Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:( Neovascularization Pathologic metabolism 60). Showing records 1 – 30 of 12345 total matches.

[1] [2] [3] [4] [5] … [412]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters

1. Khotskaya, Yekaterina B. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.

Degree: PhD, 2009, University of Alabama – Birmingham

Syndecan-1 (CD138), a transmembrane heparan sulfate-bearing proteoglycan, is expressed at high levels on most myeloma cells and is shed into the microenvironment. In patients, high… (more)

Subjects/Keywords: Gene Expression Regulation, Neoplastic<; br>; Melanoma  – metabolism<; br>; Neoplasm Proteins  – biosynthesis<; br>; Neovascularization, Pathologic  – metabolism<; br>; Syndecan-1  – biosynthesis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khotskaya, Y. B. (2009). Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,766

Chicago Manual of Style (16th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,766.

MLA Handbook (7th Edition):

Khotskaya, Yekaterina B. “Role of syndecan-1 as key regulator of multiple myeloma pathogenesis.” 2009. Web. 24 Jan 2020.

Vancouver:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766.

Council of Science Editors:

Khotskaya YB. Role of syndecan-1 as key regulator of multiple myeloma pathogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,766

2. McFarland, Braden Cox. Targeting angiogenesis with plasminogen kringle 5.

Degree: PhD, 2009, University of Alabama – Birmingham

The recombinant fifth kringle domain of plasminogen (rK5) has been shown to induce apoptosis of dermal microvessel endothelial cells (MvEC), and this pro-apoptotic effect required… (more)

Subjects/Keywords: Angiogenesis Inhibitors  – metabolism<; br>; Apoptosis<; br>; Glioblastoma  – blood supply<; br>; Neovascularization, Pathologic  – drug therapy<; br>; Plasminogen  – therapeutic use

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McFarland, B. C. (2009). Targeting angiogenesis with plasminogen kringle 5. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,399

Chicago Manual of Style (16th Edition):

McFarland, Braden Cox. “Targeting angiogenesis with plasminogen kringle 5.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,399.

MLA Handbook (7th Edition):

McFarland, Braden Cox. “Targeting angiogenesis with plasminogen kringle 5.” 2009. Web. 24 Jan 2020.

Vancouver:

McFarland BC. Targeting angiogenesis with plasminogen kringle 5. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,399.

Council of Science Editors:

McFarland BC. Targeting angiogenesis with plasminogen kringle 5. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,399

3. Splittgerber, Ryan C. Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells.

Degree: PhD, 2008, University of Alabama – Birmingham

Neuronal nicotinic acetylcholine receptors (nAChRs) are widely expressed in neural and non-neural tissues and have been reported to play a role in neovascularization and vascular… (more)

Subjects/Keywords: Angiogenesis Inducing Agents<; br>; Endothelial Cells  – metabolism<; br>; Neovascularization, Pathologic<; br>; Nicotine  – pharmacology<; br>; Receptors, Nicotinic  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Splittgerber, R. C. (2008). Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,835

Chicago Manual of Style (16th Edition):

Splittgerber, Ryan C. “Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,835.

MLA Handbook (7th Edition):

Splittgerber, Ryan C. “Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells.” 2008. Web. 24 Jan 2020.

Vancouver:

Splittgerber RC. Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,835.

Council of Science Editors:

Splittgerber RC. Expression and function of nicotinic acetylcholine receptors in human vascular endothelial cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,835


University of Texas Southwestern Medical Center

4. Arnold, Shanna Alexandria. SPARC: a Matricellular Regulator of the Tumor Microenvironment.

Degree: 2010, University of Texas Southwestern Medical Center

 SPARC, secreted protein acidic and rich in cysteine, is a matricellular protein that governs extracellular matrix (ECM) deposition and maturation during times of tissue remodeling… (more)

Subjects/Keywords: Osteonectin; Pancreatic Neoplasms; Neovascularization, Pathologic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arnold, S. A. (2010). SPARC: a Matricellular Regulator of the Tumor Microenvironment. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arnold, Shanna Alexandria. “SPARC: a Matricellular Regulator of the Tumor Microenvironment.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed January 24, 2020. http://hdl.handle.net/2152.5/867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arnold, Shanna Alexandria. “SPARC: a Matricellular Regulator of the Tumor Microenvironment.” 2010. Web. 24 Jan 2020.

