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You searched for subject:( Histones metabolism 60). Showing records 1 – 30 of 12565 total matches.

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1. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 09 Apr 2020.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

2. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 09 Apr 2020.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956


Wesleyan University

3. Ryznar, Rebecca Jean. Tdh2 and Tdh3 Regulate Sir2-dependent Transcriptional Silencing in 𝘚. 𝘤𝘦𝘳𝘦𝘷𝘪𝘴𝘪𝘢𝘦.

Degree: Molecular Biology and Biochemistry, 2014, Wesleyan University

  Cells are constantly changing their metabolic status in response to extracellular signaling and availability of nutrients. These adjustments involve distinct transcriptional responses that reflect… (more)

Subjects/Keywords: Epigenetics; Metabolism; sirtuins; Histones; Chromatin-modifying enzymes; Glyceraldehyde 3-phosphate dehydrogenase

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APA (6th Edition):

Ryznar, R. J. (2014). Tdh2 and Tdh3 Regulate Sir2-dependent Transcriptional Silencing in 𝘚. 𝘤𝘦𝘳𝘦𝘷𝘪𝘴𝘪𝘢𝘦. (Doctoral Dissertation). Wesleyan University. Retrieved from https://wesscholar.wesleyan.edu/etd_diss/37

Chicago Manual of Style (16th Edition):

Ryznar, Rebecca Jean. “Tdh2 and Tdh3 Regulate Sir2-dependent Transcriptional Silencing in 𝘚. 𝘤𝘦𝘳𝘦𝘷𝘪𝘴𝘪𝘢𝘦.” 2014. Doctoral Dissertation, Wesleyan University. Accessed April 09, 2020. https://wesscholar.wesleyan.edu/etd_diss/37.

MLA Handbook (7th Edition):

Ryznar, Rebecca Jean. “Tdh2 and Tdh3 Regulate Sir2-dependent Transcriptional Silencing in 𝘚. 𝘤𝘦𝘳𝘦𝘷𝘪𝘴𝘪𝘢𝘦.” 2014. Web. 09 Apr 2020.

Vancouver:

Ryznar RJ. Tdh2 and Tdh3 Regulate Sir2-dependent Transcriptional Silencing in 𝘚. 𝘤𝘦𝘳𝘦𝘷𝘪𝘴𝘪𝘢𝘦. [Internet] [Doctoral dissertation]. Wesleyan University; 2014. [cited 2020 Apr 09]. Available from: https://wesscholar.wesleyan.edu/etd_diss/37.

Council of Science Editors:

Ryznar RJ. Tdh2 and Tdh3 Regulate Sir2-dependent Transcriptional Silencing in 𝘚. 𝘤𝘦𝘳𝘦𝘷𝘪𝘴𝘪𝘢𝘦. [Doctoral Dissertation]. Wesleyan University; 2014. Available from: https://wesscholar.wesleyan.edu/etd_diss/37

4. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 09 Apr 2020.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

5. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

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APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 09 Apr 2020.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150

6. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 09 Apr 2020.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

7. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

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APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 09 Apr 2020.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

8. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

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APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 09 Apr 2020.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

9. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 09 Apr 2020.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

10. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

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APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 09 Apr 2020.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

11. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

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APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 09 Apr 2020.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

12. Durham, Carolyn G. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.

Degree: PhD, 2010, University of Alabama – Birmingham

The inverse correlation between the industrialization and disease prevalence is termed the "hygiene hypothesis." Supporting this, immunological studies show Th1 cytokines modulate Th2 immune responses.… (more)

Subjects/Keywords: Asthma<; br>; Gastric Mucosa  – metabolism<; br>; Gastritis<; br>; Helicobacter Infections  – metabolism<; br>; Helicobacter felis<; br>; Mucus  – metabolism

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APA (6th Edition):

Durham, C. G. (2010). Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,707

Chicago Manual of Style (16th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,707.

MLA Handbook (7th Edition):

Durham, Carolyn G. “Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma.” 2010. Web. 09 Apr 2020.

