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You searched for subject:( Oncogene). Showing records 1 – 30 of 289 total matches.

[1] [2] [3] [4] [5] [6] [7] [8] [9] [10]

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Washington University in St. Louis

1. Chen, David. Taspase1 is a Non-oncogene Mediator of Tumorigenesis and Maintenance.

Degree: PhD, Biology and Biomedical Sciences: Molecular Cell Biology, 2012, Washington University in St. Louis

 The clinical success of oncogene-targeted therapies substantiates the continued reliance of certain cancers upon the continued function of apical oncogenes involved in its genesis – a… (more)

Subjects/Keywords: Cellular biology; non-oncogene; oncogene; Taspase1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, D. (2012). Taspase1 is a Non-oncogene Mediator of Tumorigenesis and Maintenance. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/etd/563

Chicago Manual of Style (16th Edition):

Chen, David. “Taspase1 is a Non-oncogene Mediator of Tumorigenesis and Maintenance.” 2012. Doctoral Dissertation, Washington University in St. Louis. Accessed August 26, 2019. https://openscholarship.wustl.edu/etd/563.

MLA Handbook (7th Edition):

Chen, David. “Taspase1 is a Non-oncogene Mediator of Tumorigenesis and Maintenance.” 2012. Web. 26 Aug 2019.

Vancouver:

Chen D. Taspase1 is a Non-oncogene Mediator of Tumorigenesis and Maintenance. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2012. [cited 2019 Aug 26]. Available from: https://openscholarship.wustl.edu/etd/563.

Council of Science Editors:

Chen D. Taspase1 is a Non-oncogene Mediator of Tumorigenesis and Maintenance. [Doctoral Dissertation]. Washington University in St. Louis; 2012. Available from: https://openscholarship.wustl.edu/etd/563


University of Rochester

2. Aldersley, Jordan. Shared Role for Critical Mediator Genes in Ras-Dependent and Ras-Independent Malignant Cell Transformation.

Degree: PhD, 2015, University of Rochester

 Diverse types of cancer cells share a common set of critical properties that emerge in response to distinct oncogenic mutations. This suggests a role for… (more)

Subjects/Keywords: Acquired Oncogene Independence; Oncogene Addiction Recurrence

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APA (6th Edition):

Aldersley, J. (2015). Shared Role for Critical Mediator Genes in Ras-Dependent and Ras-Independent Malignant Cell Transformation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/29353

Chicago Manual of Style (16th Edition):

Aldersley, Jordan. “Shared Role for Critical Mediator Genes in Ras-Dependent and Ras-Independent Malignant Cell Transformation.” 2015. Doctoral Dissertation, University of Rochester. Accessed August 26, 2019. http://hdl.handle.net/1802/29353.

MLA Handbook (7th Edition):

Aldersley, Jordan. “Shared Role for Critical Mediator Genes in Ras-Dependent and Ras-Independent Malignant Cell Transformation.” 2015. Web. 26 Aug 2019.

Vancouver:

Aldersley J. Shared Role for Critical Mediator Genes in Ras-Dependent and Ras-Independent Malignant Cell Transformation. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/1802/29353.

Council of Science Editors:

Aldersley J. Shared Role for Critical Mediator Genes in Ras-Dependent and Ras-Independent Malignant Cell Transformation. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/29353


Texas A&M University

3. Suchodolski, Paulette F. Role of the Leucine Zipper of Marek's Disease Virus Oncoprotein Meq in Pathogenesis.

Degree: 2010, Texas A&M University

 Marek's disease virus (MDV), the etiologic agent of Marek's disease, is a potent oncogenic herpesvirus. MDV is highly contagious and elicits a rapid onset of… (more)

Subjects/Keywords: Marek's; Herpesvirus; transformation; oncogene

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APA (6th Edition):

Suchodolski, P. F. (2010). Role of the Leucine Zipper of Marek's Disease Virus Oncoprotein Meq in Pathogenesis. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2009-05-614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suchodolski, Paulette F. “Role of the Leucine Zipper of Marek's Disease Virus Oncoprotein Meq in Pathogenesis.” 2010. Thesis, Texas A&M University. Accessed August 26, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2009-05-614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suchodolski, Paulette F. “Role of the Leucine Zipper of Marek's Disease Virus Oncoprotein Meq in Pathogenesis.” 2010. Web. 26 Aug 2019.

Vancouver:

Suchodolski PF. Role of the Leucine Zipper of Marek's Disease Virus Oncoprotein Meq in Pathogenesis. [Internet] [Thesis]. Texas A&M University; 2010. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2009-05-614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suchodolski PF. Role of the Leucine Zipper of Marek's Disease Virus Oncoprotein Meq in Pathogenesis. [Thesis]. Texas A&M University; 2010. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2009-05-614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

4. Wu, Wen-Ren. Amplification-driven BCL6 overexpression in urothelial carcinoma of urinary bladder.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Urinary bladder urothelial carcinoma is the most common cancer of the urinary tract. About 70% of the diagnosed tumors classified as Non-invasive tumor, which is… (more)

Subjects/Keywords: BCL6; aCGH; oncogene; proliferation; migration

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APA (6th Edition):

Wu, W. (2012). Amplification-driven BCL6 overexpression in urothelial carcinoma of urinary bladder. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810112-224956

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Wen-Ren. “Amplification-driven BCL6 overexpression in urothelial carcinoma of urinary bladder.” 2012. Thesis, NSYSU. Accessed August 26, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810112-224956.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Wen-Ren. “Amplification-driven BCL6 overexpression in urothelial carcinoma of urinary bladder.” 2012. Web. 26 Aug 2019.

Vancouver:

Wu W. Amplification-driven BCL6 overexpression in urothelial carcinoma of urinary bladder. [Internet] [Thesis]. NSYSU; 2012. [cited 2019 Aug 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810112-224956.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu W. Amplification-driven BCL6 overexpression in urothelial carcinoma of urinary bladder. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0810112-224956

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hawaii – Manoa

5. Yoshioka, Masahiro. Downregulation of MYCN oncogene by retinoic acid in neuroblastoma.

Degree: 2015, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2014.

