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Dept: Microbiology and Immunology

You searched for subject:( Microbiology). Showing records 61 – 90 of 245 total matches.

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University of North Carolina

61. McNally, Richard Sean. The effects of DJ-1 and A20 family members on inflammation and oxidative response.

Degree: Microbiology and Immunology, 2010, University of North Carolina

 Dysregulation of the balance between cell survival and cell death is seen in the loss of dopaminergic neurons in Parkinson's disease and the inappropriate cell… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McNally, R. S. (2010). The effects of DJ-1 and A20 family members on inflammation and oxidative response. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:20769187-189f-4364-98f7-5e6bbc7929d2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McNally, Richard Sean. “The effects of DJ-1 and A20 family members on inflammation and oxidative response.” 2010. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:20769187-189f-4364-98f7-5e6bbc7929d2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McNally, Richard Sean. “The effects of DJ-1 and A20 family members on inflammation and oxidative response.” 2010. Web. 18 Nov 2019.

Vancouver:

McNally RS. The effects of DJ-1 and A20 family members on inflammation and oxidative response. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:20769187-189f-4364-98f7-5e6bbc7929d2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McNally RS. The effects of DJ-1 and A20 family members on inflammation and oxidative response. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:20769187-189f-4364-98f7-5e6bbc7929d2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

62. Aybar, Lydia. Characterization of Regulatory B cells in Patients with Anti-Neutrophil Cytoplasmic Autoantibody Vasculitis.

Degree: Microbiology and Immunology, 2015, University of North Carolina

 ANCA-associated vasculitis (AAV) is B cell dependent; however, which B cell subsets modulate immunopathogenesis remains unknown. Although their phenotype is controversial, regulatory B cells (Bregs)… (more)

Subjects/Keywords: Immunology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Aybar, L. (2015). Characterization of Regulatory B cells in Patients with Anti-Neutrophil Cytoplasmic Autoantibody Vasculitis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e0f10cf6-b137-4664-a7c2-b215b554e5d9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aybar, Lydia. “Characterization of Regulatory B cells in Patients with Anti-Neutrophil Cytoplasmic Autoantibody Vasculitis.” 2015. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:e0f10cf6-b137-4664-a7c2-b215b554e5d9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aybar, Lydia. “Characterization of Regulatory B cells in Patients with Anti-Neutrophil Cytoplasmic Autoantibody Vasculitis.” 2015. Web. 18 Nov 2019.

Vancouver:

Aybar L. Characterization of Regulatory B cells in Patients with Anti-Neutrophil Cytoplasmic Autoantibody Vasculitis. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:e0f10cf6-b137-4664-a7c2-b215b554e5d9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aybar L. Characterization of Regulatory B cells in Patients with Anti-Neutrophil Cytoplasmic Autoantibody Vasculitis. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:e0f10cf6-b137-4664-a7c2-b215b554e5d9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

63. Barton, Kirston Mandy Lang. TARGETING THE CLASS I HDACS TO DISRUPT QUIESCENT HIV-1 PROVIRUSES.

Degree: Microbiology and Immunology, 2013, University of North Carolina

 Infection with the human immunodeficiency virus type 1 (HIV-1) was first described in a report in the Morbidity and Mortality Weekly Report in June of… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Barton, K. M. L. (2013). TARGETING THE CLASS I HDACS TO DISRUPT QUIESCENT HIV-1 PROVIRUSES. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c9369af2-60fc-44f7-b1ff-b42ffbebb07c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barton, Kirston Mandy Lang. “TARGETING THE CLASS I HDACS TO DISRUPT QUIESCENT HIV-1 PROVIRUSES.” 2013. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:c9369af2-60fc-44f7-b1ff-b42ffbebb07c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barton, Kirston Mandy Lang. “TARGETING THE CLASS I HDACS TO DISRUPT QUIESCENT HIV-1 PROVIRUSES.” 2013. Web. 18 Nov 2019.

Vancouver:

Barton KML. TARGETING THE CLASS I HDACS TO DISRUPT QUIESCENT HIV-1 PROVIRUSES. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:c9369af2-60fc-44f7-b1ff-b42ffbebb07c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barton KML. TARGETING THE CLASS I HDACS TO DISRUPT QUIESCENT HIV-1 PROVIRUSES. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:c9369af2-60fc-44f7-b1ff-b42ffbebb07c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

64. Bolles, Meagan Elise. Evaluations Of Severe Acute Respiratory Syndrome Coronavirus Therapeutics And A Viral Capacity For Plasticity And Escape.