Vancouver:

Arnold SA. SPARC: a Matricellular Regulator of the Tumor Microenvironment. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/2152.5/867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arnold SA. SPARC: a Matricellular Regulator of the Tumor Microenvironment. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Rivera, Lee Benjamin. Regulation of Pericyte Behavior by Secreted Protein Acidic and Rich in Cysteine.

Degree: 2011, University of Texas Southwestern Medical Center

 Pericytes migrate to newly formed vessels where they induce vessel quiescence and promote vessel stability. Secreted Protein Acidic and Rich in Cysteine (SPARC) is a… (more)

Subjects/Keywords: Pancreatic Neoplasms; Neovascularization, Pathologic; Osteonectin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rivera, L. B. (2011). Regulation of Pericyte Behavior by Secreted Protein Acidic and Rich in Cysteine. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rivera, Lee Benjamin. “Regulation of Pericyte Behavior by Secreted Protein Acidic and Rich in Cysteine.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed January 24, 2020. http://hdl.handle.net/2152.5/863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rivera, Lee Benjamin. “Regulation of Pericyte Behavior by Secreted Protein Acidic and Rich in Cysteine.” 2011. Web. 24 Jan 2020.

Vancouver:

Rivera LB. Regulation of Pericyte Behavior by Secreted Protein Acidic and Rich in Cysteine. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/2152.5/863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rivera LB. Regulation of Pericyte Behavior by Secreted Protein Acidic and Rich in Cysteine. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Anderson, Joshua C. Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis.

Degree: PhD, 2008, University of Alabama – Birmingham

Thrombospondin-1 (TSP-1) is a 420 kilodalton homotrimeric protein; it is one of the first identified anti-angiogenic proteins found to be expressed in some normal tissues.… (more)

Subjects/Keywords: Angiogenesis Inhibitors  – therapeutic use <; br>; Brain Neoplasms <; br>; Extracellular Matrix  – physiology <; br>; Glioblastoma  – blood supply <; br>; Glioma <; br>; Neovascularization, Pathologic <; br>; Oligopeptides  – therapeutic use

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anderson, J. C. (2008). Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,318

Chicago Manual of Style (16th Edition):

Anderson, Joshua C. “Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,318.

MLA Handbook (7th Edition):

Anderson, Joshua C. “Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis.” 2008. Web. 24 Jan 2020.

Vancouver:

Anderson JC. Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,318.

Council of Science Editors:

Anderson JC. Thrombospondin type-1 repeats and their potential role in inhibiting glioblastoma angiogenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,318

7. Andrade, Luciana Nogueira de Sousa. Evidência da dualidade funcional de galectina-3 no crescimento de melanoma murino.

Degree: PhD, Oncologia, 2007, University of São Paulo

Tumores são definidos como microambientes compostos não só pelas células malignas, mas também por células endoteliais, fibroblastos e leucócitos, que promovem o crescimento tumoral e… (more)

Subjects/Keywords: Galectin-3; Galectina-3; Melanoma; Melanoma; Neovascularização patológica; Neovascularization pathologic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andrade, L. N. d. S. (2007). Evidência da dualidade funcional de galectina-3 no crescimento de melanoma murino. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-21062007-095821/ ;

Chicago Manual of Style (16th Edition):

Andrade, Luciana Nogueira de Sousa. “Evidência da dualidade funcional de galectina-3 no crescimento de melanoma murino.” 2007. Doctoral Dissertation, University of São Paulo. Accessed January 24, 2020. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-21062007-095821/ ;.

