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You searched for subject:( Kruppel Like Transcription Factors metabolism 60). Showing records 1 – 30 of 31582 total matches.

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1. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 22 Oct 2019.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

2. Liu, Zhaoli. KLF4 regulates notch1 expression and signaling during epithelial transformation.

Degree: PhD, 2006, University of Alabama – Birmingham

Notch1 and KLF4 function in the specification of epithelial cell fates, and each can act as a context-dependent oncogene or tumor suppressor. We report that… (more)

Subjects/Keywords: Breast Neoplasms <; br>; Epithelial Cells  – metabolism <; br>; Kruppel-Like Transcription Factors  – genetics <; br>; Receptor, Notch1  – metabolism

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APA (6th Edition):

Liu, Z. (2006). KLF4 regulates notch1 expression and signaling during epithelial transformation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,378

Chicago Manual of Style (16th Edition):

Liu, Zhaoli. “KLF4 regulates notch1 expression and signaling during epithelial transformation.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,378.

MLA Handbook (7th Edition):

Liu, Zhaoli. “KLF4 regulates notch1 expression and signaling during epithelial transformation.” 2006. Web. 22 Oct 2019.

Vancouver:

Liu Z. KLF4 regulates notch1 expression and signaling during epithelial transformation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,378.

Council of Science Editors:

Liu Z. KLF4 regulates notch1 expression and signaling during epithelial transformation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,378

3. Pandya, Ashka Y. Structural and functional analysis of KLF4.

Degree: PhD, 2007, University of Alabama – Birmingham

KLF4, a C2H2 type zinc finger transcription factor, plays an essential role in maturation of normal stratified squamous epithelium. In normal squamous epithelium its expression… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Nucleus  – metabolism<; br>; DNA-Binding Proteins  – biosynthesis<; br>; Kruppel-Like Transcription Factors<; br>; Prognosis Transcription Factors  – biosynthesis

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APA (6th Edition):

Pandya, A. Y. (2007). Structural and functional analysis of KLF4. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,531

Chicago Manual of Style (16th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,531.

MLA Handbook (7th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Web. 22 Oct 2019.

Vancouver:

Pandya AY. Structural and functional analysis of KLF4. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531.

Council of Science Editors:

Pandya AY. Structural and functional analysis of KLF4. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531

4. Shi, Lei. Molecular Mechanisms of Neurite Complexity in the Drosophila Brain: A Dissertation.

Degree: Neuroscience, Department of Neurobiology; Tzumin Lee Lab, 2010, U of Massachusetts : Med

  Development of functional neural circuits involves a series of complicated steps, including neurogenesis and neuronal morphogenesis. To understand the molecular mechasnims of neurite complexity,… (more)

Subjects/Keywords: Drosophila; neurite complexity; neuronal morphogenesis; Drosophila Proteins; Neurites; Cell Adhesion Molecules; Kruppel-Like Transcription Factors; Amino Acids, Peptides, and Proteins; Animal Experimentation and Research; Cells; Nervous System; Neuroscience and Neurobiology

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APA (6th Edition):

Shi, L. (2010). Molecular Mechanisms of Neurite Complexity in the Drosophila Brain: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/474

Chicago Manual of Style (16th Edition):

Shi, Lei. “Molecular Mechanisms of Neurite Complexity in the Drosophila Brain: A Dissertation.” 2010. Doctoral Dissertation, U of Massachusetts : Med. Accessed October 22, 2019. https://escholarship.umassmed.edu/gsbs_diss/474.

MLA Handbook (7th Edition):

Shi, Lei. “Molecular Mechanisms of Neurite Complexity in the Drosophila Brain: A Dissertation.” 2010. Web. 22 Oct 2019.

Vancouver:

Shi L. Molecular Mechanisms of Neurite Complexity in the Drosophila Brain: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2010. [cited 2019 Oct 22]. Available from: https://escholarship.umassmed.edu/gsbs_diss/474.

Council of Science Editors:

Shi L. Molecular Mechanisms of Neurite Complexity in the Drosophila Brain: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2010. Available from: https://escholarship.umassmed.edu/gsbs_diss/474

5. Ho, Shiuh-Rong. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.

Degree: PhD, 2010, University of Alabama – Birmingham

O-GlcNAcylation is an abundant and dynamic post-translational modification on serine and threonine residues of nuclear and cytoplasmic proteins. O-GlcNAc Transferase (OGT) and Nuclear Cytoplasmic O-GlcNAcase… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Gene Expression Regulation<; br>; Oxidative Stress  – genetics<; br>; Transcription Factors  – metabolism<; br>; Transcription, Genetic

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APA (6th Edition):

Ho, S. (2010). O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1162

Chicago Manual of Style (16th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1162.