Neuroblastoma is the most common extracranial solid tumor for children. Up to 30% of neuroblastoma tumors show the… (more)

Subjects/Keywords: MYCN oncogene; retinoic acid; neuroblastoma

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APA (6th Edition):

Yoshioka, M. (2015). Downregulation of MYCN oncogene by retinoic acid in neuroblastoma. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/101161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yoshioka, Masahiro. “Downregulation of MYCN oncogene by retinoic acid in neuroblastoma.” 2015. Thesis, University of Hawaii – Manoa. Accessed August 26, 2019. http://hdl.handle.net/10125/101161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yoshioka, Masahiro. “Downregulation of MYCN oncogene by retinoic acid in neuroblastoma.” 2015. Web. 26 Aug 2019.

Vancouver:

Yoshioka M. Downregulation of MYCN oncogene by retinoic acid in neuroblastoma. [Internet] [Thesis]. University of Hawaii – Manoa; 2015. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/10125/101161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yoshioka M. Downregulation of MYCN oncogene by retinoic acid in neuroblastoma. [Thesis]. University of Hawaii – Manoa; 2015. Available from: http://hdl.handle.net/10125/101161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Edelman, Lauren Alexis. Genetic and Molecular Dissection of Rhabdomyosarcoma Tumorigenesis.

Degree: 2011, University of Texas Southwestern Medical Center

 Rhabdomyosarcoma is a tumor of skeletal muscle-type histogenesis and the most common pediatric soft tissue cancer. Rhabdomyosarcoma is often caused by one of two chromosomal… (more)

Subjects/Keywords: Rhabdomyosarcoma; Oncogene Proteins, Fusion; Myoblasts

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APA (6th Edition):

Edelman, L. A. (2011). Genetic and Molecular Dissection of Rhabdomyosarcoma Tumorigenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/932

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Edelman, Lauren Alexis. “Genetic and Molecular Dissection of Rhabdomyosarcoma Tumorigenesis.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed August 26, 2019. http://hdl.handle.net/2152.5/932.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Edelman, Lauren Alexis. “Genetic and Molecular Dissection of Rhabdomyosarcoma Tumorigenesis.” 2011. Web. 26 Aug 2019.

Vancouver:

Edelman LA. Genetic and Molecular Dissection of Rhabdomyosarcoma Tumorigenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/2152.5/932.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Edelman LA. Genetic and Molecular Dissection of Rhabdomyosarcoma Tumorigenesis. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/932

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

7. Alotaibi, Faizah. An immune modulatory role for the fes proto-oncogene .

Degree: Pathology and Molecular Medicine, 2015, Queens University

 The Fes protein tyrosine kinase is abundantly expressed in phagocytic immune cells, including tumor associated macrophages. Fes knockout mice (fes-/-) display enhanced sensitivity to LPS,… (more)

Subjects/Keywords: fes oncogene; immune system

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APA (6th Edition):

Alotaibi, F. (2015). An immune modulatory role for the fes proto-oncogene . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alotaibi, Faizah. “An immune modulatory role for the fes proto-oncogene .” 2015. Thesis, Queens University. Accessed August 26, 2019. http://hdl.handle.net/1974/13420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alotaibi, Faizah. “An immune modulatory role for the fes proto-oncogene .” 2015. Web. 26 Aug 2019.

Vancouver:

Alotaibi F. An immune modulatory role for the fes proto-oncogene . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/1974/13420.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alotaibi F. An immune modulatory role for the fes proto-oncogene . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13420

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Collins, N. BRCA2 in familial and sporadic breast cancer.

Degree: PhD, 2000, Institute of Cancer Research (University Of London)

 The breast cancer susceptibility gene BRCA2 is located on chromosome 13q 12-13. Using breast and ovarian cancers from a BRCA2-linked family, loss of the wild… (more)

Subjects/Keywords: 572.8; Oncogene; Chromosomes

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APA (6th Edition):

Collins, N. (2000). BRCA2 in familial and sporadic breast cancer. (Doctoral Dissertation). Institute of Cancer Research (University Of London). Retrieved from http://publications.icr.ac.uk/10070/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327100

Chicago Manual of Style (16th Edition):

Collins, N. “BRCA2 in familial and sporadic breast cancer.” 2000. Doctoral Dissertation, Institute of Cancer Research (University Of London). Accessed August 26, 2019. http://publications.icr.ac.uk/10070/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327100.

MLA Handbook (7th Edition):

Collins, N. “BRCA2 in familial and sporadic breast cancer.” 2000. Web. 26 Aug 2019.

Vancouver:

Collins N. BRCA2 in familial and sporadic breast cancer. [Internet] [Doctoral dissertation]. Institute of Cancer Research (University Of London); 2000. [cited 2019 Aug 26]. Available from: http://publications.icr.ac.uk/10070/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327100.

Council of Science Editors:

Collins N. BRCA2 in familial and sporadic breast cancer. [Doctoral Dissertation]. Institute of Cancer Research (University Of London); 2000. Available from: http://publications.icr.ac.uk/10070/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327100


Université de Montréal

9. Armaos, Gregory. Proscillaridin A effects on histone acetylation and C-MYC degradation in acute lymphoblastic leukemia .

Degree: 2016, Université de Montréal

 La leucémie lymphoblastique aiguë (LLA) représente environ 25% des cancers pédiatriques diagnostiqués chaque année. Dans 80 % des cas, une rémission complète est observée. Cependant,… (more)

Subjects/Keywords: Acétylation; Histones; Oncogéne; Leucémie; Acetylation; Oncogene; Leukemia

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APA (6th Edition):

Armaos, G. (2016). Proscillaridin A effects on histone acetylation and C-MYC degradation in acute lymphoblastic leukemia . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/16276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Armaos, Gregory. “Proscillaridin A effects on histone acetylation and C-MYC degradation in acute lymphoblastic leukemia .” 2016. Thesis, Université de Montréal. Accessed August 26, 2019. http://hdl.handle.net/1866/16276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Armaos, Gregory. “Proscillaridin A effects on histone acetylation and C-MYC degradation in acute lymphoblastic leukemia .” 2016. Web. 26 Aug 2019.

