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You searched for subject:( Mammary Neoplasms Experimental pathology 60). Showing records 1 – 30 of 24291 total matches.

[1] [2] [3] [4] [5] … [810]

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1. Szafran, April Adams. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

The development of molecular imaging technologies has allowed biomedical researchers to study the process of cancer metastasis in animal models of disease. Bioluminescence imaging has… (more)

Subjects/Keywords: Antibodies, Monoclonal  – pharmacology<; br>; Antineoplastic Combined Chemotherapy<; br>; Protocols  – pharmacology<; br>; Bone Neoplasms<; br>; Breast Neoplasms<; br>; Diphosphonates  – pharmacology<; br>; Imidazoles  – pharmacology<; br>; Mammary Neoplasms, Experimental  – pathology<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Szafran, A. A. (2008). The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,927

Chicago Manual of Style (16th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,927.

MLA Handbook (7th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Web. 29 Mar 2020.

Vancouver:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927.

Council of Science Editors:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927

2. Beck, Benjamin H. (Benjamin Hester). Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.

Degree: PhD, 2009, University of Alabama – Birmingham

Unlike antigen-specific αβ-T cells, γδ-T cells can recognize and lyse cancerous cells rapidly upon encounter in a manner that does not require the recognition of… (more)

Subjects/Keywords: Adenocarcinoma  – therapy<; br>; Breast Neoplasms  – pathology<; br>; Immunotherapy, Adoptive<; br>; Mammary Neoplasms, Experimental  – therapy<; br>; Receptors, Antigen, T-Cell, gamma-delta  – immunology T-Lymphocyte Subsets  – transplantation

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APA (6th Edition):

Beck, B. H. (. H. (2009). Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1087

Chicago Manual of Style (16th Edition):

Beck, Benjamin H (Benjamin Hester). “Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1087.

MLA Handbook (7th Edition):

Beck, Benjamin H (Benjamin Hester). “Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.” 2009. Web. 29 Mar 2020.

Vancouver:

Beck BH(H. Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1087.

Council of Science Editors:

Beck BH(H. Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1087

3. Whitsett, Timothy Glynn. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.

Degree: PhD, 2007, University of Alabama – Birmingham

It has been established that the environment, including the diet, plays a critical role in a woman’s risk of breast cancer. Two dietary polyphenols that… (more)

Subjects/Keywords: Chemoprevention  – methods<; br>; Gene Expression Regulation  – drug effects<; br>; Genistein  – therapeutic use<; br>; Mammary Neoplasms, Experimental  – prevention & control<; br>; Stilbenes  – therapeutic use

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APA (6th Edition):

Whitsett, T. G. (2007). Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,539

Chicago Manual of Style (16th Edition):

Whitsett, Timothy Glynn. “Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,539.

MLA Handbook (7th Edition):

Whitsett, Timothy Glynn. “Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination.” 2007. Web. 29 Mar 2020.

Vancouver:

Whitsett TG. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,539.

Council of Science Editors:

Whitsett TG. Breast cancer chemoprevention with the natural polyphenols resveratrol and genistein, alone and in combination. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,539

4. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 29 Mar 2020.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045


University of Texas Southwestern Medical Center

5. Lynn, Kristi Dawn. Anti-VEGF Therapy Modulates Immune Cell Infiltration and Function in Multiple Breast Cancer Models.

Degree: 2011, University of Texas Southwestern Medical Center

 Breast cancer is the most frequently diagnosed malignancy in women in North America. Advancements in standard treatment regimens have improved the overall outlook for breast… (more)

Subjects/Keywords: Vascular Endothelial Growth Factor Receptor-2; Mammary Neoplasms, Experimental; Peptides

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APA (6th Edition):

Lynn, K. D. (2011). Anti-VEGF Therapy Modulates Immune Cell Infiltration and Function in Multiple Breast Cancer Models. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/948

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lynn, Kristi Dawn. “Anti-VEGF Therapy Modulates Immune Cell Infiltration and Function in Multiple Breast Cancer Models.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed March 29, 2020. http://hdl.handle.net/2152.5/948.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lynn, Kristi Dawn. “Anti-VEGF Therapy Modulates Immune Cell Infiltration and Function in Multiple Breast Cancer Models.” 2011. Web. 29 Mar 2020.

