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You searched for subject:( Receptors TNF Related Apoptosis Inducing Ligand agonists). Showing records 1 – 30 of 15400 total matches.

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1. Belenky, Michael L. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.

Degree: PhD, 2011, University of Alabama – Birmingham

Our laboratory reported that a combination of ionizing radiation (IR) or temozolomide (Tmz), a DNA methylating agent clinically approved against glioblastoma multiforme (GBM), a type… (more)

Subjects/Keywords: Antibodies, Monoclonal  – therapeutic use<; br>; Apoptosis  – physiology<; br>; Brain Neoplasms  – drug therapy<; br>; Brain Neoplasms  – radiotherapy<; br>; Cell Cycle<; br>; Glioma  – drug therapy<; br>; Glioma  – radiotherapy<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Belenky, M. L. (2011). Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1032

Chicago Manual of Style (16th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1032.

MLA Handbook (7th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Web. 29 Mar 2020.

Vancouver:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032.

Council of Science Editors:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032

2. Szafran, April Adams. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

The development of molecular imaging technologies has allowed biomedical researchers to study the process of cancer metastasis in animal models of disease. Bioluminescence imaging has… (more)

Subjects/Keywords: Antibodies, Monoclonal  – pharmacology<; br>; Antineoplastic Combined Chemotherapy<; br>; Protocols  – pharmacology<; br>; Bone Neoplasms<; br>; Breast Neoplasms<; br>; Diphosphonates  – pharmacology<; br>; Imidazoles  – pharmacology<; br>; Mammary Neoplasms, Experimental  – pathology<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Szafran, A. A. (2008). The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,927

Chicago Manual of Style (16th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,927.

MLA Handbook (7th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Web. 29 Mar 2020.

Vancouver:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927.

Council of Science Editors:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927

3. Amm, Hope. Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer.

Degree: PhD, 2010, University of Alabama – Birmingham

Breast cancer is the second leading cause of cancer-related death in American women and metastatic breast cancer has a 5-year survival rate of only 26%.… (more)

Subjects/Keywords: Antibodies, Monoclonal  – therapeutic use<; br>; Antineoplastic Combined Chemotherapy Protocols<; br>; Boronic Acids  – administration & dosage<; br>; Breast Neoplasms  – drug therapy<; br>; Doxorubicin  – administration & dosage<; br>; Drug Resistance, Neoplasm<; br>; Pyrazines  – administration & dosage<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Amm, H. (2010). Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,997

Chicago Manual of Style (16th Edition):

Amm, Hope. “Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,997.

MLA Handbook (7th Edition):

Amm, Hope. “Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer.” 2010. Web. 29 Mar 2020.

Vancouver:

Amm H. Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,997.

Council of Science Editors:

Amm H. Mechanisms by which TRA-8 anti-DR5 antibody and chemotherapy enhance cytotoxicity in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,997

4. Crowder, Roslyn Nicole. Death receptor 5 (DR5) and CD19 signaling in B cells.

Degree: PhD, 2008, University of Alabama – Birmingham

This dissertation research describes the independent examination of B cell signaling mediated by two cell surface molecules, CD19 and Death receptor 5 (DR5). In previous… (more)

Subjects/Keywords: Antigens, CD19  – immunology<; br>; Apoptosis<; br>; Lupus Erythematosus, Systemic<; br>; Neoplasms<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand<; br>; Signal Transduction  – immunology

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APA (6th Edition):

Crowder, R. N. (2008). Death receptor 5 (DR5) and CD19 signaling in B cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,751

Chicago Manual of Style (16th Edition):

Crowder, Roslyn Nicole. “Death receptor 5 (DR5) and CD19 signaling in B cells.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,751.

MLA Handbook (7th Edition):

Crowder, Roslyn Nicole. “Death receptor 5 (DR5) and CD19 signaling in B cells.” 2008. Web. 29 Mar 2020.

Vancouver:

Crowder RN. Death receptor 5 (DR5) and CD19 signaling in B cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,751.

Council of Science Editors:

Crowder RN. Death receptor 5 (DR5) and CD19 signaling in B cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,751


University of Oulu

5. Jääskeläinen, M. (Minna). Apoptosis-regulating factors in developing and adult ovaries.

Degree: 2010, University of Oulu

 Abstract Apoptosis plays a crucial part in human ovarian function from fetal development to the end of reproductive potential. Failures in the regulation of ovarian… (more)

Subjects/Keywords: TNF-related apoptosis-inducing ligand; Wnt proteins; apoptosis; caspase 3; estrogens; granulosa cells; oocytes; ovary; proto-oncogen proteins c-Bcl-2; tumor necrosis factor- alpha

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APA (6th Edition):

Jääskeläinen, M. (. (2010). Apoptosis-regulating factors in developing and adult ovaries. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514263477

Chicago Manual of Style (16th Edition):

Jääskeläinen, M (Minna). “Apoptosis-regulating factors in developing and adult ovaries.” 2010. Doctoral Dissertation, University of Oulu. Accessed March 29, 2020. http://urn.fi/urn:isbn:9789514263477.

MLA Handbook (7th Edition):

Jääskeläinen, M (Minna). “Apoptosis-regulating factors in developing and adult ovaries.” 2010. Web. 29 Mar 2020.

Vancouver:

Jääskeläinen M(. Apoptosis-regulating factors in developing and adult ovaries. [Internet] [Doctoral dissertation]. University of Oulu; 2010. [cited 2020 Mar 29]. Available from: http://urn.fi/urn:isbn:9789514263477.

Council of Science Editors:

Jääskeläinen M(. Apoptosis-regulating factors in developing and adult ovaries. [Doctoral Dissertation]. University of Oulu; 2010. Available from: http://urn.fi/urn:isbn:9789514263477


Freie Universität Berlin

6. Knie, Bettina. The influence of the death ligand TRAIL in central and peripheral nervous system after traumatic injury.

