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You searched for subject:( HCV transgenic mouse model). Showing records 1 – 30 of 47028 total matches.

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University of Ottawa

1. Soare, Catalina P. Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus .

Degree: 2011, University of Ottawa

 Hepatitis C Virus (HCV) is one of the most important causes of chronic liver disease, affecting more than 170 million people worldwide. The mechanisms of… (more)

Subjects/Keywords: Hepatitis C virus; Alcohol; HCV-transgenic mouse model; Dendritic cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Soare, C. P. (2011). Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20343

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Soare, Catalina P. “Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus .” 2011. Thesis, University of Ottawa. Accessed December 16, 2019. http://hdl.handle.net/10393/20343.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Soare, Catalina P. “Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus .” 2011. Web. 16 Dec 2019.

Vancouver:

Soare CP. Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/10393/20343.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Soare CP. Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/20343

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Mergy, Marc Andrew. Generation and Characterization of the First Construct-Valid Model of ADHD, the DAT Val559 Knock-In Mouse.

Degree: PhD, Neuroscience, 2013, Vanderbilt University

 Attention-deficit/hyperactivity disorder (ADHD) is the most commonly diagnosed childhood neuropsychiatric disorder. More than twenty years of genetic, behavioral, and pharmacological research support the hypothesis that… (more)

Subjects/Keywords: transporter; dopamine; ADHD; transgenic; mouse; animal model

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APA (6th Edition):

Mergy, M. A. (2013). Generation and Characterization of the First Construct-Valid Model of ADHD, the DAT Val559 Knock-In Mouse. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-11192013-140238/ ;

Chicago Manual of Style (16th Edition):

Mergy, Marc Andrew. “Generation and Characterization of the First Construct-Valid Model of ADHD, the DAT Val559 Knock-In Mouse.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed December 16, 2019. http://etd.library.vanderbilt.edu/available/etd-11192013-140238/ ;.

MLA Handbook (7th Edition):

Mergy, Marc Andrew. “Generation and Characterization of the First Construct-Valid Model of ADHD, the DAT Val559 Knock-In Mouse.” 2013. Web. 16 Dec 2019.

Vancouver:

Mergy MA. Generation and Characterization of the First Construct-Valid Model of ADHD, the DAT Val559 Knock-In Mouse. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2019 Dec 16]. Available from: http://etd.library.vanderbilt.edu/available/etd-11192013-140238/ ;.

Council of Science Editors:

Mergy MA. Generation and Characterization of the First Construct-Valid Model of ADHD, the DAT Val559 Knock-In Mouse. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://etd.library.vanderbilt.edu/available/etd-11192013-140238/ ;


University of Alberta

3. Hsi Dickie, Belinda. Advancing the Alb-uPA/SCID/Bg Chimeric Mouse.

Degree: PhD, Department of Surgery, 2009, University of Alberta

 The feasibility of the Alb-uPA/SCID/Bg chimeric mouse as a model for Hepatitis C Virus (HCV) infection was assessed experimentally by (1) the infection and treatment… (more)

Subjects/Keywords: Hepatitis C; Mouse Model; Transgenic mice; Alb-uPA/SCID/Beige mouse

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APA (6th Edition):

Hsi Dickie, B. (2009). Advancing the Alb-uPA/SCID/Bg Chimeric Mouse. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/rr171x54d

Chicago Manual of Style (16th Edition):

Hsi Dickie, Belinda. “Advancing the Alb-uPA/SCID/Bg Chimeric Mouse.” 2009. Doctoral Dissertation, University of Alberta. Accessed December 16, 2019. https://era.library.ualberta.ca/files/rr171x54d.

MLA Handbook (7th Edition):

Hsi Dickie, Belinda. “Advancing the Alb-uPA/SCID/Bg Chimeric Mouse.” 2009. Web. 16 Dec 2019.

Vancouver:

Hsi Dickie B. Advancing the Alb-uPA/SCID/Bg Chimeric Mouse. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2019 Dec 16]. Available from: https://era.library.ualberta.ca/files/rr171x54d.

Council of Science Editors:

Hsi Dickie B. Advancing the Alb-uPA/SCID/Bg Chimeric Mouse. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/rr171x54d


Dalhousie University

4. Lin, Liang-Tzung. HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS.

Degree: PhD, Department of Microbiology & Immunology, 2010, Dalhousie University

 Hepatitis C virus (HCV) is a serious global health problem with an estimate of 170 million carriers worldwide. Most individuals exposed to this blood-borne pathogen… (more)

Subjects/Keywords: HCV; host factors; IRF-3; miR-122; mouse model

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APA (6th Edition):

Lin, L. (2010). HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/13159

Chicago Manual of Style (16th Edition):

Lin, Liang-Tzung. “HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS.” 2010. Doctoral Dissertation, Dalhousie University. Accessed December 16, 2019. http://hdl.handle.net/10222/13159.

MLA Handbook (7th Edition):

Lin, Liang-Tzung. “HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS.” 2010. Web. 16 Dec 2019.

Vancouver:

Lin L. HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS. [Internet] [Doctoral dissertation]. Dalhousie University; 2010. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/10222/13159.

Council of Science Editors:

Lin L. HOST FACTOR REGULATION OF HEPATITIS C VIRUS REPLICATION IN RODENT CELLS. [Doctoral Dissertation]. Dalhousie University; 2010. Available from: http://hdl.handle.net/10222/13159

5. Da Costa, Daniel. Study of cell host factors involved in Hepatitis C virus tropism : Etude des facteurs cellulaires de l'hôte impliqués dans le tropisme du virus de l'hépatite C.

Degree: Docteur es, Virologie : aspects moléculaires et médicaux, 2012, Université de Strasbourg

Le virus de l’hépatite C (HCV) est un problème majeur de santé publique. Le développement de nouveaux traitements pour lutter contre le HCV a été… (more)

Subjects/Keywords: Intéraction virus-hôte; Tropisme du HCV; Spécificité d’espèce du HCV; Entrée du HCV; Replication du HCV; Assemblage du HCV et modèle murin du HCV; Hepatitis C virus infection; Virus-host interaction; HCV tropism; HCV species specificity; HCV entry; HCV replication; HCV assembly and HCV mouse model; 579.2; 616.91; 572.8

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APA (6th Edition):

Da Costa, D. (2012). Study of cell host factors involved in Hepatitis C virus tropism : Etude des facteurs cellulaires de l'hôte impliqués dans le tropisme du virus de l'hépatite C. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ071

Chicago Manual of Style (16th Edition):

Da Costa, Daniel. “Study of cell host factors involved in Hepatitis C virus tropism : Etude des facteurs cellulaires de l'hôte impliqués dans le tropisme du virus de l'hépatite C.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed December 16, 2019. http://www.theses.fr/2012STRAJ071.