Vancouver:

Lynn KD. Anti-VEGF Therapy Modulates Immune Cell Infiltration and Function in Multiple Breast Cancer Models. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2152.5/948.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lynn KD. Anti-VEGF Therapy Modulates Immune Cell Infiltration and Function in Multiple Breast Cancer Models. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/948

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cape Town

6. Tiltman, Andrew John. The histogenesis of experimental bladder cancer.

Degree: Image, Division of Chemical Pathology, 1972, University of Cape Town

Subjects/Keywords: Neoplasms; Experimental - Pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tiltman, A. J. (1972). The histogenesis of experimental bladder cancer. (Thesis). University of Cape Town. Retrieved from http://hdl.handle.net/11427/26458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tiltman, Andrew John. “The histogenesis of experimental bladder cancer.” 1972. Thesis, University of Cape Town. Accessed March 29, 2020. http://hdl.handle.net/11427/26458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tiltman, Andrew John. “The histogenesis of experimental bladder cancer.” 1972. Web. 29 Mar 2020.

Vancouver:

Tiltman AJ. The histogenesis of experimental bladder cancer. [Internet] [Thesis]. University of Cape Town; 1972. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/11427/26458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tiltman AJ. The histogenesis of experimental bladder cancer. [Thesis]. University of Cape Town; 1972. Available from: http://hdl.handle.net/11427/26458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 29 Mar 2020.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

8. Hensel, Jonathan Adam. The role of LL-37 in prostate cancer and its potential as a therapeutic target.

Degree: 2011, University of Alabama – Birmingham

LL-37 is the only cathelicidin-derived anti-microbial peptide in humans and has been shown to stimulate proliferation, angiogenesis, cellular migration and inhibit apoptosis, in addition to… (more)

Subjects/Keywords: Antimicrobial Cationic Peptides  – metabolism<; br>; Cathelicidins<; br>; Molecular Targeted Therapy  – methods<; br>; Neoplasms, Hormone-Dependent<; br>; Prostatic Neoplasms  – genetics<; br>; Prostatic Neoplasms  – metabolism<; br>; Prostatic Neoplasms  – pathology

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APA (6th Edition):

Hensel, J. A. (2011). The role of LL-37 in prostate cancer and its potential as a therapeutic target. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Web. 29 Mar 2020.

Vancouver:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Internet] [Thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Patrícia Sayuri Suzuki. Papel do torque Teno Vírus (TTV) e do polimorfismo do gene CCR5 no desenvolvimento do câncer cervical.

Degree: 2008, Universidade Estadual de Londrina

High-risk human papillomaviruses (HPVs) are a required factor for cervical uterine cancer development. However, only a small proportion of HPV-infected women will develop cervical cancer;… (more)

Subjects/Keywords: Patologia experimental; Colo uterino - Câncer; Polimorfismo (Genética); Experimental pathology; Cervix Uteri - Neoplasms; Genetic polymorphisms

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APA (6th Edition):

Suzuki, P. S. (2008). Papel do torque Teno Vírus (TTV) e do polimorfismo do gene CCR5 no desenvolvimento do câncer cervical. (Thesis). Universidade Estadual de Londrina. Retrieved from http://www.bibliotecadigital.uel.br/document/?code=vtls000128854

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suzuki, Patrícia Sayuri. “Papel do torque Teno Vírus (TTV) e do polimorfismo do gene CCR5 no desenvolvimento do câncer cervical.” 2008. Thesis, Universidade Estadual de Londrina. Accessed March 29, 2020. http://www.bibliotecadigital.uel.br/document/?code=vtls000128854.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suzuki, Patrícia Sayuri. “Papel do torque Teno Vírus (TTV) e do polimorfismo do gene CCR5 no desenvolvimento do câncer cervical.” 2008. Web. 29 Mar 2020.