Degree: 2011, Freie Universität Berlin

 The death ligand TRAIL has both beneficial and inhibitory effects on neuroregeneration in the central and peripheral nervous system. In neuronal cell cultures the lack… (more)

Subjects/Keywords: TNF-related apoptosis-inducing ligand; central nervous system; peripheral nervous system; traumatic injury; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Knie, B. (2011). The influence of the death ligand TRAIL in central and peripheral nervous system after traumatic injury. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-13844

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Knie, Bettina. “The influence of the death ligand TRAIL in central and peripheral nervous system after traumatic injury.” 2011. Thesis, Freie Universität Berlin. Accessed March 29, 2020. http://dx.doi.org/10.17169/refubium-13844.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Knie, Bettina. “The influence of the death ligand TRAIL in central and peripheral nervous system after traumatic injury.” 2011. Web. 29 Mar 2020.

Vancouver:

Knie B. The influence of the death ligand TRAIL in central and peripheral nervous system after traumatic injury. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2020 Mar 29]. Available from: http://dx.doi.org/10.17169/refubium-13844.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Knie B. The influence of the death ligand TRAIL in central and peripheral nervous system after traumatic injury. [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-13844

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Warke, Rajas V. Molecular Dissection of the Cellular Reponse to Dengue Virus Infection.

Degree: Immunology and Microbiology, Center for Infectious Disease and Vaccine Research, 2008, U of Massachusetts : Med

  The immune response to viral infection involves a complexity of both innate and adaptive pathways at the cellular and the molecular level. There are… (more)

Subjects/Keywords: Dengue Virus; Gene Expression Regulation; Receptors; Cell Surface; TNF-Related Apoptosis-Inducing Ligand; Cells; Chemical Actions and Uses; Genetic Phenomena; Hemic and Immune Systems; Pathology; Viruses

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APA (6th Edition):

Warke, R. V. (2008). Molecular Dissection of the Cellular Reponse to Dengue Virus Infection. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/366

Chicago Manual of Style (16th Edition):

Warke, Rajas V. “Molecular Dissection of the Cellular Reponse to Dengue Virus Infection.” 2008. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 29, 2020. https://escholarship.umassmed.edu/gsbs_diss/366.

MLA Handbook (7th Edition):

Warke, Rajas V. “Molecular Dissection of the Cellular Reponse to Dengue Virus Infection.” 2008. Web. 29 Mar 2020.

Vancouver:

Warke RV. Molecular Dissection of the Cellular Reponse to Dengue Virus Infection. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2008. [cited 2020 Mar 29]. Available from: https://escholarship.umassmed.edu/gsbs_diss/366.

Council of Science Editors:

Warke RV. Molecular Dissection of the Cellular Reponse to Dengue Virus Infection. [Doctoral Dissertation]. U of Massachusetts : Med; 2008. Available from: https://escholarship.umassmed.edu/gsbs_diss/366


University of Melbourne

8. CRETNEY, ERIKA. The role of TNF-related apoptosis-inducing ligand in immune function.

Degree: 2004, University of Melbourne

 Tumor necrosis factor (TNF)-related apoptosis-inducing ligand [TRAIL or Apo-2 ligand (L)] is a member of the TNF superfamily of ligands that can induce cellular responses… (more)

Subjects/Keywords: TRAIL; TNF-related apoptosis-inducing ligand; tumor; NK cells; natural killer cells; EAE; experimental autoimmune encephalomyelitis; death receptors; negative selection; autoimmune disease; lymphoproliferative disorders; immunoregulation

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APA (6th Edition):

CRETNEY, E. (2004). The role of TNF-related apoptosis-inducing ligand in immune function. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36726

Chicago Manual of Style (16th Edition):

CRETNEY, ERIKA. “The role of TNF-related apoptosis-inducing ligand in immune function.” 2004. Doctoral Dissertation, University of Melbourne. Accessed March 29, 2020. http://hdl.handle.net/11343/36726.

MLA Handbook (7th Edition):

CRETNEY, ERIKA. “The role of TNF-related apoptosis-inducing ligand in immune function.” 2004. Web. 29 Mar 2020.

Vancouver:

CRETNEY E. The role of TNF-related apoptosis-inducing ligand in immune function. [Internet] [Doctoral dissertation]. University of Melbourne; 2004. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/11343/36726.

Council of Science Editors:

CRETNEY E. The role of TNF-related apoptosis-inducing ligand in immune function. [Doctoral Dissertation]. University of Melbourne; 2004. Available from: http://hdl.handle.net/11343/36726


Kyoto University / 京都大学

9. Shinmi, Daisuke. Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity : 薬学的活性を改善するための抗体および抗体技術に関する研究.

Degree: 博士(工学), 2018, Kyoto University / 京都大学

新制・論文博士

乙第13145号

論工博第4163号

Subjects/Keywords: antibody; antibody-drug conjugate; site-specific conjugation; carcinoembryonic antigen; TNF-related apoptosis-inducing ligand receptor 2

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APA (6th Edition):

Shinmi, D. (2018). Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity : 薬学的活性を改善するための抗体および抗体技術に関する研究. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/230980 ; http://dx.doi.org/10.14989/doctor.r13145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shinmi, Daisuke. “Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity : 薬学的活性を改善するための抗体および抗体技術に関する研究.” 2018. Thesis, Kyoto University / 京都大学. Accessed March 29, 2020. http://hdl.handle.net/2433/230980 ; http://dx.doi.org/10.14989/doctor.r13145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shinmi, Daisuke. “Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity : 薬学的活性を改善するための抗体および抗体技術に関する研究.” 2018. Web. 29 Mar 2020.

Vancouver:

Shinmi D. Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity : 薬学的活性を改善するための抗体および抗体技術に関する研究. [Internet] [Thesis]. Kyoto University / 京都大学; 2018. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2433/230980 ; http://dx.doi.org/10.14989/doctor.r13145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shinmi D. Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity : 薬学的活性を改善するための抗体および抗体技術に関する研究. [Thesis]. Kyoto University / 京都大学; 2018. Available from: http://hdl.handle.net/2433/230980 ; http://dx.doi.org/10.14989/doctor.r13145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Shinmi, Daisuke. Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity .

Degree: 2018, Kyoto University

Subjects/Keywords: antibody; antibody-drug conjugate; site-specific conjugation; carcinoembryonic antigen; TNF-related apoptosis-inducing ligand receptor 2

Page 1 Page 2 Page 3 Page 4

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APA (6th Edition):

Shinmi, D. (2018). Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/230980

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shinmi, Daisuke. “Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity .” 2018. Thesis, Kyoto University. Accessed March 29, 2020. http://hdl.handle.net/2433/230980.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shinmi, Daisuke. “Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity .” 2018. Web. 29 Mar 2020.