Degree: Microbiology and Immunology, 2013, University of North Carolina

 The Severe Acute Respiratory Syndrome Coronavirus (SARS-CoV) emerged in 2002/2003, causing the deaths of almost a tenth of the 8000 individuals infected worldwide before it… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Bolles, M. E. (2013). Evaluations Of Severe Acute Respiratory Syndrome Coronavirus Therapeutics And A Viral Capacity For Plasticity And Escape. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:633a1918-3e90-4518-ad98-a979d4e65695

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bolles, Meagan Elise. “Evaluations Of Severe Acute Respiratory Syndrome Coronavirus Therapeutics And A Viral Capacity For Plasticity And Escape.” 2013. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:633a1918-3e90-4518-ad98-a979d4e65695.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bolles, Meagan Elise. “Evaluations Of Severe Acute Respiratory Syndrome Coronavirus Therapeutics And A Viral Capacity For Plasticity And Escape.” 2013. Web. 18 Nov 2019.

Vancouver:

Bolles ME. Evaluations Of Severe Acute Respiratory Syndrome Coronavirus Therapeutics And A Viral Capacity For Plasticity And Escape. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:633a1918-3e90-4518-ad98-a979d4e65695.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bolles ME. Evaluations Of Severe Acute Respiratory Syndrome Coronavirus Therapeutics And A Viral Capacity For Plasticity And Escape. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:633a1918-3e90-4518-ad98-a979d4e65695

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

65. Roberts, Reid Austin. Harnessing What Lies Within: Programming Immunity With Biocompatible Devices to Treat Human Disease.

Degree: Microbiology and Immunology, 2013, University of North Carolina

 Advances in our mechanistic insight of cellular function and how this relates to host physiology have revealed a world which is intimately connected at the… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Roberts, R. A. (2013). Harnessing What Lies Within: Programming Immunity With Biocompatible Devices to Treat Human Disease. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:fd354fc8-989a-4e1a-a010-de147d81f2d8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roberts, Reid Austin. “Harnessing What Lies Within: Programming Immunity With Biocompatible Devices to Treat Human Disease.” 2013. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:fd354fc8-989a-4e1a-a010-de147d81f2d8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roberts, Reid Austin. “Harnessing What Lies Within: Programming Immunity With Biocompatible Devices to Treat Human Disease.” 2013. Web. 18 Nov 2019.

Vancouver:

Roberts RA. Harnessing What Lies Within: Programming Immunity With Biocompatible Devices to Treat Human Disease. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:fd354fc8-989a-4e1a-a010-de147d81f2d8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roberts RA. Harnessing What Lies Within: Programming Immunity With Biocompatible Devices to Treat Human Disease. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:fd354fc8-989a-4e1a-a010-de147d81f2d8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

66. Zhou, Yang. Mechanisms of Antibody Mediated Neutralization of Dengue Virus.

Degree: Microbiology and Immunology, 2013, University of North Carolina

 Dengue virus (DENV) is the most significant arthropod-borne virus in this world, affecting 2.5 billion people. Pre-existing sub-neutralizing antibodies (Abs) can enhance virus infection, leading… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Zhou, Y. (2013). Mechanisms of Antibody Mediated Neutralization of Dengue Virus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:1f7b499d-ff29-4351-ab2a-a3e6c5a4dca8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Yang. “Mechanisms of Antibody Mediated Neutralization of Dengue Virus.” 2013. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:1f7b499d-ff29-4351-ab2a-a3e6c5a4dca8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Yang. “Mechanisms of Antibody Mediated Neutralization of Dengue Virus.” 2013. Web. 18 Nov 2019.

Vancouver:

Zhou Y. Mechanisms of Antibody Mediated Neutralization of Dengue Virus. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:1f7b499d-ff29-4351-ab2a-a3e6c5a4dca8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou Y. Mechanisms of Antibody Mediated Neutralization of Dengue Virus. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:1f7b499d-ff29-4351-ab2a-a3e6c5a4dca8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

67. Clark, Martha A. Iron Deficiency and Iron Supplementation Conspire to Mediate Susceptibility to Erythrocytic Stage Plasmodium falciparum Infection.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Malaria and iron deficiency are interconnected public health concerns, which disproportionally affect children and pregnant women. Malaria causes an estimated 250 million infections and 1… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Clark, M. A. (2014). Iron Deficiency and Iron Supplementation Conspire to Mediate Susceptibility to Erythrocytic Stage Plasmodium falciparum Infection. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7b042c68-c7a8-4b31-8ab4-6f73d368ee72

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Clark, Martha A. “Iron Deficiency and Iron Supplementation Conspire to Mediate Susceptibility to Erythrocytic Stage Plasmodium falciparum Infection.” 2014. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:7b042c68-c7a8-4b31-8ab4-6f73d368ee72.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Clark, Martha A. “Iron Deficiency and Iron Supplementation Conspire to Mediate Susceptibility to Erythrocytic Stage Plasmodium falciparum Infection.” 2014. Web. 18 Nov 2019.

