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You searched for subject:( Genetic Diseases Inborn enzymology 60). Showing records 1 – 30 of 32072 total matches.

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1. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

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APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 16 Sep 2019.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

2. Lax, Peter J. Congenital xerostomia and associated anomalies : a proposed mode of inheritance.

Degree: Certificate in Orthodontics, Orthodontics, 1975, Oregon Health Sciences University

Subjects/Keywords: Xerostomia  – genetics; Genetic Diseases, Inborn

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APA (6th Edition):

Lax, P. J. (1975). Congenital xerostomia and associated anomalies : a proposed mode of inheritance. (Thesis). Oregon Health Sciences University. Retrieved from doi:10.6083/M48S4N4D ; http://digitalcommons.ohsu.edu/etd/2490

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lax, Peter J. “Congenital xerostomia and associated anomalies : a proposed mode of inheritance.” 1975. Thesis, Oregon Health Sciences University. Accessed September 16, 2019. doi:10.6083/M48S4N4D ; http://digitalcommons.ohsu.edu/etd/2490.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lax, Peter J. “Congenital xerostomia and associated anomalies : a proposed mode of inheritance.” 1975. Web. 16 Sep 2019.

Vancouver:

Lax PJ. Congenital xerostomia and associated anomalies : a proposed mode of inheritance. [Internet] [Thesis]. Oregon Health Sciences University; 1975. [cited 2019 Sep 16]. Available from: doi:10.6083/M48S4N4D ; http://digitalcommons.ohsu.edu/etd/2490.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lax PJ. Congenital xerostomia and associated anomalies : a proposed mode of inheritance. [Thesis]. Oregon Health Sciences University; 1975. Available from: doi:10.6083/M48S4N4D ; http://digitalcommons.ohsu.edu/etd/2490

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

3. Hill, John Stuart. Genetic and environmental factors affecting the incidence of coronary artery disease in heterozygous familial hypercholesterolemia .

Degree: 1990, University of British Columbia

 Familial hypercholesterolemia (FH) is an autosomal dominant disorder in which the primary defect is a mutation in the LDL receptor. Heterozygous FH is among the… (more)

Subjects/Keywords: Hypercholesteremia; Coronary heart disease; Genetic disorders; Hypercholesterolemia; Coronary Disease; Genetic Diseases, Inborn

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APA (6th Edition):

Hill, J. S. (1990). Genetic and environmental factors affecting the incidence of coronary artery disease in heterozygous familial hypercholesterolemia . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/28810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hill, John Stuart. “Genetic and environmental factors affecting the incidence of coronary artery disease in heterozygous familial hypercholesterolemia .” 1990. Thesis, University of British Columbia. Accessed September 16, 2019. http://hdl.handle.net/2429/28810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hill, John Stuart. “Genetic and environmental factors affecting the incidence of coronary artery disease in heterozygous familial hypercholesterolemia .” 1990. Web. 16 Sep 2019.

Vancouver:

Hill JS. Genetic and environmental factors affecting the incidence of coronary artery disease in heterozygous familial hypercholesterolemia . [Internet] [Thesis]. University of British Columbia; 1990. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2429/28810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hill JS. Genetic and environmental factors affecting the incidence of coronary artery disease in heterozygous familial hypercholesterolemia . [Thesis]. University of British Columbia; 1990. Available from: http://hdl.handle.net/2429/28810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Marshall University

4. Belalcazar Maya, Andrea Del Pilar. The Transsulfuration Pathway Significantly Contributes to Glutathione Biosynthesis in Human Mammary Epithelial Cells.

Degree: 2009, Marshall University

 Cellular methylation and antioxidant metabolism are linked by the transsulfuration pathway, which converts the methionine cycle intermediate homocysteine to cysteine, a precursor for glutathione biosynthesis,… (more)

Subjects/Keywords: <; p>; Nervous system - Diseases - Genetic aspects.<; /p>; <; p>; Nervous system - Diseases - Molecular aspects.<; /p>;

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APA (6th Edition):

Belalcazar Maya, A. D. P. (2009). The Transsulfuration Pathway Significantly Contributes to Glutathione Biosynthesis in Human Mammary Epithelial Cells. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/795

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Belalcazar Maya, Andrea Del Pilar. “The Transsulfuration Pathway Significantly Contributes to Glutathione Biosynthesis in Human Mammary Epithelial Cells.” 2009. Thesis, Marshall University. Accessed September 16, 2019. http://mds.marshall.edu/etd/795.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Belalcazar Maya, Andrea Del Pilar. “The Transsulfuration Pathway Significantly Contributes to Glutathione Biosynthesis in Human Mammary Epithelial Cells.” 2009. Web. 16 Sep 2019.