Vancouver:

Suzuki PS. Papel do torque Teno Vírus (TTV) e do polimorfismo do gene CCR5 no desenvolvimento do câncer cervical. [Internet] [Thesis]. Universidade Estadual de Londrina; 2008. [cited 2020 Mar 29]. Available from: http://www.bibliotecadigital.uel.br/document/?code=vtls000128854.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suzuki PS. Papel do torque Teno Vírus (TTV) e do polimorfismo do gene CCR5 no desenvolvimento do câncer cervical. [Thesis]. Universidade Estadual de Londrina; 2008. Available from: http://www.bibliotecadigital.uel.br/document/?code=vtls000128854

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 29 Mar 2020.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906


McGill University

11. Lam, Sonya Hoan Linh. Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes.

Degree: MS, Department of Biochemistry., 2008, McGill University

 ErbB2/Neu overexpression is observed in 20 – 30% of human mammary carcinomas and correlates with poor prognosis. We have demonstrated that four ErbB2/Neu tyrosine autophosphorylation sites… (more)

Subjects/Keywords: Mammary Neoplasms, Experimental  – metabolism.; Receptor, erbB-2  – metabolism.; Phosphorylation.

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APA (6th Edition):

Lam, S. H. L. (2008). Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile112529.pdf

Chicago Manual of Style (16th Edition):

Lam, Sonya Hoan Linh. “Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes.” 2008. Masters Thesis, McGill University. Accessed March 29, 2020. http://digitool.library.mcgill.ca/thesisfile112529.pdf.

MLA Handbook (7th Edition):

Lam, Sonya Hoan Linh. “Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes.” 2008. Web. 29 Mar 2020.

Vancouver:

Lam SHL. Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes. [Internet] [Masters thesis]. McGill University; 2008. [cited 2020 Mar 29]. Available from: http://digitool.library.mcgill.ca/thesisfile112529.pdf.

Council of Science Editors:

Lam SHL. Neu tyrosine autophosphorylation site mutants exhibit similar and distinct mammary tumour phenotypes. [Masters Thesis]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile112529.pdf


University of Texas Southwestern Medical Center

12. Cheng, Wing Yin. Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone.

Degree: 2016, University of Texas Southwestern Medical Center

 Tumor-associated macrophage (TAM) significantly contributes to tumorigenesis. Human cancer is enhanced by PPARgamma loss-of-function mutations, and inhibited by the thiazolidinedione (TZD) class of synthetic PPARgamma… (more)

Subjects/Keywords: Antineoplastic Agents; Cell Cycle Proteins; Macrophages; Mammary Neoplasms, Experimental; PPAR gamma; Receptors, G-Protein-Coupled; Thiazolidinediones

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APA (6th Edition):

Cheng, W. Y. (2016). Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheng, Wing Yin. “Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed March 29, 2020. http://hdl.handle.net/2152.5/5295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheng, Wing Yin. “Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone.” 2016. Web. 29 Mar 2020.

Vancouver:

Cheng WY. Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2152.5/5295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheng WY. Macrophage PPAR-Gamma Inhibits Gpr132 to Mediate the Anti-Tumor Effects of Rosiglitazone. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Bomben, Valerie Christine. Role of transient receptor potential canonical channels in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Gliomas, primary brain tumors derived from glial cells, constitute the majority of malignant tumors within the central nervous system. The most malignant of these tumors,… (more)

Subjects/Keywords: Brain Neoplasms  – pathology<; br>; Cell Cycle<; br>; Glioma  – pathology<; br>; Transient Receptor Potential Channels  – antagonists & inhibitors

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APA (6th Edition):

Bomben, V. C. (2010). Role of transient receptor potential canonical channels in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,642

Chicago Manual of Style (16th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,642.

MLA Handbook (7th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Web. 29 Mar 2020.

Vancouver:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642.

Council of Science Editors:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642


McGill University

14. Petkiewicz, Stephanie L. The Met receptor tyrosine kinase in mammary gland tumorigenesis and development.

Degree: PhD, Division of Experimental Medicine., 2007, McGill University

The Met receptor tyrosine kinase (RTK) is expressed in the mammary gland under both normal and neoplastic conditions. Overexpression of the Met receptor is found… (more)

Subjects/Keywords: Receptor Protein-Tyrosine Kinases.; Mammary Neoplasms, Experimental  – genetics.; Mammary Tumor Virus, Mouse  – genetics.

