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You searched for subject:( Pancreas pathology 60). Showing records 1 – 30 of 12262 total matches.

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1. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 23 Oct 2019.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

2. Wang, Kai. Involvement of O-glcnacylation in lens development and cataract formation.

Degree: PhD, 2008, University of Alabama – Birmingham

O-GlcNAcylation is a ubiquitous and dynamic post-translational modification whereby the monosaccharide N-acetylglucosamine (GlcNAc) is affixed to serine or threonine hydroxyls via a beta linkage on… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Cataract  – pathology<; br>; Cell Differentiation<; br>; Lens, Crystalline  – cytology<; br>; Lens, Crystalline  – pathology<; br>; gamma-Crystallins  – metabolism

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APA (6th Edition):

Wang, K. (2008). Involvement of O-glcnacylation in lens development and cataract formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,691

Chicago Manual of Style (16th Edition):

Wang, Kai. “Involvement of O-glcnacylation in lens development and cataract formation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,691.

MLA Handbook (7th Edition):

Wang, Kai. “Involvement of O-glcnacylation in lens development and cataract formation.” 2008. Web. 23 Oct 2019.

Vancouver:

Wang K. Involvement of O-glcnacylation in lens development and cataract formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,691.

Council of Science Editors:

Wang K. Involvement of O-glcnacylation in lens development and cataract formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,691

3. Karki, Anju. Investigating the role of the basic helix-loop-helix transcription factor MIST1 in pancreatic diseases.

Degree: PhD, Biological Science, 2016, Purdue University

  Acinar cells of the exocrine pancreas are dedicated to synthesize, package and secrete immense quantities of pro-digestive enzymes to maintain proper metabolic homeostasis for… (more)

Subjects/Keywords: Biological sciences; Health and environmental sciences; Acinar cells; MIST1; Pancreas; Pathology; Physiology

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APA (6th Edition):

Karki, A. (2016). Investigating the role of the basic helix-loop-helix transcription factor MIST1 in pancreatic diseases. (Doctoral Dissertation). Purdue University. Retrieved from https://docs.lib.purdue.edu/open_access_dissertations/665

Chicago Manual of Style (16th Edition):

Karki, Anju. “Investigating the role of the basic helix-loop-helix transcription factor MIST1 in pancreatic diseases.” 2016. Doctoral Dissertation, Purdue University. Accessed October 23, 2019. https://docs.lib.purdue.edu/open_access_dissertations/665.

MLA Handbook (7th Edition):

Karki, Anju. “Investigating the role of the basic helix-loop-helix transcription factor MIST1 in pancreatic diseases.” 2016. Web. 23 Oct 2019.

Vancouver:

Karki A. Investigating the role of the basic helix-loop-helix transcription factor MIST1 in pancreatic diseases. [Internet] [Doctoral dissertation]. Purdue University; 2016. [cited 2019 Oct 23]. Available from: https://docs.lib.purdue.edu/open_access_dissertations/665.

Council of Science Editors:

Karki A. Investigating the role of the basic helix-loop-helix transcription factor MIST1 in pancreatic diseases. [Doctoral Dissertation]. Purdue University; 2016. Available from: https://docs.lib.purdue.edu/open_access_dissertations/665

4. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 23 Oct 2019.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

5. Prucka, William R. Wavelet-based regression and classification for longitudinal diffusion tensor imaging data.

Degree: PhD, 2008, University of Alabama – Birmingham

Diffusion tensor imaging (DTI) is a magnetic resonance imaging (MRI) technique capable of in vivo characterization of the spatial and angular dependence of free water… (more)

Subjects/Keywords: Brain  – pathology <; br>; Diffusion Magnetic Resonance Imaging  – instrumentation <; br>; Disease Progression <; br>; Image Interpretation, Computer-Assisted  – methods <; br>; Longitudinal Studies <; br>; Multiple Sclerosis  – pathology <; br>; Nerve Fibers, Myelinated  – pathology

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APA (6th Edition):

Prucka, W. R. (2008). Wavelet-based regression and classification for longitudinal diffusion tensor imaging data. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,335

Chicago Manual of Style (16th Edition):

Prucka, William R. “Wavelet-based regression and classification for longitudinal diffusion tensor imaging data.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,335.