Vancouver:

Clark MA. Iron Deficiency and Iron Supplementation Conspire to Mediate Susceptibility to Erythrocytic Stage Plasmodium falciparum Infection. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:7b042c68-c7a8-4b31-8ab4-6f73d368ee72.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Clark MA. Iron Deficiency and Iron Supplementation Conspire to Mediate Susceptibility to Erythrocytic Stage Plasmodium falciparum Infection. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:7b042c68-c7a8-4b31-8ab4-6f73d368ee72

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

68. Gonzalez, Rodrigo J. Dissemination of Yersinia pestis During Bubonic Plague.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Yersinia pestis is a highly virulent bacterial pathogen with remarkable abilities to disseminate in a host. The ability of this Gram-negative bacterium to spread and… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Gonzalez, R. J. (2014). Dissemination of Yersinia pestis During Bubonic Plague. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9934d35d-863f-4f0f-88b6-4b73c1756f3d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonzalez, Rodrigo J. “Dissemination of Yersinia pestis During Bubonic Plague.” 2014. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:9934d35d-863f-4f0f-88b6-4b73c1756f3d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonzalez, Rodrigo J. “Dissemination of Yersinia pestis During Bubonic Plague.” 2014. Web. 18 Nov 2019.

Vancouver:

Gonzalez RJ. Dissemination of Yersinia pestis During Bubonic Plague. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:9934d35d-863f-4f0f-88b6-4b73c1756f3d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonzalez RJ. Dissemination of Yersinia pestis During Bubonic Plague. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:9934d35d-863f-4f0f-88b6-4b73c1756f3d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

69. Jensen, Kara. Characterization of a Novel Mycobacterium Tuberculosis-Simian Immunodeficiency Virus Vaccine to Prevent Oral Pediatric HIV Transmission.

Degree: Microbiology and Immunology, 2014, University of North Carolina

 Over 3.3 million children are living with HIV, infected primarily by mother-to-child transmission (MTCT). Breast milk exposure of HIV accounts for up to 44% of… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Jensen, K. (2014). Characterization of a Novel Mycobacterium Tuberculosis-Simian Immunodeficiency Virus Vaccine to Prevent Oral Pediatric HIV Transmission. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:69f8b681-3b05-4c40-baef-a69a3c301440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jensen, Kara. “Characterization of a Novel Mycobacterium Tuberculosis-Simian Immunodeficiency Virus Vaccine to Prevent Oral Pediatric HIV Transmission.” 2014. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:69f8b681-3b05-4c40-baef-a69a3c301440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jensen, Kara. “Characterization of a Novel Mycobacterium Tuberculosis-Simian Immunodeficiency Virus Vaccine to Prevent Oral Pediatric HIV Transmission.” 2014. Web. 18 Nov 2019.

Vancouver:

Jensen K. Characterization of a Novel Mycobacterium Tuberculosis-Simian Immunodeficiency Virus Vaccine to Prevent Oral Pediatric HIV Transmission. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:69f8b681-3b05-4c40-baef-a69a3c301440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jensen K. Characterization of a Novel Mycobacterium Tuberculosis-Simian Immunodeficiency Virus Vaccine to Prevent Oral Pediatric HIV Transmission. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:69f8b681-3b05-4c40-baef-a69a3c301440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

70. Kannan, Arthi. VAGINAL MUCOSAL IMMUNITY BASED ON IgG-MUCIN CROSSLINKING.

Degree: Microbiology and Immunology, 2013, University of North Carolina

 In this thesis, I investigated whether IgG, the predominant antibody in cervicovaginal mucus (CVM), can interact with CVM constituents to protect against Herpes Simplex Virus… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Kannan, A. (2013). VAGINAL MUCOSAL IMMUNITY BASED ON IgG-MUCIN CROSSLINKING. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b25ba223-db6b-4551-96ab-afd842820bd9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kannan, Arthi. “VAGINAL MUCOSAL IMMUNITY BASED ON IgG-MUCIN CROSSLINKING.” 2013. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:b25ba223-db6b-4551-96ab-afd842820bd9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kannan, Arthi. “VAGINAL MUCOSAL IMMUNITY BASED ON IgG-MUCIN CROSSLINKING.” 2013. Web. 18 Nov 2019.

Vancouver:

Kannan A. VAGINAL MUCOSAL IMMUNITY BASED ON IgG-MUCIN CROSSLINKING. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:b25ba223-db6b-4551-96ab-afd842820bd9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kannan A. VAGINAL MUCOSAL IMMUNITY BASED ON IgG-MUCIN CROSSLINKING. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:b25ba223-db6b-4551-96ab-afd842820bd9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

71. Liesman, Rachael. Impact of the Non-structural 2 Protein on Respiratory Syncytial Virus Pathogenesis.

Degree: Microbiology and Immunology, 2013, University of North Carolina

 Respiratory syncytial virus (RSV) infection is the major cause of bronchiolitis in young children. The factors contributing to increased propensity of RSV-induced distal airway disease… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Liesman, R. (2013). Impact of the Non-structural 2 Protein on Respiratory Syncytial Virus Pathogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:dc257304-ed7c-4258-bb1f-9f4bb29deee8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liesman, Rachael. “Impact of the Non-structural 2 Protein on Respiratory Syncytial Virus Pathogenesis.” 2013. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:dc257304-ed7c-4258-bb1f-9f4bb29deee8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liesman, Rachael. “Impact of the Non-structural 2 Protein on Respiratory Syncytial Virus Pathogenesis.” 2013. Web. 18 Nov 2019.

