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You searched for subject:( NRF2). Showing records 1 – 30 of 163 total matches.

[1] [2] [3] [4] [5] [6]

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Penn State University

1. Emmert, Sans Wellington. Role of Nuclear factor-erythroid 2-Related Factor 2 in Glutamate Cysteine Ligase Induction by Organoselenium Compounds.

Degree: PhD, Molecular Medicine, 2008, Penn State University

 ABSTRACT Levels of dietary selenium are inversely associated with cancer risk in humans, and selenium has played a major role in the field of chemoprevention.… (more)

Subjects/Keywords: glutathione; organoselenium; Nrf2

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APA (6th Edition):

Emmert, S. W. (2008). Role of Nuclear factor-erythroid 2-Related Factor 2 in Glutamate Cysteine Ligase Induction by Organoselenium Compounds. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8177

Chicago Manual of Style (16th Edition):

Emmert, Sans Wellington. “Role of Nuclear factor-erythroid 2-Related Factor 2 in Glutamate Cysteine Ligase Induction by Organoselenium Compounds.” 2008. Doctoral Dissertation, Penn State University. Accessed January 29, 2020. https://etda.libraries.psu.edu/catalog/8177.

MLA Handbook (7th Edition):

Emmert, Sans Wellington. “Role of Nuclear factor-erythroid 2-Related Factor 2 in Glutamate Cysteine Ligase Induction by Organoselenium Compounds.” 2008. Web. 29 Jan 2020.

Vancouver:

Emmert SW. Role of Nuclear factor-erythroid 2-Related Factor 2 in Glutamate Cysteine Ligase Induction by Organoselenium Compounds. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2020 Jan 29]. Available from: https://etda.libraries.psu.edu/catalog/8177.

Council of Science Editors:

Emmert SW. Role of Nuclear factor-erythroid 2-Related Factor 2 in Glutamate Cysteine Ligase Induction by Organoselenium Compounds. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8177


Oregon State University

2. Smith, Eric Jonathan. Age-related loss of Nrf2, a novel mechanism for the potential attenuation of xenobiotic detoxification capacity.

Degree: PhD, Biochemistry and Biophysics, 2014, Oregon State University

 Nuclear Factor, Erythroid Derived 2, Like 2 (NFE2L2 or Nrf2) is the primary transcription factor in cellular defense against oxidative and xenobiotic stresses in higher… (more)

Subjects/Keywords: Nrf2; Transcription factors

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APA (6th Edition):

Smith, E. J. (2014). Age-related loss of Nrf2, a novel mechanism for the potential attenuation of xenobiotic detoxification capacity. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/50007

Chicago Manual of Style (16th Edition):

Smith, Eric Jonathan. “Age-related loss of Nrf2, a novel mechanism for the potential attenuation of xenobiotic detoxification capacity.” 2014. Doctoral Dissertation, Oregon State University. Accessed January 29, 2020. http://hdl.handle.net/1957/50007.

MLA Handbook (7th Edition):

Smith, Eric Jonathan. “Age-related loss of Nrf2, a novel mechanism for the potential attenuation of xenobiotic detoxification capacity.” 2014. Web. 29 Jan 2020.

Vancouver:

Smith EJ. Age-related loss of Nrf2, a novel mechanism for the potential attenuation of xenobiotic detoxification capacity. [Internet] [Doctoral dissertation]. Oregon State University; 2014. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/1957/50007.

Council of Science Editors:

Smith EJ. Age-related loss of Nrf2, a novel mechanism for the potential attenuation of xenobiotic detoxification capacity. [Doctoral Dissertation]. Oregon State University; 2014. Available from: http://hdl.handle.net/1957/50007


University of Illinois – Chicago

3. Richardson, Benjamin G. Chemical Biological Probes of the KEAP1/NRF2 Interaction.

Degree: 2018, University of Illinois – Chicago

 Kelch ECH-Associated Protein 1 (KEAP1) is an adaptor protein which functions as a negative regulator to Nuclear factor (erythroid-derived 2)-like 2 (NRF2). KEAP1 complexes NRF2(more)

Subjects/Keywords: KEAP1; NRF2; PROTAC

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APA (6th Edition):

Richardson, B. G. (2018). Chemical Biological Probes of the KEAP1/NRF2 Interaction. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richardson, Benjamin G. “Chemical Biological Probes of the KEAP1/NRF2 Interaction.” 2018. Thesis, University of Illinois – Chicago. Accessed January 29, 2020. http://hdl.handle.net/10027/23327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richardson, Benjamin G. “Chemical Biological Probes of the KEAP1/NRF2 Interaction.” 2018. Web. 29 Jan 2020.

Vancouver:

Richardson BG. Chemical Biological Probes of the KEAP1/NRF2 Interaction. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10027/23327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richardson BG. Chemical Biological Probes of the KEAP1/NRF2 Interaction. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/23327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

4. Robertson, Holly. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.

Degree: PhD, 2019, University of Dundee

 Lung cancer is the leading cause of cancer related mortality worldwide and since the discovery of the important role that NRF2 plays in regulating the… (more)

Subjects/Keywords: NRF2; Lung Cancer

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APA (6th Edition):

Robertson, H. (2019). Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364

Chicago Manual of Style (16th Edition):

Robertson, Holly. “Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.” 2019. Doctoral Dissertation, University of Dundee. Accessed January 29, 2020. https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364.

MLA Handbook (7th Edition):

Robertson, Holly. “Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.” 2019. Web. 29 Jan 2020.

Vancouver:

Robertson H. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. [Internet] [Doctoral dissertation]. University of Dundee; 2019. [cited 2020 Jan 29]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364.

Council of Science Editors:

Robertson H. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. [Doctoral Dissertation]. University of Dundee; 2019. Available from: https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364


University of Ottawa

5. Ao, Hei Sio. Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin .

Degree: 2015, University of Ottawa

 Parkin gene is associated with the development of autosomal recessive juvenile parkinsonism (Kitada et al., 1998) which is a common form of familial Parkinson’s disease… (more)

Subjects/Keywords: Parkinson's disease; Parkin; Nrf2

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APA (6th Edition):

Ao, H. S. (2015). Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/33368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ao, Hei Sio. “Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin .” 2015. Thesis, University of Ottawa. Accessed January 29, 2020. http://hdl.handle.net/10393/33368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ao, Hei Sio. “Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin .” 2015. Web. 29 Jan 2020.

