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You searched for subject:( Neurons metabolism). Showing records 1 – 30 of 35 total matches.

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University of Texas Southwestern Medical Center

1. Javadi, Christopher. Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus.

Degree: 2015, University of Texas Southwestern Medical Center

 Neuropeptide Y (NPY)-expressing neurons in the arcuate nucleus of the hypothalamus are part of a neuroendocrine feedback loop that regulates feeding behavior and glucose homeostasis.… (more)

Subjects/Keywords: Arcuate Nucleus of Hypothalamus; Metabolism; Neurons; Neuropeptide Y

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APA (6th Edition):

Javadi, C. (2015). Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Javadi, Christopher. “Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed December 11, 2019. http://hdl.handle.net/2152.5/4222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Javadi, Christopher. “Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus.” 2015. Web. 11 Dec 2019.

Vancouver:

Javadi C. Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2019 Dec 11]. Available from: http://hdl.handle.net/2152.5/4222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Javadi C. Hormone Regulation of NPY Neuron Activity in the Arcuate Nucleus of the Hypothalamus. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McGill University

2. Buscarlet, Manuel. The neural progenitor to neuron transition : role and regulation of GrouchoTLE proteins.

Degree: PhD, Division of Neuroscience., 2008, McGill University

Groucho/transducin-like Enhancer of split (Gro/TLE) family proteins are corepressors found as part of multiple transcriptional complexes that play significant roles during many developmental processes, including… (more)

Subjects/Keywords: Neurons  – metabolism.; Cerebral Cortex  – metabolism.; Repressor Proteins  – metabolism.

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APA (6th Edition):

Buscarlet, M. (2008). The neural progenitor to neuron transition : role and regulation of GrouchoTLE proteins. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile115670.pdf

Chicago Manual of Style (16th Edition):

Buscarlet, Manuel. “The neural progenitor to neuron transition : role and regulation of GrouchoTLE proteins.” 2008. Doctoral Dissertation, McGill University. Accessed December 11, 2019. http://digitool.library.mcgill.ca/thesisfile115670.pdf.

MLA Handbook (7th Edition):

Buscarlet, Manuel. “The neural progenitor to neuron transition : role and regulation of GrouchoTLE proteins.” 2008. Web. 11 Dec 2019.

Vancouver:

Buscarlet M. The neural progenitor to neuron transition : role and regulation of GrouchoTLE proteins. [Internet] [Doctoral dissertation]. McGill University; 2008. [cited 2019 Dec 11]. Available from: http://digitool.library.mcgill.ca/thesisfile115670.pdf.

Council of Science Editors:

Buscarlet M. The neural progenitor to neuron transition : role and regulation of GrouchoTLE proteins. [Doctoral Dissertation]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile115670.pdf


University of Toronto

3. Kim, Ja Hyun. Molecular Mechanism and Effects of Furan Fatty Acid Metabolite CMPF on Adipocytes.

Degree: 2017, University of Toronto

In rodents, increased plasma level of CMPF caused preferential fatty acid utilization over glucose in the pancreatic β cells. Here, we aimed to investigate whether… (more)

Subjects/Keywords: Adipose tissue/adipocytes; CMPF; Diabetes; Fatty acid Metabolism; Hypothalamic NPY/AgRP neurons; Hypothalamic POMC neurons; 0719

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APA (6th Edition):

Kim, J. H. (2017). Molecular Mechanism and Effects of Furan Fatty Acid Metabolite CMPF on Adipocytes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/92815

Chicago Manual of Style (16th Edition):

Kim, Ja Hyun. “Molecular Mechanism and Effects of Furan Fatty Acid Metabolite CMPF on Adipocytes.” 2017. Masters Thesis, University of Toronto. Accessed December 11, 2019. http://hdl.handle.net/1807/92815.

MLA Handbook (7th Edition):

Kim, Ja Hyun. “Molecular Mechanism and Effects of Furan Fatty Acid Metabolite CMPF on Adipocytes.” 2017. Web. 11 Dec 2019.

Vancouver:

Kim JH. Molecular Mechanism and Effects of Furan Fatty Acid Metabolite CMPF on Adipocytes. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2019 Dec 11]. Available from: http://hdl.handle.net/1807/92815.

Council of Science Editors:

Kim JH. Molecular Mechanism and Effects of Furan Fatty Acid Metabolite CMPF on Adipocytes. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/92815

4. Schneider, Lonnie E. Mitochondrial morphology and function in neuronal cells under stress.

Degree: PhD, 2012, University of Alabama – Birmingham

Neurodegenerative disease encompasses a wide range of conditions and pathologies that can manifest at any age depending on the etiology. A major factor in both… (more)

Subjects/Keywords: Cathepsin D <; br>; Cell Differentiation<; br>; Dopaminergic Neurons <; br>; Energy Metabolism<; br>; Neurons – metabolism<; br>; Oxidative Stress

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APA (6th Edition):

Schneider, L. E. (2012). Mitochondrial morphology and function in neuronal cells under stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1369

Chicago Manual of Style (16th Edition):

Schneider, Lonnie E. “Mitochondrial morphology and function in neuronal cells under stress.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1369.

MLA Handbook (7th Edition):

Schneider, Lonnie E. “Mitochondrial morphology and function in neuronal cells under stress.” 2012. Web. 11 Dec 2019.

Vancouver:

Schneider LE. Mitochondrial morphology and function in neuronal cells under stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1369.