Vancouver:

Liesman R. Impact of the Non-structural 2 Protein on Respiratory Syncytial Virus Pathogenesis. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:dc257304-ed7c-4258-bb1f-9f4bb29deee8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liesman R. Impact of the Non-structural 2 Protein on Respiratory Syncytial Virus Pathogenesis. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:dc257304-ed7c-4258-bb1f-9f4bb29deee8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

72. Scott, Eric. Immune Cells and Type I Interferon in Systemic Lupus Erythematosus Patient Urine and Kidney Immunopathology.

Degree: Microbiology and Immunology, 2015, University of North Carolina

 Murine models have demonstrated that systemic lupus erythematosus (SLE) results in kidney damage due to antibody mediated tissue destruction. Studies of peripheral blood immune subsets… (more)

Subjects/Keywords: Immunology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Scott, E. (2015). Immune Cells and Type I Interferon in Systemic Lupus Erythematosus Patient Urine and Kidney Immunopathology. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c3470669-53a7-40db-a40a-ad4e934d9b66

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Scott, Eric. “Immune Cells and Type I Interferon in Systemic Lupus Erythematosus Patient Urine and Kidney Immunopathology.” 2015. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:c3470669-53a7-40db-a40a-ad4e934d9b66.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Scott, Eric. “Immune Cells and Type I Interferon in Systemic Lupus Erythematosus Patient Urine and Kidney Immunopathology.” 2015. Web. 18 Nov 2019.

Vancouver:

Scott E. Immune Cells and Type I Interferon in Systemic Lupus Erythematosus Patient Urine and Kidney Immunopathology. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:c3470669-53a7-40db-a40a-ad4e934d9b66.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Scott E. Immune Cells and Type I Interferon in Systemic Lupus Erythematosus Patient Urine and Kidney Immunopathology. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:c3470669-53a7-40db-a40a-ad4e934d9b66

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

73. Ziehr, Benjamin. TRANSLATION REGULATION DURING HCMV INFECTION.

Degree: Microbiology and Immunology, 2015, University of North Carolina

 During viral infection competition for resources is inevitable. One of the fiercest areas of competition is mRNA translation. Host ribosomes are utilized by both host… (more)

Subjects/Keywords: Virology; Biochemistry; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Ziehr, B. (2015). TRANSLATION REGULATION DURING HCMV INFECTION. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:24dde334-ea8d-4a3b-9354-dfb019afa339

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ziehr, Benjamin. “TRANSLATION REGULATION DURING HCMV INFECTION.” 2015. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:24dde334-ea8d-4a3b-9354-dfb019afa339.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ziehr, Benjamin. “TRANSLATION REGULATION DURING HCMV INFECTION.” 2015. Web. 18 Nov 2019.

Vancouver:

Ziehr B. TRANSLATION REGULATION DURING HCMV INFECTION. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:24dde334-ea8d-4a3b-9354-dfb019afa339.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ziehr B. TRANSLATION REGULATION DURING HCMV INFECTION. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:24dde334-ea8d-4a3b-9354-dfb019afa339

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

74. Giffin, Louise. Manipulation and Exploitation of the Host Cell by Kaposi's Sarcoma-Associated Herpesvirus.

Degree: Microbiology and Immunology, 2015, University of North Carolina

 Kaposi’s sarcoma-associated herpesvirus (KSHV) is a human gamma-herpesvirus that is the causative agent of three human malignancies: Kaposi’s sarcoma, primary effusion lymphoma, and multicentric Castleman’s… (more)

Subjects/Keywords: Virology; Cytology; Immunology; School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Giffin, L. (2015). Manipulation and Exploitation of the Host Cell by Kaposi's Sarcoma-Associated Herpesvirus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b0e6a869-b24d-4405-8884-fed4f95d62b1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Giffin, Louise. “Manipulation and Exploitation of the Host Cell by Kaposi's Sarcoma-Associated Herpesvirus.” 2015. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:b0e6a869-b24d-4405-8884-fed4f95d62b1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Giffin, Louise. “Manipulation and Exploitation of the Host Cell by Kaposi's Sarcoma-Associated Herpesvirus.” 2015. Web. 18 Nov 2019.