Vancouver:

Shinmi D. Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity . [Internet] [Thesis]. Kyoto University; 2018. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2433/230980.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shinmi D. Characterization of the antibodies and antibody technologies to improve the pharmaceutical activity . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/230980

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

11. Girkin, Jason Leslie Nicholas. The role of rhinovirus and novel molecular mechanisms in allergic airways disease.

Degree: PhD, 2017, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Rhinovirus (RV) infections are common ailments and are the most common precipitant of asthma exacerbations. This thesis contains… (more)

Subjects/Keywords: rhinovirus; asthma; toll-like receptor 7; interferon regulatory factor 7; CC-motiff ligand 7; tumour necrosis factor-related apoptosis-inducing ligand; salmeterol; protein phosphatase 2a; animal models; thesis by publication

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APA (6th Edition):

Girkin, J. L. N. (2017). The role of rhinovirus and novel molecular mechanisms in allergic airways disease. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1335581

Chicago Manual of Style (16th Edition):

Girkin, Jason Leslie Nicholas. “The role of rhinovirus and novel molecular mechanisms in allergic airways disease.” 2017. Doctoral Dissertation, University of Newcastle. Accessed March 29, 2020. http://hdl.handle.net/1959.13/1335581.

MLA Handbook (7th Edition):

Girkin, Jason Leslie Nicholas. “The role of rhinovirus and novel molecular mechanisms in allergic airways disease.” 2017. Web. 29 Mar 2020.

Vancouver:

Girkin JLN. The role of rhinovirus and novel molecular mechanisms in allergic airways disease. [Internet] [Doctoral dissertation]. University of Newcastle; 2017. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/1959.13/1335581.

Council of Science Editors:

Girkin JLN. The role of rhinovirus and novel molecular mechanisms in allergic airways disease. [Doctoral Dissertation]. University of Newcastle; 2017. Available from: http://hdl.handle.net/1959.13/1335581

12. Justus, Calvin Richard. Activation of the proton sensing G-protein coupled receptor, GPR4, regulates focal adhesion dynamics and delays cell spreading due to increased cytoskeletal tension.

Degree: 2013, East Carolina University

 The tumor microenvironment is characteristically acidic due to insufficient blood perfusion, chronic inflammation, hypoxia, and altered cell metabolism. The low pH found in the tumor… (more)

Subjects/Keywords: Oncology; Biology, Cellular; Cellular biology; Rho-Associated Kinases; Melanoma; Receptors, G-Protein-Coupled; Models, Animal; Myosin-Light-Chain Kinase; Y 27632; TNF-Related Apoptosis-Inducing Ligand; GTP-Binding Proteins

…Rho-Kinase STA -Staurosporine TG -Thapsigargin TRAIL -TNF-Related Apoptosis Inducing… …sensitization to TNF-related apoptosisinducing ligand (TRAIL) directed treatments (54… …12-14 G-PROTEIN COUPLED RECEPTORS… …14-17 PROTON SENSING G-PROTEIN COUPLED RECEPTORS… …Exchange Factor GPCR -G-protein Coupled Receptor GRK -G-protein Related Kinase xiv GTP… 

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APA (6th Edition):

Justus, C. R. (2013). Activation of the proton sensing G-protein coupled receptor, GPR4, regulates focal adhesion dynamics and delays cell spreading due to increased cytoskeletal tension. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4246

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Justus, Calvin Richard. “Activation of the proton sensing G-protein coupled receptor, GPR4, regulates focal adhesion dynamics and delays cell spreading due to increased cytoskeletal tension.” 2013. Thesis, East Carolina University. Accessed March 29, 2020. http://hdl.handle.net/10342/4246.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Justus, Calvin Richard. “Activation of the proton sensing G-protein coupled receptor, GPR4, regulates focal adhesion dynamics and delays cell spreading due to increased cytoskeletal tension.” 2013. Web. 29 Mar 2020.

Vancouver:

Justus CR. Activation of the proton sensing G-protein coupled receptor, GPR4, regulates focal adhesion dynamics and delays cell spreading due to increased cytoskeletal tension. [Internet] [Thesis]. East Carolina University; 2013. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/10342/4246.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Justus CR. Activation of the proton sensing G-protein coupled receptor, GPR4, regulates focal adhesion dynamics and delays cell spreading due to increased cytoskeletal tension. [Thesis]. East Carolina University; 2013. Available from: http://hdl.handle.net/10342/4246

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Londono-Joshi, Angelina Ix-Ik'. Investigation Of Death Receptor-5 Mediated Apoptosis And Inhibition Of Wnt/β-Catenin Signaling In Basal-Like Breast Cancer Stem Cells.

Degree: 2013, University of Alabama – Birmingham

Basal-like breast cancers (BLBC) display aggressive clinical behavior attributed to the presence of cancer stem cells (CSCs). TRA-8, a monoclonal antibody that binds specifically to… (more)

Subjects/Keywords: Antibodies, Monoclonal – toxicity.<; /br>; Antineoplastic Agents – toxicity<; /br>; Breast Neoplasms – metabolism<; /br>; Neoplasms, Basal Cell – metabolism.<; /br>; Neoplastic Stem Cells – drug effects.<; /br>; Receptors, TNF-Related Apoptosis-Inducing Ligand – antagonists & inhibitors.

…TOPflash TCF luciferase reporter constructs TRAIL TNF-Related Apoptosis-Inducing Ligand UAB… …related apoptosis-inducing ligand (TRAIL) is a biologically active protein that… …and DR5 induce apoptosis, while the three decoy receptors (receptor 3, receptor 4 and… …This results in arrest of proliferation and induction of apoptosis. When Wnt ligand proteins… …Immunoglobulin G LRP6 Low-density Lipoprotein Receptor-Related Protein 6 mAb Monoclonal Antibody… 

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APA (6th Edition):

Londono-Joshi, A. I. (2013). Investigation Of Death Receptor-5 Mediated Apoptosis And Inhibition Of Wnt/β-Catenin Signaling In Basal-Like Breast Cancer Stem Cells. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1655

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Londono-Joshi, Angelina Ix-Ik'. “Investigation Of Death Receptor-5 Mediated Apoptosis And Inhibition Of Wnt/β-Catenin Signaling In Basal-Like Breast Cancer Stem Cells.” 2013. Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1655.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Londono-Joshi, Angelina Ix-Ik'. “Investigation Of Death Receptor-5 Mediated Apoptosis And Inhibition Of Wnt/β-Catenin Signaling In Basal-Like Breast Cancer Stem Cells.” 2013. Web. 29 Mar 2020.

