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You searched for subject:( Glycogen Synthase Kinase 3 metabolism 60). Showing records 1 – 30 of 34806 total matches.

[1] [2] [3] [4] [5] … [1161]

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1. Stella, David Ray. Lens Cataract: Biochemical Analysis Of The Alpha Crystallins.

Degree: PhD, 2010, University of Alabama – Birmingham

The cataract is a common ailment affecting the aged population. It appears over time and affects the quality of one’s life by the eventual loss… (more)

Subjects/Keywords: Alzheimer Disease<; br>; Brain – enzymology<; br>; Glycogen Synthase Kinase 3 – metabolism.<; br>; Microscopy, Electron<; br>; Subcellular Fractions – enzymology.

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APA (6th Edition):

Stella, D. R. (2010). Lens Cataract: Biochemical Analysis Of The Alpha Crystallins. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1401

Chicago Manual of Style (16th Edition):

Stella, David Ray. “Lens Cataract: Biochemical Analysis Of The Alpha Crystallins.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1401.

MLA Handbook (7th Edition):

Stella, David Ray. “Lens Cataract: Biochemical Analysis Of The Alpha Crystallins.” 2010. Web. 17 Oct 2019.

Vancouver:

Stella DR. Lens Cataract: Biochemical Analysis Of The Alpha Crystallins. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1401.

Council of Science Editors:

Stella DR. Lens Cataract: Biochemical Analysis Of The Alpha Crystallins. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1401

2. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 17 Oct 2019.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

3. Sun, Mianen. The role of DDX3 in regulating apoptosis, p53 and Snail.

Degree: PhD, 2008, University of Alabama – Birmingham

Cancer is a common disease that causes high rates of lethality. Resistance to cancer therapy is one major obstacle for producing an effective cancer treatment.… (more)

Subjects/Keywords: Apoptosis<; br>; Genes, p53<; br. Glycogen Synthase Kinase 3  – metabolism<; br>; Inhibitor of Apoptosis Proteins  – metabolism<; br>; Protein Kinase Inhibitors  – pharmacology<; br>; Receptors, Death Domain  – metabolism

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APA (6th Edition):

Sun, M. (2008). The role of DDX3 in regulating apoptosis, p53 and Snail. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,827

Chicago Manual of Style (16th Edition):

Sun, Mianen. “The role of DDX3 in regulating apoptosis, p53 and Snail.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,827.

MLA Handbook (7th Edition):

Sun, Mianen. “The role of DDX3 in regulating apoptosis, p53 and Snail.” 2008. Web. 17 Oct 2019.

Vancouver:

Sun M. The role of DDX3 in regulating apoptosis, p53 and Snail. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,827.

Council of Science Editors:

Sun M. The role of DDX3 in regulating apoptosis, p53 and Snail. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,827

4. Polter, Abigail Marie. Regulation of GSK3 in the pathophysiology and treatment of mood disorders.

Degree: PhD, 2010, University of Alabama – Birmingham

Mood disorders are devastating psychiatric illnesses that will affect as many as one in every five persons worldwide over the course of their lifetime. Significant… (more)

Subjects/Keywords: Behavior, Animal  – physiology<; br>; Brain  – metabolism<; br>; Exploratory Behavior  – physiology<; br>; Forkhead Transcription Factors  – metabolism<; br>; Glycogen Synthase Kinase 3<; br>; Mood Disorders<; br>; Receptor, Serotonin, 5-HT1A  – metabolism<; br>; Serine  – metabolism<; br>; Serotonin  – metabolism<; br>; Signal Transduction

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APA (6th Edition):

Polter, A. M. (2010). Regulation of GSK3 in the pathophysiology and treatment of mood disorders. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1108

Chicago Manual of Style (16th Edition):

Polter, Abigail Marie. “Regulation of GSK3 in the pathophysiology and treatment of mood disorders.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1108.

MLA Handbook (7th Edition):

Polter, Abigail Marie. “Regulation of GSK3 in the pathophysiology and treatment of mood disorders.” 2010. Web. 17 Oct 2019.

Vancouver:

Polter AM. Regulation of GSK3 in the pathophysiology and treatment of mood disorders. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1108.

Council of Science Editors:

Polter AM. Regulation of GSK3 in the pathophysiology and treatment of mood disorders. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1108


Boston University

5. Rolfs, Hillary. Investigation of the anti-migratory properties of GSK-3 inhibitors in glioblastoma.

Degree: MS, Medical Sciences, 2016, Boston University

 Glioblastoma is the most malignant form of brain cancer. Due to its aggressive nature, extensive research has been performed, but little progress has been made… (more)

Subjects/Keywords: Oncology; GSK-3; Glycogen synthase kinase-3

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APA (6th Edition):

Rolfs, H. (2016). Investigation of the anti-migratory properties of GSK-3 inhibitors in glioblastoma. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/19472

Chicago Manual of Style (16th Edition):

Rolfs, Hillary. “Investigation of the anti-migratory properties of GSK-3 inhibitors in glioblastoma.” 2016. Masters Thesis, Boston University. Accessed October 17, 2019. http://hdl.handle.net/2144/19472.

MLA Handbook (7th Edition):

Rolfs, Hillary. “Investigation of the anti-migratory properties of GSK-3 inhibitors in glioblastoma.” 2016. Web. 17 Oct 2019.

Vancouver:

Rolfs H. Investigation of the anti-migratory properties of GSK-3 inhibitors in glioblastoma. [Internet] [Masters thesis]. Boston University; 2016. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2144/19472.