Vancouver:

Giffin L. Manipulation and Exploitation of the Host Cell by Kaposi's Sarcoma-Associated Herpesvirus. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:b0e6a869-b24d-4405-8884-fed4f95d62b1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Giffin L. Manipulation and Exploitation of the Host Cell by Kaposi's Sarcoma-Associated Herpesvirus. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:b0e6a869-b24d-4405-8884-fed4f95d62b1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

75. Linz, Brandon. Cellular Mechanisms of Immune and Hematopoietic Dysfunction Following Radiation and Burn Injuries.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 The immune system has evolved to protect the body against damage from infection, disease, or injury. Severe injuries, such as large burns or radiation exposures,… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Linz, B. (2016). Cellular Mechanisms of Immune and Hematopoietic Dysfunction Following Radiation and Burn Injuries. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9b4df721-2d66-4912-8c43-a5d5e1081246

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Linz, Brandon. “Cellular Mechanisms of Immune and Hematopoietic Dysfunction Following Radiation and Burn Injuries.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:9b4df721-2d66-4912-8c43-a5d5e1081246.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Linz, Brandon. “Cellular Mechanisms of Immune and Hematopoietic Dysfunction Following Radiation and Burn Injuries.” 2016. Web. 18 Nov 2019.

Vancouver:

Linz B. Cellular Mechanisms of Immune and Hematopoietic Dysfunction Following Radiation and Burn Injuries. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:9b4df721-2d66-4912-8c43-a5d5e1081246.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Linz B. Cellular Mechanisms of Immune and Hematopoietic Dysfunction Following Radiation and Burn Injuries. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:9b4df721-2d66-4912-8c43-a5d5e1081246

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

76. Anacker, Daniel. Manipulation of the Host Cell DNA Damage Pathways by Human Papillomavirus.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Human papilloma virus (HPV) is thought to be the most common sexually transmitted viral infection in the United States. It poses a major public health… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Anacker, D. (2016). Manipulation of the Host Cell DNA Damage Pathways by Human Papillomavirus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:547744d8-eda7-4d27-8d66-40b962f238f9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anacker, Daniel. “Manipulation of the Host Cell DNA Damage Pathways by Human Papillomavirus.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:547744d8-eda7-4d27-8d66-40b962f238f9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anacker, Daniel. “Manipulation of the Host Cell DNA Damage Pathways by Human Papillomavirus.” 2016. Web. 18 Nov 2019.

Vancouver:

Anacker D. Manipulation of the Host Cell DNA Damage Pathways by Human Papillomavirus. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:547744d8-eda7-4d27-8d66-40b962f238f9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anacker D. Manipulation of the Host Cell DNA Damage Pathways by Human Papillomavirus. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:547744d8-eda7-4d27-8d66-40b962f238f9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

77. Rackoff, Lauren. Tissue Reservoirs of HIV-1: Insights from the Central Nervous System.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Cellular and anatomical reservoirs of HIV-1 preclude a cure to infection. Efforts to characterize these reservoirs are an important part of developing a strategy to… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Rackoff, L. (2016). Tissue Reservoirs of HIV-1: Insights from the Central Nervous System. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rackoff, Lauren. “Tissue Reservoirs of HIV-1: Insights from the Central Nervous System.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rackoff, Lauren. “Tissue Reservoirs of HIV-1: Insights from the Central Nervous System.” 2016. Web. 18 Nov 2019.

Vancouver:

Rackoff L. Tissue Reservoirs of HIV-1: Insights from the Central Nervous System. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rackoff L. Tissue Reservoirs of HIV-1: Insights from the Central Nervous System. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:48631bb1-0b98-49b9-bbf1-d6f59831a65c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

78. Dunn, Julia. Modulating Innate Immunity Improves Outcomes in Acute Lung Injury.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Pulmonary inflammation following traumatic injury disrupts the lung architecture, leading to impaired oxygen exchange and rendering patients susceptible to life threatening bacterial infections. Innate immunity… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Dunn, J. (2016). Modulating Innate Immunity Improves Outcomes in Acute Lung Injury. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7dd0c558-47f5-42a9-b14f-ec602549e6a4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dunn, Julia. “Modulating Innate Immunity Improves Outcomes in Acute Lung Injury.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:7dd0c558-47f5-42a9-b14f-ec602549e6a4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dunn, Julia. “Modulating Innate Immunity Improves Outcomes in Acute Lung Injury.” 2016. Web. 18 Nov 2019.