MLA Handbook (7th Edition):

Andrade, Luciana Nogueira de Sousa. “Evidência da dualidade funcional de galectina-3 no crescimento de melanoma murino.” 2007. Web. 24 Jan 2020.

Vancouver:

Andrade LNdS. Evidência da dualidade funcional de galectina-3 no crescimento de melanoma murino. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2020 Jan 24]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-21062007-095821/ ;.

Council of Science Editors:

Andrade LNdS. Evidência da dualidade funcional de galectina-3 no crescimento de melanoma murino. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-21062007-095821/ ;


Universidade do Rio Grande do Sul

8. Castro Júnior, Cyro. Alterações histológicas secundárias à interrupção dos vasa vasorum na aorta descendente com o uso de terapia antiangiogênica : resultados em modelo suíno.

Degree: 2016, Universidade do Rio Grande do Sul

OBJETIVO: demonstrar as alterações histológicas secundárias ao uso de bevacizumabe na aorta descendente de suínos submetida à interrupção dos vasa vasorum. MATERIAIS E MÉTODOS: em… (more)

Subjects/Keywords: Neovascularização patológica; Neovascularization; Pathologic; Inibidores da angiogênese; Vasa Vasorum; Angiogenesis inhibitors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Castro Júnior, C. (2016). Alterações histológicas secundárias à interrupção dos vasa vasorum na aorta descendente com o uso de terapia antiangiogênica : resultados em modelo suíno. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/143080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Castro Júnior, Cyro. “Alterações histológicas secundárias à interrupção dos vasa vasorum na aorta descendente com o uso de terapia antiangiogênica : resultados em modelo suíno.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed January 24, 2020. http://hdl.handle.net/10183/143080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Castro Júnior, Cyro. “Alterações histológicas secundárias à interrupção dos vasa vasorum na aorta descendente com o uso de terapia antiangiogênica : resultados em modelo suíno.” 2016. Web. 24 Jan 2020.

Vancouver:

Castro Júnior C. Alterações histológicas secundárias à interrupção dos vasa vasorum na aorta descendente com o uso de terapia antiangiogênica : resultados em modelo suíno. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/10183/143080.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Castro Júnior C. Alterações histológicas secundárias à interrupção dos vasa vasorum na aorta descendente com o uso de terapia antiangiogênica : resultados em modelo suíno. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/143080

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Zhou, Heling 1985-. Multimodality Imaging of Tumor Vasculature and Metabolism.

Degree: 2013, University of Texas Southwestern Medical Center

 Cancer is the second leading cause of death in America. A number of abnormal features in tumor vasculature and metabolism have been identified as imaging… (more)

Subjects/Keywords: Brain Neoplasms; Breast Neoplasms; Magnetic Resonance Imaging; Neovascularization, Pathologic; Phosphatidylserines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, H. 1. (2013). Multimodality Imaging of Tumor Vasculature and Metabolism. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-115

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Heling 1985-. “Multimodality Imaging of Tumor Vasculature and Metabolism.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 24, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-115.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Heling 1985-. “Multimodality Imaging of Tumor Vasculature and Metabolism.” 2013. Web. 24 Jan 2020.

Vancouver:

Zhou H1. Multimodality Imaging of Tumor Vasculature and Metabolism. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jan 24]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-115.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou H1. Multimodality Imaging of Tumor Vasculature and Metabolism. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-115

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Frizell, Carl Antonio. Investigating Potential Novel Angiogenic And Immunogenic Properties Of Heme Oxygenase-1.