Council of Science Editors:

Rolfs H. Investigation of the anti-migratory properties of GSK-3 inhibitors in glioblastoma. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/19472

6. Eom, Tae-Yeon. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.

Degree: PhD, 2009, University of Alabama – Birmingham

Neurogenesis is a crucial process for development, plasticity, and regenerative capacity of the developing and adult brain. Impairment of neurogenesis has been implicated in the… (more)

Subjects/Keywords: Apoptosis<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; Mice, Inbred C57BL<; br>; Mice, Knockout<; br>; Mice, Transgenic<; br>; Neurons  – cytology<; br>; Phosphorylation

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APA (6th Edition):

Eom, T. (2009). Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,390

Chicago Manual of Style (16th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,390.

MLA Handbook (7th Edition):

Eom, Tae-Yeon. “Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3.” 2009. Web. 17 Oct 2019.

Vancouver:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390.

Council of Science Editors:

Eom T. Regulation of neural precursor cell apoptosis and proliferation by glycogen synthase kinase-3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,390


University of Hong Kong

7. 潘彥伶; Pan, Yanling. GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury.

Degree: M. Phil., 2015, University of Hong Kong

 Spinal cord injury (SCI) results in extensive demyelination, leading to deleterious axon degeneration and inability of functional recovery. Remyelination has become a part of the… (more)

Subjects/Keywords: Spinal cord - Regeneration; Glycogen synthase kinase-3

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APA (6th Edition):

潘彥伶; Pan, Y. (2015). GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. (Masters Thesis). University of Hong Kong. Retrieved from Pan, Y. [潘彥伶]. (2015). GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435673 ; http://dx.doi.org/10.5353/th_b5435673 ; http://hdl.handle.net/10722/209467

Chicago Manual of Style (16th Edition):

潘彥伶; Pan, Yanling. “GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury.” 2015. Masters Thesis, University of Hong Kong. Accessed October 17, 2019. Pan, Y. [潘彥伶]. (2015). GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435673 ; http://dx.doi.org/10.5353/th_b5435673 ; http://hdl.handle.net/10722/209467.

MLA Handbook (7th Edition):

潘彥伶; Pan, Yanling. “GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury.” 2015. Web. 17 Oct 2019.

Vancouver:

潘彥伶; Pan Y. GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Oct 17]. Available from: Pan, Y. [潘彥伶]. (2015). GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435673 ; http://dx.doi.org/10.5353/th_b5435673 ; http://hdl.handle.net/10722/209467.

Council of Science Editors:

潘彥伶; Pan Y. GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. [Masters Thesis]. University of Hong Kong; 2015. Available from: Pan, Y. [潘彥伶]. (2015). GSK-3β inhibition promotes oligodendroglial differentiation and remyelination after spinal cord injury. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435673 ; http://dx.doi.org/10.5353/th_b5435673 ; http://hdl.handle.net/10722/209467


Boston University

8. Sepulveda, Sean Matthew. The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes.

Degree: 2015, Boston University

Glycogen synthase kinase-3 (GSK3) is a ubiquitously expressed protein kinase with key roles in controlling proliferation, differentiation and survival of a wide variety of mammalian… (more)

Subjects/Keywords: Biology; Cardiac hypertrophy; Glycogen synthase kinase 3

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APA (6th Edition):

Sepulveda, S. M. (2015). The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes. (Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sepulveda, Sean Matthew. “The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes.” 2015. Thesis, Boston University. Accessed October 17, 2019. http://hdl.handle.net/2144/15686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sepulveda, Sean Matthew. “The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes.” 2015. Web. 17 Oct 2019.

Vancouver:

Sepulveda SM. The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes. [Internet] [Thesis]. Boston University; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2144/15686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sepulveda SM. The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes. [Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/15686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Yuskaitis, Christopher Joseph. Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3.

Degree: PhD, 2009, University of Alabama – Birmingham

Neuroinflammation and Fragile X syndrome (FXS) are two particularly devastating neurologic conditions for which no adequate treatment exists and much is still unknown about the… (more)

Subjects/Keywords: Fragile X Mental Retardation Protein  – genetics<; br>; Gene Expression Regulation  – genetics<; br>; Glycogen Synthase Kinase 3  – antagonists & inhibitors<; br>; Inflammation Mediators  – metabolism<; br>; Lithium Chloride  – pharmacology<; br>; Microglia  – drug effects

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APA (6th Edition):

Yuskaitis, C. J. (2009). Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,507

Chicago Manual of Style (16th Edition):

Yuskaitis, Christopher Joseph. “Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,507.

MLA Handbook (7th Edition):

Yuskaitis, Christopher Joseph. “Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3.” 2009. Web. 17 Oct 2019.

Vancouver:

Yuskaitis CJ. Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,507.

Council of Science Editors:

Yuskaitis CJ. Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,507

10. Meares, Gordon P. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.

Degree: PhD, 2007, University of Alabama – Birmingham

Proper regulation of survival signaling is critical for all organisms. One important signaling cascade involved in the coordinated effort to control signals influencing cell fate… (more)

Subjects/Keywords: Apoptosis  – physiology <; br>; Cell Nucleus  – metabolism <; br>; Glycogen Synthase Kinase 3  – metabolism <; br>; HSP90 Heat-Shock Proteins  – physiology <; br>; Insulin  – physiology <; br>; Insulin-Like Growth Factor I  – physiology <; br>; Nuclear Localization Signals <; br>; Signal Transduction  – physiology

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APA (6th Edition):

Meares, G. P. (2007). Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,134

Chicago Manual of Style (16th Edition):

Meares, Gordon P. “Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,134.

MLA Handbook (7th Edition):

Meares, Gordon P. “Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.” 2007. Web. 17 Oct 2019.

Vancouver:

Meares GP. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,134.