MLA Handbook (7th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Web. 22 Oct 2019.

Vancouver:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162.

Council of Science Editors:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162

6. Sarof, Arjun. Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells.

Degree: MS, 2009, University of Alabama – Birmingham

Runx2 is a runt domain transcription factor essential for bone development and tooth morphogenesis and is required for epithelial-mesenchymal interactions that regulate skeletogenesis. Fibroblast growth… (more)

Subjects/Keywords: Epithelium  – physiology<; br>; Fibroblast Growth Factor 3<; br>; Tooth  – embryology<; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Sarof, A. (2009). Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,802

Chicago Manual of Style (16th Edition):

Sarof, Arjun. “Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,802.

MLA Handbook (7th Edition):

Sarof, Arjun. “Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells.” 2009. Web. 22 Oct 2019.

Vancouver:

Sarof A. Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,802.

Council of Science Editors:

Sarof A. Runx2 mediated transcriptional regulation of FGF3 in epithelial and mesenchymal cells. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,802

7. Hock, Thomas D. Regulation of the human heme oxygenase-1 gene.

Degree: PhD, 2007, University of Alabama – Birmingham

The heme oxygenase-1 (HO-1) gene encodes a microsomal enzyme that catalyzes the conversion of heme to carbon monoxide, Iron, and biliverdin. HO-1 transcription is induced… (more)

Subjects/Keywords: Heme Oxygenase-1  – genetics <; br>; Proto-Oncogene Proteins c-jun  – metabolism <; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Hock, T. D. (2007). Regulation of the human heme oxygenase-1 gene. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,126

Chicago Manual of Style (16th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,126.

MLA Handbook (7th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Web. 22 Oct 2019.

Vancouver:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126.

Council of Science Editors:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126

8. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 22 Oct 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

9. He, Ti. Molecular regulation of Pax5-mediated biological functions.

Degree: PhD, 2008, University of Alabama – Birmingham

B lineage cells are major players in the adaptive immune system. Pax5 is essential for B lineage cell development and function. Pax5 controls B lineage… (more)

Subjects/Keywords: B-Cell-Specific Activator Protein <; br>; Gene Expression Regulation <; br>; Histone Acetyltransferases  – metabolism <; br>; Transcription Factors <; br>; Transcription, Genetic

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APA (6th Edition):

He, T. (2008). Molecular regulation of Pax5-mediated biological functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,329

Chicago Manual of Style (16th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,329.

MLA Handbook (7th Edition):

He, Ti. “Molecular regulation of Pax5-mediated biological functions.” 2008. Web. 22 Oct 2019.

Vancouver:

He T. Molecular regulation of Pax5-mediated biological functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329.

Council of Science Editors:

He T. Molecular regulation of Pax5-mediated biological functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,329

10. Ahronian, Leanne G. Identification and Characteristics of Factors Regulating Hepatocellular Carcinoma Progression and Metastasis: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology Department, 2014, U of Massachusetts : Med

  Hepatocellular carcinoma (HCC) is a common malignancy of the liver that is one of the most frequent causes of cancer-related death in the world.… (more)

Subjects/Keywords: Hepatocellular Carcinoma; Gene Expression Profiling; p53 Genes; Kruppel-Like Transcription Factors; Liver Neoplasms; Point Mutation; rho GTP-Binding Proteins; Transcription Factors; Tumor Suppressor Protein p53; Cancer Biology; Digestive System Diseases; Molecular Genetics; Neoplasms

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APA (6th Edition):

Ahronian, L. G. (2014). Identification and Characteristics of Factors Regulating Hepatocellular Carcinoma Progression and Metastasis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/705

Chicago Manual of Style (16th Edition):

Ahronian, Leanne G. “Identification and Characteristics of Factors Regulating Hepatocellular Carcinoma Progression and Metastasis: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed October 22, 2019. http://escholarship.umassmed.edu/gsbs_diss/705.

MLA Handbook (7th Edition):

Ahronian, Leanne G. “Identification and Characteristics of Factors Regulating Hepatocellular Carcinoma Progression and Metastasis: A Dissertation.” 2014. Web. 22 Oct 2019.