MLA Handbook (7th Edition):

Prucka, William R. “Wavelet-based regression and classification for longitudinal diffusion tensor imaging data.” 2008. Web. 23 Oct 2019.

Vancouver:

Prucka WR. Wavelet-based regression and classification for longitudinal diffusion tensor imaging data. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,335.

Council of Science Editors:

Prucka WR. Wavelet-based regression and classification for longitudinal diffusion tensor imaging data. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,335

6. Walls, Ken C. Molecular characterization of neural apoptosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Neural cell death plays a critical role in normal nervous system development and dysregulated neural stem cell death contributes to brain malformation, tumorgenesis, and possibly,… (more)

Subjects/Keywords: Apoptosis<; br>; Autophagy  – genetics<; br>; Cell Death<; br>; Lysosomes <; br>; physiology Neurons  – pathology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Walls, K. C. (2009). Molecular characterization of neural apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,504

Chicago Manual of Style (16th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,504.

MLA Handbook (7th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Web. 23 Oct 2019.

Vancouver:

Walls KC. Molecular characterization of neural apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504.

Council of Science Editors:

Walls KC. Molecular characterization of neural apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504

7. Keel, Theresa L. Lipomatous tissue: variation in treatment procedures, comorbidities, and outcomes.

Degree: PhD, 2008, University of Alabama – Birmingham

Benign lipomas are the most common soft tissue tumors. They exhibit great variation in severity and are associated with significant comorbidity. Advanced understanding of genetic… (more)

Subjects/Keywords: Comorbidity<; br>; Lipoma  – pathology<; br>; Retrospective Studies<; br>; Treatment Outcome

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APA (6th Edition):

Keel, T. L. (2008). Lipomatous tissue: variation in treatment procedures, comorbidities, and outcomes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,820

Chicago Manual of Style (16th Edition):

Keel, Theresa L. “Lipomatous tissue: variation in treatment procedures, comorbidities, and outcomes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,820.

MLA Handbook (7th Edition):

Keel, Theresa L. “Lipomatous tissue: variation in treatment procedures, comorbidities, and outcomes.” 2008. Web. 23 Oct 2019.

Vancouver:

Keel TL. Lipomatous tissue: variation in treatment procedures, comorbidities, and outcomes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,820.

Council of Science Editors:

Keel TL. Lipomatous tissue: variation in treatment procedures, comorbidities, and outcomes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,820

8. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

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APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 23 Oct 2019.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194


Marshall University

9. Rutherford, Kelly Jean. Exploration of growth mindset application in communication sciences and disorders.

Degree: 2019, Marshall University

 As early as 1988, Carol Dweck began a line of research connecting motivation and intelligence that later became known as mindset theory (Dweck & Leggett,… (more)

Subjects/Keywords: administration; growth mindset; higher education; speech language pathology; <; p>; Thought and thinking.<; /p>; <; p>; Communicative disorders.<; /p>; <; p>; Performance.<; /p>; <; p>; Universities and colleges  – Administration.<; /p>;

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APA (6th Edition):

Rutherford, K. J. (2019). Exploration of growth mindset application in communication sciences and disorders. (Thesis). Marshall University. Retrieved from https://mds.marshall.edu/etd/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rutherford, Kelly Jean. “Exploration of growth mindset application in communication sciences and disorders.” 2019. Thesis, Marshall University. Accessed October 23, 2019. https://mds.marshall.edu/etd/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rutherford, Kelly Jean. “Exploration of growth mindset application in communication sciences and disorders.” 2019. Web. 23 Oct 2019.