Vancouver:

Armaos G. Proscillaridin A effects on histone acetylation and C-MYC degradation in acute lymphoblastic leukemia . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/1866/16276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Armaos G. Proscillaridin A effects on histone acetylation and C-MYC degradation in acute lymphoblastic leukemia . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/16276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

10. Jiang, Xiaolei. Genomic Analysis of Cancer Heterogeneity and Oncogenic Mechanisms .

Degree: 2014, Duke University

  The development of cancer is a process by which an accumulation of genetic changes leads to uncontrolled replication of cells. Since the process of… (more)

Subjects/Keywords: Genetics; Molecular biology; Cancer; Mechanism; Oncogene; Therapeutics

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APA (6th Edition):

Jiang, X. (2014). Genomic Analysis of Cancer Heterogeneity and Oncogenic Mechanisms . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jiang, Xiaolei. “Genomic Analysis of Cancer Heterogeneity and Oncogenic Mechanisms .” 2014. Thesis, Duke University. Accessed August 26, 2019. http://hdl.handle.net/10161/9421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jiang, Xiaolei. “Genomic Analysis of Cancer Heterogeneity and Oncogenic Mechanisms .” 2014. Web. 26 Aug 2019.

Vancouver:

Jiang X. Genomic Analysis of Cancer Heterogeneity and Oncogenic Mechanisms . [Internet] [Thesis]. Duke University; 2014. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/10161/9421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jiang X. Genomic Analysis of Cancer Heterogeneity and Oncogenic Mechanisms . [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/9421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

11. Clark, Christopher. Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2018, University of Minnesota

 The studies performed in this dissertation focused on the characterization of two candidate cancer genes, TM9SF2 and WAC, and their role in colorectal cancer (CRC).… (more)

Subjects/Keywords: Cancer; Driver gene; Genetics; Oncogene; Tumor suppressor

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APA (6th Edition):

Clark, C. (2018). Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/201708

Chicago Manual of Style (16th Edition):

Clark, Christopher. “Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes.” 2018. Doctoral Dissertation, University of Minnesota. Accessed August 26, 2019. http://hdl.handle.net/11299/201708.

MLA Handbook (7th Edition):

Clark, Christopher. “Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes.” 2018. Web. 26 Aug 2019.

Vancouver:

Clark C. Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes. [Internet] [Doctoral dissertation]. University of Minnesota; 2018. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/11299/201708.

Council of Science Editors:

Clark C. Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes. [Doctoral Dissertation]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/201708


Uppsala University

12. Jonasson, Jennifer. Analysis of PIK3CA mutations in tumours from patients with non-small cell lung cancer using pyrosequencing.

Degree: Women's and Children's Health, 2014, Uppsala University

  A subgroup of non-small cell lung cancer (NSCLC) cases harbour mutations in classical oncogenes, which can affect therapy response and prognosis. By therapeutically targeting… (more)

Subjects/Keywords: PI3K; tumour marker; pseudogene; oncogene; targeted therapy

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APA (6th Edition):

Jonasson, J. (2014). Analysis of PIK3CA mutations in tumours from patients with non-small cell lung cancer using pyrosequencing. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-227762

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jonasson, Jennifer. “Analysis of PIK3CA mutations in tumours from patients with non-small cell lung cancer using pyrosequencing.” 2014. Thesis, Uppsala University. Accessed August 26, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-227762.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jonasson, Jennifer. “Analysis of PIK3CA mutations in tumours from patients with non-small cell lung cancer using pyrosequencing.” 2014. Web. 26 Aug 2019.

Vancouver:

Jonasson J. Analysis of PIK3CA mutations in tumours from patients with non-small cell lung cancer using pyrosequencing. [Internet] [Thesis]. Uppsala University; 2014. [cited 2019 Aug 26]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-227762.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jonasson J. Analysis of PIK3CA mutations in tumours from patients with non-small cell lung cancer using pyrosequencing. [Thesis]. Uppsala University; 2014. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-227762

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

13. Lee, Ya-Hua. Studies on the roles and mechanisms of the STMN3 gene in human bladder cancer-derived cell lines.

Degree: Master, Institute of Biomedical Sciences, 2018, NSYSU

 Our study explored the function and regulatory mechanisms of STMN3 which is the microtubule-destabilizing phosphoprotein in the bladder cancer. Our results showed that stable transfection… (more)

Subjects/Keywords: microtubule; stathmin 3(STMN3); Bladder cancer; oncogene

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APA (6th Edition):

Lee, Y. (2018). Studies on the roles and mechanisms of the STMN3 gene in human bladder cancer-derived cell lines. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612118-141151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Ya-Hua. “Studies on the roles and mechanisms of the STMN3 gene in human bladder cancer-derived cell lines.” 2018. Thesis, NSYSU. Accessed August 26, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612118-141151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Ya-Hua. “Studies on the roles and mechanisms of the STMN3 gene in human bladder cancer-derived cell lines.” 2018. Web. 26 Aug 2019.

Vancouver:

Lee Y. Studies on the roles and mechanisms of the STMN3 gene in human bladder cancer-derived cell lines. [Internet] [Thesis]. NSYSU; 2018. [cited 2019 Aug 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612118-141151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee Y. Studies on the roles and mechanisms of the STMN3 gene in human bladder cancer-derived cell lines. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0612118-141151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. Wu, Nan. Cohesin, the SMC5/6 Complex and SUMO in DNA Repair.