Vancouver:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707.

Council of Science Editors:

Durham CG. Role of toll-like receptors 2 and 4 in Helicobacter-associated gastritis and cockroach-induced asthma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,707

13. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 09 Apr 2020.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

14. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 09 Apr 2020.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

15. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 09 Apr 2020.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

16. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

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APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 09 Apr 2020.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

17. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

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APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 09 Apr 2020.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194

18. Moore, Carlene Drucilla. The role of centaurin alpha-1 in the regulation of neuronal differentiation.

Degree: PhD, 2008, University of Alabama – Birmingham

In the nervous system, PI 3-kinase has been implicated in neuronal differentiation. My studies have focused on a candidate neuronal PI 3-kinase target centaurin alpha-1,… (more)

Subjects/Keywords: 1-Phosphatidylinositol 3-Kinase <; br>; Adaptor Proteins, Signal Transducing  – metabolism <; br>; Dendrites  – metabolism <; br>; Dendrites  – ultrastructure <; br>; GTPase-Activating Proteins  – metabolism <; br>; Hippocampus  – cytology <; br>; Nerve Tissue Proteins  – metabolism <; br>; Neurons  – metabolism

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APA (6th Edition):

Moore, C. D. (2008). The role of centaurin alpha-1 in the regulation of neuronal differentiation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,208

Chicago Manual of Style (16th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,208.

MLA Handbook (7th Edition):

Moore, Carlene Drucilla. “The role of centaurin alpha-1 in the regulation of neuronal differentiation.” 2008. Web. 09 Apr 2020.

Vancouver:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208.

Council of Science Editors:

Moore CD. The role of centaurin alpha-1 in the regulation of neuronal differentiation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,208

19. Davenport, James Robert. The role of the primary cilium in energy and glucose metabolism.

Degree: PhD, 2007, University of Alabama – Birmingham

Virtually ignored for years as a useless organelle, the primary cilium has emerged as an essential signaling center in both development and maintenance of tissues… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Flagella  – metabolism<; br>; Kidney  – metabolism<; br>; Kidney Diseases, Cystic  – metabolism<; br>; Obesity  – metabolism<; br>; Pancreas  – abnormalities<; br>; Pancreas  – pathology<; br>; Tumor Suppressor Proteins

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APA (6th Edition):

Davenport, J. R. (2007). The role of the primary cilium in energy and glucose metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,516

Chicago Manual of Style (16th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,516.

MLA Handbook (7th Edition):

Davenport, James Robert. “The role of the primary cilium in energy and glucose metabolism.” 2007. Web. 09 Apr 2020.

Vancouver:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516.

Council of Science Editors:

Davenport JR. The role of the primary cilium in energy and glucose metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,516

20. Mavalli, Mahendra D. Mechanisms of growth hormone action in skeletal muscle.

Degree: PhD, 2009, University of Alabama – Birmingham

Growth hormone (GH) and insulin like growth factor-1 (IGF-1) exert profound growth promoting actions during pre and postnatal skeletal muscle development. GH and IGF-1 seem… (more)

Subjects/Keywords: Growth Hormone  – metabolism<; br>; Insulin  – metabolism<; br>; Insulin Resistance<; br>; Muscle, Skeletal  – growth & development<; br>; Muscle, Skeletal  – metabolism<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptors, Somatotropin  – metabolism<; br>; Sarcopenia

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APA (6th Edition):

Mavalli, M. D. (2009). Mechanisms of growth hormone action in skeletal muscle. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1103

Chicago Manual of Style (16th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1103.

MLA Handbook (7th Edition):

Mavalli, Mahendra D. “Mechanisms of growth hormone action in skeletal muscle.” 2009. Web. 09 Apr 2020.

Vancouver:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103.