Vancouver:

Rutherford KJ. Exploration of growth mindset application in communication sciences and disorders. [Internet] [Thesis]. Marshall University; 2019. [cited 2019 Oct 23]. Available from: https://mds.marshall.edu/etd/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rutherford KJ. Exploration of growth mindset application in communication sciences and disorders. [Thesis]. Marshall University; 2019. Available from: https://mds.marshall.edu/etd/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Bantug, Glenn Robert Burgner. CD8+ T-lymphocytes and the control of cytomegalovirus infection of the newborn central nervous system.

Degree: PhD, 2007, University of Alabama – Birmingham

Congenital HCMV infection of the developing brain is the leading viral cause of mental retardation and sensorineural hearing loss. To elucidate the pathogenesis of congenital… (more)

Subjects/Keywords: Brain  – pathology <; br>; CD8-Positive T-Lymphocytes  – immunology <; br>; Central Nervous System Diseases  – immunology <; br>; Central Nervous System Diseases  – virology <; br>; Cytomegalovirus Infections  – pathology <; br>; Muromegalovirus  – physiology <; br>; Virus Replication

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APA (6th Edition):

Bantug, G. R. B. (2007). CD8+ T-lymphocytes and the control of cytomegalovirus infection of the newborn central nervous system. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,359

Chicago Manual of Style (16th Edition):

Bantug, Glenn Robert Burgner. “CD8+ T-lymphocytes and the control of cytomegalovirus infection of the newborn central nervous system.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,359.

MLA Handbook (7th Edition):

Bantug, Glenn Robert Burgner. “CD8+ T-lymphocytes and the control of cytomegalovirus infection of the newborn central nervous system.” 2007. Web. 23 Oct 2019.

Vancouver:

Bantug GRB. CD8+ T-lymphocytes and the control of cytomegalovirus infection of the newborn central nervous system. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,359.

Council of Science Editors:

Bantug GRB. CD8+ T-lymphocytes and the control of cytomegalovirus infection of the newborn central nervous system. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,359

11. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 23 Oct 2019.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045

12. Bomben, Valerie Christine. Role of transient receptor potential canonical channels in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Gliomas, primary brain tumors derived from glial cells, constitute the majority of malignant tumors within the central nervous system. The most malignant of these tumors,… (more)

Subjects/Keywords: Brain Neoplasms  – pathology<; br>; Cell Cycle<; br>; Glioma  – pathology<; br>; Transient Receptor Potential Channels  – antagonists & inhibitors

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APA (6th Edition):

Bomben, V. C. (2010). Role of transient receptor potential canonical channels in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,642

Chicago Manual of Style (16th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,642.

MLA Handbook (7th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Web. 23 Oct 2019.

Vancouver:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642.

Council of Science Editors:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642

13. Hovater, Michael (Michael Brian). Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.

Degree: MS, 2006, University of Alabama – Birmingham

This thesis concerns purinergic signaling in renal epithelial cells of normal and polycystic kidneys. The first section discusses first principles of “purinergic signaling” as they… (more)

Subjects/Keywords: Epithelial Cells  – physiology <; br>; Kidney Tubules  – physiology <; br>; Polycystic Kidney Diseases  – pathology <; br>; Receptors, Purinergic P2  – metabolism <; br>; Signal Transduction  – physiology

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APA (6th Edition):

Hovater, M. (. B. (2006). Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,30

Chicago Manual of Style (16th Edition):

Hovater, Michael (Michael Brian). “Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.” 2006. Masters Thesis, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,30.

MLA Handbook (7th Edition):

Hovater, Michael (Michael Brian). “Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.” 2006. Web. 23 Oct 2019.

Vancouver:

Hovater M(B. Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,30.