Council of Science Editors:

Meares GP. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,134


University of Louisville

11. Wang, Huizhi, 1975-. The role of glycogen synthase kinase 3-beta in interferon beta biology.

Degree: PhD, 2009, University of Louisville

 It has been shown that GSK3 ß plays a critical role in the inflammation response by differentially regulating MyD88-dependent pro- and anti-inflammatory cytokines production in… (more)

Subjects/Keywords: Interferon; Glycogen synthase kinase 3; Toll-like receptors; IL-10

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APA (6th Edition):

Wang, Huizhi, 1. (2009). The role of glycogen synthase kinase 3-beta in interferon beta biology. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1516 ; https://ir.library.louisville.edu/etd/1516

Chicago Manual of Style (16th Edition):

Wang, Huizhi, 1975-. “The role of glycogen synthase kinase 3-beta in interferon beta biology.” 2009. Doctoral Dissertation, University of Louisville. Accessed October 17, 2019. 10.18297/etd/1516 ; https://ir.library.louisville.edu/etd/1516.

MLA Handbook (7th Edition):

Wang, Huizhi, 1975-. “The role of glycogen synthase kinase 3-beta in interferon beta biology.” 2009. Web. 17 Oct 2019.

Vancouver:

Wang, Huizhi 1. The role of glycogen synthase kinase 3-beta in interferon beta biology. [Internet] [Doctoral dissertation]. University of Louisville; 2009. [cited 2019 Oct 17]. Available from: 10.18297/etd/1516 ; https://ir.library.louisville.edu/etd/1516.

Council of Science Editors:

Wang, Huizhi 1. The role of glycogen synthase kinase 3-beta in interferon beta biology. [Doctoral Dissertation]. University of Louisville; 2009. Available from: 10.18297/etd/1516 ; https://ir.library.louisville.edu/etd/1516


University of Alberta

12. Omar, Mohamed Abdalla. Cardioprotection by Drug-Induced Changes in Glucose and Glycogen Metabolism.

Degree: PhD, Department of Pharmacology, 2011, University of Alberta

 Myocardial energy substrate metabolism is subjected to significant changes during ischemia and reperfusion (I/R), which can greatly influence post-ischemic recovery of left-ventricular (LV) mechanical function.… (more)

Subjects/Keywords: isolated perfused working rat heart; myocardial glycolysis; phosphofructokinase; protein phosphatases; Myocardial glycogen metabolism; glycogen synthase kinase-3; p38 mitogen-activated protein kinase; myocardial glucose uptake; 5'AMP-activated protein kinase; Myocardial energy substrate metabolism; Myocardial Ischemia/reperfusion injury; adenosine; Myocardial glucose metabolism

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APA (6th Edition):

Omar, M. A. (2011). Cardioprotection by Drug-Induced Changes in Glucose and Glycogen Metabolism. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/0z708w682

Chicago Manual of Style (16th Edition):

Omar, Mohamed Abdalla. “Cardioprotection by Drug-Induced Changes in Glucose and Glycogen Metabolism.” 2011. Doctoral Dissertation, University of Alberta. Accessed October 17, 2019. https://era.library.ualberta.ca/files/0z708w682.

MLA Handbook (7th Edition):

Omar, Mohamed Abdalla. “Cardioprotection by Drug-Induced Changes in Glucose and Glycogen Metabolism.” 2011. Web. 17 Oct 2019.

Vancouver:

Omar MA. Cardioprotection by Drug-Induced Changes in Glucose and Glycogen Metabolism. [Internet] [Doctoral dissertation]. University of Alberta; 2011. [cited 2019 Oct 17]. Available from: https://era.library.ualberta.ca/files/0z708w682.

Council of Science Editors:

Omar MA. Cardioprotection by Drug-Induced Changes in Glucose and Glycogen Metabolism. [Doctoral Dissertation]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/0z708w682

13. Franklin, Aimee Vinson. Elucidating The Role Of Gsk3 In Synaptic And Cognitive Deficits In Fragile X Syndrome.

Degree: 2014, University of Alabama – Birmingham

Fragile X Syndrome (FX) is the most common inherited form of mental retarda-tion. Prominent characteristics of FX are mimicked in a mouse model with deleted… (more)

Subjects/Keywords: Disease Models; Animal Fragile X Mental Retardation Protein – genetics. Fragile X Syndrome – drug therapy. Glycogen Synthase Kinase 3 – antagonists & inhibitors Glycogen Synthase Kinase 3 – metabolism. Lithium Chloride – therapeutic use.

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APA (6th Edition):

Franklin, A. V. (2014). Elucidating The Role Of Gsk3 In Synaptic And Cognitive Deficits In Fragile X Syndrome. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Franklin, Aimee Vinson. “Elucidating The Role Of Gsk3 In Synaptic And Cognitive Deficits In Fragile X Syndrome.” 2014. Thesis, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Franklin, Aimee Vinson. “Elucidating The Role Of Gsk3 In Synaptic And Cognitive Deficits In Fragile X Syndrome.” 2014. Web. 17 Oct 2019.

Vancouver:

Franklin AV. Elucidating The Role Of Gsk3 In Synaptic And Cognitive Deficits In Fragile X Syndrome. [Internet] [Thesis]. University of Alabama – Birmingham; 2014. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Franklin AV. Elucidating The Role Of Gsk3 In Synaptic And Cognitive Deficits In Fragile X Syndrome. [Thesis]. University of Alabama – Birmingham; 2014. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

14. Fallah, Mosoka Papa. ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES.

Degree: 2011, University of Kentucky

 Immunosenescence results in reduced immune response to infections with Streptococcus pneumoniae as well as to pneumococcal polysaccharide vaccines. The antibody response to the capsular polysaccharide… (more)

Subjects/Keywords: Toll-like receptor; PI3 kinase; Glycogen synthase kinase-3; macrophages; aging; Immunity; Immunopathology; Medical Immunology

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APA (6th Edition):

Fallah, M. P. (2011). ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/205

Chicago Manual of Style (16th Edition):

Fallah, Mosoka Papa. “ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES.” 2011. Doctoral Dissertation, University of Kentucky. Accessed October 17, 2019. https://uknowledge.uky.edu/gradschool_diss/205.

MLA Handbook (7th Edition):

Fallah, Mosoka Papa. “ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES.” 2011. Web. 17 Oct 2019.