MLA Handbook (7th Edition):

Da Costa, Daniel. “Study of cell host factors involved in Hepatitis C virus tropism : Etude des facteurs cellulaires de l'hôte impliqués dans le tropisme du virus de l'hépatite C.” 2012. Web. 16 Dec 2019.

Vancouver:

Da Costa D. Study of cell host factors involved in Hepatitis C virus tropism : Etude des facteurs cellulaires de l'hôte impliqués dans le tropisme du virus de l'hépatite C. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2019 Dec 16]. Available from: http://www.theses.fr/2012STRAJ071.

Council of Science Editors:

Da Costa D. Study of cell host factors involved in Hepatitis C virus tropism : Etude des facteurs cellulaires de l'hôte impliqués dans le tropisme du virus de l'hépatite C. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ071


University of Adelaide

6. Hendrix, Ivanka. Regulation of gene expression of the 25-Hydroxyvitamin D 1α-Hydroxylase (CYP27B1) promoter : study of a transgenic mouse model.

Degree: 2004, University of Adelaide

 The enzyme 25-hydroxyvitamin D la-hydroxylase or CYP27Bl is the key enzyme in the two-step activation process by which vitamin D is converted to its biologically… (more)

Subjects/Keywords: transgenic; mouse model; genetics

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APA (6th Edition):

Hendrix, I. (2004). Regulation of gene expression of the 25-Hydroxyvitamin D 1α-Hydroxylase (CYP27B1) promoter : study of a transgenic mouse model. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/59207

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hendrix, Ivanka. “Regulation of gene expression of the 25-Hydroxyvitamin D 1α-Hydroxylase (CYP27B1) promoter : study of a transgenic mouse model.” 2004. Thesis, University of Adelaide. Accessed December 16, 2019. http://hdl.handle.net/2440/59207.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hendrix, Ivanka. “Regulation of gene expression of the 25-Hydroxyvitamin D 1α-Hydroxylase (CYP27B1) promoter : study of a transgenic mouse model.” 2004. Web. 16 Dec 2019.

Vancouver:

Hendrix I. Regulation of gene expression of the 25-Hydroxyvitamin D 1α-Hydroxylase (CYP27B1) promoter : study of a transgenic mouse model. [Internet] [Thesis]. University of Adelaide; 2004. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/2440/59207.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hendrix I. Regulation of gene expression of the 25-Hydroxyvitamin D 1α-Hydroxylase (CYP27B1) promoter : study of a transgenic mouse model. [Thesis]. University of Adelaide; 2004. Available from: http://hdl.handle.net/2440/59207

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

7. Stringer, Colin D.M. Development of an inducible and reversible mouse model of podocyte effacement .

Degree: 2011, University of Guelph

 Podocytes are specialized epithelial cells which wrap glomerular capillaries with numerous interdigitating foot processes (FP). Between adjacent FPs a unique junction, the slit diaphragm (SD),… (more)

Subjects/Keywords: nephropathy; proteinuria; glomerular filtration; podocyte; slit diaphragm; Nck; transgenic mouse model

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APA (6th Edition):

Stringer, C. D. M. (2011). Development of an inducible and reversible mouse model of podocyte effacement . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stringer, Colin D M. “Development of an inducible and reversible mouse model of podocyte effacement .” 2011. Thesis, University of Guelph. Accessed December 16, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stringer, Colin D M. “Development of an inducible and reversible mouse model of podocyte effacement .” 2011. Web. 16 Dec 2019.

Vancouver:

Stringer CDM. Development of an inducible and reversible mouse model of podocyte effacement . [Internet] [Thesis]. University of Guelph; 2011. [cited 2019 Dec 16]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stringer CDM. Development of an inducible and reversible mouse model of podocyte effacement . [Thesis]. University of Guelph; 2011. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

8. Schneeberger, Valentina. Novel Roles of the Protein Tyrosine Phosphatase SHP2 in Non-small Cell Lung Cancer.

Degree: 2014, University of South Florida

 The gene PTPN11 was identified in the early 1990s, and encodes the non-transmembrane protein tyrosine phosphatase SHP2. SHP2 is expressed ubiquitously in cells, and plays… (more)

Subjects/Keywords: mouse model; NSCLC; SHP2; Transgenic; Molecular Biology; Oncology

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APA (6th Edition):

Schneeberger, V. (2014). Novel Roles of the Protein Tyrosine Phosphatase SHP2 in Non-small Cell Lung Cancer. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/5306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schneeberger, Valentina. “Novel Roles of the Protein Tyrosine Phosphatase SHP2 in Non-small Cell Lung Cancer.” 2014. Thesis, University of South Florida. Accessed December 16, 2019. https://scholarcommons.usf.edu/etd/5306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schneeberger, Valentina. “Novel Roles of the Protein Tyrosine Phosphatase SHP2 in Non-small Cell Lung Cancer.” 2014. Web. 16 Dec 2019.

Vancouver:

Schneeberger V. Novel Roles of the Protein Tyrosine Phosphatase SHP2 in Non-small Cell Lung Cancer. [Internet] [Thesis]. University of South Florida; 2014. [cited 2019 Dec 16]. Available from: https://scholarcommons.usf.edu/etd/5306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schneeberger V. Novel Roles of the Protein Tyrosine Phosphatase SHP2 in Non-small Cell Lung Cancer. [Thesis]. University of South Florida; 2014. Available from: https://scholarcommons.usf.edu/etd/5306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

9. Pang, Daniel. To HAV or not to HAV: Novel hepatitis A virus (HAV) infection in a chimeric mouse model.

Degree: MS, Department of Medical Microbiology and Immunology, 2013, University of Alberta

 Chimpanzees have been previously used to study HAV infection in vivo but they are expensive and difficult to maintain and thus, there is a need… (more)

Subjects/Keywords: small animal model; hcv; hepatitis a virus; chimeric mouse model; hav; SCID-beige/Alb-uPA; hepatitis c virus

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APA (6th Edition):

Pang, D. (2013). To HAV or not to HAV: Novel hepatitis A virus (HAV) infection in a chimeric mouse model. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/9p290b669

Chicago Manual of Style (16th Edition):

Pang, Daniel. “To HAV or not to HAV: Novel hepatitis A virus (HAV) infection in a chimeric mouse model.” 2013. Masters Thesis, University of Alberta. Accessed December 16, 2019. https://era.library.ualberta.ca/files/9p290b669.