Vancouver:

Dunn J. Modulating Innate Immunity Improves Outcomes in Acute Lung Injury. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:7dd0c558-47f5-42a9-b14f-ec602549e6a4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dunn J. Modulating Innate Immunity Improves Outcomes in Acute Lung Injury. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:7dd0c558-47f5-42a9-b14f-ec602549e6a4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

79. Grosser, Melinda. Determinants of Nitric Oxide Resistance in Staphylococcus aureus.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Staphylococcus aureus is a prolific human pathogen capable of circumventing host innate immunity by resisting the antimicrobial radical nitric oxide (NO·). NO· is critical for… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Grosser, M. (2016). Determinants of Nitric Oxide Resistance in Staphylococcus aureus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:45c1ffea-7de0-4c9e-8515-1e5f5e21226e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grosser, Melinda. “Determinants of Nitric Oxide Resistance in Staphylococcus aureus.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:45c1ffea-7de0-4c9e-8515-1e5f5e21226e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grosser, Melinda. “Determinants of Nitric Oxide Resistance in Staphylococcus aureus.” 2016. Web. 18 Nov 2019.

Vancouver:

Grosser M. Determinants of Nitric Oxide Resistance in Staphylococcus aureus. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:45c1ffea-7de0-4c9e-8515-1e5f5e21226e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grosser M. Determinants of Nitric Oxide Resistance in Staphylococcus aureus. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:45c1ffea-7de0-4c9e-8515-1e5f5e21226e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

80. Arrildt, Kathryn. Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 HIV-1 preferentially attaches and enters specific cell types by recognizing cell-specific receptors. HIV-1 typically targets memory CD4+ T cells by restricting attachment to cells expressing… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Arrildt, K. (2016). Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arrildt, Kathryn. “Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arrildt, Kathryn. “Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1.” 2016. Web. 18 Nov 2019.

Vancouver:

Arrildt K. Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arrildt K. Phenotypic and Genotypic Features of Macrophage Tropism in HIV-1. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:650b39d7-9d1e-4d34-8e5b-a219db67813e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

81. Anders, Penny. Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus.

Degree: Microbiology and Immunology, 2016, University of North Carolina

 Kaposi’s sarcoma associated-herpesvirus (KSHV) is a double-strand DNA gamma herpesvirus that establishes lifelong latent infection in the human host. KSHV infection is associated with several… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Anders, P. (2016). Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anders, Penny. “Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus.” 2016. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anders, Penny. “Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus.” 2016. Web. 18 Nov 2019.

Vancouver:

Anders P. Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anders P. Modulation of Signal Transduction and Transformation by Kaposi’s Sarcoma-Associated Herpesvirus. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:d18be56b-2603-463f-8fc4-b155ab1e432a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

82. Johnson, Bryan. THE ACTIVATION AND CONSEQUENCES OF THE ATM MEDIATED DNA DAMAGE RESPONSE IN HPV INFECTED CELLS.

Degree: Microbiology and Immunology, 2017, University of North Carolina

 Infection with Human papillomavirus (HPV) is the most prevalent sexually transmitted disease in the world. From a public health standpoint, a subset of mucosa-tropic HPVs… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Johnson, B. (2017). THE ACTIVATION AND CONSEQUENCES OF THE ATM MEDIATED DNA DAMAGE RESPONSE IN HPV INFECTED CELLS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:07d90cfc-db78-45d3-8378-a72a865372e9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johnson, Bryan. “THE ACTIVATION AND CONSEQUENCES OF THE ATM MEDIATED DNA DAMAGE RESPONSE IN HPV INFECTED CELLS.” 2017. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:07d90cfc-db78-45d3-8378-a72a865372e9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johnson, Bryan. “THE ACTIVATION AND CONSEQUENCES OF THE ATM MEDIATED DNA DAMAGE RESPONSE IN HPV INFECTED CELLS.” 2017. Web. 18 Nov 2019.

Vancouver:

Johnson B. THE ACTIVATION AND CONSEQUENCES OF THE ATM MEDIATED DNA DAMAGE RESPONSE IN HPV INFECTED CELLS. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:07d90cfc-db78-45d3-8378-a72a865372e9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johnson B. THE ACTIVATION AND CONSEQUENCES OF THE ATM MEDIATED DNA DAMAGE RESPONSE IN HPV INFECTED CELLS. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:07d90cfc-db78-45d3-8378-a72a865372e9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

83. Dant, Trisha. Examining Factors Critical to T Cell Suppression and Migration During Acute Graft-Versus-Host Disease.

Degree: Microbiology and Immunology, 2017, University of North Carolina

 Allogeneic stem cell transplant (allo-SCT) is a curative therapy for malignant and non-malignant diseases. Unfortunately, acute Graft-versus-Host Disease (aGvHD) is a complication of allo-SCT, and… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Dant, T. (2017). Examining Factors Critical to T Cell Suppression and Migration During Acute Graft-Versus-Host Disease. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:60e87d06-bfe3-43b7-9d31-40ca2895bdb9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dant, Trisha. “Examining Factors Critical to T Cell Suppression and Migration During Acute Graft-Versus-Host Disease.” 2017. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:60e87d06-bfe3-43b7-9d31-40ca2895bdb9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dant, Trisha. “Examining Factors Critical to T Cell Suppression and Migration During Acute Graft-Versus-Host Disease.” 2017. Web. 18 Nov 2019.