Vancouver:

Belalcazar Maya ADP. The Transsulfuration Pathway Significantly Contributes to Glutathione Biosynthesis in Human Mammary Epithelial Cells. [Internet] [Thesis]. Marshall University; 2009. [cited 2019 Sep 16]. Available from: http://mds.marshall.edu/etd/795.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Belalcazar Maya ADP. The Transsulfuration Pathway Significantly Contributes to Glutathione Biosynthesis in Human Mammary Epithelial Cells. [Thesis]. Marshall University; 2009. Available from: http://mds.marshall.edu/etd/795

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Assumpção, Tatiana Malheiros. Avaliação do perfil psiquiátrico de pacientes com mucopolissacaridoses.

Degree: Mestrado, Psicologia Clínica, 2013, University of São Paulo

As mucopolissacaridoses (MPS) são um grupo de doenças metabólicas hereditárias causadas pela deficiência de enzimas lisossomais específicas, que causam alterações físicas e/ou comportamentais crônicas e… (more)

Subjects/Keywords: Behavioral genetics; Developmental disabilities; Doenças genéticas inatas; Genética comportamental; Inborn genetic diseases; Mental disorders; Mucopolissacaridoses; Mucopolysaccharidoses; Transtornos do desenvolvimento infantil; Transtornos mentais

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APA (6th Edition):

Assumpção, T. M. (2013). Avaliação do perfil psiquiátrico de pacientes com mucopolissacaridoses. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/47/47133/tde-24022014-094625/ ;

Chicago Manual of Style (16th Edition):

Assumpção, Tatiana Malheiros. “Avaliação do perfil psiquiátrico de pacientes com mucopolissacaridoses.” 2013. Masters Thesis, University of São Paulo. Accessed September 16, 2019. http://www.teses.usp.br/teses/disponiveis/47/47133/tde-24022014-094625/ ;.

MLA Handbook (7th Edition):

Assumpção, Tatiana Malheiros. “Avaliação do perfil psiquiátrico de pacientes com mucopolissacaridoses.” 2013. Web. 16 Sep 2019.

Vancouver:

Assumpção TM. Avaliação do perfil psiquiátrico de pacientes com mucopolissacaridoses. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2019 Sep 16]. Available from: http://www.teses.usp.br/teses/disponiveis/47/47133/tde-24022014-094625/ ;.

Council of Science Editors:

Assumpção TM. Avaliação do perfil psiquiátrico de pacientes com mucopolissacaridoses. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/47/47133/tde-24022014-094625/ ;

6. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 16 Sep 2019.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960


Marshall University

7. Nande, Rounak. Gene transfer in a newly established spontaneous feline fibrosarcoma cell line.

Degree: 2013, Marshall University

 Fibrosarcoma is a deadly disease in cats and is most often located at classical vaccine injections sites. More rare forms of spontaneous non-vaccination site (NSV)… (more)

Subjects/Keywords: Adenovirus; Feline; Fibrosarcoma; Gene therapy; gene transfer; spontaneous; <; p>; Gene therapy  – Research.<; /p>; <; p>; Genetic transformation  – Research.<; /p>; <; p>; Animals<; strong>; - <; /strong>; Diseases<; strong>; - <; /strong>; Molecular aspects.<; /p>;

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APA (6th Edition):

Nande, R. (2013). Gene transfer in a newly established spontaneous feline fibrosarcoma cell line. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nande, Rounak. “Gene transfer in a newly established spontaneous feline fibrosarcoma cell line.” 2013. Thesis, Marshall University. Accessed September 16, 2019. http://mds.marshall.edu/etd/429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nande, Rounak. “Gene transfer in a newly established spontaneous feline fibrosarcoma cell line.” 2013. Web. 16 Sep 2019.

Vancouver:

Nande R. Gene transfer in a newly established spontaneous feline fibrosarcoma cell line. [Internet] [Thesis]. Marshall University; 2013. [cited 2019 Sep 16]. Available from: http://mds.marshall.edu/etd/429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nande R. Gene transfer in a newly established spontaneous feline fibrosarcoma cell line. [Thesis]. Marshall University; 2013. Available from: http://mds.marshall.edu/etd/429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

8. Dinchong, Rachelle. Reducing the psychosocial impact of a false positive newborn screen for inborn errors of metabolism.

Degree: Biochemistry and Medical Genetics, 2019, University of Manitoba

 Newborn screening (NBS) is standard practice for neonatal care in Canada and screens for over 20 inborn errors of metabolism (IEM). Education about NBS is… (more)

Subjects/Keywords: newborn screening; inborn errors of metabolism; psychosocial; genetic counselling

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APA (6th Edition):

Dinchong, R. (2019). Reducing the psychosocial impact of a false positive newborn screen for inborn errors of metabolism. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34019

Chicago Manual of Style (16th Edition):

Dinchong, Rachelle. “Reducing the psychosocial impact of a false positive newborn screen for inborn errors of metabolism.” 2019. Masters Thesis, University of Manitoba. Accessed September 16, 2019. http://hdl.handle.net/1993/34019.

MLA Handbook (7th Edition):

Dinchong, Rachelle. “Reducing the psychosocial impact of a false positive newborn screen for inborn errors of metabolism.” 2019. Web. 16 Sep 2019.

Vancouver:

Dinchong R. Reducing the psychosocial impact of a false positive newborn screen for inborn errors of metabolism. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1993/34019.