MLA Handbook (7th Edition):

Pang, Daniel. “To HAV or not to HAV: Novel hepatitis A virus (HAV) infection in a chimeric mouse model.” 2013. Web. 16 Dec 2019.

Vancouver:

Pang D. To HAV or not to HAV: Novel hepatitis A virus (HAV) infection in a chimeric mouse model. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2019 Dec 16]. Available from: https://era.library.ualberta.ca/files/9p290b669.

Council of Science Editors:

Pang D. To HAV or not to HAV: Novel hepatitis A virus (HAV) infection in a chimeric mouse model. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/9p290b669


University of Alberta

10. Tennese, Alysa. Role of the Prader-Willi syndrome proteins necdin and Magel2 in the nervous system.

Degree: PhD, Medical Sciences - Medical Genetics, 2010, University of Alberta

 Prader-Willi syndrome (PWS) is a rare, neurodevelopmental disorder with multiple features caused by hypothalamic deficiency, including infantile failure to thrive, hyperphagia leading to obesity, growth… (more)

Subjects/Keywords: necdin; Prader-Willi syndrome; homeostasis; Magel2; development; transgenic mouse model; autonomic nervous system; hypothalamic-pituitary

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APA (6th Edition):

Tennese, A. (2010). Role of the Prader-Willi syndrome proteins necdin and Magel2 in the nervous system. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/m326m1960

Chicago Manual of Style (16th Edition):

Tennese, Alysa. “Role of the Prader-Willi syndrome proteins necdin and Magel2 in the nervous system.” 2010. Doctoral Dissertation, University of Alberta. Accessed December 16, 2019. https://era.library.ualberta.ca/files/m326m1960.

MLA Handbook (7th Edition):

Tennese, Alysa. “Role of the Prader-Willi syndrome proteins necdin and Magel2 in the nervous system.” 2010. Web. 16 Dec 2019.

Vancouver:

Tennese A. Role of the Prader-Willi syndrome proteins necdin and Magel2 in the nervous system. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2019 Dec 16]. Available from: https://era.library.ualberta.ca/files/m326m1960.

Council of Science Editors:

Tennese A. Role of the Prader-Willi syndrome proteins necdin and Magel2 in the nervous system. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/m326m1960


University of Alberta

11. Kelly, Stephanie Alicia Wilhelmina. Examining the origin of peripheral self-reactive T cells and the contribution of Gadd45beta to T cell selection events in the HYcd4 TCR transgenic mouse model.

Degree: MS, Department of Medical Microbiology and Immunology, 2014, University of Alberta

 Thymic negative selection is important for preventing self-reactive T cells from entering the circulation. However, some self-reactive T cell clones can escape negative selection and… (more)

Subjects/Keywords: TCR transgenic mouse model; T cell negative selection; PD-1 and Gadd45beta

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APA (6th Edition):

Kelly, S. A. W. (2014). Examining the origin of peripheral self-reactive T cells and the contribution of Gadd45beta to T cell selection events in the HYcd4 TCR transgenic mouse model. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/s1784m85d

Chicago Manual of Style (16th Edition):

Kelly, Stephanie Alicia Wilhelmina. “Examining the origin of peripheral self-reactive T cells and the contribution of Gadd45beta to T cell selection events in the HYcd4 TCR transgenic mouse model.” 2014. Masters Thesis, University of Alberta. Accessed December 16, 2019. https://era.library.ualberta.ca/files/s1784m85d.

MLA Handbook (7th Edition):

Kelly, Stephanie Alicia Wilhelmina. “Examining the origin of peripheral self-reactive T cells and the contribution of Gadd45beta to T cell selection events in the HYcd4 TCR transgenic mouse model.” 2014. Web. 16 Dec 2019.

Vancouver:

Kelly SAW. Examining the origin of peripheral self-reactive T cells and the contribution of Gadd45beta to T cell selection events in the HYcd4 TCR transgenic mouse model. [Internet] [Masters thesis]. University of Alberta; 2014. [cited 2019 Dec 16]. Available from: https://era.library.ualberta.ca/files/s1784m85d.

Council of Science Editors:

Kelly SAW. Examining the origin of peripheral self-reactive T cells and the contribution of Gadd45beta to T cell selection events in the HYcd4 TCR transgenic mouse model. [Masters Thesis]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/s1784m85d


Case Western Reserve University

12. Wolfram, Julie Ann. Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy.

Degree: PhD, Pathology, 2012, Case Western Reserve University

 Cardiovascular disease continues to be the leading cause of morbidity and mortality in the world. Among cardiovascular diseases in general, heart failure is the fastest… (more)

Subjects/Keywords: Cellular Biology; Medicine; Molecular Biology; hypertrophic cardiomyopathy; c-Myc; cell cycle; transgenic mouse model

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APA (6th Edition):

Wolfram, J. A. (2012). Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1315603810

Chicago Manual of Style (16th Edition):

Wolfram, Julie Ann. “Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy.” 2012. Doctoral Dissertation, Case Western Reserve University. Accessed December 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1315603810.

MLA Handbook (7th Edition):

Wolfram, Julie Ann. “Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy.” 2012. Web. 16 Dec 2019.

Vancouver:

Wolfram JA. Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2012. [cited 2019 Dec 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1315603810.

