Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:( Receptors Growth Factor). Showing records 1 – 30 of 30942 total matches.

[1] [2] [3] [4] [5] … [1032]

Search Limiters

Last 2 Years | English Only

Degrees

Languages

Country

▼ Search Limiters


Rutgers University

1. Rathod, Trushar, 1979-. Generation of anti-tumorigenic MET receptor variants by splicing interference.

Degree: PhD, Pharmacology, Cellular and Molecular, 2019, Rutgers University

Aberrant activation of MET, a receptor tyrosine kinase (RTK), leads to tumor growth, invasion and metastasis and is implicated in multiple types of cancers. Hence,… (more)

Subjects/Keywords: Hepatocyte growth factor  – Receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rathod, Trushar, 1. (2019). Generation of anti-tumorigenic MET receptor variants by splicing interference. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60961/

Chicago Manual of Style (16th Edition):

Rathod, Trushar, 1979-. “Generation of anti-tumorigenic MET receptor variants by splicing interference.” 2019. Doctoral Dissertation, Rutgers University. Accessed October 21, 2019. https://rucore.libraries.rutgers.edu/rutgers-lib/60961/.

MLA Handbook (7th Edition):

Rathod, Trushar, 1979-. “Generation of anti-tumorigenic MET receptor variants by splicing interference.” 2019. Web. 21 Oct 2019.

Vancouver:

Rathod, Trushar 1. Generation of anti-tumorigenic MET receptor variants by splicing interference. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2019 Oct 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60961/.

Council of Science Editors:

Rathod, Trushar 1. Generation of anti-tumorigenic MET receptor variants by splicing interference. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60961/


University of Hong Kong

2. Chen, Jinna. EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma.

Degree: PhD, 2015, University of Hong Kong

Since the vast majority of Nasopharyngeal carcinoma (NPC) is sensitive to radiotherapy and chemotherapy, the overall survival rate of stage I NPC is nearly 100%.… (more)

Subjects/Keywords: Epidermal growth factor - Receptors; Nasopharynx - Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, J. (2015). EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Chen, J. [陳金娜]. (2015). EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719441 ; http://dx.doi.org/10.5353/th_b5719441 ; http://hdl.handle.net/10722/233722

Chicago Manual of Style (16th Edition):

Chen, Jinna. “EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. Chen, J. [陳金娜]. (2015). EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719441 ; http://dx.doi.org/10.5353/th_b5719441 ; http://hdl.handle.net/10722/233722.

MLA Handbook (7th Edition):

Chen, Jinna. “EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma.” 2015. Web. 21 Oct 2019.

Vancouver:

Chen J. EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Oct 21]. Available from: Chen, J. [陳金娜]. (2015). EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719441 ; http://dx.doi.org/10.5353/th_b5719441 ; http://hdl.handle.net/10722/233722.

Council of Science Editors:

Chen J. EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Chen, J. [陳金娜]. (2015). EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719441 ; http://dx.doi.org/10.5353/th_b5719441 ; http://hdl.handle.net/10722/233722


University of Hong Kong

3. Ho, Ka-yan, Rebecca Lucinda. Mutations of epidermal growth factor receptor (EGFR) pathway genes andMET in primary lung adenocarcinoma.

Degree: Master of Medical Sciences, 2012, University of Hong Kong

This study completed the analysis of mutational frequencies and clinicopathological patterns of six EGFR pathway-related genes (EGFR, HER2, HER4, KRAS, BRAF and MET) in 212… (more)

Subjects/Keywords: Epidermal growth factor - Receptors.; Lungs - Cancer - Diagnosis.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ho, Ka-yan, R. L. (2012). Mutations of epidermal growth factor receptor (EGFR) pathway genes andMET in primary lung adenocarcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Ho, K. R. L. [何嘉茵]. (2012). Mutations of epidermal growth factor receptor (EGFR) pathway genes and MET in primary lung adenocarcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4833390 ; http://dx.doi.org/10.5353/th_b4833390 ; http://hdl.handle.net/10722/173950

Chicago Manual of Style (16th Edition):

Ho, Ka-yan, Rebecca Lucinda. “Mutations of epidermal growth factor receptor (EGFR) pathway genes andMET in primary lung adenocarcinoma.” 2012. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. Ho, K. R. L. [何嘉茵]. (2012). Mutations of epidermal growth factor receptor (EGFR) pathway genes and MET in primary lung adenocarcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4833390 ; http://dx.doi.org/10.5353/th_b4833390 ; http://hdl.handle.net/10722/173950.

MLA Handbook (7th Edition):

Ho, Ka-yan, Rebecca Lucinda. “Mutations of epidermal growth factor receptor (EGFR) pathway genes andMET in primary lung adenocarcinoma.” 2012. Web. 21 Oct 2019.

Vancouver:

Ho, Ka-yan RL. Mutations of epidermal growth factor receptor (EGFR) pathway genes andMET in primary lung adenocarcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2012. [cited 2019 Oct 21]. Available from: Ho, K. R. L. [何嘉茵]. (2012). Mutations of epidermal growth factor receptor (EGFR) pathway genes and MET in primary lung adenocarcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4833390 ; http://dx.doi.org/10.5353/th_b4833390 ; http://hdl.handle.net/10722/173950.

Council of Science Editors:

Ho, Ka-yan RL. Mutations of epidermal growth factor receptor (EGFR) pathway genes andMET in primary lung adenocarcinoma. [Masters Thesis]. University of Hong Kong; 2012. Available from: Ho, K. R. L. [何嘉茵]. (2012). Mutations of epidermal growth factor receptor (EGFR) pathway genes and MET in primary lung adenocarcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4833390 ; http://dx.doi.org/10.5353/th_b4833390 ; http://hdl.handle.net/10722/173950


Oregon State University

4. Larsen, Christine A. (Christine Ann). Suppression of Met signaling by the green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG).