Council of Science Editors:

Hovater M(B. Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. [Masters Thesis]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,30

14. Koontz, Thadeus B. MCMV induced cerebellar maldevelopment.

Degree: PhD, 2007, University of Alabama – Birmingham

Congenital Human Cytomegalovirus (HCMV) infection represents a significant cause of morbidity and mortality in infants and children. While most of the systemic damage incurred by… (more)

Subjects/Keywords: Central Nervous System Diseases  – virology <; br>; Cerebellum  – abnormalities <; br>; Cytomegalovirus Infections  – immunology <; br>; Cytomegalovirus Infections  – pathology <; br>; Disease Models, Animal

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APA (6th Edition):

Koontz, T. B. (2007). MCMV induced cerebellar maldevelopment. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,130

Chicago Manual of Style (16th Edition):

Koontz, Thadeus B. “MCMV induced cerebellar maldevelopment.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,130.

MLA Handbook (7th Edition):

Koontz, Thadeus B. “MCMV induced cerebellar maldevelopment.” 2007. Web. 23 Oct 2019.

Vancouver:

Koontz TB. MCMV induced cerebellar maldevelopment. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,130.

Council of Science Editors:

Koontz TB. MCMV induced cerebellar maldevelopment. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,130

15. Atkinson, George P. (George Prescott). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma (GBM) is an incurable tumor of the central nervous system (CNS). Over the past 50 years, little progress has made in improving the quality… (more)

Subjects/Keywords: Glioblastoma  – pathology<; br>; Neoplasms<; br>; NF-kappa B  – metabolism<; br>; Peptidylprolyl Isomerase<; br>; STAT3 Transcription Factor

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APA (6th Edition):

Atkinson, G. P. (. P. (2009). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,812

Chicago Manual of Style (16th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,812.

MLA Handbook (7th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Web. 23 Oct 2019.

Vancouver:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812.

Council of Science Editors:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812

16. Ritchie, Joseph P. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.

Degree: PhD, 2010, University of Alabama – Birmingham

Heparanase, an endoglycosidase which cleaves heparan sulfate chains at specific sites, is rarely expressed in normal tissues but becomes evident in many human cancers. We… (more)

Subjects/Keywords: Heparin  – metabolism<; br>; Heparitin Sulfate  – metabolism<; br>; Multiple Myeloma  – pathology<; br>; Neoplasm Invasiveness<; br>; Syndecan-1  – metabolism

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APA (6th Edition):

Ritchie, J. P. (2010). Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,852

Chicago Manual of Style (16th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,852.

MLA Handbook (7th Edition):

Ritchie, Joseph P. “Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma.” 2010. Web. 23 Oct 2019.

Vancouver:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852.

Council of Science Editors:

Ritchie JP. Heparanase drives the aggressive myeloma phenotype: preclinical development of a heparanase inhibitor for the treatment of multiple myeloma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,852


Marshall University

17. Valentine, Meagan Elisabeth. Chmp1 negatively regulates Epidermal Growth Factor signaling in the Drosophila wing.

Degree: 2014, Marshall University

 A critical step in cellular signaling through transmembrane receptors is the down-regulation of activated receptors through the multivesicular body (MVB) pathway to the lysosome. MVB… (more)

Subjects/Keywords: Chmp1; DER; Drosophila; EGFR; MVB; <; p>; Pancreas - Tumors.<; /p>; <; p>; Tumor suppressor proteins.<; /p>;

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APA (6th Edition):

Valentine, M. E. (2014). Chmp1 negatively regulates Epidermal Growth Factor signaling in the Drosophila wing. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/802

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Valentine, Meagan Elisabeth. “Chmp1 negatively regulates Epidermal Growth Factor signaling in the Drosophila wing.” 2014. Thesis, Marshall University. Accessed October 23, 2019. http://mds.marshall.edu/etd/802.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Valentine, Meagan Elisabeth. “Chmp1 negatively regulates Epidermal Growth Factor signaling in the Drosophila wing.” 2014. Web. 23 Oct 2019.