Council of Science Editors:

Wolfram JA. Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy. [Doctoral Dissertation]. Case Western Reserve University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1315603810


University of Manchester

13. Davis, Katherine. Hippocampal dysfunction in the 3xTgAD mouse model of Alzheimer's disease.

Degree: PhD, 2012, University of Manchester

 Alzheimer’s disease (AD) is a neurodegenerative disorder, characterised by severe memory loss and the accumulation of amyloid-beta (Aβ) and tau pathology within neocortex and medial… (more)

Subjects/Keywords: 616.831; Alzheimer's Disease; Transgenic Mouse Model; Hippocampus; Episodic Memory; In vivo Electrophysiology

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APA (6th Edition):

Davis, K. (2012). Hippocampal dysfunction in the 3xTgAD mouse model of Alzheimer's disease. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/hippocampal-dysfunction-in-the-3xtgad-mouse-model-of-alzheimers-disease(ba2d4704-9e22-4213-8bc7-a1c55a0e0ccd).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555525

Chicago Manual of Style (16th Edition):

Davis, Katherine. “Hippocampal dysfunction in the 3xTgAD mouse model of Alzheimer's disease.” 2012. Doctoral Dissertation, University of Manchester. Accessed December 16, 2019. https://www.research.manchester.ac.uk/portal/en/theses/hippocampal-dysfunction-in-the-3xtgad-mouse-model-of-alzheimers-disease(ba2d4704-9e22-4213-8bc7-a1c55a0e0ccd).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555525.

MLA Handbook (7th Edition):

Davis, Katherine. “Hippocampal dysfunction in the 3xTgAD mouse model of Alzheimer's disease.” 2012. Web. 16 Dec 2019.

Vancouver:

Davis K. Hippocampal dysfunction in the 3xTgAD mouse model of Alzheimer's disease. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2019 Dec 16]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/hippocampal-dysfunction-in-the-3xtgad-mouse-model-of-alzheimers-disease(ba2d4704-9e22-4213-8bc7-a1c55a0e0ccd).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555525.

Council of Science Editors:

Davis K. Hippocampal dysfunction in the 3xTgAD mouse model of Alzheimer's disease. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/hippocampal-dysfunction-in-the-3xtgad-mouse-model-of-alzheimers-disease(ba2d4704-9e22-4213-8bc7-a1c55a0e0ccd).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555525


University of Southern California

14. Zhu, Genyuan. The role of endoplasmic reticulum chaperones in adipogenesis, liver cancer and mammary gland development.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2015, University of Southern California

 The endoplasmic reticulum (ER) is an essential cellular compartment for protein folding and secretion, and lipid synthesis and transport. Recent studies show that the function… (more)

Subjects/Keywords: endoplasmic reticulum; adipose tissue; liver cancer; mammary gland; transgenic mouse model; GRP78; GRP94

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APA (6th Edition):

Zhu, G. (2015). The role of endoplasmic reticulum chaperones in adipogenesis, liver cancer and mammary gland development. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/528266/rec/7207

Chicago Manual of Style (16th Edition):

Zhu, Genyuan. “The role of endoplasmic reticulum chaperones in adipogenesis, liver cancer and mammary gland development.” 2015. Doctoral Dissertation, University of Southern California. Accessed December 16, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/528266/rec/7207.

MLA Handbook (7th Edition):

Zhu, Genyuan. “The role of endoplasmic reticulum chaperones in adipogenesis, liver cancer and mammary gland development.” 2015. Web. 16 Dec 2019.

Vancouver:

Zhu G. The role of endoplasmic reticulum chaperones in adipogenesis, liver cancer and mammary gland development. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2019 Dec 16]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/528266/rec/7207.

Council of Science Editors:

Zhu G. The role of endoplasmic reticulum chaperones in adipogenesis, liver cancer and mammary gland development. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/528266/rec/7207


University of Western Ontario

15. Zeppa, Joseph J. Superantigen responsive T cells are required for nasopharyngeal infection by Streptococcus pyogenes.

Degree: 2017, University of Western Ontario

 Streptococcus pyogenes is a human-specific pathogen that is responsible for serious morbidity and mortality worldwide despite being susceptible to common antibiotics. Furthermore, there is currently… (more)

Subjects/Keywords: Streptococcus pyogenes; superantigens; transgenic mouse model; nasopharyngeal infection; vaccination; passive immunization; Immunology of Infectious Disease

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APA (6th Edition):

Zeppa, J. J. (2017). Superantigen responsive T cells are required for nasopharyngeal infection by Streptococcus pyogenes. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4435

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zeppa, Joseph J. “Superantigen responsive T cells are required for nasopharyngeal infection by Streptococcus pyogenes.” 2017. Thesis, University of Western Ontario. Accessed December 16, 2019. https://ir.lib.uwo.ca/etd/4435.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zeppa, Joseph J. “Superantigen responsive T cells are required for nasopharyngeal infection by Streptococcus pyogenes.” 2017. Web. 16 Dec 2019.

Vancouver:

Zeppa JJ. Superantigen responsive T cells are required for nasopharyngeal infection by Streptococcus pyogenes. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2019 Dec 16]. Available from: https://ir.lib.uwo.ca/etd/4435.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zeppa JJ. Superantigen responsive T cells are required for nasopharyngeal infection by Streptococcus pyogenes. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4435

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

16. Razalli, Nurul Huda. Effect Of Garcinol On Change In Gene Expression In Pancreatic Cancer Mouse Model.

Degree: MS, Nutrition and Food Science, 2013, Wayne State University

  Pancreatic cancer is currently ranked as the fourth leading cause of cancer-related deaths in The United States making it so important to develop new… (more)

Subjects/Keywords: Garcinol; gene expression; KPC mouse model; Pancreatic cancer; Transgenic mice; Medicine and Health Sciences; Nutrition

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APA (6th Edition):

Razalli, N. H. (2013). Effect Of Garcinol On Change In Gene Expression In Pancreatic Cancer Mouse Model. (Masters Thesis). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_theses/273

Chicago Manual of Style (16th Edition):

Razalli, Nurul Huda. “Effect Of Garcinol On Change In Gene Expression In Pancreatic Cancer Mouse Model.” 2013. Masters Thesis, Wayne State University. Accessed December 16, 2019. https://digitalcommons.wayne.edu/oa_theses/273.

MLA Handbook (7th Edition):

Razalli, Nurul Huda. “Effect Of Garcinol On Change In Gene Expression In Pancreatic Cancer Mouse Model.” 2013. Web. 16 Dec 2019.

