2007, University of Alabama – Birmingham

The heme oxygenase-1 (HO-1) gene encodes a microsomal enzyme that catalyzes the conversion of heme to carbon monoxide, Iron, and biliverdin. HO-1 transcription is induced… (more)

Subjects/Keywords: Heme Oxygenase-1  – genetics <; br>; Proto-Oncogene Proteins c-jun  – metabolism <; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Hock, T. D. (2007). Regulation of the human heme oxygenase-1 gene. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,126

Chicago Manual of Style (16th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,126.

MLA Handbook (7th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Web. 18 Oct 2019.

Vancouver:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126.

Council of Science Editors:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126

5. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 18 Oct 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

6. He, Ti. Molecular regulation of Pax5-mediated biological functions.

Degree: PhD, 2008, University of Alabama – Birmingham

B lineage cells are major players in the adaptive immune system. Pax5 is essential for B lineage cell development and function. Pax5 controls B lineage… (more)

Subjects/Keywords: B-Cell-Specific Activator Protein <; br>; Gene Expression Regulation <; br>; Histone Acetyltransferases  – metabolism <; br>; Transcription Factors <; br>; Transcription, Genetic

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APA (6th Edition):

He, T. (2008). Molecular regulation of Pax5-mediated biological functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,329

Chicago Manual of Style (16th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,329.

MLA Handbook (7th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Web. 18 Oct 2019.

Vancouver:

He T. Molecular regulation of Pax5-mediated biological functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329.

Council of Science Editors:

He T. Molecular regulation of Pax5-mediated biological functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329

7. Pandya, Ashka Y. Structural and functional analysis of KLF4.

Degree: PhD, 2007, University of Alabama – Birmingham

KLF4, a C2H2 type zinc finger transcription factor, plays an essential role in maturation of normal stratified squamous epithelium. In normal squamous epithelium its expression… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Nucleus  – metabolism<; br>; DNA-Binding Proteins  – biosynthesis<; br>; Kruppel-Like Transcription Factors<; br>; Prognosis Transcription Factors  – biosynthesis

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APA (6th Edition):

Pandya, A. Y. (2007). Structural and functional analysis of KLF4. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,531

Chicago Manual of Style (16th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,531.

MLA Handbook (7th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Web. 18 Oct 2019.

Vancouver:

Pandya AY. Structural and functional analysis of KLF4. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531.

Council of Science Editors:

Pandya AY. Structural and functional analysis of KLF4. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531

8. Zheng, Ying. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Janus Kinase (JAK)-Signal Transducer and Activator of Transcription (STAT) signaling is involved in regulation of cell survival, proliferation and differentiation. JAK tyrosine kinases can be… (more)

Subjects/Keywords: Casein Kinase II  – metabolism<; br>; Hematologic Neoplasms  – metabolism<; br>; JNK Mitogen-Activated Protein Kinases  – metabolism<; br>; Polycythemia Vera  – metabolism<; br>; STAT Transcription Factors  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Zheng, Y. (2010). A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1168

Chicago Manual of Style (16th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1168.

MLA Handbook (7th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Web. 18 Oct 2019.

Vancouver:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168.

Council of Science Editors:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168

9. Polter, Abigail Marie. Regulation of GSK3 in the pathophysiology and treatment of mood disorders.

Degree: PhD, 2010, University of Alabama – Birmingham

Mood disorders are devastating psychiatric illnesses that will affect as many as one in every five persons worldwide over the course of their lifetime. Significant… (more)

Subjects/Keywords: Behavior, Animal  – physiology<; br>; Brain  – metabolism<; br>; Exploratory Behavior  – physiology<; br>; Forkhead Transcription Factors  – metabolism<; br>; Glycogen Synthase Kinase 3<; br>; Mood Disorders<; br>; Receptor, Serotonin, 5-HT1A  – metabolism<; br>; Serine  – metabolism<; br>; Serotonin  – metabolism<; br>; Signal Transduction

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APA (6th Edition):

Polter, A. M. (2010). Regulation of GSK3 in the pathophysiology and treatment of mood disorders. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1108

Chicago Manual of Style (16th Edition):

Polter, Abigail Marie. “Regulation of GSK3 in the pathophysiology and treatment of mood disorders.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1108.