Degree: 2013, University of Texas Southwestern Medical Center

 DNA double-strand breaks (DSBs) fuel cancer-driving chromosome translocations. Two related structural maintenance of chromosomes (Smc) complexes, cohesin and Smc5/6, promote DSB repair through sister-chromatid homologous… (more)

Subjects/Keywords: Chromatids; Proto-Oncogene Proteins; Recombination, Genetic

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APA (6th Edition):

Wu, N. (2013). Cohesin, the SMC5/6 Complex and SUMO in DNA Repair. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Nan. “Cohesin, the SMC5/6 Complex and SUMO in DNA Repair.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed August 26, 2019. http://hdl.handle.net/2152.5/1259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Nan. “Cohesin, the SMC5/6 Complex and SUMO in DNA Repair.” 2013. Web. 26 Aug 2019.

Vancouver:

Wu N. Cohesin, the SMC5/6 Complex and SUMO in DNA Repair. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/2152.5/1259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu N. Cohesin, the SMC5/6 Complex and SUMO in DNA Repair. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

15. Razumovskaya, Elena. Studies on signaling pathways induced by FLT3, an important oncogene in AML.

Degree: 2011, University of Lund

 FLT3, a receptor tyrosine kinase, is expressed in hematopoietic progenitor cells. FLT3-ITD (internal tandem duplication) and D835 mutations are found in approximately 30% and 7%… (more)

Subjects/Keywords: Läkemedelskemi; FLT3; AML; RTK; oncogene; ERK5; hematopoiesis

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APA (6th Edition):

Razumovskaya, E. (2011). Studies on signaling pathways induced by FLT3, an important oncogene in AML. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/2255711 ; http://portal.research.lu.se/ws/files/3306098/2255723.pdf

Chicago Manual of Style (16th Edition):

Razumovskaya, Elena. “Studies on signaling pathways induced by FLT3, an important oncogene in AML.” 2011. Doctoral Dissertation, University of Lund. Accessed August 26, 2019. http://lup.lub.lu.se/record/2255711 ; http://portal.research.lu.se/ws/files/3306098/2255723.pdf.

MLA Handbook (7th Edition):

Razumovskaya, Elena. “Studies on signaling pathways induced by FLT3, an important oncogene in AML.” 2011. Web. 26 Aug 2019.

Vancouver:

Razumovskaya E. Studies on signaling pathways induced by FLT3, an important oncogene in AML. [Internet] [Doctoral dissertation]. University of Lund; 2011. [cited 2019 Aug 26]. Available from: http://lup.lub.lu.se/record/2255711 ; http://portal.research.lu.se/ws/files/3306098/2255723.pdf.

Council of Science Editors:

Razumovskaya E. Studies on signaling pathways induced by FLT3, an important oncogene in AML. [Doctoral Dissertation]. University of Lund; 2011. Available from: http://lup.lub.lu.se/record/2255711 ; http://portal.research.lu.se/ws/files/3306098/2255723.pdf


University of Sydney

16. Kyung Chan, Park. Pleiotropic roles, post-translational regulation and pharmacological targeting of NDRG1 in cancer .

Degree: 2019, University of Sydney

 The metastasis suppressor, NDRG1, is a promising therapeutic target for cancer treatment. In past decade, much of the research has elucidated the anti-oncogenic molecular functions… (more)

Subjects/Keywords: cancer; metastasis; oncogene; anti-cancer drug

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APA (6th Edition):

Kyung Chan, P. (2019). Pleiotropic roles, post-translational regulation and pharmacological targeting of NDRG1 in cancer . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kyung Chan, Park. “Pleiotropic roles, post-translational regulation and pharmacological targeting of NDRG1 in cancer .” 2019. Thesis, University of Sydney. Accessed August 26, 2019. http://hdl.handle.net/2123/20146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kyung Chan, Park. “Pleiotropic roles, post-translational regulation and pharmacological targeting of NDRG1 in cancer .” 2019. Web. 26 Aug 2019.

Vancouver:

Kyung Chan P. Pleiotropic roles, post-translational regulation and pharmacological targeting of NDRG1 in cancer . [Internet] [Thesis]. University of Sydney; 2019. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/2123/20146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kyung Chan P. Pleiotropic roles, post-translational regulation and pharmacological targeting of NDRG1 in cancer . [Thesis]. University of Sydney; 2019. Available from: http://hdl.handle.net/2123/20146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Silva, Flavio Sousa. Clonagem, expressão, purificação e caracterização estrutural da região AP-1 da oncoproteína Jun.

Degree: Mestrado, Tecnologia Nuclear - Aplicações, 2014, University of São Paulo

A proteína jun é um dos principais integrantes do complexo AP-1 e está envolvido nos processos inflamatórios, diferenciação, apoptose e migração celular. Esta proteína pode… (more)

Subjects/Keywords: AP-1; AP-1; cancer; câncer; Jun; Jun; oncogene; oncogene; oncoprotein; oncoproteína

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APA (6th Edition):

Silva, F. S. (2014). Clonagem, expressão, purificação e caracterização estrutural da região AP-1 da oncoproteína Jun. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/85/85131/tde-18092014-091537/ ;

Chicago Manual of Style (16th Edition):

Silva, Flavio Sousa. “Clonagem, expressão, purificação e caracterização estrutural da região AP-1 da oncoproteína Jun.” 2014. Masters Thesis, University of São Paulo. Accessed August 26, 2019. http://www.teses.usp.br/teses/disponiveis/85/85131/tde-18092014-091537/ ;.

MLA Handbook (7th Edition):

Silva, Flavio Sousa. “Clonagem, expressão, purificação e caracterização estrutural da região AP-1 da oncoproteína Jun.” 2014. Web. 26 Aug 2019.

Vancouver:

Silva FS. Clonagem, expressão, purificação e caracterização estrutural da região AP-1 da oncoproteína Jun. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2019 Aug 26]. Available from: http://www.teses.usp.br/teses/disponiveis/85/85131/tde-18092014-091537/ ;.

