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You searched for subject:( Inflammation). Showing records 1 – 30 of 5781 total matches.

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1. Millet, Patrick. INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES.

Degree: 2015, Wake Forest University

 Sepsis is serious medical condition which kills millions of people worldwide each year. In the United States, severe sepsis has a mortality rate of 20-30%,… (more)

Subjects/Keywords: Inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Millet, P. (2015). INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/57421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Millet, Patrick. “INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES.” 2015. Thesis, Wake Forest University. Accessed September 23, 2020. http://hdl.handle.net/10339/57421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Millet, Patrick. “INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES.” 2015. Web. 23 Sep 2020.

Vancouver:

Millet P. INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES. [Internet] [Thesis]. Wake Forest University; 2015. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10339/57421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Millet P. INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES. [Thesis]. Wake Forest University; 2015. Available from: http://hdl.handle.net/10339/57421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Neyrinck-Leglantier, Déborah. Implication de zonula occludens-1 dans les processus pro-inflammatoires associés à la progression métastatique des cancers broncho-pulmonaires : Involvement of zonula occludens-1 in pro-inflammatory processes associated to metastatic progression of lung cancers.

Degree: Docteur es, Médecine, 2018, Reims

Zonula occludens 1 (ZO-1) est une protéine sous-membranaire des jonctions serrées impliquée dans l’organisation structurale des cellules épithéliales. Au cours de la progression tumorale, associée… (more)

Subjects/Keywords: Inflammation; Inflammation; 570

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APA (6th Edition):

Neyrinck-Leglantier, D. (2018). Implication de zonula occludens-1 dans les processus pro-inflammatoires associés à la progression métastatique des cancers broncho-pulmonaires : Involvement of zonula occludens-1 in pro-inflammatory processes associated to metastatic progression of lung cancers. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2018REIMM212

Chicago Manual of Style (16th Edition):

Neyrinck-Leglantier, Déborah. “Implication de zonula occludens-1 dans les processus pro-inflammatoires associés à la progression métastatique des cancers broncho-pulmonaires : Involvement of zonula occludens-1 in pro-inflammatory processes associated to metastatic progression of lung cancers.” 2018. Doctoral Dissertation, Reims. Accessed September 23, 2020. http://www.theses.fr/2018REIMM212.

MLA Handbook (7th Edition):

Neyrinck-Leglantier, Déborah. “Implication de zonula occludens-1 dans les processus pro-inflammatoires associés à la progression métastatique des cancers broncho-pulmonaires : Involvement of zonula occludens-1 in pro-inflammatory processes associated to metastatic progression of lung cancers.” 2018. Web. 23 Sep 2020.

Vancouver:

Neyrinck-Leglantier D. Implication de zonula occludens-1 dans les processus pro-inflammatoires associés à la progression métastatique des cancers broncho-pulmonaires : Involvement of zonula occludens-1 in pro-inflammatory processes associated to metastatic progression of lung cancers. [Internet] [Doctoral dissertation]. Reims; 2018. [cited 2020 Sep 23]. Available from: http://www.theses.fr/2018REIMM212.

Council of Science Editors:

Neyrinck-Leglantier D. Implication de zonula occludens-1 dans les processus pro-inflammatoires associés à la progression métastatique des cancers broncho-pulmonaires : Involvement of zonula occludens-1 in pro-inflammatory processes associated to metastatic progression of lung cancers. [Doctoral Dissertation]. Reims; 2018. Available from: http://www.theses.fr/2018REIMM212

3. Santinon, François. Rôle du TNFR2 exprimé à la surface des lymphocytes T régulateurs dans l’inflammation dépendante du TNFα : Role of TNFR2 on Tregs in TNF-α-mediated inflammation.

Degree: Docteur es, Biologie, 2018, Sorbonne Paris Cité

La polyarthrite rhumatoïde (PR) est une maladie inflammatoire chronique d’étiologie inconnue. L’inflammation présente dans cette pathologie est fortement dépendante de la cytokine pro-inflammatoire qu’est le… (more)

Subjects/Keywords: Inflammation chronique; Chronic inflammation

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APA (6th Edition):

Santinon, F. (2018). Rôle du TNFR2 exprimé à la surface des lymphocytes T régulateurs dans l’inflammation dépendante du TNFα : Role of TNFR2 on Tregs in TNF-α-mediated inflammation. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCD006

Chicago Manual of Style (16th Edition):

Santinon, François. “Rôle du TNFR2 exprimé à la surface des lymphocytes T régulateurs dans l’inflammation dépendante du TNFα : Role of TNFR2 on Tregs in TNF-α-mediated inflammation.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed September 23, 2020. http://www.theses.fr/2018USPCD006.

MLA Handbook (7th Edition):

Santinon, François. “Rôle du TNFR2 exprimé à la surface des lymphocytes T régulateurs dans l’inflammation dépendante du TNFα : Role of TNFR2 on Tregs in TNF-α-mediated inflammation.” 2018. Web. 23 Sep 2020.

