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You searched for subject:( Receptors Somatotropin metabolism 60). Showing records 1 – 30 of 16143 total matches.

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1. Deng, Luqin. Determinants of growth hormone receptor downregulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Growth hormone (GH), a 22 kD polypeptide primarily produced in the anterior pituitary gland, is a key regulator of postnatal growth and affects carbohydrate, protein… (more)

Subjects/Keywords: Down-Regulation  – drug effects<; br>; Human Growth Hormone  – pharmacology<; br>; Janus Kinase 2  – chemistry<; br>; Janus Kinase 2  – genetics<; br>; Janus Kinase 2  – metabolism<; br>; Receptors, Somatotropin  – genetics<; br>; Receptors, Somatotropin  – metabolism

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APA (6th Edition):

Deng, L. (2008). Determinants of growth hormone receptor downregulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,389

Chicago Manual of Style (16th Edition):

Deng, Luqin. “Determinants of growth hormone receptor downregulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,389.

MLA Handbook (7th Edition):

Deng, Luqin. “Determinants of growth hormone receptor downregulation.” 2008. Web. 20 Oct 2019.

Vancouver:

Deng L. Determinants of growth hormone receptor downregulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,389.

Council of Science Editors:

Deng L. Determinants of growth hormone receptor downregulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,389

2. Mavalli, Mahendra D. Mechanisms of growth hormone action in skeletal muscle.

Degree: PhD, 2009, University of Alabama – Birmingham

Growth hormone (GH) and insulin like growth factor-1 (IGF-1) exert profound growth promoting actions during pre and postnatal skeletal muscle development. GH and IGF-1 seem… (more)

Subjects/Keywords: Growth Hormone  – metabolism<; br>; Insulin  – metabolism<; br>; Insulin Resistance<; br>; Muscle, Skeletal  – growth & development<; br>; Muscle, Skeletal  – metabolism<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptors, Somatotropin  – metabolism<; br>; Sarcopenia

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APA (6th Edition):

Mavalli, M. D. (2009). Mechanisms of growth hormone action in skeletal muscle. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1103

Chicago Manual of Style (16th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1103.

MLA Handbook (7th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Web. 20 Oct 2019.

Vancouver:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103.

Council of Science Editors:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103

3. Cowan, Jon Walter. Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate.

Degree: PhD, 2007, University of Alabama – Birmingham

Growth hormone (GH) is a powerful promoter of postnatal longitudinal bone growth in mammals. GH signaling is mediated exclusively by the GH receptor (GHR), and… (more)

Subjects/Keywords: Acetylcysteine  – analogs & derivatives<; br>; ADAM Proteins<; br>; Growth Hormone  – metabolism<; br>; Membrane Proteins  – chemistry<; br>; Protein-Tyrosine Kinases  – pharmacology<; br>; Receptors, Somatotropin  – metabolism

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APA (6th Edition):

Cowan, J. W. (2007). Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,513

Chicago Manual of Style (16th Edition):

Cowan, Jon Walter. “Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,513.

MLA Handbook (7th Edition):

Cowan, Jon Walter. “Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate.” 2007. Web. 20 Oct 2019.

Vancouver:

Cowan JW. Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,513.

Council of Science Editors:

Cowan JW. Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,513

4. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

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APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 20 Oct 2019.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

5. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 20 Oct 2019.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

6. Sweetwyne, Mariya T. Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1.

Degree: PhD, 2009, University of Alabama – Birmingham

Thrombospondin-1 (TSP1) is a multifunctional matricellular protein released by platelets in response to injury and secreted by cells under stress. TSP1 is cleaved into functional… (more)

Subjects/Keywords: Anoikis  – physiology<; br>; Calreticulin  – metabolism<; br>; Fibroplasts  – metabolism<; br>; Mice, Knockout<; br>; Receptors, LDL  – agonists<; br>; Thrombospondin 1  – pharmacology

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APA (6th Edition):

Sweetwyne, M. T. (2009). Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,747

Chicago Manual of Style (16th Edition):

Sweetwyne, Mariya T. “Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,747.

MLA Handbook (7th Edition):

Sweetwyne, Mariya T. “Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1.” 2009. Web. 20 Oct 2019.

Vancouver:

Sweetwyne MT. Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,747.