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APA (6th Edition):

Petkiewicz, S. L. (2007). The Met receptor tyrosine kinase in mammary gland tumorigenesis and development. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile103278.pdf

Chicago Manual of Style (16th Edition):

Petkiewicz, Stephanie L. “The Met receptor tyrosine kinase in mammary gland tumorigenesis and development.” 2007. Doctoral Dissertation, McGill University. Accessed March 29, 2020. http://digitool.library.mcgill.ca/thesisfile103278.pdf.

MLA Handbook (7th Edition):

Petkiewicz, Stephanie L. “The Met receptor tyrosine kinase in mammary gland tumorigenesis and development.” 2007. Web. 29 Mar 2020.

Vancouver:

Petkiewicz SL. The Met receptor tyrosine kinase in mammary gland tumorigenesis and development. [Internet] [Doctoral dissertation]. McGill University; 2007. [cited 2020 Mar 29]. Available from: http://digitool.library.mcgill.ca/thesisfile103278.pdf.

Council of Science Editors:

Petkiewicz SL. The Met receptor tyrosine kinase in mammary gland tumorigenesis and development. [Doctoral Dissertation]. McGill University; 2007. Available from: http://digitool.library.mcgill.ca/thesisfile103278.pdf

15. Atkinson, George P. (George Prescott). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma (GBM) is an incurable tumor of the central nervous system (CNS). Over the past 50 years, little progress has made in improving the quality… (more)

Subjects/Keywords: Glioblastoma  – pathology<; br>; Neoplasms<; br>; NF-kappa B  – metabolism<; br>; Peptidylprolyl Isomerase<; br>; STAT3 Transcription Factor

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APA (6th Edition):

Atkinson, G. P. (. P. (2009). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,812

Chicago Manual of Style (16th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,812.

MLA Handbook (7th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Web. 29 Mar 2020.

Vancouver:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812.

Council of Science Editors:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812

16. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 29 Mar 2020.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

17. Gavin, Cristin. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.

Degree: PhD, 2012, University of Alabama – Birmingham

Dynamic changes to the actin cytoskeleton are required for synaptic plasticity and long-term memory formation. However, the molecular mechanisms that mediate filamentous actin (F-actin) dynamics… (more)

Subjects/Keywords: Bone Neoplasms – secondary<; br>; Breast Neoplasms – pathology<; br>; Cell Communication<; br>; Cyclic AMP-Dependent Protein Kinases – metabolism.<; br>; Gap Junctions – metabolism.<; br>; Osteoblasts – cytology.<; br>; Phosphatidylinositol 3-Kinases – metabolism.

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APA (6th Edition):

Gavin, C. (2012). Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1396

Chicago Manual of Style (16th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1396.

MLA Handbook (7th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Web. 29 Mar 2020.

Vancouver:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396.

Council of Science Editors:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396


McGill University

18. Ponzo, Marisa Grace, 1980-. Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors.

Degree: PhD, Division of Experimental Medicine., 2009, McGill University

Breast cancer is a heterogeneous disease comprised of distinct biological entities that correlate with diverse clinical outcomes. Gene expression profiling has divided this heterogeneity into… (more)

Subjects/Keywords: Breast Neoplasms  – etiology.; Breast Neoplasms  – genetics.; Mammary Neoplasms, Experimental  – etiology.; Mammary Neoplasms, Experimental  – genetics.; Proto-Oncogene Proteins c-met  – physiology.; Mice, Transgenic.; Mice.

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APA (6th Edition):

Ponzo, Marisa Grace, 1. (2009). Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile115881.pdf

Chicago Manual of Style (16th Edition):

Ponzo, Marisa Grace, 1980-. “Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors.” 2009. Doctoral Dissertation, McGill University. Accessed March 29, 2020. http://digitool.library.mcgill.ca/thesisfile115881.pdf.

MLA Handbook (7th Edition):

Ponzo, Marisa Grace, 1980-. “Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors.” 2009. Web. 29 Mar 2020.

Vancouver:

Ponzo, Marisa Grace 1. Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors. [Internet] [Doctoral dissertation]. McGill University; 2009. [cited 2020 Mar 29]. Available from: http://digitool.library.mcgill.ca/thesisfile115881.pdf.