Council of Science Editors:

Silva FS. Clonagem, expressão, purificação e caracterização estrutural da região AP-1 da oncoproteína Jun. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/85/85131/tde-18092014-091537/ ;

18. A. Cerutti. ONCOGENE-INDUCED ALTERED DNA REPLICATION DYNAMICS.

Degree: 2014, Università degli Studi di Milano

Oncogene Induced Senescence (OIS) is a tumor suppressive barrier that blocks cell cycle permanently. OIS results from a robust DNA damage response (DDR) activation due… (more)

Subjects/Keywords: oncogene activation; DNA replication stress; DNA damage response; oncogene-induced senescence; DNA molecular combing; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Cerutti, A. (2014). ONCOGENE-INDUCED ALTERED DNA REPLICATION DYNAMICS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/234135

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cerutti, A.. “ONCOGENE-INDUCED ALTERED DNA REPLICATION DYNAMICS.” 2014. Thesis, Università degli Studi di Milano. Accessed August 26, 2019. http://hdl.handle.net/2434/234135.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cerutti, A.. “ONCOGENE-INDUCED ALTERED DNA REPLICATION DYNAMICS.” 2014. Web. 26 Aug 2019.

Vancouver:

Cerutti A. ONCOGENE-INDUCED ALTERED DNA REPLICATION DYNAMICS. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/2434/234135.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cerutti A. ONCOGENE-INDUCED ALTERED DNA REPLICATION DYNAMICS. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/234135

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

19. Williams, Alistair Robert William. Expression of oncogenes in human colorectal neoplasms.

Degree: Thesis (M.D.), 1988, University of Edinburgh

Subjects/Keywords: 572.8; Oncogene expression/cell level

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APA (6th Edition):

Williams, A. R. W. (1988). Expression of oncogenes in human colorectal neoplasms. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/19415

Chicago Manual of Style (16th Edition):

Williams, Alistair Robert William. “Expression of oncogenes in human colorectal neoplasms.” 1988. Doctoral Dissertation, University of Edinburgh. Accessed August 26, 2019. http://hdl.handle.net/1842/19415.

MLA Handbook (7th Edition):

Williams, Alistair Robert William. “Expression of oncogenes in human colorectal neoplasms.” 1988. Web. 26 Aug 2019.

Vancouver:

Williams ARW. Expression of oncogenes in human colorectal neoplasms. [Internet] [Doctoral dissertation]. University of Edinburgh; 1988. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/1842/19415.

Council of Science Editors:

Williams ARW. Expression of oncogenes in human colorectal neoplasms. [Doctoral Dissertation]. University of Edinburgh; 1988. Available from: http://hdl.handle.net/1842/19415


Seton Hall University

20. DeFelice, Chelsea Rose. Correlation of Environmental Exposure to Polycyclic Aromatic Hydrocarbons and Polymorphisms in the Proto-oncogene v-Rel using Wild Atlantic Menhaden.

Degree: MS Biology, Biology, 2018, Seton Hall University

  Hurricane Sandy critically damaged the Atlantic coast of New Jersey in the fall of 2012. This was recorded as the largest storm to hit… (more)

Subjects/Keywords: PAHs; fluorescence; menhaden; proto-oncogene; v-rel; polymorphisms; Biology; Marine Biology

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APA (6th Edition):

DeFelice, C. R. (2018). Correlation of Environmental Exposure to Polycyclic Aromatic Hydrocarbons and Polymorphisms in the Proto-oncogene v-Rel using Wild Atlantic Menhaden. (Doctoral Dissertation). Seton Hall University. Retrieved from http://scholarship.shu.edu/dissertations/2512

Chicago Manual of Style (16th Edition):

DeFelice, Chelsea Rose. “Correlation of Environmental Exposure to Polycyclic Aromatic Hydrocarbons and Polymorphisms in the Proto-oncogene v-Rel using Wild Atlantic Menhaden.” 2018. Doctoral Dissertation, Seton Hall University. Accessed August 26, 2019. http://scholarship.shu.edu/dissertations/2512.

MLA Handbook (7th Edition):

DeFelice, Chelsea Rose. “Correlation of Environmental Exposure to Polycyclic Aromatic Hydrocarbons and Polymorphisms in the Proto-oncogene v-Rel using Wild Atlantic Menhaden.” 2018. Web. 26 Aug 2019.

Vancouver:

DeFelice CR. Correlation of Environmental Exposure to Polycyclic Aromatic Hydrocarbons and Polymorphisms in the Proto-oncogene v-Rel using Wild Atlantic Menhaden. [Internet] [Doctoral dissertation]. Seton Hall University; 2018. [cited 2019 Aug 26]. Available from: http://scholarship.shu.edu/dissertations/2512.

Council of Science Editors:

DeFelice CR. Correlation of Environmental Exposure to Polycyclic Aromatic Hydrocarbons and Polymorphisms in the Proto-oncogene v-Rel using Wild Atlantic Menhaden. [Doctoral Dissertation]. Seton Hall University; 2018. Available from: http://scholarship.shu.edu/dissertations/2512

21. 井山, 慶大. Identification of Three Novel Fusion Oncogenes, SQSTM1/NTRK3, AFAP1L2/RET, and PPFIBP2/RET, in Thyroid Cancers of Young Patients in Fukushima : 福島の若年甲状腺乳頭癌における3つの新規融合癌遺伝子(SQSTM1/NTRK3, AFAP1L2/RET, PPFIBP2/RET)の同定.

Degree: 博士(医学), 2017, Nagasaki University / 長崎大学

 Background: The BRAFV600E mutation is the most frequent genetic abnormality in adult papillary thyroid carcinomas (PTCs). On the other hand, various chromosomal rearrangements are more… (more)

Subjects/Keywords: fusion gene; rearrangement; oncogene; NTRK3; RET; papillary thyroid carcinoma

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APA (6th Edition):

井山, . (2017). Identification of Three Novel Fusion Oncogenes, SQSTM1/NTRK3, AFAP1L2/RET, and PPFIBP2/RET, in Thyroid Cancers of Young Patients in Fukushima : 福島の若年甲状腺乳頭癌における3つの新規融合癌遺伝子(SQSTM1/NTRK3, AFAP1L2/RET, PPFIBP2/RET)の同定. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/38111

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

井山, 慶大. “Identification of Three Novel Fusion Oncogenes, SQSTM1/NTRK3, AFAP1L2/RET, and PPFIBP2/RET, in Thyroid Cancers of Young Patients in Fukushima : 福島の若年甲状腺乳頭癌における3つの新規融合癌遺伝子(SQSTM1/NTRK3, AFAP1L2/RET, PPFIBP2/RET)の同定.” 2017. Thesis, Nagasaki University / 長崎大学. Accessed August 26, 2019. http://hdl.handle.net/10069/38111.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

井山, 慶大. “Identification of Three Novel Fusion Oncogenes, SQSTM1/NTRK3, AFAP1L2/RET, and PPFIBP2/RET, in Thyroid Cancers of Young Patients in Fukushima : 福島の若年甲状腺乳頭癌における3つの新規融合癌遺伝子(SQSTM1/NTRK3, AFAP1L2/RET, PPFIBP2/RET)の同定.” 2017. Web. 26 Aug 2019.