Council of Science Editors:

Sweetwyne MT. Regulation of tissue remodeling through the calreticulin binding domain of thrombospondin-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,747

7. Read, Russell W. The role of complement in experimental autoimmune uveitis.

Degree: PhD, 2007, University of Alabama – Birmingham

The complement system has been increasingly implicated in the pathophysiology of autoimmune disease. Complement expression in the normal human eye had not been previously completely… (more)

Subjects/Keywords: Autoimmune Diseases  – immunology <; br>; Complement C3  – deficiency <; br>; Receptors, Complement  – metabolism <; br>; Retina  – metabolism <; br>; Uveitis  – immunology

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APA (6th Edition):

Read, R. W. (2007). The role of complement in experimental autoimmune uveitis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,137

Chicago Manual of Style (16th Edition):

Read, Russell W. “The role of complement in experimental autoimmune uveitis.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,137.

MLA Handbook (7th Edition):

Read, Russell W. “The role of complement in experimental autoimmune uveitis.” 2007. Web. 20 Oct 2019.

Vancouver:

Read RW. The role of complement in experimental autoimmune uveitis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,137.

Council of Science Editors:

Read RW. The role of complement in experimental autoimmune uveitis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,137

8. Genovese, Nicholas J. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.

Degree: PhD, 2010, University of Alabama – Birmingham

Though human papillomavirus infection of the human epidermis is epidemiologically widespread and typically benign, manipulation of the cell cycle within host tissues during infections can… (more)

Subjects/Keywords: Cell Cycle<; br>; Cell Transformation, Viral<; br>; Human papillomavirus 16  – metabolism<; br>; Keratinocytes<; br>; Oncogene Proteins, Viral  – metabolism<; br>; Papillomaviridae  – physiology<; br>; Papillomavirus E7 Proteins  – metabolism<; br>; Receptors, Estrogen  – metabolism<; br>; Retinoblastoma-Like Protein p130  – metabolism<; br>; S Phase

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APA (6th Edition):

Genovese, N. J. (2010). The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1158

Chicago Manual of Style (16th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1158.

MLA Handbook (7th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Web. 20 Oct 2019.

Vancouver:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158.

Council of Science Editors:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158

9. Taylor, Erica Wynette. The Role Of O-Glcnacylation In Synaptic Function.

Degree: 2013, University of Alabama – Birmingham

O-GlcNAcylation is a dynamic protein posttranslational modification that adds the monosaccharide β-N-acetylglucosamine (GlcNAc) to specific serine and threonine residues on nucleocytoplasmic proteins. The hippocampus is… (more)

Subjects/Keywords: Acetylglucosaminidase – metabolism.<; br>; Hippocampus – metabolism<; br>; Long-Term Synaptic Depression – physiology.<; br>; N-Acetylglucosaminyltransferases – metabolism<; br>; Receptors, AMPA – metabolism.<; br>; Synapses – metabolism.

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APA (6th Edition):

Taylor, E. W. (2013). The Role Of O-Glcnacylation In Synaptic Function. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taylor, Erica Wynette. “The Role Of O-Glcnacylation In Synaptic Function.” 2013. Thesis, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taylor, Erica Wynette. “The Role Of O-Glcnacylation In Synaptic Function.” 2013. Web. 20 Oct 2019.

Vancouver:

Taylor EW. The Role Of O-Glcnacylation In Synaptic Function. [Internet] [Thesis]. University of Alabama – Birmingham; 2013. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taylor EW. The Role Of O-Glcnacylation In Synaptic Function. [Thesis]. University of Alabama – Birmingham; 2013. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Schreeder, Daniel M. Biological characterization of Fc receptor-like 6 (FCRL6).

Degree: PhD, 2009, University of Alabama – Birmingham

Members of the Fc receptor-like (FCRL) family are cell-surface proteins with ancient conservation, distinct lymphocyte expression patterns, and tyrosine-based signaling capabilities that imply a fundamental… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes  – immunology<; br>; HLA-DR Antigens  – metabolism<; br>; Killer Cells, Natural  – immunology<; br>; Leukemia, Lymphocytic, Chronic, B-Cell  – immunology<; br>; Receptors, Cell Surface  – physiology<; br>; Receptors, Fc  – metabolism

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APA (6th Edition):

Schreeder, D. M. (2009). Biological characterization of Fc receptor-like 6 (FCRL6). (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1112

Chicago Manual of Style (16th Edition):

Schreeder, Daniel M. “Biological characterization of Fc receptor-like 6 (FCRL6).” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1112.

MLA Handbook (7th Edition):

Schreeder, Daniel M. “Biological characterization of Fc receptor-like 6 (FCRL6).” 2009. Web. 20 Oct 2019.