Degree: PhD, Molecular and Cellular Biology, 2010, Oregon State University

 Met is a prognostic indicator of colorectal cancer patient survival. Therapies that target Met may therefore have beneficial outcomes in the clinic. Recently, EGCG was… (more)

Subjects/Keywords: epigallocatechin-3-gallate; Hepatocyte growth factor  – Receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Larsen, C. A. (. A. (2010). Suppression of Met signaling by the green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG). (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/14999

Chicago Manual of Style (16th Edition):

Larsen, Christine A (Christine Ann). “Suppression of Met signaling by the green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG).” 2010. Doctoral Dissertation, Oregon State University. Accessed October 21, 2019. http://hdl.handle.net/1957/14999.

MLA Handbook (7th Edition):

Larsen, Christine A (Christine Ann). “Suppression of Met signaling by the green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG).” 2010. Web. 21 Oct 2019.

Vancouver:

Larsen CA(A. Suppression of Met signaling by the green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG). [Internet] [Doctoral dissertation]. Oregon State University; 2010. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1957/14999.

Council of Science Editors:

Larsen CA(A. Suppression of Met signaling by the green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG). [Doctoral Dissertation]. Oregon State University; 2010. Available from: http://hdl.handle.net/1957/14999


University of Texas Southwestern Medical Center

5. Iqbal, Nida S. Mechanisms of p19Arf-Mediated Regulation of Perivascular Cell Biology During Mammalian Eye Development.

Degree: 2015, University of Texas Southwestern Medical Center

 Since its discovery in 1995, p19Arf has been under critical interrogation for its role as a potent cell cycle regulator and tumor suppressor. In the… (more)

Subjects/Keywords: Eye; MicroRNAs; Receptors, Platelet-Derived Growth Factor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Iqbal, N. S. (2015). Mechanisms of p19Arf-Mediated Regulation of Perivascular Cell Biology During Mammalian Eye Development. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iqbal, Nida S. “Mechanisms of p19Arf-Mediated Regulation of Perivascular Cell Biology During Mammalian Eye Development.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed October 21, 2019. http://hdl.handle.net/2152.5/1736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iqbal, Nida S. “Mechanisms of p19Arf-Mediated Regulation of Perivascular Cell Biology During Mammalian Eye Development.” 2015. Web. 21 Oct 2019.

Vancouver:

Iqbal NS. Mechanisms of p19Arf-Mediated Regulation of Perivascular Cell Biology During Mammalian Eye Development. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/2152.5/1736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iqbal NS. Mechanisms of p19Arf-Mediated Regulation of Perivascular Cell Biology During Mammalian Eye Development. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/1736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

6. Newcomb, Susan Elizabeth. Multi-level analysis of regulation of EGFR signalling during Drosophila melanogaster leg proximal-distal axis patterning.

Degree: 2018, Columbia University

 A major pursuit of Developmental Biology is to determine how organisms composed of cells containing a single genome generate stereotyped body plans with diverse, complex… (more)

Subjects/Keywords: Biology; Genetics; Growth factors – Receptors; Epidermal growth factor; Drosophila melanogaster

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Newcomb, S. E. (2018). Multi-level analysis of regulation of EGFR signalling during Drosophila melanogaster leg proximal-distal axis patterning. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D82V3ZZJ

Chicago Manual of Style (16th Edition):

Newcomb, Susan Elizabeth. “Multi-level analysis of regulation of EGFR signalling during Drosophila melanogaster leg proximal-distal axis patterning.” 2018. Doctoral Dissertation, Columbia University. Accessed October 21, 2019. https://doi.org/10.7916/D82V3ZZJ.

MLA Handbook (7th Edition):

Newcomb, Susan Elizabeth. “Multi-level analysis of regulation of EGFR signalling during Drosophila melanogaster leg proximal-distal axis patterning.” 2018. Web. 21 Oct 2019.

Vancouver:

Newcomb SE. Multi-level analysis of regulation of EGFR signalling during Drosophila melanogaster leg proximal-distal axis patterning. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2019 Oct 21]. Available from: https://doi.org/10.7916/D82V3ZZJ.

Council of Science Editors:

Newcomb SE. Multi-level analysis of regulation of EGFR signalling during Drosophila melanogaster leg proximal-distal axis patterning. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D82V3ZZJ


University of Hong Kong

7. 顏韻玲; Ngan, Wan-ling. Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation.

Degree: PhD, 2015, University of Hong Kong

 The forkhead box transcription factor M1 (FOXM1) is ubiquitously expressed in proliferating cells and regulates expression of genes controlling cell cycle progression. Its overexpression was… (more)

Subjects/Keywords: Transcription factors; Epidermal growth factor - Receptors; Cell proliferation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

顏韻玲; Ngan, W. (2015). Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ngan, W. [顏韻玲]. (2015). Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5570778 ; http://dx.doi.org/10.5353/th_b5570778 ; http://hdl.handle.net/10722/226677

Chicago Manual of Style (16th Edition):

顏韻玲; Ngan, Wan-ling. “Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. Ngan, W. [顏韻玲]. (2015). Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5570778 ; http://dx.doi.org/10.5353/th_b5570778 ; http://hdl.handle.net/10722/226677.

MLA Handbook (7th Edition):

顏韻玲; Ngan, Wan-ling. “Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation.” 2015. Web. 21 Oct 2019.

Vancouver:

顏韻玲; Ngan W. Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Oct 21]. Available from: Ngan, W. [顏韻玲]. (2015). Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5570778 ; http://dx.doi.org/10.5353/th_b5570778 ; http://hdl.handle.net/10722/226677.

Council of Science Editors:

顏韻玲; Ngan W. Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Ngan, W. [顏韻玲]. (2015). Novel nuclear partnering role of EPS8 with FOXM1 in regulating cell proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5570778 ; http://dx.doi.org/10.5353/th_b5570778 ; http://hdl.handle.net/10722/226677


University of Hong Kong

8. 方旭熙; Fong, Yuk-hei. The role of EGFR pathway in the pathogenesis of cigarette smoke-related airway mucus hypersecretion.