Vancouver:

Ao HS. Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin . [Internet] [Thesis]. University of Ottawa; 2015. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10393/33368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ao HS. Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin . [Thesis]. University of Ottawa; 2015. Available from: http://hdl.handle.net/10393/33368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

6. Sun, Zhiyuan. Mass Spectrometric Studies of KEAP1-NRF2 Binding Interactions.

Degree: 2012, University of Illinois – Chicago

 Chemoprevention is an active cancer preventive strategy to inhibit, delay or reverse human carcinogenesis using naturally occurring or synthetic chemical agents. In recent years, it… (more)

Subjects/Keywords: Mass Spectrometry; Ultrafiltration; KEAP1; NRF2

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APA (6th Edition):

Sun, Z. (2012). Mass Spectrometric Studies of KEAP1-NRF2 Binding Interactions. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9103

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sun, Zhiyuan. “Mass Spectrometric Studies of KEAP1-NRF2 Binding Interactions.” 2012. Thesis, University of Illinois – Chicago. Accessed January 29, 2020. http://hdl.handle.net/10027/9103.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sun, Zhiyuan. “Mass Spectrometric Studies of KEAP1-NRF2 Binding Interactions.” 2012. Web. 29 Jan 2020.

Vancouver:

Sun Z. Mass Spectrometric Studies of KEAP1-NRF2 Binding Interactions. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10027/9103.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sun Z. Mass Spectrometric Studies of KEAP1-NRF2 Binding Interactions. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9103

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Ebisine, Kimimuepigha. Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer.

Degree: PhD, 2019, University of Dundee

 Cancer is one of the foremost causes of death worldwide with about 14.1 million new incidences and 8.2 cancer related deaths occurring globally. NF-E2 p45-related… (more)

Subjects/Keywords: Nrf2; Keap1; beta-TrCP

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APA (6th Edition):

Ebisine, K. (2019). Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/20338051-4e92-417e-aeb1-4cd3dc86eda4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763002

Chicago Manual of Style (16th Edition):

Ebisine, Kimimuepigha. “Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer.” 2019. Doctoral Dissertation, University of Dundee. Accessed January 29, 2020. https://discovery.dundee.ac.uk/en/studentTheses/20338051-4e92-417e-aeb1-4cd3dc86eda4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763002.

MLA Handbook (7th Edition):

Ebisine, Kimimuepigha. “Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer.” 2019. Web. 29 Jan 2020.

Vancouver:

Ebisine K. Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer. [Internet] [Doctoral dissertation]. University of Dundee; 2019. [cited 2020 Jan 29]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/20338051-4e92-417e-aeb1-4cd3dc86eda4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763002.

Council of Science Editors:

Ebisine K. Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer. [Doctoral Dissertation]. University of Dundee; 2019. Available from: https://discovery.dundee.ac.uk/en/studentTheses/20338051-4e92-417e-aeb1-4cd3dc86eda4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763002


Colorado State University

8. Laurin, Jaime L. Evaluation of a novel phytochemical Nrf2 activator on cytoprotective gene expression and proteostasis in vivo.

Degree: MS(M.S.), Health and Exercise Science, 2017, Colorado State University

 Aging is associated with increases in oxidative stress. Redox imbalance occurs when chronic production of reactive oxygen species (ROS) exceeds the capacity of antioxidant enzymes… (more)

Subjects/Keywords: Oxidative Stress; Proteostasis; Phytochemicals; Nrf2

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APA (6th Edition):

Laurin, J. L. (2017). Evaluation of a novel phytochemical Nrf2 activator on cytoprotective gene expression and proteostasis in vivo. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/183999

Chicago Manual of Style (16th Edition):

Laurin, Jaime L. “Evaluation of a novel phytochemical Nrf2 activator on cytoprotective gene expression and proteostasis in vivo.” 2017. Masters Thesis, Colorado State University. Accessed January 29, 2020. http://hdl.handle.net/10217/183999.

MLA Handbook (7th Edition):

Laurin, Jaime L. “Evaluation of a novel phytochemical Nrf2 activator on cytoprotective gene expression and proteostasis in vivo.” 2017. Web. 29 Jan 2020.

Vancouver:

Laurin JL. Evaluation of a novel phytochemical Nrf2 activator on cytoprotective gene expression and proteostasis in vivo. [Internet] [Masters thesis]. Colorado State University; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10217/183999.

Council of Science Editors:

Laurin JL. Evaluation of a novel phytochemical Nrf2 activator on cytoprotective gene expression and proteostasis in vivo. [Masters Thesis]. Colorado State University; 2017. Available from: http://hdl.handle.net/10217/183999

9. Santos, Ana Luísa. Avaliação da proteção conferida pela via de sinalização do Nrf2 num modelo experimental de sobrecarga de ferro in vivo.

Degree: 2012, Instituto Politécnico do Porto

O ferro é encontrado em praticamente todos os seres vivos, sendo um cofator para proteínas que desempenham funções essenciais à vida. Nos mamíferos, a maioria… (more)

Subjects/Keywords: Ferro; Dieta; Nrf2; Fígado; Hemocromatose; Iron; Diet; Nrf2; Liver; Hemochromatosis

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APA (6th Edition):

Santos, A. L. (2012). Avaliação da proteção conferida pela via de sinalização do Nrf2 num modelo experimental de sobrecarga de ferro in vivo. (Thesis). Instituto Politécnico do Porto. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:recipp.ipp.pt:10400.22/1122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, Ana Luísa. “Avaliação da proteção conferida pela via de sinalização do Nrf2 num modelo experimental de sobrecarga de ferro in vivo.” 2012. Thesis, Instituto Politécnico do Porto. Accessed January 29, 2020. http://www.rcaap.pt/detail.jsp?id=oai:recipp.ipp.pt:10400.22/1122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, Ana Luísa. “Avaliação da proteção conferida pela via de sinalização do Nrf2 num modelo experimental de sobrecarga de ferro in vivo.” 2012. Web. 29 Jan 2020.

Vancouver:

Santos AL. Avaliação da proteção conferida pela via de sinalização do Nrf2 num modelo experimental de sobrecarga de ferro in vivo. [Internet] [Thesis]. Instituto Politécnico do Porto; 2012. [cited 2020 Jan 29]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:recipp.ipp.pt:10400.22/1122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos AL. Avaliação da proteção conferida pela via de sinalização do Nrf2 num modelo experimental de sobrecarga de ferro in vivo. [Thesis]. Instituto Politécnico do Porto; 2012. Available from: http://www.rcaap.pt/detail.jsp?id=oai:recipp.ipp.pt:10400.22/1122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Genard, Romain. Rôle du facteur de transcription Nrf2 dans l'immunomodulation induit par les adjuvants vaccinaux : Role of the transcription factor Nrf2 in immunomodulation induce by vaccine adjuvants.