Vancouver:

Ahronian LG. Identification and Characteristics of Factors Regulating Hepatocellular Carcinoma Progression and Metastasis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2019 Oct 22]. Available from: http://escholarship.umassmed.edu/gsbs_diss/705.

Council of Science Editors:

Ahronian LG. Identification and Characteristics of Factors Regulating Hepatocellular Carcinoma Progression and Metastasis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/705

11. Zheng, Ying. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Janus Kinase (JAK)-Signal Transducer and Activator of Transcription (STAT) signaling is involved in regulation of cell survival, proliferation and differentiation. JAK tyrosine kinases can be… (more)

Subjects/Keywords: Casein Kinase II  – metabolism<; br>; Hematologic Neoplasms  – metabolism<; br>; JNK Mitogen-Activated Protein Kinases  – metabolism<; br>; Polycythemia Vera  – metabolism<; br>; STAT Transcription Factors  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Zheng, Y. (2010). A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1168

Chicago Manual of Style (16th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1168.

MLA Handbook (7th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Web. 22 Oct 2019.

Vancouver:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168.

Council of Science Editors:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168

12. Polter, Abigail Marie. Regulation of GSK3 in the pathophysiology and treatment of mood disorders.

Degree: PhD, 2010, University of Alabama – Birmingham

Mood disorders are devastating psychiatric illnesses that will affect as many as one in every five persons worldwide over the course of their lifetime. Significant… (more)

Subjects/Keywords: Behavior, Animal  – physiology<; br>; Brain  – metabolism<; br>; Exploratory Behavior  – physiology<; br>; Forkhead Transcription Factors  – metabolism<; br>; Glycogen Synthase Kinase 3<; br>; Mood Disorders<; br>; Receptor, Serotonin, 5-HT1A  – metabolism<; br>; Serine  – metabolism<; br>; Serotonin  – metabolism<; br>; Signal Transduction

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APA (6th Edition):

Polter, A. M. (2010). Regulation of GSK3 in the pathophysiology and treatment of mood disorders. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1108

Chicago Manual of Style (16th Edition):

Polter, Abigail Marie. “Regulation of GSK3 in the pathophysiology and treatment of mood disorders.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1108.

MLA Handbook (7th Edition):

Polter, Abigail Marie. “Regulation of GSK3 in the pathophysiology and treatment of mood disorders.” 2010. Web. 22 Oct 2019.

Vancouver:

Polter AM. Regulation of GSK3 in the pathophysiology and treatment of mood disorders. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1108.

Council of Science Editors:

Polter AM. Regulation of GSK3 in the pathophysiology and treatment of mood disorders. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1108

13. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 22 Oct 2019.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913


University of Queensland

14. Ilsley, Melissa. Investigating the Molecular Pathogenesis of Inherited Childhood Anaemia.

Degree: School of Biomedical Sciences, 2017, University of Queensland

Subjects/Keywords: Transcription factors; Kruppel-like factors; Erythropoiesis; KLF1; KLF3; E2f2; Transcription; ChIP-seq; 4sU-RNA-seq; transcription factor competition; 0601 Biochemistry and Cell Biology; 0604 Genetics

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APA (6th Edition):

Ilsley, M. (2017). Investigating the Molecular Pathogenesis of Inherited Childhood Anaemia. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:683437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ilsley, Melissa. “Investigating the Molecular Pathogenesis of Inherited Childhood Anaemia.” 2017. Thesis, University of Queensland. Accessed October 22, 2019. http://espace.library.uq.edu.au/view/UQ:683437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ilsley, Melissa. “Investigating the Molecular Pathogenesis of Inherited Childhood Anaemia.” 2017. Web. 22 Oct 2019.

Vancouver:

Ilsley M. Investigating the Molecular Pathogenesis of Inherited Childhood Anaemia. [Internet] [Thesis]. University of Queensland; 2017. [cited 2019 Oct 22]. Available from: http://espace.library.uq.edu.au/view/UQ:683437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ilsley M. Investigating the Molecular Pathogenesis of Inherited Childhood Anaemia. [Thesis]. University of Queensland; 2017. Available from: http://espace.library.uq.edu.au/view/UQ:683437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Harms, Paul William. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.