Vancouver:

Santinon F. Rôle du TNFR2 exprimé à la surface des lymphocytes T régulateurs dans l’inflammation dépendante du TNFα : Role of TNFR2 on Tregs in TNF-α-mediated inflammation. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Sep 23]. Available from: http://www.theses.fr/2018USPCD006.

Council of Science Editors:

Santinon F. Rôle du TNFR2 exprimé à la surface des lymphocytes T régulateurs dans l’inflammation dépendante du TNFα : Role of TNFR2 on Tregs in TNF-α-mediated inflammation. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCD006

4. Lyroni, Konstantina. Regulation of inflammation in obesity.

Degree: 2018, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Inflammation underlies a variety of physiological and pathological processes. Over the years tremendous progress has been made in understanding the cellular and molecular events that… (more)

Subjects/Keywords: φλεγμονή; inflammation

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APA (6th Edition):

Lyroni, K. (2018). Regulation of inflammation in obesity. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/43821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lyroni, Konstantina. “Regulation of inflammation in obesity.” 2018. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed September 23, 2020. http://hdl.handle.net/10442/hedi/43821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lyroni, Konstantina. “Regulation of inflammation in obesity.” 2018. Web. 23 Sep 2020.

Vancouver:

Lyroni K. Regulation of inflammation in obesity. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2018. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10442/hedi/43821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lyroni K. Regulation of inflammation in obesity. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2018. Available from: http://hdl.handle.net/10442/hedi/43821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

5. Metzger, Corinne Elizabeth. Inflammatory Bowel Disease and Bone: Mechanisms and Interventions for Inflammation-Induced Bone Alterations.

Degree: PhD, Kinesiology, 2018, Texas A&M University

 Inflammatory bowel disease (IBD) effects approximately 1.6 million people in the United States with the incidence and prevalence increasing worldwide. All current treatments for IBD… (more)

Subjects/Keywords: Inflammation; Bone

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APA (6th Edition):

Metzger, C. E. (2018). Inflammatory Bowel Disease and Bone: Mechanisms and Interventions for Inflammation-Induced Bone Alterations. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/173354

Chicago Manual of Style (16th Edition):

Metzger, Corinne Elizabeth. “Inflammatory Bowel Disease and Bone: Mechanisms and Interventions for Inflammation-Induced Bone Alterations.” 2018. Doctoral Dissertation, Texas A&M University. Accessed September 23, 2020. http://hdl.handle.net/1969.1/173354.

MLA Handbook (7th Edition):

Metzger, Corinne Elizabeth. “Inflammatory Bowel Disease and Bone: Mechanisms and Interventions for Inflammation-Induced Bone Alterations.” 2018. Web. 23 Sep 2020.

Vancouver:

Metzger CE. Inflammatory Bowel Disease and Bone: Mechanisms and Interventions for Inflammation-Induced Bone Alterations. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1969.1/173354.

Council of Science Editors:

Metzger CE. Inflammatory Bowel Disease and Bone: Mechanisms and Interventions for Inflammation-Induced Bone Alterations. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/173354


University of Rochester

6. Saperstein, Sara C. Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors.

Degree: PhD, 2009, University of Rochester

 Tumor necrosis factor-α (TNF-α) and interleukin-1β (IL-1β) are key mediators of numerous lung inflammatory diseases. Induced by similar stimuli, in many cell types IL-1β modifies… (more)

Subjects/Keywords: Inflammation; Receptors

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APA (6th Edition):

Saperstein, S. C. (2009). Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8631

Chicago Manual of Style (16th Edition):

Saperstein, Sara C. “Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors.” 2009. Doctoral Dissertation, University of Rochester. Accessed September 23, 2020. http://hdl.handle.net/1802/8631.

MLA Handbook (7th Edition):

Saperstein, Sara C. “Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors.” 2009. Web. 23 Sep 2020.

Vancouver:

Saperstein SC. Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1802/8631.

Council of Science Editors:

Saperstein SC. Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8631


University of Otago

7. Park, Sun-Jin. RAGE Biology and Inflammation .

Degree: 2011, University of Otago

 The receptor for advanced glycation end products (RAGE) is a cell surface signal transduction receptor that amplifies inflammation, principally through the activation of nuclear factor… (more)

Subjects/Keywords: Inflammation; RAGE

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APA (6th Edition):

Park, S. (2011). RAGE Biology and Inflammation . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/1651

Chicago Manual of Style (16th Edition):

Park, Sun-Jin. “RAGE Biology and Inflammation .” 2011. Doctoral Dissertation, University of Otago. Accessed September 23, 2020. http://hdl.handle.net/10523/1651.

MLA Handbook (7th Edition):

Park, Sun-Jin. “RAGE Biology and Inflammation .” 2011. Web. 23 Sep 2020.

Vancouver:

Park S. RAGE Biology and Inflammation . [Internet] [Doctoral dissertation]. University of Otago; 2011. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10523/1651.