Degree: Master of Medical Sciences, 2017, University of Hong Kong

Chronic obstructive pulmonary disease (COPD) has been the fourth leading cause of death worldwide and is predicted to be the third by 2030. COPD is… (more)

Subjects/Keywords: Respiratory organs - Diseases; Epidermal growth factor - Receptors; Mucus

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

方旭熙; Fong, Y. (2017). The role of EGFR pathway in the pathogenesis of cigarette smoke-related airway mucus hypersecretion. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/251326

Chicago Manual of Style (16th Edition):

方旭熙; Fong, Yuk-hei. “The role of EGFR pathway in the pathogenesis of cigarette smoke-related airway mucus hypersecretion.” 2017. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. http://hdl.handle.net/10722/251326.

MLA Handbook (7th Edition):

方旭熙; Fong, Yuk-hei. “The role of EGFR pathway in the pathogenesis of cigarette smoke-related airway mucus hypersecretion.” 2017. Web. 21 Oct 2019.

Vancouver:

方旭熙; Fong Y. The role of EGFR pathway in the pathogenesis of cigarette smoke-related airway mucus hypersecretion. [Internet] [Masters thesis]. University of Hong Kong; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10722/251326.

Council of Science Editors:

方旭熙; Fong Y. The role of EGFR pathway in the pathogenesis of cigarette smoke-related airway mucus hypersecretion. [Masters Thesis]. University of Hong Kong; 2017. Available from: http://hdl.handle.net/10722/251326


University of Hong Kong

9. 陳嘉豪; Chan, Ka-ho. ARID1A variations in EGFR mutated lung adenocarcinomas and its relation with resistance to targeted therapy.

Degree: Master of Medical Sciences, 2017, University of Hong Kong

Lung cancer causes the highest cancer mortality worldwide and amongst lung cancers, adenocarcinoma (AD) is the commonest histological type. EGFR mutation occurs in around 45%… (more)

Subjects/Keywords: Lung - Cancer; Adenocarcinoma; Epidermal growth factor - Receptors; DNA-binding proteins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

陳嘉豪; Chan, K. (2017). ARID1A variations in EGFR mutated lung adenocarcinomas and its relation with resistance to targeted therapy. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/251336

Chicago Manual of Style (16th Edition):

陳嘉豪; Chan, Ka-ho. “ARID1A variations in EGFR mutated lung adenocarcinomas and its relation with resistance to targeted therapy.” 2017. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. http://hdl.handle.net/10722/251336.

MLA Handbook (7th Edition):

陳嘉豪; Chan, Ka-ho. “ARID1A variations in EGFR mutated lung adenocarcinomas and its relation with resistance to targeted therapy.” 2017. Web. 21 Oct 2019.

Vancouver:

陳嘉豪; Chan K. ARID1A variations in EGFR mutated lung adenocarcinomas and its relation with resistance to targeted therapy. [Internet] [Masters thesis]. University of Hong Kong; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10722/251336.

Council of Science Editors:

陳嘉豪; Chan K. ARID1A variations in EGFR mutated lung adenocarcinomas and its relation with resistance to targeted therapy. [Masters Thesis]. University of Hong Kong; 2017. Available from: http://hdl.handle.net/10722/251336


University of Edinburgh

10. Castle, Andrew Richard. Investigating the cell biological mechanisms regulated by the cellular prion protein.

Degree: PhD, 2017, University of Edinburgh

 Transmissible spongiform encephalopathies (TSEs) are rare, uniformly fatal neurodegenerative disorders that can affect many mammalian species, including humans. A hallmark of these diseases is the… (more)

Subjects/Keywords: PrPC; cellular prion protein; nerve growth factor signalling; NGF receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Castle, A. R. (2017). Investigating the cell biological mechanisms regulated by the cellular prion protein. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28809

Chicago Manual of Style (16th Edition):

Castle, Andrew Richard. “Investigating the cell biological mechanisms regulated by the cellular prion protein.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed October 21, 2019. http://hdl.handle.net/1842/28809.

MLA Handbook (7th Edition):

Castle, Andrew Richard. “Investigating the cell biological mechanisms regulated by the cellular prion protein.” 2017. Web. 21 Oct 2019.

Vancouver:

Castle AR. Investigating the cell biological mechanisms regulated by the cellular prion protein. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1842/28809.

Council of Science Editors:

Castle AR. Investigating the cell biological mechanisms regulated by the cellular prion protein. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28809

11. Cazares, Tareian Alonzo. Characterization of the effects of NRG-1 on HepG2 cell metabolism.

Degree: Thesis (M.S.), 2015, Ball State University

 Neuregulin-1 (NRG-1) is an epidermal growth factor-like ligand that binds to the human epidermal growth factor receptor 3 (HER3) resulting in increased glucose uptake, mitochondrial… (more)

Subjects/Keywords: Ligands (Biochemistry); Epidermal growth factor  – Receptors.; Liver cells.; Liver  – Physiology.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cazares, T. A. (2015). Characterization of the effects of NRG-1 on HepG2 cell metabolism. (Masters Thesis). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/200064

Chicago Manual of Style (16th Edition):

Cazares, Tareian Alonzo. “Characterization of the effects of NRG-1 on HepG2 cell metabolism.” 2015. Masters Thesis, Ball State University. Accessed October 21, 2019. http://cardinalscholar.bsu.edu/handle/123456789/200064.

MLA Handbook (7th Edition):

Cazares, Tareian Alonzo. “Characterization of the effects of NRG-1 on HepG2 cell metabolism.” 2015. Web. 21 Oct 2019.

Vancouver:

Cazares TA. Characterization of the effects of NRG-1 on HepG2 cell metabolism. [Internet] [Masters thesis]. Ball State University; 2015. [cited 2019 Oct 21]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/200064.