Council of Science Editors:

Schneider LE. Mitochondrial morphology and function in neuronal cells under stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1369

5. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 11 Dec 2019.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439


University of Florida

6. Nangle, Shannon. The effect of housing conditions on the metabolic activity of the cortico-basal ganglia-thalamic circuit in American mink (Neovison vison).

Degree: 2010, University of Florida

 Repetitive motor behavior is characterized by repetitive stereotypic movements and manipulation of objects and is a common feature of developmental disorders. The current knowledge of… (more)

Subjects/Keywords: Animal models; Autistic disorder; Basal ganglia; Cytochromes; Environmental enrichment; Mink; Neurons; Oxidases; Receptors; Stereotypies; American mink; Basal ganglia; Metabolism

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APA (6th Edition):

Nangle, S. (2010). The effect of housing conditions on the metabolic activity of the cortico-basal ganglia-thalamic circuit in American mink (Neovison vison). (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00059700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nangle, Shannon. “The effect of housing conditions on the metabolic activity of the cortico-basal ganglia-thalamic circuit in American mink (Neovison vison).” 2010. Thesis, University of Florida. Accessed December 11, 2019. http://ufdc.ufl.edu/AA00059700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nangle, Shannon. “The effect of housing conditions on the metabolic activity of the cortico-basal ganglia-thalamic circuit in American mink (Neovison vison).” 2010. Web. 11 Dec 2019.

Vancouver:

Nangle S. The effect of housing conditions on the metabolic activity of the cortico-basal ganglia-thalamic circuit in American mink (Neovison vison). [Internet] [Thesis]. University of Florida; 2010. [cited 2019 Dec 11]. Available from: http://ufdc.ufl.edu/AA00059700.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nangle S. The effect of housing conditions on the metabolic activity of the cortico-basal ganglia-thalamic circuit in American mink (Neovison vison). [Thesis]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/AA00059700

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université du Luxembourg

7. Delcambre, Sylvie. In vitro Metabolic Studies of Dopamine Synthesis and the Toxicity of L-DOPA in Human Cells.

Degree: 2016, Université du Luxembourg

 This work is divided in two parts. In the first, I investigated the effects of 2,3- dihydroxy-L-phenylalanine (L-DOPA) on the metabolism of human tyrosine hydroxylase… (more)

Subjects/Keywords: Dopamine; Metabolism; Neurons; Life sciences :: Biochemistry, biophysics & molecular biology [F05]; Sciences du vivant :: Biochimie, biophysique & biologie moléculaire [F05]

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APA (6th Edition):

Delcambre, S. (2016). In vitro Metabolic Studies of Dopamine Synthesis and the Toxicity of L-DOPA in Human Cells. (Doctoral Dissertation). Université du Luxembourg. Retrieved from http://orbilu.uni.lu/handle/10993/28204

Chicago Manual of Style (16th Edition):

Delcambre, Sylvie. “In vitro Metabolic Studies of Dopamine Synthesis and the Toxicity of L-DOPA in Human Cells.” 2016. Doctoral Dissertation, Université du Luxembourg. Accessed December 11, 2019. http://orbilu.uni.lu/handle/10993/28204.

MLA Handbook (7th Edition):

Delcambre, Sylvie. “In vitro Metabolic Studies of Dopamine Synthesis and the Toxicity of L-DOPA in Human Cells.” 2016. Web. 11 Dec 2019.

Vancouver:

Delcambre S. In vitro Metabolic Studies of Dopamine Synthesis and the Toxicity of L-DOPA in Human Cells. [Internet] [Doctoral dissertation]. Université du Luxembourg; 2016. [cited 2019 Dec 11]. Available from: http://orbilu.uni.lu/handle/10993/28204.

Council of Science Editors:

Delcambre S. In vitro Metabolic Studies of Dopamine Synthesis and the Toxicity of L-DOPA in Human Cells. [Doctoral Dissertation]. Université du Luxembourg; 2016. Available from: http://orbilu.uni.lu/handle/10993/28204


Macquarie University

8. Bou Farah, Lama. Intrinsic properties and network configuration of sympathetic premotor neurons in the rostral ventrolateral medulla.

Degree: 2015, Macquarie University

Thesis by publication.

1. Literature review 2. Glial ATP release, but not activation of heme oxygenase 2, contributes to the sensitivity of sympathetic premotor neurons(more)

Subjects/Keywords: Neurochemistry  – Research; Neurotransmitters  – Physiology; Neurotransmitters  – Metabolism; Sympathetic nervous system  – Physiology; rostral ventrolateral medulla; bulbospinal neurons; ympathetic; blood pressure

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APA (6th Edition):

Bou Farah, L. (2015). Intrinsic properties and network configuration of sympathetic premotor neurons in the rostral ventrolateral medulla. (Doctoral Dissertation). Macquarie University. Retrieved from http://hdl.handle.net/1959.14/1053257

Chicago Manual of Style (16th Edition):

Bou Farah, Lama. “Intrinsic properties and network configuration of sympathetic premotor neurons in the rostral ventrolateral medulla.” 2015. Doctoral Dissertation, Macquarie University. Accessed December 11, 2019. http://hdl.handle.net/1959.14/1053257.

MLA Handbook (7th Edition):

Bou Farah, Lama. “Intrinsic properties and network configuration of sympathetic premotor neurons in the rostral ventrolateral medulla.” 2015. Web. 11 Dec 2019.