Degree: Docteur es, Toxicologie, 2015, Paris Saclay

Les adjuvants vaccinaux permettent d’augmenter la réponse immunitaire dirigée contre un antigène donné. Certains de ces adjuvants miment des signaux de danger, tels que des… (more)

Subjects/Keywords: Nrf2; Adjuvants; Vaccin; Cellule Dendritique; Nrf2; Adjuvants; Vaccine; Dendritic cell

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APA (6th Edition):

Genard, R. (2015). Rôle du facteur de transcription Nrf2 dans l'immunomodulation induit par les adjuvants vaccinaux : Role of the transcription factor Nrf2 in immunomodulation induce by vaccine adjuvants. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2015SACLS230

Chicago Manual of Style (16th Edition):

Genard, Romain. “Rôle du facteur de transcription Nrf2 dans l'immunomodulation induit par les adjuvants vaccinaux : Role of the transcription factor Nrf2 in immunomodulation induce by vaccine adjuvants.” 2015. Doctoral Dissertation, Paris Saclay. Accessed January 29, 2020. http://www.theses.fr/2015SACLS230.

MLA Handbook (7th Edition):

Genard, Romain. “Rôle du facteur de transcription Nrf2 dans l'immunomodulation induit par les adjuvants vaccinaux : Role of the transcription factor Nrf2 in immunomodulation induce by vaccine adjuvants.” 2015. Web. 29 Jan 2020.

Vancouver:

Genard R. Rôle du facteur de transcription Nrf2 dans l'immunomodulation induit par les adjuvants vaccinaux : Role of the transcription factor Nrf2 in immunomodulation induce by vaccine adjuvants. [Internet] [Doctoral dissertation]. Paris Saclay; 2015. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2015SACLS230.

Council of Science Editors:

Genard R. Rôle du facteur de transcription Nrf2 dans l'immunomodulation induit par les adjuvants vaccinaux : Role of the transcription factor Nrf2 in immunomodulation induce by vaccine adjuvants. [Doctoral Dissertation]. Paris Saclay; 2015. Available from: http://www.theses.fr/2015SACLS230


University of Rochester

11. Hochmuth, Christine E. (1982 - ). CncC/Keap1 signaling pathway in the regulation of intestinal stem cells in Drosophila.

Degree: PhD, 2012, University of Rochester

 Organisms are faced with the challenge of maintaining tissue homeostasis despite various environmental and intracellular assaults that damage cells. Programmed cell death can eliminate cells… (more)

Subjects/Keywords: Stem cells; Drosophila; Nrf2; Redox; ROS

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APA (6th Edition):

Hochmuth, C. E. (. -. ). (2012). CncC/Keap1 signaling pathway in the regulation of intestinal stem cells in Drosophila. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/19263

Chicago Manual of Style (16th Edition):

Hochmuth, Christine E (1982 - ). “CncC/Keap1 signaling pathway in the regulation of intestinal stem cells in Drosophila.” 2012. Doctoral Dissertation, University of Rochester. Accessed January 29, 2020. http://hdl.handle.net/1802/19263.

MLA Handbook (7th Edition):

Hochmuth, Christine E (1982 - ). “CncC/Keap1 signaling pathway in the regulation of intestinal stem cells in Drosophila.” 2012. Web. 29 Jan 2020.

Vancouver:

Hochmuth CE(-). CncC/Keap1 signaling pathway in the regulation of intestinal stem cells in Drosophila. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/1802/19263.

Council of Science Editors:

Hochmuth CE(-). CncC/Keap1 signaling pathway in the regulation of intestinal stem cells in Drosophila. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/19263


University of Florida

12. Tang, Lanlan. Characterization of the Roles of WDR-23/SKN-1 in Stress Resistance/Longevity and Growth/Reproduction.

Degree: PhD, Zoology - Biology, 2015, University of Florida

Subjects/Keywords: aging; detoxification; nrf2

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APA (6th Edition):

Tang, L. (2015). Characterization of the Roles of WDR-23/SKN-1 in Stress Resistance/Longevity and Growth/Reproduction. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0049526

Chicago Manual of Style (16th Edition):

Tang, Lanlan. “Characterization of the Roles of WDR-23/SKN-1 in Stress Resistance/Longevity and Growth/Reproduction.” 2015. Doctoral Dissertation, University of Florida. Accessed January 29, 2020. http://ufdc.ufl.edu/UFE0049526.

MLA Handbook (7th Edition):

Tang, Lanlan. “Characterization of the Roles of WDR-23/SKN-1 in Stress Resistance/Longevity and Growth/Reproduction.” 2015. Web. 29 Jan 2020.

Vancouver:

Tang L. Characterization of the Roles of WDR-23/SKN-1 in Stress Resistance/Longevity and Growth/Reproduction. [Internet] [Doctoral dissertation]. University of Florida; 2015. [cited 2020 Jan 29]. Available from: http://ufdc.ufl.edu/UFE0049526.

Council of Science Editors:

Tang L. Characterization of the Roles of WDR-23/SKN-1 in Stress Resistance/Longevity and Growth/Reproduction. [Doctoral Dissertation]. University of Florida; 2015. Available from: http://ufdc.ufl.edu/UFE0049526


University of Sydney

13. Anzovino, Amy Christine. The Down‐Regulation of Frataxin Compromises Nrf2 Signalling and Redox Homeostasis to Promote Cardiac Remodelling .

Degree: 2017, University of Sydney

 Oxidative stress is a key contributor to the neurodegeneration in Friedreich’s ataxia (FA). While studies in neuronal models of FA have reported a diminished antioxidant… (more)

Subjects/Keywords: lMitochondria; Cardiomyopathy; Frataxin; Oxidative Stress; Nrf2

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APA (6th Edition):

Anzovino, A. C. (2017). The Down‐Regulation of Frataxin Compromises Nrf2 Signalling and Redox Homeostasis to Promote Cardiac Remodelling . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anzovino, Amy Christine. “The Down‐Regulation of Frataxin Compromises Nrf2 Signalling and Redox Homeostasis to Promote Cardiac Remodelling .” 2017. Thesis, University of Sydney. Accessed January 29, 2020. http://hdl.handle.net/2123/17576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anzovino, Amy Christine. “The Down‐Regulation of Frataxin Compromises Nrf2 Signalling and Redox Homeostasis to Promote Cardiac Remodelling .” 2017. Web. 29 Jan 2020.

Vancouver:

Anzovino AC. The Down‐Regulation of Frataxin Compromises Nrf2 Signalling and Redox Homeostasis to Promote Cardiac Remodelling . [Internet] [Thesis]. University of Sydney; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2123/17576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anzovino AC. The Down‐Regulation of Frataxin Compromises Nrf2 Signalling and Redox Homeostasis to Promote Cardiac Remodelling . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Montana Tech

14. Karikari, Akua Afriyie. DIFFERENTIATION-INDUCED CHANGES IN ANTIOXIDANT CAPACITY AND ANTIOXIDANT RESPONSE IN SH-SY5Y CELLS: ROLE OF NRF2.