Degree: PhD, 2006, University of Alabama – Birmingham

Growth factor signals often regulate similar cellular processes both during embryogenesis and in adult homeostasis. Stringent control of these signals ensures proper embryonic development and… (more)

Subjects/Keywords: Homeodomain Proteins <; br>; Membrane Proteins  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Proteins <; br>; Signal Transduction  – physiology <; br>; Transcription Factors <; br>; Xenopus Proteins

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APA (6th Edition):

Harms, P. W. (2006). Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,369

Chicago Manual of Style (16th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,369.

MLA Handbook (7th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Web. 22 Oct 2019.

Vancouver:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369.

Council of Science Editors:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369

16. Lai, Yun-Ju. Role of TRIP6 in LPA-induced cell migration.

Degree: PhD, 2007, University of Alabama – Birmingham

The LIM domain-containing Thyroid Receptor-Interacting Protein 6 (TRIP6) is a zyxin family member that has been implicated in actin dynamics and cell motility. In this… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing<; br>; Carrier Proteins  – physiology<; br>; Cell Movement<; br>; Feedback, Biochemical<; br>; Lysophospholipids<; br>; Protein-Tyrosine Kinases  – physiology<; br>; Receptors, G-Protein-Coupled  – metabolism<; br>; Transcription Factors

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APA (6th Edition):

Lai, Y. (2007). Role of TRIP6 in LPA-induced cell migration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,453

Chicago Manual of Style (16th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,453.

MLA Handbook (7th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Web. 22 Oct 2019.

Vancouver:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453.

Council of Science Editors:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453

17. Winkelbauer, Marlene Elizabeth. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.

Degree: PhD, 2007, University of Alabama – Birmingham

Numerous disorders are characterized by the presence of cystic lesions within the kidney. The proteins associated with these disorders often localize to cilia, and improper… (more)

Subjects/Keywords: Caenorhabditis elegans  – genetics<; br>; Caenorhabditis elegans Proteins  – genetics<; br>; Cilia  – metabolism<; br>; Neurons, Afferent  – physiology<; br>; Protein Transport<; br>; Transcription Factors  – metabolism

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APA (6th Edition):

Winkelbauer, M. E. (2007). Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,619

Chicago Manual of Style (16th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,619.

MLA Handbook (7th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Web. 22 Oct 2019.

Vancouver:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619.

Council of Science Editors:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619

18. Zhao, Xueyan. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.

Degree: PhD, 2008, University of Alabama – Birmingham

Matrix metalloprotinases are zinc-dependent endopeptidases with broad substrates from extracellular matrix proteins to bioactive molecules. Physiologically, they regulate tissue remodeling and immune responses. However, in… (more)

Subjects/Keywords: Gene Expression Regulation, Enzymologic<; br>; Interferon-beta  – pharmacology<; br>; Interferon-Stimulated Gene Factor 3  – metabolism<; br>; Interferon-beta/pharmacology<; br>; Matrix Metalloproteinase 9  – genetics<; br>; Matrix Metalloproteinase 9  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Transcriptional Activation  – genetics

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APA (6th Edition):

Zhao, X. (2008). Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,542

Chicago Manual of Style (16th Edition):

Zhao, Xueyan. “Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,542.

MLA Handbook (7th Edition):

Zhao, Xueyan. “Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.” 2008. Web. 22 Oct 2019.

Vancouver:

Zhao X. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,542.

Council of Science Editors:

Zhao X. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,542

19. Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye.

Degree: PhD, 2008, University of Alabama – Birmingham

Development of the vertebrate eye is controlled by a network of regulatory genes including those in the basic loop-helix-loop (bHLH) family. In this study, we… (more)

Subjects/Keywords: Amacrine Cells  – physiology<; br>; Avian Proteins  – metabolism<; br>; Basic Helix-Loop-Helix Transcription Factors  – metabolism<; br>; Gene Expression Regulation, Developmental  – physiology<; br>; Nerve Tissue Proteins  – genetics<; br>; Nuclear Reprogramming<; br>; Retina  – cytology<; br>; Retinal Neurons  – cytology Retinal Pigment Epithelium  – cytology<; br>; Stem Cells  – cytology

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APA (6th Edition):

Mao,Weiming. (2008). The role of bHLH gene ash1 in the developing chick eye. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Mao,Weiming. “The role of bHLH gene ash1 in the developing chick eye.” 2008. Web. 22 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Mao,Weiming. The role of bHLH gene ash1 in the developing chick eye. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,527