Vancouver:

Bou Farah L. Intrinsic properties and network configuration of sympathetic premotor neurons in the rostral ventrolateral medulla. [Internet] [Doctoral dissertation]. Macquarie University; 2015. [cited 2019 Dec 11]. Available from: http://hdl.handle.net/1959.14/1053257.

Council of Science Editors:

Bou Farah L. Intrinsic properties and network configuration of sympathetic premotor neurons in the rostral ventrolateral medulla. [Doctoral Dissertation]. Macquarie University; 2015. Available from: http://hdl.handle.net/1959.14/1053257


Vanderbilt University

9. Palubinsky, Amy Marie. To Fold or Not to Fold: The Role of Protein Triage in Dictating Neural Cell Fate Following Acute Injury.

Degree: PhD, Neuroscience, 2019, Vanderbilt University

 Neuronal protein refolding and degradation in response to stress is largely mediated by the HSP70 chaperone complex and is critical for maintaining cell function and… (more)

Subjects/Keywords: Protein Triage; Protein Folding; Neuronal Metabolism; CHIP; Chaperones; Primary Neurons; Oxygen and Glucose Deprivation; Ischemia; Stroke

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APA (6th Edition):

Palubinsky, A. M. (2019). To Fold or Not to Fold: The Role of Protein Triage in Dictating Neural Cell Fate Following Acute Injury. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06172019-101033/ ;

Chicago Manual of Style (16th Edition):

Palubinsky, Amy Marie. “To Fold or Not to Fold: The Role of Protein Triage in Dictating Neural Cell Fate Following Acute Injury.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed December 11, 2019. http://etd.library.vanderbilt.edu/available/etd-06172019-101033/ ;.

MLA Handbook (7th Edition):

Palubinsky, Amy Marie. “To Fold or Not to Fold: The Role of Protein Triage in Dictating Neural Cell Fate Following Acute Injury.” 2019. Web. 11 Dec 2019.

Vancouver:

Palubinsky AM. To Fold or Not to Fold: The Role of Protein Triage in Dictating Neural Cell Fate Following Acute Injury. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2019 Dec 11]. Available from: http://etd.library.vanderbilt.edu/available/etd-06172019-101033/ ;.

Council of Science Editors:

Palubinsky AM. To Fold or Not to Fold: The Role of Protein Triage in Dictating Neural Cell Fate Following Acute Injury. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://etd.library.vanderbilt.edu/available/etd-06172019-101033/ ;


University of Western Sydney

10. Steele, Megan. Effect of proinflammatory activation on the neurosupportive functions of astrocytes.

Degree: 2011, University of Western Sydney

 A complex relationship exists between astrocytes and neurons involving astrocytic uptake of glucose and glutathione (GSH) precursors (cysteine, glycine and glutamate) and the release of… (more)

Subjects/Keywords: Thesis (Ph.D.) – University of Western Sydney, 2011; astrocytes; neurons; lactates; glucose; metabolism; neurodegenerative diseases; Alzheimer's disease; nervous system; degeneration

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APA (6th Edition):

Steele, M. (2011). Effect of proinflammatory activation on the neurosupportive functions of astrocytes. (Thesis). University of Western Sydney. Retrieved from http://handle.uws.edu.au:8081/1959.7/506974

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steele, Megan. “Effect of proinflammatory activation on the neurosupportive functions of astrocytes.” 2011. Thesis, University of Western Sydney. Accessed December 11, 2019. http://handle.uws.edu.au:8081/1959.7/506974.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steele, Megan. “Effect of proinflammatory activation on the neurosupportive functions of astrocytes.” 2011. Web. 11 Dec 2019.

Vancouver:

Steele M. Effect of proinflammatory activation on the neurosupportive functions of astrocytes. [Internet] [Thesis]. University of Western Sydney; 2011. [cited 2019 Dec 11]. Available from: http://handle.uws.edu.au:8081/1959.7/506974.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steele M. Effect of proinflammatory activation on the neurosupportive functions of astrocytes. [Thesis]. University of Western Sydney; 2011. Available from: http://handle.uws.edu.au:8081/1959.7/506974

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Moore, Carlene Drucilla. The role of centaurin alpha-1 in the regulation of neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In the nervous system, PI 3-kinase has been implicated in neuronal differentiation. My studies have focused on a candidate neuronal PI 3-kinase target centaurin alpha-1,… (more)

Subjects/Keywords: 1-Phosphatidylinositol 3-Kinase <; br>; Adaptor Proteins, Signal Transducing  – metabolism <; br>; Dendrites  – metabolism <; br>; Dendrites  – ultrastructure <; br>; GTPase-Activating Proteins  – metabolism <; br>; Hippocampus  – cytology <; br>; Nerve Tissue Proteins  – metabolism <; br>; Neurons  – metabolism

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APA (6th Edition):

Moore, C. D. (2008). The role of centaurin alpha-1 in the regulation of neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,208

Chicago Manual of Style (16th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,208.

MLA Handbook (7th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Web. 11 Dec 2019.

Vancouver:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208.

Council of Science Editors:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208


McGill University

12. Murshid, Ayesha. Membrane trafficking and endocytosis in neurons.

Degree: PhD, Department of Anatomy and Cell Biology., 2008, McGill University

In mammalian cells, two main membrane trafficking pathways, the secretory and endocytic pathways are important for many biological functions. Secretory proteins are synthesized on ribosomes… (more)

Subjects/Keywords: Adaptor Protein Complex 2  – metabolism.; Clathrin  – metabolism.; Endocytosis.; Neurons  – physiology.