MLA Handbook (7th Edition):

Polter, Abigail Marie. “Regulation of GSK3 in the pathophysiology and treatment of mood disorders.” 2010. Web. 18 Oct 2019.

Vancouver:

Polter AM. Regulation of GSK3 in the pathophysiology and treatment of mood disorders. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1108.

Council of Science Editors:

Polter AM. Regulation of GSK3 in the pathophysiology and treatment of mood disorders. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1108

10. Liu, Zhaoli. KLF4 regulates notch1 expression and signaling during epithelial transformation.

Degree: PhD, 2006, University of Alabama – Birmingham

Notch1 and KLF4 function in the specification of epithelial cell fates, and each can act as a context-dependent oncogene or tumor suppressor. We report that… (more)

Subjects/Keywords: Breast Neoplasms <; br>; Epithelial Cells  – metabolism <; br>; Kruppel-Like Transcription Factors  – genetics <; br>; Receptor, Notch1  – metabolism

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APA (6th Edition):

Liu, Z. (2006). KLF4 regulates notch1 expression and signaling during epithelial transformation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,378

Chicago Manual of Style (16th Edition):

Liu, Zhaoli. “KLF4 regulates notch1 expression and signaling during epithelial transformation.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,378.

MLA Handbook (7th Edition):

Liu, Zhaoli. “KLF4 regulates notch1 expression and signaling during epithelial transformation.” 2006. Web. 18 Oct 2019.

Vancouver:

Liu Z. KLF4 regulates notch1 expression and signaling during epithelial transformation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,378.

Council of Science Editors:

Liu Z. KLF4 regulates notch1 expression and signaling during epithelial transformation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,378

11. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 18 Oct 2019.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

12. Harms, Paul William. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.

Degree: PhD, 2006, University of Alabama – Birmingham

Growth factor signals often regulate similar cellular processes both during embryogenesis and in adult homeostasis. Stringent control of these signals ensures proper embryonic development and… (more)

Subjects/Keywords: Homeodomain Proteins <; br>; Membrane Proteins  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Proteins <; br>; Signal Transduction  – physiology <; br>; Transcription Factors <; br>; Xenopus Proteins

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APA (6th Edition):

Harms, P. W. (2006). Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,369

Chicago Manual of Style (16th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,369.

MLA Handbook (7th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Web. 18 Oct 2019.

Vancouver:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369.

Council of Science Editors:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369

13. Lai, Yun-Ju. Role of TRIP6 in LPA-induced cell migration.

Degree: PhD, 2007, University of Alabama – Birmingham

The LIM domain-containing Thyroid Receptor-Interacting Protein 6 (TRIP6) is a zyxin family member that has been implicated in actin dynamics and cell motility. In this… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing<; br>; Carrier Proteins  – physiology<; br>; Cell Movement<; br>; Feedback, Biochemical<; br>; Lysophospholipids<; br>; Protein-Tyrosine Kinases  – physiology<; br>; Receptors, G-Protein-Coupled  – metabolism<; br>; Transcription Factors

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APA (6th Edition):

Lai, Y. (2007). Role of TRIP6 in LPA-induced cell migration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,453

Chicago Manual of Style (16th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,453.

MLA Handbook (7th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Web. 18 Oct 2019.

Vancouver:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453.

Council of Science Editors:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453

14. Winkelbauer, Marlene Elizabeth. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.

Degree: PhD, 2007, University of Alabama – Birmingham

Numerous disorders are characterized by the presence of cystic lesions within the kidney. The proteins associated with these disorders often localize to cilia, and improper… (more)

Subjects/Keywords: Caenorhabditis elegans  – genetics<; br>; Caenorhabditis elegans Proteins  – genetics<; br>; Cilia  – metabolism<; br>; Neurons, Afferent  – physiology<; br>; Protein Transport<; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Winkelbauer, M. E. (2007). Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,619

Chicago Manual of Style (16th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,619.