Vancouver:

Londono-Joshi AI. Investigation Of Death Receptor-5 Mediated Apoptosis And Inhibition Of Wnt/β-Catenin Signaling In Basal-Like Breast Cancer Stem Cells. [Internet] [Thesis]. University of Alabama – Birmingham; 2013. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1655.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Londono-Joshi AI. Investigation Of Death Receptor-5 Mediated Apoptosis And Inhibition Of Wnt/β-Catenin Signaling In Basal-Like Breast Cancer Stem Cells. [Thesis]. University of Alabama – Birmingham; 2013. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1655

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

14. Williams, Paul Daniel. The effect of potential anticancer gene products on breast cancer cell lines.

Degree: MS, Integrated Biosciences, 2013, University of Minnesota

 Cellular or viral delivery of anti-cancer gene therapeutics typically has been investigated as single agents. More recently combinatorial agents have been investigated for which some… (more)

Subjects/Keywords: CHO-K1; Decorin; Interferon Beta; TRAIL; Tumor necrosis factor related apoptotic inducing ligand

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APA (6th Edition):

Williams, P. D. (2013). The effect of potential anticancer gene products on breast cancer cell lines. (Masters Thesis). University of Minnesota. Retrieved from http://hdl.handle.net/11299/162414

Chicago Manual of Style (16th Edition):

Williams, Paul Daniel. “The effect of potential anticancer gene products on breast cancer cell lines.” 2013. Masters Thesis, University of Minnesota. Accessed March 29, 2020. http://hdl.handle.net/11299/162414.

MLA Handbook (7th Edition):

Williams, Paul Daniel. “The effect of potential anticancer gene products on breast cancer cell lines.” 2013. Web. 29 Mar 2020.

Vancouver:

Williams PD. The effect of potential anticancer gene products on breast cancer cell lines. [Internet] [Masters thesis]. University of Minnesota; 2013. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/11299/162414.

Council of Science Editors:

Williams PD. The effect of potential anticancer gene products on breast cancer cell lines. [Masters Thesis]. University of Minnesota; 2013. Available from: http://hdl.handle.net/11299/162414

15. Γεωργιάδου, Μαρία. Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.

Degree: 2011, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Tumor necrosis factor (TNF), a pleiotropic cytokine involved in a broad spectrum of inflammatory and immune responses, exerts its biological effects by engaging two distinct… (more)

Subjects/Keywords: Ηπατοκυτταρικό καρκίνωμα; Οκτρεοτίδη; Υποδοχείς TNF; Απόπτωση; Hepatocellular carcinoma; Kupffer cells; Octreotide; TNF; TNF receptors; p65; Apoptosis

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APA (6th Edition):

Γεωργιάδου, . . (2011). Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/26069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Γεωργιάδου, Μαρία. “Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.” 2011. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed March 29, 2020. http://hdl.handle.net/10442/hedi/26069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Γεωργιάδου, Μαρία. “Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα.” 2011. Web. 29 Mar 2020.

Vancouver:

Γεωργιάδου . Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/10442/hedi/26069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Γεωργιάδου . Μελέτη των υποδοχέων του TNFα στα κύτταρα Kupffer και στο ηπατοκυτταρικό καρκίνωμα. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. Available from: http://hdl.handle.net/10442/hedi/26069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UNIVERSIDADE FEDERAL DE OURO PRETO

16. Jamille Fernandes Lula. Níveis plasmáticos de TNF-alfa, TRAIL e FasLigante/CD95L correlacionados com a disfunção ventricular em pacientes com cardiopatia chagásica crônica OURO PRETO 2008.

Degree: 2008, UNIVERSIDADE FEDERAL DE OURO PRETO

Apoptose de cardiomiócitos tem sido reportado estar envolvido na patogênese da cardiomiopatia chagásica crônica (CCC). Para determinar se a maquinaria apoptótica está correlacionada aos distúrbios… (more)

Subjects/Keywords: apoptose; cardiomiopatia chagásica; TRAIL; Fas ligante; apoptosis; Chagas cardiomyopathy; TNF-alpha; TRAIL; Fas ligand; PARASITOLOGIA; TNF-alfa

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APA (6th Edition):

Lula, J. F. (2008). Níveis plasmáticos de TNF-alfa, TRAIL e FasLigante/CD95L correlacionados com a disfunção ventricular em pacientes com cardiopatia chagásica crônica OURO PRETO 2008. (Thesis). UNIVERSIDADE FEDERAL DE OURO PRETO. Retrieved from http://www.tede.ufop.br/tde_busca/arquivo.php?codArquivo=698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lula, Jamille Fernandes. “Níveis plasmáticos de TNF-alfa, TRAIL e FasLigante/CD95L correlacionados com a disfunção ventricular em pacientes com cardiopatia chagásica crônica OURO PRETO 2008.” 2008. Thesis, UNIVERSIDADE FEDERAL DE OURO PRETO. Accessed March 29, 2020. http://www.tede.ufop.br/tde_busca/arquivo.php?codArquivo=698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lula, Jamille Fernandes. “Níveis plasmáticos de TNF-alfa, TRAIL e FasLigante/CD95L correlacionados com a disfunção ventricular em pacientes com cardiopatia chagásica crônica OURO PRETO 2008.” 2008. Web. 29 Mar 2020.

Vancouver:

Lula JF. Níveis plasmáticos de TNF-alfa, TRAIL e FasLigante/CD95L correlacionados com a disfunção ventricular em pacientes com cardiopatia chagásica crônica OURO PRETO 2008. [Internet] [Thesis]. UNIVERSIDADE FEDERAL DE OURO PRETO; 2008. [cited 2020 Mar 29]. Available from: http://www.tede.ufop.br/tde_busca/arquivo.php?codArquivo=698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lula JF. Níveis plasmáticos de TNF-alfa, TRAIL e FasLigante/CD95L correlacionados com a disfunção ventricular em pacientes com cardiopatia chagásica crônica OURO PRETO 2008. [Thesis]. UNIVERSIDADE FEDERAL DE OURO PRETO; 2008. Available from: http://www.tede.ufop.br/tde_busca/arquivo.php?codArquivo=698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. 秋田,護. Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis : ミトコンドリア分裂阻害剤Mdivi-1によるミトコンドリアの過剰融合を介したヒトがん細胞のTRAIL誘導性アポトーシスに対する増強機構の研究.