Council of Science Editors:

Park S. RAGE Biology and Inflammation . [Doctoral Dissertation]. University of Otago; 2011. Available from: http://hdl.handle.net/10523/1651

8. Schnegg, Caroline Isabel. RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO.

Degree: 2012, Wake Forest University

 As a result of improvements in cancer therapy and health care, the population of long-term cancer survivors is growing. For these approximately 12 million long-term… (more)

Subjects/Keywords: Inflammation

…and Inflammation Classically, radiation-induced late normal tissue injury was thought to be… …which contribute to increased oxidative stress and inflammation [36-38]. This model… …act as second messengers to induce inflammation [43]. Although it was once… …believed that the brain was not susceptible to inflammation, it is now recognized that… …and/or inflammation, therefore, appears to be one of the mechanisms underlying radiation… 

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APA (6th Edition):

Schnegg, C. I. (2012). RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/37442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schnegg, Caroline Isabel. “RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO.” 2012. Thesis, Wake Forest University. Accessed September 23, 2020. http://hdl.handle.net/10339/37442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schnegg, Caroline Isabel. “RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO.” 2012. Web. 23 Sep 2020.

Vancouver:

Schnegg CI. RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO. [Internet] [Thesis]. Wake Forest University; 2012. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10339/37442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schnegg CI. RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO. [Thesis]. Wake Forest University; 2012. Available from: http://hdl.handle.net/10339/37442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Gauthier, Alexandre. Rôle des protéases à sérine du polynucléaire neutrophile dans l'inflammation associée à la mucoviscidose : No title available.

Degree: Docteur es, Sciences de la vie et de la santé, spécialité Biochimie, 2009, Université François-Rabelais de Tours

La mucoviscidose est une maladie génétique caractérisée par une inflammation pulmonairepersistante du poumon résultant d’un recrutement massif de polynucléaires neutrophiles quisécrètent trois protéases à sérine,… (more)

Subjects/Keywords: NETs; Inflammation

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APA (6th Edition):

Gauthier, A. (2009). Rôle des protéases à sérine du polynucléaire neutrophile dans l'inflammation associée à la mucoviscidose : No title available. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2009TOUR4040

Chicago Manual of Style (16th Edition):

Gauthier, Alexandre. “Rôle des protéases à sérine du polynucléaire neutrophile dans l'inflammation associée à la mucoviscidose : No title available.” 2009. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed September 23, 2020. http://www.theses.fr/2009TOUR4040.

MLA Handbook (7th Edition):

Gauthier, Alexandre. “Rôle des protéases à sérine du polynucléaire neutrophile dans l'inflammation associée à la mucoviscidose : No title available.” 2009. Web. 23 Sep 2020.

Vancouver:

Gauthier A. Rôle des protéases à sérine du polynucléaire neutrophile dans l'inflammation associée à la mucoviscidose : No title available. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2009. [cited 2020 Sep 23]. Available from: http://www.theses.fr/2009TOUR4040.

Council of Science Editors:

Gauthier A. Rôle des protéases à sérine du polynucléaire neutrophile dans l'inflammation associée à la mucoviscidose : No title available. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2009. Available from: http://www.theses.fr/2009TOUR4040


Tulane University

10. Sriyotha, Phanuwat. Ethanol-mediated dysregulation of cytokines and human anti-microbial peptide cathelicidin.

Degree: 2020, Tulane University

[email protected]

Ethanol consumption is known to increase the prevalence and severity of respiratory infection and impaired immunity. The relationships between ethanol exposure, vitamin D levels,… (more)

Subjects/Keywords: Systemic inflammation

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APA (6th Edition):

Sriyotha, P. (2020). Ethanol-mediated dysregulation of cytokines and human anti-microbial peptide cathelicidin. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:120547

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sriyotha, Phanuwat. “Ethanol-mediated dysregulation of cytokines and human anti-microbial peptide cathelicidin.” 2020. Thesis, Tulane University. Accessed September 23, 2020. https://digitallibrary.tulane.edu/islandora/object/tulane:120547.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sriyotha, Phanuwat. “Ethanol-mediated dysregulation of cytokines and human anti-microbial peptide cathelicidin.” 2020. Web. 23 Sep 2020.

Vancouver:

Sriyotha P. Ethanol-mediated dysregulation of cytokines and human anti-microbial peptide cathelicidin. [Internet] [Thesis]. Tulane University; 2020. [cited 2020 Sep 23]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:120547.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sriyotha P. Ethanol-mediated dysregulation of cytokines and human anti-microbial peptide cathelicidin. [Thesis]. Tulane University; 2020. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:120547

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

11. Balhara, Jyoti. Role of PTX3 in allergic airway inflammation.

Degree: Immunology, 2013, University of Manitoba

 Thesis Abstract: Pentraxin 3 (PTX3) is a pattern recognition receptor that plays an important role in providing immunity against various pulmonary inflammatory conditions. Having known… (more)

Subjects/Keywords: asthma; inflammation

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APA (6th Edition):

Balhara, J. (2013). Role of PTX3 in allergic airway inflammation. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balhara, Jyoti. “Role of PTX3 in allergic airway inflammation.” 2013. Thesis, University of Manitoba. Accessed September 23, 2020. http://hdl.handle.net/1993/31935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balhara, Jyoti. “Role of PTX3 in allergic airway inflammation.” 2013. Web. 23 Sep 2020.