Council of Science Editors:

Ponzo, Marisa Grace 1. Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors. [Doctoral Dissertation]. McGill University; 2009. Available from: http://digitool.library.mcgill.ca/thesisfile115881.pdf

19. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 29 Mar 2020.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

20. Pandya, Ashka Y. Structural and functional analysis of KLF4.

Degree: PhD, 2007, University of Alabama – Birmingham

KLF4, a C2H2 type zinc finger transcription factor, plays an essential role in maturation of normal stratified squamous epithelium. In normal squamous epithelium its expression… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Nucleus  – metabolism<; br>; DNA-Binding Proteins  – biosynthesis<; br>; Kruppel-Like Transcription Factors<; br>; Prognosis Transcription Factors  – biosynthesis

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APA (6th Edition):

Pandya, A. Y. (2007). Structural and functional analysis of KLF4. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,531

Chicago Manual of Style (16th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,531.

MLA Handbook (7th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Web. 29 Mar 2020.

Vancouver:

Pandya AY. Structural and functional analysis of KLF4. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531.

Council of Science Editors:

Pandya AY. Structural and functional analysis of KLF4. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531

21. Baxley, Sarah. The role of WNT5A in mammary gland development.

Degree: PhD, 2010, University of Alabama – Birmingham

Transforming growth factor {esc}gb {esc}s(TGF-{esc}gb{esc}s) negatively regulates mammary gland development and requires Wnt5a to exert some of these effects on mammary gland development. Wnt5a is… (more)

Subjects/Keywords: Breast Neoplasms<; br>; Lactation<; br>; Mammary Glands, Animal – metabolism<; br>; Mammary Neoplasms, Experimental – metabolism.<; br>; Transforming Growth Factor beta – metabolism<; br>; Wnt Proteins – metabolism.

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APA (6th Edition):

Baxley, S. (2010). The role of WNT5A in mammary gland development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1812

Chicago Manual of Style (16th Edition):

Baxley, Sarah. “The role of WNT5A in mammary gland development.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1812.

MLA Handbook (7th Edition):

Baxley, Sarah. “The role of WNT5A in mammary gland development.” 2010. Web. 29 Mar 2020.

Vancouver:

Baxley S. The role of WNT5A in mammary gland development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1812.

Council of Science Editors:

Baxley S. The role of WNT5A in mammary gland development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1812


McGill University

22. Rossdeutscher, Lionel Philip David. The role of tumoral 1,25 dihydroxyvitamin D3 in inhibition of tumor growth and progression in the PyVMT MMTV#634 transgenic breast cancer model.

Degree: MS, Division of Experimental Medicine., 2007, McGill University

 min D3 must be metabolically activated by the liver to 25-hydroxyvitamin D3 (25OHD3) and then by the kidney 1alphahydroxylase (1alphaOHase) to become 1,25dihydroxyvitamin D 3… (more)

Subjects/Keywords: Mammary Neoplasms, Experimental  – genetics.; Mammary Tumor Virus, Mouse  – genetics.; Cell Transformation, Neoplastic  – genetics.; Hyperplasia.; Adenocarcinoma.; Lung Neoplasms  – secondary.; Calcitriol  – pharmacology.

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APA (6th Edition):

Rossdeutscher, L. P. D. (2007). The role of tumoral 1,25 dihydroxyvitamin D3 in inhibition of tumor growth and progression in the PyVMT MMTV#634 transgenic breast cancer model. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile112354.pdf

Chicago Manual of Style (16th Edition):

Rossdeutscher, Lionel Philip David. “The role of tumoral 1,25 dihydroxyvitamin D3 in inhibition of tumor growth and progression in the PyVMT MMTV#634 transgenic breast cancer model.” 2007. Masters Thesis, McGill University. Accessed March 29, 2020. http://digitool.library.mcgill.ca/thesisfile112354.pdf.

MLA Handbook (7th Edition):

Rossdeutscher, Lionel Philip David. “The role of tumoral 1,25 dihydroxyvitamin D3 in inhibition of tumor growth and progression in the PyVMT MMTV#634 transgenic breast cancer model.” 2007. Web. 29 Mar 2020.