Vancouver:

井山 . Identification of Three Novel Fusion Oncogenes, SQSTM1/NTRK3, AFAP1L2/RET, and PPFIBP2/RET, in Thyroid Cancers of Young Patients in Fukushima : 福島の若年甲状腺乳頭癌における3つの新規融合癌遺伝子(SQSTM1/NTRK3, AFAP1L2/RET, PPFIBP2/RET)の同定. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2017. [cited 2019 Aug 26]. Available from: http://hdl.handle.net/10069/38111.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

井山 . Identification of Three Novel Fusion Oncogenes, SQSTM1/NTRK3, AFAP1L2/RET, and PPFIBP2/RET, in Thyroid Cancers of Young Patients in Fukushima : 福島の若年甲状腺乳頭癌における3つの新規融合癌遺伝子(SQSTM1/NTRK3, AFAP1L2/RET, PPFIBP2/RET)の同定. [Thesis]. Nagasaki University / 長崎大学; 2017. Available from: http://hdl.handle.net/10069/38111

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Colorado

22. Hu, Tianjing. Control of Cell Invasion in Melanoma by a New Gene, FAM129B.

Degree: PhD, Chemistry & Biochemistry, 2015, University of Colorado

  Approximately 70% human malignant melanomas contain oncogenic mutations in B-Raf, which constitutively activate this kinase, and result in constitutive activation of the B-RAF/MKK/ERK pathway.… (more)

Subjects/Keywords: skin cancer; oncogene; molecular determinants; Biochemistry; Cell Biology

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APA (6th Edition):

Hu, T. (2015). Control of Cell Invasion in Melanoma by a New Gene, FAM129B. (Doctoral Dissertation). University of Colorado. Retrieved from http://scholar.colorado.edu/chem_gradetds/172

Chicago Manual of Style (16th Edition):

Hu, Tianjing. “Control of Cell Invasion in Melanoma by a New Gene, FAM129B.” 2015. Doctoral Dissertation, University of Colorado. Accessed August 26, 2019. http://scholar.colorado.edu/chem_gradetds/172.

MLA Handbook (7th Edition):

Hu, Tianjing. “Control of Cell Invasion in Melanoma by a New Gene, FAM129B.” 2015. Web. 26 Aug 2019.

Vancouver:

Hu T. Control of Cell Invasion in Melanoma by a New Gene, FAM129B. [Internet] [Doctoral dissertation]. University of Colorado; 2015. [cited 2019 Aug 26]. Available from: http://scholar.colorado.edu/chem_gradetds/172.

Council of Science Editors:

Hu T. Control of Cell Invasion in Melanoma by a New Gene, FAM129B. [Doctoral Dissertation]. University of Colorado; 2015. Available from: http://scholar.colorado.edu/chem_gradetds/172


Cranfield University

23. Stebbeds, William Joshua David. The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression.

Degree: MSc by Research, 2011, Cranfield University

 G-quadruplexes (G4-DNA) are a class of secondary structures formed from Guanine rich sequences. In recent years these structures have been implicated in both telomere maintenance… (more)

Subjects/Keywords: G-quadruplexes; G-quadruplexes; oncogene expression; Carcinogenesis; C-Myc; c-Kit

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APA (6th Edition):

Stebbeds, W. J. D. (2011). The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression. (Masters Thesis). Cranfield University. Retrieved from http://dspace.lib.cranfield.ac.uk/handle/1826/7179

Chicago Manual of Style (16th Edition):

Stebbeds, William Joshua David. “The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression.” 2011. Masters Thesis, Cranfield University. Accessed August 26, 2019. http://dspace.lib.cranfield.ac.uk/handle/1826/7179.

MLA Handbook (7th Edition):

Stebbeds, William Joshua David. “The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression.” 2011. Web. 26 Aug 2019.

Vancouver:

Stebbeds WJD. The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression. [Internet] [Masters thesis]. Cranfield University; 2011. [cited 2019 Aug 26]. Available from: http://dspace.lib.cranfield.ac.uk/handle/1826/7179.

Council of Science Editors:

Stebbeds WJD. The effect of oxidation on the stability of G-quadruplex DNA : implications for oncogene expression. [Masters Thesis]. Cranfield University; 2011. Available from: http://dspace.lib.cranfield.ac.uk/handle/1826/7179


NSYSU

24. Huang, Yen-Lin. The Role of Chibby as a Potential Tumor Suppressor Gene in Human Cervical Cancer.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 The Wnt signaling pathway is highly conserved and participates in many important cellular functions including differentiation, embryonic development and tissue generations. β-catenin, the central mediator… (more)

Subjects/Keywords: Wnt; oncogene; β-catenin; adenovirus; SiHa; HeLa; cervical cancer; chibby

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APA (6th Edition):

Huang, Y. (2010). The Role of Chibby as a Potential Tumor Suppressor Gene in Human Cervical Cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902110-170133

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yen-Lin. “The Role of Chibby as a Potential Tumor Suppressor Gene in Human Cervical Cancer.” 2010. Thesis, NSYSU. Accessed August 26, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902110-170133.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yen-Lin. “The Role of Chibby as a Potential Tumor Suppressor Gene in Human Cervical Cancer.” 2010. Web. 26 Aug 2019.