Vancouver:

Schreeder DM. Biological characterization of Fc receptor-like 6 (FCRL6). [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1112.

Council of Science Editors:

Schreeder DM. Biological characterization of Fc receptor-like 6 (FCRL6). [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1112

11. Hovater, Michael (Michael Brian). Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.

Degree: MS, 2006, University of Alabama – Birmingham

This thesis concerns purinergic signaling in renal epithelial cells of normal and polycystic kidneys. The first section discusses first principles of “purinergic signaling” as they… (more)

Subjects/Keywords: Epithelial Cells  – physiology <; br>; Kidney Tubules  – physiology <; br>; Polycystic Kidney Diseases  – pathology <; br>; Receptors, Purinergic P2  – metabolism <; br>; Signal Transduction  – physiology

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APA (6th Edition):

Hovater, M. (. B. (2006). Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,30

Chicago Manual of Style (16th Edition):

Hovater, Michael (Michael Brian). “Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.” 2006. Masters Thesis, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,30.

MLA Handbook (7th Edition):

Hovater, Michael (Michael Brian). “Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.” 2006. Web. 20 Oct 2019.

Vancouver:

Hovater M(B. Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,30.

Council of Science Editors:

Hovater M(B. Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. [Masters Thesis]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,30

12. Erkkila, Brian E. (Brian Eric). Molecular determinants of picrotoxin inhibition.

Degree: PhD, 2007, University of Alabama – Birmingham

PTX is a non-competitive antagonist of many LGICs, and its site of action is believed to be within the ion-conducting pore. This study will examine,… (more)

Subjects/Keywords: Cholinergic Antagonists  – pharmacology <; br>; GABA Antagonists  – pharmacology <; br>; Peptide Fragments  – physiology <; br>; Picrotoxin  – pharmacology <; br>; Receptors, Nicotinic  – metabolism

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APA (6th Edition):

Erkkila, B. E. (. E. (2007). Molecular determinants of picrotoxin inhibition. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,252

Chicago Manual of Style (16th Edition):

Erkkila, Brian E (Brian Eric). “Molecular determinants of picrotoxin inhibition.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,252.

MLA Handbook (7th Edition):

Erkkila, Brian E (Brian Eric). “Molecular determinants of picrotoxin inhibition.” 2007. Web. 20 Oct 2019.

Vancouver:

Erkkila BE(E. Molecular determinants of picrotoxin inhibition. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,252.

Council of Science Editors:

Erkkila BE(E. Molecular determinants of picrotoxin inhibition. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,252

13. Splittgerber, Ryan C. Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells.

Degree: PhD, 2008, University of Alabama – Birmingham

Neuronal nicotinic acetylcholine receptors (nAChRs) are widely expressed in neural and non-neural tissues and have been reported to play a role in neovascularization and vascular… (more)

Subjects/Keywords: Angiogenesis Inducing Agents<; br>; Endothelial Cells  – metabolism<; br>; Neovascularization, Pathologic<; br>; Nicotine  – pharmacology<; br>; Receptors, Nicotinic  – physiology

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APA (6th Edition):

Splittgerber, R. C. (2008). Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,835

Chicago Manual of Style (16th Edition):

Splittgerber, Ryan C. “Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,835.

MLA Handbook (7th Edition):

Splittgerber, Ryan C. “Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells.” 2008. Web. 20 Oct 2019.

Vancouver:

Splittgerber RC. Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,835.

Council of Science Editors:

Splittgerber RC. Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,835

14. Haga, Christopher L. Analysis of the role of FCRL5 and FIGLERs in B cell development, signaling and malignancy.

Degree: PhD, 2008, University of Alabama – Birmingham

The regulation of signaling and migration is critical to B lymphocyte development, activation, and proliferation. This dissertation reports a functional role for Fc receptor-like molecule… (more)

Subjects/Keywords: B-Lymphocytes  – immunology <; br>; Fibronectins  – genetics <; br>; Immunoglobulins  – genetics <; br>; Lymphocyte Activation  – immunology <; br>; Multigene Family <; br>; Proteins  – genetics <; br>; Protein Tyrosine Phosphatase, Non-Receptor Type 6  – metabolism <; br>; Receptors, Antigen, B-Cell  – metabolism <; br>; Receptors, Fc  – metabolism <; br>; Receptors, Interleukin-7  – genetics<; br>; Signal Transduction  – immunology

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APA (6th Edition):

Haga, C. L. (2008). Analysis of the role of FCRL5 and FIGLERs in B cell development, signaling and malignancy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,291

Chicago Manual of Style (16th Edition):

Haga, Christopher L. “Analysis of the role of FCRL5 and FIGLERs in B cell development, signaling and malignancy.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,291.