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

20. Maynard, Craig Lueland. IL-10-competent regulatory T cells: development, phenotype and function.

Degree: PhD, 2007, University of Alabama – Birmingham

In order to avoid chronic cell activation and inflammation the immune system has developed numerous mechanisms that cooperate to induce and/or maintain what is known… (more)

Subjects/Keywords: Cell Differentiation  – immunology<; br>; Forkhead Transcription Factors  – immunology<; br>; Inflammation  – immunology<; br>; Interleukin-10  – metabolism<; br>; T-Lymphocyte Subsets  – cytology<; br>; Transforming Growth Factor beta

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APA (6th Edition):

Maynard, C. L. (2007). IL-10-competent regulatory T cells: development, phenotype and function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,529

Chicago Manual of Style (16th Edition):

Maynard, Craig Lueland. “IL-10-competent regulatory T cells: development, phenotype and function.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,529.

MLA Handbook (7th Edition):

Maynard, Craig Lueland. “IL-10-competent regulatory T cells: development, phenotype and function.” 2007. Web. 22 Oct 2019.

Vancouver:

Maynard CL. IL-10-competent regulatory T cells: development, phenotype and function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,529.

Council of Science Editors:

Maynard CL. IL-10-competent regulatory T cells: development, phenotype and function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,529


University of New South Wales

21. Dewi, Vitri Aryani. Phosphorylation of KLF3 affects its DNA binding activity and biological function.

Degree: Biotechnology & Biomolecular Sciences, 2012, University of New South Wales

 Krüppel-Like Factor 3 (KLF3) is a broadly expressed zinc-finger transcriptional repressor, which binds to CACCC-boxes and GC-rich regions in the promoters and enhancers of its… (more)

Subjects/Keywords: DNA binding; Kruppel-Like Factor; KLF

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APA (6th Edition):

Dewi, V. A. (2012). Phosphorylation of KLF3 affects its DNA binding activity and biological function. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52940 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11618/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Dewi, Vitri Aryani. “Phosphorylation of KLF3 affects its DNA binding activity and biological function.” 2012. Doctoral Dissertation, University of New South Wales. Accessed October 22, 2019. http://handle.unsw.edu.au/1959.4/52940 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11618/SOURCE01?view=true.

MLA Handbook (7th Edition):

Dewi, Vitri Aryani. “Phosphorylation of KLF3 affects its DNA binding activity and biological function.” 2012. Web. 22 Oct 2019.

Vancouver:

Dewi VA. Phosphorylation of KLF3 affects its DNA binding activity and biological function. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2019 Oct 22]. Available from: http://handle.unsw.edu.au/1959.4/52940 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11618/SOURCE01?view=true.

Council of Science Editors:

Dewi VA. Phosphorylation of KLF3 affects its DNA binding activity and biological function. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52940 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11618/SOURCE01?view=true


University of New South Wales

22. Knights, Alexander. Understanding the transcriptional landscape in immunity and metabolism.

Degree: Biotechnology & Biomolecular Sciences, 2018, University of New South Wales

 Recent decades have heralded a greater appreciation of the intricate crosstalk that exists between immunity and metabolism, significantly expanding therapeutic horizons. A fundamental layer of… (more)

Subjects/Keywords: Gene regulation; Immunity; Metabolism; Transcription factors

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APA (6th Edition):

Knights, A. (2018). Understanding the transcriptional landscape in immunity and metabolism. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60437

Chicago Manual of Style (16th Edition):

Knights, Alexander. “Understanding the transcriptional landscape in immunity and metabolism.” 2018. Doctoral Dissertation, University of New South Wales. Accessed October 22, 2019. http://handle.unsw.edu.au/1959.4/60437.

MLA Handbook (7th Edition):

Knights, Alexander. “Understanding the transcriptional landscape in immunity and metabolism.” 2018. Web. 22 Oct 2019.

Vancouver:

Knights A. Understanding the transcriptional landscape in immunity and metabolism. [Internet] [Doctoral dissertation]. University of New South Wales; 2018. [cited 2019 Oct 22]. Available from: http://handle.unsw.edu.au/1959.4/60437.