Vancouver:

Huang Y. The Role of Chibby as a Potential Tumor Suppressor Gene in Human Cervical Cancer. [Internet] [Thesis]. NSYSU; 2010. [cited 2019 Aug 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902110-170133.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang Y. The Role of Chibby as a Potential Tumor Suppressor Gene in Human Cervical Cancer. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0902110-170133

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Liu, Shujing. Activation of osteoblast-like phenotype in microcell hybrids and characterization of MDM2 functions in a rhabdomyosarcoma.

Degree: MS, 1997, Oregon Health Sciences University

Subjects/Keywords: Rhabdomyosarcoma; Oncogene Proteins; Phenotype; Osteoblasts

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APA (6th Edition):

Liu, S. (1997). Activation of osteoblast-like phenotype in microcell hybrids and characterization of MDM2 functions in a rhabdomyosarcoma. (Thesis). Oregon Health Sciences University. Retrieved from doi:10.6083/M4Q81B9N ; http://digitalcommons.ohsu.edu/etd/2626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Shujing. “Activation of osteoblast-like phenotype in microcell hybrids and characterization of MDM2 functions in a rhabdomyosarcoma.” 1997. Thesis, Oregon Health Sciences University. Accessed August 26, 2019. doi:10.6083/M4Q81B9N ; http://digitalcommons.ohsu.edu/etd/2626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Shujing. “Activation of osteoblast-like phenotype in microcell hybrids and characterization of MDM2 functions in a rhabdomyosarcoma.” 1997. Web. 26 Aug 2019.

Vancouver:

Liu S. Activation of osteoblast-like phenotype in microcell hybrids and characterization of MDM2 functions in a rhabdomyosarcoma. [Internet] [Thesis]. Oregon Health Sciences University; 1997. [cited 2019 Aug 26]. Available from: doi:10.6083/M4Q81B9N ; http://digitalcommons.ohsu.edu/etd/2626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu S. Activation of osteoblast-like phenotype in microcell hybrids and characterization of MDM2 functions in a rhabdomyosarcoma. [Thesis]. Oregon Health Sciences University; 1997. Available from: doi:10.6083/M4Q81B9N ; http://digitalcommons.ohsu.edu/etd/2626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

26. 鄧惠雯; Tang, Wai-man. The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer.

Degree: M. Phil., 2015, University of Hong Kong

Ovarian cancer is one of the leading causes of cancer-related death in females. Cancer metastasis is the major reason for the resultant mortality of patients… (more)

Subjects/Keywords: Ovaries - Cancer - Genetic aspects; Omentum; Oncogene; Interleukin-1

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APA (6th Edition):

鄧惠雯; Tang, W. (2015). The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. (Masters Thesis). University of Hong Kong. Retrieved from Tang, W. [鄧惠雯]. (2015). The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719478 ; http://hdl.handle.net/10722/223567

Chicago Manual of Style (16th Edition):

鄧惠雯; Tang, Wai-man. “The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer.” 2015. Masters Thesis, University of Hong Kong. Accessed August 26, 2019. Tang, W. [鄧惠雯]. (2015). The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719478 ; http://hdl.handle.net/10722/223567.

MLA Handbook (7th Edition):

鄧惠雯; Tang, Wai-man. “The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer.” 2015. Web. 26 Aug 2019.

Vancouver:

鄧惠雯; Tang W. The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Aug 26]. Available from: Tang, W. [鄧惠雯]. (2015). The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719478 ; http://hdl.handle.net/10722/223567.

Council of Science Editors:

鄧惠雯; Tang W. The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. [Masters Thesis]. University of Hong Kong; 2015. Available from: Tang, W. [鄧惠雯]. (2015). The significance of GRO-α and IL-8 in promoting omental metastasis of ovarian cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5719478 ; http://hdl.handle.net/10722/223567


Kent State University

27. Tschantz Chaney, Deidra Renee. Mechanisms of Interfering with HPV-Induced Carcinogenesis by Natural Products.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2009, Kent State University

 Human papillomavirus (HPV)-induced uterine cervical cancer is one of the most prevalent cancers in women world-wide. HPV is a small DNA tumor virus that can… (more)

Subjects/Keywords: Molecular Biology; human papillomavirus; natural lignans; E6 oncogene; p53

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APA (6th Edition):

Tschantz Chaney, D. R. (2009). Mechanisms of Interfering with HPV-Induced Carcinogenesis by Natural Products. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1259084071

Chicago Manual of Style (16th Edition):

Tschantz Chaney, Deidra Renee. “Mechanisms of Interfering with HPV-Induced Carcinogenesis by Natural Products.” 2009. Doctoral Dissertation, Kent State University. Accessed August 26, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1259084071.

MLA Handbook (7th Edition):

Tschantz Chaney, Deidra Renee. “Mechanisms of Interfering with HPV-Induced Carcinogenesis by Natural Products.” 2009. Web. 26 Aug 2019.

Vancouver:

Tschantz Chaney DR. Mechanisms of Interfering with HPV-Induced Carcinogenesis by Natural Products. [Internet] [Doctoral dissertation]. Kent State University; 2009. [cited 2019 Aug 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1259084071.

Council of Science Editors:

Tschantz Chaney DR. Mechanisms of Interfering with HPV-Induced Carcinogenesis by Natural Products. [Doctoral Dissertation]. Kent State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1259084071


Kent State University

28. Allen, Kristi Lynne. Therapeutic Reactivation of the p53 Tumor Suppressor Protein in HPV-Positive Cervical Cancer Cells by the Creosote Bush Lignan 3’-O-Methyl-Nordihydroguaiaretic Acid.