Vancouver:

Balhara J. Role of PTX3 in allergic airway inflammation. [Internet] [Thesis]. University of Manitoba; 2013. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1993/31935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balhara J. Role of PTX3 in allergic airway inflammation. [Thesis]. University of Manitoba; 2013. Available from: http://hdl.handle.net/1993/31935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Molnarfi, Nicolas. Production of pro- and anti-inflammatory cytokines in human monocytes: regulation and signaling.

Degree: 2005, Université de Genève

 Les tissus vivants répondent aux agressions par l'inflammation qui élimine les agents préjudiciables et initie la guérison des tissus lésés. Les dérèglements de l'inflammation entraînent… (more)

Subjects/Keywords: inflammation

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APA (6th Edition):

Molnarfi, N. (2005). Production of pro- and anti-inflammatory cytokines in human monocytes: regulation and signaling. (Thesis). Université de Genève. Retrieved from http://doc.rero.ch/record/5559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Molnarfi, Nicolas. “Production of pro- and anti-inflammatory cytokines in human monocytes: regulation and signaling.” 2005. Thesis, Université de Genève. Accessed September 23, 2020. http://doc.rero.ch/record/5559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Molnarfi, Nicolas. “Production of pro- and anti-inflammatory cytokines in human monocytes: regulation and signaling.” 2005. Web. 23 Sep 2020.

Vancouver:

Molnarfi N. Production of pro- and anti-inflammatory cytokines in human monocytes: regulation and signaling. [Internet] [Thesis]. Université de Genève; 2005. [cited 2020 Sep 23]. Available from: http://doc.rero.ch/record/5559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Molnarfi N. Production of pro- and anti-inflammatory cytokines in human monocytes: regulation and signaling. [Thesis]. Université de Genève; 2005. Available from: http://doc.rero.ch/record/5559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

13. Boyanapalli, Sarandeep S S. Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects.

Degree: PhD, Pharmaceutical Science, 2016, Rutgers University

Chronic inflammation is considered to play an important factor in neoplastic progression via the induction of reactive oxygen, reactive nitrogen species and induction of growth… (more)

Subjects/Keywords: Phytochemicals; Inflammation

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APA (6th Edition):

Boyanapalli, S. S. S. (2016). Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/49917/

Chicago Manual of Style (16th Edition):

Boyanapalli, Sarandeep S S. “Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects.” 2016. Doctoral Dissertation, Rutgers University. Accessed September 23, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/49917/.

MLA Handbook (7th Edition):

Boyanapalli, Sarandeep S S. “Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects.” 2016. Web. 23 Sep 2020.

Vancouver:

Boyanapalli SSS. Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects. [Internet] [Doctoral dissertation]. Rutgers University; 2016. [cited 2020 Sep 23]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/49917/.

Council of Science Editors:

Boyanapalli SSS. Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects. [Doctoral Dissertation]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/49917/


University of Houston

14. Zambrano Cobo, Amarayca. Inflammation in Zebrafish Models.

Degree: MS, Biomedical Engineering, 2015, University of Houston

 The zebrafish is an outstanding model for in-vivo imaging of inflammation due to its optical translucency and the ability of transgenic lines to express fluorescent… (more)

Subjects/Keywords: Zebrafish; Inflammation

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APA (6th Edition):

Zambrano Cobo, A. (2015). Inflammation in Zebrafish Models. (Masters Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/4848

Chicago Manual of Style (16th Edition):

Zambrano Cobo, Amarayca. “Inflammation in Zebrafish Models.” 2015. Masters Thesis, University of Houston. Accessed September 23, 2020. http://hdl.handle.net/10657/4848.

MLA Handbook (7th Edition):

Zambrano Cobo, Amarayca. “Inflammation in Zebrafish Models.” 2015. Web. 23 Sep 2020.

Vancouver:

Zambrano Cobo A. Inflammation in Zebrafish Models. [Internet] [Masters thesis]. University of Houston; 2015. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10657/4848.

Council of Science Editors:

Zambrano Cobo A. Inflammation in Zebrafish Models. [Masters Thesis]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/4848

15. Benkdane, Merieme. Rôle protecteur des cellules de Küpffer de phénotype M2 anti-inflammatoire dans la maladie alcoolique du foie : Protective role of M2 anti-inflammatory Kupffer cells in alcoholic liver disease.

Degree: Docteur es, Physiopathologie, 2012, Université Paris-Est

 L'alcool est la première cause de maladie hépatique en France, et la maladie alcoolique du foie est responsable de près de 7000 décès par an.… (more)

Subjects/Keywords: Alcool; Macrophages; Inflammation; Alcohol; Macrophages; Inflammation; 616.3

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APA (6th Edition):

Benkdane, M. (2012). Rôle protecteur des cellules de Küpffer de phénotype M2 anti-inflammatoire dans la maladie alcoolique du foie : Protective role of M2 anti-inflammatory Kupffer cells in alcoholic liver disease. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0062

Chicago Manual of Style (16th Edition):

Benkdane, Merieme. “Rôle protecteur des cellules de Küpffer de phénotype M2 anti-inflammatoire dans la maladie alcoolique du foie : Protective role of M2 anti-inflammatory Kupffer cells in alcoholic liver disease.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed September 23, 2020. http://www.theses.fr/2012PEST0062.