Council of Science Editors:

Cazares TA. Characterization of the effects of NRG-1 on HepG2 cell metabolism. [Masters Thesis]. Ball State University; 2015. Available from: http://cardinalscholar.bsu.edu/handle/123456789/200064


University of Texas Southwestern Medical Center

12. Dutchak, Paul Anthony. Metabolic Regulation by Fibroblast Growth Factor 21.

Degree: 2011, University of Texas Southwestern Medical Center

 Fibroblast growth factor 21 (FGF21) is a secreted hormone that can beneficially regulate glucose and lipid homeostasis. Through a reverse endocrinology approach, we uncovered that… (more)

Subjects/Keywords: Fibroblast Growth Factor; Liver; Peroxisome Proliferator-Activated Receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dutchak, P. A. (2011). Metabolic Regulation by Fibroblast Growth Factor 21. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dutchak, Paul Anthony. “Metabolic Regulation by Fibroblast Growth Factor 21.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed October 21, 2019. http://hdl.handle.net/2152.5/935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dutchak, Paul Anthony. “Metabolic Regulation by Fibroblast Growth Factor 21.” 2011. Web. 21 Oct 2019.

Vancouver:

Dutchak PA. Metabolic Regulation by Fibroblast Growth Factor 21. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/2152.5/935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dutchak PA. Metabolic Regulation by Fibroblast Growth Factor 21. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Carbajal, Liliana. The Role of EGFR Signaling in Gonadotropin-Induced Steroidogenesis.

Degree: 2010, University of Texas Southwestern Medical Center

 Recent evidence has demonstrated that cross talk between G protein-coupled receptors and Epidermal Growth Factor Receptor (EGFR) is critical for steroidogenesis in all three major… (more)

Subjects/Keywords: Ovary; Polycystic Ovary Syndrome; Receptor, Epidermal Growth Factor; Receptors, Steroid

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carbajal, L. (2010). The Role of EGFR Signaling in Gonadotropin-Induced Steroidogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/804

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carbajal, Liliana. “The Role of EGFR Signaling in Gonadotropin-Induced Steroidogenesis.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed October 21, 2019. http://hdl.handle.net/2152.5/804.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carbajal, Liliana. “The Role of EGFR Signaling in Gonadotropin-Induced Steroidogenesis.” 2010. Web. 21 Oct 2019.

Vancouver:

Carbajal L. The Role of EGFR Signaling in Gonadotropin-Induced Steroidogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/2152.5/804.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carbajal L. The Role of EGFR Signaling in Gonadotropin-Induced Steroidogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/804

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

14. Bae, James Jangho. Increased Transforming Growth Factor-β1 Modulates Hippocampal Glutamatergic Synaptic Protein Expression and Synaptic Transmission.

Degree: 2010, University of Toronto

Transforming growth factor-beta 1 (TGF-β1) is a multifunctional cytokine that orchestrates key events of development, disease and repair in the central nervous system (CNS). To… (more)

Subjects/Keywords: growth factor; brain; hippocampus; synapse; glutamate receptors; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bae, J. J. (2010). Increased Transforming Growth Factor-β1 Modulates Hippocampal Glutamatergic Synaptic Protein Expression and Synaptic Transmission. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24239

Chicago Manual of Style (16th Edition):

Bae, James Jangho. “Increased Transforming Growth Factor-β1 Modulates Hippocampal Glutamatergic Synaptic Protein Expression and Synaptic Transmission.” 2010. Masters Thesis, University of Toronto. Accessed October 21, 2019. http://hdl.handle.net/1807/24239.

MLA Handbook (7th Edition):

Bae, James Jangho. “Increased Transforming Growth Factor-β1 Modulates Hippocampal Glutamatergic Synaptic Protein Expression and Synaptic Transmission.” 2010. Web. 21 Oct 2019.

Vancouver:

Bae JJ. Increased Transforming Growth Factor-β1 Modulates Hippocampal Glutamatergic Synaptic Protein Expression and Synaptic Transmission. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1807/24239.

Council of Science Editors:

Bae JJ. Increased Transforming Growth Factor-β1 Modulates Hippocampal Glutamatergic Synaptic Protein Expression and Synaptic Transmission. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24239


Laurentian University

15. Wahby, Shahira Ahmed. Potential analysis of gene expression in nerve growth factor treated cells using semi quantitative PCR technique .

Degree: 2018, Laurentian University

 Over sixty years ago, nerve growth factor (NGF) was originally discovered as a neurotrophic factor essential for the survival of sensory and sympathetic neurons during… (more)

Subjects/Keywords: Nerve growth factor; TrkA, p75 receptors; PCR; gene expression

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wahby, S. A. (2018). Potential analysis of gene expression in nerve growth factor treated cells using semi quantitative PCR technique . (Thesis). Laurentian University. Retrieved from https://zone.biblio.laurentian.ca/handle/10219/3007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wahby, Shahira Ahmed. “Potential analysis of gene expression in nerve growth factor treated cells using semi quantitative PCR technique .” 2018. Thesis, Laurentian University. Accessed October 21, 2019. https://zone.biblio.laurentian.ca/handle/10219/3007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wahby, Shahira Ahmed. “Potential analysis of gene expression in nerve growth factor treated cells using semi quantitative PCR technique .” 2018. Web. 21 Oct 2019.

Vancouver:

Wahby SA. Potential analysis of gene expression in nerve growth factor treated cells using semi quantitative PCR technique . [Internet] [Thesis]. Laurentian University; 2018. [cited 2019 Oct 21]. Available from: https://zone.biblio.laurentian.ca/handle/10219/3007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wahby SA. Potential analysis of gene expression in nerve growth factor treated cells using semi quantitative PCR technique . [Thesis]. Laurentian University; 2018. Available from: https://zone.biblio.laurentian.ca/handle/10219/3007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Moore, Lisa. FGF signaling in Xenopus laevis lens regeneration.