Vancouver:

Valentine ME. Chmp1 negatively regulates Epidermal Growth Factor signaling in the Drosophila wing. [Internet] [Thesis]. Marshall University; 2014. [cited 2019 Oct 23]. Available from: http://mds.marshall.edu/etd/802.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Valentine ME. Chmp1 negatively regulates Epidermal Growth Factor signaling in the Drosophila wing. [Thesis]. Marshall University; 2014. Available from: http://mds.marshall.edu/etd/802

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 23 Oct 2019.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906

19. Li, Yichao. Functional imaging of excitable cells : retinal neurons and pancreatic islet beta cells.

Degree: MS, 2011, University of Alabama – Birmingham

Stimulus-evoked fast intrinsic optical signals (IOSs) have time courses compara-ble to electrophysiological kinetics, and thus promise a high-spatial and high-temporal resolution method for functional evaluation… (more)

Subjects/Keywords: Retina  – Imaging  – Equipment and supplies  – Design and construction <; br>; Pancreas  – Imaging  – Equipment and supplies  – Design and construction <; br>; Retinal ganglion cells <; br>; Pancreatic beta cells <; br>; Nerve tissue  – Optical properties  – Measurement <; br>; Diagnostic imaging <; br>; Excitation (Physiology)

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APA (6th Edition):

Li, Y. (2011). Functional imaging of excitable cells : retinal neurons and pancreatic islet beta cells. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,976

Chicago Manual of Style (16th Edition):

Li, Yichao. “Functional imaging of excitable cells : retinal neurons and pancreatic islet beta cells.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,976.

MLA Handbook (7th Edition):

Li, Yichao. “Functional imaging of excitable cells : retinal neurons and pancreatic islet beta cells.” 2011. Web. 23 Oct 2019.

Vancouver:

Li Y. Functional imaging of excitable cells : retinal neurons and pancreatic islet beta cells. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,976.

Council of Science Editors:

Li Y. Functional imaging of excitable cells : retinal neurons and pancreatic islet beta cells. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,976


Marshall University

20. Carper, Miranda B. Identification and characterization of downstream effector protein(s) regulated by p53 and pRb.

Degree: 2014, Marshall University

 A commonality among cancer types is the high frequency of mutations that inhibit or alter signaling of the p53 and pRb (Retinoblastoma) tumor suppressors. These… (more)

Subjects/Keywords: Cancer; epidermal growth factor; migration; RB1; RGS16; TP53; <; p>; Cancer cells - Growth.<; /p>; <; p>; Cancer cells - Research.<; /p>; <; p>; Pancreas  – Cysts- Research.<; /p>;

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APA (6th Edition):

Carper, M. B. (2014). Identification and characterization of downstream effector protein(s) regulated by p53 and pRb. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carper, Miranda B. “Identification and characterization of downstream effector protein(s) regulated by p53 and pRb.” 2014. Thesis, Marshall University. Accessed October 23, 2019. http://mds.marshall.edu/etd/897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carper, Miranda B. “Identification and characterization of downstream effector protein(s) regulated by p53 and pRb.” 2014. Web. 23 Oct 2019.

Vancouver:

Carper MB. Identification and characterization of downstream effector protein(s) regulated by p53 and pRb. [Internet] [Thesis]. Marshall University; 2014. [cited 2019 Oct 23]. Available from: http://mds.marshall.edu/etd/897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carper MB. Identification and characterization of downstream effector protein(s) regulated by p53 and pRb. [Thesis]. Marshall University; 2014. Available from: http://mds.marshall.edu/etd/897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Flynn, Robert Jason. The role of the classical NF-κB pathway in HSC self-renewal and acute myleloid leukemia.

Degree: PhD, 2011, University of Alabama – Birmingham

Acute myeloid leukemia (AML) comprises approximately 25% of newly diagnosed cases of leukemia each year. The constitutive activation of the classical NF-κB signaling pathway has… (more)

Subjects/Keywords: Hematopoietic Stem Cells  – pathology<; br>; I-kappa B Proteins<; br>; Leukemia, Myeloid, Acute  – genetics<; br>; Leukemia, Myeloid, Acute  – pathology<; br>; Mice, Knockout<; br>; NF-kappa B  – antagonists & inhibitors NF-kappa B p50 Subunit  – genetics

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APA (6th Edition):

Flynn, R. J. (2011). The role of the classical NF-κB pathway in HSC self-renewal and acute myleloid leukemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,862

Chicago Manual of Style (16th Edition):

Flynn, Robert Jason. “The role of the classical NF-κB pathway in HSC self-renewal and acute myleloid leukemia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,862.