Degree: 博士(医学), 2015, Nihon University / 日本大学

Subjects/Keywords: 腫瘍選択制細胞死; tumor-selective killing; TRAIL; tumor necrosis factor-related apoptosis-inducing ligand; Mdivi-1; mitochondrial division inhibitor-1; ミトコンドリアの分裂; mitochondrial fission; 活性酸素; ROS

Page 1

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APA (6th Edition):

秋田,護. (2015). Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis : ミトコンドリア分裂阻害剤Mdivi-1によるミトコンドリアの過剰融合を介したヒトがん細胞のTRAIL誘導性アポトーシスに対する増強機構の研究. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/522 ; http://dx.doi.org/10.15006/32665A4916

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

秋田,護. “Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis : ミトコンドリア分裂阻害剤Mdivi-1によるミトコンドリアの過剰融合を介したヒトがん細胞のTRAIL誘導性アポトーシスに対する増強機構の研究.” 2015. Thesis, Nihon University / 日本大学. Accessed March 29, 2020. http://repository.nihon-u.ac.jp/xmlui/handle/11263/522 ; http://dx.doi.org/10.15006/32665A4916.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

秋田,護. “Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis : ミトコンドリア分裂阻害剤Mdivi-1によるミトコンドリアの過剰融合を介したヒトがん細胞のTRAIL誘導性アポトーシスに対する増強機構の研究.” 2015. Web. 29 Mar 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

秋田,護. Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis : ミトコンドリア分裂阻害剤Mdivi-1によるミトコンドリアの過剰融合を介したヒトがん細胞のTRAIL誘導性アポトーシスに対する増強機構の研究. [Internet] [Thesis]. Nihon University / 日本大学; 2015. [cited 2020 Mar 29]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/522 ; http://dx.doi.org/10.15006/32665A4916.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

秋田,護. Mitochondrial division inhibitor-1 induces mitochondrial hyperfusion and sensitizes human cancer cells to TRAIL-induced apoptosis : ミトコンドリア分裂阻害剤Mdivi-1によるミトコンドリアの過剰融合を介したヒトがん細胞のTRAIL誘導性アポトーシスに対する増強機構の研究. [Thesis]. Nihon University / 日本大学; 2015. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/522 ; http://dx.doi.org/10.15006/32665A4916

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

18. Jheng, Huei-Ying. Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

 The study was to evaluate the effect of ABT-751 on apoptosis and autophagy in Hep-3B cells, a human hepatocellular carcinoma (HCC)-derived cell line. ABT-751 is… (more)

Subjects/Keywords: Microtubules; apoptosis; apoptosis-inducing factor; autophagy; caspase-independent pathway; ABT-751

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APA (6th Edition):

Jheng, H. (2014). Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jheng, Huei-Ying. “Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells.” 2014. Thesis, NSYSU. Accessed March 29, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jheng, Huei-Ying. “Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells.” 2014. Web. 29 Mar 2020.

Vancouver:

Jheng H. Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2020 Mar 29]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jheng H. Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Hansen, Stephen T. Nicotinic acetylcholine receptors and cocaine reward.

Degree: PhD, 2008, Oregon Health Sciences University

Subjects/Keywords: Cocaine-Related Disorders  – therapy; Self Administration; Receptors, Nicotinic; Nicotinic Agonists

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APA (6th Edition):

Hansen, S. T. (2008). Nicotinic acetylcholine receptors and cocaine reward. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M41C1TVF ; http://digitalcommons.ohsu.edu/etd/526

Chicago Manual of Style (16th Edition):

Hansen, Stephen T. “Nicotinic acetylcholine receptors and cocaine reward.” 2008. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 29, 2020. doi:10.6083/M41C1TVF ; http://digitalcommons.ohsu.edu/etd/526.

MLA Handbook (7th Edition):

Hansen, Stephen T. “Nicotinic acetylcholine receptors and cocaine reward.” 2008. Web. 29 Mar 2020.

Vancouver:

Hansen ST. Nicotinic acetylcholine receptors and cocaine reward. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2008. [cited 2020 Mar 29]. Available from: doi:10.6083/M41C1TVF ; http://digitalcommons.ohsu.edu/etd/526.

Council of Science Editors:

Hansen ST. Nicotinic acetylcholine receptors and cocaine reward. [Doctoral Dissertation]. Oregon Health Sciences University; 2008. Available from: doi:10.6083/M41C1TVF ; http://digitalcommons.ohsu.edu/etd/526


University of Ontario Institute of Technology

20. Kaji, Mark. A molecular characterization of agonists that bind to Hco-UNC-49, a GABA-gated chloride channel from Haemonchus contortus.

Degree: 2012, University of Ontario Institute of Technology

 Haemonchus contortus is a blood feeding parasitic nematode infecting ruminants causing anemia and poor health at great economic cost. The ability to pharmaceutically control infection… (more)

Subjects/Keywords: Haemonchus contortus; GABAA agonists; GABAA receptors; Homology modelling; Cys-loop receptors

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APA (6th Edition):

Kaji, M. (2012). A molecular characterization of agonists that bind to Hco-UNC-49, a GABA-gated chloride channel from Haemonchus contortus. (Thesis). University of Ontario Institute of Technology. Retrieved from http://hdl.handle.net/10155/298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kaji, Mark. “A molecular characterization of agonists that bind to Hco-UNC-49, a GABA-gated chloride channel from Haemonchus contortus.” 2012. Thesis, University of Ontario Institute of Technology. Accessed March 29, 2020. http://hdl.handle.net/10155/298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kaji, Mark. “A molecular characterization of agonists that bind to Hco-UNC-49, a GABA-gated chloride channel from Haemonchus contortus.” 2012. Web. 29 Mar 2020.