Degree: 2012, University of Alabama – Birmingham

INVESTIGATING POTENTIAL NOVEL ANGIOGENIC AND IMMUNOGENIC PROPERTIES OF HEME OXYGENASE-1According to the American Heart Association and Centers of Disease Control and Prevention, cardiovascular diseases comprise… (more)

Subjects/Keywords: Angiogenic Proteins – physiology.<; br>; Carbon Monoxide – metabolism<; br>; Dendritic Cells – immunology<; br>; Heme Oxygenase-1 – metabolism.<; br>; Neovascularization, Pathologic<; br>; Transplantation, Homologous

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Frizell, C. A. (2012). Investigating Potential Novel Angiogenic And Immunogenic Properties Of Heme Oxygenase-1. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Frizell, Carl Antonio. “Investigating Potential Novel Angiogenic And Immunogenic Properties Of Heme Oxygenase-1.” 2012. Thesis, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Frizell, Carl Antonio. “Investigating Potential Novel Angiogenic And Immunogenic Properties Of Heme Oxygenase-1.” 2012. Web. 24 Jan 2020.

Vancouver:

Frizell CA. Investigating Potential Novel Angiogenic And Immunogenic Properties Of Heme Oxygenase-1. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Frizell CA. Investigating Potential Novel Angiogenic And Immunogenic Properties Of Heme Oxygenase-1. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Wang, Ying. The role of the hypoxia-inducible factor pathway in bone development and repair.

Degree: PhD, 2007, University of Alabama – Birmingham

Osteogenesis and angiogenesis are tightly coupled during bone formation and repair. Blood vessels not only carry oxygen and nutrients to the developing bone, but also… (more)

Subjects/Keywords: Bone Development  – physiology<; br>; Bone Regeneration  – physiology<; br>; Gene Expression Regulation<; br>; Hypoxia-Inducible Factor 1, alpha Subunit<; br>; Neovascularization, Physiologic<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, Y. (2007). The role of the hypoxia-inducible factor pathway in bone development and repair. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,618

Chicago Manual of Style (16th Edition):

Wang, Ying. “The role of the hypoxia-inducible factor pathway in bone development and repair.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,618.

MLA Handbook (7th Edition):

Wang, Ying. “The role of the hypoxia-inducible factor pathway in bone development and repair.” 2007. Web. 24 Jan 2020.

Vancouver:

Wang Y. The role of the hypoxia-inducible factor pathway in bone development and repair. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,618.

Council of Science Editors:

Wang Y. The role of the hypoxia-inducible factor pathway in bone development and repair. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,618

12. Madi, Ana Paula. Análise microscópica quantitativa da influência do processo inflamatório na angiogênese tumoral.

Degree: Mestrado, Patologia Bucal, 2014, University of São Paulo

Os carcinomas de cabeça e pescoço representam um problema na saúde pública, sendo a oitava causa no mundo de morte por câncer. A taxa de… (more)

Subjects/Keywords: Angiogênese Patológica; Carcinoma de células escamosas; Immunohistochemistry; Imunoistoquímica; Inflamação; Inflammation; Neovascularization Pathologic; Squamous cell carcinoma

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Madi, A. P. (2014). Análise microscópica quantitativa da influência do processo inflamatório na angiogênese tumoral. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/25/25150/tde-07012015-104103/ ;

Chicago Manual of Style (16th Edition):

Madi, Ana Paula. “Análise microscópica quantitativa da influência do processo inflamatório na angiogênese tumoral.” 2014. Masters Thesis, University of São Paulo. Accessed January 24, 2020. http://www.teses.usp.br/teses/disponiveis/25/25150/tde-07012015-104103/ ;.

MLA Handbook (7th Edition):

Madi, Ana Paula. “Análise microscópica quantitativa da influência do processo inflamatório na angiogênese tumoral.” 2014. Web. 24 Jan 2020.

Vancouver:

Madi AP. Análise microscópica quantitativa da influência do processo inflamatório na angiogênese tumoral. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Jan 24]. Available from: http://www.teses.usp.br/teses/disponiveis/25/25150/tde-07012015-104103/ ;.

Council of Science Editors:

Madi AP. Análise microscópica quantitativa da influência do processo inflamatório na angiogênese tumoral. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/25/25150/tde-07012015-104103/ ;

13. Costa, Helena Olegario da. Melanomas extensivo-superficiais regressivos e não-regressivos finos: análise da densidade microvascular utilizando-se os marcadores D2-40 e CD31.