Degree: PhD, Cell and Developmental Biology, 2015, University of Illinois – Urbana-Champaign

 The larvae of the frog Xenopus laevis is capable of regenerating lenses. In this re- generative process, the corneal tissue is capable of forming a… (more)

Subjects/Keywords: Xenopus laevis; lens; cornea; regeneration; Fibroblast Growth Factors (FGF); Fibroblast Growth Factor Receptors (FGFR)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moore, L. (2015). FGF signaling in Xenopus laevis lens regeneration. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/78759

Chicago Manual of Style (16th Edition):

Moore, Lisa. “FGF signaling in Xenopus laevis lens regeneration.” 2015. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 21, 2019. http://hdl.handle.net/2142/78759.

MLA Handbook (7th Edition):

Moore, Lisa. “FGF signaling in Xenopus laevis lens regeneration.” 2015. Web. 21 Oct 2019.

Vancouver:

Moore L. FGF signaling in Xenopus laevis lens regeneration. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2015. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/2142/78759.

Council of Science Editors:

Moore L. FGF signaling in Xenopus laevis lens regeneration. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2015. Available from: http://hdl.handle.net/2142/78759


Michigan State University

17. Liang, Hongyan. Role of hepatocyte growth factor and its receptor met in the malignant transformation of human fibroblasts.

Degree: PhD, Department of Biochemistry and Molecular Biology, 2001, Michigan State University

Subjects/Keywords: Carcinogenesis; Hepatocyte growth factor; Hepatocyte growth factor – Receptors; Fibroblasts

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liang, H. (2001). Role of hepatocyte growth factor and its receptor met in the malignant transformation of human fibroblasts. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:31160

Chicago Manual of Style (16th Edition):

Liang, Hongyan. “Role of hepatocyte growth factor and its receptor met in the malignant transformation of human fibroblasts.” 2001. Doctoral Dissertation, Michigan State University. Accessed October 21, 2019. http://etd.lib.msu.edu/islandora/object/etd:31160.

MLA Handbook (7th Edition):

Liang, Hongyan. “Role of hepatocyte growth factor and its receptor met in the malignant transformation of human fibroblasts.” 2001. Web. 21 Oct 2019.

Vancouver:

Liang H. Role of hepatocyte growth factor and its receptor met in the malignant transformation of human fibroblasts. [Internet] [Doctoral dissertation]. Michigan State University; 2001. [cited 2019 Oct 21]. Available from: http://etd.lib.msu.edu/islandora/object/etd:31160.

Council of Science Editors:

Liang H. Role of hepatocyte growth factor and its receptor met in the malignant transformation of human fibroblasts. [Doctoral Dissertation]. Michigan State University; 2001. Available from: http://etd.lib.msu.edu/islandora/object/etd:31160


University of Hong Kong

18. Chen, Jinna. EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma.

Degree: PhD, 2015, University of Hong Kong

abstract

Clinical Oncology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Epidermal growth factor - Receptors; Nasopharynx - Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, J. (2015). EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/223667

Chicago Manual of Style (16th Edition):

Chen, Jinna. “EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. http://hdl.handle.net/10722/223667.

MLA Handbook (7th Edition):

Chen, Jinna. “EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma.” 2015. Web. 21 Oct 2019.

Vancouver:

Chen J. EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10722/223667.

Council of Science Editors:

Chen J. EGFL6, a potential novel ligand of EGFR, plays roles in cancer metastasis by inducing cancer cell epithelial-mesenchymal transition in nasopharyngeal carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/223667


University of Hong Kong

19. 溫啟峰; Wan, Kai-fung. Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion.

Degree: M. Phil., 2007, University of Hong Kong

published_or_final_version

abstract

Biological Sciences

Master

Master of Philosophy

Advisors/Committee Members: Wong, AST.

Subjects/Keywords: Hepatocyte growth factor - Receptors.; Nasopharynx - Cancer.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

溫啟峰; Wan, K. (2007). Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. (Masters Thesis). University of Hong Kong. Retrieved from Wan, K. [溫啟峰]. (2007). Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955702 ; http://dx.doi.org/10.5353/th_b3955702 ; http://hdl.handle.net/10722/53096

Chicago Manual of Style (16th Edition):

溫啟峰; Wan, Kai-fung. “Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion.” 2007. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. Wan, K. [溫啟峰]. (2007). Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955702 ; http://dx.doi.org/10.5353/th_b3955702 ; http://hdl.handle.net/10722/53096.

MLA Handbook (7th Edition):

溫啟峰; Wan, Kai-fung. “Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion.” 2007. Web. 21 Oct 2019.

Vancouver:

溫啟峰; Wan K. Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. [Internet] [Masters thesis]. University of Hong Kong; 2007. [cited 2019 Oct 21]. Available from: Wan, K. [溫啟峰]. (2007). Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955702 ; http://dx.doi.org/10.5353/th_b3955702 ; http://hdl.handle.net/10722/53096.

Council of Science Editors:

溫啟峰; Wan K. Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. [Masters Thesis]. University of Hong Kong; 2007. Available from: Wan, K. [溫啟峰]. (2007). Hepatocyte growth factor and met receptor signaling in nasopharyngeal carcinoma cell migration and invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3955702 ; http://dx.doi.org/10.5353/th_b3955702 ; http://hdl.handle.net/10722/53096


University of California – San Diego

20. Peterman, Marshall Clarke. Regulation of Growth Factor Signaling, Golgi Orientation, and Cell Migration by GOLPH3.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 GOLPH3 is an oncogene that is amplified in many different tumor types and its overexpression often correlates with poor survival. Previously known functions of GOLPH3… (more)

Subjects/Keywords: Biology; Cellular biology; Golgi; GOLPH3; growth factor receptors; oncogene; signal transduction; trafficking

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Peterman, M. C. (2016). Regulation of Growth Factor Signaling, Golgi Orientation, and Cell Migration by GOLPH3. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/7wv2d8kn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peterman, Marshall Clarke. “Regulation of Growth Factor Signaling, Golgi Orientation, and Cell Migration by GOLPH3.” 2016. Thesis, University of California – San Diego. Accessed October 21, 2019. http://www.escholarship.org/uc/item/7wv2d8kn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peterman, Marshall Clarke. “Regulation of Growth Factor Signaling, Golgi Orientation, and Cell Migration by GOLPH3.” 2016. Web. 21 Oct 2019.