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APA (6th Edition):

Murshid, A. (2008). Membrane trafficking and endocytosis in neurons. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile114005.pdf

Chicago Manual of Style (16th Edition):

Murshid, Ayesha. “Membrane trafficking and endocytosis in neurons.” 2008. Doctoral Dissertation, McGill University. Accessed December 11, 2019. http://digitool.library.mcgill.ca/thesisfile114005.pdf.

MLA Handbook (7th Edition):

Murshid, Ayesha. “Membrane trafficking and endocytosis in neurons.” 2008. Web. 11 Dec 2019.

Vancouver:

Murshid A. Membrane trafficking and endocytosis in neurons. [Internet] [Doctoral dissertation]. McGill University; 2008. [cited 2019 Dec 11]. Available from: http://digitool.library.mcgill.ca/thesisfile114005.pdf.

Council of Science Editors:

Murshid A. Membrane trafficking and endocytosis in neurons. [Doctoral Dissertation]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile114005.pdf

13. Filiano, Anthony J. The protective role of transglutaminase 2 in ischemic stroke.

Degree: PhD, 2009, University of Alabama – Birmingham

Stroke is a leading cause of long term disabilities in the US. Currently, administration of thrombolytics is the only approved therapy. Due to the variability,… (more)

Subjects/Keywords: Aryl Hydrocarbon Receptor Nuclear Translocator  – metabolism<; br>; Cell Hypoxia<; br>; GTP-Binding Proteins  – metabolism<; br>; Ischemia  – prevention & control<; br>; Neurons  – metabolism<; br>; Transglutaminases  – metabolism

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APA (6th Edition):

Filiano, A. J. (2009). The protective role of transglutaminase 2 in ischemic stroke. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,496

Chicago Manual of Style (16th Edition):

Filiano, Anthony J. “The protective role of transglutaminase 2 in ischemic stroke.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,496.

MLA Handbook (7th Edition):

Filiano, Anthony J. “The protective role of transglutaminase 2 in ischemic stroke.” 2009. Web. 11 Dec 2019.

Vancouver:

Filiano AJ. The protective role of transglutaminase 2 in ischemic stroke. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,496.

Council of Science Editors:

Filiano AJ. The protective role of transglutaminase 2 in ischemic stroke. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,496

14. Larimore, Jennifer Lynn. The role of centaurins in vesicular trafficking and neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In order to better understand the role of the phosphoinositide (PI) 3-Kinase (PI3K) pathway in neuronal differentiation and plasticity, I am studying two centaurins that… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing  – metabolism<; br>; Dendrites  – metabolism<; br>; Dentrites  – ultrastructure<; br>; GTPase-Activating Proteins  – metabolism<; br>; Hippocampus  – cytology<; br>; Neurons  – metabolism

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APA (6th Edition):

Larimore, J. L. (2008). The role of centaurins in vesicular trafficking and neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,823

Chicago Manual of Style (16th Edition):

Larimore, Jennifer Lynn. “The role of centaurins in vesicular trafficking and neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,823.

MLA Handbook (7th Edition):

Larimore, Jennifer Lynn. “The role of centaurins in vesicular trafficking and neuronal differentiation.” 2008. Web. 11 Dec 2019.

Vancouver:

Larimore JL. The role of centaurins in vesicular trafficking and neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,823.

Council of Science Editors:

Larimore JL. The role of centaurins in vesicular trafficking and neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,823

15. Jones, Page. Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model.

Degree: PhD, 2008, University of Alabama – Birmingham

Mutations to copper,zinc superoxide dismutase (Cu,Zn SOD or SOD1) are the only known causes of amyotrophic lateral sclerosis (ALS), a neurodegenerative disorder characterized primarily by… (more)

Subjects/Keywords: Amyotrophic Lateral Sclerosis  – enzymology <; br>; Copper  – metabolism <; br>; Disease Progression <; br>; Nerve Degeneration  – enzymology <; br>; Neurons  – metabolism <; br>; Spinal Cord  – enzymology <; br>; Superoxide Dismutase  – metabolism

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APA (6th Edition):

Jones, P. (2008). Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,370

Chicago Manual of Style (16th Edition):

Jones, Page. “Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,370.

MLA Handbook (7th Edition):

Jones, Page. “Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model.” 2008. Web. 11 Dec 2019.

Vancouver:

Jones P. Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,370.

Council of Science Editors:

Jones P. Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,370

16. Rubio, Maria Dolores. Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms.

Degree: PhD, 2011, University of Alabama – Birmingham

Cytoskeletal actin filaments underlie dendritic spine plasticity, critical for several forms of learning and memory. Therefore, understanding the mechanisms that regulate actin dynamics is essential… (more)

Subjects/Keywords: Actins  – metabolism<; br>; Cardiac Myosins  – physiology<; br>; Long-Term Potentiation  – physiology<; br>; Memory  – physiology<; br>; Myosin Heavy Chains  – physiology<; br>; Neurons  – metabolism<; br>; Nonmuscle Myosin Type IIB  – metabolism<; br>; Synapses  – physiology

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APA (6th Edition):

Rubio, M. D. (2011). Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,878

Chicago Manual of Style (16th Edition):

Rubio, Maria Dolores. “Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,878.

MLA Handbook (7th Edition):

Rubio, Maria Dolores. “Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms.” 2011. Web. 11 Dec 2019.