MLA Handbook (7th Edition):

Benkdane, Merieme. “Rôle protecteur des cellules de Küpffer de phénotype M2 anti-inflammatoire dans la maladie alcoolique du foie : Protective role of M2 anti-inflammatory Kupffer cells in alcoholic liver disease.” 2012. Web. 23 Sep 2020.

Vancouver:

Benkdane M. Rôle protecteur des cellules de Küpffer de phénotype M2 anti-inflammatoire dans la maladie alcoolique du foie : Protective role of M2 anti-inflammatory Kupffer cells in alcoholic liver disease. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2020 Sep 23]. Available from: http://www.theses.fr/2012PEST0062.

Council of Science Editors:

Benkdane M. Rôle protecteur des cellules de Küpffer de phénotype M2 anti-inflammatoire dans la maladie alcoolique du foie : Protective role of M2 anti-inflammatory Kupffer cells in alcoholic liver disease. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0062


University of Guelph

16. Hann, Melissa. Anti-inflammatory Activity of Isomaltodextrin in a C57BL/6NCrl Mouse Model with Lipopolysaccharide-induced Low-grade Chronic Inflammation.

Degree: MS, Department of Food Science, 2018, University of Guelph

 The purpose of this study was to identify the anti-inflammatory activity and mechanism of isomaltodextrin in a C57BL/6NCrl mouse model with lipopolysaccharide-induced systemic low-grade chronic… (more)

Subjects/Keywords: isomaltodextrin; low-grade inflammation; chronic inflammation; prebiotic

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APA (6th Edition):

Hann, M. (2018). Anti-inflammatory Activity of Isomaltodextrin in a C57BL/6NCrl Mouse Model with Lipopolysaccharide-induced Low-grade Chronic Inflammation. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14124

Chicago Manual of Style (16th Edition):

Hann, Melissa. “Anti-inflammatory Activity of Isomaltodextrin in a C57BL/6NCrl Mouse Model with Lipopolysaccharide-induced Low-grade Chronic Inflammation.” 2018. Masters Thesis, University of Guelph. Accessed September 23, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14124.

MLA Handbook (7th Edition):

Hann, Melissa. “Anti-inflammatory Activity of Isomaltodextrin in a C57BL/6NCrl Mouse Model with Lipopolysaccharide-induced Low-grade Chronic Inflammation.” 2018. Web. 23 Sep 2020.

Vancouver:

Hann M. Anti-inflammatory Activity of Isomaltodextrin in a C57BL/6NCrl Mouse Model with Lipopolysaccharide-induced Low-grade Chronic Inflammation. [Internet] [Masters thesis]. University of Guelph; 2018. [cited 2020 Sep 23]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14124.

Council of Science Editors:

Hann M. Anti-inflammatory Activity of Isomaltodextrin in a C57BL/6NCrl Mouse Model with Lipopolysaccharide-induced Low-grade Chronic Inflammation. [Masters Thesis]. University of Guelph; 2018. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14124


University of California – Merced

17. Al-Kuhlani, Mufadhal Mohammed. Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection.

Degree: Quantitative and Systems Biology, 2013, University of California – Merced

 Chlamydia trachomatis is the most common bacterial sexually-transmitted disease (STD) around the world. The response of the immune system to eliminate C. trachomatis infection through… (more)

Subjects/Keywords: Immunology; Chlamydia; Inflammation

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APA (6th Edition):

Al-Kuhlani, M. M. (2013). Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection. (Thesis). University of California – Merced. Retrieved from http://www.escholarship.org/uc/item/1m5036rd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Kuhlani, Mufadhal Mohammed. “Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection.” 2013. Thesis, University of California – Merced. Accessed September 23, 2020. http://www.escholarship.org/uc/item/1m5036rd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Kuhlani, Mufadhal Mohammed. “Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection.” 2013. Web. 23 Sep 2020.

Vancouver:

Al-Kuhlani MM. Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection. [Internet] [Thesis]. University of California – Merced; 2013. [cited 2020 Sep 23]. Available from: http://www.escholarship.org/uc/item/1m5036rd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Kuhlani MM. Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection. [Thesis]. University of California – Merced; 2013. Available from: http://www.escholarship.org/uc/item/1m5036rd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

18. Osborne, Amber Valeria. The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation.

Degree: PhD, Pathology;, 2010, University of Utah

 This dissertation examines the relationship between inflammatory cytokines and nicotinic acetylcholine receptors (nAChR). WT and nAChRa7 null mice were exposed to 4.8kJ/m2 of UVB on… (more)

Subjects/Keywords: Nicotine Receptors; Inflammation

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APA (6th Edition):

Osborne, A. V. (2010). The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195

Chicago Manual of Style (16th Edition):

Osborne, Amber Valeria. “The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation.” 2010. Doctoral Dissertation, University of Utah. Accessed September 23, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195.

MLA Handbook (7th Edition):

Osborne, Amber Valeria. “The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation.” 2010. Web. 23 Sep 2020.

Vancouver:

Osborne AV. The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Sep 23]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195.