MLA Handbook (7th Edition):

Flynn, Robert Jason. “The role of the classical NF-κB pathway in HSC self-renewal and acute myleloid leukemia.” 2011. Web. 23 Oct 2019.

Vancouver:

Flynn RJ. The role of the classical NF-κB pathway in HSC self-renewal and acute myleloid leukemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,862.

Council of Science Editors:

Flynn RJ. The role of the classical NF-κB pathway in HSC self-renewal and acute myleloid leukemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,862

22. Atkins, Kelly Dunham. The effect of sulfasalazine on functional recovery and neuropathic pain following spinal cord injury.

Degree: PhD, 2011, University of Alabama – Birmingham

Spinal cord injury (SCI) is a devastating condition resulting in loss of motor function as well as sensory abnormalities. Insight into the pathophysiology of SCI… (more)

Subjects/Keywords: Gliosis  – pathology<; br>; NF-kappa B  – metabolism<; br>; Pain  – metabolism<; br>; Spinal Cord Injuries  – complications<; br>; Spinal Cord Injuries  – pathology<; br>; Sulfasalazine

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APA (6th Edition):

Atkins, K. D. (2011). The effect of sulfasalazine on functional recovery and neuropathic pain following spinal cord injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1034

Chicago Manual of Style (16th Edition):

Atkins, Kelly Dunham. “The effect of sulfasalazine on functional recovery and neuropathic pain following spinal cord injury.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1034.

MLA Handbook (7th Edition):

Atkins, Kelly Dunham. “The effect of sulfasalazine on functional recovery and neuropathic pain following spinal cord injury.” 2011. Web. 23 Oct 2019.

Vancouver:

Atkins KD. The effect of sulfasalazine on functional recovery and neuropathic pain following spinal cord injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1034.

Council of Science Editors:

Atkins KD. The effect of sulfasalazine on functional recovery and neuropathic pain following spinal cord injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1034

23. Suranyi, Pal. Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction.

Degree: PhD, 2007, University of Alabama – Birmingham

Ischemic heart disease is the leading cause of mortality in western societies. Treatment needs to be tailored for each individual, which requires accurate assessment of… (more)

Subjects/Keywords: Algorithms <; br>; Contrast Media  – pharmacokinetics <; br>; Image Enhancement  – methods <; br>; Image Interpretation, Computer-Assisted  – methods <; br>; Magnetic Resonance Imaging  – methods <; br>; Myocardial Infarction  – diagnosis <; br>; Myocardial Infarction  – pathology <; br>; Myocardial Stunning  – diagnosis <; br>; Myocardium  – metabolism <; br>; Organometallic Compounds  – pharmacokinetics

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APA (6th Edition):

Suranyi, P. (2007). Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,151

Chicago Manual of Style (16th Edition):

Suranyi, Pal. “Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,151.

MLA Handbook (7th Edition):

Suranyi, Pal. “Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction.” 2007. Web. 23 Oct 2019.

Vancouver:

Suranyi P. Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,151.

Council of Science Editors:

Suranyi P. Relaxation rate-based magnetic resonance imaging quantification of myocardial infarction. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,151


Case Western Reserve University

24. Walrath, Jessica R. THE ROLE OF ALPHA 4 BETA 1 INTEGRIN (VLA-4) IN RECRUITMENT OF MYCOBACTERIUM TUBERCULOSIS-SPECIFIC TH1-LIKE RECALL RESPONSES TO THE HUMAN LUNG.