Vancouver:

Rossdeutscher LPD. The role of tumoral 1,25 dihydroxyvitamin D3 in inhibition of tumor growth and progression in the PyVMT MMTV#634 transgenic breast cancer model. [Internet] [Masters thesis]. McGill University; 2007. [cited 2020 Mar 29]. Available from: http://digitool.library.mcgill.ca/thesisfile112354.pdf.

Council of Science Editors:

Rossdeutscher LPD. The role of tumoral 1,25 dihydroxyvitamin D3 in inhibition of tumor growth and progression in the PyVMT MMTV#634 transgenic breast cancer model. [Masters Thesis]. McGill University; 2007. Available from: http://digitool.library.mcgill.ca/thesisfile112354.pdf

23. Yang, Sherry W. (Sherry Wei). A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer.

Degree: PhD, 2010, University of Alabama – Birmingham

Ovarian cancer remains the most lethal gynecological malignancy in the U.S. Conventional therapies have limited therapeutic value due to advanced stage of the dis-ease at… (more)

Subjects/Keywords: Adenoviridae  – genetics<; br>; Adenoviridae  – physiology<; br>; DNA, Viral  – metabolism<; br>; Oncolytic Virotherapy  – methods<; br>; Ovarian Neoplasms  – pathology<; br>; Tissue Inhibitor of Metalloproteinase-2  – metabolism<; br>; Virus Replication  – physiology

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APA (6th Edition):

Yang, S. W. (. W. (2010). A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1116

Chicago Manual of Style (16th Edition):

Yang, Sherry W (Sherry Wei). “A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1116.

MLA Handbook (7th Edition):

Yang, Sherry W (Sherry Wei). “A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer.” 2010. Web. 29 Mar 2020.

Vancouver:

Yang SW(W. A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1116.

Council of Science Editors:

Yang SW(W. A TIMP2-armed conditionally-replicating adenovirus for the treatment of ovarian cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1116

24. Edmonds, Mick D. Brms1 Coordinately Regulates Microrna To Suppress Breast.

Degree: PhD, 2010, University of Alabama – Birmingham

The majority of cancer related mortality is attributed to complications associated with metastatic disease. Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis of multiple cancer… (more)

Subjects/Keywords: Antineoplastic Agents – pharmacology<; br>; Colonic Neoplasms – pathology<; br>; Cyclic Nucleotide Phosphodiesterases, Type 5 – metabolism.<; br>; Phosphodiesterase 5 Inhibitors – pharmacology.<; br>; Sulindac – pharmacology.<; br>; Transcriptional Activation – drug effects.<; br>; beta Catenin – genetics.

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APA (6th Edition):

Edmonds, M. D. (2010). Brms1 Coordinately Regulates Microrna To Suppress Breast. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1417

Chicago Manual of Style (16th Edition):

Edmonds, Mick D. “Brms1 Coordinately Regulates Microrna To Suppress Breast.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1417.

MLA Handbook (7th Edition):

Edmonds, Mick D. “Brms1 Coordinately Regulates Microrna To Suppress Breast.” 2010. Web. 29 Mar 2020.

Vancouver:

Edmonds MD. Brms1 Coordinately Regulates Microrna To Suppress Breast. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1417.

Council of Science Editors:

Edmonds MD. Brms1 Coordinately Regulates Microrna To Suppress Breast. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1417

25. Bhatta, Madhav P. Impact of immunosuppression on the incidence and clearance of human papillomavirus in HIV-infected women in Alabama.

Degree: PhD, 2007, University of Alabama – Birmingham

Persistent infection with certain "high-risk" types of human papillomavirus (HPV) infection is central to the development of cervical cancer. Human immunodeficiency virus (HIV)-infected women are… (more)

Subjects/Keywords: HIV Seropositivity  – complications Human papillomavirus 16  – immunology <; br>; Immunocompromised Host <; br>; Papillomavirus Infections  – complications <; br>; Uterine Cervical Neoplasms  – pathology <; br>; Uterine Cervical Neoplasms  – virology

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APA (6th Edition):

Bhatta, M. P. (2007). Impact of immunosuppression on the incidence and clearance of human papillomavirus in HIV-infected women in Alabama. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,305

Chicago Manual of Style (16th Edition):

Bhatta, Madhav P. “Impact of immunosuppression on the incidence and clearance of human papillomavirus in HIV-infected women in Alabama.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,305.