Degree: MS, 2013, Montana Tech

 Organisms are exposed to reactive oxygen species from internal metabolism and environmental toxicant exposure, which have been linked to the initiation and progression of many… (more)

Subjects/Keywords: Atioxidants; Nrf2; Oxidative stress; SH-SY5Y cells

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APA (6th Edition):

Karikari, A. A. (2013). DIFFERENTIATION-INDUCED CHANGES IN ANTIOXIDANT CAPACITY AND ANTIOXIDANT RESPONSE IN SH-SY5Y CELLS: ROLE OF NRF2. (Masters Thesis). Montana Tech. Retrieved from https://scholarworks.umt.edu/etd/321

Chicago Manual of Style (16th Edition):

Karikari, Akua Afriyie. “DIFFERENTIATION-INDUCED CHANGES IN ANTIOXIDANT CAPACITY AND ANTIOXIDANT RESPONSE IN SH-SY5Y CELLS: ROLE OF NRF2.” 2013. Masters Thesis, Montana Tech. Accessed January 29, 2020. https://scholarworks.umt.edu/etd/321.

MLA Handbook (7th Edition):

Karikari, Akua Afriyie. “DIFFERENTIATION-INDUCED CHANGES IN ANTIOXIDANT CAPACITY AND ANTIOXIDANT RESPONSE IN SH-SY5Y CELLS: ROLE OF NRF2.” 2013. Web. 29 Jan 2020.

Vancouver:

Karikari AA. DIFFERENTIATION-INDUCED CHANGES IN ANTIOXIDANT CAPACITY AND ANTIOXIDANT RESPONSE IN SH-SY5Y CELLS: ROLE OF NRF2. [Internet] [Masters thesis]. Montana Tech; 2013. [cited 2020 Jan 29]. Available from: https://scholarworks.umt.edu/etd/321.

Council of Science Editors:

Karikari AA. DIFFERENTIATION-INDUCED CHANGES IN ANTIOXIDANT CAPACITY AND ANTIOXIDANT RESPONSE IN SH-SY5Y CELLS: ROLE OF NRF2. [Masters Thesis]. Montana Tech; 2013. Available from: https://scholarworks.umt.edu/etd/321


University of Arizona

15. Villeneuve, Nicole Frances. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .

Degree: 2011, University of Arizona

Nrf2 (NF-E2-related factor 2) is a transcription factor that regulates a battery of downstream genes that contain an antioxidant response element (ARE) in their promoters,… (more)

Subjects/Keywords: cinnamic aldehyde; Keap1; Nrf2; oridonin; ubiquitination; USP15

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APA (6th Edition):

Villeneuve, N. F. (2011). Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/145720

Chicago Manual of Style (16th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Doctoral Dissertation, University of Arizona. Accessed January 29, 2020. http://hdl.handle.net/10150/145720.

MLA Handbook (7th Edition):

Villeneuve, Nicole Frances. “Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response .” 2011. Web. 29 Jan 2020.

Vancouver:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Internet] [Doctoral dissertation]. University of Arizona; 2011. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10150/145720.

Council of Science Editors:

Villeneuve NF. Mechanistic Study of USP15-Dependent Deubiquitination and Characterization of Natural Compounds that Modulate the Nrf2-Keap1 Antioxidant Response . [Doctoral Dissertation]. University of Arizona; 2011. Available from: http://hdl.handle.net/10150/145720


University of Arizona

16. Kerins, Michael John. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .

Degree: 2019, University of Arizona

 The deadly kidney cancers associated with hereditary leiomyomatosis and renal cell cancer (HLRCC) show activation of the nuclear factor (erythroid 2)-like 2 (NFE2L2, NRF2) transcription… (more)

Subjects/Keywords: Cancer; ferroptosis; HLRCC; iron; NRF2; signaling

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APA (6th Edition):

Kerins, M. J. (2019). Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/633229

Chicago Manual of Style (16th Edition):

Kerins, Michael John. “Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .” 2019. Doctoral Dissertation, University of Arizona. Accessed January 29, 2020. http://hdl.handle.net/10150/633229.

MLA Handbook (7th Edition):

Kerins, Michael John. “Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome .” 2019. Web. 29 Jan 2020.

Vancouver:

Kerins MJ. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . [Internet] [Doctoral dissertation]. University of Arizona; 2019. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10150/633229.

Council of Science Editors:

Kerins MJ. Ironing Out Roles and Regulation of NRF2 in a Hereditary Cancer Syndrome . [Doctoral Dissertation]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/633229


Boston University

17. Lynch, Andrew John. Ligand selectivity: binding at the protein-protein interface of Keap1 and NEMO.

Degree: PhD, Chemistry, 2016, Boston University

 This dissertation comprises identifying the structural determinants of binding selectivity as demonstrated in three systems. The first system involves the structure determination of Keap1-small molecule… (more)

Subjects/Keywords: Biochemistry; Fbdd; Keap1; NEMO; Nrf2; Structure

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APA (6th Edition):

Lynch, A. J. (2016). Ligand selectivity: binding at the protein-protein interface of Keap1 and NEMO. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14550

Chicago Manual of Style (16th Edition):

Lynch, Andrew John. “Ligand selectivity: binding at the protein-protein interface of Keap1 and NEMO.” 2016. Doctoral Dissertation, Boston University. Accessed January 29, 2020. http://hdl.handle.net/2144/14550.

MLA Handbook (7th Edition):

Lynch, Andrew John. “Ligand selectivity: binding at the protein-protein interface of Keap1 and NEMO.” 2016. Web. 29 Jan 2020.

Vancouver:

Lynch AJ. Ligand selectivity: binding at the protein-protein interface of Keap1 and NEMO. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2144/14550.

Council of Science Editors:

Lynch AJ. Ligand selectivity: binding at the protein-protein interface of Keap1 and NEMO. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/14550


Universitat Rovira i Virgili

18. Lázaro López, Iolanda. Regulació de fabp4 depenent de nrf2 en macròfags.

Degree: Departament de Medicina i Cirurgia, 2010, Universitat Rovira i Virgili

 Macrophages play a crucial role in the development of atherosclerosis. It has been shown that oxidized LDL (oxLDL) induces adipocyte fatty acid-binding protein (FABP4) in… (more)

Subjects/Keywords: ASTEROSCLEROSI; MACRÒFAG; OXIDACIÓ; Nrf2; FABP4; 577; 61

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APA (6th Edition):

Lázaro López, I. (2010). Regulació de fabp4 depenent de nrf2 en macròfags. (Thesis). Universitat Rovira i Virgili. Retrieved from http://hdl.handle.net/10803/8886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lázaro López, Iolanda. “Regulació de fabp4 depenent de nrf2 en macròfags.” 2010. Thesis, Universitat Rovira i Virgili. Accessed January 29, 2020. http://hdl.handle.net/10803/8886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lázaro López, Iolanda. “Regulació de fabp4 depenent de nrf2 en macròfags.” 2010. Web. 29 Jan 2020.