Vancouver:

Rubio MD. Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,878.

Council of Science Editors:

Rubio MD. Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,878


University of Missouri – Columbia

17. He, Yan, 1979-. Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease.

Degree: 2011, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Alzheimer's disease (AD) is characterized by a progressive decline in memory and cognitive function together… (more)

Subjects/Keywords: NMDA receptor; oligomeric amyloid-beta peptides; oxidative damage; mitochondrial dysfunction; botanical antioxidants; Amyloid beta-peptides  – pharmacology; Catechin  – analogs & derivatives; Catechin  – pharmacology; N-Methylaspartate  – pharmacology|Reactive Oxygen Species  – metabolism; NADPH Oxidase  – metabolism; Arachidonic Acid  – metabolism; Neurons  – drug effects; Neuroprotective Agents  – pharmacology

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APA (6th Edition):

He, Yan, 1. (2011). Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/15839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

He, Yan, 1979-. “Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease.” 2011. Thesis, University of Missouri – Columbia. Accessed December 11, 2019. http://hdl.handle.net/10355/15839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

He, Yan, 1979-. “Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease.” 2011. Web. 11 Dec 2019.

Vancouver:

He, Yan 1. Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease. [Internet] [Thesis]. University of Missouri – Columbia; 2011. [cited 2019 Dec 11]. Available from: http://hdl.handle.net/10355/15839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

He, Yan 1. Amyloid-beta toxicity in neurons and potential botanical compounds for prevention of Alzheimer's disease. [Thesis]. University of Missouri – Columbia; 2011. Available from: http://hdl.handle.net/10355/15839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Saucisse, Nicolas. Dissection du rôle de la voie intracellulaire de mTORC1 dans les circuits hypothalamiques à la mélanocortine régulant la prise alimentaire : Dissecting the role of the intacellular mTORC1 pathway in hypothalamic melanocortin circuitry regulating food intake.

Degree: Docteur es, Neurosciences, 2016, Bordeaux

L’hypothalamus est une structure cérébrale ayant un rôle clé dans la régulation de la prise alimentaire. Parmi les différentes populations neuronales qui le composent, les… (more)

Subjects/Keywords: Neurones à POMC; MTORC1; ECS; Neurotransmetteurs; Prise alimentaire; « refeeding »; P62/SQSTM1; Métabolisme; Hypothalamus; Obésité; POMC neurons; MTORC1; ECS; Neurotransmitters; Food intake; Refeeding; P62/SQSTM1; Metabolism; Hypothalamus; Obesity

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APA (6th Edition):

Saucisse, N. (2016). Dissection du rôle de la voie intracellulaire de mTORC1 dans les circuits hypothalamiques à la mélanocortine régulant la prise alimentaire : Dissecting the role of the intacellular mTORC1 pathway in hypothalamic melanocortin circuitry regulating food intake. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2016BORD0151

Chicago Manual of Style (16th Edition):

Saucisse, Nicolas. “Dissection du rôle de la voie intracellulaire de mTORC1 dans les circuits hypothalamiques à la mélanocortine régulant la prise alimentaire : Dissecting the role of the intacellular mTORC1 pathway in hypothalamic melanocortin circuitry regulating food intake.” 2016. Doctoral Dissertation, Bordeaux. Accessed December 11, 2019. http://www.theses.fr/2016BORD0151.

MLA Handbook (7th Edition):

Saucisse, Nicolas. “Dissection du rôle de la voie intracellulaire de mTORC1 dans les circuits hypothalamiques à la mélanocortine régulant la prise alimentaire : Dissecting the role of the intacellular mTORC1 pathway in hypothalamic melanocortin circuitry regulating food intake.” 2016. Web. 11 Dec 2019.

Vancouver:

Saucisse N. Dissection du rôle de la voie intracellulaire de mTORC1 dans les circuits hypothalamiques à la mélanocortine régulant la prise alimentaire : Dissecting the role of the intacellular mTORC1 pathway in hypothalamic melanocortin circuitry regulating food intake. [Internet] [Doctoral dissertation]. Bordeaux; 2016. [cited 2019 Dec 11]. Available from: http://www.theses.fr/2016BORD0151.

Council of Science Editors:

Saucisse N. Dissection du rôle de la voie intracellulaire de mTORC1 dans les circuits hypothalamiques à la mélanocortine régulant la prise alimentaire : Dissecting the role of the intacellular mTORC1 pathway in hypothalamic melanocortin circuitry regulating food intake. [Doctoral Dissertation]. Bordeaux; 2016. Available from: http://www.theses.fr/2016BORD0151

19. Winkelbauer, Marlene Elizabeth. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.

Degree: PhD, 2007, University of Alabama – Birmingham

Numerous disorders are characterized by the presence of cystic lesions within the kidney. The proteins associated with these disorders often localize to cilia, and improper… (more)

Subjects/Keywords: Caenorhabditis elegans  – genetics<; br>; Caenorhabditis elegans Proteins  – genetics<; br>; Cilia  – metabolism<; br>; Neurons, Afferent  – physiology<; br>; Protein Transport<; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Winkelbauer, M. E. (2007). Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,619

Chicago Manual of Style (16th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,619.

MLA Handbook (7th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Web. 11 Dec 2019.

Vancouver:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619.