MLA Handbook (7th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Web. 18 Oct 2019.

Vancouver:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619.

Council of Science Editors:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619

15. Zhao, Xueyan. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.

Degree: PhD, 2008, University of Alabama – Birmingham

Matrix metalloprotinases are zinc-dependent endopeptidases with broad substrates from extracellular matrix proteins to bioactive molecules. Physiologically, they regulate tissue remodeling and immune responses. However, in… (more)

Subjects/Keywords: Gene Expression Regulation, Enzymologic<; br>; Interferon-beta  – pharmacology<; br>; Interferon-Stimulated Gene Factor 3  – metabolism<; br>; Interferon-beta/pharmacology<; br>; Matrix Metalloproteinase 9  – genetics<; br>; Matrix Metalloproteinase 9  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Transcriptional Activation  – genetics

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APA (6th Edition):

Zhao, X. (2008). Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,542

Chicago Manual of Style (16th Edition):

Zhao, Xueyan. “Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,542.

MLA Handbook (7th Edition):

Zhao, Xueyan. “Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.” 2008. Web. 18 Oct 2019.

Vancouver:

Zhao X. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,542.

Council of Science Editors:

Zhao X. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,542

16. Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye.

Degree: PhD, 2008, University of Alabama – Birmingham

Development of the vertebrate eye is controlled by a network of regulatory genes including those in the basic loop-helix-loop (bHLH) family. In this study, we… (more)

Subjects/Keywords: Amacrine Cells  – physiology<; br>; Avian Proteins  – metabolism<; br>; Basic Helix-Loop-Helix Transcription Factors  – metabolism<; br>; Gene Expression Regulation, Developmental  – physiology<; br>; Nerve Tissue Proteins  – genetics<; br>; Nuclear Reprogramming<; br>; Retina  – cytology<; br>; Retinal Neurons  – cytology Retinal Pigment Epithelium  – cytology<; br>; Stem Cells  – cytology

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APA (6th Edition):

Mao,Weiming. (2008). The role of bHLH gene ash1 in the developing chick eye. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Web. 18 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

17. Maynard, Craig Lueland. IL-10-competent regulatory T cells: development, phenotype and function.

Degree: PhD, 2007, University of Alabama – Birmingham

In order to avoid chronic cell activation and inflammation the immune system has developed numerous mechanisms that cooperate to induce and/or maintain what is known… (more)

Subjects/Keywords: Cell Differentiation  – immunology<; br>; Forkhead Transcription Factors  – immunology<; br>; Inflammation  – immunology<; br>; Interleukin-10  – metabolism<; br>; T-Lymphocyte Subsets  – cytology<; br>; Transforming Growth Factor beta

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APA (6th Edition):

Maynard, C. L. (2007). IL-10-competent regulatory T cells: development, phenotype and function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,529

Chicago Manual of Style (16th Edition):

Maynard, Craig Lueland. “IL-10-competent regulatory T cells: development, phenotype and function.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,529.

MLA Handbook (7th Edition):

Maynard, Craig Lueland. “IL-10-competent regulatory T cells: development, phenotype and function.” 2007. Web. 18 Oct 2019.

Vancouver:

Maynard CL. IL-10-competent regulatory T cells: development, phenotype and function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,529.

Council of Science Editors:

Maynard CL. IL-10-competent regulatory T cells: development, phenotype and function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,529


University of New South Wales

18. Knights, Alexander. Understanding the transcriptional landscape in immunity and metabolism.

Degree: Biotechnology & Biomolecular Sciences, 2018, University of New South Wales

 Recent decades have heralded a greater appreciation of the intricate crosstalk that exists between immunity and metabolism, significantly expanding therapeutic horizons. A fundamental layer of… (more)

Subjects/Keywords: Gene regulation; Immunity; Metabolism; Transcription factors

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APA (6th Edition):

Knights, A. (2018). Understanding the transcriptional landscape in immunity and metabolism. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60437

Chicago Manual of Style (16th Edition):

Knights, Alexander. “Understanding the transcriptional landscape in immunity and metabolism.” 2018. Doctoral Dissertation, University of New South Wales. Accessed October 18, 2019. http://handle.unsw.edu.au/1959.4/60437.