MLA Handbook (7th Edition):

Bhatta, Madhav P. “Impact of immunosuppression on the incidence and clearance of human papillomavirus in HIV-infected women in Alabama.” 2007. Web. 29 Mar 2020.

Vancouver:

Bhatta MP. Impact of immunosuppression on the incidence and clearance of human papillomavirus in HIV-infected women in Alabama. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,305.

Council of Science Editors:

Bhatta MP. Impact of immunosuppression on the incidence and clearance of human papillomavirus in HIV-infected women in Alabama. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,305


University of Western Ontario

26. Aldhafeeri, Hamad. CRISPR Screen for Identification of Kinases that Mediate Prostate Cancer Cell Invasion.

Degree: 2017, University of Western Ontario

 Metastasis is the primary cause of mortality in cancer patients. Inhibition of proteins that are involved in the regulation of metastasis are expected to suppress… (more)

Subjects/Keywords: Medical Pathology; Neoplasms

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APA (6th Edition):

Aldhafeeri, H. (2017). CRISPR Screen for Identification of Kinases that Mediate Prostate Cancer Cell Invasion. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aldhafeeri, Hamad. “CRISPR Screen for Identification of Kinases that Mediate Prostate Cancer Cell Invasion.” 2017. Thesis, University of Western Ontario. Accessed March 29, 2020. https://ir.lib.uwo.ca/etd/5205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aldhafeeri, Hamad. “CRISPR Screen for Identification of Kinases that Mediate Prostate Cancer Cell Invasion.” 2017. Web. 29 Mar 2020.

Vancouver:

Aldhafeeri H. CRISPR Screen for Identification of Kinases that Mediate Prostate Cancer Cell Invasion. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Mar 29]. Available from: https://ir.lib.uwo.ca/etd/5205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aldhafeeri H. CRISPR Screen for Identification of Kinases that Mediate Prostate Cancer Cell Invasion. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/5205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

27. Arnicke, Ulrich. Mammary carcinoma of the bitch : immunohistochemical detection of metastases and micrometastases in the axillary lymph nodes and bones.

Degree: 1999, Freie Universität Berlin

 182 samples of mammary tissue, 118 samples of axillary and axillary accessory lymph nodes as well as 526 bone samples and further routine tissue samples… (more)

Subjects/Keywords: mammary-gland-diseases; metastasis; pathology; mammary-glands; neoplasms; immunochemistry; lymph-nodes; bones; mammary-gland-neoplasms; 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft::630 Landwirtschaft und verwandte Bereiche

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APA (6th Edition):

Arnicke, U. (1999). Mammary carcinoma of the bitch : immunohistochemical detection of metastases and micrometastases in the axillary lymph nodes and bones. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8034

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arnicke, Ulrich. “Mammary carcinoma of the bitch : immunohistochemical detection of metastases and micrometastases in the axillary lymph nodes and bones.” 1999. Thesis, Freie Universität Berlin. Accessed March 29, 2020. http://dx.doi.org/10.17169/refubium-8034.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arnicke, Ulrich. “Mammary carcinoma of the bitch : immunohistochemical detection of metastases and micrometastases in the axillary lymph nodes and bones.” 1999. Web. 29 Mar 2020.

Vancouver:

Arnicke U. Mammary carcinoma of the bitch : immunohistochemical detection of metastases and micrometastases in the axillary lymph nodes and bones. [Internet] [Thesis]. Freie Universität Berlin; 1999. [cited 2020 Mar 29]. Available from: http://dx.doi.org/10.17169/refubium-8034.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arnicke U. Mammary carcinoma of the bitch : immunohistochemical detection of metastases and micrometastases in the axillary lymph nodes and bones. [Thesis]. Freie Universität Berlin; 1999. Available from: http://dx.doi.org/10.17169/refubium-8034

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Federal de Santa Maria

28. Monique Togni Martins. PREVALÊNCIA DOS TUMORES MAMÁRIOS EM GATAS NA REGIÃO CENTRAL DO RIO GRANDE DO SUL.

Degree: 2013, Universidade Federal de Santa Maria

Câncer é uma das principais causas de morte em cães e gatos, sendo o neoplasma mamário um dos mais frequentes. Embora estudos tenham sido realizados… (more)

