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You searched for subject:( Retinaldehyde metabolism 60). Showing records 1 – 30 of 11691 total matches.

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1. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

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APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 18 Sep 2019.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

2. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 18 Sep 2019.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956

3. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 18 Sep 2019.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

4. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 18 Sep 2019.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

5. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

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APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 18 Sep 2019.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

6. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 18 Sep 2019.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

7. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

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APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 18 Sep 2019.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

8. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 18 Sep 2019.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

9. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 18 Sep 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

10. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

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APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 18 Sep 2019.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

11. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

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APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 18 Sep 2019.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

12. Durham, Carolyn G. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.

Degree: PhD, 2010, University of Alabama – Birmingham

The inverse correlation between the industrialization and disease prevalence is termed the "hygiene hypothesis." Supporting this, immunological studies show Th1 cytokines modulate Th2 immune responses.… (more)

Subjects/Keywords: Asthma<; br>; Gastric Mucosa  – metabolism<; br>; Gastritis<; br>; Helicobacter Infections  – metabolism<; br>; Helicobacter felis<; br>; Mucus  – metabolism

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APA (6th Edition):

Durham, C. G. (2010). Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,707

Chicago Manual of Style (16th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,707.

MLA Handbook (7th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Web. 18 Sep 2019.

Vancouver:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707.

Council of Science Editors:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707

13. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 18 Sep 2019.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

14. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 18 Sep 2019.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

15. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 18 Sep 2019.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

16. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

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APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 18 Sep 2019.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194

17. Moore, Carlene Drucilla. The role of centaurin alpha-1 in the regulation of neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In the nervous system, PI 3-kinase has been implicated in neuronal differentiation. My studies have focused on a candidate neuronal PI 3-kinase target centaurin alpha-1,… (more)

Subjects/Keywords: 1-Phosphatidylinositol 3-Kinase <; br>; Adaptor Proteins, Signal Transducing  – metabolism <; br>; Dendrites  – metabolism <; br>; Dendrites  – ultrastructure <; br>; GTPase-Activating Proteins  – metabolism <; br>; Hippocampus  – cytology <; br>; Nerve Tissue Proteins  – metabolism <; br>; Neurons  – metabolism

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APA (6th Edition):

Moore, C. D. (2008). The role of centaurin alpha-1 in the regulation of neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,208

Chicago Manual of Style (16th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,208.

MLA Handbook (7th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Web. 18 Sep 2019.

Vancouver:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208.

Council of Science Editors:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208

18. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 18 Sep 2019.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

19. Mavalli, Mahendra D. Mechanisms of growth hormone action in skeletal muscle.

Degree: PhD, 2009, University of Alabama – Birmingham

Growth hormone (GH) and insulin like growth factor-1 (IGF-1) exert profound growth promoting actions during pre and postnatal skeletal muscle development. GH and IGF-1 seem… (more)

Subjects/Keywords: Growth Hormone  – metabolism<; br>; Insulin  – metabolism<; br>; Insulin Resistance<; br>; Muscle, Skeletal  – growth & development<; br>; Muscle, Skeletal  – metabolism<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptors, Somatotropin  – metabolism<; br>; Sarcopenia

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APA (6th Edition):

Mavalli, M. D. (2009). Mechanisms of growth hormone action in skeletal muscle. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1103

Chicago Manual of Style (16th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1103.

MLA Handbook (7th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Web. 18 Sep 2019.

Vancouver:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103.

Council of Science Editors:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103

20. Tang, Yi, Ph.D. The role of transforming growth factor beta-1 in bone remodeling.

Degree: PhD, 2009, University of Alabama – Birmingham

Bone remodeling depends on the precise coordination of bone resorption by the osteoclasts and bone formation by the osteoblasts. It is proposed that osteoclastic bone… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Bone Regeneration  – drug effects<; br>; Cadherins  – metabolism<; br>; Cell Membrane  – metabolism<; br>; Multiprotein Complexes  – metabolism<; br>; Smad7 Protein  – metabolism<; br>; Transforming Growth Factor beta  – metabolism

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APA (6th Edition):

Tang, Yi, P. D. (2009). The role of transforming growth factor beta-1 in bone remodeling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,405

Chicago Manual of Style (16th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,405.

MLA Handbook (7th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Web. 18 Sep 2019.

Vancouver:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405.

Council of Science Editors:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405

21. Balasubramani, Anand. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.

Degree: PhD, 2010, University of Alabama – Birmingham

The ability to differentially manipulate available genetic information in order to generate diverse cellular identities represents an innovation of complex multicellular eukaryotic organisms. Cis-acting modules… (more)

Subjects/Keywords: DNA Replication – physiology.<; br>; Drosophila – metabolism.<; br>; Drosophila Proteins – metabolism.<; br>; GTP Phosphohydrolases – metabolism.<; br>; Microfilament Proteins – metabolism.<; br>; Multiprotein Complexes – metabolism.<; br>; Origin Recognition Complex – metabolism.

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APA (6th Edition):

Balasubramani, A. (2010). Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1428

Chicago Manual of Style (16th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1428.

MLA Handbook (7th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Web. 18 Sep 2019.

Vancouver:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428.