Vancouver:

Lázaro López I. Regulació de fabp4 depenent de nrf2 en macròfags. [Internet] [Thesis]. Universitat Rovira i Virgili; 2010. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/10803/8886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lázaro López I. Regulació de fabp4 depenent de nrf2 en macròfags. [Thesis]. Universitat Rovira i Virgili; 2010. Available from: http://hdl.handle.net/10803/8886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

19. Kim, Maya Hwewon. Glutaredoxin-1 regulates the Keap1-Nrf2 pathway.

Degree: MS, Pathology, 2017, Boston University

 PURPOSE: The Nrf2/Keap1/ARE pathway is a major regulator of cytoprotective responses to oxidants. Gluatredoxin-1 (Glrx-1), a small thiol transferase removes glutathione (GSH) adducts from proteins… (more)

Subjects/Keywords: Medicine; Glrx; GSH; Keap1; NAFLD; NASH; Nrf2

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APA (6th Edition):

Kim, M. H. (2017). Glutaredoxin-1 regulates the Keap1-Nrf2 pathway. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/26675

Chicago Manual of Style (16th Edition):

Kim, Maya Hwewon. “Glutaredoxin-1 regulates the Keap1-Nrf2 pathway.” 2017. Masters Thesis, Boston University. Accessed January 29, 2020. http://hdl.handle.net/2144/26675.

MLA Handbook (7th Edition):

Kim, Maya Hwewon. “Glutaredoxin-1 regulates the Keap1-Nrf2 pathway.” 2017. Web. 29 Jan 2020.

Vancouver:

Kim MH. Glutaredoxin-1 regulates the Keap1-Nrf2 pathway. [Internet] [Masters thesis]. Boston University; 2017. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/2144/26675.

Council of Science Editors:

Kim MH. Glutaredoxin-1 regulates the Keap1-Nrf2 pathway. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/26675


University of Dundee

20. Liddell, Mary Katherine. Nrf2 in metabolic related inflammation in the brain.

Degree: PhD, 2015, University of Dundee

 Novel approaches are required to address Alzheimer’s disease (AD) in our ageing population, with recent interest focused on inflammation and oxidative stress (OS). Disruption of… (more)

Subjects/Keywords: 612.8; Nrf2; Alzheimer's disease; Obesity; APP

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APA (6th Edition):

Liddell, M. K. (2015). Nrf2 in metabolic related inflammation in the brain. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/071665a4-1d42-450c-ad49-854ead5d6814 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679086

Chicago Manual of Style (16th Edition):

Liddell, Mary Katherine. “Nrf2 in metabolic related inflammation in the brain.” 2015. Doctoral Dissertation, University of Dundee. Accessed January 29, 2020. https://discovery.dundee.ac.uk/en/studentTheses/071665a4-1d42-450c-ad49-854ead5d6814 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679086.

MLA Handbook (7th Edition):

Liddell, Mary Katherine. “Nrf2 in metabolic related inflammation in the brain.” 2015. Web. 29 Jan 2020.

Vancouver:

Liddell MK. Nrf2 in metabolic related inflammation in the brain. [Internet] [Doctoral dissertation]. University of Dundee; 2015. [cited 2020 Jan 29]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/071665a4-1d42-450c-ad49-854ead5d6814 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679086.

Council of Science Editors:

Liddell MK. Nrf2 in metabolic related inflammation in the brain. [Doctoral Dissertation]. University of Dundee; 2015. Available from: https://discovery.dundee.ac.uk/en/studentTheses/071665a4-1d42-450c-ad49-854ead5d6814 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679086


University of Guelph

21. Morgan, Larry. The Role of Cytochrome P450 2A5 During Endoplasmic Reticulum Stress .

Degree: 2014, University of Guelph

 Cytochrome P450 2A5 (CYP2A5) is a murine orthologue of human CYP2A6 and is predominantly found within the endoplasmic reticulum (ER) of the liver. CYP2A5 differs… (more)

Subjects/Keywords: CYP2A5; ER Stress; Hepatocytes; Nrf2; Heme; Bilirubin

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APA (6th Edition):

Morgan, L. (2014). The Role of Cytochrome P450 2A5 During Endoplasmic Reticulum Stress . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7827

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morgan, Larry. “The Role of Cytochrome P450 2A5 During Endoplasmic Reticulum Stress .” 2014. Thesis, University of Guelph. Accessed January 29, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7827.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morgan, Larry. “The Role of Cytochrome P450 2A5 During Endoplasmic Reticulum Stress .” 2014. Web. 29 Jan 2020.

Vancouver:

Morgan L. The Role of Cytochrome P450 2A5 During Endoplasmic Reticulum Stress . [Internet] [Thesis]. University of Guelph; 2014. [cited 2020 Jan 29]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7827.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morgan L. The Role of Cytochrome P450 2A5 During Endoplasmic Reticulum Stress . [Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7827

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

22. Agro, Lauren Alicia. Increasing Chemosensitivity in Colorectal Cancer.

Degree: PhD, 2019, University of Toronto

 Despite being one of the most genetically understood cancers, colorectal cancer is still one of the leading causes of death due to cancer. Here we… (more)

Subjects/Keywords: aggregation; antioxidant; cancer; colorectal; Nrf2; p53; 0379

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APA (6th Edition):

Agro, L. A. (2019). Increasing Chemosensitivity in Colorectal Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/97022

Chicago Manual of Style (16th Edition):

Agro, Lauren Alicia. “Increasing Chemosensitivity in Colorectal Cancer.” 2019. Doctoral Dissertation, University of Toronto. Accessed January 29, 2020. http://hdl.handle.net/1807/97022.

MLA Handbook (7th Edition):

Agro, Lauren Alicia. “Increasing Chemosensitivity in Colorectal Cancer.” 2019. Web. 29 Jan 2020.

Vancouver:

Agro LA. Increasing Chemosensitivity in Colorectal Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2019. [cited 2020 Jan 29]. Available from: http://hdl.handle.net/1807/97022.

Council of Science Editors:

Agro LA. Increasing Chemosensitivity in Colorectal Cancer. [Doctoral Dissertation]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/97022

23. Surya, Reggie. Red meat and colorectal cancer : lipid peroxidation-derived products induce different apoptosis, autophagy and Nrf2-related responses in normal and preneoplastic colon cells : Cancer colorectal et viande rouge : les produits dérivés de la lipoperoxydation induisent différentes réponses d'apoptose, d'autophagie et de Nrf2 dans des cellules coliques normales et prénéoplasiques.