Vancouver:

Razalli NH. Effect Of Garcinol On Change In Gene Expression In Pancreatic Cancer Mouse Model. [Internet] [Masters thesis]. Wayne State University; 2013. [cited 2019 Dec 16]. Available from: https://digitalcommons.wayne.edu/oa_theses/273.

Council of Science Editors:

Razalli NH. Effect Of Garcinol On Change In Gene Expression In Pancreatic Cancer Mouse Model. [Masters Thesis]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_theses/273


Wayne State University

17. Razalli, Nurul Huda. The Effect Of Oil Palm Phenolics (opp) On Pancreatic Ductal Adenocarcinoma (pdac) In Transgenic Mouse Model.

Degree: PhD, Nutrition and Food Science, 2017, Wayne State University

  Pancreatic ductal adenocarcinoma (PDAC) is an extremely aggressive form of pancreatic cancer with low survival rates partly due to late diagnosis and poor treatment… (more)

Subjects/Keywords: OIL PALM PHENOLICS; OPP; PANCREATIC CANCER; PANCREATIC DUCTAL ADENOCARCINOMA; PDAC; TRANSGENIC MOUSE MODEL; Nutrition

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APA (6th Edition):

Razalli, N. H. (2017). The Effect Of Oil Palm Phenolics (opp) On Pancreatic Ductal Adenocarcinoma (pdac) In Transgenic Mouse Model. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1737

Chicago Manual of Style (16th Edition):

Razalli, Nurul Huda. “The Effect Of Oil Palm Phenolics (opp) On Pancreatic Ductal Adenocarcinoma (pdac) In Transgenic Mouse Model.” 2017. Doctoral Dissertation, Wayne State University. Accessed December 16, 2019. https://digitalcommons.wayne.edu/oa_dissertations/1737.

MLA Handbook (7th Edition):

Razalli, Nurul Huda. “The Effect Of Oil Palm Phenolics (opp) On Pancreatic Ductal Adenocarcinoma (pdac) In Transgenic Mouse Model.” 2017. Web. 16 Dec 2019.

Vancouver:

Razalli NH. The Effect Of Oil Palm Phenolics (opp) On Pancreatic Ductal Adenocarcinoma (pdac) In Transgenic Mouse Model. [Internet] [Doctoral dissertation]. Wayne State University; 2017. [cited 2019 Dec 16]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1737.

Council of Science Editors:

Razalli NH. The Effect Of Oil Palm Phenolics (opp) On Pancreatic Ductal Adenocarcinoma (pdac) In Transgenic Mouse Model. [Doctoral Dissertation]. Wayne State University; 2017. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1737

18. Varnum, Megan Marissa. An anti-inflammatory glycoprotein, CD200, restores neurogenesis and enhances amyloid phagocytosis in a mouse model of Alzheimer's disease.

Degree: PhD, Pharmacology & Experimental Therapeutics, 2015, Boston University

 Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by the accumulation of amyloid-β peptide (Aβ) in the brain and intraneuronal hyperphosphorylated tau. Microglia in the… (more)

Subjects/Keywords: Pharmacology; Alzheimer's disease; CD200; Inflammation; Adeno-associated virus; Microglia; Transgenic mouse model

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APA (6th Edition):

Varnum, M. M. (2015). An anti-inflammatory glycoprotein, CD200, restores neurogenesis and enhances amyloid phagocytosis in a mouse model of Alzheimer's disease. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13725

Chicago Manual of Style (16th Edition):

Varnum, Megan Marissa. “An anti-inflammatory glycoprotein, CD200, restores neurogenesis and enhances amyloid phagocytosis in a mouse model of Alzheimer's disease.” 2015. Doctoral Dissertation, Boston University. Accessed December 16, 2019. http://hdl.handle.net/2144/13725.

MLA Handbook (7th Edition):

Varnum, Megan Marissa. “An anti-inflammatory glycoprotein, CD200, restores neurogenesis and enhances amyloid phagocytosis in a mouse model of Alzheimer's disease.” 2015. Web. 16 Dec 2019.

Vancouver:

Varnum MM. An anti-inflammatory glycoprotein, CD200, restores neurogenesis and enhances amyloid phagocytosis in a mouse model of Alzheimer's disease. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/2144/13725.

Council of Science Editors:

Varnum MM. An anti-inflammatory glycoprotein, CD200, restores neurogenesis and enhances amyloid phagocytosis in a mouse model of Alzheimer's disease. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13725


University of Western Ontario

19. Sule, Akshay. Role of the fibronectin-binding, collagen-binding, T-antigen region during nasopharyngeal infection by Streptococcus pyogenes.

Degree: 2019, University of Western Ontario

 Streptococcus pyogenes is a human-specific pathogen that uses a variety of adhesion molecules in order to initiate pharyngeal infection and maintain colonization. One genomic locus… (more)

Subjects/Keywords: Streptococcus pyogenes; pilus; collagen-binding protein; fibronectin-binding protein; acute nasopharyngeal infection; transgenic mouse model

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APA (6th Edition):

Sule, A. (2019). Role of the fibronectin-binding, collagen-binding, T-antigen region during nasopharyngeal infection by Streptococcus pyogenes. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6542

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sule, Akshay. “Role of the fibronectin-binding, collagen-binding, T-antigen region during nasopharyngeal infection by Streptococcus pyogenes.” 2019. Thesis, University of Western Ontario. Accessed December 16, 2019. https://ir.lib.uwo.ca/etd/6542.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sule, Akshay. “Role of the fibronectin-binding, collagen-binding, T-antigen region during nasopharyngeal infection by Streptococcus pyogenes.” 2019. Web. 16 Dec 2019.

Vancouver:

Sule A. Role of the fibronectin-binding, collagen-binding, T-antigen region during nasopharyngeal infection by Streptococcus pyogenes. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2019 Dec 16]. Available from: https://ir.lib.uwo.ca/etd/6542.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sule A. Role of the fibronectin-binding, collagen-binding, T-antigen region during nasopharyngeal infection by Streptococcus pyogenes. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6542

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

20. Olaya, Juan. Neurobiological investigation of the Neuregulin 1 gene and Neuregulin 1 type III overexpression in the pathophysiology of schizophrenia.