Council of Science Editors:

Dinchong R. Reducing the psychosocial impact of a false positive newborn screen for inborn errors of metabolism. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34019

9. Flint, Daniel L. Proteomic Clues to the Pathogenesis of Alexander Disease.

Degree: 2012, University of Alabama – Birmingham

Since the initial report identifying mutations in GFAP as the primary genetic defect in the astrogliopathy Alexander Disease (AxD) much progress has been made in… (more)

Subjects/Keywords: Molecular biology <; br>; Neurosciences <; br>; Pathology<; br>; Alexander disease – genetics. Astrocytes – metabolism. Glial Fibrillary Acidic Protein – genetics. RHOA protein, human. ATROGIN1 protein, human. Central nervous system – Diseases – Genetic aspects. Central nervous system – Diseases – Pathogenesis. Nervous system – Degeneration – Genetic aspects. Demyelination – Genetic aspects. Neurons – Analysis. Protein kinases – Inhibitors – Therapeutic use. Proteomics. Astrocytes.

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APA (6th Edition):

Flint, D. L. (2012). Proteomic Clues to the Pathogenesis of Alexander Disease. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1776

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flint, Daniel L. “Proteomic Clues to the Pathogenesis of Alexander Disease.” 2012. Thesis, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1776.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flint, Daniel L. “Proteomic Clues to the Pathogenesis of Alexander Disease.” 2012. Web. 16 Sep 2019.

Vancouver:

Flint DL. Proteomic Clues to the Pathogenesis of Alexander Disease. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1776.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flint DL. Proteomic Clues to the Pathogenesis of Alexander Disease. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1776

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Marshall University

10. Thornton, Jesse A. Role of iron in ethanol derived hepatic stress.

Degree: 2013, Marshall University

 Chronic alcohol abuse is the third leading cause of preventable death in the United States. Ethanol metabolism causes liver injury through alterations in hepatic metabolic… (more)

Subjects/Keywords: Alcohol abuse; alcoholic liver disease; ferritin; iron deficiency; lysine acetylation; oxidative stress; <; p>; Alcohol - Physiological effect - Research.<; /p>; <; p>; Alcoholic liver diseases.<; /p>; <; p>; Ethanol - pharmacology.<; /p>;

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APA (6th Edition):

Thornton, J. A. (2013). Role of iron in ethanol derived hepatic stress. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thornton, Jesse A. “Role of iron in ethanol derived hepatic stress.” 2013. Thesis, Marshall University. Accessed September 16, 2019. http://mds.marshall.edu/etd/727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thornton, Jesse A. “Role of iron in ethanol derived hepatic stress.” 2013. Web. 16 Sep 2019.

Vancouver:

Thornton JA. Role of iron in ethanol derived hepatic stress. [Internet] [Thesis]. Marshall University; 2013. [cited 2019 Sep 16]. Available from: http://mds.marshall.edu/etd/727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thornton JA. Role of iron in ethanol derived hepatic stress. [Thesis]. Marshall University; 2013. Available from: http://mds.marshall.edu/etd/727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Styles, Nathan Allen. The characterization of the subcellular localization of bile acid CoA:N-acyltransferase.

Degree: PhD, 2007, University of Alabama – Birmingham

The liver plays a central role in metabolism. It controls protein metabolism through deamination and transamination of amino acids, removal of ammonia through urea synthesis,… (more)

Subjects/Keywords: Acyltransferases  – metabolism <; br>; Bile Acids and Salts  – chemistry <; br>; Coenzyme A Ligases  – metabolism <; br>; Liver  – enzymology <; br>; Microsomes, Liver  – enzymology

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APA (6th Edition):

Styles, N. A. (2007). The characterization of the subcellular localization of bile acid CoA:N-acyltransferase. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,138

Chicago Manual of Style (16th Edition):

Styles, Nathan Allen. “The characterization of the subcellular localization of bile acid CoA:N-acyltransferase.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,138.

MLA Handbook (7th Edition):

Styles, Nathan Allen. “The characterization of the subcellular localization of bile acid CoA:N-acyltransferase.” 2007. Web. 16 Sep 2019.

Vancouver:

Styles NA. The characterization of the subcellular localization of bile acid CoA:N-acyltransferase. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,138.

Council of Science Editors:

Styles NA. The characterization of the subcellular localization of bile acid CoA:N-acyltransferase. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,138

12. Stella, David Ray. Lens Cataract: Biochemical Analysis Of The Alpha Crystallins.

Degree: PhD, 2010, University of Alabama – Birmingham

The cataract is a common ailment affecting the aged population. It appears over time and affects the quality of one’s life by the eventual loss… (more)

Subjects/Keywords: Alzheimer Disease<; br>; Brain – enzymology<; br>; Glycogen Synthase Kinase 3 – metabolism.<; br>; Microscopy, Electron<; br>; Subcellular Fractions – enzymology.