MLA Handbook (7th Edition):

Haga, Christopher L. “Analysis of the role of FCRL5 and FIGLERs in B cell development, signaling and malignancy.” 2008. Web. 20 Oct 2019.

Vancouver:

Haga CL. Analysis of the role of FCRL5 and FIGLERs in B cell development, signaling and malignancy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,291.

Council of Science Editors:

Haga CL. Analysis of the role of FCRL5 and FIGLERs in B cell development, signaling and malignancy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,291

15. Hanks, Lynae J. Calciotropic hormonal influence on energy homeostasis.

Degree: PhD, 2011, University of Alabama – Birmingham

Energy balance exists when intake is equivalent to expenditure. It has become evident that beyond quantitative aspects of intake, dietary components also have directive impact.… (more)

Subjects/Keywords: Adipose Tissue<; br>; Calcium, Dietary  – administration & dosage<; br>; Energy Metabolism<; br>; Parathyroid Hormone<; br>; Receptors, Calcitriol<; br>; Receptors, Calcium-Sensing<; br>; Rest  – physiology

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APA (6th Edition):

Hanks, L. J. (2011). Calciotropic hormonal influence on energy homeostasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1046

Chicago Manual of Style (16th Edition):

Hanks, Lynae J. “Calciotropic hormonal influence on energy homeostasis.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1046.

MLA Handbook (7th Edition):

Hanks, Lynae J. “Calciotropic hormonal influence on energy homeostasis.” 2011. Web. 20 Oct 2019.

Vancouver:

Hanks LJ. Calciotropic hormonal influence on energy homeostasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1046.

Council of Science Editors:

Hanks LJ. Calciotropic hormonal influence on energy homeostasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1046

16. King, R. Glenn (Rodney Glenn). On the immunological roles of TLT2 and HSH2.

Degree: PhD, 2007, University of Alabama – Birmingham

The evolution of multicellular organisms necessitated the ability to detect and remove harmful parasitic microorganisms from the host. This simple requirement for self non-self discrimination… (more)

Subjects/Keywords: B-Lymphocytes  – metabolism <; br>; Cell Lineage  – immunology <; br>; Granulocytes  – metabolism <; br>; Myeloid Cells  – metabolism <; br>; Receptors, Immunologic  – genetics <; br>; Up-Regulation  – immunology

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APA (6th Edition):

King, R. G. (. G. (2007). On the immunological roles of TLT2 and HSH2. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,256

Chicago Manual of Style (16th Edition):

King, R Glenn (Rodney Glenn). “On the immunological roles of TLT2 and HSH2.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,256.

MLA Handbook (7th Edition):

King, R Glenn (Rodney Glenn). “On the immunological roles of TLT2 and HSH2.” 2007. Web. 20 Oct 2019.

Vancouver:

King RG(G. On the immunological roles of TLT2 and HSH2. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,256.

Council of Science Editors:

King RG(G. On the immunological roles of TLT2 and HSH2. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,256

17. Richards-Williams, Clintoria (Clintoria Latrice). Roles of extracellular ATP in zinc in pancreatic β-cell physiology.

Degree: PhD, 2008, University of Alabama – Birmingham

Insulin secretory defects within pancreatic beta-cells (β-cells) of islets of Langerhans play a role in the pathogenesis of diabetes mellitus. Furthermore, it is also well… (more)

Subjects/Keywords: Adenosine Triphosphate  – metabolism<; br>; Diabetes Mellitus<; br>; Insulin  – secretion<; br>; Islets of Langerhans  – metabolism<; br>; Receptors, Purinergic P2  – metabolism<; br>; Zinc

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APA (6th Edition):

Richards-Williams, C. (. L. (2008). Roles of extracellular ATP in zinc in pancreatic β-cell physiology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,825

Chicago Manual of Style (16th Edition):

Richards-Williams, Clintoria (Clintoria Latrice). “Roles of extracellular ATP in zinc in pancreatic β-cell physiology.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,825.

MLA Handbook (7th Edition):

Richards-Williams, Clintoria (Clintoria Latrice). “Roles of extracellular ATP in zinc in pancreatic β-cell physiology.” 2008. Web. 20 Oct 2019.

Vancouver:

Richards-Williams C(L. Roles of extracellular ATP in zinc in pancreatic β-cell physiology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,825.