Council of Science Editors:

Osborne AV. The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195


University of Alberta

19. van Diepen, Sean. The prognosis and outcomes of patients with a ST-segment elevation myocardial infarction and the biochemical and clinical inflammatory response.

Degree: MS, Department of Medicine, 2013, University of Alberta

 Acute myocardial infarction, and specifically coronary plaque rupture, has been associated with a systemic inflammatory cascade. Using multiple large clinical trial datasets, we sought to… (more)

Subjects/Keywords: myocardial infarction; inflammation

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APA (6th Edition):

van Diepen, S. (2013). The prognosis and outcomes of patients with a ST-segment elevation myocardial infarction and the biochemical and clinical inflammatory response. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/h702q735f

Chicago Manual of Style (16th Edition):

van Diepen, Sean. “The prognosis and outcomes of patients with a ST-segment elevation myocardial infarction and the biochemical and clinical inflammatory response.” 2013. Masters Thesis, University of Alberta. Accessed September 23, 2020. https://era.library.ualberta.ca/files/h702q735f.

MLA Handbook (7th Edition):

van Diepen, Sean. “The prognosis and outcomes of patients with a ST-segment elevation myocardial infarction and the biochemical and clinical inflammatory response.” 2013. Web. 23 Sep 2020.

Vancouver:

van Diepen S. The prognosis and outcomes of patients with a ST-segment elevation myocardial infarction and the biochemical and clinical inflammatory response. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2020 Sep 23]. Available from: https://era.library.ualberta.ca/files/h702q735f.

Council of Science Editors:

van Diepen S. The prognosis and outcomes of patients with a ST-segment elevation myocardial infarction and the biochemical and clinical inflammatory response. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/h702q735f


Universiteit Utrecht

20. Emans, T.W. Inflammation in salt-induced hypertension.

Degree: 2014, Universiteit Utrecht

 Hypertension in the human population became like a worldwide epidemic, increasing risk for cardiovascular diseases. The most important contributor to hypertension among life style factors… (more)

Subjects/Keywords: salt; hypertension; inflammation

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APA (6th Edition):

Emans, T. W. (2014). Inflammation in salt-induced hypertension. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/289594

Chicago Manual of Style (16th Edition):

Emans, T W. “Inflammation in salt-induced hypertension.” 2014. Masters Thesis, Universiteit Utrecht. Accessed September 23, 2020. http://dspace.library.uu.nl:8080/handle/1874/289594.

MLA Handbook (7th Edition):

Emans, T W. “Inflammation in salt-induced hypertension.” 2014. Web. 23 Sep 2020.

Vancouver:

Emans TW. Inflammation in salt-induced hypertension. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2020 Sep 23]. Available from: http://dspace.library.uu.nl:8080/handle/1874/289594.

Council of Science Editors:

Emans TW. Inflammation in salt-induced hypertension. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/289594


Drexel University

21. Shenoda, Botros. A Role for XIST in the Regulation of Inflammatory Response by Female Cells.

Degree: 2017, Drexel University

Biological sex influences inflammatory response [1]. There is a greater incidence of acute inflammation in men, and chronic inflammation in women. X-inactive specific transcript (XIST)… (more)

Subjects/Keywords: Pharmacology; Physiology; Inflammation

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APA (6th Edition):

Shenoda, B. (2017). A Role for XIST in the Regulation of Inflammatory Response by Female Cells. (Thesis). Drexel University. Retrieved from https://idea.library.drexel.edu/islandora/object/idea%3A9409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shenoda, Botros. “A Role for XIST in the Regulation of Inflammatory Response by Female Cells.” 2017. Thesis, Drexel University. Accessed September 23, 2020. https://idea.library.drexel.edu/islandora/object/idea%3A9409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shenoda, Botros. “A Role for XIST in the Regulation of Inflammatory Response by Female Cells.” 2017. Web. 23 Sep 2020.

Vancouver:

Shenoda B. A Role for XIST in the Regulation of Inflammatory Response by Female Cells. [Internet] [Thesis]. Drexel University; 2017. [cited 2020 Sep 23]. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A9409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shenoda B. A Role for XIST in the Regulation of Inflammatory Response by Female Cells. [Thesis]. Drexel University; 2017. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A9409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

22. Sinha, Siddhartha. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.

Degree: M.S., Physiology, Physiology, 2014, Cornell University

 Atherosclerosis is a decades-long process whose patients often remain asymptomatic until after a heart attack or stroke. Novel therapeutic approaches focus on tuning the body's… (more)

Subjects/Keywords: Atherosclerosis; Inflammation; Macrophage

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APA (6th Edition):

Sinha, S. (2014). Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. (Masters Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/37128

Chicago Manual of Style (16th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.” 2014. Masters Thesis, Cornell University. Accessed September 23, 2020. http://hdl.handle.net/1813/37128.

MLA Handbook (7th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.” 2014. Web. 23 Sep 2020.

Vancouver:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Internet] [Masters thesis]. Cornell University; 2014. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1813/37128.