Council of Science Editors:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619

20. Markwardt, Sean J. GABAergic signaling to adult-generated neurons in hippocampus.

Degree: PhD, 2011, University of Alabama – Birmingham

In the central nervous system of adult mammals, new neurons are produced throughout life in at least two regions, the subventricular zone and dentate gyrus… (more)

Subjects/Keywords: Adult Stem Cells  – physiology<; br>; Dentate Gyrus  – cytology<; br>; gamma-Aminobutyric Acid  – metabolism<; br>; Neurons  – physiology<; br>; Receptors, GABA  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Markwardt, S. J. (2011). GABAergic signaling to adult-generated neurons in hippocampus. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1096

Chicago Manual of Style (16th Edition):

Markwardt, Sean J. “GABAergic signaling to adult-generated neurons in hippocampus.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1096.

MLA Handbook (7th Edition):

Markwardt, Sean J. “GABAergic signaling to adult-generated neurons in hippocampus.” 2011. Web. 11 Dec 2019.

Vancouver:

Markwardt SJ. GABAergic signaling to adult-generated neurons in hippocampus. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1096.

Council of Science Editors:

Markwardt SJ. GABAergic signaling to adult-generated neurons in hippocampus. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1096

21. Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye.

Degree: PhD, 2008, University of Alabama – Birmingham

Development of the vertebrate eye is controlled by a network of regulatory genes including those in the basic loop-helix-loop (bHLH) family. In this study, we… (more)

Subjects/Keywords: Amacrine Cells  – physiology<; br>; Avian Proteins  – metabolism<; br>; Basic Helix-Loop-Helix Transcription Factors  – metabolism<; br>; Gene Expression Regulation, Developmental  – physiology<; br>; Nerve Tissue Proteins  – genetics<; br>; Nuclear Reprogramming<; br>; Retina  – cytology<; br>; Retinal Neurons  – cytology Retinal Pigment Epithelium  – cytology<; br>; Stem Cells  – cytology

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APA (6th Edition):

Mao,Weiming. (2008). The role of bHLH gene ash1 in the developing chick eye. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Web. 11 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

22. Crimmins, Stephen Lewis. Characterization and functional analysis of Usp14.

Degree: PhD, 2007, University of Alabama – Birmingham

The ubiquitin proteasome system (UPS) is essential for regulated protein degrada-tion, a requirement for numerous neuronal process, including vesicle cycling, neuro-transmitter release, spine morphology, and… (more)

Subjects/Keywords: Ataxia  – enzymology <; br>; Ataxia  – genetics <; br>; Gene Expression Regulation, Enzymologic  – physiology <; br>; Neurons  – enzymology <; br>; Ubiquitin  – metabolism <; br>; Ubiquitin Thiolesterase  – biosynthesis <; br>; Ubiquitin Thiolesterase  – genetics

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APA (6th Edition):

Crimmins, S. L. (2007). Characterization and functional analysis of Usp14. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,118

Chicago Manual of Style (16th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,118.

MLA Handbook (7th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Web. 11 Dec 2019.

Vancouver:

Crimmins SL. Characterization and functional analysis of Usp14. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118.

Council of Science Editors:

Crimmins SL. Characterization and functional analysis of Usp14. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118

23. Eom, Tae-Yeon. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.

Degree: PhD, 2009, University of Alabama – Birmingham

Neurogenesis is a crucial process for development, plasticity, and regenerative capacity of the developing and adult brain. Impairment of neurogenesis has been implicated in the… (more)

Subjects/Keywords: Apoptosis<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; Mice, Inbred C57BL<; br>; Mice, Knockout<; br>; Mice, Transgenic<; br>; Neurons  – cytology<; br>; Phosphorylation

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APA (6th Edition):

Eom, T. (2009). Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,390

Chicago Manual of Style (16th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,390.

MLA Handbook (7th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Web. 11 Dec 2019.

Vancouver:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390.

Council of Science Editors:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390

24. Tower-Gilchrist, Cristy (Cristy Davette). The role of protein folding and protein trafficking in human disease.

Degree: PhD, 2011, University of Alabama – Birmingham

Project 1: Expansion of CAG repeats encoding glutamine in huntingtin and ataxin 3 causes the neurodegenerative diseases Huntington's disease (HD) and spinocerebellar ataxia 3 (SCA3),… (more)

Subjects/Keywords: Cilia<; br>; Cytomegalovirus  – enzymology<; br>; Huntington Disease  – virology<; br>; Neurons  – metabolism<; br>; Peptides  – antagonists & inhibitors<; br>; Phosphotransferases (Alcohol Group Acceptor)  – physiology<; br>; Spinocerebellar Ataxias  – virology

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APA (6th Edition):

Tower-Gilchrist, C. (. D. (2011). The role of protein folding and protein trafficking in human disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,964

Chicago Manual of Style (16th Edition):

Tower-Gilchrist, Cristy (Cristy Davette). “The role of protein folding and protein trafficking in human disease.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,964.

MLA Handbook (7th Edition):

Tower-Gilchrist, Cristy (Cristy Davette). “The role of protein folding and protein trafficking in human disease.” 2011. Web. 11 Dec 2019.

Vancouver:

Tower-Gilchrist C(D. The role of protein folding and protein trafficking in human disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,964.