Degree: Docteur es, Pathologie, toxicologie, génétique et nutrition, 2016, Université Toulouse III – Paul Sabatier

La viande rouge est un facteur de risque du cancer colorectal, considérée comme probablement cancérigène chez l'Homme. Le lien entre la viande rouge et le… (more)

Subjects/Keywords: Cancer colorectal; Fer héminique; Lipoperoxydation; 4-hydroxynonénal; Nrf2; Autophagie; Colorectal cancer; Heme iron; Lipid peroxidation; 4-hydroxynonenal; Nrf2; Autophagy

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APA (6th Edition):

Surya, R. (2016). Red meat and colorectal cancer : lipid peroxidation-derived products induce different apoptosis, autophagy and Nrf2-related responses in normal and preneoplastic colon cells : Cancer colorectal et viande rouge : les produits dérivés de la lipoperoxydation induisent différentes réponses d'apoptose, d'autophagie et de Nrf2 dans des cellules coliques normales et prénéoplasiques. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2016TOU30082

Chicago Manual of Style (16th Edition):

Surya, Reggie. “Red meat and colorectal cancer : lipid peroxidation-derived products induce different apoptosis, autophagy and Nrf2-related responses in normal and preneoplastic colon cells : Cancer colorectal et viande rouge : les produits dérivés de la lipoperoxydation induisent différentes réponses d'apoptose, d'autophagie et de Nrf2 dans des cellules coliques normales et prénéoplasiques.” 2016. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 29, 2020. http://www.theses.fr/2016TOU30082.

MLA Handbook (7th Edition):

Surya, Reggie. “Red meat and colorectal cancer : lipid peroxidation-derived products induce different apoptosis, autophagy and Nrf2-related responses in normal and preneoplastic colon cells : Cancer colorectal et viande rouge : les produits dérivés de la lipoperoxydation induisent différentes réponses d'apoptose, d'autophagie et de Nrf2 dans des cellules coliques normales et prénéoplasiques.” 2016. Web. 29 Jan 2020.

Vancouver:

Surya R. Red meat and colorectal cancer : lipid peroxidation-derived products induce different apoptosis, autophagy and Nrf2-related responses in normal and preneoplastic colon cells : Cancer colorectal et viande rouge : les produits dérivés de la lipoperoxydation induisent différentes réponses d'apoptose, d'autophagie et de Nrf2 dans des cellules coliques normales et prénéoplasiques. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2016. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2016TOU30082.

Council of Science Editors:

Surya R. Red meat and colorectal cancer : lipid peroxidation-derived products induce different apoptosis, autophagy and Nrf2-related responses in normal and preneoplastic colon cells : Cancer colorectal et viande rouge : les produits dérivés de la lipoperoxydation induisent différentes réponses d'apoptose, d'autophagie et de Nrf2 dans des cellules coliques normales et prénéoplasiques. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2016. Available from: http://www.theses.fr/2016TOU30082

24. Cabrié, Aimeric. Coopération entre les isoformes TAp73 et la signalisation TGF-β dans la régulation de l'expression de la NO Synthase inductible : TAp73 Isoforms and TGF-β Signaling Cooperate to Suppress Inducible Nitric Oxide Synthase Expression.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Paris Saclay

Le monoxyde d’azote (NO) est une molécule gazeuse synthétisée par les NO Synthases à partir de L-arginine. NO est une puissante molécule de signalisation dans… (more)

Subjects/Keywords: Monoxyde d’azote (NO); Inos; P73; TGF-β; Nrf2; Nitric oxide (NO); Inos; P73; TGF-β; Nrf2

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APA (6th Edition):

Cabrié, A. (2017). Coopération entre les isoformes TAp73 et la signalisation TGF-β dans la régulation de l'expression de la NO Synthase inductible : TAp73 Isoforms and TGF-β Signaling Cooperate to Suppress Inducible Nitric Oxide Synthase Expression. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2017SACLS397

Chicago Manual of Style (16th Edition):

Cabrié, Aimeric. “Coopération entre les isoformes TAp73 et la signalisation TGF-β dans la régulation de l'expression de la NO Synthase inductible : TAp73 Isoforms and TGF-β Signaling Cooperate to Suppress Inducible Nitric Oxide Synthase Expression.” 2017. Doctoral Dissertation, Paris Saclay. Accessed January 29, 2020. http://www.theses.fr/2017SACLS397.

MLA Handbook (7th Edition):

Cabrié, Aimeric. “Coopération entre les isoformes TAp73 et la signalisation TGF-β dans la régulation de l'expression de la NO Synthase inductible : TAp73 Isoforms and TGF-β Signaling Cooperate to Suppress Inducible Nitric Oxide Synthase Expression.” 2017. Web. 29 Jan 2020.

Vancouver:

Cabrié A. Coopération entre les isoformes TAp73 et la signalisation TGF-β dans la régulation de l'expression de la NO Synthase inductible : TAp73 Isoforms and TGF-β Signaling Cooperate to Suppress Inducible Nitric Oxide Synthase Expression. [Internet] [Doctoral dissertation]. Paris Saclay; 2017. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2017SACLS397.

Council of Science Editors:

Cabrié A. Coopération entre les isoformes TAp73 et la signalisation TGF-β dans la régulation de l'expression de la NO Synthase inductible : TAp73 Isoforms and TGF-β Signaling Cooperate to Suppress Inducible Nitric Oxide Synthase Expression. [Doctoral Dissertation]. Paris Saclay; 2017. Available from: http://www.theses.fr/2017SACLS397

25. Helou, Doumet. Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée : The role of Nrf2 transcription factor in the regulation of neutrophil functions in vitro and in cutaneous allergy.

Degree: Docteur es, Immunologie, 2018, Paris Saclay

Les neutrophiles constituent une première ligne de défense contre les agents infectieux. En revanche, leur activation incontrôlée peut exacerber certaines pathologies inflammatoires telles que les… (more)

Subjects/Keywords: Neutrophile; Hypersensibilité de contact; Allergie; Modèle murin; Stress oxydant; Nrf2; Neutrophil; Contact hypersensitivity; Allergy; Murine model; Oxidative stress; Nrf2

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APA (6th Edition):

Helou, D. (2018). Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée : The role of Nrf2 transcription factor in the regulation of neutrophil functions in vitro and in cutaneous allergy. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2018SACLS305

Chicago Manual of Style (16th Edition):

Helou, Doumet. “Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée : The role of Nrf2 transcription factor in the regulation of neutrophil functions in vitro and in cutaneous allergy.” 2018. Doctoral Dissertation, Paris Saclay. Accessed January 29, 2020. http://www.theses.fr/2018SACLS305.

MLA Handbook (7th Edition):

Helou, Doumet. “Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée : The role of Nrf2 transcription factor in the regulation of neutrophil functions in vitro and in cutaneous allergy.” 2018. Web. 29 Jan 2020.

Vancouver:

Helou D. Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée : The role of Nrf2 transcription factor in the regulation of neutrophil functions in vitro and in cutaneous allergy. [Internet] [Doctoral dissertation]. Paris Saclay; 2018. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2018SACLS305.