Vancouver:

Fallah MP. ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES. [Internet] [Doctoral dissertation]. University of Kentucky; 2011. [cited 2019 Oct 17]. Available from: https://uknowledge.uky.edu/gradschool_diss/205.

Council of Science Editors:

Fallah MP. ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES. [Doctoral Dissertation]. University of Kentucky; 2011. Available from: https://uknowledge.uky.edu/gradschool_diss/205

15. Braginskaja, Elena. Role of the Glycogen Synthase Kinase 3 for the Retinal Development and Homeostasis : Rôle de la Glycogène Synthase Kinase 3 dans le Développement et l'Homéostasie de la Rétine.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Paris Saclay

 Les modifications post-traductionnelles (MPTs) permettent un haut degré de régulation de l'expression des gènes en générant une diversité fonctionnelle au niveau du protéome. Dans le… (more)

Subjects/Keywords: Développement; Rétine; Glycogène Synthase Kinase 3; Neurodégénérescence; Photorécepteurs; Cellules ganglionnaires; Development; Retina; Glycogen Synthase Kinase 3; Neurodegeneration; Photoreceptors; Ganglion cells

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APA (6th Edition):

Braginskaja, E. (2018). Role of the Glycogen Synthase Kinase 3 for the Retinal Development and Homeostasis : Rôle de la Glycogène Synthase Kinase 3 dans le Développement et l'Homéostasie de la Rétine. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2018SACLS060

Chicago Manual of Style (16th Edition):

Braginskaja, Elena. “Role of the Glycogen Synthase Kinase 3 for the Retinal Development and Homeostasis : Rôle de la Glycogène Synthase Kinase 3 dans le Développement et l'Homéostasie de la Rétine.” 2018. Doctoral Dissertation, Paris Saclay. Accessed October 17, 2019. http://www.theses.fr/2018SACLS060.

MLA Handbook (7th Edition):

Braginskaja, Elena. “Role of the Glycogen Synthase Kinase 3 for the Retinal Development and Homeostasis : Rôle de la Glycogène Synthase Kinase 3 dans le Développement et l'Homéostasie de la Rétine.” 2018. Web. 17 Oct 2019.

Vancouver:

Braginskaja E. Role of the Glycogen Synthase Kinase 3 for the Retinal Development and Homeostasis : Rôle de la Glycogène Synthase Kinase 3 dans le Développement et l'Homéostasie de la Rétine. [Internet] [Doctoral dissertation]. Paris Saclay; 2018. [cited 2019 Oct 17]. Available from: http://www.theses.fr/2018SACLS060.

Council of Science Editors:

Braginskaja E. Role of the Glycogen Synthase Kinase 3 for the Retinal Development and Homeostasis : Rôle de la Glycogène Synthase Kinase 3 dans le Développement et l'Homéostasie de la Rétine. [Doctoral Dissertation]. Paris Saclay; 2018. Available from: http://www.theses.fr/2018SACLS060


Florida International University

16. Sun, Tong. Glycogen Synthase Kinase 3 Influences Cell Motility and Chemotaxis by Regulating Phosphatidylinositol 3 Kinase Localization in Dictyostelium discoideum.

Degree: PhD, Biology, 2013, Florida International University

Glycogen Synthase Kinase 3 (GSK3), a serine/threonine kinase initially characterized in the context of glycogen metabolism, has been repeatedly realized as a multitasking protein… (more)

Subjects/Keywords: Glycogen Synthase Kinase 3; Ras; phosphatidylinositol 3; 4; 5-triphosphate; phosphatidylinositol-3-kinase; chemotaxis; Dictyostelium discoideum

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APA (6th Edition):

Sun, T. (2013). Glycogen Synthase Kinase 3 Influences Cell Motility and Chemotaxis by Regulating Phosphatidylinositol 3 Kinase Localization in Dictyostelium discoideum. (Doctoral Dissertation). Florida International University. Retrieved from http://digitalcommons.fiu.edu/etd/807 ; 10.25148/etd.FI13040103 ; FI13040103

Chicago Manual of Style (16th Edition):

Sun, Tong. “Glycogen Synthase Kinase 3 Influences Cell Motility and Chemotaxis by Regulating Phosphatidylinositol 3 Kinase Localization in Dictyostelium discoideum.” 2013. Doctoral Dissertation, Florida International University. Accessed October 17, 2019. http://digitalcommons.fiu.edu/etd/807 ; 10.25148/etd.FI13040103 ; FI13040103.

MLA Handbook (7th Edition):

Sun, Tong. “Glycogen Synthase Kinase 3 Influences Cell Motility and Chemotaxis by Regulating Phosphatidylinositol 3 Kinase Localization in Dictyostelium discoideum.” 2013. Web. 17 Oct 2019.

Vancouver:

Sun T. Glycogen Synthase Kinase 3 Influences Cell Motility and Chemotaxis by Regulating Phosphatidylinositol 3 Kinase Localization in Dictyostelium discoideum. [Internet] [Doctoral dissertation]. Florida International University; 2013. [cited 2019 Oct 17]. Available from: http://digitalcommons.fiu.edu/etd/807 ; 10.25148/etd.FI13040103 ; FI13040103.

Council of Science Editors:

Sun T. Glycogen Synthase Kinase 3 Influences Cell Motility and Chemotaxis by Regulating Phosphatidylinositol 3 Kinase Localization in Dictyostelium discoideum. [Doctoral Dissertation]. Florida International University; 2013. Available from: http://digitalcommons.fiu.edu/etd/807 ; 10.25148/etd.FI13040103 ; FI13040103


Temple University

17. Miller, Jonathan S. GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses.

Degree: PhD, 2009, Temple University

Pharmacology

Cocaine is a highly abused psychostimulant with repeated use potential culminating in addiction, a disease associated with compulsive drug seeking, use and high rates… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Brain; Cocaine; Dopamine; Glutamate; Glycogen Synthase Kinase-3; Mouse

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APA (6th Edition):

Miller, J. S. (2009). GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,40192

Chicago Manual of Style (16th Edition):

Miller, Jonathan S. “GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses.” 2009. Doctoral Dissertation, Temple University. Accessed October 17, 2019. http://digital.library.temple.edu/u?/p245801coll10,40192.