Vancouver:

Dant T. Examining Factors Critical to T Cell Suppression and Migration During Acute Graft-Versus-Host Disease. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:60e87d06-bfe3-43b7-9d31-40ca2895bdb9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dant T. Examining Factors Critical to T Cell Suppression and Migration During Acute Graft-Versus-Host Disease. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:60e87d06-bfe3-43b7-9d31-40ca2895bdb9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

84. Fan, Ting-Jia. The Role of Enterococcus faecalis Sugar Transport in Experimental Colitis.

Degree: Microbiology and Immunology, 2017, University of North Carolina

 Inflammatory bowel diseases (IBDs), which afflict 1.6 million people in the United States, are chronic, relapsing and immune-mediated intestinal disorders caused in part by aggressive… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Fan, T. (2017). The Role of Enterococcus faecalis Sugar Transport in Experimental Colitis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c544c126-fdf7-4751-bd86-5b81584191f7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fan, Ting-Jia. “The Role of Enterococcus faecalis Sugar Transport in Experimental Colitis.” 2017. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:c544c126-fdf7-4751-bd86-5b81584191f7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fan, Ting-Jia. “The Role of Enterococcus faecalis Sugar Transport in Experimental Colitis.” 2017. Web. 18 Nov 2019.

Vancouver:

Fan T. The Role of Enterococcus faecalis Sugar Transport in Experimental Colitis. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:c544c126-fdf7-4751-bd86-5b81584191f7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fan T. The Role of Enterococcus faecalis Sugar Transport in Experimental Colitis. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:c544c126-fdf7-4751-bd86-5b81584191f7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

85. Maltez, Vivien. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.

Degree: Microbiology and Immunology, 2017, University of North Carolina

 Programmed cell death is a powerful anti-microbial defense. The innate immune system is our first line of defense against infection, and the induction of cell… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Maltez, V. (2017). Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maltez, Vivien. “Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.” 2017. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maltez, Vivien. “Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches.” 2017. Web. 18 Nov 2019.

Vancouver:

Maltez V. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maltez V. Inflammasomes and Cytotoxic Lymphocytes Clear Bacteria From Different Intracellular Niches. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:0eb5d888-178a-4a7c-b6af-78c92db7e502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

86. Arend, Kyle. REGULATION OF HUMAN CYTOMEGALOVIRUS MAJOR IMMEDIATE EARLY GENE EXPRESSION DURING LYTIC REPLICATION.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 Human cytomegalovirus (HCMV) is a significant cause of disease in immune-compromised adults and immune naïve newborns. No vaccine exists to prevent HCMV infection, and current… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Arend, K. (2018). REGULATION OF HUMAN CYTOMEGALOVIRUS MAJOR IMMEDIATE EARLY GENE EXPRESSION DURING LYTIC REPLICATION. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:834963c2-2e97-4a00-95ce-2fbddc925c14

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arend, Kyle. “REGULATION OF HUMAN CYTOMEGALOVIRUS MAJOR IMMEDIATE EARLY GENE EXPRESSION DURING LYTIC REPLICATION.” 2018. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:834963c2-2e97-4a00-95ce-2fbddc925c14.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arend, Kyle. “REGULATION OF HUMAN CYTOMEGALOVIRUS MAJOR IMMEDIATE EARLY GENE EXPRESSION DURING LYTIC REPLICATION.” 2018. Web. 18 Nov 2019.

Vancouver:

Arend K. REGULATION OF HUMAN CYTOMEGALOVIRUS MAJOR IMMEDIATE EARLY GENE EXPRESSION DURING LYTIC REPLICATION. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:834963c2-2e97-4a00-95ce-2fbddc925c14.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arend K. REGULATION OF HUMAN CYTOMEGALOVIRUS MAJOR IMMEDIATE EARLY GENE EXPRESSION DURING LYTIC REPLICATION. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:834963c2-2e97-4a00-95ce-2fbddc925c14

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

87. Jones, Jennifer. The Role of the Opal Termination Codon in Alphavirus Pathogenesis.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 Alphaviruses are members of the Togaviridae family of viruses. These viruses are arboviruses, most often transmitted by a mosquito host. Serious outbreaks of alphavirus infection… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Jones, J. (2018). The Role of the Opal Termination Codon in Alphavirus Pathogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9fd6cab0-c754-4a85-9b0b-c855e4e08ddf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jones, Jennifer. “The Role of the Opal Termination Codon in Alphavirus Pathogenesis.” 2018. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:9fd6cab0-c754-4a85-9b0b-c855e4e08ddf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jones, Jennifer. “The Role of the Opal Termination Codon in Alphavirus Pathogenesis.” 2018. Web. 18 Nov 2019.