Degree: Mestrado, Dermatologia, 2008, University of São Paulo

O significado prognóstico do fenômeno de regressão espontânea em melanoma, especialmente nas lesões finas, tem sido controverso. Estudos recentes sugerem que regressão extensa e tardia… (more)

Subjects/Keywords: Histologia; Histology; imphangiogenesis; Linfangiogênese; Melanoma; Melanoma; Neovascularização patológica; Pathologic neovascularization; Prognosis; Prognóstico; Regressão neoplásica espontânea; Spontaneous neoplasm regression

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Costa, H. O. d. (2008). Melanomas extensivo-superficiais regressivos e não-regressivos finos: análise da densidade microvascular utilizando-se os marcadores D2-40 e CD31. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5133/tde-14102008-141248/ ;

Chicago Manual of Style (16th Edition):

Costa, Helena Olegario da. “Melanomas extensivo-superficiais regressivos e não-regressivos finos: análise da densidade microvascular utilizando-se os marcadores D2-40 e CD31.” 2008. Masters Thesis, University of São Paulo. Accessed January 24, 2020. http://www.teses.usp.br/teses/disponiveis/5/5133/tde-14102008-141248/ ;.

MLA Handbook (7th Edition):

Costa, Helena Olegario da. “Melanomas extensivo-superficiais regressivos e não-regressivos finos: análise da densidade microvascular utilizando-se os marcadores D2-40 e CD31.” 2008. Web. 24 Jan 2020.

Vancouver:

Costa HOd. Melanomas extensivo-superficiais regressivos e não-regressivos finos: análise da densidade microvascular utilizando-se os marcadores D2-40 e CD31. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2020 Jan 24]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5133/tde-14102008-141248/ ;.

Council of Science Editors:

Costa HOd. Melanomas extensivo-superficiais regressivos e não-regressivos finos: análise da densidade microvascular utilizando-se os marcadores D2-40 e CD31. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/5/5133/tde-14102008-141248/ ;

14. Miyazawa, Marta. Efeito da inibição dos receptores de fator de crescimento endotelial vascular na angiogênese tumoral em carcinomas espinocelulares de cabeça e pescoço.

Degree: PhD, Patologia Bucal, 2007, University of São Paulo

O fator de crescimento endotelial vascular (VEGF) tem sido considerado o principal indutor da angiogênese tumoral por sua ligação a receptores específicos tirosina-quinase presentes nas… (more)

Subjects/Keywords: Angiogênese patológica; Angiogenesis inhibitors; Carcinoma espinocelular; Inibidores da angiogênese; Mouth neoplasms; Neoplasias bucais; Pathologic neovascularization; Squamous cell carcinoma

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Miyazawa, M. (2007). Efeito da inibição dos receptores de fator de crescimento endotelial vascular na angiogênese tumoral em carcinomas espinocelulares de cabeça e pescoço. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/25/25136/tde-16102007-142650/ ;

Chicago Manual of Style (16th Edition):

Miyazawa, Marta. “Efeito da inibição dos receptores de fator de crescimento endotelial vascular na angiogênese tumoral em carcinomas espinocelulares de cabeça e pescoço.” 2007. Doctoral Dissertation, University of São Paulo. Accessed January 24, 2020. http://www.teses.usp.br/teses/disponiveis/25/25136/tde-16102007-142650/ ;.

MLA Handbook (7th Edition):

Miyazawa, Marta. “Efeito da inibição dos receptores de fator de crescimento endotelial vascular na angiogênese tumoral em carcinomas espinocelulares de cabeça e pescoço.” 2007. Web. 24 Jan 2020.

Vancouver:

Miyazawa M. Efeito da inibição dos receptores de fator de crescimento endotelial vascular na angiogênese tumoral em carcinomas espinocelulares de cabeça e pescoço. [Internet] [Doctoral dissertation]. University of São Paulo; 2007. [cited 2020 Jan 24]. Available from: http://www.teses.usp.br/teses/disponiveis/25/25136/tde-16102007-142650/ ;.