Degree: MSs, Pathology, 2008, Case Western Reserve University

 Tuberculosis remains a major international health threat. Although BCG vaccination does not clearly protect against pulmonary tuberculosis, &#60;i>Mycobacterium tuberculosis&#60;/i>exposed individuals are relatively protected against subsequent… (more)

Subjects/Keywords: Health Sciences, Pathology; &; alpha 4&; beta 1; VLA-4; <; i>; Mycobacterium tuberculosis<; /i>;

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APA (6th Edition):

Walrath, J. R. (2008). THE ROLE OF ALPHA 4 BETA 1 INTEGRIN (VLA-4) IN RECRUITMENT OF MYCOBACTERIUM TUBERCULOSIS-SPECIFIC TH1-LIKE RECALL RESPONSES TO THE HUMAN LUNG. (Masters Thesis). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1196182534

Chicago Manual of Style (16th Edition):

Walrath, Jessica R. “THE ROLE OF ALPHA 4 BETA 1 INTEGRIN (VLA-4) IN RECRUITMENT OF MYCOBACTERIUM TUBERCULOSIS-SPECIFIC TH1-LIKE RECALL RESPONSES TO THE HUMAN LUNG.” 2008. Masters Thesis, Case Western Reserve University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1196182534.

MLA Handbook (7th Edition):

Walrath, Jessica R. “THE ROLE OF ALPHA 4 BETA 1 INTEGRIN (VLA-4) IN RECRUITMENT OF MYCOBACTERIUM TUBERCULOSIS-SPECIFIC TH1-LIKE RECALL RESPONSES TO THE HUMAN LUNG.” 2008. Web. 23 Oct 2019.

Vancouver:

Walrath JR. THE ROLE OF ALPHA 4 BETA 1 INTEGRIN (VLA-4) IN RECRUITMENT OF MYCOBACTERIUM TUBERCULOSIS-SPECIFIC TH1-LIKE RECALL RESPONSES TO THE HUMAN LUNG. [Internet] [Masters thesis]. Case Western Reserve University; 2008. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1196182534.

Council of Science Editors:

Walrath JR. THE ROLE OF ALPHA 4 BETA 1 INTEGRIN (VLA-4) IN RECRUITMENT OF MYCOBACTERIUM TUBERCULOSIS-SPECIFIC TH1-LIKE RECALL RESPONSES TO THE HUMAN LUNG. [Masters Thesis]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1196182534

25. Katre, Ashwini A. Ozone and lung fibrosis.

Degree: M.S.P.H., 2009, University of Alabama – Birmingham

Ozone is an extremely reactive gas molecule composed of three oxygen atoms. The ground-level ozone is formed when nitrogen oxides and volatile organic compounds, released… (more)

Subjects/Keywords: Actins<; br>; Collagen  – metabolism<; br>; Ozone  – toxicity<; br>; Plasminogen Activator Inhibitor 1<; br>; Pulmonary Fibrosis  – chemically induced<; br>; Pulmonary Fibrosis  – pathology<; br>; Transforming Growth Factor beta

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APA (6th Edition):

Katre, A. A. (2009). Ozone and lung fibrosis. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,485

Chicago Manual of Style (16th Edition):

Katre, Ashwini A. “Ozone and lung fibrosis.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,485.

MLA Handbook (7th Edition):

Katre, Ashwini A. “Ozone and lung fibrosis.” 2009. Web. 23 Oct 2019.

Vancouver:

Katre AA. Ozone and lung fibrosis. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,485.