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APA (6th Edition):

Stella, D. R. (2010). Lens Cataract: Biochemical Analysis Of The Alpha Crystallins. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1401

Chicago Manual of Style (16th Edition):

Stella, David Ray. “Lens Cataract: Biochemical Analysis Of The Alpha Crystallins.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1401.

MLA Handbook (7th Edition):

Stella, David Ray. “Lens Cataract: Biochemical Analysis Of The Alpha Crystallins.” 2010. Web. 16 Sep 2019.

Vancouver:

Stella DR. Lens Cataract: Biochemical Analysis Of The Alpha Crystallins. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1401.

Council of Science Editors:

Stella DR. Lens Cataract: Biochemical Analysis Of The Alpha Crystallins. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1401

13. Jones, Page. Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model.

Degree: PhD, 2008, University of Alabama – Birmingham

Mutations to copper,zinc superoxide dismutase (Cu,Zn SOD or SOD1) are the only known causes of amyotrophic lateral sclerosis (ALS), a neurodegenerative disorder characterized primarily by… (more)

Subjects/Keywords: Amyotrophic Lateral Sclerosis  – enzymology <; br>; Copper  – metabolism <; br>; Disease Progression <; br>; Nerve Degeneration  – enzymology <; br>; Neurons  – metabolism <; br>; Spinal Cord  – enzymology <; br>; Superoxide Dismutase  – metabolism

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APA (6th Edition):

Jones, P. (2008). Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,370

Chicago Manual of Style (16th Edition):

Jones, Page. “Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,370.

MLA Handbook (7th Edition):

Jones, Page. “Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model.” 2008. Web. 16 Sep 2019.

Vancouver:

Jones P. Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,370.

Council of Science Editors:

Jones P. Enzymatic and proteomic analysis of spinal cord in a G93A ALS mouse model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,370


University of Gothenburg / Göteborgs Universitet

14. Zhou, Xianghua. New Roles of Filamins in Cell Signaling, Transcription and Organ Development.

Degree: 2009, University of Gothenburg / Göteborgs Universitet

 Filamins are large actin-binding proteins that stabilize delicate three-dimensional actin networks and link them to cellular membranes. They integrate cell architectural and signaling functions and… (more)

Subjects/Keywords: filamins; cell movement; hypoxia-inducible factor 1; proto-oncogene proteins c-met; mice; knockout; F-actin-binding proteins; integrins; GTB phosphohydrolases; genetic diseases; inborn; vascular endothelial cell growth factor A; Smad2 protein; hepatocyte growth factor; cartilage; osteogenesis; neovascularization; neoplasm metastasis

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APA (6th Edition):

Zhou, X. (2009). New Roles of Filamins in Cell Signaling, Transcription and Organ Development. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/19605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Xianghua. “New Roles of Filamins in Cell Signaling, Transcription and Organ Development.” 2009. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed September 16, 2019. http://hdl.handle.net/2077/19605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Xianghua. “New Roles of Filamins in Cell Signaling, Transcription and Organ Development.” 2009. Web. 16 Sep 2019.

Vancouver:

Zhou X. New Roles of Filamins in Cell Signaling, Transcription and Organ Development. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2077/19605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou X. New Roles of Filamins in Cell Signaling, Transcription and Organ Development. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. Available from: http://hdl.handle.net/2077/19605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Cook, Ian Thomas. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.

Degree: PhD, 2011, University of Alabama – Birmingham

Sulfation is an important Phase II drug metabolism reaction catalyzed by the cytosolic sulfotransferases (SULTs). SULT2A1 is a major SULT in liver and adrenal cortex… (more)

Subjects/Keywords: Cytosol  – enzymology<; br>; Enzyme Inhibitors  – pharmacology<; br>; Liver<; br>; Sulfatases  – metabolism<; br>; Sulfates  – metabolism<; br>; Sulfotransferases  – metabolism

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APA (6th Edition):

Cook, I. T. (2011). Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1047

Chicago Manual of Style (16th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1047.

MLA Handbook (7th Edition):

Cook, Ian Thomas. “Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate.” 2011. Web. 16 Sep 2019.

Vancouver:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047.

Council of Science Editors:

Cook IT. Anaylsis [sic] of the structural and kinetic properties of human SULT2A1 induced by the binding of 3'-phosphoadenosine-5'-phosphosulfate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1047

16. Dolan, Philip J. The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau.

Degree: PhD, 2010, University of Alabama – Birmingham

Alzheimer Disease (AD) is pathologically characterized by the appearance of senile plaques composed of β-amyloid (Aβ) and neurofibrillary tangles composed of the microtubule-associated protein tau.… (more)

Subjects/Keywords: Alzheimer Disease  – enzymology<; br>; Autophagy<; br>; Caspases  – metabolism<; br>; Protein Kinases  – metabolism<; br>; Signal Transduction<; br>; tau Proteins  – physiology

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APA (6th Edition):

Dolan, P. J. (2010). The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1090

Chicago Manual of Style (16th Edition):

Dolan, Philip J. “The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1090.

MLA Handbook (7th Edition):

Dolan, Philip J. “The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau.” 2010. Web. 16 Sep 2019.