Council of Science Editors:

Knights A. Understanding the transcriptional landscape in immunity and metabolism. [Doctoral Dissertation]. University of New South Wales; 2018. Available from: http://handle.unsw.edu.au/1959.4/60437

23. Liu, Shanrun. Erythropoiesis in the absence of adult hemoglobin.

Degree: PhD, 2011, University of Alabama – Birmingham

The mammalian erythrocyte is a highly specialized blood cell that differentiates via an orderly series of committed progenitors in the bone marrow in a process… (more)

Subjects/Keywords: beta-Globins  – genetics<; br>; erythroid Kruppel-like factor<; br>; Erythropoiesis  – genetics<; br>; Fetal Hemoglobin  – genetics<; br>; gamma-Globins  – genetics<; br>; Thalassemia

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APA (6th Edition):

Liu, S. (2011). Erythropoiesis in the absence of adult hemoglobin. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1157

Chicago Manual of Style (16th Edition):

Liu, Shanrun. “Erythropoiesis in the absence of adult hemoglobin.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1157.

MLA Handbook (7th Edition):

Liu, Shanrun. “Erythropoiesis in the absence of adult hemoglobin.” 2011. Web. 22 Oct 2019.

Vancouver:

Liu S. Erythropoiesis in the absence of adult hemoglobin. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1157.

Council of Science Editors:

Liu S. Erythropoiesis in the absence of adult hemoglobin. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1157

24. Rayburn, Elizabeth R. Novel immunomodulatory oligonucleotides for cancer therapy.

Degree: PhD, 2007, University of Alabama – Birmingham

Prostate and colon cancer are two of the most prevalent cancers in the United States. Cancer therapy, including surgery, chemotherapy, radiotherapy and biological therapy, often… (more)

Subjects/Keywords: Adenocarcinoma  – drug therapy<; br>; Antineoplastic Agents  – therapeutic use<; br>; Antineoplastic Combined Chemotherapy Protocols  – therapeutic use<; br>; Apoptosis  – drug effects<; br>; Colonic Neoplasms  – drug therapy<; br>; Deoxycytidine  – analogs & derivatives<; br>; Immunologic Factors  – therapeutic use<; br>; Oligonucleotides  – therapeutic use<; br>; Prostatic Neoplasms  – drug therapy<; br>; Taxoids  – therapeutic use<; br>; Toll-Like Receptor 9  – agonists<; br>; Tumor Suppressor Protein p53  – metabolism

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APA (6th Edition):

Rayburn, E. R. (2007). Novel immunomodulatory oligonucleotides for cancer therapy. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,463

Chicago Manual of Style (16th Edition):

Rayburn, Elizabeth R. “Novel immunomodulatory oligonucleotides for cancer therapy.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,463.

MLA Handbook (7th Edition):

Rayburn, Elizabeth R. “Novel immunomodulatory oligonucleotides for cancer therapy.” 2007. Web. 22 Oct 2019.

Vancouver:

Rayburn ER. Novel immunomodulatory oligonucleotides for cancer therapy. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,463.

Council of Science Editors:

Rayburn ER. Novel immunomodulatory oligonucleotides for cancer therapy. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,463

25. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

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APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 22 Oct 2019.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

26. Atout, Reem (Reem N.). Expression of Sox 11 during tooth formation.

Degree: MS, 2009, University of Alabama – Birmingham

Our laboratory has shown that a human autosomal recessive (AR) form of radicular dentin dysplasia (RDD) is caused by an alteration in the transcription factor… (more)

Subjects/Keywords: Dental Pulp Cavity  – abnormalities<; br>; Dentin Dysplasia  – genetics<; br>; NFI Transcription Factors  – genetics<; br>; SOX Transcription Factors  – genetics<; br>; Tooth Root  – abnormalities

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APA (6th Edition):

Atout, R. (. N. ). (2009). Expression of Sox 11 during tooth formation. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1016

Chicago Manual of Style (16th Edition):

Atout, Reem (Reem N ). “Expression of Sox 11 during tooth formation.” 2009. Masters Thesis, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1016.

MLA Handbook (7th Edition):

Atout, Reem (Reem N ). “Expression of Sox 11 during tooth formation.” 2009. Web. 22 Oct 2019.

Vancouver:

Atout R(N). Expression of Sox 11 during tooth formation. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1016.

Council of Science Editors:

Atout R(N). Expression of Sox 11 during tooth formation. [Masters Thesis]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1016

27. Laver, Travis. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.

Degree: PhD, 2008, University of Alabama – Birmingham

Interleukin-8 (IL-8) is a potent chemoattractant of numerous cells, particularly neutrophils, in the innate immune response. In addition to immune functions, IL-8 is known to… (more)

Subjects/Keywords: Astrocytoma <; br>; Gene Expression Regulation <; br>; Interferon-beta  – physiology <; br>; Interferon-Stimulated Gene Factor 3, gamma Subunit  – metabolism <; br>; Interleukin-8 <; br>; STAT1 Transcription Factor  – metabolism <; br>; STAT2 Transcription Factor  – metabolism

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APA (6th Edition):

Laver, T. (2008). Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,204

Chicago Manual of Style (16th Edition):

Laver, Travis. “Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,204.