Council of Science Editors:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Masters Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/37128


Cornell University

23. Mitchell, Michael. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation.

Degree: PhD, Biomedical Engineering, 2014, Cornell University

 The application of fluid shear stress on leukocytes and tumor cells in the circulation plays a critical role in the progression of inflammation and cancer… (more)

Subjects/Keywords: Inflammation; Metastasis; Nanomedicine

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APA (6th Edition):

Mitchell, M. (2014). Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/39384

Chicago Manual of Style (16th Edition):

Mitchell, Michael. “Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation.” 2014. Doctoral Dissertation, Cornell University. Accessed September 23, 2020. http://hdl.handle.net/1813/39384.

MLA Handbook (7th Edition):

Mitchell, Michael. “Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation.” 2014. Web. 23 Sep 2020.

Vancouver:

Mitchell M. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1813/39384.

Council of Science Editors:

Mitchell M. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/39384


Vanderbilt University

24. Stouch, Ashley Nicole. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.

Degree: PhD, Cell and Developmental Biology, 2014, Vanderbilt University

 Bronchopulmonary dysplasia is a life-threatening lung disease affecting low birth weight preterm infants. While the occurrence of BPD is correlated with chorioamnionitis, the origination and… (more)

Subjects/Keywords: macrophage; inflammasome; inflammation

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APA (6th Edition):

Stouch, A. N. (2014). NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15353

Chicago Manual of Style (16th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2020. http://hdl.handle.net/1803/15353.

MLA Handbook (7th Edition):

Stouch, Ashley Nicole. “NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages.” 2014. Web. 23 Sep 2020.

Vancouver:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1803/15353.

Council of Science Editors:

Stouch AN. NF-kappa B signaling and inflammasome activation in developing fetal lung macrophages. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/15353

25. Campbell, Jennie S. Investigating novel players in inflammatory cell migration to sites of tissue damage in vivo.

Degree: PhD, 2020, University of Bristol

 The use of model organisms is vital for the improvement of our understanding of dynamic immunological processes. Previous work in the developing Drosophila embryo has… (more)

Subjects/Keywords: Drosophila; Zebrafish; Inflammation

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APA (6th Edition):

Campbell, J. S. (2020). Investigating novel players in inflammatory cell migration to sites of tissue damage in vivo. (Doctoral Dissertation). University of Bristol. Retrieved from http://hdl.handle.net/1983/e5fb59ce-3715-4679-8113-97be14546369

Chicago Manual of Style (16th Edition):

Campbell, Jennie S. “Investigating novel players in inflammatory cell migration to sites of tissue damage in vivo.” 2020. Doctoral Dissertation, University of Bristol. Accessed September 23, 2020. http://hdl.handle.net/1983/e5fb59ce-3715-4679-8113-97be14546369.

MLA Handbook (7th Edition):

Campbell, Jennie S. “Investigating novel players in inflammatory cell migration to sites of tissue damage in vivo.” 2020. Web. 23 Sep 2020.

Vancouver:

Campbell JS. Investigating novel players in inflammatory cell migration to sites of tissue damage in vivo. [Internet] [Doctoral dissertation]. University of Bristol; 2020. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1983/e5fb59ce-3715-4679-8113-97be14546369.

Council of Science Editors:

Campbell JS. Investigating novel players in inflammatory cell migration to sites of tissue damage in vivo. [Doctoral Dissertation]. University of Bristol; 2020. Available from: http://hdl.handle.net/1983/e5fb59ce-3715-4679-8113-97be14546369


University of Newcastle

26. Berthon, Bronwyn. Investigating the link between corticosteroids and diet in adults with asthma.

Degree: PhD, 2014, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Asthma is public health problem, affecting adults and children of all ages globally. As there is no cure… (more)

Subjects/Keywords: asthma; diet; inflammation

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APA (6th Edition):

Berthon, B. (2014). Investigating the link between corticosteroids and diet in adults with asthma. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1051152

Chicago Manual of Style (16th Edition):

Berthon, Bronwyn. “Investigating the link between corticosteroids and diet in adults with asthma.” 2014. Doctoral Dissertation, University of Newcastle. Accessed September 23, 2020. http://hdl.handle.net/1959.13/1051152.

MLA Handbook (7th Edition):

Berthon, Bronwyn. “Investigating the link between corticosteroids and diet in adults with asthma.” 2014. Web. 23 Sep 2020.

Vancouver:

Berthon B. Investigating the link between corticosteroids and diet in adults with asthma. [Internet] [Doctoral dissertation]. University of Newcastle; 2014. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1959.13/1051152.

Council of Science Editors:

Berthon B. Investigating the link between corticosteroids and diet in adults with asthma. [Doctoral Dissertation]. University of Newcastle; 2014. Available from: http://hdl.handle.net/1959.13/1051152


University of Toronto

27. Choi, So Jung. Effect of Remote Ischemic Conditioning on Lipopolysaccharide-induced Pulmonary Inflammation.

Degree: 2016, University of Toronto

Remote ischemic preconditioning (RIPC), an intervention whereby an extremity undergoes brief repeated cycles of ischemia/reperfusion (I/R), has been shown to exert protective effects on distant… (more)

Subjects/Keywords: Inflammation; leukocytes; 0564

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APA (6th Edition):

Choi, S. J. (2016). Effect of Remote Ischemic Conditioning on Lipopolysaccharide-induced Pulmonary Inflammation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/72691

Chicago Manual of Style (16th Edition):

Choi, So Jung. “Effect of Remote Ischemic Conditioning on Lipopolysaccharide-induced Pulmonary Inflammation.” 2016. Masters Thesis, University of Toronto. Accessed September 23, 2020. http://hdl.handle.net/1807/72691.