Degree: Psychiatry, 2019, University of New South Wales

 Schizophrenia is a severe psychiatric disorder where patients suffer from hallucinations, delusions, social deficits and cognitive impairments. Perturbations in the maturation, expression and function of… (more)

Subjects/Keywords: Transgenic mouse; Schizophrenia; Neuregulin 1

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APA (6th Edition):

Olaya, J. (2019). Neurobiological investigation of the Neuregulin 1 gene and Neuregulin 1 type III overexpression in the pathophysiology of schizophrenia. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/61976

Chicago Manual of Style (16th Edition):

Olaya, Juan. “Neurobiological investigation of the Neuregulin 1 gene and Neuregulin 1 type III overexpression in the pathophysiology of schizophrenia.” 2019. Doctoral Dissertation, University of New South Wales. Accessed December 16, 2019. http://handle.unsw.edu.au/1959.4/61976.

MLA Handbook (7th Edition):

Olaya, Juan. “Neurobiological investigation of the Neuregulin 1 gene and Neuregulin 1 type III overexpression in the pathophysiology of schizophrenia.” 2019. Web. 16 Dec 2019.

Vancouver:

Olaya J. Neurobiological investigation of the Neuregulin 1 gene and Neuregulin 1 type III overexpression in the pathophysiology of schizophrenia. [Internet] [Doctoral dissertation]. University of New South Wales; 2019. [cited 2019 Dec 16]. Available from: http://handle.unsw.edu.au/1959.4/61976.

Council of Science Editors:

Olaya J. Neurobiological investigation of the Neuregulin 1 gene and Neuregulin 1 type III overexpression in the pathophysiology of schizophrenia. [Doctoral Dissertation]. University of New South Wales; 2019. Available from: http://handle.unsw.edu.au/1959.4/61976


University of California – Irvine

21. Dong, Tobias Xiao. Visualizing Calcium Dynamics in the Regulation of T Lymphocyte Motility and Activation.

Degree: Biomedical Sciences, 2017, University of California – Irvine

 Calcium is a ubiquitous cellular messenger that is essential in regulating a wide variety of functions in living organisms. Our understanding of Ca2+ signaling was… (more)

Subjects/Keywords: Immunology; Physiology; Cellular biology; Calcium signaling; Genetically encoded calcium indicators; Regulatory T cells; T cell motility; Transgenic mouse model

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APA (6th Edition):

Dong, T. X. (2017). Visualizing Calcium Dynamics in the Regulation of T Lymphocyte Motility and Activation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/13x4w6v6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dong, Tobias Xiao. “Visualizing Calcium Dynamics in the Regulation of T Lymphocyte Motility and Activation.” 2017. Thesis, University of California – Irvine. Accessed December 16, 2019. http://www.escholarship.org/uc/item/13x4w6v6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dong, Tobias Xiao. “Visualizing Calcium Dynamics in the Regulation of T Lymphocyte Motility and Activation.” 2017. Web. 16 Dec 2019.

Vancouver:

Dong TX. Visualizing Calcium Dynamics in the Regulation of T Lymphocyte Motility and Activation. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2019 Dec 16]. Available from: http://www.escholarship.org/uc/item/13x4w6v6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dong TX. Visualizing Calcium Dynamics in the Regulation of T Lymphocyte Motility and Activation. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/13x4w6v6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Victoria

22. Martin, Michele. Adoptive T cell therapy of breast cancer: defining and circumventing barriers to T cell infiltration in the tumour microenvironment.

Degree: Dept. of Biology, 2011, University of Victoria

 In the era of personalized cancer treatment, adoptive T cell therapy (ACT) shows promise for the treatment of solid cancers. However, partial or mixed responses… (more)

Subjects/Keywords: adoptive cell therapy; breast cancer; tumor; transgenic mouse model; HER2/neu; immunotherapy; T cell; infiltration; bioinformatics; microenvironment

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APA (6th Edition):

Martin, M. (2011). Adoptive T cell therapy of breast cancer: defining and circumventing barriers to T cell infiltration in the tumour microenvironment. (Thesis). University of Victoria. Retrieved from http://hdl.handle.net/1828/3655

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Michele. “Adoptive T cell therapy of breast cancer: defining and circumventing barriers to T cell infiltration in the tumour microenvironment.” 2011. Thesis, University of Victoria. Accessed December 16, 2019. http://hdl.handle.net/1828/3655.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Michele. “Adoptive T cell therapy of breast cancer: defining and circumventing barriers to T cell infiltration in the tumour microenvironment.” 2011. Web. 16 Dec 2019.

Vancouver:

Martin M. Adoptive T cell therapy of breast cancer: defining and circumventing barriers to T cell infiltration in the tumour microenvironment. [Internet] [Thesis]. University of Victoria; 2011. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/1828/3655.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin M. Adoptive T cell therapy of breast cancer: defining and circumventing barriers to T cell infiltration in the tumour microenvironment. [Thesis]. University of Victoria; 2011. Available from: http://hdl.handle.net/1828/3655

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

23. Bretana, Neil. Understanding the dynamics of hepatitis C virus transmission in a high-risk environment.

Degree: Medical Sciences, 2017, University of New South Wales

HCV is mainly transmitted between people who inject drugs in developed countries. In this population, HCV prevalence is high and incarceration is recognised to be… (more)

Subjects/Keywords: Prison; HCV; Transmission; Model; Phylogenetics

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APA (6th Edition):

Bretana, N. (2017). Understanding the dynamics of hepatitis C virus transmission in a high-risk environment. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/57161 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42740/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Bretana, Neil. “Understanding the dynamics of hepatitis C virus transmission in a high-risk environment.” 2017. Doctoral Dissertation, University of New South Wales. Accessed December 16, 2019. http://handle.unsw.edu.au/1959.4/57161 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42740/SOURCE02?view=true.

MLA Handbook (7th Edition):

Bretana, Neil. “Understanding the dynamics of hepatitis C virus transmission in a high-risk environment.” 2017. Web. 16 Dec 2019.

Vancouver:

Bretana N. Understanding the dynamics of hepatitis C virus transmission in a high-risk environment. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2019 Dec 16]. Available from: http://handle.unsw.edu.au/1959.4/57161 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42740/SOURCE02?view=true.