Council of Science Editors:

Miyazawa M. Efeito da inibição dos receptores de fator de crescimento endotelial vascular na angiogênese tumoral em carcinomas espinocelulares de cabeça e pescoço. [Doctoral Dissertation]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/25/25136/tde-16102007-142650/ ;

15. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 24 Jan 2020.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956


University of North Carolina – Greensboro

16. Ford, Shavon L. Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure.

Degree: 2009, University of North Carolina – Greensboro

 The imported fire ants, Solenopsis invicta, were introduced into the United States by way of Alabama in the mid 1930s. Their devastating impact on the… (more)

Subjects/Keywords: Cancer – Treatment – Research.; Neovascularization inhibitors.; Neovascularization, Pathologic.; Neovascularization, Physiologic.; Solenopsis invicta.; Fire ants – Research.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ford, S. L. (2009). Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=2530

Chicago Manual of Style (16th Edition):

Ford, Shavon L. “Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure.” 2009. Masters Thesis, University of North Carolina – Greensboro. Accessed January 24, 2020. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=2530.

MLA Handbook (7th Edition):

Ford, Shavon L. “Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure.” 2009. Web. 24 Jan 2020.

Vancouver:

Ford SL. Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2009. [cited 2020 Jan 24]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=2530.

Council of Science Editors:

Ford SL. Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure. [Masters Thesis]. University of North Carolina – Greensboro; 2009. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=2530

17. Ford, Shavon L. Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure.

Degree: 2009, NC Docks

 The imported fire ants, Solenopsis invicta, were introduced into the United States by way of Alabama in the mid 1930s. Their devastating impact on the… (more)

Subjects/Keywords: Cancer $x Treatment $x Research.; Neovascularization inhibitors.; Neovascularization, Pathologic.; Neovascularization, Physiologic.; Solenopsis invicta.; Fire ants $x Research.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ford, S. L. (2009). Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/uncg/f/Ford_uncg_0154M_10176.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ford, Shavon L. “Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure.” 2009. Thesis, NC Docks. Accessed January 24, 2020. http://libres.uncg.edu/ir/uncg/f/Ford_uncg_0154M_10176.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ford, Shavon L. “Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure.” 2009. Web. 24 Jan 2020.

Vancouver:

Ford SL. Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure. [Internet] [Thesis]. NC Docks; 2009. [cited 2020 Jan 24]. Available from: http://libres.uncg.edu/ir/uncg/f/Ford_uncg_0154M_10176.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ford SL. Synthetic and molecular modeling studies of antiangiogenic compounds based on Solenopsin A lead structure. [Thesis]. NC Docks; 2009. Available from: http://libres.uncg.edu/ir/uncg/f/Ford_uncg_0154M_10176.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Jureidini, Ricardo. Análise da densidade da microvasculatura e da expressão do gene p53 no adenocarcinoma pancreático.

Degree: PhD, Cirurgia do Aparelho Digestivo, 2009, University of São Paulo

O adenocarcinoma pancreático é a neoplasia maligna mais comum do pâncreas. A alta taxa de mortalidade deve-se ao diagnóstico tardio e a alta agressividade do… (more)

Subjects/Keywords: Adenocarcinoma; Adenocarcinoma; Neoplasias pancreáticas; Neoplasias pancreáticas/genética; Neovascularização patológica; Neovascularization pathologic; Pancreatic neoplasms; Pancreatic neoplasms/genetics; Proteína supressora de tumor p53/análise; Tumor suppressor protein p53/analysis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jureidini, R. (2009). Análise da densidade da microvasculatura e da expressão do gene p53 no adenocarcinoma pancreático. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5154/tde-25022010-170949/ ;

Chicago Manual of Style (16th Edition):

Jureidini, Ricardo. “Análise da densidade da microvasculatura e da expressão do gene p53 no adenocarcinoma pancreático.” 2009. Doctoral Dissertation, University of São Paulo. Accessed January 24, 2020. http://www.teses.usp.br/teses/disponiveis/5/5154/tde-25022010-170949/ ;.