Vancouver:

Dolan PJ. The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1090.

Council of Science Editors:

Dolan PJ. The role of post-translational modifications and valosin-containing protein in the turnover and stability of the microtubule-associated protein tau. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1090

17. Crimmins, Stephen Lewis. Characterization and functional analysis of Usp14.

Degree: PhD, 2007, University of Alabama – Birmingham

The ubiquitin proteasome system (UPS) is essential for regulated protein degrada-tion, a requirement for numerous neuronal process, including vesicle cycling, neuro-transmitter release, spine morphology, and… (more)

Subjects/Keywords: Ataxia  – enzymology <; br>; Ataxia  – genetics <; br>; Gene Expression Regulation, Enzymologic  – physiology <; br>; Neurons  – enzymology <; br>; Ubiquitin  – metabolism <; br>; Ubiquitin Thiolesterase  – biosynthesis <; br>; Ubiquitin Thiolesterase  – genetics

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APA (6th Edition):

Crimmins, S. L. (2007). Characterization and functional analysis of Usp14. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,118

Chicago Manual of Style (16th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,118.

MLA Handbook (7th Edition):

Crimmins, Stephen Lewis. “Characterization and functional analysis of Usp14.” 2007. Web. 16 Sep 2019.

Vancouver:

Crimmins SL. Characterization and functional analysis of Usp14. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118.

Council of Science Editors:

Crimmins SL. Characterization and functional analysis of Usp14. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,118

18. Bolisetty, Subhashini. Role Of Heme Oxygenase-1 In Acute Kidney Injury.

Degree: PhD, 2010, University of Alabama – Birmingham

Acute kidney injury (AKI), defined as the rapid loss of kidney function, is often seen in the setting of multiple organ failure in critically ill… (more)

Subjects/Keywords: Acute Kidney Injury – enzymology.<; br>; Autophagy.<; br>; Cisplatin – adverse effects.<; br>; Heme Oxygenase-1 – metabolism.<; br>; Kidney Tubules, Proximal – enzymology.<; br>; Mitochondria – physiology<; br>; Reactive Oxygen Species – metabolism

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APA (6th Edition):

Bolisetty, S. (2010). Role Of Heme Oxygenase-1 In Acute Kidney Injury. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1402

Chicago Manual of Style (16th Edition):

Bolisetty, Subhashini. “Role Of Heme Oxygenase-1 In Acute Kidney Injury.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1402.

MLA Handbook (7th Edition):

Bolisetty, Subhashini. “Role Of Heme Oxygenase-1 In Acute Kidney Injury.” 2010. Web. 16 Sep 2019.

Vancouver:

Bolisetty S. Role Of Heme Oxygenase-1 In Acute Kidney Injury. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1402.

Council of Science Editors:

Bolisetty S. Role Of Heme Oxygenase-1 In Acute Kidney Injury. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1402

19. Hu, Junbin. Structural and functional studies on heat shock protein Hsp40-Hdj1 and Golgi ER trafficking protein Get3.

Degree: PhD, 2009, University of Alabama – Birmingham

Molecular chaperones are a large group of proteins that bind and stabilize nascent polypeptides and facilitate protein folding. One major function of chaperones is to… (more)

Subjects/Keywords: Adenosine Triphosphatases  – chemistry<; br>; Guanine Nucleotide Exchange Factors  – chemistry<; br>; HSP40 Heat-Shock Proteins  – chemistry<; br>; Saccharomyces cerevisiae  – enzymology<; br>; Sequence Alignment

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APA (6th Edition):

Hu, J. (2009). Structural and functional studies on heat shock protein Hsp40-Hdj1 and Golgi ER trafficking protein Get3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,566

Chicago Manual of Style (16th Edition):

Hu, Junbin. “Structural and functional studies on heat shock protein Hsp40-Hdj1 and Golgi ER trafficking protein Get3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,566.

MLA Handbook (7th Edition):

Hu, Junbin. “Structural and functional studies on heat shock protein Hsp40-Hdj1 and Golgi ER trafficking protein Get3.” 2009. Web. 16 Sep 2019.

Vancouver:

Hu J. Structural and functional studies on heat shock protein Hsp40-Hdj1 and Golgi ER trafficking protein Get3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,566.

Council of Science Editors:

Hu J. Structural and functional studies on heat shock protein Hsp40-Hdj1 and Golgi ER trafficking protein Get3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,566

20. Chen, Ping-Chung. The role of USP14 in regulating synaptic development and function of the neuromuscular junctions.

Degree: PhD, 2010, University of Alabama – Birmingham

In the nervous system, the ubiquitin proteasome system (UPS) plays critical roles in regulating a wide diversity of cellular process such as synaptic transmission, axon… (more)

Subjects/Keywords: Neuromuscular Junction  – enzymology<; br>; Neuromuscular Junction  – growth & development<; br>; Proteasome Endopeptidase Complex  – metabolism<; br>; Synapses  – enzymology<; br>; Ubiquitin  – metabolism<; br>; Ubiquitin Thiolesterase  – physiology

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APA (6th Edition):

Chen, P. (2010). The role of USP14 in regulating synaptic development and function of the neuromuscular junctions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,992

Chicago Manual of Style (16th Edition):

Chen, Ping-Chung. “The role of USP14 in regulating synaptic development and function of the neuromuscular junctions.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,992.