MLA Handbook (7th Edition):

Miller, Jonathan S. “GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses.” 2009. Web. 17 Oct 2019.

Vancouver:

Miller JS. GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 17]. Available from: http://digital.library.temple.edu/u?/p245801coll10,40192.

Council of Science Editors:

Miller JS. GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,40192


East Carolina University

18. Sokolosky, Melissa Lynn. The Role of GSK-3[beta] in MCF-7 Breast Cancer Cell Signaling and Drug Resistance.

Degree: 2011, East Carolina University

Glycogen synthase kinase- 3beta (GSK-3beta) is well documented to participate in a complex array of critical cellular processes. This versatile protein is involved in numerous… (more)

Subjects/Keywords: Breast – Cancer – Research; Cancer – Treatment – Research; Glycogen synthase kinase-3; Protein kinases

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APA (6th Edition):

Sokolosky, M. L. (2011). The Role of GSK-3[beta] in MCF-7 Breast Cancer Cell Signaling and Drug Resistance. (Masters Thesis). East Carolina University. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14762

Chicago Manual of Style (16th Edition):

Sokolosky, Melissa Lynn. “The Role of GSK-3[beta] in MCF-7 Breast Cancer Cell Signaling and Drug Resistance.” 2011. Masters Thesis, East Carolina University. Accessed October 17, 2019. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14762.

MLA Handbook (7th Edition):

Sokolosky, Melissa Lynn. “The Role of GSK-3[beta] in MCF-7 Breast Cancer Cell Signaling and Drug Resistance.” 2011. Web. 17 Oct 2019.

Vancouver:

Sokolosky ML. The Role of GSK-3[beta] in MCF-7 Breast Cancer Cell Signaling and Drug Resistance. [Internet] [Masters thesis]. East Carolina University; 2011. [cited 2019 Oct 17]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14762.

Council of Science Editors:

Sokolosky ML. The Role of GSK-3[beta] in MCF-7 Breast Cancer Cell Signaling and Drug Resistance. [Masters Thesis]. East Carolina University; 2011. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=14762


Columbia University

19. De, Arnab. Understanding two inhibitors of NF-κB: A20 and IκBβ.

Degree: 2014, Columbia University

 While prompt activation of NF-κB is essential for optimal immune response, it is equally important to terminate the response to avoid tissue damage and perhaps… (more)

Subjects/Keywords: Glycogen synthase kinase-3; NF-kappa B (DNA-binding protein); Immunology; Biochemistry; Chemistry

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APA (6th Edition):

De, A. (2014). Understanding two inhibitors of NF-κB: A20 and IκBβ. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8QC01JX

Chicago Manual of Style (16th Edition):

De, Arnab. “Understanding two inhibitors of NF-κB: A20 and IκBβ.” 2014. Doctoral Dissertation, Columbia University. Accessed October 17, 2019. https://doi.org/10.7916/D8QC01JX.

MLA Handbook (7th Edition):

De, Arnab. “Understanding two inhibitors of NF-κB: A20 and IκBβ.” 2014. Web. 17 Oct 2019.

Vancouver:

De A. Understanding two inhibitors of NF-κB: A20 and IκBβ. [Internet] [Doctoral dissertation]. Columbia University; 2014. [cited 2019 Oct 17]. Available from: https://doi.org/10.7916/D8QC01JX.

Council of Science Editors:

De A. Understanding two inhibitors of NF-κB: A20 and IκBβ. [Doctoral Dissertation]. Columbia University; 2014. Available from: https://doi.org/10.7916/D8QC01JX


University of Illinois – Urbana-Champaign

20. Scavuzzo, Claire. Use it and boost it: cognitive and physical activities modulate cognition via BDNF-TrkB signaling.

Degree: PhD, 0323, 2014, University of Illinois – Urbana-Champaign

 Our motto “Use it and boost it” suggests that stimulating experiences enhance neural functioning. Previous work has shown that cognition is enhanced by mental and… (more)

Subjects/Keywords: hippocampus; striatum; place learning; response learning; multiple memory systems; glycogen synthase kinase 3 beta

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APA (6th Edition):

Scavuzzo, C. (2014). Use it and boost it: cognitive and physical activities modulate cognition via BDNF-TrkB signaling. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/50428

Chicago Manual of Style (16th Edition):

Scavuzzo, Claire. “Use it and boost it: cognitive and physical activities modulate cognition via BDNF-TrkB signaling.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 17, 2019. http://hdl.handle.net/2142/50428.

MLA Handbook (7th Edition):

Scavuzzo, Claire. “Use it and boost it: cognitive and physical activities modulate cognition via BDNF-TrkB signaling.” 2014. Web. 17 Oct 2019.

Vancouver:

Scavuzzo C. Use it and boost it: cognitive and physical activities modulate cognition via BDNF-TrkB signaling. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2142/50428.

Council of Science Editors:

Scavuzzo C. Use it and boost it: cognitive and physical activities modulate cognition via BDNF-TrkB signaling. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/50428


University of Miami

21. Grieco, Steven F. Antidepressant and Molecular Responses to Ketamine Linked to its Inhibition of Glycogen Synthase Kinase-3 (GSK3).