Vancouver:

Peterman MC. Regulation of Growth Factor Signaling, Golgi Orientation, and Cell Migration by GOLPH3. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Oct 21]. Available from: http://www.escholarship.org/uc/item/7wv2d8kn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peterman MC. Regulation of Growth Factor Signaling, Golgi Orientation, and Cell Migration by GOLPH3. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/7wv2d8kn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

21. Garay, Camilo. Regulation of epidermal growth factor receptor signaling by clathrin-coated membrane microdomains.

Degree: 2015, Ryerson University

 The phosphatidylinositol-3-kinase (PI3K)-Akt signaling axis controls cell survival, proliferation and metabolism, and is activated by receptor tyrosine kinases (RTKs) such as the epidermal growth factor(more)

Subjects/Keywords: Epidermal growth factor  – Receptors; Coated vesicles; Cancer  – Molecular aspects; Endocytosis; Cells  – Motility; Proteins  – Analysis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garay, C. (2015). Regulation of epidermal growth factor receptor signaling by clathrin-coated membrane microdomains. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A3721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garay, Camilo. “Regulation of epidermal growth factor receptor signaling by clathrin-coated membrane microdomains.” 2015. Thesis, Ryerson University. Accessed October 21, 2019. https://digital.library.ryerson.ca/islandora/object/RULA%3A3721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garay, Camilo. “Regulation of epidermal growth factor receptor signaling by clathrin-coated membrane microdomains.” 2015. Web. 21 Oct 2019.

Vancouver:

Garay C. Regulation of epidermal growth factor receptor signaling by clathrin-coated membrane microdomains. [Internet] [Thesis]. Ryerson University; 2015. [cited 2019 Oct 21]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garay C. Regulation of epidermal growth factor receptor signaling by clathrin-coated membrane microdomains. [Thesis]. Ryerson University; 2015. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

22. Judge, Gurjeet Singh. Clathrin and TOM1L1 regulate epidermal growth factor receptor signaling at the plasma membrane.

Degree: 2015, Ryerson University

 Epidermal growth factor (EGF) receptor (EGFR) controls many aspects of cell physiology via the activation of intracellular signaling pathways. Aberrant EGFR signaling and overexpression of… (more)

Subjects/Keywords: Epidermal growth factor; Cell receptors; Receptor-ligand complexes; Ligand binding (Biochemistry); Protein-tyrosine kinase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Judge, G. S. (2015). Clathrin and TOM1L1 regulate epidermal growth factor receptor signaling at the plasma membrane. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A4678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Judge, Gurjeet Singh. “Clathrin and TOM1L1 regulate epidermal growth factor receptor signaling at the plasma membrane.” 2015. Thesis, Ryerson University. Accessed October 21, 2019. https://digital.library.ryerson.ca/islandora/object/RULA%3A4678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Judge, Gurjeet Singh. “Clathrin and TOM1L1 regulate epidermal growth factor receptor signaling at the plasma membrane.” 2015. Web. 21 Oct 2019.

Vancouver:

Judge GS. Clathrin and TOM1L1 regulate epidermal growth factor receptor signaling at the plasma membrane. [Internet] [Thesis]. Ryerson University; 2015. [cited 2019 Oct 21]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A4678.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Judge GS. Clathrin and TOM1L1 regulate epidermal growth factor receptor signaling at the plasma membrane. [Thesis]. Ryerson University; 2015. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A4678

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

23. Chen, Jennifer Ying. Quartz Crystal Microbalance with Dissipation Monitoring: Sensing Beyond Cell-Substrate Adhesion.

Degree: 2017, Drexel University

The quartz crystal microbalance with dissipation monitoring (QCM-D) is an ultrasensitive mechanical sensing device that is capable of providing real-time, non-invasive measurements of changes in… (more)

Subjects/Keywords: Chemistry; Biochemistry; Cell adhesion; Cellular signal transduction; Energy dissipation; Epidermal growth factor; G proteins – Receptors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, J. Y. (2017). Quartz Crystal Microbalance with Dissipation Monitoring: Sensing Beyond Cell-Substrate Adhesion. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7560

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jennifer Ying. “Quartz Crystal Microbalance with Dissipation Monitoring: Sensing Beyond Cell-Substrate Adhesion.” 2017. Thesis, Drexel University. Accessed October 21, 2019. http://hdl.handle.net/1860/idea:7560.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jennifer Ying. “Quartz Crystal Microbalance with Dissipation Monitoring: Sensing Beyond Cell-Substrate Adhesion.” 2017. Web. 21 Oct 2019.

Vancouver:

Chen JY. Quartz Crystal Microbalance with Dissipation Monitoring: Sensing Beyond Cell-Substrate Adhesion. [Internet] [Thesis]. Drexel University; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1860/idea:7560.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen JY. Quartz Crystal Microbalance with Dissipation Monitoring: Sensing Beyond Cell-Substrate Adhesion. [Thesis]. Drexel University; 2017. Available from: http://hdl.handle.net/1860/idea:7560

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

24. Ng, Yu Pong. Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells.

Degree: 2004, Hong Kong University of Science and Technology

 Nerve growth factor (NGF) is required for the development of sympathetic neurons and subsets of sensory neurons. Our current knowledge on the molecular mechanisms underlying… (more)

Subjects/Keywords: Cell receptors; Leukemia inhibitory factor; Nerve growth factor; Neurons

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ng, Y. P. (2004). Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b818132 ; http://repository.ust.hk/ir/bitstream/1783.1-1913/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ng, Yu Pong. “Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells.” 2004. Thesis, Hong Kong University of Science and Technology. Accessed October 21, 2019. https://doi.org/10.14711/thesis-b818132 ; http://repository.ust.hk/ir/bitstream/1783.1-1913/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ng, Yu Pong. “Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells.” 2004. Web. 21 Oct 2019.