Council of Science Editors:

Richards-Williams C(L. Roles of extracellular ATP in zinc in pancreatic β-cell physiology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,825

18. Sun, Mianen. The role of DDX3 in regulating apoptosis, p53 and Snail.

Degree: PhD, 2008, University of Alabama – Birmingham

Cancer is a common disease that causes high rates of lethality. Resistance to cancer therapy is one major obstacle for producing an effective cancer treatment.… (more)

Subjects/Keywords: Apoptosis<; br>; Genes, p53<; br. Glycogen Synthase Kinase 3  – metabolism<; br>; Inhibitor of Apoptosis Proteins  – metabolism<; br>; Protein Kinase Inhibitors  – pharmacology<; br>; Receptors, Death Domain  – metabolism

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APA (6th Edition):

Sun, M. (2008). The role of DDX3 in regulating apoptosis, p53 and Snail. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,827

Chicago Manual of Style (16th Edition):

Sun, Mianen. “The role of DDX3 in regulating apoptosis, p53 and Snail.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,827.

MLA Handbook (7th Edition):

Sun, Mianen. “The role of DDX3 in regulating apoptosis, p53 and Snail.” 2008. Web. 20 Oct 2019.

Vancouver:

Sun M. The role of DDX3 in regulating apoptosis, p53 and Snail. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,827.

Council of Science Editors:

Sun M. The role of DDX3 in regulating apoptosis, p53 and Snail. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,827

19. Seo, Hwa-Seon. The role of TGF[beta] signaling in skeletal development.

Degree: PhD, 2008, University of Alabama – Birmingham

Each skeletal element is essential for body movement along with the joints, muscles, tendons and ligaments. They also protect internal organs and serve as a… (more)

Subjects/Keywords: Bone and Bones  – abnormalities <; br>; Homeodomain Proteins  – genetics <; br>; Joints  – abnormalities <; br>; Mesoderm  – embryology <; br>; Protein-Serine-Threonine Kinases  – Metabolism <; br>; Receptors, Transforming Growth Factor beta  – metabolism <; br>; Transforming Growth Factor beta1  – physiology

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APA (6th Edition):

Seo, H. (2008). The role of TGF[beta] signaling in skeletal development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,338

Chicago Manual of Style (16th Edition):

Seo, Hwa-Seon. “The role of TGF[beta] signaling in skeletal development.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,338.

MLA Handbook (7th Edition):

Seo, Hwa-Seon. “The role of TGF[beta] signaling in skeletal development.” 2008. Web. 20 Oct 2019.

Vancouver:

Seo H. The role of TGF[beta] signaling in skeletal development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,338.

Council of Science Editors:

Seo H. The role of TGF[beta] signaling in skeletal development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,338

20. Nash, Kevin T. (Kevin Tyler). KISS1 metastasis suppressor secretion is required for metastasis suppression.

Degree: PhD, 2006, University of Alabama – Birmingham

Failure to reduce the number of cancer deaths over the last 50 years is due to the inability to selectively target metastatic disease. Recently, the… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic <; br>; Melanoma  – metabolism <; br>; Melanoma  – secretion <; br>; Neoplasm Metastasis  – prevention & control <; br>; Proteins  – physiology <; br>; Receptors, G-Protein-Coupled  – metabolism <; br>; Tumor Suppressor Proteins  – secretion

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APA (6th Edition):

Nash, K. T. (. T. (2006). KISS1 metastasis suppressor secretion is required for metastasis suppression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,380

Chicago Manual of Style (16th Edition):

Nash, Kevin T (Kevin Tyler). “KISS1 metastasis suppressor secretion is required for metastasis suppression.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,380.

MLA Handbook (7th Edition):

Nash, Kevin T (Kevin Tyler). “KISS1 metastasis suppressor secretion is required for metastasis suppression.” 2006. Web. 20 Oct 2019.

Vancouver:

Nash KT(T. KISS1 metastasis suppressor secretion is required for metastasis suppression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,380.