Council of Science Editors:

Helou D. Rôle du facteur de transcription Nrf2 dans la régulation des fonctions du neutrophile in vitro et dans l’allergie cutanée : The role of Nrf2 transcription factor in the regulation of neutrophil functions in vitro and in cutaneous allergy. [Doctoral Dissertation]. Paris Saclay; 2018. Available from: http://www.theses.fr/2018SACLS305


Université Paris-Sud – Paris XI

26. Poisson-Moreau de Lizorieux, Clémentine. Rôle du stress oxydant au niveau hépatique et rénal dans la toxicité de l’uranium après exposition chronique : Role of oxidative stress in liver and kidney in uranium toxicity after chronic exposure.

Degree: Docteur es, Toxicologie, 2013, Université Paris-Sud – Paris XI

L’Uranium (U) est un métal lourd radioactif dispersé dans l’environnement. Du fait de cette présence naturelle mais aussi de ses applications civiles et militaires, la… (more)

Subjects/Keywords: Uranium; Contamination chronique; Nrf2; Système pro/anti-oxydant; Foie; Rein; Uranium; Chronic exposure; Nrf2; Pro/anti-oxidaitve system; Liver; Kidney

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Poisson-Moreau de Lizorieux, C. (2013). Rôle du stress oxydant au niveau hépatique et rénal dans la toxicité de l’uranium après exposition chronique : Role of oxidative stress in liver and kidney in uranium toxicity after chronic exposure. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA114849

Chicago Manual of Style (16th Edition):

Poisson-Moreau de Lizorieux, Clémentine. “Rôle du stress oxydant au niveau hépatique et rénal dans la toxicité de l’uranium après exposition chronique : Role of oxidative stress in liver and kidney in uranium toxicity after chronic exposure.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed January 29, 2020. http://www.theses.fr/2013PA114849.

MLA Handbook (7th Edition):

Poisson-Moreau de Lizorieux, Clémentine. “Rôle du stress oxydant au niveau hépatique et rénal dans la toxicité de l’uranium après exposition chronique : Role of oxidative stress in liver and kidney in uranium toxicity after chronic exposure.” 2013. Web. 29 Jan 2020.

Vancouver:

Poisson-Moreau de Lizorieux C. Rôle du stress oxydant au niveau hépatique et rénal dans la toxicité de l’uranium après exposition chronique : Role of oxidative stress in liver and kidney in uranium toxicity after chronic exposure. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2013PA114849.

Council of Science Editors:

Poisson-Moreau de Lizorieux C. Rôle du stress oxydant au niveau hépatique et rénal dans la toxicité de l’uranium après exposition chronique : Role of oxidative stress in liver and kidney in uranium toxicity after chronic exposure. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA114849

27. Zgheib, Elias. Bioinformatic and modelling approaches for a system-level understanding of oxidative stress toxicity : Approches de bio-informatique et de modélisation pour une compréhension du stress oxydant au niveau systémique.

Degree: Docteur es, Bio-ingénierie et Mathématiques Appliquées : Unité de Recherche Biomécanique et Bio-ingénierie (UMR-7338), 2018, Compiègne

Avec les nouvelles avancées en biologie et toxicologie, on constate de plus en plus la complexité des mécanismes et le grand nombre de voies de… (more)

Subjects/Keywords: Toxicologie prédictive; Toxicogénomique; Voies des effets indésirables; Nrf2; Toxicology; Oxidative stress; Systems biology; Bioinformatics; Toxicogenomics; Adverse outcome pathways; Nrf2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zgheib, E. (2018). Bioinformatic and modelling approaches for a system-level understanding of oxidative stress toxicity : Approches de bio-informatique et de modélisation pour une compréhension du stress oxydant au niveau systémique. (Doctoral Dissertation). Compiègne. Retrieved from http://www.theses.fr/2018COMP2464

Chicago Manual of Style (16th Edition):

Zgheib, Elias. “Bioinformatic and modelling approaches for a system-level understanding of oxidative stress toxicity : Approches de bio-informatique et de modélisation pour une compréhension du stress oxydant au niveau systémique.” 2018. Doctoral Dissertation, Compiègne. Accessed January 29, 2020. http://www.theses.fr/2018COMP2464.

MLA Handbook (7th Edition):

Zgheib, Elias. “Bioinformatic and modelling approaches for a system-level understanding of oxidative stress toxicity : Approches de bio-informatique et de modélisation pour une compréhension du stress oxydant au niveau systémique.” 2018. Web. 29 Jan 2020.

Vancouver:

Zgheib E. Bioinformatic and modelling approaches for a system-level understanding of oxidative stress toxicity : Approches de bio-informatique et de modélisation pour une compréhension du stress oxydant au niveau systémique. [Internet] [Doctoral dissertation]. Compiègne; 2018. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2018COMP2464.

Council of Science Editors:

Zgheib E. Bioinformatic and modelling approaches for a system-level understanding of oxidative stress toxicity : Approches de bio-informatique et de modélisation pour une compréhension du stress oxydant au niveau systémique. [Doctoral Dissertation]. Compiègne; 2018. Available from: http://www.theses.fr/2018COMP2464

28. Saudrais, Élodie. Mécanismes de neuroprotection liés au glutathion dans la barrière sang - liquide céphalorachidien choroïdienne au cours du développement périnatal : Mécanismes de neuroprotection liés au glutathion dans la barrière sang-liquide céphalorachidien choroïdienne au cours du développement périnatal.

Degree: Docteur es, Neurotoxicologie, 2019, Lyon

Plus de 50 % des handicaps neurodéveloppementaux sont dus à une exposition périnatale à des stress toxiques ou oxydants. Comprendre comment le cerveau est protégé… (more)

Subjects/Keywords: Plexus choroïdes; Glutathion transférases; Glutathion peroxydase; Liquide céphalorachidien; Nrf2; Diméthylfumarate; Choroid plexuses; Glutathione transferases; Glutathione peroxidases; Cerebrospinal fluid; Nrf2; Dimethylfumarate; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saudrais, E. (2019). Mécanismes de neuroprotection liés au glutathion dans la barrière sang - liquide céphalorachidien choroïdienne au cours du développement périnatal : Mécanismes de neuroprotection liés au glutathion dans la barrière sang-liquide céphalorachidien choroïdienne au cours du développement périnatal. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSE1026

Chicago Manual of Style (16th Edition):

Saudrais, Élodie. “Mécanismes de neuroprotection liés au glutathion dans la barrière sang - liquide céphalorachidien choroïdienne au cours du développement périnatal : Mécanismes de neuroprotection liés au glutathion dans la barrière sang-liquide céphalorachidien choroïdienne au cours du développement périnatal.” 2019. Doctoral Dissertation, Lyon. Accessed January 29, 2020. http://www.theses.fr/2019LYSE1026.

MLA Handbook (7th Edition):

Saudrais, Élodie. “Mécanismes de neuroprotection liés au glutathion dans la barrière sang - liquide céphalorachidien choroïdienne au cours du développement périnatal : Mécanismes de neuroprotection liés au glutathion dans la barrière sang-liquide céphalorachidien choroïdienne au cours du développement périnatal.” 2019. Web. 29 Jan 2020.