MLA Handbook (7th Edition):

Knights, Alexander. “Understanding the transcriptional landscape in immunity and metabolism.” 2018. Web. 18 Oct 2019.

Vancouver:

Knights A. Understanding the transcriptional landscape in immunity and metabolism. [Internet] [Doctoral dissertation]. University of New South Wales; 2018. [cited 2019 Oct 18]. Available from: http://handle.unsw.edu.au/1959.4/60437.

Council of Science Editors:

Knights A. Understanding the transcriptional landscape in immunity and metabolism. [Doctoral Dissertation]. University of New South Wales; 2018. Available from: http://handle.unsw.edu.au/1959.4/60437

19. Atout, Reem (Reem N.). Expression of Sox 11 during tooth formation.

Degree: MS, 2009, University of Alabama – Birmingham

Our laboratory has shown that a human autosomal recessive (AR) form of radicular dentin dysplasia (RDD) is caused by an alteration in the transcription factor… (more)

Subjects/Keywords: Dental Pulp Cavity  – abnormalities<; br>; Dentin Dysplasia  – genetics<; br>; NFI Transcription Factors  – genetics<; br>; SOX Transcription Factors  – genetics<; br>; Tooth Root  – abnormalities

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APA (6th Edition):

Atout, R. (. N. ). (2009). Expression of Sox 11 during tooth formation. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1016

Chicago Manual of Style (16th Edition):

Atout, Reem (Reem N ). “Expression of Sox 11 during tooth formation.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1016.

MLA Handbook (7th Edition):

Atout, Reem (Reem N ). “Expression of Sox 11 during tooth formation.” 2009. Web. 18 Oct 2019.

Vancouver:

Atout R(N). Expression of Sox 11 during tooth formation. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1016.

Council of Science Editors:

Atout R(N). Expression of Sox 11 during tooth formation. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1016

20. Laver, Travis. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.

Degree: PhD, 2008, University of Alabama – Birmingham

Interleukin-8 (IL-8) is a potent chemoattractant of numerous cells, particularly neutrophils, in the innate immune response. In addition to immune functions, IL-8 is known to… (more)

Subjects/Keywords: Astrocytoma <; br>; Gene Expression Regulation <; br>; Interferon-beta  – physiology <; br>; Interferon-Stimulated Gene Factor 3, gamma Subunit  – metabolism <; br>; Interleukin-8 <; br>; STAT1 Transcription Factor  – metabolism <; br>; STAT2 Transcription Factor  – metabolism

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APA (6th Edition):

Laver, T. (2008). Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,204

Chicago Manual of Style (16th Edition):

Laver, Travis. “Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,204.

MLA Handbook (7th Edition):

Laver, Travis. “Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.” 2008. Web. 18 Oct 2019.

Vancouver:

Laver T. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,204.

Council of Science Editors:

Laver T. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,204

21. Szul, Tomasz J. The role of GBF1 in Golgi biogenesis and secretory traffic.

Degree: PhD, 2009, University of Alabama – Birmingham

The secretory pathway within a cell consists of series of membrane compartments connected by shuttling secretory vesicles containing cargo that travel from endoplasmic reticulum (ER)… (more)

Subjects/Keywords: ADP-Ribosylation Factors  – metabolism<; br>; Coat Protein Complex I  – metabolism<; br>; Endoplasmic Reticulum  – metabolism<; br>; Golgi Apparatus  – metabolism<; br>; Guanine Nucleotide Exchange Factors  – metabolism<; br>; Membrane Proteins  – metabolism

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APA (6th Edition):

Szul, T. J. (2009). The role of GBF1 in Golgi biogenesis and secretory traffic. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,577

Chicago Manual of Style (16th Edition):

Szul, Tomasz J. “The role of GBF1 in Golgi biogenesis and secretory traffic.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,577.