Vancouver:

Ng YP. Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2004. [cited 2019 Oct 21]. Available from: https://doi.org/10.14711/thesis-b818132 ; http://repository.ust.hk/ir/bitstream/1783.1-1913/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ng YP. Leukemia inhibitory factor receptor signaling in NGF-induced neuronal differentiation of PC12 cells. [Thesis]. Hong Kong University of Science and Technology; 2004. Available from: https://doi.org/10.14711/thesis-b818132 ; http://repository.ust.hk/ir/bitstream/1783.1-1913/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

25. Costa, Michelle. Unraveling the intricacies of spatial organization of the ErbB receptors and downstream signaling pathways.

Degree: Chemical and Biological Engineering, 2009, University of New Mexico

 Faced with the complexity of diseases such as cancer which has 1012 mutations, altering gene expression, and disrupting regulatory networks, there has been a paradigm… (more)

Subjects/Keywords: Monte Carlo; EGFR; Spatial Modeling; Receptor-sharing; Cellular signal transduction – Computer simulation; Cell receptors – Computer simulation.; Tumor proteins – Receptors – Computer simulation; Epidermal growth factor – Receptors – Computer simulation; Monte Carlo method

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Costa, M. (2009). Unraveling the intricacies of spatial organization of the ErbB receptors and downstream signaling pathways. (Doctoral Dissertation). University of New Mexico. Retrieved from http://hdl.handle.net/1928/9956

Chicago Manual of Style (16th Edition):

Costa, Michelle. “Unraveling the intricacies of spatial organization of the ErbB receptors and downstream signaling pathways.” 2009. Doctoral Dissertation, University of New Mexico. Accessed October 21, 2019. http://hdl.handle.net/1928/9956.

MLA Handbook (7th Edition):

Costa, Michelle. “Unraveling the intricacies of spatial organization of the ErbB receptors and downstream signaling pathways.” 2009. Web. 21 Oct 2019.

Vancouver:

Costa M. Unraveling the intricacies of spatial organization of the ErbB receptors and downstream signaling pathways. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1928/9956.

Council of Science Editors:

Costa M. Unraveling the intricacies of spatial organization of the ErbB receptors and downstream signaling pathways. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: http://hdl.handle.net/1928/9956

26. Fanganiello, Roberto Dalto. Estudo de expressão gênica e de comportamento celular em células de indivíduos portadores de craniossinostoses sindrômicas.

Degree: PhD, Biologia (Genética), 2010, University of São Paulo

 Um dos grupos de doenças mais importante que acomete o desenvolvimento da caixa craniana humana é o das craniossinostoses, caracterizado pelo fechamento prematuro de uma… (more)

Subjects/Keywords: Craniossinostoses sindrômicas; Fibroblast growth factor receptors signaling; Sinalização por receptores de fatores de crescimento de fibroblastos; Syndromic craniosynostosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fanganiello, R. D. (2010). Estudo de expressão gênica e de comportamento celular em células de indivíduos portadores de craniossinostoses sindrômicas. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41131/tde-16042010-162021/ ;

Chicago Manual of Style (16th Edition):

Fanganiello, Roberto Dalto. “Estudo de expressão gênica e de comportamento celular em células de indivíduos portadores de craniossinostoses sindrômicas.” 2010. Doctoral Dissertation, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/41/41131/tde-16042010-162021/ ;.

MLA Handbook (7th Edition):

Fanganiello, Roberto Dalto. “Estudo de expressão gênica e de comportamento celular em células de indivíduos portadores de craniossinostoses sindrômicas.” 2010. Web. 21 Oct 2019.

Vancouver:

Fanganiello RD. Estudo de expressão gênica e de comportamento celular em células de indivíduos portadores de craniossinostoses sindrômicas. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-16042010-162021/ ;.

Council of Science Editors:

Fanganiello RD. Estudo de expressão gênica e de comportamento celular em células de indivíduos portadores de craniossinostoses sindrômicas. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-16042010-162021/ ;


Univerzitet u Beogradu

27. Robajac, Dragana B., 1985-. N-glikom membranskih proteina i receptora za insulin i faktore rasta slične insulinu, izolovanih iz humane placente u različitim (pato)fiziološkim stanjima.

Degree: Hemijski fakultet, 2016, Univerzitet u Beogradu

Biohemija - Biohemija proteina / Biochemistry - Biochemistry of proteins

Funkcije membranskih proteina su brojne: međućelijska komunikacija, adhezija, signalna transdukcija. Većina membranskih proteina je glikozilovana… (more)

Subjects/Keywords: membrane proteins; N-glycans; N-glycome; insulin receptor; insulin-like growth factor receptors type 1 and 2; gestational changes; aging; placenta

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robajac, Dragana B., 1. (2016). N-glikom membranskih proteina i receptora za insulin i faktore rasta slične insulinu, izolovanih iz humane placente u različitim (pato)fiziološkim stanjima. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:12455/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robajac, Dragana B., 1985-. “N-glikom membranskih proteina i receptora za insulin i faktore rasta slične insulinu, izolovanih iz humane placente u različitim (pato)fiziološkim stanjima.” 2016. Thesis, Univerzitet u Beogradu. Accessed October 21, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:12455/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robajac, Dragana B., 1985-. “N-glikom membranskih proteina i receptora za insulin i faktore rasta slične insulinu, izolovanih iz humane placente u različitim (pato)fiziološkim stanjima.” 2016. Web. 21 Oct 2019.