MLA Handbook (7th Edition):

Chen, Ping-Chung. “The role of USP14 in regulating synaptic development and function of the neuromuscular junctions.” 2010. Web. 16 Sep 2019.

Vancouver:

Chen P. The role of USP14 in regulating synaptic development and function of the neuromuscular junctions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,992.

Council of Science Editors:

Chen P. The role of USP14 in regulating synaptic development and function of the neuromuscular junctions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,992

21. Buckoreelall, Kajal. Identification and characterization of novel adenosine cleavage enzymes in mycobacteria.

Degree: PhD, 2011, University of Alabama – Birmingham

Tuberculosis (TB) is one of the leading infectious diseases in the world. An estimated one third of the world’s population is infected with Mycobacterium tuberculosis,… (more)

Subjects/Keywords: Bacterial Proteins  – chemistry<; br>; Bacterial Proteins  – metabolism<; br>; Mycobacterium smegmatis  – enzymology<; br>; Mycobacterium tuberculosis  – enzymology<; br>; Purine-Nucleoside Phosphorylase  – chemistry<; br>; Purine-Nucleoside Phosphorylase  – metabolism

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APA (6th Edition):

Buckoreelall, K. (2011). Identification and characterization of novel adenosine cleavage enzymes in mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1136

Chicago Manual of Style (16th Edition):

Buckoreelall, Kajal. “Identification and characterization of novel adenosine cleavage enzymes in mycobacteria.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1136.

MLA Handbook (7th Edition):

Buckoreelall, Kajal. “Identification and characterization of novel adenosine cleavage enzymes in mycobacteria.” 2011. Web. 16 Sep 2019.

Vancouver:

Buckoreelall K. Identification and characterization of novel adenosine cleavage enzymes in mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1136.

Council of Science Editors:

Buckoreelall K. Identification and characterization of novel adenosine cleavage enzymes in mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1136

22. Woodard-Grice, Alencia V. (Alencia Vanay). Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.

Degree: PhD, 2008, University of Alabama – Birmingham

During monocyte activation and differentiation along the macrophage lineage, the activity of [alpha]4[beta]1 integrins is upregulated, which promotes extravasation from the vasculature and migration through… (more)

Subjects/Keywords: Amyloid Precursor Protein Secretases  – metabolism<; br>; Antigens, CD  – biosynthesis<; br>; Aspartic Endopeptidases  – metabolism<; br>; Cell Differentiation  – physiology<; br>; Integrin alpha4beta1  – metabolism<; br>; Macrophages  – enzymology<; br>; Monocytes  – enzymology<; br>; Protein Modification, Translational  – physiology<; br>; Sialyltransferases  – biosynthesis

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APA (6th Edition):

Woodard-Grice, A. V. (. V. (2008). Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,473

Chicago Manual of Style (16th Edition):

Woodard-Grice, Alencia V (Alencia Vanay). “Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,473.

MLA Handbook (7th Edition):

Woodard-Grice, Alencia V (Alencia Vanay). “Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.” 2008. Web. 16 Sep 2019.

Vancouver:

Woodard-Grice AV(V. Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,473.

Council of Science Editors:

Woodard-Grice AV(V. Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,473

23. Gaggar, Amit. Protease dysregulation: role in neutrophilic inflammation in cystic fibrosis.

Degree: PhD, 2007, University of Alabama – Birmingham

Cystic fibrosis (CF) is a lethal disorder characterized by abnormal epithelial ion transport; this disorder is characterized by an ongoing airway remodeling and neutrophilic inflammation.… (more)

Subjects/Keywords: Bodily Secretions  – enzymology <; br>; Cystic Fibrosis <; br>; Extracellular Matrix Proteins  – metabolism <; br>; Francisella tularensis  – physiology <; br>; Inflammation  – metabolism <; br>; Matrix Metalloproteinase 9  – metabolism <; br>; Neutrophils  – metabolism <; br>; Oligopeptides  – metabolism <; br>; Pneumonia, Bacterial  – enzymology <; br>; Proline  – analogs & derivatives <; br>; Respiratory System <; br>; Serine Endopeptidases  – metabolism <; br>; Tularemia  – microbiology

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APA (6th Edition):

Gaggar, A. (2007). Protease dysregulation: role in neutrophilic inflammation in cystic fibrosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,364

Chicago Manual of Style (16th Edition):

Gaggar, Amit. “Protease dysregulation: role in neutrophilic inflammation in cystic fibrosis.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,364.

MLA Handbook (7th Edition):

Gaggar, Amit. “Protease dysregulation: role in neutrophilic inflammation in cystic fibrosis.” 2007. Web. 16 Sep 2019.

Vancouver:

Gaggar A. Protease dysregulation: role in neutrophilic inflammation in cystic fibrosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,364.