Council of Science Editors:

Nash KT(T. KISS1 metastasis suppressor secretion is required for metastasis suppression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,380

21. Cao, Shuwen. Cytokine Signaling In A Mouse Model Of Parkinson's Disease.

Degree: 2012, University of Alabama – Birmingham

The protein alpha-synuclein (α-SYN), which is found in the Lewy bodies of dopaminergic (DA) neurons in the substantia nigra (SN), has an important role in… (more)

Subjects/Keywords: alpha-Synuclein.<; br>; Brain – immunology.<; br>; Cytokines – metabolism<; br>; Gene Expression Regulation – drug effects<; br>; Immune System Processes – immunology.<; br>; Lymphocyte Activation – immunology<; br>; Microglia<; br>; Parkinson Disease – immunology.<; br>; Receptors, IgG – metabolism.<; br>; Signal Transduction – drug effects

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APA (6th Edition):

Cao, S. (2012). Cytokine Signaling In A Mouse Model Of Parkinson's Disease. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cao, Shuwen. “Cytokine Signaling In A Mouse Model Of Parkinson's Disease.” 2012. Thesis, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cao, Shuwen. “Cytokine Signaling In A Mouse Model Of Parkinson's Disease.” 2012. Web. 20 Oct 2019.

Vancouver:

Cao S. Cytokine Signaling In A Mouse Model Of Parkinson's Disease. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cao S. Cytokine Signaling In A Mouse Model Of Parkinson's Disease. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Renna, Jordan Michael. The role of strychnine-sensitive nACHRS in rabbit retinal OFF ganglion cells.

Degree: PhD, 2008, University of Alabama – Birmingham

Strychnine is considered a selective competitive antagonist of glycine gated Cl- channels (Saitoh et al., 1994) and studies have used strychnine at low micromolar concentrations… (more)

Subjects/Keywords: Glycine Agents  – pharmacology <; br>; Phenylalanine  – analogs & derivatives <; br>; Phosphonic Acids  – pharmacology <; br>; Rabbits <; br>; Receptors, Nicotinic  – drug effects <; br>; Retinal Ganglion Cells  – drug effects <; br>; Retinal Ganglion Cells  – metabolism <; br>; Strychnine  – pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Renna, J. M. (2008). The role of strychnine-sensitive nACHRS in rabbit retinal OFF ganglion cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,336

Chicago Manual of Style (16th Edition):

Renna, Jordan Michael. “The role of strychnine-sensitive nACHRS in rabbit retinal OFF ganglion cells.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,336.

MLA Handbook (7th Edition):

Renna, Jordan Michael. “The role of strychnine-sensitive nACHRS in rabbit retinal OFF ganglion cells.” 2008. Web. 20 Oct 2019.

Vancouver:

Renna JM. The role of strychnine-sensitive nACHRS in rabbit retinal OFF ganglion cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,336.

Council of Science Editors:

Renna JM. The role of strychnine-sensitive nACHRS in rabbit retinal OFF ganglion cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,336

23. Lai, Yun-Ju. Role of TRIP6 in LPA-induced cell migration.

Degree: PhD, 2007, University of Alabama – Birmingham

The LIM domain-containing Thyroid Receptor-Interacting Protein 6 (TRIP6) is a zyxin family member that has been implicated in actin dynamics and cell motility. In this… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing<; br>; Carrier Proteins  – physiology<; br>; Cell Movement<; br>; Feedback, Biochemical<; br>; Lysophospholipids<; br>; Protein-Tyrosine Kinases  – physiology<; br>; Receptors, G-Protein-Coupled  – metabolism<; br>; Transcription Factors

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APA (6th Edition):

Lai, Y. (2007). Role of TRIP6 in LPA-induced cell migration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,453

Chicago Manual of Style (16th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,453.

MLA Handbook (7th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Web. 20 Oct 2019.

Vancouver:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453.

Council of Science Editors:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453

24. Lai, Jen-Fang. The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae.

Degree: PhD, 2009, University of Alabama – Birmingham

Several reports have suggested that Mycoplasma pneumoniae (Mp) can contribute importantly to the expression of symptoms in asthmatic human subjects. As a foundation for understanding… (more)

Subjects/Keywords: Antigens, CD11b  – metabolism<; br>; Macrophages  – immunology<; br>; Mice<; br>; Myeloid Differentiation Factor 88<; br>; Pneumonia, Mycoplasma  – metabolism<; br>; Toll-Like Receptors

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APA (6th Edition):

Lai, J. (2009). The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,680

Chicago Manual of Style (16th Edition):

Lai, Jen-Fang. “The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,680.

MLA Handbook (7th Edition):

Lai, Jen-Fang. “The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae.” 2009. Web. 20 Oct 2019.

Vancouver:

Lai J. The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,680.