Vancouver:

Kaji M. A molecular characterization of agonists that bind to Hco-UNC-49, a GABA-gated chloride channel from Haemonchus contortus. [Internet] [Thesis]. University of Ontario Institute of Technology; 2012. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/10155/298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kaji M. A molecular characterization of agonists that bind to Hco-UNC-49, a GABA-gated chloride channel from Haemonchus contortus. [Thesis]. University of Ontario Institute of Technology; 2012. Available from: http://hdl.handle.net/10155/298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

21. Prtenjaca, Anita. Cerebellar degeneration in harlequin mice is associated with inflammation unaltered by low-dose phenobarbital treatment.

Degree: 2012, University of Western Ontario

 Canadian population demographics are shifting to an increase in aged individuals and an increase in the prevalence of neurodegenerative diseases. The post-mitotic nature of most… (more)

Subjects/Keywords: harlequin mouse; cerebellar degeneration; Apoptosis-inducing factor; phenobarbital; hormesis; inflammation; Biology

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APA (6th Edition):

Prtenjaca, A. (2012). Cerebellar degeneration in harlequin mice is associated with inflammation unaltered by low-dose phenobarbital treatment. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Prtenjaca, Anita. “Cerebellar degeneration in harlequin mice is associated with inflammation unaltered by low-dose phenobarbital treatment.” 2012. Thesis, University of Western Ontario. Accessed March 29, 2020. https://ir.lib.uwo.ca/etd/1068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Prtenjaca, Anita. “Cerebellar degeneration in harlequin mice is associated with inflammation unaltered by low-dose phenobarbital treatment.” 2012. Web. 29 Mar 2020.

Vancouver:

Prtenjaca A. Cerebellar degeneration in harlequin mice is associated with inflammation unaltered by low-dose phenobarbital treatment. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2020 Mar 29]. Available from: https://ir.lib.uwo.ca/etd/1068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Prtenjaca A. Cerebellar degeneration in harlequin mice is associated with inflammation unaltered by low-dose phenobarbital treatment. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/1068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

22. Wong, Aaron. Dissecting The Role Of TNFα In Kawasaki Disease: Alteration Of Cell Fate By TNFα After Superantigen Activation.

Degree: 2011, University of Toronto

Kawasaki disease (KD) is an acute inflammatory disease characterized by persistent inflammation of the coronary arteries. KD is characterized by the release of cytokines such… (more)

Subjects/Keywords: Kawasaki Disease; TNF; Superantigen; apoptosis; 0982; 0379

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APA (6th Edition):

Wong, A. (2011). Dissecting The Role Of TNFα In Kawasaki Disease: Alteration Of Cell Fate By TNFα After Superantigen Activation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/31634

Chicago Manual of Style (16th Edition):

Wong, Aaron. “Dissecting The Role Of TNFα In Kawasaki Disease: Alteration Of Cell Fate By TNFα After Superantigen Activation.” 2011. Masters Thesis, University of Toronto. Accessed March 29, 2020. http://hdl.handle.net/1807/31634.

MLA Handbook (7th Edition):

Wong, Aaron. “Dissecting The Role Of TNFα In Kawasaki Disease: Alteration Of Cell Fate By TNFα After Superantigen Activation.” 2011. Web. 29 Mar 2020.

Vancouver:

Wong A. Dissecting The Role Of TNFα In Kawasaki Disease: Alteration Of Cell Fate By TNFα After Superantigen Activation. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/1807/31634.

Council of Science Editors:

Wong A. Dissecting The Role Of TNFα In Kawasaki Disease: Alteration Of Cell Fate By TNFα After Superantigen Activation. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31634


University of Cincinnati

23. Waltmann, Meaghan D. Apolipoprotein E receptor 2 deficiency alters smooth muscle cell and macrophage characteristics to promote atherosclerotic lesion necrosis.

Degree: PhD, Medicine: Pathobiology and Molecular Medicine, 2013, University of Cincinnati

 Cardiovascular disease is the leading cause of death in the U.S. Atherosclerosis, a progressive, multi-factorial disease that is characterized by the accumulation of lipoprotein particles,… (more)

Subjects/Keywords: Pathology; lipoprotein receptors; atherosclerosis; apoptosis

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APA (6th Edition):

Waltmann, M. D. (2013). Apolipoprotein E receptor 2 deficiency alters smooth muscle cell and macrophage characteristics to promote atherosclerotic lesion necrosis. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1384851103

Chicago Manual of Style (16th Edition):

Waltmann, Meaghan D. “Apolipoprotein E receptor 2 deficiency alters smooth muscle cell and macrophage characteristics to promote atherosclerotic lesion necrosis.” 2013. Doctoral Dissertation, University of Cincinnati. Accessed March 29, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1384851103.

MLA Handbook (7th Edition):

Waltmann, Meaghan D. “Apolipoprotein E receptor 2 deficiency alters smooth muscle cell and macrophage characteristics to promote atherosclerotic lesion necrosis.” 2013. Web. 29 Mar 2020.

Vancouver:

Waltmann MD. Apolipoprotein E receptor 2 deficiency alters smooth muscle cell and macrophage characteristics to promote atherosclerotic lesion necrosis. [Internet] [Doctoral dissertation]. University of Cincinnati; 2013. [cited 2020 Mar 29]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1384851103.

Council of Science Editors:

Waltmann MD. Apolipoprotein E receptor 2 deficiency alters smooth muscle cell and macrophage characteristics to promote atherosclerotic lesion necrosis. [Doctoral Dissertation]. University of Cincinnati; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1384851103


University of Florida

24. Chen, Chao. [3H]-Dihydroerysovine As a Molecular Probe of the Resting State Alpha4beta2 Nicotinic Acetylcholine Receptor.

Degree: MS, Medical Sciences - Medicine, 2009, University of Florida

 [3H]-DIHYDROERYSOVINE AS A MOLECULAR PROBE OF THE RESTING STATE ALPHA4BETA2 NICOTINIC ACETYLCHOLINE RECEPTOR The ?4?2 neuronal nicotinic acetylcholine receptor (nAChR) is the most abundant heteromeric… (more)

Subjects/Keywords: Agonists; Alkaloids; Binding sites; Cell membranes; Ligands; Nicotinic receptors; Oocytes; pH; Rats; Receptors

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APA (6th Edition):

Chen, C. (2009). [3H]-Dihydroerysovine As a Molecular Probe of the Resting State Alpha4beta2 Nicotinic Acetylcholine Receptor. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0025079

Chicago Manual of Style (16th Edition):

Chen, Chao. “[3H]-Dihydroerysovine As a Molecular Probe of the Resting State Alpha4beta2 Nicotinic Acetylcholine Receptor.” 2009. Masters Thesis, University of Florida. Accessed March 29, 2020. http://ufdc.ufl.edu/UFE0025079.