Council of Science Editors:

Bretana N. Understanding the dynamics of hepatitis C virus transmission in a high-risk environment. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/57161 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:42740/SOURCE02?view=true

24. Lau, Bonnie W. CXCR4/SDF1 in recruitment of stem cells to orthotopic murine malignant mesothelioma spheroids.

Degree: PhD, Division of Biology and Medicine. Pathobiology, 2009, Brown University

 Malignant mesothelioma is an aggressive cancer of the mesothelial lining causally linked to asbestos exposure and is highly resistant to current therapies. We hypothesize a… (more)

Subjects/Keywords: mouse model

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APA (6th Edition):

Lau, B. W. (2009). CXCR4/SDF1 in recruitment of stem cells to orthotopic murine malignant mesothelioma spheroids. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:148/

Chicago Manual of Style (16th Edition):

Lau, Bonnie W. “CXCR4/SDF1 in recruitment of stem cells to orthotopic murine malignant mesothelioma spheroids.” 2009. Doctoral Dissertation, Brown University. Accessed December 16, 2019. https://repository.library.brown.edu/studio/item/bdr:148/.

MLA Handbook (7th Edition):

Lau, Bonnie W. “CXCR4/SDF1 in recruitment of stem cells to orthotopic murine malignant mesothelioma spheroids.” 2009. Web. 16 Dec 2019.

Vancouver:

Lau BW. CXCR4/SDF1 in recruitment of stem cells to orthotopic murine malignant mesothelioma spheroids. [Internet] [Doctoral dissertation]. Brown University; 2009. [cited 2019 Dec 16]. Available from: https://repository.library.brown.edu/studio/item/bdr:148/.

Council of Science Editors:

Lau BW. CXCR4/SDF1 in recruitment of stem cells to orthotopic murine malignant mesothelioma spheroids. [Doctoral Dissertation]. Brown University; 2009. Available from: https://repository.library.brown.edu/studio/item/bdr:148/


Universitat Rovira i Virgili

25. Reverté Soler, Ingrid. Neurobehavioural effects associated with postnatal exposure to decabromodiphenyl ether in apoe2, apoe3 and apoe4 transgenic mice.

Degree: Departament de Química Analítica i Química Orgànica, 2012, Universitat Rovira i Virgili

 Decabromodiphenyl Ether (BDE-209) is a flame retardant widely used and source of concern because ofthe toxicity showed by other Polibrominateddiphenyl ethers (PBDEs). The presenceof PBDEs… (more)

Subjects/Keywords: Toxicity; APOE; PBDE; Transgenic mouse; behaviour; 159.9

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APA (6th Edition):

Reverté Soler, I. (2012). Neurobehavioural effects associated with postnatal exposure to decabromodiphenyl ether in apoe2, apoe3 and apoe4 transgenic mice. (Thesis). Universitat Rovira i Virgili. Retrieved from http://hdl.handle.net/10803/76782

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reverté Soler, Ingrid. “Neurobehavioural effects associated with postnatal exposure to decabromodiphenyl ether in apoe2, apoe3 and apoe4 transgenic mice.” 2012. Thesis, Universitat Rovira i Virgili. Accessed December 16, 2019. http://hdl.handle.net/10803/76782.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reverté Soler, Ingrid. “Neurobehavioural effects associated with postnatal exposure to decabromodiphenyl ether in apoe2, apoe3 and apoe4 transgenic mice.” 2012. Web. 16 Dec 2019.

Vancouver:

Reverté Soler I. Neurobehavioural effects associated with postnatal exposure to decabromodiphenyl ether in apoe2, apoe3 and apoe4 transgenic mice. [Internet] [Thesis]. Universitat Rovira i Virgili; 2012. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/10803/76782.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reverté Soler I. Neurobehavioural effects associated with postnatal exposure to decabromodiphenyl ether in apoe2, apoe3 and apoe4 transgenic mice. [Thesis]. Universitat Rovira i Virgili; 2012. Available from: http://hdl.handle.net/10803/76782

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

26. Cholanian, Marina. Effects of Estrogen on Morphological and Electrophysiological Properties of Arcuate NKB Neurons .

Degree: 2013, University of Arizona

 Infundibular (arcuate) neurokinin B (NKB) neurons play a critical role in neuroendocrine control of reproduction. Specifically, a local network of arcuate neurons that co-express kisspeptin,… (more)

Subjects/Keywords: hypothalamus; neurokinin B; transgenic mouse; Neuroscience; estrogen

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APA (6th Edition):

Cholanian, M. (2013). Effects of Estrogen on Morphological and Electrophysiological Properties of Arcuate NKB Neurons . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/311477

Chicago Manual of Style (16th Edition):

Cholanian, Marina. “Effects of Estrogen on Morphological and Electrophysiological Properties of Arcuate NKB Neurons .” 2013. Doctoral Dissertation, University of Arizona. Accessed December 16, 2019. http://hdl.handle.net/10150/311477.

MLA Handbook (7th Edition):

Cholanian, Marina. “Effects of Estrogen on Morphological and Electrophysiological Properties of Arcuate NKB Neurons .” 2013. Web. 16 Dec 2019.

Vancouver:

Cholanian M. Effects of Estrogen on Morphological and Electrophysiological Properties of Arcuate NKB Neurons . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/10150/311477.

Council of Science Editors:

Cholanian M. Effects of Estrogen on Morphological and Electrophysiological Properties of Arcuate NKB Neurons . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/311477


University of Kansas

27. Nair, Lakshmy. Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research.

Degree: MS, Pharmacology & Toxicology, 2010, University of Kansas

 Diabetic peripheral neuropathy (DPN), a common complication of diabetes, involves nerve damage in the arms and legs. Segmental demyelination is one of the basic patterns… (more)

Subjects/Keywords: Molecular biology; Biology; Neurosciences; Cell biology; Diabetic peripheral neuropathy; Schwann cells; Tet-on gene regulation system; Transgene construction; Transgene expression; Transgenic mouse model

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APA (6th Edition):

Nair, L. (2010). Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/6300

Chicago Manual of Style (16th Edition):

Nair, Lakshmy. “Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research.” 2010. Masters Thesis, University of Kansas. Accessed December 16, 2019. http://hdl.handle.net/1808/6300.

MLA Handbook (7th Edition):

Nair, Lakshmy. “Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research.” 2010. Web. 16 Dec 2019.