Vancouver:

Robajac, Dragana B. 1. N-glikom membranskih proteina i receptora za insulin i faktore rasta slične insulinu, izolovanih iz humane placente u različitim (pato)fiziološkim stanjima. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2019 Oct 21]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12455/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robajac, Dragana B. 1. N-glikom membranskih proteina i receptora za insulin i faktore rasta slične insulinu, izolovanih iz humane placente u različitim (pato)fiziološkim stanjima. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12455/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

28. Fender, Alexander W. Role of Notch signaling in tumorigenesis, stemness, and epithelial to mesenchymal transtion in colorectal cancer.

Degree: 2015, East Carolina University

 Colorectal cancer is the third most commonly diagnosed cancer in both men and women in the United States. Surgical resection and combination chemotherapy are often… (more)

Subjects/Keywords: Biology; Biochemistry; Molecular biology;

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fender, A. W. (2015). Role of Notch signaling in tumorigenesis, stemness, and epithelial to mesenchymal transtion in colorectal cancer. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4948

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fender, Alexander W. “Role of Notch signaling in tumorigenesis, stemness, and epithelial to mesenchymal transtion in colorectal cancer.” 2015. Thesis, East Carolina University. Accessed October 21, 2019. http://hdl.handle.net/10342/4948.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fender, Alexander W. “Role of Notch signaling in tumorigenesis, stemness, and epithelial to mesenchymal transtion in colorectal cancer.” 2015. Web. 21 Oct 2019.

Vancouver:

Fender AW. Role of Notch signaling in tumorigenesis, stemness, and epithelial to mesenchymal transtion in colorectal cancer. [Internet] [Thesis]. East Carolina University; 2015. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10342/4948.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fender AW. Role of Notch signaling in tumorigenesis, stemness, and epithelial to mesenchymal transtion in colorectal cancer. [Thesis]. East Carolina University; 2015. Available from: http://hdl.handle.net/10342/4948

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

29. 紀思思; Kee, Francis. Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-likepeptide.

Degree: M. Phil., 2000, University of Hong Kong

published_or_final_version

Zoology

Master

Master of Philosophy

Subjects/Keywords: Goldfish - Physiology.; Growth hormone releasing factor.; Hormone receptors.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

紀思思; Kee, F. (2000). Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-likepeptide. (Masters Thesis). University of Hong Kong. Retrieved from Kee, F. [紀思思]. (2000). Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-like peptide. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122276 ; http://dx.doi.org/10.5353/th_b3122276 ; http://hdl.handle.net/10722/33371

Chicago Manual of Style (16th Edition):

紀思思; Kee, Francis. “Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-likepeptide.” 2000. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. Kee, F. [紀思思]. (2000). Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-like peptide. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122276 ; http://dx.doi.org/10.5353/th_b3122276 ; http://hdl.handle.net/10722/33371.

MLA Handbook (7th Edition):

紀思思; Kee, Francis. “Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-likepeptide.” 2000. Web. 21 Oct 2019.

Vancouver:

紀思思; Kee F. Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-likepeptide. [Internet] [Masters thesis]. University of Hong Kong; 2000. [cited 2019 Oct 21]. Available from: Kee, F. [紀思思]. (2000). Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-like peptide. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122276 ; http://dx.doi.org/10.5353/th_b3122276 ; http://hdl.handle.net/10722/33371.

Council of Science Editors:

紀思思; Kee F. Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-likepeptide. [Masters Thesis]. University of Hong Kong; 2000. Available from: Kee, F. [紀思思]. (2000). Aspartic acid scanning mutation analysis of a receptor isolated from goldfish specific to the growth hormone releasing hormone salmon-like peptide. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122276 ; http://dx.doi.org/10.5353/th_b3122276 ; http://hdl.handle.net/10722/33371


University of Hong Kong

30. Zhou, Hongyan. Hepatocyte growth factor-met signaling in ovarian cancer progression.

Degree: PhD, 2007, University of Hong Kong

abstract

published_or_final_version

Zoology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Wong, AST, Lee, WWM.

Subjects/Keywords: Hepatocyte growth factor - Receptors; Ovaries - Cancer - Genetic aspects.; Cytogenetics.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, H. (2007). Hepatocyte growth factor-met signaling in ovarian cancer progression. (Doctoral Dissertation). University of Hong Kong. Retrieved from Zhou, H. [周紅艷]. (2007). Hepatocyte growth factor-met signaling in ovarian cancer progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3793798 ; http://dx.doi.org/10.5353/th_b3793798 ; http://hdl.handle.net/10722/52868

Chicago Manual of Style (16th Edition):

Zhou, Hongyan. “Hepatocyte growth factor-met signaling in ovarian cancer progression.” 2007. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. Zhou, H. [周紅艷]. (2007). Hepatocyte growth factor-met signaling in ovarian cancer progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3793798 ; http://dx.doi.org/10.5353/th_b3793798 ; http://hdl.handle.net/10722/52868.

MLA Handbook (7th Edition):

Zhou, Hongyan. “Hepatocyte growth factor-met signaling in ovarian cancer progression.” 2007. Web. 21 Oct 2019.

Vancouver:

Zhou H. Hepatocyte growth factor-met signaling in ovarian cancer progression. [Internet] [Doctoral dissertation]. University of Hong Kong; 2007. [cited 2019 Oct 21]. Available from: Zhou, H. [周紅艷]. (2007). Hepatocyte growth factor-met signaling in ovarian cancer progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3793798 ; http://dx.doi.org/10.5353/th_b3793798 ; http://hdl.handle.net/10722/52868.

Council of Science Editors:

Zhou H. Hepatocyte growth factor-met signaling in ovarian cancer progression. [Doctoral Dissertation]. University of Hong Kong; 2007. Available from: Zhou, H. [周紅艷]. (2007). Hepatocyte growth factor-met signaling in ovarian cancer progression. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3793798 ; http://dx.doi.org/10.5353/th_b3793798 ; http://hdl.handle.net/10722/52868

[1] [2] [3] [4] [5] … [1032]

.