MLA Handbook (7th Edition):

Szul, Tomasz J. “The role of GBF1 in Golgi biogenesis and secretory traffic.” 2009. Web. 18 Oct 2019.

Vancouver:

Szul TJ. The role of GBF1 in Golgi biogenesis and secretory traffic. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,577.

Council of Science Editors:

Szul TJ. The role of GBF1 in Golgi biogenesis and secretory traffic. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,577

22. Lenarcic, Erik Michael. In vivo interactions between virus genomes and host proteins for members of the order Picornavirales.

Degree: PhD, 2011, University of Alabama – Birmingham

A significant number of pathogens of economical and medical importance are positive sense RNA viruses. Their genomes enter host cells and subvert them to support… (more)

Subjects/Keywords: Eukaryotic Initiation Factor-3<; br>; Peptide Initiation Factors  – metabolism<; br>; Picornaviridae  – chemistry<; br>; Poliovirus<; br>; Protein Biosynthesis<; br>; Ribosomes  – metabolism

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APA (6th Edition):

Lenarcic, E. M. (2011). In vivo interactions between virus genomes and host proteins for members of the order Picornavirales. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,949

Chicago Manual of Style (16th Edition):

Lenarcic, Erik Michael. “In vivo interactions between virus genomes and host proteins for members of the order Picornavirales.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,949.

MLA Handbook (7th Edition):

Lenarcic, Erik Michael. “In vivo interactions between virus genomes and host proteins for members of the order Picornavirales.” 2011. Web. 18 Oct 2019.

Vancouver:

Lenarcic EM. In vivo interactions between virus genomes and host proteins for members of the order Picornavirales. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,949.

Council of Science Editors:

Lenarcic EM. In vivo interactions between virus genomes and host proteins for members of the order Picornavirales. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,949

23. Anderson, Mark Edwin. Exploring Health Disparities For Children In The City Of Milwaukee.

Degree: PhD, 2012, University of Alabama – Birmingham

This study investigated whether school-based oral health programs as a public policy intervention increased dental sealant applications among children from low-income families and minorities in… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes – metabolism.<; br>; Cytokines<; br>; Epigenomics.<; br>; STAT4 Transcription Factor – metabolism.<; br>; T-Box Domain Proteins – metabolism.<; br>; T-Lymphocytes, Helper-Inducer<; br>; Th1 Cells – metabolism.<; br>; Transcription Factor RelA – metabolism.

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APA (6th Edition):

Anderson, M. E. (2012). Exploring Health Disparities For Children In The City Of Milwaukee. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1398

Chicago Manual of Style (16th Edition):

Anderson, Mark Edwin. “Exploring Health Disparities For Children In The City Of Milwaukee.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1398.

MLA Handbook (7th Edition):

Anderson, Mark Edwin. “Exploring Health Disparities For Children In The City Of Milwaukee.” 2012. Web. 18 Oct 2019.

Vancouver:

Anderson ME. Exploring Health Disparities For Children In The City Of Milwaukee. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1398.

Council of Science Editors:

Anderson ME. Exploring Health Disparities For Children In The City Of Milwaukee. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1398


Hong Kong University of Science and Technology

24. Li, Xiumao LIFS. Exploring the roles of PICK1 and ICA69 complex in diseases.

Degree: 2013, Hong Kong University of Science and Technology

 PICK1 (Protein Interacting with C-kinase 1) and ICA69 (Islet Cell Autoantigen 69kD) are BAR (Bin/Amphiphysin/Rvs) domain-containing proteins that tightly associate with each other in brain,… (more)

Subjects/Keywords: Proteins; Metabolism; Physiological transport; Transcription factors; Autism spectrum disorders