Vancouver:

Saudrais E. Mécanismes de neuroprotection liés au glutathion dans la barrière sang - liquide céphalorachidien choroïdienne au cours du développement périnatal : Mécanismes de neuroprotection liés au glutathion dans la barrière sang-liquide céphalorachidien choroïdienne au cours du développement périnatal. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2019LYSE1026.

Council of Science Editors:

Saudrais E. Mécanismes de neuroprotection liés au glutathion dans la barrière sang - liquide céphalorachidien choroïdienne au cours du développement périnatal : Mécanismes de neuroprotection liés au glutathion dans la barrière sang-liquide céphalorachidien choroïdienne au cours du développement périnatal. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSE1026

29. Raffalli, Chloé. Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D. : Skin allergy to fragances : mechanism of action and role of the transcription factor Nrf2. From 2D to 3D models.

Degree: Docteur es, Toxicologie, 2018, Paris Saclay

La dermatite allergique de contact (DAC) est une réaction inflammatoire aiguë, médiée par les cellules dendritiques (DCs) survenant suite à l’exposition répétée de la peau… (more)

Subjects/Keywords: Allergie cutanée; Fragrance; Nrf2; In vitro; Kératinocyes; Cellules dendritiques; Skin allergy; Fragrance; Nrf2; In vitro; Keratinocytes; Dendritic cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raffalli, C. (2018). Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D. : Skin allergy to fragances : mechanism of action and role of the transcription factor Nrf2. From 2D to 3D models. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2018SACLS045

Chicago Manual of Style (16th Edition):

Raffalli, Chloé. “Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D. : Skin allergy to fragances : mechanism of action and role of the transcription factor Nrf2. From 2D to 3D models.” 2018. Doctoral Dissertation, Paris Saclay. Accessed January 29, 2020. http://www.theses.fr/2018SACLS045.

MLA Handbook (7th Edition):

Raffalli, Chloé. “Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D. : Skin allergy to fragances : mechanism of action and role of the transcription factor Nrf2. From 2D to 3D models.” 2018. Web. 29 Jan 2020.

Vancouver:

Raffalli C. Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D. : Skin allergy to fragances : mechanism of action and role of the transcription factor Nrf2. From 2D to 3D models. [Internet] [Doctoral dissertation]. Paris Saclay; 2018. [cited 2020 Jan 29]. Available from: http://www.theses.fr/2018SACLS045.

Council of Science Editors:

Raffalli C. Les allergies cutanées aux fragrances : mécanisme d'action et rôle du facteur de transcription Nrf2. Du modèle 2D au modèle 3D. : Skin allergy to fragances : mechanism of action and role of the transcription factor Nrf2. From 2D to 3D models. [Doctoral Dissertation]. Paris Saclay; 2018. Available from: http://www.theses.fr/2018SACLS045

30. 厳, 向紅. The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-.

Degree: 博士(理工学), 2016, Konan University / 甲南大学

皮膚は人体最大の臓器であり、外部刺激や環境因子から生体内部を防御する上で重要な役割を担う。その皮膚老化は、外見に影響を与えるだけでなく、生体防御機構の乱れとして一連の病患を引き起こし、個人の生活の質(QOL; Quality of Life)の低下をもたらすことから、皮膚老化の進行を制御することがますます重要となっている。そこで、皮膚老化に大きく影響する要素を解明し、それぞれの要素の皮膚老化メカニズムを分子生物学的に解析することで、皮膚に対するアンチエイジング効果を持つ生体機能性成分の作用機序について理解することを目的として研究を行った。はじめに、皮膚老化過程を経年的に調査することが被験者の個人差などを排除できる有効な調査方法であることを示し、その経年的な疫学調査から、肌理、シワ、シミの三点が皮膚の見た目の老化にとって最も重要な要素だと認めるとともに、皮膚老化の有効な指標となることを示した。そこでまず、肌理に関わる角化細胞に対してバリア機能を高める効果があるとして知られるOpuntia ficus-indica抽出物(OFIE)の作用について検討を行った。その結果、OFIEはAhR-Nrf2-NQO1(アリール炭化水素受容体—NF-E2関連因子—NAD(P)H酸化還元酵素、キノン1)経路を活性化することで抗酸化作用を持つことが示された。さらに、OFIEがフィラグリンやロリクリンの発現をAhR依存的に増強することで皮膚バリアを保護することを示した。さらに、ヒトの老化した真皮および若い真皮におけるマイクロRNAの発現を網羅的に比較するとともに、生体機能性成分によるマイクロRNA発現様式への影響を調べた。その結果、miRNA 34および29が、線維芽細胞の老化および細胞外マトリックスの再構築を制御する上で中心的な役割を果たし、シワの形成に関与することが明らかとなった。最後に、角化細胞におけるメラニンの生物学的作用を明らかにするため、単純化細胞モデルを構築し、生体機能性成分を評価したところ、ナイアシンアミドおよびトリプシン阻害剤が角化細胞におけるメラニン取り込みを抑制することが示された。さらに、取り込まれたメラニンは角化細胞の細胞周期を停止させ、色素細胞の機能を抑制することが知られるDickkopf 1遺伝子の発現低下を引き起こすことが明らかとなった。これらの結果は、皮膚老化要素に影響を与える角化細胞および線維芽細胞の働きについて新たな知見を提供するものであり、ヒトにおける皮膚老化の抑制に向けた新たな戦略を立てるうえで大きな示唆を与えている。さらに、生体機能性成分が持つ皮膚老化要素に対する有効性をより強固にサポートしている。これらの成果は、今後の新たな皮膚アンチエイジング技術の開発を通して、肌理の乱れ、シワやシミといった多くの人の皮膚老化への懸念を解消し、高齢化社会における人々のQOLの向上に資するものと期待される。

平成27年(2015年度)

Subjects/Keywords: Skin; aging; anti-aging; AhR-Nrf2 signaling; microRNA; melanin uptake

Page 1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

厳, . (2016). The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-. (Thesis). Konan University / 甲南大学. Retrieved from http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

厳, 向紅. “The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-.” 2016. Thesis, Konan University / 甲南大学. Accessed January 29, 2020. http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

厳, 向紅. “The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-.” 2016. Web. 29 Jan 2020.

Vancouver:

厳 . The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-. [Internet] [Thesis]. Konan University / 甲南大学; 2016. [cited 2020 Jan 29]. Available from: http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

厳 . The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients- : The studies of skin aging and anti-aging -New mechanism and efficacious biofunctional ingredients-; 皮膚の老化および抗老化に関する研究-新規メカニズムと生体機能性有効成分-. [Thesis]. Konan University / 甲南大学; 2016. Available from: http://id.nii.ac.jp/1260/00001761/ ; http://dx.doi.org/10.14990/00001761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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