Council of Science Editors:

Lai J. The essential role of macrophages and TLR signaling in the host response to Mycoplasma pneumoniae. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,680

25. Markwardt, Sean J. GABAergic signaling to adult-generated neurons in hippocampus.

Degree: PhD, 2011, University of Alabama – Birmingham

In the central nervous system of adult mammals, new neurons are produced throughout life in at least two regions, the subventricular zone and dentate gyrus… (more)

Subjects/Keywords: Adult Stem Cells  – physiology<; br>; Dentate Gyrus  – cytology<; br>; gamma-Aminobutyric Acid  – metabolism<; br>; Neurons  – physiology<; br>; Receptors, GABA  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Markwardt, S. J. (2011). GABAergic signaling to adult-generated neurons in hippocampus. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1096

Chicago Manual of Style (16th Edition):

Markwardt, Sean J. “GABAergic signaling to adult-generated neurons in hippocampus.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1096.

MLA Handbook (7th Edition):

Markwardt, Sean J. “GABAergic signaling to adult-generated neurons in hippocampus.” 2011. Web. 20 Oct 2019.

Vancouver:

Markwardt SJ. GABAergic signaling to adult-generated neurons in hippocampus. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1096.

Council of Science Editors:

Markwardt SJ. GABAergic signaling to adult-generated neurons in hippocampus. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1096

26. Speed, Haley E. Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus.

Degree: PhD, 2008, University of Alabama – Birmingham

Hippocampal CA1 pyramidal neurons integrate and filter spatial, temporal, and sensory information from the entorhinal cortex (EC) for memory storage. Synaptic transmission mediates the processing… (more)

Subjects/Keywords: Action Potentials  – physiology <; br>; Hippocampus  – cytology <; br>; Hippocampus  – growth & development <; br>; Hippocampus  – metabolism <; br>; Neuronal Plasticity  – physiology <; br>; Receptors, Metabotropic Glutamate  – physiology <; br>; Synapses  – metabolism <; br>; Synapses  – physiology <; br>; Synaptic Transmission  – physiology

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APA (6th Edition):

Speed, H. E. (2008). Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,243

Chicago Manual of Style (16th Edition):

Speed, Haley E. “Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,243.

MLA Handbook (7th Edition):

Speed, Haley E. “Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus.” 2008. Web. 20 Oct 2019.

Vancouver:

Speed HE. Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,243.

Council of Science Editors:

Speed HE. Changes in short-term facilitation are opposite at Schaffer collateral and Temporoammonic CA1 synapses in the developing rat hippocampus. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,243


University of Alberta

27. McCarthy, Amanda Marie. An investigation of endogenous ghrelin and growth hormone-releasing hormone following the consumption of two different relative doses of oral l-arginine.

Degree: MS, Physical Education and Recreation, 2011, University of Alberta

 Individuals consume the amino acid L-arginine (L-arg) to obtain an anabolic response. However, little is known concerning the mechanism for using an oral dose. The… (more)

Subjects/Keywords: Growth hormone releasing factor; Ghrelin  – Metabolism; Arginine  – Metabolism; Proteins  – Metabolism; Somatotropin

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APA (6th Edition):

McCarthy, A. M. (2011). An investigation of endogenous ghrelin and growth hormone-releasing hormone following the consumption of two different relative doses of oral l-arginine. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/05741s32t

Chicago Manual of Style (16th Edition):

McCarthy, Amanda Marie. “An investigation of endogenous ghrelin and growth hormone-releasing hormone following the consumption of two different relative doses of oral l-arginine.” 2011. Masters Thesis, University of Alberta. Accessed October 20, 2019. https://era.library.ualberta.ca/files/05741s32t.

MLA Handbook (7th Edition):

McCarthy, Amanda Marie. “An investigation of endogenous ghrelin and growth hormone-releasing hormone following the consumption of two different relative doses of oral l-arginine.” 2011. Web. 20 Oct 2019.

Vancouver:

McCarthy AM. An investigation of endogenous ghrelin and growth hormone-releasing hormone following the consumption of two different relative doses of oral l-arginine. [Internet] [Masters thesis]. University of Alberta; 2011. [cited 2019 Oct 20]. Available from: https://era.library.ualberta.ca/files/05741s32t.