MLA Handbook (7th Edition):

Jureidini, Ricardo. “Análise da densidade da microvasculatura e da expressão do gene p53 no adenocarcinoma pancreático.” 2009. Web. 24 Jan 2020.

Vancouver:

Jureidini R. Análise da densidade da microvasculatura e da expressão do gene p53 no adenocarcinoma pancreático. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Jan 24]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5154/tde-25022010-170949/ ;.

Council of Science Editors:

Jureidini R. Análise da densidade da microvasculatura e da expressão do gene p53 no adenocarcinoma pancreático. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5154/tde-25022010-170949/ ;


Freie Universität Berlin

19. Dege, Sabrina. Complement factor C5a reduces retinal vaso-obliteration, neovascularisation and number of mononuclear phagocytes in the mouse model of oxygen-induced retinopathy.

Degree: 2017, Freie Universität Berlin

 INTRODUCTION Retinopathy of prematurity (ROP) is a vascular disease of the retina characterized by vaso-obliteration (phase I) and, subsequently, neovascularisation (phase II). In the industrialized… (more)

Subjects/Keywords: mice; animal models; man; retinopathy; blood vessels; neovascularization, pathologic; surface antigens; immunohistochemistry; 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft::630 Landwirtschaft und verwandte Bereiche

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dege, S. (2017). Complement factor C5a reduces retinal vaso-obliteration, neovascularisation and number of mononuclear phagocytes in the mouse model of oxygen-induced retinopathy. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-13046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dege, Sabrina. “Complement factor C5a reduces retinal vaso-obliteration, neovascularisation and number of mononuclear phagocytes in the mouse model of oxygen-induced retinopathy.” 2017. Thesis, Freie Universität Berlin. Accessed January 24, 2020. http://dx.doi.org/10.17169/refubium-13046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dege, Sabrina. “Complement factor C5a reduces retinal vaso-obliteration, neovascularisation and number of mononuclear phagocytes in the mouse model of oxygen-induced retinopathy.” 2017. Web. 24 Jan 2020.

Vancouver:

Dege S. Complement factor C5a reduces retinal vaso-obliteration, neovascularisation and number of mononuclear phagocytes in the mouse model of oxygen-induced retinopathy. [Internet] [Thesis]. Freie Universität Berlin; 2017. [cited 2020 Jan 24]. Available from: http://dx.doi.org/10.17169/refubium-13046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dege S. Complement factor C5a reduces retinal vaso-obliteration, neovascularisation and number of mononuclear phagocytes in the mouse model of oxygen-induced retinopathy. [Thesis]. Freie Universität Berlin; 2017. Available from: http://dx.doi.org/10.17169/refubium-13046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 24 Jan 2020.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

21. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 24 Jan 2020.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

22. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 24 Jan 2020.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

23. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 24 Jan 2020.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

24. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 24 Jan 2020.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

25. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 24 Jan 2020.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

26. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 24 Jan 2020.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

27. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 24 Jan 2020.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

28. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 24 Jan 2020.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

29. Durham, Carolyn G. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.

Degree: PhD, 2010, University of Alabama – Birmingham

The inverse correlation between the industrialization and disease prevalence is termed the "hygiene hypothesis." Supporting this, immunological studies show Th1 cytokines modulate Th2 immune responses.… (more)

Subjects/Keywords: Asthma<; br>; Gastric Mucosa  – metabolism<; br>; Gastritis<; br>; Helicobacter Infections  – metabolism<; br>; Helicobacter felis<; br>; Mucus  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Durham, C. G. (2010). Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,707

Chicago Manual of Style (16th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,707.

MLA Handbook (7th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Web. 24 Jan 2020.

Vancouver:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707.

Council of Science Editors:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707

30. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 24, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 24 Jan 2020.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 24]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

[1] [2] [3] [4] [5] … [412]

.