Council of Science Editors:

Tower-Gilchrist C(D. The role of protein folding and protein trafficking in human disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,964

25. Geng, Ying. p53-dependent neural stem cell death regulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Regulation of neural precursor cell (NPC) death is important for both normal brain development and prevention of brain tumor formation. The tumor suppressor p53 is… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – physiology bcl-2-Associated X Protein  – physiology<; br>; Brain Neoplasms  – genetics Cerebellum  – cytology<; br>; Chloroquine  – pharmacology<; br>; Cytoplasm  – metabolism<; br>; Glioma  – genetics Neurons  – cytology<; br>; Stem Cells  – cytology Transcriptional Activation  – physiology<; br>; Tumor Suppressor Protein p53  – genetics

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APA (6th Edition):

Geng, Y. (2008). p53-dependent neural stem cell death regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,767

Chicago Manual of Style (16th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,767.

MLA Handbook (7th Edition):

Geng, Ying. “p53-dependent neural stem cell death regulation.” 2008. Web. 11 Dec 2019.

Vancouver:

Geng Y. p53-dependent neural stem cell death regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767.

Council of Science Editors:

Geng Y. p53-dependent neural stem cell death regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,767


Queens University

26. Ong, Edmund. Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization .

Degree: Pharmacology and Toxicology, Queens University

 Opioid receptors are the sites of action for morphine and most other clinically-used opioid drugs. Abundant evidence now demonstrates that different opioid receptor types can… (more)

Subjects/Keywords: Delta Opioid Receptor; Mu Opioid Receptor; Trafficking; Internalization; Dorsal Root Ganglia; Morphine; DAMGO; Deltorphin II; SNC80; SDM-25N; Adenylate Cyclase; Adenylate Cyclase: Metabolism; Animals; Antibodies; Antibodies: Metabolism; Blotting; Brain; Brain: Metabolism; CHO Cells; Cricetinae; Cricetulus; Enzyme-Linked Immunosorbent Assay; Gene Expression Regulation; Gene Expression Regulation: Physiology; Immunohistochemistry; Immunocytochemistry; Immunoprecipitation; Inbred c57bl; Knockout; Mice; Morphine: Administration & Dosage; Morphine: Pharmacology; Multiprotein Complexes; Multiprotein Complexes: Metabolism; Neurons; Neurons: Metabolism; Opioid; Rats; Receptors; Signal Transduction; Signal Transduction: Physiology; Western; Delta; Delta: Genetics; Delta: Metabolism; Mu; Mu: Metabolism; Colocalization; Colocalizational Analysis; Degradation; G-Protein Coupled Receptor; Microscopy; Molecular Biology; Receptor Post-Internalization Trafficking; Recycling; Oligomerization; Receptor Signaling

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APA (6th Edition):

Ong, E. (n.d.). Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/15437

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ong, Edmund. “Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization .” Thesis, Queens University. Accessed December 11, 2019. http://hdl.handle.net/1974/15437.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ong, Edmund. “Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization .” Web. 11 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Ong E. Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization . [Internet] [Thesis]. Queens University; [cited 2019 Dec 11]. Available from: http://hdl.handle.net/1974/15437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Ong E. Investigating the Effects of Prolonged Mu Opioid Receptor Activation Upon Opioid Receptor Heteromerization . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/15437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

27. Ghosh, Arindam P. Regulation of neuronal cell death by BH3-only molecules.

Degree: PhD, 2012, University of Alabama – Birmingham

Apoptosis in metazoan organisms plays critical roles in normal development, tissue homeostasis and immunity, and its disturbed regulation leads to many pathological states, including cancer,… (more)

Subjects/Keywords: Apoptosis  – physiology<; br>; Apoptosis Regulatory Proteins  – metabolism<; br>; Ethanol  – toxicity<; br>; Nervous System  – embryology<; br>; Neurons  – physiology<; br>; Neuropeptides  – metabolism<; br>; Tumor Suppressor Protein p53  – metabolism<; br>; Tumor Suppressor Proteins  – metabolism<; br>; bcl-2-Associated X Protein  – metabolism<; br>; bcl-X Protein  – metabolism

…42 6. NOXA deficiency on its own does not protect neurons from the toxic effects of ethanol… …reduces apoptosis of immature neurons in the developing DRG in comparison to wild-type… …triggers the intrinsic apoptotic pathway in neurons and that this requires the multi-BH domain… …neurons; it may act as a stimulant on them, rather than as a generalized CNS depressant9… …Extensive studies have shown that neurons develop resistance to the effects of ethanol as they… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ghosh, A. P. (2012). Regulation of neuronal cell death by BH3-only molecules. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1138

Chicago Manual of Style (16th Edition):

Ghosh, Arindam P. “Regulation of neuronal cell death by BH3-only molecules.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 11, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1138.

MLA Handbook (7th Edition):

Ghosh, Arindam P. “Regulation of neuronal cell death by BH3-only molecules.” 2012. Web. 11 Dec 2019.

Vancouver:

Ghosh AP. Regulation of neuronal cell death by BH3-only molecules. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Dec 11]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1138.