MLA Handbook (7th Edition):

Chen, Chao. “[3H]-Dihydroerysovine As a Molecular Probe of the Resting State Alpha4beta2 Nicotinic Acetylcholine Receptor.” 2009. Web. 29 Mar 2020.

Vancouver:

Chen C. [3H]-Dihydroerysovine As a Molecular Probe of the Resting State Alpha4beta2 Nicotinic Acetylcholine Receptor. [Internet] [Masters thesis]. University of Florida; 2009. [cited 2020 Mar 29]. Available from: http://ufdc.ufl.edu/UFE0025079.

Council of Science Editors:

Chen C. [3H]-Dihydroerysovine As a Molecular Probe of the Resting State Alpha4beta2 Nicotinic Acetylcholine Receptor. [Masters Thesis]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0025079


University of Florida

25. Sun, Zhuming. Novel Phenylaminotetralin (PAT) Analogs Multifunctional Serotonin 5HT2 Receptor Drugs for Neuropsychiatric Disorders.

Degree: PhD, Pharmaceutical Sciences - Medicinal Chemistry, 2010, University of Florida

 NOVEL PHENYLAMINOTETRALIN (PAT) ANALOGS: MULTIFUNCTIONAL SEROTONIN 5HT2 RECEPTOR DRUGS FOR NEUROPSYCHIATRIC DISORDERS By Zhuming Sun August 2010 Chair: Raymond Booth Major: Pharmaceutical Science Medicinal Chemistry… (more)

Subjects/Keywords: Agonists; Amines; Antipsychotic agents; Hexanes; Isomers; Ligands; Phenyls; Receptors; Serotonin receptors; Solvents; 5ht2c, agonist, neurodisorders

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sun, Z. (2010). Novel Phenylaminotetralin (PAT) Analogs Multifunctional Serotonin 5HT2 Receptor Drugs for Neuropsychiatric Disorders. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0042056

Chicago Manual of Style (16th Edition):

Sun, Zhuming. “Novel Phenylaminotetralin (PAT) Analogs Multifunctional Serotonin 5HT2 Receptor Drugs for Neuropsychiatric Disorders.” 2010. Doctoral Dissertation, University of Florida. Accessed March 29, 2020. http://ufdc.ufl.edu/UFE0042056.

MLA Handbook (7th Edition):

Sun, Zhuming. “Novel Phenylaminotetralin (PAT) Analogs Multifunctional Serotonin 5HT2 Receptor Drugs for Neuropsychiatric Disorders.” 2010. Web. 29 Mar 2020.

Vancouver:

Sun Z. Novel Phenylaminotetralin (PAT) Analogs Multifunctional Serotonin 5HT2 Receptor Drugs for Neuropsychiatric Disorders. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2020 Mar 29]. Available from: http://ufdc.ufl.edu/UFE0042056.

Council of Science Editors:

Sun Z. Novel Phenylaminotetralin (PAT) Analogs Multifunctional Serotonin 5HT2 Receptor Drugs for Neuropsychiatric Disorders. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0042056


University of Florida

26. Kasper, James M. Characterization of the Effects of Novel 5-Ht2c Receptor Agonists on Neurotransmission and Voluntary Alcohol Consumption in Rats.

Degree: PhD, Pharmaceutical Sciences - Pharmacodynamics, 2012, University of Florida

 This dissertation project studies the ability of 5-HT2C receptor modulators to alter voluntary ethanol intake and to investigate the changes in neurotransmission that accompany 5-HT2C… (more)

Subjects/Keywords: Agonists; Alcoholism; Dosage; Ethanol; Gelatins; Gels; Neurons; Rats; Receptors; Serotonin receptors; addiction  – alcoholism  – operant  – serotonin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kasper, J. M. (2012). Characterization of the Effects of Novel 5-Ht2c Receptor Agonists on Neurotransmission and Voluntary Alcohol Consumption in Rats. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044635

Chicago Manual of Style (16th Edition):

Kasper, James M. “Characterization of the Effects of Novel 5-Ht2c Receptor Agonists on Neurotransmission and Voluntary Alcohol Consumption in Rats.” 2012. Doctoral Dissertation, University of Florida. Accessed March 29, 2020. http://ufdc.ufl.edu/UFE0044635.

MLA Handbook (7th Edition):

Kasper, James M. “Characterization of the Effects of Novel 5-Ht2c Receptor Agonists on Neurotransmission and Voluntary Alcohol Consumption in Rats.” 2012. Web. 29 Mar 2020.

Vancouver:

Kasper JM. Characterization of the Effects of Novel 5-Ht2c Receptor Agonists on Neurotransmission and Voluntary Alcohol Consumption in Rats. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2020 Mar 29]. Available from: http://ufdc.ufl.edu/UFE0044635.

Council of Science Editors:

Kasper JM. Characterization of the Effects of Novel 5-Ht2c Receptor Agonists on Neurotransmission and Voluntary Alcohol Consumption in Rats. [Doctoral Dissertation]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/UFE0044635


University of Wollongong

27. Inamdar, Amar P. Ligand-based pharmacophore studies in the dopaminergic system.

Degree: PhD, 2011, University of Wollongong

  The dopamine receptor complex and the dopamine transporter system (DAT) are implicated in neuropsychological disorders. The aim of our research project was to design,… (more)

Subjects/Keywords: pharmacophores; ligand synthesis; dopamine receptors; transporters

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Inamdar, A. P. (2011). Ligand-based pharmacophore studies in the dopaminergic system. (Doctoral Dissertation). University of Wollongong. Retrieved from 0304 MEDICINAL AND BIOMOLECULAR CHEMISTRY, 0305 ORGANIC CHEMISTRY ; https://ro.uow.edu.au/theses/3535

Chicago Manual of Style (16th Edition):

Inamdar, Amar P. “Ligand-based pharmacophore studies in the dopaminergic system.” 2011. Doctoral Dissertation, University of Wollongong. Accessed March 29, 2020. 0304 MEDICINAL AND BIOMOLECULAR CHEMISTRY, 0305 ORGANIC CHEMISTRY ; https://ro.uow.edu.au/theses/3535.