Council of Science Editors:

Mavalli MD. Mechanisms of growth hormone action in skeletal muscle. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1103

21. Tang, Yi, Ph.D. The role of transforming growth factor beta-1 in bone remodeling.

Degree: PhD, 2009, University of Alabama – Birmingham

Bone remodeling depends on the precise coordination of bone resorption by the osteoclasts and bone formation by the osteoblasts. It is proposed that osteoclastic bone… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Bone Regeneration  – drug effects<; br>; Cadherins  – metabolism<; br>; Cell Membrane  – metabolism<; br>; Multiprotein Complexes  – metabolism<; br>; Smad7 Protein  – metabolism<; br>; Transforming Growth Factor beta  – metabolism

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APA (6th Edition):

Tang, Yi, P. D. (2009). The role of transforming growth factor beta-1 in bone remodeling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,405

Chicago Manual of Style (16th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,405.

MLA Handbook (7th Edition):

Tang, Yi, Ph D. “The role of transforming growth factor beta-1 in bone remodeling.” 2009. Web. 09 Apr 2020.

Vancouver:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405.

Council of Science Editors:

Tang, Yi PD. The role of transforming growth factor beta-1 in bone remodeling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,405

22. Balasubramani, Anand. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.

Degree: PhD, 2010, University of Alabama – Birmingham

The ability to differentially manipulate available genetic information in order to generate diverse cellular identities represents an innovation of complex multicellular eukaryotic organisms. Cis-acting modules… (more)

Subjects/Keywords: DNA Replication – physiology.<; br>; Drosophila – metabolism.<; br>; Drosophila Proteins – metabolism.<; br>; GTP Phosphohydrolases – metabolism.<; br>; Microfilament Proteins – metabolism.<; br>; Multiprotein Complexes – metabolism.<; br>; Origin Recognition Complex – metabolism.

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APA (6th Edition):

Balasubramani, A. (2010). Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1428

Chicago Manual of Style (16th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1428.

MLA Handbook (7th Edition):

Balasubramani, Anand. “Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng.” 2010. Web. 09 Apr 2020.

Vancouver:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428.

Council of Science Editors:

Balasubramani A. Evolutionarily Conserved Cis-Acting Elements Regulate Lineage-Specific Expression Of Ifng. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1428

23. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 09 Apr 2020.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

24. Jiang, Shaoning. Molecular mechanisms of hepatic insulin resistance following injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Insulin resistance commonly occurs following injuries or critical illness independent of previous diabetic status. The development of insulin resistance and hyperglycemia is associated with increased… (more)

Subjects/Keywords: Adenoviridae – metabolism<; br>; Adenoviridae Infections – metabolism<; br>; Diabetes Mellitus, Type 2 – physiopathology<; br>; Glucose – metabolism<; br>; I-kappa B Kinase – physiology <; br>; Insulin – metabolism<; br>; JNK Mitogen-Activated Protein Kinases – physiology <; br>; Liver

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APA (6th Edition):

Jiang, S. (2010). Molecular mechanisms of hepatic insulin resistance following injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1374

Chicago Manual of Style (16th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1374.

MLA Handbook (7th Edition):

Jiang, Shaoning. “Molecular mechanisms of hepatic insulin resistance following injury.” 2010. Web. 09 Apr 2020.

Vancouver:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374.

Council of Science Editors:

Jiang S. Molecular mechanisms of hepatic insulin resistance following injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1374

25. Yu, Jei-Hwa. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.

Degree: PhD, 2008, University of Alabama – Birmingham

Papillomaviruses (PV) are prevalent pathogens that infect human or animal squamous epithelia. Its genome is a double strand circular DNA of approximately 7.9 kb. It… (more)

Subjects/Keywords: DNA Helicases  – metabolism <; br>; DNA-Binding Proteins  – metabolism <; br>; Human papillomavirus 11  – physiology <; br>; Mitogen-Activated Protein Kinases  – metabolism <; br>; Nuclear Localization Signals  – metabolism <; br>; Replication Origin <; br>; Viral Proteins  – metabolism

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APA (6th Edition):

Yu, J. (2008). MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,213

Chicago Manual of Style (16th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,213.

MLA Handbook (7th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Web. 09 Apr 2020.

Vancouver:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213.