Council of Science Editors:

Gaggar A. Protease dysregulation: role in neutrophilic inflammation in cystic fibrosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,364

24. Bennett, Allison E. Characterization of sortase and its effect on the virulence of Streptococcus pneumoniae.

Degree: PhD, 2008, University of Alabama – Birmingham

Sortases are transpeptidases that covalently link surface proteins to the stempeptide on the cell wall peptidoglycan of Gram-positive acteria. This work endeavored to characterize the… (more)

Subjects/Keywords: Amino Acid Sequence <; br>; Binding Sites <; br>; Hyaluronic Acid  – metabolism <; br>; Nasopharynx  – microbiology <; br>; Streptococcus pneumoniae  – enzymology <; br>; Streptococcus pneumoniae  – genetics <; br>; Streptococcus pneumoniae  – pathogenicity <; br>; Virulence

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APA (6th Edition):

Bennett, A. E. (2008). Characterization of sortase and its effect on the virulence of Streptococcus pneumoniae. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,191

Chicago Manual of Style (16th Edition):

Bennett, Allison E. “Characterization of sortase and its effect on the virulence of Streptococcus pneumoniae.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,191.

MLA Handbook (7th Edition):

Bennett, Allison E. “Characterization of sortase and its effect on the virulence of Streptococcus pneumoniae.” 2008. Web. 16 Sep 2019.

Vancouver:

Bennett AE. Characterization of sortase and its effect on the virulence of Streptococcus pneumoniae. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,191.

Council of Science Editors:

Bennett AE. Characterization of sortase and its effect on the virulence of Streptococcus pneumoniae. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,191

25. Shonsey, Erin Mary. Structural and functional studies of human bile acid CoA:amino acid N-acyltransferase (hBAT).

Degree: PhD, 2008, University of Alabama – Birmingham

The liver is involved in a variety of biological functions including the synthesis and biliary secretion of bile acids (BAs). These biomolecules are essential in… (more)

Subjects/Keywords: Acyltransferases  – antagonists & inhibitors<; br>; Aldehydes  – chemistry<; br>; Bile Acids and Salts  – metabolism<; br>; Chemistry Techniques, Analytical  – methods<; br>; Coenzyme A Ligases<; br>; Glycine – chemistry<; br>; Liver  – enzymology<; br>; Microsomes, Liver  – enzymology<; br>; Molecular Structure<; br>; Recombinant Proteins<; br>; Spectrometry, Mass, Electrospray Ionization<; br>

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APA (6th Edition):

Shonsey, E. M. (2008). Structural and functional studies of human bile acid CoA:amino acid N-acyltransferase (hBAT). (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,830

Chicago Manual of Style (16th Edition):

Shonsey, Erin Mary. “Structural and functional studies of human bile acid CoA:amino acid N-acyltransferase (hBAT).” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,830.

MLA Handbook (7th Edition):

Shonsey, Erin Mary. “Structural and functional studies of human bile acid CoA:amino acid N-acyltransferase (hBAT).” 2008. Web. 16 Sep 2019.

Vancouver:

Shonsey EM. Structural and functional studies of human bile acid CoA:amino acid N-acyltransferase (hBAT). [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,830.

Council of Science Editors:

Shonsey EM. Structural and functional studies of human bile acid CoA:amino acid N-acyltransferase (hBAT). [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,830


Michigan State University

26. Alkhayat, Aisha Hassan. Human [beta]-mannosidase : cDNA characterization and identification of mutations associated with [beta]-mannosidosis.

Degree: PhD, Department of Genetics, 1997, Michigan State University

Subjects/Keywords: Mannosidases; Lysosomal storage diseases; Metabolism, Inborn errors of

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APA (6th Edition):

Alkhayat, A. H. (1997). Human [beta]-mannosidase : cDNA characterization and identification of mutations associated with [beta]-mannosidosis. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:26789

Chicago Manual of Style (16th Edition):

Alkhayat, Aisha Hassan. “Human [beta]-mannosidase : cDNA characterization and identification of mutations associated with [beta]-mannosidosis.” 1997. Doctoral Dissertation, Michigan State University. Accessed September 16, 2019. http://etd.lib.msu.edu/islandora/object/etd:26789.

MLA Handbook (7th Edition):

Alkhayat, Aisha Hassan. “Human [beta]-mannosidase : cDNA characterization and identification of mutations associated with [beta]-mannosidosis.” 1997. Web. 16 Sep 2019.

Vancouver:

Alkhayat AH. Human [beta]-mannosidase : cDNA characterization and identification of mutations associated with [beta]-mannosidosis. [Internet] [Doctoral dissertation]. Michigan State University; 1997. [cited 2019 Sep 16]. Available from: http://etd.lib.msu.edu/islandora/object/etd:26789.