Degree: PhD, Biochemistry and Molecular Biology (Medicine), 2016, University of Miami

  Herein are described two novel outcomes, the up-regulation of 5HTR2C cluster miRNAs and IGF2, as a result of GSK3 inhibition by an antidepressant dose… (more)

Subjects/Keywords: Depression; Glycogen Synthase Kinase-3; Ketamine; Hippocampus, microRNA; Insulin-Like Growth Factor 2

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APA (6th Edition):

Grieco, S. F. (2016). Antidepressant and Molecular Responses to Ketamine Linked to its Inhibition of Glycogen Synthase Kinase-3 (GSK3). (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1737

Chicago Manual of Style (16th Edition):

Grieco, Steven F. “Antidepressant and Molecular Responses to Ketamine Linked to its Inhibition of Glycogen Synthase Kinase-3 (GSK3).” 2016. Doctoral Dissertation, University of Miami. Accessed October 17, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/1737.

MLA Handbook (7th Edition):

Grieco, Steven F. “Antidepressant and Molecular Responses to Ketamine Linked to its Inhibition of Glycogen Synthase Kinase-3 (GSK3).” 2016. Web. 17 Oct 2019.

Vancouver:

Grieco SF. Antidepressant and Molecular Responses to Ketamine Linked to its Inhibition of Glycogen Synthase Kinase-3 (GSK3). [Internet] [Doctoral dissertation]. University of Miami; 2016. [cited 2019 Oct 17]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1737.

Council of Science Editors:

Grieco SF. Antidepressant and Molecular Responses to Ketamine Linked to its Inhibition of Glycogen Synthase Kinase-3 (GSK3). [Doctoral Dissertation]. University of Miami; 2016. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1737


McGill University

22. Danek, Eric Ian. Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42.

Degree: PhD, Department of Anatomy and Cell Biology., 2008, McGill University

 The Rho GTPases form a diverse family of proteins which regulates numerous cell processes from cytoskeletal reorganization to control of gene expression. They are negatively… (more)

Subjects/Keywords: GTPase-Activating Proteins  – metabolism.; Mitogen-Activated Protein Kinase 3  – metabolism.; Glycogen Synthase Kinase 3  – metabolism.; Phosphorylation.; rac1 GTP-Binding Protein  – metabolism.; cdc42 GTP-Binding Protein  – metabolism.

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APA (6th Edition):

Danek, E. I. (2008). Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile113853.pdf

Chicago Manual of Style (16th Edition):

Danek, Eric Ian. “Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42.” 2008. Doctoral Dissertation, McGill University. Accessed October 17, 2019. http://digitool.library.mcgill.ca/thesisfile113853.pdf.

MLA Handbook (7th Edition):

Danek, Eric Ian. “Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42.” 2008. Web. 17 Oct 2019.

Vancouver:

Danek EI. Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42. [Internet] [Doctoral dissertation]. McGill University; 2008. [cited 2019 Oct 17]. Available from: http://digitool.library.mcgill.ca/thesisfile113853.pdf.

Council of Science Editors:

Danek EI. Characterization of regulatory mechanisms of CdGAP, a negative regulator of the small GTPases Rac1 and Cdc42. [Doctoral Dissertation]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile113853.pdf


East Carolina University

23. Davis, Nicole Marie. Targeting aberrant signaling in the EGFR1/HER2/PI3K/PTEN/Akt/mTORC1/GSK-3 pathway : Novel therapeutic interventions in breast cancer.

Degree: 2015, East Carolina University

 Breast cancer is attributed to being the second most deadly cancer in American women. Approximately 1 in 8 women will be diagnosed with breast cancer… (more)

Subjects/Keywords: Immunology; Biology; Cellular biology; Breast cancer; Glycogen synthase-3 beta; Targeted therapy; Therapy resistance; Cells – Growth – Regulation; Glycogen synthase kinase-3 – Research; Breast – Cancer – Research

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APA (6th Edition):

Davis, N. M. (2015). Targeting aberrant signaling in the EGFR1/HER2/PI3K/PTEN/Akt/mTORC1/GSK-3 pathway : Novel therapeutic interventions in breast cancer. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4924

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davis, Nicole Marie. “Targeting aberrant signaling in the EGFR1/HER2/PI3K/PTEN/Akt/mTORC1/GSK-3 pathway : Novel therapeutic interventions in breast cancer.” 2015. Thesis, East Carolina University. Accessed October 17, 2019. http://hdl.handle.net/10342/4924.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davis, Nicole Marie. “Targeting aberrant signaling in the EGFR1/HER2/PI3K/PTEN/Akt/mTORC1/GSK-3 pathway : Novel therapeutic interventions in breast cancer.” 2015. Web. 17 Oct 2019.

Vancouver:

Davis NM. Targeting aberrant signaling in the EGFR1/HER2/PI3K/PTEN/Akt/mTORC1/GSK-3 pathway : Novel therapeutic interventions in breast cancer. [Internet] [Thesis]. East Carolina University; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/10342/4924.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davis NM. Targeting aberrant signaling in the EGFR1/HER2/PI3K/PTEN/Akt/mTORC1/GSK-3 pathway : Novel therapeutic interventions in breast cancer. [Thesis]. East Carolina University; 2015. Available from: http://hdl.handle.net/10342/4924

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

24. McAlpine, Cameron. THE ROLE OF GLYCOGEN SYNTHASE KINASE-3α/β IN ENDOPLASMIC RETICULUM STRESS AND ATHEROSCLEROSIS.

Degree: PhD, 2015, McMaster University

Atherosclerosis is a multifactorial inflammatory disease of the arterial wall and its clinical manifestations, including myocardial infarction and stroke, are the leading causes of death… (more)

Subjects/Keywords: GLYCOGEN SYNTHASE KINASE-3α/β; ATHEROSCLEROSIS

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APA (6th Edition):

McAlpine, C. (2015). THE ROLE OF GLYCOGEN SYNTHASE KINASE-3α/β IN ENDOPLASMIC RETICULUM STRESS AND ATHEROSCLEROSIS. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/18111

Chicago Manual of Style (16th Edition):

McAlpine, Cameron. “THE ROLE OF GLYCOGEN SYNTHASE KINASE-3α/β IN ENDOPLASMIC RETICULUM STRESS AND ATHEROSCLEROSIS.” 2015. Doctoral Dissertation, McMaster University. Accessed October 17, 2019. http://hdl.handle.net/11375/18111.