MLA Handbook (7th Edition):

Inamdar, Amar P. “Ligand-based pharmacophore studies in the dopaminergic system.” 2011. Web. 29 Mar 2020.

Vancouver:

Inamdar AP. Ligand-based pharmacophore studies in the dopaminergic system. [Internet] [Doctoral dissertation]. University of Wollongong; 2011. [cited 2020 Mar 29]. Available from: 0304 MEDICINAL AND BIOMOLECULAR CHEMISTRY, 0305 ORGANIC CHEMISTRY ; https://ro.uow.edu.au/theses/3535.

Council of Science Editors:

Inamdar AP. Ligand-based pharmacophore studies in the dopaminergic system. [Doctoral Dissertation]. University of Wollongong; 2011. Available from: 0304 MEDICINAL AND BIOMOLECULAR CHEMISTRY, 0305 ORGANIC CHEMISTRY ; https://ro.uow.edu.au/theses/3535


Rochester Institute of Technology

28. Myers, Laurel. Mechanisms of psoriatic arthritis.

Degree: 2012, Rochester Institute of Technology

  Psoriasis (Ps) is the most common chronic autoimmune disease in the United States. The immune system releases proinflammatory cytokines and growth factors that accelerate… (more)

Subjects/Keywords: Osteclast; Osteoblast; Osteocytes; Psoriatic arthritis; RANK ligand; TNF-alpha

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Myers, L. (2012). Mechanisms of psoriatic arthritis. (Thesis). Rochester Institute of Technology. Retrieved from https://scholarworks.rit.edu/theses/4319

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Myers, Laurel. “Mechanisms of psoriatic arthritis.” 2012. Thesis, Rochester Institute of Technology. Accessed March 29, 2020. https://scholarworks.rit.edu/theses/4319.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Myers, Laurel. “Mechanisms of psoriatic arthritis.” 2012. Web. 29 Mar 2020.

Vancouver:

Myers L. Mechanisms of psoriatic arthritis. [Internet] [Thesis]. Rochester Institute of Technology; 2012. [cited 2020 Mar 29]. Available from: https://scholarworks.rit.edu/theses/4319.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Myers L. Mechanisms of psoriatic arthritis. [Thesis]. Rochester Institute of Technology; 2012. Available from: https://scholarworks.rit.edu/theses/4319

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. SILVA, Maria Leonilda Gondim. Caracterização Molecular do mecanismo de morte celular programada via TNF Alfa/TNFR1 na resposta ao tratamento antirretroviral na Infecção pelo Vírus da Imunodeficiência Humana Tipo 1 (HIV-1) .

Degree: 2016, Universidade Federal de Pernambuco

 A infecção pelo Vírus da Imunodeficiência Humana 1 (HIV-1) tem como característica clássica a depleção de linfócitos T (LT) CD4+, que quando não tratada culmina… (more)

Subjects/Keywords: Apoptose; SNPs; TNF-α/TNFR1; HIV; TARV; Apoptosis; SNPs; TNF-α / TNFR1; HIV; HAART

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SILVA, M. L. G. (2016). Caracterização Molecular do mecanismo de morte celular programada via TNF Alfa/TNFR1 na resposta ao tratamento antirretroviral na Infecção pelo Vírus da Imunodeficiência Humana Tipo 1 (HIV-1) . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/17867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SILVA, Maria Leonilda Gondim. “Caracterização Molecular do mecanismo de morte celular programada via TNF Alfa/TNFR1 na resposta ao tratamento antirretroviral na Infecção pelo Vírus da Imunodeficiência Humana Tipo 1 (HIV-1) .” 2016. Thesis, Universidade Federal de Pernambuco. Accessed March 29, 2020. http://repositorio.ufpe.br/handle/123456789/17867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SILVA, Maria Leonilda Gondim. “Caracterização Molecular do mecanismo de morte celular programada via TNF Alfa/TNFR1 na resposta ao tratamento antirretroviral na Infecção pelo Vírus da Imunodeficiência Humana Tipo 1 (HIV-1) .” 2016. Web. 29 Mar 2020.

Vancouver:

SILVA MLG. Caracterização Molecular do mecanismo de morte celular programada via TNF Alfa/TNFR1 na resposta ao tratamento antirretroviral na Infecção pelo Vírus da Imunodeficiência Humana Tipo 1 (HIV-1) . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2016. [cited 2020 Mar 29]. Available from: http://repositorio.ufpe.br/handle/123456789/17867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SILVA MLG. Caracterização Molecular do mecanismo de morte celular programada via TNF Alfa/TNFR1 na resposta ao tratamento antirretroviral na Infecção pelo Vírus da Imunodeficiência Humana Tipo 1 (HIV-1) . [Thesis]. Universidade Federal de Pernambuco; 2016. Available from: http://repositorio.ufpe.br/handle/123456789/17867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Papazian, Irini. Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ.

Degree: 2019, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

TNF is a member of a large family of cytokines and has multiple roles in the CNS. Regulated TNF production is necessary for successful host… (more)

Subjects/Keywords: TNF υποδοχείς; TNF; Νευροεκφυλισμός; Νευροπροστασία; Σκλήρυνση κατά πλάκας; Γλουταμινικοί υποδοχείς; Απομυελίνωση απο κουπριζόνη; Πειραματική αυτοάνοση εγκεφαλομυελίτιδα; TNF receptors; TNF; Neurodegeneration; Neuroprotection; Multiple sclerosis; Glutamate receptors; Cuprizone model; Experimental autoimmune encephalomyelitis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Papazian, I. (2019). Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/45595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Papazian, Irini. “Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ.” 2019. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 29, 2020. http://hdl.handle.net/10442/hedi/45595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Papazian, Irini. “Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ.” 2019. Web. 29 Mar 2020.

Vancouver:

Papazian I. Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2019. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/10442/hedi/45595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Papazian I. Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2019. Available from: http://hdl.handle.net/10442/hedi/45595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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