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APA (6th Edition):

Li, X. L. (2013). Exploring the roles of PICK1 and ICA69 complex in diseases. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b1274423 ; http://repository.ust.hk/ir/bitstream/1783.1-95273/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Xiumao LIFS. “Exploring the roles of PICK1 and ICA69 complex in diseases.” 2013. Thesis, Hong Kong University of Science and Technology. Accessed October 18, 2019. https://doi.org/10.14711/thesis-b1274423 ; http://repository.ust.hk/ir/bitstream/1783.1-95273/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Xiumao LIFS. “Exploring the roles of PICK1 and ICA69 complex in diseases.” 2013. Web. 18 Oct 2019.

Vancouver:

Li XL. Exploring the roles of PICK1 and ICA69 complex in diseases. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2013. [cited 2019 Oct 18]. Available from: https://doi.org/10.14711/thesis-b1274423 ; http://repository.ust.hk/ir/bitstream/1783.1-95273/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li XL. Exploring the roles of PICK1 and ICA69 complex in diseases. [Thesis]. Hong Kong University of Science and Technology; 2013. Available from: https://doi.org/10.14711/thesis-b1274423 ; http://repository.ust.hk/ir/bitstream/1783.1-95273/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Nichols, Kimberly. Relationships among anger, patterns of anger expression and blood pressure, glucose, and cortisol in overweight school-aged children.

Degree: PhD, 2008, University of Alabama – Birmingham

Overweight and obesity in school-aged children have become a major health issue. With overweight and obesity, children have increased risk of developing elevated blood pressure… (more)

Subjects/Keywords: Blood Pressure <; br>; Child <; br>; Glucose <; br>; Hydrocortisone  – metabolism <; br>; Sex Factors Risk Factors Temperament  – physiology

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APA (6th Edition):

Nichols, K. (2008). Relationships among anger, patterns of anger expression and blood pressure, glucose, and cortisol in overweight school-aged children. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,313

Chicago Manual of Style (16th Edition):

Nichols, Kimberly. “Relationships among anger, patterns of anger expression and blood pressure, glucose, and cortisol in overweight school-aged children.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,313.

MLA Handbook (7th Edition):

Nichols, Kimberly. “Relationships among anger, patterns of anger expression and blood pressure, glucose, and cortisol in overweight school-aged children.” 2008. Web. 18 Oct 2019.

Vancouver:

Nichols K. Relationships among anger, patterns of anger expression and blood pressure, glucose, and cortisol in overweight school-aged children. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,313.

Council of Science Editors:

Nichols K. Relationships among anger, patterns of anger expression and blood pressure, glucose, and cortisol in overweight school-aged children. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,313

26. Maitra, Sushmit. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulated mRNA decay is a highly important process for the tight control of gene expression. Inherently unstable mRNAs contain AU-rich elements (AREs) in the 3’… (more)

Subjects/Keywords: Intracellular Signaling Peptides and Proteins  – metabolism <; br>; Protein-Serine-Threonine Kinases  – metabolism <; br>; RNA, Messenger  – metabolism <; br>; RNA Stability  – physiology <; br>; RNA-Binding Proteins  – metabolism <; br>; TATA-Binding Protein Associated Factors  – metabolism

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APA (6th Edition):

Maitra, S. (2008). The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,379

Chicago Manual of Style (16th Edition):

Maitra, Sushmit. “The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,379.

MLA Handbook (7th Edition):

Maitra, Sushmit. “The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase.” 2008. Web. 18 Oct 2019.

Vancouver:

Maitra S. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,379.