Vancouver:

Jones J. The Role of the Opal Termination Codon in Alphavirus Pathogenesis. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:9fd6cab0-c754-4a85-9b0b-c855e4e08ddf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jones J. The Role of the Opal Termination Codon in Alphavirus Pathogenesis. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:9fd6cab0-c754-4a85-9b0b-c855e4e08ddf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

88. HWANG, BIN-JIN. The hemidesmosomal protein BP180 (collagen XVII) in skin cancer and inflammation.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 BP180, also known as collagen XVII, is a transmembrane glycoprotein located in the hemidesmosome of basal keratinocytes, and functions as a key cell-matrix adhesion molecule.… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

HWANG, B. (2018). The hemidesmosomal protein BP180 (collagen XVII) in skin cancer and inflammation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4d7fa3fb-910e-49c4-a6fb-32a7c4836eca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

HWANG, BIN-JIN. “The hemidesmosomal protein BP180 (collagen XVII) in skin cancer and inflammation.” 2018. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:4d7fa3fb-910e-49c4-a6fb-32a7c4836eca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

HWANG, BIN-JIN. “The hemidesmosomal protein BP180 (collagen XVII) in skin cancer and inflammation.” 2018. Web. 18 Nov 2019.

Vancouver:

HWANG B. The hemidesmosomal protein BP180 (collagen XVII) in skin cancer and inflammation. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:4d7fa3fb-910e-49c4-a6fb-32a7c4836eca.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

HWANG B. The hemidesmosomal protein BP180 (collagen XVII) in skin cancer and inflammation. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:4d7fa3fb-910e-49c4-a6fb-32a7c4836eca

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

89. Chen, Liang. The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 The nucleotide-binding domain, leucine-rich repeat containing proteins, also known as NOD-like receptors (NLRs), are pattern recognition receptors that play important roles in innate immunity (Aganna… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Chen, L. (2018). The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Liang. “The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity.” 2018. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Liang. “The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity.” 2018. Web. 18 Nov 2019.

Vancouver:

Chen L. The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen L. The innate regulatory protein NLRP12 maintains commensal bacterial symbiosis, and mitigates intestinal inflammation and obesity. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:e755bc43-2da6-4229-b7f8-c4584012348a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

90. Hong, Lee. CD30-REDIRECTED CHIMERIC ANTIGEN RECEPTOR T CELLS TARGET EMBRYONAL CARCINOMA VIA ANTIGEN-DEPENDENT AND FAS/FASL INTERACTIONS.

Degree: Microbiology and Immunology, 2018, University of North Carolina

 Embryonal carcinomas (ECs) and mixed testicular germ cell tumors (TGCTs) containing EC express CD30 and are the most aggressive TGCT subtypes. Chimeric antigen receptor T… (more)

Subjects/Keywords: School of Medicine; Department of Microbiology and Immunology

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APA (6th Edition):

Hong, L. (2018). CD30-REDIRECTED CHIMERIC ANTIGEN RECEPTOR T CELLS TARGET EMBRYONAL CARCINOMA VIA ANTIGEN-DEPENDENT AND FAS/FASL INTERACTIONS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:519f1372-1638-487e-b82e-0681308db3e0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hong, Lee. “CD30-REDIRECTED CHIMERIC ANTIGEN RECEPTOR T CELLS TARGET EMBRYONAL CARCINOMA VIA ANTIGEN-DEPENDENT AND FAS/FASL INTERACTIONS.” 2018. Thesis, University of North Carolina. Accessed November 18, 2019. https://cdr.lib.unc.edu/record/uuid:519f1372-1638-487e-b82e-0681308db3e0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hong, Lee. “CD30-REDIRECTED CHIMERIC ANTIGEN RECEPTOR T CELLS TARGET EMBRYONAL CARCINOMA VIA ANTIGEN-DEPENDENT AND FAS/FASL INTERACTIONS.” 2018. Web. 18 Nov 2019.

Vancouver:

Hong L. CD30-REDIRECTED CHIMERIC ANTIGEN RECEPTOR T CELLS TARGET EMBRYONAL CARCINOMA VIA ANTIGEN-DEPENDENT AND FAS/FASL INTERACTIONS. [Internet] [Thesis]. University of North Carolina; 2018. [cited 2019 Nov 18]. Available from: https://cdr.lib.unc.edu/record/uuid:519f1372-1638-487e-b82e-0681308db3e0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hong L. CD30-REDIRECTED CHIMERIC ANTIGEN RECEPTOR T CELLS TARGET EMBRYONAL CARCINOMA VIA ANTIGEN-DEPENDENT AND FAS/FASL INTERACTIONS. [Thesis]. University of North Carolina; 2018. Available from: https://cdr.lib.unc.edu/record/uuid:519f1372-1638-487e-b82e-0681308db3e0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] [6] [7] [8] [9]

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