Council of Science Editors:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428

22. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 18 Sep 2019.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

23. Jiang, Shaoning. Molecular mechanisms of hepatic insulin resistance following injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Insulin resistance commonly occurs following injuries or critical illness independent of previous diabetic status. The development of insulin resistance and hyperglycemia is associated with increased… (more)

Subjects/Keywords: Adenoviridae – metabolism<; br>; Adenoviridae Infections – metabolism<; br>; Diabetes Mellitus, Type 2 – physiopathology<; br>; Glucose – metabolism<; br>; I-kappa B Kinase – physiology <; br>; Insulin – metabolism<; br>; JNK Mitogen-Activated Protein Kinases – physiology <; br>; Liver

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APA (6th Edition):

Jiang, S. (2010). Molecular mechanisms of hepatic insulin resistance following injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1374

Chicago Manual of Style (16th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1374.

MLA Handbook (7th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Web. 18 Sep 2019.

Vancouver:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374.

Council of Science Editors:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374

24. Yu, Jei-Hwa. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.

Degree: PhD, 2008, University of Alabama – Birmingham

Papillomaviruses (PV) are prevalent pathogens that infect human or animal squamous epithelia. Its genome is a double strand circular DNA of approximately 7.9 kb. It… (more)

Subjects/Keywords: DNA Helicases  – metabolism <; br>; DNA-Binding Proteins  – metabolism <; br>; Human papillomavirus 11  – physiology <; br>; Mitogen-Activated Protein Kinases  – metabolism <; br>; Nuclear Localization Signals  – metabolism <; br>; Replication Origin <; br>; Viral Proteins  – metabolism

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APA (6th Edition):

Yu, J. (2008). MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,213

Chicago Manual of Style (16th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,213.

MLA Handbook (7th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Web. 18 Sep 2019.

Vancouver:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213.

Council of Science Editors:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213

25. Byon, Chang Hyun. Oxidative stress-stimulated vascular calcification.

Degree: PhD, 2009, University of Alabama – Birmingham

Oxidative stress plays a critical role in pathogenesis of atherosclerosis. However, the effect of oxidative stress-induced molecular signaling in development of vascular calcification, a feature… (more)

Subjects/Keywords: Calcinosis  – metabolism<; br>; Core Binding Factor Alpha 1 Subunit  – metabolism<; br>; Hydrogen Peroxide  – metabolism<; br>; Muscle, smooth, Vascular  – metabolism<; br>; Oxidative Stress<; br>; Proto-Oncogene Proteins c-akt  – metabolism<; br>; Signal Transduction

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APA (6th Edition):

Byon, C. H. (2009). Oxidative stress-stimulated vascular calcification. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,662

Chicago Manual of Style (16th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,662.

MLA Handbook (7th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Web. 18 Sep 2019.

Vancouver:

Byon CH. Oxidative stress-stimulated vascular calcification. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662.

Council of Science Editors:

Byon CH. Oxidative stress-stimulated vascular calcification. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662

26. Zhai, Lidong. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.

Degree: PhD, 2009, University of Alabama – Birmingham

Hyperglycemia develops in the intensive care unit in many patients following injury, infection and critical illness. However, little is known about the mechanism of acute… (more)

Subjects/Keywords: Aging  – metabolism<; br>; Insulin Resistance<; br>; Liver  – metabolism<; br>; Muscle, Skeletal  – metabolism<; br>; Oxidative Stress<; br>; Shock, Hemorrhagic  – metabolism<; br>; Wounds and Injuries  – metabolism

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APA (6th Edition):

Zhai, L. (2009). Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,834

Chicago Manual of Style (16th Edition):

Zhai, Lidong. “Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,834.

MLA Handbook (7th Edition):

Zhai, Lidong. “Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.” 2009. Web. 18 Sep 2019.

Vancouver:

Zhai L. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,834.

Council of Science Editors:

Zhai L. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,834

27. Fulzele, Keertik. Insulin signaling and function in osteoblasts.

Degree: PhD, 2009, University of Alabama – Birmingham

Insulin and insulin-like growth factor-1 (IGF-1) are evolutionarily conserved hormonal signaling pathways with structurally similar ligands and receptors. Recent studies suggest that that insulin and… (more)

Subjects/Keywords: Insulin  – metabolism<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptor, Insulin  – metabolism<; br>; Signal Transduction  – physiology<; br>; Skull  – metabolism

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APA (6th Edition):

Fulzele, K. (2009). Insulin signaling and function in osteoblasts. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,999

Chicago Manual of Style (16th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,999.

MLA Handbook (7th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Web. 18 Sep 2019.

Vancouver:

Fulzele K. Insulin signaling and function in osteoblasts. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999.

Council of Science Editors:

Fulzele K. Insulin signaling and function in osteoblasts. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999

28. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 18 Sep 2019.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

29. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

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APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 18 Sep 2019.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

30. Grabski, Robert. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.

Degree: PhD, 2008, University of Alabama – Birmingham

Metazoan cells are characterized with elaborate network of intracellular membranous compartments. These membranes allow the cell to spatially separate antagonistic processes and environments, and maintain… (more)

Subjects/Keywords: Carrier Proteins  – metabolism <; br>; Golgi Apparatus  – metabolism <; br>; Membrane Proteins  – metabolism <; br>; Protein Transport <; br>; RNA Interference

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APA (6th Edition):

Grabski, R. (2008). Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,328

Chicago Manual of Style (16th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 18, 2019. http://contentdm.mhsl.uab.edu/u?/etd,328.

MLA Handbook (7th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Web. 18 Sep 2019.

Vancouver:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 18]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328.

Council of Science Editors:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328

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