MLA Handbook (7th Edition):

Laver, Travis. “Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression.” 2008. Web. 22 Oct 2019.

Vancouver:

Laver T. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,204.

Council of Science Editors:

Laver T. Mechanism of inteferon-beta-mediated inhibition of IL-8 gene expression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,204

28. Szul, Tomasz J. The role of GBF1 in Golgi biogenesis and secretory traffic.

Degree: PhD, 2009, University of Alabama – Birmingham

The secretory pathway within a cell consists of series of membrane compartments connected by shuttling secretory vesicles containing cargo that travel from endoplasmic reticulum (ER)… (more)

Subjects/Keywords: ADP-Ribosylation Factors  – metabolism<; br>; Coat Protein Complex I  – metabolism<; br>; Endoplasmic Reticulum  – metabolism<; br>; Golgi Apparatus  – metabolism<; br>; Guanine Nucleotide Exchange Factors  – metabolism<; br>; Membrane Proteins  – metabolism

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APA (6th Edition):

Szul, T. J. (2009). The role of GBF1 in Golgi biogenesis and secretory traffic. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,577

Chicago Manual of Style (16th Edition):

Szul, Tomasz J. “The role of GBF1 in Golgi biogenesis and secretory traffic.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,577.

MLA Handbook (7th Edition):

Szul, Tomasz J. “The role of GBF1 in Golgi biogenesis and secretory traffic.” 2009. Web. 22 Oct 2019.

Vancouver:

Szul TJ. The role of GBF1 in Golgi biogenesis and secretory traffic. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,577.

Council of Science Editors:

Szul TJ. The role of GBF1 in Golgi biogenesis and secretory traffic. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,577

29. Genovese, Nicholas J. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.

Degree: PhD, 2010, University of Alabama – Birmingham

Though human papillomavirus infection of the human epidermis is epidemiologically widespread and typically benign, manipulation of the cell cycle within host tissues during infections can… (more)

Subjects/Keywords: Cell Cycle<; br>; Cell Transformation, Viral<; br>; Human papillomavirus 16  – metabolism<; br>; Keratinocytes<; br>; Oncogene Proteins, Viral  – metabolism<; br>; Papillomaviridae  – physiology<; br>; Papillomavirus E7 Proteins  – metabolism<; br>; Receptors, Estrogen  – metabolism<; br>; Retinoblastoma-Like Protein p130  – metabolism<; br>; S Phase

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APA (6th Edition):

Genovese, N. J. (2010). The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1158

Chicago Manual of Style (16th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1158.

MLA Handbook (7th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Web. 22 Oct 2019.

Vancouver:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158.

Council of Science Editors:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158

30. Lenarcic, Erik Michael. In vivo interactions between virus genomes and host proteins for members of the order Picornavirales.

Degree: PhD, 2011, University of Alabama – Birmingham

A significant number of pathogens of economical and medical importance are positive sense RNA viruses. Their genomes enter host cells and subvert them to support… (more)

Subjects/Keywords: Eukaryotic Initiation Factor-3<; br>; Peptide Initiation Factors  – metabolism<; br>; Picornaviridae  – chemistry<; br>; Poliovirus<; br>; Protein Biosynthesis<; br>; Ribosomes  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lenarcic, E. M. (2011). In vivo interactions between virus genomes and host proteins for members of the order Picornavirales. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,949

Chicago Manual of Style (16th Edition):

Lenarcic, Erik Michael. “In vivo interactions between virus genomes and host proteins for members of the order Picornavirales.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 22, 2019. http://contentdm.mhsl.uab.edu/u?/etd,949.

MLA Handbook (7th Edition):

Lenarcic, Erik Michael. “In vivo interactions between virus genomes and host proteins for members of the order Picornavirales.” 2011. Web. 22 Oct 2019.

Vancouver:

Lenarcic EM. In vivo interactions between virus genomes and host proteins for members of the order Picornavirales. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 22]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,949.

Council of Science Editors:

Lenarcic EM. In vivo interactions between virus genomes and host proteins for members of the order Picornavirales. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,949

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