MLA Handbook (7th Edition):

McAlpine, Cameron. “THE ROLE OF GLYCOGEN SYNTHASE KINASE-3α/β IN ENDOPLASMIC RETICULUM STRESS AND ATHEROSCLEROSIS.” 2015. Web. 17 Oct 2019.

Vancouver:

McAlpine C. THE ROLE OF GLYCOGEN SYNTHASE KINASE-3α/β IN ENDOPLASMIC RETICULUM STRESS AND ATHEROSCLEROSIS. [Internet] [Doctoral dissertation]. McMaster University; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/11375/18111.

Council of Science Editors:

McAlpine C. THE ROLE OF GLYCOGEN SYNTHASE KINASE-3α/β IN ENDOPLASMIC RETICULUM STRESS AND ATHEROSCLEROSIS. [Doctoral Dissertation]. McMaster University; 2015. Available from: http://hdl.handle.net/11375/18111

25. Ladeira, Rodolfo Braga. Atividade da enzima GSK-3B em pacientes idosos portadores de transtorno bipolar medicados.

Degree: Mestrado, Psiquiatria, 2012, University of São Paulo

Objetivo: A glicogênio sintase quinase-3 beta (GSK-3B) é uma enzima presente em diversos sistemas biológicos e está envolvida na fisiopatologia de vários transtornos neuropsiquiátricos, incluindo… (more)

Subjects/Keywords: Aged; Bipolar disorder/physiopathology; Glycogen synthase kinase 3; Idoso; Lithium; Lítio; Quinase 3 da glicogênio sintase; Transtorno bipolar/fisiopatologia

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APA (6th Edition):

Ladeira, R. B. (2012). Atividade da enzima GSK-3B em pacientes idosos portadores de transtorno bipolar medicados. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5142/tde-01102012-165130/ ;

Chicago Manual of Style (16th Edition):

Ladeira, Rodolfo Braga. “Atividade da enzima GSK-3B em pacientes idosos portadores de transtorno bipolar medicados.” 2012. Masters Thesis, University of São Paulo. Accessed October 17, 2019. http://www.teses.usp.br/teses/disponiveis/5/5142/tde-01102012-165130/ ;.

MLA Handbook (7th Edition):

Ladeira, Rodolfo Braga. “Atividade da enzima GSK-3B em pacientes idosos portadores de transtorno bipolar medicados.” 2012. Web. 17 Oct 2019.

Vancouver:

Ladeira RB. Atividade da enzima GSK-3B em pacientes idosos portadores de transtorno bipolar medicados. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2019 Oct 17]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5142/tde-01102012-165130/ ;.

Council of Science Editors:

Ladeira RB. Atividade da enzima GSK-3B em pacientes idosos portadores de transtorno bipolar medicados. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5142/tde-01102012-165130/ ;

26. Diniz, Breno Satler de Oliveira. A atividade da enzima Glicogênio Sintase Quinase 3 Beta (GSK-3B) em pacientes idosos com depressão maior: associação com parâmetros clínicos, psicopatológicos e cognitivos.

Degree: PhD, Psiquiatria, 2011, University of São Paulo

Apesar da elevada prevalência dos transtornos depressivos em idosos, os mecanismos fisiopatológicos subjacentes a estes quadros são pouco conhecidos. Atualmente, o principal foco dos estudos… (more)

Subjects/Keywords: Cognição; Cognition; Depressão/fisiopatologia; Depression/physiopathology; Elderly; Glicogênio sintase quinase 3; Glycogen synthase kinase 3; Idoso

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APA (6th Edition):

Diniz, B. S. d. O. (2011). A atividade da enzima Glicogênio Sintase Quinase 3 Beta (GSK-3B) em pacientes idosos com depressão maior: associação com parâmetros clínicos, psicopatológicos e cognitivos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5142/tde-17062011-164159/ ;

Chicago Manual of Style (16th Edition):

Diniz, Breno Satler de Oliveira. “A atividade da enzima Glicogênio Sintase Quinase 3 Beta (GSK-3B) em pacientes idosos com depressão maior: associação com parâmetros clínicos, psicopatológicos e cognitivos.” 2011. Doctoral Dissertation, University of São Paulo. Accessed October 17, 2019. http://www.teses.usp.br/teses/disponiveis/5/5142/tde-17062011-164159/ ;.

MLA Handbook (7th Edition):

Diniz, Breno Satler de Oliveira. “A atividade da enzima Glicogênio Sintase Quinase 3 Beta (GSK-3B) em pacientes idosos com depressão maior: associação com parâmetros clínicos, psicopatológicos e cognitivos.” 2011. Web. 17 Oct 2019.

Vancouver:

Diniz BSdO. A atividade da enzima Glicogênio Sintase Quinase 3 Beta (GSK-3B) em pacientes idosos com depressão maior: associação com parâmetros clínicos, psicopatológicos e cognitivos. [Internet] [Doctoral dissertation]. University of São Paulo; 2011. [cited 2019 Oct 17]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5142/tde-17062011-164159/ ;.

Council of Science Editors:

Diniz BSdO. A atividade da enzima Glicogênio Sintase Quinase 3 Beta (GSK-3B) em pacientes idosos com depressão maior: associação com parâmetros clínicos, psicopatológicos e cognitivos. [Doctoral Dissertation]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5142/tde-17062011-164159/ ;

27. Ferreira, Aline Siqueira. Uso contínuo de antipsicóticos modula fosfolipase A2 e glicogênico sintase quinase-3 beta em plaquetas de pacientes com esquizofrenia.