Degree: PhD, College of Biomedical Sciences, 2007, Kent State University

 Persistent Human Papillomavirus (HPV) infection is the primary risk factor for the development of cervical carcinoma, a major disease burden in women worldwide. Over 90… (more)

Subjects/Keywords: Biology, Molecular; Human Papillomavirus; E6 oncogene; lignan; p53; apoptosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Allen, K. L. (2007). Therapeutic Reactivation of the p53 Tumor Suppressor Protein in HPV-Positive Cervical Cancer Cells by the Creosote Bush Lignan 3’-O-Methyl-Nordihydroguaiaretic Acid. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1176432164

Chicago Manual of Style (16th Edition):

Allen, Kristi Lynne. “Therapeutic Reactivation of the p53 Tumor Suppressor Protein in HPV-Positive Cervical Cancer Cells by the Creosote Bush Lignan 3’-O-Methyl-Nordihydroguaiaretic Acid.” 2007. Doctoral Dissertation, Kent State University. Accessed August 26, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1176432164.

MLA Handbook (7th Edition):

Allen, Kristi Lynne. “Therapeutic Reactivation of the p53 Tumor Suppressor Protein in HPV-Positive Cervical Cancer Cells by the Creosote Bush Lignan 3’-O-Methyl-Nordihydroguaiaretic Acid.” 2007. Web. 26 Aug 2019.

Vancouver:

Allen KL. Therapeutic Reactivation of the p53 Tumor Suppressor Protein in HPV-Positive Cervical Cancer Cells by the Creosote Bush Lignan 3’-O-Methyl-Nordihydroguaiaretic Acid. [Internet] [Doctoral dissertation]. Kent State University; 2007. [cited 2019 Aug 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1176432164.

Council of Science Editors:

Allen KL. Therapeutic Reactivation of the p53 Tumor Suppressor Protein in HPV-Positive Cervical Cancer Cells by the Creosote Bush Lignan 3’-O-Methyl-Nordihydroguaiaretic Acid. [Doctoral Dissertation]. Kent State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1176432164


Kent State University

29. Awad, Keytam Salem. INHIBITON OF HUMAN PAPILLOMA VIRUS E6 ONCOGENE FUNCTION BY MAMMALIAN LIGNANS ACTIVATES THE P53 TUMOR SUPPRESSOR PROTEIN AND INDUCES APOPTOSIS IN CERVICAL CANCER CELLS.

Degree: PhD, College of Biomedical Sciences, 2007, Kent State University

 Invasive uterine cervical cancer represents a major disease burden in women worldwide. Persistent Human Papillomavirus (HPV) infection is the primary risk factor for the development… (more)

Subjects/Keywords: Human papilloma virus; mammalian lignans; p53; E6 oncogene

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Awad, K. S. (2007). INHIBITON OF HUMAN PAPILLOMA VIRUS E6 ONCOGENE FUNCTION BY MAMMALIAN LIGNANS ACTIVATES THE P53 TUMOR SUPPRESSOR PROTEIN AND INDUCES APOPTOSIS IN CERVICAL CANCER CELLS. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1183405761

Chicago Manual of Style (16th Edition):

Awad, Keytam Salem. “INHIBITON OF HUMAN PAPILLOMA VIRUS E6 ONCOGENE FUNCTION BY MAMMALIAN LIGNANS ACTIVATES THE P53 TUMOR SUPPRESSOR PROTEIN AND INDUCES APOPTOSIS IN CERVICAL CANCER CELLS.” 2007. Doctoral Dissertation, Kent State University. Accessed August 26, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1183405761.

MLA Handbook (7th Edition):

Awad, Keytam Salem. “INHIBITON OF HUMAN PAPILLOMA VIRUS E6 ONCOGENE FUNCTION BY MAMMALIAN LIGNANS ACTIVATES THE P53 TUMOR SUPPRESSOR PROTEIN AND INDUCES APOPTOSIS IN CERVICAL CANCER CELLS.” 2007. Web. 26 Aug 2019.

Vancouver:

Awad KS. INHIBITON OF HUMAN PAPILLOMA VIRUS E6 ONCOGENE FUNCTION BY MAMMALIAN LIGNANS ACTIVATES THE P53 TUMOR SUPPRESSOR PROTEIN AND INDUCES APOPTOSIS IN CERVICAL CANCER CELLS. [Internet] [Doctoral dissertation]. Kent State University; 2007. [cited 2019 Aug 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1183405761.

Council of Science Editors:

Awad KS. INHIBITON OF HUMAN PAPILLOMA VIRUS E6 ONCOGENE FUNCTION BY MAMMALIAN LIGNANS ACTIVATES THE P53 TUMOR SUPPRESSOR PROTEIN AND INDUCES APOPTOSIS IN CERVICAL CANCER CELLS. [Doctoral Dissertation]. Kent State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1183405761

30. Ghisays, Valentina. Exploring the Role of the DEK Oncogene as a Novel Player in Learning and Memory.

Degree: PhD, Arts and Sciences: Psychology, 2017, University of Cincinnati

 Alzheimer’s disease (AD) is an aberrant aging process characterized by progressive decline in cognition and dramatic behavioral changes. AD has no treatment to slow or… (more)

Subjects/Keywords: Neurology; Oncogene; Alzheimers Disease; Learning and Memory; Cognition; Sex differences; Hippocampus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ghisays, V. (2017). Exploring the Role of the DEK Oncogene as a Novel Player in Learning and Memory. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505205454357126

Chicago Manual of Style (16th Edition):

Ghisays, Valentina. “Exploring the Role of the DEK Oncogene as a Novel Player in Learning and Memory.” 2017. Doctoral Dissertation, University of Cincinnati. Accessed August 26, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505205454357126.

MLA Handbook (7th Edition):

Ghisays, Valentina. “Exploring the Role of the DEK Oncogene as a Novel Player in Learning and Memory.” 2017. Web. 26 Aug 2019.

Vancouver:

Ghisays V. Exploring the Role of the DEK Oncogene as a Novel Player in Learning and Memory. [Internet] [Doctoral dissertation]. University of Cincinnati; 2017. [cited 2019 Aug 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505205454357126.

Council of Science Editors:

Ghisays V. Exploring the Role of the DEK Oncogene as a Novel Player in Learning and Memory. [Doctoral Dissertation]. University of Cincinnati; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505205454357126

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