Council of Science Editors:

Ghosh AP. Regulation of neuronal cell death by BH3-only molecules. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1138


University of Florida

28. Yu, Kan, 1963-. Regulation of neuromodulatory and neurotrophic actions of angiotensin II in brain neurons.

Degree: 1996, University of Florida

Subjects/Keywords: Brain; Catecholamines; Complementary DNA; Gene expression; Hypertension; Messenger RNA; Neurons; Plasminogen activators; Rats; Receptors; Angiotensin II  – metabolism ( mesh ); Angiotensin II  – physiology ( mesh ); Cells, Cultured ( mesh ); Department of Physiology thesis Ph.D ( mesh ); Gene Expression Regulation ( mesh ); Neurons  – physiology ( mesh ); Plasminogen Activator Inhibitor 1  – metabolism ( mesh ); Rats, Inbred SHR ( mesh ); Rats, Inbred WKY ( mesh ); Research ( mesh ); Tyrosine 3-Monooxygenase  – metabolism ( mesh )

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APA (6th Edition):

Yu, Kan, 1. (1996). Regulation of neuromodulatory and neurotrophic actions of angiotensin II in brain neurons. (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00053441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Kan, 1963-. “Regulation of neuromodulatory and neurotrophic actions of angiotensin II in brain neurons.” 1996. Thesis, University of Florida. Accessed December 11, 2019. http://ufdc.ufl.edu/AA00053441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Kan, 1963-. “Regulation of neuromodulatory and neurotrophic actions of angiotensin II in brain neurons.” 1996. Web. 11 Dec 2019.

Vancouver:

Yu, Kan 1. Regulation of neuromodulatory and neurotrophic actions of angiotensin II in brain neurons. [Internet] [Thesis]. University of Florida; 1996. [cited 2019 Dec 11]. Available from: http://ufdc.ufl.edu/AA00053441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu, Kan 1. Regulation of neuromodulatory and neurotrophic actions of angiotensin II in brain neurons. [Thesis]. University of Florida; 1996. Available from: http://ufdc.ufl.edu/AA00053441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Vallès Ortega, Jordi. Estudi del metabolisme del glicogen en la funció neuronal i la seva implicació en la malaltia de Lafora i l’envelliment.

Degree: Departament de Bioquímica i Biologia Molecular (Biologia), 2012, Universitat de Barcelona

 Lafora disease (LD) is caused by mutations in either the laforin or malin gene. The hallmark of the disease is the accumulation of polyglucosan inclusions… (more)

Subjects/Keywords: Glicogen; Glucógeno; Glycogen; Metabolisme; Metabolismo; Metabolism; Neurones; Neuronas; Neurons; Envelliment; Envejecimiento; Aging; Malaltia de Lafora; Enfermedad de Lafora; Lafora disease; Ciències Experimentals i Matemàtiques; 577

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vallès Ortega, J. (2012). Estudi del metabolisme del glicogen en la funció neuronal i la seva implicació en la malaltia de Lafora i l’envelliment. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/117739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vallès Ortega, Jordi. “Estudi del metabolisme del glicogen en la funció neuronal i la seva implicació en la malaltia de Lafora i l’envelliment.” 2012. Thesis, Universitat de Barcelona. Accessed December 11, 2019. http://hdl.handle.net/10803/117739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vallès Ortega, Jordi. “Estudi del metabolisme del glicogen en la funció neuronal i la seva implicació en la malaltia de Lafora i l’envelliment.” 2012. Web. 11 Dec 2019.

Vancouver:

Vallès Ortega J. Estudi del metabolisme del glicogen en la funció neuronal i la seva implicació en la malaltia de Lafora i l’envelliment. [Internet] [Thesis]. Universitat de Barcelona; 2012. [cited 2019 Dec 11]. Available from: http://hdl.handle.net/10803/117739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vallès Ortega J. Estudi del metabolisme del glicogen en la funció neuronal i la seva implicació en la malaltia de Lafora i l’envelliment. [Thesis]. Universitat de Barcelona; 2012. Available from: http://hdl.handle.net/10803/117739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

30. Baughan, Travis, 1980-. Gene therapy in spinal muscular atrophy: RNA-based strategies to modulate the pre-mRNA splicing of survival motor neuron.

Degree: 2008, University of Missouri – Columbia

 Gene expression is the required process in eukaryotes in which DNA is transcribed into pre-mRNA, spliced to produce "mature" mRNA and translated into proteins. Inaccuracies… (more)

Subjects/Keywords: Spinal muscular atrophy  – Treatment; RNA splicing; Genetic regulation; Protein precursors; Motor neurons  – Diseases; Antisense RNA; Muscular Atrophy, Spinal  – therapy; RNA Splicing; Gene Expression Regulation; RNA Precursors; Survival of Motor Neuron 2 Protein  – metabolism; RNA, Antisense  – therapeutic use

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baughan, Travis, 1. (2008). Gene therapy in spinal muscular atrophy: RNA-based strategies to modulate the pre-mRNA splicing of survival motor neuron. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/6686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baughan, Travis, 1980-. “Gene therapy in spinal muscular atrophy: RNA-based strategies to modulate the pre-mRNA splicing of survival motor neuron.” 2008. Thesis, University of Missouri – Columbia. Accessed December 11, 2019. https://doi.org/10.32469/10355/6686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baughan, Travis, 1980-. “Gene therapy in spinal muscular atrophy: RNA-based strategies to modulate the pre-mRNA splicing of survival motor neuron.” 2008. Web. 11 Dec 2019.

Vancouver:

Baughan, Travis 1. Gene therapy in spinal muscular atrophy: RNA-based strategies to modulate the pre-mRNA splicing of survival motor neuron. [Internet] [Thesis]. University of Missouri – Columbia; 2008. [cited 2019 Dec 11]. Available from: https://doi.org/10.32469/10355/6686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baughan, Travis 1. Gene therapy in spinal muscular atrophy: RNA-based strategies to modulate the pre-mRNA splicing of survival motor neuron. [Thesis]. University of Missouri – Columbia; 2008. Available from: https://doi.org/10.32469/10355/6686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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