Degree: PhD, Fisiopatologia Experimental, 2012, University of São Paulo

Duas enzimas têm-se destacado como possíveis marcadores biológicos periféricos na esquizofrenia: a fosfolipase A2 (PLA2) e a glicogênio sintase quinase-3 beta (GSK-3B). Essas moléculas exercem… (more)

Subjects/Keywords: Esquizofrenia; Fosfolipases A2; Glicogênio sintase quinase 3; Glycogen synthase kinase 3; Phospholipases A2; Plaquetas; Platelets; Schizophrenia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ferreira, A. S. (2012). Uso contínuo de antipsicóticos modula fosfolipase A2 e glicogênico sintase quinase-3 beta em plaquetas de pacientes com esquizofrenia. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052012-163420/ ;

Chicago Manual of Style (16th Edition):

Ferreira, Aline Siqueira. “Uso contínuo de antipsicóticos modula fosfolipase A2 e glicogênico sintase quinase-3 beta em plaquetas de pacientes com esquizofrenia.” 2012. Doctoral Dissertation, University of São Paulo. Accessed October 17, 2019. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052012-163420/ ;.

MLA Handbook (7th Edition):

Ferreira, Aline Siqueira. “Uso contínuo de antipsicóticos modula fosfolipase A2 e glicogênico sintase quinase-3 beta em plaquetas de pacientes com esquizofrenia.” 2012. Web. 17 Oct 2019.

Vancouver:

Ferreira AS. Uso contínuo de antipsicóticos modula fosfolipase A2 e glicogênico sintase quinase-3 beta em plaquetas de pacientes com esquizofrenia. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Oct 17]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052012-163420/ ;.

Council of Science Editors:

Ferreira AS. Uso contínuo de antipsicóticos modula fosfolipase A2 e glicogênico sintase quinase-3 beta em plaquetas de pacientes com esquizofrenia. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052012-163420/ ;


University of Pennsylvania

28. Shinde, Mansi. Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3.

Degree: 2016, University of Pennsylvania

Glycogen Synthase Kinase-3 (GSK-3) is a constitutively active, ubiquitously expressed kinase that acts as a critical regulator of many signaling pathways. These pathways, when dysregulated,… (more)

Subjects/Keywords: Alternative Splicing; Glycogen Synthase Kinase-3; GSK-3; Murine embryonic stem cells; Phosphoproteome; Cell Biology; Developmental Biology; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shinde, M. (2016). Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shinde, Mansi. “Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3.” 2016. Thesis, University of Pennsylvania. Accessed October 17, 2019. https://repository.upenn.edu/edissertations/2583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shinde, Mansi. “Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3.” 2016. Web. 17 Oct 2019.

Vancouver:

Shinde M. Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2019 Oct 17]. Available from: https://repository.upenn.edu/edissertations/2583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shinde M. Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/2583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Mcguire, Donald. Cd5 Regulation Of T Helper Cell Differentiation And Cytokine Signaling.

Degree: 2014, University of Alabama – Birmingham

CD5 is expressed on T and B1a cells and is an important regulator of cell survival and activation. Engagement of CD5 promotes cell survival through… (more)

Subjects/Keywords: Antigens; CD5 – physiology. Casein Kinase II – metabolism. Cell Differentiation Glycogen Synthase Kinase 3. Interleukin-10 Signal Transduction – immunology. Th17 Cells. T-Lymphocyte Subsets

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mcguire, D. (2014). Cd5 Regulation Of T Helper Cell Differentiation And Cytokine Signaling. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1757

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mcguire, Donald. “Cd5 Regulation Of T Helper Cell Differentiation And Cytokine Signaling.” 2014. Thesis, University of Alabama – Birmingham. Accessed October 17, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1757.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mcguire, Donald. “Cd5 Regulation Of T Helper Cell Differentiation And Cytokine Signaling.” 2014. Web. 17 Oct 2019.

Vancouver:

Mcguire D. Cd5 Regulation Of T Helper Cell Differentiation And Cytokine Signaling. [Internet] [Thesis]. University of Alabama – Birmingham; 2014. [cited 2019 Oct 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1757.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mcguire D. Cd5 Regulation Of T Helper Cell Differentiation And Cytokine Signaling. [Thesis]. University of Alabama – Birmingham; 2014. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1757

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

30. Mok, Sue-Ann. Retrograde signaling mechanisms of nerve growth factor regulating the survival and apoptosis of sympathetic neurons.

Degree: PhD, Department of Cell Biology, 2009, University of Alberta

 The survival of several neuron populations during development, including sympathetic neurons, is strictly regulated by neurotrophins such as nerve growth factor (NGF) released from innervation… (more)

Subjects/Keywords: neurotrophin; retrograde signaling; c-jun; apoptosis; NGF; nerve growh factor; glycogen synthase kinase-3; sympathetic neuron; retrograde apoptotic signal; axon

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mok, S. (2009). Retrograde signaling mechanisms of nerve growth factor regulating the survival and apoptosis of sympathetic neurons. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cj82k883x

Chicago Manual of Style (16th Edition):

Mok, Sue-Ann. “Retrograde signaling mechanisms of nerve growth factor regulating the survival and apoptosis of sympathetic neurons.” 2009. Doctoral Dissertation, University of Alberta. Accessed October 17, 2019. https://era.library.ualberta.ca/files/cj82k883x.

MLA Handbook (7th Edition):

Mok, Sue-Ann. “Retrograde signaling mechanisms of nerve growth factor regulating the survival and apoptosis of sympathetic neurons.” 2009. Web. 17 Oct 2019.

Vancouver:

Mok S. Retrograde signaling mechanisms of nerve growth factor regulating the survival and apoptosis of sympathetic neurons. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2019 Oct 17]. Available from: https://era.library.ualberta.ca/files/cj82k883x.

Council of Science Editors:

Mok S. Retrograde signaling mechanisms of nerve growth factor regulating the survival and apoptosis of sympathetic neurons. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/cj82k883x

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