Council of Science Editors:

Katre AA. Ozone and lung fibrosis. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,485

26. Wright, Shelton W. (Shelton Wiley). Gastric TH17 response to Helicobacter pylori infection in children.

Degree: MS, 2010, University of Alabama – Birmingham

Background & Aims: Helicobacter pylori infection in children infrequently causes gastroduodenal mucosal ulceration. Since H. pylori induces T cell (Th1/Th17)-dependent gastric inflammation in adults and… (more)

Subjects/Keywords: Child<; br>; Gastritis  – immunology<; br>; Gastritis  – virology<; br>; Helicobacter Infections  – immunology<; br>; Helicobacter pylori  – pathogenicity<; br>; T-Lymphocytes, Regulatory  – pathology<; br>; T-Lymphocytes, Regulatory  – physiology

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APA (6th Edition):

Wright, S. W. (. W. (2010). Gastric TH17 response to Helicobacter pylori infection in children. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,739

Chicago Manual of Style (16th Edition):

Wright, Shelton W (Shelton Wiley). “Gastric TH17 response to Helicobacter pylori infection in children.” 2010. Masters Thesis, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,739.

MLA Handbook (7th Edition):

Wright, Shelton W (Shelton Wiley). “Gastric TH17 response to Helicobacter pylori infection in children.” 2010. Web. 23 Oct 2019.

Vancouver:

Wright SW(W. Gastric TH17 response to Helicobacter pylori infection in children. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,739.

Council of Science Editors:

Wright SW(W. Gastric TH17 response to Helicobacter pylori infection in children. [Masters Thesis]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,739

27. Hensel, Jonathan Adam. The role of LL-37 in prostate cancer and its potential as a therapeutic target.

Degree: 2011, University of Alabama – Birmingham

LL-37 is the only cathelicidin-derived anti-microbial peptide in humans and has been shown to stimulate proliferation, angiogenesis, cellular migration and inhibit apoptosis, in addition to… (more)

Subjects/Keywords: Antimicrobial Cationic Peptides  – metabolism<; br>; Cathelicidins<; br>; Molecular Targeted Therapy  – methods<; br>; Neoplasms, Hormone-Dependent<; br>; Prostatic Neoplasms  – genetics<; br>; Prostatic Neoplasms  – metabolism<; br>; Prostatic Neoplasms  – pathology

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APA (6th Edition):

Hensel, J. A. (2011). The role of LL-37 in prostate cancer and its potential as a therapeutic target. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Thesis, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Web. 23 Oct 2019.

Vancouver:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Internet] [Thesis]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 23 Oct 2019.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

29. Yang, Sherry W. (Sherry Wei). A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer.

Degree: PhD, 2010, University of Alabama – Birmingham

Ovarian cancer remains the most lethal gynecological malignancy in the U.S. Conventional therapies have limited therapeutic value due to advanced stage of the dis-ease at… (more)

Subjects/Keywords: Adenoviridae  – genetics<; br>; Adenoviridae  – physiology<; br>; DNA, Viral  – metabolism<; br>; Oncolytic Virotherapy  – methods<; br>; Ovarian Neoplasms  – pathology<; br>; Tissue Inhibitor of Metalloproteinase-2  – metabolism<; br>; Virus Replication  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, S. W. (. W. (2010). A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1116

Chicago Manual of Style (16th Edition):

Yang, Sherry W (Sherry Wei). “A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1116.

MLA Handbook (7th Edition):

Yang, Sherry W (Sherry Wei). “A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer.” 2010. Web. 23 Oct 2019.

Vancouver:

Yang SW(W. A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1116.

Council of Science Editors:

Yang SW(W. A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1116

30. Gavin, Cristin. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.

Degree: PhD, 2012, University of Alabama – Birmingham

Dynamic changes to the actin cytoskeleton are required for synaptic plasticity and long-term memory formation. However, the molecular mechanisms that mediate filamentous actin (F-actin) dynamics… (more)

Subjects/Keywords: Bone Neoplasms – secondary<; br>; Breast Neoplasms – pathology<; br>; Cell Communication<; br>; Cyclic AMP-Dependent Protein Kinases – metabolism.<; br>; Gap Junctions – metabolism.<; br>; Osteoblasts – cytology.<; br>; Phosphatidylinositol 3-Kinases – metabolism.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gavin, C. (2012). Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1396

Chicago Manual of Style (16th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1396.

MLA Handbook (7th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Web. 23 Oct 2019.

Vancouver:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396.

Council of Science Editors:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396

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