Council of Science Editors:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213

26. Byon, Chang Hyun. Oxidative stress-stimulated vascular calcification.

Degree: PhD, 2009, University of Alabama – Birmingham

Oxidative stress plays a critical role in pathogenesis of atherosclerosis. However, the effect of oxidative stress-induced molecular signaling in development of vascular calcification, a feature… (more)

Subjects/Keywords: Calcinosis  – metabolism<; br>; Core Binding Factor Alpha 1 Subunit  – metabolism<; br>; Hydrogen Peroxide  – metabolism<; br>; Muscle, smooth, Vascular  – metabolism<; br>; Oxidative Stress<; br>; Proto-Oncogene Proteins c-akt  – metabolism<; br>; Signal Transduction

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APA (6th Edition):

Byon, C. H. (2009). Oxidative stress-stimulated vascular calcification. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,662

Chicago Manual of Style (16th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,662.

MLA Handbook (7th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Web. 09 Apr 2020.

Vancouver:

Byon CH. Oxidative stress-stimulated vascular calcification. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662.

Council of Science Editors:

Byon CH. Oxidative stress-stimulated vascular calcification. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662

27. Zhai, Lidong. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.

Degree: PhD, 2009, University of Alabama – Birmingham

Hyperglycemia develops in the intensive care unit in many patients following injury, infection and critical illness. However, little is known about the mechanism of acute… (more)

Subjects/Keywords: Aging  – metabolism<; br>; Insulin Resistance<; br>; Liver  – metabolism<; br>; Muscle, Skeletal  – metabolism<; br>; Oxidative Stress<; br>; Shock, Hemorrhagic  – metabolism<; br>; Wounds and Injuries  – metabolism

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APA (6th Edition):

Zhai, L. (2009). Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,834

Chicago Manual of Style (16th Edition):

Zhai, Lidong. “Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,834.

MLA Handbook (7th Edition):

Zhai, Lidong. “Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage.” 2009. Web. 09 Apr 2020.

Vancouver:

Zhai L. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,834.

Council of Science Editors:

Zhai L. Involvement of reactive oxygen species in the acute development of insulin resistance following trauma and hemorrhage. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,834

28. Fulzele, Keertik. Insulin signaling and function in osteoblasts.

Degree: PhD, 2009, University of Alabama – Birmingham

Insulin and insulin-like growth factor-1 (IGF-1) are evolutionarily conserved hormonal signaling pathways with structurally similar ligands and receptors. Recent studies suggest that that insulin and… (more)

Subjects/Keywords: Insulin  – metabolism<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptor, Insulin  – metabolism<; br>; Signal Transduction  – physiology<; br>; Skull  – metabolism

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APA (6th Edition):

Fulzele, K. (2009). Insulin signaling and function in osteoblasts. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,999

Chicago Manual of Style (16th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,999.

MLA Handbook (7th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Web. 09 Apr 2020.

Vancouver:

Fulzele K. Insulin signaling and function in osteoblasts. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999.

Council of Science Editors:

Fulzele K. Insulin signaling and function in osteoblasts. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999

29. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 09 Apr 2020.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

30. Pawar, Pritish Subhash. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Cholangiocarcinoma, a fatal tumor arising from biliary epithelium, has very poor 5-year survival rate due to lack of early diagnosis and effective therapies. Induction of… (more)

Subjects/Keywords: Antigens, CD95  – metabolism <; br>; CASP8 and FADD-Like Apoptosis Regulating Protein  – metabolism <; br>; Calmodulin  – metabolism <; br>; Cholangiocarcinoma <; br>; Signal Transduction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pawar, P. S. (2008). Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,301

Chicago Manual of Style (16th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,301.

MLA Handbook (7th Edition):

Pawar, Pritish Subhash. “Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma.” 2008. Web. 09 Apr 2020.

Vancouver:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301.

Council of Science Editors:

Pawar PS. Calmodulin binding to cellular FLICE like inhibitory protein modulates Fas-induced signaling and tumorigenesis in cholangiocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,301

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