Subjects/Keywords: tumores em felinos; neoplasmas mamários; glândula mamária; MEDICINA VETERINARIA; mammary neoplasms; mammary gland; feline tumors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martins, M. T. (2013). PREVALÊNCIA DOS TUMORES MAMÁRIOS EM GATAS NA REGIÃO CENTRAL DO RIO GRANDE DO SUL. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=5083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martins, Monique Togni. “PREVALÊNCIA DOS TUMORES MAMÁRIOS EM GATAS NA REGIÃO CENTRAL DO RIO GRANDE DO SUL.” 2013. Thesis, Universidade Federal de Santa Maria. Accessed March 29, 2020. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=5083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martins, Monique Togni. “PREVALÊNCIA DOS TUMORES MAMÁRIOS EM GATAS NA REGIÃO CENTRAL DO RIO GRANDE DO SUL.” 2013. Web. 29 Mar 2020.

Vancouver:

Martins MT. PREVALÊNCIA DOS TUMORES MAMÁRIOS EM GATAS NA REGIÃO CENTRAL DO RIO GRANDE DO SUL. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2013. [cited 2020 Mar 29]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=5083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martins MT. PREVALÊNCIA DOS TUMORES MAMÁRIOS EM GATAS NA REGIÃO CENTRAL DO RIO GRANDE DO SUL. [Thesis]. Universidade Federal de Santa Maria; 2013. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=5083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

29. Azhar, Raed A. Histological analysis of the kidney tumor-parenchyma interface.

Degree: MS, Clinical, Biomedical and Translational Investigations, 2014, University of Southern California

 Purpose: During enucleative partial nephrectomy (PN), excision is performed adjacent to the tumor edge. To better inform the oncological propriety of enucleative PN, we histologically… (more)

Subjects/Keywords: renal neoplasms; pathology; capsule; nephrectomy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Azhar, R. A. (2014). Histological analysis of the kidney tumor-parenchyma interface. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/512074/rec/3190

Chicago Manual of Style (16th Edition):

Azhar, Raed A. “Histological analysis of the kidney tumor-parenchyma interface.” 2014. Masters Thesis, University of Southern California. Accessed March 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/512074/rec/3190.

MLA Handbook (7th Edition):

Azhar, Raed A. “Histological analysis of the kidney tumor-parenchyma interface.” 2014. Web. 29 Mar 2020.

Vancouver:

Azhar RA. Histological analysis of the kidney tumor-parenchyma interface. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Mar 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/512074/rec/3190.

Council of Science Editors:

Azhar RA. Histological analysis of the kidney tumor-parenchyma interface. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/512074/rec/3190


East Carolina University

30. Corbin, Joshua Moses. TMEFF2 is an epigenetic modulator that promotes androgen independent growth in castration-resistant prostate cancer cells.

Degree: 2014, East Carolina University

 While the ability to detect PCa has improved significantly due to PSA screenings, the survival rate for men diagnosed with PCa has remained stagnant, and… (more)

Subjects/Keywords: Oncology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Corbin, J. M. (2014). TMEFF2 is an epigenetic modulator that promotes androgen independent growth in castration-resistant prostate cancer cells. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Corbin, Joshua Moses. “TMEFF2 is an epigenetic modulator that promotes androgen independent growth in castration-resistant prostate cancer cells.” 2014. Thesis, East Carolina University. Accessed March 29, 2020. http://hdl.handle.net/10342/4662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Corbin, Joshua Moses. “TMEFF2 is an epigenetic modulator that promotes androgen independent growth in castration-resistant prostate cancer cells.” 2014. Web. 29 Mar 2020.

Vancouver:

Corbin JM. TMEFF2 is an epigenetic modulator that promotes androgen independent growth in castration-resistant prostate cancer cells. [Internet] [Thesis]. East Carolina University; 2014. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/10342/4662.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Corbin JM. TMEFF2 is an epigenetic modulator that promotes androgen independent growth in castration-resistant prostate cancer cells. [Thesis]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4662

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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