Council of Science Editors:

Alkhayat AH. Human [beta]-mannosidase : cDNA characterization and identification of mutations associated with [beta]-mannosidosis. [Doctoral Dissertation]. Michigan State University; 1997. Available from: http://etd.lib.msu.edu/islandora/object/etd:26789

27. Mitchell, Natalie E. Genome-wide DNA methylation changes during breast tumorigenesis.

Degree: MS, 2012, University of Alabama – Birmingham

Neoplastic reprogramming generates cancerous cells from normal derivatives, enabling researchers to create a model of the early stages of oncogenesis and analyze molecular changes in… (more)

Subjects/Keywords: Breast – Cancer – Genetic aspects<; br>; DNA – Methylation<; br>; Carcinogenesis<; br>; Breast – Precancerous conditions – Genetic aspects

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APA (6th Edition):

Mitchell, N. E. (2012). Genome-wide DNA methylation changes during breast tumorigenesis. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1309

Chicago Manual of Style (16th Edition):

Mitchell, Natalie E. “Genome-wide DNA methylation changes during breast tumorigenesis.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1309.

MLA Handbook (7th Edition):

Mitchell, Natalie E. “Genome-wide DNA methylation changes during breast tumorigenesis.” 2012. Web. 16 Sep 2019.

Vancouver:

Mitchell NE. Genome-wide DNA methylation changes during breast tumorigenesis. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1309.

Council of Science Editors:

Mitchell NE. Genome-wide DNA methylation changes during breast tumorigenesis. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1309

28. D'Alessandro, Tracy Lyn. Metabolism of neutrophil-derived chlorinated isoflavones.

Degree: PhD, 2008, University of Alabama – Birmingham

Despite medical advances in both drug therapy and surgical techniques, Americans mostly die from heart disease. Many belong to the low- and middle-class families who… (more)

Subjects/Keywords: Antioxidants  – pharmacology<; br>; Chlorine  – metabolism<; br>; Isoflavones  – metabolism<; br>; Mass Spectrometry<; br>; Neutrophils  – enzymology<; br>; Nitrates  – metabolism<; br>; Peroxidase  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

D'Alessandro, T. L. (2008). Metabolism of neutrophil-derived chlorinated isoflavones. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,819

Chicago Manual of Style (16th Edition):

D'Alessandro, Tracy Lyn. “Metabolism of neutrophil-derived chlorinated isoflavones.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,819.

MLA Handbook (7th Edition):

D'Alessandro, Tracy Lyn. “Metabolism of neutrophil-derived chlorinated isoflavones.” 2008. Web. 16 Sep 2019.

Vancouver:

D'Alessandro TL. Metabolism of neutrophil-derived chlorinated isoflavones. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,819.

Council of Science Editors:

D'Alessandro TL. Metabolism of neutrophil-derived chlorinated isoflavones. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,819

29. Tower-Gilchrist, Cristy (Cristy Davette). The role of protein folding and protein trafficking in human disease.

Degree: PhD, 2011, University of Alabama – Birmingham

Project 1: Expansion of CAG repeats encoding glutamine in huntingtin and ataxin 3 causes the neurodegenerative diseases Huntington's disease (HD) and spinocerebellar ataxia 3 (SCA3),… (more)

Subjects/Keywords: Cilia<; br>; Cytomegalovirus  – enzymology<; br>; Huntington Disease  – virology<; br>; Neurons  – metabolism<; br>; Peptides  – antagonists & inhibitors<; br>; Phosphotransferases (Alcohol Group Acceptor)  – physiology<; br>; Spinocerebellar Ataxias  – virology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tower-Gilchrist, C. (. D. (2011). The role of protein folding and protein trafficking in human disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,964

Chicago Manual of Style (16th Edition):

Tower-Gilchrist, Cristy (Cristy Davette). “The role of protein folding and protein trafficking in human disease.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,964.

MLA Handbook (7th Edition):

Tower-Gilchrist, Cristy (Cristy Davette). “The role of protein folding and protein trafficking in human disease.” 2011. Web. 16 Sep 2019.

Vancouver:

Tower-Gilchrist C(D. The role of protein folding and protein trafficking in human disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,964.

Council of Science Editors:

Tower-Gilchrist C(D. The role of protein folding and protein trafficking in human disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,964

30. Kim, Junghyun. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.

Degree: PhD, 2010, University of Alabama – Birmingham

Heme oxygenase-1 (HO-1) is a critical enzyme catalyzing the degradation of heme and generating carbon monoxide, iron, and biliverdin. In addition to heme degradation, HO-1… (more)

Subjects/Keywords: Acute Kidney Injury<; br>; Chromatin  – chemistry<; br>; Gene Expression Regulation, Enzymologic<; br>; Heme Oxygenase-1  – biosynthesis<; br>; Kidney  – enzymology<; br>; Mice<; br>; Sp1 Transcription Factor  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, J. (2010). Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1100

Chicago Manual of Style (16th Edition):

Kim, Junghyun. “Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 16, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1100.

MLA Handbook (7th Edition):

Kim, Junghyun. “Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells.” 2010. Web. 16 Sep 2019.

Vancouver:

Kim J. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1100.

Council of Science Editors:

Kim J. Transcriptional regulation of the human heme oxygenase-1 via chromatin looping in renal cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1100

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