Vancouver:

Nair L. Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research. [Internet] [Masters thesis]. University of Kansas; 2010. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/1808/6300.

Council of Science Editors:

Nair L. Development and validation of transgene constructs in order to generate bi-transgenic mice models for Diabetic Peripheral Neuropathy research. [Masters Thesis]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6300


Université Montpellier II

28. Lahmy, Valentine. Validation préclinique de l'efficacité de l'ANAVEX2-73 dans des modèles transgénique et non transgénique de la maladie d'Alzheimer : Preclinical efficacy of ANAVEX2-73 in transgenic and non-transgenic of Alzheimer's disease.

Degree: Docteur es, Biologie Santé, 2014, Université Montpellier II

La maladie d'Alzheimer est la démence neurodégénérative la plus fréquente, en particulier chez les personnes âgées. A l'heure actuelle, aucun traitement ne permet d'arrêter efficacement… (more)

Subjects/Keywords: Ligand Sigma-1; Ligand muscarinique; Maladie d'Alzheimer; Souris transgénique; Amyloïde B25-35; Altération mitochondriale; Sigma-1 Ligand; Muscarinic ligand; Alzheimer's disease; Transgenic mouse model; Amyloid B25-35; Mitochondrial alteration

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APA (6th Edition):

Lahmy, V. (2014). Validation préclinique de l'efficacité de l'ANAVEX2-73 dans des modèles transgénique et non transgénique de la maladie d'Alzheimer : Preclinical efficacy of ANAVEX2-73 in transgenic and non-transgenic of Alzheimer's disease. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2014MON20084

Chicago Manual of Style (16th Edition):

Lahmy, Valentine. “Validation préclinique de l'efficacité de l'ANAVEX2-73 dans des modèles transgénique et non transgénique de la maladie d'Alzheimer : Preclinical efficacy of ANAVEX2-73 in transgenic and non-transgenic of Alzheimer's disease.” 2014. Doctoral Dissertation, Université Montpellier II. Accessed December 16, 2019. http://www.theses.fr/2014MON20084.

MLA Handbook (7th Edition):

Lahmy, Valentine. “Validation préclinique de l'efficacité de l'ANAVEX2-73 dans des modèles transgénique et non transgénique de la maladie d'Alzheimer : Preclinical efficacy of ANAVEX2-73 in transgenic and non-transgenic of Alzheimer's disease.” 2014. Web. 16 Dec 2019.

Vancouver:

Lahmy V. Validation préclinique de l'efficacité de l'ANAVEX2-73 dans des modèles transgénique et non transgénique de la maladie d'Alzheimer : Preclinical efficacy of ANAVEX2-73 in transgenic and non-transgenic of Alzheimer's disease. [Internet] [Doctoral dissertation]. Université Montpellier II; 2014. [cited 2019 Dec 16]. Available from: http://www.theses.fr/2014MON20084.

Council of Science Editors:

Lahmy V. Validation préclinique de l'efficacité de l'ANAVEX2-73 dans des modèles transgénique et non transgénique de la maladie d'Alzheimer : Preclinical efficacy of ANAVEX2-73 in transgenic and non-transgenic of Alzheimer's disease. [Doctoral Dissertation]. Université Montpellier II; 2014. Available from: http://www.theses.fr/2014MON20084


University of South Florida

29. Lebson, Lori Ann. Monocytes as Gene Therapy Vectors for the Treatment of Alzheimer’s Disease.

Degree: 2008, University of South Florida

 The accumulation of amyloid-ß; protein (Aß) in Alzheimer's disease (AD) is a well known pathological event. Decreasing the production or increasing the degradation of Aß;… (more)

Subjects/Keywords: Transgenic mouse model; Cell therapy; Abeta; Neprilysin; Protease; American Studies; Arts and Humanities

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APA (6th Edition):

Lebson, L. A. (2008). Monocytes as Gene Therapy Vectors for the Treatment of Alzheimer’s Disease. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lebson, Lori Ann. “Monocytes as Gene Therapy Vectors for the Treatment of Alzheimer’s Disease.” 2008. Thesis, University of South Florida. Accessed December 16, 2019. https://scholarcommons.usf.edu/etd/356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lebson, Lori Ann. “Monocytes as Gene Therapy Vectors for the Treatment of Alzheimer’s Disease.” 2008. Web. 16 Dec 2019.

Vancouver:

Lebson LA. Monocytes as Gene Therapy Vectors for the Treatment of Alzheimer’s Disease. [Internet] [Thesis]. University of South Florida; 2008. [cited 2019 Dec 16]. Available from: https://scholarcommons.usf.edu/etd/356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lebson LA. Monocytes as Gene Therapy Vectors for the Treatment of Alzheimer’s Disease. [Thesis]. University of South Florida; 2008. Available from: https://scholarcommons.usf.edu/etd/356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

30. Nabi, Md. Zinnatun. Characterization of RTEL/PCNA interaction in the maintenance of genomic stability.

Degree: Biochemistry and Medical Genetics, 2010, University of Manitoba

 Previously, we have demonstrated that a DNA helicase-like protein, termed RTEL (regulator of telomere length) is essential for the maintenance of genomic stability. RTEL deficiency… (more)

Subjects/Keywords: Molecular Biology; Transgenic model

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nabi, M. Z. (2010). Characterization of RTEL/PCNA interaction in the maintenance of genomic stability. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4198

Chicago Manual of Style (16th Edition):

Nabi, Md Zinnatun. “Characterization of RTEL/PCNA interaction in the maintenance of genomic stability.” 2010. Masters Thesis, University of Manitoba. Accessed December 16, 2019. http://hdl.handle.net/1993/4198.

MLA Handbook (7th Edition):

Nabi, Md Zinnatun. “Characterization of RTEL/PCNA interaction in the maintenance of genomic stability.” 2010. Web. 16 Dec 2019.

Vancouver:

Nabi MZ. Characterization of RTEL/PCNA interaction in the maintenance of genomic stability. [Internet] [Masters thesis]. University of Manitoba; 2010. [cited 2019 Dec 16]. Available from: http://hdl.handle.net/1993/4198.

Council of Science Editors:

Nabi MZ. Characterization of RTEL/PCNA interaction in the maintenance of genomic stability. [Masters Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4198

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