Council of Science Editors:

Maitra S. The AU-rich element mRNA decay-promoting activity of BRF1 is regulated by mitogen-activated protein kinase activated protein kinase. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,379

27. Li, Xingnan. Regulation of [beta]-catenin by Gli1 in epithelial transformation.

Degree: PhD, 2006, University of Alabama – Birmingham

Gli family members-mediated continuous Hedgehog (Hh) pathway activity plays a role in the growth of a number of human cancers, including the common malignancy of… (more)

Subjects/Keywords: beta Catenin  – metabolism <; br>; Cell Transformation, Neoplastic  – genetics <; br>; Oncogene Proteins <; br>; Trans-Activators  – genetics <; br>; Transcription, Genetic

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APA (6th Edition):

Li, X. (2006). Regulation of [beta]-catenin by Gli1 in epithelial transformation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,90

Chicago Manual of Style (16th Edition):

Li, Xingnan. “Regulation of [beta]-catenin by Gli1 in epithelial transformation.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,90.

MLA Handbook (7th Edition):

Li, Xingnan. “Regulation of [beta]-catenin by Gli1 in epithelial transformation.” 2006. Web. 18 Oct 2019.

Vancouver:

Li X. Regulation of [beta]-catenin by Gli1 in epithelial transformation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,90.

Council of Science Editors:

Li X. Regulation of [beta]-catenin by Gli1 in epithelial transformation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,90

28. Atkinson, George P. (George Prescott). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma (GBM) is an incurable tumor of the central nervous system (CNS). Over the past 50 years, little progress has made in improving the quality… (more)

Subjects/Keywords: Glioblastoma  – pathology<; br>; Neoplasms<; br>; NF-kappa B  – metabolism<; br>; Peptidylprolyl Isomerase<; br>; STAT3 Transcription Factor

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APA (6th Edition):

Atkinson, G. P. (. P. (2009). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,812

Chicago Manual of Style (16th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,812.

MLA Handbook (7th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Web. 18 Oct 2019.

Vancouver:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812.

Council of Science Editors:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812

29. Qian, Yingjuan, Ph.D. The role of DEC1 in P53-dependent cellular senescence.

Degree: PhD, 2008, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly mutated gene in human cancers. As a transcription factor, p53 exerts its tumor suppressor function through the… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors  – genetics<; br>; Basic Helix-Loop-Helix Transcription Factors  – physiology<; br>; Cell Aging  – physiology<; br>; DNA Damage<; br>; Tumor Suppressor Protein p53  – physiology<; br>; Tumor Suppressor Proteins  – genetics

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APA (6th Edition):

Qian, Yingjuan, P. D. (2008). The role of DEC1 in P53-dependent cellular senescence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,462

Chicago Manual of Style (16th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,462.

MLA Handbook (7th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Web. 18 Oct 2019.

Vancouver:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462.

Council of Science Editors:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462

30. Bhakta, Sushma Jyotika. A functional analysis of the human immunodeficiency virus envelope glycoprotein cytoplasmic domain.

Degree: PhD, 2009, University of Alabama – Birmingham

The retroviral life cycle is separated into two distinct phases of infection. In the first phase, viral enzymes and proteins allow the virus to establish… (more)

Subjects/Keywords: Amino Acid Motifs<; br>; HIV-1  – physiology<; br>; Mutagenesis, Site-Directed<; br>; Viral Fusion Proteins  – metabolism<; br>; Virulence Factors  – metabolism<; br>; Virus Internalization<; br>; env Gene Products, Human Immunodeficiency Virus  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bhakta, S. J. (2009). A functional analysis of the human immunodeficiency virus envelope glycoprotein cytoplasmic domain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1086

Chicago Manual of Style (16th Edition):

Bhakta, Sushma Jyotika. “A functional analysis of the human immunodeficiency virus envelope glycoprotein cytoplasmic domain.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1086.

MLA Handbook (7th Edition):

Bhakta, Sushma Jyotika. “A functional analysis of the human immunodeficiency virus envelope glycoprotein cytoplasmic domain.” 2009. Web. 18 Oct 2019.

Vancouver:

Bhakta SJ. A functional analysis of the human immunodeficiency virus envelope glycoprotein cytoplasmic domain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1086.

Council of Science Editors:

Bhakta SJ. A functional analysis of the human immunodeficiency virus envelope glycoprotein cytoplasmic domain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1086

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