Council of Science Editors:

McCarthy AM. An investigation of endogenous ghrelin and growth hormone-releasing hormone following the consumption of two different relative doses of oral l-arginine. [Masters Thesis]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/05741s32t

28. Penton, Rachel E. (Rachel Elizabeth). Changes in hippocampal excitability during withdrawal from chronic nicotine.

Degree: PhD, 2008, University of Alabama – Birmingham

Drug-seeking behavior following chronic drug use lasts for many months or even years. Short-term withdrawal experiments have suggested that the neuroadaptations thought to underlie learning… (more)

Subjects/Keywords: Brain  – drug effects <; br>; Hippocampus  – drug effects <; br>; Nicotine  – adverse effects <; br>; Rats <; br>; Receptors, Nicotinic  – metabolism <; br>; Substance Withdrawal Syndrome  – physiopathology <; br>; Time Factors

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APA (6th Edition):

Penton, R. E. (. E. (2008). Changes in hippocampal excitability during withdrawal from chronic nicotine. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,334

Chicago Manual of Style (16th Edition):

Penton, Rachel E (Rachel Elizabeth). “Changes in hippocampal excitability during withdrawal from chronic nicotine.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,334.

MLA Handbook (7th Edition):

Penton, Rachel E (Rachel Elizabeth). “Changes in hippocampal excitability during withdrawal from chronic nicotine.” 2008. Web. 20 Oct 2019.

Vancouver:

Penton RE(E. Changes in hippocampal excitability during withdrawal from chronic nicotine. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,334.

Council of Science Editors:

Penton RE(E. Changes in hippocampal excitability during withdrawal from chronic nicotine. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,334


Marshall University

29. Butts, Molly Rae. The effect of moderate alcohol consumption on sodium-dependent nutrient co-transport in intestinal epithelial cells in vitro and in vivo.

Degree: 2019, Marshall University

 Background: Alcohol consumption leads to a variety of different health consequences including cardiovascular disease, cancer and malnutrition. This malnutrition is in part due to a… (more)

Subjects/Keywords: Alcohol; B0AT1; Ethanol; Nutrient co-transport; SGLT1; <; p>; Alcohol  – Physiological effect.<; /p>; <; p>; Membranes (Biology)  – Research.<; /p>; <; p>; Cell receptors  – Research.<; /p>;

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APA (6th Edition):

Butts, M. R. (2019). The effect of moderate alcohol consumption on sodium-dependent nutrient co-transport in intestinal epithelial cells in vitro and in vivo. (Thesis). Marshall University. Retrieved from https://mds.marshall.edu/etd/1215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Butts, Molly Rae. “The effect of moderate alcohol consumption on sodium-dependent nutrient co-transport in intestinal epithelial cells in vitro and in vivo.” 2019. Thesis, Marshall University. Accessed October 20, 2019. https://mds.marshall.edu/etd/1215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Butts, Molly Rae. “The effect of moderate alcohol consumption on sodium-dependent nutrient co-transport in intestinal epithelial cells in vitro and in vivo.” 2019. Web. 20 Oct 2019.

Vancouver:

Butts MR. The effect of moderate alcohol consumption on sodium-dependent nutrient co-transport in intestinal epithelial cells in vitro and in vivo. [Internet] [Thesis]. Marshall University; 2019. [cited 2019 Oct 20]. Available from: https://mds.marshall.edu/etd/1215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Butts MR. The effect of moderate alcohol consumption on sodium-dependent nutrient co-transport in intestinal epithelial cells in vitro and in vivo. [Thesis]. Marshall University; 2019. Available from: https://mds.marshall.edu/etd/1215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Zhu, Xiaolin. Roles of NR4A3 in insulin sensitivity in skeletal muscle.

Degree: PhD, 2010, University of Alabama – Birmingham

For the past several decades, the prevalence of Type 2 Diabetes and the metabolic syndrome has been increasing. Insulin resistance is the central pathogenic event… (more)

Subjects/Keywords: Adipocytes  – drug effects<; br>; DNA-Binding Proteins  – metabolism<; br>; Hypoglycemic Agents  – pharmacology<; br>; Insulin  – pharmacology<; br>; Insulin Resistance<; br>; Muscle, Skeletal<; br>; Nuclear Receptor Subfamily 4, Group A, Member 3<; br>; Receptors, Steroid  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhu, X. (2010). Roles of NR4A3 in insulin sensitivity in skeletal muscle. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1117

Chicago Manual of Style (16th Edition):

Zhu, Xiaolin. “Roles of NR4A3 in insulin sensitivity in skeletal muscle.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 20, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1117.

MLA Handbook (7th Edition):

Zhu, Xiaolin. “Roles of NR4A3 in insulin sensitivity in skeletal muscle.” 2010. Web. 20 Oct 2019.

Vancouver:

Zhu X. Roles of NR4A3 in insulin sensitivity in skeletal muscle. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 20]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1117.

Council of Science Editors:

Zhu X. Roles of NR4A3 in insulin sensitivity in skeletal muscle. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1117

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