MLA Handbook (7th Edition):

Choi, So Jung. “Effect of Remote Ischemic Conditioning on Lipopolysaccharide-induced Pulmonary Inflammation.” 2016. Web. 23 Sep 2020.

Vancouver:

Choi SJ. Effect of Remote Ischemic Conditioning on Lipopolysaccharide-induced Pulmonary Inflammation. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/1807/72691.

Council of Science Editors:

Choi SJ. Effect of Remote Ischemic Conditioning on Lipopolysaccharide-induced Pulmonary Inflammation. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72691

28. Al-Rifai, Sarah. Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique : Role of hypothalamic resistin in the onset of hypothalamic inflammation and insulin resistance : impact of acute or chronic high fat diet consumption.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2019, Université Paris-Saclay (ComUE)

 La prévalence de l’obésité est en net progrès et constitue un problème majeur de santé publique. Cette pathologie est d’autant plus dangereuse qu’elle s’accompagne d’un… (more)

Subjects/Keywords: Résistine; Inflammation; Insulino-Résistance; Diabète; Inflammation; Obésité; Resistin; Inflammation; Insulin resistance; Diabetes; Inflammation; Obesity

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APA (6th Edition):

Al-Rifai, S. (2019). Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique : Role of hypothalamic resistin in the onset of hypothalamic inflammation and insulin resistance : impact of acute or chronic high fat diet consumption. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS095

Chicago Manual of Style (16th Edition):

Al-Rifai, Sarah. “Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique : Role of hypothalamic resistin in the onset of hypothalamic inflammation and insulin resistance : impact of acute or chronic high fat diet consumption.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed September 23, 2020. http://www.theses.fr/2019SACLS095.

MLA Handbook (7th Edition):

Al-Rifai, Sarah. “Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique : Role of hypothalamic resistin in the onset of hypothalamic inflammation and insulin resistance : impact of acute or chronic high fat diet consumption.” 2019. Web. 23 Sep 2020.

Vancouver:

Al-Rifai S. Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique : Role of hypothalamic resistin in the onset of hypothalamic inflammation and insulin resistance : impact of acute or chronic high fat diet consumption. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2020 Sep 23]. Available from: http://www.theses.fr/2019SACLS095.

Council of Science Editors:

Al-Rifai S. Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique : Role of hypothalamic resistin in the onset of hypothalamic inflammation and insulin resistance : impact of acute or chronic high fat diet consumption. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS095


University of Waikato

29. Newland, Julia. In vitro cell culture to study microglial inflammation .

Degree: 2016, University of Waikato

Inflammation of the central nervous system (CNS) characterised by release of cytokines such as tumour necrosis factor-alpha (TNF-a) and interleukin-1 (IL-1) is an event that… (more)

Subjects/Keywords: CNs; inflammation; Microglial

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Newland, J. (2016). In vitro cell culture to study microglial inflammation . (Masters Thesis). University of Waikato. Retrieved from http://hdl.handle.net/10289/12053

Chicago Manual of Style (16th Edition):

Newland, Julia. “In vitro cell culture to study microglial inflammation .” 2016. Masters Thesis, University of Waikato. Accessed September 23, 2020. http://hdl.handle.net/10289/12053.

MLA Handbook (7th Edition):

Newland, Julia. “In vitro cell culture to study microglial inflammation .” 2016. Web. 23 Sep 2020.

Vancouver:

Newland J. In vitro cell culture to study microglial inflammation . [Internet] [Masters thesis]. University of Waikato; 2016. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10289/12053.

Council of Science Editors:

Newland J. In vitro cell culture to study microglial inflammation . [Masters Thesis]. University of Waikato; 2016. Available from: http://hdl.handle.net/10289/12053


Virginia Tech

30. Wendelsdorf, Katherine Veronica. Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis.

Degree: PhD, Genetics, Bioinformatics, and Computational Biology, 2011, Virginia Tech

 The scientific method requires the creation of a unifying hypothesis that reconciles an observed health outcome of infection with experimental data gathered about the disease… (more)

Subjects/Keywords: inflammation; simulation; immunopathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wendelsdorf, K. V. (2011). Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/29717

Chicago Manual of Style (16th Edition):

Wendelsdorf, Katherine Veronica. “Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis.” 2011. Doctoral Dissertation, Virginia Tech. Accessed September 23, 2020. http://hdl.handle.net/10919/29717.

MLA Handbook (7th Edition):

Wendelsdorf, Katherine Veronica. “Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis.” 2011. Web. 23 Sep 2020.

Vancouver:

Wendelsdorf KV. Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis. [Internet] [Doctoral dissertation]. Virginia Tech; 2011. [cited 2020 Sep 23]. Available from: http://hdl.handle.net/10919/29717.

Council of Science Editors:

Wendelsdorf KV. Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis. [Doctoral Dissertation]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/29717

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